FN Thomson Reuters Web of Science™
VR 1.0
PT J
AU Myrbakk, E
Von Tetzchner, S
AF Myrbakk, Even
Von Tetzchner, Stephen
TI The Prevalence of Behavior Problems Among People with Intellectual
Disability Living in Community Settings
SO JOURNAL OF MENTAL HEALTH RESEARCH IN INTELLECTUAL DISABILITIES
LA English
DT Article
DE community settings; challenging behavior; intellectual disability;
behavior problems
AB With the desegregation processes of services for people with intellectual disability (ID) that is taking place in most Western countries there is a need for more knowledge related to the prevalence of behavior problems among people living in community settings. This study investigates the prevalence of behavior problems among 140 adolescents and adults with ID living in 5 municipalities in Norway using the Aberrant Behavior Checklist (Aman & Singh, 1986, 1994). Twenty percent of the sample were reported to show severe behavior disorder or "challenging behavior," and this gives a prevalence for challenging behavior of 63.6 people per 100,000 base population. On the average, people showing challenging behavior had 7 behaviors rated as severe problems. A total of 60 participants were reported to have moderate or severe behavior problems, giving a prevalence for moderate and severe behavior problems of 136.3 per 100,000 base population. There were more people with profound ID and more people with autism in the group with challenging behavior and more people with Down syndrome in the group with mild or no problems. Those with severe behavior problems were perceived to be significantly less satisfied with their life situation than people without severe behavior problems. Challenging behavior seems to have a significant negative impact on the quality of life of people with ID. The results of this and other studies of challenging behavior among individuals with ID who live in areas with different service structures, using base population information, are important for analyzing the impact of reforms and different service systems on the life quality and well-being of people with ID.
C1 [Myrbakk, Even] Nordland Hosp Trust, N-8038 Bodo, Norway.
[Von Tetzchner, Stephen] Univ Oslo, Dept Psychol, N-0316 Oslo, Norway.
RP Myrbakk, E (reprint author), Nordland Hosp Trust, Helse Nord RHF Northern Norway Reg Hlth Author, N-8038 Bodo, Norway.
EM even.myrbakk@nlsh.no
FU Northern Norwegian Research Centre for Psychiatry
FX The project was supported by a grant from the Northern Norwegian
Research Centre for Psychiatry.
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TC 8
Z9 8
PU ROUTLEDGE JOURNALS, TAYLOR & FRANCIS LTD
PI ABINGDON
PA 4 PARK SQUARE, MILTON PARK, ABINGDON OX14 4RN, OXFORDSHIRE, ENGLAND
SN 1931-5864
EI 1931-5872
J9 J MENT HEALTH RES IN
JI J. Ment. Health Res. Intellect. Disabil.
PD JUN 12
PY 2008
VL 1
IS 3
BP 205
EP 222
DI 10.1080/19315860802115607
PG 18
WC Education, Special; Psychiatry; Rehabilitation
SC Education & Educational Research; Psychiatry; Rehabilitation
GA V37ZY
UT WOS:000209314900006
ER
PT J
AU Hines, RM
Wu, LJ
Hines, DJ
Steenland, H
Mansour, S
Dahlhaus, R
Singaraja, RR
Cao, XY
Sammler, E
Hormuzdi, SG
Zhuo, M
El-Husseini, A
AF Hines, Rochelle M.
Wu, Longjun
Hines, Dustin J.
Steenland, Hendrik
Mansour, Souraya
Dahlhaus, Regina
Singaraja, Roshni R.
Cao, Xiaoyan
Sammler, Esther
Hormuzdi, Sheriar G.
Zhuo, Min
El-Husseini, Alaa
TI Synaptic imbalance, stereotypies, and impaired social interactions in
mice with altered neuroligin 2 expression
SO JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE neuroligin; transgenic; synapse; excitatory/inhibitory ratio;
neurodevelopmental disorder; autism
ID AUTISM-SPECTRUM DISORDERS; RETT-SYNDROME; INHIBITORY SYNAPSES;
CELL-ADHESION; BETA-NEUREXINS; FRONTAL-CORTEX; LOBE EPILEPSY; MOUSE;
BEHAVIOR; AMYGDALA
AB The level of excitation in the brain is kept under control through inhibitory signals mainly exerted by GABA neurons. However, the molecular machinery that regulates the balance between excitation and inhibition (E/I) remains unclear. Candidate molecules implicated in this process are neuroligin (NL) adhesion molecules, which are differentially enriched at either excitatory or inhibitory contacts. In this study, we use transgenic mouse models expressing NL1 or NL2 to examine whether enhanced expression of specific NLs results in synaptic imbalance and altered neuronal excitability and animal behavior. Our analysis reveals several abnormalities selectively manifested in transgenic mice with enhanced expression of NL2 but not NL1. A small change in NL2 expression results in enlarged synaptic contact size and vesicle reserve pool in frontal cortex synapses and an overall reduction in the E/I ratio. The frequency of miniature inhibitory synaptic currents was also found to be increased in the frontal cortex of transgenic NL2 mice. These animals also manifested stereotyped jumping behavior, anxiety, impaired social interactions, and enhanced incidence of spike-wave discharges, as depicted by EEG analysis in freely moving animals. These findings may provide the neural basis for E/I imbalance and altered behavior associated with neurodevelopmental disorders.
C1 [Hines, Rochelle M.; Hines, Dustin J.; Mansour, Souraya; Dahlhaus, Regina; El-Husseini, Alaa] Univ British Columbia, Dept Psychiat, Brain Res Ctr, Fac Med, Vancouver, BC V6T 2B5, Canada.
[Wu, Longjun; Steenland, Hendrik; Cao, Xiaoyan; Zhuo, Min] Univ Toronto, Dept Physiol, Fac Med, Toronto, ON M5S 1A8, Canada.
[Singaraja, Roshni R.] Univ British Columbia, Dept Med Genet, Ctr Mol Med & Therapeut, Vancouver, BC V5Z 4H4, Canada.
[Sammler, Esther; Hormuzdi, Sheriar G.] Univ Dundee, Ninewells Hosp, Dept Neurol, Dundee DD1 9SY, Scotland.
[Sammler, Esther; Hormuzdi, Sheriar G.] Univ Dundee, Sch Med, Dundee DD1 9SY, Scotland.
RP Hines, RM (reprint author), Univ British Columbia, 2255 Wesbrook Mall, Vancouver, BC V6T 1Z3, Canada.
EM rbruneau@interchange.ubc.ca
RI Zhuo, Min/A-2072-2008; Wu, Long-Jun /E-2684-2012
OI Zhuo, Min/0000-0001-9062-3241;
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NR 84
TC 75
Z9 78
PU SOC NEUROSCIENCE
PI WASHINGTON
PA 11 DUPONT CIRCLE, NW, STE 500, WASHINGTON, DC 20036 USA
SN 0270-6474
J9 J NEUROSCI
JI J. Neurosci.
PD JUN 11
PY 2008
VL 28
IS 24
BP 6055
EP 6067
DI 10.1523/JNEUROSCI.0032-08.2008
PG 13
WC Neurosciences
SC Neurosciences & Neurology
GA 312JU
UT WOS:000256668500001
PM 18550748
ER
PT J
AU Martin, PM
Carnaud, M
del Cano, GG
Irondelle, M
Irinopoulou, T
Girault, JA
Dargent, B
Goutebroze, L
AF Martin, Pierre-Marie
Carnaud, Michele
Garcia del Cano, Gontzal
Irondelle, Marie
Irinopoulou, Theano
Girault, Jean-Antoine
Dargent, Benedicte
Goutebroze, Laurence
TI Schwannomin-interacting protein-1 isoform IQCJ-SCHIP-1 is a late
component of nodes of Ranvier and axon initial segments
SO JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE myelinated fibers; schwannomin/merlin; calmodulin; ankyrin G; beta
IV-spectrin; voltage-gated sodium channels; language disorder
ID BETA-IV-SPECTRIN; SODIUM-CHANNELS; ANKYRIN-G; MYELINATED AXONS;
NERVOUS-SYSTEM; IQ MOTIF; AUTISM; DOMAINS; NEUROFASCIN; ASSOCIATION
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C1 [Martin, Pierre-Marie; Carnaud, Michele; Irinopoulou, Theano; Girault, Jean-Antoine; Goutebroze, Laurence] INSERM, UMR S 839, F-75005 Paris, France.
[Martin, Pierre-Marie; Carnaud, Michele; Irinopoulou, Theano; Girault, Jean-Antoine; Goutebroze, Laurence] Univ Paris 06, F-75005 Paris, France.
[Martin, Pierre-Marie; Carnaud, Michele; Irinopoulou, Theano; Girault, Jean-Antoine; Goutebroze, Laurence] Inst Fer Moulin, F-75005 Paris, France.
[Garcia del Cano, Gontzal; Irondelle, Marie; Dargent, Benedicte] INSERM, U641, F-13916 Marseille, France.
[Garcia del Cano, Gontzal; Irondelle, Marie; Dargent, Benedicte] Univ Aix Marseille 2, Fac Med Secteur Nord, Inst Fed Rech 11, F-13916 Marseille, France.
[Garcia del Cano, Gontzal] Univ Basque Country, Fac Pharm, Dept Neurosci, Vitoria 01006, Araba, Spain.
RP Goutebroze, L (reprint author), INSERM, UMR S 839, F-75005 Paris, France.
EM girault@fer-a-moulin.inserm.fr; goutebro@fer-a-moulin.inserm.fr
RI Garcia del Cano, Gontzal/H-4710-2012; Girault, Jean-Antoine/F-7518-2013
OI Garcia del Cano, Gontzal/0000-0002-7098-0986; Girault,
Jean-Antoine/0000-0002-7900-1705
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NR 43
TC 14
Z9 16
PU SOC NEUROSCIENCE
PI WASHINGTON
PA 11 DUPONT CIRCLE, NW, STE 500, WASHINGTON, DC 20036 USA
SN 0270-6474
J9 J NEUROSCI
JI J. Neurosci.
PD JUN 11
PY 2008
VL 28
IS 24
BP 6111
EP 6117
DI 10.1523/JNEUROSCI.1044-08.2008
PG 7
WC Neurosciences
SC Neurosciences & Neurology
GA 312JU
UT WOS:000256668500006
PM 18550753
ER
PT J
AU Ke, XY
Hong, SS
Tang, TY
Zou, B
Li, HG
Hang, YY
Zhou, ZY
Ruan, ZC
Lu, ZH
Tao, GT
Liu, YJ
AF Ke, Xiaoyan
Hong, Shanshan
Tang, Tianyu
Zou, Bing
Li, Huiguo
Hang, Yueyue
Zhou, Zhenyu
Ruan, Zongcai
Lu, Zuhong
Tao, Guotai
Liu, Yijun
TI Voxel-based morphometry study on brain structure in children with
high-functioning autism
SO NEUROREPORT
LA English
DT Review
DE brain structure; children; high-functioning autism; magnetic resonance
imaging; morphometry
ID SPECTRUM DISORDER; ADOLESCENTS; MRI; INDIVIDUALS; ANATOMY
AB Earlier studies have suggested abnormal brain volumes in autism, but inconsistencies exist. Using voxel-based morphometry, we compared global and regional brain volumes in 17 high-functioning autistic children with 15 matched controls. We identified significant reduction in left white matter volume and white/gray matter ratio in autism. Regional brain volume reductions were detected for right anterior cingulate, left superior parietal lobule white matter volumes, and right parahippocampal gyrus gray matter volume, whereas enlargements in bilateral supramarginal gyrus, right postcentral gyrus, right medial frontal gyrus, and right posterior lobe of cerebellum gray matter in autism. Our findings showed global and regional brain volumes abnormality in high-functioning autism.
C1 [Ke, Xiaoyan; Zou, Bing; Li, Huiguo; Hang, Yueyue; Tao, Guotai] Nanjing Med Univ, Nanjing Brain Hosp, Child Mental Hlth Res Ctr, Nanjing 210029, Peoples R China.
[Ke, Xiaoyan; Hong, Shanshan; Tang, Tianyu; Zhou, Zhenyu; Ruan, Zongcai; Lu, Zuhong] Southeast Univ, Key Lab Child Dev & Learning Sci, Minist Educ, Nanjing, Peoples R China.
[Liu, Yijun] Univ Florida, McKnight Brain Inst, Dept Psychiat & Neurosci, Gainesville, FL 32611 USA.
RP Ke, XY (reprint author), Nanjing Med Univ, Nanjing Brain Hosp, Child Mental Hlth Res Ctr, Nanjing GuangZhou Rd 264, Nanjing 210029, Peoples R China.
EM kexynj@hotmail.com
RI Lu, Zuhong/A-5448-2013
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NR 25
TC 36
Z9 37
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0959-4965
J9 NEUROREPORT
JI Neuroreport
PD JUN 11
PY 2008
VL 19
IS 9
BP 921
EP 925
DI 10.1097/WNR.0b013e328300edf3
PG 5
WC Neurosciences
SC Neurosciences & Neurology
GA 313LJ
UT WOS:000256741800005
PM 18520994
ER
PT J
AU Kawabe, K
Miyamoto, E
AF Kawabe, Kouichi
Miyamoto, Eri
TI Effects of neonatal repeated MK-801 treatment on delayed non
matching-to-position responses in rats
SO NEUROREPORT
LA English
DT Article
DE delayed non matching-to-position task; N-methyl-D-aspartate receptors;
neonatal repeated treatment; psychomimetic responses; rats; working
memory
ID D-ASPARTATE RECEPTOR; WORKING-MEMORY; MAZE PERFORMANCE; ADULT-RAT;
SCHIZOPHRENIA; HIPPOCAMPAL; BLOCKADE; ANTAGONIST; AUTISM; BRAIN
AB This study was carried out to investigate the long-term effects of chronic neonatal antagonism of N-methyl-D-aspartate (NMDA) receptors, a subtype of glutamate receptors, on working memory. Rats were tested on the delayed nonmatching-to-position task in adulthood after repeated treatment of a noncompetitive NMDA antagonist MK-801 in postnatal days 7-20. As a result, this treatment led to deficits in learning and/or performance of delayed non matching-to-position responses, suggesting that chronic neo-natal NMDA antagonism persistently impairs working memory. Furthermore, it decreased body and brain weight, and induced stereotyped head-rotation behavior. As working memory deficits are shown in several mental disorders such as schizophrenia and developmental disorders, rats with chronic neonatal NMDA antagonism might be useful for a better understanding of these disorders.
C1 [Kawabe, Kouichi; Miyamoto, Eri] Osaka City Univ, Grad Sch Literature & Human Sci, Sumiyoshi Ku, Osaka 5588585, Japan.
RP Kawabe, K (reprint author), Osaka City Univ, Grad Sch Literature & Human Sci, Sumiyoshi Ku, Osaka 5588585, Japan.
EM kawabe@lit.osaka-cu.ac.jp
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NR 25
TC 13
Z9 14
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0959-4965
J9 NEUROREPORT
JI Neuroreport
PD JUN 11
PY 2008
VL 19
IS 9
BP 969
EP 973
DI 10.1097/WNR.0b013e328302ee31
PG 5
WC Neurosciences
SC Neurosciences & Neurology
GA 313LJ
UT WOS:000256741800014
PM 18521003
ER
PT J
AU Roohi, J
Tegay, DH
Pomeroy, JC
Burkett, S
Stone, G
Stanyon, R
Hatchwell, E
AF Roohi, Jasmin
Tegay, David H.
Pomeroy, John C.
Burkett, Sandra
Stone, Gary
Stanyon, Roscoe
Hatchwell, Eli
TI A de novo apparently balanced translocation [46AY,t(2;9)(p13;p24)]
interrupting RAB11FIP5 identifies a potential candidate gene for autism
spectrum disorder
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE autism; translocation, genetic; RAB11 family interacting protein 5
(class I), human; doublestranded DNA break; type I DNA topoisomerase
ID RAB11-INTERACTING PROTEINS; RECYCLING ENDOSOMES; TRAFFICKING; DNA;
SEQUENCES; LINKAGE; MOTIFS; FAMILY
AB Autism spectrum disorder (ASD) is a severe developmental disorder of the central nervous system characterized by impairments in social interaction, communication, and range of interests and behaviors. The syndrome's prevalence is estimated to be as high as 1 in 150 American children yet its etiology remains largely unknown. Examination of observed cytogenetic variants in individuals with ASD may identify genes involved in its pathogenesis. As part of a multidisciplinary study, an apparently balanced de novo translocation between chromosomes 2 and 9 [46,X-Y,t(2;9)(p13;p24)] was identified in a subject with pervasive developmental disorder not otherwise specified (PDD-NOS), and no distinctive dysmorphic features. Molecular characterization of the rearrangement revealed direct interruption of the RAB11 family interacting protein 5 (RAB11FIP5) gene. RAB11FIP5 is a Rab effector involved in protein trafficking from apical recycling endosomes to the apical plasma membrane. It is ubiquitously expressed and reported to contribute to both neurotransmitter release and neurotransmitter uptake at the synaptic junction. Detailed analysis of the rearrangement breakpoints suggests that the reciprocal translocation may have formed secondary to incorrect repair of double strand breaks (DSBs) by nonhomologous end-joining (NHEJ). (c) 2008 Wiley-Liss, Inc.
C1 [Hatchwell, Eli] SUNY Stony Brook, Dept Pathol, Stony Brook, NY 11794 USA.
[Roohi, Jasmin; Hatchwell, Eli] SUNY Stony Brook, Dept Genet, Stony Brook, NY 11794 USA.
[Tegay, David H.; Pomeroy, John C.] SUNY Stony Brook, Dept Pediat, Med Ctr, Stony Brook, NY 11794 USA.
[Tegay, David H.] New York Coll Osteopath Med, Dept Med & Med Genet, Old Westbury, NY USA.
[Burkett, Sandra; Stone, Gary; Stanyon, Roscoe] NCI, Frederick, MD 21701 USA.
[Stanyon, Roscoe] Univ Florence, Dept Anim Biol & Genet, Florence, Italy.
RP Hatchwell, E (reprint author), SUNY Stony Brook, Dept Pathol, BST-9, Stony Brook, NY 11794 USA.
EM eli.hatchwell@stonybrook.edu
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NR 34
TC 6
Z9 6
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD JUN 5
PY 2008
VL 147B
IS 4
BP 411
EP 417
DI 10.1002/ajmg.b.30755
PG 7
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 307PO
UT WOS:000256331000001
PM 18384058
ER
PT J
AU Losh, M
Childress, D
Lam, K
Piven, J
AF Losh, Molly
Childress, Debra
Lam, Kristen
Piven, Joseph
TI Defining key features of the broad autism phenotype: A comparison across
parents of multiple- and single-incidence autism families
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE autism; genetic; broad autism phenotype
ID NEO PERSONALITY-INVENTORY; PSYCHIATRIC GENETICS; GENERAL-POPULATION;
CHILDREN; SIBLINGS; TRAITS; TWIN; INDIVIDUALS; HISTORY; SCHIZOPHRENIA
AB This study examined the frequency of personality, language, and social-behavioral characteristics believed to comprise the broad autism phenotype (BAP), across families differing in genetic liability to autism. We hypothesized that within this unique sample comprised of multiple-incidence autism families (MIAF), single-incidence autism families (SIAF), and control Down syndrome families (DWNS), a graded expression would be observed for the principal characteristics conferring genetic susceptibility to autism, in which such features would express most profoundly among parents from MIAFs, less strongly among SIAFs, and least of all among comparison parents from DWNS families, who should display population base rates. Analyses detected linear expression of traits in line with hypotheses, and further suggested differential intrafamilial expression across family types. In the vast majority of MIAFs both parents displayed BAP characteristics, whereas within SIAFs, it was equally likely that one, both, or neither parent show BAP features. The significance of these findings is discussed in relation to etiologic mechanisms in autism and relevance to molecular genetic studies. (c) 2007 Wiley-Liss, Inc.
C1 [Losh, Molly; Childress, Debra; Lam, Kristen; Piven, Joseph] Univ N Carolina, Neurodev Disorders Res Ctr, Chapel Hill, NC 27599 USA.
RP Losh, M (reprint author), Univ N Carolina, Neurodev Disorders Res Ctr, CB 3366, Chapel Hill, NC 27599 USA.
EM losh@med.unc.edu
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NR 62
TC 78
Z9 79
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD JUN 5
PY 2008
VL 147B
IS 4
BP 424
EP 433
DI 10.1002/ajmg.b.30612
PG 10
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 307PO
UT WOS:000256331000003
PM 17948871
ER
PT J
AU Wang, LF
Jia, MX
Yue, WH
Tang, FL
Qu, M
Ruan, Y
Lu, TL
Zhang, H
Yan, H
Liu, J
Guo, YQ
Zhang, J
Yang, XL
Zhang, D
AF Wang, Lifang
Jia, Meixiang
Yue, Weihua
Tang, Fulei
Qu, Mei
Ruan, Yan
Lu, Tianlan
Zhang, Handi
Yan, Hao
Liu, Jing
Guo, Yanqing
Zhang, Jishui
Yang, Xiaoling
Zhang, Dai
TI Association of the ENGRAILED 2 (EN2) gene with autism in Chinese Han
population
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE single nucleotide polymorphism (SNP); family-based association test
(FBAT); haplotype
ID HOMEOBOX-TRANSCRIPTION-FACTOR; INFANTILE-AUTISM; SPECTRUM-DISORDER;
CEREBELLAR DEVELOPMENT; SUSCEPTIBILITY LOCUS; GENOMEWIDE SCREEN; CELL
LOSS; HYPOPLASIA; BRAIN; BEHAVIOR
AB Human ENGRAILED 2 (EN2) gene is localized to 7q36, an autism susceptibility locus. En2 knockout mice display hypoplasia of cerebellum and a decrease in the number of Purkinje cell, which are similar to those reported for individuals with autism. Furthermore, deficits in social behavior were detected in En2(-/-) mice. Two recent studies have demonstrated that two intronic SNPs (rs1861972, rs1861973) in the EN2 gene are significantly associated with autism. To investigate whether this finding could be replicated in Chinese Han population, we performed the association study between eight single nueleotide polymorphisms (SNPs) of the EN2 gene and autism in 210 Chinese Han trios, using the family-based association test (FBAT). The present study demonstrated that a preferential transmission of the rs3824068 A-allele to affected offspring (A > G: Z = 2.399, P = 0.0165). After the Bonferroni correction, this statistical significance of preferential transmission did not remain. However, when haplotypes were constructed with multiple markers, a number of haplotypes including three two-marker haplotypes, nine three-marker haplotypes, one four-marker haplotype, and one six-marker haplotype, all of which contain the major allele A of rs3824068, displayed significantly associated with autism. These results were still significant after using the permutation method to obtain empirical P values. Thus, our data provide evidence that the EN2 gene may be implicated in the predisposition to autism in the Chinese Han population. (c) 2007 Wiley-Liss, Inc.
C1 [Wang, Lifang; Jia, Meixiang; Yue, Weihua; Tang, Fulei; Qu, Mei; Ruan, Yan; Lu, Tianlan; Zhang, Handi; Yan, Hao; Liu, Jing; Guo, Yanqing; Yang, Xiaoling; Zhang, Dai] Peking Univ, Inst Natl Hlth, Beijing 100083, Peoples R China.
[Wang, Lifang; Jia, Meixiang; Yue, Weihua; Tang, Fulei; Qu, Mei; Ruan, Yan; Lu, Tianlan; Zhang, Handi; Yan, Hao; Liu, Jing; Guo, Yanqing; Yang, Xiaoling; Zhang, Dai] Minist Hlth, Key Lab Mental Hlth, Beijing, Peoples R China.
[Zhang, Jishui] Capital Univ Med Sci, Beijing Childrens Hosp, Beijing, Peoples R China.
RP Yang, XL (reprint author), Peking Univ, Inst Natl Hlth, 51,Hua Yuan Bei Rd, Beijing 100083, Peoples R China.
EM yangxl@public.fhnet.cn.net
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NR 38
TC 28
Z9 29
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD JUN 5
PY 2008
VL 147B
IS 4
BP 434
EP 438
DI 10.1002/ajmg.b.30623
PG 5
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 307PO
UT WOS:000256331000004
PM 17948901
ER
PT J
AU Wermter, AK
Kamp-Becker, I
Strauch, K
Schulte-Koerne, G
Remschmidt, H
AF Wermter, Anne-Kathrin
Kamp-Becker, Inge
Strauch, Konstantin
Schulte-Koerne, Gerd
Remschmidt, Helmut
TI No evidence for involvement of genetic variants in the X-linked
neuroligin genes NLGN3 and NLGN4X in probands with autism spectrum
disorder on high functioning level
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE neuroligin 3; neuroligin 4; autism spectrum disorder; Asperger syndrome;
X-chromosome
ID PERVASIVE DEVELOPMENTAL DISORDERS; MUTATIONS
AB Several lines of evidence indicate a role of mutations in the two X-linked genes neuroligin 3 (NLGN3) and neuroligin 4 (NLGN4X) in the etiology of autistic spectrum disorders. To analyze whether genetic variants in the NLGN3 and NLGN4X genes occurs in patients with autistic disorders on high functioning level, we performed a mutation screen of both genes using SSCP in 107 probands with Asperger syndrome, high-functioning autism and atypical autism. We identified four polymorphisms (rs2290488, rs7049300, rs3747333, rs3747334) and one novel synonymous variant (A558) in the NLGN4X. The polymorphisms rs7049300, rs3747333, and rs3747334 did not cause any amino acid substitutions in the total of the eight detected carriers. A family-based association study for rs2290488 in 101 trios did not reveal association of this polymorphism with autistic disorders on high functioning level. We conclude that there is no evidence for an involvement of NLGN3 and NLGN4X genetic variants with autism spectrum disorder on high functioning level in our study group. (C) 2008 Wiley-Liss, Inc.
C1 [Wermter, Anne-Kathrin] Univ Marburg, Dept Child & Adolescent Psychiat & Psychotherapy, Clin Res Grp, D-35033 Marburg, Germany.
[Strauch, Konstantin] Univ Marburg, Inst Med Biometry & Epidemiol, D-35033 Marburg, Germany.
RP Wermter, AK (reprint author), Univ Marburg, Dept Child & Adolescent Psychiat & Psychotherapy, Clin Res Grp, Schutzenstr 49, D-35033 Marburg, Germany.
EM Anne-Kathrin.Wermter@med.uni-marburg.de
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NR 23
TC 22
Z9 24
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD JUN 5
PY 2008
VL 147B
IS 4
BP 535
EP 537
DI 10.1002/ajmg.b.30618
PG 3
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 307PO
UT WOS:000256331000023
PM 18189281
ER
PT J
AU Gonzalez, JR
Carrasco, JL
Armengol, L
Villatoro, S
Jover, L
Yasui, Y
Estivill, X
AF Gonzalez, Juan R.
Carrasco, Josep L.
Armengol, Lluis
Villatoro, Sergi
Jover, Lluis
Yasui, Yutaka
Estivill, Xavier
TI Probe-specific mixed-model approach to detect copy number differences
using multiplex ligation-dependent probe amplification (MLPA)
SO BMC BIOINFORMATICS
LA English
DT Article
ID SHORT FLUORESCENT FRAGMENTS; HUMAN GENOME; SEGMENTAL DUPLICATIONS;
STRUCTURAL VARIATION; MENTAL-RETARDATION; EXON DELETIONS; LOCUS;
REARRANGEMENTS; HYBRIDIZATION; GENES
AB Background: MLPA method is a potentially useful semi-quantitative method to detect copy number alterations in targeted regions. In this paper, we propose a method for the normalization procedure based on a non-linear mixed-model, as well as a new approach for determining the statistical significance of altered probes based on linear mixed-model. This method establishes a threshold by using different tolerance intervals that accommodates the specific random error variability observed in each test sample.
Results: Through simulation studies we have shown that our proposed method outperforms two existing methods that are based on simple threshold rules or iterative regression. We have illustrated the method using a controlled MLPA assay in which targeted regions are variable in copy number in individuals suffering from different disorders such as Prader-Willi, DiGeorge or Autism showing the best performace.
Conclusion: Using the proposed mixed-model, we are able to determine thresholds to decide whether a region is altered. These threholds are specific for each individual, incorporating experimental variability, resulting in improved sensitivity and specificity as the examples with real data have revealed.
C1 [Gonzalez, Juan R.] Ctr Res Environm Epidemiol CREAL, Barcelona, Spain.
[Gonzalez, Juan R.; Carrasco, Josep L.; Jover, Lluis] Univ Barcelona, Biostat Unit, Dept Publ Hlth, E-08007 Barcelona, Spain.
[Armengol, Lluis; Villatoro, Sergi; Estivill, Xavier] CRG, Genes & Dis Program, Barcelona, Spain.
[Yasui, Yutaka] Univ Alberta, Dept Publ Hlth Sci, Sch Publ Hlth, Edmonton, AB T6G 2M7, Canada.
RP Gonzalez, JR (reprint author), Ctr Res Environm Epidemiol CREAL, Barcelona, Spain.
EM jrgonzalez@imim.es; jlcarrasco@ub.edu; lluis.armengol@crg.es;
sergi.villatoro@crg.es; lluis_jover@ub.edu; yyasui@ualberta.ca;
xavier.estivill@crg.es
RI Estivill, Xavier/E-2957-2012; Jover, Lluis/D-2192-2011; Carrasco,
Josep/D-3660-2011; Estivill, Xavier/A-3125-2013; Yasui,
Yutaka/E-2564-2015
OI Jover, Lluis/0000-0003-0631-1398; Carrasco, Josep/0000-0003-1184-0753;
Estivill, Xavier/0000-0002-0723-2256; Yasui, Yutaka/0000-0002-7717-8638
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NR 26
TC 11
Z9 11
PU BIOMED CENTRAL LTD
PI LONDON
PA 236 GRAYS INN RD, FLOOR 6, LONDON WC1X 8HL, ENGLAND
SN 1471-2105
J9 BMC BIOINFORMATICS
JI BMC Bioinformatics
PD JUN 4
PY 2008
VL 9
AR 261
DI 10.1186/1471-2105-9-261
PG 15
WC Biochemical Research Methods; Biotechnology & Applied Microbiology;
Mathematical & Computational Biology
SC Biochemistry & Molecular Biology; Biotechnology & Applied Microbiology;
Mathematical & Computational Biology
GA 333YD
UT WOS:000258188600001
PM 18522760
ER
PT J
AU Tanimura, Y
Yang, MC
Lewis, MH
AF Tanimura, Yoko
Yang, Mark C.
Lewis, Mark H.
TI Procedural learning and cognitive flexibility in a mouse model of
restricted, repetitive behaviour
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Article
DE stereotypy; autism; environmental enrichment; cortico-basal ganglia
circuitry
ID ENVIRONMENTAL ENRICHMENT; STEREOTYPED BEHAVIOR; MEMORY-SYSTEMS;
PREFRONTAL CORTEX; CAUDATE-NUCLEUS; SPATIAL MEMORY; DEVELOPMENTAL
PSYCHOPATHOLOGY; PEROMYSCUS-MANICULATUS; SYNAPTOPHYSIN LEVELS; DOUBLE
DISSOCIATION
AB Restricted, repetitive behaviours (e.g., stereotypies, compulsions, rituals) in neurodevelopmental disorders have been linked to alterations in cortico-basal ganglia circuitry. Cognitive processes mediated by this circuitry (e.g., procedural learning, executive function) are likely to be impaired in individuals exhibiting high rates of repetitive behaviour. To test this hypothesis, we assessed both procedural learning and cognitive flexibility (reversal learning) using a T-maze task in deer mice (Peromyscus maniculatus) exhibiting various rates of repetitive behaviour (vertical jumping and backward somersaulting). These mice exhibited high rates of stereotypy when reared in standard rodent cages, and such behaviour was significantly attenuated by housing them in larger more complex environments. Mice reared in complex environments exhibited significantly better procedural and reversal learning than standard caged mice. Thus, early experience associated with the prevention and attenuation of stereotypy was associated with better striatally mediated learning and cognitive flexibility. Stereotypy score was significantly correlated with the number of errors made in reversal learning, and interacted with housing condition to affect overall cognitive performance. Our findings support the applicability of the deer mouse model of spontaneous stereotypy to a wider range of restricted, repetitive behaviour (e.g., insistence on sameness) typical of neurodevelopmental disorders. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Tanimura, Yoko; Lewis, Mark H.] Univ Florida, Dept Psychiat, McKnight Brain Inst, Gainesville, FL 32611 USA.
[Tanimura, Yoko; Lewis, Mark H.] Univ Florida, Dept Psychol, McKnight Brain Inst, Gainesville, FL 32611 USA.
[Yang, Mark C.] Univ Florida, Dept Stat, Gainesville, FL 32611 USA.
RP Lewis, MH (reprint author), 100 S Newell Dr, Gainesville, FL 32610 USA.
EM marklewis@ufl.edu
RI Tanimura, Yoko/D-3141-2014
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NR 55
TC 33
Z9 33
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD JUN 3
PY 2008
VL 189
IS 2
BP 250
EP 256
DI 10.1016/j.bbr.2008.01.001
PG 7
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 297GE
UT WOS:000255604300003
PM 18272239
ER
PT J
AU Whitehouse, CR
Boullata, J
McCauley, LA
AF Whitehouse, Christina R.
Boullata, Joseph
McCauley, Linda A.
TI The Potential Toxicity of Artificial Sweeteners
SO AAOHN JOURNAL
LA English
DT Article
AB Since their discovery, the safety of artificial sweeteners has been controversial. Artificial sweeteners provide the sweetness of sugar without the calories. As public health attention has turned to reversing the obesity epidemic in the United States, more individuals of all ages are choosing to use these products. These choices may be beneficial for those who cannot tolerate sugar in their diets (e.g., diabetics). However, scientists disagree about the relationships between sweeteners and lymphomas, leukemias, cancers of the bladder and brain, chronic fatigue syndrome, Parkinson's disease, Alzheimer's disease, multiple sclerosis, autism, and systemic lupus. Recently these substances have received increased attention due to their effects on glucose regulation. Occupational health nurses need accurate and timely information to counsel individuals regarding the use of these substances. This article provides an overview of types of artificial sweeteners, sweetener history, chemical structure, biological fate, physiological effects, published animal and human studies, and current standards and regulations.
C1 [Whitehouse, Christina R.; Boullata, Joseph; McCauley, Linda A.] Univ Penn, Sch Nursing, Philadelphia, PA 19104 USA.
RP Whitehouse, CR (reprint author), Univ Penn, Sch Nursing, Philadelphia, PA 19104 USA.
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World Health Organization, 2004, 1 WHO, V1, P10
NR 51
TC 22
Z9 22
PU SLACK INC
PI THOROFARE
PA 6900 GROVE RD, THOROFARE, NJ 08086 USA
SN 0891-0162
J9 AAOHN J
JI AAOHN J.
PD JUN
PY 2008
VL 56
IS 6
BP 251
EP 259
PG 9
WC Public, Environmental & Occupational Health; Nursing
SC Public, Environmental & Occupational Health; Nursing
GA V15BU
UT WOS:000207778300004
PM 18604921
ER
PT J
AU Segui, JD
Ortiz-Tallo, M
De Diego, Y
AF David Segui, Jose
Ortiz-Tallo, Margarita
De Diego, Yolanda
TI Stress related factors in caregivers of children with autism: Overload,
psychopathology and health
SO ANALES DE PSICOLOGIA
LA Spanish
DT Article
DE children's caregivers; autism; overload; psychopathology; health
ID MENTAL-RETARDATION; FAMILY STRESS; SPECTRUM DISORDERS; SOCIAL SUPPORT;
MOTHERS; INDIVIDUALS; DEPRESSION; SERVICES; PARENTS; VERSION
AB The objective of this study was to evaluate if caregivers were overloaded when their children were diagnosed with autism disorder, how it may influence their mental and physical health. 40 caregivers have been participating in this study, mainly mothers. The parameters studied include sociodemographic factors, caregivers burden overload (Zarit Scale, adapted to the Spanish language), Psychopathology (SCL-90) and health status (SF-60).
The results indicate that caregivers were overloaded, and in a worse state of mental and physical health compared to the general population. A strong positive correlation was observed between overwhelmed carers and the evaluated health and pathopsicological parameters. These results arc in accordance with previous findings that were published by other groups, supporting the idea that specific health programs are needed for caregivers of children with chronic diseases.
C1 [Ortiz-Tallo, Margarita] Univ Malaga, Fac Psicol, E-29071 Malaga, Spain.
Hosp Reg Univ Carlos Haya, Malaga, Spain.
RP Ortiz-Tallo, M (reprint author), Univ Malaga, Fac Psicol, Campus Teatinos, E-29071 Malaga, Spain.
EM mortiztallo@uma.es
RI Ortiz-Tallo Alarcon, Margarita/G-8807-2011; de Diego,
yolanda/I-6070-2012
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NR 31
TC 7
Z9 11
PU UNIV MURCIA
PI MURCIA
PA SERVICIO DE PUBLICACIONES, CALLE VISTALEGRE S/N, MURCIA, 30007, SPAIN
SN 0212-9728
J9 AN PSICOL
JI An. Psicol.
PD JUN
PY 2008
VL 24
IS 1
BP 100
EP 105
PG 6
WC Psychology; Psychology, Multidisciplinary
SC Psychology
GA 326LM
UT WOS:000257660600012
ER
PT J
AU Caronna, EB
Milunsky, JM
Tager-Flusberg, H
AF Caronna, E. B.
Milunsky, J. M.
Tager-Flusberg, H.
TI Autism spectrum disorders: clinical and research frontiers
SO ARCHIVES OF DISEASE IN CHILDHOOD
LA English
DT Review
ID CHROMOSOMAL REARRANGEMENTS; HEAD CIRCUMFERENCE; ASPERGER-SYNDROME;
AMYGDALA VOLUME; GAZE-FIXATION; CHILDREN; BRAIN; PREVALENCE; DIAGNOSIS;
EPIDEMIOLOGY
AB Autism spectrum disorders (ASD) are common neurodevelopmental disorders that occur along a broad continuum of severity with impairments in social interactions, communication and behaviour. This review highlights recent advances in autism research that shed light on the causes of the disorder and that have implications for clinical practice. It focuses on (1) the rising prevalence of ASD with attention given to recent epidemiological studies, (2) important genetic discoveries that may affect clinical evaluation of children with ASD, (3) active areas of research in cognitive neuroscience that seek to explain the underlying mechanisms of a complex disorder and (4) important studies on clinical populations with implications for screening and early identification of infants and toddlers with ASD.
C1 [Caronna, E. B.] Boston Univ, Sch Med, Boston Med Ctr, Dept Pediat,Div Dev & Behav Pediat, Boston, MA 02118 USA.
[Milunsky, J. M.] Boston Univ, Sch Med, Ctr Human Genet, Dept Pediat & Genet, Boston, MA 02118 USA.
[Milunsky, J. M.] Boston Univ, Sch Med, Ctr Human Genet, Dept Genom, Boston, MA 02118 USA.
[Tager-Flusberg, H.] Boston Univ, Sch Med, Dept Anat & Neurobiol, Boston, MA 02118 USA.
[Tager-Flusberg, H.] Boston Univ, Sch Med, Dept Pediat, Boston, MA 02118 USA.
RP Caronna, EB (reprint author), Boston Univ, Sch Med, Boston Med Ctr, Dept Pediat,Div Dev & Behav Pediat, 88 E Newton St,Vose Hall 4, Boston, MA 02118 USA.
EM Elizabeth.Caronna@bmc.org
RI Tager-Flusberg, Helen/D-5265-2009
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NR 79
TC 38
Z9 39
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0003-9888
J9 ARCH DIS CHILD
JI Arch. Dis. Child.
PD JUN
PY 2008
VL 93
IS 6
BP 518
EP 523
DI 10.1136/adc.2006.115337
PG 6
WC Pediatrics
SC Pediatrics
GA 303TY
UT WOS:000256065000015
PM 18305076
ER
PT J
AU Gerkensmeyer, JE
Perkins, SM
Scott, EL
Wu, JW
AF Gerkensmeyer, Janis E.
Perkins, Susan M.
Scott, Eric L.
Wu, Jingwei
TI Depressive symptoms among primary caregivers of children with mental
health needs: Mediating and moderating variables
SO ARCHIVES OF PSYCHIATRIC NURSING
LA English
DT Article
ID MATERNAL DEPRESSION; SOCIAL SUPPORT; PARENTAL DEPRESSION; PRIMARY-CARE;
STRESS; FAMILIES; AUTISM; DISORDERS; BEHAVIOR; MOTHERS
AB The prevalence and severity of depressive symptoms among 155 primary caregivers of children with mental health problems were examined along with variables that mediated or moderated the association of child behavior problems with caregivers' depressive symptoms. Forty percent of participants scored 22 or higher on the Center for Epidemiological Studies Depression Scale, indicating that they might have a serious depression. Perceived personal control, subjective distress, and role disruption mediated the association between internalizing child behavior problems and depressive symptoms. Tangible social support moderated the association between internalizing behavior problems and depressive symptoms. Intangible social support moderated the association between externalizing behavior problems and depressive symptoms. (C) 2008 Elsevier Inc. All rights reserved.
C1 [Gerkensmeyer, Janis E.] Indiana Univ, Sch Nursing, Indianapolis, IN 46202 USA.
Indiana Univ, Sch Med, Div Biostat, Indianapolis, IN 46202 USA.
Indiana Univ, Sch Med, Riley Hosp, Child Psychiat Clin, Indianapolis, IN 46202 USA.
RP Gerkensmeyer, JE (reprint author), Indiana Univ, Sch Nursing, 1111 Middle Dr, Indianapolis, IN 46202 USA.
EM jgerkens@iupui.edu
RI Vollrath, Margarete/G-1297-2011
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NR 65
TC 10
Z9 11
PU W B SAUNDERS CO-ELSEVIER INC
PI PHILADELPHIA
PA 1600 JOHN F KENNEDY BOULEVARD, STE 1800, PHILADELPHIA, PA 19103-2899 USA
SN 0883-9417
J9 ARCH PSYCHIAT NURS
JI Arch. Psychiatr. Nurs.
PD JUN
PY 2008
VL 22
IS 3
BP 135
EP 146
DI 10.1016/j.apnu.2007.06.016
PG 12
WC Nursing; Psychiatry
SC Nursing; Psychiatry
GA 315GK
UT WOS:000256866200004
PM 18505695
ER
PT J
AU Bailey, AJ
Karr, V
AF Bailey, Anthony J.
Karr, Valerie
TI Autism as a Global Challenge
SO AUTISM RESEARCH
LA English
DT Editorial Material
C1 [Bailey, Anthony J.; Karr, Valerie] Columbia Univ, Coll Teachers, New York, NY 10027 USA.
RP Bailey, AJ (reprint author), Columbia Univ, Coll Teachers, New York, NY 10027 USA.
RI Bailey, Anthony/J-2860-2014
OI Bailey, Anthony/0000-0003-4257-972X
NR 0
TC 0
Z9 0
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD JUN
PY 2008
VL 1
IS 3
BP 145
EP 146
DI 10.1002/aur.28
PG 2
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CJ
UT WOS:000270031700001
PM 19360661
ER
PT J
AU Chadman, KK
Gong, SC
Scattoni, ML
Boltuck, SE
Gandhy, SU
Heintz, N
Crawley, JN
AF Chadman, Kathryn K.
Gong, Shiaoching
Scattoni, Maria L.
Boltuck, Sarah E.
Gandhy, Shruti U.
Heintz, Nathaniel
Crawley, Jacqueline N.
TI Minimal Aberrant Behavioral Phenotypes of Neuroligin-3 R451C Knockin
Mice
SO AUTISM RESEARCH
LA English
DT Article
DE autism; mouse model; neuroligin; behavior phenotyping; social behavior
ID AUTISM SPECTRUM DISORDER; SOCIAL APPROACH BEHAVIORS; BTBR-T+TF/J MICE;
INBRED STRAINS; GENES NLGN3; MUTATIONS; SYNAPSE; PROTEIN; PROBANDS;
REVEALS
AB Neuroligin-3 is a member of the class of cell adhesion proteins that mediate synapse development and have been implicated in autism. Mice with the human R451C mutation (NL3), identical to the point mutation found in two brothers with autism spectrum disorders, were generated and phenotyped in multiple behavioral assays with face validity to the diagnostic symptoms of autism. No differences between NL3 and their wildtype (WT) littermate controls were detected on measures of juvenile reciprocal social interaction, adult social approach, cognitive abilities, and resistance to change in a spatial habit, findings which were replicated in several cohorts of males and females. physical and procedural abilities were similar across genotypes on measures of general health, sensory abilities, sensorimotor gating, motor functions, and anxiety-related traits. Minor developmental differences were detected between NL3 and WT, including slightly different rates of somatic growth, slower righting reflexes at postnatal days 2-6, faster homing reflexes in females, and less vocalizations on postnatal day 8 in males. Significant differences in NI,3 adults included somewhat longer latencies to fall from the rotarod, less vertical activity in the open field, and less acoustic startle to high decibel tones. The humanized R451C mutation in mice did not result in apparent autism-like phenotypes, but produced detectable functional consequences that may be interpreted in terms of physical development and/or reduced sensitivity to stimuli.
C1 [Chadman, Kathryn K.] NIMH, Lab Behav Neurosci, IRP, NIH, Bethesda, MD 20892 USA.
[Gong, Shiaoching; Heintz, Nathaniel] Howard Hughes Med Inst, Mol Biol Lab, Chevy Chase, MD USA.
[Gong, Shiaoching; Heintz, Nathaniel] Rockefeller Univ, GENSAT Project, New York, NY 10021 USA.
[Scattoni, Maria L.] Ist Super Sanita, Behav Neurosci Sect, I-00161 Rome, Italy.
RP Chadman, KK (reprint author), NIMH, Lab Behav Neurosci, IRP, NIH, Bldg 35 Room 1C-909,Mail Stop 3730, Bethesda, MD 20892 USA.
EM kathryn.chadman@gmail.com
FU National Institute of Mental Health Intramural Research Program, Simons
Foundation [ISS-NIH 0F14]
FX Grant sponsors: National Institute of Mental Health Intramural Research
Program, Simons Foundation, ISS-NIH 0F14
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NR 44
TC 107
Z9 108
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD JUN
PY 2008
VL 1
IS 3
BP 147
EP 158
DI 10.1002/aur.22
PG 12
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CJ
UT WOS:000270031700002
PM 19360662
ER
PT J
AU Campbell, DB
Li, C
Sutcliffe, JS
Persico, AM
Levitt, P
AF Campbell, Daniel B.
Li, Chun
Sutcliffe, James S.
Persico, Antonio M.
Levitt, Pat
TI Genetic Evidence Implicating Multiple Genes in the MET Receptor Tyrosine
Kinase Pathway in Autism Spectrum Disorder
SO AUTISM RESEARCH
LA English
DT Article
DE genetic; association; plasminogen; PLAUR; uPAR; SERPINE1; brain;
cerebral cortex
ID INTERNEURON DEVELOPMENT; GASTROINTESTINAL SYMPTOMS; ASSOCIATION;
DISRUPTION; TELENCEPHALON; MUTATIONS; INTEGRINS; GENOTYPE; CHILDREN;
DEFICITS
AB A functional promoter variant of the gene encoding the MET receptor tyrosine kinase alters SP1 and SUB1 transcription factor binding, and is associated with autism spectrum disorder (ASD). Recent analyses of postmortem cerebral cortex from ASD patients revealed altered expression of MET protein and three transcripts encoding proteins that regulate MET signaling, hepatocyte growth factor (HGF), urokinase plasminogen activator receptor (PLAUR) and plasminogen activator inhibitor-1 (SERPINE1). To address potential risk conferred by multiple genes in the MET signaling pathway, we screened all exons and 5' promoter regions for variants in the five genes encoding proteins that regulate MET expression and activity Identified variants were genotyped in 664 families (2,712 individuals including 1,228 with ASD) and 312 unrelated controls. Replicating our initial findings, family-based association test (FBAT) analyses demonstrated that the MET promoter variant rs1858830 C allele was associated with ASD in 101 new families (P = 0.033). Two other genes in the MET signaling pathway also may confer risk. A haplotype of the SERPINE1 gene exhibited significant association. In addition, the PLAUR promoter variant rs344781 T allele was associated with ASD by both FBAT (P = 0.006) and case-control analyses (P = 0.007). The PLAUR promoter rs344781 relative risk was 1.93 (95% confidence interval [CI]: 1.12-3.31) for genotype TT and 2.42 (95% CI: 1.38-4.25) for genotype CT compared to genotype CC. Gene-gene interaction analyses suggested a significant interaction between MET and PLAUR. These data further support our hypothesis that genetic susceptibility impacting multiple components of the MET signaling pathway contributes to ASD risk.
C1 [Campbell, Daniel B.; Levitt, Pat] Vanderbilt Univ, Dept Pharmacol, Nashville, TN 37232 USA.
[Campbell, Daniel B.; Sutcliffe, James S.; Levitt, Pat] Vanderbilt Univ, Vanderbilt Kennedy Ctr Res Human Dev, Nashville, TN 37232 USA.
[Li, Chun] Vanderbilt Univ, Dept Biostat, Nashville, TN 37232 USA.
[Sutcliffe, James S.] Vanderbilt Univ, Dept Mol Physiol & Biophys, Nashville, TN 37232 USA.
[Persico, Antonio M.] Univ Campus Biomed, Lab Mol Psychiat & Neurogenet, Rome, Italy.
[Persico, Antonio M.] IRCCS Fdn S Lucia, Rome, Italy.
RP Campbell, DB (reprint author), Vanderbilt Univ, Dept Pharmacol, 8114 MRB3,465 21st Ave S, Nashville, TN 37232 USA.
EM daniel.campbell@vanderbilt.edu
RI Li, Chun/B-8388-2012; Sutcliffe, James/C-1348-2012
OI Sutcliffe, James/0000-0001-5200-6007
FU NIMH [MH064547, MH055135, MH065299]; NICHD [HD015052]; The Marino Autism
Research Institute; Cure Autism Now; National Alliance for Autism
Research; Fondation Jerome Lejeune; NIH [MH061009, NS049261]
FX Grant sponsor: NIMH; Grant numbers: MH064547, MH055135, MH065299; Grant
sponsor: NICHD; Grant number: HD015052; Grant sponsors: The Marino
Autism Research Institute, Cure Autism Now, National Alliance for Autism
Research, Fondation Jerome Lejeune; Grant sponsor: NIH; Grant numbers:
MH061009, NS049261.
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NR 31
TC 71
Z9 73
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD JUN
PY 2008
VL 1
IS 3
BP 159
EP 168
DI 10.1002/aur.27
PG 10
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CJ
UT WOS:000270031700003
PM 19360663
ER
PT J
AU Nagarajan, RP
Patzel, KA
Martin, M
Yasui, DH
Swanberg, SE
Hertz-Picciotto, I
Hansen, RL
Van de Water, J
Pessah, IN
Jiang, R
Robinson, WP
LaSalle, JM
AF Nagarajan, Raman P.
Patzel, Katherine A.
Martin, Michelle
Yasui, Dag H.
Swanberg, Susan E.
Hertz-Picciotto, Irva
Hansen, Robin L.
Van de Water, Judy
Pessah, Isaac N.
Jiang, Ruby
Robinson, Wendy P.
LaSalle, Janine M.
TI MECP2 Promoter Methylation and X Chromosome Inactivation in Autism
SO AUTISM RESEARCH
LA English
DT Article
DE epigenetics; X chromosome inactivation; MECP2; postmortem brain
ID RETT-SYNDROME; SPECTRUM DISORDERS; CTCF; EXPRESSION; BOUNDARIES;
SEQUENCES; DOMAINS; BRAIN; GENE
AB Epigenetic mechanisms have been proposed to play a role in the etiology of autism. This hypothesis is supported by the discovery of increased MECP2 promoter methylation associated with decreased MeCP2 protein expression in autism male brain. To further understand the influence of female X chromosome inactivation (XCI) and neighboring methylation patterns on aberrant MECP2 promoter methylation in autism, multiple methylation analyses were performed on brain and blood samples from individuals with autism. Bisulfite sequencing analyses of a region 0.6 kb upstream of MECP2 in brain DNA samples revealed an abrupt transition from a highly methylated region in both sexes to a region unmethylated in males and subject to XCI in females. Chromatin immunoprecipitation analysis demonstrated that the CCCTC-binding factor (CTCF) is bound to this transition region in neuronal cells, consistent with a chromatin boundary at the methylation transition. Male autism brain DNA samples displayed a slight increase in methylation in this transition region, suggesting a possible aberrant spreading of methylation into the MECP2 promoter in autism males across this boundary element. In addition, autistic female brain DNA samples showed evidence for aberrant MECP2 promoter methylation as ail increase in the number of bisulfite sequenced clones with undefined XCI status for MECP2 but not androgen receptor (AR). To further investigate the specificity of MECP2 methylation alterations in autism, blood DNA samples from females and mothers of males with autism were also examined for XCI skewing at AR, but no significant increase in XCI skewing was observed compared to controls. These results suggest that the aberrant MECP2 methylation in autism brain DNA samples is due to locus-specific rather than global X chromosome methylation changes.
C1 [Nagarajan, Raman P.; Patzel, Katherine A.; Martin, Michelle; Yasui, Dag H.; Swanberg, Susan E.; LaSalle, Janine M.] Univ Calif Davis, Med Microbiol & Immunol & Rowe Program Human Gene, Davis, CA 95616 USA.
[Hertz-Picciotto, Irva] Univ Calif Davis, Dept Publ Hlth Sci, Davis, CA 95616 USA.
[Hertz-Picciotto, Irva] Univ Calif Davis, Div Epidemiol, Davis, CA 95616 USA.
[Hertz-Picciotto, Irva] Univ Calif Davis, Div Environm, Davis, CA 95616 USA.
[Hertz-Picciotto, Irva] Univ Calif Davis, Div Occupat Hlth, Davis, CA 95616 USA.
[Hertz-Picciotto, Irva; Hansen, Robin L.; Van de Water, Judy; Pessah, Isaac N.] Univ Calif Davis, Med Inst Neurodev Disorders, Davis, CA 95616 USA.
[Hansen, Robin L.] Univ Calif Davis, Dept Pediat, Davis, CA 95616 USA.
[Van de Water, Judy] Univ Calif Davis, Div Rheumatol Allergy & Clin Immunol, Davis, CA 95616 USA.
[Pessah, Isaac N.] Univ Calif Davis, Dept Vet Mol Biosci, Davis, CA 95616 USA.
[Jiang, Ruby; Robinson, Wendy P.] Univ British Columbia, Dept Med Genet, Child & Family Res Inst, Vancouver, BC, Canada.
RP LaSalle, JM (reprint author), Univ Calif Davis, Med Microbiol & Immunol & Rowe Program Human Gene, 1 Shields Ave, Davis, CA 95616 USA.
EM jmlasalle@ucdavis.edu
RI LaSalle, Janine/A-4643-2008; Robinson, Wendy/I-9590-2014
OI LaSalle, Janine/0000-0002-3480-2031;
FU NIH [1R01HD048799, R01ES015171]
FX Grant sponsors: NIH; Grant numbers: 1R01HD048799, R01ES015171.
CR Amir RE, 1999, NAT GENET, V23, P185
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Lewis A, 2004, CURR BIOL, V14, pR284, DOI 10.1016/j.cub.2004.03.026
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LYON MF, 1961, NATURE, V190, P372, DOI 10.1038/190372a0
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Thatcher KN, 2006, EPIGENETICS-US, V1, P24
NR 27
TC 37
Z9 39
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD JUN
PY 2008
VL 1
IS 3
BP 169
EP 178
DI 10.1002/aur.24
PG 10
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CJ
UT WOS:000270031700004
PM 19132145
ER
PT J
AU Loth, E
Gomez, JC
Happe, F
AF Loth, Eva
Gomez, Juan Carlos
Happe, Francesca
TI Detecting Changes in Naturalistic Scenes: Contextual Inconsistency Does
not Influence Spontaneous Attention in High-Functioning People With
Autism Spectrum Disorder
SO AUTISM RESEARCH
LA English
DT Article
ID ASPERGER-SYNDROME; CENTRAL COHERENCE; VISUAL-ATTENTION; SOCIAL-STIMULI;
EYE-MOVEMENTS; PICTURES; CHILDREN; OBJECTS; KNOWLEDGE; SALIENCY
AB Individuals with autism spectrum disorders (ASD) are often reported to be good at detecting minute changes in the environment. This study tested two factors in this phenomenon; detail-focus and reduced top-down influence of scene-schema expectations on spontaneous attention to visual scene elements. Using a change blindness paradigm, adults with ASD and matched typically developing (TD) adults were presented with images of naturalistic scenes (e.g., living room). Scene changes involved three types of object substitution: an object was replaced with (i) all unexpected scene-unrelated object, (ii) a scene-related object of a different basic-level category, (iii) or a different exemplar of the original object category. Top-down effects of scene-schema expectations should render scene-unrelated (i) substitutions easiest to recognize; detail focus should increase detection of exemplar changes. The TD group showed the expected condition effects, detecting scene-unrelated substitutions significantly better than both types of scene-related changes. By contrast, the ASD group showed no condition effect, and was only significantly slower and less accurate than the TD group in detecting scene-unrelated objects. These findings suggest reduced influence of schematic expectations on spontaneous attention in individuals with ASD. Together with other factors, this may contribute to the tendency to notice "irrelevant" changes in the environment.
C1 [Loth, Eva] Univ Cambridge, Dept Expt Psychol, Behav & Clin Neurosci Inst, Cambridge CB2 3EB, England.
[Gomez, Juan Carlos] Univ St Andrews, Sch Psychol, St Andrews, Fife, Scotland.
[Happe, Francesca] Kings Coll London, MRC, Social Genet & Dev Psychiat Ctr, Inst Psychiat, London WC2R 2LS, England.
RP Loth, E (reprint author), Univ Cambridge, Dept Expt Psychol, Behav & Clin Neurosci Inst, Cambridge CB2 3EB, England.
EM el266@cam.ac.uk
RI Happe, Francesca/D-5544-2012; Loth, Eric/C-5805-2008
FU Economic and Social Research Council (ESRC) [RES-000-22-1123]; Nuffield
Foundation
FX Grant sponsors: Economic and Social Research Council
(ESRC)RES-000-22-1123, Nuffield Foundation
CR ANTES JR, 1974, J EXP PSYCHOL, V103, P62, DOI 10.1037/h0036799
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LOTH E, PERSON KNOWLED UNPUB
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NR 36
TC 17
Z9 18
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD JUN
PY 2008
VL 1
IS 3
BP 179
EP 188
DI 10.1002/aur.19
PG 10
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CJ
UT WOS:000270031700005
PM 19360664
ER
PT J
AU Turunen, JA
Rehnstrom, K
Kilpinen, H
Kuokkanen, M
Kempas, E
Ylisaukko-oja, T
AF Turunen, Joni A.
Rehnstrom, Karola
Kilpinen, Helena
Kuokkanen, Mikko
Kempas, Elli
Ylisaukko-oja, Tero
TI Mitochondrial Aspartate/Glutamate Carrier SLC25A12 Gene Is Associated
With Autism
SO AUTISM RESEARCH
LA English
DT Article
DE autism; Asperger syndrome; SLC25A12; association; SNP
ID LINKAGE DISEQUILIBRIUM; ASPERGER-SYNDROME; DISORDER
AB Two single nucleotide polymorphisms (SNP) within Mitochondrial Aspartate/Glutamate Carrier SLC25A12 gene have recently shown to be strongly associated with autism. Here, we attempted to replicate this finding in two separate Finnish samples with autism spectrum disorders. Family-based association analysis of two SNPs, rs2056202 and rs2292813, previously shown to be associated with autism was performed in two samples with different phenotypic characteristics. The samples included 97 families with strictly defined autism and 29 extended families with Asperger syndrome (AS). We detected association at rs2292813 (FBAT, P=0.0018) in the Finnish autism sample. In, addition other family-based analysis methods supported this finding. By contrast, analysis of the AS sample yielded no evidence for association. This study shows further Support that genetic variants within SLC25A12 gene contribute to the etiology of autism.
C1 [Turunen, Joni A.] Natl Publ Hlth Inst, Dept Mol Med, Biomedicum, Helsinki 00290, Finland.
[Turunen, Joni A.; Rehnstrom, Karola; Kilpinen, Helena; Kuokkanen, Mikko; Kempas, Elli; Ylisaukko-oja, Tero] Natl Publ Hlth Inst, FIMM, Helsinki 00290, Finland.
[Rehnstrom, Karola; Kuokkanen, Mikko; Ylisaukko-oja, Tero] Univ Helsinki, Dept Med Genet, FIN-00014 Helsinki, Finland.
RP Turunen, JA (reprint author), Natl Publ Hlth Inst, Dept Mol Med, Biomedicum, Haartmaninkatu 8, Helsinki 00290, Finland.
EM joni.turunen@helsinki.fi
FU Paivikki and Sakari Sohlberg Foundation; Center of Excellence of Disease
Genetics of the Academy of Finland; Finnish Cultural Foundation; Cure
Autism Now
FX Grant sponsors: Paivikki and Sakari Sohlberg Foundation, Center of
Excellence of Disease Genetics of the Academy of Finland, Finnish
Cultural Foundation, Cure Autism Now.
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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Ylisaukko-oja T, 2004, MOL PSYCHIATR, V9, P161, DOI 10.1038/sj.mp.4001385
NR 17
TC 16
Z9 17
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD JUN
PY 2008
VL 1
IS 3
BP 189
EP 192
DI 10.1002/aur.25
PG 4
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CJ
UT WOS:000270031700006
PM 19360665
ER
PT J
AU Mazefsky, CA
Folstein, SE
Lainhart, JE
AF Mazefsky, Carla A.
Folstein, Susan E.
Lainhart, Janet E.
TI Overrepresentation of Mood and Anxiety Disorders in Adults With Autism
and Their First-Degree Relatives: What Does it Mean?
SO AUTISM RESEARCH
LA English
DT Article
DE familial aggregation; affective disorders; family history; autism;
co-morbidity
ID PSYCHIATRIC-DISORDERS; ASPERGER-SYNDROME; CHILDREN; DEPRESSION;
PSYCHOPATHOLOGY; INDIVIDUALS; INTERVIEW; PARENTS; FAMILY
AB Research indicates that relatives of individuals with autism have higher rates of affective disorders than both the general population and families of children with other developmental disabilities. In addition, individuals with autism have high rates of co-morbid mood and anxiety disorders. This study sought to identify possible reasons for these previous findings by documenting the presence of affective disorders in both probands (the individuals with autism) and their family members. A sub-sample of 17 adults with autism and their first-degree relatives from the Baltimore Family Study of Autism completed a structured psychiatric interview. The results indicated that the rates of mood and anxiety disorders were 35 and 77%, respectively, for probands, and these disorders were present in at least one first-degree relative at rates of 71 and 29%, respectively. Exploring patterns within families revealed that 80% of probands with a mother who had a mood disorder history also had a mood disorder themselves, compared to only 16% of probands whose mothers did not have a mood disorder history. The results must be considered preliminary given the small sample size. Replicating these findings in a larger sample would help clarify whether a true increased risk of mood disorder exists, which would have potential implications for prevention efforts and understanding possible genetic mechanisms.
C1 [Mazefsky, Carla A.] Univ Pittsburgh, Dept Pediat, Pittsburgh, PA 15260 USA.
[Mazefsky, Carla A.] Univ Pittsburgh, Dept Psychiat, Pittsburgh, PA USA.
[Folstein, Susan E.] Johns Hopkins Univ, Dept Psychiat & Behav Sci, Baltimore, MD USA.
[Lainhart, Janet E.] Univ Utah, Dept Psychiat, Salt Lake City, UT USA.
RP Mazefsky, CA (reprint author), Childrens Hosp Pittsburgh, Child Dev Unit, 3705 5th Ave, Pittsburgh, PA 15213 USA.
EM Carla.Mazefsky@chp.edu
FU [NIMHR01]; [MI-139936-041101]; [HD35476-02]
FX Grant sponsors: NIMHR01 MI-139936-041101 HD35476-02
CR Bolton PF, 1998, PSYCHOL MED, V28, P385, DOI 10.1017/S0033291797006004
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NR 19
TC 20
Z9 20
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD JUN
PY 2008
VL 1
IS 3
BP 193
EP 197
DI 10.1002/aur.23
PG 5
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CJ
UT WOS:000270031700007
PM 19360666
ER
PT J
AU Jennett, HK
Hagopian, LP
AF Jennett, Heather K.
Hagopian, Louis P.
TI Identifying empirically supported treatments for phobic avoidance in
individuals with intellectual disabilities
SO BEHAVIOR THERAPY
LA English
DT Article
ID SHAPING APPROACH RESPONSES; MENTALLY-RETARDED ADULTS; DSM-IV DISORDERS;
CHILDREN; DESENSITIZATION; AUTISM; RETARDATION; REINFORCEMENT;
PREVALENCE; BEHAVIOR
AB This paper reviews the literature regarding the treatment of phobic avoidance in individuals with intellectual disabilities. Criteria for classifying interventions as empirically supported, developed by the American Psychological Association (APA) Division 12 Task Force on Promotion and Dissemination of Psychological Procedures, were used. For studies employing single case experimental designs, criteria developed by APA Division 16 (Kratochwill & Stoiber, 2002; Shernoff, Kratochwill, & Stoiber, 2002) were used to supplement Division 12 criteria. Results indicate that behavioral treatment can be designated as a well-established treatment for phobic avoidance in individuals with intellectual disabilities.
C1 [Jennett, Heather K.] Kennedy Krieger Inst, Dept Behav Psychol, Baltimore, MD 21205 USA.
[Jennett, Heather K.; Hagopian, Louis P.] Johns Hopkins Sch Med, Baltimore, MD 21218 USA.
RP Jennett, HK (reprint author), Kennedy Krieger Inst, Dept Behav Psychol, 707 N Broadway, Baltimore, MD 21205 USA.
EM jennett@kennedykrieger.org
CR Altabet SC, 2002, J DEV PHYS DISABIL, V14, P297, DOI 10.1023/A:1016032623478
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NR 33
TC 14
Z9 14
PU ASSOC ADV BEHAVIOR THERAPY
PI NEW YORK
PA 305 7TH AVE #16A, NEW YORK, NY 10001-6008 USA
SN 0005-7894
J9 BEHAV THER
JI Behav. Therapy
PD JUN
PY 2008
VL 39
IS 2
BP 151
EP 161
DI 10.1016/j.beth.2007.06.003
PG 11
WC Psychology, Clinical
SC Psychology
GA 323BN
UT WOS:000257419100005
PM 18502248
ER
PT J
AU Crespi, B
Badcock, C
AF Crespi, Bernard
Badcock, Christopher
TI Psychosis and autism as diametrical disorders of the social brain
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Review
DE autism; cognition; genomic conflict; genomic imprinting;
hyper-mentalism; psychosis; schizophrenia
ID PRADER-WILLI-SYNDROME; HIGH-FUNCTIONING AUTISM; WHITE-MATTER VOLUME;
OBSESSIVE-COMPULSIVE DISORDER; EARLY-ONSET SCHIZOPHRENIA; MIRROR NEURON
SYSTEM; PERVASIVE DEVELOPMENTAL DISORDER; SCHIZOTYPAL
PERSONALITY-DISORDER; LANGUAGE-ASSOCIATION CORTEX; SUPERIOR TEMPORAL
GYRUS
AB Autistic-spectrum conditions and psychotic-spectrum conditions (mainly schizophrenia, bipolar disorder, and major depression) represent two major suites of disorders of human cognition, affect, and behavior that involve altered development and function of the social brain. We describe evidence that a large set of phenotypic traits exhibit diametrically opposite phenotypes in autistic-spectrum versus psychotic-spectrum conditions, with a focus on schizophrenia. This suite of traits is inter-correlated, in that autism involves a general pattern of constrained overgrowth, whereas schizophrenia involves undergrowth. These disorders also exhibit diametric patterns for traits related to social brain development, including aspects of gaze, agency, social cognition, local versus global processing, language, and behavior. Social cognition is thus underdeveloped in autistic-spectrum conditions and hyper-developed on the psychotic spectrum.
We propose and evaluate a novel hypothesis that may help to explain these diametric phenotypes: that the development of these two sets of conditions is mediated in part by alterations of genomic imprinting. Evidence regarding the genetic, physiological, neurological, and psychological underpinnings of psychotic-spectrum conditions supports the hypothesis that the etiologies of these conditions involve biases towards increased relative effects from imprinted genes with maternal expression, which engender a general pattern of undergrowth. By contrast, autistic-spectrum conditions appear to involve increased relative bias towards effects of paternally expressed genes, which mediate overgrowth. This hypothesis provides a simple yet comprehensive theory, grounded in evolutionary biology and genetics, for understanding the causes and phenotypes of autistic-spectrum and psychotic-spectrum conditions.
C1 [Crespi, Bernard] Simon Fraser Univ, Dept Biosci, Burnaby, BC V5A 1S6, Canada.
[Badcock, Christopher] London Sch Econ, Dept Sociol, London WC2A 2AE, England.
RP Crespi, B (reprint author), Simon Fraser Univ, Dept Biosci, Burnaby, BC V5A 1S6, Canada.
EM crespi@sfu.ca; C.Badcock@lse.ac.uk
FU NSERC; Canada Council for the Arts
FX Bernard Crespi is grateful to NSERC and the Canada Council for the Arts
for financial support. Both authors thank Paul Bloom, Martin Brune, Will
Davies, Clyde Francks, David Haig, Randy Jirtle, Randy Nesse, Daniel
Nettle, Sophie van Rijn, and Alfonso Troisi for helpful comments and
discussions.
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NR 700
TC 145
Z9 148
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
EI 1469-1825
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 241
EP +
DI 10.1017/S0140525X08004214
PG 46
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200001
PM 18578904
ER
PT J
AU Abu-Akel, A
AF Abu-Akel, Ahmad
TI Theory of mind in autism, schizophrenia, and in-between
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
AB Autism and schizophrenia are presented as the extremes of disorders affecting the social brain. By viewing human cognition impairment in terms of competence and performance, a variety of social brain disorders can be identified along the autistic-psychotic continuum.
RP Abu-Akel, A (reprint author), 8015 Airlane Ave, Los Angeles, CA 90045 USA.
EM abuakel@hotmail.com
CR Abu-Akel A, 2003, BRAIN RES REV, V43, P29, DOI 10.1016/S0165-0173(03)00190-5
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NR 4
TC 5
Z9 5
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 261
EP +
DI 10.1017/S0140525X08004226
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200002
ER
PT J
AU Behrendt, RP
AF Behrendt, Ralf-Peter
TI Mapping autism and schizophrenia onto the ontogenesis of social
behaviour: A hierarchical-developmental rather than diametrical
perspective
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID SPECTRUM DISORDER; CHILDREN; PSYCHOPATHOLOGY; HALLUCINATIONS;
ABNORMALITIES; RECOGNITION; EXPRESSIONS; ATTENTION; OXYTOCIN
AB Co-morbidity of schizophrenia and autism is low because interpersonal concerns of schizophrenic patients presuppose developmental achievements that are absent in autism. Autism may arise if primary anxiety is not overcome at a key developmental stage by affective synchronisation between infant and caregiver. Schizophrenic patients will have learned to regulate primitive anxiety by affectively attuning to narrow social networks but remain highly vulnerable to exclusion from larger groups.
C1 [Behrendt, Ralf-Peter] Retreat Hosp, York YO10 5BN, N Yorkshire, England.
RP Behrendt, RP (reprint author), Retreat Hosp, York YO10 5BN, N Yorkshire, England.
EM rp.behrendt@btinternet.com
CR Adolphs R, 2000, J NEUROSCI, V20, P2683
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Behrendt RP, 2006, BEHAV BRAIN SCI, V29, P226
Behrendt RP, 2006, J PSYCHOPHARMACOL, V20, P356, DOI 10.1177/0269881105057696
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NR 39
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
EI 1469-1825
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 262
EP +
DI 10.1017/S0140525X08004238
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200003
ER
PT J
AU Belmonte, MK
AF Belmonte, Matthew K.
TI The "mechanism" of human cognitive variation
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID AUTISM; SCHIZOPHRENIA; DISORDER; HIPPOCAMPUS; ATTENTION; SPECTRUM;
DEFICITS; AMYGDALA; VOLUMES; ADULTS
AB The theory of psychosis and autism as diametrical disorders offers a tractable and testable view of normal and abnormal human cognitive variation as a function of opposing traits grouped by their selection for maternal and paternal reproductive fitness. The theory could be usefully rooted and developed with reference to the lower-level perceptual and attentional phenomena from which social cognitive modules are developmentally refined.
C1 [Belmonte, Matthew K.] Cornell Univ, Dept Human Dev, Ithaca, NY 14853 USA.
RP Belmonte, MK (reprint author), Cornell Univ, Dept Human Dev, Ithaca, NY 14853 USA.
EM belmonte@mit.edu
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NR 21
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 263
EP +
DI 10.1017/S0140525X0800424X
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200004
ER
PT J
AU Burne, THJ
Eyles, DW
McGrath, JJ
AF Burne, Thomas H. J.
Eyles, Darryl W.
McGrath, John J.
TI Animal models may help fractionate shared and discrete pathways
underpinning schizophrenia and autism
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID ADVANCING PATERNAL AGE; GENOMIC REARRANGEMENTS; DYNAMIC MUTATIONS;
BRAIN-DEVELOPMENT; DISORDERS; INFECTION; RISK; RELEVANT; SYMPTOMS;
DAMAGE
AB Crespi & Badcock (C&B) present all appealing and parsimonious synthesis arguing that schizophrenia and autism are differentially regulated by maternal versus paternal genomic imprinting, respectively. We argue that animal models related to schizophrenia and autism provide a useful platform to explore the mechanisms outlined by C&B. We also note that schizophrenia and autism share certain risk factors such as advanced paternal age. Apart from genomic imprinting, copy number variants related to advanced paternal age may also contribute to the differential trajectory of brain development associated with autism and schizophrenia.
C1 [Burne, Thomas H. J.; Eyles, Darryl W.; McGrath, John J.] Univ Queensland, Queensland Ctr Mental Hlth Res, Queensland Brain Inst, Brisbane, Qld 4072, Australia.
RP Burne, THJ (reprint author), Univ Queensland, Queensland Ctr Mental Hlth Res, Queensland Brain Inst, Brisbane, Qld 4072, Australia.
EM t.burne@uq.edu.au; eyles@uq.edu.au; john_mcgrath@qcmhr.uq.edu.au
RI Burne, Thomas/C-5656-2009; McGrath, John/G-5493-2010
OI Burne, Thomas/0000-0003-3502-9789; McGrath, John/0000-0002-4792-6068
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NR 29
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 264
EP +
DI 10.1017/S0140525X08004251
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200005
ER
PT J
AU Deutsch, CK
Ludwig, WW
McIlvane, WJ
AF Deutsch, Curtis K.
Ludwig, Wesley W.
McIlvane, William J.
TI Heterogeneity and hypothesis testing in neuropsychiatric illness
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID AUTISM SPECTRUM DISORDERS; HUMAN GENOME; PHENOTYPE; INDIVIDUALS; GENES
AB The confounding effects of heterogeneity in biological psychiatry and psychiatric genetics have been widely discussed in the literature. We suggest an approach in which heterogeneity may be put to use in hypothesis testing, and may find application in evaluation of the Crespi & Badcock (C&B) imprinting hypothesis. Here we consider three potential sources of etiologic subtypes for analysis.
C1 [Deutsch, Curtis K.; McIlvane, William J.] Univ Massachusetts, Sch Med, Eunice Kennedy Shriver Ctr Mental Retardat Inc, MRDDRC,Off Director, Waltham, MA 02452 USA.
[Deutsch, Curtis K.] Harvard Univ, Sch Med, Psychobiol Program, Boston, MA 02115 USA.
[Ludwig, Wesley W.] McLean Hosp, Mailman Res Ctr, Neuroregenerat Labs, Belmont, MA 02478 USA.
RP Deutsch, CK (reprint author), Univ Massachusetts, Sch Med, Eunice Kennedy Shriver Ctr Mental Retardat Inc, MRDDRC,Off Director, Waltham, MA 02452 USA.
EM curtis.deutsch@umassmed.edu; wludwig@mclean.harvard.edu;
william.mcilvane@umassmed.edu
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NR 27
TC 2
Z9 2
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 266
EP +
DI 10.1017/S0140525X08004275
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200007
ER
PT J
AU Dickins, BJA
Dickins, DW
Dickins, TE
AF Dickins, Benjamin James Alexander
Dickins, David William
Dickins, Thomas Edmund
TI Is this conjectural phenotypic dichotomy a plausible outcome of genomic
imprinting?
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID ANGELMAN-SYNDROME; GENE-EXPRESSION; EVOLUTION; BEHAVIOR; AUTISM; BRAIN
AB What is the status of the dichotomy proposed and the nosological validity of the contrasting pathologies described in the target article? How plausibly can dysregulated imprinting explain the array of features described, compared with other genetic models? We believe that considering alternative models is more likely to lead in the long term to the correct classification and explanation of the component behaviours.
C1 [Dickins, Benjamin James Alexander] Penn State Univ, Wartik Lab 505, Ctr Comparat Genom & Bioinformat, University Pk, PA 16802 USA.
[Dickins, David William] Univ Liverpool, Sch Psychol, ERB, Liverpool L69 7ZA, Merseyside, England.
[Dickins, Thomas Edmund] Univ E London, Sch Psychol, London E15 4LZ, England.
[Dickins, Thomas Edmund] London Sch Econ, Ctr Philosophy Nat & Social Sci, London WC2A 2AE, England.
RP Dickins, BJA (reprint author), Penn State Univ, Wartik Lab 505, Ctr Comparat Genom & Bioinformat, University Pk, PA 16802 USA.
EM ben@bx.psu.edu; dickins@liverpool.ac.uk; t.dickins@uel.ac.uk
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NR 17
TC 2
Z9 2
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 267
EP +
DI 10.1017/S0140525X08004287
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200008
ER
PT J
AU Fitzgerald, M
Hawi, Z
AF Fitzgerald, Michael
Hawi, Ziarih
TI Creativity, psychosis, autism, and the social brain
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
AB In the target article, Crespi & Badcock (C&B) propose a novel hypothesis based on observations that a large set of phenotypic traits exhibit diametrically opposite phenotypes in autism-spectrum versus psychotic-spectrum conditions. They propose that development of these conditions is mediated in part by alterations in "genomic imprinting." This hypothesis is based on the model of the Prader-Willi and Angelman syndromes. The authors have produced a masterful discussion of the differences between psychosis and autism. Of course, another article could be written on the similarities.
C1 [Fitzgerald, Michael; Hawi, Ziarih] Trinity Coll Dublin, Dept Psychiat, Dublin 2, Ireland.
RP Fitzgerald, M (reprint author), Trinity Coll Dublin, Dept Psychiat, Dublin 2, Ireland.
EM fitzi@iol.ie; zhhawi@tcd.ie
CR McAlonan GM, 2002, BRAIN, V125, P1594, DOI 10.1093/brain/awf150
NR 1
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 268
EP +
DI 10.1017/S0140525X08004299
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200009
ER
PT J
AU Frawley, W
AF Frawley, William
TI Private speech, cognitive-computational control, and the
autism-psychosis continuum
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID INNER SPEECH; LANGUAGE; TASK
AB Autism and psychosis manifest private speech disruptions analogous to their diametrical opposition along the autism-psychosis continuum. Autism has naturally suppressed private speech with predictable structural deficits when it does surface; psychosis has overt but ineffectual private speech with similar structural deficits. These private speech oppositions are best understood in the context of the control processes of cognitive-computational architectures.
C1 [Frawley, William] Ctr Appl Linguist, Washington, DC 20016 USA.
RP Frawley, W (reprint author), Ctr Appl Linguist, Washington, DC 20016 USA.
EM bfrawley@cal.org
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NR 11
TC 1
Z9 1
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 269
EP +
DI 10.1017/S0140525X08004305
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200010
ER
PT J
AU Keller, MC
AF Keller, Matthew C.
TI Problems with the imprinting hypothesis of schizophrenia and autism
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID REPRODUCTIVE FITNESS; 1ST-DEGREE RELATIVES; SPECTRUM DISORDERS;
FERTILITY; EPIDEMIOLOGY; METAANALYSIS; MUTATIONS; SIBLINGS; COMMON; GENE
AB Crespi & Badcock (C&B) convincingly argue that autism and schizophrenia are diametric malfunctions of the social brain, but their core imprinting hypothesis is less persuasive. Much of the evidence they cite is unrelated to their hypothesis, is selective, or is overstated; their hypothesis lacks a clearly explained mechanism; and it is unclear how their explanation fits in with known aspects of the disorders.
C1 [Keller, Matthew C.] Univ Colorado, Dept Psychol, Boulder, CO 80309 USA.
[Keller, Matthew C.] Univ Colorado, Inst Behav Genet, Boulder, CO 80309 USA.
RP Keller, MC (reprint author), Univ Colorado, Dept Psychol, Campus Box 345, Boulder, CO 80309 USA.
EM matthew.c.keller@gmail.com
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NR 22
TC 3
Z9 3
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
EI 1469-1825
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 273
EP +
DI 10.1017/S0140525X08004342
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200014
ER
PT J
AU Langdon, R
Brock, J
AF Langdon, Robyn
Brock, Jon
TI Hypo- or hyper-mentalizing: It all depends upon what one means by
"mentalizing"
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID AUTISM; SCHIZOPHRENIA; GAZE
AB By conceiving of autism and psychosis as diametrically opposite phenotypes of underactive and overactive mentalizing, respectively, Crespi & Badcock (C&B) commit themselves to a continuum view of intercorrelated mentalizing functions. This view fails to acknowledge dissociations between mentalizing functions and that psychotic people show a mixture of both hypo- and hyper-mentalizing.
C1 [Langdon, Robyn; Brock, Jon] Macquarie Univ, Macquarie Ctr Cognit Sci, Sydney, NSW 2109, Australia.
RP Langdon, R (reprint author), Macquarie Univ, Macquarie Ctr Cognit Sci, Sydney, NSW 2109, Australia.
EM rlangdon@maccs.mq.edu.au; jbrock@maccs.mq.edu.au
CR ABELL F, 2000, J COGNITIVE DEV, V15, P1
Blair RJR, 2005, CONSCIOUS COGN, V14, P698, DOI 10.1016/j.concog.2005.06.004
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NR 14
TC 5
Z9 5
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 274
EP +
DI 10.1017/S0140525X08004354
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200015
ER
PT J
AU Oberman, LM
Pascual-Leone, A
AF Oberman, Lindsay M.
Pascual-Leone, Alvaro
TI Cortical plasticity: A proposed mechanism by which genomic factors lead
to the behavioral and neurological phenotype of autism spectrum and
psychotic-spectrum disorders
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID SCHIZOPHRENIA; CORTEX; BDNF
AB Crespi & Badcock (C&B) hypothesize that biases toward expression of paternally or maternally imprinted genes lead to the symptoms of autism spectrum disorders (ASD) and psychotic-spectrum disorders (PSD), respectively. We suggest that such genetic risk factors may act by inducing abnormalities in developmental and learning-related plasticity. We provide preliminary evidence of abnormal plasticity in ASD and suggest transcranial magnetic stimulation as a useful tool to investigate as well as influence cortical plasticity.
C1 [Oberman, Lindsay M.; Pascual-Leone, Alvaro] Beth Israel Deaconess Med Ctr, Berenson Allen Ctr Noninvas Brain Stimulat, Dept Neurol, Boston, MA 02215 USA.
[Oberman, Lindsay M.; Pascual-Leone, Alvaro] Harvard Univ, Sch Med, Boston, MA 02215 USA.
RP Oberman, LM (reprint author), Beth Israel Deaconess Med Ctr, Berenson Allen Ctr Noninvas Brain Stimulat, Dept Neurol, Boston, MA 02215 USA.
EM loberman@bidmc.harvard.edu; apleone@bidmc.harvard.edu
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Weickert CS, 2003, MOL PSYCHIATR, V8, P592, DOI 10.1038/sj.mp.4001308
NR 6
TC 2
Z9 2
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 276
EP +
DI 10.1017/S0140525X08004378
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200017
ER
PT J
AU Thakkar, KN
Matthews, N
Park, S
AF Thakkar, Katharine N.
Matthews, Natasha
Park, Sohee
TI A complete theory of psychosis and autism as diametric disorders of
social brain must consider full range of clinical syndromes
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID SCHIZOPHRENIA
AB We argue that autism and psychosis spectrum disorders cannot be conceptualized as polar extremes of mentalizing ability. We raise two main objections: (1) the autistic-psychotic continuum, as conceptualized by the authors, excludes defining features of schizophrenia spectrum: negative symptoms, which correlate more strongly with mentalizing impairments; and (2) little evidence exists for a relationship between mentalizing ability and positive symptoms.
C1 [Thakkar, Katharine N.; Matthews, Natasha; Park, Sohee] Vanderbilt Univ, Dept Psychol, Nashville, TN 37240 USA.
RP Thakkar, KN (reprint author), Vanderbilt Univ, Dept Psychol, 221 Kirkland Hall, Nashville, TN 37240 USA.
EM katy.thakkar@vanderbilt.edu; natasha.matthews@vanderbilt.edu;
sohee.park@vanderbilt.edu
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NR 10
TC 1
Z9 1
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 277
EP +
DI 10.1017/S0140525X0800438X
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200018
ER
PT J
AU Tordjman, S
AF Tordjman, Sylvie
TI Reunifying autism and early-onset schizophrenia in terms of social
communication disorders
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID SEROTONIN TRANSPORTER GENE; PREMORBID ADJUSTMENT; CHILDHOOD;
POLYMORPHISM; CHILDREN; PSYCHOSIS; VALIDITY; CORTISOL; TRAITS
AB Autism and early-onset schizophrenia share common dimensions of social communication deficits. The possible role of common genetic factors has to be seriously considered, such as the serotonin transporter gene that influences the severity of social communication impairments (negative symptoms) and hallucinations (positive symptoms). Autism and the negative symdrome of schizophrenia might be at one extreme of a continuum, and paranoid schizophrenia (positive symptoms) at the other extreme.
C1 [Tordjman, Sylvie] Univ Rennes 1, Dept Child & Adolescent Psychiat, F-35014 Rennes, France.
[Tordjman, Sylvie] Univ Paris 05, Lab Psychol Percept, CNRS, FRE 2929, F-75270 Paris 06, France.
RP Tordjman, S (reprint author), Univ Rennes 1, Dept Child & Adolescent Psychiat, F-35014 Rennes, France.
EM s.tordjman@ch-guillaumeregnier.fr
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NR 31
TC 5
Z9 5
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 278
EP +
DI 10.1017/S0140525X08004391
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200019
ER
PT J
AU van Rijn, S
Swaab, H
Aleman, A
AF van Rijn, Sophie
Swaab, Hanna
Aleman, Andre
TI Psychosis and autism as two developmental windows on a disordered social
brain
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID SCHIZOPHRENIA; 47,XXY; MIND; MEN
AB With regard to social-cognitive deficits in autism and psychosis, Crespi & Badcock's (C&B's) theory does not incorporate the developmental context of the disorders. We propose that there is significant overlap in social-cognitive impairments, but that the exact manifestation of social-cognitive deficits is highly dependent on the dynamics of cognitive development and hence different in autism as compared to psychosis.
C1 [van Rijn, Sophie; Swaab, Hanna] Leiden Univ, Dept Clin Child & Adolescent Studies, Ctr Study Dev Disorders, NL-2333 AK Leiden, Netherlands.
[Aleman, Andre] Univ Groningen, BCN Neuroimaging Ctr, NL-9713 AW Groningen, Netherlands.
RP van Rijn, S (reprint author), Leiden Univ, Dept Clin Child & Adolescent Studies, Ctr Study Dev Disorders, NL-2333 AK Leiden, Netherlands.
EM srijn@fsw.leidenuniv.nl; hswaab@fsw.leidenuniv.nl; a.aleman@med.umcg.nl
RI Aleman, Andre/E-1341-2012
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NR 15
TC 2
Z9 2
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 280
EP +
DI 10.1017/S0140525X0800441X
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200021
ER
PT J
AU Venkatasubramanian, G
AF Venkatasubramanian, Ganesan
TI Evolutionary perspectives on psychoses and autism: Does genomic
imprinting contribute to phenomenological antithesis?
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID BIPOLAR DISORDER; CORPUS-CALLOSUM; PATERNAL AGE; SCHIZOPHRENIA;
SPECTRUM; INSULIN; DISC1
AB Crespi & Badcock (C&B) have presented a novel view that the influence of genomic imprinting causes diametrically opposite disorders: namely, psychoses and autism. I propose an extended hypothesis that while genomic imprinting is likely to have an influence on the pathogenesis of psychoses and autism, it might contribute to phenomenological antithesis between as well as within these disorders.
C1 [Venkatasubramanian, Ganesan] Natl Inst Mental Hlth & Neurosci, Dept Psychiat, Bangalore 560029, Karnataka, India.
RP Venkatasubramanian, G (reprint author), Natl Inst Mental Hlth & Neurosci, Dept Psychiat, Bangalore 560029, Karnataka, India.
EM venkat.nimhans@gmail.com
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NR 28
TC 1
Z9 1
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 281
EP +
DI 10.1017/S0140525X08004421
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200022
ER
PT J
AU Vladusich, T
AF Vladusich, Tony
TI Towards a computational neuroscience of autism-psychosis spectrum
disorders
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID HALLUCINATIONS
AB Crespi & Badcock (C&B) hypothesize that psychosis and autism represent opposite poles of human social cognition. I briefly outline how computational models of cognitive brain function may be used as a resource to further develop and experimentally test hypotheses concerning "autism-psychosis spectrum disorders."(1).
C1 [Vladusich, Tony] Boston Univ, Dept Cognit & Neural Syst, Boston, MA 02215 USA.
[Vladusich, Tony] Boston Univ, Ctr Excellence Learning Educ Sci & Technol, Boston, MA 02215 USA.
RP Vladusich, T (reprint author), Boston Univ, Dept Cognit & Neural Syst, Boston, MA 02215 USA.
EM thevlad@bu.edu
CR Aleman A, 2003, SCHIZOPHR RES, V64, P175, DOI 10.1016/S0920-9964(03)00060-4
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Molesworth CJ, 2005, J CHILD PSYCHOL PSYC, V46, P661, DOI 10.1111/j.1469-7610.2004.00383.x
VERCAMMEN A, PSYCHOL MED IN PRESS
NR 6
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 282
EP +
DI 10.1017/S0140525X08004433
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200023
ER
PT J
AU Voracek, M
AF Voracek, Martin
TI Digit ratio (2D:4D) as a marker for mental disorders: Low (masculinized)
2D:4D in autism-spectrum disorders, high (feminized) 2D:4D in
schizophrenic-spectrum disorders
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID FINGER LENGTH; 2D-4D; 2ND; HORMONE; ADHD; HERITABILITY; SAMPLE; TWINS
AB Augmenting and supplementing the arguments of Crespi & Badcock (C&B), I show that digit ratio (2D:4D), a putative marker of prenatal androgen action, indeed appears differentially altered in autism-spectrum disorders (lower/masculinized) versus schizophrenic-spectrum disorders (higher/feminized). Consistent with C&B's framework, some evidence (substantial heritability, assortative mating, sex-specific familial transmission) points to possible sex chromosome and imprinted genes effects on 2D:4D expression.
C1 [Voracek, Martin] Univ Vienna, Dept Basic Psychol Res, Sch Psychol, A-1010 Vienna, Austria.
RP Voracek, M (reprint author), Univ Vienna, Dept Basic Psychol Res, Sch Psychol, Waehringer Guertel 18, A-1010 Vienna, Austria.
EM martin.voracek@univie.ac.at
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NR 31
TC 13
Z9 14
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 283
EP +
DI 10.1017/S0140525X08004445
PG 27
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200024
ER
PT J
AU Crespi, B
Badcock, C
AF Crespi, Bernard
Badcock, Christopher
TI The evolutionary social brain: From genes to psychiatric conditions
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
ID SMITH-MAGENIS-SYNDROME; PRADER-WILLI-SYNDROME; FRAGILE-X-SYNDROME;
CHILDHOOD-ONSET SCHIZOPHRENIA; BIPOLAR AFFECTIVE-DISORDER; AUTISM
SPECTRUM DISORDERS; HEAD CIRCUMFERENCE GROWTH; HIGH-FUNCTIONING AUTISM;
SYNDROME DEL 17P11.2; TURNER-SYNDROME
AB The commentaries on our target article, "Psychosis and Autism as Diametrical Disorders of the Social Brain," reflect the multidisciplinary yet highly fragmented state of current studies of human social cognition. Progress in our understanding of the human social brain must come from studies that integrate across diverse analytic levels, using conceptual frameworks grounded in evolutionary biology.
C1 [Crespi, Bernard] Simon Fraser Univ, Dept Biosci, Burnaby, BC V5A 1S6, Canada.
[Badcock, Christopher] London Sch Econ, Dept Sociol, London WC2A 2AE, England.
RP Crespi, B (reprint author), Simon Fraser Univ, Dept Biosci, Burnaby, BC V5A 1S6, Canada.
EM crespi@sfu.ca; C.Badcock@lse.ac.uk
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NR 219
TC 13
Z9 13
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD JUN
PY 2008
VL 31
IS 3
BP 284
EP 320
DI 10.1017/S0140525X08004457
PG 37
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 338XH
UT WOS:000258542200025
ER
PT J
AU Bonilha, L
Cendes, F
Rorden, C
Eckert, M
Dalgalarrondo, P
Li, LM
Steiner, CE
AF Bonilha, Leonardo
Cendes, Fernando
Rorden, Chris
Eckert, Mark
Dalgalarrondo, Paulo
Li, Li Min
Steiner, Carlos E.
TI Gray and white matter imbalance - Typical structural abnormality
underlying classic autism?
SO BRAIN & DEVELOPMENT
LA English
DT Article
DE autism; brain structure; gray matter; white matter; connectivity
ID VOXEL-BASED MORPHOMETRY; SPECTRUM DISORDERS; FRONTAL-CORTEX; BRAIN; MRI;
PREVALENCE; LIFE
AB Recent evidence supports increased cortical activity and impaired brain connectivity in autism, but the structural correlates of these abnormalities are not yet defined. We performed a voxel based morphometry analysis of brain MRI from patients with autism selected from a group of 103 subjects with pervasive developmental disorders. Twelve male patients with mean age of 12.4 +/- 4 years were compared with 16 matched controls. Patients with autism exhibited increase in gray matter in medial and dorsolateral frontal areas, in the lateral and medial parts of the temporal lobes, in the parietal lobes, cerebellum and claustrum. Patients also showed decrease in frontal, parietal, temporal and occipital white matter. The combination of enlarged cortex and reduced white matter is possibly the structural basis of some symptoms of classic autism. (C) 2007 Elsevier B.V. All rights reserved.
C1 [Bonilha, Leonardo; Rorden, Chris] Univ S Carolina, Dept Commun Sci & Disorders, Columbia, SC 29208 USA.
[Bonilha, Leonardo] Univ S Carolina, Dept Neuropsychiat, Columbia, SC 29208 USA.
[Cendes, Fernando; Li, Li Min] Univ Estadual Campinas, Dept Psychiat, Campinas, Brazil.
[Eckert, Mark; Dalgalarrondo, Paulo] Med Univ S Carolina, Dept Otolaryngol, Charleston, SC 29425 USA.
[Steiner, Carlos E.] Univ Estadual Campinas, Dept Med Genet, Campinas, Brazil.
RP Bonilha, L (reprint author), Univ S Carolina, Dept Commun Sci & Disorders, 1621 Greene St,6th Floor,Room 630B, Columbia, SC 29208 USA.
EM bonilha@gwm.sc.edu
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NR 25
TC 39
Z9 40
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0387-7604
J9 BRAIN DEV-JPN
JI Brain Dev.
PD JUN
PY 2008
VL 30
IS 6
BP 396
EP 401
DI 10.1016/j.braindev.2007.11.006
PG 6
WC Clinical Neurology
SC Neurosciences & Neurology
GA 303UH
UT WOS:000256065900004
PM 18362056
ER
PT J
AU Heaton, P
Allen, R
Williams, K
Cummins, O
Happe, F
AF Heaton, Pamela
Allen, Rory
Williams, Kerry
Cummins, Omar
Happe, Francesca
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from autism and Down syndrome
SO BRITISH JOURNAL OF DEVELOPMENTAL PSYCHOLOGY
LA English
DT Article
ID CHILDREN; DISORDERS; EMOTION
AB Children with autism experience difficulties in understanding social affective cues, and it has been suggested that such deficits will generalize to music. In order to investigate this proposal, typically developing individuals and children with autism and Down syndrome were compared on tasks measuring perception of affective and movement states in music. The results showed that discrimination performance on both experimental conditions depended on chronological or verbal mental age rather than diagnosis. The findings suggest that emotion-processing deficits in the social domain do not generalize to music, and that musical understanding is closely related to the level of language development.
C1 [Heaton, Pamela; Allen, Rory; Williams, Kerry; Cummins, Omar] Univ London, Univ London Goldsmiths Coll, London SE14 6NW, England.
[Happe, Francesca] Univ London, Inst Psychiat, London SE14 6NW, England.
RP Heaton, P (reprint author), Univ London, Univ London Goldsmiths Coll, London SE14 6NW, England.
EM P.Heaton@gold.ac.uk
RI Allen, Rory/E-8660-2011; Happe, Francesca/D-5544-2012
OI Allen, Rory/0000-0003-3434-9772;
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NR 25
TC 18
Z9 18
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0261-510X
J9 BRIT J DEV PSYCHOL
JI Br. J. Dev. Psychol.
PD JUN
PY 2008
VL 26
BP 171
EP 182
DI 10.1348/026151007X206776
PN 2
PG 12
WC Psychology, Developmental
SC Psychology
GA 306DD
UT WOS:000256227600003
ER
PT J
AU Ford, RM
Rees, EL
AF Ford, Ruth M.
Rees, Elen Lord
TI Representational drawing and the transition from intellectual to visual
realism in children with autism
SO BRITISH JOURNAL OF DEVELOPMENTAL PSYCHOLOGY
LA English
DT Article
ID HIGH-FUNCTIONING AUTISM; MIND; INDIVIDUALS; DIMENSIONALITY; ACCURACY;
FEATURES; CONTEXT; SAVANTS; ABILITY; OBJECT
AB The present study compared the representational drawings of children with autism, children with Down syndrome and typically developing children. Participants were asked to draw a series of objects and their depictions were scored for the incidence of intellectual realism. The tasks sought evidence of conceptual as opposed to episodic influences on intellectual realism in terms of (1) omission errors; for the visible decorative attributes of isolated objects and (2) commission errors for the hidden categorical attributes of contextually situated objects. All groups evinced sensitivity to meaning as gauged by the former measure but the autistic group were impaired in their performance on the latter measure. Whereas children without autism were less likely to depict the occluded handle of a cup when the model was presented in context rather than independently, this trend was lacking in the autistic sample. We consider these findings in terms of weak central coherence and alternative accounts of impaired conceptualization in autism.
C1 [Ford, Ruth M.] Griffith Univ, Sch Psychol, Brisbane, Qld 4111, Australia.
[Rees, Elen Lord] Swansea Univ, Swansea, W Glam, Wales.
RP Ford, RM (reprint author), Griffith Univ, Sch Psychol, Brisbane, Qld 4111, Australia.
EM r.ford@griffith.edu.au
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NR 51
TC 3
Z9 3
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0261-510X
J9 BRIT J DEV PSYCHOL
JI Br. J. Dev. Psychol.
PD JUN
PY 2008
VL 26
BP 197
EP 219
DI 10.1348/026151007X209917
PN 2
PG 23
WC Psychology, Developmental
SC Psychology
GA 306DD
UT WOS:000256227600005
ER
PT J
AU Bigham, S
AF Bigham, Sally
TI Comprehension of pretence in children with autism
SO BRITISH JOURNAL OF DEVELOPMENTAL PSYCHOLOGY
LA English
DT Article
ID SYMBOLIC PLAY; DEFICITS; MIND; TRANSFORMATIONS; INFANCY; OBJECTS; DELAY
AB Impairments of pretend play are a diagnostic characteristic of autism. This has been interpreted in terms of a generative impairment. Specifically, children with autism are unable to generate the ideas for pretend play despite an intact underlying ability to understand pretence. The notion of a performance deficit affecting production only has, in part, been based upon observations that children with autism appear to have no difficulty understanding pretence. However, research investigating comprehension is somewhat limited and has only investigated a specific type of pretend play, namely pretend properties. The research reported here investigated the ability of children with autism to understand object substitution pretence and pretend gestures (i.e. using no substitutes). Children with autism (N = 36) matched for verbal ability (between 3 and 6 years) with typically developing children (N = 55) and children with moderate learning difficulties (N = 37) were questioned about a series of pretend actions, such as writing. The results revealed three main findings. First, children with autism appear to experience no notable difficulties comprehending functional play. Second, children with autism do encounter difficulties comprehending pretend play. A performance deficit is unable to explain these findings and it is suggested that there may be a more critical impairment in understanding how absent objects are represented. Finally, there was a trend for some kinds of object substitutions and gestures to be more impaired than others. One tentative interpretation is that as the degree of similarity between the referent and the substitute diminishes the representational connection becomes more difficult to understand, particularly when there is a need to inhibit salient but irrelevant cues.
C1 Thames Valley Univ, Dept Psychol, London W5 5RF, England.
RP Bigham, S (reprint author), Thames Valley Univ, Dept Psychol, St Marys Rd, London W5 5RF, England.
EM Sally.Bigham@tvu.oc.uk
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NR 37
TC 3
Z9 3
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0261-510X
J9 BRIT J DEV PSYCHOL
JI Br. J. Dev. Psychol.
PD JUN
PY 2008
VL 26
BP 265
EP 280
DI 10.1348/026151007X235855
PN 2
PG 16
WC Psychology, Developmental
SC Psychology
GA 306DD
UT WOS:000256227600009
ER
PT J
AU Botting, N
Conti-Ramsden, G
AF Botting, Nicola
Conti-Ramsden, Gina
TI The role of language, social cognition, and social skill in the
functional social outcomes of young adolescents with and without a
history of SLI
SO BRITISH JOURNAL OF DEVELOPMENTAL PSYCHOLOGY
LA English
DT Article
ID IMPAIRMENT SLI; FOLLOW-UP; SPECTRUM DISORDER; ASPERGER-SYNDROME; ADULT
LIFE; CHILDREN; AUTISM; MIND; PERSPECTIVES; QUESTIONNAIRE
AB Social skill and language are known to relate, not least in the example of those with specific language impairment (SLI). However, most of the research examining this trend has been conducted on young primary school age children and the nature of the relationships is unclear. Furthermore, little is known about which young people in general have social difficulties and whether language, social cognition, and social skills are directly associated at this age. In this study, a large cohort made up of young people with a history of SLI (N = 134) and a typically developing (TD) group (N = 124) of the same age were followed up in their final year of compulsory schooling (aged 16). Language, social cognition, social skills, and functional social outcomes (friendships and levels of social activity) were assessed using tasks and questionnaires. Modest associations were found between social cognition, language, and social behaviours, the strongest being between language and social cognition. Regression analyses showed that as a combined group, the adolescents' functional social outcomes were most associated with expressive language, social skill, and social cognitive ability. However, the patterns differed when the groups were analysed separately, with social cognition playing more of a role for those with SLI. These findings suggest that poor language may play a complex role in adolescents' social development.
C1 [Botting, Nicola] City Univ London, London EC1V 0HB, England.
[Conti-Ramsden, Gina] Univ Manchester, Manchester, Lancs, England.
RP Botting, N (reprint author), City Univ London, Northampton Sq, London EC1V 0HB, England.
EM nicola.botting.l@cq.ac.uk
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NR 54
TC 25
Z9 27
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0261-510X
J9 BRIT J DEV PSYCHOL
JI Br. J. Dev. Psychol.
PD JUN
PY 2008
VL 26
BP 281
EP 300
DI 10.1348/026151007X235891
PN 2
PG 20
WC Psychology, Developmental
SC Psychology
GA 306DD
UT WOS:000256227600010
ER
PT J
AU Russell, RL
Grizzle, KL
AF Russell, Robert L.
Grizzle, Kenneth L.
TI Assessing child and adolescent pragmatic language competencies: Toward
evidence-based assessments
SO CLINICAL CHILD AND FAMILY PSYCHOLOGY REVIEW
LA English
DT Review
DE pragmatic language competence; assessment of language skills; childhood
disorders; social communication skills; childhood language disorders
ID PSYCHIATRICALLY DISTURBED-CHILDREN; BEHAVIORAL-CHARACTERISTICS;
COMMUNICATION-CHECKLIST; FOLLOW-UP; CONVERSATIONAL CHARACTERISTICS;
DISORDERS; IMPAIRMENT; MIND; AUTISM; ADHD
AB Using language appropriately and effectively in social contexts requires pragmatic language competencies (PLCs). Increasingly, deficits in PLCs are linked to child and adolescent disorders, including autism spectrum, externalizing, and internalizing disorders. As the role of PLCs expands in diagnosis and treatment of developmental psychopathology, psychologists and educators will need to appraise and select clinical and research PLC instruments for use in assessments and/or studies. To assist in this appraisal, 24 PLC instruments, containing 1,082 items, are assessed by addressing four questions: (1) Can PLC domains targeted by assessment items be reliably identified?, (2) What are the core PLC domains that emerge across the 24 instruments?, (3) Do PLC questionnaires and tests assess similar PLC domains?, and (4) Do the instruments achieve content, structural, diagnostic, and ecological validity? Results indicate that test and questionnaire items can be reliably categorized into PLC domains, that PLC domains featured in questionnaires and tests significantly differ, and that PLC instruments need empirical confirmation of their dimensional structure, content validity across all developmental age bands, and ecological validity. Progress in building a better evidence base for PLC assessments should be a priority in future research.
C1 [Russell, Robert L.; Grizzle, Kenneth L.] Med Coll Wisconsin, Dept Pediat, Milwaukee, WI 53226 USA.
RP Russell, RL (reprint author), Med Coll Wisconsin, Dept Pediat, 8701 Watertown Plank Rd,POB 26509, Milwaukee, WI 53226 USA.
EM rrussell@mcw.edu
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NR 115
TC 8
Z9 8
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1096-4037
J9 CLIN CHILD FAM PSYCH
JI Clin. Child Fam. Psychol. Rev.
PD JUN
PY 2008
VL 11
IS 1-2
BP 59
EP 73
DI 10.1007/s10567-008-0032-1
PG 15
WC Psychology, Clinical
SC Psychology
GA 299IW
UT WOS:000255750300004
PM 18386177
ER
PT J
AU Van Der Linde, AAA
Pillen, S
Gerrits, GPM
Bavinck, JNB
AF Van Der Linde, A. A. A.
Pillen, S.
Gerrits, G. PJ. M.
Bavinck, J. N. Bouwes
TI Stevens-Johnson syndrome in a child with chronic mercury exposure and
2,3-dimercaptopropane-1-sulfonate (DMPS) therapy
SO CLINICAL TOXICOLOGY
LA English
DT Article
DE Stevens-Johnson syndrome; child; chelation therapy; DMPS; mercury
intoxication
AB Introduction. Stevens-Johnson syndrome (SJS) is an uncommon and potentially serious mucocutaneous disease. The most important step in the management of SJS is early recognition and immediate withdrawal of the causative agent. We present a patient with SJS associated with dimercaptopropane-l-sulfonate (DMPS) therapy. Case Report. An asymptomatic 11-year old boy who had been exposed chronically to mercury vapour had a 24-hour urine mercury concentration of 37 microgram/L (reference value < 10 microgram/L). Exposure to the mercury vapour was stopped and treatment with oral DMPS was begun. After two weeks of therapy, he developed a disseminated cutaneous eruption of red pruritic macules on his chest and back, which three days later had spread all over his body with the discrete maculae becoming confluent; erosions and crusts developed on his lips and he had blisters in his mouth. The diagnosis of SJS was made, the DMPS was stopped, and the SJS resolved gradually. Discussion. Chelation agents like DMPS or DMSA are increasingly used and are available over the counter in some countries. These drugs are used in patients with complaints that are attributed to mercury-containing dental amalgams and in children with autism. Conclusion. The reported association suggests that SJS may be a potential complication of DMPS therapy, and this should be considered in the risk-benefit analysis of chelation. The reported association suggests that SJS may be a potential complication of DMPS therapy, and this should be considered in the risk-benefit analysis of chelation.
C1 [Van Der Linde, A. A. A.; Pillen, S.; Gerrits, G. PJ. M.] Canisius Wilhelmina Hosp, NL-6500 GS Nijmegen, Netherlands.
[Bavinck, J. N. Bouwes] Leiden Univ, Med Ctr, Leiden, Netherlands.
RP Van Der Linde, AAA (reprint author), Canisius Wilhelmina Hosp, NL-6500 GS Nijmegen, Netherlands.
EM annelieke@hotmail.com
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NR 7
TC 7
Z9 7
PU INFORMA HEALTHCARE
PI NEW YORK
PA 52 VANDERBILT AVE, NEW YORK, NY 10017 USA
SN 1556-3650
J9 CLIN TOXICOL
JI Clin. Toxicol.
PD JUN
PY 2008
VL 46
IS 5
BP 479
EP 481
DI 10.1080/15563650701779687
PG 3
WC Toxicology
SC Toxicology
GA 315YV
UT WOS:000256917000327
PM 18568806
ER
PT J
AU Bauminger, N
Chomsky-Smolkin, L
Orbach-Caspi, E
Zachor, D
Levy-Shiff, R
AF Bauminger, Nirit
Chomsky-Smolkin, Liza
Orbach-Caspi, Efrat
Zachor, Ditza
Levy-Shiff, Rachel
TI Jealousy and emotional responsiveness in young children with ASD
SO COGNITION & EMOTION
LA English
DT Article
ID DEPRESSED MOTHERS; JOINT ATTENTION; AUTISM; INFANTS; RESPONSES;
DISTRESS; CONTEXT; OTHERS
AB We investigated manifestations of jealousy in preschoolers (n = 32) with autism spectrum disorder (ASD) and in a group of typically developing children (n = 18) matched on mental age, verbal mental age, nonverbal mental age, and mother's education. Main results revealed explicit indices of jealousy in two thirds of the children with ASD compared with 94.5% in the typical group. In addition, different manifestations of this emotion emerged in the ASD group compared with the matched control group. Regarding mental and affective correlates of jealousy, expressions of jealousy correlated with IQ only for the children in the ASD group, and the ASD group revealed deficient emotional responsiveness (ER) capabilities. Significant correlations emerged between jealousy and ER in both the ASD and control groups. Discussion focuses on implications of these findings for understanding the core emotional deficit in autism.
C1 [Bauminger, Nirit] Bar Ilan Univ, Sch Educ, IL-52900 Ramat Gan, Israel.
[Zachor, Ditza] Tel Aviv Univ, IL-69978 Tel Aviv, Israel.
RP Bauminger, N (reprint author), Bar Ilan Univ, Sch Educ, IL-52900 Ramat Gan, Israel.
EM bauminn@mail.biu.ac.il
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NR 45
TC 5
Z9 5
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0269-9931
J9 COGNITION EMOTION
JI Cogn. Emot.
PD JUN
PY 2008
VL 22
IS 4
BP 595
EP 619
DI 10.1080/02699930701439986
PG 25
WC Psychology, Experimental
SC Psychology
GA 315NY
UT WOS:000256887900002
ER
PT J
AU Zinck, A
AF Zinck, Alexandra
TI Self-referential emotions
SO CONSCIOUSNESS AND COGNITION
LA English
DT Article; Proceedings Paper
CT Interdisciplinary Conference on Consciousness and Cognition
CY 2007
CL Delmenhorst, GERMANY
HO Hanse Inst Adv Studies
DE emotion; self-referential emotion; self-representation;
self-consciousness; mentalizing; interaction; 'theory of mind'
ID REPRESENTATION; CONSCIOUSNESS; EXPRESSIONS; EMBARRASSMENT; IMITATION;
COGNITION; CHILDREN; ORIGINS; AUTISM; OTHERS
AB The aim of this paper is to examine a special subgroup of emotion: self-referential emotions such as shame, pride and guilt. Self-referential emotions are usually conceptualized as (i) essentially involving the subject herself and as (ii) having complex conditions such as the capacity to represent others' thoughts. I will show that rather than depending on a fully fledged 'theory of mind' and an explicit language-based self-representation, (i) pre-forms of self-referential emotions appear at early developmental stages already exhibiting their characteristic structure of the intentional object of the emotion being identical with or intricately related to the subject experiencing the emotional state and that (ii) they precede and substantially contribute to the development of more complex representations and to the development of a self-concept, to social interaction and to ways of understanding of other minds. (C) 2008 Elsevier Inc. All rights reserved.
C1 Ruhr Univ Bochum, Inst Philosophie, D-44801 Bochum, Germany.
RP Zinck, A (reprint author), Ruhr Univ Bochum, Inst Philosophie, Univ Str 150, D-44801 Bochum, Germany.
EM alexandra.zinck@rub.de
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NR 74
TC 11
Z9 11
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1053-8100
J9 CONSCIOUS COGN
JI Conscious. Cogn.
PD JUN
PY 2008
VL 17
IS 2
BP 496
EP 505
DI 10.1016/j.concog.2008.03.014
PG 10
WC Psychology, Experimental
SC Psychology
GA 309WE
UT WOS:000256490100010
PM 18436451
ER
PT J
AU David, N
Newen, A
Vogeley, K
AF David, Nicole
Newen, Albert
Vogeley, Kai
TI The "sense of agency" and its underlying cognitive and neural mechanisms
SO CONSCIOUSNESS AND COGNITION
LA English
DT Article; Proceedings Paper
CT Interdisciplinary Conference on Consciousness and Cognition
CY 2007
CL Delmenhorst, GERMANY
HO Hanse Inst Adv Studies
DE sense of agency; perspective taking; simulation; central monitoring;
pre-reflective consciousness
ID POSTERIOR PARIETAL CORTEX; AUTISM SPECTRUM DISORDERS; EXTRASTRIATE BODY
AREA; SELF-CONSCIOUSNESS; SCHIZOPHRENIC-PATIENTS; GENERATED MOVEMENTS;
ACTION RECOGNITION; VOLUNTARY ACTION; HAND MOVEMENTS; OTHERS ACTIONS
AB The sense of agency is a central aspect of human self-consciousness and refers to the experience of oneself as the agent of one's own actions. Several different cognitive theories on the sense of agency have been proposed implying divergent empirical approaches and results, especially with respect to neural correlates. A multifactorial and multilevel model of the sense of agency may provide the most constructive framework for integrating divergent theories and findings, meeting the complex nature of this intriguing phenomenon. (C) 2008 Elsevier Inc. All rights reserved.
C1 [David, Nicole] Univ Med Ctr Hamburg Eppendorf, Ctr Med Expt, Dept Neurophysiol & Pathophysiol, D-20246 Hamburg, Germany.
[David, Nicole; Vogeley, Kai] Univ Cologne, Dept Psychiat & Psychotherapy, D-50924 Cologne, Germany.
[Newen, Albert] Ruhr Univ Bochum, Dept Philosophy, D-44801 Bochum, Germany.
RP David, N (reprint author), Univ Med Ctr Hamburg Eppendorf, Ctr Med Expt, Dept Neurophysiol & Pathophysiol, Martinistr 52, D-20246 Hamburg, Germany.
EM ndavid@uke.de
RI David, Nicole/H-1682-2012; Vogeley, K/E-4860-2012
OI Vogeley, K/0000-0002-5891-5831
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NR 104
TC 83
Z9 84
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1053-8100
J9 CONSCIOUS COGN
JI Conscious. Cogn.
PD JUN
PY 2008
VL 17
IS 2
BP 523
EP 534
DI 10.1016/j.concog.2008.03.004
PG 12
WC Psychology, Experimental
SC Psychology
GA 309WE
UT WOS:000256490100013
PM 18424080
ER
PT J
AU Hay, DA
AF Hay, David A.
TI Fragile X - A challenge to models of the mind and to best clinical
practice
SO CORTEX
LA English
DT Editorial Material
DE fragile X; response inhibition; selective attention; ageing; ADHD;
ataxia
AB Cornish et al. (2008, this issue) provide an excellent review of Fragile X a common but very complex cause of intellectual disability. They report on a cohort of such males of normal intelligence quotient (IQ) and socioeconomic status (SES), but who have deficits in selective attention and growing impairment in response inhibition. This paper has theoretical views for our models of the mind and clinical implications for families where Fragile X may never have been considered as a possible cause of some of the problems in male and female family members and possibly as well for other disorders such as attention deficit hyperactivity disorder (ADHD) and autism. (C) 2007 Elsevier Masson Srl. All rights reserved.
C1 Curtin Univ Technol, Sch Psychol, Perth, WA 6845, Australia.
RP Hay, DA (reprint author), Curtin Univ Technol, Sch Psychol, GPO Box U1987, Perth, WA 6845, Australia.
EM d.hay@curtin.edu.au
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NR 8
TC 5
Z9 5
PU ELSEVIER MASSON
PI MILANO
PA VIA PALEOCAPA 7, 20121 MILANO, ITALY
SN 0010-9452
J9 CORTEX
JI Cortex
PD JUN
PY 2008
VL 44
IS 6
BP 626
EP 627
DI 10.1016/j.cortex.2007.05.001
PG 2
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 310CG
UT WOS:000256505900002
PM 18472032
ER
PT J
AU Flint, J
Shifman, S
AF Flint, Jonathan
Shifman, Sagiv
TI Animal models of psychiatric disease
SO CURRENT OPINION IN GENETICS & DEVELOPMENT
LA English
DT Review
ID ADULT HIPPOCAMPAL NEUROGENESIS; ANXIETY-RELATED BEHAVIOR;
MATERNAL-BEHAVIOR; LIFE STRESS; MICE; DEPRESSION; AUTISM; SCHIZOPHRENIA;
GENES; MOUSE
AB Animal models of psychiatric diseases are useful tools for screening new drugs and for investigating the mechanisms of those disorders. Despite the difficulties inherent in modelling human psychiatric phenotypes in animals, there has been recent success identifying mutations in mice that give rise to some of the characteristic features of anxiety, depression, schizophrenia, autism, obsessive-compulsive disorder and bipolar disorder. In some cases these models have the additional strength that drugs used to treat the human condition alleviate the symptoms in mice. Robust genetic evidence of the involvement of multiple susceptibility genes in psychiatric disease will enable future studies to move from single-gene models to models with multiple modified loci, with the promise of better representing the complexity of the human diseases.
C1 [Flint, Jonathan] Univ Oxford, Wellcome Trust Ctr Human Genet, Oxford, England.
[Shifman, Sagiv] Hebrew Univ Jerusalem, Inst Life Sci, Dept Genet, IL-91904 Jerusalem, Israel.
RP Flint, J (reprint author), Univ Oxford, Wellcome Trust Ctr Human Genet, Oxford, England.
EM jf@well.ox.ac.uk
FU Wellcome Trust
FX Jonathan Flint is funded by the Wellcome Trust.
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NR 42
TC 26
Z9 27
PU CURRENT BIOLOGY LTD
PI LONDON
PA 84 THEOBALDS RD, LONDON WC1X 8RR, ENGLAND
SN 0959-437X
J9 CURR OPIN GENET DEV
JI Curr. Opin. Genet. Dev.
PD JUN
PY 2008
VL 18
IS 3
BP 235
EP 240
DI 10.1016/j.gde.2008.07.002
PG 6
WC Cell Biology; Genetics & Heredity
SC Cell Biology; Genetics & Heredity
GA 366AM
UT WOS:000260451800003
PM 18657615
ER
PT J
AU Martin, JR
Arici, A
AF Martin, J. Ryan
Arici, Aydin
TI Fragile X and reproduction
SO CURRENT OPINION IN OBSTETRICS & GYNECOLOGY
LA English
DT Article
DE fragile X; premature ovarian failure; reproduction
ID PREMATURE OVARIAN FAILURE; PREIMPLANTATION GENETIC DIAGNOSIS; CGG REPEAT
NUMBER; PREMUTATION CARRIERS; FMR1 PREMUTATION; FULL-MUTATION; WOMEN;
INTERMEDIATE; MENOPAUSE; SPERMATOGENESIS
AB Purpose of review
To review the recent literature on fragile X and the genotypic and phenotypic implications on human reproduction.
Recent findings
Fragile X syndrome is the most common inherited cause of mental retardation and the most common genetic cause of autism. The fully expanded form of the mutation leads to mental retardation and autism, whereas the premutation can lead to a neurological disorder called fragile X-associated tremor/ataxia, macroorchidism after puberty and premature ovarian failure. Fragile X is also a major cause of premature ovarian failure and irregular menses, and it can subsequently affect fertility. Couples who carry the premutation or full mutation should be offered genetic and preconceptual counseling prior to attempting to conceive. This allows the patient full disclosure about the risks of transmitting the mutation and possible preventive measures, which allows them to formulate educated decisions about their reproductive future.
Summary
Clinicians should identify and recognize clinical situations that warrant fragile X testing to properly counsel and inform patients about their reproductive possibilities and ways to increase their chances of a successful reproductive outcome.
C1 [Martin, J. Ryan; Arici, Aydin] Yale Univ, Sch Med, Div Reprod Endocrinol & Infertil, Dept Obstet Gynecol & Reprod Sci, New Haven, CT 06520 USA.
RP Martin, JR (reprint author), Yale Univ, Sch Med, Div Reprod Endocrinol & Infertil, Dept Obstet Gynecol & Reprod Sci, 333 Cedar St, New Haven, CT 06520 USA.
EM ryan.martin@yale.edu; aydin.arici@yale.edus
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NR 55
TC 7
Z9 8
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 1040-872X
J9 CURR OPIN OBSTET GYN
JI Curr. Opin. Obstet. Gynecol.
PD JUN
PY 2008
VL 20
IS 3
BP 216
EP 220
DI 10.1097/GCO.0b013e3282fe7254
PG 5
WC Obstetrics & Gynecology
SC Obstetrics & Gynecology
GA 304XP
UT WOS:000256142700004
PM 18460934
ER
PT J
AU Dawson, G
AF Dawson, Geraldine
TI Early behavioral intervention, brain plasticity, and the prevention of
autism spectrum disorder
SO DEVELOPMENT AND PSYCHOPATHOLOGY
LA English
DT Review
ID ENVIRONMENTAL ENRICHMENT REVERSES; PERVASIVE DEVELOPMENTAL DISORDERS;
HUMAN EXTRASTRIATE CORTEX; FUSIFORM FACE AREA; RECEPTOR GENE OXTR;
YOUNG-CHILDREN; GENOMEWIDE SCREEN; INFANTILE-AUTISM; JOINT ATTENTION;
SOCIAL BRAIN
AB Advances in the fields of cognitive and affective developmental neuroscience, developmental psychopathology, neurobiology, genetics, and applied behavior analysis have contributed to a more optimistic outcome for individuals with autism spectrum disorder (ASD). These advances have led to new methods for early detection and more effective treatments. For the first time, prevention of ASD is plausible. Prevention will entail detecting infants at risk before the full syndrome is present and implementing treatments designed to alter the course of early behavioral and brain development. This article describes a developmental model of risk, risk processes, symptom emergence, and adaptation in ASD that offers a framework for understanding early brain plasticity in ASD and its role in prevention of the disorder.
C1 Autism Speaks, Hillsborough, NC 27278 USA.
RP Dawson, G (reprint author), Autism Speaks, 1311 Lawrence Dr, Hillsborough, NC 27278 USA.
EM gdawson@autismspeaks.org
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NR 265
TC 218
Z9 223
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0954-5794
J9 DEV PSYCHOPATHOL
JI Dev. Psychopathol.
PD SUM
PY 2008
VL 20
IS 3
BP 775
EP 803
DI 10.1017/S0954579408000370
PG 29
WC Psychology, Developmental
SC Psychology
GA 327NP
UT WOS:000257736100003
PM 18606031
ER
PT J
AU Selassie, GRH
Viggedal, G
Olsson, I
Jennische, M
AF Selassie, G. Rejno-Habte
Viggedal, G.
Olsson, I.
Jennische, M.
TI Speech, language, and cognition in preschool children with epilepsy
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Article
ID CHILDHOOD EPILEPSY; DISORDERS; HANDICAP; SEIZURES; SPIKES; MEMORY
AB We studied expressive and receptive language, oral motor ability, attention, memory, and intelligence in 20 6-year- old children with epilepsy (14 females, six males; mean age 6y 5mo, range 6y-6y 11mo) without learning disability, cerebral palsy (CP), and/or autism, and in 30 reference children without epilepsy (18 females, 12 males; mean age 6y 5mo, range 6y-6y 11mo). Ten children had partial, six primarily generalized, and four unclassified epilepsy. Fourteen were having monotherapy and six were taking two or more antiepileptic drugs; 13 children were free from seizures 3 months before the assessment. Results show no statistically significant difference between the groups concerning Verbal IQ, expressive and receptive grammar, and receptive vocabulary. The children with epilepsy had a significantly lower Performance IQ and lower scores in tests of oral motor ability, articulation, emerging literacy, auditory attention, short-term memory, and rapid word retrieval. Parent ratings revealed no significant difference in communicative ability. Polytherapy and early onset of epilepsy influenced some results. Preschool children with epilepsy without learning disability, CP, and/or autism may have receptive verbal ability within the normal range but visuoperceptual, auditory attentional, and speech-language difficulties that could affect school achievement. Careful testing of children with epilepsy who appear to be functioning within the normal range is needed because this may reveal specific impairments that require appropriate professional input.
C1 [Selassie, G. Rejno-Habte] Univ Gothenburg, Sahlgrenska Acad, Inst Neurosci & Physiol, Dept Speech & Language Pathol, SE-40530 Gothenburg, Sweden.
[Viggedal, G.; Olsson, I.] Univ Gothenburg, Sahlgrenska Acad, Dept Paediat, SE-40530 Gothenburg, Sweden.
[Jennische, M.] Uppsala Univ, Fac Med, Dept Neurosci Speech & Language Pathol, Uppsala, Sweden.
RP Selassie, GRH (reprint author), Univ Gothenburg, Sahlgrenska Acad, Inst Neurosci & Physiol, Dept Speech & Language Pathol, Box 452, SE-40530 Gothenburg, Sweden.
EM gunilla.rejno-habte-selassie@vgregion.se
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NR 30
TC 15
Z9 15
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD JUN
PY 2008
VL 50
IS 6
BP 432
EP 438
DI 10.1111/j.1469-8749.2008.02060.x
PG 7
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 302AC
UT WOS:000255937200011
PM 18422681
ER
PT J
AU Fernyhough, C
AF Fernyhough, Charles
TI Getting Vygotskian about theory of mind: Mediation, dialogue, and the
development of social understanding
SO DEVELOPMENTAL REVIEW
LA English
DT Review
DE dialogue; inner speech; mentalizing; private speech; semiotic mediation;
social understanding; Vygotsky's theory
ID PRIVATE SPEECH; INDIVIDUAL-DIFFERENCES; TASK-PERFORMANCE; FALSE BELIEF;
VERBAL MEDIATION; MENTAL STATE; INNER SPEECH; LANGUAGE; CHILDREN; AUTISM
AB The ideas of Vygotsky [Vygotsky, L. S. (1987). Thinking and speech. In The collected works of L. S. Vygotsky, (Vol. 1). New York: Plenum. (Original work published 1934.)] have been increasingly influential in accounting for social-environmental influences on the development of social understanding (SU). In the first part of this article, I examine how Vygotskian ideas have to date been recruited to explanations of the development of SU. Next, I present a model of SU development which draws on two implications of Vygotsky's ideas: the importance of semiotic mediation for mental functioning, and the dialogic nature of the higher mental functions. I then consider the value of the proposed model in accounting for evidence from three areas of enquiry: the typical development of SU in infancy and early childhood, relations between individual differences in SU and social-environmental variables, and atypical development. The model is suggested to be particularly helpful in understanding the transition from intentional-agent to mental-agent understanding, and the role of language in SU. Remaining challenges include a need to specify further the cognitive processes underlying internalization, and to gather more extensive evidence on the roles of typical and atypical social experience in SU development. (C) 2007 Elsevier Inc. All rights reserved.
C1 Univ Durham, Dept Psychol, Durham DH1 3LE, England.
RP Fernyhough, C (reprint author), Univ Durham, Dept Psychol, South Rd, Durham DH1 3LE, England.
EM c.p.fernyhough@durham.ac.uk
RI Fernyhough, Charles/A-1057-2010
OI Fernyhough, Charles/0000-0002-3822-710X
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NR 151
TC 43
Z9 43
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0273-2297
J9 DEV REV
JI Dev. Rev.
PD JUN
PY 2008
VL 28
IS 2
BP 225
EP 262
DI 10.1016/j.dr.2007.03.001
PG 38
WC Psychology, Developmental
SC Psychology
GA 303BO
UT WOS:000256014700002
ER
PT J
AU Moreno, J
Aguilera-Jimenez, A
Saldana, D
AF Moreno, Javier
Aguilera-Jimenez, Antonio
Saldana, David
TI Do Spanish parents prefer special schools for their children with
autism?
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID INCLUSION; EDUCATION; PERSPECTIVES
AB The social and communication difficulties of children with Autism Spectrum Disorders (ASD) pose a special challenge to educational inclusion. Previous research has suggested that, because of this, parents of children with ASD might be less favorable to educating their children in inclusive settings. In this study, 60 parents of children with ASD in the city of Seville (Spain) were interviewed about their perception of educational provision. Parents were from three different groups, according to the children's educational placement: mainstream non-segregated settings (regular schools and sharing time with other children without disabilities), mainstream segregated settings (special classes in regular schools) and special schools. These contexts differ in teacher training, resources and contact with other children in ways that allow a comparison of the relative influence of these variables on parental perception. Overall parental satisfaction was high. However, parents in mainstream segregated settings were less satisfied than those in special schools. There were no differences between the satisfaction of parents with children in mainstream segregated and non-segregated settings. Results seem to indicate that it is resources and teacher training, rather than severity of the disorder, the classroom structure or fear of contact with other children without ASD, that determine positive parental perception.
C1 [Moreno, Javier; Aguilera-Jimenez, Antonio; Saldana, David] Univ Seville, Dev & Educ Psychol, Seville 41018, Spain.
RP Saldana, D (reprint author), Univ Seville, Dev & Educ Psychol, Avda Camilo Jose Cela S-N, Seville 41018, Spain.
RI Saldana, David/F-2067-2010
OI Saldana, David/0000-0002-4192-7924
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NR 23
TC 5
Z9 5
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD JUN
PY 2008
VL 43
IS 2
BP 162
EP 173
PG 12
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 296GW
UT WOS:000255532500003
ER
PT J
AU Boyd, BA
Conroy, MA
Asmus, JM
McKenney, ELW
Mancil, GR
AF Boyd, Brian A.
Conroy, Maureen A.
Asmus, Jennifer M.
McKenney, Elizabeth L. W.
Mancil, G. Richmond
TI Descriptive analysis of classroom setting events on the social behaviors
of children with autism spectrum disorder
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID PEER INTERACTIONS; YOUNG-CHILDREN; BRAIN ACTIVITY; RECOMMENDATIONS;
INTERVENTIONS; ANTECEDENTS; PRESCHOOL; STUDENTS; SYSTEM; SKILLS
AB Children with Autism Spectrum Disorder (ASD) are characterized by extreme deficits in social relatedness with same-age peers. The purpose of this descriptive study was to identify naturally occurring antecedent variables (i.e., setting events) in the classroom environments of children with ASD that promoted their engagement in peer-related social behaviors. Over a 12-week period, seven preschool-aged children were individually observed an average of 3.4 - 5.9 hours across the following classroom setting events: (a) contexts with varying peer group sizes, (b) contexts in which the adult or child directed the activities, and (c) contexts with varying levels of teacher engagement. Results based on a continuous, sequential behavioral coding system showed that for the majority of participants; small group sizes, child. directed activities, and limited teacher engagement most influenced the occurrence of target children's social behaviors. Implications of this study to practice are addressed.
C1 [Boyd, Brian A.] Univ N Carolina, Frank Porter Graham Child Dev Inst, Chapel Hill, NC 27599 USA.
[Conroy, Maureen A.] Virginia Commonwealth Univ, Richmond, VA 23284 USA.
[Asmus, Jennifer M.] Univ Wisconsin, Madison, WI 53706 USA.
[McKenney, Elizabeth L. W.] Univ Florida, Gainesville, FL 32611 USA.
[Mancil, G. Richmond] Univ Cent Florida, Orlando, FL 32816 USA.
RP Boyd, BA (reprint author), Univ N Carolina, Frank Porter Graham Child Dev Inst, 105 Smith Level Rd,Campus Box 8180, Chapel Hill, NC 27599 USA.
EM boyd@mail.fpg.unc.edu
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NR 38
TC 10
Z9 10
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD JUN
PY 2008
VL 43
IS 2
BP 186
EP 197
PG 12
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 296GW
UT WOS:000255532500005
ER
PT J
AU Beck, AR
Stoner, JB
Bock, SJ
Parton, T
AF Beck, Ann R.
Stoner, Julia B.
Bock, Stacey J.
Parton, Tom
TI Comparison of PECS and the use of a VOCA: A replication
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID EXCHANGE COMMUNICATION-SYSTEM; YOUNG-CHILDREN; AUTISM; ACQUISITION;
SPEECH
AB This study compares use of the Picture Exchange Communication System (PECS) and a Voice Output Communication Aide (VOCA) with four preschool children who were either non-speaking or limited in their ability to speak and did not use an AAC system to communicate functionally. An alternating treatment single subject design was used to measure participants' preferences for each system and the verbalizations of the participants during system use. Results indicated that participants Learned PECS in a relatively short time period, preferences for one mode of communication are not predictable, and the influence of the communication systems on each panicipant's verbalizations varied.
C1 [Beck, Ann R.; Stoner, Julia B.; Bock, Stacey J.; Parton, Tom] Illinois State Univ, Coll Arts & Sci 4100, Normal, IL 61790 USA.
RP Beck, AR (reprint author), Illinois State Univ, Coll Arts & Sci 4100, Normal, IL 61790 USA.
EM arbeck@ilstu.edu
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NR 23
TC 23
Z9 23
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD JUN
PY 2008
VL 43
IS 2
BP 198
EP 216
PG 19
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 296GW
UT WOS:000255532500006
ER
PT J
AU Saugstad, LF
AF Saugstad, Letten F.
TI What is a psychosis and where is it located?
SO EUROPEAN ARCHIVES OF PSYCHIATRY AND CLINICAL NEUROSCIENCE
LA English
DT Article
DE psychosis; medial frontal lobe system; Autism; regressive events
ID SCHIZOPHRENIA
AB Kraepelin's dichotomy, manic-depressive insanity and dementia praecox, are contrasting and true endogenous disease entities which affect excitability, the fundamental property of the CNS. Kraepelin wanted to establish a valid classification and hit the extremes in brain structure and function at a time when we had no knowledge of brain dysfunction in "functional'' psychoses. The aetiology is now known: the psychoses are part of human growth and maturation and might be classified according to their brain dysfunction, which is exactly what Kraepelin wanted. However, presumably to reduce the stigma attached to the word "psychosis'', there is currently a strong initiative to eliminate the concept. But knowledge of what is happening in the brain in a psychosis might be more helpful in reducing stigma. It is suggested that psychosis is due to an affection of the supplementary motor area ( SMA), located at the centre of the Medial Frontal Lobe network. The SMA is one of the rare universally connected areas of the brain, as should be the case for such a key structure that makes decisions as to the right moment for action. This important network, which partly has continuous neurogenesis, has sufficiently widespread connections. The SMA, a premotor area located on the medial side of the frontal lobes, is one of the last regions to reach a concurrence of synaptogenesis. An affection of the SMA, a deficient or abolished Delayed Response Task, seriously disturbs our relation and adaptation to the surroundings. We usually master the Delayed Response Task around the age of 7 months, a time at which the second CNS regressive event takes place, which proceeds from the posterior to the anterior of the brain. In very late maturation, a persistent affection of the SMA might occur. We experience a chronic psychosis: infantile autism (IA), a chronic inability to act consciously, which contrasts with the episodic SMA affection post-puberty, when excitation is reduced due to excessive pruning of excitatory synapses. Silent spots are the result of insufficient fill-in mechanisms following a breakdown of circuitry. They may affect the SMA in the case of very late puberty. An acute reduction in excitation and concomitantly a marked increase in silent spots might lead to an acute psychosis. A frontal preference is likely, given that a reduction might occur anywhere in the cortex, but particularly in the areas maturing latest. The varying localisations probably explain the difficulty in accepting schizophrenia as a disease entity. The multifactorial inheritance of the dichotomy implies that the genetics are not fate, a psychotic development might be prevented given enough epigenetic factors: brain food ( omega 3). Might the present dietary adversity, with its lack of brain food, be responsible for a rising incidence in psychosis? A psychosis is an understandable and preventable dysfunction of the brain, and its mechanisms are known. Primarily a disorder of reduced excitation in an attenuated CNS, this explains why all the neuroleptics are convulsants, raising excitation, in contrast to all antidepressives, which are anti-epileptic.
C1 [Saugstad, Letten F.] Letten Fdn, N-0257 Oslo, Norway.
[Saugstad, Letten F.] Univ Oslo, Inst Neurosci, Oslo, Norway.
RP Saugstad, LF (reprint author), Letten Fdn, Behrensgate 5, N-0257 Oslo, Norway.
EM kari_horn@hotmail.com
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NR 31
TC 8
Z9 10
PU DR DIETRICH STEINKOPFF VERLAG
PI DARMSTADT
PA PO BOX 10 04 62, D-64204 DARMSTADT, GERMANY
SN 0940-1334
J9 EUR ARCH PSY CLIN N
JI Eur. Arch. Psych. Clin. Neurosci.
PD JUN
PY 2008
VL 258
SU 2
BP 111
EP 117
DI 10.1007/s00406-008-2014-1
PG 7
WC Clinical Neurology; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 316RI
UT WOS:000256967000015
PM 18516523
ER
PT J
AU Keen, DV
AF Keen, Daphne V.
TI Childhood autism, feeding problems and failure to thrive in early
infancy - Seven case studies
SO EUROPEAN CHILD & ADOLESCENT PSYCHIATRY
LA English
DT Article
DE autism; feeding problems; failure to thrive
ID LOW BODY-WEIGHT; NON-ORGANIC FAILURE; ANOREXIA-NERVOSA;
ASPERGERS-DISORDER; INFANTILE-AUTISM; YOUNG-CHILDREN; MASS INDEX;
ADOLESCENTS; POPULATION; GROWTH
AB Despite longstanding clinical experience of unusual feeding difficulties in children with autism, there is no published literature describing their association with early onset FTT. This paper examines literature that may link feeding problems and abnormal growth with developmental and psychiatric conditions and describes seven cases of children with autism, who showed growth failure caused by severe feeding problems starting in the first year of life. Inadequacies in existing classifications systems are highlighted. The presence of severe or atypical feeding problems and FTT in infancy should alert professionals to possible underlying ASD. The aetiology of feeding disorders in autism appears to involve an unusually complex interactional model with biological vulnerabilities due to dysfunction in sensory, cognitive and emotional response interacting with dysfunctional attachment and learnt behaviours to produce a severe and intractable problem. Effective treatment therefore requires a novel multifaceted approach that can address each of these areas.
C1 St George Hosp, St Georges Healthcare NHS Trust, London SW17 0QT, England.
RP Keen, DV (reprint author), St George Hosp, St Georges Healthcare NHS Trust, Rm 2 35,2nd Gloor Clare House,Blackshaw Rd, London SW17 0QT, England.
EM daphne.keen@stgeorges.nhs.uk
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NR 43
TC 17
Z9 17
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1018-8827
J9 EUR CHILD ADOLES PSY
JI Eur. Child Adolesc. Psych.
PD JUN
PY 2008
VL 17
IS 4
BP 209
EP 216
DI 10.1007/s00787-007-0655-7
PG 8
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 292GO
UT WOS:000255254700003
PM 17876499
ER
PT J
AU Garber, KB
Visootsak, J
Warren, ST
AF Garber, Kathryn B.
Visootsak, Jeannie
Warren, Stephen T.
TI Fragile X syndrome
SO EUROPEAN JOURNAL OF HUMAN GENETICS
LA English
DT Article
DE fragile X syndrome; FMR1; permutation; full mutation; autism
ID FMR-I LOCUS; MENTAL-RETARDATION; CGG REPEAT; TREMOR/ATAXIA SYNDROME;
FULL MUTATION; PREMUTATION; AUTISM; MALES; DISORDERS; BEHAVIOR
AB Fragile X syndrome, an X-linked dominant disorder with reduced penetrance, is associated with intellectual and emotional disabilities ranging from learning problems to mental retardation, and mood instability to autism. It is most often caused by the transcriptional silencing of the FMR1 gene, due to an expansion of a CGG repeat found in the 5'-untranslated region. The FMR1 gene product, FMRP, is a selective RNA-binding protein that negatively regulates local protein synthesis in neuronal dendrites. In its absence, the transcripts normally regulated by FMRP are over translated. The resulting over abundance of certain proteins results in reduced synaptic strength due to AMPA receptor trafficking abnormalities that lead, at least in part, to the fragile X phenotype.
C1 [Garber, Kathryn B.; Visootsak, Jeannie; Warren, Stephen T.] Emory Univ, Sch Med, Dept Human Genet, Atlanta, GA 30322 USA.
RP Warren, ST (reprint author), Emory Univ, Sch Med, Dept Human Genet, 615 Michael St,Room 301 Whitehead Bldg, Atlanta, GA 30322 USA.
EM swarren@emory.edu
RI Warren, Stephen/A-2498-2012
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NR 50
TC 121
Z9 122
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1018-4813
J9 EUR J HUM GENET
JI Eur. J. Hum. Genet.
PD JUN
PY 2008
VL 16
IS 6
BP 666
EP 672
DI 10.1038/ejhg.2008.61
PG 7
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 304MN
UT WOS:000256113700003
PM 18398441
ER
PT J
AU Floris, C
Rassu, S
Boccone, L
Gasperini, D
Cao, A
Crisponi, L
AF Floris, Chiara
Rassu, Stefania
Boccone, Loredana
Gasperini, Daniela
Cao, Antonio
Crisponi, Laura
TI Two patients with balanced translocations and autistic disorder: CSMD3
as a candidate gene for autism found in their common 8q23 breakpoint
area
SO EUROPEAN JOURNAL OF HUMAN GENETICS
LA English
DT Article
DE autistic spectrum disorder; balanced translocation; FISH; 8q23; CSMD3;
position effect
ID FAMILIAL MYOCLONIC EPILEPSY; CHROMOSOME 8Q23.3-Q24.1; IDENTIFICATION;
ASSOCIATION; LOCALIZATION; VARIANTS; PROTEIN; MEF2C; MAP
AB Recent studies estimated a rate of 3-5% of cytogenetic abnormalities involving many different chromosomes in autistic spectrum disorders (ASDs). Here, we report on two unrelated male patients with de novo translocations, autistic behaviour and psychomotor delay. These two patients carry a balanced chromosome translocation t(5; 8)(q14.3; q23.3) and t(6; 8)(q13; q23.2), respectively. A detailed physical map covering the regions involved in the translocations was constructed using BAC clones mapping on chromosomes 5q14.3, 6q13 and 8q23. Fluorescence in situ hybridisation (FISH) analyses were carried out using these genomic clones. We fine mapped the two translocation breakpoints on chromosomes 8 identifying their position within a short 5 Mb genomic region. Breakpoints on chromosomes 8 in both patients do not interrupt any known gene but both map in a region containing the CSMD3 gene, which thereby can be considered as a candidate for ASDs.
C1 [Crisponi, Laura] Cittadella Univ Monserrato, INN, CNR, I-09042 Cagliari, Italy.
[Rassu, Stefania] Univ Cagliari, Dipartimento Sci Biomed & Biotecnol, Cagliari, Italy.
[Boccone, Loredana; Gasperini, Daniela] Osped Reg Microcitemie, Clin Pediat 2, Cagliari, Italy.
RP Crisponi, L (reprint author), Cittadella Univ Monserrato, INN, CNR, I-09042 Cagliari, Italy.
EM laura.crisponi@inn.cnr.it
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NR 38
TC 12
Z9 16
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1018-4813
J9 EUR J HUM GENET
JI Eur. J. Hum. Genet.
PD JUN
PY 2008
VL 16
IS 6
BP 696
EP 704
DI 10.1038/ejhg.2008.7
PG 9
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 304MN
UT WOS:000256113700007
PM 18270536
ER
PT J
AU Young, DJ
Bebbington, A
Anderson, A
Ravine, D
Ellaway, C
Kulkarni, A
de Klerk, N
Kaufmann, WE
Leonard, H
AF Young, Deidra J.
Bebbington, Ami
Anderson, Alison
Ravine, David
Ellaway, Carolyn
Kulkarni, Alpana
de Klerk, Nick
Kaufmann, Walter E.
Leonard, Helen
TI The diagnosis of autism in a female: could it be Rett syndrome?
SO EUROPEAN JOURNAL OF PEDIATRICS
LA English
DT Article
DE MECP2; Rett syndrome; autistic spectrum disorders; autism
ID SEVERE MENTAL-RETARDATION; CPG BINDING-PROTEIN; MECP2 MUTATIONS;
DIFFERENTIAL-DIAGNOSIS; PRESERVED SPEECH; SYNDROME PHENOTYPE; DISORDERS;
GIRLS; GENE; SPECTRUM
AB The overlap between autism and Rett syndrome clinical features has led to many cases of Rett syndrome being initially diagnosed with infantile autism or as having some autistic features. Both conditions seriously disrupt social and language development and are often accompanied by repetitive, nonpurposeful stereotypic hand movements. The aims of this study were to compare the early and subsequent clinical courses of female subjects with Rett syndrome categorised by whether or not a diagnosis of autism had been proposed before Rett syndrome had been diagnosed and compare the spectrum of methyl-CpG binding protein 2 (MECP2) mutations identified among the two groups. This study made use of a total of 313 cases recorded in two databases: the Australian Rett Syndrome Database (ARSD) and the International Rett Syndrome Phenotype Database (InterRett). Cases with an initial diagnosis of autism had significantly milder Rett syndrome symptoms and were more likely to remain ambulant, to have some functional hand use and not to have developed a scoliosis. Females with the p.R306C or p.T158M mutations in the MECP2 gene were more likely to have an initial diagnosis of autism, and the specific Rett syndrome symptoms were noted at a later age. We recommend that females who are initially considered to have autism be carefully monitored for the evolution of the signs and symptoms of Rett syndrome.
C1 [Young, Deidra J.] Telethon Inst Child Hlth Res, Perth, WA 6872, Australia.
[Kaufmann, Walter E.] Johns Hopkins Univ, Sch Med, Baltimore, MD USA.
[Kaufmann, Walter E.] Kennedy Krieger Inst, Baltimore, MD USA.
[Kulkarni, Alpana] Princess Margaret Hosp Children, Perth, WA, Australia.
[Young, Deidra J.; Bebbington, Ami; Anderson, Alison; de Klerk, Nick; Leonard, Helen] Univ Western Australia, Telethon Inst Child Hlth Res, Ctr Child Hlth Res, Perth, WA 6009, Australia.
[Ravine, David] Univ Western Australia, Med Res Ctr, Western Australian Inst Med Res, Perth, WA 6009, Australia.
[Ravine, David] Univ Western Australia, Sch Med & Pharmacol, Perth, WA 6009, Australia.
[Ellaway, Carolyn] Univ Sydney, Sch Paediat & Child Hlth, Sydney, NSW 2006, Australia.
[Ellaway, Carolyn] Childrens Hosp Westmead, Sydney, NSW, Australia.
RP Young, DJ (reprint author), Telethon Inst Child Hlth Res, POB 855, Perth, WA 6872, Australia.
EM Deidray@ichr.uwa.edu.au
RI Anderson, Alison/J-6786-2014; Leonard, Helen/A-1010-2013
OI Anderson, Alison/0000-0002-1490-2262; Leonard, Helen/0000-0001-6405-5834
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NR 54
TC 26
Z9 27
PU SPRINGER
PI NEW YORK
PA 233 SPRING STREET, NEW YORK, NY 10013 USA
SN 0340-6199
J9 EUR J PEDIATR
JI Eur. J. Pediatr.
PD JUN
PY 2008
VL 167
IS 6
BP 661
EP 669
DI 10.1007/s00431-007-0569-x
PG 9
WC Pediatrics
SC Pediatrics
GA 285VN
UT WOS:000254804400009
PM 17684768
ER
PT J
AU Nettle, D
Liddle, B
AF Nettle, Daniel
Liddle, Bethany
TI Agreeableness is related to social-cognitive, but not social-perceptual,
theory of mind
SO EUROPEAN JOURNAL OF PERSONALITY
LA English
DT Article
DE theory of mind; empathy; empathising; Agreeableness; five-factor model
ID HIGH-FUNCTIONING AUTISM; PERSONALITY-TRAITS; PERSPECTIVE-TAKING; GENDER
DIFFERENCES; ASPERGER-SYNDROME; SEX-DIFFERENCES; PSYCHOPATHY;
MOTIVATION; EVOLUTION; ACCURACY
AB We hypothesise on a number of grounds that the personality dimension of Agreeableness may be associated with inter-individual differences in theory of mind (ToM) functioning. However it is important to distinguish social-perceptual from social-cognitive ToM. Previous findings on ToM in psychopathic individuals, sex differences in ToM and the associations between ToM and social relationships, all suggest that social-cognitive ToM is more likely than social-perceptual ToM to relate to Agreeableness. In separate empirical studies, we find that Agreeableness is substantially correlated with social-cognitive ToM performance, but uncorrelated with social-perceptual ToM performance. We suggest that the propensity or motivation to attend to the mental states of others may be central to the personality dimension of Agreeableness. Copyright (C) 2008 John Wiley & Sons, Ltd.
C1 [Nettle, Daniel; Liddle, Bethany] Univ Newcastle, Ctr Behaviour & Evolut, Newcastle Upon Tyne NE2 4HH, Tyne & Wear, England.
RP Nettle, D (reprint author), Univ Newcastle, Ctr Behaviour & Evolut, Henry Wellcome Bldg,Framlington Pl, Newcastle Upon Tyne NE2 4HH, Tyne & Wear, England.
EM daniel.nettle@ncl.ac.uk
RI Nettle, Daniel/B-2259-2008
OI Nettle, Daniel/0000-0001-9089-2599
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NR 43
TC 22
Z9 24
PU JOHN WILEY & SONS LTD
PI CHICHESTER
PA THE ATRIUM, SOUTHERN GATE, CHICHESTER PO19 8SQ, W SUSSEX, ENGLAND
SN 0890-2070
J9 EUR J PERSONALITY
JI Eur. J. Personal.
PD JUN
PY 2008
VL 22
IS 4
BP 323
EP 335
DI 10.1002/per.672
PG 13
WC Psychology, Social
SC Psychology
GA 327ZB
UT WOS:000257765900003
ER
PT J
AU Stanfield, AC
McIntosh, AM
Spencer, MD
Philip, R
Gaur, S
Lawrie, SM
AF Stanfield, Andrew C.
McIntosh, Andrew M.
Spencer, Michael D.
Philip, Ruth
Gaur, Sonia
Lawrie, Stephen M.
TI Towards a neuroanatomy of autism: A systematic review and meta-analysis
of structural magnetic resonance imaging studies
SO EUROPEAN PSYCHIATRY
LA English
DT Review
DE autism; MRI; meta-analysis
ID PERVASIVE DEVELOPMENTAL DISORDERS; POSTERIOR-FOSSA STRUCTURES;
INFANTILE-AUTISM; SPECTRUM DISORDERS; BRAIN SIZE; CORPUS-CALLOSUM; BASAL
GANGLIA; DIAGNOSTIC INSTRUMENTS; PRESCHOOL-CHILDREN; HEAD CIRCUMFERENCE
AB Background. - Structural brain abnormalities have been described in autism but studies are often small and contradictory. We aimed to identify which brain regions can reliably be regarded as different in autism compared to healthy controls.
Method. - A systematic search was conducted for magnetic resonance imaging studies of regional brain size in autism. Data were extracted and combined using random effects meta-analysis. The modifying effects of age and IQ were investigated using meta-regression.
Results. - The total brain, cerebral hemispheres, cerebellum and caudate nucleus were increased in volume, whereas the corpus callosurn area was reduced. There was evidence for a modifying effect of age and IQ on the cerebellar vermal lobules VI-VII and for age on the arnygdala.
Conclusions. - Autism may result from abnormalities in specific brain regions and a global lack of integration due to brain enlargement. Inconsistencies in the literature partly relate to differences in the age and IQ of study populations. Some regions may show abnormal growth trajectories. (c) 2007 Elsevier Masson SAS. All rights reserved.
C1 [Stanfield, Andrew C.; McIntosh, Andrew M.; Spencer, Michael D.; Philip, Ruth; Lawrie, Stephen M.] Univ Edinburgh, Royal Edinburgh Hosp, Sch Mol & Clin Med, Div Psychiat, Edinburgh EH10 5HF, Midlothian, Scotland.
RP Stanfield, AC (reprint author), Univ Edinburgh, Royal Edinburgh Hosp, Sch Mol & Clin Med, Div Psychiat, Edinburgh EH10 5HF, Midlothian, Scotland.
EM andrew.stanfield@cd.ac.uk
RI McIntosh, Andrew/B-9379-2008
OI McIntosh, Andrew/0000-0002-0198-4588
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NR 83
TC 158
Z9 162
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD JUN
PY 2008
VL 23
IS 4
BP 289
EP 299
DI 10.1016/j.eurpsy.2007.05.006
PG 11
WC Psychiatry
SC Psychiatry
GA 330CH
UT WOS:000257916400009
PM 17765485
ER
PT J
AU Yurgelun-Todd, DA
Rogowska, J
Gruber, SA
Bogorodzki, P
Simpson, NS
Irvin, RW
Jauregui, KA
Strong, RA
Rusche, JR
AF Yurgelun-Todd, Deborah A.
Rogowska, Jadwiga
Gruber, Staci A.
Bogorodzki, Piotr
Simpson, Norah S.
Irvin, Robert W.
Jauregui, Karen A.
Strong, Richard A.
Rusche, James R.
TI Increased amygdala fMRI activation after secretin administration
SO EXPERIMENTAL AND CLINICAL PSYCHOPHARMACOLOGY
LA English
DT Article
DE amygdala; fMRI secretin; affect; social behavior
ID FACIAL EXPRESSIONS; AUTISM; RECOGNITION; SYSTEM; INTELLIGENCE;
CEREBELLUM; RESPONSES; CHILDREN; FEARFUL; BRAIN
AB It has recently been reported that secretin activates gene expression in the central nucleus Of the amygdala in rats. To examine the neurophysiological effects of secretin on amygdalar activation in humans, the authors measured Blood Oxygen Level Dependent functional magnetic resonance imaging signal change during facial affect processing in a placebo-controlled double-blind study. The authors studied 12 healthy male subjects who were presented with three stimulus conditions: viewing happy, fearful, and neutral faces, before and after infusion with either secretin or placebo. To test whether treatment was associated with distinct patterns of activation, the two conditions (Pre and Post) were subjected to a subtraction analyses in SPM99 and hypotheses regarding the activation of the left and right amygdala were tested using a region-of-interest approach. Subtraction of treatment minus baseline activation during the fear condition yielded significant (p=.001) activation in the right amygdala and a nonsignificant increase in activation in the left amygdala. No significant differences were seen between the treatment conditions for the amygdala when viewing happy or neutral faces. These preliminary findings indicate that secretin may after responsivity to affective stimuli. The presence of increased activation of the amygdala during the viewing of fearful faces is consistent with findings from animal studies and suggests a mechanism by which secretin may modulate social behavior.
C1 [Yurgelun-Todd, Deborah A.; Rogowska, Jadwiga; Gruber, Staci A.; Bogorodzki, Piotr; Simpson, Norah S.; Irvin, Robert W.] Harvard Univ, McLean Hosp, Sch Med, Cognit Neuroimaging Lab, Belmont, MA 02478 USA.
RP Yurgelun-Todd, DA (reprint author), Harvard Univ, McLean Hosp, Sch Med, Cognit Neuroimaging Lab, 115 Mill St, Belmont, MA 02478 USA.
EM ytodd@mclean.harvard.edu
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NR 46
TC 3
Z9 3
PU AMER PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST NE, WASHINGTON, DC 20002-4242 USA
SN 1064-1297
J9 EXP CLIN PSYCHOPHARM
JI Exp. Clin. Psychopharmacol.
PD JUN
PY 2008
VL 16
IS 3
BP 191
EP 198
DI 10.1037/1064-1297.16.3.191
PG 8
WC Psychology, Biological; Psychology, Clinical; Pharmacology & Pharmacy;
Psychiatry
SC Psychology; Pharmacology & Pharmacy; Psychiatry
GA 308HE
UT WOS:000256378300001
PM 18540778
ER
PT J
AU Russo, N
Larson, C
Kraus, N
AF Russo, Nicole
Larson, Charles
Kraus, Nina
TI Audio-vocal system regulation in children with autism spectrum disorders
SO EXPERIMENTAL BRAIN RESEARCH
LA English
DT Article
DE autism; vocal production; auditory feedback
ID EVENT-RELATED POTENTIALS; HIGH-FUNCTIONING AUTISM; BRAIN-STEM RESPONSES;
COCHLEAR IMPLANT; AUDITORY-FEEDBACK; ASPERGER-SYNDROME;
FUNDAMENTAL-FREQUENCY; SPEECH SOUNDS; VOICE F-0; LANGUAGE DISORDERS
AB Do children with autism spectrum disorders (ASD) respond similarly to perturbations in auditory feedback as typically developing (TD) children? Presentation of pitch-shifted voice auditory feedback to vocalizing participants reveals a close coupling between the processing of auditory feedback and vocal motor control. This paradigm was used to test the hypothesis that abnormalities in the audio-vocal system would negatively impact ASD compensatory responses to perturbed auditory feedback. Voice fundamental frequency (F-0) was measured while children produced an /a/ sound into a microphone. The voice signal was fed back to the subjects in real time through headphones. During production, the feedback was pitch shifted (-100 cents, 200 ms) at random intervals for 80 trials. Averaged voice F-0 responses to pitch-shifted stimuli were calculated and correlated with both mental and language abilities as tested via standardized tests. A subset of children with ASD produced larger responses to perturbed auditory feedback than TD children, while the other children with ASD produced significantly lower response magnitudes. Furthermore, robust relationships between language ability, response magnitude and time of peak magnitude were identified. Because auditory feedback helps to stabilize voice F-0 (a major acoustic cue of prosody) and individuals with ASD have problems with prosody, this study identified potential mechanisms of dysfunction in the audio-vocal system for voice pitch regulation in some children with ASD. Objectively quantifying this deficit may inform both the assessment of a subgroup of ASD children with prosody deficits, as well as remediation strategies that incorporate pitch training.
C1 [Russo, Nicole; Larson, Charles; Kraus, Nina] Northwestern Univ, Roxelyn & Richard Paper Dept Commun Sci, Evanston, IL 60208 USA.
[Russo, Nicole; Larson, Charles; Kraus, Nina] Northwestern Univ, Interdeptartmental Neurosci Program, Evanston, IL 60208 USA.
[Kraus, Nina] Northwestern Univ, Dept Neurobiol & Physiol, Evanston, IL 60208 USA.
[Larson, Charles; Kraus, Nina] Northwestern Univ, Dept Otolaryngol, Evanston, IL 60208 USA.
RP Russo, N (reprint author), Northwestern Univ, Roxelyn & Richard Paper Dept Commun Sci, Frances Searle Bldg,2240 Campus Dr, Evanston, IL 60208 USA.
EM n-russo@northwestern.edu
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PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0014-4819
J9 EXP BRAIN RES
JI Exp. Brain Res.
PD JUN
PY 2008
VL 188
IS 1
BP 111
EP 124
DI 10.1007/s00221-008-1348-2
PG 14
WC Neurosciences
SC Neurosciences & Neurology
GA 308ZO
UT WOS:000256429600010
PM 18347784
ER
PT J
AU Haubenreisser, O
Chiocchetti, A
Zhou, J
Oender, K
Bauer, J
Kellermann, J
Lottspeich, F
Klauck, S
Poustka, A
Hintner, H
Breitenbach, M
Breitenbach-Koller, H
AF Haubenreisser, O.
Chiocchetti, A.
Zhou, J.
Oender, K.
Bauer, J.
Kellermann, J.
Lottspeich, F.
Klauck, S.
Poustka, A.
Hintner, H.
Breitenbach, M.
Breitenbach-Koller, H.
TI What a difference a ribosomal protein makes: rpL10 in aging and autism
SO FEBS JOURNAL
LA English
DT Meeting Abstract
CT Joint Conference of the 33rd FEBS Congress/11th IUBMB Conference
CY JUN 28-JUL 03, 2008
CL Athens, GREECE
C1 [Haubenreisser, O.; Chiocchetti, A.; Zhou, J.; Oender, K.; Breitenbach, M.; Breitenbach-Koller, H.] Salzburg Univ, A-5020 Salzburg, Austria.
[Chiocchetti, A.; Klauck, S.; Poustka, A.] DKFZ, Heidelberg, Germany.
[Oender, K.; Bauer, J.; Hintner, H.] St Johanns Spital PMU Salzburg, Salzburg, Austria.
[Kellermann, J.; Lottspeich, F.] MPI Biochem, Munich, Germany.
NR 0
TC 0
Z9 0
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1742-464X
J9 FEBS J
JI FEBS J.
PD JUN
PY 2008
VL 275
SU 1
BP 72
EP 72
PG 1
WC Biochemistry & Molecular Biology
SC Biochemistry & Molecular Biology
GA 311XI
UT WOS:000256633300197
ER
PT J
AU Ganz, JB
Kaylor, M
Bourgeois, B
Hadden, K
AF Ganz, Jennifer B.
Kaylor, Maria
Bourgeois, Bethany
Hadden, Kathy
TI The Impact of Social Scripts and Visual Cues on Verbal Communication in
Three Children With Autism Spectrum Disorders
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism; scripts; verbal communication; visual strategies; conversation;
perseverative speech; unscripted speech
AB Social script and visual cue use with students with autism spectrum disorders (ASD) were examined. A multiple baseline design across activities with embedded withdrawal was used to measure student acquisition of verbal communication skills. Three children with ASD, two boys and one girl, were taught a series of scripts and were shown a "quiet" picture when they engaged in perseverative speech (e. g., repetitive phrases or words). The number of scripted statements increased during treatment, with reductions in perseverative speech for all three students. One student's unscripted statements increased during intervention. Analysis of percentage of nonoverlapping data indicated that the intervention was highly effective for scripted statements, ineffective for unscripted statements, and produced variable results for total communicative statements.
C1 [Ganz, Jennifer B.] Univ Texas San Antonio, Dept ILT, San Antonio, TX 78249 USA.
[Bourgeois, Bethany] Male Family Support Ctr, Buffalo, NY USA.
RP Ganz, JB (reprint author), Univ Texas San Antonio, Dept ILT, 1 UTSA Circle, San Antonio, TX 78249 USA.
EM jennifer.ganz@utsa.edu
CR Agosta E, 2004, INTERV SCH CLIN, V39, P276, DOI 10.1177/10534512040390050401
(APA) APA, 2000, DIAGN STAT MAN MENT
Baron-Cohen Simon, 2004, Pediatr Rehabil, V7, P73, DOI 10.1080/13638490310001654790
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Charlop-Christy M.H., 2003, EDUC TREAT CHILD, V26, P108
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Dooley P., 2001, J POSIT BEHAV INTERV, V3, P57, DOI 10.1177/109830070100300108
Goldstein H, 2002, J AUTISM DEV DISORD, V32, P373, DOI 10.1023/A:1020589821992
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Janzen J. E., 2003, UNDERSTANDING NATURE
Kazdin A. E., 1982, SINGLE CASE RES DESI
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Kuttler S., 1998, FOCUS AUTISM OTHER D, V13, P176, DOI DOI 10.1177/108835769801300306
Lorimer PA, 2002, J POSIT BEHAV INTERV, V4, P53, DOI 10.1177/109830070200400109
MACDUFF GS, 1993, J APPL BEHAV ANAL, V26, P89, DOI 10.1901/jaba.1993.26-89
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Morrison RS, 2002, J EARLY INTERVENTION, V25, P58, DOI 10.1177/105381510202500106
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Norris C, 1999, FOCUS AUTISM OTHER D, V14, P180, DOI DOI 10.1177/108835769901400307
Olive M. L., 2005, ED PSYCHOL, V25, P313, DOI DOI 10.1080/0144341042000301238
Rogers MF, 2001, INTERV SCH CLIN, V36, P310
Sarokoff RA, 2001, J APPL BEHAV ANAL, V34, P81, DOI 10.1901/jaba.2001.34-81
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Swaggart B. L., 1995, FOCUS AUTISTIC BEHAV, V10, P1
NR 31
TC 13
Z9 13
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD JUN
PY 2008
VL 23
IS 2
BP 79
EP 94
DI 10.1177/1088357607311447
PG 16
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10FZ
UT WOS:000207451200002
ER
PT J
AU Belfiore, PJ
Fritts, KM
Herman, BC
AF Belfiore, Phillip J.
Fritts, Kevin M.
Herman, Brian C.
TI The Role of Procedural Integrity Using Self-Monitoring to Enhance
Discrete Trial Instruction (DTI)
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE evidence-based practices; autism; auditory processing; autism spectrum
disorders; applied behavior analysis; personnel preparation
AB The purpose of this study was to examine the effects of staff video self-monitoring (SM) training on the accuracy of delivering Discrete Trial Instruction (DTI) to students enrolled in a classroom for children with autism. Staff were trained to self-monitor a five-step DTI trial: (a) delivery of discriminative stimulus, (b) wait time for student response, (c) response-specific feedback, (d) immediacy of specific feedback, and (e) latency before delivery of next discriminative stimulus. The dependent measure was the accuracy of completing the five-step trial. Prior to intervention, staff were trained to criteria using videotapes of their teaching in scoring their DTI delivery. A multiple baseline design across staff was used to evaluate the effectiveness of SM. Results showed that video self-monitoring and self-evaluation increased the accuracy of DTI and, more specifically, the accuracy of managing the intertrial interval.
C1 [Belfiore, Phillip J.] Mercyhurst Coll, Dept Educ, Erie, PA 16546 USA.
[Fritts, Kevin M.; Herman, Brian C.] Dr Gertrude Barber Natl Inst, Erie, PA USA.
RP Belfiore, PJ (reprint author), Mercyhurst Coll, Dept Educ, 501 E 38th St, Erie, PA 16546 USA.
EM pbelfiore@mercyhurst.edu
CR BARLOW DH, 1984, SINGLE SUBJECT EXPT
Belfiore P. J., 1998, SELF REGULATED LEARN, P184
CAUTILLI J, 2000, BEHAV ANAL TODAY, V1, P42
Ferraioli S., 2005, J EARLY INTENSIVE BE, V2, P224
Green G., 1996, BEHAV INTERVENTIONS
Iwata BA, 2000, J APPL BEHAV ANAL, V33, P181, DOI 10.1901/jaba.2000.33-181
Jacobson JW, 2000, BEHAV ANALYST, V23, P149
KOEGEL RL, 1980, J APPL BEHAV ANAL, V13, P91, DOI 10.1901/jaba.1980.13-91
LATIES VG, 1993, J APPL BEHAV ANAL, V26, P513, DOI 10.1901/jaba.1993.26-513
Lavie T, 2002, J APPL BEHAV ANAL, V35, P209, DOI 10.1901/jaba.2002.35-209
Leblanc M., 2005, EDUC TREAT CHILD, V28, P76
Lerman DC, 2004, SCHOOL PSYCHOL REV, V33, P510
Lovaas I. O, 2003, TEACHING INDIVIDUALS
McBride BJ, 2003, TOP EARLY CHILD SPEC, V23, P5, DOI 10.1177/027112140302300102
National Research Council, 2001, ED CHILDR AUT
NEEF NA, 1995, J APPL BEHAV ANAL, V28, P297, DOI 10.1901/jaba.1995.28-297
Odom S. L., 2003, FOCUS AUTISM OTHER D, V18, P166, DOI DOI 10.1177/10883576030180030401
PETERSON L, 1982, J APPL BEHAV ANAL, V15, P477, DOI 10.1901/jaba.1982.15-477
SARAKOFF RA, 2004, J APPL BEHAV ANAL, V37, P353
Scheuermann B., 2003, FOCUS AUTISM OTHER D, V18, P197, DOI DOI 10.1177/10883576030180030801
SCHOEN AA, 2003, J INSTRUCTIONAL PSYC, V30, P125
Schreibman L, 2000, J AUTISM DEV DISORD, V30, P373, DOI 10.1023/A:1005535120023
Smith T., 2001, FOCUS AUTISM OTHER D, V16, P86, DOI 10.1177/108835760101600204
NR 23
TC 8
Z9 8
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD JUN
PY 2008
VL 23
IS 2
BP 95
EP 102
DI 10.1177/1088357607311445
PG 8
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10FZ
UT WOS:000207451200003
ER
PT J
AU Bolton, J
Mayer, MD
AF Bolton, Janice
Mayer, Michele D.
TI Promoting the Generalization of Paraprofessional Discrete Trial Teaching
Skills
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism; staff training; generalization; general case instruction;
discrete trial instruction; autism spectrum disorders; personnel
preparation; applied behavior analysis
ID SEVERE DISABILITIES; YOUNG-CHILDREN; SUPPORT STAFF; IMPLEMENTATION;
INTERVENTION; FEEDBACK; PROGRAM; AUTISM; PEOPLE
AB This study investigated the effectiveness of a brief staff training procedure aimed at promoting the generalization of accurate implementation of discrete trial instruction from the training environment to the teaching environment. Three bachelor's-level paraprofessionals received classroom training using a training package that included didactic instruction, modeling, general case instruction, and practice with specific performance feedback. Participants were required to meet performance criteria of 98% accuracy or better to exit training. After successfully completing the training program, participants were followed into the treatment environment and shown to maintain a high level of treatment integrity in implementing varied discrete trial programs with children in early intervention and school-age services for periods of up to 23 weeks.
C1 [Bolton, Janice; Mayer, Michele D.] HMEA, Childrens & Transit Serv, Hudson, MA 01749 USA.
RP Bolton, J (reprint author), HMEA, Childrens & Transit Serv, 15 South St, Hudson, MA 01749 USA.
EM jbolton@hmea.org
CR ARCO L, 1990, BEHAV RESIDENTIAL TR, V5, P207, DOI 10.1002/bin.2360050307
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Cooper J., 1987, APPL BEHAV ANAL
DAY HM, 1989, J APPL BEHAV ANAL, V22, P223, DOI 10.1901/jaba.1989.22-223
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GREENWOOD CR, 1994, BEHAVIOR ANALYSIS IN EDUCATION, P213
HEWARD WL, 1994, BEHAV ANAL ED, P213
Hopkins B., 1989, BEHAV RESIDENTIAL TR, V4, P331, DOI 10.1002/bin.2360040405
IWATA BA, 1982, J APPL BEHAV ANAL, V15, P191, DOI 10.1901/jaba.1982.15-191
Lerman DC, 2004, SCHOOL PSYCHOL REV, V33, P510
MAYER MD, 2004, ENSURING TREATMENT I
MCBRIDE BJ, 2003, TOP EARLY CHILD SPEC, V2, P5
McDonough K. A., 1996, BEHAV INTERVENTION Y, P63
Mortenson BP, 1998, SCHOOL PSYCHOL REV, V27, P613
NEEF NA, 1990, J APPL BEHAV ANAL, V23, P447, DOI 10.1901/jaba.1990.23-447
NEEF NA, 1995, J APPL BEHAV ANAL, V28, P297, DOI 10.1901/jaba.1995.28-297
PAGE TJ, 1982, J APPL BEHAV ANAL, V15, P335, DOI 10.1901/jaba.1982.15-335
Parsons MB, 1996, RES DEV DISABIL, V17, P467, DOI 10.1016/S0891-4222(96)00031-5
PARSONS MB, 1995, J APPL BEHAV ANAL, V28, P317, DOI 10.1901/jaba.1995.28-317
Richards S. B., 1999, SINGLE SUBJECT RES A
Ryan C. S., 2005, BEHAV ANAL TODAY, V6, P1
Sarokoff RA, 2004, J APPL BEHAV ANAL, V37, P535, DOI 10.1901/jaba.2004.37-535
Schepis M. M., 2000, J POSIT BEHAV INTERV, V2, P170, DOI 10.11-77/109830070000200305
Schepis MM, 2001, J APPL BEHAV ANAL, V34, P313, DOI 10.1901/jaba.2001.34-313
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*US GOV ACC OFF, 2005, PUBL US GOV ACC OFF, P60
NR 28
TC 9
Z9 9
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD JUN
PY 2008
VL 23
IS 2
BP 103
EP 111
DI 10.1177/1088357608316269
PG 9
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10FZ
UT WOS:000207451200004
ER
PT J
AU Goodwin, MS
AF Goodwin, Matthew S.
TI Enhancing and Accelerating the Pace of Autism Research and Treatment The
Promise of Developing Innovative Technology
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Editorial Material
DE autism; innovative technology
C1 [Goodwin, Matthew S.] Groden Ctr Inc, Providence, RI USA.
RP Goodwin, MS (reprint author), 86 Mt Hope Ave, Providence, RI 02906 USA.
EM msgoodwin@earthlink.net
CR *AUT SOC AM, 2008, WHAT IS AUT
de la Cruz B., 2006, FOCUS AUTISM OTHER D, V21, P245, DOI 10.1177/10883576060210040601
Ganz ML, 2007, ARCH PEDIAT ADOL MED, V161, P343, DOI 10.1001/archpedi.161.4.343
Hofmann S. G., 2006, FOCUS AUTISM OTHER D, V21, P100, DOI DOI 10.1177/10883576060210020101
Kimball J. W., 2007, FOCUS AUTISM OTHER D, V22, P131, DOI 10.1177/10883576070220020501
Randell T, 2007, J AUTISM DEV DISORD, V37, P637, DOI 10.1007/s10803-006-0193-z
NR 6
TC 24
Z9 24
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD JUN
PY 2008
VL 23
IS 2
BP 125
EP 128
DI 10.1177/1088357608316678
PG 4
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10FZ
UT WOS:000207451200006
ER
PT J
AU Yoo, JH
Tarbox, J
Granpeesheh, D
AF Yoo, J. Helen
Tarbox, Jonathan
Granpeesheh, Doreen
TI Using stimulus fading to teach a young child with autism to ingest
wireless capsule endoscopy
SO GASTROINTESTINAL ENDOSCOPY
LA English
DT Editorial Material
C1 [Yoo, J. Helen] New York State Inst Basic Res Dev Disabil, Dept Psychol, Staten Isl, NY 10314 USA.
[Tarbox, Jonathan; Granpeesheh, Doreen] Ctr Autism & Related Disorders, Austin, TX USA.
RP Yoo, JH (reprint author), New York State Inst Basic Res Dev Disabil, Dept Psychol, 1050 Forest Hill Rd, Staten Isl, NY 10314 USA.
CR BABBITT RL, 1991, J DEV BEHAV PEDIATR, V12, P229
Barth BA, 2004, GASTROINTEST ENDOSC, V60, P818, DOI 10.1016/S0016-5107(04)02052-8
NR 2
TC 2
Z9 2
PU MOSBY-ELSEVIER
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0016-5107
J9 GASTROINTEST ENDOSC
JI Gastrointest. Endosc.
PD JUN
PY 2008
VL 67
IS 7
BP 1203
EP 1204
DI 10.1016/j.gie.2007.10.048
PG 2
WC Gastroenterology & Hepatology
SC Gastroenterology & Hepatology
GA 310GE
UT WOS:000256516100048
PM 18249403
ER
PT J
AU Schaefer, GB
Mendelsohn, NJ
AF Schaefer, G. B.
Mendelsohn, N. J.
TI Genetics evaluation for the etiologic diagnosis of autism spectrum
disorders (vol 10, pg 4, 2008)
SO GENETICS IN MEDICINE
LA English
DT Correction
CR Schaefer GB, 2008, GENET MED, V10, P4, DOI 10.1097/GIM.0b013e31815efdd7
NR 1
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 1098-3600
J9 GENET MED
JI Genet. Med.
PD JUN
PY 2008
VL 10
IS 6
BP 464
EP 464
PG 1
WC Genetics & Heredity
SC Genetics & Heredity
GA 316OP
UT WOS:000256959900015
ER
PT J
AU Schaefer, GB
Mendelsohn, NJ
AF Schaefer, G. B.
Mendelsohn, N. J.
CA Professional Practice Guidelines
TI Clinical genetics evaluation in identifying the etiology of autism
spectrum disorders (vol 10, pg 301, 2008)
SO GENETICS IN MEDICINE
LA English
DT Correction
CR Schaefer GB, 2008, GENET MED, V10, P301, DOI 10.1097/GIM.0b013e31816b5cc9
NR 1
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 1098-3600
J9 GENET MED
JI Genet. Med.
PD JUN
PY 2008
VL 10
IS 6
BP 464
EP 464
PG 1
WC Genetics & Heredity
SC Genetics & Heredity
GA 316OP
UT WOS:000256959900016
ER
PT J
AU Holburn, CS
AF Holburn, Charles Steven
TI Detrimental effects of overestimating the occurrence of autism
SO INTELLECTUAL AND DEVELOPMENTAL DISABILITIES
LA English
DT Editorial Material
ID SPECTRUM DISORDERS; DEVELOPMENTAL-DISABILITIES; MENTAL-RETARDATION;
PREVALENCE; EPIDEMIOLOGY; CHILDREN
C1 New York State Inst Basic Res Dev Disabil, Staten Isl, NY 10314 USA.
RP Holburn, CS (reprint author), New York State Inst Basic Res Dev Disabil, 1050 Forest Hill Rd, Staten Isl, NY 10314 USA.
EM holbursc@infionline.net
CR Braddock D, 2001, MENT RETARD DEV D R, V7, P115, DOI 10.1002/mrdd.1016
Brown W., 2002, FRAGILE X SYNDROME D, P110
Corbett BA, 2007, MOL PSYCHIATR, V12, P292, DOI 10.1038/sj.mp.4001943
Croen LA, 2002, J AUTISM DEV DISORD, V32, P207, DOI 10.1023/A:1015453830880
FOMBONNE E, 2005, 2007 2007 DISTINGUIS
Fombonne E, 1999, PSYCHOL MED, V29, P769, DOI 10.1017/S0033291799008508
Fombonne E, 2003, JAMA-J AM MED ASSOC, V289, P87, DOI 10.1001/jama.289.1.87
Ganz M.L., 2006, UNDERSTANDING AUTISM, P475
Geschwind DH, 2007, CURR OPIN NEUROBIOL, V17, P103, DOI 10.1016/j.conb.2007.01.009
Gillberg C., 2000, BIOL AUTISTIC SYNDRO
Grether JK, 2006, CLIN NEUROSCI RES, V6, P119, DOI 10.1016/j.cnr.2006.06.009
Gupta AR, 2007, BIOL PSYCHIAT, V61, P429, DOI 10.1016/j.biopsych.2006.06.020
Jacobson John W., 1998, BEHAV INTERVENT, V13, P202
Kanner L, 1943, NERV CHILD, V2, P217
Kanner L, 1944, J PEDIATR-US, V25, P211, DOI 10.1016/S0022-3476(44)80156-1
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MAURICE C, 1996, BEHAV INTERVENTION Y, P3
REHFELDT RA, 2004, DEV DISABILITIES ADV, P177
Shattuck PT, 2006, PEDIATRICS, V117, P1028, DOI 10.1542/peds.2005-1516
Shattuck PT, 2007, MENT RETARD DEV D R, V13, P129, DOI 10.1002/mrdd.20143
Sigman M., 1987, HDB AUTISM PERVASIVE, P103
Skellern C, 2005, J PAEDIATR CHILD H, V41, P407, DOI 10.1111/j.1440-1754.2005.00634.x
Talebizadeh Z, 2005, J AUTISM DEV DISORD, V35, P675, DOI 10.1007/s10803-005-0011-z
*US DEP HHS CDCP, 2007, CDC REL NEW DAT AUT
2007, CBS NEWS 1216
NR 25
TC 3
Z9 3
PU AMER ASSOC INTELLECTUAL DEVELOPMENTAL DISABILITIES
PI WASHINGTON
PA 444 N CAPITOL ST, NW STE 846, WASHINGTON, DC 20001-1512 USA
SN 1934-9491
J9 INTELLECT DEV DISAB
JI Intellect. Dev. Disabil.
PD JUN
PY 2008
VL 46
IS 3
BP 243
EP 246
DI 10.1352/2008.46:243-246
PG 4
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 327ZJ
UT WOS:000257766700009
PM 18578583
ER
PT J
AU Asberg, J
Dahlgren, SO
Dahlgren, SA
AF Asberg, Jakob
Dahlgren, Sven Olof
Dahlgren, Sandberg Annika
TI Is theory of mind; associated with reading skills among high-functioning
children with autism spectrum conditions?
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Asberg, Jakob; Dahlgren, Sandberg Annika] Univ Gothenburg, Dept Psychol, S-40020 Gothenburg, Sweden.
[Dahlgren, Sven Olof] Stockholm Habilitat Serv, Autismforum, Stockholm, Sweden.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 82
EP 82
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264300892
ER
PT J
AU Fischer, C
Probst, P
AF Fischer, Christian
Probst, Paul
TI Obsessive compulsive symptoms in Asperger's disorder and high
functioning autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Fischer, Christian; Probst, Paul] Univ Hamburg, Dept Psychol, Hamburg, Germany.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 94
EP 94
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264301037
ER
PT J
AU Koolaee, AK
AF Koolaee, Khodabakhshi Anahite
TI Mothers expressed emotion towards children with and without autism:
Study in Iran
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Koolaee, Khodabakhshi Anahite] Allamhe Tabatabaee Univ, Tehran, Iran.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 104
EP 104
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264301147
ER
PT J
AU Laine, F
Gepner, B
AF Laine, France
Gepner, Bruno
TI impact of the speed of visual and auditory stimuli on the perceptive and
comprehensive aspects of communication in children with autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Laine, France] Ctr Psycle, Aix En Provence, France.
[Gepner, Bruno] CNRS, Lab Parole & Langage, UMR 6057, Aix En Provence, France.
NR 0
TC 1
Z9 1
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 106
EP 106
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264301166
ER
PT J
AU Nagasaki, T
Maruyama, D
Nagasaki, Y
AF Nagasaki, Tsutomu
Maruyama, Daiki
Nagasaki, Yuko
TI Relationship of narrative and theory of mind in children with high
function autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Nagasaki, Tsutomu] Univ Tsukuba, Narashino, Chiba, Japan.
[Maruyama, Daiki] Kotoni Elementary Sch, Sapporo, Hokkaido, Japan.
[Nagasaki, Yuko] Dai Ni Jr High Sch, Narashino, Chiba, Japan.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 112
EP 112
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264301233
ER
PT J
AU Pouretemad, H
Mamaghaniah, M
Ahamadi, F
Khoshabi, K
AF Pouretemad, Hamid
Mamaghaniah, Maryam
Ahamadi, Fatamah
Khoshabi, Katayoun
TI The effect of picture exchange communication system on behavioral
problems of children with Autism spectrum Disorders
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Pouretemad, Hamid; Mamaghaniah, Maryam] Shahid Beheshti Univ, Tehran, Iran.
[Ahamadi, Fatamah] Ctr Treatment Au, PECS, Tehran, Iran.
[Khoshabi, Katayoun] Univ Social Welf, Tehran, Iran.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 115
EP 115
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264301270
ER
PT J
AU Malkoc, G
AF Malkoc, Gokhan
TI Local and global process of stimuli in autism using face
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Malkoc, Gokhan] Dogus Univ, Istanbul, Turkey.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 151
EP 151
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264301653
ER
PT J
AU Zhou, SJ
Yang, J
AF Zhou Shijie
Yang Juan
TI Theory of mind in children with autism spectrum disorders
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Zhou Shijie; Yang Juan] Cent S Univ, Xiangya Hosp 2, Changsha, Hunan, Peoples R China.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 167
EP 167
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264301825
ER
PT J
AU Lawson, C
AF Lawson, Christine
TI Successful innovations for young people with autism spectrum/disorders
(dispositions)
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Lawson, Christine] CAMHS, Dept Psychol, Isle Of Man, Southam, England.
NR 0
TC 2
Z9 2
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 241
EP 241
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264302683
ER
PT J
AU Muller, C
Nussbeck, S
AF Mueller, Christoph
Nussbeck, Susanne
TI Autism: Is there a spontaneous preference for details and not meaning?
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Nussbeck, Susanne] Univ Cologne, Dept Human Sci, Cologne, Germany.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 241
EP 241
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264302681
ER
PT J
AU Oakland, T
Woolf, S
AF Oakland, Thomas
Woolf, Steve
TI Adaptive behavior and skills of young children with autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Oakland, Thomas] Univ Florida, Dept Educ Psychol, Gainesville, FL USA.
[Woolf, Steve] Beacon Serv, Psychol Serv, Milford, MA USA.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 291
EP 291
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264303393
ER
PT J
AU Annen, Y
AF Annen, Yasumasa
TI Prisoner's Dilemma Games and the Autism-Spectrum Quotient
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Annen, Yasumasa] Aichi Mizuho Coll, Dept Human Sci, Toyota, Japan.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 304
EP 304
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264303534
ER
PT J
AU Probst, P
AF Probst, Paul
TI Outcomes of a social communication enhancement program for individuals
with autism within a combined one-to-one and small group setting
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 331
EP 331
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264303821
ER
PT J
AU Konrad, K
Kohls, G
Herpertz-Dahlmann, B
AF Konrad, Kerstin
Kohls, Gregor
Herpertz-Dahlmann, Beate
TI Typical development of cognitve control and abnormal development in ADHD
and autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Konrad, Kerstin; Kohls, Gregor; Herpertz-Dahlmann, Beate] Univ Klinikum Aachen, Klin Neuropsychol, Aachen, Germany.
RI Konrad, Kerstin/H-7747-2013
OI Konrad, Kerstin/0000-0001-9039-2615
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 358
EP 358
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264304247
ER
PT J
AU Fleming, M
Martin, A
AF Fleming, Mitchel
Martin, Aoife
TI Supporting the inclusion of children with autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Fleming, Mitchel; Martin, Aoife] St Paul Serv, Dublin, Ireland.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 379
EP 379
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264304492
ER
PT J
AU Leppert, T
Bagh, M
AF Leppert, Tobias
Bagh, Muna
TI Implementation of structured teaching methods in a classroom for pupils
with autism in the United Arab Emirates (Shajah Autism Center)
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Leppert, Tobias] Autism Treatment Ctr, Inst Klin Psychol, Hamburg, Germany.
[Bagh, Muna] Sharjah Autism Ctr, Inst Klin, Sharjah, U Arab Emirates.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 564
EP 564
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264306616
ER
PT J
AU Probst, P
Leppert, T
AF Probst, Paul
Leppert, Tobias
TI Outcomes of a parent group training and a teacher group training for
autism spectrum disorders in Germany
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Leppert, Tobias] Autism Treatment Ctr, Hamburg, Germany.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 564
EP 564
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264306615
ER
PT J
AU Probst, P
AF Probst, Paul
TI Interventions for autism spectrum disorders: Concepts and outcomes
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 564
EP 564
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264306612
ER
PT J
AU Schwartz, C
Bente, G
Vogeley, K
AF Schwartz, Caroline
Bente, Gary
Vogeley, Kai
TI Using virtual characters to study gaze perception in high-functioning
autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Bente, Gary] Univ Cologne, Dept Psychol, Cologne, Germany.
[Vogeley, Kai] Univ Cologne, Dept Psychiat, Cologne, Germany.
RI Vogeley, K/E-4860-2012
OI Vogeley, K/0000-0002-5891-5831
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 590
EP 590
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264306897
ER
PT J
AU Chano, J
Sarnrattana, U
Pinpradit, N
AF Chano, Jiraporn
Sarnrattana, Unchalee
Pinpradit, Neon
TI Reducing behavioral problems through a communicative instructional model
for preschool children with autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Chano, Jiraporn] Srinagarind Hosp, ENT 6, Speech Clin, Khon Kaen, Thailand.
[Sarnrattana, Unchalee; Pinpradit, Neon] Khon Kaen Univ, Khon Kaen, Thailand.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 592
EP 592
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264306918
ER
PT J
AU Krebs, J
Biswas, A
Pascalis, O
Schwarzer, G
AF Krebs, Julia
Biswas, Ajanta
Pascalis, Oliver
Schwarzer, Gudrun
TI Face processing in children with autism spectrum disorder: Independent
or interactive processing of facial identity and facial expression?
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Krebs, Julia] Univ Giessen, Inst Psychol, Giessen, Germany.
[Biswas, Ajanta; Pascalis, Oliver] Univ Sheffield, Sheffield, S Yorkshire, England.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 592
EP 592
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264306920
ER
PT J
AU Tang, JC
Lee, SH
Wang, MC
AF Tang Jung-Chang
Lee Shu-Hui
Wang Ming-Chua
TI The effects of pivotal response training on communicative behavior of
preschoolers with autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Lee Shu-Hui] Natl Kaohsiung Normal Uni, Chiayi, Taiwan.
[Wang Ming-Chua] Natl Taitung Univ, Chiayi, Taiwan.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 592
EP 592
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264306919
ER
PT J
AU Banerjee, M
AF Banerjee, Mallika
TI Effect of affective stimulation on cognitive-affective impairments of
autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Banerjee, Mallika] Univ Calcutta, Dept Psychol, Kolkata, India.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
EI 1464-066X
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 609
EP 609
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264307165
ER
PT J
AU Strid, K
Tjus, T
Heimann, M
AF Strid, Karin
Tjus, Tomas
Heimann, Mikael
TI Parents' verbal comments in relation to their child's diagnosis and
language level: Comparing children with Down syndrome, autism and
typical development
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Strid, Karin; Tjus, Tomas] Gothenburg Univ, Dept Psychol, Gothenburg, Sweden.
[Heimann, Mikael] Linkoping Univ, Dept Behav Sci, Linkoping, Sweden.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 698
EP 698
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264308233
ER
PT J
AU Roper, D
Sheppard, E
Mitchell, P
AF Roper, Danielle
Sheppard, Elizabeth
Mitchell, Peter
TI The impact of knowledge on copying and drawing accuracy in individuals
with autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Roper, Danielle; Sheppard, Elizabeth; Mitchell, Peter] Univ Nottingham, Dept Psychol, Beeston, England.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 716
EP 716
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264308427
ER
PT J
AU Pineda, J
AF Pineda, Jaime
TI Inducing neural plasticity in neurological rehabilitation:
Brain-computer interface use for the treatment of autism spectrum
disorders
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Pineda, Jaime] Univ Calif San Diego, Dept Cognit Sci, La Jolla, CA 92093 USA.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 724
EP 724
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264308530
ER
PT J
AU Martin, G
Arnal, L
Thiessen, C
Fazzio, D
Yu, D
AF Martin, Garry
Arnal, Lindsay
Thiessen, Carly
Fazzio, Daniela
Yu, Dickie
TI Evaluation of a self-instructional manual to teach instructors to
conduct discrete-trials teaching to children with autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Martin, Garry; Arnal, Lindsay; Thiessen, Carly; Fazzio, Daniela; Yu, Dickie] Univ Manitoba, St Pauls Coll, Winnipeg, MB, Canada.
RI Yu, C.T./D-1731-2014
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 794
EP 794
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264309399
ER
PT J
AU Prokofyev, A
Orekhova, E
Posikera, I
Morozov, A
Morozov, V
Obukhov, Y
Stroganova, T
AF Prokofyev, Andrey
Orekhova, Elena
Posikera, Irina
Morozov, Alexey
Morozov, Vladimir
Obukhov, Yuriy
Stroganova, Tatiana
TI Inverted event-related potentials response to illusory contour in boys
with autism
SO INTERNATIONAL JOURNAL OF PSYCHOLOGY
LA English
DT Meeting Abstract
C1 [Prokofyev, Andrey] MSUPE, Moscow, Russia.
[Orekhova, Elena] Sahlgrens Univ Hosp, Moscow, Sweden.
[Posikera, Irina] PIRAO, Moscow, Russia.
[Morozov, Alexey; Morozov, Vladimir; Obukhov, Yuriy] IREERAS, Lab 144, Moscow, Russia.
[Stroganova, Tatiana] PIRAE, Moscow, Russia.
NR 0
TC 0
Z9 0
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 0020-7594
J9 INT J PSYCHOL
JI Int. J. Psychol.
PD JUN-AUG
PY 2008
VL 43
IS 3-4
BP 803
EP 803
PG 1
WC Psychology, Multidisciplinary
SC Psychology
GA 349EO
UT WOS:000259264309498
ER
PT J
AU Suto, K
AF Suto, Kunihiko
TI Establishing helping behavior in a child with autism : Acquisition of
observing responses
SO JAPANESE JOURNAL OF EDUCATIONAL PSYCHOLOGY
LA Japanese
DT Article
DE helping behavior; observing response; verbal stimuli; environmental
stimuli; child with autism
ID DISORDER
AB Various discriminative stimuli set the occasion for helping behavior. In the present study, an 8-year-old boy with autism was trained to help a person who is confronted with some difficulty. The discriminative stimulus conditions in the present study were (1) a verbal stimulus condition : a person requests help from others, (2) a environmental stimulus condition a person needs a tool that others have, and (3) a compound stimulus condition : verbal stimuli and observing responses. As a result of the training, the boy acquired helping behavior in the presence of all 3 classes of discriminative stimuli. This result suggests that children with autism can understand not only verbal and environmental stimuli, but also their own observing responses, so that these various discriminative stimuli can be used to establish helping behavior.
C1 Iwaki Meisei Univ, Fac Humanities, Iwaki, Fukushima, Japan.
RP Suto, K (reprint author), Iwaki Meisei Univ, Fac Humanities, Iwaki, Fukushima, Japan.
EM hikokuni@rr.iij4u.or.jp
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NR 20
TC 1
Z9 1
PU JAPANESE ASSOC EDUCATIONAL PSYCHOLOGY
PI TOKYO
PA 5-24-6-7F HONGO, BUMKYO-KU, TOKYO, 113-0033, JAPAN
SN 0021-5015
J9 JPN J EDUC PSYCHOL
JI Jpn. J. Educ. Psychol.
PD JUN
PY 2008
VL 56
IS 2
BP 268
EP 277
PG 10
WC Psychology, Educational
SC Psychology
GA 326XJ
UT WOS:000257692500011
ER
PT J
AU Glover, AC
Roane, HS
Kadey, HJ
AF Glover, Ashley C.
Roane, Henry S.
Kadey, Heather J.
TI Preference for reinforcers under progressive and fixed-ratio schedules:
A comparison of single and concurrent arrangements
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; concurrent schedules; fixed ratio; mental retardation; positive
reinforcement; progressive ratio
ID STIMULUS PREFERENCE; ASSESSMENTS; REQUIREMENTS; DISABILITIES;
DISPLACEMENT; LEISURE; FOOD
AB Progressive-ratio (PR) schedules were used to identify the break point (i.e., the last schedule value completed) for 2 reinforcers under single and concurrent schedules. After the respective break points were established, the same reinforcers were presented under concurrent fixed-ratio (FR) schedules that were yoked to the break points obtained with the PR schedules. Results suggested that the participants responded more for the high-preference item than for the low-preference item, regardless of the presentation arrangement (single or concurrent presentations). This pattern of responding was maintained when the reinforcers were presented under dissimilar FR schedules. The results suggest that responding for differentially preferred stimuli may vary as a function of differences in schedule requirements.
C1 [Roane, Henry S.; Kadey, Heather J.] Munroe Meyer Inst, Ctr Autism Spectrum Disorders, Omaha, NE 68198 USA.
[Roane, Henry S.; Kadey, Heather J.] Univ Nebraska Med Ctr, Omaha, NE 68198 USA.
[Glover, Ashley C.] Georgia State Univ, Atlanta, GA 30303 USA.
RP Roane, HS (reprint author), Munroe Meyer Inst, Ctr Autism Spectrum Disorders, 985450 Nebraska Med Ctr, Omaha, NE 68198 USA.
EM hroane@unmc.edu
CR BARON A, 1992, J EXP ANAL BEHAV, V58, P377, DOI 10.1901/jeab.1992.58-377
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TUSTIN RD, 1994, J APPL BEHAV ANAL, V27, P597, DOI 10.1901/jaba.1994.27-597
NR 16
TC 16
Z9 16
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD SUM
PY 2008
VL 41
IS 2
BP 163
EP 176
DI 10.1901/jaba.2008.41-163
PG 14
WC Psychology, Clinical
SC Psychology
GA 309LL
UT WOS:000256461800002
PM 18595281
ER
PT J
AU Betz, A
Higbee, TS
Reagon, KA
AF Betz, Alison
Higbee, Thomas S.
Reagon, Kara A.
TI Using joint activity schedules to promote peer engagement in
preschoolers with autism
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE activity schedules; autism; peer play; social interaction
ID SCRIPT-FADING PROCEDURE; SOCIAL-INTERACTION SKILLS; TEACHING-CHILDREN
AB We assessed the use of a joint activity schedule to increase peer engagement for preschoolers with autism. We taught 3 dyads of preschoolers with autism to follow joint activity schedules that cued both members of the pair to play a sequence of interactive games together. Results indicated that joint activity schedules increased peer engagement and the number of games completed for all dyads. Schedule following was maintained without additional prompting when activities were resequenced and when new games were introduced for 2 of the 3 dyads.
C1 [Betz, Alison; Higbee, Thomas S.; Reagon, Kara A.] Utah State Univ, Logan, UT 84322 USA.
RP Higbee, TS (reprint author), Utah State Univ, 2865 Old Main Hill, Logan, UT 84322 USA.
EM tom.higbee@usu.edu
RI Higbee, Thomas/F-5157-2010
CR Krantz PJ, 1998, J APPL BEHAV ANAL, V31, P191, DOI 10.1901/jaba.1998.31-191
KRANTZ PJ, 1993, J APPL BEHAV ANAL, V26, P121, DOI 10.1901/jaba.1993.26-121
MACDUFF GS, 1993, J APPL BEHAV ANAL, V26, P89, DOI 10.1901/jaba.1993.26-89
McClannahan L. E., 1999, ACTIVITY SCHEDULES C
Stevenson CL, 2000, BEHAV INTERVENT, V15, P1, DOI 10.1002/(SICI)1099-078X(200001/03)15:1<1::AID-BIN41>3.0.CO;2-V
NR 5
TC 20
Z9 21
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD SUM
PY 2008
VL 41
IS 2
BP 237
EP 241
DI 10.1901/jaba.2008.41-237
PG 5
WC Psychology, Clinical
SC Psychology
GA 309LL
UT WOS:000256461800008
PM 18595287
ER
PT J
AU Lerman, DC
Tetreault, A
Hovanetz, A
Strobel, M
Garro, J
AF Lerman, Dorothea C.
Tetreault, Allison
Hovanetz, Alyson
Strobel, Margaret
Garro, Joanie
TI Further evaluation of a brief, intensive teacher-training model
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; direct teaching; generalization; preference assessment; teacher
training
AB The purpose of this study was to further evaluate the outcomes of a model program that was designed to train current teachers of children with autism. Nine certified special education teachers participating in an intensive 5-day summer training program were taught a relatively large number of specific skills in two areas (preference assessment and direct teaching). The teachers met the mastery criteria for all of the skills during the summer training. Follow-up observations up to 6 months after training suggested that the skills generalized to their classrooms and were maintained for most teachers with brief feedback only.
C1 [Lerman, Dorothea C.; Tetreault, Allison; Hovanetz, Alyson; Strobel, Margaret; Garro, Joanie] Univ Houston Clear Lake, Houston, TX 77058 USA.
RP Lerman, DC (reprint author), Univ Houston Clear Lake, 2700 Bay Area Blvd,Campus Box 245, Houston, TX 77058 USA.
EM lerman@uhcl.edu
CR Cooper J. O., 2007, APPL BEHAV ANAL
DeLeon IG, 1996, J APPL BEHAV ANAL, V29, P519, DOI 10.1901/jaba.1996.29-519
Lerman DC, 2004, SCHOOL PSYCHOL REV, V33, P510
National Research Council, 2001, ED CHILDR AUT
Noell GH, 2002, SCHOOL PSYCHOL REV, V31, P217
REID DH, 1990, HDB BEHAV MODIFICATI, P71
NR 6
TC 20
Z9 20
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD SUM
PY 2008
VL 41
IS 2
BP 243
EP 248
DI 10.1901/jaba.2008.41-243
PG 6
WC Psychology, Clinical
SC Psychology
GA 309LL
UT WOS:000256461800009
PM 18595288
ER
PT J
AU Athens, ES
Vollmer, TR
Sloman, KN
Pipkin, CS
AF Athens, Elizabeth S.
Vollmer, Timothy R.
Sloman, Kimberly N.
Pipkin, Claire St. Peter
TI An analysis of vocal stereotypy and therapyst fading
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; Down syndrome; response cost; treatment fading; vocal stereotypy
ID DEVELOPMENTAL-DISABILITIES; AUTOMATIC REINFORCEMENT; ABERRANT BEHAVIOR;
RESPONSE COST; PREFERENCE; CHILDREN; AUTISM
AB A functional analysis for a boy with Down syndrome and autism suggested that vocal stereotypy was maintained by automatic reinforcement. The analysis also showed that instructions and noncontingent attention suppressed vocal stereotypy. A treatment package consisting of noncontingent attention, contingent demands, and response cost effectively reduced vocal stereotypy. The treatment package remained effective even when noncontingent attention was removed, making the procedure easier to implement. Also, the presence of the therapist in the room with the participant was faded systematically. After completion of fading, vocal stereotypy remained low during conditions similar to the no-consequence phase of the functional analysis.
C1 [Vollmer, Timothy R.] Univ Florida, Dept Psychol, Gainesville, FL 32611 USA.
[Pipkin, Claire St. Peter] W Virginia Univ, Morgantown, WV 26506 USA.
RP Vollmer, TR (reprint author), Univ Florida, Dept Psychol, Gainesville, FL 32611 USA.
EM vollmera@ufl.edu
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BOSTOW DE, 1969, J APPL BEHAV ANAL, V2, P31, DOI 10.1901/jaba.1969.2-31
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IWATA BA, 1994, J APPL BEHAV ANAL, V27, P197, DOI 10.1901/jaba.1994.27-197
Keeney KM, 2000, J APPL BEHAV ANAL, V33, P255, DOI 10.1901/jaba.2000.33-255
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WAGAMAN JR, 1993, J APPL BEHAV ANAL, V26, P53, DOI 10.1901/jaba.1993.26-53
NR 13
TC 21
Z9 21
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD SUM
PY 2008
VL 41
IS 2
BP 291
EP 297
DI 10.1901/jaba.2008.41-291
PG 7
WC Psychology, Clinical
SC Psychology
GA 309LL
UT WOS:000256461800017
PM 18595296
ER
PT J
AU Aman, MG
Hollway, JA
McDougle, CJ
Scahill, L
Tierney, E
McCracken, JT
Arnold, LE
Vitiello, B
Ritz, L
Gavaletz, A
Cronin, P
Swiezy, N
Wheeler, C
Koenig, K
Ghuman, JK
Pose, DJ
AF Aman, Michael G.
Hollway, Jill A.
McDougle, Christopher J.
Scahill, Lawrence
Tierney, Elaine
McCracken, James T.
Arnold, L. Eugene
Vitiello, Benedetto
Ritz, Louise
Gavaletz, Allison
Cronin, Pegeen
Swiezy, Naomi
Wheeler, Courtney
Koenig, Kathleen
Ghuman, Jaswinder K.
Pose, David J.
TI Cognitive effects of risperidone in children with autism and irritable
behavior
SO JOURNAL OF CHILD AND ADOLESCENT PSYCHOPHARMACOLOGY
LA English
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDERS; SPATIAL WORKING-MEMORY;
SCHIZOPHRENIA; METHYLPHENIDATE; PERFORMANCE; ADOLESCENTS; CLOZAPINE
AB Objective: The objective of this research was to explore the effects of risperidone on cognitive processes in children with autism and irritable behavior.
Method: Thirty-eight children, ages 5-17 years with autism and severe behavioral disturbance, were randomly assigned to risperidone (0.5 to 3.5 mg/day) or placebo for 8 weeks. This sample of 38 was a subset of 101 subjects who participated in the clinical trial; 63 were unable to perform the cognitive tasks. A double-blind placebo-controlled parallel groups design was used. Dependent measures included tests of sustained attention, verbal learning, hand-eye coordination, and spatial memory assessed before, during, and after the 8-week treatment. Changes in performance were compared by repeated measures ANOVA.
Results: Twenty-nine boys and 9 girls with autism and severe behavioral disturbance and a mental age >= 18 months completed the cognitive part of the study. No decline in performance occurred with risperidone. Performance on a cancellation task (number of correct detections) and a verbal learning task (word recognition) was better on risperidone than on placebo (without correction for multiplicity). Equivocal improvement also occurred on a spatial memory task. There were no significant differences between treatment conditions on the Purdue Pegboard (hand-eye coordination) task or the Analog Classroom Task (timed math test).
Conclusion: Risperidone given to children with autism at doses up to 3.5 mg for up to 8 weeks appears to have no detrimental effect on cognitive performance.
C1 [Hollway, Jill A.] Ohio State Univ, UAP, Nisonger Ctr, Columbus, OH 43210 USA.
[McDougle, Christopher J.; Swiezy, Naomi; Pose, David J.] Indiana Univ, James Whitcomb Riley Hosp Children, Indianapolis, IN 46204 USA.
[Scahill, Lawrence; Gavaletz, Allison; Koenig, Kathleen; Pose, David J.] Yale Univ, Yale Child Study Ctr, New Haven, CT USA.
[Tierney, Elaine; Wheeler, Courtney; Ghuman, Jaswinder K.] Johns Hopkins Univ, Kennedy Krieger Inst, Baltimore, MD USA.
[McCracken, James T.; Cronin, Pegeen] Univ Calif Los Angeles, Inst Neuropsychiat, Dept Psychiat, Los Angeles, CA 90024 USA.
[Vitiello, Benedetto; Ritz, Louise] Univ Calif Los Angeles, Inst Neuropsychiat, Dept Psychiat, Los Angeles, CA 90024 USA.
RP Hollway, JA (reprint author), Ohio State Univ, UAP, Nisonger Ctr, 1581 Dodd Dr, Columbus, OH 43210 USA.
EM Jill.Hollway@osumc.edu
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Yoo JH, 2003, J APPL BEHAV ANAL, V36, P245, DOI 10.1901/jaba.2003.36-245
NR 32
TC 23
Z9 23
PU MARY ANN LIEBERT INC
PI NEW ROCHELLE
PA 140 HUGUENOT STREET, 3RD FL, NEW ROCHELLE, NY 10801 USA
SN 1044-5463
J9 J CHILD ADOL PSYCHOP
JI J. Child Adolesc. Psychopharmacol.
PD JUN
PY 2008
VL 18
IS 3
BP 227
EP 236
DI 10.1089/cap.2007.0133
PG 10
WC Pediatrics; Pharmacology & Pharmacy; Psychiatry
SC Pediatrics; Pharmacology & Pharmacy; Psychiatry
GA 326RI
UT WOS:000257676200001
PM 18582177
ER
PT J
AU Chiat, S
Roy, P
AF Chiat, Shula
Roy, Penny
TI Early phonological and sociocognitive skills as predictors of later
language and social communication outcomes
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE delayed language; early clinical predictors; repetition; sociocognition;
social communication; longitudinal
ID NONWORD REPETITION; YOUNG-CHILDREN; IMPAIRMENT; 4-YEAR-OLDS; DISORDER;
PATTERN; AUTISM; WORD
AB Background: Previous studies of outcome for children with early language delay have focused on measures of early language as predictors of language outcome. This study investigates whether very early processing skills (VEPS) known to underpin language development will be better predictors of specific language and social communication outcomes than measures of language itself. Method: Participants were 163 children referred to clinical services with concerns about language at 2;6-3;6 years and followed up at 4-5 years. Novel assessments of phonological and sociocognitive processing were administered at Time 1 (T1), together with a standardised test of receptive and expressive language, and parental report of expressive vocabulary. The language test was re-administered at Time 2 (T2), together with assessments of morphosyntax and parental reports of social communication. Results: Intercorrelations at and between T1 and T2 were high, and dissociations were rare. Ordinal regressions were run, entering predictors singly and simultaneously. With the exception of the phonological task, every early measure on its own was significantly predictive of most outcomes, and receptive language was the strongest all-round predictor. Results of simultaneous entry, controlling for the effect of other predictors, showed that early language was the strongest predictor of general language outcome, but early phonology was the strongest predictor of a measure of morphosyntax, and early sociocognition the strongest predictor of social communication. Conclusions: Language measures which draw on a wide range of skills were the strongest overall predictors of general language outcomes. However, our VEPS measures were stronger predictors of specific outcomes. The clinical and theoretical implications of these findings are discussed.
C1 [Chiat, Shula; Roy, Penny] City Univ London, London EC1V 0HB, England.
RP Chiat, S (reprint author), City Univ London, Northampton Sq, London EC1V 0HB, England.
EM shula.chiat.1@city.ac.uk
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NR 40
TC 36
Z9 37
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD JUN
PY 2008
VL 49
IS 6
BP 635
EP 645
DI 10.1111/j.1469-7610.2008.01881.x
PG 11
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 303RB
UT WOS:000256057100006
PM 18341547
ER
PT J
AU Kuznetsova, AY
Deth, RC
AF Kuznetsova, Anna Y.
Deth, Richard C.
TI A model for modulation of neuronal synchronization by D4 dopamine
receptor-mediated phospholipid methylation
SO JOURNAL OF COMPUTATIONAL NEUROSCIENCE
LA English
DT Review
DE gamma oscillations; potassium channel; attention; autism; schizophrenia;
membrane fluidity; attention-deficit hyperactivity disorder
ID DEFICIT HYPERACTIVITY DISORDER; ATTENTION-DEFICIT/HYPERACTIVITY
DISORDER; DEPENDENT K+ CHANNEL; LIPID-PROTEIN INTERACTIONS;
BETA-ADRENERGIC-RECEPTOR; PREFRONTAL CORTEX; SELECTIVE ATTENTION;
SPECTRUM DISORDERS; ALZHEIMERS-DISEASE; GAMMA-OSCILLATIONS
AB We describe a new molecular mechanism of dopamine-induced membrane protein modulation that can tune neuronal oscillation frequency to attention-related gamma rhythm. This mechanism is based on the unique ability of D4 dopamine receptors (D4R) to carry out phospholipid methylation (PLM) that may affect the kinetics of ion channels. We show that by deceasing the inertia of the delayed rectifier potassium channel, a transition to 40 Hz oscillations can be achieved. Decreased potassium channel inertia shortens spike duration and decreases the interspike interval via its influence on the calcium-dependent potassium current. This mechanism leads to a transition to attention-related gamma oscillations in a pyramidal cell-interneuron network. The higher frequency and better synchronization is observed with PLM affecting pyramidal neurons only, and recurrent excitation between pyramidal neurons is important for synchronization. Thus dopamine-stimulated methylation of membrane phospholipids may be an important mechanism for modulating firing activity, while impaired methylation can contribute to disorders of attention.
C1 [Deth, Richard C.] Northeastern Univ, Dept Pharmaceut Sci, Boston, MA 02115 USA.
[Kuznetsova, Anna Y.] Louisiana State Univ, Hlth Sci Ctr, Neurosci Ctr Excellence, New Orleans, LA 70112 USA.
[Kuznetsova, Anna Y.] Saratov NG Chernyshevskii State Univ, Dept Nonlinear Proc, Saratov 410026, Russia.
RP Deth, RC (reprint author), Northeastern Univ, Dept Pharmaceut Sci, 312 Mugar Hall,306 Huntington Ave, Boston, MA 02115 USA.
EM r.deth@neu.edu
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NR 131
TC 12
Z9 12
PU SPRINGER
PI DORDRECHT
PA VAN GODEWIJCKSTRAAT 30, 3311 GZ DORDRECHT, NETHERLANDS
SN 0929-5313
J9 J COMPUT NEUROSCI
JI J. Comput. Neurosci.
PD JUN
PY 2008
VL 24
IS 3
BP 314
EP 329
DI 10.1007/s10827-007-0057-3
PG 16
WC Mathematical & Computational Biology; Neurosciences
SC Mathematical & Computational Biology; Neurosciences & Neurology
GA 289ZV
UT WOS:000255093300004
PM 17929154
ER
PT J
AU Liptak, GS
Benzoni, LB
Mruzek, DW
Nolan, KW
Thingvoll, MA
Wade, CM
Fryer, GE
AF Liptak, Gregory S.
Benzoni, Lauren B.
Mruzek, Daniel W.
Nolan, Karen W.
Thingvoll, Melissa A.
Wade, Christine M.
Fryer, G. Edgar
TI Disparities in diagnosis and access to health services for children with
autism: Data from the National Survey of Children's Health
SO JOURNAL OF DEVELOPMENTAL AND BEHAVIORAL PEDIATRICS
LA English
DT Article
DE autistic disorder; health services accessibility; prevalence
ID PERVASIVE DEVELOPMENTAL DISORDERS; SCHOOL-AGE-CHILDREN; SPECTRUM
DISORDERS; MENTAL-HEALTH; YOUNG-CHILDREN; BEHAVIORAL TREATMENT;
ETHNIC-DIFFERENCES; CARE UTILIZATION; INSURANCE STATUS; PARENTAL REPORT
AB Objective: Earlier assessment of autism improves outcomes. In addition, children with autism have significant need for medical care. Therefore, identification of factors associated with delays in the early diagnosis of autism and with decreased access to care has the potential to lead to interventions that will improve health and well-being. The aim of this study was to determine whether differences occur in the age-specific prevalence of autism or in access to health care in children of traditionally underserved populations. Method: Data from the National Survey of Children's Health of 2003/2004 were used. Diagnosis of autism and its severity were based on parental report. Results: The prevalence of autism was lower for Latinos (26/10,000) than for non-Latinos (51/10,000). Whites and blacks had comparable rates. The lowest preschool rate of autism (16/10,000) occurred in poor children. Latinos and poor families rated their children's autism as more severe. Being black, Latino, or poor was associated with decreased access to services, while having Medicaid or State Children's Health Insurance Program was linked with better access to some services. Conclusions: Disparities in the prevalence and parent-reported severity of autism and in access to health care were found for children with autism. Programs for children in general (e.g., universal screening for autism) and programs that target traditionally underserved. groups of children, their families, and their health care providers should be tested and implemented to optimize case finding of children with autism and to eliminate disparities in access to care and to early intervention.
C1 [Liptak, Gregory S.] Upstate Med Univ, Dept Pediat, Rochester, NY USA.
[Liptak, Gregory S.; Benzoni, Lauren B.; Mruzek, Daniel W.; Nolan, Karen W.; Thingvoll, Melissa A.; Wade, Christine M.] Univ Rochester, Med Ctr, Dept Pediat, Rochester, NY 14642 USA.
[Fryer, G. Edgar] NYU, Med Ctr, Dept Pediat, New York, NY 10016 USA.
RP Liptak, GS (reprint author), Univ Hosp, Dept Pediat, 750 E Adams St, Syracuse, NY 13210 USA.
EM Liptakg@upstate.edu
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NR 66
TC 71
Z9 71
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0196-206X
J9 J DEV BEHAV PEDIATR
JI J. Dev. Behav. Pediatr.
PD JUN
PY 2008
VL 29
IS 3
BP 152
EP 160
DI 10.1097/DBP.0b013e318165c7a0
PG 9
WC Behavioral Sciences; Psychology, Developmental; Pediatrics
SC Behavioral Sciences; Psychology; Pediatrics
GA 314JE
UT WOS:000256804900002
PM 18349708
ER
PT J
AU Dodd, S
Hupp, SDA
Jewell, JD
Krohn, E
AF Dodd, Sara
Hupp, Stephen D. A.
Jewell, Jeremy D.
Krohn, Emily
TI Using parents and siblings during a social story intervention for two
children diagnosed with PDD-NOS
SO JOURNAL OF DEVELOPMENTAL AND PHYSICAL DISABILITIES
LA English
DT Article
DE autism; pervasive developmental disorder; social stories
ID AUTISM; BEHAVIORS
AB Very few experimental studies have examined the use of Social Stories to modify the social skills of children with autism spectrum disorders. The behaviors targeted for the present study include a problem social skill (i.e., excessive directions) and a prosocial skill (i.e., compliments). The study used both a multiple-baseline-design-across-behaviors and a multiple-baseline-design-across-participants with two children diagnosed with Pervasive Developmental Disorder-Not Otherwise Specified. The main dependent variables were frequencies of directions and compliments. Results demonstrated that Social Stories were effective at modifying these social skills, and child and parent evaluations of the intervention were positive.
C1 [Dodd, Sara; Hupp, Stephen D. A.; Jewell, Jeremy D.; Krohn, Emily] So Illinois Univ, Dept Psychol, Edwardsville, IL 62026 USA.
RP Hupp, SDA (reprint author), So Illinois Univ, Dept Psychol, Edwardsville, IL 62026 USA.
EM sthupp@siue.edu
CR Adamian GG, 2004, ACTA PHYS HUNG NS-H, V19, P87, DOI 10.1556/APH.19.2004.1-2.13
Barry L. M., 2004, FOCUS AUTISM OTHER D, V19, P45, DOI DOI 10.1177/10883576040190010601
Brownell MD, 2002, J MUSIC THER, V39, P117
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GRAY C, 2000, NEW SOCIAL STORY HDB
Gray C. A., 1993, FOCUS AUTISTIC BEHAV, V8, P1, DOI DOI 10.1177/108835769300800101
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Kuttler S., 1998, FOCUS AUTISM OTHER D, V13, P176, DOI DOI 10.1177/108835769801300306
Lorimer PA, 2002, J POSIT BEHAV INTERV, V4, P53, DOI 10.1177/109830070200400109
Myles B. S., 1999, FOCUS AUTISM OTHER D, V14, P82, DOI 10.1177/108835769901400203
Nichols S, 2005, J EVIDENCE BASED PRA, V6, P90
Sansosti FJ, 2006, J POSIT BEHAV INTERV, V8, P43, DOI 10.1177/10983007060080010601
Scattone D, 2002, J AUTISM DEV DISORD, V32, P535, DOI 10.1023/A:1021250813367
Thiemann KS, 2001, J APPL BEHAV ANAL, V34, P425, DOI 10.1901/jaba.2001.34-425
NR 16
TC 18
Z9 18
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1056-263X
J9 J DEV PHYS DISABIL
JI J. Dev. Phys. Disabil.
PD JUN
PY 2008
VL 20
IS 3
BP 217
EP 229
DI 10.1007/s10882-007-9090-4
PG 13
WC Rehabilitation
SC Rehabilitation
GA 291KD
UT WOS:000255195000002
ER
PT J
AU Williams, KE
Hendy, H
Knecht, S
AF Williams, Keith E.
Hendy, Helen
Knecht, Stacey
TI Parent feeding practices and child variables associated with childhood
feeding problems
SO JOURNAL OF DEVELOPMENTAL AND PHYSICAL DISABILITIES
LA English
DT Article
DE feeding problems; parent practices; mealtime practices
ID PHYSICAL-ACTIVITY; SELECTIVE EATERS; EATING BEHAVIOR; RELATIVE WEIGHT;
YOUNG-CHILDREN; FOOD-INTAKE; CONSUMPTION; EVENTS
AB The present study identifies dimensions of feeding practices used by parents of children referred for feeding problems. These dimensions were derived from parent reported use of 17 feeding practices. This study compares these parent dimensions with child variables (age, gender, diagnosis of autism, medical conditions) as predictors of three measures of feeding problems: weight status, diet variety, and mealtime behavior problems. Participants included a clinical sample of 240 children with feeding problems. Results indicated that only child variables predicted weight status, while both dimensions of parent feeding practices and child variables predicted diet variety and mealtime behavior problems.
C1 [Williams, Keith E.] Penn State Hershey Med Ctr, Feeding Program, Hershey, PA 17033 USA.
[Hendy, Helen] Penn State Univ, Psychol Program, Schuylkill Haven, PA 17972 USA.
[Knecht, Stacey] Penn State Univ, Appl Behav Anal Program, Middletown, PA 17047 USA.
RP Williams, KE (reprint author), Penn State Hershey Med Ctr, Feeding Program, 905 W Governor Rd, Hershey, PA 17033 USA.
EM feedingprogram@hmc.psu.edu
CR ANLIKER JA, 1992, J NUTR EDUC, V24, P285
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Drucker RR, 1999, J DEV BEHAV PEDIATR, V20, P88, DOI 10.1097/00004703-199904000-00003
Faith MS, 2004, OBES RES, V12, P1711, DOI 10.1038/oby.2004.212
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GALLOWAY AT, 2007, APPETITE, V46, P318
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KEDESDY JH, 1998, CHILDREN FEEDING DIS
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MCKENZIE TL, 1991, J APPL BEHAV ANAL, V24, P141, DOI 10.1901/jaba.1991.24-141
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Williams KE, 2005, J DEV PHYS DISABIL, V17, P299, DOI 10.1007/s10882-005-4387-7
NR 30
TC 11
Z9 11
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1056-263X
J9 J DEV PHYS DISABIL
JI J. Dev. Phys. Disabil.
PD JUN
PY 2008
VL 20
IS 3
BP 231
EP 242
DI 10.1007/s10882-007-9091-3
PG 12
WC Rehabilitation
SC Rehabilitation
GA 291KD
UT WOS:000255195000003
ER
PT J
AU Johnson, CR
Rakison, DH
AF Johnson, Cynthia R.
Rakison, D. H.
TI Early categorization of Animate/Inanimate concepts in young children
with autism (vol 20, pg 315, 2008)
SO JOURNAL OF DEVELOPMENTAL AND PHYSICAL DISABILITIES
LA English
DT Correction
C1 [Johnson, Cynthia R.] Univ Pittsburgh, Sch Med, Childrens Hosp Pittsburgh, Autism Ctr, Pittsburgh, PA 15213 USA.
[Rakison, D. H.] Carnegie Mellon Univ, Pittsburgh, PA 15213 USA.
RP Johnson, CR (reprint author), Univ Pittsburgh, Sch Med, Childrens Hosp Pittsburgh, Autism Ctr, 3705 5th Ave, Pittsburgh, PA 15213 USA.
EM Cynthia.Johnson@chp.edu
CR Johnson CR, 2008, J DEV PHYS DISABIL, V20, P315, DOI 10.1007/s10882-007-9094-0
NR 1
TC 1
Z9 1
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1056-263X
J9 J DEV PHYS DISABIL
JI J. Dev. Phys. Disabil.
PD JUN
PY 2008
VL 20
IS 3
BP 315
EP 316
DI 10.1007/s10882-007-9094-0
PG 2
WC Rehabilitation
SC Rehabilitation
GA 291KD
UT WOS:000255195000010
ER
PT J
AU Jung, S
Sainato, DM
Davis, CA
AF Jung, Sunhwa
Sainato, Diane M.
Davis, Carol A.
TI Using High-Probability Request Sequences to Increase Social Interactions
in Young Children With Autism
SO JOURNAL OF EARLY INTERVENTION
LA English
DT Article
DE autism; high-probability requests; social interactions; social validity;
peer-mediated interventions
ID BEHAVIORAL MOMENTUM; FACILITATE SOCIALIZATION; INTERSPERSED REQUESTS;
INTEGRATED PRESCHOOL; INCLUSIVE SETTINGS; STUDENT COMPLIANCE; TEACHER
PROMPTS; DISABILITIES; INTERVENTIONS; MAINTENANCE
AB We investigated the effects of an intervention using high-probability request sequences with embedded peer modeling to increase social interactions of children with autism in a classroom. The effects of the intervention on compliant responding to social requests and social behaviors were monitored using a single-subject multiple baseline design across children. Additionally, social validity regarding the intervention goals, procedures, and outcomes was measured by relevant consumers. The results of this study indicate that all three children's compliant responding to low-probability requests and social behaviors increased with the intervention and were maintained. Furthermore, the target children's social behaviors generalized to untrained peers and nontraining settings. The social validity results indicated a high level of consumer acceptability and usability among relevant consumers.
C1 [Jung, Sunhwa] Otterbein Coll, Westerville, OH 43081 USA.
[Sainato, Diane M.] Ohio State Univ, Columbus, OH 43210 USA.
[Davis, Carol A.] Univ Washington, Seattle, WA 98195 USA.
RP Jung, S (reprint author), Otterbein Coll, 456 Roush Hall, Westerville, OH 43081 USA.
EM sjung@otterbein.edu
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NR 62
TC 12
Z9 12
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1053-8151
J9 J EARLY INTERVENTION
JI J. Early Interv.
PD JUN
PY 2008
VL 30
IS 3
BP 163
EP 187
DI 10.1177/1053815108317970
PG 25
WC Education, Special; Psychology, Educational; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA 479EO
UT WOS:000268642700001
ER
PT J
AU Stanton-Chapman, TL
Kaiser, AP
Vijay, P
Chapman, C
AF Stanton-Chapman, Tina L.
Kaiser, Ann P.
Vijay, Prathibha
Chapman, Carol
TI A Multicomponent Intervention to Increase Peer-Directed Communication in
Head Start Children
SO JOURNAL OF EARLY INTERVENTION
LA English
DT Article
DE early intervention; developmental disabilities; Head Start; peer
interaction; preschoolers
ID SOCIAL-COMMUNICATION; YOUNG-CHILDREN; PRESCHOOLERS; SKILLS;
DISABILITIES; AUTISM; PLAY
AB The effects of a multicomponent intervention strategy to increase peer-directed social communication in eight Head Start children at risk for poor language and social skill development were examined. The intervention consisted of three components: (a) a planning period, including reading a storybook that illustrated the play theme and use of the social pragmatic communication strategies, practice using vocabulary to be used in play, and choosing roles for the thematic activity; (b) a 10-min play session in which the interventionist coached children to interact while they played with the thematic related toys; and (c) a brief reporting period in which children reviewed their use of the social pragmatic strategies and specific vocabulary. A multiple-baseline design across dyads with pre- and postgeneralization probes was employed. Children increased their use of peer-directed communication over baseline levels, target vocabulary words, and other social pragmatic skills.
C1 [Stanton-Chapman, Tina L.; Kaiser, Ann P.; Vijay, Prathibha; Chapman, Carol] Vanderbilt Univ, Nashville, TX USA.
RP Stanton-Chapman, TL (reprint author), Univ Virginia, Curry Sch Educ, 405 Emmet St, S Charlottesville, VA 22904 USA.
EM stantonchapman@virginia.edu
CR Achenbach T. M., 1997, GUIDE CAREGIVER TEAC
Barnett D. W., 1995, DESIGNING PRESCHOOL
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NR 52
TC 3
Z9 3
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1053-8151
J9 J EARLY INTERVENTION
JI J. Early Interv.
PD JUN
PY 2008
VL 30
IS 3
BP 188
EP 212
DI 10.1177/1053815108318746
PG 25
WC Education, Special; Psychology, Educational; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA 479EO
UT WOS:000268642700002
ER
PT J
AU Duda, MA
Clarke, S
Fox, L
Dunlap, G
AF Duda, Michelle A.
Clarke, Shelley
Fox, Lise
Dunlap, Glen
TI Implementation of Positive Behavior Support With a Sibling Set in a Home
Environment
SO JOURNAL OF EARLY INTERVENTION
LA English
DT Article
DE positive behavior support; home setting; young children; procedural
fidelity; sibling set; challenging behavior
ID YOUNG-CHILDREN; CHALLENGING BEHAVIOR; ABERRANT BEHAVIOR; CONDUCT
DISORDER; INTERVENTION; PERSPECTIVES; DISABILITIES; PREVENTION;
OUTCOMES; AUTISM
AB This study provides a demonstration of the process of positive behavior support (PBS) within a home setting to address the challenging behavior of a sibling set within family routines. Although a growing data base is demonstrating the feasibility and effectiveness of conducting functional behavioral assessment and implementing assessment-based interventions for young children in family contexts, the vast majority of these studies have focused on dyadic parent child interactions. The presence of more than one child, particularly when all children display challenging behaviors, can amplify levels of parent stress and complicate the logistics of assessment, planning, and systematic intervention. In this study, three young children presented challenging behaviors that were highly disruptive to home routines. Functional assessments were followed by implementation of multicomponent, family-centered PBS plans individualized to four activity routines. Multiple-baseline across-routines and quasiexperimental (A-B) within-routines designs demonstrated promising effects of the interventions in reducing aggregate levels of challenging behavior.
C1 [Duda, Michelle A.; Clarke, Shelley; Fox, Lise; Dunlap, Glen] Univ S Florida, Tampa, FL USA.
EM mduda@fmhi.usf.edu
CR Achenbach T, 2000, CHILD BEHAV CHECKLIS
Albin R. W., 1996, POSITIVE BEHAV SUPPO, P81
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NR 42
TC 4
Z9 4
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1053-8151
J9 J EARLY INTERVENTION
JI J. Early Interv.
PD JUN
PY 2008
VL 30
IS 3
BP 213
EP 236
DI 10.1177/1053815108319124
PG 24
WC Education, Special; Psychology, Educational; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA 479EO
UT WOS:000268642700003
ER
PT J
AU Cornish, K
Turk, J
Hagerman, R
AF Cornish, K.
Turk, J.
Hagerman, R.
TI The fragile X continuum: new advances and perspectives
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Review
DE autism; fragile X syndrome; FXTAS; FMRP; intellectual disability;
premutation
ID PREMATURE OVARIAN FAILURE; TREMOR/ATAXIA SYNDROME FXTAS; MULTIPLE SYSTEM
ATROPHY; FMR1 MESSENGER-RNA; TREMOR-ATAXIA SYNDROME; PREMUTATION
CARRIERS; MENTAL-RETARDATION; MOVEMENT-DISORDERS; FULL MUTATION; MOUSE
MODEL
AB Fragile X syndrome is the world's most common hereditary cause of intellectual disability in men and to a lesser extent in women. The disorder is caused by the silencing of a single gene on the X chromosome, the Fragile X Mental Retardation Gene-I. A substantial body of research across the disciplines of molecular genetics, child psychiatry and developmental neuroscience bears testament to a decade of exciting and innovative science that has advanced our knowledge about the fragile X 'signature' or influence across cognitive and social development. The core aims of this review are to first discuss fragile X syndrome and premutation involvement in the context of current advances that demonstrate the dynamic nature of the genotype on phenotypic outcomes. Second, to discuss the implications of these recent advances for the development of clinical and educational interventions and resource tools that target specific phenotypic 'signatures' within the fragile X continuum.
C1 [Cornish, K.] McGill Univ, McGill Child Lab Res & Educ Dev Disorders, Montreal, PQ H3A 1Y2, Canada.
[Turk, J.] St Georges Univ London, Div Clin Dev Sci, London, England.
[Hagerman, R.] Univ Calif Davis, Sch Med, Dept Pediat, Sacramento, CA 95817 USA.
[Hagerman, R.] Univ Calif Davis, Sch Med, MIND Inst, Sacramento, CA 95817 USA.
RP Cornish, K (reprint author), McGill Univ, McGill Child Lab Res & Educ Dev Disorders, 3700 McTavish St, Montreal, PQ H3A 1Y2, Canada.
EM kim.cornish@mcgill.ca
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NR 129
TC 66
Z9 68
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD JUN
PY 2008
VL 52
BP 469
EP 482
DI 10.1111/j.1365-2788.2008.01056.x
PN 6
PG 14
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 301VV
UT WOS:000255925000001
PM 18444988
ER
PT J
AU Didden, R
Sigafoos, J
Green, VA
Korzilius, H
Mouws, C
Lancioni, GE
O'Reilly, MF
Curfs, LMG
AF Didden, R.
Sigafoos, J.
Green, V. A.
Korzilius, H.
Mouws, C.
Lancioni, G. E.
O'Reilly, M. F.
Curfs, L. M. G.
TI Behavioural flexibility in individuals with Angelman syndrome, Down
syndrome, non-specific intellectual disability and Autism spectrum
disorder
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Article
DE Angelman syndrome; Autism spectrum disorder; Behavioural flexibility;
Behavioural phenotype; Down syndrome
AB Background Little is known about behavioural flexibility in children and adults with Angelman syndrome and whether people with this syndrome have more or less problems in being behaviourally flexible as compared with other people.
Method Behavioural flexibility scores were assessed in 129 individuals with Angelman syndrome using 11 items from the Behavioural Flexibility Rating Scale-Revised (Green et al. 2007). Level of behavioural flexibility scores in individuals with Angelman syndrome (N = 129) was compared with that of people with non-specific intellectual disability (ID) ( N = 90), Down syndrome (N = 398) and Autism spectrum disorder (N = 235).
Results Comparative analyses show that individuals with Angelman syndrome were more flexible than those with non-specific ID (P < 0.001) and those with Autism spectrum disorder (P < 0.01). There were no differences in behavioural flexibility scores between individuals with Angelman syndrome and those with Down syndrome (P = 0.94).
Conclusion It is concluded that individuals with Angelman syndrome are comparatively flexible in their behaviour.
C1 [Didden, R.; Korzilius, H.; Mouws, C.] Radboud Univ Nijmegen, NL-6500 HE Nijmegen, Netherlands.
[Sigafoos, J.; Green, V. A.] Victoria Univ Wellington, Wellington, New Zealand.
[Lancioni, G. E.] Univ Bari, Bari, Italy.
[O'Reilly, M. F.] Univ Texas Austin, Austin, TX 78712 USA.
[Curfs, L. M. G.] Univ Maastricht, Maastricht, Netherlands.
RP Didden, R (reprint author), Radboud Univ Nijmegen, POB 9104, NL-6500 HE Nijmegen, Netherlands.
EM R.Didden@pwo.ru.nl
CR Berry RJ, 2005, AM J MED GENET A, V132A, P8, DOI 10.1002/ajmg.a.30154
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Kraijer DW, 2004, SOCIALE REDZAAMHEIDS
PETERSSCHEFFER N, 2008, IN PRESS RES DEV DIS
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Williams MM, 2005, WEED SCI, V53, P62, DOI 10.1614/WS-04-079R1
NR 13
TC 10
Z9 10
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD JUN
PY 2008
VL 52
BP 503
EP 509
DI 10.1111/j.1365-2788.2008.01055.x
PN 6
PG 7
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 301VV
UT WOS:000255925000004
PM 18384537
ER
PT J
AU [Anonymous]
AF [Anonymous]
TI $8.5 million awarded to Massachusetts Institute of Technology for autism
and dyslexia research
SO JOURNAL OF INVESTIGATIVE MEDICINE
LA English
DT News Item
NR 0
TC 0
Z9 0
PU B C DECKER INC
PI HAMILTON
PA 50 KING STREET EAST, 2ND FLOOR, PO BOX 620, L C D 1, HAMILTON, ONTARIO
L8N 3K7, CANADA
SN 1081-5589
J9 J INVEST MED
JI J. Invest. Med.
PD JUN
PY 2008
VL 56
IS 5
BP 742
EP 743
PG 2
WC Medicine, General & Internal; Medicine, Research & Experimental
SC General & Internal Medicine; Research & Experimental Medicine
GA 311FP
UT WOS:000256586400007
ER
PT J
AU Gothelf, D
Goraly, O
Avni, S
Stawski, M
Hartmann, I
Basel-Vanagaite, L
Apter, A
AF Gothelf, Doron
Goraly, Olga
Avni, Sari
Stawski, Mike
Hartmann, Inbar
Basel-Vanagaite, Lina
Apter, Alan
TI Psychiatric morbidity with focus on obsessive-compulsive disorder in an
Israeli cohort of adolescents with mild to moderate mental retardation
SO JOURNAL OF NEURAL TRANSMISSION
LA English
DT Article
DE adolescence; mental retardation; intellectual disability; psychiatric
disorders; obsessive-compulsive disorder; autism; antipsychotics
ID K-SADS-PL; INTELLECTUAL DISABILITY; PSYCHOMETRIC PROPERTIES;
SUICIDAL-BEHAVIOR; CHILDREN; PREVALENCE; PSYCHOPATHOLOGY; PEOPLE;
ADULTS; VALIDITY
AB The study evaluated the prevalence of DSM-IV-TR-defined psychiatric disorders in adolescents with mental retardation, with a focus on obsessive-compulsive disorder (OCD), for which data at present are sparse. Eighty-seven adolescents with mild to moderate mental retardation attending the Israeli special-education system were screened for psychiatric disorders in general and obsessive-compulsive symptoms in particular. Sixty-one percent had at least one psychiatric disorder. Of the 13 participants receiving antipsychotic medication, none had an underlying psychotic disorder and most had anxiety or depressive disorders. OCD was detected in 11% of participants and was characterized by high rates of psychiatric comorbidities. The severity of autistic symptoms predicted 39% of the variance in the severity of OCD symptoms. Adolescents with mild to moderate mental retardation have high rates of psychiatric morbidities that are often inappropriately treated. OCD is prevalent in this population and is strongly associated with autistic symptoms. Further studies are required in adolescents with mental retardation to better delineate psychiatric morbidities and their appropriate treatment in this at-risk population.
C1 [Gothelf, Doron; Goraly, Olga; Stawski, Mike; Apter, Alan] Schneider Childrens Med Ctr, Feinberg Dept Child Psychiat, Behav Neurogenet Ctr, IL-49202 Petah Tiqwa, Israel.
[Gothelf, Doron; Basel-Vanagaite, Lina; Apter, Alan] Tel Aviv Univ, Sackler Fac Med, IL-69978 Tel Aviv, Israel.
[Avni, Sari] Lev Ha Sharon Psychiat Hosp, Pardesia, Israel.
[Hartmann, Inbar] Asaf Harofe Med Ctr, Dept Pediat, Zerifin, Israel.
[Basel-Vanagaite, Lina] Rabin Med Ctr, Raphael Recanati Genet Inst, Petah Tiqwa, Israel.
RP Gothelf, D (reprint author), Schneider Childrens Med Ctr, Feinberg Dept Child Psychiat, Behav Neurogenet Ctr, 14 Kaplan St, IL-49202 Petah Tiqwa, Israel.
EM gothelf@post.tau.ac.il
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NR 39
TC 8
Z9 8
PU SPRINGER WIEN
PI WIEN
PA SACHSENPLATZ 4-6, PO BOX 89, A-1201 WIEN, AUSTRIA
SN 0300-9564
J9 J NEURAL TRANSM
JI J. Neural Transm.
PD JUN
PY 2008
VL 115
IS 6
BP 929
EP 936
DI 10.1007/s00702-008-0037-4
PG 8
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 309PJ
UT WOS:000256472400019
PM 18351287
ER
PT J
AU Neumann, ID
AF Neumann, I. D.
TI Brain oxytocin: A key regulator of emotional and social behaviours in
both females and males
SO JOURNAL OF NEUROENDOCRINOLOGY
LA English
DT Review
DE anxiety; maternal care; aggression; sexual behaviour; cognition; stress
coping
ID PITUITARY-ADRENAL AXIS; HYPOTHALAMIC-NEUROHYPOPHYSEAL SYSTEM;
ANXIETY-RELATED BEHAVIOR; PARAVENTRICULAR NUCLEUS; MALE RATS; DEFICIENT
MICE; KNOCKOUT MICE; VASOPRESSIN RECEPTOR; CENTRAL AMYGDALA;
SEXUAL-BEHAVIOR
AB In addition to various reproductive stimuli, the neuropeptide oxytocin (OXT) is released both from the neurohypophysial terminal into the blood stream and within distinct brain regions in response to stressful or social stimuli. Brain OXT receptor-mediated actions were shown to be significantly involved in the regulation of a variety of behaviours. Here, complementary methodological approaches are discussed which were utilised to reveal, for example, anxiolytic and anti-stress effects of OXT, both in females and in males, effects that were localised within the central amygdala and the hypothalamic paraventricular nucleus. Also, in male rats, activation of the brain OXT system is essential for the regulation of sexual behaviour, and increased OXT system activity during mating is directly linked to an attenuated anxiety-related behaviour. Moreover, in late pregnancy and during lactation, central OXT is involved in the establishment and fine-tuned maintenance of maternal care and maternal aggression. In monogamous prairie voles, brain OXT is important for mating-induced pair bonding, especially in females. Another example of behavioural actions of intracerebral OXT is the promotion of social memory processes and recognition of con-specifics, as revealed in rats, mice, sheep and voles. Experimental evidence suggests that, in humans, brain OXT exerts similar behavioural effects. Thus, the brain OXT system seems to be a potential target for the development of therapeutics to treat anxiety and depression-related diseases or abnormal social behaviours including autism.
C1 Univ Regensburg, Dept Behav & Mol Neuroendocrinol, D-93053 Regensburg, Germany.
RP Neumann, ID (reprint author), Univ Regensburg, Dept Behav & Mol Neuroendocrinol, D-93053 Regensburg, Germany.
EM inga.neumann@biologie.uni-regensburg.de
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NR 113
TC 272
Z9 275
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0953-8194
J9 J NEUROENDOCRINOL
JI J. Neuroendocrinol.
PD JUN
PY 2008
VL 20
IS 6
BP 858
EP 865
DI 10.1111/j.1365-2826.2008.01726.x
PG 8
WC Endocrinology & Metabolism; Neurosciences
SC Endocrinology & Metabolism; Neurosciences & Neurology
GA 311EG
UT WOS:000256582900032
PM 18601710
ER
PT J
AU Wang, PS
AF Wang, Peishi
TI Effects of a Parent Training Program on the Interactive Skills of
Parents of Children with Autism in China
SO JOURNAL OF POLICY AND PRACTICE IN INTELLECTUAL DISABILITIES
LA English
DT Article
DE autism; intellectual disabilities; parent interactive skills; parent
training; parent-child interaction
AB This study evaluated the effects of a parent training program on the interactive skills of parents of children with autism in the People's Republic of China. Twenty-seven families of children with autism in a northeastern city of China were randomly assigned to either the training or the control group. Parents received a total of 20 h of training in a format that included both group and individual sessions. Parents' interactive skills during free play with their children with autism were examined using videotaped observations at their homes and analyzed using the Maternal Behavior Rating Scales. One-way analysis of covariance indicated that following the training, parents in the training group, compared with those in the control group, were more sensitive to their children's interests, responded to their children's behavior more appropriately, were more accepting of their children and their behavior, showed more enjoyment of interacting with their children, and expressed more warmth toward their children throughout the free play interactions. More research is needed to investigate whether parental responsiveness and affect promote more active engagement by their children in their interactions.
C1 CUNY Queens Coll, Grad Programs Special Educ, Dept Educ & Community Programs, Flushing, NY 11367 USA.
RP Wang, PS (reprint author), CUNY Queens Coll, Grad Programs Special Educ, Dept Educ & Community Programs, 65-30 Kissena Blvd, Flushing, NY 11367 USA.
EM peishi.wang@qc.cuny.edu
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1996, ADVOCATE JUL
NR 42
TC 10
Z9 10
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1741-1122
J9 J POLICY PRACT INTEL
JI J. Policy Pract. Intellect. Disabil.
PD JUN
PY 2008
VL 5
IS 2
BP 96
EP 104
DI 10.1111/j.1741-1130.2008.00154.x
PG 9
WC Health Policy & Services; Rehabilitation
SC Health Care Sciences & Services; Rehabilitation
GA V13SL
UT WOS:000207686400004
ER
PT J
AU Bottroff, V
AF Bottroff, Verity
TI Sensory Perceptual Issues in Autism and Asperger's Syndrome. Different
Sensory Experiences-Different Perceptual Worlds
SO JOURNAL OF POLICY AND PRACTICE IN INTELLECTUAL DISABILITIES
LA English
DT Book Review
C1 [Bottroff, Verity] Flinders Univ S Australia, Dept Disabil Studies, Sch Med, Adelaide, SA, Australia.
RP Bottroff, V (reprint author), Flinders Univ S Australia, Dept Disabil Studies, Sch Med, Adelaide, SA, Australia.
EM verity.bottroff@flinders.edu.au
CR Bogdashina O, 2003, SENSORY PERCEPTUAL I
NR 1
TC 0
Z9 0
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1741-1122
J9 J POLICY PRACT INTEL
JI J. Policy Pract. Intellect. Disabil.
PD JUN
PY 2008
VL 5
IS 2
BP 145
EP 145
DI 10.1111/j.1741-1130.2008.00164.x
PG 1
WC Health Policy & Services; Rehabilitation
SC Health Care Sciences & Services; Rehabilitation
GA V13SL
UT WOS:000207686400014
ER
PT J
AU Krakowiak, P
Goodlin-Jones, B
Hertz-Picciotto, I
Croen, LA
Hansen, RL
AF Krakowiak, Paula
Goodlin-Jones, Beth
Hertz-Picciotto, Irva
Croen, Lisa A.
Hansen, Robin L.
TI Sleep problems in children with autism spectrum disorders, developmental
delays, and typical development: a population-based study
SO JOURNAL OF SLEEP RESEARCH
LA English
DT Article
DE autism; autistic spectrum disorder; developmental delay; sleep problems
ID SCHOOL-AGE-CHILDREN; YOUNG-CHILDREN; ASPERGERS-DISORDER; BEHAVIOR
PROBLEMS; DAYTIME BEHAVIOR; PATTERNS; INFANTS; DISTURBANCES;
RESTRICTION; PREVALENCE
AB This study compared parent-reported sleep characteristics in 2- to 5-year-old children with autism spectrum disorders (ASD) to children with other developmental delays (DD) and typical development (TD). We included 529 children (303 ASD [167 males], 63 DD [46 males], and 163 TD [134 males]) enrolled in the CHARGE study, an ongoing population-based case-control study. The mean age of participants was 3.6 years (standard deviation, 0.8 years). ASD diagnosis was confirmed with Autism Diagnostic Interview-Revised (ADI-R) and Autism Diagnostic Observation Schedules (ADOS). Cognitive and adaptive functioning was assessed using Mullen Scales of Early Learning (MSEL) and Vineland Adaptive Behavior Scales (VABS), respectively. Demographic, medical and sleep history information were ascertained from California birth records, telephone interview, medical assessments at clinic visit, and parent-administered questionnaires. Fifty-three percent of children with ASD had at least one frequent sleep problem, followed by 46% of children with DD, and 32% of the TD group (P < 0.0001). Exploratory factor analyses of sleep history data yielded two factors: sleep onset problems and night waking. Children with ASD had marginally higher sleep onset factor scores and significantly higher night waking factor scores compared with the TD group. Factor scores for children with DD were intermediate between the ASD and TD groups. Cognitive or adaptive development did not predict severity of sleep problems in the ASD group.
C1 [Krakowiak, Paula; Hertz-Picciotto, Irva; Hansen, Robin L.] Univ Calif Davis, Sch Med, Dept Publ Hlth Sci, Div Epidemiol, Davis, CA 95616 USA.
[Krakowiak, Paula; Goodlin-Jones, Beth; Hertz-Picciotto, Irva; Croen, Lisa A.] Univ Calif Davis, Med Invest Neurodev Disorders Inst, Davis, CA 95616 USA.
[Goodlin-Jones, Beth] Univ Calif Davis, Sch Med, Dept Psychiat & Behav Sci, Davis, CA 95616 USA.
[Hansen, Robin L.] Univ Calif Davis, Sch Med, Dept Pediat, Davis, CA 95616 USA.
[Croen, Lisa A.] Kaiser Permanente, Kaiser Fdn Res Inst, Div Res, Oakland, CA USA.
RP Hansen, RL (reprint author), MIND Inst, 2825 50th St, Sacramento, CA USA.
EM robin.hansen@ucdmc.ucdavis.edu
CR ALI NJ, 1993, ARCH DIS CHILD, V68, P360
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NR 53
TC 92
Z9 96
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0962-1105
J9 J SLEEP RES
JI J. Sleep Res.
PD JUN
PY 2008
VL 17
IS 2
BP 197
EP 206
DI 10.1111/j.1365-2869.2008.00650.x
PG 10
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 300OR
UT WOS:000255835300009
PM 18482108
ER
PT J
AU Wakschlag, LS
Hill, C
Carter, AS
Danis, B
Egger, HL
Keenan, K
Leventhal, BL
Cicchetti, D
Maskowitz, K
Burns, J
Briggs-Gowan, MJ
AF Wakschlag, Lauren S.
Hill, Carri
Carter, Alice S.
Danis, Barbara
Egger, Helen L.
Keenan, Kate
Leventhal, Bennett L.
Cicchetti, Domenic
Maskowitz, Katie
Burns, James
Briggs-Gowan, Margaret J.
TI Observational assessment of preschool disruptive behavior, part I:
Reliability of the Disruptive Behavior Diagnostic Observation Schedule
(DB-DOS)
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Article
DE disruptive behavior; diagnostic observation; developmental
psychopathology; preschool behavior problems
ID CHILD INTERACTION; OPPOSITIONAL DEFIANT; CONDUCT DISORDER; PERCEPTIONS;
VALIDITY; AUTISM
AB Objective: To examine the reliability of the Disruptive Behavior Diagnostic Observation Schedule (DB-DOS), a new observational method for assessing preschool disruptive behavior. Method: The DB-DOS is a structured clinic-based assessment designed to elicit clinically salient behaviors relevant to the diagnosis of disruptive behavior in preschoolers. Child behavior is assessed in three interactional contexts that vary by partner (parent versus examiner) and level of support provided. Twenty-one disruptive behaviors are coded within two domains: problems in Behavioral Regulation and problems in Anger Modulation. A total of 364 referred and nonreferred preschoolers participated: interrater reliability and internal consistency were assessed on a primary sample (n = 335) and test-retest reliability was assessed in a separate sample (n = 29). Results: The DB-DOS demonstrated good interrater and test-retest reliability. Confirmatory factor analysis demonstrated an excellent fit of the DB-DOS multidomain model of disruptive behavior. Conclusions: The DB-DOS is a reliable observational tool for clinic-based assessment of preschool disruptive behavior. This standardized assessment method holds promise for advancing developmentally sensitive characterization of preschool psychopathology.
C1 [Wakschlag, Lauren S.; Hill, Carri; Danis, Barbara; Leventhal, Bennett L.; Maskowitz, Katie; Burns, James] Univ Illinois, Dept Psychiat, Inst Juvenile Res, Chicago, IL 60608 USA.
[Keenan, Kate] Univ Chicago, Dept Psychiat, Chicago, IL 60637 USA.
[Egger, Helen L.] Duke Univ, Dept Psychiat, Durham, NC 27706 USA.
[Cicchetti, Domenic] Yale Univ, Dept Psychiat, New Haven, CT 06520 USA.
[Briggs-Gowan, Margaret J.] Univ Connecticut, Dept Psychiat, Storrs, CT 06269 USA.
[Carter, Alice S.] Univ Massachusetts, Dept Psychol, Amherst, MA 01003 USA.
RP Wakschlag, LS (reprint author), Univ Illinois, Dept Psychiat, Inst Juvenile Res, 1747 W Roosevelt Rd,MC 747, Chicago, IL 60608 USA.
EM lwakschlag@psych.uic.edu
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NR 44
TC 32
Z9 32
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD JUN
PY 2008
VL 47
IS 6
BP 622
EP 631
DI 10.1097/CHI.0b013c31816c5bdb
PG 10
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 304LM
UT WOS:000256111000005
PM 18434926
ER
PT J
AU Gotham, K
Risi, S
Dawson, G
Tager-Flusberg, H
Joseph, R
Carter, A
Hepburn, S
McMahon, W
Rodier, P
Hyman, SL
Sigman, M
Rogers, S
Landa, R
Spence, MA
Osann, K
Flodman, P
Volkmar, F
Hollander, E
Buxbaum, J
Pickles, A
Lord, C
AF Gotham, Katherine
Risi, Susan
Dawson, Geraldine
Tager-Flusberg, Helen
Joseph, Robert
Carter, Alice
Hepburn, Susan
McMahon, William
Rodier, Patricia
Hyman, Susan L.
Sigman, Marian
Rogers, Sally
Landa, Rebecca
Spence, M. Anne
Osann, Kathryn
Flodman, Pamela
Volkmar, Fred
Hollander, Eric
Buxbaum, Joseph
Pickles, Andrew
Lord, Catherine
TI A replication of the Autism Diagnostic Observation Schedule (ADOS)
revised algorithms
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Article
DE autism; pervasive developmental disorders not otherwise specified;
Autism Diagnostic Observation Schedule; diagnosis
ID SPECTRUM; DISORDER; CHILDREN
AB Objective: To replicate the factor structure and predictive validity of revised Autism Diagnostic Observation Schedule algorithms in an independent dataset (N = 1,282). Method: Algorithm revisions were replicated using data from children ages 18 months to 16 years collected at 11 North American sites participating in the Collaborative Programs for Excellence in Autism and the Studies to Advance Autism Research and Treatment. Results: Sensitivities and specificities approximated or exceeded those of the old algorithms except for young children with phrase speech and a clinical diagnosis of pervasive developmental disorders not otherwise specified. Conclusions: Revised algorithms increase comparability between modules and improve the predictive validity of the Autism Diagnostic Observation Schedule for autism cases compared to the original algorithms.
C1 [Gotham, Katherine; Risi, Susan; Lord, Catherine] Univ Michigan, Autism & Commun Disorders Ctr, Ann Arbor, MI 48109 USA.
[Dawson, Geraldine] Univ Washington, Seattle, WA 98195 USA.
[Tager-Flusberg, Helen; Joseph, Robert] Boston Univ, Sch Med, Boston, MA 02215 USA.
[Carter, Alice] Univ Massachusetts, Amherst, MA 01003 USA.
[Hepburn, Susan] Univ Colorado, Hlth Sci Ctr, Boulder, CO 80309 USA.
[McMahon, William] Univ Utah, Salt Lake City, UT 84112 USA.
[Rodier, Patricia; Hyman, Susan L.] Univ Rochester, Med Ctr, Rochester, NY 14627 USA.
[Sigman, Marian] Univ Calif Los Angeles, Los Angeles, CA USA.
[Rogers, Sally] Univ Calif Davis, MIND Inst, Davis, CA USA.
[Spence, M. Anne; Osann, Kathryn; Flodman, Pamela] Univ Calif Irvine, Irvine, CA USA.
[Volkmar, Fred] Yale Child Study Ctr, New Haven, CT USA.
[Hollander, Eric; Buxbaum, Joseph] Mt Sinai Sch Med, New York, NY USA.
[Pickles, Andrew] Univ Manchester, Manchester M13 9PL, Lancs, England.
RP Gotham, K (reprint author), Univ Michigan, Autism & Commun Disorders Ctr, 1111 E Catherine St, Ann Arbor, MI 48109 USA.
EM kog@umich.edu
RI Pickles, Andrew/A-9625-2011; Tager-Flusberg, Helen/D-5265-2009; Joseph,
Roy/D-8530-2015
OI Pickles, Andrew/0000-0003-1283-0346;
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Browne MW, 1993, TESTING STRUCTURAL E, P136, DOI DOI 10.1177/0049124192021002001
Constantino JN, 2004, J CHILD PSYCHOL PSYC, V45, P719, DOI 10.1111/j.1469-7610.2004.00266.x
de Bildt A, 2004, J AUTISM DEV DISORD, V34, P129
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Wechsler D., 2003, WECHSLER INTELLIGENC
NR 18
TC 83
Z9 84
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD JUN
PY 2008
VL 47
IS 6
BP 642
EP 651
DI 10.1097/CHI.0b013e31816bffb7
PG 10
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 304LM
UT WOS:000256111000007
PM 18434924
ER
PT J
AU Pine, DS
Guyer, AE
Goldwin, M
Towbin, KA
Leibenluft, E
AF Pine, Daniel S.
Guyer, Amanda E.
Goldwin, Michelle
Towbin, Kenneth A.
Leibenluft, Ellen
TI Autism spectrum disorder scale scores in pediatric mood and anxiety
disorders
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Article
DE mood disorder; anxiety disorder; autism spectrum; impairment
ID PERVASIVE DEVELOPMENTAL DISORDERS; BIPOLAR DISORDER; DIAGNOSTIC
INTERVIEW; LANGUAGE IMPAIRMENT; BROAD AUTISM; CHILDREN; TRAITS;
FLEXIBILITY; ADOLESCENTS; POPULATION
AB Objective: To compare scores on autism spectrum disorder (ASD) symptom scales in healthy youths and youths with mood or anxiety disorders. Method: A total of 352 youths were recruited (107 healthy participants, 88 with an anxiety disorder, 32 with major depressive disorder, 62 with bipolar disorder, and 63 with a mood disorder characterized by severe nonepisodic irritability). Participants received structured psychiatric interviews and parent ratings on at least one of three ASD symptom scales: Children's Communication Checklist, Social Communication Questionnaire, and Social Responsiveness Scale. Results: Relative to healthy youths, youths with mood or anxiety disorders exhibited higher scores on each ASD symptom scale. ASD symptom scale scores also showed an association with impairment severity and attention-cleficit/hyperactivity disorder. Among patients with mood disorders but not those with anxiety disorders, consistent, statistically significant associations between diagnosis and ASD symptom scale scores remained even after controlling for potential confounders. Conclusions: Patients with mood disorders exhibit higher scores on ASD symptom scales than healthy youths or youths with anxiety disorders. These data should alert clinicians to the importance of assessing ASD symptoms to identify social reciprocity and communication deficits as possible treatment targets in pediatric mood and anxiety disorders.
C1 [Pine, Daniel S.; Guyer, Amanda E.; Goldwin, Michelle; Towbin, Kenneth A.] NIMH, Sect Dev & Affect Neurosci, Emot & Dev Branch, MAP,NIH,Dept Hlth & Human Serv, Bethesda, MD 20892 USA.
[Leibenluft, Ellen] NIMH, Sect Bipolar Spectrum Disorders, Emot & Dev Branch, MAP,NIH,Dept Hlth & Human Serv, Bethesda, MD 20892 USA.
RP Pine, DS (reprint author), NIMH, Sect Dev & Affect Neurosci, Emot & Dev Branch, MAP,NIH,Dept Hlth & Human Serv, 15K North Dr, Bethesda, MD 20892 USA.
EM pined@mail.nih.gov
CR Baird G, 2006, LANCET, V368, P210, DOI 10.1016/S0140-6736(06)69041-7
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NR 35
TC 47
Z9 47
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD JUN
PY 2008
VL 47
IS 6
BP 652
EP 661
DI 10.1097/CHI.0b013e31816bffa5
PG 10
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 304LM
UT WOS:000256111000008
PM 18434923
ER
PT J
AU Reiersen, AM
Constantino, JN
Todd, RD
AF Reiersen, Angela M.
Constantino, John N.
Todd, Richard D.
TI Co-occurrence of motor problems and autistic symptoms in
attention-deficit/hyperactivity disorder
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Article
DE attention-deficit/hyperactivity disorder; autism; DAMP; motor
coordination
ID DEFICIT-HYPERACTIVITY DISORDER; DEVELOPMENTAL COORDINATION DISORDER;
RECIPROCAL SOCIAL-BEHAVIOR; NEUROLOGICAL SOFT SIGNS; POPULATION-BASED
SAMPLE; DEFINED ADHD SUBTYPES; LOW-BIRTH-WEIGHT; DSM-IV; LATENT CLASS;
GENERAL-POPULATION
AB Objective: To investigate the relation between parent reports of motor problems and clinically significant autistic symptoms in children with and without attention-deficit/hyperactivity disorder (ADHD). Method: Subjects were male (n = 521) and female (n = 330) twins from an epidemiological study of ADHD, ages 7 to 19 years at assessment using the Child Behavior Checklist and semistructured psychiatric diagnostic interviews. Parent-rated Social Responsiveness Scale questionnaires were returned for 62% of 1,647 individuals who participated in interviews. After exclusion of subjects with incomplete data or evidence of mental retardation, 851 subjects (52%) were available for the present study analysis. Each subject was classified by DSM-IVADHD subtype and assigned to one of seven population-defined ADHD subtypes based on latent class analysis of DSM-IVADHD symptoms. Within each ADHD subtype, we examined the relation between Child Behavior Checklist motor problem endorsement and elevated autistic symptoms on the Social Responsiveness Scale. Results: Motor problems and high levels of autistic traits were most common in individuals with combined-type ADHD. Within each of the clinically relevant DSM-IV and latent class ADHD subtypes, individuals with the combination of motor problems and ADHD were more likely to have high levels of autistic traits than those with ADHD alone. Conclusions: Children with the combination of ADHD and parent-reported motor coordination deficits have elevated levels of autistic symptoms. Targeted treatment and prevention interventions may be warranted. The exclusion criteria for DSM-IVADHD should be revised to reflect these population-based findings.
C1 [Reiersen, Angela M.; Constantino, John N.; Todd, Richard D.] Washington Univ, Sch Med, Dept Psychiat, St Louis, MO 63110 USA.
RP Reiersen, AM (reprint author), Washington Univ, Sch Med, Dept Psychiat, Box 8134,660 S Euclid Ave, St Louis, MO 63110 USA.
EM reiersea@psychiatry.wustl.edu
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NR 66
TC 41
Z9 42
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD JUN
PY 2008
VL 47
IS 6
BP 662
EP 672
DI 10.1097/CHI.0b013e31816bff88
PG 11
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 304LM
UT WOS:000256111000009
PM 18434922
ER
PT J
AU Bolte, S
Holtmann, M
Poustka, F
AF Boelte, Sven
Holtmann, Martin
Poustka, Fritz
TI The Social Communicaton Questionnaire (SCQ) as a screener for autism
spectrum disorders: Additional evidence and cross-cultural validity
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Letter
C1 [Boelte, Sven; Holtmann, Martin; Poustka, Fritz] Univ Frankfurt, Dept Child & Adolescent Psychiat & Psychotherapy, Frankfurt, Germany.
RP Bolte, S (reprint author), Univ Frankfurt, Dept Child & Adolescent Psychiat & Psychotherapy, Frankfurt, Germany.
CR Berument SK, 1999, BRIT J PSYCHIAT, V175, P444, DOI 10.1192/bjp.175.5.444
Bolte S., 2006, FRAGEBOGEN SOZIALEN
Bolte S, 2000, DIAGNOSTICA, V46, P149, DOI 10.1026//0012-1924.46.3.149
Chandler S, 2007, J AM ACAD CHILD PSY, V46, P1324, DOI 10.1097/chi.0b013e31812f7d8d
Rutter M., 2003, SOCIAL COMMUNICATION
NR 5
TC 16
Z9 16
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD JUN
PY 2008
VL 47
IS 6
BP 719
EP 720
DI 10.1097/CHI.0b013e31816c42bd
PG 2
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 304LM
UT WOS:000256111000015
PM 18496329
ER
PT J
AU Charman, T
Chandler, S
Baird, G
Simonoff, E
Loucas, T
Meldrum, D
Scott, M
Pickles, A
AF Charman, Tony
Chandler, Susie
Baird, Gillian
Simonoff, Emily
Loucas, Tom
Meldrum, David
Scott, Mimi
Pickles, Andrew
TI The Social Communicaton Questionnaire (SCQ) as a screener for autism
spectrum disorders: Additional evidence and cross-cultural validity -
Reply
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Letter
C1 [Charman, Tony; Chandler, Susie] UCL Inst Child Hlth, London, England.
[Baird, Gillian] Guys & St Thomas NHS Fdn Trust, London, England.
[Simonoff, Emily] Kings Coll London, Inst Psychiat, London WC2R 2LS, England.
[Loucas, Tom] Univ Reading, Sch Psychol & Clin Language Sci, Reading, Berks, England.
[Meldrum, David] Chatswood Assessment Ctr, Sydney, NSW, Australia.
[Pickles, Andrew] Univ Manchester, Div Epidemiol & Hlth Sci, Manchester, Lancs, England.
RP Charman, T (reprint author), UCL Inst Child Hlth, London, England.
RI Pickles, Andrew/A-9625-2011; Simonoff, Emily/B-7593-2011; Charman,
Tony/A-2085-2014
OI Pickles, Andrew/0000-0003-1283-0346; Charman, Tony/0000-0003-1993-6549
CR Berument SK, 1999, BRIT J PSYCHIAT, V175, P444, DOI 10.1192/bjp.175.5.444
Bolte S, 2000, DIAGNOSTICA, V46, P149, DOI 10.1026//0012-1924.46.3.149
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Rutter M., 2003, SOCIAL COMMUNICATION
NR 6
TC 0
Z9 0
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD JUN
PY 2008
VL 47
IS 6
BP 720
EP 721
DI 10.1097/CHI.0b013e31816c42cf
PG 2
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 304LM
UT WOS:000256111000016
ER
PT J
AU Noordhof, P
AF Noordhof, Paul
TI Expressive perception as projective imagining
SO MIND & LANGUAGE
LA English
DT Article
ID MUSIC; EMOTION; ART
AB I argue that our experience of expressive properties (such as the joyfulness or sadness of a piece of music) essentially involves the sensuous imagination (through simulation) of an emotion-guided process which would result in the production of the properties which constitute the realisation of the expressive properties experienced. I compare this proposal with arousal theories, Wollheim's Freudian account, and other more closely related theories appealing to imagination such as Kendall Walton's. I explain why the proposal is most naturally developed in terms of simulation and briefly comment upon the impact of work on cross-cultural perception of facial expression, modularity and autism for the proposal.
C1 Univ York, Dept Philosophy, York YO10 5DD, N Yorkshire, England.
RP Noordhof, P (reprint author), Univ York, Dept Philosophy, York YO10 5DD, N Yorkshire, England.
EM pjpn500@york.ac.uk
RI Noordhof, Paul/H-1604-2013
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NR 70
TC 5
Z9 5
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0268-1064
J9 MIND LANG
JI Mind Lang.
PD JUN
PY 2008
VL 23
IS 3
BP 329
EP 358
DI 10.1111/j.1468-0017.2008.00346.x
PG 30
WC Linguistics; Psychology, Experimental
SC Linguistics; Psychology
GA 301WB
UT WOS:000255925600007
ER
PT J
AU Hammond, P
Forster-Gibson, C
Chudley, AE
Allanson, JE
Hutton, TJ
Farrell, SA
McKenzie, J
Holden, J
Lewis, MES
AF Hammond, P.
Forster-Gibson, C.
Chudley, A. E.
Allanson, J. E.
Hutton, T. J.
Farrell, S. A.
McKenzie, J.
Holden, J. J. A.
Lewis, M. E. S.
TI Face-brain asymmetry in autism spectrum disorders
SO MOLECULAR PSYCHIATRY
LA English
DT Article
DE autism; face-brain asymmetry; dense surface-modelling; dysmorphology;
endophenotype
ID PERVASIVE DEVELOPMENTAL DISORDERS; FACIAL MORPHOLOGY; LANGUAGE DISORDER;
SHAPE; INDIVIDUALS; MRI
AB The heterogeneity of autism spectrum disorders (ASDs) confounds attempts to identify causes and pathogenesis. Identifiable endophenotypes and reliable biomarkers within ASDs would help to focus molecular research and uncover genetic causes and developmental mechanisms. We used dense surface-modelling techniques to compare the facial morphology of 72 boys with ASD and 128 first-degree relatives to that of 254 unrelated controls. Pattern-matching algorithms were able to discriminate between the faces of ASD boys and those of matched controls (AUC = 0.82) and also discriminate between the faces of unaffected mothers of ASD children and matched female controls (AUC = 0.76). We detected significant facial asymmetry in boys with ASD (P < 0.01), notably depth-wise in the supra-and periorbital regions anterior to the frontal pole of the right hemisphere of the brain. Unaffected mothers of children with ASD display similar significant facial asymmetry, more exaggerated than that in matched controls (P < 0.03) and, in particular, show vertical asymmetry of the periorbital region. Unaffected fathers of children with ASD did not show facial asymmetry to a significant degree compared to controls. Two thirds of unaffected male siblings tested were classified unseen as more facially similar to unrelated boys with ASD than to unrelated controls. These unaffected male siblings and two small groups of girls with ASD and female siblings, all show overall directional asymmetry, but without achieving statistical significance in two-tailed t-tests of individual asymmetry of ASD family and matched control groups. We conclude that previously identified right dominant asymmetry of the frontal poles of boys with ASD could explain their facial asymmetry through the direct effect of brain growth. The atypical facial asymmetry of unaffected mothers of children with ASD requires further brain studies before the same explanation can be proposed. An alternative explanation, not mutually exclusive, is a simultaneous and parallel action on face and brain growth by genetic factors. Both possibilities suggest the need for coordinated face and brain studies on ASD probands and their first-degree relatives, especially on unaffected mothers, given that their unusual facial asymmetry suggests an ASD susceptibility arising from maternal genes.
C1 [Hammond, P.] UCL, Inst Child Hlth, London WC1N 1EH, England.
[Hammond, P.; Hutton, T. J.] UCL, Eastman Dent Inst, London WC1N 1EH, England.
[Forster-Gibson, C.; McKenzie, J.; Holden, J. J. A.] Queens Univ, Kingston, ON, Canada.
[Chudley, A. E.] Univ Manitoba, Dept Pediat & Child Hlth, Winnipeg, MB R3T 2N2, Canada.
[Allanson, J. E.] Univ Ottawa, Childrens Hosp Eastern Ontario, Dept Genet, Ottawa, ON, Canada.
[Farrell, S. A.] Credit Valley Hosp, Mississauga, ON, Canada.
[Lewis, M. E. S.] Univ British Columbia, Child & Family Res Inst, Dept Med Genet, Vancouver, BC V5Z 1M9, Canada.
RP Hammond, P (reprint author), UCL, Inst Child Hlth, 30 Guilford St, London WC1N 1EH, England.
EM p.hammond@ucl.ac.uk; sume@interchange.ubc.ca
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NR 26
TC 34
Z9 35
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1359-4184
J9 MOL PSYCHIATR
JI Mol. Psychiatr.
PD JUN
PY 2008
VL 13
IS 6
BP 614
EP 623
DI 10.1038/mp.2008.18
PG 10
WC Biochemistry & Molecular Biology; Neurosciences; Psychiatry
SC Biochemistry & Molecular Biology; Neurosciences & Neurology; Psychiatry
GA 302FE
UT WOS:000255953400006
PM 18317467
ER
PT J
AU Abrahams, BS
Geschwind, DH
AF Abrahams, Brett S.
Geschwind, Daniel H.
TI Advances in autism genetics: on the threshold of a new neurobiology (vol
9, pg 341, 2008)
SO NATURE REVIEWS GENETICS
LA English
DT Correction
CR Abrahams BS, 2008, NAT REV GENET, V9, P341, DOI 10.1038/nrg2346
NR 1
TC 2
Z9 2
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1471-0056
J9 NAT REV GENET
JI Nat. Rev. Genet.
PD JUN
PY 2008
VL 9
IS 6
PG 1
WC Genetics & Heredity
SC Genetics & Heredity
GA 302FF
UT WOS:000255953500017
ER
PT J
AU Garbett, K
Ebert, PJ
Mitchell, A
Lintas, C
Manzi, B
Mirnics, K
Persico, AM
AF Garbett, Krassimira
Ebert, Philip J.
Mitchell, Amanda
Lintas, Carla
Manzi, Barbara
Mirnics, Karoly
Persico, Antonio M.
TI Immune transcriptome alterations in the temporal cortex of subjects with
autism
SO NEUROBIOLOGY OF DISEASE
LA English
DT Article
DE DNA microarray; gene expression; transcriptoine; autism; qPCR; post
mortem; temporal cortex
ID SPECTRUM DISORDERS; POSTMORTEM BRAIN; EXPRESSION; GENE; INFECTION;
SCHIZOPHRENIA; ASSOCIATION; ACTIVATION; PROTEINS; AMYGDALA
AB Autism is a severe disorder that involves both genetic and environmental factors. Expression profiling of the superior temporal gyrus of six autistic subjects and matched controls revealed increased transcript levels of many immune system-related genes. We also noticed changes in transcripts related to cell communication, differentiation, cell cycle regulation and chaperone systems. Critical expression changes were confirmed by qPCR (BCL6, CHI3L1, CYR61, IF116, IFITM3, MAP2K3, PTDSR, RFX4, SPP1, RELN, NOTCH2, RIT1, SFN, GADD45B, HSPA6, HSPB8 and SERPINH1). Overall, these expression patterns appear to be more associated with the late recovery phase of autoimmune brain disorders, than with the innate immune response characteristic of neurodegenerative diseases. Moreover, a variance-based analysis revealed much greater transcript variability in brains from autistic subjects compared to the control group, suggesting that these genes may represent autism susceptibility genes and should be assessed in follow-up genetic studies. (C) 2008 Elsevier Inc. All rights reserved.
C1 [Garbett, Krassimira; Ebert, Philip J.; Mitchell, Amanda; Mirnics, Karoly] Vanderbilt Univ, Dept Psychiat, Nashville, TN 37203 USA.
[Lintas, Carla; Persico, Antonio M.] Univ Campus Biomed, Lab Mol Psychiat & Neurogenet, Rome, Italy.
[Lintas, Carla; Persico, Antonio M.] IRCCS Fdn Santa Lucia, Dept Expt Neurosci, Lab Mol Psychiat & Psychiat Genet, Rome, Italy.
[Manzi, Barbara] Univ Roma Tor Vergata, Dept Child Neuropsychiat, Rome, Italy.
[Mirnics, Karoly] Vanderbilt Univ, Vanderbilt Kennedy Ctr Res Human Dev, Nashville, TN 37203 USA.
RP Mirnics, K (reprint author), Vanderbilt Univ, Dept Psychiat, 8130A MRB 3,465 21st Ave S, Nashville, TN 37203 USA.
EM karoly.mirnics@vanderbilt.edu; a.persico@unicampus.it
RI Mirnics, Karoly/E-6730-2010
OI Mirnics, Karoly/0000-0002-5521-0254
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NR 41
TC 131
Z9 140
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0969-9961
J9 NEUROBIOL DIS
JI Neurobiol. Dis.
PD JUN
PY 2008
VL 30
IS 3
BP 303
EP 311
DI 10.1016/j.nbd.2008.01.012
PG 9
WC Neurosciences
SC Neurosciences & Neurology
GA 306WM
UT WOS:000256278600003
PM 18378158
ER
PT J
AU Hrdlicka, M
AF Hrdlicka, Michal
TI Structural neuroimaging in autism
SO NEUROENDOCRINOLOGY LETTERS
LA English
DT Review
DE autism; MRI; brain volume; cerebellum; caudate nucleus; thalamus; corpus
callosum; amygdala; hippocampus
ID RESONANCE-IMAGING MRI; CORPUS-CALLOSUM; SPECTRUM DISORDERS;
INFANTILE-AUTISM; YOUNG-CHILDREN; CEREBELLAR HYPOPLASIA;
CAUDATE-NUCLEUS; AMYGDALA VOLUME; BASAL GANGLIA; BRAIN VOLUME
AB Structural neuroimaging studies done by means of magnetic resonance imaging (MRI) have provided important insights into the neurobiological basis for autism. The aim of this article is to review the current state of knowledge regarding structural brain abnormalities in autism. Results of MRI studies dealing with total brain volume, the volume of the cerebellum, caudate nucleus, thalamus, amygdala, hippocampus and the area of the corpus callosum are summarized. Existing research suggests that autistic individuals have larger total brain, cerebellar and caudate nucleus volumes; however, the area of the corpus callosum is reduced. Results of studies involving the amygdala and hippocampus volume in autistic subjects remain inconsistent and no changes have been detected in thalamic volume.
C1 [Hrdlicka, Michal] Charles Univ Prague, Fac Med 2, Dept Child Psychiat, Prague 15006, Czech Republic.
[Hrdlicka, Michal] Charles Univ Prague, Fac Med 1, Prague 12108, Czech Republic.
RP Hrdlicka, M (reprint author), Charles Univ Prague, Fac Med 2, Dept Child Psychiat, V Uvalu 84, Prague 15006, Czech Republic.
EM michal.hrdlicka@lfmotol.cuni.cz
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NR 71
TC 15
Z9 15
PU MAGHIRA & MAAS PUBLICATIONS
PI STOCKHOLM
PA PO BOX 26132, S-100 41 STOCKHOLM, SWEDEN
SN 0172-780X
J9 NEUROENDOCRINOL LETT
JI Neuroendocrinol. Lett.
PD JUN
PY 2008
VL 29
IS 3
BP 281
EP 286
PG 6
WC Endocrinology & Metabolism; Neurosciences
SC Endocrinology & Metabolism; Neurosciences & Neurology
GA 326RY
UT WOS:000257677800001
PM 18580841
ER
PT J
AU Croonenberghs, J
Spaas, K
Wauters, A
Verkerk, R
Scharpe, S
Deboutte, D
Maes, M
AF Croonenberghs, Jan
Spaas, Kristien
Wauters, Annick
Verkerk, Robert
Scharpe, Simon
Deboutte, Dirk
Maes, Michael
TI Faulty serotonin - DHEA interactions in autism: results of the
5-hydroxytryptophan challenge test
SO NEUROENDOCRINOLOGY LETTERS
LA English
DT Article
DE autism; serotonin; cortisol; DHEA; L-5-hydroxytryptophan; neurogenesis;
neurodegeneration; neurotoxic; stress
ID PERINATAL RISK-FACTORS; DEHYDROEPIANDROSTERONE-SULFATE;
HIPPOCAMPAL-NEURONS; CEREBROSPINAL-FLUID; OXIDATIVE STRESS;
SEX-DIFFERENCES; DISORDERS; CORTISOL; NEUROSTEROIDS; DISTURBANCES
AB BACKGROUND: Autism is accompanied by peripheral and central disorders in the metabolism of serotonin (5-HT). The present study examines plasma dehydroepiandrosterone-sulphate (DHEA-S) and the cortisol/DHEA-S ratio following administration of L-5-hydroxytryptophan (5-HTP), the direct precursor of 5-HT, to autistic patients.
METHODS: Plasma DHEA-S levels were determined both before and after administration of 5-HTP or placebo, on two consecutive days in a single blind order in 18 male autistic patients and 22 matched healthy controls.
RESULTS: The 5-HTP-induced DHEA-S responses were significantly higher in autistic patients than in controls. In baseline conditions, the cortisol/DHEA-S ratio was significantly higher in autistic patients than in controls. Discussion: The results suggest that autism is accompanied by a major disequilibrium in the serotonergic system. The increased Cortisol (neurotoxic) versus DHEA-S (neuroprotective) ratio suggests that an increased neurotoxic potential occurs in autism.
CONCLUSIONS: It is concluded that a disequilibrium in the peripheral and central turnover of serotonin and an increased neurotoxic capacity by glucocorticoids are important pathways in autism.
C1 [Croonenberghs, Jan; Spaas, Kristien; Deboutte, Dirk] Univ Antwerp, Fac Med, AZ Middelheim, Univ Ctr Child & Adolescent Psychiat, B-2020 Antwerp, Belgium.
[Wauters, Annick] AZ Middelheim, Lab Clin Biol, Antwerp, Belgium.
[Verkerk, Robert; Scharpe, Simon] Univ Antwerp, Dept Med Biochem, Edegem, Belgium.
[Maes, Michael] Clin Res Ctr Mental Hlth, Antwerp, Belgium.
RP Croonenberghs, J (reprint author), Univ Antwerp, Fac Med, AZ Middelheim, Univ Ctr Child & Adolescent Psychiat, Lindendreef 1, B-2020 Antwerp, Belgium.
EM jan.croonenberghs@zna.be
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NR 45
TC 11
Z9 12
PU MAGHIRA & MAAS PUBLICATIONS
PI STOCKHOLM
PA PO BOX 26132, S-100 41 STOCKHOLM, SWEDEN
SN 0172-780X
J9 NEUROENDOCRINOL LETT
JI Neuroendocrinol. Lett.
PD JUN
PY 2008
VL 29
IS 3
BP 385
EP 390
PG 6
WC Endocrinology & Metabolism; Neurosciences
SC Endocrinology & Metabolism; Neurosciences & Neurology
GA 326RY
UT WOS:000257677800017
PM 18580847
ER
PT J
AU Schendel, D
Bhasin, TK
AF Schendel, Diana
Bhasin, Tanya Karapurkar
TI Birth weight and gestational age characteristics of children with
autism, including a comparison with other developmental disabilities
SO PEDIATRICS
LA English
DT Article
DE autism; birth weight; gestational age; developmental disabilities;
gender distribution
ID PERINATAL RISK-FACTORS; INFANTILE-AUTISM; OBSTETRIC COMPLICATIONS;
NEONATAL COMPLICATIONS; EPIDEMIOLOGIC SURVEY; ASPERGER-SYNDROME; ADULT
DISEASE; UNITED-STATES; DISORDER; FETAL
AB OBJECTIVES. The objectives of this study were to compare the birth weight and gestational age distributions and prevalence rates of autism with those of other developmental disabilities and to estimate the birth weight - and gestational age-specific risks for autism.
METHODS. For the first objective, a retrospective cohort of children born in Atlanta, Georgia, in 1981-1993 who survived to 3 years of age was identified through vital records. Children in the cohort who had developmental disabilities (autism, mental retardation, cerebral palsy, hearing loss, or vision impairment) and were still residing in metropolitan Atlanta at 3 to 10 years of age were identified through the Metropolitan Atlanta Developmental Disabilities Surveillance Program. A nested case-control sample from the cohort was used for the second objective; all cohort children identified with autism were case participants, and control participants were cohort children who were not identified as having developmental disabilities or receiving special education services.
RESULTS. The prevalence of autism in low birth weight or preterm children was markedly lower than those of other developmental disabilities. In multivariate analyses, birth weight of <2500 g and preterm birth at <33 weeks' gestation were associated with an approximately twofold increased risk for autism, although the magnitude of risk from these factors varied according to gender (higher in girls) and autism subgroup (higher for autism accompanied by other developmental disabilities). For example, a significant fourfold increased risk was observed in low birth weight girls for autism accompanied by mental retardation, whereas there was no significantly increased risk observed in low birth weight boys for autism alone.
CONCLUSIONS. Gender and autism subgroup differences in birth weight and gestational age, resulting in lower gender ratios with declining birth weight or gestational age across all autism subgroups, might be markers for etiologic heterogeneity in autism.
C1 [Schendel, Diana; Bhasin, Tanya Karapurkar] Ctr Dis Control & Prevent, Natl Ctr Birth Defects & Dev Disabil, Atlanta, GA 30333 USA.
RP Schendel, D (reprint author), Ctr Dis Control & Prevent, Natl Ctr Birth Defects & Dev Disabil, 1600 Clifton Rd,Mail Stop E-80, Atlanta, GA 30333 USA.
EM dschendel@cdc.gov
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NR 61
TC 72
Z9 73
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD JUN
PY 2008
VL 121
IS 6
BP 1155
EP 1164
DI 10.1542/peds.2007-1049
PG 10
WC Pediatrics
SC Pediatrics
GA 307IX
UT WOS:000256313700046
PM 18519485
ER
PT J
AU Altevogt, BM
Hanson, SL
Leshner, AI
AF Altevogt, Bruce M.
Hanson, Sarah L.
Leshner, Alan I.
TI Autism and the environment: Challenges and opportunities for research
SO PEDIATRICS
LA English
DT Review
DE autism; autism spectrum disorder; environmental biology; epidemiology;
research agenda; public-private partnership
ID DISORDERS; TRENDS
AB Autism spectrum disorder is a complex developmental disorder that dramatically affects the lives of patients and their families and the broader community. The causes of autism are unknown; however, evidence increasingly suggests that a complex interplay among environmental stressors, genetic mutations, and other biological factors likely plays a significant role in the development and/or progression of autism spectrum disorder. On April 18 and 19, 2007, the Institute of Medicine's Forum on Neuroscience and Nervous System Disorders hosted a workshop to provide a venue to bring together scientists; major sponsors of autism-related research; and members of the autism patient, family, and advocacy community to discuss the most promising and urgent scientific questions and opportunities. Broad participation by the autism community enriched the meeting significantly by contributing a valuable and personal perspective that is often missing from scientific meetings. It also began a much improved public-private partnership in which all stakeholders are represented. On the basis of the presentations and the discussions that followed, an array of important scientific opportunities were identified in 5 general categories: (1) opportunities to advance clinical research; (2) opportunities to enhance epidemiologic studies; (3) opportunities to improve the understanding of autism's pathology and etiology; (4) tools and infrastructure needs; and (5) opportunities for public-private partnerships. This workshop demonstrated that full public engagement can greatly enhance activities such as this workshop and its outcomes. Furthermore, we expect that this listing of scientific challenges, needs, and opportunities will help to frame a more comprehensive research agenda.
C1 [Altevogt, Bruce M.; Hanson, Sarah L.; Leshner, Alan I.] Inst Med, Washington, DC 20001 USA.
[Leshner, Alan I.] Amer Assoc Advancement Sci, Washington, DC USA.
RP Altevogt, BM (reprint author), Inst Med, 500 5th St NW, Washington, DC 20001 USA.
EM baltevogt@nas.edu
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NR 14
TC 24
Z9 24
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD JUN
PY 2008
VL 121
IS 6
BP 1225
EP 1229
DI 10.1542/peds.2007-3000
PG 5
WC Pediatrics
SC Pediatrics
GA 307IX
UT WOS:000256313700054
PM 18519493
ER
PT J
AU Sparaci, L
AF Sparaci, Laura
TI Embodying gestures: The Social Orienting Model and the study of early
gestures in autism
SO PHENOMENOLOGY AND THE COGNITIVE SCIENCES
LA English
DT Article
DE Autism; Social Orienting Model; Gestures; Intersubjectivity; Embodiment
ID MIRROR NEURONS; FUNCTIONAL-ORGANIZATION; SPECTRUM DISORDERS; INFERIOR
AREA-6; MACAQUE MONKEY; DOWNS-SYNDROME; CHILDREN; LANGUAGE; MIND;
COMMUNICATION
AB Autistic spectrum disorders impair the ability to interact socially. Detecting and understanding their onset is not only an empirical enterprise, but also a theoretical one, often linked to studies on intersubjectivity. Different theoretical perspectives have been elaborated in the past to account for the deficit. The main purpose of this paper is to reinforce and offer empirical grounding to a recent approach, termed Social Orienting Model, by presenting the main theoretical approaches to autism and contrasting them to this view, as well as considering its possible effect on empirical research, focusing on current literature analyzing gestures in children with autism.
C1 Univ Siena, I-53100 Siena, Italy.
RP Sparaci, L (reprint author), Univ Siena, Via Laterina 8, I-53100 Siena, Italy.
EM laurasparaci@hotmail.com
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NR 96
TC 4
Z9 5
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1568-7759
EI 1572-8676
J9 PHENOMENOL COGN SCI
JI Phenomenol. Cogn. Sci.
PD JUN
PY 2008
VL 7
IS 2
BP 203
EP 223
DI 10.1007/s11097-007-9084-9
PG 21
WC Philosophy
SC Philosophy
GA 418WK
UT WOS:000264180100004
ER
PT J
AU Ashoori, A
Jankovic, J
AF Ashoori, A.
Jankovic, J.
TI Mozart's movements and behaviour: a case of Tourette's syndrome?
SO POSTGRADUATE MEDICAL JOURNAL
LA English
DT Review
ID ALCOHOL DEPENDENCE; MEDICAL HISTORY; DISORDER; SAVANT; AUTISM; MUSIC;
BRAIN; ASSOCIATION; PREVALENCE; CHILDREN
AB In this review, we intend to explore the often asked question: "Did Mozart have Tourette's syndrome?'' Although there are numerous reports attributing Mozart's peculiar personality and behaviour to a spectrum of neurobehavioural disorders such as Tourette's syndrome, autistic disorder, Asperger's syndrome, attention deficit hyperactivity disorder, obsessive-compulsive disorder and paediatric autoimmune neuropsychiatric disorders associated with streptococcal infection, the evidence for any of these disorders is lacking. Whether Mozart's behaviour was nothing more than a reflection of his unique personality or a more complex neurological disorder, aggravated later in life by enormous demands by his father and society, his behaviour has been the subject of many biographies. It will also remain unknown to what extent his accomplishments and failures were shaped by his childhood experiences, pressured lifestyle, and his innate genius and extraordinary talent. Lessons from his life may have important implications for other gifted individuals and savants whose special attributes may lead them to succeed or, on the other hand, suppress their emotional growth and make them more vulnerable to stress and failure.
C1 Baylor Coll Med, Parkinsons Dis Ctr, Dept Neurol, Houston, TX 77030 USA.
Baylor Coll Med, Movement Disorder Clin, Dept Neurol, Houston, TX 77030 USA.
RP Jankovic, J (reprint author), Baylor Coll Med, Parkinsons Dis Ctr, Dept Neurol, Smith Tower,Suite 1801,6550 Fannin, Houston, TX 77030 USA.
EM josephj@bcm.tmc.edu
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NR 69
TC 1
Z9 1
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0032-5473
J9 POSTGRAD MED J
JI Postgrad. Med. J.
PD JUN
PY 2008
VL 84
IS 992
BP 313
EP 317
DI 10.1136/jnnp.2007.114520
PG 5
WC Medicine, General & Internal
SC General & Internal Medicine
GA 328IB
UT WOS:000257790500007
PM 18644922
ER
PT J
AU Vincent, JB
Choufani, S
Horike, S
Stachowriak, B
Li, M
Dill, FJ
Marshal, C
Hrynchak, M
Pewsey, E
Ukadike, KC
Friedman, JM
Srivastava, AK
Scherer, SW
AF Vincent, John B.
Choufani, Sanaa
Horike, Shin-ichi
Stachowriak, Beata
Li, Martin
Dill, Fred J.
Marshal, Christian
Hrynchak, Monica
Pewsey, Elizabeth
Ukadike, Kennedy C.
Friedman, Jan M.
Srivastava, Anand K.
Scherer, Stephen W.
TI A translocation t(6;7)(p11-p12;q22) associated with autism and mental
retardation: localization and identification of candidate genes at the
breakpoints
SO PSYCHIATRIC GENETICS
LA English
DT Article
DE autism; chromosome 7; dystonin; mental retardation; pentraxin;
translocation
ID CHROMOSOME-7; DISORDER; DYSTONIN; CLONING; ARRAYS; NARP; 7Q31
AB Objectives Our aim is to use information from cytogenetic anomalies to identify candidate genes for autism.
Methods We have identified a male patient with mental retardation and autism who has a balanced translocation involving chromosomes 6 and 7, described as t(6;7)(P11-p12;q22). This translocation was inherited from an apparently normal father.
Results Using fluorescence in situ hybridization, we have localized the breakpoints on both the chromosomes; and using bioinformatic genomic analysis, we have identified a number of potential candidate genes at these loci. These include the neural pentraxin 2 gene, NPTX2, and a novel gene encoding a transmembrane protein, TMEM130, which contains a polycystic kidney domain on 7q22. On 6p12 the breakpoint directly interrupts isoform 2 of the human homologue of the mouse dystonin gene. We also performed a 250 K single nucleotide polymorphism microarray analysis and comparative genomic hybridization using a bacterial artificial chromosome microarray to look for minor genomic deletions or duplications in the proband's DNA. The single nucleotide polymorphism microarray analysis identified a number of copy number variants, remote from the translocation breakpoints, containing potential candidate genes.
Conclusion It is conceivable that one or more of the copy number variant regions or either of the two breakpoint locations and the dystonin gene, in particular, may be a new locus for a form of mental retardation, which may also include autistic features. Psychiatr Genet 18:101-109 (c) 2008 Wolters Kluwer Health vertical bar Lippincott Williams & Wilkins.
C1 [Vincent, John B.; Stachowriak, Beata] Ctr Addict & Mental Hlth, Neurogenet Sect, Toronto, ON M5T 1R8, Canada.
[Choufani, Sanaa; Horike, Shin-ichi; Li, Martin; Marshal, Christian; Scherer, Stephen W.] Hosp Sick Children, Program Genet & Genom Biol, Toronto, ON M5G 1X8, Canada.
[Dill, Fred J.; Friedman, Jan M.] Univ British Columbia, Dept Med Genet, Vancouver, BC, Canada.
[Pewsey, Elizabeth] Childrens & Womens Hlth Ctr British Columbia, Prov Med Genet Programme, Columbia, BC, Canada.
[Hrynchak, Monica] Royal Columbian Hosp, Cytogenet Lab, Columbia, BC, Canada.
[Ukadike, Kennedy C.; Srivastava, Anand K.] Greenwood Genet Ctr, JC Self Res Inst Human Genet, Greenwood, SC 29646 USA.
RP Vincent, JB (reprint author), Ctr Addict & Mental Hlth, Neurogenet Sect, R30,Clarke Site,250 Coll St, Toronto, ON M5T 1R8, Canada.
EM john_vincent@camh.net
RI Howe, Jennifer/I-9013-2012; Scherer, Stephen /B-3785-2013
OI Scherer, Stephen /0000-0002-8326-1999
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NR 24
TC 5
Z9 6
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0955-8829
J9 PSYCHIAT GENET
JI Psychiatr. Genet.
PD JUN
PY 2008
VL 18
IS 3
BP 101
EP 109
DI 10.1097/YPG.0b013e3282f97df7
PG 9
WC Genetics & Heredity; Neurosciences
SC Genetics & Heredity; Neurosciences & Neurology
GA 304QZ
UT WOS:000256125300001
PM 18496206
ER
PT J
AU Kato, C
Tochigi, M
Koishi, S
Kawakubo, Y
Yamamoto, K
Matsumoto, H
Hashimoto, O
Kim, SY
Watanabe, K
Kano, Y
Nanba, E
Kato, N
Sasaki, T
AF Kato, Chieko
Tochigi, Mamoru
Koishi, Shinko
Kawakubo, Yuki
Yamamoto, Kenji
Matsumoto, Hideo
Hashimoto, Ohiko
Kim, Soo-Yung
Watanabe, Keiichiro
Kano, Yukiko
Nanba, Eiji
Kato, Nobumasa
Sasaki, Tsukasa
TI Association study of the commonly recognized breakpoints in chromosome
15q11-q13 in Japanese autistic patients
SO PSYCHIATRIC GENETICS
LA English
DT Article
DE annyloid precursor protein-binding protein A2; Angelman syndrome;
autism; breakpoint; chromosome 15; Prader-Willi syndrome
ID HAPLOTYPE FREQUENCIES; DISORDERS; POPULATION; ANGELMAN; DELETION;
GENETICS; BLOCKS; ORIGIN
AB Objective Chromosome 15q11-q13 has been proposed to harbor a gene for autism susceptibility because deletions of the region lead to Prader-Willi syndrome and Angelman syndrome, whose phenotypes overlap with autism. These deletions generally occur with the use of three commonly recognized breakpoints (BP1, BP2, and BP3); therefore, it may be possible that genes located in the breakpoints are impaired and contribute to autism susceptibility. No study, however, has investigated the genetic association between the breakpoints and autism, to our knowledge. Here, we investigated the association between the common breakpoints of chromosome 15q11-q13 and autism in a Japanese population.
Methods We genotyped 12 single nucleotide polymorphisms (SNPs) in 166 patients with autistic disorder and 415 healthy controls. The SNPs are located in two additional distal breakpoints (BP4 and BP5), involved in duplications and triplications of the region, as well as in BP1 and BP3.
Results No significant difference was observed between the controls and patients in allelic frequencies or genotypic distributions of the 12 SNPs. In the analyses of the suggested five haplotypes, no significant difference between the controls and patients was observed in the distributions of any estimated haplotypes. When confining the patients to only males, a difference was observed in a two-marker haplotype in BP3 between the controls and patients (global permutation P value= 0.006), although the statistical level became insignificant after correction for multiple testing.
Conclusion This study provides no positive evidence of the association between the common breakpoints of chromosome 15q11-q13 and autism in the Japanese population.
C1 [Sasaki, Tsukasa] Univ Tokyo, Hlth Serv Ctr, Bunkyo Ku, Tokyo 1138655, Japan.
[Kato, Chieko; Tochigi, Mamoru; Kawakubo, Yuki; Kim, Soo-Yung; Watanabe, Keiichiro; Kano, Yukiko; Kato, Nobumasa; Sasaki, Tsukasa] Univ Tokyo, Grad Sch Med, Dept Neuropsychiat, Tokyo 1138655, Japan.
[Kim, Soo-Yung; Watanabe, Keiichiro; Kano, Yukiko] Tokyo Univ Hosp, Dept Child Psychiat, Bunkyo Ku, Tokyo 113, Japan.
[Koishi, Shinko; Yamamoto, Kenji; Matsumoto, Hideo] Tokai Univ, Sch Med, Dept Psychiat, Kanagawa 2591100, Japan.
[Hashimoto, Ohiko] Aino Univ, Dept Med Technol, Osaka, Japan.
[Nanba, Eiji] Tottori Univ, Ctr Gene Res, Tottori 680, Japan.
RP Sasaki, T (reprint author), Univ Tokyo, Hlth Serv Ctr, Bunkyo Ku, 7-3-1 Hongo, Tokyo 1138655, Japan.
EM psytokyo@yahoo.co.jp
CR Biederer T, 2000, J BIOL CHEM, V275, P39803, DOI 10.1074/jbc.C000656200
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NR 17
TC 1
Z9 1
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0955-8829
J9 PSYCHIAT GENET
JI Psychiatr. Genet.
PD JUN
PY 2008
VL 18
IS 3
BP 133
EP 136
DI 10.1097/YPG.0b013e3282fb0064
PG 4
WC Genetics & Heredity; Neurosciences
SC Genetics & Heredity; Neurosciences & Neurology
GA 304QZ
UT WOS:000256125300006
PM 18496211
ER
PT J
AU Tochigi, M
Kato, C
Ohashi, J
Koishi, S
Kawakubo, Y
Yamamoto, K
Matsumoto, H
Hashimoto, O
Kim, SY
Watanabe, K
Kano, Y
Nanba, E
Kato, N
Sasaki, T
AF Tochigi, Mamoru
Kato, Chieko
Ohashi, Jun
Koishi, Shinko
Kawakubo, Yuki
Yamamoto, Kenji
Matsumoto, Hideo
Hashimoto, Ohiko
Kim, Soo-Yung
Watanabe, Keiichiro
Kano, Yukiko
Nanba, Eiji
Kato, Nobumasa
Sasaki, Tsukasa
TI No association between the ryanodine receptor 3 gene and autism in a
Japanese population
SO PSYCHIATRY AND CLINICAL NEUROSCIENCES
LA English
DT Article
DE association study; autism; chromosome 15; ryanodine receptor; single
nucleotide polymorphism
ID HAPLOTYPE BLOCKS; DELETION; HYBRIDIZATION; DISORDERS; ANGELMAN; ORIGIN;
RYR3
AB Aim: Autism is a neurodevelopmental disorder with a complex genetic etiology. Chromosome 15q11-q14 has been proposed to harbor a gene for autism susceptibility because deletion of the region leads to Prader-Willi syndrome or Angelman syndrome, having phenotypic overlap with autism. Here we studied the association between autism and the ryanodine receptor 3 (RyR3) gene, which is located in the region. This is the first study, to our knowledge, that has investigated the association.
Methods: We genotyped 14 tag single nucleotide polymorphisms (SNPs) in 166 Japanese patients with autism and 375 controls.
Results: No significant difference was observed between the patients and controls in allelic frequencies or genotypic distributions of the 14 SNPs. Analysis after confining the subjects to males showed similar results.
Conclusions: The present study provides no positive evidence for the association between the RyR3 gene and autism in the Japanese population.
C1 [Sasaki, Tsukasa] Univ Tokyo, Hlth Serv Ctr, Bunkyo Ku, Tokyo 1138655, Japan.
[Tochigi, Mamoru; Kato, Chieko; Kawakubo, Yuki; Kim, Soo-Yung; Watanabe, Keiichiro; Kano, Yukiko; Kato, Nobumasa; Sasaki, Tsukasa] Univ Tokyo, Dept Neuropsychiat, Tokyo 1138655, Japan.
[Ohashi, Jun] Univ Tokyo, Dept Human Genet, Grad Sch Med, Tokyo 1138655, Japan.
[Koishi, Shinko; Yamamoto, Kenji; Matsumoto, Hideo] Tokai Univ, Sch Med, Dept Psychiat, Kanagawa 2591100, Japan.
[Hashimoto, Ohiko] Aino Univ, Dept Med Technol, Osaka, Japan.
[Nanba, Eiji] Tottori Univ, Ctr Gene Res, Tottori 680, Japan.
RP Sasaki, T (reprint author), Univ Tokyo, Hlth Serv Ctr, Bunkyo Ku, 7-3-1 Hongo, Tokyo 1138655, Japan.
EM psytokyo@yahoo.co.jp
CR Balschun D, 1999, EMBO J, V18, P5264, DOI 10.1093/emboj/18.19.5264
Barrett JC, 2005, BIOINFORMATICS, V21, P263, DOI 10.1093/bioinformatics/bth457
CHONG SS, 1994, AM J MED GENET, V51, P522, DOI 10.1002/ajmg.1320510447
Folstein SE, 2001, NAT REV GENET, V2, P943, DOI 10.1038/35103559
Freitag CM, 2007, MOL PSYCHIATR, V12, P2, DOI 10.1038/sj.mp.4001896
Gabriel SB, 2002, SCIENCE, V296, P2225, DOI 10.1126/science.1069424
Honda H, 1996, BRIT J PSYCHIAT, V169, P228, DOI 10.1192/bjp.169.2.228
Gibbs RA, 2003, NATURE, V426, P789, DOI 10.1038/nature02168
IZUTSU T, 2001, RINSYO SEISHIN IGAKU, V30, P525
KNOLL JHM, 1989, AM J MED GENET, V32, P285, DOI 10.1002/ajmg.1320320235
Kouzu Y, 2000, MOL BRAIN RES, V76, P142, DOI 10.1016/S0169-328X(99)00344-7
LEWONTIN RC, 1988, GENETICS, V120, P849
Nakashima Y, 1997, FEBS LETT, V417, P157, DOI 10.1016/S0014-5793(97)01275-1
Peters SU, 2004, CLIN GENET, V66, P530, DOI 10.1111/j.1399-0004.2004.00362.x
Purcell S, 2003, BIOINFORMATICS, V19, P149, DOI 10.1093/bioinformatics/19.1.149
SORRENTINO V, 1993, GENOMICS, V18, P163, DOI 10.1006/geno.1993.1446
Veltman MWM, 2004, EUR CHILD ADOLES PSY, V13, P42, DOI 10.1007/s00787-004-0354-6
Wang N, 2002, AM J HUM GENET, V71, P1227, DOI 10.1086/344398
Wang NJ, 2004, AM J HUM GENET, V75, P267, DOI 10.1086/422854
NR 19
TC 2
Z9 2
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1323-1316
J9 PSYCHIAT CLIN NEUROS
JI Psychiatry Clin. Neurosci.
PD JUN
PY 2008
VL 62
IS 3
BP 341
EP 344
DI 10.1111/j.1440-1819.2008.01802.x
PG 4
WC Clinical Neurology; Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 312RW
UT WOS:000256689500013
PM 18588595
ER
PT J
AU Belmonte, MK
AF Belmonte, Matthew K.
TI Does the experimental scientist have a "Theory of Mind"?
SO REVIEW OF GENERAL PSYCHOLOGY
LA English
DT Article; Proceedings Paper
CT Conference on From the Brain to Human Culture - Intersections Between
the Humanities and Neuroscience
CY APR 20-22, 2007
CL Lewisburg, PA
HO Bucknell Univ
DE theory of mind; cognitive literary theory; autism; schizophrenia;
narrative
ID NEURAL CIRCUITRY; AUTISTIC-CHILD; PERCEPTION; BRAIN; SCHIZOPHRENIA;
GAZE; REPRESENTATION; INTENTIONS; MOVEMENT
AB The concept of a "theory of mind" was widely used in developmental and evolutionary psychology and neuroscience in the wake of Premack and Woodruff's 1978 article "Does the Chimpanzee Have a Theory of Mind?" and Baron-Cohen, Leslie, and Frith's 1985 follow-up "Does the Autistic Child Have a 'Theory of Mind?"' The subsequent confluence of cognitive science and narrative theory brought "theory of mind" to literary critics. Only a very small set of people, however, have read both the neuropsychological and the literary texts on "theory of mind"; as a result of this lack of interdisciplinary expertise, the term has acquired subtly differing senses in the literary and neuroscientific communities. Because of this terminological slippage, neuroscientists and literary critics who argue in terms of "theory of mind" may believe that they are speaking with each other when they actually are speaking past each other. If proponents of cognitive literary theory are to realize the interdisciplinary fusion to which we aspire, then we must ensure that we speak in the same idiom.
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RP Belmonte, MK (reprint author), Cornell Univ, Dept Human Dev, G77 Martha Van Rensselaer Hall, Ithaca, NY 14853 USA.
EM belmonte@mit.edu
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NR 72
TC 6
Z9 7
PU EDUCATIONAL PUBLISHING FOUNDATION-AMERICAN PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST, NE, WASHINGTON, DC 20002-4242 USA
SN 1089-2680
J9 REV GEN PSYCHOL
JI Rev. Gen. Psychol.
PD JUN
PY 2008
VL 12
IS 2
BP 192
EP 204
DI 10.1037/1089-2680.12.2.192
PG 13
WC Psychology, Multidisciplinary
SC Psychology
GA 313YH
UT WOS:000256775400011
ER
PT J
AU Cheng, G
AF Cheng, Glen
TI CARING FOR NEW JERSEY'S CHILDREN WITH AUTISM: A MULTIFACETED STRUGGLE
FOR PARITY
SO RUTGERS LAW REVIEW
LA English
DT Review
ID SPECIAL-EDUCATION; PREVALENCE; LAWS; INTERVENTION; IMPACT
AB This Note will examine both health insurance coverage for children with autism in New Jersey under the federal and state Mental Health Parity statutes, Medicaid, and the State Children's Health Insurance Program (SCRIP), and special education coverage for children with autism in New Jersey under the Individuals with Disabilities Education Act (IDEA). This Note ultimately proposes that despite overlapping coverage in the various acts providing mental health, insurance and special education benefits for children with autism, the New Jersey Legislature should pass Assembly Bill 2238 in order to ensure the provision of urgently-needed therapies for children with autism.
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NR 117
TC 0
Z9 0
PU RUTGERS UNIV
PI NEWARK
PA SCHOOL LAW 123 WASHINGTON ST, NEWARK, NJ 07102 USA
SN 0036-0465
J9 RUTGERS LAW REV
JI Rutgers Law Rev.
PD SUM
PY 2008
VL 60
IS 4
BP 997
EP 1037
PG 41
WC Law
SC Government & Law
GA 384ZV
UT WOS:000261783600004
ER
PT J
AU Volker, MA
Lopata, C
AF Volker, Martin A.
Lopata, Christopher
TI Autism: A review of biological bases, assessment, and intervention
SO SCHOOL PSYCHOLOGY QUARTERLY
LA English
DT Article
DE autism; autistic spectrum; genetics; assessment; intervention
ID PERVASIVE DEVELOPMENTAL DISORDERS; PLATELET SEROTONIN LEVELS;
HIGH-FUNCTIONING AUTISM; YOUNG-CHILDREN; SPECTRUM DISORDERS; BEHAVIORAL
TREATMENT; LANGUAGE IMPAIRMENT; MENTAL-RETARDATION; ASPERGERS-SYNDROME;
BRAIN
AB The number of children classified with autism in US schools has risen sharply over the past decade. School psychologists are being called upon with increasing frequency to assist in the identification, assessment, and treatment of these children. The diagnostic complexities and heterogeneity of the disorder make dealing effectively with this condition a considerable challenge for school personnel. Additionally, the biological basis of the disorder necessitates the involvement of medical personnel in both assessment and intervention. This article reviews recent findings regarding the genetics of autism, associated neurological features, best practice assessment, and empirically supported interventions.
C1 [Volker, Martin A.] SUNY Buffalo, Grad Sch Educ, Dept Counseling, Sch & Educ Psychol, Buffalo, NY 14260 USA.
RP Volker, MA (reprint author), SUNY Buffalo, Grad Sch Educ, Dept Counseling, Sch & Educ Psychol, 409 Christopher Baldy Hall, Buffalo, NY 14260 USA.
EM mvolker@buffalo.edu
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NR 94
TC 12
Z9 12
PU AMER PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST NE, WASHINGTON, DC 20002-4242 USA
SN 1045-3830
J9 SCHOOL PSYCHOL QUART
JI Sch. Psychol. Q.
PD JUN
PY 2008
VL 23
IS 2
BP 258
EP 270
DI 10.1037/1045-3830.23.2.258
PG 13
WC Psychology, Educational
SC Psychology
GA 316BI
UT WOS:000256923500008
ER
PT J
AU Schwarte, AR
AF Schwarte, Andrea R.
TI Fragile X Syndrome
SO SCHOOL PSYCHOLOGY QUARTERLY
LA English
DT Article
DE Fragile X Syndrome; cognitive; neuropsychology; treatment; education;
recommendations
ID YOUNG MALES; MENTAL-RETARDATION; AUTISTIC BEHAVIOR; EEG FINDINGS; BOYS;
CHILDREN; PROFILES; LANGUAGE; MUTATION; COMMUNICATION
AB This article provides an overview of current research on Fragile X Syndrome, and how that knowledge can be used to guide successful intervention. The genetic etiology of Fragile X is reviewed and the physical, cognitive, adaptive, behavioral, and emotional phenotypes of children with the disorder are described, highlighting the differences in presentation among males and females. Comorbidity with autism is addressed. Typical strengths and weaknesses of children with Fragile X are delineated as a basis for educational assessment and intervention. A multidisciplinary approach to treatment is advocated, including medical management, educational accommodations, and speech-language and occupational therapy. The social and emotional challenges of those with the syndrome are considered in recommended interventions.
C1 [Schwarte, Andrea R.] St Vincents Hosp, Indiana Neurosci Inst, Indianapolis, IN 46260 USA.
RP Schwarte, AR (reprint author), St Vincents Hosp, Neuropsychol Dept, 2001 W 86th St,3rd Floor, Indianapolis, IN 46260 USA.
EM ARSchwar@stvincent.org
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NR 60
TC 4
Z9 4
PU AMER PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST NE, WASHINGTON, DC 20002-4242 USA
SN 1045-3830
J9 SCHOOL PSYCHOL QUART
JI Sch. Psychol. Q.
PD JUN
PY 2008
VL 23
IS 2
BP 290
EP 300
DI 10.1037/1045-3830.23.2.290
PG 11
WC Psychology, Educational
SC Psychology
GA 316BI
UT WOS:000256923500011
ER
PT J
AU Zak, PJ
AF Zak, Paul J.
TI The neurobiology of trust
SO SCIENTIFIC AMERICAN
LA English
DT Article
ID OXYTOCIN
AB The development of trust is essential for appropriate social interactions. Using an experimental task called the trust game, researchers have found that oxytocin, a hormone and neurochemical, enhances an individual's propensity to trust a stranger when that person exhibits non-threatening signals. Greater understanding of oxytocin's functions and interactions with other key brain chemicals could lead to insights into many disorders marked by impaired social interactions, such as autism and to find how people decide whether to trust a new acquaintance or potential business partner. According to evolutionary biologists, vasotocin appeared first in fish and it facilitates sexual reproduction by reducing a female's natural fear of being approached by a male when she is ovulating. It is observed that oxytocin rise boosts trust, increases trustworthiness and that flaws in the brain's ability to respond to oxytocin might contribute to social disorders.
C1 [Zak, Paul J.] Claremont Grad Univ, Ctr Neuroeconom Studies, Claremont, CA USA.
[Zak, Paul J.] Loma Linda Univ, Med Ctr, Loma Linda, CA 92350 USA.
RP Zak, PJ (reprint author), Claremont Grad Univ, Ctr Neuroeconom Studies, Claremont, CA USA.
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Zak PJ, 2007, PLOS ONE, V2, DOI 10.1371/journal.pone.0001128
NR 5
TC 24
Z9 24
PU SCI AMERICAN INC
PI NEW YORK
PA 415 MADISON AVE, NEW YORK, NY 10017 USA
SN 0036-8733
J9 SCI AM
JI Sci.Am.
PD JUN
PY 2008
VL 298
IS 6
BP 88
EP +
PG 6
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 305SF
UT WOS:000256197400038
PM 18642547
ER
PT J
AU Garwrylewski, A
AF Garwrylewski, Andrea
TI Inducing autism
SO SCIENTIST
LA English
DT News Item
NR 0
TC 0
Z9 0
PU SCIENTIST INC
PI PHILADELPHIA
PA 400 MARKET ST, STE 1250, PHILADELPHIA, PA 19106 USA
SN 0890-3670
J9 SCIENTIST
JI Scientist
PD JUN
PY 2008
VL 22
IS 6
BP 26
EP 27
PG 2
WC Information Science & Library Science; Multidisciplinary Sciences
SC Information Science & Library Science; Science & Technology - Other
Topics
GA 303FZ
UT WOS:000256027700018
ER
PT J
AU Ivanenko, A
Johnson, K
AF Ivanenko, Anna
Johnson, Kyle
TI Sleep Disturbances in Children With Psychiatric Disorders
SO SEMINARS IN PEDIATRIC NEUROLOGY
LA English
DT Review
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; DEFICIT HYPERACTIVITY
DISORDER; LIMB MOVEMENT-DISORDER; RESTLESS LEGS SYNDROME; PERVASIVE
DEVELOPMENTAL DISORDERS; CONTROLLED-RELEASE MELATONIN; ADOLESCENT
BIPOLAR DISORDER; MAJOR DEPRESSION; EEG SLEEP; YOUNG-ADULTS
AB Sleep disturbances are highly prevalent among children with psychiatric disorders, making recognition and management of pediatric sleep disorders an important step in improving treatment outcome and preventing relapse of mental illness. This chapter will review the research data on the epidemiology, clinical presentation and treatment approaches for sleep disorders frequently seen in the context of common psychiatric conditions in children and adolescents. Readers will learn about interaction between sleep related problems and symptoms of Attention Deficit/Hyperactivity Disorder, Anxiety and Mood Disorders and Autism Spectrum Disorders, and about the impact of sleep loss and sleep fragmentation on the emotional and behavioral development in children. Semin Pediatr Neurol 15:70-78 (c) 2008 Elsevier Inc. All rights reserved.
C1 [Ivanenko, Anna] Childrens Mem Hosp, Div Child & Adolescent Psychiat, Chicago, IL 60614 USA.
[Johnson, Kyle] Oregon Hlth & Sci Univ, Dept Psychiat, Portland, OR 97201 USA.
RP Ivanenko, A (reprint author), Childrens Mem Hosp, Div Child & Adolescent Psychiat, 800 Biesterfield Rd,Suite 510, Elk Grove Village, IL 60007 USA.
EM aivanenko@sbcglobal.net
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J9 SIGNS
JI Signs
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ER
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AF Wicker, Bruno
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Tardif, Carole
Gepner, Bruno
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LA English
DT Article
DE emotion; autism; prefrontal; effective connectivity; fMRI
ID HIGH-FUNCTIONING AUTISM; FUSIFORM FACE AREA; PREFRONTAL CORTEX;
WHITE-MATTER; ASPERGER-SYNDROME; FRONTAL-CORTEX; SOCIAL INTELLIGENCE;
FACIAL EXPRESSIONS; NEURAL CIRCUITRY; BRAIN
AB Several recent studies suggest that autism may result from abnormal communication between brain regions. We directly assessed this hypothesis by testing the presence of abnormalities in a model of the functional cerebral network engaged during explicit emotion processing in adults with high functioning autism or Asperger syndrome. Comparison of structural equation models revealed abnormal patterns of effective connectivity, with the prefrontal cortex as a key site of dysfunction. These findings provide evidence that abnormal long-range connectivity between structures of the 'social brain' could explain the socio-emotional troubles that characterize the autistic pathology.
C1 [Wicker, Bruno; Hubert, Benedicte; Deruelle, Christine] Univ Aix Marseille 2, CNRS, Mediterranean Inst Cognit Neurosci, F-13402 Marseille 20, France.
[Fonlupt, Pierre] Univ Lyon 1, Unite 280, INSERM, F-69000 Lyon, France.
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RP Wicker, B (reprint author), Univ Aix Marseille 2, CNRS, Mediterranean Inst Cognit Neurosci, 31 Chemin Joseph Aiguier, F-13402 Marseille 20, France.
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Z9 45
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
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PY 2008
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IS 2
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EP 143
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SC Neurosciences & Neurology; Psychology
GA 310JO
UT WOS:000256525000007
PM 19015104
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PT J
AU Kennedy, DP
Courchesne, E
AF Kennedy, Daniel P.
Courchesne, Eric
TI Functional abnormalities of the default network during self- and
other-reflection in autism
SO SOCIAL COGNITIVE AND AFFECTIVE NEUROSCIENCE
LA English
DT Article
DE autism spectrum disorders; retrosplenial cortex; posterior cingulate
cortex; default mode; rest
ID POSTERIOR CINGULATE CORTEX; ASPERGER-SYNDROME; BASE-LINE; PREFRONTAL
CORTEX; BRAIN-FUNCTION; FMRI; FACE; INDIVIDUALS; ATTRIBUTION; ACTIVATION
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[Courchesne, Eric] Rady Childrens Hosp, Res Ctr, Ctr Autism Res, La Jolla, CA 92037 USA.
RP Kennedy, DP (reprint author), CALTECH, Div Humanities & Social Sci, HSS 228-77, Pasadena, CA 91125 USA.
EM kennedy@caltech.edu
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NR 43
TC 99
Z9 100
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 1749-5016
J9 SOC COGN AFFECT NEUR
JI Soc. Cogn. Affect. Neurosci.
PD JUN
PY 2008
VL 3
IS 2
BP 177
EP 190
DI 10.1093/scan/nsn011
PG 14
WC Neurosciences; Psychology; Psychology, Experimental
SC Neurosciences & Neurology; Psychology
GA 310JO
UT WOS:000256525000011
PM 19015108
ER
PT J
AU Silani, G
Bird, G
Brindley, R
Singer, T
Frith, C
Frith, U
AF Silani, Giorgia
Bird, Geoffrey
Brindley, Rachel
Singer, Tania
Frith, Chris
Frith, Uta
TI Levels of emotional awareness and autism: An fMRI study
SO SOCIAL NEUROSCIENCE
LA English
DT Article
ID ASPERGER-SYNDROME; NEURAL BASIS; INTEROCEPTIVE AWARENESS; PHYSIOLOGICAL
CONDITION; ALEXITHYMIA; ACTIVATION; EXPERIENCE; FEELINGS; PERCEPTION;
MODULATION
AB Autism is associated with an inability to identify and distinguish one's own feelings. We assessed this inability using alexithymia and empathy questionnaires, and used fMRI to investigate brain activity while introspecting on emotion. Individuals with high functioning autism/Asperger syndrome (HFA/AS) were compared with matched controls. Participants rated stimuli from the International Affective Picture System twice, once according to the degree of un/pleasantness that the pictures induced, and once according to their color balance. The groups differed significantly on both alexithymia and empathy questionnaires. Alexithymia and lack of empathy were correlated, indicating a link between understanding one's own and others' emotions. For both groups a strong relationship between questionnaire scores and brain activity was found in the anterior insula (AI), when participants were required to assess their feelings to unpleasant pictures. Regardless of self-reported degree of emotional awareness, individuals with HFA/AS differed from controls when required to introspect on their feelings by showing reduced activation in self-reflection/mentalizing regions. Thus, we conclude that difficulties in emotional awareness are related to hypoactivity in AI in both individuals with HFA/AS and controls, and that the particular difficulties in emotional awareness in individuals with HFA/AS are not related to their impairments in self-reflection/mentalizing.
C1 [Bird, Geoffrey] UCL, Dept Psychol, London WC1N 3AR, England.
[Silani, Giorgia; Singer, Tania] Univ Zurich, Zurich, Switzerland.
RP Bird, G (reprint author), UCL, Dept Psychol, 26 Bedford Way, London WC1N 3AR, England.
EM g.bird@ucl.ac.uk
RI Frith, Chris/A-2171-2009; Frith, Uta/C-1757-2008; Bonefeld,
Birgit/B-7936-2010
OI Frith, Chris/0000-0002-8665-0690; Frith, Uta/0000-0002-9063-4466;
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NR 45
TC 146
Z9 149
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 1747-0919
J9 SOC NEUROSCI
JI Soc. Neurosci.
PD JUN
PY 2008
VL 3
IS 2
BP 97
EP 112
DI 10.1080/17470910701577020
PG 16
WC Neurosciences; Psychology
SC Neurosciences & Neurology; Psychology
GA 321AY
UT WOS:000257277700001
PM 18633852
ER
PT J
AU Carter, EJ
Pelphrey, KA
AF Carter, Elizabeth J.
Pelphrey, Kevin A.
TI Friend or foe? Brain systems involved in the perception of dynamic
signals of menacing and friendly social approaches
SO SOCIAL NEUROSCIENCE
LA English
DT Article
ID SUPERIOR TEMPORAL SULCUS; BILATERAL AMYGDALA DAMAGE; HAPPY FACIAL
EXPRESSIONS; FUSIFORM FACE AREA; BIOLOGICAL MOTION; NEURAL RESPONSES;
EMOTION; CORTEX; AUTISM; RECOGNITION
AB During every social approach, humans must assess each other's intentions. Facial expressions provide cues to assist in these assessments via associations with emotion, the likelihood of affiliation, and personality. In this functional magnetic resonance imaging (fMRI) study, participants viewed animated male characters approaching them in a hallway and making either a happy or an angry facial expression. An expected increase in amygdala and superior temporal sulcus activation to the expression of anger was found. Notably, two other social brain regions also had an increased hemodynamic response to anger relative to happiness, including the lateral fusiform gyrus and a region centered in the middle temporal gyrus. Other brain regions showed little differentiation or an increased level of activity to the happy stimuli. These findings provide insight into the brain mechanisms involved in reading the intentions of other human beings in an overtly social context. In particular, they demonstrate brain regions sensitive to social signals of dominance and affiliation.
C1 [Pelphrey, Kevin A.] Carnegie Mellon Univ, Dept Psychol, Pittsburgh, PA 15213 USA.
RP Pelphrey, KA (reprint author), Carnegie Mellon Univ, Dept Psychol, Baker Hall 356D, Pittsburgh, PA 15213 USA.
EM kpelphrey@cmu.edu
RI Carter, Elizabeth/G-6958-2012
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NR 61
TC 12
Z9 13
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 1747-0919
J9 SOC NEUROSCI
JI Soc. Neurosci.
PD JUN
PY 2008
VL 3
IS 2
BP 151
EP 163
DI 10.1080/17470910801903431
PG 13
WC Neurosciences; Psychology
SC Neurosciences & Neurology; Psychology
GA 321AY
UT WOS:000257277700005
PM 18633856
ER
PT J
AU Lombard, J
AF Lombard, Jay
TI Synchrnoic consciousness from a neurological point of view: the
philosophical foundations for neuroethics
SO SYNTHESE
LA English
DT Article
DE consciousness; neurology; psychiatry; dissociation; personal identity;
the subject; phenomenology; neuroscience; Kolak; Freud; autism;
aspergers; schizophrenia; stem cells; glutamic acid decarboxylase (GAD);
GABA; neural darwinism; korsakoff's; entanglement; cognitive dynamics;
apoptosis; identification disorder syndrome (IDS); fact of exclusive
conjoinment (FEC); neuropsychopharmacology; synchronic consciousness;
neuroethics
AB Daniel Kolak's theory of synchronic consciousness according to which the entire range of dissociative phenomena, from pathologies such as MPD and schizophrenia to normal dream states, are best explained in terms of consciousness becoming simultaneously identified as many selves, has revolutionary therapeutic implications for neurology and psychiatry. All these selves, according to Kolak-even the purely imaginary ones that exist as such only in our dreams-are not just conscious but also self-conscious, with beliefs, intentions, living lives informed by memories (confabulatory, in the case of the fictional ones) and personal histories. Kolak's derivation of psychiatrically relevant aspects of his theory-a neurological rendition of a Kantian transcendental argument-can be given a straightforward neurological, and therefore open to scientific scrutiny, interpretation that would then more easily lend itself to the clinical setting in which these perplexing phenomena, along with their purveyors, must live and cope. This will be the main focus of this paper.
C1 [Lombard, Jay] Weill Cornell Med Coll, New York, NY 10021 USA.
[Lombard, Jay] Bronx Lebanon Hosp Ctr, Bronx, NY 10456 USA.
[Lombard, Jay] Brain Behav Ctr, Pomona, NJ 10970 USA.
RP Lombard, J (reprint author), Weill Cornell Med Coll, 525 E 68th St, New York, NY 10021 USA.
EM braincures@yahoo.com
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KOLAK D, 2007, COGNITIVE SCI
NR 5
TC 0
Z9 0
PU SPRINGER
PI DORDRECHT
PA VAN GODEWIJCKSTRAAT 30, 3311 GZ DORDRECHT, NETHERLANDS
SN 0039-7857
J9 SYNTHESE
JI Synthese
PD JUN
PY 2008
VL 162
IS 3
BP 439
EP 450
DI 10.1007/s11229-007-9246-x
PG 12
WC History & Philosophy Of Science; Philosophy
SC History & Philosophy of Science; Philosophy
GA 292HZ
UT WOS:000255258400009
ER
PT J
AU Southgate, V
Hamilton, AFD
AF Southgate, Victoria
Hamilton, Antonia F. de C.
TI Unbroken mirrors: challenging a theory of Autism
SO TRENDS IN COGNITIVE SCIENCES
LA English
DT Review
ID NEURON SYSTEM; SPECTRUM DISORDERS; IMITATION; CHILDREN; INDIVIDUALS;
SIMULATION; OTHERS; RECOGNITION; DYSFUNCTION; MECHANISMS
AB The 'broken mirror' theory of autism has received considerable attention far beyond the scientific community. This theory proposes that the varied social-cognitive difficulties characteristic of autism could be explained by dysfunction of the mirror neuron system, thought to play a role in imitation. We examine this theory and argue that explaining typical imitation behavior, and the failure to imitate in autism, requires much more than the mirror neuron system. Furthermore, evidence for the role of the mirror neuron system in autism is weak. We suggest the broken mirror theory of autism is premature and that better cognitive models of social behavior within and beyond the mirror neuron system are required to understand the causes of poor social interaction in autism.
C1 [Southgate, Victoria] Univ London Birkbeck Coll, Ctr Brain & Cognit Dev, Sch Psychol, London WC1E 7HX, England.
[Hamilton, Antonia F. de C.] Univ Nottingham, Sch Psychol, Nottingham NG7 2RD, England.
RP Southgate, V (reprint author), Univ London Birkbeck Coll, Ctr Brain & Cognit Dev, Sch Psychol, London WC1E 7HX, England.
EM v.southgate@bbk.ac.uk
RI Hamilton, Antonia/B-3612-2008; Southgate, Victoria/C-4957-2008
OI Hamilton, Antonia/0000-0001-8000-0219;
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2008, Q J EXP PSYCHOL COLC, V61, P101
NR 50
TC 60
Z9 62
PU ELSEVIER SCIENCE LONDON
PI LONDON
PA 84 THEOBALDS RD, LONDON WC1X 8RR, ENGLAND
SN 1364-6613
J9 TRENDS COGN SCI
JI TRENDS COGN. SCI.
PD JUN
PY 2008
VL 12
IS 6
BP 225
EP 229
DI 10.1016/j.tics.2008.03.005
PG 5
WC Behavioral Sciences; Neurosciences; Psychology, Experimental
SC Behavioral Sciences; Neurosciences & Neurology; Psychology
GA 317EO
UT WOS:000257002800006
PM 18479959
ER
PT J
AU Farzin, F
Whitney, D
Hagerman, RJ
Rivera, SM
AF Farzin, F.
Whitney, D.
Hagerman, R. J.
Rivera, S. M.
TI Contrast detection in infants with fragile X syndrome
SO VISION RESEARCH
LA English
DT Article
DE fragile X syndrome; contrast detection; second-order; motion; threshold;
forced-choice preferential looking
ID 2ND-ORDER MOTION PATTERNS; 1ST-ORDER MOTION; VISUAL-MOTION;
BRAIN-DAMAGE; DIFFERENTIAL IMPACT; ATTENTIVE TRACKING;
WILLIAMS-SYNDROME; FMR1 GENE; PERCEPTION; SENSITIVITY
AB Studies have reported that a selective deficit in visual motion processing is present in certain developmental disorders, including Williams syndrome and autism. More recent evidence suggests a visual motion impairment is also present in adults with fragile X syndrome (FXS), the most common form of inherited mental retardation. The goal of the current study was to examine low-level cortical visual processing in infants diagnosed with FXS in order to explore the developmental origin of this putative deficit. We measured contrast detection of first-order (luminance-defined) and second-order (contra st-cle fined) gratings at two levels of temporal frequency, 0 Hz (static) and 4 Hz (moving). Results indicate that infants with FXS display significantly higher detection thresholds only for the second-order, moving stimuli compared to mental age-matched typically developing controls. (c) 2008 Elsevier Ltd. All rights reserved.
C1 [Farzin, F.; Whitney, D.; Rivera, S. M.] Univ Calif Davis, Ctr Mind & Brain, Davis, CA 95618 USA.
[Farzin, F.; Whitney, D.; Rivera, S. M.] Univ Calif Davis, Dept Psychol, Davis, CA 95616 USA.
[Hagerman, R. J.; Rivera, S. M.] Univ Calif Davis, Med Ctr, MIND Inst, Sacramento, CA 95817 USA.
[Hagerman, R. J.] Univ Calif Davis, Med Ctr, Dept Pediat, Sacramento, CA 95817 USA.
RP Farzin, F (reprint author), Univ Calif Davis, Ctr Mind & Brain, 202 Cousteau Pl Suite 250, Davis, CA 95618 USA.
EM ffarzin@ucdavis.edu
RI Farzin, Faraz/I-6170-2012
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NR 75
TC 26
Z9 26
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0042-6989
J9 VISION RES
JI Vision Res.
PD JUN
PY 2008
VL 48
IS 13
BP 1471
EP 1478
DI 10.1016/j.visres.2008.03.019
PG 8
WC Neurosciences; Ophthalmology
SC Neurosciences & Neurology; Ophthalmology
GA 323WT
UT WOS:000257479800004
PM 18457856
ER
PT J
AU Phelan, MC
AF Phelan, Mary C.
TI Deletion 22q13.3 syndrome
SO ORPHANET JOURNAL OF RARE DISEASES
LA English
DT Review
ID AUTISM SPECTRUM DISORDERS; PERICENTRIC-INVERSION; IDENTIFICATION;
REARRANGEMENTS; CHROMOSOME-22; FREQUENCY; SHANK3; GENE
AB The deletion 22q13.3 syndrome ( deletion 22q13 syndrome or Phelan-McDermid syndrome) is a chromosome microdeletion syndrome characterized by neonatal hypotonia, global developmental delay, normal to accelerated growth, absent to severely delayed speech, and minor dysmorphic features. The deletion occurs with equal frequency in males and females and has been reported in mosaic and non-mosaic forms. Due to lack of clinical recognition and often insufficient laboratory testing, the syndrome is under- diagnosed and its true incidence remains unknown. Common physical traits include long eye lashes, large or unusual ears, relatively large hands, dysplastic toenails, full brow, dolicocephaly, full cheeks, bulbous nose, and pointed chin. Behavior is autistic-like with decreased perception of pain and habitual chewing or mouthing. The loss of 22q13.3 can result from simple deletion, translocation, ring chromosome formation and less common structural changes affecting the long arm of chromosome 22, specifically the region containing the SHANK3 gene. The diagnosis of deletion 22q13 syndrome should be considered in all cases of hypotonia of unknown etiology and in individuals with absent speech. Although the deletion can sometimes be detected by high resolution chromosome analysis, fluorescence in situ hybridization ( FISH) or array comparative genomic hybridization (CGH) is recommended for confirmation. Differential diagnosis includes syndromes associated with hypotonia, developmental delay, speech delay and/or autistic-like affect (Prader-Willi, Angelman, Williams, Smith-Magenis, Fragile X, Sotos, FG, trichorhinophalangeal and velocardiofacial syndromes, autism spectrum disorders, cerebral palsy). Genetic counseling is recommended and parental laboratory studies should be considered to identify cryptic rearrangements and detect parental mosaicism. Prenatal diagnosis should be offered for future pregnancies in those families with inherited rearrangements. Individuals with deletion 22q13 should have routine examinations by the primary care physician as well as genetic evaluations with referral to specialists if neurological, gastrointestinal, renal, or other systemic problems are suspected. Affected individuals benefit from early intervention programs, intense occupational and communication therapies, adaptive exercise and sport programs, and other therapies to strengthen their muscles and increase their communication skills. No apparent life-threatening organic abnormalities accompany the diagnosis of deletion 22q13.
C1 Mol Pathol Lab Network, Cytogenet Lab, Maryville, TN 37804 USA.
RP Phelan, MC (reprint author), Mol Pathol Lab Network, Cytogenet Lab, 250 E Broadway, Maryville, TN 37804 USA.
EM kphelan@mplnet.com
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NR 19
TC 70
Z9 72
PU BIOMED CENTRAL LTD
PI LONDON
PA 236 GRAYS INN RD, FLOOR 6, LONDON WC1X 8HL, ENGLAND
SN 1750-1172
J9 ORPHANET J RARE DIS
JI Orphanet J. Rare Dis.
PD MAY 27
PY 2008
VL 3
AR 14
DI 10.1186/1750-1172-3-14
PG 6
WC Genetics & Heredity; Medicine, Research & Experimental
SC Genetics & Heredity; Research & Experimental Medicine
GA 324CP
UT WOS:000257495600001
PM 18505557
ER
PT J
AU Geddes, L
AF Geddes, Linda
TI Earlier the better for autism therapy?
SO NEW SCIENTIST
LA English
DT News Item
AB Parents are often aware of initial signs of autism when their kids are very young and yet the average age of diagnosis remains at 3 years or 4 or older. Early detection leads to earlier intervention. Behavioural interventions often build on the child's own interests, in order to help them practise establishing a social and emotional connection with others. Children with autism are born with an average or slightly below average head circumference but then experience faster than average head growth in the first three years. Using behavioural therapies, researchers hope to override the underlying genetic causes of autism and push the brain in a different direction. Significant improvements in core behaviours such as making eye contact and sharing a smile compared with children who began therapy were observed.
NR 0
TC 1
Z9 1
PU REED BUSINESS INFORMATION LTD
PI SUTTON
PA QUADRANT HOUSE THE QUADRANT, SUTTON SM2 5AS, SURREY, ENGLAND
SN 0262-4079
J9 NEW SCI
JI New Sci.
PD MAY 24
PY 2008
VL 198
IS 2657
BP 6
EP 7
PG 2
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 304QA
UT WOS:000256122800003
ER
PT J
AU Baumgartner, T
Heinrichs, M
Vonlanthen, A
Fischbacher, U
Fehr, E
AF Baumgartner, Thomas
Heinrichs, Markus
Vonlanthen, Aline
Fischbacher, Urs
Fehr, Ernst
TI Oxytocin shapes the neural circuitry of trust and trust adaptation in
humans
SO NEURON
LA English
DT Article
ID ANTERIOR CINGULATE CORTEX; EMOTIONAL FACES; AMYGDALA RESPONSES;
SENSATION-SEEKING; SOCIAL COGNITION; DECISION-MAKING; HUMAN BRAIN;
BEHAVIOR; FAIRNESS; ECONOMICS
AB Trust and betrayal of trust are ubiquitous in human societies. Recent behavioral evidence shows that the neuropeptide oxytocin increases trust among humans, thus offering a unique chance of gaining a deeper understanding of the neural mechanisms underlying trust and the adaptation to breach of trust. We examined the neural circuitry of trusting behavior by combining the intranasal, double-blind, administration of oxytocin with fMRI. We find that subjects in the oxytocin group show no change in their trusting behavior after they learned that their trust had been breached several times while subjects receiving placebo decrease their trust. This difference in trust adaptation is associated with a specific reduction in activation in the amygdala, the midbrain regions, and the dorsal striatum in subjects receiving oxytocin, suggesting that neural systems mediating fear processing (amygdala and midbrain regions) and behavioral adaptations to feedback information (dorsal striatum) modulate oxytocin's effect on trust. These findings may help to develop deeper insights into mental disorders such as social phobia and autism, which are characterized by persistent fear or avoidance of social interactions.
C1 [Baumgartner, Thomas; Vonlanthen, Aline; Fischbacher, Urs; Fehr, Ernst] Univ Zurich, Inst Empir Res Econ, Ctr Study Social & Neural Syst, CH-8006 Zurich, Switzerland.
[Heinrichs, Markus] Univ Zurich, Dept Psychol Clin Psychol & Psychobiol, CH-8050 Zurich, Switzerland.
[Fehr, Ernst] Coll Helveticum, CH-8092 Zurich, Switzerland.
RP Baumgartner, T (reprint author), Univ Zurich, Inst Empir Res Econ, Ctr Study Social & Neural Syst, Blumlisalpstr 10, CH-8006 Zurich, Switzerland.
EM t.baumgartner@iew.uzh.ch; efehr@iew.uzh.ch
RI Fischbacher, Urs/H-2693-2014
OI Fischbacher, Urs/0000-0002-5115-8815
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NR 76
TC 422
Z9 431
PU CELL PRESS
PI CAMBRIDGE
PA 600 TECHNOLOGY SQUARE, 5TH FLOOR, CAMBRIDGE, MA 02139 USA
SN 0896-6273
J9 NEURON
JI Neuron
PD MAY 22
PY 2008
VL 58
IS 4
BP 639
EP 650
DI 10.1016/j.neuron.2008.04.009
PG 12
WC Neurosciences
SC Neurosciences & Neurology
GA 304PD
UT WOS:000256120500016
PM 18498743
ER
PT J
AU Janusonis, S
AF Janusonis, Skirmantas
TI Origin of the blood hyperserotonemia of autism
SO THEORETICAL BIOLOGY AND MEDICAL MODELLING
LA English
DT Article
ID PLATELET SEROTONIN LEVELS; PERVASIVE DEVELOPMENTAL DISORDERS; PROMOTER
VARIANTS; RETARDED-CHILDREN; TRANSPORTER; BRAIN; MICE; EXPRESSION;
PLASMA; SYSTEM
AB Background: Research in the last fifty years has shown that many autistic individuals have elevated serotonin (5-hydroxytryptamine, 5-HT) levels in blood platelets. This phenomenon, known as the platelet hyperserotonemia of autism, is considered to be one of the most well-replicated findings in biological psychiatry. Its replicability suggests that many of the genes involved in autism affect a small number of biological networks. These networks may also play a role in the early development of the autistic brain.
Results: We developed an equation that allows calculation of platelet 5-HT concentration as a function of measurable biological parameters. It also provides information about the sensitivity of platelet 5- HT levels to each of the parameters and their interactions.
Conclusion: The model yields platelet 5-HT concentrations that are consistent with values reported in experimental studies. If the parameters are considered independent, the model predicts that platelet 5-HT levels should be sensitive to changes in the platelet 5-HT uptake rate constant, the proportion of free 5-HT cleared in the liver and lungs, the gut 5-HT production rate and its regulation, and the volume of the gut wall. Linear and non-linear interactions among these and other parameters are specified in the equation, which may facilitate the design and interpretation of experimental studies.
C1 Univ Calif Santa Barbara, Dept Psychol, Santa Barbara, CA 93106 USA.
RP Janusonis, S (reprint author), Univ Calif Santa Barbara, Dept Psychol, Santa Barbara, CA 93106 USA.
EM janusonis@psych.ucsb.edu
FU Santa Barbara Cottage Hospital UCSB Special Research l
FX This study was supported, in part, by the Santa Barbara Cottage Hospital
UCSB Special Research Award. I thank the anonymous reviewers for their
constructive comments and Vaiva for her support.
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SC Mathematical & Computational Biology
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PT J
AU Meikle, L
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Egnor, A
Kramvis, I
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Sahin, M
Kwiatkowski, DJ
AF Meikle, Lynsey
Pollizzi, Kristen
Egnor, Anna
Kramvis, Ioannis
Lane, Heidi
Sahin, Mustafa
Kwiatkowski, David J.
TI Response of a neuronal model of tuberous sclerosis to mammalian target
of rapamycin (mTOR) inhibitors: Effects on mTORC1 and Akt signaling lead
to improved survival and function
SO JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE tuberous sclerosis; TSC; TSC1; TSC2; rapamycin; RAD001
ID KINASE-B-GAMMA; MOUSE MODEL; CORTICAL DYSPLASIA; GLUCOSE-HOMEOSTASIS;
MUTATIONAL ANALYSIS; HUMAN-DISEASE; IN-VIVO; COMPLEX; TSC1; ACTIVATION
AB Tuberous sclerosis (TSC) is a hamartoma syndrome attributable to mutations in either TSC1 or TSC2 in which brain involvement causes epilepsy, mental retardation, and autism. We have reported recently (Meikle et al., 2007) a mouse neuronal model of TSC in which Tsc1 is ablated in most neurons during cortical development. We have tested rapamycin and RAD001 [40-O-(2-hydroxyethyl)-rapamycin], both mammalian target of rapamycin mTORC1 inhibitors, as potential therapeutic agents in this model. Median survival is improved from 33 d to more than 100 d; behavior, phenotype, and weight gain are all also markedly improved. There is brain penetration of both drugs, with accumulation over time with repetitive treatment, and effective reduction of levels of phospho-S6, a downstream target of mTORC1. In addition, there is restoration of phospho-Akt and phospho-glycogen synthase kinase 3 levels in the treated mice, consistent with restoration of Akt function. Neurofilament abnormalities, myelination, and cell enlargement are all improved by the treatment. However, dysplastic neuronal features persist, and there are only modest changes in dendritic spine density and length. Strikingly, mice treated with rapamycin or RAD001 for 23 d only (postnatal days 7-30) displayed a persistent improvement in phenotype, with median survival of 78 d. In summary, rapamycin/RAD001 are highly effective therapies for this neuronal model of TSC, with benefit apparently attributable to effects on mTORC1 and Akt signaling and, consequently, cell size and myelination. Although caution is appropriate, the results suggest the possibility that rapamycin/RAD001 may have benefit in the treatment of TSC brain disease, including infantile spasms.
C1 [Meikle, Lynsey; Pollizzi, Kristen; Egnor, Anna; Kwiatkowski, David J.] Harvard Univ, Sch Med, Brigham & Womens Hosp, Dept Med,Div Translat Med, Boston, MA 02115 USA.
[Kramvis, Ioannis; Sahin, Mustafa] Harvard Univ, Sch Med, Childrens Hosp, Dept Neurol, Boston, MA 02115 USA.
[Lane, Heidi] Nova Pharma AG, Novartis Inst Biomed Res, CH-4002 Basel, Switzerland.
[Lane, Heidi] Nova Pharma AG, Novartis Inst Oncol Basel, CH-4002 Basel, Switzerland.
RP Kwiatkowski, DJ (reprint author), Harvard Univ, Sch Med, Brigham & Womens Hosp, Dept Med,Div Translat Med, 1 Blackfan Circle 6-216, Boston, MA 02115 USA.
EM dk@rics.bwh.harvard.edu
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PI WASHINGTON
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PT J
AU Muehlmann, AM
Devine, DP
AF Muehlmann, Amber M.
Devine, Darragh P.
TI Glutamate-mediated neuroplasticity in an animal model of self-injurious
behaviour
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Review
DE self-injurious behaviour; neuroplasticity; pemoline; glutamate; MK-801;
memantine
ID VENTRAL TEGMENTAL AREA; PRADER-WILLI-SYNDROME; LESCH-NYHAN-SYNDROME;
LONG-TERM POTENTIATION; RAT HIPPOCAMPAL SLICES; EXCITATORY AMINO-ACIDS;
NUCLEUS-ACCUMBENS; IN-VITRO; PSYCHOMOTOR STIMULANTS; SYNAPTIC
TRANSMISSION
AB Self-injurious behaviour (SIB) is exhibited by individuals with a broad variety of developmental disorders and genetic abnormalities, including autism and Lesch-Nyhan, Prader-Willi and Rett syndromes. Most research has focused on environmental factors that reinforce SIB, and less is known about the biological basis of this behaviour disorder. However, animal models have been developed to study the neurochemical pathology that underlies SIB. In one model, rats exhibit self-biting after repeated daily administration of moderately high doses of pemoline (100-200 mg/kg). Dopaminergic and glutamatergic neurotransmission have been implicated in this model. Accordingly, we investigated the role of glutamatergic neurotransmission in pemoline-induced SIB, using the N-methyl-D-aspartate (NMDA) receptor antagonists MK-801 and memantine. MK-801 is a high affinity antagonist which blocks glutamate-mediated neuroplasticity and behavioural sensitization to other psychostimulants. It lessened the incidence of SIB, the time spent self-injuring, and the area of tissue damage in the pemoline model. Memantine, on the other hand, is a low affinity antagonist which does not disrupt glutamate-mediated neuroplasticity, and it had little if any effect on any measure of pemoline-induced SIB. These results suggest that repeated pemoline administration induces glutamate-mediated neuroplastic changes that lead to the eventual expression of SIB. Further investigation of these changes may reveal specific neurochemical factors that contribute to SIB in this animal model of self-injury. (c) 2007 Elsevier B.V. All rights reserved.
C1 [Muehlmann, Amber M.; Devine, Darragh P.] Univ Florida, Dept Psychol, Behav Neurosci Program, Gainesville, FL 32611 USA.
RP Devine, DP (reprint author), Univ Florida, Dept Psychol, Behav Neurosci Program, POB 112250, Gainesville, FL 32611 USA.
EM dpdevine@ufl.edu
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NR 103
TC 8
Z9 8
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD MAY 16
PY 2008
VL 189
IS 1
BP 32
EP 40
DI 10.1016/j.bbr.2007.12.001
PG 9
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 289PG
UT WOS:000255065800004
PM 18243356
ER
PT J
AU Sutcliffe, JS
AF Sutcliffe, James S.
TI Affiliative behaviors and beyond: It's the phenotype, stupid
SO BIOLOGICAL PSYCHIATRY
LA English
DT Editorial Material
ID AUTISM; OXYTOCIN
C1 [Sutcliffe, James S.] Vanderbilt Univ, Ctr Mol Neurosci, Nashville, TN 37232 USA.
[Sutcliffe, James S.] Vanderbilt Univ, Vanderblit Kennedy Ctr, Nashville, TN 37232 USA.
RP Sutcliffe, JS (reprint author), Vanderbilt Univ, Ctr Mol Neurosci, 221 Kirkland Hall, Nashville, TN 37232 USA.
EM james.s.sutcliffe@vanderbilt.edu
RI Sutcliffe, James/C-1348-2012
OI Sutcliffe, James/0000-0001-5200-6007
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Jacob S, 2007, NEUROSCI LETT, V417, P6, DOI 10.1016/j.neulet.2007.02.001
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NR 10
TC 2
Z9 2
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0006-3223
J9 BIOL PSYCHIAT
JI Biol. Psychiatry
PD MAY 15
PY 2008
VL 63
IS 10
BP 909
EP 910
DI 10.1016/j.biopsych.2008.03.027
PG 2
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 297GB
UT WOS:000255604000001
PM 18452756
ER
PT J
AU Yrigollen, CM
Han, SS
Kochetkova, A
Babitz, T
Chang, JT
Volkmar, FR
Leckman, JF
Grigorenko, EL
AF Yrigollen, Carolyn M.
Han, Summer S.
Kochetkova, Anna
Babitz, Tammy
Chang, Joseph T.
Volkmar, Fred R.
Leckman, James F.
Grigorenko, Elena L.
TI Genes controlling affiliative behavior as candidate genes for autism
SO BIOLOGICAL PSYCHIATRY
LA English
DT Article
DE affiliative behaviors; allelic association studies; autism; spectrum
disorders; D beta H; FOSB; OXT OXTR; PRL; PRLR
ID GENOTYPE-PHENOTYPE ASSOCIATIONS; DIAGNOSTIC OBSERVATION SCHEDULE;
SPECTRUM DISORDERS; RECEPTOR GENE; REPETITIVE BEHAVIORS; QUANTITATIVE
TRAITS; PITOCIN INDUCTION; PLASMA OXYTOCIN; INDIVIDUALS; CHILDREN
AB Background: Autism spectrum disorders (ASD) are neurodevelopmental disorders of complex etiology, with a recognized substantial contribution of heterogeneous genetic factors; one of the core features of ASD is a lack of affiliative behaviors.
Methods: On the basis of the existing literature, in this study we examined the hypothesis of allelic associations between genetic variants in six genes involved in control of maternal and affiliative behaviors (OX; OXTR, PRL, PRLR, D beta H, and FOSB). One hundred and seventy-seven probands with ASID from 151 families (n = 527) were assessed with a set of related instruments capturing multiplefacets of ASID. Multivariate and univariate phenotypes were constructed from these assessments and subjected to genetic linkage and association analyses with PBAT and FBAT software.
Results: The resulting pattern of findings, in general, confirmed the hypotheses of the significance of the genes involved in the development of affiliative behaviors in the manifestation of ASID (p values ranging from .000005 to .05); statistically speaking, the strongest results were obtained for allelic associations with the PRL, PRLR, and OXTR genes.
Conclusions: These preliminary data provide additional support for the hypothesis that the allelic variants of genes necessary for the development of species-typical affiliative behaviors are associated with ASID. Independent replication of these findings is needed and studies of other genes associated with affiliative behaviors are indicated.
C1 [Yrigollen, Carolyn M.; Babitz, Tammy; Volkmar, Fred R.; Leckman, James F.; Grigorenko, Elena L.] Yale Univ, Ctr Child Study, Dept Psychol, Dept Epidemiol & Publ Hlth, New Haven, CT 06519 USA.
[Han, Summer S.; Kochetkova, Anna; Chang, Joseph T.] Yale Univ, Dept Stat, New Haven, CT 06519 USA.
[Grigorenko, Elena L.] Moscow MV Lomonosov State Univ, Dept Psychol, Moscow, Russia.
RP Grigorenko, EL (reprint author), Yale Univ, Ctr Child Study, Dept Psychol, Dept Epidemiol & Publ Hlth, 230 S Frontage Rd, New Haven, CT 06519 USA.
EM elena.grigorenko@yale.edu
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NR 58
TC 80
Z9 83
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0006-3223
J9 BIOL PSYCHIAT
JI Biol. Psychiatry
PD MAY 15
PY 2008
VL 63
IS 10
BP 911
EP 916
DI 10.1016/j.biopsych.2007.11.015
PG 6
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 297GB
UT WOS:000255604000002
PM 18207134
ER
PT J
AU Shafritz, KM
Dichter, GS
Baranek, GT
Belger, A
AF Shafritz, Keith M.
Dichter, Gabriel S.
Baranek, Grace T.
Belger, Aysenil
TI The neural circuitry mediating shifts in behavioral response and
cognitive set in autism
SO BIOLOGICAL PSYCHIATRY
LA English
DT Article
DE autism; cognitive set; executive function; fMRI
ID INFERIOR PREFRONTAL CORTEX; PERVASIVE DEVELOPMENTAL DISORDERS; CARD
SORTING TEST; EXECUTIVE FUNCTION; COMMUNICATION DEFICITS; SPECTRUM
DISORDERS; PARKINSONS-DISEASE; WORKING-MEMORY; FUNCTIONAL MRI; ATTENTION
AB Background: Recent studies have suggested that the social and cognitive impairments in autism are associated with neural processing deficits in specific brain regions. However, these studies have primarily focused on neural systems responsible for face processing and social behaviors. Although repetitive, stereotyped behaviors are a hallmark of autism, little is known about the neural mechanisms underlying these behaviors in the disorder.
Methods: We used functional magnetic resonance imaging (fMRI) to investigate the neural correlates of shifts in behavioral response and cognitive set in 18 individuals with high-functioning autism and 15 neurotypical control participants. Participants performed a target detection task specifically designed to distinguish shifts in response from shifts in cognitive set.
Results: Individuals with autism showed lower accuracy on response shifting trials, independent of whether those trials also required a shift in cognitive set. Compared with control subjects, participants with autism showed reduced activation in frontal, striatal, and parietal regions during these trials. In addition, within the autism group, the severity of restricted, repetitive behaviors was negatively correlated with activation in anterior cingulate and posterior parietal regions.
Conclusions: These results suggest that executive deficits and, by extension, repetitive behaviors associated with autism might reflect a core dysfunction within the brain's executive circuitry.
C1 [Dichter, Gabriel S.; Belger, Aysenil] Univ N Carolina, Sch Med, Dept Psychiat, Chapel Hill, NC 27599 USA.
[Shafritz, Keith M.] Hofstra Univ, Dept Psychol, Hempstead, NY 11550 USA.
[Shafritz, Keith M.; Dichter, Gabriel S.; Belger, Aysenil] Duke Univ, Med Ctr, Duke UNC Brain Imaging & Anal Ctr, Durham, NC 27706 USA.
[Dichter, Gabriel S.; Baranek, Grace T.; Belger, Aysenil] Neurodev Disorders Res Ctr, Chapel Hill, NC USA.
[Baranek, Grace T.] Univ N Carolina, Dept Allied Hlth Sci, Chapel Hill, NC USA.
RP Belger, A (reprint author), Univ N Carolina, Sch Med, Dept Psychiat, CB 7160,101 Manning Dr, Chapel Hill, NC 27599 USA.
EM abelger@med.unc.edu
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NR 48
TC 69
Z9 70
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0006-3223
J9 BIOL PSYCHIAT
JI Biol. Psychiatry
PD MAY 15
PY 2008
VL 63
IS 10
BP 974
EP 980
DI 10.1016/j.biopsych.2007.06.028
PG 7
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 297GB
UT WOS:000255604000009
PM 17916328
ER
PT J
AU Offit, PA
AF Offit, Paul A.
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LA English
DT Editorial Material
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Univ Penn, Sch Med, Philadelphia, PA 19104 USA.
RP Offit, PA (reprint author), Childrens Hosp Philadelphia, Philadelphia, PA 19104 USA.
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NR 4
TC 34
Z9 34
PU MASSACHUSETTS MEDICAL SOC
PI WALTHAM
PA WALTHAM WOODS CENTER, 860 WINTER ST,, WALTHAM, MA 02451-1413 USA
SN 0028-4793
J9 NEW ENGL J MED
JI N. Engl. J. Med.
PD MAY 15
PY 2008
VL 358
IS 20
BP 2089
EP 2091
DI 10.1056/NEJMp0802904
PG 3
WC Medicine, General & Internal
SC General & Internal Medicine
GA 300TY
UT WOS:000255849300001
PM 18480200
ER
PT J
AU Virji-Babul, N
Moiseev, A
Cheung, T
Weeks, D
Cheyne, D
Ribary, U
AF Virji-Babul, Naznin
Moiseev, Alexander
Cheung, Teresa
Weeks, Daniel
Cheyne, Douglas
Ribary, Urs
TI Changes in mu rhythm during action observation and execution in adults
with Down syndrome: Implications for action representation
SO NEUROSCIENCE LETTERS
LA English
DT Article
DE mu rhythm; action representation; reach to grasp; Down syndrome; mirror
neurons
ID IMITATION; CHILDREN; INDIVIDUALS; PERCEPTION; AUTISM
AB The human mirror neuron system is thought to be the underlying basis of perception-action coupling involved in imitation and action understanding. In order to examine this issue we examined the recruitment of the mirror neuron system, as reflected in mu rhythm suppression in a population of adults with Down syndrome (DS) with known strengths in imitation but with impairments in perceptual-motor coupling. Ten healthy adults and 10 age-matched adults with (DS) participated in the study. Subjects were asked to make self-paced movements (execution), and view movements made by the experimenter (observation). The action consisted of reaching with the dominant hand to grasp and lift a cup. Cortical responses were recorded with a whole head magnetoencephalography (MEG) system. Both groups demonstrated significant attenuation of the mu rhythm in bilateral sensorimotor areas when executing the action. Typical adults also demonstrated significant mu suppression in bilateral sensorimotor areas during observation of the action. In contrast, when observing the movement, adults with DS showed a significantly reduced overall attenuation of mu activity with a distinct laterality in the pattern of mu suppression. These results suggest that there is a dysfunction in the execution/observation matching system in adults with DS and has implications for the functional role of the human mirror neuron system. (C) 2008 Elsevier Ireland Ltd. All rights reserved.
C1 [Virji-Babul, Naznin; Moiseev, Alexander; Cheung, Teresa; Ribary, Urs] Down Syndrome Res Fdn, MEG Lab, Burnaby, BC, Canada.
[Virji-Babul, Naznin; Weeks, Daniel; Ribary, Urs] Simon Fraser Univ, Dept Psychol, Burnaby, BC V5A 1S6, Canada.
[Cheyne, Douglas] Hosp Sick Children, Program Neurosci & Mental Hlth, Toronto, ON M5G 1X8, Canada.
RP Virji-Babul, N (reprint author), Down Syndrome Res Fdn, MEG Lab, 1409 Sperling Ave, Burnaby, BC, Canada.
EM naznin@dsrf.org
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NR 26
TC 20
Z9 20
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0304-3940
J9 NEUROSCI LETT
JI Neurosci. Lett.
PD MAY 9
PY 2008
VL 436
IS 2
BP 177
EP 180
DI 10.1016/j.neulet.2008.03.022
PG 4
WC Neurosciences
SC Neurosciences & Neurology
GA 305TN
UT WOS:000256200800020
PM 18394804
ER
PT J
AU Cheng, YW
Lee, PL
Yang, CY
Lin, CP
Hung, D
Decety, J
AF Cheng, Yawei
Lee, Po-Lei
Yang, Chia-Yen
Lin, Ching-Po
Hung, Daisy
Decety, Jean
TI Gender Differences in the Mu Rhythm of the Human Mirror-Neuron System
SO PLOS ONE
LA English
DT Article
ID PRIMARY MOTOR CORTEX; AUTISM SPECTRUM DISORDERS; HIGH-FUNCTIONING
AUTISM; NORMAL SEX-DIFFERENCES; SOCIAL NEUROSCIENCE; FACIAL EXPRESSIONS;
ASPERGER-SYNDROME; PREMOTOR CORTEX; HUMAN EMPATHY; ACTIVATION
AB Background: Psychologically, females are usually thought to be superior in interpersonal sensitivity than males. The human mirror-neuron system is considered to provide the basic mechanism for social cognition. However, whether the human mirror-neuron system exhibits gender differences is not yet clear.
Methodology/Principal Findings: We measured the electroencephalographic mu rhythm, as a reliable indicator of the human mirror-neuron system activity, when female (N = 20) and male (N = 20) participants watched either hand actions or a moving dot. The display of the hand actions included androgynous, male, and female characteristics. The results demonstrate that females displayed significantly stronger mu suppression than males when watching hand actions. Instead, mu suppression was similar across genders when participants observed the moving dot and between the perceived sex differences (same-sex vs. opposite-sex). In addition, the mu suppressions during the observation of hand actions positively correlated with the personal distress subscale of the interpersonal reactivity index and negatively correlated with the systemizing quotient.
Conclusions/Significance: The present findings indirectly lend support to the extreme male brain theory put forward by Baron-Cohen (2005), and may cast some light on the mirror-neuron dysfunction in autism spectrum disorders. The mu rhythm in the human mirror-neuron system can be a potential biomarker of empathic mimicry.
C1 [Cheng, Yawei; Lin, Ching-Po] Natl Yang Ming Univ, Sch Life Sci, Inst Neurosci, Taipei 112, Taiwan.
[Cheng, Yawei] Natl Yang Ming Univ Hosp, Dept Rehabil, Yilan, Taiwan.
[Lee, Po-Lei] Natl Cent Univ, Dept Elect Engn, Tao Yuan, Taiwan.
[Yang, Chia-Yen] Ching Yun Univ, Inst Comp, Commun & Syst Engn, Chungli, Taiwan.
[Hung, Daisy] Natl Cent Univ, Coll Sci, Inst Cognitive Neurosci, Tao Yuan, Taiwan.
[Decety, Jean] Univ Chicago, Ctr Cognitive & Social Neurosci, Dept Psychiat, Chicago, IL USA.
[Decety, Jean] Univ Chicago, Ctr Cognitive & Social Neurosci, Dept Psychol, Chicago, IL USA.
RP Cheng, YW (reprint author), Natl Yang Ming Univ, Sch Life Sci, Inst Neurosci, Taipei 112, Taiwan.
EM decety@uchicago.edu
FU National Science Council [95-2752-H-010-004-PAE, 96-2314-B-532-001,
96-2221-E-008-122-MY3]; Department of Health, Taipei City Government
[96001-62-044]; Ministry of Education, Taiwan [96-2752-B-010-008-PAE];
NSF [BCS- 0718480]
FX This study was sponsored by the National Science Council
(95-2752-H-010-004-PAE; 96-2314-B-532-001; 96-2221-E-008-122-MY3), the
Department of Health, Taipei City Government (96001-62-044), and
Ministry of Education (96-2752-B-010-008-PAE), Taiwan. Dr. Jean Decety
was supported by an NSF grant (BCS- 0718480).
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NR 56
TC 51
Z9 54
PU PUBLIC LIBRARY SCIENCE
PI SAN FRANCISCO
PA 185 BERRY ST, STE 1300, SAN FRANCISCO, CA 94107 USA
SN 1932-6203
J9 PLOS ONE
JI PLoS One
PD MAY 7
PY 2008
VL 3
IS 5
AR e2113
DI 10.1371/journal.pone.0002113
PG 7
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 382YX
UT WOS:000261642400035
PM 18461176
ER
PT J
AU Liao, WL
Tsai, HC
Wang, HF
Chang, J
Lu, KM
Wu, HL
Lee, YC
Tsai, TF
Takahashi, H
Wagner, M
Ghyselinck, NB
Chambon, P
Liu, FC
AF Liao, Wen-Lin
Tsai, Hsiu-Chao
Wang, Hsiao-Fang
Chang, Josephine
Lu, Kuan-Ming
Wu, Hsiao-Lin
Lee, Yi-Chao
Tsai, Ting-Fen
Takahashi, Hiroshi
Wagner, Michael
Ghyselinck, Norbert B.
Chambon, Pierre
Liu, Fu-Chin
TI Modular patterning of structure and function of the striatum by retinoid
receptor signaling
SO PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF
AMERICA
LA English
DT Article
DE basal ganglia; cell proliferation; retinoic acid; stereotypic behavior
ID RAR-BETA; DEVELOPMENTAL EXPRESSION; MATRIX COMPARTMENTS; ACID;
TELENCEPHALON; MOUSE; DIFFERENTIATION; ACTIVATION; NEURONS; AUTISM
AB Retinoid signaling plays a crucial role in patterning rhombomeres in the hindbrain and motor neurons in the spinal cord during development. A fundamentally interesting question is whether retinoids can pattern functional organization in the forebrain that generates a high order of cognitive behavior. The striatum contains a compartmental structure of striosome (or "patch") and intervening matrix. How this highly complex mosaic design is patterned by the genetic programs during development remains elusive. We report a developmental mechanism by which retinoid receptor signaling controls compartmental formation in the striatum. We analyzed RAR beta(-/-) mutant mice and found a selective loss of striosomal compartmentalization in the rostral mutant striatum. The loss of RAR beta signaling in the mutant mice resulted in reduction of cyclin E2, a cell cycle protein regulating transition from G(1) to S phase, and also reduction of the proneural gene Mash1, which led to defective neurogenesis of late-born striosomal cells. Importantly, during striatal neurogenesis, endogenous levels of retinoic acid were spatiotemporally regulated such that transduction of high levels of retinoic acid through RAR beta selectively expanded the population of late-born striosomal progenitors, which evolved into a highly elaborate compartment in the rostral striatum. RAR beta(-/-) mutant mice, which lacked such enlarged compartment, displayed complex alternations of dopamine agonist-induced stereotypic motor behavior, including exaggeration of head bobbing movement and reduction of rearing activity. RAR beta signaling thus plays a crucial role in setting up striatal compartments that may engage in neural circuits of psychomotor control.
C1 [Ghyselinck, Norbert B.; Chambon, Pierre] Univ Strasbourg 1, Coll France, CNRS, INSERM,IGBMC, F-67404 Illkirch Graffenstaden, CU Strasbourg, France.
[Liao, Wen-Lin; Tsai, Hsiu-Chao; Wang, Hsiao-Fang; Chang, Josephine; Lu, Kuan-Ming; Wu, Hsiao-Lin; Liu, Fu-Chin] Natl Yang Ming Univ, Inst Neurosci, Taipei 112, Taiwan.
[Liu, Fu-Chin] Natl Yang Ming Univ, Ctr Neurosci, Taipei 112, Taiwan.
[Tsai, Ting-Fen] Natl Yang Ming Univ, Dept Life Sci, Taipei 112, Taiwan.
[Lee, Yi-Chao] Natl Cheng Kung Univ, Ctr Gene Regulat & Signal Transduct Res, Tainan 701, Taiwan.
[Takahashi, Hiroshi] Mitsubishi Kagaku Inst Life Sci, Dev Neurobiol Grp, Tokyo 1948511, Japan.
[Wagner, Michael] SUNY Hlth Sci Ctr, Coll Med, Dept Anat & Cell Biol, Brooklyn, NY 11203 USA.
RP Chambon, P (reprint author), Univ Strasbourg 1, Coll France, CNRS, INSERM,IGBMC, F-67404 Illkirch Graffenstaden, CU Strasbourg, France.
EM chambon@titus.u-strasbg.fr; fuchin@ym.edu.tw
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NR 40
TC 24
Z9 25
PU NATL ACAD SCIENCES
PI WASHINGTON
PA 2101 CONSTITUTION AVE NW, WASHINGTON, DC 20418 USA
SN 0027-8424
J9 P NATL ACAD SCI USA
JI Proc. Natl. Acad. Sci. U. S. A.
PD MAY 6
PY 2008
VL 105
IS 18
BP 6765
EP 6770
DI 10.1073/pnas.0802109105
PG 6
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 300QZ
UT WOS:000255841600046
PM 18443282
ER
PT J
AU Dietert, RR
Dietert, JM
AF Dietert, Rodney R.
Dietert, Janice M.
TI Possible role for early-life immune insult including developmental
immunotoxicity in chronic fatigue syndrome (CFS) or myalgic
encephalomyelitis (ME)
SO TOXICOLOGY
LA English
DT Editorial Material
DE early-life immune insults (ELII); developmental immunotoxicity (DIT);
chronic fatigue syndrome (CFS); myalgic encephalomyelitis (ME); immune
dysfunction; inflammation; infections; prenatal; neonatal; neurological
system; endocrine; postnatal stress
ID ENVIRONMENTAL TOBACCO-SMOKE; REGULATORY T-CELLS; IN-UTERO EXPOSURE;
OXIDATIVE STRESS; NITRIC-OXIDE; HUMAN HERPESVIRUS-6; CYTOKINE
PRODUCTION; PARKINSONS-DISEASE; PRENATAL EXPOSURE; RISK-FACTORS
AB Chronic fatigue syndrome (CFS), also known as myalgic encephalomyelitis (ME) in some countries, is a debilitating disease with a constellation of multi-system dysfunctions primarily involving the neurological, endocrine and immune systems. While substantial information is available concerning the complex dysfunction-associated symptoms of CFS, environmental origins of the disease have yet to be determined. Part of the dilemma in identifying the cause(s) has been the focus on biomarkers (hormones, neurotransmitters, cytokines, infectious agents) that are contemporary with later-life CFS episodes. Yet, recent investigations on the origins of environmental diseases of the neurological, endocrine, reproductive, respiratory and immune systems suggest that early life toxicologic and other insults are pivotal in producing later-life onset of symptoms. As with autism and childhood asthma, CFS can also occur in children where the causes are certainly early-life events. Immune dysfunction is recognized as part of the CFS phenotype but has received comparatively less attention than aberrant neurological or endocrine function. However, recent research results suggest that early life immune insults (ELII) including developmental immunotoxicity (DIT), which is induced by xenobiotics, may offer an important clue to the origin(s) of CFS. The developing immune system is a sensitive and novel target for environmental insult (xenobiotic, infectious agents, stress) with major ramifications for postnatal health risks. Additionally, many prenatal and early postnatal neurological lesions associated with postnatal neurobehavioral diseases are now recognized as linked to prenatal immune insult and inflammatory dysregulation. This review considers the potential role of ELII including DIT as an early-life component of later-life CFS. (C) 2008 Elsevier Ireland Ltd. All rights reserved.
C1 [Dietert, Rodney R.] Cornell Univ, Coll Vet Med, Dept Microbiol & Immunol, Ithaca, NY 14853 USA.
[Dietert, Janice M.] Performace Plus Consulting, Lansing, NY 14882 USA.
RP Dietert, RR (reprint author), Cornell Univ, Coll Vet Med, Dept Microbiol & Immunol, C5-135 VMC,N Tower Rd, Ithaca, NY 14853 USA.
EM rrd1@cornell.edu
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NR 194
TC 11
Z9 12
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0300-483X
J9 TOXICOLOGY
JI Toxicology
PD MAY 2
PY 2008
VL 247
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BP 61
EP 72
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WC Pharmacology & Pharmacy; Toxicology
SC Pharmacology & Pharmacy; Toxicology
GA 297JN
UT WOS:000255613100008
PM 18336982
ER
PT J
AU Wallis, KE
Pinto-Martin, J
AF Wallis, Kate E.
Pinto-Martin, Jennifer
TI The challenge of screening for autism spectrum disorder in a culturally
diverse society
SO ACTA PAEDIATRICA
LA English
DT Editorial Material
ID CHILDREN
C1 [Wallis, Kate E.; Pinto-Martin, Jennifer] Univ Penn, Sch Nursing, Div Biobehav Hlth Sci, Philadelphia, PA 19104 USA.
[Pinto-Martin, Jennifer] Univ Penn, Sch Med, Dept Biostat & Epidemiol, Philadelphia, PA 19104 USA.
RP Wallis, KE (reprint author), Univ Penn, Sch Nursing, Div Biobehav Hlth Sci, 418 Curie Blvd,Claire M Fagin Hall,Room 445L, Philadelphia, PA 19104 USA.
EM wallisk@nursing.upenn.edu
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NR 6
TC 9
Z9 9
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
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J9 ACTA PAEDIATR
JI Acta Paediatr.
PD MAY
PY 2008
VL 97
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BP 539
EP 540
DI 10.1111/j.1651-2227.2008.00720.x
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SC Pediatrics
GA 288LT
UT WOS:000254988600006
PM 18373717
ER
PT J
AU Beaudet, AL
AF Beaudet, Arthur L.
TI Allan award lecture: Rare patients leading to epigenetics and back to
genetics - Arthur L. Beaudet
SO AMERICAN JOURNAL OF HUMAN GENETICS
LA English
DT Biographical-Item
ID PRADER-WILLI-SYNDROME; ANGELMAN SYNDROME; AUTISM; INHERITANCE;
MUTATIONS; UBE3A
C1 Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
RP Beaudet, AL (reprint author), Baylor Coll Med, Dept Mol & Human Genet, 1 Baylor Plaza,BCM225, Houston, TX 77030 USA.
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DI 10.1016/j.ajhg.2008.04.009
PG 5
WC Genetics & Heredity
SC Genetics & Heredity
GA 301VJ
UT WOS:000255923600003
PM 18468009
ER
PT J
AU Miles, JH
Takahashi, TN
Hong, J
Munden, N
Flournoy, N
Braddock, SR
Martin, RA
Spence, MA
Hillman, RE
Farmer, JE
AF Miles, Judith H.
Takahashi, T. Nicole
Hong, Julie
Munden, Nicole
Flournoy, Nancy
Braddock, Stephen R.
Martin, Rick A.
Spence, M. Anne
Hillman, Richard E.
Farmer, Janet E.
TI Development and validation of a measure of dysmorphology: Useful for
autism subgroup classification
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Article
DE autism; dysmorphology; congenital anomalies; screening; CART; complex
autism
ID MINOR PHYSICAL ANOMALIES; CONGENITAL-ANOMALIES; SPECTRUM DISORDERS;
CHILDHOOD AUTISM; CHILDREN; SCHIZOPHRENIA; ABNORMALITIES; MALFORMATIONS;
GENETICS; DEFECT
AB Autism spectrum disorders (ASD) comprise a class of neuro-developmental disorders that can originate from a variety of genetic and environmental causes. To delineate autism's heterogeneity we have looked for biologically-based phenotypes found in consistent proportions of ASD individuals. One informative phenotype is that of generalized dysmorphology, based on whole body examinations by medical geneticists trained in the nuances of anomalous embryologic development. We identified a need for a dysmorphology measure that could be completed by medical clinicians not extensively trained in dysmorphology that would still retain the level of sensitivity and specificity of the comprehensive dysmorphology examination. Based on expert-derived consensus dysmorphology designation of 222 autism patients and a classification validation study of 30 subjects by four dysmorphologists, we determined that dysmorphology designations based on body areas provided superior inter-rater reliability. Using 34 body area designations, we performed a classification and regression tree (CART) analysis to construct a scoring algorithm. Compared to the consensus classification, the model performed with 81% sensitivity and 99% specificity, and classification of a replication dataset of 31 ASD individuals performed well, with 82% sensitivity and 95% specificity. The autism dysmorphology measure (ADM) directs the clinician to score 12 body areas sequentially to arrive at a determination of "dysmorphic" or "nondysmorphic." We anticipate the ADM will permit clinicians to differentiate accurately between dysmorphic and nondysmorphic individuals-allowing better diagnostic classification, prognostication, recurrence risk assessment, and laboratory analysis decisions - and research scientists to better define more homogeneous autism subtypes. (C) 2008 Wiley-Liss, Inc.
C1 [Miles, Judith H.; Hillman, Richard E.] Univ Missouri, Hosp & Clin, Dept Child Hlth, Thompson Ctr Autism & Neurodev Disorders, Columbia, MO 65211 USA.
[Hong, Julie] Univ Tennessee, Hlth Sci Ctr, Dept Pediat, Memphis, TN USA.
[Munden, Nicole; Flournoy, Nancy] Univ Missouri, Dept Stat, Columbia, MO 65211 USA.
[Braddock, Stephen R.] Univ Virginia Hlth Syst, Dept Pediat, Div Genet, Charlottesville, VA USA.
[Martin, Rick A.] Cardinal Glennon Childrens Med Ctr, Div Med Genet, St Louis, MO USA.
[Spence, M. Anne] Univ Calif Irvine, Dept Pediat, Div Med Genet, Irvine, CA 92717 USA.
RP Miles, JH (reprint author), Univ Missouri, Hosp & Clin, Dept Child Hlth, Thompson Ctr Autism & Neurodev Disorders, 300 Portland St,Suite 110, Columbia, MO 65211 USA.
EM milesjh@missouri.edu
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NR 54
TC 21
Z9 21
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD MAY 1
PY 2008
VL 146A
IS 9
BP 1101
EP 1116
DI 10.1002/ajmg.a.32244
PG 16
WC Genetics & Heredity
SC Genetics & Heredity
GA 294RJ
UT WOS:000255422900001
PM 18383511
ER
PT J
AU Gamerdinger, U
Eggermann, T
Schubert, R
Schwanitz, G
Kreiss-Nachtsheim, M
AF Gamerdinger, Ulrilke
Eggermann, Thomas
Schubert, Regine
Schwanitz, Gesa
Kreiss-Nachtsheim, Martina
TI Rare Interstitial Deletion 9q31.2 to q33.1 de novo: Longitudinal study
in a patient over a period of more than 20 years
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Article
DE interstitial deletion 9q31.2q33.1; breakpoint characterization; parental
origin; phenotype-genotype correlation; longitudinal study
ID GORLIN-SYNDROME; CHROMOSOME-9; 9Q; ARM
AB The female carrier of a de novo interstitial deletion 9q [karyotype 46,XX,del(9q31.2q33.1)] was followed up over a period of more than 20 years. She shows facial dysmorphisms and significant growth retardation. Motor abilities are restricted by muscular hypotonia and malposition of the feet. She has mental retardation. There was no speech development and phases of autism were reported. By analyses with FISH and short tandem repeat markers, the interstitial deletion was confirmed and characterized to span 9q31.2q33.1, comprising at least 7.07 Mb. The aberration is of paternal origin. (C) 2008 Wiley-Liss, Inc.
C1 [Gamerdinger, Ulrilke] Univ Med Ctr Giessen & Marburg, Inst Pathol, D-35392 Giessen, Germany.
[Eggermann, Thomas] Rhein Westfal TH Aachen, Inst Human Genet, Aachen, Germany.
[Schubert, Regine] Praxis Human Genet, Cologne, Germany.
[Schwanitz, Gesa; Kreiss-Nachtsheim, Martina] Univ Bonn, Inst Human Genet, D-5300 Bonn, Germany.
RP Gamerdinger, U (reprint author), Univ Med Ctr Giessen & Marburg, Inst Pathol, Langhansstr 10, D-35392 Giessen, Germany.
EM ulrike.gamerdinger@patho.med.uni-giessen.de
RI Eggermann, Thomas/F-3807-2014
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NR 11
TC 6
Z9 6
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD MAY 1
PY 2008
VL 146A
IS 9
BP 1180
EP 1184
DI 10.1002/ajmg.a.32122
PG 5
WC Genetics & Heredity
SC Genetics & Heredity
GA 294RJ
UT WOS:000255422900010
PM 18386807
ER
PT J
AU Rajendra, K
Bringman, JJ
Ward, J
Phillips, OP
AF Rajendra, Krishna
Bringman, Jay J.
Ward, Jewel
Phillips, Owen P.
TI Who should be tested for fragile X carriership? A review of 1 center's
pedigrees
SO AMERICAN JOURNAL OF OBSTETRICS AND GYNECOLOGY
LA English
DT Article
DE fragile X syndrome; pedigree
ID WOMEN
AB OBJECTIVE: To determine whether following American College of Obstetricians and Gynecologists and American College of Medical Genetics recommendations would have detected carriers in pedigrees of patients diagnosed with fragile X.
STUDY DESIGN: Using a database of patients referred to the UT genetics clinic for evaluation of fragile X, pedigrees of cases of fragile X syndrome were analyzed.
RESULTS: Eight of 17 cases identified had a family history of unexplained mental retardation (MR) or fragile X MR and would have been diagnosed using current guidelines. Other findings noted in the pedigrees included autism, speech or hearing problems, attention deficit hyperactivity syndrome and behavioural disorders. No risk factors were found in 4 cases.
CONCLUSION: Using current guidelines, less than one half of fragile X carriers would have been identified during a prenatal assessment. Using other risk factors in screening would likely increase carrier detection rate.
C1 [Rajendra, Krishna; Bringman, Jay J.; Phillips, Owen P.] Univ Tennessee, Hlth Sci Ctr, Div Maternal Fetal Med, Dept Obstet & Gynecol, Memphis, TN 38103 USA.
[Ward, Jewel] Univ Tennessee, Hlth Sci Ctr, Dept Pediat, Div Genet, Memphis, TN 38103 USA.
RP Bringman, JJ (reprint author), Univ Tennessee, Hlth Sci Ctr, Div Maternal Fetal Med, Dept Obstet & Gynecol, 853 Jefferson Ave,Suite E102, Memphis, TN 38103 USA.
EM jbringma@utmem.edu
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NR 10
TC 2
Z9 2
PU MOSBY-ELSEVIER
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0002-9378
J9 AM J OBSTET GYNECOL
JI Am. J. Obstet. Gynecol.
PD MAY
PY 2008
VL 198
IS 5
AR e51
DI 10.1016/j.ajog.2007.12.023
PG 3
WC Obstetrics & Gynecology
SC Obstetrics & Gynecology
GA 295YV
UT WOS:000255511600076
PM 18358452
ER
PT J
AU Pinkham, AE
Sasson, NJ
Calkins, ME
Richard, J
Hughett, P
Gur, RE
Gur, RC
AF Pinkham, Amy E.
Sasson, Noah J.
Calkins, Monica E.
Richard, Jan
Hughett, Paul
Gur, Raquel E.
Gur, Ruben C.
TI The other-race effect in face processing among African American and
Caucasian individuals with schizophrenia
SO AMERICAN JOURNAL OF PSYCHIATRY
LA English
DT Article
ID FACIAL EMOTION RECOGNITION; IN-GROUP ADVANTAGE; SOCIAL COGNITION;
OWN-RACE; AUTISM; MEMORY; PERFORMANCE; IMPAIRMENT; PERCEPTION;
ACTIVATION
AB Objective: Studies of emotion recognition abilities in schizophrenia show greater impairment for non-Caucasians with schizophrenia compared with Caucasians. These studies, however, included only Caucasian faces as stimuli. There is evidence from healthy individuals for a performance disadvantage on face memory and emotion recognition when processing faces from a different ethnicity. The authors sought to measure the "other-race effect" in schizophrenia, which could account for previous findings and provide information about sensitivity to such social cues in patients.
Method: The study included 540 participants from four groups: African Americans with schizophrenia (N=135), Caucasians with schizophrenia (N=135), African American community comparison subjects (N=135), and Caucasian community comparison subjects (N=135). All participants completed face recognition and facial emotion identification tasks that included both Caucasian and African American faces as stimuli.
Results: Although comparison participants performed better than individuals with schizophrenia across all tasks, both comparison participants and participants with schizophrenia exhibited a strong and significant other-race effect for face memory and emotion recognition. The magnitude of the other-race effect did not differ between these two groups.
Conclusions: These findings reveal an intact other-race effect in patients with schizophrenia and highlight a methodological concern in the measurement of face processing abilities in schizophrenia, namely, that findings of greater impairment in African American patients are spurious when Caucasian faces are used as stimuli. Despite overall impairments in face memory and emotion recognition, the presence of a normative other-race effect in schizophrenia may reflect typical experiences with faces during development.
C1 Univ Penn, Dept Neuropsychiat, Philadelphia, PA 19104 USA.
RP Pinkham, AE (reprint author), Univ Penn, Dept Neuropsychiat, 10th Floor,Gates Pavill, Philadelphia, PA 19104 USA.
EM amypi@upenn.edu
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NR 39
TC 33
Z9 33
PU AMER PSYCHIATRIC PUBLISHING, INC
PI ARLINGTON
PA 1000 WILSON BOULEVARD, STE 1825, ARLINGTON, VA 22209-3901 USA
SN 0002-953X
J9 AM J PSYCHIAT
JI Am. J. Psychiat.
PD MAY
PY 2008
VL 165
IS 5
BP 639
EP 645
DI 10.1176/appi.ajp.2007.07101604
PG 7
WC Psychiatry
SC Psychiatry
GA 295YT
UT WOS:000255511400019
PM 18347000
ER
PT J
AU Wilkinson, K
Carlin, M
Thistle, J
AF Wilkinson, Krista
Carlin, Michael
Thistle, Jennifer
TI The role of color cues in facilitating accurate and rapid location of
aided symbols by children with and without down syndrome
SO AMERICAN JOURNAL OF SPEECH-LANGUAGE PATHOLOGY
LA English
DT Article
DE aided symbols; visual processing; visual search; symbol identification
ID STIMULUS OVER-SELECTIVITY; AUTISM SPECTRUM DISORDERS; LONG-TERM-MEMORY;
MENTAL-RETARDATION; INTELLECTUAL DISABILITIES; YOUNG-CHILDREN;
VISUAL-SEARCH; INDIVIDUALS; ATTENTION; RECOGNITION
AB Purpose: This research examined how the color distribution of symbols within a visual aided augmentative and alternative communication array influenced the speed and accuracy with which participants with and without Down syndrome located a target picture symbol.
Method: Eight typically developing children below the age of 4 years, 8 typically developing children over the age of 4 years, and 10 children with Down syndrome participated. Participants were asked to find a target line drawing among an array of 12. Line drawings represented either foods (e.g., grapes, cherries), clothing (e.g., a red shirt, a yellow shirt), or activities (e.g., soccer, swimming). In one condition, symbols that shared a color were clustered together, creating a subgroup within which to search. In another condition, symbols that shared a color were distributed across the display, allowing each to appear individually. Dependent measures were accuracy and speed of finding the target symbol.
Results: Clustering same-color symbols facilitated the speed of locating the target for all participants, and facilitated search accuracy in the younger preschool children and participants with Down syndrome. These effects held when targets were foods, clothing, or activities.
Conclusion: Clinicians should consider the internal color of visual symbols when constructing aided symbol displays, at least for children with Down syndrome. Further research is needed on a number of dimensions, however, including visual processing in other etiological categories, the role of background color, and the relation of color to other stimulus dimensions.
C1 [Wilkinson, Krista; Thistle, Jennifer] Emerson Coll, Boston, MA 02116 USA.
[Wilkinson, Krista; Carlin, Michael] Univ Massachusetts, Sch Med, Eunice Kennedy Shriver Ctr, Waltham, MA USA.
RP Wilkinson, K (reprint author), Emerson Coll, 120 Boylston St, Boston, MA 02116 USA.
EM krista-wilkinson@emerson.edu
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NR 54
TC 13
Z9 13
PU AMER SPEECH-LANGUAGE-HEARING ASSOC
PI ROCKVILLE
PA 10801 ROCKVILLE PIKE, ROCKVILLE, MD 20852-3279 USA
SN 1058-0360
J9 AM J SPEECH-LANG PAT
JI Am. J. Speech-Lang. Pathol.
PD MAY
PY 2008
VL 17
IS 2
BP 179
EP 193
DI 10.1044/1058-0360(2008/018)
PG 15
WC Audiology & Speech-Language Pathology; Linguistics; Rehabilitation
SC Audiology & Speech-Language Pathology; Linguistics; Rehabilitation
GA 295OZ
UT WOS:000255485100008
PM 18448605
ER
PT J
AU Carlin, MT
Toglia, MP
Wakeford, Y
Jakway, A
Sullivan, K
Hasel, L
AF Carlin, Michael T.
Toglia, Michael P.
Wakeford, Yvonne
Jakway, Allison
Sullivan, Kate
Hasel, Lisa
TI Veridical and false pictorial memory in individuals with and without
mental retardation
SO AMERICAN JOURNAL ON MENTAL RETARDATION
LA English
DT Article
ID CHILDREN; RECALL; RECOGNITION; ADULTS; DISTINCTIVENESS; INTERVIEWS;
AUTISM; WORDS; ACQUIESCENCE; ILLUSION
AB Veridical and false pictorial recognition were assessed in individuals with mental retardation; groups were matched for MA and CA. Pictures were viewed in either a generative or static format at acquisition. The individuals with mental retardation and those in the MA-matched group had higher rates of false memories for critical items and lower hit rates than did their CA-matched peers. The mental retardation group demonstrated an acquiescent response bias (i.e., high novel false-alarm rate). When data were corrected for this bias, those with mental retardation had significantly lower hit rates but equivalent false-alarm rates to the MA-matched participants. Results are discussed in terms of pictorial distinctiveness and within the frameworks of activation monitoring and fuzzy trace theory.
C1 [Carlin, Michael T.] Rider Univ, Dept Psychol, Lawrenceville, NJ 08648 USA.
[Toglia, Michael P.] SUNY Coll Cortland, Cortland, NY 13045 USA.
[Wakeford, Yvonne] Tufts Univ, Medford, MA 02155 USA.
[Jakway, Allison; Sullivan, Kate] Univ Massachusetts, Eunice Kennedy Shriver Ctr, Sch Med, Amherst, MA 01003 USA.
[Hasel, Lisa] Iowa State Univ, Ames, IA 50011 USA.
RP Carlin, MT (reprint author), Rider Univ, Dept Psychol, Lawrenceville, NJ 08648 USA.
EM mcarlin@rider.edu
RI Hasel, Lisa/A-2518-2011
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NR 41
TC 4
Z9 4
PU AMER ASSOC MENTAL RETARDATION
PI WASHINGTON
PA 444 N CAPITOL ST, NW, STE 846, WASHINGTON, DC 20001-1512 USA
SN 0895-8017
J9 AM J MENT RETARD
JI Am. J. Ment. Retard.
PD MAY
PY 2008
VL 113
IS 3
BP 201
EP 213
DI 10.1352/0895-8017(2008)113[201:VAFPMI]2.0.CO;2
PG 13
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 296OP
UT WOS:000255556000005
PM 18407722
ER
PT J
AU Abbecluto, L
Murphy, MM
Kover, ST
Giles, ND
Karaclottir, S
Amman, A
Bruno, L
Kim, JS
Schroeder, S
Anderson, JA
Nollin, KA
AF Abbecluto, Leonard
Murphy, Melissa M.
Kover, Sara T.
Giles, Nancy D.
Karaclottir, Selma
Amman, Adrienne
Bruno, Loreclana
Kim, Jee-Seon
Schroeder, Susen
Anderson, Julie A.
Nollin, Kathryn A.
TI Signaling noncomprehension of language: A comparison of fragile X
syndrome and Down syndrome
SO AMERICAN JOURNAL ON MENTAL RETARDATION
LA English
DT Article
ID COMPREHENSION MONITORING SKILLS; MENTAL-RETARDATION; REFERENTIAL
COMMUNICATION; RETARDED-ADULTS; CHILDREN; INDIVIDUALS; AUTISM; MIND;
ADOLESCENTS; SPECIFICITY
AB Signaling noncomprehension of the spoken messages of others was examined for youth with fragile X or Down syndrome in comparison with each other and nonverbal MA-matched typically developing children. A direction-following task was used in which some of the directions were inadequate. Both syndrome groups signaled noncomprehension less often than did the typically developing children. The ability to signal noncomprehension appropriately was related to a measure of receptive vocabulary and syntax. Preliminary analyses indicated that males with fragile X syndrome signaled noncomprehension less often than did their female peers, even after controlling for differences in nonverbal MA.
C1 [Abbecluto, Leonard; Kover, Sara T.; Giles, Nancy D.; Karaclottir, Selma; Amman, Adrienne; Bruno, Loreclana; Kim, Jee-Seon; Schroeder, Susen; Anderson, Julie A.; Nollin, Kathryn A.] Univ Wisconsin, Waisman Ctr, Madison, WI 53705 USA.
[Murphy, Melissa M.] Johns Hopkins Univ, Kennedy Krieger Res Inst, Baltimore, MD 21218 USA.
RP Abbecluto, L (reprint author), Univ Wisconsin, Waisman Ctr, 1500 Highland Ave, Madison, WI 53705 USA.
EM Abbeduto@waisman.wisc.edu
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NR 62
TC 20
Z9 20
PU AMER ASSOC MENTAL RETARDATION
PI WASHINGTON
PA 444 N CAPITOL ST, NW, STE 846, WASHINGTON, DC 20001-1512 USA
SN 0895-8017
J9 AM J MENT RETARD
JI Am. J. Ment. Retard.
PD MAY
PY 2008
VL 113
IS 3
BP 214
EP 230
DI 10.1352/0895-8017(2008)113[214:SNOLAC]2.0.CO;2
PG 17
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 296OP
UT WOS:000255556000006
PM 18407723
ER
PT J
AU Preissler, MA
AF Preissler, Melissa Allen
TI Associative learning of pictures and words by low-functioning children
with autism
SO AUTISM
LA English
DT Article
DE associative learning; autism; symbolic development
ID INFANTS ABILITY; LANGUAGE; COMMUNICATION; DEFICITS
AB This research investigates whether children with autism learn picture, word and object relations as associative pairs or whether they understand such relations as referential. In Experiment 1, children were taught a new word (e.g. 'whisk') repeatedly paired with a novel picture. When given the picture and a previously unseen real whisk and asked to indicate a whisk, children with autism, unlike typically developing peers matched on receptive language, associated the word with the picture rather than the object. Subsequent experiments respectively confirmed that neither a bias for selecting pictures nor perseverative responding accounted for these results. Taken together, these results suggest that children with autism with cognitive difficulties are learning picture-word and picture-object relations via an associative mechanism and have difficulty understanding the symbolic nature of pictures.
C1 Univ Lancaster, Dept Psychol, Fylde Coll, Lancaster LA1 4YF, England.
RP Preissler, MA (reprint author), Univ Lancaster, Dept Psychol, Fylde Coll, Lancaster LA1 4YF, England.
EM melissa.allen@lancaster.ac.uk
RI Allen, Melissa/F-9711-2011
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NR 35
TC 15
Z9 15
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAY
PY 2008
VL 12
IS 3
BP 231
EP 248
DI 10.1177/1362361307088753
PG 18
WC Psychology, Developmental
SC Psychology
GA 301MP
UT WOS:000255900600002
PM 18445733
ER
PT J
AU Rinehart, NJ
Bradshaw, JL
Moss, SA
Brereton, AV
Tonge, BJ
AF Rinehart, Nicole J.
Bradshaw, John L.
Moss, Simon A.
Brereton, Avril V.
Tonge, Bruce J.
TI Brief report: Inhibition of return in young people with autism and
Asperger's disorder
SO AUTISM
LA English
DT Article
DE Asperger's disorder; autism; inhibition of return; visual search
ID HIGH-FUNCTIONING AUTISM; VISUAL-SEARCH; CHILDREN
AB The aim of this study was to investigate whether the superior search abilities observed in autism/Asperger's disorder may in part be a consequence of a more pronounced inhibition of return (IOR). Contrary to our prediction, IOR in individuals with autism was comparable to the matched comparison group. However, the autism group committed more false alarm responses than the matched comparison group; this may reflect a possible inhibitory deficit, or suggest that individuals with autism rely more on probabilities to determine their behavioural responses. There was a borderline-significant trend (p = 0.052) to indicate that IOR may be more pronounced in individuals with Asperger's disorder. In contrast to the autism group, the Asperger's disorder group had a pattern of false alarm responses similar to that of the comparison group. The findings further inform Minshew's complex information processing theory which seeks to establish which areas of neuropsychological functioning are preserved and deficit in autism.
C1 [Rinehart, Nicole J.] Monash Univ, Sch Psychol Psychiat & Psychol Med, Ctr Dev Psychiat & Psychol, Notting Hill, Vic 3168, Australia.
RP Rinehart, NJ (reprint author), Monash Univ, Sch Psychol Psychiat & Psychol Med, Ctr Dev Psychiat & Psychol, Bldg 1,270 Ferntree Gully Rd, Notting Hill, Vic 3168, Australia.
EM nicole.rinehart@med.monash.edu.au
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NR 23
TC 9
Z9 9
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAY
PY 2008
VL 12
IS 3
BP 249
EP 260
DI 10.1177/1362361307088754
PG 12
WC Psychology, Developmental
SC Psychology
GA 301MP
UT WOS:000255900600003
PM 18445734
ER
PT J
AU Parron, C
Da Fonseca, D
Santos, A
Moore, DG
Monfardini, E
Deruelle, C
AF Parron, Carole
Da Fonseca, David
Santos, Andreia
Moore, David G.
Monfardini, Elisa
Deruelle, Christine
TI Recognition of biological motion in children with autistic spectrum
disorders
SO AUTISM
LA English
DT Article
DE autistic children; biological motion; configural processing; emotion
ID POINT-LIGHT DISPLAYS; MENTAL-RETARDATION; ASPERGER-SYNDROME;
DEVELOPMENTAL DISORDERS; EMOTION RECOGNITION; FACIAL EXPRESSIONS;
PERCEPTION; INDIVIDUALS; FACE; DISCRIMINATION
AB It is widely accepted that autistic children experience difficulties in processing and recognizing emotions. Most relevant studies have explored the perception of faces. However, context and bodily gestures are also sources from which we derive emotional meanings. We tested 23 autistic children and 23 typically developing control children on their ability to recognize point-light displays of a person's actions, subjective states and emotions. In a control task, children had to recognize point-light displays of everyday objects. The children with autism only differed from the control children in their ability to name the emotional point-light displays. This suggests that children with autism can extract complex meanings from bodily movements but may be less sensitive to higher-order emotional information conveyed by human movement. The results are discussed in the context of a specific deficit in emotion perception in children with autism.
C1 [Deruelle, Christine] CNRS INCM, F-13402 Marseille 20, France.
[Da Fonseca, David] Ste Marguerite Hosp, Marseille, France.
[Moore, David G.] Univ E London, London E15 4LZ, England.
RP Deruelle, C (reprint author), CNRS INCM, 31 Chemin Joseph Aiguier, F-13402 Marseille 20, France.
EM deruelle@incm.cnrs-mrs.fr
RI deruelle, christine/E-2130-2015
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NR 33
TC 46
Z9 47
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAY
PY 2008
VL 12
IS 3
BP 261
EP 274
DI 10.1177/1362361307089520
PG 14
WC Psychology, Developmental
SC Psychology
GA 301MP
UT WOS:000255900600004
PM 18445735
ER
PT J
AU Roos, EM
McDuffie, AS
Weismer, SE
Gernsbacher, MA
AF Roos, Elizabeth M.
McDuffie, Andrea S.
Weismer, Susan Ellis
Gernsbacher, Morton Ann
TI A comparison of contexts for assessing joint attention in toddlers on
the autism spectrum
SO AUTISM
LA English
DT Article
DE autism; joint attention; naturalistic play; social communication
ID EARLY RECOGNITION; YOUNG-CHILDREN; DISORDERS; COMMUNICATION; INFANTS;
DEFICITS; SKILL; 1ST
AB Children on the autism spectrum often demonstrate atypical joint attention, leading some researchers to consider joint attention defecits a core feature of the autism spectrum. Structured measures, such as the Early Social Communication Scales (ESCS), are commonly used to provide a metric of joint attention. To explore the assessment of joint attention in multiple contexts, we implemented an alternative system for coding joint attention behaviors. We compared initiation of joint attention (IJA) and response to joint attention (RJA) behaviors coded from naturalistic examiner-child play samples with similar IJA and RJA behaviors elicited within the structured ESCS protocol. Participants were 20 toddlers on the autism spectrum. Levels of IJA and RJA within the two assessment contexts were significantly and positively correlated, providing support for the use of naturalistic sampling of joint attention skills as a viable alternative, or supplement, to structured measures.
C1 [Roos, Elizabeth M.] Univ Wisconsin, Waisman Ctr, Madison, WI 53705 USA.
RP Roos, EM (reprint author), Univ Wisconsin, Waisman Ctr, Room 441,1500 Highland Ave, Madison, WI 53705 USA.
EM eroos@wisc.edu
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NR 29
TC 10
Z9 11
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAY
PY 2008
VL 12
IS 3
BP 275
EP 291
DI 10.1177/1362361307089521
PG 17
WC Psychology, Developmental
SC Psychology
GA 301MP
UT WOS:000255900600005
PM 18445736
ER
PT J
AU Schultz, ST
Klonoff-Cohen, HS
Wingard, DL
Akshoomoff, NA
Macera, CA
Ji, M
AF Schultz, Stephen T.
Klonoff-Cohen, Hillary S.
Wingard, Deborah L.
Akshoomoff, Natacha A.
Macera, Caroline A.
Ji, Ming
TI Acetaminophen (paracetamol) use, measles-mumps-rubella vaccination, and
autistic disorder - The results of a parent survey
SO AUTISM
LA English
DT Article
DE acetaminophen; autism; paracetamol; vaccination
ID NO EVIDENCE; CHILDREN; MMR; POPULATION; ANTIBODIES; REGRESSION
AB The present study was performed to determine whether acetaminophen (paracetamol) use after the measles-mumps-rubella vaccination could be associated with autistic disorder. This case-control study used the results of an online parental survey conducted from 16 July 2005 to 30 January 2006, consisting of 83 children with autistic disorder and 80 control children. Acetaminophen use after measles-mumps-rubella vaccination was significantly associated with autistic disorder when considering children 5 years of age or less (OR 6.11, 95% CI 1.42-26.3), after limiting cases to children with regression in development (OR 3.97, 95% CI 1.11-14.3), and when considering only children who had post-vaccination sequelae (OR 8.23, 95% CI 1.56-43.3), adjusting for age, gender, mother's ethnicity, and the presence of illness concurrent with measles-mumps-rubella vaccination. Ibuprofen use after measles-mumps-rubella vaccination was not associated with autistic disorder. This preliminary study found that acetaminophen use after measles-mumps-rubella vaccination was associated with autistic disorder.
C1 [Schultz, Stephen T.; Klonoff-Cohen, Hillary S.; Wingard, Deborah L.; Akshoomoff, Natacha A.] Univ Calif San Diego, La Jolla, CA 92093 USA.
[Schultz, Stephen T.; Macera, Caroline A.; Ji, Ming] San Diego State Univ, San Diego, CA 92182 USA.
RP Schultz, ST (reprint author), 943 Water Thrush Court, Antioch, IL 60002 USA.
EM Stephen.schultz@med.navy.mil
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NR 32
TC 24
Z9 25
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAY
PY 2008
VL 12
IS 3
BP 293
EP 307
DI 10.1177/1362361307089518
PG 15
WC Psychology, Developmental
SC Psychology
GA 301MP
UT WOS:000255900600006
PM 18445737
ER
PT J
AU Osborne, LA
Reed, P
AF Osborne, Lisa A.
Reed, Phil
TI Parents' perceptions of communication with professionals during the
diagnosis of autism
SO AUTISM
LA English
DT Article
DE autism; diagnosis; information giving; parental experience
ID SPECTRUM DISORDER; YOUNG-CHILDREN; STRESS; DISABILITY; IMPACT
AB In order to obtain the views of parents concerning their perceptions of the process of getting a diagnosis of an autistic spectrum disorder (ASD) for their child, 15 focus groups were conducted across a range of locations in England. These groups were split into parents of preschool-, primary- and secondary-aged children who had recently received an ASD diagnosis. At the time of diagnosis, most of the parents wished for a quicker and easier process. In particular, they would prefer the procedure to have a more coherent structure and content. They also requested greater professional training about ASD, in particular, regarding the information that professionals possess, and the interpersonal skills of some professionals. The idea of broad information sheets to be provided to parents at the time of diagnosis would be of value, especially to combat negative information provided from other sources.
C1 [Reed, Phil] Univ Coll Swansea, Dept Psychol, Swansea SA2 8PP, W Glam, Wales.
RP Reed, P (reprint author), Univ Coll Swansea, Dept Psychol, Singleton Pk, Swansea SA2 8PP, W Glam, Wales.
EM p.reed@swansea.ac.uk
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Randall P., 1999, SUPPORTING FAMILIES
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Vaughn S., 1996, FOCUS GROUP INTERVIE
WOLF LC, 1989, J AUTISM DEV DISORD, V19, P157, DOI 10.1007/BF02212727
NR 25
TC 22
Z9 22
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAY
PY 2008
VL 12
IS 3
BP 309
EP 324
DI 10.1177/1362361307089517
PG 16
WC Psychology, Developmental
SC Psychology
GA 301MP
UT WOS:000255900600007
PM 18445738
ER
PT J
AU Meyer, U
Nyffeler, M
Yee, BK
Knuesel, I
Feldon, J
AF Meyer, Urs
Nyffeler, Myriel
Yee, Benjamin K.
Knuesel, Irene
Feldon, Joram
TI Adult brain and behavioral pathological markers of prenatal immune
challenge during early/middle and late fetal development in mice
SO BRAIN BEHAVIOR AND IMMUNITY
LA English
DT Article
DE animal model; autism; cytokines; infection; neurodevelopment; pregnancy;
schizophrenia
ID METHYL-D-ASPARTATE; NEURODEVELOPMENTAL ANIMAL-MODEL; SPATIAL
WORKING-MEMORY; NMDA RECEPTOR FUNCTION; NUCLEUS-ACCUMBENS; PREPULSE
INHIBITION; DOPAMINE RELEASE; VENTRAL HIPPOCAMPUS; DIZOCILPINE MK-801;
HEALTHY-VOLUNTEERS
AB Maternal infection during pregnancy increases the risk for neurodevelopmental disorders such as schizophrenia and autism in the offspring. This association appears to be critically dependent on the precise prenatal timing. However, the extent to which distinct adult psychopathological and neuropathological traits may be sensitive to the precise times of prenatal immune activation remains to be further characterized. Here, we evaluated in a mouse model of prenatal immune challenge by the viral mimic, polyriboinosinic-polyribocytidilic acid (PolyIC), whether prenatal immune activation in early/middle and late gestation may influence the susceptibility to some of the critical cognitive, pharmacological, and neuroanatomical dysfunctions implicated in schizophrenia and autism. We revealed that PolyIC-induced prenatal immune challenge on gestation day (GD) 9 but not GD17 significantly impaired sensorimotor gating and reduced prefrontal dopamine D1 receptors in adulthood, whereas prenatal immune activation specifically in late gestation impaired working memory, potentiated the locomotor reaction to the NMDA-receptor antagonist dizocilpine, and reduced hippocampal NMDA-receptor subunit 1 expression. On the other hand, potentiation of the locomotor reaction to the dopamine-receptor agonist amphetamine and reduction in Reelin- and Parvalbumin-expressing prefrontal neurons emerged independently of the precise times of prenatal immune challenge. Our findings thus highlight that prenatal immune challenge during early/middle and late fetal development in mice leads to distinct brain and behavioral pathological symptom clusters in adulthood. Further examination and evaluation of in utero immune challenge at different times of gestation may provide important new insight into the neuroimmunological and neuropathological mechanisms underlying the segregation of different symptom clusters in heterogeneous neuropsychiatric disorders such as schizophrenia and autism. (c) 2007 Elsevier Inc. All rights reserved.
C1 [Meyer, Urs; Nyffeler, Myriel; Yee, Benjamin K.; Feldon, Joram] Swiss Fed Inst Technol, Lab Behav Neurobiol, Schwerzenbach, Switzerland.
[Knuesel, Irene] Univ Zurich, Inst Pharmacol & Toxicol, CH-8006 Zurich, Switzerland.
RP Feldon, J (reprint author), Swiss Fed Inst Technol, Lab Behav Neurobiol, Schwerzenbach, Switzerland.
EM feldon@behav.biol.ethz.ch
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NR 75
TC 168
Z9 170
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0889-1591
J9 BRAIN BEHAV IMMUN
JI Brain Behav. Immun.
PD MAY
PY 2008
VL 22
IS 4
BP 469
EP 486
DI 10.1016/j.bbi.2007.09.012
PG 18
WC Immunology; Neurosciences
SC Immunology; Neurosciences & Neurology
GA 298ZE
UT WOS:000255724800008
PM 18023140
ER
PT J
AU Turkstra, LS
AF Turkstra, Lyn S.
TI (Conversation-based assessment of social cognition in adults with
traumatic brain injury
SO BRAIN INJURY
LA English
DT Article
DE social cognition; theory of mind; brain injury; pragmatic language;
inference
ID CLOSED-HEAD-INJURY; WORKING-MEMORY CAPACITY; HIGH-FUNCTIONING AUTISM;
FALSE-BELIEF TASK; INDIVIDUAL-DIFFERENCES; LOBE CONTRIBUTIONS;
ASPERGER-SYNDROME; ELICITATION TASK; FOLLOW-UP; MIND
AB Primary objective: The purpose of this study was to characterize performance of individuals with traumatic brain injury (TBI) on a novel video-based test, the Video Social Inference Test (VSIT). The VSIT was designed to capture social inference processes that would be engaged in daily conversations. The test required both initial social inferences and also predictions or explanations of subsequent behaviours.
Research design: Between-groups comparison.
Methods: Adults with TBI (n=19) and typical controls matched for age and sex (n=19) completed the VSIT, as well as a working memory test and the Eyes Test, a widely-used picture-based test of social cognition.
Results: VSIT scores were lower in the TBI group and higher than on the Eyes Test. Participants in both groups had lower scores when required to predict or explain future behaviours based on an initial social inference.
Conclusions and implications: The results suggest that conversation-based stimuli may yield unique and useful information about social cognition beyond the laboratory.
C1 [Turkstra, Lyn S.] Univ Wisconsin, Dept Communicat Disorders, Madison, WI 53706 USA.
RP Turkstra, LS (reprint author), Univ Wisconsin, Dept Communicat Disorders, 1975 Willow Dr, Madison, WI 53706 USA.
EM lsturkstra@wisc.edu
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NR 80
TC 14
Z9 14
PU TAYLOR & FRANCIS LTD
PI ABINGDON
PA 4 PARK SQUARE, MILTON PARK, ABINGDON OX14 4RN, OXON, ENGLAND
SN 0269-9052
J9 BRAIN INJURY
JI Brain Inj.
PD MAY
PY 2008
VL 22
IS 5
BP 397
EP 409
DI 10.1080/02699050802027059
PG 13
WC Neurosciences; Rehabilitation
SC Neurosciences & Neurology; Rehabilitation
GA 302RH
UT WOS:000255986500005
PM 18415720
ER
PT J
AU Berney, T
AF Berney, Tom
TI New Developments in Autism: The Future is Today
SO CHILD AND ADOLESCENT MENTAL HEALTH
LA English
DT Book Review
C1 [Berney, Tom] Prudhoe Hosp, Prudhoe, Northd, England.
RP Berney, T (reprint author), Prudhoe Hosp, Prudhoe, Northd, England.
CR PEREZ JM, 2006, NEW DEV AUTISM FUTUR
NR 1
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1475-357X
J9 CHILD ADOL MENT H-UK
JI Child Adolesc. Ment. Health
PD MAY
PY 2008
VL 13
IS 2
BP 99
EP 99
DI 10.1111/j.1475-3588.2008.00486_2.x
PG 1
WC Psychology, Clinical; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA V16EY
UT WOS:000207854100012
ER
PT J
AU Wyatt, TH
Hauenstein, E
AF Wyatt, Tami H.
Hauenstein, Emily
TI Enhancing children's health through digital story
SO CIN-COMPUTERS INFORMATICS NURSING
LA English
DT Article
DE child health; computer-assisted instruction; digital storytelling;
health education; personal narrative
ID SOCIAL STORIES; MULTIMEDIA; AUTISM
AB Stories in all of their many forms, including books, plays, skits, movies, poems, and songs, appeal to individuals of all ages but especially the young. Children are easily engaged in stories, and today's generation of children, the millennium generation, demands interactive, multimedia-rich environments. Story as a teaching and learning technique is pervasive in the classroom but is infrequently used to promote health. Because of advancing technology, it is possible to create interactive digital storytelling programs that teach children health topics. Using digital storytelling in an interactive environment to promote health has not been tested, but there is empirical support for using story in health education and interactive technology to promote health. This article briefly reviews the literature and discusses how technology and storytelling can be joined to promote positive health outcomes.
C1 [Wyatt, Tami H.] Univ Tennessee, Coll Nursing, Knoxville, TN 37996 USA.
[Hauenstein, Emily] Univ Virginia, SE Rural Mental Hlth Res Ctr, Charlottesville, VA USA.
RP Wyatt, TH (reprint author), Univ Tennessee, Coll Nursing, 1200 Volunteer Blvd, Knoxville, TN 37996 USA.
EM twyatt@utk.edu
RI Hauenstein, Emily/A-2338-2012
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NR 46
TC 3
Z9 3
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 1538-2931
J9 CIN-COMPUT INFORM NU
JI CIN-Comput. Inform. Nurs.
PD MAY-JUN
PY 2008
VL 26
IS 3
BP 142
EP 148
DI 10.1097/01.NCN.0000304779.49941.44
PG 7
WC Computer Science, Interdisciplinary Applications; Medical Informatics;
Nursing
SC Computer Science; Medical Informatics; Nursing
GA 297FB
UT WOS:000255601400007
PM 18438149
ER
PT J
AU Coben, R
Clarke, AR
Hudspeth, W
Barry, RJ
AF Coben, Robert
Clarke, Adam R.
Hudspeth, William
Barry, Robert J.
TI EEG power and coherence in autistic spectrum disorder
SO CLINICAL NEUROPHYSIOLOGY
LA English
DT Article
DE autism; children; EEG; coherence; diagnosis
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; INFANTILE-AUTISM; 2 SUBTYPES;
CONNECTIVITY; CHILDREN; BRAIN; UNDERCONNECTIVITY; LATERALIZATION;
INDIVIDUALS; MORPHOMETRY
AB Objective: Autistic spectrum disorder (ASD) has been defined as a neurodevelopmental disorder with associated deficits in executive function, language, emotional, and social function. ASD has been associated with pathophysiology in cerebral organization. The current study investigated quantitative EEG findings in twenty children diagnosed with autistic disorders as compared to 20 controls matched for gender, age and IQ.
Methods: The EEG was recorded during an eyes-closed resting condition and topographical differences in cerebral functioning were examined using estimates of absolute, relative, and total power, as well as intrahemispheric and interhemispheric coherences.
Results: There were group differences in power, intrahemispheric and interhemispheric coherences. Findings included excessive theta, primarily in right posterior regions, in autistics. There was also a pattern of deficient delta over the frontal cortex and excessive midline beta. More significantly, there was a pattern of underconnectivity in autistics compared to controls. This included decreased intrahemispheric delta and theta coherences across short to medium and long inter-electrode distances. Interhemispherically, delta and theta coherences were low across the frontal region. Delta, theta and alpha hypocoherence was also evident over the temporal regions. Lastly, there were low delta, theta and beta coherence measurements across posterior regions.
Conclusions: These results suggest dysfunctional integration of frontal and posterior brain regions in autistics along with a pattern of neural underconnectivity. This is consistent with other EEG, MRI and fMRI research suggesting that neural connectivity anomalies are a major deficit leading to autistic symptomatology.
Significance: This paper reports the largest integrated study of EEG power and coherence during a resting state in children suffering autism spectrum disorder. (c) 2008 International Federation of Clinical Neurophysiology. Published by Elsevier Ireland Ltd. All rights reserved.
C1 [Coben, Robert] Neurorehabil & Neuropsychol Serv, Massapequa Pk, NY 11762 USA.
[Clarke, Adam R.; Barry, Robert J.] Univ Wollongong, Brain & Behav Res Inst, Wollongong, NSW 2522, Australia.
[Clarke, Adam R.; Barry, Robert J.] Univ Wollongong, Sch Psychol, Wollongong, NSW 2522, Australia.
[Hudspeth, William] Neuropsychometrix, Los Osos, CA 93402 USA.
RP Coben, R (reprint author), Neurorehabil & Neuropsychol Serv, 1035 Pk Blvd,Suite 2B, Massapequa Pk, NY 11762 USA.
EM robcoben@optonline.net
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TABACHNICK BARBARA G., 1989, USING MULTIVARIATE S, V2d
NR 38
TC 85
Z9 86
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 1388-2457
J9 CLIN NEUROPHYSIOL
JI Clin. Neurophysiol.
PD MAY
PY 2008
VL 119
IS 5
BP 1002
EP 1009
DI 10.1016/j.clinph.2008.01.013
PG 8
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 298VW
UT WOS:000255716200005
PM 18331812
ER
PT J
AU Bull, R
Phillips, LH
Conway, CA
AF Bull, Rebecca
Phillips, Louise H.
Conway, Claire A.
TI The role of control functions in mentalizing: Dual-task studies of
Theory of Mind and executive function
SO COGNITION
LA English
DT Article
DE theory of mind; executive functioning; dual-task
ID NEUROPSYCHOLOGICAL EVIDENCE; WORKING-MEMORY; FRONTOTEMPORAL DEMENTIA;
STORY COMPREHENSION; DOMAIN-SPECIFICITY; OLD-AGE; AUTISM; DISSOCIATION;
PRESCHOOLERS; SELECTION
AB Conflicting evidence has arisen from correlational studies regarding the role of executive control functions in Theory of Mind. The current study used dual-task manipulations of executive functions (inhibition, updating and switching) to investigate the role of these control functions ill mental state and non-mental state tasks. The 'Eyes' pictorial test of Theory of Mind showed specific dual-task costs when concurrently performed with an inhibitory secondary task. In contrast, interference effects on a verbal 'Stories' task were general, occurring on both mental state and non-mental state tasks, and across all types of executive function. These findings from healthy functioning adults should help to guide decisions about appropriate methods of assessing ToM in clinical populations, and interpreting deficits in performance in such tasks in the context of more general cognitive dysfunction. (C) 2007 Elsevier B.V. All rights reserved.
C1 [Bull, Rebecca; Phillips, Louise H.; Conway, Claire A.] Univ Aberdeen, Sch Psychol, Aberdeen AB24 2UB, Scotland.
RP Bull, R (reprint author), Univ Aberdeen, Sch Psychol, William Guild Bldg, Aberdeen AB24 2UB, Scotland.
EM r.bull@abdn.ac.uk
RI Bull, Rebecca/A-7895-2008; Phillips, Louise/A-7952-2008
OI Phillips, Louise/0000-0003-1005-8567
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NR 39
TC 42
Z9 42
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0010-0277
J9 COGNITION
JI Cognition
PD MAY
PY 2008
VL 107
IS 2
BP 663
EP 672
DI 10.1016/j.cognition.2007.07.015
PG 10
WC Psychology, Experimental
SC Psychology
GA 298RL
UT WOS:000255704700013
PM 17765214
ER
PT J
AU Buccino, G
Amore, M
AF Buccino, Giovanni
Amore, Mario
TI Mirror neurons and the understanding of behavioural symptoms in
psychiatric disorders
SO CURRENT OPINION IN PSYCHIATRY
LA English
DT Article
DE autism; empathy; mirror neuron system; schizophrenia
ID SHARED MANIFOLD HYPOTHESIS; AUTISM SPECTRUM DISORDERS; SOCIAL COGNITION;
HAND ACTIONS; NEURAL BASIS; SYSTEM; MOTOR; SCHIZOPHRENIA; EMPATHY;
IMITATION
AB Purpose of review
Recent findings show that we can understand other people's actions, intentions and emotions through a mirror mechanism as if we performed the same actions and felt the same intentions or emotions (embodied simulation). The present paper reviews experimental evidence that this mechanism may be broken in some psychiatric disorders.
Recent findings
A mirror neuron system has been described in both monkeys and humans that allows one to map an observed action on a correspondent motor representation in the observer's brain. This mechanism has been involved in many higher motor functions ranging from action understanding to imitation and intention coding. A mirror mechanism has also been invoked in empathy, through an embodied simulation.
Summary
A dysfunction of the mirror neuron system may be at the root of the inability to empathize in patients with autism and may play a role in some negative and positive symptoms found in patients with schizophrenia. This opens up new perspectives in the interpretation of psychotic symptoms and possibly in developing therapeutic strategies.
C1 [Buccino, Giovanni; Amore, Mario] Univ Parma, Dept Neurosci, Physiol Sect, I-43100 Parma, Italy.
RP Buccino, G (reprint author), Univ Parma, Dept Neurosci, Physiol Sect, Via Volturno 39, I-43100 Parma, Italy.
EM giovanni.buccino@unipr.it
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NR 48
TC 24
Z9 25
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0951-7367
J9 CURR OPIN PSYCHIATR
JI Curr. Opin. Psychiatr.
PD MAY
PY 2008
VL 21
IS 3
BP 281
EP 285
DI 10.1097/YCO.0b013e3282fbcd32
PG 5
WC Psychiatry
SC Psychiatry
GA 291YK
UT WOS:000255233200010
PM 18382228
ER
PT J
AU Howlin, P
AF Howlin, Patricia
TI Autism and diagnostic substitution
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Editorial Material
ID INFANTILE-AUTISM; FOLLOW-UP; DISORDER
C1 Kings Coll London, Inst Psychiat, London WC2R 2LS, England.
RP Howlin, P (reprint author), Kings Coll London, Inst Psychiat, London WC2R 2LS, England.
RI Howlin, Patricia/A-7622-2011
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NR 6
TC 1
Z9 1
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD MAY
PY 2008
VL 50
IS 5
BP 325
EP 325
DI 10.1111/j.1469-8749.2008.00325.x
PG 1
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 288MC
UT WOS:000254989500003
PM 18416720
ER
PT J
AU Bishop, DVM
Whitehouse, AJO
Watt, HJ
Line, EA
AF Bishop, Dorothy V. M.
Whitehouse, Andrew J. O.
Watt, Helen J.
Line, Elizabeth A.
TI Autism and diagnostic substitution: evidence from a study of adults with
a history of developmental language disorder
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Article
ID SEMANTIC-PRAGMATIC DISORDER; CHILDRENS COMMUNICATION CHECKLIST; SPECTRUM
DISORDERS; FOLLOW-UP; IMPAIRMENT; PREVALENCE; DEFICITS; OUTCOMES; LIFE;
SLI
AB Rates of diagnosis of autism have risen since 1980, raising the question of whether some children who previously had other diagnoses are now being diagnosed with autism. We applied contemporary diagnostic criteria for autism to adults with a history of developmental language disorder, to discover whether diagnostic substitution has taken place. A total of 38 adults (aged 15-31y; 31 males, seven females) who had participated in studies of developmental language disorder during childhood were given the Autism Diagnostic Observation Schedule - Generic. Their parents completed the Autism Diagnostic Interview - Revised, which relies largely on symptoms present at age 4 to 5 years to diagnose autism. Eight individuals met criteria for autism on both instruments, and a further four met criteria for milder forms of autistic spectrum disorder. Most individuals with autism had been identified with pragmatic impairments in childhood. Some children who would nowadays be diagnosed unambiguously with autistic disorder had been diagnosed with developmental language disorder in the past. This finding has implications for our understanding of the epidemiology of autism.
C1 [Bishop, Dorothy V. M.; Whitehouse, Andrew J. O.; Watt, Helen J.; Line, Elizabeth A.] Univ Oxford, Dept Expt Psychol, Oxford OX1 3UD, England.
RP Bishop, DVM (reprint author), Univ Oxford, Dept Expt Psychol, S Parks Rd, Oxford OX1 3UD, England.
EM dorothy.bishop@psy.ox.ac.uk
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NR 29
TC 53
Z9 55
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD MAY
PY 2008
VL 50
IS 5
BP 341
EP 345
DI 10.1111/j.1469-8749.2008.02057.x
PG 5
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 288MC
UT WOS:000254989500007
PM 18384386
ER
PT J
AU Sabbagh, MA
Seamans, EL
AF Sabbagh, Mark A.
Seamans, Elizabeth L.
TI Intergenerational transmission of theory-of-mind
SO DEVELOPMENTAL SCIENCE
LA English
DT Article
ID INDIVIDUAL-DIFFERENCES; ASPERGER-SYNDROME; AUTISM; PHENOTYPE; CHILDREN
AB We examined whether individual differences in children's performance on a scaled battery of theory-of-mind tasks was predicted by parents' performance on an adult theory-of-mind task. Forty-six 3-year-old children and their parents participated in this study when children were aged 2;11 to 4;0. Thirty dyads returned 6 months later for a second assessment. After statistically controlling for relevant covariates, we detected a positive correlation between parents' and children's theory-of-mind scores. The correlation was significant at both time points, and was robust when data were aggregated across the two sessions. These results provide some evidence for intergenerational transmission of theory-of-mind abilities in a typically developing sample, and possible mechanisms underlying this relation are discussed.
C1 [Sabbagh, Mark A.] Queens Univ, Dept Psychol, Kingston, ON K7L 3N6, Canada.
[Seamans, Elizabeth L.] Univ New Brunswick, Dept Psychol, Fredericton, NB E3B 5A3, Canada.
RP Sabbagh, MA (reprint author), Queens Univ, Dept Psychol, Kingston, ON K7L 3N6, Canada.
EM sabbagh@post.queensu.ca
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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Wellman HM, 2004, DEVELOPMENTAL SCI, V7, P283, DOI 10.1111/j.1467-7687.2004.00347.x
NR 27
TC 11
Z9 12
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1363-755X
J9 DEVELOPMENTAL SCI
JI Dev. Sci.
PD MAY
PY 2008
VL 11
IS 3
BP 354
EP 360
DI 10.1111/j.1467-7687.2008.00680.x
PG 7
WC Psychology, Developmental; Psychology, Experimental
SC Psychology
GA 297UY
UT WOS:000255644100004
PM 18466369
ER
PT J
AU Bolte, S
Poustka, F
Holtmann, M
AF Boelte, Sven
Poustka, Fritz
Holtmann, Martin
TI Trends in autism spectrum disorder referrals
SO EPIDEMIOLOGY
LA English
DT Letter
C1 [Boelte, Sven; Poustka, Fritz; Holtmann, Martin] Univ Frankfurt, Dept Child & Adolescent Psychiat, Frankfurt, Germany.
RP Bolte, S (reprint author), Univ Frankfurt, Dept Child & Adolescent Psychiat, Frankfurt, Germany.
EM Boelte@em.uni-frankfurt-de
CR Baird G, 2006, LANCET, V368, P210, DOI 10.1016/S0140-6736(06)69041-7
Böhm K, 2004, Bundesgesundheitsblatt Gesundheitsforschung Gesundheitsschutz, V47, P457, DOI 10.1007/s00103-004-0832-5
*CAL DEP DEV SERV, 2002, AUT SPECTR DIS CHANG
Fombonne E, 1999, PSYCHOL MED, V29, P769, DOI 10.1017/S0033291799008508
World Health Organisation, 1992, ICD 10 CLASS MENT BE
NR 5
TC 2
Z9 2
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 1044-3983
J9 EPIDEMIOLOGY
JI Epidemiology
PD MAY
PY 2008
VL 19
IS 3
BP 519
EP 520
PG 2
WC Public, Environmental & Occupational Health
SC Public, Environmental & Occupational Health
GA 293DF
UT WOS:000255314400030
PM 18414094
ER
PT J
AU Lawson-Yuen, A
Saldivar, JS
Sommer, S
Picker, J
AF Lawson-Yuen, Amy
Saldivar, Juan-Sebastian
Sommer, Steve
Picker, Jonathan
TI Familial deletion within NLGN4 associated with autism and Tourette
syndrome
SO EUROPEAN JOURNAL OF HUMAN GENETICS
LA English
DT Article
DE neuroligin 4; autism; Tourette syndrome; tic disorder
ID NEUROLIGIN GENES; BETA-NEUREXINS; MUTATIONS; SYNAPSE; ISOFORMS; BINDING;
PSD-95
AB Neuroligin 4 ( NLGN4) is a member of a cell adhesion protein family that appears to play a role in the maturation and function of neuronal synapses. Mutations in the X- linked NLGN4 gene are a potential cause of autistic spectrum disorders, and mutations have been reported in several patients with autism, Asperger syndrome, and mental retardation. We describe here a family with a wide variation in neuropsychiatric illness associated with a deletion of exons 4, 5, and 6 of NLGN4. The proband is an autistic boy with a motor tic. His brother has Tourette syndrome and attention deficit hyperactivity disorder. Their mother, a carrier, has a learning disorder, anxiety, and depression. This family demonstrates that NLGN4 mutations can be associated with a wide spectrum of neuropsychiatric conditions and that carriers may be affected with milder symptoms.
C1 [Lawson-Yuen, Amy; Picker, Jonathan] Childrens Hosp, Dept Genet, Boston, MA 02115 USA.
[Saldivar, Juan-Sebastian; Sommer, Steve] City Hope Natl Med Ctr, Dept Mol Genet, Clin Miol Diagnost Lab, Duarte, CA 91010 USA.
RP Picker, J (reprint author), Childrens Hosp, Dept Genet, Fegan 10,300 Longwood Ave, Boston, MA 02115 USA.
EM jonathan.picker@childrens.harvard.edu
CR Blasi F, 2006, AM J MED GENET B, V141B, P220, DOI 10.1002/ajmg.b.30287
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NR 18
TC 125
Z9 131
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1018-4813
J9 EUR J HUM GENET
JI Eur. J. Hum. Genet.
PD MAY
PY 2008
VL 16
IS 5
BP 614
EP 618
DI 10.1038/sj.ejhg.5202006
PG 5
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 292IU
UT WOS:000255260500012
PM 18231125
ER
PT J
AU Thompson, RR
Walton, JC
Bhalla, R
George, KC
Beth, EH
AF Thompson, R. R.
Walton, J. C.
Bhalla, R.
George, K. C.
Beth, E. H.
TI A primitive social circuit: vasotocin-substance P interactions modulate
social behavior through a peripheral feedback mechanism in goldfish
SO EUROPEAN JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE autism; autonomic; emotion; hindbrain; polyvagal; vasopressin
ID ANXIETY-RELATED BEHAVIOR; VASOPRESSIN RECEPTOR; CARASSIUS-AURATUS;
SPINAL-CORD; NEURONS; RESPONSES; AUTISM; BRAIN; RATS; NEUROPEPTIDES
AB At its core, the polyvagal theory proposes that peptides affect simple social behaviors through influences on hindbrain autonomic processes. To test this mechanism, we compared the effects of fore- and hindbrain infusions of vasotocin (VT) on social approach behavior in goldfish. VT infusions into the 4th ventricle, which ink infusions verified did not move rostrally to the forebrain, inhibited social approach at a lower dose than did infusions into the 3rd ventricle, which did diffuse to the hindbrain. Thus, VT actions in the hindbrain appear to modulate this simple social behavior. We then identified a population of substance P (SP)-immunoreactive cells in the hindbrain that are encapsulated by putative VT terminals, and determined that those cells project to the periphery. Injecting SP peripherally, as with infusing VT centrally, inhibited social approach, and peripheral injections of an SP antagonist, but not central infusions, abolished the behavioral effects of central VT infusions. We therefore propose that VT inhibits social approach by activating SP cells in the hindbrain, which then induce changes in body state that feed back to the brain. Central VT infusions did not inhibit feeding, suggesting that this VT mechanism selectively affects appetitive social responses. Because VT projections to the hindbrain are highly conserved in vertebrates, influences on peripheral feedback processes like the one we have described in goldfish may reflect how VT affected simple social behaviors in ancestral vertebrates and thus preadapted members of this peptide family to play increasingly complex roles in social and emotional regulation in modern animals.
C1 [Thompson, R. R.] Bowdoin Coll, Dept Psychol, Brunswick, ME 04011 USA.
Bowdoin Coll, Neurosci Program, Brunswick, ME 04011 USA.
RP Thompson, RR (reprint author), Bowdoin Coll, Dept Psychol, Brunswick, ME 04011 USA.
EM rthompso@bowdoin.edu
RI Walton, James/F-7435-2010
OI Walton, James/0000-0002-3049-1029
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NR 48
TC 20
Z9 21
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0953-816X
J9 EUR J NEUROSCI
JI Eur. J. Neurosci.
PD MAY
PY 2008
VL 27
IS 9
BP 2285
EP 2293
DI 10.1111/j.1460-9568.2008.06210.x
PG 9
WC Neurosciences
SC Neurosciences & Neurology
GA 292SG
UT WOS:000255285800008
PM 18445219
ER
PT J
AU Stack, CM
Lim, MA
Cuasay, K
Stone, MA
Seibert, KM
Spivak-Pohis, I
Crawley, JN
Waschek, JA
Hill, JM
AF Stack, Conor M.
Lim, Maria A.
Cuasay, Katrina
Stone, Madeleine A.
Seibert, Kimberly M.
Spivak-Pohis, Irit
Crawley, Jacqueline N.
Waschek, James A.
Hill, Joanna M.
TI Deficits in social behavior and reversal learning are more prevalent in
male offspring of VIP deficient female mice
SO EXPERIMENTAL NEUROLOGY
LA English
DT Article
DE autism; Morris water maze; mouse model; neurodevelopmental disorder;
neuropeptide; sex differences; sociability; social approach; spatial
reversal learning; vasoactive intestinal peptide
ID VASOACTIVE-INTESTINAL-PEPTIDE; DEPENDENT NEUROTROPHIC FACTOR; EMBRYONIC
GROWTH; DORSOMEDIAL STRIATUM; NEUROPROTECTIVE PEPTIDE; KNOCKOUT MICE;
MOUSE; RECEPTOR; RAT; AUTISM
AB Blockage of vasoactive intestinal peptide (VIP) receptors during early embryogenesis in the mouse has been shown to result in developmental delays in neonates, and social behavior deficits selectively in adult male offspring. Offspring of VIP deficient mothers (VIP +/-) also exhibited developmental delays, and reductions in maternal affiliation and play behavior. In the current study, comparisons among the offspring of VIP deficient mothers (VIP +/-) mated to VIP +/- males with the offspring of wild type (WT) mothers mated to VIP +/- males allowed assessment of the contributions of both maternal and offspring VIP genotype to general health measures, social behavior, fear conditioning, and spatial learning and memory in the water maze. These comparisons revealed few differences in general health among offspring of WT and VIP deficient mothers, and all offspring exhibited normal responses in fear conditioning and in the acquisition phase of spatial discrimination in the water maze. WT mothers produced offspring that were normal in all tests; the reduced VIP in their VIP +/- offspring apparently did not contribute to any defects in the measures under study. However, regardless of their own VIP genotype, all male offspring of VIP deficient mothers exhibited severe deficits in social approach behavior and reversal learning. The deficits in these behaviors in the female offspring of VIP deficient mothers were less severe than in their male littermates, and the extent of their impairment was related to their own VIP genotype. This study has shown that intrauterine conditions had a greater influence on behavioral outcome than did genetic inheritance. In addition, the greater prevalence of deficits in social behavior and the resistance to change seen in reversal learning in the male offspring of VIP deficient mothers indicate a potential usefulness of the VIP knockout mouse in furthering the understanding of neurodevelopmental disorders such as autism. Published by Elsevier Inc.
C1 [Stack, Conor M.; Lim, Maria A.; Cuasay, Katrina; Stone, Madeleine A.; Seibert, Kimberly M.; Crawley, Jacqueline N.; Hill, Joanna M.] NIH, NIMH, Lab Behav Neurosci, Bethesda, MD 20892 USA.
[Spivak-Pohis, Irit] Tel Aviv Univ, Sackler Sch Med, Dept Clin Biochem, IL-69978 Tel Aviv, Israel.
[Waschek, James A.] Univ Calif Los Angeles, Dept Psychiat & Behav Sci, Los Angeles, CA USA.
RP Hill, JM (reprint author), NIH, NIMH, Lab Behav Neurosci, 35-1C903, Bethesda, MD 20892 USA.
EM hiilljoa@mail.nih.gov
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NR 67
TC 19
Z9 20
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0014-4886
J9 EXP NEUROL
JI Exp. Neurol.
PD MAY
PY 2008
VL 211
IS 1
BP 67
EP 84
DI 10.1016/j.expneurol.2008.01.003
PG 18
WC Neurosciences
SC Neurosciences & Neurology
GA 298ZM
UT WOS:000255725600010
PM 18316078
ER
PT J
AU Davis, LK
Meyer, KJ
Rudd, DS
Librant, AL
Epping, EA
Sheffield, VC
Wassink, TH
AF Davis, L. K.
Meyer, K. J.
Rudd, D. S.
Librant, A. L.
Epping, E. A.
Sheffield, V. C.
Wassink, T. H.
TI Pax6 3 ' deletion results in aniridia, autism and mental retardation
SO HUMAN GENETICS
LA English
DT Article
ID MISSENSE MUTATIONS; CONGENITAL ANIRIDIA; HUMAN GENOME; GENE;
POLYMORPHISM; EXPRESSION; REGION; EYE; MALFORMATIONS; FAMILY
AB The PAX6 gene is a transcription factor expressed early in development, predominantly in the eye, brain and gut. It is well known that mutations in PAX6 may result in aniridia, Peter's anomaly and kertatisis. Here, we present mutation analysis of a patient with aniridia, autism and mental retardation. We identified and characterized a 1.3 Mb deletion that disrupts PAX6 transcriptional activity and deletes additional genes expressed in the brain. Our findings provide continued evidence for the role of PAX6 in neural phenotypes associated with aniridia.
C1 [Davis, L. K.; Meyer, K. J.; Rudd, D. S.; Librant, A. L.; Epping, E. A.; Wassink, T. H.] Univ Iowa, Carver Coll Med, Dept Psychiat, Iowa City, IA 52242 USA.
[Sheffield, V. C.] Univ Iowa, Carver Coll Med, Dept Pediat, Iowa City, IA USA.
[Sheffield, V. C.] Univ Iowa, Howard Hughes Med Inst, Iowa City, IA 52242 USA.
[Davis, L. K.; Meyer, K. J.; Sheffield, V. C.; Wassink, T. H.] Univ Iowa, Interdisciplinary Program Genet, Iowa City, IA USA.
RP Davis, LK (reprint author), Univ Iowa, Carver Coll Med, Dept Psychiat, Iowa City, IA 52242 USA.
EM lea.k.davis@gmail.com
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NR 44
TC 33
Z9 35
PU SPRINGER
PI NEW YORK
PA 233 SPRING STREET, NEW YORK, NY 10013 USA
SN 0340-6717
J9 HUM GENET
JI Hum. Genet.
PD MAY
PY 2008
VL 123
IS 4
BP 371
EP 378
DI 10.1007/s00439-008-0484-x
PG 8
WC Genetics & Heredity
SC Genetics & Heredity
GA 288AW
UT WOS:000254959600005
PM 18322702
ER
PT J
AU Mehrotra, N
Vaidya, S
AF Mehrotra, Nilika
Vaidya, Shubhangi
TI Exploring Constructs of Intellectual Disability and. Personhood in
Haryana and Delhi
SO INDIAN JOURNAL OF GENDER STUDIES
LA English
DT Article
AB Intellectual disability is one of the least researched areas in social science inquiry. This paper traces the complex interplay between the concepts of intellectual disability, gender and personhood. It outlines the socio-historical and cross-cultural variability of intellectual disability, and its connections with class, urbanisation and modernisation. Based on ethnographic material on the disabled in Delhi and the neighbouring state of Haryana, it presents case studies of two NGOs working with the intellectually disabled, namely, Arpan, a school for the mentally retarded in Rohtak, Haryana, and Action for Autism in New Delhi. It engages specifically with the notion of masculinity and the manner in which intellectually disabled male adults are feminised and in-fantilised. An attempt is made to understand how disabled individuals and their families seek social spaces for themselves and negotiate the social compulsions for 'normalcy' and competent adulthood.
C1 [Mehrotra, Nilika] Jawaharlal Nehru Univ, Ctr Study Social Syst, New Delhi 110067, India.
[Vaidya, Shubhangi] Indira Gandhi Natl Open Univ, Reg Serv Div, New Delhi, India.
RP Mehrotra, N (reprint author), Jawaharlal Nehru Univ, Ctr Study Social Syst, New Delhi 110067, India.
EM nilika2l@yahoo.co.in; shubhangi_v2000@yahoo.co.in
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NR 46
TC 2
Z9 2
PU SAGE PUBLICATIONS INDIA PVT LTD
PI NEW DELHI
PA POST BOX 4125 M-32 MARKET, GREATER KAILASH-I, NEW DELHI 110 048, INDIA
SN 0971-5215
J9 INDIAN J GEND STUD
JI Indian J. Gend. Stud.
PD MAY-AUG
PY 2008
VL 15
IS 2
SI SI
BP 317
EP 340
DI 10.1177/097152150801500206
PG 24
WC Women's Studies
SC Women's Studies
GA 360PJ
UT WOS:000260069400006
ER
PT J
AU Botting, N
AF Botting, Nicola
TI Children without language: From dysphasia to autism
SO INFANT AND CHILD DEVELOPMENT
LA English
DT Book Review
C1 [Botting, Nicola] City Univ London, London EC1V 0HB, England.
RP Botting, N (reprint author), City Univ London, London EC1V 0HB, England.
CR DANONBOILEAU L, 2006, CHILDREN WITHOUT LAN
NR 1
TC 0
Z9 0
PU JOHN WILEY & SONS LTD
PI CHICHESTER
PA THE ATRIUM, SOUTHERN GATE, CHICHESTER PO19 8SQ, W SUSSEX, ENGLAND
SN 1522-7227
J9 INFANT CHILD DEV
JI Infant Child Dev.
PD MAY-JUN
PY 2008
VL 17
IS 3
BP 306
EP 307
DI 10.1002/icd.556
PG 2
WC Psychology, Developmental
SC Psychology
GA 314JQ
UT WOS:000256806100009
ER
PT J
AU Lopez, CA
Tchanturia, K
Stahl, D
Treasure, J
AF Lopez, Carolina A.
Tchanturia, Kate
Stahl, Daniel
Treasure, Janet
TI Central coherence in women with bulimia nervosa
SO INTERNATIONAL JOURNAL OF EATING DISORDERS
LA English
DT Article
DE bulimia; central coherence; information processing; neuropsychology;
endophenotype; eating disorders; cognition; anorexia
ID ANOREXIA-NERVOSA; EATING-DISORDERS; COGNITIVE-STYLE; SPECTRUM DISORDERS;
AUTISM; PREDICTORS; PHENOTYPE; SEVERITY; MEMORY; ADULTS
AB Objective: To examine the concept of central coherence in women with bulimia nervosa (BN) and address similarities and difference with those with anorexia nervosa (AN)
Method: Forty two women with BN and 42 matched healthy women, completed neuropsychological testing measuring aspects of central coherence: Rey-Osterrieth Complex Figure (RCFT), Embedded Figures Test (EFT), Block Design Test (BD), Homograph Reading Test (HRT), and Sentence Completion Task (SCT).
Results: The BN group showed superiority in local processing as measured by EFT and lesser relative advantage from segmentation in BID, and difficulties in global processing in both visual and verbal domains as examined by RCFT, HRT, and SCT. Anxiety levels were associated with low central coherence indices in RCFT.
Conclusion: People with BN displayed a profile consistent with the weak central coherence hypothesis. Their pattern of cognitive performance resembles that seen in AN although some differences are apparent. (C) 2008 by Wiley Periodicals, Inc.
C1 [Lopez, Carolina A.; Tchanturia, Kate] Kings Coll London, Dept Psychol Med & Psychiat, Inst Psychiat, London WC2R 2LS, England.
[Stahl, Daniel] Kings Coll London, Dept Biostat & Comp, London WC2R 2LS, England.
RP Lopez, CA (reprint author), Guys Hosp, Dept Acad Psychiat, 5th Floor, London SE1 9RT, England.
EM c.lopez@iop.kcl.ac.uk
RI Stahl, Daniel/B-9713-2011; Tchanturia, Kate/H-1474-2011
OI Stahl, Daniel/0000-0001-7987-6619;
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NR 54
TC 32
Z9 32
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 0276-3478
J9 INT J EAT DISORDER
JI Int. J. Eating Disord.
PD MAY
PY 2008
VL 41
IS 4
BP 340
EP 347
DI 10.1002/eat.20511
PG 8
WC Psychology, Clinical; Nutrition & Dietetics; Psychiatry; Psychology
SC Psychology; Nutrition & Dietetics; Psychiatry
GA 290UE
UT WOS:000255147100007
PM 18306347
ER
PT J
AU Huotilainen, M
Shestakova, A
Hukki, J
AF Huotilainen, Minna
Shestakova, Anna
Hukki, Jyri
TI Using magnetoencephalography in assessing auditory skills in infants and
children
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Article
DE magnetoencephalography; language; infant; child; auditory
ID EVENT-RELATED POTENTIALS; EVOKED MAGNETIC-FIELDS; MISMATCH NEGATIVITY;
PHONEME REPRESENTATIONS; DEVELOPMENTAL-CHANGES; SPEECH-PERCEPTION; BRAIN
RESPONSES; AUTISM DISORDER; MEG; CORTEX
AB In this paper, the use of magnetoencephalography (MEG) in studying the basic auditory skills in infants and children is reviewed. The auditory skills are related to perceiving sound onsets and offsets of sounds, extracting rules and regularities in sound environments, perceiving differences and changes in sounds, categorizing sound elements, allocating attention towards certain sounds or sound streams, and attaching semantic information into sounds. Studying each of these auditory skills with MEG in particular stimulation paradigms is shortly reviewed, including two examples of data sets in children. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Huotilainen, Minna; Shestakova, Anna] Univ Helsinki, Dept Psychol, Cognit Brain Res Unit, FIN-00014 Helsinki, Finland.
[Huotilainen, Minna] Univ Helsinki, Helsinki Brain Res Ctr, FIN-00014 Helsinki, Finland.
[Shestakova, Anna] UCL, Dept Human Commun Sci, Ctr Dev Disorders & Cognit Neurosci, London, England.
[Hukki, Jyri] Helsinki Univ Hosp, Dept Plast Surg, Cleft Lip & Palate & Craniofacial Ctr, Helsinki, Finland.
RP Huotilainen, M (reprint author), Univ Helsinki, Dept Psychol, Cognit Brain Res Unit, POB 9, FIN-00014 Helsinki, Finland.
EM minna.huotilainen@helsinki.fi
RI Shestakova, Anna/H-3329-2013
OI Shestakova, Anna/0000-0001-8323-7270
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NR 70
TC 8
Z9 8
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD MAY
PY 2008
VL 68
IS 2
BP 123
EP 129
DI 10.1016/j.ijpsycho.2007.12.007
PG 7
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 305VM
UT WOS:000256205900006
PM 18394734
ER
PT J
AU Roberts, TPL
Schmidt, GL
Egeth, M
Blaskey, L
Rey, MM
Edgar, JC
Levy, SE
AF Roberts, Timothy P. L.
Schmidt, Gwen L.
Egeth, Marc
Blaskey, Lisa
Rey, Michael M.
Edgar, J. Christopher
Levy, Susan E.
TI Electrophysiological signatures: Magnetoencephalographic studies of the
neural correlates of language impairment in autism spectrum disorders
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Article
DE autism spectrum disorder; magnetoencephalography; language impairment;
auditory
ID AUDITORY MAGNETIC-FIELDS; MISMATCH NEGATIVITY MMN; NON-SPEECH SOUNDS;
HEMISPHERIC-ASYMMETRY; PLANUM TEMPORALE; CEREBRAL LATERALIZATION;
PERCEPTION DEFICITS; ASSOCIATION CORTEX; EPILEPSY SURGERY; BRAIN
RESPONSES
AB While magnetoencephalography (MEG) is of increasing utility in the assessment of pediatric patients with seizure disorders, this reflects only a part of the clinical potential of the technology. Beyond epilepsy, a broad range of developmental psychiatric disorders require the spatial and temporal resolution of brain activity offered by MEG. This article reviews the application of MEG in the study of auditory processing as an aspect of language impairment in children. Specifically, the potential application of MEG is elaborated in autism spectrum disorders (ASD), a devastating disorder with prevalence of I in 150. Results demonstrate the sensitivity of MEG for detection of abnormalities of auditory processing in ASD ('electrophysiological signatures') and their clinical correlates. These findings offer promise for the comprehensive assessment of developmental neuropsychiatric disorders. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Roberts, Timothy P. L.; Schmidt, Gwen L.; Egeth, Marc; Blaskey, Lisa; Rey, Michael M.; Edgar, J. Christopher] Childrens Hosp Philadelphia, Dept Radiol, Philadelphia, PA 19104 USA.
[Blaskey, Lisa; Levy, Susan E.] Childrens Hosp Philadelphia, Dept Pediat, Philadelphia, PA 19104 USA.
RP Roberts, TPL (reprint author), Childrens Hosp Philadelphia, Dept Radiol, Wood Bldg,Suite 2115,34th St & Civic Ctr Blvd, Philadelphia, PA 19104 USA.
EM robertstim@email.chop.edu
RI Egeth, Marc/D-1289-2010
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NR 82
TC 23
Z9 24
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD MAY
PY 2008
VL 68
IS 2
BP 149
EP 160
DI 10.1016/j.ijpsycho.2008.01.012
PG 12
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 305VM
UT WOS:000256205900009
PM 18336941
ER
PT J
AU Cardy, JEO
Flagg, EJ
Roberts, W
Roberts, TPL
AF Cardy, Janis E. Oram
Flagg, Elissa J.
Roberts, Wendy
Roberts, Timothy P. L.
TI Auditory evoked fields predict language ability and impairment in
children
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Article
DE autism; specific language impairment; magnetoencephalography; auditory
evoked fields
ID AUTISTIC SPECTRUM; MAGNETIC-FIELDS; DISORDER; PHENOTYPES; RESPONSES;
STIMULI; SPEECH
AB Recent evidence suggests that a subgroup of children with autism show similarities to children with Specific Language Impairment (SLI) in the pattern of their linguistic impairments, but the source of this overlap is unclear. We examined the ability of auditory evoked magnetic fields to predict language and other developmental abilities in children and adolescents. Following standardized assessment of language ability, nonverbal IQ, and autism-associated behaviors, I 10 trails of a tone were binaurally presented to 45 7-18 year olds who had typical development, autism (with LI), Asperger Syndrome (i.e., without LI), or SLI. Using a 15 1 -channel MEG system, latency of left hemisphere (LH) and right hemisphere (RH) auditory M50 and M100 peaks was recorded. RH M50 latency (and to a lesser extent, RH M100 latency) predicted overall oral language ability, accounting for 36% of the variance. Nonverbal IQ and autism behavior ratings were not predicted by any of the evoked fields. Latency of the RH M50 was the best predictor of clinical LI (i.e., irrespective of autism diagnosis), and demonstrated 82% accuracy in predicting Receptive LI; a cutoff of 84.6 ms achieved 92% specificity and 70% sensitivity in classifying children with and without Receptive LI. Auditory evoked responses appear to reflect language functioning and impairment rather than non-specific brain (dys)function (e.g., IQ, behavior). RH M50 latency proved to be a relatively useful indicator of impaired language comprehension, suggesting that delayed auditory perceptual processing in the RH may be a key neural dysfunction underlying the overlap between subgroups of children with autism and SLI. (C) 2008 Elsevier B.V All rights reserved.
C1 [Cardy, Janis E. Oram] Univ Western Ontario, Elbom Coll, Sch Commun Sci & Disorders, London, ON N6G 1H1, Canada.
[Flagg, Elissa J.] York Univ, Dept Languages Literatures & Linguist, Toronto, ON M3J 2R7, Canada.
[Roberts, Wendy] Univ Toronto, Hosp Sick Children, Autism Res Unit, Toronto, ON M5G 1X8, Canada.
[Roberts, Wendy] Univ Toronto, Dept Paediat, Toronto, ON M5S 1A1, Canada.
[Roberts, Timothy P. L.] Childrens Hosp Philadelphia, Dept Radiol, Philadelphia, PA 19104 USA.
[Roberts, Timothy P. L.] Univ Penn, Sch Med, Philadelphia, PA 19104 USA.
RP Cardy, JEO (reprint author), Univ Western Ontario, Elbom Coll, Sch Commun Sci & Disorders, London, ON N6G 1H1, Canada.
EM joramcar@uwo.ca
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NR 40
TC 31
Z9 32
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD MAY
PY 2008
VL 68
IS 2
BP 170
EP 175
DI 10.1016/j.ijpsycho.2007.10.015
PG 6
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 305VM
UT WOS:000256205900011
ER
PT J
AU Mazefsky, CA
Williams, DL
Minshew, NJ
AF Mazefsky, Carla A.
Williams, Diane L.
Minshew, Nancy J.
TI Variability in adaptive behavior in autism: Evidence for the importance
of family history
SO JOURNAL OF ABNORMAL CHILD PSYCHOLOGY
LA English
DT Article
DE autism; adaptive behavior; family history; Vineland adaptive behavior
scales; broader autism phenotype
ID PERVASIVE DEVELOPMENTAL DISORDERS; PSYCHIATRIC-DISORDERS; SPECTRUM
DISORDERS; MENTAL-RETARDATION; BROADER PHENOTYPE; CHILDREN; INDIVIDUALS;
PARENTS; VINELAND; EXPRESSION
AB Adaptive behavior in autism is highly variable and strongly related to prognosis. This study explored family history as a potential source of variability in adaptive behavior in autism. Participants included 77 individuals (mean age = 18) with average or better intellectual ability and autism. Parents completed the Family History Interview about the presence of broader autism phenotype symptoms and major psychiatric disorders in first degree relatives. Adaptive behavior was assessed via the Vineland Adaptive Behavior Scales (VABS). Based on family history variables, age, and intelligence quotient (IQ), 87% of participants were correctly classified as having impaired or average VABS scores. Family history of depression and shyness accounted for the most variance in VABS scores, and they had the greatest influence on VABS Socialization scores in particular. Possible underlying mechanisms include genetics, psychosocial factors, and social resources. This study provides initial evidence of the importance of family history to adaptive behavior in autism and has implications for genetics and treatment.
C1 [Mazefsky, Carla A.] Univ Pittsburgh, Dept Pediat & Psychiat, Pittsburgh, PA USA.
[Williams, Diane L.] Duquesne Univ, Dept Speech & Language Pathol, Pittsburgh, PA 15219 USA.
[Minshew, Nancy J.] Univ Pittsburgh, Dept Psychiat & Neurol, Pittsburgh, PA USA.
RP Mazefsky, CA (reprint author), Childrens Hosp Pittsburgh, Child Dev Unit, 3705 5th Ave, Pittsburgh, PA 15213 USA.
EM Carla.Mazefsky@chp.edu
CR Aldenderfer MS, 1984, CLUSTER ANAL
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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Starr E, 2001, J AUTISM DEV DISORD, V31, P89, DOI 10.1023/A:1005669915105
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Tabachnick B. G., 2006, USING MULTIVARIATE S, V5th
NR 44
TC 13
Z9 13
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0091-0627
J9 J ABNORM CHILD PSYCH
JI J. Abnorm. Child Psychol.
PD MAY
PY 2008
VL 36
IS 4
BP 591
EP 599
DI 10.1007/s10802-007-9202-8
PG 9
WC Psychology, Clinical; Psychology, Developmental
SC Psychology
GA 283GI
UT WOS:000254624300010
PM 18188537
ER
PT J
AU Porter, MA
Coltheart, M
Langdon, R
AF Porter, Melanie A.
Coltheart, Max
Langdon, Robyn
TI Theory of mind in Williams syndrome assessed using a nonverbal task
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE Williams syndrome; Theory of Mind (ToM); nonverbal; heterogeneity
ID CHILDREN; AUTISM; ADULTS; HYPERSOCIABILITY; LANGUAGE
AB This study examined Theory of Mind in Williams syndrome (WS) and in normal chronological age-matched and mental age-matched control groups, using a picture sequencing task. This task assesses understanding of pretence, intention and false belief, while controlling for social-script knowledge and physical cause-and-effect reasoning. The task was selected because it is entirely non-verbal, so that the WS individuals could not rely on their good verbal skills when performing the task. Results indicated a specific deficit in understanding of false belief within the WS group. There was also evidence of heterogeneity in the WS group, with the false belief impairment restricted to only a particular subgroup of WS individuals identified originally by Porter, M., & Coltheart, M. (2005). Cognitive heterogeneity in Williams syndrome.
C1 [Porter, Melanie A.; Coltheart, Max; Langdon, Robyn] Macquarie Univ, Macquarie Ctr Cognit Sci, Sydney, NSW 2109, Australia.
RP Porter, MA (reprint author), Macquarie Univ, Macquarie Ctr Cognit Sci, Sydney, NSW 2109, Australia.
EM mporter@maccs.mq.edu.au
CR Achenbach TM, 2001, MANUAL ASEBA SCH AGE
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Perner Josef, 1991, UNDERSTANDING REPRES
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Porter MA, 2005, DEV NEUROPSYCHOL, V27, P275, DOI 10.1207/s15326942dn2702_5
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NR 30
TC 22
Z9 22
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 806
EP 814
DI 10.1007/s10803-007-0447-4
PG 9
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700002
PM 17874179
ER
PT J
AU Harper, CB
Symon, JBG
Frea, WD
AF Harper, Christena Blauvelt
Symon, Jennifer B. G.
Frea, William D.
TI Recess is time-in: Using peers to improve social skills of children with
autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; peer-mediated strategies; inclusion; social skills; school
intervention; peer interactions; Pivotal Response Training; initiations
ID YOUNG-CHILDREN; PLAY SKILLS; INTERVENTION; BEHAVIOR; DISABILITIES;
IMITATION; STUDENTS
AB Children with autism face enormous struggles when attempting to interact with their typically developing peers. More children are educated in integrated settings; however, play skills usually need to be explicitly taught, and play environments must be carefully prepared to support effective social interactions. This study incorporated the motivational techniques of Pivotal Response Training through peer-mediated practice to improve social interactions for children with autism during recess activities. A multiple baseline design across subjects was used to assess social skills gains in two elementary school children. The results demonstrated an increase in important social skills, namely social initiations and turn taking, during recess.
C1 [Symon, Jennifer B. G.] Calif State Univ Los Angeles, Div Special Educ & Counselling, Los Angeles, CA 90032 USA.
[Harper, Christena Blauvelt] Calif State Univ Los Angeles, Garden Groove Unified Sch Dist Orange Cty, Los Angeles, CA 90032 USA.
[Frea, William D.] Austin Spectrum Therapies, Los Angeles, CA USA.
RP Symon, JBG (reprint author), Calif State Univ Los Angeles, Div Special Educ & Counselling, 5151 State Univ Dr, Los Angeles, CA 90032 USA.
EM jsymon@calstatela.edu
CR Attwood T., 1998, ASPERGERS SYNDROME G
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NR 44
TC 46
Z9 46
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 815
EP 826
DI 10.1007/s10803-007-0449-2
PG 12
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700003
PM 17874290
ER
PT J
AU Kleinman, JM
Robins, DL
Ventola, PE
Pandey, J
Boorstein, HC
Esser, EL
Wilson, LB
Rosenthal, MA
Sutera, S
Verbalis, AD
Barton, M
Hodgson, S
Green, J
Dumont-Mathieu, T
Volkmar, F
Chawarska, K
Klin, A
Fein, D
AF Kleinman, Jamie M.
Robins, Diana L.
Ventola, Pamela E.
Pandey, Juhi
Boorstein, Hilary C.
Esser, Emma L.
Wilson, Leandra B.
Rosenthal, Michael A.
Sutera, Saasha
Verbalis, Alyssa D.
Barton, Marianne
Hodgson, Sarah
Green, James
Dumont-Mathieu, Thyde
Volkmar, Fred
Chawarska, Katarzyna
Klin, Ami
Fein, Deborah
TI The Modified Checklist for Autism in Toddlers: A follow-up study
investigating the early detection of autism spectrum disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; early identification; pediatric screening
ID PERVASIVE DEVELOPMENTAL DISORDERS; TRAITS QUESTIONNAIRE ESAT;
YOUNG-CHILDREN; DIAGNOSTIC INSTRUMENTS; BEHAVIORAL-PROBLEMS;
PRESCHOOL-CHILDREN; AGE; POPULATION; PREVALENCE; AGREEMENT
AB Autism spectrum disorders (ASD) often go undetected in toddlers. The Modified Checklist for Autism in Toddlers (M-CHAT) was used to screen 3,793 children aged 16-30 months from low- and high-risk sources; screen positive cases were diagnostically evaluated. Re-screening was performed on 1,416 children aged 42-54 months. Time1 Positive Predictive Value (PPV) was .36 for the initial screening and .74 for the screening plus follow-up telephone interview; values were similar for Time2 PPV. When separating referral sources, PPV was low for the low-risk sample but acceptable with the follow-up telephone interview. Children with ASD from the low-risk and high-risk samples were highly similar. Results indicate that the M-CHAT continues to be a promising instrument for the early detection of ASD.
C1 [Kleinman, Jamie M.; Pandey, Juhi; Boorstein, Hilary C.; Esser, Emma L.; Wilson, Leandra B.; Rosenthal, Michael A.; Sutera, Saasha; Verbalis, Alyssa D.; Barton, Marianne; Hodgson, Sarah; Green, James; Dumont-Mathieu, Thyde; Fein, Deborah] Univ Connecticut, Dept Psychol, Storrs, CT 06269 USA.
[Robins, Diana L.] Georgia State Univ, Atlanta, GA 30303 USA.
[Ventola, Pamela E.; Volkmar, Fred; Chawarska, Katarzyna; Klin, Ami] Yale Univ, Sch Med, New Haven, CT USA.
RP Kleinman, JM (reprint author), Univ Connecticut, Dept Psychol, 406 Babbidge Rd, Storrs, CT 06269 USA.
EM jamie_kleinman@yahoo.com
RI Robins, Diana/D-9959-2011
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 51
TC 104
Z9 105
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
EI 1573-3432
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 827
EP 839
DI 10.1007/s10803-007-0450-9
PG 13
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700004
PM 17882539
ER
PT J
AU Best, CS
Moffat, VJ
Power, MJ
Owens, DGC
Johnstone, EC
AF Best, Catherine S.
Moffat, Vivien J.
Power, Michael J.
Owens, David G. C.
Johnstone, Eve C.
TI The boundaries of the cognitive phenotype of autism: Theory of mind,
central coherence and ambiguous figure perception in young people with
autistic traits
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autistic spectrum; continuum; Theory of Mind; central coherence;
ambiguous figures
ID SPECTRUM DISORDERS; INDIVIDUALS; CHILDREN; PERFORMANCE; ILLUSIONS
AB Theory of Mind, Weak Central Coherence and executive dysfunction, were investigated as a function of behavioural markers of autism. This was irrespective of the presence or absence of a diagnosis of an autistic spectrum disorder. Sixty young people completed the Social Communication Questionnaire (SCQ), false belief tests, the block design test, viewed visual illusions and an ambiguous figure. A logistic regression was performed and it was found that Theory of Mind, central coherence and ambiguous figure variables significantly contributed to prediction of behavioural markers of autism. These findings provide support for the continuum hypothesis of autism. That is, mild autistic behavioural traits are distributed through the population and these behavioural traits may have the same underlying cognitive determinants as autistic disorder.
C1 [Best, Catherine S.; Moffat, Vivien J.; Power, Michael J.; Owens, David G. C.; Johnstone, Eve C.] Univ Edinburgh, Royal Edinburgh Hosp, Dept Psychiat, Edinburgh EH10 5HF, Midlothian, Scotland.
RP Best, CS (reprint author), Univ Edinburgh, Royal Edinburgh Hosp, Dept Psychiat, Kennedy Tower,Morningside Pk, Edinburgh EH10 5HF, Midlothian, Scotland.
EM s0344764@sms.ed.ac.uk
CR [Anonymous], 2000, DSMIVTR
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NR 28
TC 19
Z9 20
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 840
EP 847
DI 10.1007/s10803-007-0451-8
PG 8
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700005
PM 18004653
ER
PT J
AU Hediger, ML
England, LJ
Molloy, CA
Yu, KF
Manning-Courtney, P
Mills, JL
AF Hediger, Mary L.
England, Lucinda J.
Molloy, Cynthia A.
Yu, Kai F.
Manning-Courtney, Patricia
Mills, James L.
TI Reduced bone cortical thickness in boys with autism or autism spectrum
disorder
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; autism spectrum disorder; boys; bone growth; calcium intake;
dietary intake
ID RETT-SYNDROME; GASTROINTESTINAL SYMPTOMS; DIETARY INTERVENTION;
METACARPAL INDEX; HEALTHY-CHILDREN; MINERAL DENSITY; ABNORMALITIES;
ASSOCIATION; PREVALENCE; ADRENARCHE
AB Bone development, casein-free diet use, supplements, and medications were assessed for 75 boys with autism or autism spectrum disorder, ages 4-8 years. Second metacarpal bone cortical thickness (BCT), measured on hand-wrist radiographs, and % deviations in BCT from reference medians were derived. BCT increased with age, but % deviations evidenced a progressive fall-off (p = .02): +3.1 +/- 4.7%, -6.5 +/- 4.0%, -16.6 +/- 3.4%, -19.4 +/- 3.7%, -24.1 +/- 4.4%, at ages 4-8, respectively, adjusting for height. The 12% of the boys on casein-free diets had an overall % deviation of -18.9 +/- 3.7%, nearly twice that of boys on minimally restricted or unrestricted diets (-10.5 +/- 1.3%, p < .04), although even for boys on minimally restricted or unrestricted diets the % deviation was highly significant (p < .001). Our data suggest that the bone development of autistic boys should be monitored as part of routine care, especially if they are on casein-free diets.
C1 [Hediger, Mary L.; Yu, Kai F.; Mills, James L.] NICHHD, NIH, DESPR, Dept Hlth & Human Serv, Bethesda, MD 20892 USA.
[England, Lucinda J.] Natl Ctr Chron Dis Prevent & Hlth Promot, Ctr Dis Control & Prevent, Dept Hlth & Human Serv, Div Reprod Hlth, Atlanta, GA USA.
[Molloy, Cynthia A.] Univ Cincinnati, Childrens Hosp, Coll Med, Med Ctr,Ctr Epidemiol & Biostat, Cincinnati, OH USA.
[Manning-Courtney, Patricia] Cincinnati Childrens Hosp, Kelly O Leary Ctr Austin Spectrum Disorder, Div Dev Disablilities, Cincinnati, OH USA.
RP Hediger, ML (reprint author), NICHHD, NIH, DESPR, Dept Hlth & Human Serv, Blbg 6100,Rm 7B03,MSC 7510,9000 Rockville Pike, Bethesda, MD 20892 USA.
EM hedigerm@exchange.nih.gov
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NR 48
TC 53
Z9 56
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 848
EP 856
DI 10.1007/s10803-007-0453-6
PG 9
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700006
PM 17879151
ER
PT J
AU Naber, FBA
Bakermans-Kranenburg, MJ
van IJzendoorn, MH
Swinkels, SHN
Buitelaar, JK
Dietz, C
van Daalen, E
van Engeland, H
AF Naber, Fabienne B. A.
Bakermans-Kranenburg, Marian J.
van IJzendoorn, Marinus H.
Swinkels, Sophie H. N.
Buitelaar, Jan K.
Dietz, Claudine
van Daalen, Emma
van Engeland, Herman
TI Play behavior and attachment in toddlers with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE attachment; autism; developmental disorder; play behavior
ID PERVASIVE DEVELOPMENTAL DISORDERS; TRAITS QUESTIONNAIRE ESAT; SYMBOLIC
PLAY; PRETEND PLAY; JOINT ATTENTION; EARLY-CHILDHOOD; CHILDREN;
SPECTRUM; INTERVENTION; PROGRESSION
AB Play helps to develop social skills. Children with autism show deviances in their play behavior that may be associated with delays in their social development. In this study, we investigated manipulative, functional and symbolic play behavior of toddlers with and without autism (mean age: 26.45, SD 5.63). The results showed that the quality of interaction between the child and the caregiver was related to the development of play behavior. In particular, security of attachment was related to better play behavior. When the developmental level of the child is taken into account, the attachment relationship of the child with the caregiver at this young age is a better predictor of the level of play behavior than the child's disorder.
C1 [Naber, Fabienne B. A.; Bakermans-Kranenburg, Marian J.; van IJzendoorn, Marinus H.] Leiden Univ, Ctr Family & Child Studies, NL-2300 RB Leiden, Netherlands.
[Swinkels, Sophie H. N.; Buitelaar, Jan K.] Radboud Univ Nijmegen Med Ctr, Dept Psychiat, Nijmegen, Netherlands.
[Naber, Fabienne B. A.; Dietz, Claudine; van Daalen, Emma; van Engeland, Herman] Univ Med Ctr Utrecht, Rudolf Magnus Inst Neurosci, Dept Child & Adolscent Psychiat, Utrecht, Netherlands.
RP Naber, FBA (reprint author), Leiden Univ, Ctr Family & Child Studies, NL-2300 RB Leiden, Netherlands.
EM Fnaber@fsw.leidenuniv.nl
RI van IJzendoorn, Marinus/I-1379-2012; Buitelaar, Jan/E-4584-2012
OI Buitelaar, Jan/0000-0001-8288-7757
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NR 58
TC 12
Z9 12
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 857
EP 866
DI 10.1007/s10803-007-0454-5
PG 10
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700007
PM 17896172
ER
PT J
AU Baker, AEZ
Lane, A
Angley, MT
Young, RL
AF Baker, Amy E. Z.
Lane, Alison
Angley, Manya T.
Young, Robyn L.
TI The relationship between sensory processing patterns and behavioural
responsiveness in autistic disorder: A pilot study
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE sensory processing; autistic disorder; pervasive developmental disorder;
behaviour; short sensory profile
ID YOUNG-CHILDREN; OCCUPATIONAL-THERAPY; SPECTRUM DISORDERS; PROFILE;
DISABILITIES; DISRUPTION; TODDLERS
AB Sensory processing (SP) difficulties have been reported in as many as 95% of children with autism, however, empirical research examining the existence of specific patterns of SP difficulties within this population is scarce. Furthermore, little attention has been given to examining the relationship between SP and either the core symptoms or secondary manifestations of autism. In the current study, SP patterns in children with autistic disorder (AD) were investigated via a caregiver questionnaire and findings were correlated with the social, emotional and behavioural responsiveness of participants. Results indicated the presence of specific SP patterns in this sample of children with AD and several significant relationships were found between SP and social, emotional and behavioural function.
C1 [Baker, Amy E. Z.] Univ S Australia, Sch Nursing & Midwifery, Adelaide, SA 5000, Australia.
[Young, Robyn L.] Flinders Univ S Australia, Sch Psychol, Adelaide, SA 5001, Australia.
[Angley, Manya T.] Univ S Australia, Sch Pharm & Med Sci, Sansom Inst, Adelaide, SA 5001, Australia.
[Baker, Amy E. Z.; Lane, Alison] Univ S Australia, Sch Hlth Sci, Sansom Inst, Adelaide, SA 5001, Australia.
RP Baker, AEZ (reprint author), Univ S Australia, Sch Nursing & Midwifery, Adelaide, SA 5000, Australia.
EM amy.baker@postgrads.unisa.edu.au
RI Angley, Manya/D-2645-2009; Lane, Alison/E-3460-2011
CR Aarons M., 1999, HDB AUTISM GUIDE PAR
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 38
TC 57
Z9 57
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 867
EP 875
DI 10.1007/s10803-007-0459-0
PG 9
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700008
PM 17899349
ER
PT J
AU Smith, LE
Seltzer, MM
Tager-Flusberg, H
Greenberg, JS
Carter, AS
AF Smith, Leann E.
Seltzer, Marsha Mailick
Tager-Flusberg, Helen
Greenberg, Jan S.
Carter, Alice S.
TI A comparative analysis of well-being and coping among mothers of
toddlers and mothers of adolescents with ASD
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE coping; autism symptoms; maternal well-being; toddlers; adolescents
ID AUTISM SPECTRUM DISORDERS; PERVASIVE DEVELOPMENTAL DISORDERS;
SCHOOL-AGE-CHILDREN; INTELLECTUAL DISABILITY; MENTAL-RETARDATION;
POSITIVE PERCEPTIONS; SYNDROME SPECIFICITY; PRESCHOOL-CHILDREN; BEHAVIOR
PROBLEMS; SOCIAL SUPPORT
AB The present study examined the impact of autism symptoms and coping strategies on the well-being of mothers of children with autism spectrum disorder (ASD). The sample consisted of 153 mothers of toddlers and 201 mothers of adolescents drawn from two ongoing, longitudinal studies of families of individuals with ASD. For mothers of toddlers, lower levels of emotion-focused coping and higher levels of problem-focused coping were generally associated with better maternal well-being, regardless of the level of child symptomatology. For mothers of adolescents, coping often acted as a buffer when autism symptoms were high. Although there was evidence of maternal distress in both groups, the presence of significant buffering effects reflects adaptation in the face of stress, particularly for mothers of adolescents.
C1 [Smith, Leann E.; Seltzer, Marsha Mailick; Greenberg, Jan S.] Univ Wisconsin, Waisman Ctr, Madison, WI 53705 USA.
[Tager-Flusberg, Helen; Carter, Alice S.] Boston Univ, Sch Med, Dept Anat & Neurobiol, Boston, MA 02118 USA.
[Carter, Alice S.] Univ Massachusetts, Dept Psychol, Boston, MA 02125 USA.
RP Smith, LE (reprint author), Univ Wisconsin, Waisman Ctr, 1500 Highland Ave, Madison, WI 53705 USA.
EM lsmith@waisman.wisc.edu
RI Tager-Flusberg, Helen/D-5265-2009
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NR 39
TC 42
Z9 45
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 876
EP 889
DI 10.1007/s10803-007-0461-6
PG 14
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700009
PM 17924181
ER
PT J
AU Lopata, C
Thomeer, ML
Volker, MA
Nida, RE
Lee, GK
AF Lopata, Christopher
Thomeer, Marcus L.
Volker, Martin A.
Nida, Robert E.
Lee, Gloria K.
TI Effectiveness of a manualized summer social treatment program for
high-functioning children with autism spectrum disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE social skills groups; intervention; high-functioning autism; Asperger's;
PDDNOS
ID PERVASIVE DEVELOPMENTAL DISORDER; ASPERGER-SYNDROME; INTERVENTION;
ADOLESCENTS; ADULTS
AB This paper presents findings from the final two years of a four-year study investigating a manualized social treatment program for high-functioning children with autism spectrum disorders. The study sought to (1) replicate and expand findings from years one and two; (2) compare outcomes of participants who received response-cost feedback versus non-categorical feedback; and (3) provide further evidence of program feasibility. Results indicated significant improvements in social skills and problem behaviors, however no significant differences for face emotion recognition. Measures of several socially-related behaviors yielded mixed results based on rater. While parent ratings did not appear to favor one feedback format, staff ratings appeared to favor the response-cost format on some measures. Results also provided support for program feasibility.
C1 [Lopata, Christopher; Thomeer, Marcus L.; Lee, Gloria K.] SUNY Buffalo, Sch Educ Psychol, Dept Counseling, Buffalo, NY 14260 USA.
[Thomeer, Marcus L.] Summit Educ Resources, Getzville, NY USA.
[Nida, Robert E.] Canisius Coll, Dept Teacher Educ, Buffalo, NY 14208 USA.
RP Lopata, C (reprint author), SUNY Buffalo, Sch Educ Psychol, Dept Counseling, 409 Baldy Hall, Buffalo, NY 14260 USA.
EM cjlopata@buffalo.edu
CR Abell F, 2005, AUTISM, V9, P515, DOI 10.1177/1362361305057857
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Attwood T., 2000, AUTISM, V4, P85, DOI DOI 10.1177/1362361300004001006
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NR 47
TC 36
Z9 36
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 890
EP 904
DI 10.1007/s10803-007-0460-7
PG 15
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700010
PM 18058012
ER
PT J
AU Ponnet, K
Buysse, A
Roeyers, H
De Clercq, A
AF Ponnet, Koen
Buysse, Ann
Roeyers, Herbert
De Clercq, Armand
TI Mind-reading in young adults with ASD: Does structure matter?
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; ASD; empathic accuracy; mind-reading
ID NATURALISTIC SOCIAL COGNITION; HIGH-FUNCTIONING CHILDREN; EMPATHIC
ACCURACY; ASPERGER-SYNDROME; STORY CHARACTERS; AUTISM; INDIVIDUALS;
INTELLIGENCE; RECOGNITION; STATES
AB This study further elaborates on the mind-reading impairments of young adults with autism spectrum disorder (ASD). The hypothesis is that differences in mind-reading abilities between subjects with ASD and control subjects become more apparent when they have to infer thoughts and feelings of other persons in a less structured or more chaotic conversation, than when they have to do so in a more structured conversation. Conform to the empathic accuracy design, subjects viewed two videotaped interactions depicting two strangers and attempted to infer thoughts and feelings. One of the videotaped conversations was less structured than in the other. The results underscore the significance of structure to the mind-reading abilities of young adults with ASD.
C1 [Ponnet, Koen; Buysse, Ann; Roeyers, Herbert] Univ Ghent, Dept Psychol, B-9000 Ghent, Belgium.
[De Clercq, Armand] Univ Ghent, Dept Appl Math & Comp Sci, Ghent, Belgium.
RP Ponnet, K (reprint author), Univ Antwerp, Res Ctr Longitudinal & Life Course Studies, B-2020 Antwerp, Belgium.
EM Koen_Ponnet@yahoo.com
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NR 53
TC 8
Z9 8
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 905
EP 918
DI 10.1007/s10803-007-0462-5
PG 14
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700011
PM 17929156
ER
PT J
AU Rosset, DB
Rondan, C
Da Fonseca, D
Santos, A
Assouline, B
Deruelle, C
AF Rosset, Delphine B.
Rondan, Cecilie
Da Fonseca, David
Santos, Andreia
Assouline, Brigitte
Deruelle, Christine
TI Typical emotion processing for cartoon but not for real faces in
children with autistic spectrum disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; face; emotional expressions; cartoon; children; inversion effect
ID ASPERGER-SYNDROME; FACIAL EXPRESSIONS; YOUNG-CHILDREN; NORMAL ADULTS;
RECOGNITION; PERCEPTION; AMYGDALA; PEOPLE; DEFICITS; EYES
AB This study evaluated whether atypical face processing in autism extends from human to cartoon faces for which they show a greater interest. Twenty children with autistic spectrum disorders (ASD) were compared to two groups of typically developing children, matched on chronological and mental age. They processed the emotional expressions of real faces, human cartoon and non-human cartoon faces. Children with ASD were as capable as controls in processing emotional expressions, but strategies differed according to the type of face. Controls relied on a configural strategy with all faces. By contrast, ASD children exploited this typical configural strategy with cartoons but used a local strategy with real faces. This atypical visual processing style is discussed in the context of face expertise.
C1 [Rosset, Delphine B.; Rondan, Cecilie; Santos, Andreia; Deruelle, Christine] CNRS, INCM, F-13402 Marseille 20, France.
[Rosset, Delphine B.; Da Fonseca, David] Hosp Ste Marguerite, Ctr Resources Autisme, Marseille, France.
[Rondan, Cecilie; Assouline, Brigitte] Hosp St Egreve, Ctr Alpin Diagnost Precoce Autisme, St Egreve, France.
RP Rosset, DB (reprint author), CNRS, INCM, 31 Chemin Joseph Aiguier, F-13402 Marseille 20, France.
EM delphinerosset@yahoo.fr
RI deruelle, christine/E-2130-2015
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 52
TC 30
Z9 32
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 919
EP 925
DI 10.1007/s10803-007-0465-2
PG 7
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700012
PM 17952583
ER
PT J
AU Ganz, JB
Flores, MM
AF Ganz, Jennifer B.
Flores, Margaret M.
TI Effects of the use of visual strategies in play groups for children with
autism spectrum disorders and their peers
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; scripts; verbal communication; visual strategies; unscripted
speech
ID SCRIPT-FADING PROCEDURE; SOCIAL-INTERACTION SKILLS; ACTIVITY SCHEDULES;
TEACHING-CHILDREN; BEHAVIORS; CLASSROOM; MODEL; TASK
AB The purpose of this study was to investigate the impact of visual strategies with preschool children with autism spectrum disorders (ASD) and their peers during play group sessions. A changing-criterion design was implemented with three preschool-aged children with ASD while they participated in play groups with four typically-developing peers. Results indicated improvements in the use of script phrases, context-related comments, and intervals in which speech occurred for all three participants. Results regarding unscripted phrases, responses, and use of prompts were variable and are discussed.
C1 [Ganz, Jennifer B.] Univ Texas San Antonio, Dept ILT, San Antonio, TX 78249 USA.
[Ganz, Jennifer B.; Flores, Margaret M.] Univ Texas San Antonio, Dept Special Educ, San Antonio, TX USA.
RP Ganz, JB (reprint author), Univ Texas San Antonio, Dept ILT, 1 UTSA Circle, San Antonio, TX 78249 USA.
EM jennifer.ganz@utsa.edu
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NR 35
TC 15
Z9 15
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 926
EP 940
DI 10.1007/s10803-007-0463-4
PG 15
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700013
PM 17932735
ER
PT J
AU Vandenbroucke, MWG
Scholte, HS
van Engeland, H
Lamme, VAF
Kemner, C
AF Vandenbroucke, Myriam W. G.
Scholte, H. Steven
van Engeland, Herman
Lamme, Victor A. F.
Kemner, Chantal
TI Coherent versus component motion perception in autism spectrum disorder
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE asperger; integration; synchronization; global; stimulus rivalry
ID HIGH-FUNCTIONING AUTISM; WEAK CENTRAL COHERENCE; PRIMARY VISUAL-CORTEX;
BINOCULAR-RIVALRY; COMPLEX CELLS; CHILDREN; INDIVIDUALS; SEGREGATION;
SYNCHRONY; ATTENTION
AB Research on visual perception in Autism Spectrum Disorder (ASD) tries to reveal the underlying mechanisms of aberrant local and global processing. Global motion perception is one way to study this aspect of ASD. We used plaid motion stimuli, which can be perceived as a coherently moving pattern, requiring feature integration, or as two transparent gratings sliding over each other. If global motion detection is impaired in ASD, this would lead to a decrease of the total time that a coherent pattern is perceived. However, in contrast to other studies in the literature, our results gave no evidence of impaired global motion perception in people with ASD. A reconciliation of the different outcomes is proposed based on spatial frequency processing in ASD.
C1 [Vandenbroucke, Myriam W. G.; van Engeland, Herman; Kemner, Chantal] Univ Med Ctr Utrecht, Rudolf Magnus Inst Neurosci, Dept Child & Adolescent Psychiat, NL-3584 CX Utrecht, Netherlands.
[Vandenbroucke, Myriam W. G.; Scholte, H. Steven; Lamme, Victor A. F.] Univ Amsterdam, Dept Psychol, Amsterdam, Netherlands.
[Lamme, Victor A. F.] Netherlands Inst Neurosci, Royal Acad Arts & Sci KNAW, Amsterdam, Netherlands.
[Kemner, Chantal] Maastricht Univ, Fac Psychol, Dept Neurocognit, Maastricht, Netherlands.
RP Vandenbroucke, MWG (reprint author), Univ Med Ctr Utrecht, Rudolf Magnus Inst Neurosci, Dept Child & Adolescent Psychiat, B01-201,Hedelberglaan 100, NL-3584 CX Utrecht, Netherlands.
EM m.w.g.vandenbroucke@umcutrecht.nl
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NR 38
TC 15
Z9 15
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 941
EP 949
DI 10.1007/s10803-007-0467-0
PG 9
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700014
PM 17952582
ER
PT J
AU Ingram, DG
Takahashi, TN
Miles, JH
AF Ingram, David G.
Takahashi, T. Nicole
Miles, Judith H.
TI Defining autism subgroups: A taxometric solution
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism subgroups; taxometrics
ID HIGHER FUNCTIONING INDIVIDUALS; COMMUNICATION ABILITIES; PHENOTYPIC
HOMOGENEITY; REPETITIVE BEHAVIORS; DIMENSIONAL MODELS; PSYCHOPATHOLOGY;
DISORDER; CHROMOSOME-2; DISABILITIES; SPECTRUM
AB The purpose of the present study was to determine which behavioral and physical phenotypes would be most likely to divide the ASD population into discrete subgroups. The taxometric methods of Maximum Covariance (MAXCOV) and Minus Mean Below A Cut (MAMBAC) were employed to test for categorical versus continuous variation of each phenotype across the ASD population. Data was retrieved from the Autism Genetic Resource Exchange and the University of Missouri Autism Database. The results of our analyses support subgrouping subjects based on variation in social interaction/communication, intelligence, and essential/complex phenotype; in contrast, subjects varied continuously in insistence on sameness, repetitive sensory motor actions, language acquisition, and, tentatively, adaptive functioning. Stratifying ASD samples based on taxometric results should increase power in gene-finding studies and aid in treatment efficacy research.
C1 [Ingram, David G.; Takahashi, T. Nicole; Miles, Judith H.] Univ Missouri, Thompson Ctr Autsim & Neurodev Disorder, Columbia, MO 65201 USA.
RP Miles, JH (reprint author), Univ Missouri, Thompson Ctr Autsim & Neurodev Disorder, 300 Portland St,Suite 110, Columbia, MO 65201 USA.
EM milesjh@missouri.edu
CR *AGR, AGRE AFF STAT CAT
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Beglinger LJ, 2001, J AUTISM DEV DISORD, V31, P411, DOI 10.1023/A:1010616719877
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NR 34
TC 18
Z9 18
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 950
EP 960
DI 10.1007/s10803-007-0469-y
PG 11
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700015
PM 17985224
ER
PT J
AU Hess, KL
Morrier, MJ
Heflin, LJ
Ivey, ML
AF Hess, Kristen L.
Morrier, Michael J.
Heflin, L. Juane
Ivey, Michelle L.
TI Autism treatment survey: Services received by children with autism
spectrum disorders in public school classrooms
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; best practices; intervention strategies; public schools
AB The Autism Treatment Survey was developed to identify strategies used in education of children with autism spectrum disorders (ASD) in Georgia. Respondents of the web-based survey included a representative sample of 185 teachers across the state, reporting on 226 children with ASD in grades preschool-12th. The top five strategies being used in Georgia (Gentle Teaching, sensory integration, cognitive behavioral modification, assistive technology, and Social Stories (TM)) are recognized as lacking a scientific basis for implementation. Analysis revealed the choice of strategies varied by grade level and classroom type (e.g., general education, special education). Results highlight clear implications for preservice and inservice educator training, and the need for continued research to document evidence-based strategy use in public schools for students with ASD.
C1 [Hess, Kristen L.; Morrier, Michael J.; Heflin, L. Juane; Ivey, Michelle L.] Georgia State Univ, Dept Educ Psychol & Special Educ, Atlanta, GA 30302 USA.
RP Hess, KL (reprint author), Georgia State Univ, Dept Educ Psychol & Special Educ, POB 3979, Atlanta, GA 30302 USA.
EM ecekhh@langate.gsu.edu
CR American Psychiatric Association, 2004, DIAGN STAT MAN MENT
*AP SOFTW FDN, 2000, SPSS WIND VERS 13 0
GREEN G, 1999, AUTISM BEHAV ANAL PE
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Yeargin-Allsopp M, 2003, JAMA-J AM MED ASSOC, V289, P49, DOI 10.1001/jama.289.1.49
NR 15
TC 65
Z9 65
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 961
EP 971
DI 10.1007/s10803-007-0470-5
PG 11
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700016
PM 17929155
ER
PT J
AU Wiggins, LD
Robins, DL
AF Wiggins, Lisa D.
Robins, Diana L.
TI Brief report: Excluding the ADI-R behavioral domain improves diagnostic
agreement in toddlers
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE assessment; diagnosis; toddlers; ADI-R
ID AUTISM SPECTRUM DISORDERS; PERVASIVE DEVELOPMENTAL DISORDERS;
OBSERVATION SCHEDULE; FOLLOW-UP; CHILDREN; INTERVIEW; AGE; RELIABILITY;
INSTRUMENTS; VALIDITY
AB Past research shows poor agreement between the Autism Diagnostic Interview-Revised (ADI-R) and other diagnostic measures in toddlers. Our goal was to examine whether exclusion of the ADI-R behavioral domain results in improved diagnostic agreement. Toddlers aged 16-37 months (M = 26 months) received an evaluation because they failed the Modified Checklist for Autism in Toddlers (n = 142). Evaluations included the ADI-R, Autism Diagnostic Observation Schedule, Childhood Autism Rating Scale, and clinical judgment. Results found poor to fair agreement between the ADI-R and other measures; agreement improved when the ADI-R behavioral domain was excluded. These findings suggest that stereotyped interests and behaviors are not as relevant to the ADI-R as other diagnostic criteria when evaluating toddlers for autism spectrum disorders.
C1 [Wiggins, Lisa D.; Robins, Diana L.] Georgia State Univ, Dept Psychol, Atlanta, GA 30302 USA.
RP Wiggins, LD (reprint author), Georgia State Univ, Dept Psychol, POB 5010, Atlanta, GA 30302 USA.
EM lwiggins@cdc.gov
RI Robins, Diana/D-9959-2011
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NR 28
TC 16
Z9 17
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 972
EP 976
DI 10.1007/s10803-007-0456-3
PG 5
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700017
PM 17879150
ER
PT J
AU Lehmkuhl, HD
Storch, EA
Bodfish, JW
Geffken, GR
AF Lehmkuhl, Heather D.
Storch, Eric A.
Bodfish, James W.
Geffken, Gary R.
TI Brief report: Exposure and response prevention for obsessive compulsive
disorder in a 12-year-old with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE cognitive behavioral therapy; exposure and response prevention; autism;
obsessive compulsive disorder
ID MEDICATION-INDUCED HYPOMANIA; ASPERGERS-DISORDER; CHILDREN; ADOLESCENTS;
CBT
AB Obsessive Compulsive Disorder (OCD) involves exaggerated or excessive worry about threatening and non-threatening stimuli coupled with impairing rituals believed to reduce anxiety. Autism Spectrum Disorders (ASD) are characterized by impairment in social and communicative activities as well as restricted and repetitive behaviors. Approximately 2% of children with ASD are also diagnosed with OCD. Although there is extensive research demonstrating the effectiveness of behavioral interventions for pediatric OCD, little is known about how effective these treatments are for children who have a dual diagnosis of OCD and ASD. This report describes a 12-year-old male with Autism who was treated successfully with cognitive behavioral therapy with exposure and response prevention. This case study provides initial support that cognitive-behavioral therapy is effective in symptom reduction for children with comorbid autism and OCD.
C1 [Lehmkuhl, Heather D.; Storch, Eric A.; Geffken, Gary R.] Univ Florida, Dept Psychiat, Gainesville, FL 32611 USA.
[Storch, Eric A.; Geffken, Gary R.] Univ Florida, Dept Pediat, Gainesville, FL USA.
[Bodfish, James W.] Univ N Carolina, Dept Psychiat, Chapel Hill, NC USA.
[Geffken, Gary R.] Univ Florida, Dept Clin Hlth, Gainesville, FL USA.
RP Lehmkuhl, HD (reprint author), Univ Florida, Dept Psychiat, Gainesville, FL 32611 USA.
EM lehmkuhl@psychiatry.ufl.edu
RI Storch, Eric/I-4935-2012
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 29
TC 25
Z9 25
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 977
EP 981
DI 10.1007/s10803-007-0457-2
PG 5
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700018
PM 17885801
ER
PT J
AU Lajiness-O'Neill, R
Menard, P
AF Lajiness-O'Neill, Renee
Menard, Philip
TI Brief report: An autistic spectrum subtype revealed through familial
psychopathology coupled with cognition in ASD
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE pervasive development disorder; autistic disorder; Asperger syndrome;
neuropsychology; cognition; phenotype; endophenotype
ID WEAK CENTRAL COHERENCE; EXECUTIVE FUNCTION; ASPERGER-SYNDROME;
INFANTILE-AUTISM; RISK-FACTORS; CHILDREN; PARENTS; PHENOTYPE; SIBLINGS;
DISORDERS
AB This study identified a possible autistic spectrum subtype expressed through family psychopathology coupled with autistic probands' cognitive functioning (i.e., an endophenotypic profile). Participants included 24 children with Autism Spectrum Disorder (ASD) and 49 children with Learning Disorder (LD). There were significantly higher rates of Mood and Anxiety Disorder in first degree maternal relatives and of LD and Attention-Deficit Hyperactivity Disorder in first degree paternal relatives of ASD probands. Significantly higher visuospatial functioning was noted in all ASD probands for which there were higher rates of Mood Disorder on the maternal side suggesting a possible marker for an ASD subtype and indicating that maternal psychopathology may have a neuroprotective effect on visuospatial functioning.
C1 [Lajiness-O'Neill, Renee; Menard, Philip] Eastern Michigan Univ, Dept Psychol, Ypsilanti, MI 48197 USA.
[Lajiness-O'Neill, Renee] Henry Food Hlth Syst, Div Neuropsychol, Dept Behav Sci, Detroit, MI USA.
RP Lajiness-O'Neill, R (reprint author), Eastern Michigan Univ, Dept Psychol, 537F Marl Jefferson, Ypsilanti, MI 48197 USA.
EM rlajines@emich.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 38
TC 1
Z9 1
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 982
EP 987
DI 10.1007/s10803-007-0464-3
PG 6
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700019
PM 18034295
ER
PT J
AU Knickmeyer, RC
Baron-Cohen, S
Auyeung, B
Ashwin, E
AF Knickmeyer, Rebecca C.
Baron-Cohen, Simon
Auyeung, Bonnie
Ashwin, Emma
TI How to test the Extreme Male Brain Theory of Autism in terms of foetal
androgens?
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Letter
ID CONGENITAL ADRENAL-HYPERPLASIA; 4TH DIGIT RATIO; SEX; CHILDREN; 2ND
C1 [Knickmeyer, Rebecca C.] Univ N Carolina, Dept Psychiat, Chapel Hill, NC USA.
[Knickmeyer, Rebecca C.] Neurosci Hosp, Chapel Hill, NC 27599 USA.
[Baron-Cohen, Simon; Auyeung, Bonnie; Ashwin, Emma] Univ Cambridge, Autism Res Ctr, Cambridge, England.
RP Knickmeyer, RC (reprint author), Univ N Carolina, Dept Psychiat, CB 7160, Chapel Hill, NC USA.
EM rebecca_knickmeyer@med.unc.edu
RI Knickmeyer, Rebecca/G-8128-2014
OI Knickmeyer, Rebecca/0000-0001-7708-1388
CR Baron-Cohen S, 2005, SCIENCE, V310, P819, DOI 10.1126/science.1115455
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NR 8
TC 7
Z9 7
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 995
EP 996
DI 10.1007/s10803-008-0553-y
PG 2
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700021
PM 18327635
ER
PT J
AU Falter, CM
Plaisted, KC
Davis, G
AF Falter, Christine M.
Plaisted, Kate C.
Davis, Greg
TI Male brains, androgen, and the cognitive profile in autism: Convergent
evidence from 2D : 4D and Congenital Adrenal Hyperplasia
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Letter
C1 [Falter, Christine M.; Plaisted, Kate C.; Davis, Greg] Univ Cambridge, Cambridge CB2 3EB, England.
RP Davis, G (reprint author), Univ Cambridge, Downing St, Cambridge CB2 3EB, England.
EM gjd1000@cam.ac.uk
RI Davis, Gregory/G-9954-2012
CR FALTER CM, 2007, J AUTISM DEV DISORDE
Falter CM, 2006, BIOL PSYCHOL, V73, P132, DOI 10.1016/j.biopsycho.2006.01.011
Hines M, 2003, PSYCHONEUROENDOCRINO, V28, P1010, DOI 10.1016/S0306-4530(02)00121-X
RESNICK SM, 1986, DEV PSYCHOL, V22, P191, DOI 10.1037/0012-1649.22.2.191
NR 4
TC 5
Z9 6
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 997
EP 998
DI 10.1007/s10803-008-0552-z
PG 2
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700022
PM 18324462
ER
PT J
AU Grigorenko, EL
AF Grigorenko, Elena L.
TI P. Kluth and L. Chandler-Olcott, a land we can share: Teaching literacy
to students with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Book Review
C1 Yale Univ, Sch Med, Yale Child Study Ctr, New Haven, CT 06520 USA.
RP Grigorenko, EL (reprint author), Yale Univ, Sch Med, Yale Child Study Ctr, 230 S Frontage Rd,POB 207900, New Haven, CT 06520 USA.
EM Elena.grigorenko@yale.edu
CR *PAUL H BROOK PUBL, 2008, P KLUTH L CHANDLER O
NR 1
TC 0
Z9 0
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAY
PY 2008
VL 38
IS 5
BP 999
EP 1000
DI 10.1007/s10803-008-0562-x
PG 2
WC Psychology, Developmental
SC Psychology
GA 294NL
UT WOS:000255412700023
ER
PT J
AU Nagai, K
Natori, T
Nishino, T
Kodaira, F
AF Nagai, Kaoru
Natori, Takamitsu
Nishino, Toru
Kodaira, Fumiaki
TI Epigenetic disregulation induces cell growth retardation in primary
cultured glial cells
SO JOURNAL OF BIOSCIENCE AND BIOENGINEERING
LA English
DT Article
DE glia; epigeneties; DNA methylation; histone deacetylation; brain
development
ID RETT-SYNDROME; DNA METHYLATION; SCHIZOPHRENIA; POSTMORTEM; METABOLISM;
MECHANISMS; AUTISM; CORTEX; MECP2
AB Some epigenetic mechanisms, including DNA methylation and histone deacetylation, act as transcriptional repression signals. In this study, we examined whether DNA methylation dependent transcriptional control regulates glial cell growth. Primary cultured mouse cortical glial cells were treated with the DNA methylation inhibitor 5-aza-deoxycytidine (5adC) or the histone deacetylase inhibitor sodium valproate (VPA), which inhibits DNA-methylation-dependent transcriptional repression. 5adC significantly reduced methylated C level determined by reversed-phase high-performance liquid chromatography (HPLC), while VPA did not. Treatments with these inhibitors significantly reduced cell number determined by MTT assay after 48 h. Both 5adC and VPA showed little cellular toxicity observed by live and dead cell staining. In contrast, both 5adC and VPA induced an abnormality in the cell cycle. Cells treated with the inhibitors represented a significantly higher ratio in the G2+M-phase and 5adC-treated cells showed a significantly lower ratio in the S-phase. Regarding the in vivo effect, prenatal treatment with VPA, which is an autistic model in rodents, significantly reduced the brain/body weight ratio in early postnatal days. Our data indicate that DNA-methylation- and histone-deacetylation-dependent transcriptional control is crucial for the regulation of glial cell growth. Our data suggest that abnormalities of epigenetic transcriptional regulatory mechanisms in glial cells cause an abnormal brain size, which may in turn cause mental diseases.
C1 [Nagai, Kaoru; Natori, Takamitsu; Nishino, Toru; Kodaira, Fumiaki] Univ Yamanashi, Dept Epigenet Med, Interdisciplinary Grad Sch Med & Engn, Yamanashi 4093898, Japan.
RP Nagai, K (reprint author), Univ Yamanashi, Dept Epigenet Med, Interdisciplinary Grad Sch Med & Engn, 1110 Shimokato, Yamanashi 4093898, Japan.
EM kaoru@yamanashi.ac.jp
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NR 25
TC 3
Z9 3
PU SOC BIOSCIENCE BIOENGINEERING JAPAN
PI OSAKA
PA OSAKA UNIV, FACULTY ENGINEERING, 2-1 YAMADAOKA, SUITA, OSAKA, 565-0871,
JAPAN
SN 1389-1723
J9 J BIOSCI BIOENG
JI J. Biosci. Bioeng.
PD MAY
PY 2008
VL 105
IS 5
BP 470
EP 475
DI 10.1263/jbb.105.470
PG 6
WC Biotechnology & Applied Microbiology; Food Science & Technology
SC Biotechnology & Applied Microbiology; Food Science & Technology
GA 330GY
UT WOS:000257930000005
PM 18558336
ER
PT J
AU Hendriksen, JGM
Vles, JSH
AF Hendriksen, Joseph G. M.
Vles, Johan S. H.
TI Neuropsychiatric disorders in males with Duchenne muscular dystrophy:
Frequency rate of attention-deficit hyperactivity disorder (ADHD),
autism spectrum disorder, and obsessive-compulsive disorder
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Article
DE attention-deficit hyperactivity disorder; autism spectrum disorder;
obsessive-compulsive disorder; males; Duchenne muscular dystrophy
ID DEFICIT/HYPERACTIVITY DISORDER; SCALE; RELIABILITY; VALIDITY; BEHAVIOR
AB Using a questionnaire-based study, we assessed the parent-reported prevalence of attention-deficit hyperactivity disorders (ADHDs), autism spectrum disorders, and obsessive-compulsive disorders in a group of 351 males with Duchenne muscular dystrophy. Of the 351 males with Duchenne muscular dystrophy, 11.7% were reported to have a comorbid diagnosis of ADHD, 3.1% had autism spectrum disorder, and 4.8% had obsessive-compulsive disorder. It can be concluded that the incidence of these neuropsychiatric disorders is higher in Duchenne males than in the normal population. This finding, together with recent reports on the higher prevalence of cognitive and learning problems in Duchenne, supports the view that Duchenne muscular dystrophy is not only a muscular disorder but also a disorder affecting the brain. It is important for clinical practice to take in account this heightened association. More research is needed to examine this association and its consequences.
C1 [Hendriksen, Joseph G. M.] Kempenhaeghe Epilepsy Ctr, Dept Behav Sci, NL-5590 AB Heeze, Netherlands.
[Hendriksen, Joseph G. M.; Vles, Johan S. H.] Maastricht Univ Hosp, Dept Neurol, Maastricht, Netherlands.
RP Hendriksen, JGM (reprint author), Kempenhaeghe Epilepsy Ctr, Dept Behav Sci, POB 61, NL-5590 AB Heeze, Netherlands.
EM hendriksenj@kempenhaeghe.nl
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NR 23
TC 62
Z9 66
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD MAY
PY 2008
VL 23
IS 5
BP 477
EP 481
DI 10.1177/0883073807309775
PG 5
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 292XO
UT WOS:000255299600001
PM 18354150
ER
PT J
AU Andersen, IM
Kaczmarska, J
McGrevv, SG
Malow, BA
AF Andersen, Ivy M.
Kaczmarska, JoAnna
McGrevv, Susan G.
Malow, Beth A.
TI Melatonin for insomnia in children with autism spectrum disorders
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Article; Proceedings Paper
CT 20th Annual Meeting of the Associated-Professional-Sleep-Societies
CY JUN 17-22, 2006
CL Salt Lake City, UT
SP Associated Profess Sleep Soc
DE sleep; pervasive developmental disorder-not otherwise specified;
Asperger syndrome; melatonin
ID CONTROLLED-RELEASE MELATONIN; SLEEP DISORDERS
AB We describe our experience in using melatonin to treat insomnia, a common sleep concern, in children with autism spectrum disorders. One hundred seven children (2-18 years of age) with a confirmed diagnosis of autism spectrum disorders who received melatonin were identified by reviewing the electronic medical records of a single pediatrician. All parents were counseled on sleep hygiene techniques. Clinical response to melatonin, based on parental report, was categorized as (1) sleep no longer a concern, (2) improved sleep but continued parental concerns, (3) sleep continues to be a major concern, and (4) worsened sleep. The melatonin dose varied from 0.75 to 6 mg. After initiation of melatonin, parents of 27 children (25%) no longer reported sleep concerns at follow-up visits. Parents of 64 children (60%) reported improved sleep, although continued to have concerns regarding sleep. Parents of 14 children (13%) continued to report sleep problems as a major concern, with only 1 child having worse sleep after starting melatonin (1%), and 1 child having undetermined response (1%). Only 3 children had mild side-effects after starting melatonin, which included morning sleepiness and increased enuresis. There was no reported increase in seizures after starting melatonin in children with pre-existing epilepsy and no new-onset seizures. The majority of children were taking psychotropic medications. Melatonin appears to be a safe and well-tolerated treatment for insomnia in children with autism spectrum disorders. Controlled trials to determine efficacy appear warranted.
C1 [Andersen, Ivy M.; Kaczmarska, JoAnna; Malow, Beth A.] Vanderbilt Univ, Sch Med, Vanderbilt Childrens Hosp, Sleep Disorders Div, Nashville, TN 37212 USA.
[McGrevv, Susan G.] Vanderbilt Univ, Sch Med, Vanderbilt Childrens Hosp, Dept Neurol, Nashville, TN 37212 USA.
[McGrevv, Susan G.] Vanderbilt Univ, Sch Med, Vanderbilt Childrens Hosp, Kennedy Ctr, Nashville, TN 37212 USA.
RP Malow, BA (reprint author), Vanderbilt Univ, Dept Neurol, Med Ctr North, Room A-0118,1161 21st Ave S, Nashville, TN 37232 USA.
EM beth.malow@vanderbilt.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Giannotti F, 2006, J AUTISM DEV DISORD, V36, P741, DOI 10.1007/s10803-006-0116-z
JAN JE, 1994, DEV MED CHILD NEUROL, V36, P97
Jan JE, 2000, J PINEAL RES, V29, P34, DOI 10.1034/j.1600-079X.2000.290105.x
Malow BA, 2006, SLEEP, V29, P1563
Miyamoto A, 1999, BRAIN DEV-JPN, V21, P59, DOI 10.1016/S0387-7604(98)00072-2
Paavonen EJ, 2003, J CHILD ADOL PSYCHOP, V13, P83, DOI 10.1089/104454603321666225
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SHELDON SH, 1998, LANCET, V351, P1963
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Wheeler B, 2005, SLEEP, V28, P1609
Williams PG, 2004, J SLEEP RES, V13, P265
Zhdanova IV, 1999, J PEDIATR ENDOCR MET, V12, P57
NR 13
TC 60
Z9 62
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD MAY
PY 2008
VL 23
IS 5
BP 482
EP 485
DI 10.1177/0883073807309783
PG 4
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 292XO
UT WOS:000255299600002
PM 18182647
ER
PT J
AU Jeste, SS
Sahin, M
Bolton, P
Ploubidis, GB
Humphrey, A
AF Jeste, Shafali S.
Sahin, Mustafa
Bolton, Patrick
Ploubidis, George B.
Humphrey, Ayla
TI Characterization of autism in young children with tuberous sclerosis
complex
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Article
DE tuberous sclerosis complex; autism; Mullen Scale of Early Learning
ID FRAGILE-X-SYNDROME; MENTAL-RETARDATION; DISORDERS; SPECTRUM;
COMMUNICATION; ADOLESCENTS; BEHAVIORS
AB Both cognitive impairment and autism are common in the tuberous sclerosis complex, but the relationship between the 2 diagnoses has not been formally explored. The authors evaluated 20 clinic-referred children with tuberous sclerosis complex at ages 18, 24, 36, and 60 months and classified them as autism, autism spectrum disorder, or normal on the basis of the Autism Diagnostic Observation Schedule. Using the Mullen Scale of Early Learning, cognitive function in each subgroup was assessed. The authors then analyzed the subscores of the Autism Diagnostic Observation Schedule in children with autism. Children with autism showed significantly more global cognitive impairment than those without autism. In addition, all children had some baseline cognitive impairment and the majority had deficits in play scores. The authors conclude that clinic-referred children with tuberous sclerosis complex and autism are at considerable risk for cognitive impairment. These characteristics may help to guide more tailored services for these high-risk children.
C1 [Jeste, Shafali S.; Sahin, Mustafa] Childrens Hosp, Dept Neurol, Boston, MA 02115 USA.
[Jeste, Shafali S.; Sahin, Mustafa] Harvard Univ, Sch Med, Boston, MA USA.
[Ploubidis, George B.] London Sch Hyg & Trop Med, Ctr Populat Studies, Dept Epidemiol & Populat Hlth, London, England.
[Humphrey, Ayla] Univ Cambridge, Dept Psychiat, Cambridge, England.
RP Jeste, SS (reprint author), Childrens Hosp, Dept Neurol, 300 Longwood Ave,Fegan 11, Boston, MA 02115 USA.
EM shafali.jeste@childrens.harvard.edu
RI Ploubidis, George/G-9531-2011; Bolton, Patrick/E-8501-2010
OI Bolton, Patrick/0000-0002-5270-6262
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Wiznitzer M, 2004, J CHILD NEUROL, V19, P675
NR 21
TC 44
Z9 45
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD MAY
PY 2008
VL 23
IS 5
BP 520
EP 525
DI 10.1177/0883073807309788
PG 6
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 292XO
UT WOS:000255299600007
PM 18160549
ER
PT J
AU Skovgaard, AM
Olsen, EM
Christiansen, E
Houmann, T
Landorph, SL
Jorgensen, T
AF Skovgaard, A. M.
Olsen, E. M.
Christiansen, E.
Houmann, T.
Landorph, S. L.
Jorgensen, T.
CA CCC 2000 Study Grp
TI Predictors (0-10 months) of psychopathology at age 1 1/2 years - a
general population study in The Copenhagen Child Cohort CCC 2000
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE predictors; psychopathology; infant mental health; birth cohort;
epidemiology; longitudinal studies
ID PSYCHIATRIC-DISORDERS; MENTAL-HEALTH; BIRTH COHORT; AGE; INFANCY;
EPIDEMIOLOGY; VALIDITY; AUTISM; THRIVE; LIFE
AB Background: Epidemiological studies of mental health problems in the first years of life are few. This study aims to investigate infancy predictors of psychopathology in the second year of life. Methods: A random general population sample of 210 children from the Copenhagen Child Birth Cohort CCC 2000 was investigated by data from National Danish registers and data collected prospectively from birth in a general child health surveillance programme. Mental health outcome at 11/2 years was assessed by clinical and standardised measures including the Child Behavior Check List 11/2-5 (CBCL 11/2-5), Infant Toddler Symptom Check List (ITSCL), Checklist for Autism in Toddlers (CHAT), Bayley Scales of Infant Development (BSID II), Mannheim Eltern Interview (MEI), Parent Child Early Relational Assessment (PC ERA) and Parent Infant Relationship Global Assessment Scale (PIR-GAS), and disordered children were diagnosed according to the International Classification of Diseases (ICD-10) and Diagnostic Classification Zero to Three (DC: 0-3). Results: Deviant language development in the first 10 months of life predicted the child having any disorder at 11/2 years, OR 3.3 (1.4-8.0). Neuro-developmental disorders were predicted by deviant neuro-cognitive functioning, OR 6.8 (2.2-21.4), deviant language development, OR 5.9 (1.9-18.7) and impaired social interaction and communication, OR 3.8 (1.3-11.4). Unwanted pregnancy and parents' negative expectations of the child recorded in the first months of the child's life were significant predictors of relationship disturbances at 11/2 years. Conclusions: Predictors of neuro-developmental disorders and parent-child relationship disturbances can be identified in the first 10 months of life in children from the general population.
C1 [Skovgaard, A. M.; Olsen, E. M.; Houmann, T.; Landorph, S. L.] Univ Hosp Copenhagen, Child & Adolescent Psychiat Ctr, DK-2600 Glostrup, Denmark.
[Olsen, E. M.; Jorgensen, T.] Univ Hosp Copenhagen, Res Ctr Prevent & Hlth, DK-2600 Glostrup, Denmark.
[Christiansen, E.] Univ Hosp Copenhagen, Dept Child & Adolescent Psychiat, Bispebjerg, Denmark.
RP Skovgaard, AM (reprint author), Univ Hosp Copenhagen, Child & Adolescent Psychiat Ctr, DK-2600 Glostrup, Denmark.
EM ames@glo.regionH.dk
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NR 46
TC 22
Z9 23
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD MAY
PY 2008
VL 49
IS 5
BP 553
EP 562
DI 10.1111/j.1469-7610.2007.01860.x
PG 10
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 285QJ
UT WOS:000254791000010
PM 18341552
ER
PT J
AU Honey, K
AF Honey, Karen
TI Attention focuses on autism
SO JOURNAL OF CLINICAL INVESTIGATION
LA English
DT News Item
ID VACCINATION
CR *ASS PRESS, 2008, VACC SETTL COMPL MAY
Immunization Safety Review Committee, 2004, IMM SAF REV VACC AUT
Madsen KM, 2002, NEW ENGL J MED, V347, P1477, DOI 10.1056/NEJMoa021134
Murch SH, 2004, LANCET, V363, P750, DOI 10.1016/S0140-6736(04)15715-2
PEARCE A, 2008, BMJ
Schechter R, 2008, ARCH GEN PSYCHIAT, V65, P19, DOI 10.1001/archgenpsychiatry.2007.1
Smeeth L, 2004, LANCET, V364, P963, DOI 10.1016/S0140-6736(04)17020-7
STEINHAUER J, 2008, NY TIMES 0321
Wakefield AJ, 1998, LANCET, V351, P637, DOI 10.1016/S0140-6736(97)11096-0
NR 9
TC 2
Z9 2
PU AMER SOC CLINICAL INVESTIGATION INC
PI ANN ARBOR
PA 35 RESEARCH DR, STE 300, ANN ARBOR, MI 48103 USA
SN 0021-9738
J9 J CLIN INVEST
JI J. Clin. Invest.
PD MAY
PY 2008
VL 118
IS 5
BP 1586
EP 1587
DI 10.1172/JCI35821
PG 2
WC Medicine, Research & Experimental
SC Research & Experimental Medicine
GA 295QX
UT WOS:000255490100001
PM 18451989
ER
PT J
AU Esau, L
Kaur, M
Adonis, L
Arieff, Z
AF Esau, Luke
Kaur, Mandeep
Adonis, Lucinda
Arieff, Zainunisha
TI The 5-HTTLPR polymorphism in South African healthy populations: a global
comparison
SO JOURNAL OF NEURAL TRANSMISSION
LA English
DT Article
DE serotonin transporter; polymorphism; 5-HTTLPR; autism; South African
populations
ID SEROTONIN TRANSPORTER; ALLELIC VARIATION; GENE; ASSOCIATION; DISORDERS;
AUTISM; DEPRESSION; VARIANTS; REGION
AB The serotonin transporter promoter length polymorphism (5-hydroxytryptamine transporter length polymorphism, 5-HTTLPR) in serotonin transporter gene has been implicated in numerous psychiatric disorders. Having a high affinity for the neurotransmitter serotonin (5-hydroxytryptamine, 5-HT), serotonin transporter controls the duration, availability and signaling capacity of 5-HT in the synapse. Association studies have focused extensively on this polymorphic region as the frequencies of long- and short-alleles of this gene differ greatly amongst populations and association studies have either reported conflicting results or nothing significant at all. In this study, the genotype and allele frequencies of 5-HTTLPR polymorphism were determined in the healthy South African (SA) individuals belonging to diverse ethnic backgrounds. Cheek cell samples were collected from the three major ethnic groups namely: Caucasians, Africans and coloreds/Mixed population. The DNA was extracted and genotyped for the 5-HTTLPR. Genotypes were compared amongst the three major ethnic groups from SA as well as to that of other studies around the world. This is the first study to report significant differences in the 5-HTTLPR genotype and allelic frequencies among various ethnic groups in SA. Future studies will target larger population groups and the estimation of frequency of these alleles in individuals with autism.
C1 [Kaur, Mandeep] Univ Western Cape, SANBI, ZA-7535 Bellville, South Africa.
[Esau, Luke; Adonis, Lucinda; Arieff, Zainunisha] Univ Western Cape, Dept Biotechnol, ZA-7535 Bellville, South Africa.
RP Kaur, M (reprint author), Univ Western Cape, SANBI, Private Bag X17, ZA-7535 Bellville, South Africa.
EM mandeep@sanbi.ac.za
CR Arbelle S, 2003, AM J PSYCHIAT, V160, P671, DOI 10.1176/appi.ajp.160.4.671
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Hong CJ, 2003, J NEURAL TRANSM, V110, P345, DOI 10.1007/s00702-002-0790-8
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Stoneking M, 1997, GENOME RES, V7, P1061
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Yu YWY, 2002, MOL PSYCHIATR, V7, P1115, DOI 10.1038/sj.mp.4001141
NR 25
TC 11
Z9 12
PU SPRINGER WIEN
PI WIEN
PA SACHSENPLATZ 4-6, PO BOX 89, A-1201 WIEN, AUSTRIA
SN 0300-9564
J9 J NEURAL TRANSM
JI J. Neural Transm.
PD MAY
PY 2008
VL 115
IS 5
BP 755
EP 760
DI 10.1007/s00702-007-0012-5
PG 6
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 308ZY
UT WOS:000256430700012
PM 18193379
ER
PT J
AU Jyonouchi, H
Geng, L
Cushing-Ruby, A
Quraishi, H
AF Jyonouchi, Harumi
Geng, Lee
Cushing-Ruby, Agnes
Quraishi, Huma
TI Evaluation of atopy and immune functions in children with autism
spectrum disorders (ASD): Identification of an ASD subset with distinct
clinical and immunological findings
SO JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY
LA English
DT Meeting Abstract
CT 84th Annual Meeting of the American-Association-of-Neuropathologists
CY APR 04-07, 2008
CL San Diego, CA
SP Amer Assoc Neuropathologists
C1 [Quraishi, Huma] Univ Med & Dent New Jersey, New Jersey Med Sch, Newark, NJ 07103 USA.
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0022-3069
J9 J NEUROPATH EXP NEUR
JI J. Neuropathol. Exp. Neurol.
PD MAY
PY 2008
VL 67
IS 5
BP 518
EP 519
PG 2
WC Clinical Neurology; Neurosciences; Pathology
SC Neurosciences & Neurology; Pathology
GA 294YN
UT WOS:000255442300157
ER
PT J
AU Meltzer, LJ
AF Meltzer, Lisa J.
TI Brief report: Sleep in parents of children with autism spectrum
disorders
SO JOURNAL OF PEDIATRIC PSYCHOLOGY
LA English
DT Article
DE autism; children; fathers; mothers; sleep
ID PERVASIVE DEVELOPMENTAL DISORDERS; ASPERGERS-DISORDER; QUALITY INDEX;
ACTIGRAPHY; PATTERNS; DISABILITIES; BEHAVIOR
AB Objective To examine sleep quality and sleepwake patterns in parents of children with autism spectrum disorders (ASDs) and parents of typically developing (TD) children. Methods Thirty-five mothers and 22 fathers completed the Pittsburgh Sleep Quality Index, a 7-day sleep diary, and wore an actigraph for 1 week. Results Parents of children with ASDs reported poorer sleep quality compared to the TD group. In addition, parents of children with ASDs had objectively different sleep patterns, with an earlier wake time and shorter total sleep time than parents of TD children. Finally, regardless of group, fathers had significantly shorter sleep time compared to mothers. Conclusions This study is one of the first to demonstrate poorer sleep quality and shorter sleep quantity in parents of children with ASDs using validated measures of sleep. Future studies should examine the relationship between chronic sleep loss and stress in parents of children with ASDs.
C1 [Meltzer, Lisa J.] Childrens Hosp Philadelphia, Philadelphia, PA 19104 USA.
[Meltzer, Lisa J.] Univ Penn, Dept Pediat, Philadelphia, PA 19104 USA.
[Meltzer, Lisa J.] Univ Penn, Div Pulm Med, Philadelphia, PA 19104 USA.
RP Meltzer, LJ (reprint author), Childrens Hosp Philadelphia, 3535 Market St,14th Floor, Philadelphia, PA 19104 USA.
EM meltzerl@email.chop.edu
CR Allik H, 2006, J AUTISM DEV DISORD, V36, P585, DOI 10.1007/s10803-006-0099-9
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NR 25
TC 20
Z9 20
PU OXFORD UNIV PRESS INC
PI CARY
PA JOURNALS DEPT, 2001 EVANS RD, CARY, NC 27513 USA
SN 0146-8693
J9 J PEDIATR PSYCHOL
JI J. Pediatr. Psychol.
PD MAY
PY 2008
VL 33
IS 4
BP 380
EP 386
DI 10.1093/jpepsy/jsn005
PG 7
WC Psychology, Developmental
SC Psychology
GA 284NW
UT WOS:000254714100006
PM 18250091
ER
PT J
AU Corbett, BA
Mendoza, S
Wegelin, JA
Carmean, V
Levine, S
AF Corbett, Blythe A.
Mendoza, Sally
Wegelin, Jacob A.
Carmean, Vanessa
Levine, Seymour
TI Variable cortisol circadian rhythms in children with autism and
anticipatory stress
SO JOURNAL OF PSYCHIATRY & NEUROSCIENCE
LA English
DT Article
DE autistic disorder; cortisol; circadian rhythm; stress
ID COMPLEX DEVELOPMENTAL DISORDER; DEXAMETHASONE SUPPRESSION TEST; SALIVARY
CORTISOL; PSYCHOSOCIAL STRESS; MAJOR DEPRESSION; SECRETION; EXPOSURE;
AXIS
AB Objective: Autism is characterized by impairment in communication and social interaction, by repetitive behaviours and by difficulty in adapting to novel experiences. The objective of the current investigation was to replicate and extend our previous findings showing variable circadian rhythm and significant elevations in cortisol following exposure to a novel stimulus (mock magnetic resonance imaging [MRI]). Methods: Circadian rhythms of cortisol were estimated in 22 children with and 22 children without autism via analysis of salivary samples collected in the morning, afternoon and evening over 6 separate days. We assessed hypothalamic-pituitary-ad renal (HPA) responsiveness by examining changes in salivary cortisol in response to a mock MRI. One-half of the children were re-exposed to the MRI environment. Results: Children with autism showed a decrease in cortisol in the morning over 6 days while maintaining higher evening values. Children with autism also showed more within- and between-subject variability in circadian rhythms. Although the cortisol values tended to be higher in some of the children with autism, a statistically significant elevation in cortisol in response to the initial mock MRI was not observed. Rather, both groups showed heightened cortisol at the arrival to the second visit to the imaging centre, suggesting an anticipatory response to the re-exposure to the mock MRI. Conclusion: Children with autism showed dysregulation of the circadian rhythm evidenced by variability between groups, between children and within individual child comparisons. Both groups demonstrated increased salivary cortisol in anticipation of re-exposure to the perceived stressor.
C1 [Corbett, Blythe A.; Carmean, Vanessa; Levine, Seymour] Virginia Commonwealth Univ, Dept Psychiat & Behav Sci, Richmond, VA USA.
[Corbett, Blythe A.; Carmean, Vanessa] Virginia Commonwealth Univ, MIND Inst, Richmond, VA USA.
[Mendoza, Sally] Virginia Commonwealth Univ, Dept Psychol, Richmond, VA 23284 USA.
[Wegelin, Jacob A.] Virginia Commonwealth Univ, Dept Biostat, Richmond, VA USA.
RP Corbett, BA (reprint author), Univ Calif Davis, Dept Psychiat & Behav Sci, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM blythe.corbett@ucdmc.ucdavis.edu
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NR 32
TC 45
Z9 45
PU CMA-CANADIAN MEDICAL ASSOC
PI OTTAWA
PA 1867 ALTA VISTA DR, OTTAWA, ONTARIO K1G 3Y6, CANADA
SN 1180-4882
J9 J PSYCHIATR NEUROSCI
JI J. Psychiatry Neurosci.
PD MAY
PY 2008
VL 33
IS 3
BP 227
EP 234
PG 8
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 306HB
UT WOS:000256237800006
PM 18592041
ER
PT J
AU Loukusa, S
Ryder, N
Leinonen, E
AF Loukusa, Soile
Ryder, Nuala
Leinonen, Eeva
TI Answering questions and explaining answers: A study of Finnish-speaking
children
SO JOURNAL OF PSYCHOLINGUISTIC RESEARCH
LA English
DT Article
DE comprehension strategies; inference; pragmatic processing; relevance
theory
ID ASPERGER-SYNDROME; RELEVANCE THEORY; LANGUAGE; COMPREHENSION;
PRAGMATICS; CONTEXT; AUTISM; MIND; STRATEGIES; KNOWLEDGE
AB This research explores, within the framework of Relevance Theory, how children's ability to answer questions and explain their answers develops between the ages of 3 and 9 years. Two hundred and ten normally developing Finnish-speaking children participated in this study. The children were asked questions requiring processing of inferential meanings and routines, and were asked to explain their correct answers to elicit understanding about their awareness of how they had derived the answers from the context. The results indicated that the number of correct answers increased rapidly between the ages of 3 years and 4-5 years. Familiarity of context had a significant effect on young children's ability to answer questions. Becoming aware of the information used in inferencing developed gradually over time between the ages of 3 and 9. Analysis of the children's incorrect answers and explanations showed that, as children develop, their unsophisticated answer strategies diminish and they increasingly utilize context even in incorrect answers and explanations.
C1 [Loukusa, Soile] Univ Oulu, Informat Studies & Logoped, Dept Finnish, Oulu 90014, Finland.
[Ryder, Nuala; Leinonen, Eeva] Univ Hertfordshire, Dept Psychol, Hatfield AL10 9AB, Herts, England.
RP Loukusa, S (reprint author), Univ Oulu, Informat Studies & Logoped, Dept Finnish, Oulu 90014, Finland.
EM soile.loukusa@oulu.fi
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NR 53
TC 7
Z9 7
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0090-6905
J9 J PSYCHOLINGUIST RES
JI J. Psycholinguist. Res.
PD MAY
PY 2008
VL 37
IS 3
BP 219
EP 241
DI 10.1007/s10936-007-9067-6
PG 23
WC Linguistics; Psychology, Experimental
SC Linguistics; Psychology
GA 270YH
UT WOS:000253755600004
PM 17990120
ER
PT J
AU Cleavinger, HB
Bigler, ED
Johnson, JL
Lu, J
McMahon, W
Lainhart, JE
AF Cleavinger, Howard B.
Bigler, Erin D.
Johnson, Jamie L.
Lu, Jeffrey
McMahon, William
Lainhart, Janet E.
TI Quantitative magnetic resonance image analysis of the cerebellum in
macrocephalic and normocephalic children and adults with autism
SO JOURNAL OF THE INTERNATIONAL NEUROPSYCHOLOGICAL SOCIETY
LA English
DT Review
DE neuroimaging; autism; cerebellar; brain volumes; head size
ID PERVASIVE DEVELOPMENTAL DISORDERS; POSTERIOR-FOSSA STRUCTURES; EARLY
INFANTILE-AUTISM; HEAD CIRCUMFERENCE; SPECTRUM DISORDER; BRAIN
STRUCTURE; ASPERGER-SYNDROME; CORPUS-CALLOSUM; MRI; ABNORMALITIES
AB A detailed morphometric analysis of the cerebellum ill autism with and without macrocephaly. Four subject groups (N = 65; male, IQs >= 65 age 7 to 26 years) were studied with quantitative MRI; normocephalic and macrocephalic individuals with autism without mental retardation were compared to normocephalic and benign macrocephalic typically developing individuals. Total cerebellum volumes and surface areas of four lobular midsagittal groups were measured. Independent t-tests between autism and control Subjects matched for head size revealed no significant differences. Multivariate analyses of variance were also performed, using the diagnostic group as the fixed factor, cerebellar measures as the dependent variables and total intracranial Volume, total brain Volume, age, verbal IQ, and performance IQ as covariates. No significant differences Were found however, a trend was noted in which macrocephalic individuals with autism consistently exhibited slightly smaller cerebellar volume or surface area when compared to individuals with benign macrocephaly. In autism, with and without macrocephaly, cerebellar Structures were found to be proportional to head size and did not differ from typically developing subjects.
C1 [Cleavinger, Howard B.; Bigler, Erin D.; Johnson, Jamie L.] Brigham Young Univ, Dept Psychol, Provo, UT 84602 USA.
[Cleavinger, Howard B.; Bigler, Erin D.; Johnson, Jamie L.] Brigham Young Univ, Dept Neurosci, Provo, UT 84602 USA.
[Bigler, Erin D.; McMahon, William; Lainhart, Janet E.] Univ Utah, Dept Psychiat, Salt Lake City, UT USA.
[Lu, Jeffrey] Univ Utah, Sch Med, Dept Anesthesiol, Salt Lake City, UT USA.
[Bigler, Erin D.; McMahon, William; Lainhart, Janet E.] Univ Utah, Inst Brain, Salt Lake City, UT USA.
RP Bigler, ED (reprint author), Brigham Young Univ, Dept Psychol, 1001 SWKT, Provo, UT 84602 USA.
EM erin_bigler@byu.edu
FU NICHD [U19 HD35476]; NICHD/NIDCD Collaborative Pro-rams of Excellence in
Autism (CPEA); Ira Fulton Foundation; Utah Autism Foundation; Valley
Mental Health
FX This work was supported by NICHD U19 HD35476 (University of Utah), the
NICHD/NIDCD Collaborative Pro-rams of Excellence in Autism (CPEA), the
Ira Fulton Foundation, the Utah Autism Foundation, and Valley Mental
Health. The technical assistance of Tracy Abildskov and the editorial
assistance of Jo Ann Petrie are gratefully acknowledged. The authors
thank the staff of the Utah Autism Research Program. particularly
Michael Johnson. Jubel Morgan, Lori Krasny, Barbara Young, Dr. Sally J.
Ozonoff (now of the UC Davis MIND Institute). Henry Buswell and Dr.
Dennis Parker of the University of Utah Research Radiology. The authors
particularly thank the children and adults who participated in this
study and their parents.
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NR 108
TC 6
Z9 8
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1355-6177
J9 J INT NEUROPSYCH SOC
JI J. Int. Neuropsychol. Soc.
PD MAY
PY 2008
VL 14
IS 3
BP 401
EP 413
DI 10.1017/S1355617708080594
PG 13
WC Clinical Neurology; Neurosciences; Psychiatry; Psychology
SC Neurosciences & Neurology; Psychiatry; Psychology
GA 338VK
UT WOS:000258537300006
PM 18419839
ER
PT J
AU Seeman, C
AF Seeman, Corey
TI Voices of autism: The healing companion; stories for courage, comfort
and strength
SO LIBRARY JOURNAL
LA English
DT Book Review
C1 [Seeman, Corey] Univ Michigan, Kresge Business Admn, Ann Arbor, MI 48109 USA.
RP Seeman, C (reprint author), Univ Michigan, Kresge Business Admn, Ann Arbor, MI 48109 USA.
CR LACHANCE, 2008, VOICE AUTISM HEALING
NR 1
TC 0
Z9 0
PU REED BUSINESS INFORMATION
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010 USA
SN 0363-0277
J9 LIBR J
JI Libr. J.
PD MAY 1
PY 2008
VL 133
IS 8
BP 88
EP 88
PG 1
WC Information Science & Library Science
SC Information Science & Library Science
GA 293TY
UT WOS:000255359400156
ER
PT J
AU Pan, CY
Oh, HK
Chu, CH
AF Pan, Chien-Yu
Oh, Hyun-Kyoung
Chu, Chia-Hua
TI PE and Recess Differences in Social Engagement and Physical Activity in
Children with Autism
SO MEDICINE AND SCIENCE IN SPORTS AND EXERCISE
LA English
DT Meeting Abstract
C1 [Pan, Chien-Yu; Chu, Chia-Hua] Natl Kaohsiung Normal Univ, Kaohsiung, Taiwan.
[Oh, Hyun-Kyoung] Calif State Univ San Bernardino, San Bernardino, CA 92407 USA.
EM chpan@nknucc.nknu.edu.tw
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0195-9131
J9 MED SCI SPORT EXER
JI Med. Sci. Sports Exerc.
PD MAY
PY 2008
VL 40
IS 5
SU S
BP S411
EP S412
DI 10.1249/01.mss.0000322754.15528.11
PG 2
WC Sport Sciences
SC Sport Sciences
GA V19KI
UT WOS:000208070903333
ER
PT J
AU Ivanov, I
Charney, A
AF Ivanov, Iliyan
Charney, Alexander
TI Treating pediatric patients with antipsychotic drugs: Balancing benefits
and safety
SO MOUNT SINAI JOURNAL OF MEDICINE
LA English
DT Article
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; CHILDHOOD-ONSET SCHIZOPHRENIA;
DISRUPTIVE BEHAVIOR DISORDER; AUTISM SPECTRUM DISORDERS; ATYPICAL
ANTIPSYCHOTICS; METABOLIC SYNDROME; DOUBLE-BLIND; BIPOLAR DISORDER;
PITUITARY INCIDENTALOMAS; PSYCHIATRIC-PATIENTS
C1 [Ivanov, Iliyan; Charney, Alexander] Mt Sinai Sch Med, Dept Psychiat, New York, NY 10029 USA.
RP Ivanov, I (reprint author), Mt Sinai Sch Med, Dept Psychiat, New York, NY 10029 USA.
EM iliyan.ivanov@mssm.edu
FU NIDA/AACAP K23 [PA-00-003]
FX This work was supported by NIDA/AACAP K23 (PA-00-003).
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CBS EVENING NEW 1111
NR 70
TC 2
Z9 2
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 0027-2507
J9 MT SINAI J MED
JI Mt. Sinai J. Med.
PD MAY-JUN
PY 2008
VL 75
IS 3
BP 276
EP 286
DI 10.1002/msj.20051
PG 11
WC Medicine, General & Internal
SC General & Internal Medicine
GA 343MT
UT WOS:000258858900007
PM 18704980
ER
PT J
AU Abrahams, BS
Geschwind, DH
AF Abrahams, Brett S.
Geschwind, Daniel H.
TI Advances in autism genetics: on the threshold of a new neurobiology
SO NATURE REVIEWS GENETICS
LA English
DT Review
ID FRAGILE-X-SYNDROME; QUANTITATIVE-TRAIT LOCUS;
HOMEOBOX-TRANSCRIPTION-FACTOR; SEROTONIN TRANSPORTER GENE;
LYMPHOBLASTOID CELL-LINES; FAMILY-BASED ASSOCIATION; CARRIER SLC25A12
GENE; SPECTRUM DISORDERS; GENOMEWIDE SCREEN; SUSCEPTIBILITY LOCUS
AB Autism is a heterogeneous syndrome defined by impairments in three core domains: social interaction, language and range of interests. Recent work has led to the identification of several autism susceptibility genes and an increased appreciation of the contribution of de novo and inherited copy number variation. Promising strategies are also being applied to identify common genetic risk variants. Systems biology approaches, including array-based expression profiling, are poised to provide additional insights into this group of disorders, in which heterogeneity, both genetic and phenotypic, is emerging as a dominant theme.
C1 [Abrahams, Brett S.; Geschwind, Daniel H.] Univ Calif Los Angeles, David Geffen Sch Med, Dept Neurol, Program Neurogenet, Los Angeles, CA 90095 USA.
[Abrahams, Brett S.; Geschwind, Daniel H.] Univ Calif Los Angeles, David Geffen Sch Med, Dept Neurol, Program Neurobehav Genet, Los Angeles, CA 90095 USA.
[Abrahams, Brett S.; Geschwind, Daniel H.] Univ Calif Los Angeles, David Geffen Sch Med, Semel Inst Neurosci & Behav, Los Angeles, CA 90095 USA.
RP Abrahams, BS (reprint author), Univ Calif Los Angeles, David Geffen Sch Med, Dept Neurol, Program Neurogenet, Los Angeles, CA 90095 USA.
EM brett.abrahams@gmail.com; dhg@ucla.edu
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NR 152
TC 739
Z9 760
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1471-0056
EI 1471-0064
J9 NAT REV GENET
JI Nat. Rev. Genet.
PD MAY
PY 2008
VL 9
IS 5
BP 341
EP 355
DI 10.1038/nrg2346
PG 15
WC Genetics & Heredity
SC Genetics & Heredity
GA 289LZ
UT WOS:000255057300011
PM 18414403
ER
PT J
AU Shultz, SR
MacFabe, DE
Ossenkopp, KP
Scratch, S
Whelan, J
Taylor, R
Cain, DP
AF Shultz, Sandy R.
MacFabe, Deffick E.
Ossenkopp, Klaus-Peter
Scratch, Shannon
Whelan, Jennifer
Taylor, Roy
Cain, Donald P.
TI Intracerebroventricular injection of propionic acid, an enteric
bacterial metabolic end-product, impairs social behavior in the rat:
Implications for an animal model of autism
SO NEUROPHARMACOLOGY
LA English
DT Article
DE intracerebroventricular; behavioral tracking; play behavior; autism;
animal model; neuroinflammatory; sodium acetate; propanol; short chain
fatty acids
ID 3-NITROPROPIONIC ACID; SPECTRUM DISORDER; VALPROIC ACID; FATTY-ACIDS;
SHORT-CHAIN; CHILDREN; PLAY; BRAIN; ABNORMALITIES; COMPLEXITY
AB Environmental, dietary, and gastrointestinal factors may contribute to autism spectrum disorders (ASD). Propionic acid (PPA) is a short chain fatty acid, a metabolic end-product of enteric bacteria in the gut, and a common food preservative. Recent evidence indicates that PPA can cause behavioral abnormalities and a neuroinflammatory response in rats. Social behavior was examined in similarly-treated pairs of adult male Long-Evans rats placed in an open field following intracerebroventricular (ICV) injection of PPA (4 mu l of 0.26 M solution) or control compounds. Behavior was analyzed using both the EthoVision behavior tracking system and by blind scoring of videotapes of social behaviors. Compared to controls, rats treated with PPA displayed social behavior impairments as indicated by significantly greater mean distance apart, reduced time spent in close proximity, reduced playful interaction, and altered responses to playful initiations. Treatment with another short chain fatty acid, sodium acetate, produced similar impairments, but treatment with the alcohol analog of PPA, 1-propanol, did not produce impairments. Immunohistochemical analysis of brain tissue taken from rats treated with PPA revealed reactive astrogliosis, indicating a neuroinflammatory response. These findings suggest that PPA can change both brain and behavior in the laboratory rat in a manner that is consistent with symptoms of human ASD. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Shultz, Sandy R.; MacFabe, Deffick E.] Univ Western Ontario, Dept Psychol, Kilee Patchell Evans Autism Res Grp, Div Dev Disabil, London, ON N6A 5B8, Canada.
[Shultz, Sandy R.; Ossenkopp, Klaus-Peter; Cain, Donald P.] Univ Western Ontario, Grad Program Neurosci, London, ON N6A 5B8, Canada.
[MacFabe, Deffick E.] Univ Western Ontario, Dept Psychiat, Kilee Patchell Evans Autism Res Grp, Div Dev Disabil, London, ON N6A 5B8, Canada.
RP Shultz, SR (reprint author), Univ Western Ontario, Dept Psychol, Kilee Patchell Evans Autism Res Grp, Div Dev Disabil, 1151 Richmond St,SSC Room 7418, London, ON N6A 5B8, Canada.
EM sshultz@uwo.ca
RI Ossenkopp, Klaus-Peter/A-4810-2008
OI Ossenkopp, Klaus-Peter/0000-0001-7391-3239
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NR 54
TC 48
Z9 50
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0028-3908
J9 NEUROPHARMACOLOGY
JI Neuropharmacology
PD MAY
PY 2008
VL 54
IS 6
BP 901
EP 911
DI 10.1016/j.neuropharm.2008.01.013
PG 11
WC Neurosciences; Pharmacology & Pharmacy
SC Neurosciences & Neurology; Pharmacology & Pharmacy
GA 305TL
UT WOS:000256200600001
PM 18395759
ER
PT J
AU Goldstein, G
Allen, DN
Minshew, NJ
Williams, DL
Volkmar, F
Klin, A
Schultz, RT
AF Goldstein, Gerald
Allen, Daniel N.
Minshew, Nancy J.
Williams, Diane L.
Volkmar, Fred
Klin, Ami
Schultz, Robert T.
TI The structure of intelligence in children and adults with high
functioning autism
SO NEUROPSYCHOLOGY
LA English
DT Article
DE autism; intelligence testing; confirmatory factor analysis
ID CONFIRMATORY FACTOR-ANALYSIS; DIAGNOSTIC OBSERVATION SCHEDULE; SENTENCE
COMPREHENSION; WAIS-R; SCHIZOPHRENIA; INDIVIDUALS; FIT;
UNDERCONNECTIVITY; CONNECTIVITY; VALIDATION
AB Confirmatory factor analyses of the commonly used 11 subtests of the Wechsler child and adult intelligence scales were accomplished for 137 children and 117 adults with high functioning autism (HFA) and for comparable age groups from the standardization samples contained in the Wechsler manuals. The objectives were to determine whether the structure of intelligence in HFA groups was similar to that found in the normative samples, and whether a separate "social context" factor would emerge that was unique to HFA. Four-factor models incorporating a Social Context factor provided the best fit in both the autism and normative samples, but the subtest intercorrelations were generally lower in the autism samples. Findings suggest similar organization of cognitive abilities in HFA, but with the possibility of underconnectivity or reduced communication among brain regions in autism.
C1 [Goldstein, Gerald] VA Pittsburgh Hlth Care Syst, Res Serv 151R, Pittsburgh, PA 15206 USA.
[Allen, Daniel N.] Univ Nevada Las Vegas, Dept Psychol, Las Vegas, NV USA.
[Minshew, Nancy J.] Univ Pittsburgh, Sch Med, Dept Psychiat, Pittsburgh, PA 15260 USA.
[Minshew, Nancy J.] Univ Pittsburgh, Sch Med, Dept Neurol, Pittsburgh, PA 15260 USA.
[Williams, Diane L.] Duquesne Univ, Dept Speech Language Pathol, Pittsburgh, PA USA.
[Volkmar, Fred; Klin, Ami] Yale Univ, Sch Med, Dept Psychiat, New Haven, CT 06520 USA.
[Schultz, Robert T.] Childrens Hosp Philadelphia, Dept Pediat, Philadelphia, PA USA.
RP Goldstein, G (reprint author), VA Pittsburgh Hlth Care Syst, Res Serv 151R, 7180 Highland Dr, Pittsburgh, PA 15206 USA.
EM ggold@nb.net
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NR 44
TC 22
Z9 23
PU AMER PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST NE, WASHINGTON, DC 20002-4242 USA
SN 0894-4105
J9 NEUROPSYCHOLOGY
JI Neuropsychology
PD MAY
PY 2008
VL 22
IS 3
BP 301
EP 312
DI 10.1037/0894-4105.22.3.301
PG 12
WC Psychology, Clinical; Neurosciences; Psychology
SC Psychology; Neurosciences & Neurology
GA 294AX
UT WOS:000255377600003
PM 18444708
ER
PT J
AU Pessah, IN
Seegal, RF
Lein, PJ
LaSalle, J
Yee, BK
Van De Water, J
Berman, RF
AF Pessah, Isaac N.
Seegal, Richard F.
Lein, Pamela J.
LaSalle, Janine
Yee, Benjamin K.
Van De Water, Judy
Berman, Robert F.
TI Immunologic and neurodevelopmental susceptibilities of autism
SO NEUROTOXICOLOGY
LA English
DT Article; Proceedings Paper
CT 11th Meeting of the International-Neurotoxicology-Association
CY JUN, 2007
CL Pacific Grove, CA
SP Int Neurotoxicol Assoc
DE autism; ADHD; organochlorines; mercury; polychlorinated biphenyls;
organophosphate; neuroimmunology
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; RECEPTOR SUBUNIT GENES;
DEFICIT HYPERACTIVITY DISORDER; AMINOBUTYRIC-ACID RECEPTOR; POSTNATAL
MATERNAL CONTRIBUTIONS; MYELIN BASIC-PROTEIN; POLY I-C;
POLYCHLORINATED-BIPHENYLS; SPECTRUM DISORDERS; ANGELMAN-SYNDROME
AB Symposium 5 focused on research approaches that are aimed at understanding common patterns of immunological and neurological dysfunction contributing to neurodevelopmental disorders such as autism and ADHD. The session focused on genetic, epigenetic, and environmental factors that might act in concert to influence autism risk, severity and co-morbidities, and immunological and neurobiological targets as etiologic contributors. The immune system of children at risk of autism may be therefore especially susceptible to psychological stressors, exposure to chemical triggers, and infectious agents. Identifying early biomarkers of risk provides tangible approaches toward designing studies in animals and humans that yield a better understanding of environmental risk factors, and can help identify rational intervention strategies to mitigate these risks. (C) 2008 Elsevier Inc. All rights reserved.
C1 [Pessah, Isaac N.] Univ Calif Davis, Sch Vet Med, Dept Mol Biosci, Davis, CA 95616 USA.
[Pessah, Isaac N.; Van De Water, Judy; Berman, Robert F.] Univ Calif Davis, NIEHS EPA Ctr Childrens Environm Hlth, Davis, CA 95616 USA.
[Seegal, Richard F.] Wadsworth Ctr, New York State Dept Hlth, Albany, NY USA.
[Lein, Pamela J.] Swiss Fed Inst Technol, Zurich, Switzerland.
[LaSalle, Janine] Oregon Hlth & Sci Univ, CROET, Portland, OR 97201 USA.
[Yee, Benjamin K.; Van De Water, Judy; Berman, Robert F.] Univ Calif Davis, Sch Med, Davis, CA 95616 USA.
RP Pessah, IN (reprint author), Univ Calif Davis, Sch Vet Med, Dept Mol Biosci, 1 Shields Ave, Davis, CA 95616 USA.
EM inpessah@ucdavis.edu
RI LaSalle, Janine/A-4643-2008
OI LaSalle, Janine/0000-0002-3480-2031
CR Aman MG, 2000, J AUTISM DEV DISORD, V30, P451, DOI 10.1023/A:1005559725475
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 210
TC 18
Z9 18
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0161-813X
EI 1872-9711
J9 NEUROTOXICOLOGY
JI Neurotoxicology
PD MAY
PY 2008
VL 29
IS 3
SI SI
BP 532
EP 545
DI 10.1016/j.neuro.2008.02.006
PG 14
WC Neurosciences; Pharmacology & Pharmacy; Toxicology
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Toxicology
GA 317MV
UT WOS:000257025200021
PM 18394707
ER
PT J
AU Hertz-Picciotto, I
Green, P
Delwiche, L
Pessah, I
Hansen, R
AF Hertz-Picciotto, Irva
Green, Peter
Delwiche, Lora
Pessah, Isaac
Hansen, Robin
TI Role of metal exposures in autism
SO NEUROTOXICOLOGY AND TERATOLOGY
LA English
DT Meeting Abstract
CT 32nd Annual Meeting of the Neurobehavioral-Teratology-Society/48th
Annual Meeting of the Teratology-Society/21st Annual Meeting of the
Organization of Teratology Information Specialists
CY JUN 28-JUL 02, 2008
CL Monterey, CA
SP Neurobehav Teratol Soc, Teratol Soc
NR 0
TC 0
Z9 0
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0892-0362
J9 NEUROTOXICOL TERATOL
JI Neurotoxicol. Teratol.
PD MAY-JUN
PY 2008
VL 30
IS 3
MA NBTS16
BP 247
EP 248
DI 10.1016/j.ntt.2008.03.019
PG 2
WC Neurosciences; Toxicology
SC Neurosciences & Neurology; Toxicology
GA 321KH
UT WOS:000257303100027
ER
PT J
AU Rani, F
Murray, ML
Byrne, PJ
Wong, ICK
AF Rani, Fariz
Murray, Macey L.
Byrne, Patrick J.
Wong, Ian C. K.
TI Epidemiologic features of antipsychotic prescribing to children and
adolescents in primary care in the United Kingdom
SO PEDIATRICS
LA English
DT Article
DE drug utilization; pharmacoepidemiology; pediatric; antipsychotics
ID PRACTICE RESEARCH DATABASE; PSYCHOTROPIC MEDICATIONS; HALOPERIDOL;
DISORDERS; SCHIZOPHRENIA; PIMOZIDE; PLACEBO; AUTISM; TRENDS; DRUGS
AB OBJECTIVE. The goal was to investigate the epidemiologic features of antipsychotic prescribing to children and adolescents in general practice in the United Kingdom.
METHODS. A total of 384 participating general practices from the United Kingdom General Practice Research Database were used to identify patients 0 to 18 years of age who were prescribed >= 1 antipsychotic medication between January 1, 1992, and December 31, 2005. Annual age-specific prevalences and incidences of antipsychotic prescribing were calculated.
RESULTS. The overall prevalence of use of all antipsychotics increased from 1992 ( 0.39 users per 1000 patient-years) to 2005 ( 0.77 users per 1000 patient-years). The prescribing prevalence for patients 7 to 12 years of age almost tripled between 1992 ( 0.23 users per 1000 patient-years) and 2005 ( 0.61 users per 1000 patient-years). Atypical antipsychotic prescribing increased 60-fold from 1994 ( 0.01 users per 1000 patient-years) to 2005 ( 0.61 users per 1000 patient-years). However, typical antipsychotic prescribing decreased significantly from 2000 ( 0.44 users per 1000 patient-years) to 2005 ( 0.18 users per 1000 patient-years). The incidences for typical and atypical antipsychotics showed trends similar to those of the respective prevalences. However, the overall incidence ( number of new starters) for all antipsychotics was relatively stable between 1992 and 2005, which suggests that patients remain on treatment longer.
CONCLUSIONS. The overall prevalence of antipsychotics almost doubled between 1992 and 2005; however, the rate of increase was much lower than the reported figures in the United States. The prescribing of atypical antipsychotic drugs has increased despite the lack of conclusive evidence showing their superiority over older conventional antipsychotics. Additional investigation is required to evaluate their efficacy and safety in children and adolescents.
C1 [Rani, Fariz; Murray, Macey L.; Wong, Ian C. K.] Univ London, Sch Pharm, Ctr Pediat Pharm Res, London WC1N 1AX, England.
[Rani, Fariz; Murray, Macey L.; Wong, Ian C. K.] UCL, Inst Child Hlth, London, England.
[Murray, Macey L.] TEDDY, S London, England.
[Byrne, Patrick J.] Bethlem Royal & Maudsley Hosp, Dept Adolescent Psychiat, S London, England.
[Byrne, Patrick J.] Maudsley Natl Hlth Serv Trust, Beckenham, Kent, England.
RP Wong, ICK (reprint author), Univ London, Sch Pharm, Ctr Pediat Pharm Res, 29-39 Brunswick Sq, London WC1N 1AX, England.
EM ian.wong@pharmacy.ac.uk
RI Rani, Fariz /B-4331-2009; Wong, Ian/B-8141-2011; Research Datalink,
Clinical Practice/H-2477-2013
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Wood L, 2004, DRUG SAFETY, V27, P871, DOI 10.2165/00002018-200427120-00004
NR 30
TC 78
Z9 80
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD MAY
PY 2008
VL 121
IS 5
BP 1002
EP 1009
DI 10.1542/peds.2007-2008
PG 8
WC Pediatrics
SC Pediatrics
GA 295VL
UT WOS:000255501900018
PM 18450906
ER
PT J
AU Daniels, JL
Forssen, U
Hultman, CM
Cnattingius, S
Savitz, DA
Feychting, M
Sparen, P
AF Daniels, Julie L.
Forssen, Ulla
Hultman, Christina M.
Cnattingius, Sven
Savitz, David A.
Feychting, Maria
Sparen, Par
TI Parental psychiatric disorders associated with autism spectrum disorders
in the offspring
SO PEDIATRICS
LA English
DT Article
DE autism; psychiatric disorders; schizophrenia; depression; epidemiology;
registry
ID FAMILY HISTORY; RISK-FACTORS; GENERAL-POPULATION; INFANTILE-AUTISM;
PHENOTYPE; TRAITS; EPIDEMIOLOGY; AGGREGATION; INDIVIDUALS; TWIN
AB OBJECTIVE. Autism is a developmental disorder defined by impaired social interaction, communication, and behavior. Causes and correlates of autism are largely unknown, but elevated frequencies of psychiatric disorders and distinct personality traits have been reported among the family members of individuals with autism. Linkage of data from Swedish registries was used to investigate whether hospitalization for psychiatric conditions was higher among parents of children with autism compared with control subjects.
METHODS. Data sources included the Swedish Medical Birth Register (child's birth), the Swedish Multi-Generation Register (linking parents to children), and Swedish Hospital Discharge Register (hospitalization records). Children born between 1977 and 2003 who had a hospitalization record indicating autism before 10 years of age (n = 1227) were matched with 30 693 control subjects from the Swedish Medical Birth Register by gender, year of birth, and hospital. Parent diagnoses were based on an inpatient hospital diagnostic evaluation and included schizophrenia, other nonaffective psychoses, affective disorders, neurotic and personality disorders and other nonpsychotic disorders, alcohol and drug addiction and abuse, and autism. Odds ratios and 95% confidence intervals were estimated by using conditional logistic regression, adjusted for child's age, gender, hospital of birth, parents' age, country of birth and socioeconomic status, and diagnosis of a mental disorder in the other parent.
RESULTS. Parents of children with autism were more likely to have been hospitalized for a mental disorder than parents of control subjects. Schizophrenia was more common among case mothers and fathers compared with respective control parents. Depression and personality disorders were more common among case mothers but not fathers.
CONCLUSIONS. This large population study supports the potential for familial aggregation of psychiatric conditions that may provide leads for future investigations of heritable forms of autism.
C1 [Daniels, Julie L.] Univ N Carolina, Dept Epidemiol, Chapel Hill, NC 27599 USA.
[Daniels, Julie L.] Univ N Carolina, Dept Maternal & Child Hlth, Chapel Hill, NC 27599 USA.
[Forssen, Ulla] GlaxoSmithKline R&D, Worldwide Epidemiol, Collegeville, PA USA.
[Hultman, Christina M.] Karolinska Inst, Dept Med Epidemiol & Biostat, Stockholm, Sweden.
[Feychting, Maria] Karolinska Inst, Inst Environm Med, S-10401 Stockholm, Sweden.
[Savitz, David A.] Mt Sinai Sch Med, Dept Community & Prevent Med, New York, NY USA.
[Savitz, David A.] Univ N Carolina, Inst Epidemiol Biostat & Prevent, Chapel Hill, NC 27599 USA.
RP Daniels, JL (reprint author), Univ N Carolina, Dept Epidemiol, CB 7435, Chapel Hill, NC 27599 USA.
EM juliedaniels@unc.edu
RI Reis, Aline/G-9573-2012
CR American Psychiatric Association Task Force on DSM-IV, 2000, DIAGN STAT MAN MENT, V4th
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NR 29
TC 66
Z9 67
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD MAY
PY 2008
VL 121
IS 5
BP E1357
EP E1362
DI 10.1542/peds.2007-2296
PG 6
WC Pediatrics
SC Pediatrics
GA 295VL
UT WOS:000255501900085
PM 18450879
ER
PT J
AU Morton, JE
Campbell, JM
AF Morton, Jane E.
Campbell, Jonathan M.
TI Information source affects peers' initial attitudes toward autism
SO RESEARCH IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism; attitudes; persuasion; source; inclusion
ID CHILDRENS ATTITUDES; BEHAVIORAL INTENTIONS; PERCEPTIONS; STUDENTS
AB Authors examined the effects of information source on peers' cognitive and behavioral attitudes toward an unfamiliar child with autism. Children (N = 296; M age = 10.21 years) received information about an unfamiliar child with autism from one of the following sources: (a) videotape, (b) teacher, (c) hypothetical mother, (d) hypothetical father, or (e) hypothetical "doctor." Interactive effects between source, and sex and grade were found for cognitive and behavioral attitudes. Fifth-graders reported more favorable cognitive and behavioral attitudes when information was provided by extra-familial sources (i.e., "doctor") versus parent sources. Mother yielded more persuasive effects on behavioral attitudes for third-graders versus fifth-graders. Attitudes toward autism differ depending on who provides information about the disability. Persuasion theory appears useful to guide evaluation of educational interventions to improve attitudes towards autism. Implications of the findings, study limitations, and recommendations for future research are discussed. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Morton, Jane E.; Campbell, Jonathan M.] Univ Georgia, Dept Educ Psychol, Athens, GA 30602 USA.
RP Campbell, JM (reprint author), Univ Georgia, Dept Educ Psychol, 325-J Aderhold Hall, Athens, GA 30602 USA.
EM jmcmpbll@uga.edu
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NR 26
TC 14
Z9 14
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0891-4222
J9 RES DEV DISABIL
JI Res. Dev. Disabil.
PD MAY-JUN
PY 2008
VL 29
IS 3
BP 189
EP 201
DI 10.1016/j.ridd.2007.02.006
PG 13
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 313LU
UT WOS:000256742900001
PM 17391914
ER
PT J
AU Lee, LC
Harrington, RA
Chang, JJ
Connors, SL
AF Lee, Li-Ching
Harrington, Rebecca A.
Chang, Jen Jen
Connors, Susan L.
TI Increased risk of injury in children with developmental disabilities
SO RESEARCH IN DEVELOPMENTAL DISABILITIES
LA English
DT Article; Proceedings Paper
CT 2nd North American Congress of Epidemiology
CY JUN 21-24, 2006
CL Seattle, WA
DE injury; developmental disabilities; autism spectrum disorders; ADHD
ID NONFATAL INJURIES; UNINTENTIONAL INJURY; US CHILDREN; AUTISM; DISORDER;
CHILDHOOD
AB The objective of this study was to examine injury risk in children with autism, ADD/ADHD, learning disability, psychopathology, or other medical conditions. Children aged 3-5 years who participated in the National Survey of Children's Health were included. Six study groups were analyzed in this report: autism (n = 82), ADD/ADHD (n = 191), learning disability (n = 307), psychopathology (n = 210), other medical conditions (it = 1802), and unaffected controls (n = 13,398). The weighted prevalence of injury in each group was 24.2% (autism), 26.5% (ADD/ADHD), 9.3% (learning disability), 20.5% (psychopathology), 14.6% (other medical conditions), and 11.9% (unaffected controls). Compared to unaffected controls, the risk of injury was 2.15 (95% confidence interval (CI): 1.00-4.60), 2.74 (95% CI: 1.63-4.59), 2.06 (95% CI: 1.24-3.42), and 1.26 (95% CI: 1.00-1.58) in children with autism, ADD/ADHD, psychopathology, and other medical conditions, respectively, after adjusting for child sex, child age, number of children in the household, child race, and family poverty level. Children with autism, ADD/ADHD, and other psychopathology were about 2-3 times more likely to experience an injury that needs medical attention than unaffected controls. Future studies need to clarify the extent to which injuries in young children with autism, ADD/ADHD, and psychopathology are related to core symptoms, comorbid conditions, associated behaviors, or unintentional injuries due to lack of additional supervision from caregivers. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Lee, Li-Ching; Harrington, Rebecca A.] Johns Hopkins Univ, Bloomberg Sch Publ Hlth, Dept Epidemiol, Baltimore, MD 21205 USA.
[Chang, Jen Jen] St Louis Univ, Sch Publ Hlth, Dept Community Hlth Epidemiol, St Louis, MO 63103 USA.
[Connors, Susan L.] Kennedy Krieger inst, Baltimore, MD USA.
RP Lee, LC (reprint author), Johns Hopkins Univ, Bloomberg Sch Publ Hlth, Dept Epidemiol, 615 N Wolfe St,Room E6032, Baltimore, MD 21205 USA.
EM llee2@jhsph.edu
CR Bennet Murphy L. M., 2001, J PEDIATR HEALTH CAR, V15, P194
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NR 24
TC 36
Z9 36
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0891-4222
J9 RES DEV DISABIL
JI Res. Dev. Disabil.
PD MAY-JUN
PY 2008
VL 29
IS 3
BP 247
EP 255
DI 10.1016/j.ridd.2007.05.002
PG 9
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 313LU
UT WOS:000256742900005
PM 17582739
ER
PT J
AU Aussilloux, C
Baghdadli, A
AF Aussilloux, C.
Baghdadli, A.
TI Asperger syndrome: Evolution of the concept and current clinical data
SO REVUE NEUROLOGIQUE
LA French
DT Review
DE Asperger syndrome; autism; high functioning autism; pervasive
developmental disorders; autistic spectrum disorders
ID HIGH-FUNCTIONING AUTISM; PERVASIVE DEVELOPMENTAL DISORDER; CHILDREN;
CLASSIFICATION; INTERVENTION; DISABILITIES; PSYCHOPATHY; COMORBIDITY;
ADOLESCENTS; CHILDHOOD
AB Although Asperger syndrome is described by international classifications as a category of pervasive developmental disorder (PDD), its validity as a specific entity distinct from autistic disorders remains controversial. The syndrome, first described by Hans Asperger, could not be distinguished from high functioning autism (onset, symptoms, outcome...). However, international classifications propose a distinction between the two syndromes based on a delayed onset, the absence of speech delay, the presence of motor disorders and a better outcome in Asperger syndrome. This categorical differentiation is not confirmed by current studies and in the absence of biological markers, no clinical, neuropsychological or epidemiological criteria makes it possible to distinguish high functioning autism from Asperger syndrome. From a clinical perspective, it is nevertheless of interest to isolate Aspeger syndrome from other autistic disorders to propose specific assessment and therapy. (C) 2008 Elsevier Masson SAS. Tous droits reserves.
C1 [Aussilloux, C.; Baghdadli, A.] CHU Montpellier, Serv Med Psychol Enfants & Adolescents Peyre Plan, F-34295 Montpellier 5, France.
RP Aussilloux, C (reprint author), CHU Montpellier, Serv Med Psychol Enfants & Adolescents Peyre Plan, 291 Ave Doyen Gastron Giraud, F-34295 Montpellier 5, France.
EM c-aussilloux@chu-montpellier.fr
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NR 51
TC 2
Z9 2
PU MASSON EDITEUR
PI MOULINEAUX CEDEX 9
PA 21 STREET CAMILLE DESMOULINS, ISSY, 92789 MOULINEAUX CEDEX 9, FRANCE
SN 0035-3787
J9 REV NEUROL-FRANCE
JI Rev. Neurol.
PD MAY
PY 2008
VL 164
IS 5
BP 406
EP 413
DI 10.1016/j.neurol.2008.01.009
PG 8
WC Clinical Neurology
SC Neurosciences & Neurology
GA 326NP
UT WOS:000257666500002
PM 18555872
ER
PT J
AU Ramirez, AEG
Martinez, AD
Diaz-Anzaldua, A
AF Gonzalez Ramirez, Adriana Estrella
Diaz Martinez, Alejandro
Diaz-Anzaldua, Adriana
TI Epigenetics and twin studies in psychiatric domains
SO SALUD MENTAL
LA Spanish
DT Article
DE twins; epigenetics; heritability; concordonce; mental disorders
ID HYPERACTIVITY DISORDER SYMPTOMS; MONOZYGOTIC TWINS; SCHIZOPHRENIA;
EXPRESSION; DEPRESSION; DISEASE; AUTISM; HERITABILITY; PATTERNS; REELIN
AB The sequence of the human genome integrates the keystone of our life. Part of it is transcribed to RNA, which in turn provides the information required by our cells to produce proteins. Discoveries in the genetics field have been essential to medicine and have been used to develop strategies to modify, prevent and propose new therapeutic approaches for human diseases.
In the 19th Century, Gregor Johann Mendel developed a theoretical model which was able to predict in an accurate way, hereditary mechanisms; indeed, his laws still explain the basis of human inheritance. Almost ninety years later, James Watson and Francis Crick announced their double-helix model of the DNA molecule. Then, positional cloning and the polymerase chain reaction (PCR) were introduced; more recently, almost 99% of the sequence of our genome was made public. The current period of time is known as the post-genomic era, due to the fact that researchers are not only obtaining the complete sequences of thousands of genomes, but are also searching for clues that may help understand the mechanisms that affect gene activation and deactivation, in which epigenetic factors are also involved.
In medical domains, twins constitute a suitable group to study inherited disorders. Dizygotic or fraternal twins ore produced by different egg and sperm cells, and even when these two fertilization events occur simultaneously, dizygotic twins share approximately the some percentage of genetic material than any pair of siblings, that is, around 50%. Some authors have suggested that the tendency fat spontaneous dizygotic twinning could be attributed to a double ovulation which is genetically determined in an autosomal dominant manner.
Monozygotic, as opposed to dizygotic twins, are produced by a single zygote whose cells are dissociated and originate two independent organisms; approximately a third of monozygotic twins are separated before the 5(th) day after fertilization, and the rest between the 5(th) and the 15(th) day. Most monozygotic twins are very similar; nevertheless, some few exceptions prove that in fact they actually do not have to be identical.
Relatives of a person with a mental disorder tend to share traits associated with this disease, especially if the patient and the relative are monozygotic twins. However, important differences may be detected even between each pair of identical twins.
Parameters such as concordance and heritability have shown that a monozygotic twin can develop an inherited disorder while his or her co-twin,will always be disease-free. In addition to differences in susceptibility to inherited diseases, this kind of twins can display dissimilarities in somatic cell mutations (more overtly noticeable when ageing), their set of antibodies and T cell receptors, their number of mitochondrial DNA molecules, and chromosome X-inactivation patterns in women, all of which are the main subject of many ongoing studies. A recent report,shows that from 160 monozygotic twin pairs who were 3 to 74 years old, epigenetic patterns were,identical early I in life, but differences were more obvious at older ages, especially if twins were raised apart or if they had different medical history. Medical conditions, but also environmental factors such as pregnancy tobacco exposure, physical activity, and diet could contribute to differences in epigenetic patterns. It has been shown that epigenetic modifications (or epi-mutations) are more frequent than the ones that modify DNA sequence,e, so they are part of the fundamental causes of biological diversity, and they show how environment can modulate,gene expression and contribute to our phenotype.
Even when twin studies ore sometimes considered purely genetic they also give information about the influence of environmental c factors. However, it is important to consider with caution the results from this type of studies. Heritability estimates are not unchangeable fact. They depend on the sample being analyzed, the genes involved in the specific sample, the characteristics of the environmental factors which members of this group were exposed to, and the precise moment the study was clone.
Epigenetics refers to changes that do not alter the DNA sequence but affect gene function due to chemical modifications which mainly occur in DNA cytosines and in chromartin-related histones. Epigenetic processes are covalent modifications which include the addition of functional groups (methyl, acetyl, phosphate, etc.) or proteins (ubiquitin, SUMO, etc.) to the DNA molecule or to associated proteins. These modifications contribute to the activation or inhibition of transcription, which leads to changes in messenger ARN expression that con ultimately influence the onset of disease.
Pseudogenes are still being excluded while new genes are being confirmed in our genome sequence, but the current estimates indicate that each one of our nucleated cells contains almost 22000 genes (excluding mitochondrial DNA) which encode for polypeptides and more than 4,000 whose final product is RNA.
Gene expression is partially controlled by DNA coiling around globular proteins called histones, which constitute a structure known os chromatin, a DNA-protein complex that represents the packaging of 3.25 billion base pairs of our genetic information. Physical and chemical chromatin modifications can also affect gene expression by changing DNA-protein interactions; in general terms, genes are inhibited when chromatin is packed and they are active when it is free. These dynamic states are controlled by epigenetic reversible modifications on DNA methylation or by changes in histones. It has been shown that subtle epigenetic differences between any two human beings are associated with dissimilar final chromatin remodeling, as well as expression/repression of genes.
In order to explain how DNA methylation controls transcription, two mechanisms have been proposed. First, the presence of a methyl group on specific sites could simply prevent transcription factors from binding the DNA. Second, some proteins contain a specific domain that recognizes and binds methylated DNA, and works as a transcription repressor.
Regarding histone modifications, acetyl group addition occurs on the amino acid lysine; there are reports that indicate that almost 14% of lysines are susceptible to acetylation. On the other hand, methyl groups bind arginines and lysines. Different combinations of covalent modifications lead to what is known as the histone code. This review gathers information about the possible involvement of epigenetics in some mental disorders and attempts to explain how it can account for differences observed between (identical,, twins. Some examples of a plausible link between epigenetic modifications and menial disorders are discussed.
Regarding bipolar disorder, valproate efficacy has been linked to its inhibitory activity, suggesting that an epigenetic modification (that represses transcription of a gene) may play a role in the onset of some of the symptoms of the disease. Perhaps one of the most studied aspects regarding schizophrenia is the hypermethylation of Reelin (RELN) and GAD(67)genes in the prefrontal cortex, due to the over-expression of DNMT1 (DNA methyl-trasferase 1) in GABAergic cortical interneurons. Reelin is an important protein during prenatal development of the Central Nervous System and it is relevant for the expression of cortical pyramidal neurons in the adult brain; it regulates the migration of neurons during brain development and it is essential for the correct organization and plasticity of the cerebral cortex. GAD(67) is one of two molecular forms of GABA synthetizing enzymes, being GABA the main inhibitory neurotransmitter in humans.
Hypermethylation of these genes leads to transcription repression either by interference of transcription promoters by methyl groups or by MBD protein binding. The final outcome is a low production of Reelin and GAD(67), a typical feature found in brains of shchizophrenic patients.
Epigenetic analyses of autism are sometimes with Rett and Angelman's syndromes. The latter are pathological conditions that share clinical features with autism, such as development retardation, language impairments and stereotypic behaviors. Rett syndrome is caused by mutations at the MECF2gene, and Angelman's syndrome by a maternal deficiency on chromosome 15q11-q13 region, impaired methylation of DNA or maternal mutation of the ubiquitin-protein ligase E3A (UBE3A). However, there have also been reports of patients with Angelmon's syndrome or autism who have mutations at MECP2.
In conclusion, the onset of mental disorders is influenced both by environmental and genetic factors. Concordance and heritability estimates are just two of the many ways of analyzing differences between monozygotic twins. Part of the reported disparity between monozygotic twins could be caused by epigenetic factors. Epigenetic modifications could partially explain the etiology of mental disorders.
C1 [Gonzalez Ramirez, Adriana Estrella; Diaz-Anzaldua, Adriana] Inst Nacl Psiquiatria Ramon Fuente, Dept Genet, Tlalpan 14370, DF, Mexico.
[Diaz Martinez, Alejandro] Univ Nacl Autonoma Mexico, Fac Med, Dept Psicol Med Psiquiatria & Salud Mental, Mexico City, DF, Mexico.
RP Ramirez, AEG (reprint author), Inst Nacl Psiquiatria Ramon Fuente, Dept Genet, Calz Mexico Xochimilco 101, Tlalpan 14370, DF, Mexico.
EM adiaza2@imp.edu.mx
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NR 38
TC 0
Z9 0
PU INST MEX PSIQUIATRIA
PI MEXICO CITY
PA CALZ MEXICO-XOCHIMILCO #101, MEXICO CITY 22 DF, MEXICO
SN 0185-3325
J9 SALUD MENT
JI Salud Ment.
PD MAY-JUN
PY 2008
VL 31
IS 3
BP 229
EP 237
PG 9
WC Psychiatry
SC Psychiatry
GA 325VH
UT WOS:000257616000009
ER
PT J
AU Olie, JP
AF Olie, Jean-Pierre
TI Treating a psychotic syndrome in 2007
SO THERAPIE
LA French
DT Article; Proceedings Paper
CT 12th Annual Seminar of Experimental and Clinical Pharmacology
CY DEC 06-08, 2007
CL Paris, FRANCE
DE psychosis; antipsychotics; compliance; rehabilitation; schizophrenia
ID SUICIDE-PREVENTION TRIAL; ATYPICAL ANTIPSYCHOTICS; 1ST-EPISODE
PSYCHOSIS; CHRONIC-SCHIZOPHRENIA; GLUCOSE-METABOLISM; RISPERIDONE;
OLANZAPINE; EFFICACY; METAANALYSIS; HALOPERIDOL
AB It is well established that we have to consider 3 patterns of psychotic symptoms: positive ( hallucinations, delusion...), negative ( affective flatness, autism...) symptoms and disorganization ( ambivalence, incoherence...).
In the past, ECT (electroconvulsivotherapy) was the first e. ective treatment in psychiatry. Conventionnal neuroleptics have been determinant in the significant evolution of care to psychotic patients. ECT use is now better defined in terms of practise and indications.
Assessment of new antipsychotic medications is mainly focused on efficacy on positive symptoms. Atypical antipsychotics brought improvement in treating psychotic syndromes: they are better tolerated and more effective on the whole spectrum of psychotic syndromes including emotional symptoms than conventional neuroleptics.
Atypical antipsychotics have raised questions about metabolic and cardiac risks. Compliance remains a cause of failure of many antipsychotic treatments.
Treating a psychotic syndrome requires complementary strategies to medications: conditions of assistance and rehabilitation, choice of psychotherapy. Research program are currently orientated towards:
-identification of prepsychotic symptoms and endophenotypes which can be treatment targets;
-assessment of putative therapeutical means such a brain stimulation.
C1 Univ Paris 05, Hop St Anne, INSERM, Fac Med Paris Descartes,Serv Hosp Univ,U894 7, F-75014 Paris, France.
RP Olie, JP (reprint author), Univ Paris 05, Hop St Anne, INSERM, Fac Med Paris Descartes,Serv Hosp Univ,U894 7, 1 Rue Cabanis, F-75014 Paris, France.
EM jp.olie@ch-sainte-anne.fr
CR *AM PSYCH ASS, DSMIV DIAGN STAT MAN
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1992, ENCEPHALE, V18
NR 33
TC 0
Z9 0
PU EDP SCIENCES S A
PI LES ULIS CEDEX A
PA 17, AVE DU HOGGAR, PA COURTABOEUF, BP 112, F-91944 LES ULIS CEDEX A,
FRANCE
SN 0040-5957
J9 THERAPIE
JI Therapie
PD MAY-JUN
PY 2008
VL 63
IS 3
BP 165
EP 175
DI 10.2515/therapie:2008027
PG 11
WC Pharmacology & Pharmacy
SC Pharmacology & Pharmacy
GA 348TU
UT WOS:000259233800001
PM 18718208
ER
PT J
AU Kurt, O
Tekin-Iftar, E
AF Kurt, Onur
Tekin-Iftar, Elif
TI A Comparison of Constant Time Delay and Simultaneous Prompting Within
Embedded Instruction on Teaching Leisure Skills to Children With Autism
SO TOPICS IN EARLY CHILDHOOD SPECIAL EDUCATION
LA English
DT Article
DE applied behavior analysis; intervention strategies; single-subject
designs; experimental studies; research methodologies; autism spectrum
disorder (ASD); disability populations
ID GENERAL-EDUCATION CLASSES; DEVELOPMENTAL DELAYS; PRESCHOOLERS; STUDENTS
AB An adapted alternating-treatments design was used to compare the effectiveness and efficiency of constant time delay and simultaneous prompting procedures within an embedded instruction format on the acquisition of various leisure skills by four preschool students with autism. The results showed that both procedures were effective in promoting the acquisition of the skills and maintaining them over time by three students. Although the impacts of the procedures were evident for these three students, the results were not replicated With the fourth student. Furthermore, mixed results were obtained regarding the efficiency measures. However, the social validity findings of the study were positive overall. On the basis of an evaluation of the findings, implications and future research needs are discussed.
C1 [Kurt, Onur; Tekin-Iftar, Elif] Anadolu Univ, Engelliler Arastirma Enstitusu, TR-26470 Eskisehir, Turkey.
RP Tekin-Iftar, E (reprint author), Anadolu Univ, Engelliler Arastirma Enstitusu, TR-26470 Eskisehir, Turkey.
EM eltekin@anadolu.edu.tr
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NR 28
TC 20
Z9 20
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0271-1214
J9 TOP EARLY CHILD SPEC
JI Top. Early Child. Spec. Educ.
PD MAY
PY 2008
VL 28
IS 1
BP 53
EP 64
DI 10.1177/0271121408316046
PG 12
WC Education, Special
SC Education & Educational Research
GA 390LU
UT WOS:000262166600005
ER
PT J
AU Khanna, R
Madhavan, SS
AF Khanna, R.
Madhavan, S. S.
TI Trends in the prevalence of autism spectrum disorders and related health
care utilization and costs in a state Medicaid program
SO VALUE IN HEALTH
LA English
DT Meeting Abstract
C1 [Khanna, R.; Madhavan, S. S.] W Virginia Univ, Morgantown, WV 26506 USA.
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1098-3015
J9 VALUE HEALTH
JI Value Health
PD MAY-JUN
PY 2008
VL 11
IS 3
BP A137
EP A137
DI 10.1016/S1098-3015(10)70436-8
PG 1
WC Economics; Health Care Sciences & Services; Health Policy & Services
SC Business & Economics; Health Care Sciences & Services
GA 302CC
UT WOS:000255945400435
ER
PT J
AU Bardyshevskaya, MK
Trenina, MY
AF Bardyshevskaya, M. K.
Trenina, M. Yu.
TI Peculiarities of inclinations in children with affective instability
SO VOPROSY PSIKHOLOGII
LA Russian
DT Article
DE inclination disorders in children; affective instability; emotional
disorders; disruptions of attachment; adopted children; emotional
deprivation; psychological trauma
AB A study of peculiarities of inclinations and types of affective instability, peculiarities of attachments and personality identification in 61 children (40 boys and 21 girls) aged 3 to 5, 12 of them adopted is described. All the children were patients of a children's psychiatric hospital undergoing treatment for maladjustment in kindergarten, adoptive and native families. Their diagnoses included early autism, schizoaffective psychosis, the psychopathological syndrome of different origins, mental retardedness. Emotional and inclination disorders were studied by the method of systematic observation of behavior on the basis of the level organization of emotional regulation. The study has revealed that inclination disorders are caused by severe disruptions of attachment, emotional deprivation, violence, psychological traumas from the moment of birth. The authors describe types of affective instability and specific situations which provoke, form and aggravate inclination disorders in a child, also providing therapeutic recommendations for such children and their parents.
CR BARDYSHEVSKAYA MK, 2006, DEFEKTOLOGIYA, P6
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NR 21
TC 1
Z9 1
PU MEZHDUNARODNAYA KNIGA
PI MOSCOW
PA 39 DIMITROVA UL., 113095 MOSCOW, RUSSIA
SN 0042-8841
J9 VOP PSIKHOL+
JI Vopr. Psikhologii
PD MAY-JUN
PY 2008
IS 3
BP 35
EP +
PG 12
WC Psychology, Educational
SC Psychology
GA 328CD
UT WOS:000257774200004
ER
PT J
AU Obukhova, EY
Stroganova, TA
Grachiov, VV
AF Obukhova, E. Yu.
Stroganova, T. A.
Grachiov, V. V.
TI Attention disorders in children with peculiarities of development
SO VOPROSY PSIKHOLOGII
LA Russian
DT Article
DE the technique "the arbitrary attention test"; attention control
operations; performance functions; the attention deficit syndrome with
hyperactivity; children's autism
ID BRAIN POTENTIALS; AUTISM; CORTEX; MODEL
AB The paper contains findings from applying the technique "The arbitrary attention test" to assess effectiveness of basic attention control operations in pre-school and primary school children. Data from healthy children were compared with those from hyperactive children suffering from attention deficit and autistic children. The authors analyze mistakes made by the subjects (false alarms, failures to identify the signal), times of visual and auditory reactions and specificity of reactions of children from different groups. The study has revealed common and specific attention disorders in hyperactive and autistic children. Disorders common to the two syndromes are linked to general lowering of alertness. Specific to hyperactivity is high impulsiveness of reacting and partial deficit of the operation of interference control. Slowing of the simple sensory-motor reaction and specific difficulties of switching attention from the visual to the auditory modality are characteristic of autistic children.
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PU MEZHDUNARODNAYA KNIGA
PI MOSCOW
PA 39 DIMITROVA UL., 113095 MOSCOW, RUSSIA
SN 0042-8841
J9 VOP PSIKHOL+
JI Vopr. Psikhologii
PD MAY-JUN
PY 2008
IS 3
BP 61
EP +
PG 11
WC Psychology, Educational
SC Psychology
GA 328CD
UT WOS:000257774200007
ER
PT J
AU Bolliger, MF
Pei, J
Maxeiner, S
Boucard, AA
Grishin, NV
Sudhof, TC
AF Bolliger, Marc F.
Pei, Jimin
Maxeiner, Stephan
Boucard, Antony A.
Grishin, Nick V.
Sudhof, Thomas C.
TI Unusually rapid evolution of Neuroligin-4 in mice
SO PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF
AMERICA
LA English
DT Article
DE autism; cell-adhesion molecule; neurexin; synapse
ID CELL-ADHESION; INHIBITORY SYNAPSES; ALPHA-NEUREXINS; BETA-NEUREXINS;
PROTEIN; BINDING; AUTISM; MUTATION; COMPLEX; GENES
AB Neuroligins (NLs) are postsynaptic cell-adhesion molecules that are implicated in humans in autism spectrum disorders because the genes encoding NL3 and NL4 are mutated in rare cases of familial autism. NLs are highly conserved evolutionarily, except that no NL4 was detected in the currently available mouse genome sequence assemblies. We now demonstrate that mice express a distant NL4 variant that rapidly evolved from other mammalian NL4 genes and that exhibits sequence variations even between different mouse strains. Despite its divergence, mouse NL4 binds neurexins and is transported into dendritic spines, suggesting that the core properties of NLs are retained in this divergent NIL isoform. The selectively rapid evolution of NL4 in mice suggests that its function in the brain is under less stringent control than that of other NLs, shedding light on why its mutation in autism spectrum disorder patients is not lethal, but instead leads to a discrete developmental brain disorder.
C1 [Bolliger, Marc F.; Maxeiner, Stephan; Boucard, Antony A.; Sudhof, Thomas C.] Univ Texas SW Med Ctr Dallas, Dept Neurosci, Dallas, TX 75390 USA.
[Grishin, Nick V.] Univ Texas SW Med Ctr Dallas, Dept Biochem, Dallas, TX 75390 USA.
[Sudhof, Thomas C.] Univ Texas SW Med Ctr Dallas, Dept Mol Genet, Dallas, TX 75390 USA.
[Pei, Jimin; Grishin, Nick V.; Sudhof, Thomas C.] Univ Texas SW Med Ctr Dallas, Howard Hughes Med Inst, Dallas, TX 75390 USA.
RP Sudhof, TC (reprint author), Univ Texas SW Med Ctr Dallas, Dept Neurosci, 6000 Harry Hines Blvd, Dallas, TX 75390 USA.
EM thomas.sudhof@utsouthwestern.edu
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NR 27
TC 33
Z9 38
PU NATL ACAD SCIENCES
PI WASHINGTON
PA 2101 CONSTITUTION AVE NW, WASHINGTON, DC 20418 USA
SN 0027-8424
J9 P NATL ACAD SCI USA
JI Proc. Natl. Acad. Sci. U. S. A.
PD APR 29
PY 2008
VL 105
IS 17
BP 6421
EP 6426
DI 10.1073/pnas.0801383105
PG 6
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 296HM
UT WOS:000255534100038
PM 18434543
ER
PT J
AU Reed, MC
Thomas, RL
Pavisic, J
James, SJ
Ulrich, CM
Nijhout, HF
AF Reed, Michael C.
Thomas, Rachel L.
Pavisic, Jovana
James, S. Jill
Ulrich, Cornelia M.
Nijhout, H. Frederik
TI A mathematical model of glutathione metabolism
SO THEORETICAL BIOLOGY AND MEDICAL MODELLING
LA English
DT Article
ID GAMMA-GLUTAMYLCYSTEINE SYNTHETASE; CYSTATHIONINE BETA-SYNTHASE;
RAT-LIVER; METHIONINE METABOLISM; HEPATIC GLUTATHIONE; OXIDATIVE STRESS;
DOWN-SYNDROME; HOMOCYSTEINE METABOLISM; S-ADENOSYLMETHIONINE;
PROTEIN-TURNOVER
AB Background: Glutathione (GSH) plays an important role in anti-oxidant defense and detoxification reactions. It is primarily synthesized in the liver by the transsulfuration pathway and exported to provide precursors for in situ GSH synthesis by other tissues. Deficits in glutathione have been implicated in aging and a host of diseases including Alzheimer's disease, Parkinson's disease, cardiovascular disease, cancer, Down syndrome and autism.
Approach: We explore the properties of glutathione metabolism in the liver by experimenting with a mathematical model of one-carbon metabolism, the transsulfuration pathway, and glutathione synthesis, transport, and breakdown. The model is based on known properties of the enzymes and the regulation of those enzymes by oxidative stress. We explore the half-life of glutathione, the regulation of glutathione synthesis, and its sensitivity to fluctuations in amino acid input. We use the model to simulate the metabolic profiles previously observed in Down syndrome and autism and compare the model results to clinical data.
Conclusion: We show that the glutathione pools in hepatic cells and in the blood are quite insensitive to fluctuations in amino acid input and offer an explanation based on model predictions. In contrast, we show that hepatic glutathione pools are highly sensitive to the level of oxidative stress. The model shows that overexpression of genes on chromosome 21 and an increase in oxidative stress can explain the metabolic profile of Down syndrome. The model also correctly simulates the metabolic profile of autism when oxidative stress is substantially increased and the adenosine concentration is raised. Finally, we discuss how individual variation arises and its consequences for one-carbon and glutathione metabolism.
C1 [Reed, Michael C.; Thomas, Rachel L.; Pavisic, Jovana] Duke Univ, Dept Math, Durham, NC 27708 USA.
[Pavisic, Jovana; Nijhout, H. Frederik] Duke Univ, Dept Biol, Durham, NC 27708 USA.
[James, S. Jill] Univ Arkansas Med Sci Hosp, Dept Pediat, Little Rock, AR 72205 USA.
[Ulrich, Cornelia M.] Fred Hutchinson Canc Res Ctr, Seattle, WA 98109 USA.
RP Reed, MC (reprint author), Duke Univ, Dept Math, Durham, NC 27708 USA.
EM reed@math.duke.edu; rachel@math.duke.edu; jovana.pavisic@duke.edu;
JamesJill@uams.edu; nulrich@fhcrc.org; hfn@duke.edu
FU NIH [RO1 CA105437]; NSF [DMS-0616710]
FX The authors benefited greatly from the advice of Jesse Gregory, III,
University of Florida, Barry Shane, University of California, Berkeley,
and Marian Neuhouser, Fred Hutchinson Cancer Research Center. This
research was supported by NIH grant RO1 CA105437 (C.M.U.) and NSF grant
DMS-0616710 (M.C.R.).
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NR 76
TC 44
Z9 45
PU BIOMED CENTRAL LTD
PI LONDON
PA 236 GRAYS INN RD, FLOOR 6, LONDON WC1X 8HL, ENGLAND
SN 1742-4682
J9 THEOR BIOL MED MODEL
JI Theor. Biol. Med. Model.
PD APR 28
PY 2008
VL 5
AR 8
DI 10.1186/1742-4682-5-8
PG 16
WC Mathematical & Computational Biology
SC Mathematical & Computational Biology
GA 439AP
UT WOS:000265598900001
PM 18442411
ER
PT J
AU Fajardo, C
Escobar, MI
Buritica, E
Arteaga, G
Umbarila, J
Casanova, MF
Pimienta, H
AF Fajardo, C.
Escobar, M. I.
Buritica, E.
Arteaga, G.
Umbarila, J.
Casanova, M. F.
Pimienta, H.
TI Von Economo neurons are present in the dorsolateral (dysgranular)
prefrontal cortex of humans
SO NEUROSCIENCE LETTERS
LA English
DT Article
DE Von Economo neurons; spindle cells; anterior cingulate gyros;
frontoinsular cortex; dorsolateral prefrontal cortex
ID CINGULATE CORTEX; AREAS 9; CYTOARCHITECTONIC DEFINITION; SPINDLE
NEURONS; BRAIN; PATHOLOGY; AUTISM; UNIQUE; APES
AB Von Economo neurons (VENs), also known as spindle cells, have been described in layer V of the anterior cingulate (BA 24) and frontoinsular cortex (FI) of humans and other great apes. In the present study we used immunohistochemistry against two specific neuronal markers (NeuN and MAP2) in order to establish the presence of these cell types in Brodmann area 9 (BA 9) of the human prefrontal cortex. We evaluated tissue samples of eight human postmortem brains (age range 26-50) from BAs 9, 24, 4, 46, 45, 10 and 17. We identified a group of cells with similar morphology to that previously described for VENs in all specimens of BA 9 examined, albeit less frequently than in BA 24. This is the first description of this cell type in a human brain area with well developed granular layers (BA 9). (c) 2008 Elsevier Ireland Ltd. All rights reserved.
C1 [Fajardo, C.; Escobar, M. I.; Buritica, E.; Arteaga, G.; Umbarila, J.; Pimienta, H.] Univ Valle, Fac Salud, Ctr Estudios Cerebrales, Cali, Valle, Colombia.
[Casanova, M. F.] Univ Louisville, Dept Psychiat, Louisville, KY 40292 USA.
RP Pimienta, H (reprint author), Univ Valle, Fac Salud, Ctr Estudios Cerebrales, Calle 4B 36-00, Cali, Valle, Colombia.
EM hernpim@yahoo.com
RI Pimienta Jimenez, Hernan Jose/C-3147-2011; Buritica, Efrain/C-3315-2011;
Escobar, Martha Isabel/C-3310-2011; Escobar, Martha/E-4288-2015
OI Escobar, Martha/0000-0002-0036-7473
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NR 21
TC 31
Z9 31
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0304-3940
J9 NEUROSCI LETT
JI Neurosci. Lett.
PD APR 25
PY 2008
VL 435
IS 3
BP 215
EP 218
DI 10.1016/j.neulet.2008.02.048
PG 4
WC Neurosciences
SC Neurosciences & Neurology
GA 296EW
UT WOS:000255527300009
PM 18355958
ER
PT J
AU Tommerdahl, M
Tannan, V
Holden, JK
Baranek, GT
AF Tommerdahl, Mark
Tannan, Vinay
Holden, Jameson K.
Baranek, Grace T.
TI Absence of stimulus-driven synchronization effects on sensory perception
in autism: Evidence for local underconnectivity?
SO BEHAVIORAL AND BRAIN FUNCTIONS
LA English
DT Article
ID HIGH-FUNCTIONING AUTISM; TEMPORAL DISCRIMINATION; MINICOLUMNAR
ORGANIZATION; SENTENCE COMPREHENSION; ASPERGERS-DISORDER;
CAUDATE-NUCLEUS; WRITERS CRAMP; WHITE-MATTER; BRAIN SIZE; CONNECTIVITY
AB Background: A number of neurophysiological characteristics demonstrated in autism share the common theme of under-connectivity in the cerebral cortex. One of the prominent theories of the cause of the dysfunctional connectivity in autism is based on distinct anatomical structures that differ between the autistic and the neurotypical cortex. The functional minicolumn has been identified as occupying a much smaller space in the cortex of people with autism as compared to neurotypical controls, and this aberration in architecture has been proposed to lead to under-connectivity at the local or within-macrocolumn level, which in turn leads to dysfunctional connectivity globally across cortical areas in persons with autism. Numerous reports have indicated reduced synchronization of activity on a large scale in the brains of people with autism. We hypothesized that if the larger-scale aberrant dynamics in autism were due - at least in part - to a widespread propagation of the errors introduced at the level of local connectivity between minicolumns, then aberrations in local functional connectivity should also be detectable in autism.
Methods: Recently, we reported a method for measuring the perceptual changes that are impacted by the presence of synchronized conditioning stimuli on the skin. In this study, the temporal order judgment (TOJ) and temporal discriminative threshold (TDT) of 10 adult autism subjects were assessed both in the absence and presence of synchronized conditioning vibrotactile stimuli.
Results: Our previous report demonstrated that delivering simultaneous and synchronized vibrotactile stimuli to near-adjacent skin sites decreases a subject's ability to determine temporal order by 3 to 4-fold. However, results presented in this report show that subjects with autism do not demonstrate such decreased capacity in temporal order judgment (TOJ) in the presence of synchronized conditioning stimuli, although these same subjects do have TOJ thresholds well above that of controls.
Conclusion: It is speculated that the differences in sensory perceptual capacities in the presence of synchronized conditioning stimuli in autism are due to local under-connectivity in cortex at the minicolumnar organizational level, and that the above-average TOJ thresholds in autism could be attributed to structural differences that have been observed in the frontostrial system of this population.
C1 [Tommerdahl, Mark; Tannan, Vinay; Holden, Jameson K.] Univ N Carolina, Dept Biomed Engn, Chapel Hill, NC 27599 USA.
[Baranek, Grace T.] Univ N Carolina, Dept Allied Hlth Sci, Chapel Hill, NC 27599 USA.
RP Tommerdahl, M (reprint author), Univ N Carolina, Dept Biomed Engn, Chapel Hill, NC 27599 USA.
EM tommerda@med.unc.edu; tannanv@email.unc.edu; jamesonholden@unc.edu;
grace_baranek@med.unc.edu
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TC 37
Z9 37
PU BIOMED CENTRAL LTD
PI LONDON
PA 236 GRAYS INN RD, FLOOR 6, LONDON WC1X 8HL, ENGLAND
SN 1744-9081
J9 BEHAV BRAIN FUNCT
JI Behav. Brain Funct.
PD APR 24
PY 2008
VL 4
AR 19
DI 10.1186/1744-9081-4-19
PG 9
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 303KG
UT WOS:000256038800001
PM 18435849
ER
PT J
AU Cantor, RM
Geschwind, DH
AF Cantor, Rita M.
Geschwind, Daniel H.
TI Schizophrenia: Genome, interrupted
SO NEURON
LA English
DT Editorial Material
ID CHROMOSOMAL REARRANGEMENTS; MENTAL-RETARDATION; AUTISM; ASSOCIATION;
DISORDERS; GENETICS
AB Structural chromosomal variation is increasingly recognized as an important contributor to human diseases, particularly those of neurodevelopment, such as autism. A current paper makes a significant advance to schizophrenia genetics by establishing an association with rare copy number variants (CNV), which are over-represented in neurodevelopmental genes.
C1 [Cantor, Rita M.; Geschwind, Daniel H.] Univ Calif Los Angeles, Dept Human Genet, David Geffen Sch Med, Los Angeles, CA 90095 USA.
[Cantor, Rita M.; Geschwind, Daniel H.] Univ Calif Los Angeles, Ctr Neurobehav Genet, Semel Inst, Los Angeles, CA 90095 USA.
[Geschwind, Daniel H.] Univ Calif Los Angeles, Program Neurogenet, Dept Neurol, Los Angeles, CA 90095 USA.
RP Cantor, RM (reprint author), Univ Calif Los Angeles, Dept Human Genet, David Geffen Sch Med, Los Angeles, CA 90095 USA.
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TC 17
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PU CELL PRESS
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PA 600 TECHNOLOGY SQUARE, 5TH FLOOR, CAMBRIDGE, MA 02139 USA
SN 0896-6273
J9 NEURON
JI Neuron
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PY 2008
VL 58
IS 2
BP 165
EP 167
DI 10.1016/j.neuron.2008.04.007
PG 3
WC Neurosciences
SC Neurosciences & Neurology
GA 292YX
UT WOS:000255303100004
PM 18439401
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PT J
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LA English
DT Article
DE Aberrant Behavior Checklist; autism spectrum disorder; cerebellum; Down
syndrome; magnetic resonance imaging; neuroimaging; Talairach
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MRI; GROWTH; SCALE
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[Capone, George T.; Kaufmann, Walter E.] Johns Hopkins Univ, Sch Med, Dept Pediat, Baltimore, MD 21205 USA.
Johns Hopkins Univ, Sch Med, Baltimore, MD 21205 USA.
RP Kaufmann, WE (reprint author), Kennedy Krieger Inst, Ctr Genet Disorders Cognit & Behav, 3901 Greenspring Ave, Baltimore, MD 21211 USA.
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PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0959-4965
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SC Neurosciences & Neurology
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UT WOS:000255301300010
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PT J
AU Tessier, CR
Broadie, K
AF Tessier, Charles R.
Broadie, Kendal
TI Drosophila fragile X mental retardation protein developmentally
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SO DEVELOPMENT
LA English
DT Article
DE fragile X syndrome; mental retardation; autism; neural development;
translation; synaptogenesis; synaptic pruning
ID LONG-TERM POTENTIATION; SELECTIVE RNA-BINDING; FMR1 KNOCKOUT MICE;
MESSENGER-RNA; MOUSE MODEL; VISUAL-CORTEX; DENDRITIC SPINE; IN-VIVO;
SYNAPTIC PLASTICITY; MACACA-NEMESTRINA
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C1 [Tessier, Charles R.; Broadie, Kendal] Vanderbilt Univ, Kennedy Ctr Res Human Dev, Dept Biol Sci, Nashville, TN 37232 USA.
RP Broadie, K (reprint author), Vanderbilt Univ, Kennedy Ctr Res Human Dev, Dept Biol Sci, Nashville, TN 37232 USA.
EM kendal.broadie@vanderbilt.edu
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NR 92
TC 67
Z9 67
PU COMPANY OF BIOLOGISTS LTD
PI CAMBRIDGE
PA BIDDER BUILDING CAMBRIDGE COMMERCIAL PARK COWLEY RD, CAMBRIDGE CB4 4DL,
CAMBS, ENGLAND
SN 0950-1991
J9 DEVELOPMENT
JI Development
PD APR 15
PY 2008
VL 135
IS 8
BP 1547
EP 1557
DI 10.1242/dev.015867
PG 11
WC Developmental Biology
SC Developmental Biology
GA 287LD
UT WOS:000254917700016
PM 18321984
ER
PT J
AU Nomura, T
Kimura, M
Horii, T
Morita, S
Soejima, H
Kudo, S
Hatada, I
AF Nomura, Tasuku
Kimura, Mika
Horii, Takuro
Morita, Sumiyo
Soejima, Hidenobu
Kudo, Shinichi
Hatada, Izuho
TI MeCP2-dependent repression of an imprinted miR-184 released by
depolarization
SO HUMAN MOLECULAR GENETICS
LA English
DT Article
ID CPG-BINDING PROTEIN-2; X MENTAL-RETARDATION; RETT-SYNDROME BRAIN; BDNF
TRANSCRIPTION; DNA METHYLATION; MESSENGER-RNAS; NEURODEVELOPMENTAL
DISORDERS; MICROARRAY ANALYSIS; DENDRITIC SPINES; MECP2 EXPRESSION
AB Both fragile X syndrome and Rett syndrome are commonly associated with autism spectrum disorders and involve defects in synaptic plasticity. MicroRNA is implicated in synaptic plasticity because fragile X mental retardation protein was recently linked to the microRNA pathway. DNA methylation is also involved in synaptic plasticity since methyl CpG-binding protein 2 (MeCP2) is mutated in patients with Rett syndrome. Here we report that expression of miR-184, a brain-specific microRNA repressed by the binding of MeCP2 to its promoter, is upregulated by the release of MeCP2 after depolarization. The restricted release of MeCP2 from the paternal allele results in paternal allele-specific expression of miR-184. Our finding provides a clue to the link between the microRNA and DNA methylation pathways.
C1 Hokkaido Inst Publ Hlth, Kita Ku, Sapporo, Hokkaido 0600189, Japan.
[Nomura, Tasuku; Kimura, Mika; Horii, Takuro; Morita, Sumiyo; Hatada, Izuho] Gunma Univ, Biosignal Genome Resource Ctr, Lab Genome Sci, Inst Mol & Cellular Regulat, Maebashi, Gumma 3718512, Japan.
[Soejima, Hidenobu] Saga Univ, Dept Biomol Sci, Fac Med, Dept Mol Biol & Genet, Saga 8498501, Japan.
RP Hatada, I (reprint author), Gunma Univ, Biosignal Genome Resource Ctr, Lab Genome Sci, Inst Mol & Cellular Regulat, 3-39-15 Showa Machi, Maebashi, Gumma 3718512, Japan.
EM ihatada@showa.gunma-u.ac.jp
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NR 57
TC 65
Z9 69
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0964-6906
J9 HUM MOL GENET
JI Hum. Mol. Genet.
PD APR 15
PY 2008
VL 17
IS 8
BP 1192
EP 1199
DI 10.1093/hmg/ddn011
PG 8
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 284LP
UT WOS:000254708200014
PM 18203756
ER
PT J
AU Jordan, LM
AF Jordan, Lisa M.
TI Does your baby have autism?: Detecting the earliest signs of autism
SO LIBRARY JOURNAL
LA English
DT Book Review
C1 [Jordan, Lisa M.] Johnson Cty Lib, Shawnee Mission, KS 66201 USA.
RP Jordan, LM (reprint author), Johnson Cty Lib, Shawnee Mission, KS 66201 USA.
CR Teitelbaum O, 2008, DOES YOUR BABY HAVE
NR 1
TC 0
Z9 0
PU REED BUSINESS INFORMATION
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010 USA
SN 0363-0277
J9 LIBR J
JI Libr. J.
PD APR 15
PY 2008
VL 133
IS 7
BP 100
EP 101
PG 2
WC Information Science & Library Science
SC Information Science & Library Science
GA 291ZQ
UT WOS:000255236500178
ER
PT J
AU Leehey, MA
Berry-Kravis, E
Goetz, CG
Zhang, L
Hall, DA
Li, L
Rice, CD
Lara, R
Cogswell, J
Reynolds, A
Gane, L
Jacquemont, S
Tassone, F
Grigsby, J
Hagerman, RJ
Hagerman, PJ
AF Leehey, M. A.
Berry-Kravis, E.
Goetz, C. G.
Zhang, L.
Hall, D. A.
Li, L.
Rice, C. D.
Lara, R.
Cogswell, J.
Reynolds, A.
Gane, L.
Jacquemont, S.
Tassone, F.
Grigsby, J.
Hagerman, R. J.
Hagerman, P. J.
TI FMR1 CGG repeat length predicts motor dysfunction in premutation
carriers
SO NEUROLOGY
LA English
DT Article
ID FRAGILE-X PREMUTATION; TREMOR/ATAXIA-SYNDROME FXTAS; MESSENGER-RNA;
RATING-SCALE; ATAXIA; TREMOR; ONSET; CEREBELLAR; INVOLVEMENT; ALLELES
AB Background: Fragile X-associated tremor/ataxia syndrome (FXTAS) is a recently described, under-recognized neurodegenerative disorder of aging fragile X mental retardation 1 (FMR1) premutation carriers, particularly men. Core motor features are action tremor, gait ataxia, and parkinsonism. Carriers have expanded CGG repeats (55 to 200); larger expansions cause fragile X syndrome, the most common heritable cause of mental retardation and autism. This study determines whether CGG repeat length correlates with severity and type of motor dysfunction in premutation carriers.
Methods: Persons aged >= 50 years with a family history of fragile X syndrome underwent structured videotaping. Movement disorder neurologists, blinded to carrier status, scored the tapes using modified standardized rating scales. CGG repeat length analyses for women incorporated the activation ratio, which measures the percentage of normal active chromosome X alleles.
Results: Male carriers (n = 54) had significantly worse total motor scores, especially in tremor and ataxia, than age-matched male noncarriers (n = 51). There was a trend toward a difference between women carriers (n = 82) and noncarriers (n = 39). In men, increasing CGG repeat correlated with greater impairment in all motor signs. In women, when activation ratio was considered, increasing CGG correlated with greater ataxia.
Conclusions: CGG repeat size is significantly associated with overall motor impairment in premutation carriers. Whereas this association is most pronounced for men and covers overall motor impairment-tremor, ataxia, and parkinsonism-the association exists for ataxia among women carriers. This is the first report of a significant correlation between the premutation status and a motor feature of fragile X-associated tremor/ataxia syndrome in women.
C1 [Leehey, M. A.; Hall, D. A.] Univ Colorado, Dept Neurol, Denver, CO 80262 USA.
[Leehey, M. A.; Hall, D. A.; Rice, C. D.; Reynolds, A.; Grigsby, J.] Univ Colorado, Hlth Sci Ctr, Denver, CO 80262 USA.
[Berry-Kravis, E.; Goetz, C. G.; Lara, R.] Rush Univ, Med Ctr, Chicago, IL 60612 USA.
[Zhang, L.] Univ Calif Davis, Med Ctr, Dept Neurol, Sacramento, CA 95817 USA.
[Li, L.] N Carolina State Univ, Dept Stat, Raleigh, NC 27695 USA.
[Rice, C. D.; Reynolds, A.] Univ Colorado, Dept Pediat, Denver, CO 80262 USA.
[Cogswell, J.; Gane, L.; Hagerman, R. J.] Univ Calif Davis, Med Ctr, Dept Pediat, Sacramento, CA 95817 USA.
[Cogswell, J.; Gane, L.; Hagerman, R. J.] MIND Inst, Sacramento, CA USA.
[Jacquemont, S.] CHU Vaudois, Serv Genet, Lausanne, Switzerland.
[Tassone, F.; Hagerman, P. J.] Univ Calif Davis, Sch Med, Dept Biochem & Mol Med, Davis, CA 95616 USA.
[Grigsby, J.] Univ Colorado, Dept Med, Denver, CO 80262 USA.
RP Leehey, MA (reprint author), Univ Colorado, Dept Neurol, Box B182,4200 E 9th Ave, Denver, CO 80262 USA.
EM maureen.leehey@uchsc.edu
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NR 39
TC 62
Z9 64
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0028-3878
J9 NEUROLOGY
JI Neurology
PD APR 15
PY 2008
VL 70
IS 16
BP 1397
EP 1402
DI 10.1212/01.wnl.0000281692.98200.f5
PN 2
PG 6
WC Clinical Neurology
SC Neurosciences & Neurology
GA 312XW
UT WOS:000256706700006
PM 18057320
ER
PT J
AU Akiyama, T
Kato, M
Muramatsu, T
Maeda, T
Hara, T
Kashima, H
AF Akiyama, Tomoko
Kato, Motoichiro
Muramatsu, Taro
Maeda, Takaki
Hara, Tsunekatsu
Kashima, Haruo
TI Gaze-triggered orienting is reduced in chronic schizophrenia
SO PSYCHIATRY RESEARCH
LA English
DT Article
DE ambiguous stimulus; arrow; biological motion; spatial cueing; superior
temporal gyrus; superior temporal sulcus
ID SUPERIOR TEMPORAL GYRUS; CHILDHOOD-ONSET SCHIZOPHRENIA; EYE GAZE;
PERCEPTUAL ORGANIZATION; AUTISM; ATTENTION; DIRECTION; CUES;
DISCRIMINATION; ABNORMALITIES
AB Patients with schizophrenia have been reported to demonstrate subtle impairment in gaze processing, which in some cases indicates hypersensitivity to gaze, while in others, hyposensitivity. The neural correlate of gaze processing is situated in the superior temporal sulcus (STS), a major portion of which is constituted by the superior temporal gyrus (STG), and may be the underlying dysfunctional neural basis to the abnormal gaze sensitivity in schizophrenia. To identify the characteristics of gaze behavior in patients with chronic schizophrenia, in whom the STG has been reported to be smaller in volume, we tested 22 patients (mean duration of illness 29 years) in a spatial cueing paradigm using two central pictorial gaze cues, both of which effectively triggered attentional orienting in 22 age-matched normal controls. Arrow cues were also employed to determine whether any compromise in schizophrenia, if present, was gaze-specific. Results demonstrated that schizophrenic subjects benefit significantly less from congruent cues than normal subjects, which was evident for gaze cues but not for arrow cues. This finding is suggestive of a relatively gaze-specific hyposensitivity in patients with chronic schizophrenia, a finding that is in line with their clinical symptomatology and that may be associated with a hypoactive STS. (C) 2006 Elsevier Ireland Ltd. All rights reserved.
C1 [Akiyama, Tomoko; Hara, Tsunekatsu] Komagino Hosp, Dept Psychiat, Hachioji, Tokyo 1938505, Japan.
[Kato, Motoichiro; Muramatsu, Taro; Maeda, Takaki; Kashima, Haruo] Keio Univ, Sch Med, Dept Neuropsychiat, Shinjuku Ku, Tokyo, Japan.
RP Akiyama, T (reprint author), Komagino Hosp, Dept Psychiat, 273 Uratakao Cho, Hachioji, Tokyo 1938505, Japan.
EM tee-i@mxv.rnesh.nejp
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NR 46
TC 9
Z9 10
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0165-1781
J9 PSYCHIAT RES
JI Psychiatry Res.
PD APR 15
PY 2008
VL 158
IS 3
BP 287
EP 296
DI 10.1016/j.psychres.2006.12.004
PG 10
WC Psychiatry
SC Psychiatry
GA 294BL
UT WOS:000255379000003
PM 18262285
ER
PT J
AU Degirmenci, B
Miral, S
Kaya, GC
Iyilikci, L
Arslan, G
Baykara, A
Evren, I
Durak, H
AF Degirmenci, Berna
Miral, Sueha
Kaya, Game Capa
Iyilikci, Leyla
Arslan, Gulhan
Baykara, Aysen
Evren, Ismail
Durak, Hatice
TI Technetium-99m HMPAO brain SPECT in autistic children and their families
SO PSYCHIATRY RESEARCH-NEUROIMAGING
LA English
DT Article
DE autism; brain imaging; developmental disorder; single photon emission
computed tomography
ID CEREBRAL-BLOOD-FLOW; CHILDHOOD AUTISM; GLUCOSE-METABOLISM; HEAD
CIRCUMFERENCE; DISORDER; TOMOGRAPHY; PARENTS; HISTORY; SCREEN;
ASYMMETRIES
AB The purpose of the study was to investigate perfusion patterns in autistic children (AC) and their families. Ten AC (9 boys, I girl; mean age: 6.9 +/- 1.7 years) with autistic disorder defined by DSM-III-R criteria, five age-matched children (3 boys, 2 girls) as a control group, and the immediate family members of eight AC (8 mothers, 8 fathers, 7 siblings; mean ages: 39 +/- 4 years, 36 +/- 5 years and 13 +/- 5 years, respectively) were included in the study. Age- and sex-matched control groups for both the parents and the siblings were also included in the study. Brain perfusion images were obtained 1 h after the intravenous injection of an adjusted dose of Tc-99m HMPAO to children and the adults. Visual and semiquantitative evaluations were performed. Hypoperfusion was seen in the right posterior parietal cortex in three AC, in bilateral parietal cortex in one AC, bilateral frontal cortex in two AC, left parietal and temporal cortex in one AC, and right parietal and temporal cortex in one AC. Asymmetric perfusion was observed in the caudate nucleus in four AC. In semiquantitative analyses, statistically significant hypoperfusion was found in the right inferior and superior frontal, left superior frontal, right parietal, right mesial temporal and right caudate nucleus. In parents of AC, significant hypoperfusion was noted in the right parietal and bilateral inferior frontal cortex. In siblings of AC, perfusion in the right frontal cortex, right nucleus caudate and left parietal cortex was significantly decreased. This preliminary study suggests the existence of regional brain perfusion alterations in frontal, temporal, and parietal cortex and in caudate nucleus in AC and in their first-degree family members. (C) 2007 Elsevier Ireland Ltd. All rights reserved.
C1 [Degirmenci, Berna; Kaya, Game Capa; Arslan, Gulhan; Evren, Ismail; Durak, Hatice] Dokuz Eylul Univ, Sch Med, Dept Nucl Med, TR-35340 Izmir, Turkey.
[Miral, Sueha; Baykara, Aysen] Dokuz Eylul Univ, Dept Child & Adolescent Psychiat, Izmir, Turkey.
[Iyilikci, Leyla] Dokuz Eylul Univ, Dept Anesthesiol, Izmir, Turkey.
RP Degirmenci, B (reprint author), Dokuz Eylul Univ, Sch Med, Dept Nucl Med, TR-35340 Izmir, Turkey.
EM berna.degirmenci@deu.edu.tr
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NR 54
TC 9
Z9 11
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0925-4927
J9 PSYCHIAT RES-NEUROIM
JI Psychiatry Res. Neuroimaging
PD APR 15
PY 2008
VL 162
IS 3
BP 236
EP 243
DI 10.1016/j.pscychresns.2004.12.005
PG 8
WC Clinical Neurology; Neuroimaging; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 290KQ
UT WOS:000255122100007
PM 18302983
ER
PT J
AU Kring, SR
Greenberg, JS
Seltzer, MM
AF Kring, Sheilah R.
Greenberg, Jan S.
Seltzer, Marsha Mailick
TI Adolescents and Adults with Autism with and without Co-morbid
Psychiatric Disorders: Differences in Maternal Well-Being
SO JOURNAL OF MENTAL HEALTH RESEARCH IN INTELLECTUAL DISABILITIES
LA English
DT Article
DE burden; autism; co-morbidity; adolescents and adults; psychiatric
disorders
AB This study investigated the associations between the characteristics of adolescents and adults with autism spectrum disorders (ASD) and maternal well-being. Two groups were compared: mothers of adolescents and adults with ASD and co-morbid psychiatric disorders (n = 142) and mothers whose sons or daughters had a single diagnosis of ASD (n = 130). Individuals with co-morbid psychiatric disorders had higher levels of repetitive behaviors, asocial behavior, and unpredictability of behavior than their counterparts with ASD only. They also had poorer rated health as well as more frequent gastrointestinal problems and sleep problems. Mothers of sons and daughters with ASD and co-morbid psychiatric disorders reported higher levels of burden and a poorer quality parent-child relationship than mothers of sons and daughters with ASD only. Higher levels of asocial behavior, unpredictability of behavior, and poorer health in sons and daughters with ASD were predictive of greater burden in mothers and a poorer quality parent-child relationship.
C1 [Kring, Sheilah R.; Greenberg, Jan S.; Seltzer, Marsha Mailick] Univ Wisconsin Madison, Madison, WI 53705 USA.
RP Kring, SR (reprint author), Univ Wisconsin Madison, Waisman Ctr, 1500 Highland Ave, Madison, WI 53705 USA.
EM kring@waisman.wisc.edu
FU National Institute on Aging [R01 AG08768]; National Institute of Mental
Health [T32 MH065185]; National Institute of Child Health and Human
Development [P30 HD03352]; National Institute for Disability and
Rehabilitation Research through the Rehabilitation Research and Training
Center on Aging and Developmental Disabilities at the University of
Illinois at Chicago [H133B031134]
FX This article was prepared with support from the National Institute on
Aging (R01 AG08768), the National Institute of Mental Health (T32
MH065185), the National Institute of Child Health and Human Development
(P30 HD03352), and the National Institute for Disability and
Rehabilitation Research (H133B031134) through the Rehabilitation
Research and Training Center on Aging and Developmental Disabilities at
the University of Illinois at Chicago.
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NR 60
TC 5
Z9 5
PU ROUTLEDGE JOURNALS, TAYLOR & FRANCIS LTD
PI ABINGDON
PA 4 PARK SQUARE, MILTON PARK, ABINGDON OX14 4RN, OXFORDSHIRE, ENGLAND
SN 1931-5864
EI 1931-5872
J9 J MENT HEALTH RES IN
JI J. Ment. Health Res. Intellect. Disabil.
PD APR 10
PY 2008
VL 1
IS 2
BP 53
EP 74
DI 10.1080/19315860801988228
PG 22
WC Education, Special; Psychiatry; Rehabilitation
SC Education & Educational Research; Psychiatry; Rehabilitation
GA V37ZP
UT WOS:000209314000001
ER
PT J
AU Kalueff, AV
Ren-Patterson, RF
LaPorte, JL
Murphy, DL
AF Kalueff, Allan V.
Ren-Patterson, Renee F.
LaPorte, Justin L.
Murphy, Dennis L.
TI Domain interplay concept in animal models of neuropsychiatric disorders:
A new strategy for high-throughput neurophenotyping research
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Review
DE genetic animal models; brain disorders; behavioral/psychiatric
phenotypes; domain interplay; comorbidity; gene x environment
interaction; mutant and transgenic animals
ID TRANSPORTER KNOCKOUT MICE; OBSESSIVE-COMPULSIVE DISORDER; FACTOR BDNF
GENE; SEROTONIN TRANSPORTER; BIPOLAR SPECTRUM; INBRED STRAINS; ANXIETY;
DEPRESSION; AUTISM; PHENOTYPES
AB Genetic and environmental factors play a key role in psychiatric disorders. While some disorders display exceptionally high heritability, others show gene x experience x personality interactions, contributing complexity to psychiatric phenotypes. As some brain disorders frequently overlap and co-occur (representing a continuum Or spectrum of phenomena), modern psychiatry is shifting front "artificial" heterogeneity to the recognition of common elements in the pathogenesis of emotional, personality and behavioral disorders. Genetic animal models of these disorders represent an important direction of research, and are widely used to explore the role of different genes in brain mechanisms. Several concepts (such as endophenotypes, gene x environment interactions, and cross-species trait genetics) have been suggested for animal experimentation in this field. Here we develop a new concept based on targeting the complex interplay between different behavioral domains, meant to foster high-throughput phenotyping and integrative modeling of psychiatric disorders. Published by Elsevier B.V.
C1 [Kalueff, Allan V.; Ren-Patterson, Renee F.; LaPorte, Justin L.; Murphy, Dennis L.] NIMH, Clin Sci Lab, Bethesda, MD 20892 USA.
RP Kalueff, AV (reprint author), NIMH, Clin Sci Lab, Bldg 10,Room 3D41,10 Ctr Dr,MSC 1264, Bethesda, MD 20892 USA.
EM kalueva@mail.nih.gov
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NR 73
TC 34
Z9 34
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD APR 9
PY 2008
VL 188
IS 2
BP 243
EP 249
DI 10.1016/j.bbr.2007.11.011
PG 7
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 277MT
UT WOS:000254217700001
PM 18164476
ER
PT J
AU Yochum, CL
Dowling, P
Reuhl, KR
Wagner, GC
Ming, X
AF Yochum, Carrie L.
Dowling, Peter
Reuhl, Kenneth R.
Wagner, George C.
Ming, Xue
TI VPA-induced apoptosis and behavioral deficits in neonatal mice
SO BRAIN RESEARCH
LA English
DT Article
DE autism; sodium valproate; apoptosis; cerebellum
ID FETAL VALPROATE SYNDROME; DEVELOPMENTAL DISORDERS; ANIMAL-MODELS;
AUTISM; CEREBELLUM; HUMANS; CORTEX; EXPOSURE; ONTOGENY; BRAIN
AB Sodium valproate (VPA) administered to neonatal mice causes cognitive and motor deficits similar to those observed in humans with autism. In an effort to further evaluate similarities between early VPA exposure and autism, the present study examined treated mice for deficits in social behavior and neuronal damage. BALB/c mice injected on P14 with 400 mg/kg VPA engaged in fewer social interactions (including ano-genital sniffs, allogrooming, and crawl-under/over behaviors) than control mice. Treated mice also exhibited reduced motor activity in a social context but were not significantly different from controls when motor activity was assessed in non-social settings. A second set of BALB/c mice were treated with VPA on P14 and sacrificed at different times thereafter for histopathological analysis. At time-points 12 and 24 h following VPA, treated mice had up to a 30-fold increase in the number of TUNEL-positive cells in the external granule cell layer of the cerebellum and a 10-fold increase in TUNEL-positive cells in the dentate gyrus of the hippocampus. These observations may provide a histopathological. correlate for the social deficits observed following post-natal VPA exposure and supports the use of early VPA administration as an animal model for the study of autism. (c) 2008 Elsevier B.V. All rights reserved.
C1 [Yochum, Carrie L.; Wagner, George C.] Rutgers State Univ, Dept Psychol, New Brunswick, NJ 08854 USA.
[Reuhl, Kenneth R.; Wagner, George C.] Rutgers State Univ, Dept Pharmacol & Toxicol, New Brunswick, NJ 08854 USA.
[Dowling, Peter; Ming, Xue] Univ Med & Dent New Jersey, Dept Neurol & Neurosci, Newark, NJ 07103 USA.
RP Wagner, GC (reprint author), Rutgers State Univ, Dept Psychol, New Brunswick, NJ 08854 USA.
EM gcwagner@rci.rutgers.edu
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NR 29
TC 31
Z9 33
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0006-8993
J9 BRAIN RES
JI Brain Res.
PD APR 8
PY 2008
VL 1203
BP 126
EP 132
DI 10.1016/j.brainres.2008.01.055
PG 7
WC Neurosciences
SC Neurosciences & Neurology
GA 288DR
UT WOS:000254966900015
PM 18316065
ER
PT J
AU Looi, JCL
AF Looi, Jeffrey C. L.
TI Empathy and competence
SO MEDICAL JOURNAL OF AUSTRALIA
LA English
DT Article
ID PHYSICIAN EMPATHY; MEDICAL-STUDENTS; PERFORMANCE; PREDICTORS; DOCTORS;
SCHOOL; TRUST
AB There has been a call to include empathy as a selection criterion in medical training.
Empathy is a complex construct currently assessed by self-rating and observational scales, which may be complicated by the subjectivity of such measurements.
Neuroscientific research into disorders of empathy such as autism should be encouraged to help further refine the evolving construct of empathy.
Empathy may be more common in females, and selection for higher empathy may discriminate against males unless sex-specific adjustments are included in selection criteria.
Physician empathy may lead to greater patient satisfaction and confidence in physicians, but more evidence is needed to support links to physician competence. In contrast, academic performance and conscientiousness have been clearly linked to physician competence.
Competence and empathy may be independent qualities developed by different aspects of medical training.
Provision of better work conditions and environments for physicians may forestall erosion of empathy, reducing the need to predict and enhance its development.
Empathy should be valued in medical students and doctors, but more research is needed into the nature, assessment, and correlates of empathy before its adoption as a selection criterion for medical students.
C1 Australian Natl Univ, Sch Med, Acad Unit Psychol Med, Canberra, ACT, Australia.
RP Looi, JCL (reprint author), Australian Natl Univ, Sch Med, Acad Unit Psychol Med, Canberra, ACT, Australia.
EM jefFrey.looi@anu.edu.au
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NR 22
TC 0
Z9 1
PU AUSTRALASIAN MED PUBL CO LTD
PI PYRMONT
PA LEVEL 2, 26-32 PYRMONT BRIDGE RD, PYRMONT, NSW 2009, AUSTRALIA
SN 0025-729X
J9 MED J AUSTRALIA
JI Med. J. Aust.
PD APR 7
PY 2008
VL 188
IS 7
BP 414
EP 416
PG 3
WC Medicine, General & Internal
SC General & Internal Medicine
GA 300CV
UT WOS:000255802800012
PM 18393746
ER
PT J
AU Silverman, JM
Buxbaum, JD
Ramoz, N
Schmeidler, J
Reichenberg, A
Hollander, E
Angelo, G
Smith, CJ
Kryzak, LA
AF Silverman, Jeremy M.
Buxbaum, Joseph D.
Ramoz, Nicolas
Schmeidler, James
Reichenberg, Abraham
Hollander, Eric
Angelo, Gary
Smith, Christopher J.
Kryzak, Lauren A.
TI Autism-related routines and rituals associated with a mitochondrial
aspartate/glutamate carrier SLC25A12 polymorphism
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE repetitive behaviors; familial traits; autism spectrum disorders
ID GENE; LINKAGE; CHROMOSOME-2
AB Evidence for a genetic association between autism and two single nucleotide polymorphisms (SNPs), rs2056202 and rs2292813, in the mitochondrial aspartate/glutamate carrier (SLC25A12) gene led us to ask whether any of the four previously identified familial traits in autism spectrum disorders (ASD) varied by these SNPs. In 355 ASD cases from 170 sibships we examined levels of the four traits in these SNPs using ANCOVA models. The primary models selected unrelated affected cases and used age and sex as covariates. An ancillary set of models used all affected siblings and included "sibship" as a random effects independent variable. We found significantly lower levels of routines and rituals associated with the presence of the less frequent A allele in rs2056206. No other significant differences were observed. The rs2056202 polymorphism may be associated with levels of routines and rituals in autism and related disorders. (C) 2007 Wiley-Liss, Inc.
C1 [Silverman, Jeremy M.; Buxbaum, Joseph D.; Schmeidler, James; Reichenberg, Abraham; Hollander, Eric; Angelo, Gary; Smith, Christopher J.; Kryzak, Lauren A.] Mt Sinai Sch Med, Dept Psychiat, New York, NY 10029 USA.
[Ramoz, Nicolas] INSERM, Paris, France.
[Reichenberg, Abraham] Inst Psychiat, London, England.
RP Silverman, JM (reprint author), Mt Sinai Sch Med, Dept Psychiat, Box 1230,1 Gustave L Levy Pl, New York, NY 10029 USA.
EM jeremy.silverman@mssm.edu
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NR 12
TC 24
Z9 24
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD APR 5
PY 2008
VL 147B
IS 3
BP 408
EP 410
DI 10.1002/ajmg.b.30614
PG 3
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 282LN
UT WOS:000254569400021
PM 17894412
ER
PT J
AU Wirojanan, J
Angkustsiri, K
Tassone, F
Gane, LW
Hagerman, RJ
AF Wirojanan, Juthamas
Angkustsiri, Kathleen
Tassone, Flora
Gane, Louise W.
Hagerman, Randi J.
TI A girl with fragile X premutation from sperm donation
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Article
DE fragile X syndrome; premutation; sperm donation; genetic screening
ID TREMOR/ATAXIA SYNDROME FXTAS; FMR1 PREMUTATION; CGG-REPEAT; INTERMEDIATE
ALLELES; CARRIERS; AUTISM; INDIVIDUALS; POPULATION; PHENOTYPE; GENE
AB We present a girl with the fragile X premutation who obtained the premutation allele from donated sperm. Our patient has clinical characteristics of fragile X syndrome including emotional problems and neuropsychological difficulties presenting as learning disabilities. She is also at high risk for premature ovarian failure and low risk for the fragile X-associated tremor ataxia (FXTAS). We suggest fragile X DNA screening in gamete donor candidates to decrease the chance of fragile X involvement in their offspring. (c) 2008 Wiley-Liss, Inc.
C1 [Wirojanan, Juthamas; Angkustsiri, Kathleen; Tassone, Flora; Gane, Louise W.; Hagerman, Randi J.] Univ Calif Davis, Med Ctr, MIND Inst, Sacramento, CA 95817 USA.
[Wirojanan, Juthamas] Prince Songkla Univ, Fac Med, Dept Pediat, Hat Yai, Songkhla, Thailand.
[Angkustsiri, Kathleen; Hagerman, Randi J.] Univ Calif Davis, Med Ctr, Dept Pediat, Sacramento, CA 95817 USA.
[Tassone, Flora] Univ Calif Davis, Sch Med, Dept Biol Chem, Sacramento, CA 95817 USA.
RP Hagerman, RJ (reprint author), Univ Calif Davis Hlth Syst, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM randi.hagerman@ucdmc.ucdavis
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NR 40
TC 4
Z9 4
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD APR 1
PY 2008
VL 146A
IS 7
BP 888
EP 892
DI 10.1002/ajmg.a.31876
PG 5
WC Genetics & Heredity
SC Genetics & Heredity
GA 282SL
UT WOS:000254587400011
PM 18286596
ER
PT J
AU Harpin, VA
AF Harpin, V. A.
TI Medication options when treating children and adolescents with ADHD:
interpreting the NICE guidance 2006
SO ARCHIVES OF DISEASE IN CHILDHOOD-EDUCATION AND PRACTICE EDITION
LA English
DT Article
ID ATTENTION-DEFICIT/HYPERACTIVITY-DISORDER; DEFICIT HYPERACTIVITY
DISORDER; AUTISM SPECTRUM DISORDERS; ONCE-DAILY ATOMOXETINE; OPEN-LABEL
TRIAL; TREATMENT STRATEGIES; STIMULANT TREATMENT; DOUBLE-BLIND;
METHYLPHENIDATE; SYMPTOMS
AB In recent years the medication options for the treatment of ADHD in children and young people have increased. The National Institute for Health and Clinical Excellence (NICE) produced updated guidelines in 2006. This paper aims to interpret these guidelines by reviewing the medication options in light of relevant research and clinical practice. The properties of methylphenidate, dexamfetamine and atomoxetine are discussed.
C1 Sheffield Childrens NHS Fdn Trust, Ryegate Childrens Ctr, Sheffield S10 5DD, S Yorkshire, England.
RP Harpin, VA (reprint author), Sheffield Childrens NHS Fdn Trust, Ryegate Childrens Ctr, Tapton Crescent Rd, Sheffield S10 5DD, S Yorkshire, England.
EM val.harpin@sch.nhs.uk
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NR 71
TC 4
Z9 4
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 1743-0585
J9 ARCH DIS CHILDHOOD-E
JI Arch. Dis. Childhood-Educ. Pract. Ed.
PD APR
PY 2008
VL 93
IS 2
BP 58
EP 65
DI 10.1136/adc.2006.106864
PG 8
WC Pediatrics
SC Pediatrics
GA 283XE
UT WOS:000254669000004
PM 18356307
ER
PT J
AU Bailey, A
Sutcliffe, JS
Schultz, R
Rogers, S
AF Bailey, Anthony
Sutcliffe, James S.
Schultz, Robert
Rogers, Sally
TI Our Vision for Autism Research
SO AUTISM RESEARCH
LA English
DT Editorial Material
RI Bailey, Anthony/J-2860-2014
OI Bailey, Anthony/0000-0003-4257-972X
NR 0
TC 0
Z9 0
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD APR
PY 2008
VL 1
IS 2
BP 71
EP 72
DI 10.1002/aur.18
PG 2
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CI
UT WOS:000270031600001
PM 19360653
ER
PT J
AU Williams, JHG
AF Williams, Justin H. G.
TI Self-Other Relations in Social Development and Autism: Multiple Roles
for Mirror Neurons and Other Brain Bases
SO AUTISM RESEARCH
LA English
DT Review
DE autism; autistic disorder; Asperger's syndrome; imitation; gaze;
attention; motor control; perception; action; mirror neurons; fMRI;
parietal cortex; Broca's area; pSTS; 'theory of mind' (TOM)
ID TEMPORO-PARIETAL JUNCTION; EVENT-RELATED FMRI; SPECTRUM DISORDERS;
ASPERGER-SYNDROME; JOINT ATTENTION; BIOLOGICAL MOTION; YOUNG-CHILDREN;
WHITE-MATTER; FUNCTIONAL-ORGANIZATION; NEURAL REPRESENTATION
AB Mirror neuron system dysfunction may underlie a self-other matching impairment, which has previously been suggested to account for autism. Embodied Cognition Theory, which proposes that action provides a foundation for cognition has tent further credence to these ideas. The hypotheses of a self-other matching deficit and impaired mirror neuron function in autism have now been well supported by studies employing a range of methodologies. However, underlying mechanisms require further exploration to explain how mirror neurons may be involved in attentional and mentalizing processes. Impairments in self-other matching and mirror neuron function are not necessarily inextricably linked and it seems possible that different sub-populations of mirror neurons, located in several regions, contribute differentially to social cognitive functions. It is hypothesized that mirror neuron coding for action-direction may be required for developing attentional sensitivity to self-directed actions, and consequently for person-oriented, stimulus-driven attention. Mirror neuron networks may vary for different types of social learning such as "automatic" imitation and imitation learning. Imitation learning may be more reliant on self-other comparison processes (based on mirror neurons) that identify differences as well as similarities between actions. Differential connectivity with the amygdala-orbitofrontal system may also be important. This could have implications for developing "theory of mind," with intentional self-other comparison being relevant to meta-representational abilities, and "automatic" imitation being more relevant to empathy. While it seems clear that autism is associated with impaired development of embodied aspects of cognition, the ways that mirror neurons contribute to these brain-behavior links are likely to be complex.
C1 Univ Aberdeen, Sch Med, Dept Child Hlth, Royal Aberdeen Childrens Hosp, Aberdeen AB25 2ZG, Scotland.
RP Williams, JHG (reprint author), Univ Aberdeen, Sch Med, Dept Child Hlth, Royal Aberdeen Childrens Hosp, Aberdeen AB25 2ZG, Scotland.
EM justin.williams@abdn.ac.uk
FU Northwood Charitable Trust
FX This research is supported by the Northwood Charitable Trust. I am also
grateful to Nina Williams, David Perrett, Morven McWhirr, Michael Arbib,
and Sally Rogers and to two anonymous reviewers for helpful comments on
earlier drafts.
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NR 170
TC 53
Z9 55
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD APR
PY 2008
VL 1
IS 2
BP 73
EP 90
DI 10.1002/aur.15
PG 18
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CI
UT WOS:000270031600002
PM 19360654
ER
PT J
AU Deruelle, C
Hubert, B
Santos, A
Wicker, B
AF Deruelle, C.
Hubert, B.
Santos, A.
Wicker, B.
TI Negative Emotion Does Not Enhance Recall Skills in Adults With Autistic
Spectrum Disorders
SO AUTISM RESEARCH
LA English
DT Article
DE emotion; memory; valence; autism
ID PERVASIVE DEVELOPMENTAL DISORDERS; HIGH-FUNCTIONING AUTISM; MEMORY
ENHANCEMENT; WORKING-MEMORY; LONG-TERM; AMYGDALA; CHILDREN; INDIVIDUALS;
RECOGNITION; ATTENTION
AB Recent empirical findings suggest a significant influence of emotion on memory processes. Surprisingly, although emotion-processing difficulties appear to be a hallmark feature in autism spectrum disorders (ASD), their impact on higher-level cognitive functions, such as memory, has not been directly studied in this population. The aim of this study was to address this issue by assessing whether the emotional valence of visual scenes affects recall skills in high-functioning individuals with ASD. To this purpose, their recall performance of neutral and emotional pictures was compared with that of typically developing adults (control group). Results revealed that while typically developing individuals showed enhanced recall skills for negative relative to positive and neutral pictures, individuals with ASD recalled the neutral pictures as well as the emotional ones. Findings of this study thus point to reduced influence of emotion on memory processes in ASD than in typically developing individuals, possibly owing to amygdala dysfunctions.
C1 [Deruelle, C.; Hubert, B.; Santos, A.; Wicker, B.] CNRS, Inst Neurosci Cognit Mediterranee, F-13402 Marseille 20, France.
RP Deruelle, C (reprint author), CNRS, Inst Neurosci Cognit Mediterranee, 31 Chemin Joseph Aiguier, F-13402 Marseille 20, France.
EM deruelle@incm.cnrs-mrs.fr
FU FCT-MCTES (Portugal) [SFRH/BD/18820/2004]
FX We thank all individuals who participated in this study, as well as
their family. A. Santos was supported by a grant from the FCT-MCTES
(Portugal, SFRH/BD/18820/2004) to conduct this study.
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NR 43
TC 11
Z9 11
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD APR
PY 2008
VL 1
IS 2
BP 91
EP 96
DI 10.1002/aur.13
PG 6
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CI
UT WOS:000270031600003
PM 19360655
ER
PT J
AU Paul, R
Chawarska, K
Cicchetti, D
Volkmar, F
AF Paul, Rhea
Chawarska, Katarzyna
Cicchetti, Domenic
Volkmar, Fred
TI Language Outcomes of Toddlers With Autism Spectrum Disorders: A Two Year
Follow-Up
SO AUTISM RESEARCH
LA English
DT Article
DE autism; language; communication; toddlers
ID PERVASIVE DEVELOPMENTAL DISORDERS; YOUNG-CHILDREN; ADAPTIVE-BEHAVIOR;
JOINT ATTENTION; PRELINGUISTIC PREDICTORS; NONVERBAL-COMMUNICATION;
3-YEAR-OLD CHILDREN; DOWN-SYNDROME; 2ND YEAR; AGE
AB Thirty-seven children 15-25 months of age received clinical diagnoses of autism spectrum disorder (ASD) and were re-evaluated two years later. All subjects were judged to have retained a diagnosis of ASD at the follow-up evaluation. Communication scores for the group as a whole during the first visit were significantly lower than nonverbal IQ. However, by the second visit, verbal and nonverbal scores were no longer significantly different. The group was divided into two subgroups, based on expressive language (EL) outcome at the second visit. The two groups were similar in the second year of life in terms of expressive communication skills and autistic symptoms, except for a trend toward more stereotypic and repetitive behavior in the worse outcome group. By the second visit, however, the groups differed significantly on all standard measures of expression and reception, as well as on autistic symptomotology and nonverbal IQ. When assessed during their second year, children who ended up in the better outcome group showed higher average nonverbal cognitive level, receptive language (RL) scores, number of sounds and words produced, use of symbolic play schemes, and response to joint attention bids. Regression analysis revealed that the variables for which significant differences between the two outcome groups in their second year of life were found provided significant prediction of EL outcome at age four. Stepwise regression identified RL and presence of stereotypic and repetitive at the first visit as significantly associated with EL outcome. Implications of these findings for early identification and intervention are discussed.
C1 [Paul, Rhea; Chawarska, Katarzyna; Cicchetti, Domenic; Volkmar, Fred] Yale Univ, Sch Med, Yale Child Study Ctr, New Haven, CT 06510 USA.
[Paul, Rhea] So Connecticut State Univ, Dept Commun Disorders, New Haven, CT 06515 USA.
RP Paul, R (reprint author), Yale Univ, Sch Med, Yale Child Study Ctr, 40 Temple St 6B, New Haven, CT 06510 USA.
EM rhea.paul@yale.edu
FU NIDCD [K24 HD045576]
FX Preparation of this article was supported by a MidCareer Development
grant to Dr. Paul, K24 HD045576 funded by NIDCD. We also thank Carolyn
Gosse, Kelly Cardona, Kate Elliot, and Elizabeth Schoen for their
assistance in collecting data and preparing this manuscript, as well as
the families who participated in our research.
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NR 77
TC 24
Z9 25
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD APR
PY 2008
VL 1
IS 2
BP 97
EP 107
DI 10.1002/aur.12
PG 11
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CI
UT WOS:000270031600004
PM 19360656
ER
PT J
AU Brune, CW
Kim, SJ
Hanna, GL
Courchesne, E
Lord, C
Leventhal, BL
Cook, EH
AF Brune, Camille W.
Kim, Soo-Jeong
Hanna, Gregory L.
Courchesne, Eric
Lord, Catherine
Leventhal, Bennett L.
Cook, Edwin H.
TI Family-Based Association Testing of OCD-Associated SNPs of SLC1A1 in an
Autism Sample
SO AUTISM RESEARCH
LA English
DT Article
DE autism; SLC1A1; OCD; association
ID OBSESSIVE-COMPULSIVE DISORDER; PERVASIVE DEVELOPMENTAL DISORDERS;
TRANSPORTER GENE; PSYCHIATRIC-DISORDERS; REPETITIVE BEHAVIOR; SPECTRUM
DISORDERS; LINKAGE ANALYSIS; GLUTAMATE; CHILDREN; SUSCEPTIBILITY
AB Reports identified the neuronal glutamate transporter gene, SLC1A1 (OMIM 133550, chromosome 9p24), as a positional and functional candidate gene for obsessive-compulsive disorder (OCD). The presence of obsessions and compulsions similar to OCD in autism, the identification of this region in a genome-wide linkage analysis of individuals with autism spectrum disorders (ASDs), and the hypothesized role of glutamate in ASDs make SLC1A1 a candidate gene for ASD as well. To test for association between SLC1A1 and autism, we typed three single nucleotide polymorphisms (SNPs, rs301430, rs301979, rs301434) previously associated with OCD in 86 strictly defined trios with autism. Family-Based Association Tests (FBAT) with additive and recessive models were used to check for association. Additionally, an rs301430-rs301979 haplotype identified for OCD was investigated. FBAT revealed nominally significant association between autism and one SNP under a recessive model. The G allele of rs301979 was undertransmitted (equivalent to overtransmission of the C allele under a dominant model) to individuals with autism (Z = -2.47, P = 0.01). The G allele was also undertransmitted in the T-G haplotype under the recessive model (Z = -2.41, P = 0.02). Both findings were also observed in the male-only sample. However, they did not withstand correction for multiple comparisons.
C1 [Brune, Camille W.; Leventhal, Bennett L.; Cook, Edwin H.] Univ Illinois, Inst Juvenile Res, Dept Psychiat, Chicago, IL 60608 USA.
[Kim, Soo-Jeong] Univ Florida, Coll Med, Dept Psychiat, Gainesville, FL 32611 USA.
[Hanna, Gregory L.] Univ Michigan, Dept Psychiat, Ann Arbor, MI 48109 USA.
[Courchesne, Eric] Univ Calif San Diego, Dept Neurosci, San Diego, CA 92103 USA.
[Lord, Catherine] Univ Michigan, Autism & Commun Disorders Ctr, Ann Arbor, MI 48109 USA.
RP Cook, EH (reprint author), Univ Illinois, Inst Juvenile Res, Dept Psychiat MC 747, 1747 W Roosevelt Rd,Room 155, Chicago, IL 60608 USA.
EM ecook@psych.uic.edu
FU Autism Speaks postdoctoral fellowship; NARSAD; PWSA (USA) Research
Award; National Alliance for Autism Research; NIH [U19 HD35482]
FX C.W.B. is supported in part by an Autism Speaks postdoctoral fellowship.
S.-J.K. is an Advanced Postgraduate Program in Clinical Investigation
(APPCI) fellow at the University of Florida and is supported in part by
a 2007 NARSAD young investigator award and the 2008 PWSA (USA) Research
Award. E.H.C. is supported in part from the National Alliance for Autism
Research (currently Autism Speaks) and NIH U19 HD35482.
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NR 43
TC 11
Z9 11
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD APR
PY 2008
VL 1
IS 2
BP 108
EP 113
DI 10.1002/aur.11
PG 6
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CI
UT WOS:000270031600005
PM 19360657
ER
PT J
AU Scherf, KS
Luna, B
Kimchi, R
Minshew, N
Behrmann, M
AF Scherf, K. Suzanne
Luna, Beatriz
Kimchi, Ruth
Minshew, Nancy
Behrmann, Marlene
TI Missing the Big Picture: Impaired Development of Global Shape Processing
in Autism
SO AUTISM RESEARCH
LA English
DT Article
DE perceptual grouping; global/local processing; vision; global advantage
ID HIGH-FUNCTIONING AUTISM; FUSIFORM FACE AREA; PERCEPTUAL ORGANIZATION;
HIERARCHICAL PATTERNS; SPECTRUM DISORDER; ASPERGER-SYNDROME;
VISUAL-SEARCH; SELECTIVE ATTENTION; CHILDRENS ANALYSIS; TASK
AB Individuals with autism exhibit hypersensitivity to local elements of the input, which may interfere with the ability to group visual elements perceptually. We investigated the development of perceptual grouping abilities in high-functioning individuals with autism (HFA) across a wide age range (8-30 years) using a classic compound letter global/local (GL) task and a more fine-grained microgenetic prime paradigm (MPP), including both few- and many-element hierarchical displays. In the GL task, contrary to the typically developing (TD) controls, HFA participants did not develop an increasing sensitivity to the global information with age. In the MPP, like the TD controls, individuals with autism at all three age groups evinced a bias to individuate the few-element displays. However, contrary to the TD controls, the HFA group failed to show age-related improvements in the ability to encode the global shape of the many-element displays. In fact, across the age range, the HFA group was consistently faster than the TD controls at perceiving the local elements in these displays. These results indicate that in autism the full process of garnering shape information from perceptual grouping, which is essential for the ability to do fast and efficient object recognition and identification, never matures, and this is especially evident in adolescence when this ability begins to improve in TD individuals. The atypical development of these perceptual organizational abilities may disrupt processing of visually presented objects, which may, in turn, fundamentally impede the development of major aspects of the social and emotional behaviors in individuals with autism.
C1 [Scherf, K. Suzanne; Behrmann, Marlene] Carnegie Mellon Univ, Dept Psychol, Pittsburgh, PA 15213 USA.
[Luna, Beatriz; Minshew, Nancy] Univ Pittsburgh, Dept Psychiat, Pittsburgh, PA USA.
[Kimchi, Ruth] Univ Haifa, Dept Psychol, IL-31999 Haifa, Israel.
RP Scherf, KS (reprint author), Carnegie Mellon Univ, Dept Psychol, 330 Baker Hall,5000 Forbes Ave, Pittsburgh, PA 15213 USA.
EM scherf@pitt.edu
RI Luna, Beatriz/F-1201-2010
FU NIH [NICHD/NIDCD PO1/U19 HD35469-07]; National Alliance for Autism
Research; [T32 HD049354]
FX Grant Sponsors: NAAR638, NICHD/NIDCD PO1/U195U19 HD35469, NIMH5 T32
HD049354-03, National Alliance for Autism Research
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NR 79
TC 27
Z9 27
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD APR
PY 2008
VL 1
IS 2
BP 114
EP 129
DI 10.1002/aur.17
PG 16
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CI
UT WOS:000270031600006
PM 19360658
ER
PT J
AU Croen, LA
Goines, P
Braunschweig, D
Yolken, R
Yoshida, CK
Grether, JK
Fireman, B
Kharrazi, M
Hansen, RL
Van de Water, J
AF Croen, Lisa A.
Goines, Paula
Braunschweig, Daniel
Yolken, Robert
Yoshida, Cathleen K.
Grether, Judith K.
Fireman, Bruce
Kharrazi, Martin
Hansen, Robin L.
Van de Water, Judy
TI Brain-Derived Neurotrophic Factor and Autism: Maternal and Infant
Peripheral Blood Levels in the Early Markers for Autism (EMA) Study
SO AUTISM RESEARCH
LA English
DT Article
DE biologic markers; neurotrophin; autism; BDNF; prenatal
ID PERVASIVE DEVELOPMENTAL DISORDERS; SPECTRUM DISORDERS;
MENTAL-RETARDATION; NEONATAL BLOOD; CHILDREN; SERUM; BDNF;
NEUROPEPTIDES; DEPRESSION; PREVALENCE
AB To investigate levels of brain-derived neurotrophic factor (BDNF) in mid-pregnancy and neonatal blood specimens as early biologic markers for autism, we conducted a population-based case-control study nested within the cohort of infants born from July 2000 to September 2001 to women who participated in the prenatal screening program in Orange County, CA. Cases (n = 84) were all children receiving services for autism at the Regional Center of Orange County. Two comparison groups from the same study population were included: children with mental retardation or developmental delay (n = 49) receiving services at the same regional center, and children not receiving services for developmental disabilities, randomly sampled from the California birth certificate files (n = 159), and frequency matched to autism cases on sex, birth year, and birth month. BDNF concentrations were measured in archived mid-pregnancy and neonatal blood specimens drawn during routine prenatal and newborn screening using a highly sensitive bead-based assay (Luminex, Biosource Human BDNF Antibody Bead Kit, Invitrogen-Biosource, Carlsbad, CA). The concentration of BDNF in maternal mid-pregnancy and neonatal specimens was similar across all three study groups. These data do not support previous findings of an association between BDNF and autism and suggest that the concentration of BDNF during critical periods of early neurodevelopment is not likely to be a useful biomarker for autism susceptibility.
C1 [Croen, Lisa A.; Yoshida, Cathleen K.; Fireman, Bruce] Kaiser Permanente No Calif, Div Res, Oakland, CA 94612 USA.
[Goines, Paula; Braunschweig, Daniel; Van de Water, Judy] Univ Calif Davis, Div Rheumatol Allergy & Clin Immunol, Davis, CA 95616 USA.
[Yolken, Robert] Johns Hopkins Univ, Div Dev Neurovirol, Baltimore, MD USA.
[Grether, Judith K.] Calif Dept Publ Hlth, Environm Hlth Invest Branch, Richmond, CA USA.
[Kharrazi, Martin] Calif Dept Publ Hlth, Genet Dis Screening Program, Richmond, CA USA.
[Hansen, Robin L.] Univ Calif Davis, MIND Inst, Davis, CA 95616 USA.
RP Croen, LA (reprint author), Kaiser Permanente No Calif, Div Res, 2000 Broadway, Oakland, CA 94612 USA.
EM Lisa.A.Croen@kp.org
FU National Institute of Mental Health [R01-MH72565]; National Alliance for
Autism Research [824/LC/01-201-004-00-00]; CaliforniaTobacco-Related
Disease Research Program [8RT-0115]
FX Funding was provided by grants from the National Institute of Mental
Health (R01-MH72565, L. Croen, PI), the National Alliance for Autism
Research (824/LC/01-201-004-00-00, L. Croen, PI), and the
CaliforniaTobacco-Related Disease Research Program (8RT-0115, M.
Kharrazi, PI). We thank Ron Torres for DNA amplification; Jack Collins,
Roxana Odouli, and Tiffany Wong for project coordination; Julie
Ruedaflores for record review and abstraction; Meredith Anderson and
Daniel Najjar for assistance with data management and analysis; and
Steve Graham and Debbie Hildebrandt for record linkage and specimen
retrieval.
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NR 23
TC 20
Z9 20
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD APR
PY 2008
VL 1
IS 2
BP 130
EP 137
DI 10.1002/aur.14
PG 8
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CI
UT WOS:000270031600007
PM 19119429
ER
PT J
AU Cook, EH
AF Cook, Edwin H.
TI Improved Resolution of Chromosomal Analysis through Microarray Studies
Leads to Identification of Relatively Large Chromosomal Deletion of
16p11.2 Associated with ASDs [Kumar et al., 2008; Marshall et al., 2008;
Weiss et al., 2008]
SO AUTISM RESEARCH
LA English
DT Review
ID AUTISM
CR Chiu PH, 2008, NEURON, V57, P463, DOI 10.1016/j.neuron.2007.12.020
Kumar RA, 2008, HUM MOL GENET, V17, P628, DOI 10.1093/hmg/ddm376
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Weiss LA, 2008, NEW ENGL J MED, V358, P667, DOI 10.1056/NEJMoa075974
NR 5
TC 0
Z9 0
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD APR
PY 2008
VL 1
IS 2
BP 138
EP 139
DI 10.1002/aur.16
PG 2
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CI
UT WOS:000270031600008
ER
PT J
AU Perrin, CJ
Perrin, SH
Hill, EA
DiNovi, K
AF Perrin, Christopher J.
Perrin, Stefanie H.
Hill, Elizabeth A.
DiNovi, Kristin
TI Brief functional anlaysis and treatment of elopement in preschoolers
with autism
SO BEHAVIORAL INTERVENTIONS
LA English
DT Article
ID BEHAVIOR; OUTCOMES
AB As a result of the impact of functional analysis methodologies in identifying maintaining variables of problem behavior, considerable research has been devoted to tailoring the methodologies to be practical for an outpatient setting. The purpose of this study was (a) to use brief functional analysis (BFA) methodologies to empirically demonstrate the function of elopement exhibited by two preschoolers diagnosed with autism and (b) to develop function-based interventions derived from the results of the BFA. In phase one, a BFA consisting of 5-min sessions with multiple repetitions of each condition was conducted with each participant. In phase two, the efficacy of function-based interventions was assessed using a pairwise design. Both participants' rates of elopement were differentially lower during the implementation of the function-based intervention relative to baseline. Collectively, these results demonstrated that brief functional methodologies could be applied successfully to the assessment and treatment of elopement. Moreover, the application of BFA and the ensuing treatment in an outpatient setting extend the existing research on functional analysis. Copyright (c) 2008 John Wiley & Sons, Ltd.
C1 [Perrin, Stefanie H.; Hill, Elizabeth A.] Bancroft NeuroHlth, Haddonfield, NJ USA.
RP Perrin, CJ (reprint author), 2463 Marblevista Blvd, Columbus, OH 43204 USA.
EM perrin.39@osu.edu
CR IWATA BA, 1994, J APPL BEHAV ANAL, V27, P215, DOI 10.1901/jaba.1994.27-215
Kahng SW, 1999, J APPL BEHAV ANAL, V32, P149, DOI 10.1901/jaba.1999.32-149
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Wallace MD, 1999, J APPL BEHAV ANAL, V32, P175, DOI 10.1901/jaba.1999.32-175
NR 7
TC 9
Z9 9
PU JOHN WILEY & SONS LTD
PI CHICHESTER
PA THE ATRIUM, SOUTHERN GATE, CHICHESTER PO19 8SQ, W SUSSEX, ENGLAND
SN 1072-0847
J9 BEHAV INTERVENT
JI Behav. Intervent.
PD APR
PY 2008
VL 23
IS 2
BP 87
EP 95
DI 10.1002/bin.256
PG 9
WC Psychology, Clinical
SC Psychology
GA 303UE
UT WOS:000256065600002
ER
PT J
AU Wilder, DA
Schadler, J
Higbee, TS
Haymes, LK
Bajagic, V
Register, M
AF Wilder, David A.
Schadler, John
Higbee, Thomas S.
Haymes, Linda K.
Bajagic, Verdrana
Register, Martisa
TI Identification of olfactory stimuli as reinforcers in individuals with
autism: A preliminary investigation
SO BEHAVIORAL INTERVENTIONS
LA English
DT Article
ID PREFERENCE
AB Preference for six olfactory stimuli was assessed via paired preference assessments with three adults with autism. Using a combination multielement and reversal design, a reinforcer assessment was then conducted to determine the extent to which three (high preference, medium preference, and low preference) of the six olfactory stimuli evaluated in the preference assessment functioned as reinforcers for responding in the context of a sorting task. Relative to baseline, all of the participants exhibited increased responding for access to their high preference olfactory stimulus. The utility of olfactory stimuli as components of behavior intervention plans is discussed. Copyright (c) 2008 John Wiley & Sons, Ltd.
C1 [Wilder, David A.; Schadler, John] Florida Inst Technol, Sch Psychol, Melbourne, FL 32901 USA.
[Higbee, Thomas S.] Utah State Univ, Logan, UT 84322 USA.
RP Wilder, DA (reprint author), Florida Inst Technol, Sch Psychol, 150 W Univ Blvd, Melbourne, FL 32901 USA.
EM dawilder@fit.edu
RI Higbee, Thomas/F-5157-2010
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NR 4
TC 3
Z9 3
PU JOHN WILEY & SONS LTD
PI CHICHESTER
PA THE ATRIUM, SOUTHERN GATE, CHICHESTER PO19 8SQ, W SUSSEX, ENGLAND
SN 1072-0847
J9 BEHAV INTERVENT
JI Behav. Intervent.
PD APR
PY 2008
VL 23
IS 2
BP 97
EP 103
DI 10.1002/bin.257
PG 7
WC Psychology, Clinical
SC Psychology
GA 303UE
UT WOS:000256065600003
ER
PT J
AU McNaughton, CH
Moon, J
Strawderman, MS
Maclean, KN
Evans, J
Strupp, BJ
AF McNaughton, Caitlyn H.
Moon, Jisook
Strawderman, Myla S.
Maclean, Kenneth N.
Evans, Jeffrey
Strupp, Barbara J.
TI Evidence for social anxiety and impaired social cognition in a mouse
model of Fragile X syndrome
SO BEHAVIORAL NEUROSCIENCE
LA English
DT Article
DE social behavior; fmr1 knockout mouse; Fragile X syndrome; autism; social
cognition
ID FMR1 KNOCKOUT MOUSE; MENTAL-RETARDATION; AUTISTIC BEHAVIOR; MUTANT
MOUSE; MICE; MALES; ABNORMALITIES; SUPPRESSION; PHENOTYPE; FEATURES
AB This study assessed social behavior in a mouse model of Fragile X syndrome (FXS), the Fmr1(im1Cgr) Fmr1 "knockout" (KO) mouse. Both the KO and wild-type (WT) mice preferred to be near a novel conspecific than to be alone. However, during the initial interaction with a novel conspecific, (1) a greater proportion of the KO mice exhibited high levels of grooming; and (2) the average duration of nose contact with the stimulus mouse was significantly shorter for the KO mice, both indicative of increased arousal and/or anxiety. Both groups exhibited a robust novelty preference when the novel animal was a "preferred" mouse. However, when the novel mouse was a "nonpreferred" animal, both groups showed a diminished novelty preference but this effect was more pronounced for the WT mice. This blunted negative reaction of the KO mice to a nonpreferred animal may indicate that they were less proficient than controls in distinguishing between positive and negative social interactions. These findings provide support for the use of this animal model to study the autistic features of FXS and autism spectrum disorders.
C1 [McNaughton, Caitlyn H.; Strupp, Barbara J.] Cornell Univ, Dept Psychol, Ithaca, NY 14853 USA.
[Moon, Jisook; Strawderman, Myla S.] Cornell Univ, Dept Nutr Sci, Ithaca, NY USA.
[Strupp, Barbara J.] Cornell Univ, Div Nutr Sci, Ithaca, NY 14853 USA.
[Maclean, Kenneth N.; Evans, Jeffrey] Univ Colorado, Hlth Sci Ctr, Boulder, CO 80309 USA.
RP Strupp, BJ (reprint author), Cornell Univ, Dept Psychol, Savage Hall, Ithaca, NY 14853 USA.
EM bjsl3@cornell.edu
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NR 35
TC 67
Z9 69
PU AMER PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST NE, WASHINGTON, DC 20002-4242 USA
SN 0735-7044
J9 BEHAV NEUROSCI
JI Behav. Neurosci.
PD APR
PY 2008
VL 122
IS 2
BP 293
EP 300
DI 10.1037/0735-7044A22.2.293
PG 8
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 283ZN
UT WOS:000254675100006
PM 18410169
ER
PT J
AU Brodkin, ES
AF Brodkin, Edward S.
TI Social behavior phenotypes in Fragile X syndrome, autism, and the Fmr1
knockout mouse: Theoretical comment on McNaughton et al. (2008)
SO BEHAVIORAL NEUROSCIENCE
LA English
DT Editorial Material
DE sociability; affiliative; anxiety; reward; cognition; genetic
ID SPECTRUM DISORDER; YOUNG MALES; METHODOLOGICAL ISSUES; INBRED STRAINS;
TASKS RELEVANT; MUTANT MOUSE; MICE; MODEL; CHILDREN; IDENTIFICATION
AB Individuals with fragile X syndrome (FXS) show varying degrees of social behavior disturbances, from social anxiety to autism. This variability of social behavior phenotypes in FXS is likely to be due to interactions of Fmr1 with other gene variants and environmental factors during brain development, although very little is known about the specific genetic and neural mechanisms involved. The Fmr1 knockout mouse is an important experimental resource for elucidating the neural mechanisms of social anxiety, social reward, and social cognition. However, studies of social behavior phenotypes in the Fmr1 knockout mouse are still in early stages. C. H. McNaughton et al. (2008) provide important new information on these phenotypes in the Fmr1 knockout mouse through their use of novel, detailed behavioral analysis to identify signs of increased social anxiety and social cognition deficits. Their significant refinements in measurement of social behavior phenotypes will help to advance future efforts to elucidate the genetic and neural mechanisms underlying social behavior disturbances in FXS and autism.
C1 Univ Penn, Sch Med, Translat Res Lab, Dept Psychiat, Philadelphia, PA 19104 USA.
RP Brodkin, ES (reprint author), Univ Penn, Sch Med, Translat Res Lab, Dept Psychiat, 125 S 31st St,Room 2220, Philadelphia, PA 19104 USA.
EM ebrodkin@mail.med.upenn.edu
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NR 92
TC 12
Z9 12
PU AMER PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST NE, WASHINGTON, DC 20002-4242 USA
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J9 BEHAV NEUROSCI
JI Behav. Neurosci.
PD APR
PY 2008
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AU Frazier, TW
Youngstrom, EA
Sinclair, L
Kubu, CS
Law, P
Rezai, A
AF Frazier, Thomas W.
Youngstrom, Eric A.
Sinclair, Leslie
Kubu, Cynthia S.
Law, Paul
Rezai, Ali
TI Autism spectrum disorders are a category, qualitatively distinct from
typical social communication
SO BIOLOGICAL PSYCHIATRY
LA English
DT Meeting Abstract
CT 63rd Annual Convention of the Society-of-Biological-Psychiatry
CY 2008
CL Washington, DC
SP Soc Biol Psychiat
C1 [Frazier, Thomas W.; Sinclair, Leslie] Cleveland Clin, Cleveland, OH 44106 USA.
[Youngstrom, Eric A.] Univ N Carolina, Chapel Hill, NC USA.
[Kubu, Cynthia S.; Rezai, Ali] Cleveland Clin, Ctr Neurol Restorat, Cleveland, OH USA.
[Law, Paul] Med Informat, Kennedy Krieger Inst Autism Speaks, Baltimore, MD USA.
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TC 0
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PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
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PD APR 1
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VL 63
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SU S
MA 94
BP 30S
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AU Hardan, AY
Keshavan, MS
Bansal, R
Fedorov, S
Minshew, NJ
AF Hardan, Antonio Y.
Keshavan, Matcheri S.
Bansal, Rahul
Fedorov, Serguei
Minshew, Nancy J.
TI Longitudinal study of the corpus callosum in autism
SO BIOLOGICAL PSYCHIATRY
LA English
DT Meeting Abstract
CT 63rd Annual Convention of the Society-of-Biological-Psychiatry
CY 2008
CL Washington, DC
SP Soc Biol Psychiat
C1 [Hardan, Antonio Y.] Stanford Univ, Stanford, CA 94305 USA.
[Keshavan, Matcheri S.; Bansal, Rahul; Fedorov, Serguei] Wayne State Univ, Detroit, MI USA.
[Minshew, Nancy J.] Univ Pittsburgh, Western Psychiat Inst & Clin, Pittsburgh, PA 15213 USA.
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Chaplin, W
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Soorya, L
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Hollander, E
AF Bartz, Jennifer
Cuellar, Jessica
Chaplin, William
Anagnostou, Evdokia
Soorya, Latha
Halpern, Danielle
Hollander, Eric
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interactions in adults with autism spectrum disorders
SO BIOLOGICAL PSYCHIATRY
LA English
DT Meeting Abstract
CT 63rd Annual Convention of the Society-of-Biological-Psychiatry
CY 2008
CL Washington, DC
SP Soc Biol Psychiat
C1 [Bartz, Jennifer; Cuellar, Jessica; Anagnostou, Evdokia; Soorya, Latha; Halpern, Danielle; Hollander, Eric] Mt Sinai Sch Med, New York, NY USA.
[Chaplin, William] St Johns Univ, Queens, NY USA.
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SO BIOLOGICAL PSYCHIATRY
LA English
DT Meeting Abstract
CT 63rd Annual Convention of the Society-of-Biological-Psychiatry
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CL Washington, DC
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AU Carpenter, KLH
Dichter, G
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AF Carpenter, Kimberly L. H.
Dichter, Gabriel
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DT Meeting Abstract
CT 63rd Annual Convention of the Society-of-Biological-Psychiatry
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C1 [Carpenter, Kimberly L. H.] Univ N Carolina, Chapel Hill, NC USA.
[Carpenter, Kimberly L. H.; Dichter, Gabriel; Belger, Aysenil] Duke UNC, Brain Imaging Anal Ctr, Durham, NC USA.
[Dichter, Gabriel; Belger, Aysenil] Univ N Carolina, Neurodev Disorders Res Ctr, Chapel Hill, NC USA.
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PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0006-3223
J9 BIOL PSYCHIAT
JI Biol. Psychiatry
PD APR 1
PY 2008
VL 63
IS 7
SU S
MA 612
BP 194S
EP 195S
PG 2
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 276SV
UT WOS:000254163700622
ER
PT J
AU Solomon, M
Frank, MJ
Kaluzhny, P
Ursu, S
Carter, CS
AF Solomon, Marjorie
Frank, Michael J.
Kaluzhny, Petrina
Ursu, Stefan
Carter, Cameron S.
TI Reward-based learning in autism spectrum disorders: Probabilistic
selection and transitive inference
SO BIOLOGICAL PSYCHIATRY
LA English
DT Meeting Abstract
CT 63rd Annual Convention of the Society-of-Biological-Psychiatry
CY 2008
CL Washington, DC
SP Soc Biol Psychiat
C1 [Solomon, Marjorie; Kaluzhny, Petrina; Ursu, Stefan; Carter, Cameron S.] Univ Calif Davis, Imaging Res Ctr, MIND Inst, Sacramento, CA 95817 USA.
[Frank, Michael J.] Univ Arizona, Dept Psychol, Program Neurosci cognit & Nrural Syst, Tucson, AZ USA.
NR 0
TC 0
Z9 0
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0006-3223
J9 BIOL PSYCHIAT
JI Biol. Psychiatry
PD APR 1
PY 2008
VL 63
IS 7
SU S
MA 690
BP 219S
EP 219S
PG 1
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 276SV
UT WOS:000254163700700
ER
PT J
AU Scheffer, IE
Turner, SJ
Dibbens, LM
Bayly, MA
Friend, K
Hodgson, B
Burrows, L
Shaw, M
Wei, C
Ullmann, R
Ropers, HH
Szepetowski, P
Haan, E
Mazarib, A
Afawi, Z
Neufeld, M
Andrews, PI
Wallace, G
Kivity, S
Lev, D
Lerman-Sagie, T
Derry, CP
Korczyn, AD
Gecz, J
Mulley, JC
Berkovic, SF
AF Scheffer, Ingrid E.
Turner, Samantha J.
Dibbens, Leanne M.
Bayly, Marta A.
Friend, Kathryn
Hodgson, Bree
Burrows, Linda
Shaw, Marie
Wei, Chen
Ullmann, Reinhard
Ropers, Hans-Hilger
Szepetowski, Pierre
Haan, Eric
Mazarib, Aziz
Afawi, Zaid
Neufeld, MiriamY.
Andrews, P. Ian
Wallace, Geoffrey
Kivity, Sara
Lev, Dorit
Lerman-Sagie, Tally
Derry, Christopher P.
Korczyn, Amos D.
Gecz, Jozef
Mulley, John C.
Berkovic, Samuel F.
TI Epilepsy and mental retardation limited to females: an under-recognized
disorder
SO BRAIN
LA English
DT Article
DE epilepsy; intellectual disability; females; X-linked inheritance;
autistic features
ID BROADER AUTISM PHENOTYPE; CRANIOFRONTONASAL SYNDROME; CONVULSIVE
DISORDER; FAMILIAL FORM; CLASSIFICATION; SEIZURES; MUTATIONS; PROPOSAL
AB Epilepsy and Mental Retardation limited to Females (EFMR) which links to Xq22 has been reported in only one family. We aimed to determine if there was a distinctive phenotype that would enhance recognition of this disorder. We ascertained four unrelated families (two Australian, two Israeli) where seizures in females were transmitted through carrier males. Detailed clinical assessment was performed on 58 individuals, using a validated seizure questionnaire, neurological examination and review of EEG and imaging studies. Gene localization was examined using Xq22 microsatellite markers. Twenty-seven affected females had a mean seizure onset of 14 months (range 6-36) typically presenting with convulsions. All had convulsive attacks at some stage, associated with fever in 17 out of 27 (63%). Multiple seizure types occurred including tonic-clonic (26), tonic (4), partial (II), absence (5), atonic (3) and myoclonic (4). Seizures ceased at mean 12 years. Developmental progress varied from normal (7), to always delayed (4) to normal followed by regression (12). Intellect ranged from normal to severe intellectual disability (ID), with 67% of females having ID or being of borderline intellect. Autistic (6), obsessive (9) and aggressive (7) features were prominent. EEGs showed generalized and focal epileptiform abnormalities. Five obligate male carriers had obsessional tendencies. Linkage to Xq22 was confirmed (maximum lod 3.5 at theta=0). We conclude that EFMR is a distinctive, under-recognized familial syndrome where girls present with convulsions in infancy, often associated with intellectual impairment and autistic features. The unique inheritance pattern with transmission by males is perplexing. Clinical recognition is straightforward in multiplex families due to the unique inheritance pattern; however, this disorder should be considered in smaller families where females alone have seizures beginning in infancy, particularly in the setting of developmental delay. In single cases, diagnosis will depend on identification of the molecular basis.
C1 [Scheffer, Ingrid E.; Turner, Samantha J.; Derry, Christopher P.; Berkovic, Samuel F.] Univ Melbourne, Heidelberg Repatriat Hosp, Dept Med, Epilepsy Res Ctr, Heidelberg, Vic 3081, Australia.
[Scheffer, Ingrid E.] Univ Melbourne, Royal Childrens Hosp, Dept Paediat, Melbourne, Vic, Australia.
[Dibbens, Leanne M.; Bayly, Marta A.; Friend, Kathryn; Hodgson, Bree; Burrows, Linda; Shaw, Marie; Haan, Eric; Gecz, Jozef; Mulley, John C.] Womens & Childrens Hosp, Dept Med Genet, Adelaide, SA, Australia.
[Dibbens, Leanne M.; Gecz, Jozef; Mulley, John C.] Sch Paediat & Reprod Hlth, Adelaide, SA, Australia.
[Wei, Chen; Ullmann, Reinhard; Ropers, Hans-Hilger] Max Planck Inst Mol Genet, Dept Human Mol Genet, Berlin, Germany.
[Szepetowski, Pierre] Univ Aix Marseille 2, Fac Med Timone, Genet Human Epilepsies Grp, INSERM,UMR 491, Marseille, France.
[Mazarib, Aziz; Afawi, Zaid; Neufeld, MiriamY.; Korczyn, Amos D.] Tel Aviv Sourasky Med Ctr, Dept Neurol, Tel Aviv, Israel.
[Mazarib, Aziz; Afawi, Zaid; Neufeld, MiriamY.; Korczyn, Amos D.] Tel Aviv Univ, Sackler Fac Med, Tel Aviv, Israel.
[Andrews, P. Ian] Sydney Childrens Hosp, Randwick, NSW, Australia.
[Wallace, Geoffrey] Mater Med Ctr, Brisbane, Qld, Australia.
[Kivity, Sara] Schneider Childrens Med Ctr, Dept Neurol, Petah Tiqwa, Israel.
[Lev, Dorit; Lerman-Sagie, Tally] Wolfson Med Ctr, Metab Neurogenet Clin, Holon, Israel.
[Gecz, Jozef; Mulley, John C.] Univ Adelaide, Sch Mol & Biomed Sci, Adelaide, SA 5005, Australia.
RP Scheffer, IE (reprint author), Univ Melbourne, Heidelberg Repatriat Hosp, Dept Med, Epilepsy Res Ctr, Neurosci Bldg,Level 1,Banksi St, Heidelberg, Vic 3081, Australia.
EM scheffer@unimelb.edu.au
RI Scheffer, Ingrid/G-1668-2013; Chen, Wei/C-1273-2011
OI Scheffer, Ingrid/0000-0002-2311-2174;
CR DREIFUSS FE, 1985, EPILEPSIA, V26, P268
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NR 18
TC 58
Z9 59
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0006-8950
J9 BRAIN
JI Brain
PD APR
PY 2008
VL 131
BP 918
EP 927
DI 10.1093/brain/awm338
PN 4
PG 10
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 284CH
UT WOS:000254682300005
PM 18234694
ER
PT J
AU van Kooten, IAJ
Palmen, SJMC
von Cappeln, P
Steinbusch, HWM
Korr, H
Heinsen, H
Hof, PR
van Engeland, H
Schmitz, C
AF van Kooten, Imke A. J.
Palmen, Saskia J. M. C.
von Cappeln, Patricia
Steinbusch, Harry W. M.
Korr, Hubert
Heinsen, Helmut
Hof, Patrick R.
van Engeland, Herman
Schmitz, Christoph
TI Neurons in the fusiform gyrus are fewer and smaller in autism
SO BRAIN
LA English
DT Article
DE fusiform gyrus; design-based stereology; autism
ID HIGH-FUNCTIONING AUTISM; FACE AREA; SPECTRUM DISORDER; ORBITOFRONTAL
CORTEX; MENTAL-RETARDATION; NEURAL SYSTEMS; BRAIN VOLUME; AMYGDALA;
PERCEPTION; SCHIZOPHRENIA
AB Abnormalities in face perception are a core feature of social disabilities in autism. Recent functional magnetic resonance imaging studies showed that patients with autism could perform face perception tasks. However, the fusiform gyrus (FG) and other cortical regions supporting face processing in controls are hypoactive in patients with autism. The neurobiological basis of this phenomenon is unknown. Here, we tested the hypothesis that the FG shows neuropathological alterations in autism, namely alterations in neuron density, total neuron number and mean perikaryal volume. We investigated the FG (analysing separately layers II, III, IV, V and VI), in seven post-mortem brains from patients with autism and 10 controls for volume, neuron density, total neuron number and mean perikaryal volume with high-precision design-based stereology. To determine whether these results were specific for the FG, the same analyses were also performed in the primary visual cortex and in the cortical grey matter as a whole. Compared to controls, patients with autism showed significant reductions in neuron densities in layer III, total neuron numbers in layers III, V and VI, and mean perikaryal volumes of neurons in layers V and VI in the FG. None of these alterations were found in the primary visual cortex or in the whole cerebral cortex. Although based on a relatively small sample of post-mortem brains from patients with autism and controls, the results of the present study may provide important insight about the cellular basis of abnormalities in face perception in autism.
C1 [van Kooten, Imke A. J.; Steinbusch, Harry W. M.; Schmitz, Christoph] Maastricht Univ, Div Cellular Neurosci, Dept Psychiat & Neuropsychol, NL-6200 MD Maastricht, Netherlands.
[van Kooten, Imke A. J.; Steinbusch, Harry W. M.; Schmitz, Christoph] European Grad Sch Neurosci EURON, Maastricht, Netherlands.
[van Kooten, Imke A. J.; Palmen, Saskia J. M. C.; van Engeland, Herman] Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, Utrecht, Netherlands.
[von Cappeln, Patricia; Korr, Hubert] Rhein Westfal TH Aachen, Dept Anat & Cell Biol, Aachen, Germany.
[Heinsen, Helmut] Univ Wurzburg, Morphol Brain Res Unit, Wurzburg, Germany.
[Hof, Patrick R.] Mt Sinai Sch Med, Dept Neurosci, New York, NY USA.
RP Schmitz, C (reprint author), Maastricht Univ, Div Cellular Neurosci, Dept Psychiat & Neuropsychol, POB 616, NL-6200 MD Maastricht, Netherlands.
EM c.schmitz@np.unimaas.nl
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NR 86
TC 77
Z9 78
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0006-8950
J9 BRAIN
JI Brain
PD APR
PY 2008
VL 131
BP 987
EP 999
DI 10.1093/brain/awn033
PN 4
PG 13
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 284CH
UT WOS:000254682300011
PM 18332073
ER
PT J
AU Kleinhans, NM
Richards, T
Sterling, L
Stegbauer, KC
Mahurin, R
Johnson, LC
Greenson, J
Dawson, G
Aylward, E
AF Kleinhans, Natalia M.
Richards, Todd
Sterling, Lindsey
Stegbauer, Keith C.
Mahurin, Roderick
Johnson, L. Clark
Greenson, Jessica
Dawson, Geraldine
Aylward, Elizabeth
TI Abnormal functional connectivity in autism spectrum disorders during
face processing
SO BRAIN
LA English
DT Article
DE autism; Asperger's disorder; functional connectivity; face processing;
brain imaging
ID WHITE-MATTER; BRAIN SIZE; SENTENCE COMPREHENSION; EMOTION RECOGNITION;
FACIAL EXPRESSIONS; HEAD CIRCUMFERENCE; ASPERGER-SYNDROME; SOCIAL
COGNITION; CORPUS-CALLOSUM; VISUAL PATHWAYS
AB Abnormalities in the interactions between functionally linked brain regions have been suggested to be associated with the clinical impairments observed in autism spectrum disorders (ASD). We investigated functional connectivity within the limbic system during face identification; a primary component of social cognition, in 19 high-functioning adults with ASD and 21 age-and IQ-matched control adults. Activation during identification of previously viewed faces and houses using a one-back paradigm was compared. The fusiform face area (FFA) was individually localized in each participant and used as the seed point for functional connectivity analyses. The degree of correlation between FFA and the extended neural circuitry involved in face identification was tested. A whole brain analysis was also conducted in order to determine whether connectivity from the FFA to aberrant brain locations was present in the ASD group. Measures of clinical severity (ADOS social score and ADI-R social score) were included as independent variables into the functional connectivity analyses. Significant FFA-amygdala and FFA-superior temporal sulcus functional connectivity was found in both the ASD and control participants. However, the control group had significantly increased connectivity to the left amygdala and the posterior cingulate compared to ASD. Post hoc analyses additionally found increased connectivity to the thalamus in the controls. A significant relationship between abnormal functional connectivity and clinical severity in the ASD group was observed. Specifically, greater social impairment was associated with reduced FFA-amygdala connectivity and increased FFA-right inferior frontal connectivity. These results suggest that abnormal neural connections within the limbic system may contribute to the social impairments observed in ASD.
C1 [Kleinhans, Natalia M.; Richards, Todd; Stegbauer, Keith C.; Mahurin, Roderick; Aylward, Elizabeth] Univ Washington, Dept Radiol, Seattle, WA 98195 USA.
[Johnson, L. Clark] Univ Washington, Dept Psychosocial & Community Hlth, Seattle, WA 98195 USA.
[Sterling, Lindsey; Dawson, Geraldine] Univ Washington, Dept Psychol, Seattle, WA 98195 USA.
[Greenson, Jessica] Univ Washington, Ctr Human Dev & Disabil, Seattle, WA 98195 USA.
RP Kleinhans, NM (reprint author), Univ Washington, Dept Radiol, Box 357115, Seattle, WA 98195 USA.
EM nkleinha@u.washington.edu
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NR 85
TC 198
Z9 202
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0006-8950
J9 BRAIN
JI Brain
PD APR
PY 2008
VL 131
BP 1000
EP 1012
DI 10.1093/brain/awm334
PN 4
PG 13
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 284CH
UT WOS:000254682300012
PM 18234695
ER
PT J
AU Vandenbroucke, MWG
Scholte, HS
van Engeland, H
Lamme, VAF
Kemner, C
AF Vandenbroucke, Myriam W. G.
Scholte, H. Steven
van Engeland, Herman
Lamme, Victor A. F.
Kemner, Chantal
TI A neural substrate for atypical low-level visual processing in autism
spectrum disorder
SO BRAIN
LA English
DT Article
DE asperger; horizontal connections; lateral inhibition; GABA; minicolumns
ID FIGURE-GROUND SEGREGATION; FUNCTIONAL ARCHITECTURE; MACAQUE MONKEY;
STRIATE CORTEX; DISTINCT MODES; INDIVIDUALS; V1; ABNORMALITIES;
CONNECTIONS; FEEDFORWARD
AB An important characteristic of autism spectrum disorder (ASD) is increased visual detail perception. Yet, there is no standing neurobiological explanation for this aspect of the disorder. We show evidence from EEG data, from 31 control subjects (three females) and 13 subjects (two females) aged 16-28 years, for a specific impairment in object boundary detection in ASD, which is present as early as 120 ms after stimulus presentation. In line with a neural network model explicating the role of feedforward, horizontal and recurrent processing in visual perception, we can attribute this deficit to a dysfunction of horizontal connections within early visual areas. Interestingly, ASD subjects showed an increase in subsequent activity at lateral occipital sites (225 ms), which might reflect a compensational mechanism. In contrast, recurrent processing between higher and lower visual areas (around 260 ms), associated with the segregation between figure and background, was normal. Our results show specific neural abnormalities in ASD related to low-level visual processing. In addition, given the reconciliation between our findings and previous neuropathology and neurochemistry research, we suggest that atypical horizontal interactions might reflect a more general neural abnormality in this disorder.
C1 [Vandenbroucke, Myriam W. G.; van Engeland, Herman; Kemner, Chantal] Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, NL-3584 CX Utrecht, Netherlands.
[Vandenbroucke, Myriam W. G.; Scholte, H. Steven; Lamme, Victor A. F.] Univ Amsterdam, Dept Psychol, Cognit Neurosci Grp, Amsterdam, Netherlands.
[Lamme, Victor A. F.] Netherlands Inst Neurosci, Amsterdam, Netherlands.
[Kemner, Chantal] Maastricht Univ, Fac Psychol, Dept Neurocognit, Maastricht, Netherlands.
RP Vandenbroucke, MWG (reprint author), Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, B01-201 Heidelberglaan 100, NL-3584 CX Utrecht, Netherlands.
EM m.w.g.vandenbroucke@umcutrecht.nl
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NR 47
TC 43
Z9 43
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0006-8950
J9 BRAIN
JI Brain
PD APR
PY 2008
VL 131
BP 1013
EP 1024
DI 10.1093/brain/awm321
PN 4
PG 12
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 284CH
UT WOS:000254682300013
PM 18192288
ER
PT J
AU Hayashi, M
Kato, M
Igarashi, K
Kashima, H
AF Hayashi, Mika
Kato, Motoichiro
Igarashi, Kazue
Kashima, Haruo
TI Superior fluid intelligence in children with Asperger's disorder
SO BRAIN AND COGNITION
LA English
DT Article
DE general fluid intelligence; Raven's progressive matrices test; abstract
reasoning ability
ID PROGRESSIVE MATRICES TEST; WORKING-MEMORY CAPACITY; HIGH-FUNCTIONING
AUTISM; GENERAL INTELLIGENCE; PERSPECTIVE; ACTIVATION; ATTENTION;
ACCOUNT; CORTEX
AB Asperger's disorder is one of autistic spectrum disorders; sharing clinical features with autism, but without developmental delay in language acquisition. There have been some studies of intellectual functioning in autism so far, but very few in Asperger's disorder. In the present study, we investigated abstract reasoning ability, whose form of intelligence has been labeled fluid intelligence in the theory of Cattell [Cattell, R. B. (1963). Theory of fluid and crystallized intelligence: A critical experiment. Journal of Educational Psychology, 54, 1-22.], in children with Asperger's disorder. A test of fluid intelligence, the Raven's Standard Progressive Matrices Test, was administered to 17 children with Asperger's disorder and 17 age-, gender-, and FIQ-matched normal children. The results showed that children with Asperger's disorder outperformed on the test of fluid reasoning than typically developing children. We suggest that individuals with Asperger's disorder have higher fluid reasoning ability than normal individuals, highlighting superior fluid intelligence. (c) 2007 Elsevier Inc. All rights reserved.
C1 [Hayashi, Mika; Kato, Motoichiro; Kashima, Haruo] Keio Univ, Sch Med, Dept Neuropsychiat, Shinjuku Ku, Tokyo 1608582, Japan.
[Igarashi, Kazue] Shirayuri Coll, Dept Child Culture, Tokyo 1828525, Japan.
RP Hayashi, M (reprint author), Keio Univ, Sch Med, Dept Neuropsychiat, Shinjuku Ku, 35 Shinanomachi, Tokyo 1608582, Japan.
EM di055010@sc.itc.keio.ac.jp
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NR 30
TC 40
Z9 41
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0278-2626
J9 BRAIN COGNITION
JI Brain Cogn.
PD APR
PY 2008
VL 66
IS 3
BP 306
EP 310
DI 10.1016/j.bandc.2007.09.008
PG 5
WC Neurosciences; Psychology, Experimental
SC Neurosciences & Neurology; Psychology
GA 288BN
UT WOS:000254961300011
PM 17980944
ER
PT J
AU Zafeiriou, DI
Ververi, A
Salomons, GS
Vargiami, E
Haas, D
Papadopoulou, V
Kontopoulos, E
Jakobs, C
AF Zafeiriou, D. I.
Ververi, A.
Salomons, G. S.
Vargiami, E.
Haas, D.
Papadopoulou, V.
Kontopoulos, E.
Jakobs, C.
TI L-2-hydroxyglutaric aciduria presenting with severe autistic features
SO BRAIN & DEVELOPMENT
LA English
DT Article
DE L-2-hydroxyglutaric aciduria; autism; macrocephaly; leukodystrophy;
L2HGDH
AB L-2-Hydroxyglutaric aciduria (L-2-HGA) is an autosomal recessive neurometabolic disorder characterized by psychomotor delay, ataxia, macrocephaly and typical neuroradiological findings of subcortical leucoencephalopathy. Recently, the disease causing gene has been discovered (L2HGDH) encoding L-2-hydroxyglutarate dehydrogenase. We present a 3-year-old boy With L-2-HGA, who demonstrated macrocephaly, noted already in utero with ultrasound. Cranial MRI demonstrated diffuse subcortical encephalopathy with increased signal of the subcortical white matter. Subsequent metabolic screening revealed increased levels Of L-2-HGA, and genomic DNA analysis demonstrated two missense mutations in L-2-HGDG. Patient's further motor development was mildly impaired, whilst his speech development was profoundly impaired (first words at the age of 2 years). Since the age of 2 years he started demonstrating autistic repetitive behaviors and movements, increasing aloofness to his environment and limitations in the variety of spontaneous activity (CARS score: 44/60-severe autism). Autism has not so far been described in L-2-HGA and may be considered as an additional feature of the phenotypic spectrum. (c) 2007 Elsevier B.V. All rights reserved.
C1 [Zafeiriou, D. I.; Ververi, A.; Vargiami, E.; Papadopoulou, V.; Kontopoulos, E.] Aristotle Univ Thessaloniki, Dept Pediat 1, Thessaloniki 54622, Greece.
[Salomons, G. S.; Jakobs, C.] Vrije Univ Amsterdam, Med Ctr, Dept Clin Chem, Metab Unit, Amsterdam, Netherlands.
[Haas, D.] Univ Hosp Pediat & Adolescent Med, Div Metab Dis, Heidelberg, Germany.
RP Zafeiriou, DI (reprint author), Aristotle Univ Thessaloniki, Dept Pediat 1, Egnatia St 106, Thessaloniki 54622, Greece.
EM jeff@med.auth.gr
RI Zafeiriou, Dimitrios/N-2641-2013
OI Zafeiriou, Dimitrios/0000-0003-2187-9299
CR Keller TA, 2007, NEUROREPORT, V18, P23, DOI 10.1097/01.wnr.0000239965.21685.99
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Topcu M, 2005, TURKISH J PEDIATR, V47, P1
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Zafeiriou DI, 2001, BRAIN DEV-JPN, V23, P255, DOI 10.1016/S0387-7604(01)00206-6
NR 5
TC 8
Z9 8
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0387-7604
J9 BRAIN DEV-JPN
JI Brain Dev.
PD APR
PY 2008
VL 30
IS 4
BP 305
EP 307
DI 10.1016/j.braindev.2007.09.005
PG 3
WC Clinical Neurology
SC Neurosciences & Neurology
GA 283ZL
UT WOS:000254674900012
PM 17981416
ER
PT J
AU Rinaldi, T
Silberberg, G
Markram, H
AF Rinaldi, Tania
Silberberg, Gilad
Markram, Henry
TI Hyperconnectivity of local neocortical microcircuitry induced by
prenatal exposure to valproic acid
SO CEREBRAL CORTEX
LA English
DT Article
DE autism; connectivity; in vitro electrophysiology; juvenile rat;
somatosensory cortex; synaptic strength
ID PYRAMIDAL NEURONS; AUTISM; CORTEX; BRAIN; RATS; MODEL
AB Exposure to valproic acid (VPA) during embryogenesis can cause several teratogenic effects, including developmental delays and in particular autism in humans if exposure occurs during the third week of gestation. We examined the postnatal effects of embryonic exposure to VPA on microcircuit properties of juvenile rat neocortex using in vitro electrophysiology. We found that a single prenatal injection of VPA on embryonic day 11.5 causes a significant enhancement of the local recurrent connectivity formed by neocortical pyramidal neurons. The study of the biophysical properties of these connections revealed weaker excitatory synaptic responses. A marked decrease of the intrinsic excitability of pyramidal neurons was also observed. Furthermore, we demonstrate a diminished number of putative synaptic contacts in connection between layer 5 pyramidal neurons. Local hyperconnectivity may render cortical modules more sensitive to stimulation and once activated, more autonomous, isolated, and more difficult to command. This could underlie some of the core symptoms observed in humans prenatally exposed to valproic acid.
C1 [Rinaldi, Tania; Silberberg, Gilad; Markram, Henry] Ecole Polytech Fed Lausanne, Brain Mind Inst, Lab Neural Microcircuitry, CH-1015 Lausanne, Switzerland.
[Silberberg, Gilad] Karolinska Inst, Dept Neurosci, Nobel Inst Neurophysiol, Stockholm, Sweden.
RP Markram, H (reprint author), Ecole Polytech Fed Lausanne, Brain Mind Inst, Lab Neural Microcircuitry, CH-1015 Lausanne, Switzerland.
EM henry.markram@epfl.ch
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NR 29
TC 59
Z9 61
PU OXFORD UNIV PRESS INC
PI CARY
PA JOURNALS DEPT, 2001 EVANS RD, CARY, NC 27513 USA
SN 1047-3211
J9 CEREB CORTEX
JI Cereb. Cortex
PD APR
PY 2008
VL 18
IS 4
BP 763
EP 770
DI 10.1093/cercor/bhm117
PG 8
WC Neurosciences
SC Neurosciences & Neurology
GA 274NL
UT WOS:000254007900004
PM 17638926
ER
PT J
AU Takahashi, H
Matsuura, M
Koeda, M
Yahata, N
Suhara, T
Kato, M
Okubo, Y
AF Takahashi, Hidehiko
Matsuura, Masato
Koeda, Michihiko
Yahata, Noriaki
Suhara, Tetsuya
Kato, Motoichiro
Okubo, Yoshiro
TI Brain activations during judgments of positive self-conscious emotion
and positive basic emotion: Pride and joy
SO CEREBRAL CORTEX
LA English
DT Article
DE medial prefrontal cortex; positive emotions; pride; superior temporal
sulcus; theory of mind; ventral striatum
ID SOCIAL COGNITION; FMRI; MIND; REWARD; MECHANISMS; AUTISM;
REPRESENTATION; PERCEPTION; DOPAMINE; APPETITE
AB We aimed to investigate the neural correlates associated with judgments of a positive self-conscious emotion, pride, and elucidate the difference between pride and a basic positive emotion, joy, at the neural basis level using functional magnetic resonance imaging. Study of the neural basis associated with pride might contribute to a better understanding of the pride-related behaviors observed in neuropsychiatric disorders. Sixteen healthy volunteers were studied. The participants read sentences expressing joy or pride contents during the scans. Pride conditions activated the right posterior superior temporal sulcus and left temporal pole, the regions implicated in the neural substrate of social cognition or theory of mind. However, against our prediction, we did not find brain activation in the medial prefrontal cortex, a region responsible for inferring others' intention or self-reflection. Joy condition produced activations in the ventral striatum and insula/operculum, the key nodes of processing of hedonic or appetitive stimuli. Our results support the idea that pride is a self-conscious emotion, requiring the ability to detect the intention of others. At the same time, judgment of pride might require less self-reflection compared with those of negative self-conscious emotions such as guilt or embarrassment.
C1 [Takahashi, Hidehiko; Suhara, Tetsuya] Natl Inst Radiol Sci, Dept Mol Neuroimaging, Mol Imaging Ctr, Inage Ku, Chiba 2638555, Japan.
[Takahashi, Hidehiko] Asai Hosp, Dept Psychiat, Tougane, Japan.
[Matsuura, Masato] Tokyo Med & Dent Univ, Grad Sch Hlth Sci, Dept Life Sci & Bioinformat, Tokyo, Japan.
[Okubo, Yoshiro] Nippon Med Coll, Dept Neuropsychiat, Tokyo, Japan.
[Yahata, Noriaki] Nippon Med Coll, Dept Pharmacol, Tokyo, Japan.
[Kato, Motoichiro] Keio Univ, Sch Med, Dept Neuropsychiat, Tokyo, Japan.
RP Takahashi, H (reprint author), Natl Inst Radiol Sci, Dept Mol Neuroimaging, Mol Imaging Ctr, Inage Ku, 9-1 4-Chome, Chiba 2638555, Japan.
EM hidehiko@nirs.go.jp
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NR 54
TC 55
Z9 58
PU OXFORD UNIV PRESS INC
PI CARY
PA JOURNALS DEPT, 2001 EVANS RD, CARY, NC 27513 USA
SN 1047-3211
J9 CEREB CORTEX
JI Cereb. Cortex
PD APR
PY 2008
VL 18
IS 4
BP 898
EP 903
DI 10.1093/cercor/bhm120
PG 6
WC Neurosciences
SC Neurosciences & Neurology
GA 274NL
UT WOS:000254007900016
PM 17638925
ER
PT J
AU Gernsbacher, MA
Stevenson, JL
Khandakar, S
Goldsmith, HH
AF Gernsbacher, Morton Ann
Stevenson, Jennifer L.
Khandakar, Suraiya
Goldsmith, H. Hill
TI Why Does Joint Attention Look Atypical in Autism?
SO CHILD DEVELOPMENT PERSPECTIVES
LA English
DT Article
DE autism; enhanced perceptual processing; gaze dyspraxia; joint attention;
intentionality; pointing
ID SPECTRUM DISORDERS; YOUNG-CHILDREN; PRESCHOOL-CHILDREN; EYE GAZE;
ENHANCED DISCRIMINATION; SOCIAL BEHAVIORS; HEAD MOVEMENTS; INFANT;
PERCEPTION; INTERVENTION
AB This essay answers the question of why autistic children are less likely to initiate joint attention (e.g., use their index finger to point to indicate interest in something) and why they are less likely to respond to bids for their joint attention (e.g., turn their heads to look at something to which another person points). It reviews empirical evidence that autistic toddlers, children, adolescents, and adults can attend covertly, even to social stimuli, such as the direction in which another person's eyes are gazing. It also reviews empirical evidence that autistics Of various ages understand the intentionality of other persons' actions. The essay suggests that autistics' atypical resistance to distraction, atypical skill at parallel perception, and atypical execution of volitional actions underlie their atypical manifestations of joint attention.
C1 [Gernsbacher, Morton Ann] Univ Wisconsin, Dept Psychol, Madison, WI 53706 USA.
RP Gernsbacher, MA (reprint author), Univ Wisconsin, Dept Psychol, 1202 W Johnson St, Madison, WI 53706 USA.
EM magernsb@wisc.edu
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NR 80
TC 14
Z9 14
PU WILEY-BLACKWELL
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN 07030-5774, NJ USA
SN 1750-8592
EI 1750-8606
J9 CHILD DEV PERSPECT
JI Child Develop. Perspect.
PD APR
PY 2008
VL 2
IS 1
BP 38
EP 45
DI 10.1111/j.1750-8606.2008.00039.x
PG 8
WC Psychology, Developmental
SC Psychology
GA 499YH
UT WOS:000270262300007
ER
PT J
AU Burack, JA
Russo, N
AF Burack, Jacob A.
Russo, Natalie
TI On Why Joint Attention Might Look Atypical in Autism: A Case for a
Strong Policy Statement but More Nuanced Empirical Story
SO CHILD DEVELOPMENT PERSPECTIVES
LA English
DT Article
DE autism; development; methodology; attention
ID CHILDREN; VARIABLES; DEFICITS
AB In the present response to Gernsbacher, Stevenson, Khandakar, and Goldsmith (2008), we support the positivistic and strength-based perspective taken by the authors in understanding the abilities and skills of persons with autism. However, we argue that a more tempered approach one that encompasses a developmental perspective, as well as a more comprehensive review of both the supporting and the contradictory empirical evidence-is warranted in advancing their conclusions.
C1 [Burack, Jacob A.; Russo, Natalie] McGill Univ, Montreal, PQ H3A 2T5, Canada.
[Russo, Natalie] CUNY City Coll, New York, NY USA.
RP Burack, JA (reprint author), Dept Educ & Counseling Psychol, 3700 McTavish St, Montreal, PQ H3A 1Y2, Canada.
EM jake.burack@mcgill.ca
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NR 13
TC 1
Z9 1
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1750-8592
J9 CHILD DEV PERSPECT
JI Child Develop. Perspect.
PD APR
PY 2008
VL 2
IS 1
BP 46
EP 48
DI 10.1111/j.1750-8606.2008.00040.x
PG 3
WC Psychology, Developmental
SC Psychology
GA 499YH
UT WOS:000270262300008
ER
PT J
AU Gernsbacher, MA
Stevenson, JL
Khandakar, S
Goldsmith, HH
AF Gernsbacher, Morton Ann
Stevenson, Jennifer L.
Khandakar, Suraiya
Goldsmith, H. Hill
TI Autistics' Atypical Joint Attention: Policy Implications and Empirical
Nuance
SO CHILD DEVELOPMENT PERSPECTIVES
LA English
DT Article
DE autism; enhanced perceptual processing; gaze dyspraxia; joint attention;
intentionality; pointing
ID VISUAL-SEARCH; SPECTRUM DISORDERS; CHILDREN; OTHERS; GAZE; INTENTIONS;
ARROW; CUES; EYES
AB Burack and Russo (2008) applaud our approach to understanding autistics' atypical joint attention (Gernsbacher, Stevenson, Khandakar, & Goldsmith, 2008) but express some concerns about the evidence we drew upon to support our thesis. In response, we underscore the empirical nuance of our thesis-that autistics' atypical manifestations of joint attention arise from their atypical resistance to distraction, atypical parallel perception, and atypical execution of volitional actions. We recap how our hypothesis derives from fresh interpretations, well-replicated findings, and underlying mechanisms.
C1 [Gernsbacher, Morton Ann] Univ Wisconsin, Dept Psychol, Madison, WI 53706 USA.
RP Gernsbacher, MA (reprint author), Univ Wisconsin, Dept Psychol, 1202 W Johnson St, Madison, WI 53706 USA.
EM magernsb@wisc.edu
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*US DEP HHS, 2003, ANT DO YOU KNOW FACT
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YOUNG GS, 2007, BIENN M SOC RES CHIL
NR 43
TC 3
Z9 3
PU WILEY-BLACKWELL
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN 07030-5774, NJ USA
SN 1750-8592
EI 1750-8606
J9 CHILD DEV PERSPECT
JI Child Develop. Perspect.
PD APR
PY 2008
VL 2
IS 1
BP 49
EP 52
DI 10.1111/j.1750-8606.2008.00041.x
PG 4
WC Psychology, Developmental
SC Psychology
GA 499YH
UT WOS:000270262300009
ER
PT J
AU Nijmeijer, JS
Minderaa, RB
Buitelaar, JK
Mulligan, A
Hartman, CA
Hoekstra, PJ
AF Nijmeijer, Judith S.
Minderaa, Ruud B.
Buitelaar, Jan K.
Mulligan, Aisling
Hartman, Catharina A.
Hoekstra, Pieter J.
TI Attention-deficit/hyperactivity disorder and social dysfunctioning
SO CLINICAL PSYCHOLOGY REVIEW
LA English
DT Review
ID DEFICIT-HYPERACTIVITY DISORDER; PERVASIVE DEVELOPMENTAL DISORDER; AUTISM
SPECTRUM DISORDERS; CHILDRENS-COMMUNICATION-CHECKLIST;
SCHOOL-AGE-CHILDREN; ADULT FOLLOW-UP; CONDUCT PROBLEMS;
GENERAL-POPULATION; EXECUTIVE FUNCTION; PEER RELATIONSHIPS
AB Attention-deficit/hyperactivity disorder (ADHD) is associated with functional impairments in different areas of daily life. One such area is social functioning. The purpose of this paper is to critically review research on social dysfunctioning in children with ADHD. Children with ADHD often have conflicts with adults and peers, and suffer from unpopularity, rejection by peers, and a lack of friendships, in part as a consequence of their ADHD symptoms. Comorbid oppositional defiant or conduct disorder aggravates these impairments. In some cases the inadequate social behavior of children with ADHD may be phenomenologically and etiologically related to pervasive developmental disorders (PDD). However, the causes and consequences of PDD symptoms in ADHD are understudied. Also, the relative contributions of ADHD, on the one hand, and comorbid disorders, on the other, to the course of social impairments are unknown. Social dysfunctioning in children with ADHD appears to increase their risk of later psychopathology other than ADHD. Thus far effective treatment for social dysfunctioning is lacking. Future research should address the exact nature and long-term consequences of social dysfunctioning in children with ADHD, and focus on development of effective treatment strategies. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Nijmeijer, Judith S.; Minderaa, Ruud B.; Hartman, Catharina A.; Hoekstra, Pieter J.] Univ Groningen, Dept Psychiat, Univ Med Ctr Groningen, NL-9713 GZ Groningen, Netherlands.
[Buitelaar, Jan K.] Radboud Univ Nijmegen Med Ctr, Dept Psychiat, NL-6500 HB Nijmegen, Netherlands.
[Buitelaar, Jan K.] Karakter Child & Adolescent Psychiat Univ Ctr Nij, NL-6500 HB Nijmegen, Netherlands.
[Mulligan, Aisling] St James Hosp, Trinity Ctr Hlth Sci, Dept Psychiat, Dublin 8, Ireland.
RP Nijmeijer, JS (reprint author), Univ Groningen, Dept Child & Adolescent Psychiat, Hanzepl 1,POB 660, NL-9700 AR Groningen, Netherlands.
EM j.nijmeijer@accare.nl
RI Buitelaar, Jan/E-4584-2012; Hoekstra, Pieter/O-4396-2014
OI Buitelaar, Jan/0000-0001-8288-7757;
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NR 156
TC 101
Z9 101
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0272-7358
J9 CLIN PSYCHOL REV
JI Clin. Psychol. Rev.
PD APR
PY 2008
VL 28
IS 4
BP 692
EP 708
DI 10.1016/j.cpr.2007.10.003
PG 17
WC Psychology, Clinical
SC Psychology
GA 287QH
UT WOS:000254931100009
PM 18036711
ER
PT J
AU Hein, G
Singer, T
AF Hein, Grit
Singer, Tania
TI I feel how you feel but not always: the empathic brain and its
modulation
SO CURRENT OPINION IN NEUROBIOLOGY
LA English
DT Article
ID SOCIAL COGNITION; OTHERS PAIN; CINGULATE CORTEX; NEURAL PROCESSES;
PREMOTOR CORTEX; AUTISM; MIND; PSYCHOPATHY; RECOGNITION; SIMULATION
AB The ability to share the other's feelings, known as empathy, has recently become the focus of social neuroscience studies. We review converging evidence that empathy with, for example, the pain of another person, activates part of the neural pain network of the empathizer, without first hand pain stimulation to the empathizer's body. The amplitude of empathic brain responses is modulated by the intensity of the displayed emotion, the appraisal of the situation, characteristics of the suffering person such as perceived fairness, and features of the empathizer such as gender or previous experience with pain-inflicting situations. Future studies in the field should address inter-individual differences in empathy, development and plasticity of the empathic brain over the life span, and the link between empathy, compassionate motivation, and prosocial behavior.
C1 [Hein, Grit; Singer, Tania] Univ Zurich, Ctr Study Social & Neural Syst, CH-8006 Zurich, Switzerland.
RP Hein, G (reprint author), Univ Zurich, Ctr Study Social & Neural Syst, Blumlisalpstr 10, CH-8006 Zurich, Switzerland.
EM ghein@iew.uzh.ch
FU Society in Science - The Branco Weiss Fellowship; UFSP
FX This work was supported by Society in Science - The Branco Weiss
Fellowship to GH, and the UFSP "Foundations of Human Social Behaviour".
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NR 51
TC 154
Z9 156
PU CURRENT BIOLOGY LTD
PI LONDON
PA 84 THEOBALDS RD, LONDON WC1X 8RR, ENGLAND
SN 0959-4388
J9 CURR OPIN NEUROBIOL
JI Curr. Opin. Neurobiol.
PD APR
PY 2008
VL 18
IS 2
BP 153
EP 158
DI 10.1016/j.conb.2008.07.012
PG 6
WC Neurosciences
SC Neurosciences & Neurology
GA 363PO
UT WOS:000260279400007
PM 18692571
ER
PT J
AU Adolphs, R
AF Adolphs, Ralph
TI Fear, faces, and the human amygdala
SO CURRENT OPINION IN NEUROBIOLOGY
LA English
DT Article
ID HIGH-FUNCTIONING AUTISM; FACIAL EXPRESSIONS; SINGLE NEURONS; EMOTION
RECOGNITION; PREFRONTAL CORTEX; NEURAL CIRCUITRY; VISUAL CORTICES; 1ST
IMPRESSIONS; MONKEY AMYGDALA; RHESUS-MONKEY
AB The amygdala's historical role in processing stimuli related to threat and fear is being modified to suggest a role that is broader and more abstract. Amygdala lesions impair the ability to seek out and make use of the eye region of faces, resulting in impaired fear perception. Other studies in rats and humans revive earlier proposals that the amygdala is important not only for fear perception as such, but also for detecting saliency and biological relevance. Debates about some features of this processing now suggest that while the amygdala can process fearful facial expressions in the absence of conscious perception, and while there is some degree of preattentive processing, this depends on the context and is not necessarily more rapid than cortical processing routes. A large current research effort extends the amygdala's putative role to a number of psychiatric illnesses.
C1 CALTECH, Pasadena, CA 91125 USA.
RP Adolphs, R (reprint author), CALTECH, Pasadena, CA 91125 USA.
EM radolphs@hss.caltech.edu
FU NIMH; Gordon and Betty Moore Foundation; Simons Foundation
FX This paper was supported by grants from NIMH, the Gordon and Betty Moore
Foundation, and the Simons Foundation.
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NR 72
TC 146
Z9 148
PU CURRENT BIOLOGY LTD
PI LONDON
PA 84 THEOBALDS RD, LONDON WC1X 8RR, ENGLAND
SN 0959-4388
J9 CURR OPIN NEUROBIOL
JI Curr. Opin. Neurobiol.
PD APR
PY 2008
VL 18
IS 2
BP 166
EP 172
DI 10.1016/j.conb.2008.06.006
PG 7
WC Neurosciences
SC Neurosciences & Neurology
GA 363PO
UT WOS:000260279400009
PM 18655833
ER
PT J
AU Rizzolatti, G
Fabbri-Destro, M
AF Rizzolatti, Giacomo
Fabbri-Destro, Maddalena
TI The mirror system and its role in social cognition
SO CURRENT OPINION IN NEUROBIOLOGY
LA English
DT Article
ID 3 MOTOR AREAS; PREMOTOR CORTEX; NEURON SYSTEM; MAGNETIC STIMULATION;
REPRESENTATIONS; MONKEY; ORGANIZATION; MODULATION; MOVEMENTS; DIRECTION
AB Experiments in monkeys have shown that coding the goal of the motor acts is a fundamental property of the cortical motor system. In area F5, goal-coding motor neurons are also activated by observing motor acts done by others (the 'classical' mirror mechanism); in area F2 and area F1, some motor neurons are activated by the mere observation of goal-directed movements of a cursor displayed on a computer screen (a 'mirror-like' mechanism). Experiments in humans and monkeys have shown that the mirror mechanism enables the observer to understand the intention behind an observed motor act, in addition to the goal of it. Growing evidence shows that a deficit in the mirror mechanism underlies some aspects of autism.
C1 [Rizzolatti, Giacomo; Fabbri-Destro, Maddalena] Univ Parma, Sez Fisiol, Dipartimento Neurosci, I-43100 Parma, Italy.
[Fabbri-Destro, Maddalena] Univ Ferrara, Sez Fisiol Umana, Dipartimento SBTA, I-44100 Ferrara, Italy.
RP Rizzolatti, G (reprint author), Univ Parma, Sez Fisiol, Dipartimento Neurosci, Via Volturno 39, I-43100 Parma, Italy.
EM giacomo.rizzolatti@unipr.it; fbbmdl@unife.it
FU Ministero dell'Istruzione, dell'Universita e della Ricerca; EU project
Neurocom; Interuniversity Attraction Poles (IAP); Fondazione Cassa di
Risparmio di Ferrara
FX This work was supported by, a grant from Ministero dell'Istruzione,
dell'Universita e della Ricerca to GR, EU project Neurocom, and
Interuniversity Attraction Poles (IAP). M.F-D. was supported by
Fondazione Cassa di Risparmio di Ferrara.
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SN 0959-4388
J9 CURR OPIN NEUROBIOL
JI Curr. Opin. Neurobiol.
PD APR
PY 2008
VL 18
IS 2
BP 179
EP 184
DI 10.1016/j.conb.2008.08.001
PG 6
WC Neurosciences
SC Neurosciences & Neurology
GA 363PO
UT WOS:000260279400011
PM 18706501
ER
PT J
AU Rapin, I
Tuchman, RF
AF Rapin, Isabelle
Tuchman, Roberto F.
TI What is new in autism?
SO CURRENT OPINION IN NEUROLOGY
LA English
DT Review
DE autism; brain imaging; electrophysiology; epidemiology; genetics;
sensorimotor deficits
ID MIRROR NEURON SYSTEM; PERVASIVE DEVELOPMENTAL DISORDERS;
OF-THE-LITERATURE; SPECTRUM DISORDERS; COMMUNICATIVE DEFICITS; ANTIBRAIN
ANTIBODIES; LANGUAGE DISORDERS; MENTAL-RETARDATION; BROADER PHENOTYPE;
YOUNG-CHILDREN
AB Purpose of review
Autism is now recognized in one out of 150 children. This review highlights the topics within the growing autism literature that are shaping current thinking on autism and advancing research and clinical understanding of autism spectrum disorders.
Recent findings
The role of single-stranded microdeletions and epigenetic influences on brain development has dramatically altered our understanding of the etiology of the autisms. Recent research has focused on the role of synapse structure and function as central to the development of autism and suggests possible targets of interventions. Brain underconnectivity has been a focus in recent imaging studies and has become a central theme in conceptualizing autism. Despite increased awareness of autism there is no 'epidemic' and no one cause for autism. Data from the sibling studies are identifying early markers of autism and defining the broader autism phenotype.
Summary
Larger datasets in genetics, a focus on the early signs of autism, and increased recognition of the importance of defining subgroups of children with autism are leading to a greater understanding of the etiologies of autism. A growing interest in defining the molecular biology of social cognition, which is at the core of autism, will lead to expansion of our presently limited choices of mechanistically based interventions.
C1 [Rapin, Isabelle] Albert Einstein Coll Med, Rose F Kennedy Ctr res Mental Retardat & Human De, Bronx, NY 10461 USA.
[Rapin, Isabelle] Albert Einstein Coll Med, Dept Pediat, Saul R Korey Dept Neurol, Bronx, NY USA.
[Tuchman, Roberto F.] Univ Miami, Miller Sch Med, Miami, FL 33152 USA.
[Tuchman, Roberto F.] Miami Childrens Hosp, Miami, FL USA.
RP Rapin, I (reprint author), Albert Einstein Coll Med, Rose F Kennedy Ctr res Mental Retardat & Human De, Room 807,1410 Pelham Pkwy S, Bronx, NY 10461 USA.
EM rapin@aecom.yu.edu
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NR 103
TC 29
Z9 30
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 1350-7540
J9 CURR OPIN NEUROL
JI Curr. Opin. Neurol.
PD APR
PY 2008
VL 21
IS 2
BP 143
EP 149
PG 7
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 288FW
UT WOS:000254972600006
PM 18317271
ER
PT J
AU Janicki, MP
Henderson, CM
Rubin, IL
AF Janicki, Matthew P.
Henderson, C. Michael
Rubin, I. Leslie
CA Neurodev Conditions Study Grp
TI Neurodevelopmental conditions and aging: Report on the Atlanta Study
Group Charrette on Neurodevelopmental Conditions and Aging
SO DISABILITY AND HEALTH JOURNAL
LA English
DT Article
DE Aging; Intellectual disabilities; Neurodevelopmental conditions; Genetic
conditions; Disabilities
AB This article provides a summary of the proceedings of the Neurodevelopmental Conditions Study Group charrette held on May 21-22, 2007, in Atlanta, Georgia (USA) and underwritten by the Developmental Disabilities Branch of the U. S. Centers for Disease Control and Prevention and the National Institute for Disability and Rehabilitation Research. The charrette was a part of the conference on "State of the Science in Aging with Developmental Disabilities: Charting Lifespan Trajectories and Supportive Environments for Healthy Living." The aim of the charrette was to examine the extant knowledge on aging-related long-term effects and interactions of a number of neurodevelopmental conditions, including autism, cerebral palsy, Down syndrome, fragile X syndrome, Prader-Willi syndrome, spina bifida, and Williams syndrome. The discussants noted that although there is some published information regarding lifespan changes with these disorders, especially cerebral palsy and Down syndrome, there is a lack of confirming evidence for most of these conditions and concluded that additional evidence-based research and investigatory clinical work are needed to better understand the long-term effects of maturation and aging upon adults with these conditions. Primary recommendations included a call for more work toward the identification and description of the presentations and courses of age-related medical disorders that are common among these conditions; determination of the comparative prevalence and incidence of specific medical conditions between persons with neurodevelopmental disabilities and the general population; use of prevalence and incidence data to better understand risk factors for concomitant conditions; promotion of surveillance, screening, and specific treatment protocols for health provision; institution of a program of translational collaborative research related to older-age associated conditions; and dissemination of information related to aging and health to providers and people affected by these conditions. (C) 2008 Elsevier Inc. All rights reserved.
C1 [Janicki, Matthew P.] Univ Illinois, Dept Human Dev & Disabil, Chicago, IL 60608 USA.
[Henderson, C. Michael] Univ Rochester, Sch Med, Dept Med, Rochester, NY 14642 USA.
[Rubin, I. Leslie] Team Ctr, Chattanooga, TN 37404 USA.
[Rubin, I. Leslie] Morehouse Sch Med, Dept Pediat, Atlanta, GA 30342 USA.
RP Janicki, MP (reprint author), Univ Illinois, Dept Human Dev & Disabil, 1640 W Roosevelt Rd, Chicago, IL 60608 USA.
EM mjanicki@uic.edu
FU University of Illinois at Chicago's Rehabilitation Research and Training
Center on Aging and Developmental Disabilities; Rehabilitation Research
and Training Center on Aging and Developmental Disabilities; National
Institute on Disability and Rehabilitation Research [H133031134]; U.S.
Centers for Disease Control and Prevention; National Center on Birth
Defects and Developmental Disabilities; National Institute for
Disability and Rehabilitation Research; American Association on
Intellectual and Developmental Disabilities
FX The Atlanta Study Group Charrette on Neurodevelopmental Conditions and
Aging, held on May 21-22, 2007 in Atlanta, Georgia (USA), and sponsored
by the University of Illinois at Chicago's Rehabilitation Research and
Training Center on Aging and Developmental Disabilities, was
underwritten by the Developmental Disabilities Branch of the United
States Centers on Disease Control and Prevention. Funding support for
the meeting and development of this publication was also provided
through the Rehabilitation Research and Training Center on Aging and
Developmental Disabilities and grant H133031134 from the National
Institute on Disability and Rehabilitation Research.Appreciation is
extended to the U.S. Centers for Disease Control and Prevention,
National Center on Birth Defects and Developmental Disabilities, and the
National Institute for Disability and Rehabilitation Research for
financial support of the meeting, and the University of Illinois at
Chicago, University of Rochester, and Emory University for provision of
personnel. Principals for the organization and management of the Atlanta
Charrette were M. Janicki of the University of Illinois at Chicago, L.
Rubin of Emory University, and C. Michael Henderson of the University of
Rochester. T. Heller of the University of Illinois at Chicago provided
overall direction. Appreciation is also extended to the American
Association on Intellectual and Developmental Disabilities for
sponsorship and logistical support and to the Tosinvest Sanita
Organization for additional support. C. Michael Henderson prepared the
substance of the original meeting report, with additional contributions
from M. Janicki, L. Rubin, and several of the participants.
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NR 50
TC 7
Z9 7
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 1936-6574
J9 DISABIL HEALTH J
JI Disabil. Health J.
PD APR
PY 2008
VL 1
IS 2
BP 116
EP 124
DI 10.1016/j.dhjo.2008.02.004
PG 9
WC Health Care Sciences & Services; Health Policy & Services; Public,
Environmental & Occupational Health; Rehabilitation
SC Health Care Sciences & Services; Public, Environmental & Occupational
Health; Rehabilitation
GA V18JS
UT WOS:000208001700007
PM 21122718
ER
PT J
AU Tripi, G
Roux, S
Canziani, T
Brithault, FB
Barthelemy, C
Canziani, F
AF Tripi, Gabriele
Roux, Sylvie
Canziani, Tatiana
Brithault, Frederique Bonnet
Barthelemy, Catherine
Canziani, Fabio
TI Minor physical anomalies in children with autism spectrum disorder
SO EARLY HUMAN DEVELOPMENT
LA English
DT Article
DE autism spectrum disorder; minor physical anomalies; neurodevelopment
ID INFANTILE-AUTISM
AB Aim: To investigate the rate and topological profile of minor physical anomalies (MPAs) (prenatal errors of morphogenesis) in a group of children with Autism Spectrum Disorder (ASD), in order to better set a temporal framing of embryological factors involved in the neurodevelopmental etiology.
Method: A new modified Waldrop scale and a mixed approach of computerized photogrammetry and classic anthroposcopy was used to detect the presence or absence of 41 MPAs in 24 children (mean age: 7 years; sex ratio: 22M:2F) with ASD and 24 healthy comparison subjects (mean age: 7 years; sex ratio: 19M:5F) selected with DSM IV and CARS.
Results: We found that children with ASD presenting MPAs (n=23; 96%) had significantly higher rates of MPAs in four body areas (head, ears, mouth, hands); interestingly three of 41 MPAs best discriminated ASD groups from comparison subjects: abnormal head circumference, abnormal cephalic index, abnormal palate. Moreover, our results suggest that most MPAs occur predominantly after the first trimester of pregnancy.
Conclusions: These results support a prenatal neurodevelopmental model of the autism spectrum disorder. (C) 2007 Elsevier Ireland Ltd. All rights reserved.
C1 [Tripi, Gabriele; Canziani, Tatiana; Canziani, Fabio] Univ Palermo, Aiuto Materno Hosp, Dept Child Neuropsychiat, I-90100 Palermo, Italy.
[Roux, Sylvie; Brithault, Frederique Bonnet; Barthelemy, Catherine] CHRU Bretonneau, INSERM, Dept Neurophysiol Explorat Pedopsychiat, U619,Equipe 1, F-37044 Tours 1, France.
[Canziani, Tatiana] Univ Palermo, Fac Med, Didact Area Sci English, I-12990127 Palermo, Italy.
RP Tripi, G (reprint author), Univ Palermo, Aiuto Materno Hosp, Dept Child Neuropsychiat, Via Lancia Brolo 10 Bis, I-90100 Palermo, Italy.
EM gabrieletripi@hotmail.com
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NR 38
TC 16
Z9 16
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0378-3782
J9 EARLY HUM DEV
JI Early Hum. Dev.
PD APR
PY 2008
VL 84
IS 4
BP 217
EP 223
DI 10.1016/j.earlhumdev.2007.04.005
PG 7
WC Obstetrics & Gynecology; Pediatrics
SC Obstetrics & Gynecology; Pediatrics
GA 303BP
UT WOS:000256014800002
PM 17566672
ER
PT J
AU Sullivan, KM
AF Sullivan, Kevin M.
TI The interaction of agricultural pesticides and marginal iodine nutrition
status as a cause of autism spectrum disorders
SO ENVIRONMENTAL HEALTH PERSPECTIVES
LA English
DT Letter
ID AMERICAN-THYROID-ASSOCIATION; UNITED-STATES; NATIONAL-HEALTH;
SUPPLEMENTATION; RECOMMENDATIONS; DISRUPTION; PREGNANCY; LACTATION;
CANADA
C1 Emory Univ, Rollins Sch Publ Hlth, Atlanta, GA 30322 USA.
RP Sullivan, KM (reprint author), Emory Univ, Rollins Sch Publ Hlth, Atlanta, GA 30322 USA.
EM cdckms@sph.emory.edu
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Zoeller RT, 2000, NEUROTOXICOLOGY, V21, P935
NR 9
TC 4
Z9 4
PU US DEPT HEALTH HUMAN SCIENCES PUBLIC HEALTH SCIENCE
PI RES TRIANGLE PK
PA NATL INST HEALTH, NATL INST ENVIRONMENTAL HEALTH SCIENCES, PO BOX 12233,
RES TRIANGLE PK, NC 27709-2233 USA
SN 0091-6765
J9 ENVIRON HEALTH PERSP
JI Environ. Health Perspect.
PD APR
PY 2008
VL 116
IS 4
BP A155
EP A155
DI 10.1289/elip.11010
PG 1
WC Environmental Sciences; Public, Environmental & Occupational Health;
Toxicology
SC Environmental Sciences & Ecology; Public, Environmental & Occupational
Health; Toxicology
GA 282KL
UT WOS:000254566500008
PM 18414608
ER
PT J
AU Naber, FBA
Bakermans-Kranenburg, MJ
van Ijzendoorn, MH
Dietz, C
van Daalen, E
Swinkels, SHN
Buitelaar, JK
van Engeland, H
AF Naber, Fabienne B. A.
Bakermans-Kranenburg, Marian J.
van Ijzendoorn, Marinus H.
Dietz, Claudine
van Daalen, Emma
Swinkels, Sophie H. N.
Buitelaar, Jan K.
van Engeland, Herman
TI Joint attention development in toddlers with autism
SO EUROPEAN CHILD & ADOLESCENT PSYCHIATRY
LA English
DT Article
DE autism developmental; trajectory; joint attention; PDD; toddlers
ID TRAITS QUESTIONNAIRE ESAT; SPECTRUM DISORDERS; CHILDREN; LANGUAGE;
BEHAVIOR; INFANT; COMMUNICATION; IMPAIRMENTS; POPULATION; CHECKLIST
AB Deficits in Joint Attention (JA) may be one of the earliest signs of Autism Spectrum Disorders (ASD). In this longitudinal study we investigated several types of JA behaviors at the age of 24 and 42 months, and their development over time. Eleven children with ASD, 10 children with other developmental disorders, and eight children without a developmental disorder participated. It was found that children with ASD showed significantly less JA at the age of 24 months. At this age, the various types of JA ( Basic Joint Attention, Associated Joint Attention, Joint Visual Attention) were correlated with developmental level and number of autistic characteristics. However, at the age of 42 months, these associations were absent. Although children with ASD may show less JA at the age of 24 months compared to other groups of children, by the age of 42 months they reach about the same level of JA, except for joint visual attention. In fact, at both ages, children with ASD differed consistently only on JVA from the other groups. JVA may be a core component of an early screening device for ASD.
C1 [Naber, Fabienne B. A.; Bakermans-Kranenburg, Marian J.; van Ijzendoorn, Marinus H.] Leiden Univ, Ctr Child & Family Studies, NL-2300 RB Leiden, Netherlands.
[Naber, Fabienne B. A.; Dietz, Claudine; van Daalen, Emma; van Engeland, Herman] Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, Utrecht, Netherlands.
[Swinkels, Sophie H. N.; Buitelaar, Jan K.] Radboud Univ Nijmegen, Dept Psychiat, NL-6525 ED Nijmegen, Netherlands.
RP Naber, FBA (reprint author), Leiden Univ, Ctr Child & Family Studies, PB 9555, NL-2300 RB Leiden, Netherlands.
EM Fnaber@fsw.leidenuniv.nl
RI van IJzendoorn, Marinus/I-1379-2012; Buitelaar, Jan/E-4584-2012
OI Buitelaar, Jan/0000-0001-8288-7757
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NR 50
TC 14
Z9 17
PU DR DIETRICH STEINKOPFF VERLAG
PI DARMSTADT
PA PO BOX 10 04 62, D-64204 DARMSTADT, GERMANY
SN 1018-8827
J9 EUR CHILD ADOLES PSY
JI Eur. Child Adolesc. Psych.
PD APR
PY 2008
VL 17
IS 3
BP 143
EP 152
DI 10.1007/s00787-007-0648-6
PG 10
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 292GM
UT WOS:000255254500003
PM 17849078
ER
PT J
AU Billstedt, E
Gillberg, C
Gillberg, IC
AF Billstedt, E.
Gillberg, C.
Gillberg, I. C.
TI Outcome of autism spectrum disorders
SO EUROPEAN PSYCHIATRY
LA English
DT Meeting Abstract
C1 [Billstedt, E.; Gillberg, C.; Gillberg, I. C.] Univ Gothenburg, Inst Neurosci & Physiol, Dept Child & Adolescent Psychiat, Gothenburg, Sweden.
[Gillberg, C.] Univ Strathclyde, Glasgow, Lanark, Scotland.
NR 0
TC 0
Z9 0
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD APR
PY 2008
VL 23
SU 2
BP S270
EP S271
DI 10.1016/j.eurpsy.2008.01.555
PG 2
WC Psychiatry
SC Psychiatry
GA 288LL
UT WOS:000254987801208
ER
PT J
AU Bloemen, IF
Tuinier, S
Verbeeck, W
AF Bloemen, I. F.
Tuinier, S.
Verbeeck, W.
TI Obsessive compulsive symptoms in autism
SO EUROPEAN PSYCHIATRY
LA English
DT Meeting Abstract
C1 [Bloemen, I. F.; Tuinier, S.; Verbeeck, W.] Vincent Van Gogh Inst, Dept Clin Res, Venray, Netherlands.
NR 0
TC 0
Z9 0
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD APR
PY 2008
VL 23
SU 2
BP S401
EP S401
DI 10.1016/j.eurpsy.2008.01.1389
PG 1
WC Psychiatry
SC Psychiatry
GA 288LL
UT WOS:000254987801664
ER
PT J
AU Ceylan, ME
Turkcan, A
Aydin, A
AF Ceylan, M. E.
Turkcan, A.
Aydin, A.
TI Autism and metabolic cytopathy
SO EUROPEAN PSYCHIATRY
LA English
DT Meeting Abstract
C1 [Ceylan, M. E.; Turkcan, A.; Aydin, A.] Bakirkoy Res Hosp, Dept Psychiat, Istanbul, Turkey.
NR 0
TC 0
Z9 0
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD APR
PY 2008
VL 23
SU 2
BP S368
EP S368
DI 10.1016/j.eurpsy.2008.01.1275
PG 1
WC Psychiatry
SC Psychiatry
GA 288LL
UT WOS:000254987801550
ER
PT J
AU Goeb, U
Bonelli, F
Jardri, R
Kechid, G
Lenfant, AY
Delion, P
AF Goeb, U.
Bonelli, F.
Jardri, R.
Kechid, G.
Lenfant, A. Y.
Delion, P.
TI Packing therapy in children and adolescents with autism and serious
behavioural problems
SO EUROPEAN PSYCHIATRY
LA English
DT Meeting Abstract
C1 [Goeb, U.; Bonelli, F.; Jardri, R.; Kechid, G.; Lenfant, A. Y.; Delion, P.] Univ Hosp, Ctr Lille, Dept Child & Adolescent Psychiat, Lille, France.
NR 0
TC 0
Z9 0
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD APR
PY 2008
VL 23
SU 2
BP S405
EP S406
PG 2
WC Psychiatry
SC Psychiatry
GA 288LL
UT WOS:000254987801680
ER
PT J
AU Orsi, P
Caputi, M
Pace, A
Di Nemi, SU
Barale, F
AF Orsi, P.
Caputi, M.
Pace, A.
Di Nemi, S. Ucelli
Barale, F.
TI Autism in adulthood: 48 months follow-up evaluation of the farmstead
community model
SO EUROPEAN PSYCHIATRY
LA English
DT Meeting Abstract
C1 [Orsi, P.; Pace, A.; Di Nemi, S. Ucelli; Barale, F.] Univ Pavia, Dept Appl & Psychobehav Hlth Sci, Sect Psychiat, I-27100 Pavia, Italy.
[Pace, A.; Di Nemi, S. Ucelli; Barale, F.] Cascina Rossago RSD, Pavia, Italy.
[Caputi, M.] Univ Pavia, Dept Psychol, I-27100 Pavia, Italy.
RI Caputi, Marcella/H-6193-2012
NR 0
TC 0
Z9 0
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD APR
PY 2008
VL 23
SU 2
BP S390
EP S391
DI 10.1016/j.eurpsy.2008.01.1351
PG 2
WC Psychiatry
SC Psychiatry
GA 288LL
UT WOS:000254987801626
ER
PT J
AU van Deurzen, L
Tuinier, S
de Mey, HR
Verbeeck, W
AF van Deurzen, L.
Tuinier, S.
de Mey, H. R.
Verbeeck, W.
TI Theory of mind and executive functioning in autism
SO EUROPEAN PSYCHIATRY
LA English
DT Meeting Abstract
C1 [van Deurzen, L.; Tuinier, S.; Verbeeck, W.] Vincent Van Gogh Inst, Dept Clin Res, Venray, Netherlands.
[de Mey, H. R.] Radboud Univ Nijmegen, Dept Psychol, NL-6525 ED Nijmegen, Netherlands.
NR 0
TC 0
Z9 0
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD APR
PY 2008
VL 23
SU 2
BP S401
EP S401
DI 10.1016/j.eurpsy.2008.01.1387
PG 1
WC Psychiatry
SC Psychiatry
GA 288LL
UT WOS:000254987801662
ER
PT J
AU Wachtel, LE
AF Wachtel, L. E.
TI Catatonia in autism: Etiology, incidence and treatment
SO EUROPEAN PSYCHIATRY
LA English
DT Meeting Abstract
C1 [Wachtel, L. E.] Johns Hopkins Sch Med, Kennedy Krieger Inst, Dept Psychiat, Baltimore, MD USA.
NR 0
TC 0
Z9 0
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD APR
PY 2008
VL 23
SU 2
BP S402
EP S402
DI 10.1016/j.eurpsy.2008.01.1391
PG 1
WC Psychiatry
SC Psychiatry
GA 288LL
UT WOS:000254987801666
ER
PT J
AU Wachtel, LE
AF Wachtel, L. E.
TI Electroconvulsive therapy in autism: Hope for severe psychopathology
SO EUROPEAN PSYCHIATRY
LA English
DT Meeting Abstract
C1 [Wachtel, L. E.] Johns Hopkins Univ Hosp, Johns Hopkins Sch Med, Dept Psychiat, Baltimore, MD 21287 USA.
NR 0
TC 0
Z9 0
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD APR
PY 2008
VL 23
SU 2
BP S288
EP S288
DI 10.1016/j.eurpsy.2008.01.615
PG 1
WC Psychiatry
SC Psychiatry
GA 288LL
UT WOS:000254987801268
ER
PT J
AU Comte-Gervais, I
Giron, A
Soares-Boucaud, I
Poussin, G
AF Comte-Gervais, Isabelle
Giron, Alain
Soares-Boucaud, Isabelle
Poussin, Gerard
TI Assessment of social intelligence in children with specific language
impairment - Presentation of an assessing scale
SO EVOLUTION PSYCHIATRIQUE
LA French
DT Article
DE specific language impairment; pervasive developmental disorders;
continuum autistic; social cognition; theory of mind; assessing scale
diagnostic
ID HIGH-LEVEL AUTISM; FALSE BELIEF; MIND; DISORDERS; BEHAVIOR
AB Performances in social abilities of everyday life were studied in children with specific language impairment or PDD (pervasive developmental disorders). Comparison was made with normal children, children with intellectual deficiencies and dyslexic children. Results concerning children with language impairment confirm a relationship exists between language abilities and social cognition abilities, development of theory of mind being likely to go along with the development of language. The second objective is to establish normative values for the EASE scale. Hence, 327 normal children of various ages were tested. Results show a significant effect of age on mentalization development and results confirm that this ability is acquired between three and five years old. Normative values have been thus established. This study may allow to propose the EASE scale as a tool to help diagnosis, in particular to help make differential diagnosis of pathologies leading to troubles of language and personality in young children, such as "PDD" (atypic autism among others) and "SLI", as well as to have clinical tools which enable to make a diagnosis in younger children. The EASE scale therefore presents greatest importance. (C) 2008 Elsevier Masson SAS. Tous droits reserves.
C1 [Comte-Gervais, Isabelle; Soares-Boucaud, Isabelle] Hop E Herriot HCL, Ctr Reference Troubles Apprentissages, Serv Prof Collet, Pavillon U, F-69437 Lyon 03, France.
[Giron, Alain] CHU Pitie Salpetriere, INSERM, U 494, F-75634 Paris, France.
[Soares-Boucaud, Isabelle] ITTAC, Serv Univ Psychiat Infantojuvenile, F-69100 Villeurbanne, France.
[Poussin, Gerard] Univ Grenoble 2, F-38040 Grenoble, France.
RP Comte-Gervais, I (reprint author), Hop E Herriot HCL, Ctr Reference Troubles Apprentissages, Serv Prof Collet, Pavillon U, Pl Arsonval, F-69437 Lyon 03, France.
EM isabelle.comte-gervais@chu-lyon.fr
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NR 24
TC 1
Z9 1
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0014-3855
J9 EVOL PSYCHIATR
JI Evol. Psychiatr.
PD APR-JUN
PY 2008
VL 73
IS 2
BP 353
EP 366
DI 10.1016/j.evopsy.2008.02.004
PG 14
WC Psychiatry
SC Psychiatry
GA 322BL
UT WOS:000257350100010
ER
PT J
AU Chatterjee, A
AF Chatterjee, Archana
TI Vaccine safety: genuine concern or a legacy of unfounded skepticism?
SO EXPERT REVIEW OF VACCINES
LA English
DT Editorial Material
ID AUTISM SPECTRUM DISORDERS; UNITED-STATES; CHILDREN
C1 Creighton Univ, Sch Med, Div Pediat Infect Dis, Dept Pediat, Omaha, NE 68131 USA.
RP Chatterjee, A (reprint author), Creighton Univ, Sch Med, Div Pediat Infect Dis, Dept Pediat, 601 N 30th St,Suite 2321, Omaha, NE 68131 USA.
EM achatter@creighton.edu
CR Ball LK, 2001, PEDIATRICS, V107, P1147, DOI 10.1542/peds.107.5.1147
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*US CDC, 1998, MMWR-MORBID MORTAL W, V48, P243
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Wakefield AJ, 2004, LANCET, V363, P750
NR 19
TC 1
Z9 1
PU EXPERT REVIEWS
PI LONDON
PA UNITEC HOUSE, 3RD FL, 2 ALBERT PLACE, FINCHLEY CENTRAL, LONDON N3 1QB,
ENGLAND
SN 1476-0584
J9 EXPERT REV VACCINES
JI Expert Rev. Vaccines
PD APR
PY 2008
VL 7
IS 3
BP 275
EP 277
DI 10.1586/14760584.7.3.275
PG 3
WC Immunology
SC Immunology
GA 360HG
UT WOS:000260048000001
PM 18393594
ER
PT J
AU De Jaco, A
Miller, M
Comoletti, D
Dubi, N
Taylor, P
AF De Jaco, Antonella
Miller, Meghan
Comoletti, Davide
Dubi, Noga
Taylor, Palmer
TI Trafficking of neuroligin mutant proteins associated with autism
SO FASEB JOURNAL
LA English
DT Meeting Abstract
C1 [De Jaco, Antonella; Miller, Meghan; Comoletti, Davide; Dubi, Noga; Taylor, Palmer] Univ Calif San Diego, La Jolla, CA 92093 USA.
NR 0
TC 0
Z9 0
PU FEDERATION AMER SOC EXP BIOL
PI BETHESDA
PA 9650 ROCKVILLE PIKE, BETHESDA, MD 20814-3998 USA
SN 0892-6638
J9 FASEB J
JI Faseb J.
PD APR
PY 2008
VL 22
PG 1
WC Biochemistry & Molecular Biology; Biology; Cell Biology
SC Biochemistry & Molecular Biology; Life Sciences & Biomedicine - Other
Topics; Cell Biology
GA V25GZ
UT WOS:000208467809225
ER
PT J
AU Goldberg, JE
Manske, J
AF Goldberg, Jodi Elisabeth
Manske, Jill
TI Reduced proliferative capacity of highly purified T cells from patients
with Autism Spectrum Disorders (ASD)
SO FASEB JOURNAL
LA English
DT Meeting Abstract
C1 [Goldberg, Jodi Elisabeth] Hamline Univ, St Paul, MN USA.
[Manske, Jill] Univ St Thomas, St Paul, MN USA.
NR 0
TC 0
Z9 0
PU FEDERATION AMER SOC EXP BIOL
PI BETHESDA
PA 9650 ROCKVILLE PIKE, BETHESDA, MD 20814-3998 USA
SN 0892-6638
J9 FASEB J
JI Faseb J.
PD APR
PY 2008
VL 22
PG 1
WC Biochemistry & Molecular Biology; Biology; Cell Biology
SC Biochemistry & Molecular Biology; Life Sciences & Biomedicine - Other
Topics; Cell Biology
GA V25GZ
UT WOS:000208467803534
ER
PT J
AU Jyonouchi, H
Geng, L
Cushing-Ruby, A
Quraishi, H
AF Jyonouchi, Harumi
Geng, Lee
Cushing-Ruby, Agnes
Quraishi, Huma
TI Evaluation of atopy and immune functions in children with autism
spectrum disorders (ASD): Identification of an ASD subset with distinct
clinical and immunological findings
SO FASEB JOURNAL
LA English
DT Meeting Abstract
C1 [Jyonouchi, Harumi; Geng, Lee; Cushing-Ruby, Agnes; Quraishi, Huma] UMDNJ New Jersey Med Sch, Newark, NJ USA.
NR 0
TC 0
Z9 0
PU FEDERATION AMER SOC EXP BIOL
PI BETHESDA
PA 9650 ROCKVILLE PIKE, BETHESDA, MD 20814-3998 USA
SN 0892-6638
J9 FASEB J
JI Faseb J.
PD APR
PY 2008
VL 22
PG 1
WC Biochemistry & Molecular Biology; Biology; Cell Biology
SC Biochemistry & Molecular Biology; Life Sciences & Biomedicine - Other
Topics; Cell Biology
GA V25GZ
UT WOS:000208467804641
ER
PT J
AU Kwack, K
Lee, SK
Kim, MH
Nam, M
Bang, HJ
Yang, JW
Choe, KS
Kim, SK
Hong, MS
Chung, JH
Kim, HG
AF Kwack, KyuBum
Lee, Seung Ku
Kim, Min-ho
Nam, Min
Bang, Hee Jung
Yang, Jae Won
Choe, Kyong-Sik
Kim, Su Kang
Hong, Mee Suk
Chung, Joo-Ho
Kim, Hyoun Geun
TI Positive association between the mesoderm specific transcript gene and
autism spectrum disorder in a Korean male population
SO FASEB JOURNAL
LA English
DT Meeting Abstract
C1 [Kwack, KyuBum; Lee, Seung Ku; Kim, Min-ho; Kim, Hyoun Geun] Pochon CHA Univ, CHA Res Inst, Seungnam Si, South Korea.
[Nam, Min; Bang, Hee Jung] Ewha Womans Univ, Dept Psychol, Seoul, South Korea.
[Yang, Jae Won] Korea Univ, Dept Neuropsychiat, Seoul, South Korea.
[Choe, Kyong-Sik] Hankuk Yukyoung Sch, Seoul, South Korea.
[Kim, Su Kang; Hong, Mee Suk; Chung, Joo-Ho] Kyung Hee Univ, Kohwang Med Res Inst, Seoul, South Korea.
NR 0
TC 0
Z9 0
PU FEDERATION AMER SOC EXP BIOL
PI BETHESDA
PA 9650 ROCKVILLE PIKE, BETHESDA, MD 20814-3998 USA
SN 0892-6638
J9 FASEB J
JI Faseb J.
PD APR
PY 2008
VL 22
PG 1
WC Biochemistry & Molecular Biology; Biology; Cell Biology
SC Biochemistry & Molecular Biology; Life Sciences & Biomedicine - Other
Topics; Cell Biology
GA V25GZ
UT WOS:000208467805311
ER
PT J
AU Waly, MIA
Deth, R
AF Waly, Mostafa Ibrahim-Ahmed
Deth, Richard
TI Neurodevelopmental Toxins Deplete Glutathione and Inhibit Folate and
Vitamin B12-Dependent Methionine Synthase Activity: A Link between
Oxidative Stress and Autism
SO FASEB JOURNAL
LA English
DT Meeting Abstract
C1 [Waly, Mostafa Ibrahim-Ahmed; Deth, Richard] Northeastern Univ, Boston, MA 02115 USA.
NR 0
TC 0
Z9 0
PU FEDERATION AMER SOC EXP BIOL
PI BETHESDA
PA 9650 ROCKVILLE PIKE, BETHESDA, MD 20814-3998 USA
SN 0892-6638
J9 FASEB J
JI Faseb J.
PD APR
PY 2008
VL 22
PG 1
WC Biochemistry & Molecular Biology; Biology; Cell Biology
SC Biochemistry & Molecular Biology; Life Sciences & Biomedicine - Other
Topics; Cell Biology
GA V25GZ
UT WOS:000208467806475
ER
PT J
AU Scearce-Levie, K
Roberson, ED
Gerstein, H
Cholfin, JA
Mandiyan, VS
Shah, NM
Rubenstein, JLR
Mucke, L
AF Scearce-Levie, K.
Roberson, E. D.
Gerstein, H.
Cholfin, J. A.
Mandiyan, V. S.
Shah, N. M.
Rubenstein, J. L. R.
Mucke, L.
TI Abnormal social behaviors in mice lacking Fgf17
SO GENES BRAIN AND BEHAVIOR
LA English
DT Article
DE autism; cortical development; fibroblast growth factor; neuropsychiatric
disease; schizophrenia; social behavior
ID MEDIAL PREFRONTAL CORTEX; ESTROGEN-RECEPTOR-ALPHA; PROTEIN TRANSGENIC
MICE; BETA KNOCKOUT MICE; RETT-SYNDROME; MOUSE MODEL; SCHIZOPHRENIA;
EXPRESSION; OXYTOCIN; GENE
AB The fibroblast growth factor family of secreted signaling molecules is essential for patterning in the central nervous system. Fibroblast growth factor 17 (Fgf17) has been shown to contribute to regionalization of the rodent frontal cortex. To determine how Fgf17 signaling modulates behavior, both during development and in adulthood, we studied mice lacking one or two copies of the Fgf17 gene. Fgf17-deficient mice showed no abnormalities in overall physical growth, activity level, exploration, anxiety-like behaviors, motor co-ordination, motor learning, acoustic startle, prepulse inhibition, feeding, fear conditioning, aggression and olfactory exploration. However, they displayed striking deficits in several behaviors involving specific social interactions. Fgf17-deficient pups vocalized less than wild-type controls when separated from their mother and siblings. Elimination of Fgf17 also decreased the interaction of adult males with a novel ovariectomized female in a social recognition test and reduced the amount of time opposite-sex pairs spent engaged in prolonged, affiliative interactions during exploration of a novel environment. After social exploration of a novel environment, Fgf17-deficient mice showed less activation of the immediate-early gene Fos in the frontal cortex than wild-type controls. Our findings show that Fgf17 is required for several complex social behaviors and suggest that disturbances in Fgf17 signaling may contribute to neuropsychiatric diseases that affect such behaviors.
C1 [Scearce-Levie, K.; Roberson, E. D.; Gerstein, H.; Mucke, L.] Gladstone Inst Neurol Dis, San Francisco, CA 94158 USA.
[Scearce-Levie, K.; Roberson, E. D.; Mucke, L.] Univ Calif San Francisco, Dept Neurol, San Francisco, CA 94143 USA.
[Cholfin, J. A.; Rubenstein, J. L. R.] Univ Calif San Francisco, Dept Psychiat, San Francisco, CA 94143 USA.
[Cholfin, J. A.; Rubenstein, J. L. R.] Univ Calif San Francisco, Nina Ireland Lab Dev Neurobiol, San Francisco, CA 94143 USA.
[Mandiyan, V. S.; Shah, N. M.] Univ Calif San Francisco, Dept Anat, San Francisco, CA 94143 USA.
RP Scearce-Levie, K (reprint author), Gladstone Inst Neurol Dis, 1650 Owens St, San Francisco, CA 94158 USA.
EM kscearce-levie@gladstone.ucsf.edu
RI Roberson, Erik/A-5718-2009
OI Roberson, Erik/0000-0002-1810-9763
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NR 37
TC 46
Z9 46
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1601-1848
J9 GENES BRAIN BEHAV
JI Genes Brain Behav.
PD APR
PY 2008
VL 7
IS 3
BP 344
EP 354
DI 10.1111/j.1601-183X.2007.00357.x
PG 11
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 283BM
UT WOS:000254611500010
PM 17908176
ER
PT J
AU Schaefer, GB
Mendelsohn, NJ
AF Schaefer, G. Bradley
Mendelsohn, Nancy J.
CA Professional Practice Guidelines
TI Clinical genetics evaluation in identifying the etiology of autism
spectrum disorders
SO GENETICS IN MEDICINE
LA English
DT Article
DE pervasive developmental disorders; tiered evaluations; diagnostic yield;
Asperger syndrome
ID PERVASIVE DEVELOPMENTAL DISORDERS; FRAGILE-X; CHILDREN; YIELD;
IDENTIFICATION; PREVALENCE; DELETION; FEMALES; MALES
AB The autism spectrum disorders are a collection of conditions, which have, in common, impaired socialization and communication in association with stereotypic behaviors. The reported incidence of autism spectrum disorders has increased markedly over the past decade. In addition, a large amount of attention has been paid to these conditions among lay and professional groups. These influences have resulted in a marked increase in the number of referrals to clinical geneticists for evaluation of persons with autism spectrum disorders. The primary role of the geneticist in this process is to define etiology, if possible, and to provide counseling and contribute to case management based on the results of such investigations. In deciding upon the appropriate evaluation scheme for a particular patient, the geneticist must consider a host of different factors. Such considerations would include (1) Assuring an accurate diagnosis of autism before proceeding with any investigation. (2) Discussing testing options, diagnostic yields, and patient investment before proceeding with an evaluation. (3) Communication and coordination with the patient's medical home. (4) Assessing the continuously expanding and evolving list of available laboratory testing modalities in light of evidence-based medicine. (5) Recognizing expanded phenotypes of well-described syndromic and metabolic conditions that encompass autism spectrum disorders. (6) Defining an individualized evaluation scheme based on the unique history and clinical features of a given patient. The guidelines in this article have been developed to assist the clinician in the consideration of these factors.
C1 [Schaefer, G. Bradley] Univ Nebraska Med Ctr, Munroe Meyer Inst Genet & Rehabil, Omaha, NE USA.
[Mendelsohn, Nancy J.] Childrens Hosp, Div Med Genet, Minneapolis, MN USA.
[Mendelsohn, Nancy J.] Clin Minnesota, Minneapolis, MN USA.
RP Schaefer, GB (reprint author), 985430 Nebraska Med Ctr, Omaha, NE 68198 USA.
EM gbschaef@unmc.edu
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NR 34
TC 46
Z9 46
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 1098-3600
J9 GENET MED
JI Genet. Med.
PD APR
PY 2008
VL 10
IS 4
BP 301
EP 305
DI 10.1097/GIM.0b013e31816b5cc9
PG 5
WC Genetics & Heredity
SC Genetics & Heredity
GA 291XP
UT WOS:000255231000010
PM 18414214
ER
PT J
AU Amor, DJ
Cameron, C
AF Amor, David J.
Cameron, Carolyn
TI PGD gender selection for non-Mendelian disorders with unequal sex
incidence
SO HUMAN REPRODUCTION
LA English
DT Article
DE preimplantation genetic diagnosis; gender selection; IVF; autism
ID PREIMPLANTATION GENETIC DIAGNOSIS; PERVASIVE DEVELOPMENTAL DISORDERS;
IN-VITRO FERTILIZATION; AUTISM; CHILDREN; PREVALENCE; SIBLINGS;
INHERITANCE; ADJUSTMENT; EXPRESSION
AB Preimplantation genetic diagnosis (PGD) was originally developed for couples whose potential offspring were at risk of severe Mendelian disorders, but has since been extended to other indications. One possible use of PGD is to perform gender selection for couples whose offspring are at increased risk of disorders that do not follow Mendelian inheritance, but which are substantially more common in one sex than another (unequal sex incidence). Here, we examine the clinical and ethical issues to be considered prior to offering PGD gender selection to reduce the risk of a child being affected by a non-Mendelian condition with unequal sex incidence. Factors to be considered include: the risk that a child of either sex will be affected by the condition; the overall reduction in risk provided by gender selection and the potential harms of the procedure. Consideration should also be given to the interests of the family and the child to be born, the seriousness of the condition and the couple's procreative autonomy. To illustrate these issues we use the example of autism, a non-Mendelian disorder that is considerably more common in males than in females.
C1 [Amor, David J.; Cameron, Carolyn] Royal Childrens Hosp, Murdoch Childrens Res Inst, Parkville, Vic 3052, Australia.
[Amor, David J.] Univ Melbourne, Royal Childrens Hosp, Dept Paediat, Parkville, Vic 3052, Australia.
[Amor, David J.] Melbourne IVF, Melbourne, Vic 3002, Australia.
RP Amor, DJ (reprint author), Royal Childrens Hosp, Murdoch Childrens Res Inst, Flemington Rd, Parkville, Vic 3052, Australia.
EM david.amor@mcri.edu.au
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NR 36
TC 5
Z9 5
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0268-1161
J9 HUM REPROD
JI Hum. Reprod.
PD APR
PY 2008
VL 23
IS 4
BP 729
EP 734
DI 10.1093/humrep/dem433
PG 6
WC Obstetrics & Gynecology; Reproductive Biology
SC Obstetrics & Gynecology; Reproductive Biology
GA 296OG
UT WOS:000255555100001
PM 18222917
ER
PT J
AU Ingersoll, B
AF Ingersoll, Brooke
TI The social role of imitation in autism - Implications for the treatment
of imitation deficits
SO INFANTS AND YOUNG CHILDREN
LA English
DT Article
DE autism; early intervention; imitation; social communication
ID YOUNG-CHILDREN; JOINT ATTENTION; PRETEND PLAY; SCHIZOPHRENIC CHILDREN;
SPECTRUM DISORDERS; PRESCHOOL-CHILDREN; SPONTANEOUS SPEECH; MOTOR
IMITATION; POSITIVE AFFECT; LANGUAGE
AB Individuals with autism exhibit significant deficits in imitation skills. This article reviews the importance of imitation in typical development, focusing on the social function of imitation and its role in the development of social communication skills. Second, it reviews evidence suggesting an association between imitation deficits and social communication impairments in children with autism. Third, it discusses limitations of the current method for teaching imitation that targets only the learning function of imitation. Finally, it describes a new imitation intervention designed to teach the social use of imitation in young children with autism.
C1 Lewis & Clark Coll, Portland, OR 97219 USA.
RP Ingersoll, B (reprint author), Michigan State Univ, 105B Psychol Bldg, E Lansing, MI 48824 USA.
EM ingers19@msu.edu
RI Ingersoll, Brooke/A-9117-2012
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NR 74
TC 16
Z9 16
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0896-3746
J9 INFANT YOUNG CHILD
JI Infants Young Child.
PD APR-JUN
PY 2008
VL 21
IS 2
BP 107
EP 119
PG 13
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA 280ZI
UT WOS:000254465100003
ER
PT J
AU Solomon, M
Ozonoff, SJ
Cummings, N
Carter, CS
AF Solomon, Marjorie
Ozonoff, Sally J.
Cummings, Neil
Carter, Cameron S.
TI Cognitive control in autism spectrum disorders
SO INTERNATIONAL JOURNAL OF DEVELOPMENTAL NEUROSCIENCE
LA English
DT Article
DE autism; Asperger syndrome; PDDNOS; executive functions; cognitive
control; neuroscience; prefrontal cortex
ID ANTERIOR CINGULATE CORTEX; HIGH-FUNCTIONING AUTISM; DEFICIT
HYPERACTIVITY DISORDER; EXECUTIVE FUNCTIONS; WORKING-MEMORY; DIAGNOSTIC
INTERVIEW; RESPONSE-INHIBITION; LIFE-SPAN; ATTENTION; CHILDREN
AB Cognitive control refers to the ability to flexibly allocate mental resources to guide thoughts and actions in light of internal goals. Given the behavioral inflexibility exhibited by individuals with autism spectrum disorders (ASDs), it would appear they experience cognitive control deficits. Cognitive correlates of this behavioral inflexibility have been elusive in previous investigations. Study goals were to investigate deficits in cognitive control in ASDs; to explore its developmental trajectory; and to test whether control deficits are related to symptoms of inflexible thoughts and/or behaviors, and attention symptoms. Thirty-one children and adolescents aged 8-17 with ASDs and 32 age, IQ, and gender matched control subjects completed cognitive, diagnostic, and behavorial assessments, as well as a measure of cognitive control involving overcoming a prepotent response tendency. Compared with typically developing control subjects, individuals with ASDs exhibited deficits in cognitive control. Younger children with ASDs did not demonstrate age-related improvements in cognitive control. Modest relationships between cognitive control, IQ, and attention problems were found for the sample. Only the relationship between cognitive control and full-scale IQ survived correction for multiple comparisons. (c) 2007 ISDN. Published by Elsevier Ltd. All rights reserved.
C1 [Solomon, Marjorie; Ozonoff, Sally J.; Cummings, Neil] UC Davis Hlth Syst, MIND Inst, Sacramento, CA 95817 USA.
[Solomon, Marjorie; Ozonoff, Sally J.; Cummings, Neil; Carter, Cameron S.] Univ Calif Davis, Dept Psychiat & Behav Sci, Davis, CA USA.
RP Solomon, M (reprint author), UC Davis Hlth Syst, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM marjorie.solomon@ucdmc.ucdavis.edu
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NR 69
TC 41
Z9 42
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0736-5748
J9 INT J DEV NEUROSCI
JI Int. J. Dev. Neurosci.
PD APR
PY 2008
VL 26
IS 2
BP 239
EP 247
DI 10.1016/j.ijdevneu.2007.11.001
PG 9
WC Developmental Biology; Neurosciences
SC Developmental Biology; Neurosciences & Neurology
GA 268UR
UT WOS:000253605700007
PM 18093787
ER
PT J
AU Katsyri, J
Sams, M
AF Katsyri, Jari
Sams, Mikko
TI The effect of dynamics on identifying basic emotions from synthetic and
natural faces
SO INTERNATIONAL JOURNAL OF HUMAN-COMPUTER STUDIES
LA English
DT Article
DE facial animation; basic emotions; movement perception
ID FACIAL EXPRESSIONS; ASPERGER-SYNDROME; RECOGNITION; ALEXITHYMIA;
IDENTITY; UNIVERSALS; PERCEPTION; MOVEMENT; AUTISM; MOTION
AB The identification of basic emotions (anger, disgust, fear, happiness, sadness and surprise) has been studied widely from pictures of facial expressions. Until recently, the role of dynamic information in identifying facial emotions has received little attention. There is evidence that dynamics improves the identification of basic emotions from synthetic (computer-animated) facial expressions [Wehrle, T., Kaiser, S., Schmidt, S., Scherer, K.R., 2000. Studying dynamic models of facial expression of emotion using synthetic animated faces. Journal of Personality and Social Psychology 78 (1), 105-119.]; however, similar result has not been confirmed with natural human faces. We compared the identification of basic emotions from both natural and synthetic dynamic vs. static facial expressions in 54 subjects. We found no significant differences in the identification of static and dynamic expressions from natural faces. In contrast, some synthetic dynamic expressions were identified much more accurately than static ones. This effect was evident only with synthetic facial expressions whose static displays were non-distinctive. Our results show that dynamics does not improve the identification of already distinctive static facial displays. On the other hand, dynamics has an important role for identifying subtle emotional expressions, particularly from computer-animated synthetic characters. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Katsyri, Jari; Sams, Mikko] Helsinki Univ Technol, Lab Computat, FIN-02015 Espoo, Finland.
RP Katsyri, J (reprint author), Helsinki Univ Technol, Lab Computat, POB 9203, FIN-02015 Espoo, Finland.
EM katsyri@1ce.hut.fi
RI Sams, Mikko/G-7060-2012
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NR 53
TC 16
Z9 16
PU ACADEMIC PRESS LTD ELSEVIER SCIENCE LTD
PI LONDON
PA 24-28 OVAL RD, LONDON NW1 7DX, ENGLAND
SN 1071-5819
J9 INT J HUM-COMPUT ST
JI Int. J. Hum.-Comput. Stud.
PD APR
PY 2008
VL 66
IS 4
BP 233
EP 242
DI 10.1016/j.ijhcs.2007.10.001
PG 10
WC Computer Science, Cybernetics; Ergonomics; Psychology, Multidisciplinary
SC Computer Science; Engineering; Psychology
GA 281CH
UT WOS:000254473500002
ER
PT J
AU Mouridsen, SE
Rich, B
Isager, T
Nedergaard, NJ
AF Mouridsen, Svend Erik
Rich, Bente
Isager, Torbert
Nedergaard, Niels Jorgen
TI Pervasive developmental disorders and criminal behaviour - A case
control study
SO INTERNATIONAL JOURNAL OF OFFENDER THERAPY AND COMPARATIVE CRIMINOLOGY
LA English
DT Article
DE arson; criminal behaviour; pervasive developmental disorders
ID ASPERGERS-SYNDROME; VIOLENCE; AUTISM
AB The prevalence and pattern of criminal behaviour in a population of 313 former child psychiatric in-patients with pervasive developmental disorders were studied. The patients were divided into three subgroups and compared with 933 matched controls from the general population. Age at follow-up was between 25 years and 59 years. An account of convictions in the nationwide Danish Register of Criminality was used as a measure of criminal behaviour. Among 113 cases with childhood autism, .9% had been convicted. In atypical autism (n = 86) and Asperger's syndrome (n = 114) the percentages were 8.1% and 18.4%, respectively. The corresponding rate of convictions in the comparison groups was 18.9%, 14.7%, and 19.6% respectively. Particular attention is given to arson in Asperger's syndrome (P = .0009).
C1 [Mouridsen, Svend Erik] Bispebjerg Hosp, Copenhagen, Denmark.
[Rich, Bente] Odense Univ Hosp, Odense, Denmark.
[Isager, Torbert] Glostrup Univ Hosp, Glostrup, Denmark.
[Nedergaard, Niels Jorgen] Aarhus Univ Hosp, Risskov, Denmark.
RP Mouridsen, SE (reprint author), Bispebjerg Hosp, Copenhagen, Denmark.
CR Analytical Software, 2003, STAT 8
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World Health Organization, 1992, ICD 10 INT CLASS MEN
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NR 31
TC 16
Z9 16
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0306-624X
J9 INT J OFFENDER THER
JI Int. J. Offender Ther. Comp. Criminol.
PD APR
PY 2008
VL 52
IS 2
BP 196
EP 205
DI 10.1177/0306624X07302056
PG 10
WC Criminology & Penology; Psychology, Applied
SC Criminology & Penology; Psychology
GA 273QR
UT WOS:000253947300006
PM 17615427
ER
PT J
AU Dawes, P
Bishop, DVM
Sirimanna, T
Bamiou, DE
AF Dawes, Piers
Bishop, Dorothy V. M.
Sirimanna, Tony
Bamiou, Doris-Eva
TI Profile and aetiology of children diagnosed with auditory processing
disorder (APD)
SO INTERNATIONAL JOURNAL OF PEDIATRIC OTORHINOLARYNGOLOGY
LA English
DT Article
DE auditory processing disorder; aetiotogy; otitis media; obstetric
optimality
ID LANGUAGE IMPAIRMENT; OBSTETRIC COMPLICATIONS; POPULATION; PREVALENCE;
AUTISM
AB Objective: Auditory processing disorder (APD) is characterised by Listening difficulties despite a normal audiogram. APD is becoming ever more widely diagnosed in children, though there is a controversy over definition, diagnosis and aetiology. This study sought to describe presenting features and investigate aeitological factors for children diagnosed with APD compared to those for whom APD was excluded.
Methods: Medical notes for children referred to a specialist hospital-based APD clinic were reviewed in relation to presenting features and potential aetiological factors.
Results: 32 children diagnosed with APD and 57 non-APD children were compared. They reported similar symptoms and similarly had high rates of co-morbid learning problems. No aetiological factor (including history of otitis media, adverse obstetric history or familial history of listening problems) predicted APD group membership.
Conclusions: Children identified with APD on the basis of commonly used APD tests cannot be distinguished on the basis of presenting features or the aetiological factors examined here. One explanation is that learning problems exist independently of auditory processing difficulties and the aetiological factors do not have a strong causal role in APD. However, no gold standard for APD testing exists and an alternative explanation is that the commonly used APD tests used as selection criteria in this study may be unreliable. (c) 2008 Elsevier Ireland Ltd. All rights reserved.
C1 [Dawes, Piers; Bishop, Dorothy V. M.] Univ Oxford, Dept Expt Psychol, Oxford OX1 3UD, England.
[Sirimanna, Tony; Bamiou, Doris-Eva] Great Ormond St Hosp Sick Children, Dept Audiol Med, London WC1N 3JH, England.
RP Dawes, P (reprint author), Univ Oxford, Dept Expt Psychol, S Parks Rd, Oxford OX1 3UD, England.
EM Piers.dawes@psy.ox.ac.uk
CR American Speech-Language-Hearing Association (ASHA), CENTR AUD PROC DIS
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NR 29
TC 26
Z9 31
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0165-5876
J9 INT J PEDIATR OTORHI
JI Int. J. Pediatr. Otorhinolaryngol.
PD APR
PY 2008
VL 72
IS 4
BP 483
EP 489
DI 10.1016/j.ijporl.2007.12.007
PG 7
WC Otorhinolaryngology; Pediatrics
SC Otorhinolaryngology; Pediatrics
GA 284SB
UT WOS:000254725000006
PM 18262288
ER
PT J
AU Martineau, J
Cochin, S
Magne, R
Barthelemy, C
AF Martineau, Joele
Cochin, Stephanie
Magne, Remy
Barthelemy, Catherine
TI Impaired cortical activation in autistic children: Is the mirror neuron
system involved?
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Article
DE autism; EEG; desynchronisation; human motion; visual perception; mirror
neuron system
ID DEVELOPMENTAL DISORDERS; PREMOTOR CORTEX; PERCEPTION; MOTION;
RECOGNITION; IMITATION; MOVEMENT; AREAS
AB The inability to imitate becomes obvious early in autistic children and seems to contribute to learning delay and to disorders of communication and contact. Posture, motility and imitation disorders in autistic syndrome might be the consequence of an abnormality of sensori-motor integration, related to the visual perception of movement, and could reflect impairment of the mirror neuron system (MNS).
We compared EEG activity during the observation of videos showing actions or still scenes in 14 right-handed autistic children and 14 right-handed, age- and gender-matched control children (3 girls and I I boys, aged 5 years 3 months-7 years I I months). We showed desynchronisation of the EEG in the motor cerebral cortex and the frontal and temporal areas during observation of human actions in the group of healthy children. No such desynchronisation was found in autistic children. Moreover, inversion of the pattern of hemispheric activation was found in autistic children, with increased cortical activity in the right hemisphere in the posterior region, including the centro-parietal and temporo-occipital sites. These results are in agreement with the hypothesis of impairment of the mirror neuron system in autistic disorder. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Martineau, Joele; Cochin, Stephanie; Magne, Remy; Barthelemy, Catherine] Univ Tours, CHRU, INSERM U930, F-37000 Tours, France.
RP Martineau, J (reprint author), CHU Bretonneau, 2 Blvd Tonnelle, F-37044 Tours, France.
EM j.martineau@chu-tours.fr
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NR 23
TC 49
Z9 50
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD APR
PY 2008
VL 68
IS 1
BP 35
EP 40
DI 10.1016/j.ijpsycho.2008.01.002
PG 6
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 293GE
UT WOS:000255322600005
PM 18313160
ER
PT J
AU Aneja, A
Tierney, E
AF Aneja, Alka
Tierney, Elaine
TI Autism: The role of cholesterol in treatment
SO INTERNATIONAL REVIEW OF PSYCHIATRY
LA English
DT Article
ID LEMLI-OPITZ-SYNDROME; SPECTRUM DISORDERS; MOUSE MODEL; SIMVASTATIN;
PHENOTYPE; INDIVIDUALS; MUTATIONS; CHILDREN; THERAPY; GENE
AB Cholesterol is essential for neuroactive steroid production, growth of myelin membranes, and normal embryonic and fetal development. It also modulates the oxytocin receptor, ligand activity and G-protein coupling of the serotonin-1A receptor. A deficit of cholesterol may perturb these biological mechanisms and thereby contribute to autism spectrum disorders (ASDs), as observed in Smith-Lemli-Opitz syndrome (SLOS) and some subjects with ASDs in the Autism Genetic Resource Exchange (AGRE). A clinical diagnosis of SLOS can be confirmed by laboratory testing with an elevated plasma 7DHC level relative to the cholesterol level and is treatable by dietary cholesterol supplementation. Individuals with SLOS who have such cholesterol treatment display fewer autistic behaviours, infections, and symptoms of irritability and hyperactivity, with improvements in physical growth, sleep and social interactions. Other behaviours shown to improve with cholesterol supplementation include aggressive behaviours, self-injury, temper outbursts and trichotillomania. Cholesterol ought to be considered as a helpful treatment approach while awaiting an improved understanding of cholesterol metabolism and ASD. There is an increasing recognition that this single-gene disorder of abnormal cholesterol synthesis may be a model for understanding genetic causes of autism and the role of cholesterol in ASD.
C1 [Aneja, Alka] Kennedy Krieger Inst, Dept Psychiat, Baltimore, MD 21211 USA.
Johns Hopkins Univ, Sch Med, Dept Psychiat & Behav Sci, Div Child & Adolescent Psychiat, Baltimore, MD 21205 USA.
RP Aneja, A (reprint author), Kennedy Krieger Inst, Dept Psychiat, 3901 Greenspring Ave, Baltimore, MD 21211 USA.
EM aneja@kennedykrieger.org
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NR 36
TC 20
Z9 21
PU ROUTLEDGE JOURNALS, TAYLOR & FRANCIS LTD
PI ABINGDON
PA 4 PARK SQUARE, MILTON PARK, ABINGDON OX14 4RN, OXFORDSHIRE, ENGLAND
SN 0954-0261
J9 INT REV PSYCHIATR
JI Int. Rev. Psych.
PD APR
PY 2008
VL 20
IS 2
BP 165
EP 170
DI 10.1080/09540260801889062
PG 6
WC Psychiatry
SC Psychiatry
GA 297DZ
UT WOS:000255598600008
PM 18386207
ER
PT J
AU Wallis, KE
Smith, SM
AF Wallis, Kate E.
Smith, Suzanne M.
TI Developmental screening in pediatric primary care: The role of nurses
SO JOURNAL FOR SPECIALISTS IN PEDIATRIC NURSING
LA English
DT Article
DE autism; developmental disabilities; health screening
ID AUTISM SPECTRUM DISORDERS; MODIFIED CHECKLIST; PARENTS CONCERNS;
YOUNG-CHILDREN; FOLLOW-UP; DIAGNOSIS; TODDLERS; IDENTIFICATION;
SURVEILLANCE; CHAT
C1 [Wallis, Kate E.; Smith, Suzanne M.] Univ Penn, Sch Nursing, Ctr Autism & Dev Disabil Res & Epidemiol, Philadelphia, PA 19104 USA.
RP Wallis, KE (reprint author), Univ Penn, Sch Nursing, Ctr Autism & Dev Disabil Res & Epidemiol, Philadelphia, PA 19104 USA.
EM wallisk@nursing.upenn.edu
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 27
TC 4
Z9 4
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1539-0136
J9 J SPEC PEDIATR NURS
JI J. Spec. Pediatr. Nurs.
PD APR
PY 2008
VL 13
IS 2
BP 130
EP 134
DI 10.1111/j.1744-6155.2008.00145.x
PG 5
WC Nursing; Pediatrics
SC Nursing; Pediatrics
GA 278SH
UT WOS:000254306100009
PM 18366381
ER
PT J
AU David, N
Gawronski, A
Santos, NS
Huff, WG
Lehnhardt, FG
Newen, A
Vogeley, K
AF David, Nicole
Gawronski, Astrid
Santos, Natacha S.
Huff, Wolfgang
Lehnhardt, Fritz-Georg
Newen, Albert
Vogeley, Kai
TI Dissociation between key processes of social cognition in autism:
Impaired mentalizing but intact sense of agency
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Review
DE mentalizing; sense of agency; high functioning autism; asperger
syndrome; action-monitoring; self-other distinction
ID HIGH-FUNCTIONING AUTISM; STRANGE STORIES TEST; ASPERGER-SYNDROME;
SPECTRUM DISORDERS; SELF-CONSCIOUSNESS; MOTOR IMPAIRMENT; NORMAL ADULTS;
SCHIZOPHRENIC-PATIENTS; ALIEN CONTROL; HUMAN BRAIN
AB Deficits in social cognition and interaction, such as in mentalizing and imitation behavior, are hallmark features of autism spectrum disorders. Both imitation and mentalizing are at the core of the sense of agency, the awareness that we are the initiators of our own behavior. Little evidence exists regarding the sense of agency in autism. Thus, we compared high-functioning adults with autism to healthy control subjects using an action monitoring and attribution task. Subjects with autism did not show deficits in this task, yet they showed significant mentalizing deficits. Our findings indicate a dissociation between the sense of agency and ascription of mental states in autism. We propose that social-cognitive deficits in autism may arise on a higher level than that of action monitoring and awareness.
C1 [David, Nicole; Gawronski, Astrid; Santos, Natacha S.; Huff, Wolfgang; Lehnhardt, Fritz-Georg; Vogeley, Kai] Univ Cologne, Dept Psychiat & Psychotherapy, D-50924 Cologne, Germany.
[Newen, Albert] Ruhr Univ Bochum, Dept Philosophy, Bochum, Germany.
RP David, N (reprint author), Univ Cologne, Dept Psychiat & Psychotherapy, Kerpener St 62, D-50924 Cologne, Germany.
EM nicole.david@uk-koeln.de
RI David, Nicole/H-1682-2012; Vogeley, K/E-4860-2012
OI Vogeley, K/0000-0002-5891-5831
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NR 121
TC 24
Z9 24
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 593
EP 605
DI 10.1007/s10803-007-0425-x
PG 13
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700001
PM 17710522
ER
PT J
AU Kleinman, JM
Ventola, PE
Pandey, J
Verbalis, AD
Barton, M
Hodgson, S
Green, J
Dumont-Mathieu, T
Robins, DL
Fein, D
AF Kleinman, Jamie M.
Ventola, Pamela E.
Pandey, Juhi
Verbalis, Alyssa D.
Barton, Marianne
Hodgson, Sarah
Green, James
Dumont-Mathieu, Thyde
Robins, Diana L.
Fein, Deborah
TI Diagnostic stability in very young children with autism spectrum
disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; PDD-NOS; diagnostic stability; early detection
ID PERVASIVE DEVELOPMENTAL DISORDERS; FOLLOW-UP; ADI-R; AGE; INFANCY;
INTERVENTION; RECOGNITION; INTERVIEW; TODDLERS; FAMILIES
AB Autism Spectrum Disorders (ASD) diagnosis in very young children may be delayed due to doubts about validity. In this study, 77 children received a diagnostic and developmental evaluation between 16 and 35 months and also between 42 and 82 months. Diagnoses based on clinical judgment, Childhood Autism Rating Scale, and the Autism Diagnostic Observation Schedule were stable over time. Diagnoses made using the Autism Diagnostic Interview were slightly less stable. According to clinical judgment, 15 children (19%) moved off the autism spectrum by the second evaluation; none moved onto the spectrum. Results indicate diagnostic stability at acceptable levels for diagnoses made at age 2. Movement off the spectrum may reflect true improvement based on maturation, intervention, or over-diagnosis at age 2.
C1 [Kleinman, Jamie M.; Ventola, Pamela E.; Pandey, Juhi; Verbalis, Alyssa D.; Barton, Marianne; Hodgson, Sarah; Green, James; Dumont-Mathieu, Thyde; Robins, Diana L.; Fein, Deborah] Univ Connecticut, Dept Psychol, Storrs, CT 06269 USA.
RP Kleinman, JM (reprint author), Univ Connecticut, Dept Psychol, 406 Babbidge Rd, Storrs, CT 06269 USA.
EM jamie_kleinman@yahoo.com
RI Robins, Diana/D-9959-2011
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NR 35
TC 60
Z9 61
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
EI 1573-3432
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 606
EP 615
DI 10.1007/s10803-007-0427-8
PG 10
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700002
PM 17924183
ER
PT J
AU Bennett, T
Szatmari, P
Bryson, S
Volden, J
Zwaigenbaum, L
Vaccarella, L
Duku, E
Boyle, M
AF Bennett, Terry
Szatmari, Peter
Bryson, Susan
Volden, Joanne
Zwaigenbaum, Lonnie
Vaccarella, Liezanne
Duku, Eric
Boyle, Michael
TI Differentiating autism and asperger syndrome on the basis of language
delay or impairment
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; asperger syndrome; language impairment; outcomes
ID BEHAVIOR CHECKLIST; DISORDERS; CHILDREN; INDIVIDUALS
AB Asperger syndrome (AS) is differentiated from high-functioning autism (HFA) largely on a history of "language delay." This study examined "specific language impairment" as a predictor of outcome. Language skills of 19 children with AS and 45 with HFA were assessed at 4-6 years of age (Time 1) and 2 years later (Time 2). Children's symptoms and functional outcome scores were assessed every 2 years (Times 3, 4, and 5) until ages 15-17 years old. Regression analysis revealed that specific language impairment at time 2 more often accounted for the greatest variation in outcome scores in adolescence than the standard diagnosis of AS versus HFA based on history of language delay. Diagnostic implications are discussed.
C1 [Bennett, Terry; Szatmari, Peter; Vaccarella, Liezanne; Duku, Eric; Boyle, Michael] McMaster Univ, Dept Psychiat & Behav Neurosci, Hamilton, ON L8N 3Z5, Canada.
[Bryson, Susan] Dalhousie Univ, Dept Psychiat, IWK Hlth Ctr, Halifax, NS, Canada.
[Volden, Joanne; Zwaigenbaum, Lonnie] Univ Alberta, Dept Pediat, Edmonton, AB, Canada.
RP Szatmari, P (reprint author), Offord Ctr Child Studies, Chedoke Site, Hamilton, ON, Canada.
EM szatmar@mcmaster.ca
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NR 31
TC 40
Z9 40
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 616
EP 625
DI 10.1007/s10803-007-0428-7
PG 10
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700003
PM 17721697
ER
PT J
AU McDermott, S
Zhou, L
Mann, J
AF McDermott, Suzanne
Zhou, Li
Mann, Joshua
TI Injury treatment among children with autism or pervasive developmental
disorder
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; injury; epidemiology; IDC9 codes; E-codes
ID INTELLECTUAL DISABILITIES; CHALLENGING BEHAVIORS; YOUNG-PEOPLE;
POPULATION; RISK
AB This study examined the differences in the frequency and type of injury for children with autism and pervasive developmental disorder (PDD) compared with typically developing peers, when both groups are insured by Medicaid. The relative rate (RR) of emergency/hospital treatment of injury for children with autism or PDD compared to controls was 1.20 [95% Confidence Interval (CI) 1.04-1.39] after controlling for age and gender. Children with autism or PDD had a higher rate for head, face, and neck injuries (RR 1.47, 95% CI 1.13-1.90) and lower rate for sprains and strains (RR 0.54, 95% CI 0.32-0.91). Treatment for poisoning was 7.6 times as frequent, and self-inflicted injury was also 7.6 times as frequent for children with autism or PDD.
C1 [McDermott, Suzanne; Mann, Joshua] Univ S Carolina, Sch Med, Dept Family & Prevent Med, Family Med Ctr, Columbia, SC 29208 USA.
[Zhou, Li] Univ S Carolina, Arnold Sch Publ Hlth, Dept Epidemiol & Biostat, Columbia, SC 29208 USA.
RP McDermott, S (reprint author), Univ S Carolina, Sch Med, Dept Family & Prevent Med, Family Med Ctr, 3209 Colonial Dr, Columbia, SC 29208 USA.
EM suzanne.mcdermott@palmettohealth.org
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT, P41
Barell V, 2002, INJ PREV, V8, P91, DOI 10.1136/ip.8.2.91
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2006, FORUM CHILD FAMILY S
NR 24
TC 15
Z9 15
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 626
EP 633
DI 10.1007/s10803-007-0426-9
PG 8
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700004
PM 17690968
ER
PT J
AU Daoust, AM
Lusignan, FA
Braun, CMJ
Mottron, L
Godbout, R
AF Daoust, Anne-Marie
Lusignan, Felix-Antoine
Braun, Claude M. J.
Mottron, Laurent
Godbout, Roger
TI Dream content analysis in persons with an autism spectrum disorder
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; dream; emotions; REM sleep; questionnaire; polysomnography
ID ASPERGERS-SYNDROME; REM-SLEEP; PRAGMATIC IMPAIRMENTS; WORKING-MEMORY;
CHILDREN; RECALL; BRAIN; NEUROPSYCHOLOGY; INDIVIDUALS; ADULTS
AB Dream questionnaires were completed by 28 young adults with autism spectrum disorder (ASD) participants. Seventy-nine typically developed individual served as the control group. In a subset of 17 persons with ASD and 11 controls matched for verbal IQ, dream narratives were obtained following REM sleep awakenings in a sleep laboratory. Questionnaires revealed that participants with ASD, compared to controls, had fewer recollections of dreaming, fewer bad dreams and fewer emotions. In the sleep laboratory, dream content narratives following REM sleep awakenings were shorter in ASD participants than in controls. ASD participants also reported fewer settings, objects, characters, social interactions, activities, and emotions. It is concluded that these characteristics of dreaming in ASD may reflect neurocognitive dimensions specific to this condition.
C1 [Godbout, Roger] Hop Riviere Prairies, Sleep Lab & Clin, Montreal, PQ H1E 1A4, Canada.
[Daoust, Anne-Marie; Lusignan, Felix-Antoine; Mottron, Laurent; Godbout, Roger] Hop Riviere Prairies, Ctr Rech Fernand Seguin, Neurodev Disorders Program, Montreal, PQ, Canada.
[Braun, Claude M. J.] Univ Quebec, Dept Psychol, Montreal, PQ H3C 3P8, Canada.
RP Godbout, R (reprint author), Hop Riviere Prairies, Sleep Lab & Clin, 7070 Perras Blvd, Montreal, PQ H1E 1A4, Canada.
EM roger.godbout@umontreal.ca
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NR 57
TC 2
Z9 2
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 634
EP 643
DI 10.1007/s10803-007-0431-z
PG 10
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700005
PM 17682931
ER
PT J
AU Ozonoff, S
Young, GS
Goldring, S
Greiss-Hess, L
Herrera, AM
Steele, J
Macari, S
Hepburn, S
Rogers, SJ
AF Ozonoff, Sally
Young, Gregory S.
Goldring, Stacy
Greiss-Hess, Laura
Herrera, Adriana M.
Steele, Joel
Macari, Suzanne
Hepburn, Susan
Rogers, Sally J.
TI Gross motor development, movement abnormalities, and early
identification of autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; motor; early identification
ID ASPERGERS-SYNDROME; CHILDREN; AGE; DISORDERS; INFANCY; REGRESSION;
DIAGNOSIS; PHENOTYPE; SYMPTOMS; LANGUAGE
AB Gross motor development (supine, prone, rolling, sitting, crawling, walking) and movement abnormalities were examined in the home videos of infants later diagnosed with autism (regression and no regression subgroups), developmental delays (DD), or typical development. Group differences in maturity were found for walking, prone, and supine, with the DD and Autism-No Regression groups both showing later developing motor maturity than typical children. The only statistically significant differences in movement abnormalities were in the DD group; the two autism groups did not differ from the typical group in rates of movement abnormalities or lack of protective responses. These findings do not replicate previous investigations suggesting that early motor abnormalities seen on home video can assist in early identification of autism.
C1 [Ozonoff, Sally; Young, Gregory S.; Goldring, Stacy; Greiss-Hess, Laura; Steele, Joel; Rogers, Sally J.] Univ Calif Davis Hlth Syst, Dept Psychiat, MIND Inst, Sacramento, CA 95817 USA.
[Herrera, Adriana M.] Univ Minnesota, Inst Child Dev, Minneapolis, MN 55455 USA.
[Macari, Suzanne] Yale Univ, Sch Med, Ctr Child Study, New Haven, CT 06510 USA.
[Hepburn, Susan] Univ Colorado, Hlth Sci Ctr, Dept Psychiat, Denver, CO 80262 USA.
RP Ozonoff, S (reprint author), Univ Calif Davis Hlth Syst, Dept Psychiat, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM sjozonoff@ucdavis.edu
CR ADRIEN JL, 1993, J AM ACAD CHILD PSY, V32, P617, DOI 10.1097/00004583-199305000-00019
American Psychiatric Association, 2000, DIAGN STAT MAN DIS
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NR 32
TC 62
Z9 64
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 644
EP 656
DI 10.1007/s10803-007-0430-0
PG 13
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700006
PM 17805956
ER
PT J
AU Gray, KM
Tonge, BJ
Sweeney, DJ
AF Gray, Kylie M.
Tonge, Bruce J.
Sweeney, Deborah J.
TI Using the autism diagnostic interview-revised and the autism diagnostic
observation schedule with young children with developmental delay:
Evaluating diagnostic validity
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE preschool children; ADI-R; ADOS; diagnostic validity
ID ADI-R; SPECTRUM DISORDERS; MENTAL-RETARDATION; PRESCHOOL-CHILDREN;
CLASSIFICATION; ADOLESCENTS; INFORMATION; INSTRUMENT; AGREEMENT;
STABILITY
AB Few studies have focused on the validity of the ADI-R and ADOS in the assessment of preschool children with developmental delay. This study aimed to evaluate the diagnostic validity of the ADI-R and the ADOS in young children. Two-hundred and nine children aged 20-55 months participated in the study, 120 of whom received a diagnosis of autism. ADI-R and ADOS diagnostic classifications were compared to consensus clinical diagnoses. Children with a clinical diagnosis of autism scored significantly higher on all algorithm domains of the ADI-R and ADOS. The ADOS performed better than the ADI-R in comparison to consensus clinical diagnosis. Characteristics of the ADI-R and ADOS false positive and false negative cases are explored. Further research is recommended in terms of examining which items of the ADI-R best predict a diagnosis of autism for very young children with developmental problems.
C1 [Gray, Kylie M.] Monash Med Ctr, Child & Adolescent Menatl Hlth Serv, Clayton, Vic 3168, Australia.
[Gray, Kylie M.; Tonge, Bruce J.; Sweeney, Deborah J.] Monash Univ, Ctr Dev Psychiat & Psychol, Sch Psychol Psychiat & Psychol Med, Clayton, Vic 3168, Australia.
RP Gray, KM (reprint author), Monash Med Ctr, Child & Adolescent Menatl Hlth Serv, 246 Clayton Rd, Clayton, Vic 3168, Australia.
EM kylie.gray@med.monash.edu.au
RI Gray, Kylie/H-3345-2014
OI Gray, Kylie/0000-0001-6518-4240
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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Schopler E, 1990, INDIVIDUALIZED ASSES
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World Health Organisation, 1992, ICD 10 CLASS MENT BE
NR 27
TC 36
Z9 37
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 657
EP 667
DI 10.1007/s10803-007-0432-y
PG 11
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700007
PM 17690967
ER
PT J
AU Idol, JR
Addington, AM
Long, RT
Rapoport, JL
Green, ED
AF Idol, Jacquelyn R.
Addington, Anjene M.
Long, Robert T.
Rapoport, Judith L.
Green, Eric D.
TI Sequencing and analyzing the t(1;7) reciprocal translocation breakpoints
associated with a case of childhood-onset schizophrenia/autistic
disorder
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE schizophrenia; autism; translocation; genetics; genome analysis;
cytogenetics
ID HUMAN INHERITED DISEASE; AUTISM SPECTRUM; PHYSICAL MAP; HUMAN GENOME;
7Q; IDENTIFICATION; CHROMOSOME-1; POPULATION; FAMILIES; CHILDREN
AB We characterized a t(1;7)(p22;q21) reciprocal translocation in a patient with childhood-onset schizophrenia (COS) and autism using genome mapping and sequencing methods. Based on genomic maps of human chromosome 7 and fluorescence in situ hybridization (FISH) studies, we delimited the region of 7q21 harboring the translocation breakpoint to a similar to 16-kb interval. A cosmid containing the translocation-associated 1:7 junction on der(1) was isolated and sequenced, revealing the positions on chromosomes 1 and 7, respectively, where the translocation occurred. PCR-based studies enabled the isolation and sequencing of the reciprocal 7:1 junction on der(7). No currently recognized gene on either chromosome appears to be disrupted by the translocation. We further found no evidence for copy-number differences in the genomic regions flanking the translocation junctions in the patient. Our efforts provide sequence-based information about a schizophrenia/autism-associated translocation, and may facilitate future studies investigating the genetic bases of these disorders.
C1 [Idol, Jacquelyn R.; Green, Eric D.] NHGRI, Genome Technol Branch, NIH, Bethesda, MD 20892 USA.
[Green, Eric D.] NHGRI, NIH Intramural Sequencing Ctr, NIH, Bethesda, MD 20892 USA.
[Addington, Anjene M.; Long, Robert T.; Rapoport, Judith L.] NIMH, Child Psychiat Branch, NIH, Bethesda, MD 20892 USA.
RP Green, ED (reprint author), NHGRI, Genome Technol Branch, NIH, 50 South Dr,Bldg 50 Rm, Bethesda, MD 20892 USA.
EM egreen@nhgri.nih.gov
CR Abeysinghe SS, 2003, HUM MUTAT, V22, P229, DOI 10.1002/humu.10254
American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Auranen M, 2000, MOL PSYCHIATR, V5, P320, DOI 10.1038/sj.mp.4000708
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NR 34
TC 4
Z9 4
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 668
EP 677
DI 10.1007/s10803-007-0435-8
PG 10
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700008
PM 17879154
ER
PT J
AU Callahan, K
Henson, RK
Cowan, AK
AF Callahan, Kevin
Henson, Robin K.
Cowan, Angela K.
TI Social validation of evidence-based practices in autism by parents,
teachers, and administrators
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; social validation; special education; evidence-based practices
ID ISSUES; DISORDERS; VALIDITY; CHILDREN
AB Relatively little attention has been devoted to the social validation of potentially effective autism interventions. Thus, it is often difficult to identify and implement evidence-based practices, and programming is often inadequate. The authors identified autism intervention components with reported effectiveness for school settings. The results of a social validation survey completed by parents, teachers, and administrators indicate strong, consistent support for program components falling within five functional areas: (a) individualized programming, (b) data collection, (c) the use of empirically-based strategies, (d) active collaboration, and (e) a focus on long-term outcomes. These socially validated interventions can be used to evaluate existing autism curricula and develop training for professionals, parents, and students in order to improve public school autism programs.
C1 [Callahan, Kevin; Henson, Robin K.; Cowan, Angela K.] Univ N Texas, Dept Educ Psychol, Denton, TX 76203 USA.
RP Callahan, K (reprint author), Univ N Texas, Dept Educ Psychol, POB 311335, Denton, TX 76203 USA.
EM Callahan@unt.edu
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NR 35
TC 90
Z9 90
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 678
EP 692
DI 10.1007/s10803-007-0434-9
PG 15
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700009
PM 17924182
ER
PT J
AU Chiang, HM
Carter, M
AF Chiang, Hsu-Min
Carter, Mark
TI Spontaneity of communication in individuals with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE spontaneous communication; autism
ID CHAIN INTERRUPTION STRATEGY; STIMULUS OVERSELECTIVITY; SPECTRUM
DISORDERS; TIME-DELAY; NATURAL-ENVIRONMENT; SOCIAL INITIATIONS;
MENTAL-RETARDATION; SPONTANEOUS SPEECH; TEACHING-CHILDREN; LANGUAGE
AB This article provides an examination of issues related to spontaneity of communication in children with autism. Deficits relating to spontaneity or initiation are frequently reported in individuals with autism, particularly in relation to communication and social behavior. Nevertheless, spontaneity is not necessarily clearly conceptualized or measured. Several approaches to conceptualization of communicative spontaneity are examined with a particular focus on the continuum model and how it might be practically applied. A range of possible explanations for deficits in spontaneity of communication in children with autism is subsequently explored, including external factors (highly structured teaching programs, failure to systematically instruct for spontaneity) and intrinsic characteristics (intellectual disability, stimulus overselectivity, weak central coherence). Possible implications for future research are presented.
C1 [Chiang, Hsu-Min; Carter, Mark] Macquarie Univ, Special Educ Ctr, Sydney, NSW 2109, Australia.
RP Chiang, HM (reprint author), Macquarie Univ, Special Educ Ctr, Sydney, NSW 2109, Australia.
EM mina.chiang@speced.sed.mq.edu.au
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NR 99
TC 12
Z9 12
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 693
EP 705
DI 10.1007/s10803-007-0436-7
PG 13
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700010
PM 17682930
ER
PT J
AU Herba, CM
de Bruin, E
Althaus, M
Verheij, F
Ferdinand, RF
AF Herba, Catherine M.
de Bruin, Esther
Althaus, Monika
Verheij, Fop
Ferdinand, Robert F.
TI Face and emotion recognition in MCDD versus PDD-NOS
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE MCDD; pervasive developmental disorders; PDD-NOS; face recognition;
facial expression recognition; emotion recognition
ID COMPLEX DEVELOPMENTAL DISORDER; FACIAL EXPRESSIONS; PSYCHOSOCIAL STRESS;
BORDERLINE SYNDROME; SPECTRUM DISORDERS; CHILDREN; AUTISM; MULTIPLE;
CHILDHOOD; SCHIZOPHRENIA
AB Previous studies indicate that Multiple Complex Developmental Disorder (MCDD) children differ from PDD-NOS and autistic children on a symptom level and on psychophysiological functioning. Children with MCDD (n = 21) and PDD-NOS (n = 62) were compared on two facets of social-cognitive functioning: identification of neutral faces and facial expressions. Few significant group differences emerged. Children with PDD-NOS demonstrated a more attention-demanding strategy of face processing, and processed neutral faces more similarly to complex patterns whereas children with MCDD showed an advantage for face recognition compared to complex patterns. Results further suggested that any disadvantage in face recognition was related more to the autistic features of the PDD-NOS group rather than characteristics specific to MCDD. No significant group differences emerged for identifying facial expressions.
C1 [Herba, Catherine M.; de Bruin, Esther; Verheij, Fop; Ferdinand, Robert F.] Sophia Childrens Univ Hosp, Dept Child & Adolescent Psychiat, Erasmus Med Ctr, NL-3000 CB Rotterdam, Netherlands.
[Althaus, Monika] Univ Groningen, Univ Ctr Child & Adolescent Psychiat, Groningen, Netherlands.
RP Herba, CM (reprint author), Sophia Childrens Univ Hosp, Dept Child & Adolescent Psychiat, Erasmus Med Ctr, POB 2060, NL-3000 CB Rotterdam, Netherlands.
EM c.herba@erasmusmc.nl
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NR 38
TC 8
Z9 8
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 706
EP 718
DI 10.1007/s10803-007-0438-5
PG 13
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700011
PM 17721812
ER
PT J
AU Lund, SK
Troha, JM
AF Lund, Shelley K.
Troha, Jeanette M.
TI Teaching young people who are blind and have autism to make requests
using a variation on the picture exchange communication system with
tactile symbols: A preliminary investigation
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE picture exchange communication system; autism; tactile symbols;
augmentative and alternative communication; communication intervention;
visual impairment
ID CHILDREN; PECS; TEACHERS; SPEECH
AB This study used a single-subject multiple baseline across participants design to evaluate the effectiveness of a modified picture exchange communication system (PECS) teaching protocol with tactile symbols. Three students (two male, one female) aged 12-17 years who had autism and were blind participated in the study. The instructional program involved three phases. First, each participant learned to exchange a tactile symbol with his/her communication partner to request a preferred item/activity. Second, the distance between the communication partner and the participant was increased. Third, the participants were required to discriminate between two dissimilar tactile symbols. One out of three participants completed all phases of the instructional program. Although the other two participants did not complete the program, they demonstrated improvement from baseline responding rates. This study provided preliminary results that using tactile symbols with strategies from PECS may be an effective method to teach requesting to youth who are blind and have autism.
C1 [Lund, Shelley K.; Troha, Jeanette M.] Univ Wisconsin, Dept Commun Sci & Disorders, Milwaukee, WI 53201 USA.
RP Lund, SK (reprint author), Univ Wisconsin, Dept Commun Sci & Disorders, POB 413, Milwaukee, WI 53201 USA.
EM sklund@uwm.edu
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NR 28
TC 25
Z9 25
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 719
EP 730
DI 10.1007/s10803-007-0439-4
PG 12
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700012
PM 17828449
ER
PT J
AU Bryson, SE
Zwaigenbaum, L
McDermott, C
Rombough, V
Brian, J
AF Bryson, Susan E.
Zwaigenbaum, Lonnie
McDermott, Catherine
Rombough, Vicki
Brian, Jessica
TI The autism observation scale for infants: Scale development and
reliability data
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autistic spectrum disorder; early detection; behaviour; assessment
ID SPECTRUM DISORDERS; PRESCHOOL-CHILDREN; AGE; DIAGNOSIS; SYMPTOMS; LIFE;
1ST; IDENTIFICATION; STAT
AB The Autism Observation Scale for Infants (AOSI) was developed to detect and monitor early signs of autism as they emerge in high-risk infants (all with an older sibling with an autistic spectrum disorder). Here we describe the scale and its development, and provide preliminary data on its reliability. Inter-rater reliability both for total scores and total number of endorsed items is good to excellent at 6, 12 and 18 months; reliability is more modest for individual items, particularly in 6-month-olds. Test-retest reliability of the AOSI at 12 months of age is within acceptable limits. Evidence that the AOSI provides reliable data is the first critical step towards evaluating its efficacy in distinguishing high-risk infants who develop ASD.
C1 [Bryson, Susan E.] IWK Hlth Ctr, Autism Res Ctr, Halifax, NS B3K 6R8, Canada.
[Bryson, Susan E.] Dalhousie Univ, Dept Pediat, Halifax, NS, Canada.
[Bryson, Susan E.] Dalhousie Univ, Dept Psychol, Halifax, NS, Canada.
[Zwaigenbaum, Lonnie] Univ Alberta, Dept Pediat, Edmonton, AB, Canada.
[McDermott, Catherine] York Univ, Toronto, ON M3J 2R7, Canada.
[Rombough, Vicki] N York Gen Hosp, Toronto, ON, Canada.
[Brian, Jessica] Hosp Sick Children & Bloorview Kids Rehab, Toronto, ON, Canada.
RP Bryson, SE (reprint author), IWK Hlth Ctr, Autism Res Ctr, 5850 Univ Ave, Halifax, NS B3K 6R8, Canada.
EM susan.bryson@iwk.nshealth.ca
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NR 36
TC 69
Z9 69
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 731
EP 738
DI 10.1007/s10803-007-0440-y
PG 8
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700013
PM 17874180
ER
PT J
AU Eaves, LC
Ho, HH
AF Eaves, Linda C.
Ho, Helena H.
TI Young adult outcome of autism spectrum disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism outcomes; young adults with autism
ID FOLLOW-UP; INFANTILE PSYCHOSIS; CHILDREN; PREVALENCE; CHILDHOOD; UPDATE
AB To learn about the lives of young adults with ASD, families with children born 1974-1984, diagnosed as preschoolers and followed into adolescence were contacted by mail. Of 76 eligible, 48 (63%) participated in a telephone interview. Global outcome scores were assigned based on work, friendships and independence. At mean age 24, half had good to fair outcome and 46% poor. Co-morbid conditions, obesity and medication use were common. Families noted unmet needs particularly in social areas. Multilinear regression indicated a combination of IQ and CARS score at age 11 predicted outcome. Earlier studies reported more adults with ASD who had poor to very poor outcomes, however current young people had more opportunities, and thus better results were expected.
C1 [Eaves, Linda C.] Sunny Hill Hlth Ctr Children, Dept Psychol, Vancouver, BC V5M 3E8, Canada.
[Ho, Helena H.] Univ British Columbia, Dept Pediat, Div Dev Pediat, Vancouver, BC V6T 1W5, Canada.
RP Eaves, LC (reprint author), Sunny Hill Hlth Ctr Children, Dept Psychol, 3644 Slocan St, Vancouver, BC V5M 3E8, Canada.
EM leaves@cw.bc.ca
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NR 36
TC 110
Z9 111
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 739
EP 747
DI 10.1007/s10803-007-0441-x
PG 9
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700014
PM 17764027
ER
PT J
AU Allen, D
Evans, C
Hider, A
Hawkins, S
Peckett, H
Morgan, H
AF Allen, David
Evans, Carys
Hider, Andrew
Hawkins, Sarah
Peckett, Helen
Morgan, Hugh
TI Offending behaviour in adults with asperger syndrome
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE asperger; offending; adults
ID DISORDER; PREVALENCE; VIOLENCE; AUTISM; DISABILITIES; PERSONALITY;
SAMPLE; PRISON
AB Considerable speculation is evident both within the scientific literature and popular media regarding possible links between Asperger syndrome and offending. A survey methodology that utilised quantitative data collection was employed to investigate the prevalence of offending behaviour amongst adults with Asperger Syndrome in a large geographical area of South Wales, UK; qualitative interviews were then conducted with a sub-sample of those identified. A small number of participants meeting the study criteria were identified. For those who had offended, their experience of the criminal justice system was essentially negative. Possible implications of the results were discussed.
C1 [Allen, David; Evans, Carys; Hawkins, Sarah; Peckett, Helen] Bro Morgannwg NHS Trust, Special Projects Team, Unit 3, Cardiff CF5 5BS, Wales.
[Allen, David] Univ Glamorgan, Pontypridd CF37 1DL, M Glam, Wales.
[Hider, Andrew] Bro Morgannwg NHS Trust, Caswell Clin, Bridgend, Wales.
[Morgan, Hugh] Autism Cymru, Aberystwyth, Dyfed, Wales.
RP Allen, D (reprint author), Bro Morgannwg NHS Trust, Special Projects Team, Unit 3, 58-62 Cowbridge Rd W, Cardiff CF5 5BS, Wales.
EM David.Allen@bromor-tr.wales.nhs.uk
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NR 39
TC 21
Z9 22
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 748
EP 758
DI 10.1007/s10803-007-0442-9
PG 11
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700015
PM 17805955
ER
PT J
AU Ghanizadeh, A
AF Ghanizadeh, Ahmad
TI A preliminary study on screening prevalence of pervasive developmental
disorder in schoolchildren in Iran
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE epidemiology; prevalence; autism; asperger's; pervasive developmental
disorder; students; Iran
ID AUTISM SPECTRUM DISORDER; CHILDREN; TRENDS
AB To study prevalence rates of pervasive developmental disorder (PDD) symptoms and differences between subtypes in school age Iranian children. A random sample of 2,000 school age children from both genders was selected. A parent-completed, DSM-IV-referenced rating scale of PDD symptoms was used. About 1.9% of the sample obtained screening cutoff scores for probable autistic disorder and 0.5 for probable asperger's disorder. The rate of probable PDD was not more in girls than the boys. The rate of suspected cases of PDD in Iran is very high and probable autistic disorder is not gender related. It shows the need for more consideration of PDD in the mental health programs planning.
C1 [Ghanizadeh, Ahmad] Hafez Hosp, Dept Psychiat, Shiraz, Iran.
[Ghanizadeh, Ahmad] Shiraz Univ Med Sci, Dept Child & Adolescent Psychiat, Hafez Hosp, Shiraz, Iran.
RP Ghanizadeh, A (reprint author), Hafez Hosp, Dept Psychiat, Shiraz, Iran.
EM ghanizad@sina.tums.ac.ir
RI Ghanizadeh, Ahmad/C-2177-2011
CR ALFVEN G, 1993, ACTA PAEDIATR, V82, P484, DOI 10.1111/j.1651-2227.1993.tb12728.x
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American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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Wazana A, 2007, J AM ACAD CHILD PSY, V46, P721, DOI 10.1097/chi.0b013e31804a7f3b
NR 18
TC 19
Z9 20
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 759
EP 763
DI 10.1007/s10803-007-0445-6
PG 5
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700016
PM 17879153
ER
PT J
AU Cicchetti, DV
Lord, C
Koenig, K
Klin, A
Volkmar, FR
AF Cicchetti, Domenic V.
Lord, Catherine
Koenig, Kathy
Klin, Ami
Volkmar, Fred R.
TI Reliability of the ADI-R: Multiple examiners evaluate a single case
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE ADI-R; reliability; single Case
ID WEIGHTED KAPPA; INTERRATER RELIABILITY; RATING-SCALES; AGREEMENT;
COEFFICIENT; BEHAVIOR; CRITERIA; ISSUES; OBJECT; RATERS
AB The authors assessed the reliability of the Autism Diagnostic Interview (ADI-R). Seven Clinical Examiners evaluated a three and one half year old female toddler suspected of being on the Autism Spectrum. Examiners showed agreement levels of 94-96% across all items, with weighted kappa (K-w) between .80 and .88. They were in 100% agreement on 74% of the items; in excellent agreement on 6% of the items (93-96%, with K-w between .78 and .85); in good agreement on 7% (89-90%, with K-w between .62 and 0.68); and in fair agreement on 3% (82 - 84%, with K-w between .40 and .47). For the remaining 10% of ADI-R items, examiners showed poor agreement (50-81% with K-w between -.67 and .37).
C1 [Cicchetti, Domenic V.] Yale Home Off, N Branford, CT 06471 USA.
[Cicchetti, Domenic V.; Koenig, Kathy; Klin, Ami; Volkmar, Fred R.] Yale Univ, Sch Med, Ctr Child Study, New Haven, CT 06510 USA.
[Lord, Catherine] Univ Michigan, Ann Arbor, MI 48109 USA.
RP Cicchetti, DV (reprint author), Yale Home Off, N Branford, CT 06471 USA.
EM dom.cicchetti@yale.edu
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NR 28
TC 18
Z9 18
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 764
EP 770
DI 10.1007/s10803-007-0448-3
PG 7
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700017
PM 18058216
ER
PT J
AU Baharav, E
Darling, R
AF Baharav, Eva
Darling, Rieko
TI Case report: Using an auditory trainer with caregiver video modeling to
enhance communication and socialization behaviors in autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; video modeling; auditory trainer; communication; social
orienting; early intervention
ID JOINT ATTENTION; YOUNG-CHILDREN; FACE; SPEECH
AB A minimally verbal child with autism was exposed to short daily sessions of watching his parents on video in conjunction with an FM auditory trainer for a period of 4 weeks. Baseline measures of verbal and social behaviors were taken pre-treatment and repeated post treatment. Results indicate substantial gains in word productions, social orienting, and increased eye contact. Results are discussed in terms of the contributions of auditory-visual processing to establishing communication and socialization in autism and early intervention effectiveness.
C1 [Baharav, Eva; Darling, Rieko] Western Washington Univ, Dept Commun Sci & Disorders, Bellingham, WA 98225 USA.
RP Baharav, E (reprint author), Western Washington Univ, Dept Commun Sci & Disorders, 516 High St, Bellingham, WA 98225 USA.
EM eva.baharav@wwu.edu
CR BAHARAV E, 2005, ANN C WASH SPEECH HE
BAHARAV E, IN PRESS USING AUDIT
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NR 21
TC 9
Z9 10
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 771
EP 775
DI 10.1007/s10803-007-0429-6
PG 5
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700018
PM 17710521
ER
PT J
AU Bolte, S
Feineis-Matthews, S
Poustka, F
AF Boelte, Sven
Feineis-Matthews, Sabine
Poustka, Fritz
TI Brief report: Emotional processing in high-functioning
autism-physiological reactivity and affective report
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE IAPS; self-assessment manikin; emotion induction; autonomic response
ID FACIAL AFFECT; GERMAN FORM; CHILDREN; RELIABILITY; RECOGNITION; DISORDER
AB This study examined physiological response and affective report in 10 adult individuals with autism and 10 typically developing controls. An emotion induction paradigm using stimuli from the International Affective Picture System was applied. Blood pressure, heart and self-ratings of experienced valence (pleasure), arousal and dominance (control) were assessed during the experiment. Physiological response profiles correlated low to significantly negative between groups. Individuals with autism experienced less arousal when viewing sad pictures but higher arousal while processing neutral stimuli. In addition, they reported more control than the normative group when viewing fearful and sad stimuli. Findings indicate altered physiological reactivity and affective report in autism, which may be related to more general impairments in socio-emotional functioning.
C1 [Boelte, Sven; Feineis-Matthews, Sabine; Poustka, Fritz] Univ Frankfurt, Klin Psychiat & Psychotherapie Kindes & Jugendalt, D-60528 Frankfurt, Germany.
RP Bolte, S (reprint author), Univ Frankfurt, Klin Psychiat & Psychotherapie Kindes & Jugendalt, Deutschordenstr 50, D-60528 Frankfurt, Germany.
EM Boelte@em.uni-frankfurt.de
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NR 24
TC 26
Z9 27
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 776
EP 781
DI 10.1007/s10803-007-0443-8
PG 6
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700019
PM 17882540
ER
PT J
AU Spek, AA
Scholte, EM
van Berckelaer-Onnes, IA
AF Spek, Antoinette A.
Scholte, Evert M.
van Berckelaer-Onnes, Ina A.
TI Brief report: The use of WAIS-III in adults with HFA and asperger
syndrome
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE high functioning autism; asperger syndrome; intelligence; WAIS-III;
processing speed
ID AUTISTIC INDIVIDUALS; CHILDREN; IV
AB The WAIS III was administered to 16 adults with high functioning autism (HFA) and 27 adults with Asperger syndrome. Differences between Verbal Intelligence (VIQ) and Performance Intelligence (PIQ) were not found. Processing Speed problems in people with HFA appeared. At the subtest level, the Asperger syndrome group performed weak on Digit Span. Comprehension and Block Design were relative strengths. In the HFA group, performance on Digit-Symbol Coding and Symbol Search was relatively poor. Strengths were found on Information and Matrix Reasoning. The results suggest that the VIQ-PIQ difference cannot distinguish between HFA and Asperger syndrome. WAIS III Factor Scale and Subtest patterning provides a more valid indicator.
C1 [Spek, Antoinette A.] GGZ Eindhoven, NL-5626 AB Eindhoven, Netherlands.
[Scholte, Evert M.; van Berckelaer-Onnes, Ina A.] Leiden Univ, Dept Clin Child & Adolescent Studies, Leiden, Netherlands.
RP Spek, AA (reprint author), GGZ Eindhoven, Boschdijk 771,Postvak 1418, NL-5626 AB Eindhoven, Netherlands.
EM aa.spek@ggze.nl
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NR 26
TC 26
Z9 27
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD APR
PY 2008
VL 38
IS 4
BP 782
EP 787
DI 10.1007/s10803-007-0446-5
PG 6
WC Psychology, Developmental
SC Psychology
GA 275XO
UT WOS:000254105700020
PM 17879152
ER
PT J
AU Parikh, MS
Kolevzon, A
Hollander, E
AF Parikh, Mihir S.
Kolevzon, Alexander
Hollander, Eric
TI Psychopharmacology of aggression in children and adolescents with
autism: A critical review of efficacy and tolerability
SO JOURNAL OF CHILD AND ADOLESCENT PSYCHOPHARMACOLOGY
LA English
DT Review
ID PERVASIVE DEVELOPMENTAL DISORDERS; DEFICIT HYPERACTIVITY DISORDER;
PLACEBO-CONTROLLED TRIAL; EARLY INFANTILE-AUTISM; WHOLE-BLOOD SEROTONIN;
MENTALLY-RETARDED CHILDREN; SELF-INJURIOUS-BEHAVIOR; PLASMA
BETA-ENDORPHIN; FATTY-ACID LEVELS; DOUBLE-BLIND
AB Background: Autism is characterized by a clinical triad of symptoms that affect social, language, and behavioral domains. Aggression and self-injury may be associated symptoms of autism and can result in significant harm to those affected as well as marked distress for their families. The precise nature of the relationship between aggressive or self-injurious behavior (SIB) and autism remains unclear and as a result, these symptoms are treated with a broad range of pharmacological approaches. This review seeks to systematically and critically examine the evidence for the pharmacological management of aggression and SIB in children with autism spectrum disorders.
Method: The entire PubMed database was searched for English language biomedical articles on clinical trials with medication in autism spectrum disorders. Studies were selected based on the following inclusion criteria: (1) randomized placebo-controlled trials; (2) a sample population that included children and adolescents; (3) at least one standardized assessment of aggression as a primary outcome measure of the study.
Results: Twenty one trials with 12 medications were identified. Five medications produced significant improvement as compared to placebo, including tianeptine, methylphenidate, risperidone, clonidine, and naltrexone. Only risperidone and methylphenidate demonstrate results that have been replicated across at least two studies.
Conclusions: Although many medications have been studied under placebo-controlled conditions, few produce significant improvement. Additional placebo-controlled trials are needed to increase the number of therapeutic options available in the treatment of aggression in autism.
C1 [Kolevzon, Alexander; Hollander, Eric] Mt Sinai Sch Med, Dept Psychiat, New York, NY 10029 USA.
[Parikh, Mihir S.] Mt Sinai Sch Med, New York, NY USA.
[Kolevzon, Alexander; Hollander, Eric] Mt Sinai Sch Med, Seaver Autism Res Ctr, New York, NY USA.
RP Kolevzon, A (reprint author), Mt Sinai Sch Med, Dept Psychiat, 1 Gustave L Levy Pl,Box 1230, New York, NY 10029 USA.
EM alexander.kolevzon@mssm.edu
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NR 129
TC 38
Z9 40
PU MARY ANN LIEBERT INC
PI NEW ROCHELLE
PA 140 HUGUENOT STREET, 3RD FL, NEW ROCHELLE, NY 10801 USA
SN 1044-5463
J9 J CHILD ADOL PSYCHOP
JI J. Child Adolesc. Psychopharmacol.
PD APR
PY 2008
VL 18
IS 2
BP 157
EP 178
DI 10.1089/cap.2007.0041
PG 22
WC Pediatrics; Pharmacology & Pharmacy; Psychiatry
SC Pediatrics; Pharmacology & Pharmacy; Psychiatry
GA 300LT
UT WOS:000255826000004
PM 18439113
ER
PT J
AU Malone, RP
Harvey, JA
AF Malone, Richard P.
Harvey, John A.
TI Abnormal movements with the addition of clindamycin to risperidone in a
girl with autism
SO JOURNAL OF CHILD AND ADOLESCENT PSYCHOPHARMACOLOGY
LA English
DT Letter
ID NEUROMUSCULAR BLOCKADE
C1 [Malone, Richard P.] Drexel Univ, Coll Med, Dept Psychiat, Philadelphia, PA 19124 USA.
[Malone, Richard P.; Harvey, John A.] Drexel Univ, Coll Med, Dept Physiol & Pharmacol, Philadelphia, PA 19104 USA.
RP Malone, RP (reprint author), Drexel Univ, Coll Med, Dept Psychiat, 4641 Roosevelt Blvd, Philadelphia, PA 19124 USA.
EM rmalone@drexelmed.edu; john.harvey@drexel.edu
CR Baldessarini RJ, 2006, GOODMAN GILMANS PHAR, P429
Best JA, 1999, J ORAL MAXIL SURG, V57, P600, DOI 10.1016/S0278-2391(99)90083-6
Sloan PA, 2002, ANESTH ANALG, V94, P123, DOI 10.1097/00000539-200201000-00023
NR 3
TC 2
Z9 2
PU MARY ANN LIEBERT INC
PI NEW ROCHELLE
PA 140 HUGUENOT STREET, 3RD FL, NEW ROCHELLE, NY 10801 USA
SN 1044-5463
J9 J CHILD ADOL PSYCHOP
JI J. Child Adolesc. Psychopharmacol.
PD APR
PY 2008
VL 18
IS 2
BP 221
EP 222
DI 10.1089/cap.2007.0134
PG 2
WC Pediatrics; Pharmacology & Pharmacy; Psychiatry
SC Pediatrics; Pharmacology & Pharmacy; Psychiatry
GA 300LT
UT WOS:000255826000011
PM 18439121
ER
PT J
AU Aschner, M
AF Aschner, Michael
TI Response to article by DeSoto and Hitlan on the relationship between
mercury exposure and autism
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Letter
DE mercury; autism
C1 Vanderbilt Univ, Med Ctr, Dept Pediat, Nashville, TN 37232 USA.
RP Aschner, M (reprint author), Vanderbilt Univ, Med Ctr, Dept Pediat, Nashville, TN 37232 USA.
CR Budtz-Jorgensen E, 2004, ENVIRON RES, V95, P385, DOI 10.1016/j.envres.2003.11.001
Desoto M Catherine, 2007, J Child Neurol, V22, P1308, DOI 10.1177/0883073807307111
Ip P, 2004, J CHILD NEUROL, V19, P431
NR 3
TC 2
Z9 2
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD APR
PY 2008
VL 23
IS 4
BP 463
EP 463
DI 10.1177/0883073808314647
PG 1
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 282ER
UT WOS:000254550900020
PM 18401039
ER
PT J
AU DeSoto, MC
Hitlan, RT
AF DeSoto, M. Catherine
Hitlan, Robert T.
TI Concerning blood mercury levels and autism: A need to clarify
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Letter
C1 [DeSoto, M. Catherine; Hitlan, Robert T.] Univ No Iowa, Dept Psychol, Cedar Falls, IA 50614 USA.
RP DeSoto, MC (reprint author), Univ No Iowa, Dept Psychol, Cedar Falls, IA 50614 USA.
CR ASCHNER M, 2008, J CHILD NEUROL, P23
BRUMBACK RA, J CHILD NEUROL, V22, P1321
DESOTO MC, 2007, AM J PSYCHOL, V9, P535
Desoto M Catherine, 2007, J Child Neurol, V22, P1308, DOI 10.1177/0883073807307111
DESOTO MC, 2007, FREQUENTLY ASKED QUE
*EPA, HUM EXP
HORNIG M, 2004, MOL PSYCHIATR, P1, DOI 10.1017/CBO9780511490576.002
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KOHANE I, BLOOD LEVELS MERCURY
Tamm C, 2006, J NEUROCHEM, V97, P69, DOI 10.1111/j.1471-4159.2006.03718.x
NR 11
TC 3
Z9 3
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD APR
PY 2008
VL 23
IS 4
BP 463
EP 465
DI 10.1177/0883073808314718
PG 3
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 282ER
UT WOS:000254550900021
ER
PT J
AU Rogers, SJ
Young, GS
Cook, I
Giolzetti, A
Ozonoff, S
AF Rogers, Sally J.
Young, Gregory S.
Cook, Ian
Giolzetti, Angelo
Ozonoff, Sally
TI Deferred and immediate imitation in regressive and early onset autism
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE autistic disorder; development; developmental delay; mental retardation;
pervasive developmental disorder; preschool children; imitation
ID SPECTRUM DISORDERS; YOUNG-CHILDREN; DEVELOPMENTAL DISORDERS; INFANT
IMITATION; JOINT ATTENTION; PRETEND PLAY; MEMORY; COMMUNICATION;
INDIVIDUALS; CPEA
AB Deferred imitation has long held a privileged position in early cognitive development, considered an early marker of representational thought with links to language development and symbolic processes. Children with autism have difficulties with several abilities generally thought to be related to deferred imitation: immediate imitation, language, and symbolic play. However, few studies have examined deferred imitation in early autism. The present study examined both deferred, spontaneous imitation and immediate, elicited imitation on a set of carefully matched tasks in 36 young children with autism: 16 with early onset autism, 20 with regressive autism and two contrast groups, younger typically developing children (n = 20) and age matched children with significant developmental delays (n = 21). Analyses of co-variance controlling for differences in verbal mental age revealed significant main effects for task, but no main effect of group and no interaction of task by group. Deferred imitation scores were lower than immediate imitation scores for all groups. Imitation performance was related to overall intellectual functioning for all groups, and there were moderate and significant relations between imitation in the immediate elicited condition and in the spontaneous deferred condition for all groups. Finally, there were no differences between onset subgroups in imitation scores, suggesting that the two share a similar phenotype involving both types of imitation.
C1 [Rogers, Sally J.; Young, Gregory S.; Cook, Ian; Giolzetti, Angelo; Ozonoff, Sally] Univ Calif Davis, MIND Inst, Sacramento, CA 95817 USA.
RP Rogers, SJ (reprint author), Univ Calif Davis, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM sally.rogers@ucdmc.ucdavis.edu
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NR 34
TC 7
Z9 7
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD APR
PY 2008
VL 49
IS 4
BP 449
EP 457
DI 10.1111/j.1469-7610.2007.01866.x
PG 9
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 278HL
UT WOS:000254275700011
PM 18221343
ER
PT J
AU Carneiro, AMD
Cook, EH
Murphy, DL
Blakely, RD
AF Carneiro, Ana Marin D.
Cook, Edwin H.
Murphy, Dennis L.
Blakely, Randy D.
TI Interactions between integrin alpha IIb beta 3 and the serotonin
transporter regulate serotonin transport and platelet aggregation in
mice and humans
SO JOURNAL OF CLINICAL INVESTIGATION
LA English
DT Article
ID ACTIVATED PROTEIN-KINASE; WHOLE-BLOOD SEROTONIN; MYOCARDIAL-INFARCTION;
REUPTAKE INHIBITORS; SELECTIVE SEROTONIN; PROTHROMBOTIC RISK;
GLYCOPROTEIN IIIA; P38 MAPK; PHOSPHORYLATION; POLYMORPHISM
AB The essential contribution of the antidepressant-sensitive serotonin (5-HT) transporter SERT (which is encoded by the SLC6A4 gene) to platelet 5-HT stores suggests an important role of this transporter in platelet function. Here, using SERT-deficient mice, we have established a role for constitutive SERT expression in efficient ADP-and thrombin-triggered platelet aggregation. Additionally, using pharmacological blockers of SERT and the vesicular monoamine transporter (VMAT), we have identified a role for ongoing 5-HT release and SERT activity in efficient human platelet aggregation. We have also demonstrated that fibrinogen, an activator of integrin alpha IIb beta 3, enhances SERT activity in human platelets and that integrin alpha IIb beta 3 interacts directly with the C terminus of SERT. Consistent with these findings, knockout mice lacking integrin beta 3 displayed diminished platelet SERT activity. Conversely, HEK293 cells engineered to express human SERT and an activated form of integrin beta 3 exhibited enhanced SERT function that coincided with elevated SERT surface expression. Our results support an unsuspected role of alpha IIb beta 3/SERT associations as well as alpha IIb beta 3 activation in control of SERT activity in vivo that may have broad implications for hyperserotonemia, cardiovascular disorders, and autism.
C1 [Carneiro, Ana Marin D.; Blakely, Randy D.] Vanderbilt Univ, Sch Med, Dept Pharmacol, Nashville, TN 37232 USA.
[Cook, Edwin H.] Univ Illinois, Chicago, IL USA.
[Murphy, Dennis L.] NIMH, Clin Sci Lab, Bethesda, MD 20892 USA.
[Blakely, Randy D.] Vanderbilt Univ, Sch Med, Dept Psychiat, Nashville, TN 37212 USA.
[Blakely, Randy D.] Vanderbilt Univ, Sch Med, Ctr Mol Neurosci, Nashville, TN 37212 USA.
RP Blakely, RD (reprint author), Vanderbilt Univ, Sch Med, Dept Pharmacol, 7140 MRBIII, Nashville, TN 37232 USA.
EM randy.blakely@vanderbilt.edu
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NR 50
TC 69
Z9 70
PU AMER SOC CLINICAL INVESTIGATION INC
PI ANN ARBOR
PA 35 RESEARCH DR, STE 300, ANN ARBOR, MI 48103 USA
SN 0021-9738
J9 J CLIN INVEST
JI J. Clin. Invest.
PD APR
PY 2008
VL 118
IS 4
BP 1544
EP 1552
DI 10.1172/JCI33374
PG 9
WC Medicine, Research & Experimental
SC Research & Experimental Medicine
GA 282SX
UT WOS:000254588600047
PM 18317590
ER
PT J
AU Nickels, KC
Katusic, SK
Colligan, RC
Weaver, AL
Voigt, RG
Barbaresi, WJ
AF Nickels, Katherine C.
Katusic, Slavica K.
Colligan, Robert C.
Weaver, Amy L.
Voigt, Robert G.
Barbaresi, William J.
TI Stimulant medication treatment of target behaviors in children with
autism: A population-based study
SO JOURNAL OF DEVELOPMENTAL AND BEHAVIORAL PEDIATRICS
LA English
DT Article
DE autism; autistic disorder; stimulant medications
ID PERVASIVE DEVELOPMENTAL DISORDERS; ATTENTION-DEFICIT/HYPERACTIVITY
DISORDER; DEFICIT HYPERACTIVITY DISORDER; MENTALLY-RETARDED CHILDREN;
METHYLPHENIDATE; EFFICACY; PATTERNS; ADHD; RETARDATION; PREVALENCE
AB Objective: This study provides detailed information about stimulant medication treatment for the target symptoms of hyperactivity, impulsivity, disinhibition, and inattention in children with autism. Methods: In a previous study, 124 subjects fulfilling DSM-IV-based research criteria for autistic disorder were identified among all 0-21 year old residents of Olmsted County, MN from 1976-1997. For each of these 124 children with research-identified autism, information was abstracted on all prescribed psychopharmacological medications. Results: Psychostimulants were used to treat 52.4% (N = 65) of the 124 subjects. The median total duration of psychostimulant treatment was 4.0 years. There were 398 episodes of psychostimulant treatment. Favorable responses were associated with 69.4% of treatment episodes. Of the 398 episodes of stimulant treatment, 16.8% were associated with a documented side effect. At least one side effect was experienced by 66% of the children. Conclusion: These results indicate that psychostimulants are commonly prescribed for children with autism, and suggest that these medications may improve the target symptoms of hyperactivity, impulsivity, disinhibition and inattention.
C1 [Voigt, Robert G.; Barbaresi, William J.] Mayo Clin, Coll Med, Dana Child Dev & Learning Disorders Program, Rochester, MN 55905 USA.
[Katusic, Slavica K.; Voigt, Robert G.; Barbaresi, William J.] Mayo Clin, Coll Med, Dept Pediat & Adolescent Med, Div Dev & Behav Pediat, Rochester, MN 55905 USA.
[Katusic, Slavica K.] Mayo Clin, Coll Med, Dept Hlth Sci Res, Div Clin Epidemiol, Rochester, MN 55905 USA.
[Colligan, Robert C.] Mayo Clin, Coll Med, Dept Psychiat & Psychol, Rochester, MN 55905 USA.
[Weaver, Amy L.] Mayo Clin, Coll Med, Dept Hlth Sci Res, Div Biostat, Rochester, MN 55905 USA.
RP Barbaresi, WJ (reprint author), Mayo Clin, Coll Med, Dana Child Dev & Learning Disorders Program, 200 1st St SW, Rochester, MN 55905 USA.
EM barbaresi.william@mayo.edu
CR Aman MG, 2003, J CHILD ADOL PSYCHOP, V13, P29, DOI 10.1089/104454603321666171
Aman MG, 2005, J CHILD ADOL PSYCHOP, V15, P116, DOI 10.1089/cap.2005.15.116
AMAN MG, 1995, J AM ACAD CHILD PSY, V34, P1672, DOI 10.1097/00004583-199512000-00018
Aman MG, 2000, J AUTISM DEV DISORD, V30, P451, DOI 10.1023/A:1005559725475
Perrin JM, 2001, PEDIATRICS, V108, P1033
American Psychiatric Association, 2000, DIAGN CRIT DSM 4 TR
American Psychiatric Association, 1994, DIAGN STAT MAN MENT, P65
Barbaresi WJ, 2006, J DEV BEHAV PEDIATR, V27, P1, DOI 10.1097/00004703-200602000-00001
Barbaresi WJ, 2005, ARCH PEDIAT ADOL MED, V159, P37, DOI 10.1001/archpedi.159.1.37
Campbell M, 1996, J AM ACAD CHILD PSY, V35, P134, DOI 10.1097/00004583-199602000-00005
Carminati GG, 2006, PROG NEURO-PSYCHOPH, V30, P312, DOI 10.1016/j.pnpbp.2005.10.002
Challman TD, 2003, J AUTISM DEV DISORD, V33, P187, DOI 10.1023/A:1022995611730
Di Martino A, 2004, J CHILD ADOL PSYCHOP, V14, P207, DOI 10.1089/1044546041649011
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Palermo MT, 2004, J CHILD NEUROL, V19, P155
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Stigler KA, 2004, J CHILD ADOL PSYCHOP, V14, P49, DOI 10.1089/104454604773840481
Volkmar FR, 2003, LANCET, V362, P1133, DOI 10.1016/S0140-6736(03)14471-6
NR 31
TC 15
Z9 15
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0196-206X
J9 J DEV BEHAV PEDIATR
JI J. Dev. Behav. Pediatr.
PD APR
PY 2008
VL 29
IS 2
BP 75
EP 81
DI 10.1097/DBP.0b013e31815f24f7
PG 7
WC Behavioral Sciences; Psychology, Developmental; Pediatrics
SC Behavioral Sciences; Psychology; Pediatrics
GA 289GF
UT WOS:000255042300001
PM 18478626
ER
PT J
AU Audet, JR
AF Audet, Jeannine R.
TI Making sense of autism: The authoritave guide for non-experts
SO JOURNAL OF DEVELOPMENTAL AND BEHAVIORAL PEDIATRICS
LA English
DT Book Review
C1 [Audet, Jeannine R.] St Annes Hosp, Fall River, MA USA.
RP Audet, JR (reprint author), St Annes Hosp, Fall River, MA USA.
CR Thompson T., 2007, MAKING SENSE AUTISM
NR 1
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0196-206X
J9 J DEV BEHAV PEDIATR
JI J. Dev. Behav. Pediatr.
PD APR
PY 2008
VL 29
IS 2
BP 81
EP 81
DI 10.1097/DBP.0b013e31816c001b
PG 1
WC Behavioral Sciences; Psychology, Developmental; Pediatrics
SC Behavioral Sciences; Psychology; Pediatrics
GA 289GF
UT WOS:000255042300002
ER
PT J
AU Goodlin-Jones, BL
Sitnick, SL
Tang, K
Liu, JY
Anders, TF
AF Goodlin-Jones, Beth L.
Sitnick, Stephanie L.
Tang, Karen
Liu, Jingyi
Anders, Thomas F.
TI The children's sleep habits questionnaire in toddlers and preschool
children
SO JOURNAL OF DEVELOPMENTAL AND BEHAVIORAL PEDIATRICS
LA English
DT Article
DE sleep problems; screening instruments; toddlers; preschool; CSHQ
ID SCREENING QUESTIONNAIRE; DEVELOPMENTAL DISORDERS; NIGHT WAKINGS; AUTISM;
DISTURBANCES; INFANTS; SPECTRUM; PATTERNS; SAMPLE; LIFE
AB Objective: Twenty to 40% of young children are reported to have behavioral insomnias of childhood. Concerns about sleep at these ages are the most common problem expressed to pediatricians at the time of well child visits. A screening questionnaire, the Children's Sleep Habits Questionnaire (CSHQ), has been used in clinical settings and in research studies to assess children ages 4 to 10 for the presence of sleep problems. A CSHQ total score has distinguished clinical populations from community samples. Methods: The current study assesses the CSHQ in a younger age group than previously reported and in a diverse population. A total of 194 children, ages 2 to 51/2 years, were recruited into 3 diagnostic groups: 68 children with autism, 57 children with developmental delay without autism, and 69 typically developing children. All children's parents completed the CSHQ and a sleep log, and all children were studied for 7 days and nights with actigraphy. The children were divided into problem sleep and non-problem sleep groups on the basis of a parent report of a generic sleep problem at the time of entry into the study. The CSHQ responses for the problem and non-problem sleep groups were then compared. Results: The results suggest that the CSHQ is clinically useful for screening of sleep problems in typically developing children at these young ages as well as in children with diverse neurodevelopmental diagnoses. Conclusions: The somewhat higher subscale scores than previously reported for older children appear to be consistent with more sleep problems in younger children.
C1 [Goodlin-Jones, Beth L.; Tang, Karen; Anders, Thomas F.] Univ Calif Davis, MIND Inst, Sacramento, CA 95817 USA.
[Goodlin-Jones, Beth L.; Tang, Karen; Anders, Thomas F.] Univ Calif Davis, Dept Psychiat & Behav Sci, Sacramento, CA 95817 USA.
[Sitnick, Stephanie L.] Univ Calif Davis, Dept Human & Community Dev, Sacramento, CA 95817 USA.
[Liu, Jingyi] Univ Calif Davis, Dept Stat, Sacramento, CA 95817 USA.
RP Anders, TF (reprint author), Univ Calif Davis, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM tfanders@ucdavis.edu
CR Acebo C, 2005, SLEEP, V28, P1568
American Academy of Sleep Medicine, 2005, INT CLASS SLEEP DIS, V2nd
ANDERS TF, 1992, PEDIATRICS, V90, P554
Archbold KH, 2002, J PEDIATR-US, V140, P97, DOI 10.1067/mpd.2002.119990
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Dahl R E, 1996, Semin Pediatr Neurol, V3, P44, DOI 10.1016/S1071-9091(96)80028-3
El-Sheikh M, 2007, J CHILD PSYCHOL PSYC, V48, P88, DOI 10.1111/j.1469-7610.2006.01604.x
El-Sheikh M, 2006, CHILD DEV, V77, P31, DOI 10.1111/j.1467-8624.2006.00854.x
Fazzi E, 2006, FUNCT NEUROL, V21, P151
Giannotti F, 2006, J AUTISM DEV DISORD, V36, P741, DOI 10.1007/s10803-006-0116-z
GOODLINJONES B, HDB DEV PSYCHOPATHOL
Hering E, 1999, J AUTISM DEV DISORD, V29, P143, DOI 10.1023/A:1023092627223
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Malow BA, 2006, SLEEP, V29, P1563
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Mindell JA, 1999, CHILD ADOL PSYCH CL, V8, P695
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SITNICK S, IN PRESS SLEEP
Sparrow SS, 2005, VINELAND ADAPTIVE BE
Stores G, 1998, CHILD CARE HLTH DEV, V24, P5
Wiggs L, 2001, J ROY SOC MED, V94, P177
NR 33
TC 65
Z9 66
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0196-206X
J9 J DEV BEHAV PEDIATR
JI J. Dev. Behav. Pediatr.
PD APR
PY 2008
VL 29
IS 2
BP 82
EP 88
DI 10.1097/DBP.0b013e318163c39a
PG 7
WC Behavioral Sciences; Psychology, Developmental; Pediatrics
SC Behavioral Sciences; Psychology; Pediatrics
GA 289GF
UT WOS:000255042300003
PM 18478627
ER
PT J
AU Castillo, H
Patterson, B
Hickey, F
Kinsman, A
Howard, JM
Mitchell, T
Molloy, CA
AF Castillo, Heidi
Patterson, Bonnie
Hickey, Francis
Kinsman, Anne
Howard, Jennifer M.
Mitchell, Terry
Molloy, Cynthia A.
TI Difference in age at regression and without Down syndrome
SO JOURNAL OF DEVELOPMENTAL AND BEHAVIORAL PEDIATRICS
LA English
DT Article
DE autism; Down syndrome; regression; Autism Diagnostic Interview-Revised
ID AUTISM-SPECTRUM DISORDER; DIAGNOSTIC INTERVIEW; CHILDREN; INDIVIDUALS;
POPULATION; PREVALENCE; PHENOTYPE; CPEA
AB Objective: Autism occurs more frequently in individuals with Down syndrome than it does in the general population. Among children with autism and Down syndrome, regression is reported to occur in up to 50%. The aim of this study was to characterize and compare regression in children with autism with and without Down syndrome. Methods: in this case-control study, children with Down syndrome and autism characterized by a history of developmental regression (n = 12) were compared to children with autism with regression who did not have Down syndrome, matched for chronologic age and gender. Comparisons were made on age at acquisition of language and age at loss of language and other skills as measured by the Autism Diagnostic interview-Revised (ADI-R). Results: The mean age at acquisition of meaningful use of single words was 40.6 months (SD = 38.0) in children with Down syndrome and autism compared to 14.9 months (SD = 8.5) in children with autism without Down syndrome (p.005). The mean age at language loss in children with autism with Down syndrome was 61.8 months (SD 22.9) compared to 19.7 months (SD = 5.8) for those with autism without Down syndrome (p =.01). The mean age at other skill loss was 46.2 months (SD = 19.1) and 19.5 months (SD = 5.6), respectively (p =.006). Conclusions: When regression occurs in children with autism and Down syndrome it is, on average, much later than is typically seen in children with autism without Down syndrome.
C1 [Mitchell, Terry; Molloy, Cynthia A.] Cincinnati Childrens Hosp, Med Ctr, Ctr Biostat & Epidemiol, Cincinnati, OH 45229 USA.
[Castillo, Heidi; Patterson, Bonnie; Hickey, Francis] Cincinnati Childrens Hosp, Med Ctr, Div Dev & Behav Pediat, Cincinnati, OH 45229 USA.
[Kinsman, Anne] Greenville Hosp Syst Childrens Hosp, Div Dev Behav Pediat, Greenville, SC USA.
[Howard, Jennifer M.] Univ Kentucky, Coll Med, Lexington, KY USA.
[Patterson, Bonnie; Hickey, Francis; Molloy, Cynthia A.] Univ Cincinnati, Coll Med, Dept Pediat, Cincinnati, OH USA.
RP Molloy, CA (reprint author), Cincinnati Childrens Hosp, Med Ctr, Ctr Biostat & Epidemiol, 3333 Burnet Ave, Cincinnati, OH 45229 USA.
EM cynthia.molloy@cchmc.org
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Bertrand J, 2001, PEDIATRICS, V108, P1155, DOI 10.1542/peds.108.5.1155
Capone GT, 2005, AM J MED GENET A, V134A, P373, DOI 10.1002/ajmg.a.30622
de Bildt A, 2004, J AUTISM DEV DISORD, V34, P129
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GHAZIUDDIN M, 1992, J INTELL DISABIL RES, V36, P449
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World Health Organization (WHO), 1992, ICD 10 INT CLASS DIS
NR 33
TC 6
Z9 6
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0196-206X
J9 J DEV BEHAV PEDIATR
JI J. Dev. Behav. Pediatr.
PD APR
PY 2008
VL 29
IS 2
BP 89
EP 93
DI 10.1097/DBP.0b013e318165c78d
PG 5
WC Behavioral Sciences; Psychology, Developmental; Pediatrics
SC Behavioral Sciences; Psychology; Pediatrics
GA 289GF
UT WOS:000255042300004
PM 18367994
ER
PT J
AU Capone, GT
Goyal, P
Grados, M
Smith, B
Kammann, H
AF Capone, George T.
Goyal, Parag
Grados, Marco
Smith, Brandon
Kammann, Heather
TI Risperidone use in children with Down syndrome, severe intellectual
disability, and comorbid autistic spectrum disorders: A naturalistic
study
SO JOURNAL OF DEVELOPMENTAL AND BEHAVIORAL PEDIATRICS
LA English
DT Article
DE Aberrant Behavior Checklist; autistic-spectrum disorder; disruptive
behavior; Down syndrome; dual diagnosis; intellectual disability;
risperidone; self-injury; trisomy 21
ID ABERRANT BEHAVIOR CHECKLIST; DISRUPTIVE BEHAVIORS; DOUBLE-BLIND;
SLEEP-APNEA; OPEN-LABEL; ADOLESCENTS; SCALE; SCHIZOPHRENIA; INDIVIDUALS;
MECHANISM
AB Objective: We report on an open-label, naturalistic study using risperidone to treat disruptive behaviors and self-injury in children with Down syndrome, severe intellectual disability, and comorbid autism spectrum disorders (DS+ASDs). We hypothesized that hyperactivity and disruptive behaviors would improve in response to risperidone treatment consistent with previous studies of children with ASD. Methods: Subjects were children (mean age, 7.8 +/- 2.6 years), consisting of 20 males and three females identified through our outpatient Down Syndrome Clinic between 2000 and 2004. Results: Using the Aberrant Behavior Checklist as the primary outcome measure, all five subscales showed significant improvement following risperidone treatment. The mean duration of treatment was 95.8 +/- 16.8 days, and mean total daily dose was 0.66 +/- 0.28 mg/day. The Hyperactivity, Stereotypy, and Lethargy subscale scores showed the most significant reduction (p <.001), followed by irritability (p <.02), and Inappropriate Speech (p <.04). Children with disruptive behavior and self-injury showed the greatest improvement. Sleep quality improved for 88% of subjects with preexisting sleep disturbance. Subjects for whom a follow-up weight was available showed a mean weight increase of 2.8 +/- 1.5 kg during the treatment period. Conclusions: These findings support our clinical impression of improvement on important target behaviors such as aggression, disruptiveness, self-injury, stereotypy, and social withdrawal. Low-dose risperidone appears to be well tolerated in children with DS+ASD, although concerns about weight gain and metabolic alterations may limit its usefulness over the long term in some children.
C1 [Capone, George T.; Goyal, Parag; Kammann, Heather] Kennedy Krieger Inst, Div Neurol & Dev Med, Baltimore, MD 21231 USA.
[Capone, George T.] Kennedy Krieger Inst, Ctr Genet Disorders Cognit & Behav, Baltimore, MD 21231 USA.
[Capone, George T.] Johns Hopkins Univ, Sch Med, Dept Pediat, Baltimore, MD 21205 USA.
[Grados, Marco] Johns Hopkins Univ, Sch Med, Dept Psychiat & Behav Sci, Baltimore, MD 21205 USA.
[Smith, Brandon] Childrens Hosp Pittsburgh, Dept Pediat, Pittsburgh, PA 15213 USA.
RP Capone, GT (reprint author), Kennedy Krieger Inst, Div Neurol & Dev Med, 1750 E Fairmount Ave, Baltimore, MD 21231 USA.
EM capone@kennedykrieger.org
CR AMAN MG, 1985, AM J MENT DEF, V89, P485
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Campbell M, 1997, J AM ACAD CHILD PSY, V36, P835, DOI 10.1097/00004583-199706000-00022
Capone GT, 2005, AM J MED GENET A, V134A, P373, DOI 10.1002/ajmg.a.30622
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NR 42
TC 14
Z9 15
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0196-206X
J9 J DEV BEHAV PEDIATR
JI J. Dev. Behav. Pediatr.
PD APR
PY 2008
VL 29
IS 2
BP 106
EP 116
DI 10.1097/DBP.0b013e318165c100
PG 11
WC Behavioral Sciences; Psychology, Developmental; Pediatrics
SC Behavioral Sciences; Psychology; Pediatrics
GA 289GF
UT WOS:000255042300008
PM 18349709
ER
PT J
AU Matson, JL
Dempsey, T
AF Matson, Johnny L.
Dempsey, Timothy
TI Stereotypy in adults with autism spectrum disorders: Relationship and
diagnostic fidelity
SO JOURNAL OF DEVELOPMENTAL AND PHYSICAL DISABILITIES
LA English
DT Article
DE stereotypy; adults; autism spectrum disorders; intellectual disability
ID INTELLECTUAL DISABILITY; BEHAVIOR PROBLEMS; YOUNG-CHILDREN; PREVALENCE;
SKILLS; INTERVENTION; TIME; RISK; AGE
AB Autism Spectrum Disorders (ASD), are characterized by severe and debilitating symptoms including stereotypic behaviors. Stereotypies constitute core features of ASD and markedly impede attempts to remediate the disorder. Little previous research has examined characteristics of stereotypies in adults with ASD, or differentiated these behaviors as they present in ASD, from persons with intellectual disability (ID) only. In the present study 336 adults, those with ASD and ID versus ID alone were evaluated with respect to the nature and extent of their stereotypied behavior. A comparison of autistic to PDD-NOS participants was also conducted to offer a more fine-grained analysis specific to the ASD population. The nature and implications of these data for differential diagnosis in adults with ASD are discussed.
C1 [Matson, Johnny L.; Dempsey, Timothy] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, 236 Audubon Hall, Baton Rouge, LA 70803 USA.
EM Johnmatson@aol.com
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NR 40
TC 45
Z9 45
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1056-263X
J9 J DEV PHYS DISABIL
JI J. Dev. Phys. Disabil.
PD APR
PY 2008
VL 20
IS 2
BP 155
EP 165
DI 10.1007/s10882-007-9086-0
PG 11
WC Rehabilitation
SC Rehabilitation
GA 269WV
UT WOS:000253682400005
ER
PT J
AU Matson, JL
Dempsey, T
AF Matson, Johnny L.
Dempsey, Timothy
TI Autism spectrum disorders: Pharmacotherapy for challenging behaviors
SO JOURNAL OF DEVELOPMENTAL AND PHYSICAL DISABILITIES
LA English
DT Review
DE autism spectrum disorders; pharmacotherapy; challenging behaviors
ID PERVASIVE DEVELOPMENTAL DISORDERS; HIGH-FUNCTIONING AUTISM; CORE SYMPTOM
DOMAINS; OF-THE-LITERATURE; OPEN-LABEL; RISPERIDONE TREATMENT;
YOUNG-CHILDREN; ATYPICAL ANTIPSYCHOTICS; MENTAL-RETARDATION;
DOUBLE-BLIND
AB Autism spectrum disorders, a group of five conditions with similar behavioral characteristics, have received increasing research attention in recent years. Treatments have been successful at least to remediate or improve some symptoms, and the amount of research on the topic has been escalating very rapidly. The two primary forms of intervention studied the most to date have been applied behavior analysis and behavior therapy, and psychopharmacology. The purpose of this paper was to review efficacy of this latter category: drug related interventions for challenging behaviors. Effectiveness of these treatments based on the available research literature are reviewed with respect to current strengths and weaknesses of various psychotropic drug interventions. Trends in the data and potential future directions for research are discussed, including types of drugs used, targets for intervention, the future of combined behavior and drug interventions, and the measurement of side effects in psychopharmacology.
C1 [Matson, Johnny L.; Dempsey, Timothy] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
EM Johnmatson@aol.com
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NR 121
TC 20
Z9 20
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1056-263X
EI 1573-3580
J9 J DEV PHYS DISABIL
JI J. Dev. Phys. Disabil.
PD APR
PY 2008
VL 20
IS 2
BP 175
EP 191
DI 10.1007/s10882-007-9088-y
PG 17
WC Rehabilitation
SC Rehabilitation
GA 269WV
UT WOS:000253682400007
ER
PT J
AU Messier, J
Ferland, F
Majnemer, A
AF Messier, Julie
Ferland, Francine
Majnemer, Annette
TI Play behavior of school age children with intellectual disability: Their
capacities, interests and attitude
SO JOURNAL OF DEVELOPMENTAL AND PHYSICAL DISABILITIES
LA English
DT Article
DE children; intellectual disability; play; ludic behavior
ID OCCUPATIONAL-THERAPY; HANDICAPPED-CHILDREN; MENTAL-RETARDATION; AUTISM;
RELIABILITY; VALIDITY; SCALE
AB This study describes play of intellectually disabled children. A sample of 27 school aged children from five to seven years of age was evaluated using the Knox Preschool Play Scale and Ferland Assessment of Ludic Behavior. Play capacities and Ludic attitude were described and analyzed in relation to intellectual capacities. Children demonstrated good abilities in use of objects and space in both assessments. Furthermore, four out of six elements of the Ludic attitude: curiosity, initiative, pleasure, and spontaneity were present irrespective of IQ level. However, sense of humor and enjoyment of challenge were less present. Interestingly, the Imitation dimension showed relative weakness suggesting that this learning method may not be optimal in the school setting. The results highlight the strengths and limitations in play behaviors of children with intellectual disability.
C1 [Ferland, Francine] Univ Montreal, Programme Ergotherapie, Ecole Readaptat, Montreal, PQ H3C 3J7, Canada.
[Majnemer, Annette] McGill Univ, Montreal, PQ H3G 1Y5, Canada.
RP Messier, J (reprint author), 1195 Chateauneuf, Quebec City, PQ J2Y 1A4, Canada.
EM maxan@videotron.ca
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NR 33
TC 5
Z9 5
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1056-263X
J9 J DEV PHYS DISABIL
JI J. Dev. Phys. Disabil.
PD APR
PY 2008
VL 20
IS 2
BP 193
EP 207
DI 10.1007/s10882-007-9089-x
PG 15
WC Rehabilitation
SC Rehabilitation
GA 269WV
UT WOS:000253682400008
ER
PT J
AU Carr, D
Felce, J
AF Carr, D.
Felce, J.
TI Teaching picture-to-object relations in picture-based requesting by
children with autism: a comparison between error prevention and error
correction teaching procedures
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Article
DE autism; conditional discriminations; error correction; exclusion;
linguistics
ID SEVERE MENTAL-RETARDATION; EQUIVALENCE CLASSES; BEHAVIOR ANALYSIS;
EXCLUSION; LANGUAGE; INDIVIDUALS
AB Background Children who have a combination of language and developmental disabilities with autism often experience major difficulties in learning relations between objects and their graphic representations. Therefore, they would benefit from teaching procedures that minimize their difficulties in acquiring these relations. This study compared two teaching procedures, an error prevention procedure and an error correction procedure, for teaching relations between objects and pictures.
Method Participants were two groups of children with autism, aged between 3 and 7 years. In the context of picture-to-object requesting, one group was taught using an error correction method and the other group with an error prevention method. The measures for each child were accuracy of correspondences between taught picture and object pairs and accuracy of delayed correspondences in learning outcome tests with all combinations of object and picture pairs presented to them throughout the study.
Results The group receiving the error prevention-based teaching made significantly fewer errors during the teaching phases and in their learning outcome test for correspondences between all combinations of pictures and objects.
Conclusions The error prevention teaching procedure would seem to provide a more efficient and ecologically valid method than the error correction procedure for teaching relations between objects and their graphic-based referents. Improvements in the methodology were suggested for providing a stronger basis for comparison between error correction and error prevention teaching methods.
C1 [Carr, D.; Felce, J.] Univ Cardiff, Sch Med, Welsh Ctr Learning Disabil, Cardiff CF14 4YS, S Glam, Wales.
RP Carr, D (reprint author), Univ Cardiff, Sch Med, Welsh Ctr Learning Disabil, Neuadd Merionydd,Hlth Pk, Cardiff CF14 4YS, S Glam, Wales.
EM debcarr60@gmail.com
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NR 35
TC 2
Z9 2
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD APR
PY 2008
VL 52
BP 309
EP 317
DI 10.1111/j.1365-2788.2007.01021.x
PN 4
PG 9
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 274CO
UT WOS:000253979600004
PM 18339093
ER
PT J
AU de Vries, PJ
Watson, P
AF de Vries, P. J.
Watson, P.
TI Attention deficits in tuberous sclerosis complex (TSC): rethinking the
pathways to the endstate
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Article
DE cognition; developmental trajectory; neuroconstructivist;
neuropsychology
ID DEVELOPMENTAL DISORDERS; BRAIN; CHILDREN; MEMORY; NEUROPSYCHOLOGY;
AUTISM
AB Background Tuberous sclerosis complex (TSC) is a genetic disorder associated with a range of neurocognitive manifestations, including neuropsychological attention deficits most notably in dual tasking/divided attention. These dual-task deficits have so far been interpreted as evidence of a vulnerable 'cognitive module' in TSC. Here, we suggest that this interpretation represents an 'adult neuropsychological' perspective, and argue that a developmental approach would be more appropriate to examine attention deficits in TSC.
Method We examined the pathway to 'endstate' dual-task deficits in twenty 6-16 year olds with TSC utilising the Test of Everyday Attention for Children (TEA-Ch). We predicted that the pattern of attentional deficits in TSC would support a 'conditional' model where the establishment of a later-maturing skill was dependent on the functional maturation of an earlier expected skill.
Results Attentional profiles showed statistical support for a conditional model. Only one child showed a deterministic pattern while one showed a hybrid pattern, attributed to the admixture of a surgically acquired lesion and a neurodevelopmental disorder.
Conclusions This preliminary study suggests that the developmental cascade in TSC may be arrested at various stages of neuropsychological development, thus leading to different developmental trajectories towards similar 'endstate' profiles.
C1 [de Vries, P. J.] Univ Cambridge, Dev Psychiat Sect, Cambridge CB2 2AH, England.
[de Vries, P. J.] Cambridgeshire & Peterborough Mental Hlth Partner, Neurodev Serv NDS, Peterborough, Cambs, England.
[Watson, P.] Univ Cambridge, MRC, Cognit & Brain Sci Unit, Cambridge, England.
RP de Vries, PJ (reprint author), Univ Cambridge, Dev Psychiat Sect, Douglas House,18B Trumpington Rd, Cambridge CB2 2AH, England.
EM pd215@cam.ac.uk
CR Agresti A., 1990, CATEGORICAL DATA ANA
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NR 37
TC 1
Z9 2
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD APR
PY 2008
VL 52
BP 348
EP 357
DI 10.1111/j.1365-2788.2007.01030.x
PN 4
PG 10
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 274CO
UT WOS:000253979600008
PM 18179508
ER
PT J
AU Zahir, FR
Baross, A
Delaney, AD
Eydoux, P
Fernandes, ND
Pugh, T
Marra, MA
Friedman, JM
AF Zahir, F. R.
Baross, A.
Delaney, A. D.
Eydoux, P.
Fernandes, N. D.
Pugh, T.
Marra, M. A.
Friedman, J. M.
TI A patient with vertebral, cognitive and behavioural abnormalities and a
de novo deletion of NRXN1 alpha
SO JOURNAL OF MEDICAL GENETICS
LA English
DT Article
ID NEUREXIN I-ALPHA; SCREENING QUESTIONNAIRE; SYNAPSE FORMATION;
BETA-NEUREXINS; CA2+ CHANNELS; CELL-ADHESION; AUTISM; DIFFERENTIATION;
RECEPTOR; GENES
AB The authors report a patient with mild mental retardation, autistic features, multiple vertebral malformations, and an unusual facial appearance who carries a de novo submicroscopic deletion of chromosome 2p16.3. The patient's deletion is similar to 320 kb in size and includes only the part of the NRXN1 gene that codes for the neurexin1 alpha promoter and initial coding exons. The more downstream neurexin1 beta promoter and the region surrounding it are intact. Neurexin1 beta has been associated with autism in several recent studies, but this is the first reported patient with loss of only neurexin1 alpha and not of neurexin1 beta. These findings suggest that neurexin1 alpha function in correct dosage is necessary for normal neurological development.
C1 [Zahir, F. R.; Marra, M. A.; Friedman, J. M.] Univ British Columbia, Womens & Childrens Hosp, Med Genet Res Unit, Vancouver, BC V6H 3N1, Canada.
[Baross, A.] Genome British Columbia, Vancouver, BC, Canada.
[Delaney, A. D.; Pugh, T.; Marra, M. A.] British Columbia Canc Agcy, Genome Sci Ctr, Vancouver, BC V5Z 4E6, Canada.
[Eydoux, P.] Childrens & Womens Hlth Ctr British Columbia, Dept Pathol & Lab Med, Vancouver, BC, Canada.
[Fernandes, N. D.] Univ British Columbia, Fac Med, Vancouver, BC V5Z 1M9, Canada.
RP Zahir, FR (reprint author), Univ British Columbia, Womens & Childrens Hosp, Med Genet Res Unit, Box 153,4500 Oak St, Vancouver, BC V6H 3N1, Canada.
EM farahz@interchange.ubc.ca
RI Marra, Marco/B-5987-2008; Tang, Macy/B-9798-2014
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NR 30
TC 65
Z9 67
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0022-2593
J9 J MED GENET
JI J. Med. Genet.
PD APR
PY 2008
VL 45
IS 4
BP 239
EP 243
DI 10.1136/jmg.2007.054437
PG 5
WC Genetics & Heredity
SC Genetics & Heredity
GA 282JB
UT WOS:000254562600008
PM 18057082
ER
PT J
AU Ferguson, HJ
Sanford, AJ
AF Ferguson, Heather J.
Sanford, Anthony J.
TI Anomalies in real and counterfactual worlds: An eye-movement
investigation
SO JOURNAL OF MEMORY AND LANGUAGE
LA English
DT Article
DE counterfactual reasoning; eye-movements; discourse processing; anomalies
ID BRAIN POTENTIALS; DISCOURSE ROLES; CONDITIONALS; SENTENCES; KNOWLEDGE;
CONTEXT; NARRATIVES; RETRIEVAL; AUTISM; MEMORY
AB Counterfactual reasoning is valid reasoning arising from premises that are true in a hypothetical model, but false in actuality. Investigations of counterfactuals have concentrated on reasoning and production, but psycholinguistic research has been more limited. We report three eye-movement studies investigating the comprehension of counterfactual information. Prior context depicted a counterfactual world (CW), or real world (RW), while a second sentence was manipulated to create RW anomalous continuations, where events included a violation of RW knowledge, and RW congruent continuations, where the events described were congruent with RW knowledge. Results showed that RW violations can be 'neutralised' within an appropriate pre-specified CW context, and RW congruent items can lead to the experience of an anomaly following an inconsistent CW context. Importantly, there was also evidence in all three studies for early processing difficulty with RW violations regardless of prior context, indicating that a proposition is rapidly evaluated against real-world knowledge, just prior to the accommodation of a proposition into a counterfactual world representation. We discuss the results in terms of a variety of accounts of the nature of counterfactual worlds. (C) 2007 Elsevier Inc. All rights reserved.
C1 [Ferguson, Heather J.; Sanford, Anthony J.] Univ Glasgow, Dept Psychol, Glasgow G12 8QQ, Lanark, Scotland.
RP Ferguson, HJ (reprint author), Univ Glasgow, Dept Psychol, Glasgow G12 8QQ, Lanark, Scotland.
EM h.ferguson@psy.gla.ac.uk
RI Ferguson, Heather/D-4308-2014
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VONFINTEL K, 2001, KEN HALE LIFE LANGUA
NR 60
TC 38
Z9 40
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0749-596X
J9 J MEM LANG
JI J. Mem. Lang.
PD APR
PY 2008
VL 58
IS 3
BP 609
EP 626
DI 10.1016/j.jml.2007.06.007
PG 18
WC Linguistics; Psychology; Psychology, Experimental
SC Linguistics; Psychology
GA 288SJ
UT WOS:000255005800001
ER
PT J
AU Lian, WB
Ying, SHK
Tean, SCH
Lin, DCK
Lian, YC
Yun, HL
AF Lian, Wee Bin
Ying, Selina Ho Kah
Tean, Sylvia Choo Henn
Lin, Daisy Chan Kwai
Lian, Yeo Cheo
Yun, Ho Lai
TI Pre-school teachers' knowledge, attitudes and practices on childhood
developmental and behavioural disorders in Singapore
SO JOURNAL OF PAEDIATRICS AND CHILD HEALTH
LA English
DT Article
DE child developmental disorder; teacher
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; DOUBLE-BLIND CROSSOVER;
RUBELLA VACCINE; FOOD COLORINGS; AUTISM; CHILDREN; MEASLES; MUMPS;
HYPERACTIVITY; PREVALENCE
AB Demands for diagnostic and intervention services in childhood developmental and behavioural disorders (CDABD) have increased in Singapore. With earlier enrolment of some 50 000 children in pre-schools, early childhood educators must be well-versed in normal development (ND) and CDABD, to help detect children with potential difficulties and refer for early diagnosis and intervention.
Knowledge, attitudes and practices in ND and CDABD were evaluated among 503 pre-school teachers, most aged 30-44 years. With a median pre-school experience of 6.0 (0.1, 40) years, most had received formal training in early childhood but not special-needs (SN) education.
A pass rate in knowledge (>= 50% total-score) was achieved in 56%, with the overall median total-score of 50 (0, 87)%. In specific blocks on ND, autistic spectrum disorder and attention deficit/hyperactive disorder, pass-rate was achieved in 66%, 68% and 32%, with median block-scores of 56 (0, 100)%, 50 (0, 100)%, 40 (0, 100)% respectively. Results on attitudes and perceptions revealed that most supported mainstream integration and aides in the classroom, agreeing that both the government and parents should pay for such support services. While most felt unequipped, further training interested them, with > 90% wanting to and feeling that they could make a difference for these children.
This study demonstrated educational deficits in CDABD among our pre-school teachers. Yet, most care and want to improve their skills to aid integration and improve SN education, calling for more training and resource support. Necessary changes in policy and resource allocation should occur to allow better-integrated adults of tomorrow.
C1 [Lian, Wee Bin; Ying, Selina Ho Kah; Tean, Sylvia Choo Henn; Lin, Daisy Chan Kwai; Lian, Yeo Cheo; Yun, Ho Lai] Singapore Gen Hosp, Dept Neonatal & Dev Med, Singapore 169608, Singapore.
[Lian, Wee Bin; Ying, Selina Ho Kah; Tean, Sylvia Choo Henn; Lin, Daisy Chan Kwai; Lian, Yeo Cheo; Yun, Ho Lai] KK Womens & Childrens Hosp, Child Dev Unit, Singapore, Singapore.
RP Lian, WB (reprint author), Singapore Gen Hosp, Dept Neonatal & Dev Med, Outram Rd, Singapore 169608, Singapore.
EM lian.wee.bin@sgh.com.sg
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NR 45
TC 4
Z9 4
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1034-4810
J9 J PAEDIATR CHILD H
JI J. Paediatr. Child Health
PD APR
PY 2008
VL 44
IS 4
BP 187
EP 194
DI 10.1111/j.1440-1754.2007.01231.x
PG 8
WC Pediatrics
SC Pediatrics
GA 281MG
UT WOS:000254501100007
PM 17927728
ER
PT J
AU Carr, EG
Ladd, MV
Schulte, CF
AF Carr, Edward G.
Ladd, Mara V.
Schulte, Christine F.
TI Validation of the Contextual Assessment Inventory for Problem Behavior
SO JOURNAL OF POSITIVE BEHAVIOR INTERVENTIONS
LA English
DT Article
DE autism; problem behavior; functional assessment; setting events;
antecedent control
ID DEVELOPMENTAL-DISABILITIES; FUNCTIONAL ASSESSMENT; SELF-INJURY;
INDIVIDUALS; STAFF
AB Problem behavior is a major barrier to successful community integration for people with developmental disabilities. Recently, there has been increased interest in identifying contextual factors involving setting events and discriminative stimuli that impact the display of problem behavior. The authors previously developed the Con textual Assessment Inventory and evaluated it for efficiency, comprehensiveness, comprehensibility, and reliability. This study further evaluated this inventory with respect to convergent and predictive validity. Convergent validity was examined for 17 participants with developmental disabilities through a review of community residence log entries that included a record of the contextual events associated with each episode of problem behavior. Predictive validity was evaluated for a subset of 5 participants through direct observation-of contextual events. Results indicated that-the inventory had both convergent and predictive validity. Implications for extending contextual assessment and using,such-information to develop intervention strategies are explored.
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[Ladd, Mara V.] Ben May Inst, Randolph, MA USA.
[Schulte, Christine F.] Dev Disabil Inst, Smithtown, NY USA.
RP Carr, EG (reprint author), SUNY Stony Brook, Dept Psychol, Stony Brook, NY 11794 USA.
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NR 43
TC 12
Z9 12
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1098-3007
J9 J POSIT BEHAV INTERV
JI J. Posit. Behav. Interv.
PD APR
PY 2008
VL 10
IS 2
BP 91
EP 104
DI 10.1177/1098300707312543
PG 14
WC Psychology, Clinical; Education, Special
SC Psychology; Education & Educational Research
GA 390MQ
UT WOS:000262168800003
ER
PT J
AU Rawlings, D
Locarnini, A
AF Rawlings, David
Locarnini, Ann
TI Dimensional schizotypy, autism, and unusual word associations in artists
and scientists
SO JOURNAL OF RESEARCH IN PERSONALITY
LA English
DT Article
DE creativity; schizotypy; autism spectrum; word association; hypomania
ID SPECTRUM QUOTIENT AQ; CREATIVITY; PERSONALITY; PSYCHOTICISM; TRAITS
AB Studies from a range of perspectives provide evidence for a relationship between creativity and the tendency to mental illness. The present study further examined this issue by administering questionnaires measuring the minor features of psychosis and autism to 31 professional "artists" (visual artists and musicians) and 28 professional "scientists" (biological scientists and physical scientists/mathematicians). The Oxford-Liverpool Inventory of Feelings and Experiences (O-LIFE), the Hypomanic Personality Scale, and the Autism-Spectrum Quotient (AQ), were administered, in addition to a shortened form of the Kent-Rosanoff Word Association Scale. The results provided strong support for the connection of artistic creativity to positive schizotypy and hypomania and the tendency to make unusual word associations, and somewhat weaker support for the connection of scientific creativity to certain components of the autism spectrum. (c) 2007 Elsevier Inc. All rights reserved.
C1 [Rawlings, David; Locarnini, Ann] Univ Melbourne, Dept Psychol, Melbourne, Vic 3010, Australia.
RP Rawlings, D (reprint author), Univ Melbourne, Dept Psychol, Melbourne, Vic 3010, Australia.
EM rawlings@unimelb.edu.au
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NR 18
TC 29
Z9 29
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0092-6566
J9 J RES PERS
JI J. Res. Pers.
PD APR
PY 2008
VL 42
IS 2
BP 465
EP 471
DI 10.1016/j.jrp.2007.06.005
PG 7
WC Psychology, Social
SC Psychology
GA 300RT
UT WOS:000255843600012
ER
PT J
AU Klin, A
AF Klin, Ami
TI In the eye of the beholden: Tracking developmental psychopathology
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Editorial Material
ID CONTACT; AUTISM; GAZE
C1 Yale Child Study Ctr, New Haven, CT 06520 USA.
RP Klin, A (reprint author), Yale Child Study Ctr, 230 S Frontage Rd, New Haven, CT 06520 USA.
EM Ami.Klin@yale.edu
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NR 14
TC 2
Z9 2
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD APR
PY 2008
VL 47
IS 4
BP 362
EP 363
DI 10.1097/CHI.0b013e3181648dd1
PG 2
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 278EF
UT WOS:000254266100002
PM 18356702
ER
PT J
AU De Jong, MC
van Engeland, H
Kemner, C
AF De Jong, Maartje Cathelijne
van Engeland, Herman
Kemner, Chantal
TI Attentional effects of gaze shifts are influenced by emotion and spatial
frequency, but not in autism
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Article
DE autism; gaze; emotion; spatial frequency; event-related potentials
ID PERVASIVE DEVELOPMENTAL DISORDER; HIGH-FUNCTIONING CHILDREN; EYE GAZE;
FACIAL EXPRESSION; FACE PERCEPTION; FEARFUL FACES; INDIVIDUALS;
ADOLESCENTS; DIRECTION; FIXATION
AB Objective: Impaired gaze following is an important hallmark of autism spectrum disorders (ASDs) in clinical settings. Yet, ASD subjects perform normally on laboratory tasks involving gaze shifts. We investigated this contradiction, hypothesizing that impaired gaze following in ASDs is not related to basic impairments in attention orienting but to impaired emotion perception and abnormal processing of spatial frequencies (i.e., local and global information). Method: We tested 30 high-functioning, school-age children with ASDs and 30 age- and IQ-matched controls on a task involving gaze shifts,that cue the location of targets. The cueing faces differed in emotionality and were filtered for different spatial frequencies. We recorded behavioral responses (reaction times) and brain responses (event-related potentials). Results: ASD subjects performed normally when neutral faces were used. However, emotional faces elicited modified face and gaze cue processing in control subjects, but not in the ASD subjects. Furthermore, the control group was biased toward the use of low spatial frequencies (global information) to process gaze cues, whereas the ASD group was biased toward the use of high spatial frequencies (local information). Conclusions: We conclude that impaired gaze following in ASDs is related to impaired emotion processing. Moreover, ASD subjects show an abnormal reliance on local information to process gaze cues'.
C1 [De Jong, Maartje Cathelijne; van Engeland, Herman; Kemner, Chantal] Univ Med Ctr Utrecht, Dept Child & Adolscent Psychiat, Rudolf Magnus Inst Neurosci, Utrecht, Netherlands.
RP De Jong, MC (reprint author), POB 85500, NL-3508 GA Utrecht, Netherlands.
EM M.C.delong1@students.uu.nl
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TC 15
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PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
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J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD APR
PY 2008
VL 47
IS 4
BP 443
EP 454
DI 10.1097/CHI.0b013e31816429a6
PG 12
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 278EF
UT WOS:000254266100013
PM 18356706
ER
PT J
AU McNeilly, LG
Sheppard, JJ
AF McNeilly, Lemmietta G.
Sheppard, Justine J.
TI Prologue - Managing dysphagia in the schools
SO LANGUAGE SPEECH AND HEARING SERVICES IN SCHOOLS
LA English
DT Article
DE pediatric; dysphagia; swallowing and feeding disorders; school service
delivery; culturally appropriate intervention; legislation; ethical
considerations
AB Purpose: This prologue introduces the clinical forum, "Managing Dysphagia in the Schools," and informs the reader of the scope of the included articles.
Method: The contributing authors are introduced and a brief summary is provided for each of the 10 articles that make up the forum.
Conclusion: The articles address a broad range of issues, with specific reference to children with dysphagia in school settings. There are comprehensive reviews of legislation, ethical issues, cultural issues, motor learning, medically fragile conditions, severe neuromuscular impairments, and autism spectrum disorder. In addition, there are systematic observations and research that address a model public school dysphagia program, school-based speech-language pathologists' perceptions of competency, family perceptions of school programs, and a program for implementing transition from tube to oral feeding.
C1 [McNeilly, Lemmietta G.] Amer Speech Language Hearing Assoc, Rockville, MD 20850 USA.
[Sheppard, Justine J.] Columbia Univ, Teachers Coll, New York, NY 10027 USA.
RP McNeilly, LG (reprint author), Amer Speech Language Hearing Assoc, 2200 Res Blvd 217, Rockville, MD 20850 USA.
EM Lmcneilly@asha.org
CR Angell ME, 2008, LANG SPEECH HEAR SER, V39, P214, DOI 10.1044/0161-1461(2008/021)
Davis-McFarland E, 2008, LANG SPEECH HEAR SER, V39, P199, DOI 10.1044/0161-1461(2008/020)
Homer EA, 2008, LANG SPEECH HEAR SER, V39, P177, DOI 10.1044/0161-1461(2008/018)
Huffman NP, 2008, LANG SPEECH HEAR SER, V39, P167, DOI 10.1044/0161-1461(2008/017)
Lefton-Greif MA, 2008, LANG SPEECH HEAR SER, V39, P237, DOI 10.1044/0161-1461(2008/023)
LOGEMANN JA, 2000, LANG SPEECH HEAR SER, V31, P26
McKirdy LS, 2008, LANG SPEECH HEAR SER, V39, P249, DOI 10.1044/0161-1461(2008/024)
O'Donoghue CR, 2008, LANG SPEECH HEAR SER, V39, P192, DOI 10.1044/0161-1461(2008/019)
Power-DeFur L, 2008, LANG SPEECH HEAR SER, V39, P160, DOI 10.1044/0161-1461(2008/016)
Sheppard JJ, 2008, LANG SPEECH HEAR SER, V39, P227, DOI 10.1044/0161-1461(2008/022)
Twachtman-Reilly J, 2008, LANG SPEECH HEAR SER, V39, P261, DOI 10.1044/0161-1461(2008/025)
NR 11
TC 0
Z9 0
PU AMER SPEECH-LANGUAGE-HEARING ASSOC
PI ROCKVILLE
PA 10801 ROCKVILLE PIKE, ROCKVILLE, MD 20852-3279 USA
SN 0161-1461
J9 LANG SPEECH HEAR SER
JI Lang. Speech Hear. Serv. Sch.
PD APR
PY 2008
VL 39
IS 2
BP 158
EP 159
DI 10.1044/0161-1461(2008/015)
PG 2
WC Linguistics; Rehabilitation
SC Linguistics; Rehabilitation
GA 286JV
UT WOS:000254843100003
PM 18420518
ER
PT J
AU Twachtman-Reilly, J
Amaral, SC
Zebrowski, PP
AF Twachtman-Reilly, Jennifer
Amaral, Sheryl C.
Zebrowski, Patrecia P.
TI Addressing feeding disorders in children on the autism spectrum in
school-based settings: Physiological and behavioral issues
SO LANGUAGE SPEECH AND HEARING SERVICES IN SCHOOLS
LA English
DT Article
DE autism; feeding; assessment; intervention
ID PERVASIVE DEVELOPMENTAL DISORDERS; CONSUMPTION; SYMPTOMS; THERAPY;
DISABILITIES; ACCEPTANCE; PATTERNS; IMPROVE
AB Purpose: The purposes of this article are to define the nature of feeding difficulties in children with autism spectrum disorder (ASD), identify important components of the assessment and treatment of feeding disorders specific to this population, and delineate specific therapeutic techniques designed to improve assessment and treatment within the school setting.
Method: Literature review and case example are used to define the predominant nature of the feeding difficulties that are experienced by some children on the autism spectrum. Characteristics of this complex disorder that can have an impact on feeding skill and behavior are also identified. These factors, are then integrated to create assessment and intervention techniques that can be used in conjunction with traditional feeding approaches to facilitate improvements in eating in this unique population.
Implications: The complex nature of ASD and its many influences on feeding skills and behavior create the need for modification to both assessment and treatment approaches. Additional research is needed to create therapeutic protocols that can be used by school-based speech-language pathologists to effectively assess and treat feeding difficulties that are commonly encountered in children with ASD.
C1 [Twachtman-Reilly, Jennifer] Autism & Dev Disabilities Consultat Ctr LLC, Higganum, CT 06441 USA.
[Amaral, Sheryl C.] Cumberland Sch Dept, Cumberland, RI USA.
[Zebrowski, Patrecia P.] Sargent Rehabil Ctr, Warwick, RI USA.
RP Twachtman-Reilly, J (reprint author), Autism & Dev Disabilities Consultat Ctr LLC, POB 709, Higganum, CT 06441 USA.
EM jtreillyslp@sbcglobal.net
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NR 69
TC 22
Z9 22
PU AMER SPEECH-LANGUAGE-HEARING ASSOC
PI ROCKVILLE
PA 10801 ROCKVILLE PIKE, ROCKVILLE, MD 20852-3279 USA
SN 0161-1461
J9 LANG SPEECH HEAR SER
JI Lang. Speech Hear. Serv. Sch.
PD APR
PY 2008
VL 39
IS 2
BP 261
EP 272
DI 10.1044/0161-1461(2008/025)
PG 12
WC Linguistics; Rehabilitation
SC Linguistics; Rehabilitation
GA 286JV
UT WOS:000254843100013
PM 18420528
ER
PT J
AU Lepagnol-Bestel, AM
Maussion, G
Boda, B
Cardona, A
Iwayama, Y
Delezoide, AL
Moalic, JM
Muller, D
Dean, B
Yoshikawa, T
Gorwood, P
Buxbaum, JD
Ramoz, N
Simonneau, M
AF Lepagnol-Bestel, A-M
Maussion, G.
Boda, B.
Cardona, A.
Iwayama, Y.
Delezoide, A-L
Moalic, J-M
Muller, D.
Dean, B.
Yoshikawa, T.
Gorwood, P.
Buxbaum, J. D.
Ramoz, N.
Simonneau, M.
TI SLC25A12 expression is associated with neurite outgrowth and is
upregulated in the prefrontal cortex of autistic subjects
SO MOLECULAR PSYCHIATRY
LA English
DT Article
DE SLC25A12; human embryo; fetal brain; in situ hybridization; expression
pattern; cortical parcellation; gene overexpression; dendrite length
ID CORTICAL DENDRITE DEVELOPMENT; SPECTRUM DISORDERS; BIPOLAR DISORDER;
HIPPOCAMPAL REGION; CEREBRAL-CORTEX; FRONTAL-CORTEX; RHESUS-MONKEY;
GENE; BRAIN; NEURONS
AB Autism is a neurodevelopmental disorder with a strong genetic component, probably involving several genes. Genome screens have provided evidence of linkage to chromosome 2q31-q33, which includes the SLC25A12 gene. Association between autism and single-nucleotide polymorphisms in SLC25A12 has been reported in various studies. SLC25A12 encodes the mitochondrial aspartate/glutamate carrier functionally important in neurons with high-metabolic activity. Neuropathological findings and functional abnormalities in autism have been reported for Brodmann's area (BA) 46 and the cerebellum. We found that SLC25A12 was expressed more strongly in the post-mortem brain tissues of autistic subjects than in those of controls, in the BA46 prefrontal cortex but not in cerebellar granule cells. SLC25A12 expression was not modified in brain subregions of bipolar and schizophrenic patients. SLC25A12 was expressed in developing human neuronal tissues, including neocortical regions containing excitatory neurons and neocortical progenitors and the ganglionic eminences that generate neocortical inhibitory interneurons. At mid-gestation, when gyri and sulci start to develop, SLC25A12 molecular gradients were identified in the lateral prefrontal and ventral temporal cortex. These fetal structures generate regions with abnormal activity in autism, including the dorsolateral prefrontal cortex (BA46), the pars opercularis of the inferior frontal cortex and the fusiform gyrus. SLC25A12 overexpression or silencing in mouse embryonic cortical neurons also modified dendrite length and the mobility of dendritic mitochondria. Our findings suggest that SLC25A12 overexpression may be involved in the pathophysiology of autism, modifying neuronal networks in specific subregions, such as the dorsolateral prefrontal cortex and fusiform gyrus, at both pre-and postnatal stages.
RP Simonneau, M (reprint author), INSERM, U675, IFR2, Fac Med Xavier Bichat, 16 Rue Henri Huchard, F-75018 Paris, France.
EM simonneaumi@wanadoo.fr
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NR 62
TC 33
Z9 39
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1359-4184
J9 MOL PSYCHIATR
JI Mol. Psychiatr.
PD APR
PY 2008
VL 13
IS 4
BP 385
EP 397
DI 10.1038/sj.mp.4002120
PG 13
WC Biochemistry & Molecular Biology; Neurosciences; Psychiatry
SC Biochemistry & Molecular Biology; Neurosciences & Neurology; Psychiatry
GA 276EH
UT WOS:000254123600007
PM 18180767
ER
PT J
AU Opar, A
AF Opar, Alisa
TI Search for potential autism treatments turns to 'trust hormone'
SO NATURE MEDICINE
LA English
DT News Item
ID RECEPTOR GENE OXTR; ASSOCIATION; CHILDREN
AB For all of the attention that autism has received, the condition still lacks pharmacological treatments. Faced with this frustrating reality, some researchers have begun exploring whether oxytocin, the so could ease symptoms of the disorder, such as repetitive behaviors and difficulties with social interactions. Researchers are conducting trials using nasal sprays of the drug. Another drug in the pipeline could potentially treat the social deficits associated with autism by reducing levels of a protein called metabotropic glutamate receptor 5.
CR Jacob S, 2007, NEUROSCI LETT, V417, P6, DOI 10.1016/j.neulet.2007.02.001
Modahl C, 1998, BIOL PSYCHIAT, V43, P270, DOI 10.1016/S0006-3223(97)00439-3
Wu SP, 2005, BIOL PSYCHIAT, V58, P74, DOI 10.1016/j.biopsych.2005.03.013
NR 3
TC 9
Z9 9
PU NATURE PUBLISHING GROUP
PI NEW YORK
PA 75 VARICK STREET, 9TH FLOOR, NEW YORK, NY 10013-1917 USA
SN 1078-8956
J9 NAT MED
JI Nat. Med.
PD APR
PY 2008
VL 14
IS 4
BP 353
EP 353
DI 10.1038/nm0408-353
PG 1
WC Biochemistry & Molecular Biology; Cell Biology; Medicine, Research &
Experimental
SC Biochemistry & Molecular Biology; Cell Biology; Research & Experimental
Medicine
GA 283ZD
UT WOS:000254674100002
PM 18391923
ER
PT J
AU Geier, DA
Mumper, E
Gladfelter, B
Coleman, L
Geier, MR
AF Geier, David A.
Mumper, Elizabeth
Gladfelter, Bambi
Coleman, Lisa
Geier, Mark R.
TI Neurodevelopmental disorders, maternal Rh-negativity, and Rho(D) immune
globulins: A multi-center assessment
SO NEUROENDOCRINOLOGY LETTERS
LA English
DT Article
DE developmental delay; ethylmercury; merthiolate; rhogam; thiomersal
ID METHYL MERCURY EXPOSURE; AUTISTIC DISORDERS; METHYLMERCURY EXPOSURE;
ANTENATAL PROPHYLAXIS; SPECTRUM DISORDERS; BLOOD MERCURY; CELL-DEATH;
IN-VITRO; THIMEROSAL; CHILDREN
AB BACKGROUND: Many formulations of Thimerosal (49.55% mercury by weight)containing Rho(D) immune globulins (TCRs) were routinely administered to Rh-negative mothers in the US prior to 2002.
OBJECTIVES: It was hypothesized: (1) if prenatal Rho(D)-immune globulin preparation exposure was a risk factor for neurodevelopmental disorders (NDs) then more children with NDs would have Rh-negative mothers compared to controls; and (2) if Thimerosal in the Rho(D)-immune globulin preparations was the ingredient associated with NDs, following the removal of Thimerosal from all manufactured Rho(D)-immune globulin preparations from 2002 in the US the frequency of maternal Rh-negativity among children with NDs should be similar to control populations.
METHODS: Maternal Rh-negativity was assessed at two sites (Clinic A-Lynchburg, VA; Clinic B-Rockville and Baltimore, MD) among 298 Caucasian children with NDs and known Rh-status. As controls, maternal Rh-negativity frequency was determined from 124 Caucasian children (born 1987-2001) without NDs at Clinic A, and the Rh-negativity frequency was determined from 1,021 Caucasian pregnant mothers that presented for prenatal genetic care at Clinic B (1980-1989). Additionally, 22 Caucasian patients with NDs born from 2002 onwards (Clinics A and B) were assessed for maternal Rh-negativity.
RESULTS: There were significant and comparable increases in maternal Rh-negativity among children with NDs (Clinic: A=24.2%), autism spectrum disorders (Clinic: A=28.3%, B=25.3%), and attention-deficit- disorder/attention-deficit hyperactivity- disorder (Clinic: A=26.3%) observed at both clinics in comparison to both control groups (Clinic: A=12.1%, B=13.9%) employed. Children with NDs born post-2001 had a maternal Rh-negativity frequency (13.6%) similar to controls.
CONCLUSION: This study associates TCR exposure with some NDs in children.
C1 [Geier, David A.] Inst Chron Illnesses Inc, Silver Spring, MD 20905 USA.
[Mumper, Elizabeth; Gladfelter, Bambi; Coleman, Lisa] Advocates Children Pediat Ltd, Lynchburg, VA 24501 USA.
RP Geier, MR (reprint author), 14 Redgate Ct, Silver Spring, MD 20905 USA.
EM mgeier@comcast.net
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NR 78
TC 15
Z9 16
PU MAGHIRA & MAAS PUBLICATIONS
PI STOCKHOLM
PA PO BOX 26132, S-100 41 STOCKHOLM, SWEDEN
SN 0172-780X
J9 NEUROENDOCRINOL LETT
JI Neuroendocrinol. Lett.
PD APR
PY 2008
VL 29
IS 2
BP 272
EP 280
PG 9
WC Endocrinology & Metabolism; Neurosciences
SC Endocrinology & Metabolism; Neurosciences & Neurology
GA 295QW
UT WOS:000255490000014
PM 18404135
ER
PT J
AU [Anonymous]
AF [Anonymous]
TI Cortical misfolding in autism
SO NEUROSCIENTIST
LA English
DT Editorial Material
CR Nordahl CW, 2007, J NEUROSCI, V27, P11725, DOI 10.1523/JNEUROSCI.0777-07.2007
NR 1
TC 0
Z9 0
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1073-8584
EI 1089-4098
J9 NEUROSCIENTIST
JI Neuroscientist
PD APR
PY 2008
VL 14
IS 2
BP 138
EP 138
PG 1
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 273PL
UT WOS:000253944100008
ER
PT J
AU Harris, KM
Mahone, EM
Singer, HS
AF Harris, Kendra M.
Mahone, E. Mark
Singer, Harvey S.
TI Nonautistic motor stereotypies: Clinical features and longitudinal
follow-up
SO PEDIATRIC NEUROLOGY
LA English
DT Article
ID AMPHETAMINE MICROINJECTION; DEVELOPMENTAL DISORDERS; REPETITIVE
BEHAVIORS; TOURETTE SYNDROME; NORMAL-CHILDREN; YOUNG-CHILDREN; STRIATUM;
MOVEMENTS; DOPAMINE; AUTISM
AB To characterize further the clinical features and long-term outcomes among children with motor stereotypies who do not manifest mental retardation or pervasive developmental disorders, a review of clinical records and semistructured telephone interviews were undertaken. The identified clinical cohort consisted of 100 typically developing children with motor stereotypies. The mean length of follow-up was 6.8 +/- 4.6 years. At most recent follow-up, movements had continued in 94% of the sample (62% for >5 years). Only six children reported complete cessation of movements, with four (3 of 4 with head nodding) doing so >1 year after their initial diagnosis. Thus the course of motor stereotypies, especially in children with arm/hand movements, appears chronic. Nearly half the children in this cohort exhibit other comorbidities, including attention-deficit-hyperactivity disorder (30%), tics (18%), and obsessive-compulsive behaviors/obsessive compulsive disorder (10%). Twenty-five percent of children with motor stereotypies reported positive family histories of motor stereotypies, suggesting an underlying genetic abnormality. Finally, evidence is emerging that the clinical course of children who exhibit head nodding may differ from those whose motor stereotypy predominantly involves the hands and arms. (C) 2008 by Elsevier Inc. All rights reserved.
C1 [Singer, Harvey S.] Johns Hopkins Univ Hosp, Sch Med, Dept Pediat, Div Pediat Neurol, Baltimore, MD 21287 USA.
[Harris, Kendra M.; Singer, Harvey S.] Johns Hopkins Univ Hosp, Sch Med, Dept Neurol, Baltimore, MD 21205 USA.
[Mahone, E. Mark] Kennedy Krieger Inst, Dept Neuropsychol, Baltimore, MD USA.
RP Singer, HS (reprint author), Johns Hopkins Univ Hosp, Sch Med, Dept Pediat, Div Pediat Neurol, David M Rubenstein Child Hlth Bldg 200 N,Wolfe St, Baltimore, MD 21287 USA.
EM hsinger@jhmi.edu
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NR 29
TC 40
Z9 41
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0887-8994
J9 PEDIATR NEUROL
JI Pediatr. Neurol.
PD APR
PY 2008
VL 38
IS 4
BP 267
EP 272
DI 10.1016/j.pediatrneurol.2007.12.008
PG 6
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 283OV
UT WOS:000254646800008
PM 18358406
ER
PT J
AU Limperopoulos, C
Bassan, H
Sullivan, NR
Soul, JS
Robertson, RL
Moore, M
Ringer, SA
Volpe, JJ
du Plessis, AJ
AF Limperopoulos, Catherine
Bassan, Haim
Sullivan, Nancy R.
Soul, Janet S.
Robertson, Richard L., Jr.
Moore, Marianne
Ringer, Steven A.
Volpe, Joseph J.
du Plessis, Adre J.
TI Positive screening for autism in ex-preterm infants: Prevalence and risk
factors
SO PEDIATRICS
LA English
DT Article
DE autism; prematurity; MRI; risk factors; outcome
ID LOW-BIRTH-WEIGHT; PERVASIVE DEVELOPMENTAL DISORDERS; SCHOOL-AGED
CHILDREN; SPECTRUM DISORDERS; PERINATAL COMPLICATIONS; NEWBORN
ENCEPHALOPATHY; PSYCHIATRIC OUTCOMES; PREMATURE-INFANTS; YOUNG-CHILDREN;
BRAIN-STEM
AB OBJECTIVE. The survival of very low birth weight infants has increased markedly in recent years. Unfortunately, the prevalence of significant and lifelong motor, cognitive, and behavioral dysfunction has remained a major problem confronting these children. The objective of this study was to perform screening tests for early autistic features in children with a history of very low birth weight and to identify risk factors associated with a positive screening result.
METHODS. We studied 91 ex-preterm infants <= 1500 g at birth. Infants underwent conventional MRI studies at preterm and/or term-adjusted age. We collected pertinent demographic, prenatal, intrapartum, acute postnatal, and short-term outcome data for all infants. Follow-up assessments were performed at a mean age of 21.9 +/- 4.7 months, using the Modified Checklist for Autism in Toddlers, the Vineland Adaptive Behavior Scale, and the Child Behavior Checklist.
RESULTS. Twenty-six percent of ex-preterm infants had a positive result on the autism screening tool. Abnormal scores correlated highly with internalizing behavioral problems on the Child Behavior Checklist and socialization and communication deficits on the Vineland Scales. Lower birth weight, gestational age, male gender, chorioamnionitis, acute intrapartum hemorrhage, illness severity on admission, and abnormal MRI studies were significantly associated with an abnormal autism screening score.
CONCLUSIONS. Early autistic behaviors seem to be an underrecognized feature of very low birth weight infants. The results from this study suggest that early screening for signs of autism may be warranted in this high-risk population followed by definitive autism testing in those with positive screening results.
C1 [Limperopoulos, Catherine] McGill Univ, Sch Phys & Occupat Therapy, Dept Neurol & Neurosurg, Montreal, PQ, Canada.
[Limperopoulos, Catherine] McGill Univ, Dept Pediat, Montreal, PQ H3A 2T5, Canada.
[Limperopoulos, Catherine; Bassan, Haim; Soul, Janet S.; Moore, Marianne; Volpe, Joseph J.; du Plessis, Adre J.] Childrens Hosp, Dept Neurol, Fetal Neonatal Neurol Res Program, Boston, MA 02115 USA.
[Sullivan, Nancy R.] Childrens Hosp, Dev Med Ctr, Boston, MA 02115 USA.
[Robertson, Richard L., Jr.] Childrens Hosp, Dept Radiol, Boston, MA 02115 USA.
Harvard Univ, Sch Med, Boston, MA USA.
[Ringer, Steven A.] Brigham & Womens Hosp, Dept Neonatol, Boston, MA 02115 USA.
RP Limperopoulos, C (reprint author), Montreal Childrens Hosp, Div Pediat Neurol, A-334,2300 Tupper St, Montreal, PQ H3H 1P3, Canada.
EM catherine.limperopoulos@childrens.harvard.edu
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NR 70
TC 113
Z9 115
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD APR
PY 2008
VL 121
IS 4
BP 758
EP 765
DI 10.1542/peds.2007-2158
PG 8
WC Pediatrics
SC Pediatrics
GA 282OJ
UT WOS:000254576800014
PM 18381541
ER
PT J
AU Greenspan, SI
Brazelton, TB
Cordero, J
Solomon, R
Bauman, ML
Robinson, R
Shanker, S
Breinbauer, C
AF Greenspan, Stanley I.
Brazelton, T. Berry
Cordero, Jose
Solomon, Richard
Bauman, Margaret L.
Robinson, Ricki
Shanker, Stuart
Breinbauer, Cecilia
TI Guidelines for early identification, screening, and clinical management
of children with autism spectrum disorders
SO PEDIATRICS
LA English
DT Editorial Material
ID YOUNG-CHILDREN; LANGUAGE INTERVENTION; TRIAL
C1 [Greenspan, Stanley I.] George Washington Univ, Dept Psychiat Behav Sci & Pediat, Washington, DC USA.
[Brazelton, T. Berry] Harvard Univ, Sch Med, Dept Pediat Emeritus, Boston, MA USA.
[Bauman, Margaret L.] Harvard Univ, Sch Med, Dept Neurol, Boston, MA 02115 USA.
[Brazelton, T. Berry] Massachusetts Gen Hosp, Dept Neurol, Boston, MA 02114 USA.
[Brazelton, T. Berry] Massachusetts Gen Hosp, Dept Pediat, Boston, MA 02114 USA.
[Brazelton, T. Berry] Massachusetts Gen Hosp, LADDERS, Boston, MA 02114 USA.
[Cordero, Jose] Univ Puerto Rico, Sch Publ Hlth, San Juan, PR 00936 USA.
[Solomon, Richard] Ann Arbor Ctr Dev & Behav Pediat, Ann Arbor, MI USA.
[Robinson, Ricki] Univ So Calif, Keck Sch Med, Dept Pediat, Los Angeles, CA 90033 USA.
[Shanker, Stuart] York Univ, Dept Philosophy & Psychol, Toronto, ON M3J 2R7, Canada.
[Breinbauer, Cecilia] ICDL Grad Sch, Interdisciplinary Council Dev & Learning Disorder, Kentfield, CA USA.
RP Greenspan, SI (reprint author), 7201 Glenbrook Rd, Bethesda, MD 20814 USA.
EM stanleygreenspan@gmail.com
CR Aldred C, 2004, J CHILD PSYCHOL PSYC, V45, P1420, DOI 10.1111/j1469-7610.2004.00338.x
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NR 23
TC 12
Z9 12
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD APR
PY 2008
VL 121
IS 4
BP 828
EP 830
DI 10.1542/peds.2007-3833
PG 3
WC Pediatrics
SC Pediatrics
GA 282OJ
UT WOS:000254576800020
PM 18381546
ER
PT J
AU Montes, G
Halterman, JS
AF Montes, Guillermo
Halterman, Jill S.
TI Association of childhood autism spectrum disorders and loss of family
income
SO PEDIATRICS
LA English
DT Article
DE autism; household income; pervasive developmental disorder; national
survey; economics; National Household Education Survey; United States
ID CHILDREN; EXPENDITURES; PARENTS
AB BACKGROUND. Parents of children with autism have significant out-of-pocket expenditures related to their child's care. The impact of having a child with autism on household income is not known.
OBJECTIVE. The purpose of this work was to estimate the loss of household income associated with childhood autism using a nationally representative sample.
METHODS. Parents of 11 684 children enrolled in kindergarten to eighth grade were surveyed by the National Household Education Survey-After School Programs and Activities in 2005. An autism spectrum disorder was defined as an affirmative response to the questions, "has a health professional told you that [child] has any of the following disabilities? 1) autism? 2) pervasive developmental disorder or PDD?" There were 131 children with autism spectrum disorder in the sample and 2775 children with other disabilities. We used ordinal logistic regression analyses to estimate the expected income of families of children with autism given their education level and demographic characteristics and compared the expected income with their reported income.
RESULTS. Both having a child with autism spectrum disorder and having a child with other disabilities were associated with decreased odds of living in a higher income household after controlling for parental education, type of family, parental age, location of the household, and minority ethnicity. The average loss of annual income associated with having a child with autism spectrum disorder was $6200 or 14% of their reported income.
CONCLUSION. Childhood autism is associated with a substantial loss of annual household income. This likely places a significant burden on families in the face of additional out-of-pocket expenditures.
C1 [Montes, Guillermo; Halterman, Jill S.] Childrens Inst, Rochester, NY 14607 USA.
[Montes, Guillermo; Halterman, Jill S.] Univ Rochester, Sch Med & Dent, Dept Pediat, Rochester, NY USA.
RP Montes, G (reprint author), Childrens Inst, 271 N Goodman st,Suite D103, Rochester, NY 14607 USA.
EM gmontes@childrensinstitute.net
CR *CDC, 2004, MMWR-MORBID MORTAL W, V55, P481
Chambers J, 2003, TOTAL EXPENDITURES S
CROEN LA, 2006, PEDIATRICS, V118
DeNavas-Walt C., 2006, INCOME POVERTY HLTH
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HAGEDORN M, 2006, NATL HOUSEHOLD ED SU
Halterman JS, 2006, J DEV BEHAV PEDIATR, V26, P375
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Lewis S, 2000, PERS REV, V29, P417, DOI 10.1108/00483480010324797
Liptak GS, 2006, J AUTISM DEV DISORD, V36, P871, DOI 10.1007/s10803-006-0119-9
Micali N, 2004, AUTISM, V8, P21, DOI 10.1177/1362361304040636
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Newacheck PW, 2004, PEDIATRICS, V114, P79, DOI 10.1542/peds.114.1.79
Rosenzweig JM, 2002, SOC WORK, V47, P415
NR 14
TC 26
Z9 26
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD APR
PY 2008
VL 121
IS 4
BP E821
EP E826
DI 10.1542/peds.2007-1594
PG 6
WC Pediatrics
SC Pediatrics
GA 282OJ
UT WOS:000254576800058
PM 18381511
ER
PT J
AU Smith, MJ
Ellenberg, SS
Bell, LM
Rubin, DM
AF Smith, Michael J.
Ellenberg, Susan S.
Bell, Louis M.
Rubin, David M.
TI Media coverage of the measles-mumps-rubella vaccine and autism
controversy and its relationship to MMR immunization rates in the United
States
SO PEDIATRICS
LA English
DT Article
DE media impact; vaccines
ID NEWS MEDIA; CHILDREN; PARENTS; ATTITUDES; PROVIDER; REFUSAL; SCIENCE;
CANCER; RISKS; SEE
AB OBJECTIVE. The purpose of this work was to assess the association between media coverage of the MMR-autism controversy and MMR immunization in the United States.
METHODS. The public-use files of the National Immunization Survey were used to estimate annual MMR coverage from 1995 to 2004. The primary outcome was selective measles-mumps-rubella nonreceipt, that is, those children who received all childhood immunizations except MMR. Media coverage was measured by using LexisNexis, a comprehensive database of national and local news media. Factors associated with MMR nonreceipt were identified by using a logistic regression model.
RESULTS. Selective MMR nonreceipt, occurring in as few as 0.77% of children in the 1995 cohort, rose to 2.1% in the 2000 National Immunization Survey. Children included in the 2000 National Immunization Survey were born when the putative link between MMR and autism surfaced in the medical literature but before any significant media attention occurred. Selective nonreceipt was more prevalent in private practices and unrelated to family characteristics. MMR nonreceipt returned to baseline before sustained media coverage of the MMR-autism story began.
CONCLUSIONS. There was a significant increase in selective MMR nonreceipt that was temporally associated with the publication of the original scientific literature, suggesting a link between MMR and autism, which preceded media coverage of the MMR-autism controversy. This finding suggests a limited influence of mainstream media on MMR immunization in the United States.
C1 [Smith, Michael J.; Bell, Louis M.] Univ Louisville, Sch Med, Div Pediat Infecdt Dis, Louisville, KY 40292 USA.
[Bell, Louis M.; Rubin, David M.] Childrens Hosp Philadelphia, Div Gen Pediat, Louisville, KY 40292 USA.
[Smith, Michael J.; Ellenberg, Susan S.; Rubin, David M.] Univ Louisville, Sch Med, Ctr Clin Epidemiol & Biostat, Louisville, KY 40292 USA.
RP Smith, MJ (reprint author), Univ Louisville, Sch Med, Div Pediat Infecdt Dis, 571 S Floyd St,Suite 321, Louisville, KY 40292 USA.
EM mjsmit22@louisville.edu
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NR 36
TC 31
Z9 31
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD APR
PY 2008
VL 121
IS 4
BP E836
EP E843
DI 10.1542/peds.2007-1760
PG 8
WC Pediatrics
SC Pediatrics
GA 282OJ
UT WOS:000254576800060
PM 18381512
ER
PT J
AU Fazlioglu, Y
Baran, G
AF Fazlioglu, Yesim
Baran, Gulen
TI A sensory integration therapy program on sensory problems for children
with autism
SO PERCEPTUAL AND MOTOR SKILLS
LA English
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDERS; OCCUPATIONAL-THERAPY; MOTOR;
INTERVENTIONS; RESPONSIVITY; TOUCH
AB The study was planned to investigate the effect of a sensory integration therapy program on sensory problems of children with autism. This study was conducted at the Trakya University Training and Research Center for Mentally and Physically Handicapped Children in Turkey. The children were separated into two groups, each comprising 15 children between 7 and 11 years of age with autism, according to DSM-IV criteria. The children in each group were assessed initially on a checklist, Sensory Evaluation Form for Children with Autism, developed to evaluate sensory characteristics of children with autism, and at the end of the study, participants were assessed again on the checklist. Statistically significant differences between groups indicated that the sensory integration therapy program positively affected treated children.
C1 [Fazlioglu, Yesim] Trakya Univ, Egitim Fak, Okuloncesi Egitim ABD, Edirne, Turkey.
[Baran, Gulen] Ankara Univ, TR-06100 Ankara, Turkey.
RP Fazlioglu, Y (reprint author), Trakya Univ, Egitim Fak, Okuloncesi Egitim ABD, Edirne, Turkey.
EM yfazli@hotmail.com
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NR 29
TC 15
Z9 17
PU AMMONS SCIENTIFIC, LTD
PI MISSOULA
PA PO BOX 9229, MISSOULA, MT 59807-9229 USA
SN 0031-5125
J9 PERCEPT MOTOR SKILL
JI Percept. Mot. Skills
PD APR
PY 2008
VL 106
IS 2
BP 415
EP 422
DI 10.2466/PMS.106.2.4t5-422
PG 8
WC Psychology, Experimental
SC Psychology
GA 301HB
UT WOS:000255885900005
PM 18556898
ER
PT J
AU Andrew, J
Cooke, M
Muncer, SJ
AF Andrew, J.
Cooke, M.
Muncer, S. J.
TI The relationship between empathy and Machiavellianism: An alternative to
empathizing-systemizing theory
SO PERSONALITY AND INDIVIDUAL DIFFERENCES
LA English
DT Article
DE empathy; Machiavellianism; confirmatory factor analysis; negative
correlation
ID ASPERGER-SYNDROME; QUOTIENT; POPULATION; AUTISM; ADULTS; SEX
AB The psychometric properties of the suggested short form EQ (Muncer & Ling, 2006) and the Mach IV (Christie & Geis, 1970) are examined. Confirmatory factor analysis Suggests that the proposed three factor structure for empathy and four factor structure for Machiavellianism fit the data better than other conceptions. From the previous research we expected empathy to be significantly higher in females, and also that the gender difference on the emotional reactivity subfactor would be highest. Males were expected to score higher on Machiavellianism. All of these results were found. From the pattern of results it is suggested that,in EQ, Machiavellianism dichotomy makes more sense and has better empirical support than the proposed EQ/SQ distinction of Baron-Cohen. The problems of assuming unidimensionality for the EQ or any scale, based on limited evidence, are discussed. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Andrew, J.; Cooke, M.; Muncer, S. J.] Univ Durham, Dept Psychol, Thornaby TS17 6BH, England.
RP Muncer, SJ (reprint author), Univ Durham, Dept Psychol, Queens Campus, Thornaby TS17 6BH, England.
EM s.j.muncer@durham.ac.uk
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NR 28
TC 21
Z9 24
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0191-8869
J9 PERS INDIV DIFFER
JI Pers. Individ. Differ.
PD APR
PY 2008
VL 44
IS 5
BP 1203
EP 1211
DI 10.1016/j.paid.2007.11.014
PG 9
WC Psychology, Social
SC Psychology
GA 282WO
UT WOS:000254598100015
ER
PT J
AU Fatemi, SH
AF Fatemi, S. H.
TI The hyperglutamatergic hypothesis of autism
SO PROGRESS IN NEURO-PSYCHOPHARMACOLOGY & BIOLOGICAL PSYCHIATRY
LA English
DT Letter
ID CEREBELLAR CORTICES; GLUTAMATE; PARIETAL; BRAIN
C1 Univ Minnesota, Sch Med, Minneapolis, MN 55455 USA.
RP Fatemi, SH (reprint author), Univ Minnesota, Sch Med, Minneapolis, MN 55455 USA.
EM fatem002@umn.edu
CR Fatemi SH, 2002, BIOL PSYCHIAT, V52, P805, DOI 10.1016/S0006-3223(02)01430-0
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YIP, 2007, ACTA NEUROPATHOL, V113, P559, DOI 10.1007/s00401-006-0176-3
NR 6
TC 24
Z9 25
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0278-5846
J9 PROG NEURO-PSYCHOPH
JI Prog. Neuro-Psychopharmacol. Biol. Psychiatry
PD APR 1
PY 2008
VL 32
IS 3
BP 911
EP 911
DI 10.1016/j.pnpbp.2007.11.004
PG 1
WC Clinical Neurology; Neurosciences; Pharmacology & Pharmacy; Psychiatry
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Psychiatry
GA 284EV
UT WOS:000254690300051
PM 18160196
ER
PT J
AU Hashimoto, K
Shinohe, A
Mori, N
AF Hashimoto, Kenji
Shinohe, Atsuko
Mori, Norio
TI Reply to: The hyperglutamatergic hypothesis of autism
SO PROGRESS IN NEURO-PSYCHOPHARMACOLOGY & BIOLOGICAL PSYCHIATRY
LA English
DT Letter
ID CARRIER SLC25A12 GENE; CEREBELLAR CORTICES; GLUTAMATE; PARIETAL;
ASSOCIATION; LINKAGE; BRAIN
C1 [Hashimoto, Kenji] Chiba Univ, Ctr Forens Mental Hlth, Div Clin Neurosci, Chiba, Japan.
[Shinohe, Atsuko; Mori, Norio] Hamamatsu Univ Sch Med, Dept Psychiat & Neurol, Hamamatsu, Shizuoka 43131, Japan.
RP Hashimoto, K (reprint author), Chiba Univ, Ctr Forens Mental Hlth, Div Clin Neurosci, Chiba, Japan.
EM hashimoto@faculty.chiba-u.jp
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NR 15
TC 0
Z9 0
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0278-5846
J9 PROG NEURO-PSYCHOPH
JI Prog. Neuro-Psychopharmacol. Biol. Psychiatry
PD APR 1
PY 2008
VL 32
IS 3
BP 912
EP 913
DI 10.1016/j.pnpbp.2007.11.005
PG 2
WC Clinical Neurology; Neurosciences; Pharmacology & Pharmacy; Psychiatry
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Psychiatry
GA 284EV
UT WOS:000254690300052
ER
PT J
AU Wassink, TH
Vieland, VJ
Sheffield, VC
Bartlett, CW
Goedken, R
Childress, D
Piven, J
AF Wassink, Thomas H.
Vieland, Veronica J.
Sheffield, Val C.
Bartlett, Christopher W.
Goedken, Rhinda
Childress, Deborah
Piven, Joseph
TI Posterior probability of linkage analysis of autism dataset identifies
linkage to chromosome 16
SO PSYCHIATRIC GENETICS
LA English
DT Article
DE autism; genetics; heterogeneity; language delay; linkage; positional
cloning
ID DIAGNOSTIC OBSERVATION SCHEDULE; MAJOR SUSCEPTIBILITY LOCUS; GENOME-WIDE
SCAN; LANGUAGE IMPAIRMENT; DATA SET; SCREEN; DISORDER; HETEROGENEITY;
TWIN; PHENOTYPES
AB Objective To apply phenotypic and statistical methods designed to account for heterogeneity to linkage analyses of the autism Collaborative Linkage Study of Autism (CLSA) affected sibling pair families.
Method The CLSA contains two sets of 57 families each; Set 1 has been analyzed previously, whereas this study presents the first analyses of Set 2. The two sets were analyzed independently, and were further split based on the degree of phrase speech delay in the siblings. Linkage analysis was carried out using the posterior probability of linkage (PPL), a Bayesian statistic that provides a mathematically rigorous mechanism for combining linkage evidence across multiple samples.
Results Two-point PPLs from Set 1 led to the follow-up genotyping of 18 markers around linkage peaks on 1q, 13p, 13q, 16q, and 17q in both sets of families. Multipoint PPLs were then calculated for the entire CLSA sample. These analyses identified four regions with at least modest evidence in support of linkage: 1q at 173 cM, PPL = 0.12; 13p at 21 cM, PPL=0.16; 16q at 63cM, PPL=0.36; Xq at 40 cM, PPL = 0.11.
Conclusion We find strengthened evidence for linkage of autism to chromosomes 1q, 13p, 16q and Xq, and, diminished evidence for linkage to 7q and 13q. The verity of these findings will be tested by continuing to update our PPL analyses with data from additional autism datasets.
C1 [Wassink, Thomas H.; Goedken, Rhinda] Univ Iowa, Carver Coll Med, Howard Hughes Med Inst, Dept Psychiat,Psychiat Res MEB, Iowa City, IA 52242 USA.
[Sheffield, Val C.] Univ Iowa, Carver Coll Med, Howard Hughes Med Inst, Dept Pediat, Iowa City, IA 52242 USA.
[Vieland, Veronica J.; Bartlett, Christopher W.] Ohio State Univ, Columbus Childrens Res Inst, Ctr Quantitat & Computat Biol, Columbus, OH 43210 USA.
[Childress, Deborah; Piven, Joseph] Univ N Carolina, Neurodev Disorders Res Ctr, Chapel Hill, NC 27515 USA.
[Childress, Deborah; Piven, Joseph] Univ N Carolina, Dept Psychiat, Chapel Hill, NC 27515 USA.
RP Wassink, TH (reprint author), Univ Iowa, Carver Coll Med, Howard Hughes Med Inst, Dept Psychiat,Psychiat Res MEB, Iowa City, IA 52242 USA.
EM thomas-wassink@uiowa.edu
RI Bartlett, Christopher/B-4958-2009
OI Bartlett, Christopher/0000-0001-7837-6348
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NR 46
TC 3
Z9 3
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0955-8829
J9 PSYCHIAT GENET
JI Psychiatr. Genet.
PD APR
PY 2008
VL 18
IS 2
BP 85
EP 91
PG 7
WC Genetics & Heredity; Neurosciences
SC Genetics & Heredity; Neurosciences & Neurology
GA 281FK
UT WOS:000254482100007
PM 18349700
ER
PT J
AU Kawamura, Y
Takahashi, O
Ishii, T
AF Kawamura, Yuichi
Takahashi, Osamu
Ishii, Takashi
TI Reevaluating the incidence of pervasive developmental disorders: Impact
of elevated rates of detection through implementation of an integrated
system of screening in Toyota, Japan
SO PSYCHIATRY AND CLINICAL NEUROSCIENCES
LA English
DT Article
DE autism; autistic disorder; epidemiology; incidence; PDD
ID TOTAL POPULATION; CUMULATIVE INCIDENCE; CHILDHOOD AUTISM; PREVALENCE;
CHILDREN
AB Aim: Although recent epidemiological studies on the pervasive development disorders (PDD) appear to be reporting higher rates of incidence than previously believed, great variation in the reported figures suggests a need for review of the methodology involved. As such, a survey on the incidence of PDD was conducted and compared with data from a previous survey to examine the effects of screening and diagnostic methodology on incidence.
Methods: The incidence of pervasive developmental disorders was surveyed in all children (12 589) born between January 1994 and December 1996 in Toyota, Japan.
Results: Incidence was 1.81% and the ratio of boys to girls was 2.80. Definitive diagnoses were made between 13 months and 7 years 2 months, the average age at diagnosis being 3 years 4 months. Among the cases of PDD, children with normal or borderline intelligence amounted to 66.4%, mild mental retardation (MR) 17.5%, moderate MR 10.3% and severe MR 5.8%.
Conclusion: An approximately 11-fold increase was noted in prevalence of PDD compared to a previous survey two decades ago, and two main factors were believed to account for this apparent sharp increase. First, inclusion of high-functioning subjects detected during infancy, and second, higher rates of diagnosis resulting from an integrated process of screening.
C1 [Kawamura, Yuichi; Takahashi, Osamu] Nagoya Univ Hosp, Toyota Municipal Child Dev Ctr, Aichi, Japan.
[Ishii, Takashi] Nagoya Univ Hosp, Dept Psychiat Parents & Children, Aichi, Japan.
RP Kawamura, Y (reprint author), 2-19 Nishiyama Cho, Toyota 4710062, Japan.
EM brt_future@yahoo.co.jp
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
American Psychiatric Association, 1980, DIAGN STAT MAN MENT
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NR 17
TC 34
Z9 34
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1323-1316
J9 PSYCHIAT CLIN NEUROS
JI Psychiatry Clin. Neurosci.
PD APR
PY 2008
VL 62
IS 2
BP 152
EP 159
DI 10.1111/j.1440-1819.2008.01748.x
PG 8
WC Clinical Neurology; Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 288ND
UT WOS:000254992200004
PM 18412836
ER
PT J
AU Kurita, H
Koyama, T
Inoue, K
AF Kurita, Hiroshi
Koyama, Tomonori
Inoue, Kanna
TI Reliability and validity of the pervasive developmental disorders
assessment system
SO PSYCHIATRY AND CLINICAL NEUROSCIENCES
LA English
DT Article
DE diagnosis; pervasive developmental disorders (PDD); reliability;
semistructured interview; validity
ID AUTISM DIAGNOSTIC INTERVIEW; CHILDREN; VERSION
AB Aim: To test the reliability and validity of the Pervasive Developmental Disorders Assessment System (PDDAS), a Japanese semistructured interview system.
Methods: The PDDAS, consisting of 91 items including 12 major items corresponding to 12 items in criterion A of DSM-IV autistic disorder criteria, 36 items on autistic symptoms and three Asperger's disorder (AS) screening items for diagnosing pervasive developmental disorders (PDD) and their subtypes and 40 items for other information including early development and past/family histories, was administered to mothers of 77 PDD children and 64 non-PDD children.
Results: The PDDAS had satisfactory interrater reliability (ranges of kappa, r and raw agreement rate were 0.69-1.00 in 76 items, 1.00 in 11 items and 0.91-1.00 in four kappa un-calculable items, respectively). Thirty-three of the 36 items and all of the 12 major items scored significantly higher in the PDD than non-PDD groups to show satisfactory discriminant validity. PDDAS and consensus DSM-IV diagnoses agreed in the 77 children in PDD diagnosis and disagreed in only two children in subtype diagnoses of autistic disorder and PDD not otherwise specified.
Conclusions: The PDDAS, which takes 1.5 h to administer, seems to have clinical and research utility, although further investigation is necessary.
C1 [Koyama, Tomonori] Natl Ctr Neurol & Psychiat, NIMH, Dept Child & Adolescent Mental Hlth, Tokyo, Japan.
[Inoue, Kanna] Nerima Welf Ctr Handicapped Persons, Tokyo, Japan.
RP Kurita, H (reprint author), Zenkoku Ryoiku Sodan Ctr, Shinjuku Ku, 2-2-8 Nishiwaseda, Tokyo 1620051, Japan.
EM hkurita@mvf.biglobe.ne.jp
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American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 9
TC 2
Z9 2
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1323-1316
J9 PSYCHIAT CLIN NEUROS
JI Psychiatry Clin. Neurosci.
PD APR
PY 2008
VL 62
IS 2
BP 226
EP 233
DI 10.1111/j.1440-1819.2008.01759.x
PG 8
WC Clinical Neurology; Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 288ND
UT WOS:000254992200015
PM 18412847
ER
PT J
AU Savaskan, E
Ehrhardt, R
Schulz, A
Walter, M
Schachinger, H
AF Savaskan, Egemen
Ehrhardt, Rike
Schulz, Andre
Walter, Marc
Schaechinger, Hartmut
TI Post-learning intranasal oxytocin modulates human memory for facial
identity
SO PSYCHONEUROENDOCRINOLOGY
LA English
DT Article
DE oxytocin; memory; facial expression; identity
ID POSTTRAUMATIC-STRESS-DISORDER; SOCIAL RECOGNITION; NEUROHYPOPHYSEAL
HORMONES; VASOPRESSIN; PEPTIDES; BEHAVIOR; AMYGDALA; RATS; AUTISM; BRAIN
AB The nanopeptide oxytocin has physiological functions during labour and lactation. In addition, oxytocin is known to modulate aggression, anxiety, social behaviour and cognition. Little is known about its effects on memory for emotional stimuli. In the present single-blind, placebo-controlled, randomised study we have investigated the short- and long-term effects of a single post-learning dose (20 IU) of intranasal oxytocin on memory for facial identity and expression in 36 healthy young females and males using a face portrait recognition test. In the acquisition phase of the test, 60 different male faces with happy, angry or neutral expressions were presented to the volunteers. Thirty minutes and 24h after oxytocin administration, recognition memory tests were performed using portraits with neutral facial expressions, only. Oxytocin improved identity recognition memory independently of participant's gender, for neutral and angry faces, whereas this effect was not present for happy faces. Oxytocin-treated subjects had a lower bias to judge not previously seen faces as being previously seen. Oxytocin had no effect on facial expression memory. In conclusion, oxytocin has distinct effects on memory performance for facial identity and may contribute to the modulation of social behaviour. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Savaskan, Egemen] Psychiat Univ Hosp, Div Psychiat Res & Psychogeriatr Med, CH-8032 Zurich, Switzerland.
[Ehrhardt, Rike; Schulz, Andre; Schaechinger, Hartmut] Univ Trier, Inst Psychobiol, Div Clin Physiol, D-54286 Trier, Germany.
[Walter, Marc] Psychiat Univ Clin, Basel, Switzerland.
RP Savaskan, E (reprint author), Psychiat Univ Hosp, Div Psychiat Res & Psychogeriatr Med, Minervastr 145,POB 1682, CH-8032 Zurich, Switzerland.
EM egemen.savaskan@puk.zh.ch
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NR 51
TC 111
Z9 114
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0306-4530
J9 PSYCHONEUROENDOCRINO
JI Psychoneuroendocrinology
PD APR
PY 2008
VL 33
IS 3
BP 368
EP 374
DI 10.1016/j.psyneuen.2007.12.004
PG 7
WC Endocrinology & Metabolism; Neurosciences; Psychiatry
SC Endocrinology & Metabolism; Neurosciences & Neurology; Psychiatry
GA 282LD
UT WOS:000254568400012
PM 18221838
ER
PT J
AU Brown, AK
Brown, JL
Poulson, CL
AF Brown, Ann K.
Brown, John L.
Poulson, Claire L.
TI Discriminating which fork to use: Teaching selective imitation to people
with autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Review
DE Autism; Generalized imitation; Imitation; Observing response
ID GENERALIZED IMITATION; CHILDREN; PEER; ACQUISITION; PERFORMANCE;
INFANTS; LIFE
AB Little empirical research has focused on teaching of imitation to learners with autism in ordinary environments. Typically-developing individuals imitate the behavior of others in ordinary social environments. One possible reason that learners with autism do not imitate in ordinary environments is they are not observing relevant discriminative stimuli that should set the occasion for imitative responding. This paper will review the operant research on generalized imitation with the goal of identifying procedures to teach learners with autism to imitate in ordinary environments. A stimulus-control account of imitation in ordinary environments is included with the goal of the development of effective teaching procedures. Imitation in ordinary environments is discussed in relation to the discriminative stimuli that occasion imitative responding. The use of differential observing responses to increase discrimination of relevant stimuli in ordinary environments is suggested as a possible strategy to increase imitation among individuals with autism in ordinary environments. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Brown, Ann K.; Brown, John L.] REED Acad, Garfield, NJ 07026 USA.
[Brown, Ann K.; Poulson, Claire L.] CUNY Grad Sch & Univ Ctr, New York, NY 10036 USA.
[Poulson, Claire L.] CUNY Queens Coll, New York, NY USA.
RP Brown, AK (reprint author), REED Acad, 85 Summit Ave, Garfield, NJ 07026 USA.
EM abrown@reedacademy.org
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NR 21
TC 4
Z9 4
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 199
EP 208
DI 10.1016/j.rasd.2007.06.001
PG 10
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800001
ER
PT J
AU Noens, ILJ
van Berckelaer-Onnes, IA
AF Noens, Ilse L. J.
van Berckelaer-Onnes, Ina A.
TI The central coherence account of autism revisited: Evidence from the
ComFor study
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Central coherence; Perception; Local; Global; Autism; Intellectual
disability
ID WEAK CENTRAL COHERENCE; HIGH-FUNCTIONING AUTISM; ASPERGER-SYNDROME;
INTELLECTUAL DISABILITY; COGNITIVE PHENOTYPE; CHILDREN; COMMUNICATION;
ADOLESCENTS; DISORDER; ADULTS
AB According to the central coherence account, people with autism have a tendency to focus oil local rather than global processing. However, there is considerable controversy about the locus of the weak drive for central coherence. Some studies support enhanced bottom-up processing, whereas others claim reduced top-down feedback. The results of the standardization study of the ComFor - a clinical instrument for the indication of augmentative communication, based on the central coherence account - were reviewed within the perspective of this debate. One hundred fifty-five individuals with intellectual disability and the autistic disorder were individually matched with 155 individuals with intellectual disability without the autistic disorder according to their level of daily living skills. The finding that individuals with the autistic disorder exhibit a higher discrepancy between the presentation and representation scores of the ComFor is consistent with expectations on the basis of the central coherence theory, but does not stipulate whether this is due to enhanced bottom-up or reduced top-down processing. Item level analyses, however, show that enhanced local processing emerges most clearly on those items whereby the establishment of meaning (global processing) is not supportive, suggesting that enhanced bottom-up processing and reduced global feedback are interconnected. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Noens, Ilse L. J.] Leiden Univ, Clin Child & Adolescent Studies, NL-2300 RB Leiden, Netherlands.
RP Noens, ILJ (reprint author), Leiden Univ, Clin Child & Adolescent Studies, NL-2300 RB Leiden, Netherlands.
EM noens@fsw.leidenuniv.nl
CR Bates E., 1979, EMERGENCE SYMBOLS CO, P33
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NR 46
TC 6
Z9 6
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 209
EP 222
DI 10.1016/j.rasd.2007.05.004
PG 14
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800002
ER
PT J
AU Olive, ML
Lang, RB
Davis, TN
AF Olive, Melissa L.
Lang, Russell B.
Davis, Tonya N.
TI An analysis of the effects of functional communication and a Voice
Output Communication Aid for a child with autism spectrum disorder
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Functional communication training; Voice output communication aid;
Pronoun reversal
ID SEVERE BEHAVIOR PROBLEMS; PERSONAL PRONOUNS; SELF-INJURY; SETTINGS;
ACCEPTABILITY; INTERVENTION
AB The purpose of this study was to examine the effects of Functional Communication Training (FCT) and a Voice Output Communication Aid (VOCA) on the challenging behavior and language development of a 4-year-old girl with autism spectrum disorder. The participant's mother implemented modified functional analysis (FA) and intervention procedures in Kerri's home. A multiple probe design across activities was used to analyze intervention effectiveness. FCT with a VOCA successfully decreased Kerri's challenging behavior and increased VOCA use. A secondary analysis revealed that Kerri increased her use of correct pronouns. Kerri's mother implemented modified FA and intervention procedures with a high level of fidelity. Social validity data indicated that Kerri's mother believed the intervention to be acceptable and effective. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Olive, Melissa L.] Univ Texas Austin, Dept Special Educ, Austin, TX 78712 USA.
RP Olive, ML (reprint author), Univ Texas Austin, Dept Special Educ, 1 Univ Stn D5300, Austin, TX 78712 USA.
EM molive@mail.utexas.edu
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NR 37
TC 14
Z9 14
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 223
EP 236
DI 10.1016/j.rasd.2007.06.002
PG 14
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800003
ER
PT J
AU Matson, JL
Boisjoli, JA
AF Matson, Johnny L.
Boisjoli, Jessica A.
TI Strategies for assessing Asperger's syndrome: A critical review of data
based methods
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Asperger's syndrome; Assessment; Children; Data based
ID AUTISM SPECTRUM DISORDERS; HIGH-FUNCTIONING AUTISM; SCHOOL-AGE-CHILDREN;
DIAGNOSTIC INTERVIEW; YOUNG-CHILDREN; RATING-SCALE;
DIFFERENTIAL-DIAGNOSIS; SOCIAL-SKILLS; CHILDHOOD AUTISM; ADULTS
AB Asperger's syndrome has gained tremendous recognition and popularity in the last 20 years. However, controversy around the nature of the disorder, whether it is distinct from high functioning autism, and whether it can be reliably and validly diagnosed has continued throughout this period unabated. Fortunately, there has been a strong tradition of developing systematic data based methods of differential diagnosis in the autism spectrum disorders. The bulk of the effort has been in autism, but there has been moderate, yet consistent efforts to develop data based methods to diagnose Asperger's syndrome as well. The present paper provides an up to date critical review of the existing literature on the topic. Strengths, weaknesses of the research, and avenues for future efforts are discussed. (C) 2007 Published by Elsevier Ltd.
C1 [Matson, Johnny L.; Boisjoli, Jessica A.] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70806 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70806 USA.
EM johnmatson@aol.com
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NR 80
TC 43
Z9 43
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
EI 1878-0237
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 237
EP 248
DI 10.1016/j.rasd.2007.06.003
PG 12
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800004
ER
PT J
AU DeQuinzio, JA
Townsend, DB
Poulson, CL
AF DeQuinzio, Jaime Ann
Townsend, Dawn Buffington
Poulson, Claire L.
TI The effects of forward chaining and contingent social interaction on the
acquisition of complex sharing responses by children with autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Forward chaining; Complex prosocial responses; Differential
reinforcement
ID PRESCHOOL-CHILDREN; SKILLS
AB Children with autism have deficits in social interaction, including the failure to engage in sharing responses. Four children with autism were taught a sharing response chain. The treatment package (manual guidance, auditory prompts, and contingent access to toy play and social interaction with the recipient instructor) was introduced successively across participants in a multiple-baseline design. None of the participants engaged in the sharing response chain during baseline. Systematic increases in responding occurred for all four participants in the presence of training stimuli. In addition, there were systematic increases in responding to non-trained probe stimuli. Also, during pre- and post-test measures, the participants demonstrated sharing in the presence of peers in a non-training classroom containing non-trained toys. Furthermore, social validity measures indicated that judges scored more post-treatment responses than baseline responses as "sharing." (C) 2007 Elsevier Ltd. All rights reserved.
C1 [DeQuinzio, Jaime Ann; Poulson, Claire L.] CUNY Queens Coll, Dept Psychol, Flushing, NY 11367 USA.
[DeQuinzio, Jaime Ann; Poulson, Claire L.] CUNY, Grad Ctr, Flushing, NY 11367 USA.
RP DeQuinzio, JA (reprint author), CUNY Queens Coll, Dept Psychol, 65-30 Kissena Blvd, Flushing, NY 11367 USA.
EM jaimedes@aol.com
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Bandura A, 1963, SOCIAL LEARNING PERS
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NR 19
TC 5
Z9 5
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 264
EP 275
DI 10.1016/j.rasd.2007.06.006
PG 12
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800006
ER
PT J
AU Matson, JL
Boisjoli, JA
AF Matson, Johnny L.
Boisjoli, Jessica A.
TI Autism spectrum disorders in adults with intellectual disability and
comorbid psychopathology: Scale development and reliability of the
ASD-CA
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Assessment; Autism; Comorbidity; Intellectual disability
ID MENTAL-RETARDATION; BEHAVIOR PROBLEMS; CHILDREN; PARENTS; DEPRESSION;
DIAGNOSIS; VALIDITY; ISSUES; SAMPLE
AB Researchers and clinicians have generally agreed that persons with autism spectrum disorders (ASD) are susceptible to other DSM Axis I disorders. However, to date, little has been done to establish the specific disorders, their rate and severity of occurrence, and their interrelationship with ASD symptoms. One reason for the lack of research has been the absence of a comorbidity measure for this population. Additionally, when research has been conducted, it typically has been on one disorder with young children. The present study assessed comorbid DSM Axis I disorders in adults with ASD, either autism or PDDNOS and intellectual disabilities, using a new scale designed for the purpose. The reliability and factor structure of the scale was evaluated and implications for future research are discussed. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Matson, Johnny L.; Boisjoli, Jessica A.] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
EM johnmatson@aol.com
CR ANDERSON JC, 1987, ARCH GEN PSYCHIAT, V44, P69
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Zachor DA, 2006, RES DEV DISABIL, V27, P400, DOI 10.1016/j.ridd.2005.05.004
NR 42
TC 19
Z9 19
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 276
EP 287
DI 10.1016/j.rasd.2007.07.002
PG 12
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800007
ER
PT J
AU Matson, JL
Wilkins, J
AF Matson, Johnny L.
Wilkins, Jonathan
TI Nosology and diagnosis of Asperger's syndrome
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Asperger's syndrome; Diagnosis; Nosology
ID HIGH-FUNCTIONING AUTISM; PERVASIVE DEVELOPMENTAL DISORDERS; SPECTRUM
DISORDERS; DIFFERENTIAL-DIAGNOSIS; YOUNG-CHILDREN; SCHIZOID PERSONALITY;
PRESCHOOL-CHILDREN; FAMILY-HISTORY; INTERVENTION; ADULTS
AB The autism spectrum disorders (ASD) have generated great interest among clinicians and researchers. Once considered rare, recent epidemiological data now suggests rates of up to I in 150 people. One of the most frequent of the ASD, Asperger's syndrome (AS), has been known as a disorder for as long as autism, which is easily the most visible of this group of conditions. Growing attention to and popularity of studying AS have made the nosology and diagnosis of the disorder a topic of growing concern in recent years. The purpose of this paper is to present an up-to-date analysis of the syndrome and the likelihood that it is actually a disorder distinct from high-functioning autism (HFA). This argument about how to diagnose people, either AS or HFA, hinges largely on the debate about whether distinct symptom patterns exist between AS and HFA and if AS can be reliably differentiated from HFA. A discussion of this topic, trends in research, and where the data appears to be leading diagnosticians is also presented, as well as research areas requiring further attention. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Matson, Johnny L.] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
EM johnmatson@aol.com
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NR 96
TC 67
Z9 67
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
EI 1878-0237
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 288
EP 300
DI 10.1016/j.rasd.2007.07.003
PG 13
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800008
ER
PT J
AU Kahana-Kalman, R
Goldman, S
AF Kahana-Kalman, Ronit
Goldman, Sylvie
TI Intermodal matching of emotional expressions in young children with
autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism; Intermodal matching; Emotional expression
ID INFANTS PERCEPTION; FACIAL EXPRESSIONS; SPECTRUM DISORDER; EARLY
RECOGNITION; FACE; BEHAVIORS; INFORMATION; PEOPLE
AB This study examined the ability of young children with autism spectrum disorders (ASD) to detect affective correspondences between facial and vocal expressions of emotion using an intermodal matching paradigm. Four-year-old children with ASD (n = 18) and their age-matched normally developing peers (n = 18) were presented pairs of videotaped facial expressions accompanied by a single soundtrack matching the affect of one of the two facial expressions. In one block of trials, the emotions were portrayed by their mothers; in another block of trials, the same emotion pairs were portrayed by an unfamiliar woman. Findings showed that ASD children were able to detect the affective correspondence between facial and vocal expressions of emotion portrayed by their mothers, but not a stranger. Furthermore. in a control condition using inanimate objects and their sounds, ASD children also showed a preference for sound-matched displays. These results suggest that children with ASD do not have a general inability to detect intermodal correspondences between visual and vocal events, however, their ability to detect affective correspondences between facial and vocal expressions of emotions may be limited to familiar displays. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Kahana-Kalman, Ronit; Goldman, Sylvie] Albert Einstein Coll Med, Rose F Kennedy Ctr Res Mental Retardat & Human De, Dept Pediat, New York, NY 10033 USA.
[Goldman, Sylvie] Yeshiva Univ Albert Einstein Coll Med, Rose F Kennedy Ctr Res Mental Retardat & Human De, Saul R Korey Dept Neurol, Bronx, NY 10461 USA.
[Goldman, Sylvie] Childrens Hosp Montefiore, Bronx, NY USA.
RP Goldman, S (reprint author), Albert Einstein Coll Med, Rose F Kennedy Ctr Res Mental Retardat & Human De, Dept Pediat, New York, NY 10033 USA.
EM rk2024@nyu.com; sylviegold@aol.com
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 37
TC 6
Z9 6
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 301
EP 310
DI 10.1016/j.rasd.2007.07.004
PG 10
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800009
ER
PT J
AU McDonnell, A
Sturmey, P
Oliver, C
Cunningham, J
Hayes, S
Galvin, M
Walshe, C
Cunningham, C
AF McDonnell, Andrew
Sturmey, Peter
Oliver, Chris
Cunningham, Joanna
Hayes, Samira
Galvin, Martin
Walshe, Caroline
Cunningham, Cathy
TI The effects of staff training on staff confidence and challenging
behavior in services for people with autism spectrum disorders
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Confidence; Training; Physical intervention
ID INTELLECTUAL DISABILITIES; PATIENT AGGRESSION; CARE STAFF; MANAGEMENT;
QUESTIONNAIRE; INTERVENTION; PERCEPTIONS; PERFORMANCE
AB The effects of a 3-day training course in the management of aggressive behavior in services for people with autism spectrum disorders were investigated using a quasi-experimental design. An experimental group received training over a 10-month period and a contrast group, which had received training before this study, did not. Staff training increased carer confidence, but there were no training effects of measures of staff coping, support or perceived control of challenging behaviors. Staff reports of service user challenging behavior management difficulties decreased in both the experimental and contrast groups. This study showed that staff training can increase staff confidence in managing aggression in people with autism spectrum disorders. (C) 2007 Published by Elsevier Ltd.
C1 [McDonnell, Andrew; Cunningham, Joanna] Studio III Training Syst, Bath, Avon, England.
[Sturmey, Peter] CUNY Queens Coll, New York, NY USA.
[Sturmey, Peter] CUNY, Grad Ctr, New York, NY USA.
[Oliver, Chris] Univ Birmingham, Dept Psychol, Birmingham B15 2TT, W Midlands, England.
[Hayes, Samira; Galvin, Martin; Walshe, Caroline; Cunningham, Cathy] Gheel Autism Serv, Dublin, Ireland.
RP McDonnell, A (reprint author), Studio III Training Syst, Bath, Avon, England.
EM Andy@studio3.org
CR Allen D., 2001, TRAINING CARERS PHYS
Allen D, 2000, MENT RETARD, V38, P97, DOI 10.1352/0047-6765(2000)038<0097:RTABIO>2.0.CO;2
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NR 40
TC 7
Z9 7
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 311
EP 319
DI 10.1016/j.rasd.2007.08.001
PG 9
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800010
ER
PT J
AU Mazefsky, CA
Goin-Kochel, RP
Riley, BP
Maes, HH
AF Mazefsky, Carla A.
Goin-Kochel, Robin P.
Riley, Brien P.
Maes, Hermine H.
TI Genetic and environmental influences on symptom domains in twins and
siblings with autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism; Autism diagnostic interview; Behavioral genetics; Twins; Social
interaction; Nonverbal communication
ID PERVASIVE DEVELOPMENTAL DISORDERS; INTENSIVE BEHAVIORAL TREATMENT;
YOUNG-CHILDREN; GENERAL-POPULATION; SPECTRUM; TRAITS; INDIVIDUALS;
LINKAGE; MODELS
AB Clarifying the sources of variation among autism symptom domains is important to the identification of homogenous subgroups for molecular genetic studies. This study explored the genetic and environmental bases of nonverbal communication and social interaction, two symptom domains that have also been related to treatment response, in 1294 child and adolescent twins and siblings with pervasive developmental disorders (PDDs) from the Autism Genetic Resource Exchange under the age of 18. Twin/sibling resemblance was assessed through correlations and behavior genetic modeling of autism diagnostic interview (ADI) nonverbal communication and social scores. Variation in these phenotypes was explained by additive genetic, dominant genetic, and unique environmental factors with no evidence for shared environmental factors. Broad heritability estimates were higher for nonverbal communication (45%) than social interaction (28%). Nonverbal communication and social scores were partially accounted for by the same underlying genetic and environmental factors. Gender differences were not supported. These results add to information on familial resemblance of these symptom domains based on correlational methods, and this study is one of the first to apply behavioral genetic modeling to a PDD population. The results have implications for molecular genetics as well as treatment. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Mazefsky, Carla A.] Univ Pittsburgh, Sch Med, Dept Pediat, Pittsburgh, PA 15261 USA.
[Mazefsky, Carla A.] Univ Pittsburgh, Sch Med, Dept Psychiat, Pittsburgh, PA 15261 USA.
[Riley, Brien P.; Maes, Hermine H.] Virginia Commonwealth Univ, Virginia Inst Psychiat & Behav Genet, Richmond, VA 23284 USA.
[Goin-Kochel, Robin P.] Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
RP Mazefsky, CA (reprint author), Univ Pittsburgh, Childrens Hosp Pittsburgh, Dept Pediat, Child Dev Unit, 3705 5th Ave, Pittsburgh, PA 15213 USA.
EM Carla.Mazefsky@chp.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Andres C, 2002, BRAIN RES BULL, V57, P109, DOI 10.1016/S0361-9230(01)00642-6
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Bimbrauer J. S., 1993, BEHAV CHANGE, V10, P63
Carpenter M, 2002, J AUTISM DEV DISORD, V32, P91, DOI 10.1023/A:1014836521114
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Goin-Kochel R., 2007, INT J DISABIL DEV ED, V54, P151, DOI 10.1080/10349120701330404
GOINKOCHEL RP, AUTISM GENE IN PRESS
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NR 31
TC 11
Z9 11
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 320
EP 331
DI 10.1016/j.rasd.2007.08.002
PG 12
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800011
ER
PT J
AU Ingersoll, B
AF Ingersoll, Brooke
TI The effect of context on imitation skills in children with autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism; Imitation; Social communication
ID YOUNG-CHILDREN; SPECTRUM DISORDERS; MOTOR IMITATION; COMMUNICATION;
ATTENTION; LANGUAGE; OBJECT
AB Children with autism exhibit deficits in imitation skills. Previous authors have suggested that they may have particular difficulty imitating in natural social interactions, but properly controlled experiments investigating this possibility have not been conducted. To investigate this possibility, children with autism and typically developing children were compared on a series of imitation tasks presented either in a structured-elicited or naturalistic-spontaneous condition. Modeled actions were counterbalanced across conditions. Results suggest children with autism imitated less than typically developing children overall; however, this difference was mainly evident when the imitation task was presented in a spontaneous context. In addition, they exhibited less coordinated joint attention during imitation than the typically developing children. These findings support the hypothesis that children with autism are particularly impaired in their ability to imitate spontaneously. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Ingersoll, Brooke] Michigan State Univ, Lewis & Clark Coll, E Lansing, MI 48824 USA.
RP Ingersoll, B (reprint author), Michigan State Univ, Lewis & Clark Coll, 105 B Psychol Bldg, E Lansing, MI 48824 USA.
EM ingers19@msu.edu
RI Ingersoll, Brooke/A-9117-2012
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Brown J. D., 1998, INTERSUBJECTIVE COMM, P260
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Ingersoll B, 2003, J AUTISM DEV DISORD, V33, P673, DOI 10.1023/B:JADD.0000006003.26667.f8
INGERSOLL B, INFANTS YOU IN PRESS
Landry R, 2004, J CHILD PSYCHOL PSYC, V45, P1115, DOI 10.1111/j.1469-7610.2004.00304.x
Lord C, 2000, J AUTISM DEV DISORD, V30, P205, DOI 10.1023/A:1005592401947
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NR 26
TC 14
Z9 14
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 332
EP 340
DI 10.1016/j.rasd.2007.08.003
PG 9
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800012
ER
PT J
AU Argott, P
Townsend, DB
Sturmey, P
Poulson, CL
AF Argott, Paul
Townsend, Dawn Buffington
Sturmey, Peter
Poulson, Claire L.
TI Increasing the use of empathic statements in the presence of a
non-verbal affective stimulus in adolescents with autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism; Empathy; Script-fading; Non-verbal stimuli
ID SCRIPT-FADING PROCEDURE; SOCIAL-INTERACTION SKILLS; TEACHING-CHILDREN;
BEHAVIOR; INFANTS
AB Previous studies have shown that most individuals with autism do not show empathic responding. The present study is an attempt to teach such skills. Script-fading procedures have been used to teach other social-interaction skills. so they arc applied here to teach empathic responding. This study included three adolescents with autism, two males and one female. A non-verbal affective stimulus was presented and students' empathic responding was recorded. Data were collected on scripted and unscripted verbal statements of empathy in the presence of training discriminative stimuli and on unscripted verbal statements of empathy in the presence of non-training discriminative stimuli. A multiple-baseline-across-participants experimental design was used to assess the effectiveness of a script-fading procedure on increasing verbal statements of empathy. With the successive introduction of scripts and a script-fading procedure across participants, the percentage of opportunities on which scripted and unscripted statements of empathy occurred, in the presence of the training stimuli, increased systematically. Additionally, an increase in the percentage of opportunities on which an unscripted statement of empathy occurred in the presence of generalization stimuli was observed. These data show that adolescents with autism can learn to differentiate non-verbal affective stimuli and display differential empathic responses with behavioral interventions. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Argott, Paul; Sturmey, Peter; Poulson, Claire L.] CUNY Queens Coll, New York, NY USA.
[Argott, Paul; Sturmey, Peter; Poulson, Claire L.] CUNY, Grad Ctr, New York, NY USA.
RP Argott, P (reprint author), 169 W Saddle River Rd, Saddle River, NJ 07458 USA.
EM p_argott@yahoo.com
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
BROWN JL, 2003, 29 ANN CONV ASS BEH
Buffington DM, 1998, J AUTISM DEV DISORD, V28, P535, DOI 10.1023/A:1026056229214
Charman T, 1997, DEV PSYCHOL, V33, P781, DOI 10.1037//0012-1649.33.5.781
Charman T, 1998, INF MENTAL HLTH J, V19, P260, DOI 10.1002/(SICI)1097-0355(199822)19:2<260::AID-IMHJ12>3.0.CO;2-W
Dyck MJ, 2001, EUR CHILD ADOLES PSY, V10, P105
Gena A, 1996, J APPL BEHAV ANAL, V29, P291, DOI 10.1901/jaba.1996.29-291
Krantz PJ, 1998, J APPL BEHAV ANAL, V31, P191, DOI 10.1901/jaba.1998.31-191
KRANTZ PJ, 1993, J APPL BEHAV ANAL, V26, P121, DOI 10.1901/jaba.1993.26-121
Sarokoff RA, 2001, J APPL BEHAV ANAL, V34, P81, DOI 10.1901/jaba.2001.34-81
SIGMAN MD, 1992, CHILD DEV, V63, P796, DOI 10.1111/j.1467-8624.1992.tb01662.x
Stevenson CL, 2000, BEHAV INTERVENT, V15, P1, DOI 10.1002/(SICI)1099-078X(200001/03)15:1<1::AID-BIN41>3.0.CO;2-V
Travis L, 2001, J AUTISM DEV DISORD, V31, P119, DOI 10.1023/A:1010705912731
YIRMIYA N, 1992, CHILD DEV, V63, P150, DOI 10.1111/j.1467-8624.1992.tb03603.x
NR 14
TC 7
Z9 7
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 341
EP 352
DI 10.1016/j.rasd.2007.08.004
PG 12
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800013
ER
PT J
AU Williams, DL
Goldstein, G
Kojkowski, N
Minshew, NJ
AF Williams, Diane L.
Goldstein, Gerald
Kojkowski, Nicole
Minshew, Nancy J.
TI Do individuals with high functioning autism have the IQ profile
associated with nonverbal learning disability?
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism; Nonverbal learning disability; Asperger syndrome; Wechsler
intelligence scales
ID PHONOLOGICAL PROCESSING DISABILITIES; DIAGNOSTIC OBSERVATION SCHEDULE;
ASPERGER-SYNDROME; RIGHT-HEMISPHERE; CHILDREN; CLASSIFICATION;
INTERVIEW; LANGUAGE; VALIDITY; SPECTRUM
AB Previously researchers have noted a high level of occurrence of the IQ profile associated with nonverbal learning disability (NLD) in Asperger syndrome (ASP) but not in high functioning autism (HFA). We examined the IQ profile scores of a large sample of children (n = 69) and adults (n = 77) with HFA. stringently diagnosed according to ADOS, ADI-R. and DSM-IV criteria. and a corresponding sample of typical child (it = 72) and adult controls (it = 107). At least one of the three primary components of the Wechsler pattern seen in NLD were found in 17-26% of the children and 20-32% of the adults with HFA. All three components occurred in slightly more than 5% of the children and adults with autism. Overall, the VIQ > PIQ profile seen in NLD occurred in 18% of the sample of individuals stringently diagnosed with HFA. Therefore, obtaining this IQ profile is not a valid clinical discriminator between NLD and HFA. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Williams, Diane L.] Duquesne Univ, Rangos Sch Hlth Sci, Dept Speech Language Pathol, Pittsburgh, PA 15282 USA.
[Goldstein, Gerald] VA Pittsburgh Healthcare Syst, Pittsburgh, PA USA.
[Minshew, Nancy J.] Univ Pittsburgh, Sch Med, Dept Psychiat, Pittsburgh, PA 15260 USA.
[Minshew, Nancy J.] Univ Pittsburgh, Sch Med, Dept Neurol, Pittsburgh, PA 15260 USA.
RP Williams, DL (reprint author), Duquesne Univ, Rangos Sch Hlth Sci, Dept Speech Language Pathol, 600 Forbes Ave, Pittsburgh, PA 15282 USA.
EM williamsd2139@duq.edu
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Drummond CR, 2005, ARCH CLIN NEUROPSYCH, V20, P171, DOI 10.1016/j.acn.2004.05.001
ELLIS HD, 1994, EUR CHILD ADOLES PSY, V3, P255
Frith U., 2003, AUTISM EXPLAINING EN
Ghaziuddin M, 2004, J AUTISM DEV DISORD, V34, P279, DOI 10.1023/B:JADD.0000029550.19098.77
Jolliffe T, 1999, COGNITION, V71, P149, DOI 10.1016/S0010-0277(99)00022-0
Joseph RM, 2002, J CHILD PSYCHOL PSYC, V43, P807, DOI 10.1111/1469-7610.00092
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LECOUTEUR A, 1989, J AUTISM DEV DISORD, V19, P363
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LORD C, 1994, J AUTISM DEV DISORD, V24, P659, DOI 10.1007/BF02172145
Lord C, 2000, J AUTISM DEV DISORD, V30, P205, DOI 10.1023/A:1005592401947
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World Health Organization, 1993, INT CLASS DIS 10 REV
Worling DE, 1999, BRAIN LANG, V70, P220, DOI 10.1006/brln.1999.2156
NR 37
TC 19
Z9 19
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 353
EP 361
DI 10.1016/j.rasd.2007.08.005
PG 9
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800014
ER
PT J
AU Bernstein, H
Sturmey, P
AF Bernstein, Haven
Sturmey, Peter
TI Effects of fixed-ratio schedule values on concurrent mands in children
with autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Concurrent schedules; Mands; Variability
AB Interventions with children with autism often involve more than one concurrent schedule or reinforcement. Manipulation of one schedule of reinforcement may affect responding on a second. We demonstrated the effect of manipulating the schedule of reinforcement for a single high-rate mand on alternative concurrently available mands with two children with autism. Teachers conducted sessions in the participant's usual teaching environment. When the schedule value for all mands was FR1, a single mand occurred at a high rate while all other mands occurred at low rates for both participants and response variability was low. When the schedule of reinforcement for the high-rate mand increased to FR10 for one participant and to FR25 for the second participant, and all other mands remained on FR1 schedules, the high-rate mand decreased while the combined rate of all other mands increased. There was also some evidence of increased response variability. We discuss the affects of schedule values during concurrent schedules and implications for increasing variability in manding. (C) 2007 Published by Elsevier Ltd.
C1 [Bernstein, Haven; Sturmey, Peter] CUNY, Queens Coll, New York, NY USA.
[Bernstein, Haven; Sturmey, Peter] CUNY, Grad Ctr, New York, NY 10021 USA.
RP Sturmey, P (reprint author), CUNY, Queens Coll, New York, NY USA.
EM petersturmey@QC.CUNY.edu
CR BERNSTEIN H, EFFECTS FIXED UNPUB
Hermstein RJ, 1970, J EXP ANAL BEHAV, V13, P243
HORNER RH, 1991, J APPL BEHAV ANAL, V24, P719, DOI 10.1901/jaba.1991.24-719
MACE FC, 1990, J APPL BEHAV ANAL, V23, P197, DOI 10.1901/jaba.1990.23-197
NR 4
TC 1
Z9 1
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 362
EP 370
DI 10.1016/j.rasd.2007.09.001
PG 9
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800015
ER
PT J
AU Esposito, G
Venuti, P
AF Esposito, Gianluca
Venuti, Paola
TI How is crying perceived in children with Autistic Spectrum Disorder
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE ASD; Episode of cry; Distress
ID EARLY RECOGNITION; INFANTS; INTERVENTION; PERCEPTIONS; POPULATION;
EXPRESSION; DISTRESS; PITCH; 1ST; AGE
AB Autistic Spectrum Disorder (ASD) is a disorder that affects language and social skills to varying degrees. While many studies have concentrated on examining patterns of behavior and development on the context of speaking and interacting, very few researchers have investigated the parameters of crying in infants with ASD. This finding is surprising since crying can be viewed as both the first communicative and social structure in human development. The aim of our study was to investigate how the crying of children with ASD, as opposed to children with intellectual disability (ID) was perceived. In particular, we tested a questionnaire to verify whether the atypical structure of autistic crying can bias parent perceptions. The atypical structure of autistic crying was highlighted. In autistic children, crying was inexplicable for their parents who could not identify causative factors. These results support the view of autism as related to a problem of expressing and sharing emotions. Parents' reactions to autistic crying were qualitatively different from non-autistic children of the same age. This difference was compounded parental attempt to share feelings and developing inter-subjectivity processes with their children. (C) 2007 Elsevier Ltd. All rights reserved.
C1 [Esposito, Gianluca; Venuti, Paola] Univ Trent, Observ & Funct Diag Lab DiSCoF, I-38100 Trento, Italy.
RP Esposito, G (reprint author), Univ Trent, Observ & Funct Diag Lab DiSCoF, I-38100 Trento, Italy.
EM gianluca.esposito@unitn.it
RI Esposito, Gianluca/B-1374-2012; Esposito, Gianluca/K-9353-2013
OI Esposito, Gianluca/0000-0002-9442-0254; Esposito,
Gianluca/0000-0002-9442-0254
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NR 49
TC 12
Z9 13
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
EI 1878-0237
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD APR-JUN
PY 2008
VL 2
IS 2
BP 371
EP 384
DI 10.1016/j.rasd.2007.09.003
PG 14
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DJ
UT WOS:000263413800016
ER
PT J
AU Geils, C
Knoetze, J
AF Geils, Catherine
Knoetze, Jan
TI Conversations with Barney: A conversation analysis of interactions with
a child with autism
SO SOUTH AFRICAN JOURNAL OF PSYCHOLOGY
LA English
DT Article
DE autism; autistic communication; construction of identity; conversation
analysis; positioning
ID COMMUNICATION; ECHOLALIA; MANAGEMENT
AB This study arose from an intervention programme aimed to develop the communication and social interaction skills of Barney, a six-year-old child diagnosed with Pervasive Developmental Disorder (Autistic Spectrum), The approach we take is a social constructionist one that challenges the assumption of a Western psychiatric approach emphasising the impairment and deficits associated with autism. Conversation analysis is employed as a method of elucidating the collaborative mechanisms employed by Barney and his co-participants in making sense of one another. The aim of this study was to closely examine the communicative behaviour and interactive styles of Barney and his co-participants, their implications for communicative success (cc-ordinated interaction) or breakdown (discordant interaction), and the implications for ways in which the child is positioned within the discourse in relation to his co-participants. Our constructions of the data suggested the following: A playful, activity-based interactive style constituted by non-verbal turns, affection, and short, simple utterances enhanced co-ordinated interaction. Discordant interaction seemed to result from a tendency of the co-participants to dominate the interaction (e.g., frequent and repetitive questioning) which directed and constrained interaction and resulted in the child's withdrawal. Other implications are highlighted. This research informs intervention efforts and encourages co-participants to reflect on ways in which interaction is co-constructed between themselves and the child with autism.
C1 [Geils, Catherine; Knoetze, Jan] Rhodes Univ, Dept Psychol, ZA-6140 Grahamstown, South Africa.
RP Geils, C (reprint author), Rhodes Univ, Dept Psychol, POB 94, ZA-6140 Grahamstown, South Africa.
EM j.knoetze@ru.ac.za
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NR 42
TC 2
Z9 3
PU UNISA PRESS
PI PRETORIA
PA PO BOX 392, PRETORIA, 0003, SOUTH AFRICA
SN 0081-2463
J9 S AFR J PSYCHOL
JI South Afr. J. Psychol.
PD APR
PY 2008
VL 38
IS 1
BP 200
EP 224
PG 25
WC Psychology, Multidisciplinary
SC Psychology
GA 329QN
UT WOS:000257884400011
ER
PT J
AU Orekhova, EV
Stroganova, TA
Prokofyev, AO
Nygren, G
Gillberg, C
Elam, M
AF Orekhova, Elena V.
Stroganova, Tatiana A.
Prokofyev, Andrey O.
Nygren, Gudrun
Gillberg, Cristopher
Elam, Mikael
TI Sensory gating in young children with autism: Relation to age, IQ, and
EEG gamma oscillations
SO NEUROSCIENCE LETTERS
LA English
DT Article
DE autism; children; ERP; P50 sensory gating; gamma oscillations
ID P50 SUPPRESSION; SCHIZOPHRENIA; INHIBITION; DISORDER; DEFICIT;
ELECTROENCEPHALOGRAM; STIMULATION; SPIKES; ADULTS; BOYS
AB Unusual reactions to auditory stimuli are often observed in autism and may relate to ineffective inhibitory modulation of sensory input (sensory gating). A previous study of P50 sensory gating did not reveal abnormalities in high-functioning school age children [C. Kemner, B. Oranje, M.N. Verbaten, H. van Engeland, Normal P50 gating in children with autism, J. Clin. Psychiatry 63 (2002) 214-217]. Sensory gating deficit may, however, characterize younger children with autism or be a feature of retarded children with autism, reflecting imbalance of neuronal excitation/inhibition in these cohorts. We applied a paired clicks paradigm to study P50 sensory gating, and its relation to IQ and EEG gamma spectral power (as a putative marker of cortical excitability), in young (3-8 years) children with autism (N = 21) and age-matched typically developing children (N = 21). P50 suppression in response to the second click was normal in high-functioning children with autism, but significantly (p < 0.03) reduced in those with mental retardation. P50 gating improved with age in both typically developing children and those with autism. Higher ongoing EEG gamma power corresponded to lower P50 suppression in autism (p < 0.02), but not in control group. The data suggest that ineffective inhibitory control of sensory processing is characteristic for retarded children with autism and may reflect excitation/inhibition imbalance in this clinical group. (C) 2008 Elsevier Ireland Ltd. All rights reserved.
C1 [Orekhova, Elena V.; Elam, Mikael] Sahlgrens Univ Hosp, Dept Clin Neurophysiol, S-41345 Gothenburg, Sweden.
[Stroganova, Tatiana A.] Russian Acad Educ, Inst Psychol, Moscow 125009, Russia.
[Stroganova, Tatiana A.; Prokofyev, Andrey O.] Moscow Univ Psychol & Educ, Moscow 103051, Russia.
[Nygren, Gudrun; Gillberg, Cristopher] Sahlgrens Univ Hosp, Dept Child & Adolescent Psychiat, Gothenburg 41345, Sweden.
RP Orekhova, EV (reprint author), Sahlgrens Univ Hosp, Dept Clin Neurophysiol, S-41345 Gothenburg, Sweden.
EM elena@neuro.gu.se
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NR 29
TC 39
Z9 40
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0304-3940
J9 NEUROSCI LETT
JI Neurosci. Lett.
PD MAR 28
PY 2008
VL 434
IS 2
BP 218
EP 223
DI 10.1016/j.neulet.2008.01.066
PG 6
WC Neurosciences
SC Neurosciences & Neurology
GA 288EC
UT WOS:000254968000013
PM 18313850
ER
PT J
AU Kulesza, RJ
Mangunay, K
AF Kulesza, Randy J.
Mangunay, Kathleen
TI Morphological features of the medial superior olive in autism
SO BRAIN RESEARCH
LA English
DT Article
DE hearing; superior olivary complex; morphometry
ID AUDITORY BRAIN-STEM; INFERIOR COLLICULUS; COCHLEAR NUCLEUS; COMPLEX;
CHILDREN; ORGANIZATION; PERCEPTION; RESPONSES; HEARING; SPEECH
AB Autism is a psychosocial disorder clinically characterized by social difficulties, impairment in communication skills and repetitive behavioral patterns. Despite the increasing reported incidence of autism, the neurobiology of this disorder is poorly understood. However, researchers have uncovered numerous structural anomalies in the brainstem, cerebellum and forebrain of autistic individuals and there is substantial support for the association of hearing deficits with autism. In an effort to discover an anatomical correlate for the functional auditory deficits found in autism, we examined the SOC, a group of brainstem nuclei that function in sound source localization, in post-mortem brain tissue from autistic individuals. The neurons of the medial superior olive (MSO), an SOC nucleus, display a precise geometric organization essential for detection of timing differences between the two ears. We examined the architecture of the MSO in five autistic brains (ages 8 to 32 years) and two age-matched controls (ages 26 and 29 years) and found a significant disruption in the morphology of MSO neurons in autistic brains, involving cell body shape and orientation. The results from this study provide evidence on the cellular level that may help to explain the hearing difficulties associated with autism. (c) 2008 Elsevier B.V. All rights reserved.
C1 [Kulesza, Randy J.; Mangunay, Kathleen] Lake Erie Coll Osteopath Med, Auditory Res Ctr, Erie, PA 16509 USA.
RP Kulesza, RJ (reprint author), Lake Erie Coll Osteopath Med, Auditory Res Ctr, 1858 W Grandview Blvd, Erie, PA 16509 USA.
EM rkulesza@lecom.edu
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NR 34
TC 23
Z9 23
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0006-8993
J9 BRAIN RES
JI Brain Res.
PD MAR 20
PY 2008
VL 1200
BP 132
EP 137
DI 10.1016/j.brainres.2008.01.009
PG 6
WC Neurosciences
SC Neurosciences & Neurology
GA 290BJ
UT WOS:000255097400014
PM 18291353
ER
PT J
AU Moy, SS
Nadler, JJ
Poe, MD
Nonneman, RJ
Young, NB
Koller, BH
Crawley, JN
Duncan, GE
Bodfish, JW
AF Moy, Sheryl S.
Nadler, Jessica J.
Poe, Michele D.
Nonneman, Randal J.
Young, Nancy B.
Koller, Beverly H.
Crawley, Jacqueline N.
Duncan, Gaxy E.
Bodfish, James W.
TI Development of a mouse test for repetitive, restricted behaviors:
Relevance to autism
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Article
DE autism; exploration; olfaction; restricted interests; social preference;
stereotypy
ID NMDA RECEPTOR FUNCTION; FREE-EXPLORATORY PARADIGM; X MENTAL-RETARDATION;
MODIFIED HOLE-BOARD; CORPUS-CALLOSUM; ANIMAL-MODEL; STEREOTYPED
BEHAVIOR; SYNAPTIC PLASTICITY; INBRED STRAINS; GENETIC MODEL
AB Repetitive behavior, a core symptom of autism, encompasses stereotyped responses, restricted interests, and resistance to change. These studies investigated whether different components of the repetitive behavior domain could be modeled in the exploratory hole-board task in mice. Four inbred mouse strains, C57BL/6J, BALB/cByJ, BTBR T(+)tf/J, and FVB/NJ, and mice with reduced expression of Grin1, leading to NMDA receptor hypofunction (NR1(neo/neo) mice), were tested for exploration and preference for olfactory stimuli in an activity chamber with a Whole floor-board. Reduced exploration and high preference for holes located in the corners of the chamber were observed in BALB/cByJ and BTBR T(+)tf/J mice. All inbred strains had initial high preference for a familiar olfactory stimulus (clean cage bedding). BTBR T(+)f/J was the only strain that did not demonstrate a shift in hole preference towards an appetitive olfactory stimulus (cereal or a chocolate chip), following home cage exposure to the food. The NR1(neo/neo) mice showed lower hole selectivity and aberrant olfactory stimulus preference, in comparison to wildtype controls. The results indicate that NR1(neo/neo), mice have repetitive nose poke responses that are less modified by environmental contingencies than responses in wildtype mice. 25-30% of NMDA receptor hypomorphic mice also show self-injurious responses. Findings from the olfactory studies suggest that resistance to change and restricted interests might be modeled in mice by a failure to alter patterns of hole preference following familiarization with an appetitive stimulus, and by high preference persistently demonstrated for one particular olfactory stimulus. Further work is required to determine the characteristics of optimal mouse social stimuli in the olfactory hole-board test. (C) 2007 Elsevier B.V. All rights reserved.
C1 [Moy, Sheryl S.; Nadler, Jessica J.; Poe, Michele D.; Nonneman, Randal J.; Young, Nancy B.; Crawley, Jacqueline N.; Duncan, Gaxy E.; Bodfish, James W.] Univ N Carolina, Sch Med, Neurodev Disorders Res Ctr, Chapel Hill, NC 27599 USA.
[Moy, Sheryl S.; Crawley, Jacqueline N.; Duncan, Gaxy E.; Bodfish, James W.] Univ N Carolina, Sch Med, Dept Psychiat, Chapel Hill, NC 27599 USA.
[Nadler, Jessica J.; Koller, Beverly H.] Univ N Carolina, Sch Med, Dept Genet, Chapel Hill, NC 27599 USA.
[Nadler, Jessica J.; Koller, Beverly H.] NIMH, Intramural Res Program, Lab Behav Neurosci, Bethesda, MD 20892 USA.
RP Moy, SS (reprint author), Univ N Carolina, Sch Med, Neurodev Disorders Res Ctr, CB 7146, Chapel Hill, NC 27599 USA.
EM ssmoy@med.unc.edu
RI Poe, Michele/K-6615-2012
OI Poe, Michele/0000-0001-9693-3638
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NR 85
TC 78
Z9 78
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD MAR 17
PY 2008
VL 188
IS 1
BP 178
EP 194
DI 10.1016/j.bbr.2007.10.029
PG 17
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 266PK
UT WOS:000253448200018
PM 18068825
ER
PT J
AU Downs, JS
de Bruin, WB
Fischhoff, B
AF Downs, Julie S.
de Bruin, Waendi Bruine
Fischhoff, Baruch
TI Parents' vaccination comprehension and decisions
SO VACCINE
LA English
DT Article
DE decision making; parents; vaccines; risk communication;
measles-mumps-rubella; autism
ID RISK-COMMUNICATION; RUBELLA VACCINE; MMR VACCINATION; MENTAL MODELS;
PERTUSSIS VACCINATION; MEASLES OUTBREAK; SAFETY CONCERNS; NO EVIDENCE;
MUMPS; IMMUNIZATION
AB We report on 30 in-depth mental models interviews with parents discussing vaccination for their children, both in general terms and in response to communications drawn from sources supporting and opposing vaccines. We found that even parents favourable to vaccination can be confused by the ongoing debate, leading them to question their choices. Many parents lack basic knowledge of how vaccines work, and do not find the standard information provided to them to be particularly helpful in explaining it. Those with the greatest need to know about vaccination seem most vulnerable to confusing information. Opportunities for education may be missed if paediatricians do not appreciate parents' specific information needs. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Downs, Julie S.; de Bruin, Waendi Bruine; Fischhoff, Baruch] Carnegie Mellon Univ, Dept Social & Decis Sci, Pittsburgh, PA 15213 USA.
RP Downs, JS (reprint author), Carnegie Mellon Univ, Dept Social & Decis Sci, Pittsburgh, PA 15213 USA.
EM downs@cmu.edu
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NR 78
TC 45
Z9 45
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 0264-410X
J9 VACCINE
JI Vaccine
PD MAR 17
PY 2008
VL 26
IS 12
BP 1595
EP 1607
DI 10.1016/j.vaccine.2008.01.011
PG 13
WC Immunology; Medicine, Research & Experimental
SC Immunology; Research & Experimental Medicine
GA 282UL
UT WOS:000254592600010
PM 18295940
ER
PT J
AU Casanova, MF
Konkachbaev, AI
Switala, AE
Elmaghraby, AS
AF Casanova, Manuel F.
Konkachbaev, Anouar I.
Switala, Andrew E.
Elmaghraby, Adel S.
TI Recursive trace line method for detecting myelinated bundles: A
comparison study with pyramidal cell arrays
SO JOURNAL OF NEUROSCIENCE METHODS
LA English
DT Article
DE computerized image analysis; minicolumns; myelin; neocortex; pyramidal
cells
ID DOUBLE BOUQUET CELL; HUMAN AUDITORY-CORTEX; CEREBRAL-CORTEX;
VISUAL-CORTEX; QUANTITATIVE-ANALYSIS; DENDRITE BUNDLES; ORGANIZATION;
NEURONS; MONKEY; NEOCORTEX
AB Minicolumns are thought to be the smallest cortical modules within the hierarchical organization of the isocortex. Several reports suggest alterations in minicolumnar morphometry may be involved in psychiatric disorders such as autism, dyslexia, and schizophrenia. Thus far anatomical studies of minicolumns have primarily relied on measurements of pyramidal cell arrays. This study expands on a recursive trace line segmentation method used to define morphometric measures for myelinated axon bundles. The results were compared against those of pyramidal cell arrays derived from immediately adjacent serial sections. Width estimates based on cell somas and myelinated axon bundles were highly correlated (r = 0.9888). Histograms of signal intensity using the recursive trace line method produced expected features of myeloarchitectonics; that is, bundles of Meynert and intervening interradiary plexus. The close correspondence of derived values for myelinated axon bundles and pyramidal cell arrays suggests their participation and interaction within the same modular arrangement of the isocortex. (c) 2007 Elsevier B.V. All rights reserved.
C1 [Casanova, Manuel F.; Switala, Andrew E.] Univ Louisville, Dept Psychiat, Louisville, KY 40202 USA.
[Konkachbaev, Anouar I.; Elmaghraby, Adel S.] Univ Louisville, Dept Comp Sci & Comp Engn, Louisville, KY 40292 USA.
RP Casanova, MF (reprint author), Univ Louisville, Dept Psychiat, 500 S Preston St,Bldg 55-A,Suite 210, Louisville, KY 40202 USA.
EM m0casa02@Louisville.edu
RI Elmaghraby, Adel/B-3353-2014
OI Elmaghraby, Adel/0000-0001-5274-8596
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NR 41
TC 6
Z9 6
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0165-0270
J9 J NEUROSCI METH
JI J. Neurosci. Methods
PD MAR 15
PY 2008
VL 168
IS 2
BP 367
EP 372
DI 10.1016/j.jneumeth.2007.10.024
PG 6
WC Biochemical Research Methods; Neurosciences
SC Biochemistry & Molecular Biology; Neurosciences & Neurology
GA 271YK
UT WOS:000253824400010
PM 18192023
ER
PT J
AU Dolen, G
Bear, MF
AF Dolen, Gul
Bear, Mark F.
TI Role for metabotropic glutamate receptor 5 (mGluR5) in the pathogenesis
of fragile X syndrome
SO JOURNAL OF PHYSIOLOGY-LONDON
LA English
DT Article; Proceedings Paper
CT Symposium on Synaptic Plasticity
CY NOV 02, 2007
CL San Diego, CA
ID LONG-TERM POTENTIATION; OCULAR DOMINANCE PLASTICITY; MENTAL-RETARDATION
PROTEIN; FREELY MOVING RATS; GLUTAMATE RECEPTORS; DENDRITIC SPINES;
VISUAL-CORTEX; MOUSE MODEL; SYNAPTIC PLASTICITY; DENTATE GYRUS
AB Metabotropic glutamate receptors (mGluRs) have been implicated in a diverse variety of neuronal functions. Studies reviewed here indicate that exaggerated signalling through mGluR5 can account for multiple cognitive and syndromic features of fragile X syndrome, the most common inherited form of mental retardation and autism. Since a reduction of mGluR5 signalling can reverse fragile X phenotypes, these studies provide a compelling rationale for the use of mGluR5 antagonists for the treatment of fragile X and related disorders.
C1 [Dolen, Gul; Bear, Mark F.] MIT, Howard Hughes Med Inst, Picower Inst Learning & Memory, Dept Brain & Cognit Sci, Cambridge, MA 02139 USA.
[Dolen, Gul] Brown Med Sch, Providence, RI USA.
RP Bear, MF (reprint author), MIT, Howard Hughes Med Inst, Picower Inst Learning & Memory, Dept Brain & Cognit Sci, 77 Massachusetts Ave, Cambridge, MA 02139 USA.
EM mbear@mit.edu
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NR 48
TC 125
Z9 126
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0022-3751
J9 J PHYSIOL-LONDON
JI J. Physiol.-London
PD MAR 15
PY 2008
VL 586
IS 6
BP 1503
EP 1508
DI 10.1113/jphysiol.2008.150722
PG 6
WC Neurosciences; Physiology
SC Neurosciences & Neurology; Physiology
GA 278IA
UT WOS:000254277300011
PM 18202092
ER
PT J
AU Lee, JE
Hsu, D
Hsu, M
Alexander, AL
DuBray, MB
Froehlich, AL
Lu, JK
Bigler, ED
Lainhart, JE
AF Lee, Jee Eun
Hsu, David
Hsu, Murielle
Alexander, Andrew L.
DuBray, Molly B.
Froehlich, Alyson L.
Lu, Jeffrey K.
Bigler, Erin D.
Lainhart, Janet E.
TI Structural underconnectivity in autism: A neural network method for
diffusion tensor tractography
SO NEUROLOGY
LA English
DT Meeting Abstract
CT 6th Annual Meeting of the American-Academy-of-Neurology
CY APR 12-19, 2008
CL Chicago, IL
SP Amer Acad Neurol
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0028-3878
J9 NEUROLOGY
JI Neurology
PD MAR 11
PY 2008
VL 70
IS 11
SU 1
BP A2
EP A2
PG 1
WC Clinical Neurology
SC Neurosciences & Neurology
GA 319XB
UT WOS:000257197200006
ER
PT J
AU Levesque, CA
AF Levesque, Claire A.
TI Behavioral manifestations of autism in adults
SO NEUROLOGY
LA English
DT Meeting Abstract
CT 6th Annual Meeting of the American-Academy-of-Neurology
CY APR 12-19, 2008
CL Chicago, IL
SP Amer Acad Neurol
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0028-3878
J9 NEUROLOGY
JI Neurology
PD MAR 11
PY 2008
VL 70
IS 11
SU 1
BP A3
EP A3
PG 1
WC Clinical Neurology
SC Neurosciences & Neurology
GA 319XB
UT WOS:000257197200009
ER
PT J
AU Newman, LK
Beversdorf, D
AF Newman, Loriana K.
Beversdorf, David
TI Gender specificity of prenatal stress effects in autism
SO NEUROLOGY
LA English
DT Meeting Abstract
CT 6th Annual Meeting of the American-Academy-of-Neurology
CY APR 12-19, 2008
CL Chicago, IL
SP Amer Acad Neurol
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0028-3878
J9 NEUROLOGY
JI Neurology
PD MAR 11
PY 2008
VL 70
IS 11
SU 1
BP A2
EP A2
PG 1
WC Clinical Neurology
SC Neurosciences & Neurology
GA 319XB
UT WOS:000257197200007
ER
PT J
AU Scaduto, MJ
White, CA
Saklayen, SS
Narayanan, A
Beversdorf, D
AF Scaduto, Mary J.
White, Catherine A.
Saklayen, Sanjida S.
Narayanan, Ananth
Beversdorf, David
TI The effect of propranolol on word fluency in autism spectrum disorder
SO NEUROLOGY
LA English
DT Meeting Abstract
CT 6th Annual Meeting of the American-Academy-of-Neurology
CY APR 12-19, 2008
CL Chicago, IL
SP Amer Acad Neurol
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0028-3878
J9 NEUROLOGY
JI Neurology
PD MAR 11
PY 2008
VL 70
IS 11
SU 1
BP A1
EP A1
PG 1
WC Clinical Neurology
SC Neurosciences & Neurology
GA 319XB
UT WOS:000257197200002
ER
PT J
AU Serajee, F
Zhong, HL
Huq, A
AF Serajee, Fatema
Zhong, Hailang
Huq, Ahm
TI Y chromosome effect in autism
SO NEUROLOGY
LA English
DT Meeting Abstract
CT 6th Annual Meeting of the American-Academy-of-Neurology
CY APR 12-19, 2008
CL Chicago, IL
SP Amer Acad Neurol
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0028-3878
J9 NEUROLOGY
JI Neurology
PD MAR 11
PY 2008
VL 70
IS 11
SU 1
BP A1
EP A2
PG 2
WC Clinical Neurology
SC Neurosciences & Neurology
GA 319XB
UT WOS:000257197200004
ER
PT J
AU Serajee, F
Zhong, HL
Huq, A
AF Serajee, Fatema
Zhong, Hailang
Huq, Ahm
TI Mitochondrial haplogroups in autism
SO NEUROLOGY
LA English
DT Meeting Abstract
CT 6th Annual Meeting of the American-Academy-of-Neurology
CY APR 12-19, 2008
CL Chicago, IL
SP Amer Acad Neurol
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0028-3878
J9 NEUROLOGY
JI Neurology
PD MAR 11
PY 2008
VL 70
IS 11
SU 1
BP A3
EP A3
PG 1
WC Clinical Neurology
SC Neurosciences & Neurology
GA 319XB
UT WOS:000257197200012
ER
PT J
AU [Anonymous]
AF [Anonymous]
TI Can autism be a mitochondrial desease?
SO NEW SCIENTIST
LA English
DT News Item
NR 0
TC 0
Z9 0
PU REED BUSINESS INFORMATION LTD
PI SUTTON
PA QUADRANT HOUSE THE QUADRANT, SUTTON SM2 5AS, SURREY, ENGLAND
SN 0262-4079
J9 NEW SCI
JI New Sci.
PD MAR 8
PY 2008
VL 197
IS 2646
BP 9
EP 9
PG 1
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 275LE
UT WOS:000254072100004
ER
PT J
AU Brune, CW
Korvatska, E
Allen-Brady, K
Cook, EH
Dawson, G
Devlin, B
Estes, A
Hennelly, M
Hyman, SL
McMahon, WM
Munson, J
Rodier, PM
Schellenberg, GD
Stodgell, CJ
Coon, H
AF Brune, Camille W.
Korvatska, Elena
Allen-Brady, Kristina
Cook, Edwin H., Jr.
Dawson, Geraldine
Devlin, Bernie
Estes, Annette
Hennelly, Meghann
Hyman, Susan L.
McMahon, William M.
Munson, Jeffrey
Rodier, Patricia M.
Schellenberg, Gerard D.
Stodgell, Christopher J.
Coon, Hilary
TI Heterogeneous association between engrailed-2 and autism in the CPEA
network
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE autism spectrum disorder; Engrailed-2 (EN2); genetic association
ID FUNCTIONAL MAGNETIC-RESONANCE; HOMEOBOX-TRANSCRIPTION-FACTOR;
FAMILY-BASED ASSOCIATION; SPECTRUM DISORDERS; CEREBELLAR DEVELOPMENT;
SEROTONIN TRANSPORTER; SUSCEPTIBILITY LOCUS; GENOMEWIDE SCREEN;
INFANTILE-AUTISM; GENERAL-CLASS
AB Autism is a neurodevelopmental disorder characterized by an early onset of abnormal social, communicative, and repetitive behavior. Engrailed-2 (EN2) was identified as an autism candidate gene because its influence on cerebellar development in mice parallels neurodevelopmental abnormalities seen in individuals with autism. Studies investigating association between markers at EN2 (chr7q36), a location associated with language disorders, and autism reveal mixed findings. Two positive reports revealed association with two intronic SNPs. Since the associated SNPs were in high linkage disequilibrium and shared similar minor allele frequencies, we chose to test whether one of the SNPs (rs1861972) was associated with autism in three recruiting sites from the NIH Collaborative Programs of Excellence in Autism (CPEA) network. A recessive model revealed significant association with broad autism spectrum disorder. Site specific analyses indicated differential allele transmission by site, despite similar ethnicity, and parental genotypes, suggesting the SNP may contribute to various risk haplotypes. No significant association with autism was found under an additive model for either a broad (autism spectrum disorder) or a narrow (autistic disorder) diagnostic group. Although our findings were not as robust as the previous studies, they suggest that rs1861972 may influence the risk for autism spectrum disorders. Future studies investigating EN2 should consider how the association of variants in this gene with autism could be influenced by differences in phenotype and possible interactions with genotypes at other autism candidate genes. Published 2007 Wiley-Liss, Inc.
C1 [Allen-Brady, Kristina; McMahon, William M.; Coon, Hilary] Univ Utah, Div Child & Adolescent Psychiat, Dept Psychiat, Salt Lake City, UT 84108 USA.
[Brune, Camille W.; Cook, Edwin H., Jr.] Univ Illinois, Dept Psychiat, Inst Juvenile Res, Chicago, IL USA.
[Korvatska, Elena; Schellenberg, Gerard D.] Univ Washington, Vet Affairs Med Ctr, Dept Med, Seattle, WA 98195 USA.
[Korvatska, Elena; Schellenberg, Gerard D.] Univ Washington, Vet Affairs Med Ctr, Dept Neurol & Pharmacol, Seattle, WA 98195 USA.
[Dawson, Geraldine; Munson, Jeffrey] Univ Washington, Dept Psychol, Seattle, WA 98195 USA.
[Dawson, Geraldine; Estes, Annette] Univ Washington, Dept Psychiat & Behav Sci, Seattle, WA 98195 USA.
[Devlin, Bernie] Univ Pittsburgh, Dept Psychiat, Pittsburgh, PA USA.
[Hennelly, Meghann] Univ Chicago, Dept Med, Chicago, IL 60637 USA.
[Hyman, Susan L.] Univ Rochester, Med Ctr, Dept Pediat, Rochester, NY 14642 USA.
[Rodier, Patricia M.] Univ Rochester, Med Ctr, Dept OB GYN, Rochester, NY 14642 USA.
RP Coon, H (reprint author), Univ Utah, Div Child & Adolescent Psychiat, Dept Psychiat, 421 Wakara Way,Suite 143, Salt Lake City, UT 84108 USA.
EM hilary@bach.med.utah.edu
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NR 56
TC 18
Z9 19
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD MAR 5
PY 2008
VL 147B
IS 2
BP 187
EP 193
DI 10.1002/ajmg.b.30585
PG 7
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 271OS
UT WOS:000253798700006
PM 17948868
ER
PT J
AU Li, H
Li, Y
Shao, J
Li, R
Qin, YF
Xie, CH
Zhao, ZY
AF Li, Hui
Li, Yun
Shao, Jie
Li, Rong
Qin, Yufeng
Xie, Chunhong
Zhao, Zhengyan
TI The association analysis of RELN and GRM8 genes with autistic spectrum
disorder in Chinese Han population
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE autistic spectrum disorder; RELN; GRM8; SNP; linkage disequilibrium
ID REELIN GENE; CHROMOSOME 7Q; GLUTAMATE RECEPTORS; GENOMIC SCREEN; CGG
REPEAT; LINKAGE; SUSCEPTIBILITY; POLYMORPHISMS; ALLELES; REGION
AB The region on chromosome 7q stands out as the region of suggestive linkage to aetiology of autism with the greatest concordance in many independent genome-wide scans. RELN and GRM8, the two genes selected in this study, are located within this region. The protein products of both genes are considered to play a pivotal role in the development of the central nervous system. In addition, biochemical and neuroanatomical data indicated that RELN and GRM8 genes are likely involved in the pathogenesis of autistic disorder. Therefore, both RELN and GRM8 genes are considered to be not only the positional but also the functional candidate genes to autism for association research. In this study, we genotyped 12 single nucleotide polymorphisms (SNPs) located within the RELN and GRM8 genes in 213 children with autistic spectrum disorder (ASD) and 160 controls. A significant genetic association between SNP2 (located in intron 59 of RELN) and ASD was observed, and the log-additive model was accepted as the best inheritance model fitting this data (OR: 0.72, 95% CI: 0.54-0.97, P = 0.03). Haplotype-specific association analysis revealed that the result was consistent with the individual SNP study; the combination of SNP1/SNP2/SNP3/SNP4 which are in strong linkage disequilibrium (LD) (D' > 0.75) showed significant association with ASD (P = 0.027). Neither the single SNP nor the haplotype analysis showed significant association between ASD and the markers of GRM8 gene. Hence, our study suggested the possible involvement of RELN gene in the susceptibility to ASD. Future replications are warranted before definitive conclusion can be drawn. (c) 2007 Wiley-Liss, Inc.
C1 [Shao, Jie; Li, Rong; Qin, Yufeng; Zhao, Zhengyan] Zhejiang Univ, Sch Med, Childrens Hosp, Dept Pediat Hlth Care, Hangzhou 310003, Peoples R China.
[Li, Hui] Zhejiang Univ, Sch Med, Childrens Hosp, Dept Cent Lab, Hangzhou, Peoples R China.
[Li, Yun] Zhejiang Univ, Sch Med, Childrens Hosp, Dept Endocrinol, Hangzhou, Peoples R China.
[Xie, Chunhong] Zhejiang Univ, Sch Med, Childrens Hosp, Dept Cardiol, Hangzhou, Peoples R China.
RP Zhao, ZY (reprint author), Zhejiang Univ, Sch Med, Childrens Hosp, Dept Pediat Hlth Care, Hangzhou 310003, Peoples R China.
EM zhaozy@zju.edu.cn
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NR 47
TC 25
Z9 29
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD MAR 5
PY 2008
VL 147B
IS 2
BP 194
EP 200
DI 10.1002/ajmg.b.30584
PG 7
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 271OS
UT WOS:000253798700007
PM 17955477
ER
PT J
AU DeLorey, TM
Sahbaie, P
Hashemi, E
Homanics, GE
Clark, JD
AF DeLorey, Timothy M.
Sahbaie, Peyman
Hashemi, Ezzat
Homanics, Gregg E.
Clark, J. David
TI Gabrb3 gene deficient mice exhibit impaired social and exploratory
behaviors, deficits in non-selective attention and hypoplasia of
cerebellar vermal lobules: A potential model of autism spectrum disorder
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Article
DE social behavior; cerebellar vermis; exploration; non-selective
attention; nest building
ID SUBUNIT KNOCKOUT MICE; GABA(A) RECEPTOR; ANGELMAN-SYNDROME; BETA(3)
SUBUNIT; LOCUS-COERULEUS; NITRIC-OXIDE; FOLLOW-UP; LACKING; CHILDREN;
ABNORMALITIES
AB Objective: GABA(A) receptors play an important regulatory role in the developmental events leading to the formation of complex neuronal networks and to the behaviors they govern. The primary aim of this study was to assess whether gabrb3 gene deficient (gabrb3(-/-)) mice exhibit abnormal social behavior, a core deficit associated with autism spectrum disorder.
Methods: Social and exploratory behaviors along with non-selective attention were assessed in gabrb3(-/-), littermates (gabrb3(-/-)) and progenitor strains, C57BL/6J and 129/SvJ. In addition, semi-quantitative assessments of the size of cerebellar vermal lobules were performed on gabrb3(-/-) and gabrb3-/- mice.
Results: Relative to controls, gabrb3(-/-) mice exhibited significant deficits in activities related to social behavior including sociability, social novelty and nesting. In addition, gabrb3(-/-) mice also exhibited differences in exploratory behavior compared to controls, as well as reductions in the frequency and duration of rearing episodes, suggested as being an index of non-selective attention. Gabrb3(-/-) mice also displayed significant hypoplasia of the cerebellar vermis compared to gabrb3(-/-) mice.
Conclusions: The observed behavioral deficits, especially regarding social behaviors, strengthens the face validity of the gabrb3 gene deficient mouse as being a model of autism spectrum disorder. (C) 2007 Elsevier B.V. All rights reserved.
C1 [DeLorey, Timothy M.; Sahbaie, Peyman; Hashemi, Ezzat] Mol Res Inst, Palo Alto, CA 94303 USA.
[Homanics, Gregg E.] Univ Pittsburgh, Dept Anesthesiol & Pharmacol, Pittsburgh, PA 15261 USA.
[Clark, J. David] Vet Affairs Palo Alto Hlth Care Syst, Palo Alto, CA 94304 USA.
[Clark, J. David] Stanford Univ, Sch Med, Dept Anesthesiol, Stanford, CA 94305 USA.
RP DeLorey, TM (reprint author), Mol Res Inst, 1000 Elwell Court,Suite 105, Palo Alto, CA 94303 USA.
EM tim@molres.org
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NR 79
TC 78
Z9 78
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD MAR 5
PY 2008
VL 187
IS 2
BP 207
EP 220
DI 10.1016/j.bbr.2007.09.009
PG 14
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 259BX
UT WOS:000252915200001
PM 17983671
ER
PT J
AU Scattoni, ML
McFarlane, HG
Zhodzishsky, V
Caldwell, HK
Young, WS
Ricceri, L
Crawley, JN
AF Scattoni, M. L.
McFarlane, H. G.
Zhodzishsky, V.
Caldwell, H. K.
Young, W. S.
Ricceri, L.
Crawley, J. N.
TI Reduced ultrasonic vocalizations in vasopressin 1b knockout mice
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Article
DE social recognition; female-female mouse interactions; maternal
potentiation; nest odor orientation; autistic spectrum disorders; mouse
models
ID V-1B RECEPTOR ANTAGONIST; RAT PUPS; SOCIAL RECOGNITION;
AGGRESSIVE-BEHAVIOR; MATERNAL POTENTIATION; MUS-MUSCULUS; OXYTOCIN;
AUTISM; MOUSE; SSR149415
AB The neuropeptides oxytocin and vasopressin have been implicated in rodent social and affiliative behaviors, including social bonding, parental care, social recognition, social memory, vocalizations, territoriality, and aggression, as well as components of human social behaviors and the etiology of autism. Previous investigations of mice with various manipulations of the oxytocin and vasopressin systems reported unusual levels of ultrasonic vocalizations in social settings. We employed a vasopressin 1b receptor (Avpr1b) knockout mouse to evaluate the role of the vasopressin 1b receptor subtype in the emission of ultrasonic vocalizations in adult and infant mice. Avpr1b null mutant female mice emitted fewer ultrasonic vocalizations, and their vocalizations were generally at lower frequencies, during a resident-intruder test. Avpr1b null mutant pups emitted ultrasonic vocalizations similar to heterozygote and wildtype littermates when separated from the nest on postnatal days 3, 6, 9, and 12. However, maternal potentiation of ultrasonic vocalizations in Avpr1b null and heterozygote mutants was absent, when tested at postnatal day 9. These results indicate that Avpr1b null mutant mice are impaired in the modulation of ultrasonic vocalizations within different social contexts at infant and adult ages. Published by Elsevier B.V.
C1 [Scattoni, M. L.; McFarlane, H. G.; Zhodzishsky, V.; Crawley, J. N.] NIMH, Lab Behav Neurosci, Bethesda, MD 20892 USA.
[Scattoni, M. L.; Ricceri, L.] Ist Super Sanita, Behav Neurosci Sect, Dept Cell Biol & Neurosci, I-00161 Rome, Italy.
[McFarlane, H. G.] Kenyon Coll, Dept Psychol, Gambier, OH 43022 USA.
[Caldwell, H. K.; Young, W. S.] NIMH, Sect Neural Gene Express, NIH, DHHS, Bethesda, MD 20892 USA.
RP Scattoni, ML (reprint author), NIMH, Lab Behav Neurosci, Bldg 35 Room 1C-909, Bethesda, MD 20892 USA.
EM scattonim@mail.nih.gov
RI Young, W/A-9333-2009
OI Young, W/0000-0001-6614-5112
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NR 58
TC 51
Z9 51
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD MAR 5
PY 2008
VL 187
IS 2
BP 371
EP 378
DI 10.1016/j.bbr.2007.09.034
PG 8
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 259BX
UT WOS:000252915200019
PM 18005969
ER
PT J
AU Glasson, EJ
MacDermott, S
Dixon, G
Cook, H
Chauvel, P
Maley-Berg, A
Wray, J
AF Glasson, Emma J.
MacDermott, Sarah
Dixon, Glenys
Cook, Hugh
Chauvel, Peter
Maley-Berg, Alana
Wray, John
TI Management of assessments and diagnoses for children with autism
spectrum disorders: the Western Australian model
SO MEDICAL JOURNAL OF AUSTRALIA
LA English
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDERS
AB Autism spectrum disorders (ASDs) are severe developmental conditions that require specialised intervention and lifelong support.
Recent increases in ASD prevalence have prompted new initiatives in Western Australia to improve the consistency of assessments and to more accurately monitor diagnostic trends within the population.
WA has implemented statewide guidelines for the assessment of ASDs, has developed an open forum for clinicians to discuss issues relating to the assessment process, and supports a statewide register of newly diagnosed cases.
These initiatives have led to improved consistency across assessments, allowed analysis of diagnoses over time, and promoted cohesiveness among autism assessors.
These strategies potentially provide an alternative model for other states and territories that wish to strengthen and assimilate ASD assessments.
C1 [Glasson, Emma J.; Dixon, Glenys] Univ Western Australia, Ctr Child Hlth Res, Telethon Inst Child Hlth Res, Perth, WA 6009, Australia.
[Glasson, Emma J.] Univ Western Australia, Sch Populat Hlth, Perth, WA 6009, Australia.
[MacDermott, Sarah; Wray, John] Western Australian Autism Diagnosticians Forum, Perth, WA, Australia.
[Cook, Hugh] Autism Assoc Western Australia, Perth, WA, Australia.
[Maley-Berg, Alana] Disabil Serv Commiss Western Australia, Perth, WA, Australia.
[Wray, John] Princess Margaret Hosp Children, Child & Adolescent Hlth Serv, State Child Dev Ctr, Perth, WA, Australia.
RP Glasson, EJ (reprint author), Univ Western Australia, Ctr Child Hlth Res, Telethon Inst Child Hlth Res, Perth, WA 6009, Australia.
EM emma.glasson@health.wa.gov.au
RI Glasson, Emma/H-5339-2013
OI Glasson, Emma/0000-0003-3996-9049
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NR 27
TC 14
Z9 14
PU AUSTRALASIAN MED PUBL CO LTD
PI PYRMONT
PA LEVEL 2, 26-32 PYRMONT BRIDGE RD, PYRMONT, NSW 2009, AUSTRALIA
SN 0025-729X
J9 MED J AUSTRALIA
JI Med. J. Aust.
PD MAR 3
PY 2008
VL 188
IS 5
BP 288
EP 291
PG 4
WC Medicine, General & Internal
SC General & Internal Medicine
GA 285WV
UT WOS:000254807800008
PM 18312193
ER
PT J
AU Cannell, JJ
Hollis, BW
AF Cannell, John J.
Hollis, Bruce W.
TI Use of vitamin D in clinical practice
SO ALTERNATIVE MEDICINE REVIEW
LA English
DT Review
ID D DEFICIENCY; D INSUFFICIENCY; HYPOVITAMINOSIS-D; 25-HYDROXYVITAMIN D;
HIGH PREVALENCE; UNITED-STATES; SUN EXPOSURE; SECONDARY
HYPERPARATHYROIDISM; SERUM 25-HYDROXYVITAMIN-D; ANTIMICROBIAL ACTIVITY
AB The recent discovery - from a meta-analysis of 18 randomized controlled trials - that supplemental cholecalciferol (vitamin D) significantly reduces all-cause mortality emphasizes the medical, ethical, and legal implications of promptly diagnosing and adequately treating vitamin D deficiency. Not only are such deficiencies common, and probably the rule, vitamin D deficiency is implicated in most of the diseases of civilization. Vitamin D's final metabolic product is a potent, pleiotropic, repair and maintenance, seco-steroid hormone that targets more than 200 human genes in a wide variety of tissues, meaning it has as many mechanisms of action as genes it targets. One of the most important genes vitamin D up-regulates is for cathelicidin, a naturally occurring broad-spectrum antibiotic. Natural vitamin D levels, those found in humans living in a sun-rich environment, are between 40-70 ng/mL, levels obtained by few modern humans. Assessing serum 25-hydroxy-vitamin D (25(OH)D) is the only way to make the diagnosis and to assure treatment is adequate and safe. Three treatment modalities exist for vitamin D deficiency: sunlight, artificial ultraviolet B (UVB) radiation, and vitamin D supplementation. Treatment of vitamin D deficiency in otherwise healthy patients with 2,000-7,000 IU vitamin D-3 per day should be sufficient to maintain year-round 25(OH)D levels between 40-70 ng/mL. In those with serious illnesses associated with vitamin D deficiency, such as cancer, heart disease, multiple sclerosis, diabetes, autism, and a host of other illnesses, doses should be sufficient to maintain year-round 25(OH)D levels between 55-70 ng/mL. Vitamin D-deficient patients with serious illness should not only be supplemented more aggressively than the well, they should have more frequent monitoring of serum 25(OH) D and serum calcium. Vitamin D should always be adjuvant treatment in patients with serious illnesses and never replace standard treatment. Theoretically, pharmacological doses of vitamin D (2,000 IU/kg/day for three days) may produce enough of the naturally occurring antibiotic cathelicidin to cure common viral respiratory infections, such as influenza and the common cold, but such a theory awaits further science.
C1 [Hollis, Bruce W.] Med Univ S Carolina, Charleston, SC 29425 USA.
RP Cannell, JJ (reprint author), 9100 San Gregorio Rd, Atascadero, CA 93422 USA.
EM jjcannell@gmail.com
RI Szaleniec, Maciej/A-1198-2013
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NR 98
TC 65
Z9 70
PU THORNE RESEARCH, INC
PI DOVER
PA PO BOX 25, DOVER, ID 83825 USA
SN 1089-5159
J9 ALTERN MED REV
JI Altern. Med. Rev.
PD MAR
PY 2008
VL 13
IS 1
BP 6
EP 20
PG 15
WC Integrative & Complementary Medicine; Pharmacology & Pharmacy
SC Integrative & Complementary Medicine; Pharmacology & Pharmacy
GA 286RU
UT WOS:000254864500002
PM 18377099
ER
PT J
AU Guardino, CA
AF Guardino, Caroline A.
TI Identification and placement for deaf students with multiple
disabilities. Choosing the path less followed
SO AMERICAN ANNALS OF THE DEAF
LA English
DT Article
ID HEARING-IMPAIRED STUDENTS; HARD-OF-HEARING; CHILDREN
AB STUDENTS WITH a hearing loss often have an additional disability. Although the number of deaf students with multiple disabilities is growing, research on this population has significantly decreased over the past quarter-century. The article reviews the literature on identification and placement of deaf students with multiple disabilities, specifically deaf students with one of the following disabilities: autism, emotional/behavior disorders, attention deficit disorders, or mental retardation. Degree of incidence is also reported, and is compared with statistics from the annual report of the Gallaudet Research Institute (2005). Included in the review are suggestions for future research and implications for professionals in the field of deafness.
C1 [Guardino, Caroline A.] Univ Arizona, Dept Special Educ Rehabil, Tucson, AZ 85721 USA.
[Guardino, Caroline A.] Univ Arizona, Sch Psychol, Tucson, AZ USA.
RP Guardino, CA (reprint author), Univ Arizona, Dept Special Educ Rehabil, Tucson, AZ 85721 USA.
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NR 41
TC 12
Z9 12
PU AMER ANNALS DEAF
PI WASHINGTON
PA FOWLER HALL 409, 800 FLORIDA AVE NE, WASHINGTON, DC 20002 USA
SN 0002-726X
J9 AM ANN DEAF
JI Am. Ann. Deaf
PD SPR
PY 2008
VL 153
IS 1
BP 55
EP 64
PG 10
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 314OL
UT WOS:000256818600006
PM 18619069
ER
PT J
AU Nakamine, A
Ouchanov, L
Jimenez, P
Manghi, ER
Esquivel, M
Monge, S
Fallas, M
Burton, BK
Szomju, B
Elsea, SH
Marshall, CR
Scherer, SW
McInnes, A
AF Nakamine, Alisa
Ouchanov, Leonid
Jimenez, Patricia
Manghi, Elina R.
Esquivel, Marcela
Monge, Silvia
Fallas, Marietha
Burton, Barbara K.
Szomju, Barbara
Elsea, Sarah H.
Marshall, Christian R.
Scherer, Stephen W.
McInnes, Alison
TI Duplication of 17(p11.2p11.2) in a male child with autism and severe
language delay
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Article
DE autism; cytogenetics; 17(p11.2p11.2); gene expression; language delay
ID SMITH-MAGENIS-SYNDROME; 17P11.2; SPECTRUM; RAI1; REARRANGEMENTS;
DISORDERS; MUTATIONS; GENOME; INDIVIDUALS; DELETIONS
AB Duplications of 17(p11.2p11.2) have been associated with various behavioral manifestations including attention deficits, obsessive-compulsive symptoms, autistic traits, and language delay. We are conducting a genetic study of autism and are screening all cases for submicroscopic chromosomal abnormalities, in addition to standard karyotyping, and fragile X testing. Using array-based comparative genomic hybridization analysis of data from the Affymetrix Gene-Chip(R) Human Mapping Array set, we detected a duplication of similar to 3.3 Mb on chromosome 17p11.2 in a male child with autism and severe expressive language delay. The duplication was confirmed by measuring the copy number of genomic DNA using quantitative polymerase chain reaction. Gene expression analyses revealed increased expression of three candidate genes for the Smith-Magenis neurobehavioral phenotype, RAI1, DRG2, and RASD1, in transformed lymphocytes from Case 81A, suggesting gene dosage effects. Our results add to a growing body of evidence suggesting that duplications of 17(p11.2p11.2) result in language delay as well as autism and related phenotypes. As Smith-Magenis syndrome is also associated with language delay, a gene involved in acquisition of language may lie within this interval. Whether a parent of origin effect, gender of the case, the presence of allelic variation, or changes in expression of genes outside the breakpoints influence the resultant phenotype remains to be determined. (C) 2007 Wiley-Liss, Inc.
C1 [Nakamine, Alisa; Ouchanov, Leonid; McInnes, Alison] Mt Sinai Sch Med, Dept Psychiat, New York, NY USA.
[Jimenez, Patricia; Esquivel, Marcela; Monge, Silvia; Fallas, Marietha] Hosp Nacl Ninos Dr Carlos Saenz Herrera, CCSs, Child Dev & Behav Unit, San Jose, Costa Rica.
[Manghi, Elina R.] Univ Illinois, Chicago, IL USA.
[Burton, Barbara K.] Northwestern Univ, Childrens Mem Hosp, Feinberg Sch Med, Div Genet, Chicago, IL 60614 USA.
[Szomju, Barbara; Elsea, Sarah H.] Virginia Commonwealth Univ, Dept Pediat, Richmond, VA USA.
[Szomju, Barbara; Elsea, Sarah H.] Virginia Commonwealth Univ, Dept Human Genet, Richmond, VA USA.
[Marshall, Christian R.; Scherer, Stephen W.] Univ Toronto, Dept Mol & Med Genet, Program Genet & Genom Biol, Hosp Sick Children, Toronto, ON, Canada.
[McInnes, Alison] Mt Sinai Sch Med, Dept Human Genet, New York, NY USA.
RP McInnes, A (reprint author), 1 Gustave L Levy Pl,Box 1229, New York, NY 10029 USA.
EM alison.mcinnes@mssm.edu
RI Howe, Jennifer/I-9013-2012; Scherer, Stephen /B-3785-2013
OI Scherer, Stephen /0000-0002-8326-1999
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NR 34
TC 12
Z9 12
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD MAR 1
PY 2008
VL 146A
IS 5
BP 636
EP 643
DI 10.1002/ajmg.a.31636
PG 8
WC Genetics & Heredity
SC Genetics & Heredity
GA 269LJ
UT WOS:000253650200013
PM 17334992
ER
PT J
AU Stiefel, I
Shields, AK
Swain, MA
Innes, WR
AF Stiefel, Ingeborg
Shields, Alexandra K.
Swain, Michelle A.
Innes, Waverney R.
TI Asperger's coming out of our ears: Making sense of a modern epidemic
SO AUSTRALIAN AND NEW ZEALAND JOURNAL OF FAMILY THERAPY
LA English
DT Article
DE Asperger's syndrome; family context; autism spectrum disorder; family
ID TOTAL POPULATION; AUTISM; CHILDREN
AB Asperger's syndrome (AS) as a diagnostic category has gained enormous popularity and the label is being applied with increasing liberality. While greater awareness of the disorder may be of advantage to certain children and their families, we argue that the over-inclusive use of the diagnosis may also lead to professional conflicts and ethical dilemmas. Perhaps most concerning of these is that diagnostic 'generosity' may exclude children from treatments they would have received if an alternative formulation had been considered. We present clinical scenarios, with special consideration of contextual and intergenerational influences on the children's early lives, and offer alternative conceptualisations. We argue that the new 'epidemic' of Asperger's should not be allowed to compromise our professional standards. Assessment should always include a detailed family and developmental history. Finally, we would be interested in feedback from clinicians working in this field.
C1 [Stiefel, Ingeborg; Shields, Alexandra K.; Swain, Michelle A.; Innes, Waverney R.] Child & Family Hlth Counselling Team, Cent Coast Hlth Serv Gateway, Gosford, NSW 2250, Australia.
RP Stiefel, I (reprint author), Child & Family Hlth Counselling Team, Cent Coast Hlth Serv Gateway, Level 1,POB 361, Gosford, NSW 2250, Australia.
CR (APA) APA, 2000, DIAGN STAT MAN MENT
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NR 19
TC 0
Z9 0
PU AUSTRALIAN ACAD PRESS
PI BOWEN HILLS
PA 32 JEAYS ST, BOWEN HILLS, QLD 4006, AUSTRALIA
SN 0814-723X
J9 AUST N Z J FAM THER
JI Aust. N. Z. J. Fam. Ther.
PD MAR
PY 2008
VL 29
IS 1
BP 1
EP 9
DI 10.1375/anft.29.1.1
PG 9
WC Family Studies
SC Family Studies
GA 318XS
UT WOS:000257126700003
ER
PT J
AU Barnard, L
Muldoon, K
Hasan, R
O'Brien, G
Stewart, M
AF Barnard, Louise
Muldoon, Kevin
Hasan, Reem
O'Brien, Gregory
Stewart, Mary
TI Profiling executive dysfunction in adults with autism and comorbid
learning disability
SO AUTISM
LA English
DT Article
DE autism; executive; functioning
ID INDIVIDUALS; PERFORMANCE; CHILDREN
AB Executive dysfunction is thought to be primary to autism. We examined differences in executive function between 20 adults with autism and learning disability and 23 individuals with learning disabilities outside the autistic spectrum. All participants were matched for chronological age and full-scale IQ, and were given a battery of tasks assessing fluency, planning, set-shifting, inhibition and working memory. Analyses of the individual tasks revealed very few significant differences between the two groups. However, analyses of composite scores derived for each executive domain revealed that the group with autism showed impaired performance on the working memory and planning tests. Together, these two measures were sufficient to classify participants into their diagnostic groups significantly better than would be expected by chance (75% of the autism group; 65% of the control group). Executive impairments were neither universal nor exclusive to the autism group, and we suggest that an alternative cognitive theory may better explain the cognitive profile we found.
C1 [Stewart, Mary] Heriot Watt Univ, Sch Life Sci, Edinburgh EH14 4AS, Midlothian, Scotland.
[Muldoon, Kevin] Royal Victoria Infirm, Newcastle Upon Tyne NE1 4LP, Tyne & Wear, England.
[Barnard, Louise] Univ Leeds, Leeds LS2 9JT, W Yorkshire, England.
[O'Brien, Gregory] Northumbria Univ, Newcastle Upon Tyne NE1 8ST, Tyne & Wear, England.
RP Stewart, M (reprint author), Heriot Watt Univ, Sch Life Sci, Edinburgh EH14 4AS, Midlothian, Scotland.
EM m.e.stewart@hw.ac.uk
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World Health Organization (WHO), 1992, INT CLASS DIS VERS 1
NR 37
TC 22
Z9 22
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAR
PY 2008
VL 12
IS 2
BP 125
EP 141
DI 10.1177/1362361307088486
PG 17
WC Psychology, Developmental
SC Psychology
GA 290MD
UT WOS:000255126000002
PM 18308763
ER
PT J
AU Herrera, G
Alcantud, F
Jordan, R
Blanquer, A
Labajo, G
De Pablo, C
AF Herrera, Gerardo
Alcantud, Francisco
Jordan, Rita
Blanquer, Amparo
Labajo, Gabriel
De Pablo, Cristina
TI Development of symbolic play through the use of virtual reality tools in
children with autistic spectrum disorders
SO AUTISM
LA English
DT Article
DE autism; imagination; pretend play; virtual reality
ID REPRESENTATION; EPIDEMIOLOGY
AB Difficulties in understanding symbolism have been documented as characteristic of autistic spectrum disorders (ASDs). In general, virtual reality (VR) environments offer a set of potential advantages for educational intervention in ASD. In particular, VR offers the advantage, for teaching pretend play and for understanding imagination, of it being possible to show these imaginary transformations explicitly. This article reports two case studies of children with autism (aged 8:6 and 15:7, both male), examining the effectiveness of using a VR tool specifically designed to work on teaching understanding of pretend play. The results, confirmed by independent observers, showed a significant advance in pretend play abilities after the intervention period in both participants, and a high degree of generalization of the acquired teaching in one of them.
C1 [Jordan, Rita] Univ Birmingham, Sch Educ, Birmingham B15 2TT, W Midlands, England.
[Herrera, Gerardo; Alcantud, Francisco] Univ Valencia Estudi Gen, Valencia, Spain.
RP Jordan, R (reprint author), Univ Birmingham, Sch Educ, Birmingham B15 2TT, W Midlands, England.
EM r.r.jordan@bham.ac.uk
RI Alcantud, Francisco/D-8573-2011
OI Alcantud, Francisco/0000-0001-6022-5437
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American Phychiatric Assocation, 2000, DIAGN STAT MAN MENT
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NR 27
TC 20
Z9 22
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAR
PY 2008
VL 12
IS 2
BP 143
EP 157
DI 10.1177/1362361307086657
PG 15
WC Psychology, Developmental
SC Psychology
GA 290MD
UT WOS:000255126000003
PM 18308764
ER
PT J
AU Lopez, B
Leekam, SR
Arts, GRJ
AF Lopez, Beatriz
Leekam, Susan R.
Arts, Gerda R. J.
TI How central is central coherence? Preliminary evidence on the link
between conceptual and perceptual processing in children with autism
SO AUTISM
LA English
DT Article
DE autism; central; coherence theory; face; perception; semantic memory
ID HIGH-FUNCTIONING AUTISM; DEFICIT; INFORMATION; ADULTS; INTERFERENCE;
ADOLESCENTS; CONTEXT; RECALL; WORDS
AB This study aimed to test the assumption drawn from weak central coherence theory that a central cognitive mechanism is responsible for integrating information at both conceptual and perceptual levels. A visual semantic memory task and a face recognition task measuring use of holistic information were administered to 15 children with autism and 16 typically developing children. If there is a central integration mechanism, performance on the two tasks should be positively associated. No relationship was found, however, between the two abilities in the comparison group and, unexpectedly, a strong significant inverse correlation was found in the autism group. Classification data further confirmed this finding and indicated the possibility of the presence of subgroups in autism. The results add to emerging evidence suggesting that central coherence is not a unitary construct.
C1 [Lopez, Beatriz] Univ W England, Sch Psychol, Bristol BS16 1QY, Avon, England.
[Leekam, Susan R.; Arts, Gerda R. J.] Univ Durham, Durham DH1 3HP, England.
RP Lopez, B (reprint author), Univ W England, Sch Psychol, Bristol BS16 1QY, Avon, England.
EM beatriz.lopez@uwe.ac.uk
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American Psychiatric Association, 1987, DIAGN STAT MAN MENT
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NR 37
TC 14
Z9 14
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAR
PY 2008
VL 12
IS 2
BP 159
EP 171
DI 10.1177/1362361307086662
PG 13
WC Psychology, Developmental
SC Psychology
GA 290MD
UT WOS:000255126000004
PM 18308765
ER
PT J
AU Muller, E
Schuler, A
Yates, GB
AF Mueller, Eve
Schuler, Adriana
Yates, Gregory B.
TI Social challenges and supports from the perspective of individuals with
Asperger syndrome and other autism spectrum disabilities
SO AUTISM
LA English
DT Article
DE Asperger syndrome; autism; social challenges; social supports
ID HIGH-FUNCTIONING CHILDREN; LONELINESS
AB The study describes the perspectives of individuals with Asperger syndrome and other autism spectrum disabilities (ASDs) regarding social challenges and supports. Eighteen adults with ASDs were individually interviewed. They were asked to describe their experiences navigating their social worlds, and recommend effective social supports and strategies for improving social connectedness. Qualitative analyses of the interview transcripts revealed a number of common experiences including a profound sense of isolation, difficulty initiating social interactions, challenges relating to communication, longing for greater intimacy, desire to contribute to one's community, and effort to develop greater social/self-awareness. Commonly recommended social supports included external supports (e.g. activities based on shared interests, highly structured or scripted social activities, and small groups or dyads); communication supports (e.g. alternative modes of communication, explicit communication, and instruction in interpreting and using social cues); and self-initiated strategies for handling social anxiety (e.g. creative/improvisational outlets, physical activity, spiritual practice/organized religion, and time spent alone).
C1 [Mueller, Eve] NASDSE, Alexandria, VA USA.
[Schuler, Adriana] San Francisco State Univ, San Francisco, CA USA.
RP Muller, E (reprint author), NASDSE, 1800 Diagonal Rd,Suite 320, Alexandria, VA USA.
EM eve.muller@nasdse.org
CR ATTWOOD T, 1995, ASPERGERS SYNDROME A
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NR 27
TC 38
Z9 38
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAR
PY 2008
VL 12
IS 2
BP 173
EP 190
DI 10.1177/1362361307086664
PG 18
WC Psychology, Developmental
SC Psychology
GA 290MD
UT WOS:000255126000005
PM 18308766
ER
PT J
AU Montiel-Nava, C
Pena, JA
AF Montiel-Nava, Cecilia
Pena, Joaquin A.
TI Epidemiological findings of pervasive developmental disorders in a
Venezuelan study
SO AUTISM
LA English
DT Article
DE autism spectrum disorders; epidemiology; pervasive developmental
disorders; prevalence; Venezuela
ID AUTISTIC SPECTRUM DISORDERS; INFANTILE-AUTISM; PREVALENCE; CHILDREN;
POPULATION; DIAGNOSIS; FRENCH; BRAZIL; AGE
AB The study aims to determine the prevalence of autism spectrum disorders (ASDs) for children receiving services in Maracaibo County, Venezuela. Children aged 3-9 with diagnosis of any ASD were recruited. We ascertained area, referral process, and definitions of ASD for each patient. A total of 430 children were identified, and 76.5 percent were boys. Prevalences were 1.7 per 1000 for all ASD, 1.1 per 1000 for autism, and 0.6 per 1000 for PDD-NOS and Asperger syndrome combined. These prevalences are lower than current reports in the literature. Differences in case-finding methods, diagnostic criteria, and lack of awareness in the general population may have in number of cases identified. An ASD prevalence of 1.7 per 1000 should alert the health and education authorities to the need to reassess the services available for children with these disorders and their families.
C1 [Montiel-Nava, Cecilia; Pena, Joaquin A.] La Univ Zulia, Maracaibo, Venezuela.
RP Montiel-Nava, C (reprint author), Calle 79 3E-31,Sector La Lago, Maracaibo 4002A, Estado Zulia, Venezuela.
EM ceciliamontiel@cantv.net
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 33
TC 13
Z9 13
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAR
PY 2008
VL 12
IS 2
BP 191
EP 202
DI 10.1177/1362361307086663
PG 12
WC Psychology, Developmental
SC Psychology
GA 290MD
UT WOS:000255126000006
PM 18308767
ER
PT J
AU Heaton, P
Williams, K
Cummins, O
Happe, F
AF Heaton, Pamela
Williams, Kerry
Cummins, Omar
Happe, Francesca
TI Autism and pitch processing splinter skills: A group and subgroup
analysis
SO AUTISM
LA English
DT Article
DE ability islands; autism; pitch processing; splinter skills; tone memory
ID SAVANT
AB Autism is characterized by an uneven profile of cognitive abilities and population studies show that approximately 10 percent of diagnosed individuals possess a skill that is significantly better than would be predicted by global IQ. Recent evidence suggests that individuals with autism who possess special skills may represent a distinct genetic group within the autism spectrum. Intellectually high- and low-functioning children and adolescents with autism, together with age- and intelligence-matched comparison participants, completed two experiments that tested pitch discrimination and pitch memory within a visuo-spatial format. The analysis of the data from the studies showed that a subgroup of individuals with autism achieved performance scores that were between four and five standard deviations above the mean for the groups. Unlike comparison participants, their performance appeared to be independent of intelligence, musical training and experience. The findings were interpreted within the context of neuroconstructivist models of typical development and delayed language acquisition characteristic of autism and other neurodevelopmental disorders.
C1 [Heaton, Pamela; Williams, Kerry; Cummins, Omar; Happe, Francesca] Univ London Goldsmiths Coll, Dept Psychol, London SE14 6NW, England.
RP Heaton, P (reprint author), Univ London Goldsmiths Coll, Dept Psychol, London SE14 6NW, England.
EM P.Heaton@gold.ac.uk
RI Happe, Francesca/D-5544-2012
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 24
TC 25
Z9 25
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD MAR
PY 2008
VL 12
IS 2
BP 203
EP 219
DI 10.1177/1362361307085270
PG 17
WC Psychology, Developmental
SC Psychology
GA 290MD
UT WOS:000255126000007
PM 18308768
ER
PT J
AU Bogels, S
Hoogstad, B
van Dun, L
de Schutter, S
Restifo, K
AF Bogels, Susan
Hoogstad, Bert
van Dun, Lieke
de Schutter, Sarah
Restifo, Kathleen
TI Mindfulness Training for Adolescents with Externalizing Disorders and
their Parents
SO BEHAVIOURAL AND COGNITIVE PSYCHOTHERAPY
LA English
DT Article
DE Mindfulness; externalizing; children and adolescents; mindful parenting;
meditation
ID DEFICIT HYPERACTIVITY DISORDER; GENERIC CORE SCALES; QUALITY-OF-LIFE;
ATTENTION-DEFICIT; ANTISOCIAL-BEHAVIOR; ANXIETY DISORDERS; CHILDREN;
CONDUCT; PEDSQL(TM)-4.0; INTERVENTIONS
AB Mindfulness training was evaluated as a new treatment for attention and impulsivity problems in adolescents with a variety of different externalizing disorders: attention deficit-hyperactivity disorder, oppositional-defiant and/or conduct disorder, and autism spectrum disorder if characterized by externalizing problem behaviour. It was argued that the large overlap between these three disorders may be partially explained by common underlying attention and behaviour control deficits. Fourteen clinically referred adolescents suffering from externalizing disorders followed mindfulness training in a group format. Parallel, their parents received mindful parenting training. Adolescents and their parents were measured before and after waitlist, after 8-week training, and at 8-week follow-up. No improvement occurred during waitlist on most variables. After mindfulness training, children self-reported substantial improvement on personal goals, internalizing and externalizing complaints, attention problems, happiness, and mindful awareness, and performed better on a sustained attention test. Likewise, parents reported improvement on children's goals, externalizing and attention problems, self-control, attunement to others and withdrawal. In addition, parents improved on their own goals. Improvement was maintained 8 weeks after the training. Consistent with mindfulness theory, increased child awareness after training predicted longer-term improvement in parent-rated child symptoms. Concomitant parent and child mindfulness training appears to be a promising approach for clinic-referred adolescents with attention and impulsivity problems.
C1 [Bogels, Susan] Univ Amsterdam, Dept Educ, NL-1090 GE Amsterdam, Netherlands.
[Hoogstad, Bert; van Dun, Lieke] Community Child & Youth Mental Hlth Ctr Maastrich, Maastricht, Netherlands.
[Restifo, Kathleen] Maastricht Univ, Maastricht, Netherlands.
RP Bogels, S (reprint author), Univ Amsterdam, Dept Educ, POB 94208, NL-1090 GE Amsterdam, Netherlands.
EM s.m.bogels@uva.nl
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NR 50
TC 56
Z9 56
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1352-4658
J9 BEHAV COGN PSYCHOTH
JI Behav. Cognit. Psychther.
PD MAR
PY 2008
VL 36
IS 2
BP 193
EP 209
DI 10.1017/S1352465808004190
PG 17
WC Psychology, Clinical
SC Psychology
GA 368IX
UT WOS:000260615900005
ER
PT J
AU Deruelle, C
Rondan, C
Salle-Collemiche, X
Bastard-Rosset, D
Da Fonseca, D
AF Deruelle, Christine
Rondan, Cecilie
Salle-Collemiche, Xavier
Bastard-Rosset, Delphine
Da Fonseca, David
TI Attention to low- and high-spatial frequencies in categorizing facial
identities, emotions and gender in children with autism
SO BRAIN AND COGNITION
LA English
DT Article
DE autism; face perception; identity; emotion; gender; spatial frequency
ID HUMAN EXTRASTRIATE CORTEX; UPSIDE-DOWN FACES; ASPERGER-SYNDROME;
FUNCTIONAL MRI; RECOGNITION; PERCEPTION; INDIVIDUALS; EXPRESSIONS;
ADULTS; PERFORMANCE
AB This study was aimed at investigating face categorization strategies in children with autistic spectrum disorders (ASD). Performance of 17 children with ASD was compared to that of 17 control children in a face-matching task, including hybrid faces (composed of two overlapping faces of different spatial bandwidths) and either low- or high-pass filtered faces. Participants were asked to match faces on the basis of identity, emotion or gender. Results revealed that children with ASD used the same strategies as controls when matching faces by gender. By contrast, in the identity and the emotion conditions, children with ASD showed a high-pass bias (i.e., preference for local information), contrary to controls. Consistent with previous studies on autism, these findings suggest that children with ASD do use atypical (local-oriented) strategies to process faces. (c) 2007 Elsevier Inc. All rights reserved.
C1 [Deruelle, Christine; Bastard-Rosset, Delphine; Da Fonseca, David] CNRS, Mediterranean Inst Cognit Neurosci, Marseille, France.
[Rondan, Cecilie] Austism Resource Ctr, Grenoble, France.
[Salle-Collemiche, Xavier; Bastard-Rosset, Delphine; Da Fonseca, David] Austism Resource Ctr, Marseille, France.
RP Deruelle, C (reprint author), CNRS, Mediterranean Inst Cognit Neurosci, Marseille, France.
EM deruelle@incm.cnrs-mrs.fr
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 48
TC 31
Z9 34
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0278-2626
J9 BRAIN COGNITION
JI Brain Cogn.
PD MAR
PY 2008
VL 66
IS 2
BP 115
EP 123
DI 10.1016/j.bandc.2007.06.001
PG 9
WC Neurosciences; Psychology, Experimental
SC Neurosciences & Neurology; Psychology
GA 275XY
UT WOS:000254106800002
PM 17693004
ER
PT J
AU Cuevas-Covarrubias, SA
Gonzalez-Huerta, LM
AF Cuevas-Covarrubias, S. A.
Gonzalez-Huerta, L. M.
TI Analysis of the VCX3A, VCX2 and VCX3B genes shows that VCX3A gene
deletion is not sufficient to result in mental retardation in X-linked
ichthyosis
SO BRITISH JOURNAL OF DERMATOLOGY
LA English
DT Article
DE mental retardation; steroid sulphatase; STS gene; VCX3A gene; X-linked
ichthyosis
ID STEROID-SULFATASE DEFICIENCY; DISTAL SHORT ARM; STS GENE; FAMILY;
CHROMOSOME; LOCUS; PATTERN; XP22.3; AUTISM; MEMBER
AB Background X-linked ichthyosis (XLI), an inborn error of metabolism, is due to steroid sulphatase (STS) deficiency. Most patients with XLI harbour complete deletion of the STS gene and flanking sequences. The presence of low copy number repeats on either side of the STS gene seems to have a major role in the high frequency of these deletions. Some patients with XLI with terminal deletions of Xp22.3 involving marker DXS1139 and the STS gene show mental retardation (MR); VCX3A is the only gene located on this critical region.
Objectives To analyse the VCX3A, VCX, VCX2 and VCX3B genes in 80 unrelated Mexican patients with XLI with normal intelligence.
Methods STS activity was measured in the leucocytes using 7-[H-3]-dehydroepiandrosterone sulphate as a substrate. Amplification of the regions from telomeric DXS89 to centromeric DXS1134 including both extremes of the STS and the VCX3A, VCX, VCX2 and VCX3B genes was performed using polymerase chain reaction.
Results No STS activity was detected in the patients with XLI (0.00 pmol mg(-1) protein h(-1)). We observed two different deletion patterns: the first group included 62 patients with deletion of VCX3A and VCX genes. The second group included 18 patients with breakpoints at several regions on either side of the STS gene not including the VCX3A gene.
Conclusions These data indicate that more complex mechanisms, apart from possible VCX3A gene participation, are occurring in the genesis of MR in XLI, at least in the sample of Mexican patients analysed.
C1 [Cuevas-Covarrubias, S. A.; Gonzalez-Huerta, L. M.] Univ Nacl Autonoma Mexico, Fac Med, Gen Hosp, Serv Genet, Mexico City, DF, Mexico.
RP Cuevas-Covarrubias, SA (reprint author), Univ Nacl Autonoma Mexico, Fac Med, Gen Hosp, Serv Genet, Dr Balmis 148,Col Doctores CP, Mexico City, DF, Mexico.
EM sergioa@servidor.unam.mx
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TC 17
Z9 17
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0007-0963
J9 BRIT J DERMATOL
JI Br. J. Dermatol.
PD MAR
PY 2008
VL 158
IS 3
BP 483
EP 486
DI 10.1111/j.1365-2133.2007.08373.x
PG 4
WC Dermatology
SC Dermatology
GA 262AF
UT WOS:000253120700008
PM 18076704
ER
PT J
AU Milne, E
Scope, A
AF Milne, Elizabeth
Scope, Alison
TI Are children with autistic spectrum disorders susceptible to contour
illusions?
SO BRITISH JOURNAL OF DEVELOPMENTAL PSYCHOLOGY
LA English
DT Article
ID WEAK CENTRAL COHERENCE; ILLUSORY CONTOURS; PERCEPTION; INDIVIDUALS;
PERFORMANCE; ADOLESCENTS; TASK
AB Children with autism have been shown to be less susceptible to Kanisza type contour illusions than children without autism (Happe, 1996). Other authors have suggested that this finding could be explained by the fact that participants with autism were required to make a potentially ambiguous verbal response which may have masked whether or not they actually perceived the illusory contours (Ropar & Mitchell, 1999). The present study tested perception of illusory contours in children with autism using a paradigm that requires participants to make a forced choice about the dimensions of a shape defined by illusory contours. It was reasoned that accuracy of the participant on this task would indicate whether or not children with autism could perceive illusory contours. A total of 18 children with autistic spectrum disorder, 16 children with special educational needs not including autism and 20 typically developing children completed an experimental task which assessed perception of Kanisza-style rectangles defined by illusory contours. There were no significant differences between the performance of the children with autism and either of the two control groups, suggesting that perception of illusory contours is intact in autism.
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RP Milne, E (reprint author), Univ Sheffield, Dept Psychol, Western Bank, Sheffield S10 2TN, S Yorkshire, England.
EM E.Mitne@Sheffield.oc.uk
RI Scope, Alison/E-5368-2010
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NR 27
TC 6
Z9 6
PU BRITISH PSYCHOLOGICAL SOC
PI LEICESTER
PA ST ANDREWS HOUSE, 48 PRINCESS RD EAST, LEICESTER LE1 7DR, LEICS, ENGLAND
SN 0261-510X
J9 BRIT J DEV PSYCHOL
JI Br. J. Dev. Psychol.
PD MAR
PY 2008
VL 26
BP 91
EP 102
DI 10.1348/026151007X202509
PN 1
PG 12
WC Psychology, Developmental
SC Psychology
GA 260CF
UT WOS:000252987700006
ER
PT J
AU Dean, SL
Mccarthy, MM
AF Dean, Shannon L.
Mccarthy, Margaret M.
TI Steroids, sex and the cerebellar cortex: implications for human disease
SO CEREBELLUM
LA English
DT Article
DE neurosteroids; cerebellum; neuropsychological diseases; gender
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; CHILDHOOD-ONSET SCHIZOPHRENIA;
POSITRON-EMISSION-TOMOGRAPHY; DEVELOPING PURKINJE-CELL;
CEREBRAL-BLOOD-FLOW; RAT CEREBELLUM; POSTNATAL-DEVELOPMENT;
MESSENGER-RNA; HUMAN-BRAIN; MAJOR DEPRESSION
AB Neurosteroids play an important role in the development of the cerebellum. In particular, estradiol and progesterone appear capable of inducing increases in dendritic spine density during development, and there is evidence that both are synthesized de novo in the cerebellum during critical developmental periods. In normal neonates and adults, there are few differences in the cerebellum between the sexes and most studies indicate that hormone and receptor levels also do not differ significantly during development. However, the sexes do differ significantly in risk of neuropsychological diseases associated with cerebellar pathology, and in animal models there are noticeable sex differences in the response to insult and genetic mutation. In both humans and animals, males tend to fare worse. Boys are more at risk for autism and Attention Deficit Hyperactivity Disorder than girls, and schizophrenia manifests at an earlier age in men. In rats males fare worse than females after perinatal exposure to polychlorinated biphenyls, and male mice heterozygous for the staggerer and reeler mutation show a more severe phenotype. Although very recent evidence suggests that differences in neurosteroid levels between the sexes in diseased animals may play a role in generating different disease phenotypes, the reason this hormonal difference occurs in diseased but not normal animals is currently unknown.
C1 [Mccarthy, Margaret M.] Univ Maryland, Sch Med, Dept Physiol, Baltimore, MD 21210 USA.
[Dean, Shannon L.; Mccarthy, Margaret M.] Univ Maryland, Sch Med, Program Neurosci, Baltimore, MD 21210 USA.
RP Mccarthy, MM (reprint author), Univ Maryland, Sch Med, Dept Physiol, 655 W Baltimore St, Baltimore, MD 21210 USA.
EM mmccarth@umaryland.edu
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NR 81
TC 27
Z9 27
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1473-4222
J9 CEREBELLUM
JI Cerebellum
PD MAR
PY 2008
VL 7
IS 1
BP 38
EP 47
DI 10.1007/s12311-008-0003-6
PG 10
WC Neurosciences
SC Neurosciences & Neurology
GA 330RL
UT WOS:000257960400006
PM 18418672
ER
PT J
AU Williams, K
Helmer, M
Duncan, GW
Peat, JK
Mellis, CM
AF Williams, K.
Helmer, M.
Duncan, G. W.
Peat, J. K.
Mellis, C. M.
TI Perinatal and maternal risk factors for autism spectrum disorders in New
South Wales, Australia
SO CHILD CARE HEALTH AND DEVELOPMENT
LA English
DT Article
DE autism; maternal; perinatal; risk factors
ID INFANTILE-AUTISM; NEONATAL FACTORS; POPULATION; HISTORY; AGE
AB Background This study was commenced in 1999 with the aim of examining risk factors for autism using established population-based data for comparison.
Methods Cases were ascertained using active surveillance and compared with birth data.
Results Four risk factors were found to be significantly associated with autism using binary logistic regression analysis; being male [adjusted odds ratio (OR) 4.7, 95% confidence interval (CI) 3.2-7.0], being born prematurely (adjusted OR 2.2, 95% CI 1.5-3.5), having maternal age >= 35 years (adjusted OR 1.7, 95% CI 1.2-2.4) and having a mother born outside Australia (adjusted OR 1.4, 95% CI 1.0-1.9). For analysis completed for pregnancies, rather than live births, multiple birth was also a significant risk factor for one or more children of the pregnancy to be affected by autism (adjusted OR 2.5, 95% CI 1.1-5.5). There was a statistically significant trend towards increasing risk with increasing risk factor 'dose' for gestational age (P = 0.019), multiple birth (P = 0.016) and maternal age (P < 0.001). For mother's country of birth the group with the highest risk were children of mother's born in south-east or north-east Asia. There was a non-significant trend towards a higher proportion of children with developmental disability having risk factors.
Conclusion Replication of risk factors from previous studies and a significant risk factor 'dose' effect add to growing evidence that maternal and perinatal factors are low magnitude risk factors for autism. The association between developmental disability and autism risk factors warrants further examination.
C1 [Williams, K.; Helmer, M.; Duncan, G. W.; Peat, J. K.; Mellis, C. M.] Childrens Hosp, Clin Epidemiol Unit, Westmead, NSW, Australia.
[Williams, K.; Peat, J. K.; Mellis, C. M.] Univ Sydney, Fac Med, Sydney, NSW 2006, Australia.
RP Williams, K (reprint author), Sydney Childrens Community Hlth Ctr, Cnr Barker & Avoca St, Randwick, NSW 2031, Australia.
EM katrina.williams@sesiahs.health.nsw.gov.au
RI Williams, Katrina/B-6828-2015
OI Williams, Katrina/0000-0002-1686-4458
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 26
TC 31
Z9 32
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0305-1862
J9 CHILD CARE HLTH DEV
JI Child Care Health Dev.
PD MAR
PY 2008
VL 34
IS 2
BP 249
EP 256
DI 10.1111/j.1365-2214.2007.00796.x
PG 8
WC Psychology, Developmental; Pediatrics
SC Psychology; Pediatrics
GA 258UV
UT WOS:000252895900016
PM 18257794
ER
PT J
AU Mouridsen, SE
Rich, B
Isager, T
AF Mouridsen, Svend Erik
Rich, Bente
Isager, Torben
TI Epilepsy and other neurological diseases in the parents of children with
infantile autism. A case control study
SO CHILD PSYCHIATRY & HUMAN DEVELOPMENT
LA English
DT Article
DE infantile autism; epilepsy; neurological diseases; broader phenotype
ID DISORDERS; PSYCHOSIS; PHENOTYPE; RELATIVES; REGISTER
AB In order to study the broader phenotype of infantile autism (IA) we compared the rates and types of epilepsy and other neurological diseases in the parents of 111 consecutively admitted patients with IA with a matched control group of parents of 330 children from the general population. All participants were screened through the nationwide Danish National Hospital Register (DNHR). We inquired about epilepsy and other neurological diseases during an observation period of 27 years. A similar proportion of case- and control mothers had a diagnosis of any neurological disease, 9.9% vs 10.6%. For case fathers the proportion was 5.7% vs 9.7%. No single neurological disease was significantly more frequent among parents of persons with IA. Our study lent support to the notion that epilepsy and other neurological diseases are not part of the broader IA phenotype.
C1 [Mouridsen, Svend Erik] Bispebjerg Hosp, Dept Child & Adolescent Psychiat, DK-2400 Copenhagen, Denmark.
[Rich, Bente] Odense Univ Hosp, Child & Adolescent Psychiat Dept, DK-5000 Odense, Denmark.
[Isager, Torben] Glostrup Univ Hosp, Ctr Child & Adolescent Psychiat, DK-2600 Glostrup, Denmark.
RP Mouridsen, SE (reprint author), Bispebjerg Hosp, Dept Child & Adolescent Psychiat, DK-2400 Copenhagen, Denmark.
EM sem01@bbh.hosp.dk
CR Andersen TF, 1999, DAN MED BULL, V46, P263
Bailey A, 1998, J AUTISM DEV DISORD, V28, P369, DOI 10.1023/A:1026048320785
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NR 23
TC 4
Z9 4
PU SPRINGER
PI NEW YORK
PA 233 SPRING STREET, NEW YORK, NY 10013 USA
SN 0009-398X
J9 CHILD PSYCHIAT HUM D
JI Child Psychiat. Hum. Dev.
PD MAR
PY 2008
VL 39
IS 1
BP 1
EP 8
DI 10.1007/s10578-007-0062-9
PG 8
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 248MF
UT WOS:000252156700001
PM 17564832
ER
PT J
AU Greco, D
Romano, C
Reitano, S
Barone, C
Benedetto, DD
Castiglia, L
Fichera, M
Galesi, O
Zingale, M
Buono, S
Uliana, V
Caselli, R
Canitano, R
Hayek, G
Renieri, A
AF Greco, D.
Romano, C.
Reitano, S.
Barone, C.
Benedetto, D. D.
Castiglia, L.
Fichera, M.
Galesi, O.
Zingale, M.
Buono, S.
Uliana, V.
Caselli, R.
Canitano, R.
Hayek, G.
Renieri, A.
TI Three new patients with dup(17)(p11.2p11.2) without autism
SO CLINICAL GENETICS
LA English
DT Letter
ID PHENOTYPE
C1 [Greco, D.; Romano, C.; Reitano, S.; Barone, C.] IRCCS, Assoc Oasi Maria Santissima, Dept Mental Retardat, Unit Pediat & Med Genet, Troina, Italy.
[Benedetto, D. D.; Castiglia, L.; Fichera, M.; Galesi, O.] IRCCS, Assoc Oasi Maria Santissima, Lab Genet Diagnost, Troina, Italy.
[Zingale, M.; Buono, S.] IRCCS, Assoc Oasi Maria Santissima, Dept Mental Retardat, Unit Psychol, Troina, Italy.
[Uliana, V.; Caselli, R.; Renieri, A.] Univ Siena, Dept Med Genet, I-53100 Siena, Italy.
[Canitano, R.; Hayek, G.] Azienda Osp Siena, Dept Child Neuropsychiat, Siena, Italy.
RP Romano, C (reprint author), IRCCS, Assoc Oasi Maria Santissima, Unit Pediat & Med Genet, Via Conte Ruggero 73, I-94018 Troina, Italy.
EM cromano@oasi.en.it
RI Romano, Corrado/B-9695-2008
OI Romano, Corrado/0000-0003-1049-0683
CR Lord C., 1999, AUTISM DIAGNOSTIC OB
Moog U, 2004, GENET COUNSEL, V15, P73
NAKAMINE A, 2007, AM J MED GENE A 0302
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WHO, 1993, ICD 10 CLASS MENT BE
NR 11
TC 6
Z9 6
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0009-9163
J9 CLIN GENET
JI Clin. Genet.
PD MAR
PY 2008
VL 73
IS 3
BP 294
EP 296
DI 10.1111/j.1399-0004.2007.00959.x
PG 3
WC Genetics & Heredity
SC Genetics & Heredity
GA 259GY
UT WOS:000252929000017
PM 18218042
ER
PT J
AU Munoz-Yunta, JA
Ortiz, T
Palau-Baduell, M
Martin-Munoz, L
Salvado-Salvado, B
Valls-Santasusana, A
Perich-Alsina, J
Cristobal, I
Fernandez, A
Maestu, F
Dursteler, C
AF Munoz-Yunta, J. A.
Ortiz, T.
Palau-Baduell, M.
Martin-Munoz, L.
Salvado-Salvado, B.
Valls-Santasusana, A.
Perich-Alsina, J.
Cristobal, I.
Fernandez, A.
Maestu, F.
Dursteler, C.
TI Magnetoencephalographic pattern of epileptiform activity in children
with early-onset autism spectrum disorders
SO CLINICAL NEUROPHYSIOLOGY
LA English
DT Article
DE magnetoencephalography; pervasive developmental disorders; autistic
spectrum disorders; epileptiform activity pattern
ID SCHOOL-AGE-CHILDREN; DIAGNOSTIC INTERVIEW; ASPERGER-SYNDROME; EEG;
ABNORMALITIES; EPILEPSY; INDIVIDUALS; CHILDHOOD; SEIZURES; BRAIN
AB Objective: To provide further data around magnetoencephalographic (MEG) findings in early-onset autism spectrum disorders (ASD).
Methods: Thirty-six children (mean age 7 years) diagnosed of PDD (DSM-IV, ICD-10) were studied. There were 22 children with autistic disorder, 9 with Asperger's syndrome.. and 5 with pervasive developmental disorder not otherwise specified (PDD-NOS). According to the Childhood Autism Rating Scale (CARS), the autistic disorder was mild to moderate in 11, and severe in 11. Neuroimaging studies using three-dimensional MRI as well as simultaneous MEG-EEG and fusion techniques through magnetic source imaging (MSI) were performed, with the aid of anesthesia in non-cooperative patients.
Results: Most patients had no EEG abnormalities. All ASD children showed common specific abnormalities in the shape of low amplitude monophasic and biphasic spikes (isolated or short bursts) as well as acute waves, predominantly distributed in the perisylvian areas. In Asperger's syndrome, epileptiform spikes were mostly found in the right hemisphere. No lateralized epileptiform activity was observed in non-Asperger's autistic patients.
Conclusions: MEG epileptiform activity is frequently documented in children with early-onset ASD.
Significance: Subclinical epileptiform activity is present especially in the perisylvian regions for many patients with ASD. (C) 2007 International Federation of Clinical Neurophysiology. Published by Elsevier Ireland Ltd. All rights reserved.
C1 [Munoz-Yunta, J. A.] Univ Autonoma Barcelona, Hosp Univ Mar, Unit Neuropediat, E-08003 Barcelona, Spain.
[Ortiz, T.; Fernandez, A.; Maestu, F.] Magnetoencephalog Ctr, Madrid, Spain.
[Palau-Baduell, M.; Martin-Munoz, L.] Neuropsychobiol Clin, Serv Neurophysiol, Barcelona, Spain.
[Salvado-Salvado, B.] Neuropsychobiol Clin, Serv Neuropsychol, Barcelona, Spain.
[Valls-Santasusana, A.] Univ Autonoma Barcelona, Hosp Univ Mar, Serv Neurophysiol, E-08003 Barcelona, Spain.
[Perich-Alsina, J.] Univ Autonoma Barcelona, Hosp Univ Mar, CRC Serv Magnet Resonance Imaging, E-08003 Barcelona, Spain.
[Cristobal, I.] Hosp Clin Univ San Carlos, Anesthesiol Serv, Madrid, Spain.
[Dursteler, C.] Univ Autonoma Barcelona, Hosp Univ Mar, Anesthesiol Serv, E-08003 Barcelona, Spain.
RP Munoz-Yunta, JA (reprint author), Univ Autonoma Barcelona, Hosp Univ Mar, Unit Neuropediat, Passeig Maritim 25-29, E-08003 Barcelona, Spain.
EM 10030amy@comb.es
RI Maestu, Fernando/E-3213-2012
OI Maestu, Fernando/0000-0002-3195-0071
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NR 39
TC 14
Z9 14
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 1388-2457
J9 CLIN NEUROPHYSIOL
JI Clin. Neurophysiol.
PD MAR
PY 2008
VL 119
IS 3
BP 626
EP 634
DI 10.1016/j.clinph.2007.11.007
PG 9
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 277ZN
UT WOS:000254253300012
PM 18164240
ER
PT J
AU Robbins, P
AF Robbins, Philip
TI Consciousness and the social mind
SO COGNITIVE SYSTEMS RESEARCH
LA English
DT Article
DE consciousness; emotion; social cognition; social pain; embodiment;
mindreading; moral cognition
ID WILLIAMS-SYNDROME; AMYGDALA THEORY; PAIN; AUTISM; LANGUAGE; INDIVIDUALS;
RECOGNITION; EXPRESSIONS; EXCLUSION; MORALITY
AB Phenomenal consciousness and social cognition are interlocking capacities, but the relations between them have yet to be systematically investigated. In this paper, I begin to develop a theoretical and empirical framework for such an investigation. I begin by describing the phenomenon known as social pain: the affect associated with the perception of actual or potential damage to one's interpersonal relations. I then adduce a related phenomenon known as affective contagion: the tendency for emotions, moods, and other affective states to spread from person to person in social contexts. Experimental studies of these two phenomena suggest that affective consciousness depends on perception of the social world in much the same way that it depends on perception of the body - in short, that consciousness is 'socially embodied'. In the second part of the paper I argue that the distinctive sociality of our species, especially its moral dimension, rests heavily on our ability to represent the conscious states of others. In closing, I put these ideas together and show how they point to a circular causal-mechanistic nexus between consciousness and social mindedness. (C) 2007 Elsevier B.V. All rights reserved.
C1 Univ Missouri, Dept Philosophy, Columbia, MO 65211 USA.
RP Robbins, P (reprint author), Univ Missouri, Dept Philosophy, 426 Gen Classroom Bldg, Columbia, MO 65211 USA.
EM robbinsp@missouri.edu
RI X, Simon/F-4678-2011
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NR 57
TC 3
Z9 3
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 1389-0417
J9 COGN SYST RES
JI Cogn. Syst. Res.
PD MAR
PY 2008
VL 9
IS 1-2
BP 15
EP 23
DI 10.1016/j.cogsys.2007.07.005
PG 9
WC Computer Science, Artificial Intelligence; Neurosciences; Psychology,
Experimental
SC Computer Science; Neurosciences & Neurology; Psychology
GA 299AN
UT WOS:000255728300003
ER
PT J
AU Glazebrook, CM
Elliott, D
Lyons, J
AF Glazebrook, Cheryl M.
Elliott, Digby
Lyons, James
TI Temporal judgements of internal and external events in persons with and
without autism
SO CONSCIOUSNESS AND COGNITION
LA English
DT Article
DE autism; feed-forward control; binding; action; movement preparation
ID CONSCIOUS INTENTION; MOTOR CONTROL; MOVEMENT; TIME; DISORDER; CHILDREN;
ONSET; TASK
AB When participants make judgments about the onset of self-initiated movements they typically report the movement occurred earlier than it had [Obhi, S. S., & Haggard, P. (2004). Free will and free won't. American Scientific, 92, 358365.]. One interpretation is that feed-forward processes lead to awareness of the movement prior to execution. Because individuals with autism experience reduced preparatory activity prior to a voluntary movement, the present study sought to determine whether these anticipatory biases are exhibited by persons with autism. Participants watched a dot move in a circle and pressed the spacebar any time after one revolution. A tone either followed the participants' voluntary movement or was computer generated. Participants in both groups made anticipatory judgements regarding movement initiation (similar to 100 ms). When the movement and tone occurred together this anticipatory bias was also present, regardless of which event participants focused on. Individuals with autism appear to have access to a similar representation of voluntary movements, however this representation may be more variable. (c) 2007 Elsevier Inc. All rights reserved.
C1 [Glazebrook, Cheryl M.; Elliott, Digby; Lyons, James] McMaster Univ, Dept Kinesiol, Hamilton, ON L8S 4K1, Canada.
RP Glazebrook, CM (reprint author), McMaster Univ, Dept Kinesiol, 1280 Main St W, Hamilton, ON L8S 4K1, Canada.
EM bezalg@mcmaster.ca
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TC 2
Z9 2
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1053-8100
J9 CONSCIOUS COGN
JI Conscious. Cogn.
PD MAR
PY 2008
VL 17
IS 1
BP 203
EP 209
DI 10.1016/j.concog.2007.03.001
PG 7
WC Psychology, Experimental
SC Psychology
GA 293IP
UT WOS:000255329300016
PM 17433718
ER
PT J
AU Marazziti, D
Dell'Osso, MC
AF Marazziti, D.
Dell'Osso, M. Catena
TI The role of oxytocin in neuropsychiatric disorders
SO CURRENT MEDICINAL CHEMISTRY
LA English
DT Review
DE oxytocin (OT); vasopressin (AVP); synthesis; receptors; physiology;
social behaviors; neuropsychiatric disorders
ID OBSESSIVE-COMPULSIVE DISORDER; PRADER-WILLI-SYNDROME; HYPOTHALAMIC
PARAVENTRICULAR NUCLEUS; CORTICOTROPIN-RELEASING-FACTOR;
CEREBROSPINAL-FLUID LEVELS; BASAL GANGLIA DYSFUNCTION; PLASMA OXYTOCIN;
ANOREXIA-NERVOSA; MAJOR DEPRESSION; BULIMIA-NERVOSA
AB Oxytocin (OT) is a neurohypophysial hormone synthesized in the paraventricular and supraoptic nuclei of the hypothalamus. Although OT-like substances have been identified in all vertebrates, OT has been found only in mammals where it plays a major role in the onset and maintaining of behaviors which are typical of these animals, such as labour and lactation. Recently, several data have suggested the involvement of OT in the formation of infant attachment, maternal behavior, pair bonding and, more generally, in linking social signals with cognition, behaviors and reward.
The aim of this paper was to review critically the role of OT in the regulation of different physiological functions and complex behaviors, as well as its possible involvement in the pathophysiology of some neuropsychiatric disorders. MEDLINE and PubMed (1972-2007) databases were searched for English language articles by using the following keywords: oxytocin, physiology, cognitive functions, attachment, psychopathology, psychiatric disorders. Papers were examined that addressed the following aspects of the OT system: synthesis and localization, receptors, physiology: In addition, latest findings showing abnormalities of OT and OT system in several neuropsychiatric disorders, including autism, obsessive-compulsive disorder, eating disorders, addiction, schizophrenia, post-traumatic stress disorder and Prader-Willy syndrome, will be also discussed together with the possible clinical use of OT or its analogues and/or antagonists.
C1 [Marazziti, D.; Dell'Osso, M. Catena] Univ Pisa, Dipartimento Psichiat Neurobiol Farmacol & Biotec, I-56100 Pisa, Italy.
RP Marazziti, D (reprint author), Univ Pisa, Dipartimento Psichiat Neurobiol Farmacol & Biotec, Via Roma 67, I-56100 Pisa, Italy.
EM dmarazzi@psico.med.unipi.it
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NR 152
TC 36
Z9 36
PU BENTHAM SCIENCE PUBL LTD
PI SHARJAH
PA EXECUTIVE STE Y26, PO BOX 7917, SAIF ZONE, 1200 BR SHARJAH, U ARAB
EMIRATES
SN 0929-8673
J9 CURR MED CHEM
JI Curr. Med. Chem.
PD MAR
PY 2008
VL 15
IS 7
BP 698
EP 704
DI 10.2174/092986708783885291
PG 7
WC Biochemistry & Molecular Biology; Chemistry, Medicinal; Pharmacology &
Pharmacy
SC Biochemistry & Molecular Biology; Pharmacology & Pharmacy
GA 274GU
UT WOS:000253990600006
PM 18336283
ER
PT J
AU Johnson, KP
Malow, BA
AF Johnson, Kyle P.
Malow, Beth A.
TI Sleep in children with autism spectrum disorders
SO CURRENT NEUROLOGY AND NEUROSCIENCE REPORTS
LA English
DT Review
ID PERVASIVE DEVELOPMENTAL DISORDERS; CONTROLLED-RELEASE MELATONIN;
EYE-MOVEMENT SLEEP; NEURODEVELOPMENTAL DISORDERS; BEHAVIORAL TREATMENT;
ASPERGERS-DISORDER; LIGHT THERAPY; YOUNG-ADULTS; EPILEPSY; DISABILITIES
AB The amount of research conducted on sleep in children and adolescents has increased dramatically over the past decade due to the recognition that many children have significant sleep problems leading to daytime dysfunction. Approximately one third of typically developing children have sleep difficulties at some point, and a similar percentage of adolescents have impaired or insufficient sleep leading to daytime impairments. Sleep problems are known to occur at even greater rates in children with special needs, such as those with developmental disabilities, psychiatric conditions, and medical illnesses. The recognition that interventions can improve sleep and may result in better daytime functioning has fueled a growing interest in more fully characterizing the sleep problems in children with special needs. This article presents a discussion of the sleep problems experienced by children with one particular group of developmental disorders-the autism spectrum disorders.
C1 [Johnson, Kyle P.] Oregon Hlth & Sci Univ, Dept Psychiat, Portland, OR 97239 USA.
RP Johnson, KP (reprint author), Oregon Hlth & Sci Univ, Dept Psychiat, DC-7P,3181 SW Sam Jackson Pk Rd, Portland, OR 97239 USA.
EM johnsoky@ohsu.edu
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NR 55
TC 8
Z9 8
PU CURRENT SCIENCE INC
PI PHILADELPHIA
PA 400 MARKET STREET, STE 700, PHILADELPHIA, PA 19106 USA
SN 1528-4042
J9 CURR NEUROL NEUROSCI
JI Curr. Neurol. Neurosci. Rep.
PD MAR
PY 2008
VL 8
IS 2
BP 155
EP 161
DI 10.1007/s11910-008-0025-y
PG 7
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 307LB
UT WOS:000256319300009
PM 18460285
ER
PT J
AU Colvert, E
Rutter, M
Beckett, C
Castle, J
Groothues, C
Hawkins, A
Kreppner, J
O'Connor, TG
Stevens, S
Sonuga-Barke, EJS
AF Colvert, Emma
Rutter, Michael
Beckett, Celia
Castle, Jenny
Groothues, Christine
Hawkins, Amanda
Kreppner, Jana
O'Connor, Thomas G.
Stevens, Suzanne
Sonuga-Barke, Edmund J. S.
TI Emotional difficulties in early adolescence following severe early
deprivation: Findings from the English and Romanian adoptees study
SO DEVELOPMENT AND PSYCHOPATHOLOGY
LA English
DT Article
ID AUTISM DIAGNOSTIC INTERVIEW; AGGRESSIVE-BEHAVIOR; INTERNATIONAL
ADOPTEES; READING-DISABILITY; CATCH-UP; CHILDREN; QUESTIONNAIRE;
DISORDERS; ADOPTION; SPECIFICITY
AB The study assessed conduct and emotional difficulties in a group of Romanian adoptees at age 11, and serves as a follow-up to assessments made when the children were 6 years old. It was found that there was a significant increase in emotional difficulties, but not conduct problems, for the Romanian sample since age 6. It was also found that emotional difficulty was significantly more prevalent at age I I in the Romanian group than in a within-UK adoptee group. Emotional difficulties in the Romanian adoptee group were found to be significantly and strongly related to previous deprivation-specific problems (disinhibited attachment, cognitive impairment, inattention/overactivity and quasi-autism); however, the presence of such early problems did not account fully for the onset of later emotional problems. Five contrasting hypotheses concerning possible mediators for later onset of emotional difficulties for the Romanian group were examined. No links were found to duration of deprivation or other deprivation-related indices, stresses/difficulties in the postadoption family environment, or educational attainment and self-esteem. There was some evidence that emotion recognition might play a role in the emergence of these problems, but other measures of social competence and theory of mind showed no associations with the onset of emotional problems.
C1 [Colvert, Emma; Rutter, Michael; Beckett, Celia; Castle, Jenny; Groothues, Christine; Hawkins, Amanda; Kreppner, Jana; Stevens, Suzanne; Sonuga-Barke, Edmund J. S.] Univ London, Univ London Kings Coll, Inst Psychiat, SGDP Ctr, London SE5 8AF, England.
[O'Connor, Thomas G.] Univ Rochester, Rochester, NY 14627 USA.
[Stevens, Suzanne; Sonuga-Barke, Edmund J. S.] Univ Southampton, Southampton SO9 5NH, Hants, England.
[Sonuga-Barke, Edmund J. S.] NYU, New York, NY 10003 USA.
RP Colvert, E (reprint author), Univ London, Univ London Kings Coll, Inst Psychiat, SGDP Ctr, POB 80,Denmark Hill, London SE5 8AF, England.
EM e.colvert@iop.kcl.ac.uk
RI Sonuga-Barke, Edmund/D-9137-2011; Rutter, Michael/C-8570-2013
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NR 63
TC 55
Z9 55
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0954-5794
J9 DEV PSYCHOPATHOL
JI Dev. Psychopathol.
PD SPR
PY 2008
VL 20
IS 2
BP 547
EP 567
DI 10.1017/S0954579408000278
PG 21
WC Psychology, Developmental
SC Psychology
GA 293HX
UT WOS:000255327500009
PM 18423094
ER
PT J
AU Dumontheil, I
Burgess, PW
Blakemore, SJ
AF Dumontheil, Iroise
Burgess, Paul W.
Blakemore, Sarah-Jayne
TI Development of rostral prefrontal cortex and cognitive and behavioural
disorders
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Review
ID STRATEGY APPLICATION DISORDER; POSITRON-EMISSION-TOMOGRAPHY;
VISUOSPATIAL WORKING-MEMORY; AUTISTIC SPECTRUM DISORDERS; EXECUTIVE
FUNCTION DEFICITS; DIFFUSION-TENSOR MRI; FRONTAL-LOBE DAMAGE;
EVENT-RELATED FMRI; LONG-TERM-MEMORY; 6 ELEMENTS TEST
AB Information on the development and functions of rostral prefrontal cortex (PFC), or Brodmann area 10, has been gathered from different fields, from anatomical development to functional neuroimaging in adults, and put forward in relation to three particular cognitive and behavioural disorders. Rostral PFC is larger and has a lower cell density in humans than in other primates. It also has a large number of dendritic spines per cell and numerous connections to the supramodal cortex. These characteristics suggest that rostral PFC is likely to support processes of integration or coordination of inputs that are particularly developed in humans. The development of rostral PFC is prolonged, with decreases in grey matter and synaptic density continuing into adolescence. Functions attributed to rostral PFC, such as prospective memory, seem similarly to follow a prolonged development until adulthood. Neuroimaging studies have generally found a reduced recruitment of rostral PFC, for example in tasks requiring response inhibition, in adults compared with children or adolescents, which is consistent with maturation of grey matter. The examples of autism, attention-deficit-hyperactivity disorder, and schizophrenia show that rostral PFC could be affected in several disorders as a result of the susceptibility of its prolonged maturation to developmental abnormalities.
C1 [Dumontheil, Iroise] UCL, Inst Cognit Neurosci, London WC1N 3AR, England.
UCL, Dept Psychol, London WC1N 3AR, England.
RP Dumontheil, I (reprint author), UCL, Inst Cognit Neurosci, 17 Queen Sq,Alexandra House, London WC1N 3AR, England.
EM i.dumontheil@ucl.ac.uk
RI Blakemore, Sarah-Jayne/A-1792-2010; Burgess, Paul/A-1811-2010
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NR 212
TC 58
Z9 59
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD MAR
PY 2008
VL 50
IS 3
BP 168
EP 181
DI 10.1111/j.1469-8749.2008.02026.x
PG 14
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 266ZL
UT WOS:000253477300006
PM 18190537
ER
PT J
AU Buschmann, A
Jooss, B
Rupp, A
Dockter, S
Blaschtikowitz, H
Heggen, I
Pietz, J
AF Buschmann, Anke
Jooss, Bettina
Rupp, Andre
Dockter, Sonja
Blaschtikowitz, Heike
Heggen, Iris
Pietz, Joachim
TI Children with developmental language delay at 24 months of age: results
of a diagnostic work-up
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Article
ID PRESCHOOL-CHILDREN; OTITIS-MEDIA; PREVALENCE; DISORDERS; POPULATION;
IMPAIRMENT; OUTCOMES; AUTISM; COHORT; LIFE
AB The aim of this study was to evaluate if a diagnostic work-up should be recommended for 2-year-old children with developmental language delay (LD), or if the widely chosen 'wait and see' strategy is adequate. Children with LD were identified in paediatric practices during routine developmental check-ups using a German parent-report screening questionnaire (adapted from the MacArthur Communicative Development Inventories). A standardized German instrument and the Netherlands version of Bayley Scales of Infant Development (2nd edn) were used to assess language ability and nonverbal cognitive development respectively in 100 children with LD (65 males, 35 females; mean age 24.7mo [SD 0.9]) and a control group of 53 children with normal language development (33 males, 20 females; mean age 24.6mo [SD 0.8]). Neurological and audiometric testing were also performed. Sixty-one per cent of the LD group had specific expressive LD and 17% specific receptive-expressive LD. In 22%, LD was associated with other neurodevelopmental problems, 6% showed significant deficits in nonverbal cognitive abilities, and in 12%, nonverbal cognitive abilities were borderline. Four per cent fulfilled the criteria of childhood autism. LD at 2 years proved to represent a sensitive marker for different developmental problems. Adequate early intervention requires a clear distinction between specific expressive or receptive-expressive LD and LD associated with other neurodevelopmental problems. Though catch-up development is to be expected in a substantial proportion of 'late talkers', our data demonstrate that a general 'wait and see' approach is not justified in young children with LD. A proposal for a rational diagnostic work-up is presented.
C1 [Buschmann, Anke; Pietz, Joachim] Univ Heidelberg, Childrens Hosp, Dept Paediat Neurol, D-69120 Heidelberg, Germany.
[Rupp, Andre] Univ Heidelberg, Childrens Hosp, Dept Neurol, Sect Biomagnetism, D-69120 Heidelberg, Germany.
[Dockter, Sonja] Univ Heidelberg, Childrens Hosp, Dept Paedaudiol, D-69120 Heidelberg, Germany.
RP Buschmann, A (reprint author), Univ Heidelberg, Childrens Hosp, Dept Paediat Neurol, Neuenheimer Feld 150, D-69120 Heidelberg, Germany.
EM anke.buschmann@med.uni-heidelberg.de
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NR 27
TC 23
Z9 26
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD MAR
PY 2008
VL 50
IS 3
BP 223
EP 229
DI 10.1111/j.1469-8749.2008.02034.x
PG 7
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 266ZL
UT WOS:000253477300013
PM 18266869
ER
PT J
AU Marsh, ED
Minarcik, J
Campbell, K
Brooks-Kayal, AR
Golden, JA
AF Marsh, Eric D.
Minarcik, Jennifer
Campbell, Kenneth
Brooks-Kayal, Amy R.
Golden, Jeffrey A.
TI FACS-array gene expression analysis during early development of mouse
telencephalic interneurons
SO DEVELOPMENTAL NEUROBIOLOGY
LA English
DT Article
DE interneuron; development; FACs; gene profiling; ganglionic eminence;
nonradial migration
ID ION CHANNEL EXPRESSION; WHITE MATTER GLIA; GANGLIONIC EMINENCE; NEURONAL
MIGRATION; GLOBAL ANALYSIS; HOMEOBOX GENES; GABA; CELL; FOREBRAIN;
PATHWAYS
AB Cortical interneuron dysfunction has been implicated in multiple human disorders including forms of epilepsy, mental retardation, and autism. Although significant advances have been made, understanding the biologic basis of these disorders will require a level of anatomic, molecular, and genetic detail of interneuron development that currently does not exist. To further delineate the pathways modulating interneuron development we performed fluorescent activated cell sorting (FACs) on genetically engineered mouse embryos that selectively express green fluorescent protein (GFP) in developing interneurons followed by whole genome microarray expression profiling on the isolated cells. Bioinformatics analysis revealed expression of both predicted and unexpected genes in developing cortical interneurons. Two unanticipated pathways discovered to be up regulated prior to interneurons differentiating in the cortex were ion channels/neurotransmitters and synaptic/vesicular related genes. A significant association of neurological disease related genes to the population of developing interneurons was found. These results have defined new and potentially important data on gene expression changes during the development of cortical interneurons. In addition, these data can be mined to uncover numerous novel genes involved in the generation of interneurons and may suggest genes/pathways potentially involved in a number of human neurological disorders. (C) 2008 Wiley Periodicals, Inc.
C1 [Marsh, Eric D.; Minarcik, Jennifer; Brooks-Kayal, Amy R.] Univ Penn, Childrens Hosp Philadelphia, Div Neurol, Philadelphia, PA 19130 USA.
[Campbell, Kenneth] Cincinnati Childrens Hosp, Cincinnati, OH 45229 USA.
[Golden, Jeffrey A.] Univ Penn, Childrens Hosp Philadelphia, Dept Pathol, Philadelphia, PA 19130 USA.
RP Marsh, ED (reprint author), Univ Penn, Childrens Hosp Philadelphia, Div Neurol, Philadelphia, PA 19130 USA.
EM marshe@email.chop.edu
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NR 45
TC 25
Z9 25
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1932-8451
J9 DEV NEUROBIOL
JI Dev. Neurobiol.
PD MAR
PY 2008
VL 68
IS 4
BP 434
EP 445
DI 10.1002/dneu.20602
PG 12
WC Developmental Biology; Neurosciences
SC Developmental Biology; Neurosciences & Neurology
GA 269OB
UT WOS:000253657400002
PM 18172891
ER
PT J
AU Kuhn, LR
Bodkin, AE
Devlin, SD
Doggett, RA
AF Kuhn, Laura R.
Bodkin, Amy E.
Devlin, Sandra D.
Doggett, R. Anthony
TI Using pivotal response training with peers in special education to
facilitate play in two children with autism
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID INTERVENTION
AB This study evaluated the ability of peers in special education to implement pivotal response training (PRT) with two students with autism in order to increase social interactions. Peers were taught the strategies using modeling, role-playing, and feedback. After training, peers implemented PRT strategies with the children with autism. Picture prompts were provided to assist peers in recalling the strategies, but were completely faded until peers could implement the procedures with no instruction from observers. Increases in opportunities to respond were observed, as well as responses and initiations of social interaction by the children with autism.
C1 [Kuhn, Laura R.; Bodkin, Amy E.; Devlin, Sandra D.; Doggett, R. Anthony] Mississippi State Univ, Dept Counselor Educ Psychol & Special Educ, Mississippi State, MS 39759 USA.
RP Devlin, SD (reprint author), Mississippi State Univ, Dept Counselor Educ Psychol & Special Educ, Box 9727, Mississippi State, MS 39759 USA.
CR Garrison-Harrell L., 1997, FOCUS AUTISM OTHER D, V12, P241, DOI DOI 10.1177/108835769701200406
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GOLDSTEIN H, 1987, J SPEECH HEAR DISORD, V52, P200
Koegel R. L., 1989, TEACH PIVOTAL BEHAV
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PIERCE K, 1995, J APPL BEHAV ANAL, V28, P285, DOI 10.1901/jaba.1995.28-285
Stahmer A. C., 1999, CHILD LANG TEACH THE, V15, P29, DOI 10.1191/026565999672332808
Strain PS, 2000, TOP EARLY CHILD SPEC, V20, P116, DOI 10.1177/027112140002000207
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THORP DM, 1995, J AUTISM DEV DISORD, V25, P265, DOI 10.1007/BF02179288
NR 10
TC 7
Z9 7
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD MAR
PY 2008
VL 43
IS 1
BP 37
EP 45
PG 9
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 264NK
UT WOS:000253294400003
ER
PT J
AU Okada, S
Ohtake, Y
Yanagihara, M
AF Okada, Shingo
Ohtake, Yoshihisa
Yanagihara, Masafumi
TI Effects of perspective sentences in Social Stories (TM) on improving the
adaptive behaviors of students with autism spectrum disorders and
related disabilities
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID INTERVENTION; CHILDREN
AB This study examined the effects of adding perspective sentences to Social Stories (TM) on improving the adaptive behaviors of students with autism spectrum disorders (ASD) and related disabilities. In Study 1, two students with ASD read two different types of Social Stories: Social Story without perspective sentences (SS without PS) and Social Story with perspective sentences (SS with PS). ABC or ABCA designs were used, with an SS without PS presented in the B phase and an SS with PS presented in the C phase. A visual inspection revealed that Social Stories were likely to be effective in reducing inappropriate behaviors even without perspective sentences. In addition, adding perspective sentences appeared to have no impact on further improving the target behaviors. In Study 2, a perspective sentence was added, characterized as specific, valuable, and contingent to a Social Story in the SS with PS condition. An AA'BA'CA' design was utilized, with a permanent visual step poster in the A' phase, an SS without PS in the B phase, and an SS with PS in the C phase for a student diagnosed with attention deficit hyperactivity disorder. A visual inspection revealed that adding a perspective sentence to a Social Story contributed to further improvement of the target behavior. Based on these findings component and parametric analyses on Social Stories are recommended in future research.
C1 [Okada, Shingo; Ohtake, Yoshihisa; Yanagihara, Masafumi] Okayama Univ, Fac Educ, Okayama 7008530, Japan.
RP Ohtake, Y (reprint author), Okayama Univ, Fac Educ, 3-1-1 Tsushima Naka, Okayama 7008530, Japan.
EM ohtake@cc.okayama-u.ac.jp
CR Adamian GG, 2004, ACTA PHYS HUNG NS-H, V19, P87, DOI 10.1556/APH.19.2004.1-2.13
Bledsoe R, 2003, AUTISM, V7, P289, DOI 10.1177/1362361303007003005
Brownell MD, 2002, J MUSIC THER, V39, P117
GRAY C, NEW SOCIAL STORY BOO
Ivey M. L., 2004, FOCUS AUTISM OTHER D, V19, P164, DOI DOI 10.1177/10883576040190030401
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Lorimer PA, 2002, J POSIT BEHAV INTERV, V4, P53, DOI 10.1177/109830070200400109
Michael J, 2000, J APPL BEHAV ANAL, V33, P401, DOI 10.1901/jaba.2000.33-401
Myles B. S., 1999, FOCUS AUTISM OTHER D, V14, P82, DOI 10.1177/108835769901400203
Myles B. S., 2004, HIDDEN CURRICULUM PR
Sansosd F. J., 2004, FOCUS AUTISM OTHER D, V19, P194, DOI DOI 10.1177/10883576040190040101
Scattone D, 2002, J AUTISM DEV DISORD, V32, P535, DOI 10.1023/A:1021250813367
Thiemann KS, 2001, J APPL BEHAV ANAL, V34, P425, DOI 10.1901/jaba.2001.34-425
NR 14
TC 3
Z9 3
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD MAR
PY 2008
VL 43
IS 1
BP 46
EP 60
PG 15
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 264NK
UT WOS:000253294400004
ER
PT J
AU Tien, KC
AF Tien, Kai-Chien
TI Effectiveness of the picture exchange communication system as a
functional communication intervention for individuals with autism
spectrum disorders: A practice-based research synthesis
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID CHILDREN; PECS; SPEECH
AB This research synthesis verifies the effectiveness of the Picture Exchange Communication System (PECS) for improving the functional communication skills of individuals with autism spectrum disorders (ASD). The research synthesis was focused on the degree to which variations in PECS training are associated with variations in functional communication outcomes (Dunst, Trivette & Cutspec, 2002). The communication consequences of PECS were examined in 13 studies, which included 125 participants with ASD who had been identified as having limited or no functional communication skills. Claims that PECS is an effective intervention for improving functional communication skills appeared to be supported by the available research evidence.
C1 Univ Kansas, Lawrence, KS 66047 USA.
RP Tien, KC (reprint author), Univ Kansas, 4227 Wimbledon Dr, Lawrence, KS 66047 USA.
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
ANDERSON AE, 2002, DISS ABSTR INT B, V62, P4269
Axelrod S., 2002, BEHAV ANAL TODAY, V2, P259
Bondy A. S., 1994, FOCUS AUTISTIC BEHAV, V9, P1, DOI DOI 10.1177/108835769400900301
BONDY AS, 1993, BEHAV ANALYST, V16, P123
Charlop-Christy MH, 2002, J APPL BEHAV ANAL, V35, P213, DOI 10.1901/jaba.2002.35-213
Cummings A., 2000, J DEV DISABILITIES, V7, P109
DOOLEY P, 2001, J POSITIVE BEHAV ANA, V35, P213
Dunst C. J., 2002, CENTERSCOPE, V1, P1
Frost L, 2002, PECS PICTURE EXCHANG
Ganz JB, 2004, J AUTISM DEV DISORD, V34, P395, DOI 10.1023/B:JADD.0000037416.59095.d7
HENEKER S, 2003, SPEECH LANGUAGE THER, P12
JONES CM, 2005, DISS ABSTR INT, V65, P4270
Kravits TR, 2002, J AUTISM DEV DISORD, V32, P225, DOI 10.1023/A:1015457931788
Liddle K, 2001, INT J LANG COMM DIS, V36, P391
Magiati I, 2003, AUTISM, V7, P297, DOI 10.1177/1362361303007003006
Mirenda P., 2001, FOCUS AUTISM OTHER D, V16, P141, DOI DOI 10.1177/108835760101600302
MIRENDA P, 2000, AUTISM SPECTRUM DISO, P333
National Research Council, 2001, ED CHILDR AUT
Schwartz IS, 1998, TOP EARLY CHILD SPEC, V18, P144
Tincani M., 2004, FOCUS AUTISM OTHER D, V19, P152, DOI DOI 10.1177/10883576040190030301
Vittimberga G. L, 2001, J POSIT BEHAV INTERV, V3, P194, DOI 10.1177/109830070100300401
NR 22
TC 8
Z9 8
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD MAR
PY 2008
VL 43
IS 1
BP 61
EP 76
PG 16
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 264NK
UT WOS:000253294400005
ER
PT J
AU Boyd, CM
Fraiman, JL
Hawkins, KA
Labin, JM
Sutter, MB
Wahl, MR
AF Boyd, Christina M.
Fraiman, Jeffrey L.
Hawkins, Kelly A.
Labin, Jennifer M.
Sutter, Mary Beth
Wahl, Meghan R.
TI Effects of the STAR intervention program on interactions between campers
with and without disabilities during inclusive summer day camp
activities
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID CHILDREN; PRESCHOOLERS; AUTISM; SKILLS
AB The purpose of this study was to examine the effects of a peer intervention program designed to increase interactions between children with and without disabilities in an inclusive summer camp. A multiple probe single subject design was used to determine the effects of the STAR intervention on six dyads of campers aged five through ten over two week sessions. Each dyad consisted of one camper with a mild to moderate disability and one camper without a disability. The results showed an overall increase in the number of interactions and demonstrated that the STAR program was effective in increasing interactions between campers with and without disabilities. Factors contributing to the success of the intervention are discussed as well as limitations.
C1 [Boyd, Christina M.; Fraiman, Jeffrey L.; Hawkins, Kelly A.; Labin, Jennifer M.; Sutter, Mary Beth; Wahl, Meghan R.] Univ Maryland, Dept Special Educ, College Pk, MD 20742 USA.
RP Boyd, CM (reprint author), Univ Maryland, Dept Special Educ, 1308 Benjamin Bldg, College Pk, MD 20742 USA.
CR Anderson L., 1997, Therapeutic Recreation Journal, V31, P214
Bedini L. A., 2000, Therapeutic Recreation Journal, V34, P55
Devine MA, 2004, J LEISURE RES, V36, P137
English K., 1996, TEACHING EXCEPTIONAL, V28, P62
English K, 1997, EXCEPT CHILDREN, V63, P229
Garfinkle AN, 2002, TOP EARLY CHILD SPEC, V22, P26, DOI 10.1177/027112140202200103
GOLDSTEIN H, 1992, J APPL BEHAV ANAL, V25, P289, DOI 10.1901/jaba.1992.25-289
Goldstein H, 1997, J SPEECH LANG HEAR R, V40, P33
Gonzalez-Lopez A., 1997, FOCUS AUTISM OTHER D, V12, P2
Herbert J. T., 2000, Therapeutic Recreation Journal, V34, P211
HUNDERT J, 1992, EXCEPT CHILDREN, V58, P311
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SASSO GM, 1998, FOCUS AUTISM OTHER D, V13, P2
Schleien S, 1996, THERAPEUTIC RECREATI, V30, P260
NR 16
TC 0
Z9 0
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD MAR
PY 2008
VL 43
IS 1
BP 92
EP 101
PG 10
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 264NK
UT WOS:000253294400007
ER
PT J
AU Bakan, MB
Koen, B
Kobylarz, F
Morgan, L
Goff, R
Kahn, S
Bakan, M
AF Bakan, Michael B.
Koen, Benjamin
Kobylarz, Fred
Morgan, Lindee
Goff, Rachel
Kahn, Sally
Bakan, Megan
TI Following Frank: Response-ability and the co-creation of culture in a
medical ethnomusicology program for children on the autism spectrum
SO ETHNOMUSICOLOGY
LA English
DT Article
ID MUSIC-THERAPY; ASPERGER-SYNDROME; SCERTS MODEL; PLAY; COMMUNICATION;
INTERVENTION; REFLECTIONS; COMPETENCE; DISORDERS; DISCOURSE
C1 [Bakan, Michael B.; Koen, Benjamin; Morgan, Lindee; Bakan, Megan] Florida State Univ, Ctr Autism & Related Disabil, Tallahassee, FL 32306 USA.
[Kobylarz, Fred] Robert Wood Johnson Med Sch, Piscataway, NJ 08854 USA.
[Goff, Rachel] Univ N Carolina, Chapel Hill, NC USA.
[Kahn, Sally] Vanderbilt Univ, Nashville, TN USA.
RP Bakan, MB (reprint author), Florida State Univ, Ctr Autism & Related Disabil, Tallahassee, FL 32306 USA.
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American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 74
TC 11
Z9 11
PU SOC ETHNOMUSICOLOGY INC
PI BLOOMINGTON
PA MORRISON HALL, ROOM 005 INDIANA UNIVERSITY, BLOOMINGTON, IN 47405 USA
SN 0014-1836
J9 ETHNOMUSICOLOGY
JI Ethnomusicology
PD SPR-SUM
PY 2008
VL 52
IS 2
BP 163
EP 202
PG 40
WC Music
SC Music
GA 303BY
UT WOS:000256015700002
ER
PT J
AU Sinzig, J
Morsch, D
Lehmkuhl, G
AF Sinzig, Judith
Morsch, Dagmar
Lehmkuhl, Gerd
TI Do hyperactivity, impulsivity and inattention have an impact on the
ability of facial affect recognition in children with autism and ADHD?
SO EUROPEAN CHILD & ADOLESCENT PSYCHIATRY
LA English
DT Article
DE ADHD; autism; emotion recognition; facial affect recognition
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; SPECTRUM DISORDERS;
PSYCHIATRIC-DISORDERS; ASPERGER-SYNDROME; CORPUS-CALLOSUM; DEFICIT;
EMOTION; MIND; PERSPECTIVES; EXPRESSIONS
AB Psychopathological, genetic and neuropsychological findings indicate an association between autism and attention deficit/hyperactivity disorder (ADHD). The goal of this study was to assess possible differences in facial affect recognition in children with autism (with and without comorbid ADHD), with ADHD and healthy controls. Children aged 6-18 years old with DSM-IV-diagnosis ADHD (n = 30) or autism (n = 40) were included consecutively in the study. Facial affect recognition was assessed with a computer-based program used for teaching emotion processing called the Frankfurt Test and Training of Social Affect (FEFA) using faces and eye-pairs as target material. Additionally three attention-tasks (Sustained attention, Inhibition, Set-Shifting) were administered. Approximately 52% of the autistic children met the criteria for the comorbid diagnosis of ADHD. A MANOVA with post-hoc Scheffe tests revealed a significant difference in the recognition of faces and eye pairs between the group ADHD and controls (P = 0.009). Children with autism and ADHD also differed significantly from healthy participants in the recognition of eye-pairs (P = 0.009). Neither correlations with PDD nor with ADHD symptom scores were able to explain these results. Sustained attention and inhibition deficits had a significant influence on emotion recognition in children with ADHD. Our findings imply that the ability of facial affect recognition is reduced in children suffering from ADHD symptoms, both in autistic and pure ADHD children. ADHD symptoms need to be taken into account in future studies assessing emotion recognition in autistic children and adolescents.
C1 [Sinzig, Judith; Morsch, Dagmar; Lehmkuhl, Gerd] Univ Cologne, Dept Child & Adolescent Psychiat & Psychotherapy, D-50931 Cologne, Germany.
RP Sinzig, J (reprint author), Univ Cologne, Dept Child & Adolescent Psychiat & Psychotherapy, Robert Koch Str 10, D-50931 Cologne, Germany.
EM ju.k.sinzig@web.de
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NR 56
TC 54
Z9 55
PU DR DIETRICH STEINKOPFF VERLAG
PI DARMSTADT
PA PO BOX 10 04 62, D-64204 DARMSTADT, GERMANY
SN 1018-8827
J9 EUR CHILD ADOLES PSY
JI Eur. Child Adolesc. Psych.
PD MAR
PY 2008
VL 17
IS 2
BP 63
EP 72
DI 10.1007/s00787-007-0637-9
PG 10
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 280CO
UT WOS:000254402700001
PM 17896119
ER
PT J
AU Gilling, M
Lauritsen, MB
Moller, M
Henriksen, KF
Vicente, A
Oliveira, G
Cintin, C
Eiberg, H
Andersen, PS
Mors, O
Rosenberg, T
Brondum-Nielsen, K
Cotterill, RMJ
Lundsteen, C
Ropers, HH
Ullmann, R
Bache, I
Tumer, Z
Tommerup, N
AF Gilling, Mette
Lauritsen, Marlene Briciet
Moller, Morten
Henriksen, Karen Friis
Vicente, Astrid
Oliveira, Guiomar
Cintin, Christina
Eiberg, Hans
Andersen, Paal Skyt
Mors, Ole
Rosenberg, Thomas
Brondum-Nielsen, Karen
Cotterill, Rodney M. J.
Lundsteen, Claes
Ropers, Hans-Hilger
Ullmann, Reinhard
Bache, Iben
Tuemer, Zeynep
Tommerup, Niels
TI A 3.2Mb deletion on 18q12 in a patient with childhood autism and
high-grade myopia
SO EUROPEAN JOURNAL OF HUMAN GENETICS
LA English
DT Article
DE translocation; deletion; autism; myopia; array CGH; chromosome 18
ID PERVASIVE DEVELOPMENTAL DISORDERS; DROSOPHILA-MELANOGASTER; HUMAN
GENOME; SPECTRUM DISORDERS; MENTAL-RETARDATION; BIPOLAR DISORDER;
FAMILY-HISTORY; COPY NUMBER; LONG ARM; EXPRESSION
AB Autism spectrum disorders (ASDs) are a heterogeneous group of disorders with unknown aetiology. Even though ASDs are suggested to be among the most heritable complex disorders, only a few reproducible mutations leading to susceptibility for ASD have been identified. In an attempt to identify ASD susceptibility genes through chromosome rearrangements, we investigated a female patient with childhood autism and high-grade myopia, and an apparently balanced de novo translocation, t(5; 18)(q34; q12.2). Further analyses revealed a 3.2Mb deletion encompassing 17 genes at the 18q break point and an additional deletion of 1.27Mb containing two genes on chromosome 4q35. Q-PCR analysis of 14 of the 17 genes deleted on chromosome 18 showed that 11 of these genes were expressed in the brain, suggesting that haploinsufficiency of one or more genes may have contributed to the childhood autism phenotype of the patient. Identification of multiple genetic changes in this patient with childhood autism agrees with the most frequently suggested genetic model of ASDs as complex, polygenic disorders.
C1 [Gilling, Mette; Henriksen, Karen Friis; Bache, Iben; Tuemer, Zeynep; Tommerup, Niels] Univ Copenhagen, Inst Cellular & Mol Med, Dept Med Genet, Wilhelm Johannsen Ctr Funct Genome Res, DK-2200 Copenhagen N, Denmark.
[Lauritsen, Marlene Briciet; Mors, Ole] Aarhus Univ Hosp, Ctr Psychiat Res, Riskov, Denmark.
[Lauritsen, Marlene Briciet] Univ Aarhus, Inst Publ Hlth, Dept Epidemiol, NANEA, Aarhus, Denmark.
[Moller, Morten] Univ Copenhagen, Dept Med Anat, DK-2200 Copenhagen, Denmark.
[Vicente, Astrid] Gulbenkian Inst Sci, Oeiras, Portugal.
[Vicente, Astrid] Inst Nacl Saude Dr Ricardo Jorge, Lisbon, Portugal.
[Oliveira, Guiomar] Hosp Pediat Coimbra, Coimbra, Portugal.
[Cintin, Christina] Hosp Psychiat, Hillerod, Denmark.
[Eiberg, Hans] Univ Copenhagen, Inst Mol & Cellular Med, Dept Med Genet, DK-2200 Copenhagen N, Denmark.
[Andersen, Paal Skyt] Statens Serum Inst, Dept Clin Biochem, DK-2300 Copenhagen S, Denmark.
[Rosenberg, Thomas; Brondum-Nielsen, Karen] John F Kennedy Inst, Natl Eye Clin, DK-2600 Glostrup, Denmark.
[Cotterill, Rodney M. J.] Danish Tech Univ, Dept Phys, Biophys Grp, Lyngby, Denmark.
[Lundsteen, Claes] Univ Copenhagen, Rigshosp, Dept Clin Genet, DK-2100 Copenhagen, Denmark.
[Ropers, Hans-Hilger; Ullmann, Reinhard] Max Planck Inst Mol Genet, Berlin, Germany.
RP Gilling, M (reprint author), Univ Copenhagen, Inst Cellular & Mol Med, Dept Med Genet, Wilhelm Johannsen Ctr Funct Genome Res, Blegdamsvej 3B, DK-2200 Copenhagen N, Denmark.
EM mette@imbg.ku.dk
RI Oliveira, Guiomar/I-7255-2013
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NR 56
TC 6
Z9 7
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1018-4813
J9 EUR J HUM GENET
JI Eur. J. Hum. Genet.
PD MAR
PY 2008
VL 16
IS 3
BP 312
EP 319
DI 10.1038/sj.ejhg.5201985
PG 8
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 266BS
UT WOS:000253406900006
PM 18183041
ER
PT J
AU Chou, IJ
Lin, KL
Wong, AM
Wang, HS
Chou, ML
Hung, PC
Hsieh, MY
Chang, MY
AF Chou, I-Jun
Lin, Kuang-Lin
Wong, Alex M.
Wang, Huei-Shyong
Chou, Min-Liang
Hung, Po-Cheng
Hsieh, Meng-Ying
Chang, Mingy Yu
TI Neuroimaging correlation with neurological severity in tuberous
sclerosis complex
SO EUROPEAN JOURNAL OF PAEDIATRIC NEUROLOGY
LA English
DT Article
DE tuberous sclerosis complex; neurological severity; neuroimaging
ID MOLECULAR PATHOGENESIS; SUBEPENDYMAL NODULES; INFANTILE SPASMS; AUTISM;
PROGNOSIS; SEIZURES; LOCATION; LESIONS; COUNT
AB Objective: To delineate the relationship between neurological severity and neuroimage of lesion load including specific topography of supratentorial cortical tubers and white matter lesions in tuberous sclerosis complex (TSC).
Methods: Twenty-five TSC patients more than 2 years of age who underwent conventional and fluid-attenuated inversion recovery sequence (FLAIR) magnetic resonance imaging (MRI) were retrospectively studied. Neurological severity score was designated for three items: seizure, developmental delay and/or mental retardation, and autism. A neuroimaging scoring system was designed to evaluate the load of the cerebrum lesions with respect to location and size of cortical tubers and white matter lesions based on FLAIR MRI.
Results: A linear trend was observed between MRI lesion score and neurological severity score (r = 0.511; p = 0.009). The lesion score in the left temporal lobe has positive correlation to neurological severity score (r = 0.609; p = 0.001).
Conclusions: The brain lesion load was positively correlated with neurological prognosis in TSC patients. Patients with larger lesion load in the left temporal lobe may be correlated with increased neurological severity in right-handed patients with TSC. (C) 2007 Published by Elsevier Ltd. on behalf of European Paediatric Neurology Society.
C1 [Chou, I-Jun; Lin, Kuang-Lin; Wang, Huei-Shyong; Chou, Min-Liang; Hung, Po-Cheng; Hsieh, Meng-Ying; Chang, Mingy Yu] Chang Gung Childrens Hosp, Div Pediat Neurol, Tao Yuan 333, Taiwan.
[Wong, Alex M.] Chang Gung Univ Coll Med, Chang Gung Mem Hosp, Dept Diagnost Radiol, Div Neurodiol, Tao Yuan, Taiwan.
RP Wang, HS (reprint author), Chang Gung Childrens Hosp, Div Pediat Neurol, 5 Fu Shin St, Tao Yuan 333, Taiwan.
EM wanghs444@cgmh.org.tw
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NR 32
TC 14
Z9 15
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1090-3798
J9 EUR J PAEDIATR NEURO
JI Eur. J. Paediatr. Neurol.
PD MAR
PY 2008
VL 12
IS 2
BP 108
EP 112
DI 10.1016/j.ejpn.2007.07.002
PG 5
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 278PM
UT WOS:000254298800008
PM 17869556
ER
PT J
AU Murray, DS
Creaghead, NA
Manning-Courtney, P
Shear, PK
Bean, J
Prendeville, JA
AF Murray, Donna S.
Creaghead, Nancy A.
Manning-Courtney, Patricia
Shear, Paula K.
Bean, Judy
Prendeville, Jo-Anne
TI The Relationship Between Joint Attention and Language in Children With
Autism Spectrum Disorders
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE joint attention; communication; autism spectrum disorders; language
AB The relationship between initiation of and response to joint attention and components of receptive and expressive language in 20 children with autism between the ages of 3 and 5 years were examined. Receptive language skills were assessed using the Mullen Scales of Early Learning (MSEL). Expressive language skills were evaluated by examining Mean Length of Utterance and Type Token Ratio. These variables, along with data on responses to joint attention bids and initiation of joint attention were analyzed using Spearman calculations. The ability to respond to the joint attention bids of others was positively correlated with receptive language scores on the MSEL and mean length of utterance in children with autism. There was no relationship between the ability to initiate joint attention and the selected components of language examined.
C1 [Murray, Donna S.] Univ Cincinnati, Cincinnati Childrens Hosp, Med Ctr, Cincinnati, OH 45221 USA.
[Creaghead, Nancy A.] Univ Cincinnati, Dept Commun Sci & Disorders, Cincinnati, OH 45221 USA.
[Manning-Courtney, Patricia] Cincinnati Childrens Hosp, Med Ctr, Kelly OLeary Ctr Autism Spectrum Disorders, Cincinnati, OH USA.
RP Murray, DS (reprint author), Cincinnati Childrens Med Ctr, Div Dev & Behav Pediat, Kelly OLeary Ctr Autism Spectrum Disorders, 3333 Burnet Ave,ML 4002, Cincinnati, OH 45229 USA.
EM donna.murray@cchmc.org
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NR 50
TC 9
Z9 10
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD MAR
PY 2008
VL 23
IS 1
BP 5
EP 14
DI 10.1177/1088357607311443
PG 10
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10FY
UT WOS:000207451100001
ER
PT J
AU Owen-DeSchryver, JS
Carr, EG
Cale, SI
Blakeley-Smith, A
AF Owen-DeSchryver, Jamie S.
Carr, Edward G.
Cale, Sanja I.
Blakeley-Smith, Audrey
TI Promoting Social Interactions Between Students With Autism Spectrum
Disorders and Their Peers in Inclusive School Settings
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism; social skills; peer training; social initiations; schools
AB This study evaluated the impact of a peer training intervention on social interactions among three students with autism spectrum disorders (ASD) and their typical peers. Two second graders and one fourth grader with ASD participated. For each student with ASD, two to four typical peers participated in training sessions that targeted increased social interactions. Data collected during lunchtime and recess showed that the peer training intervention generally resulted in increased initiations by trained peers as well as increased initiations and responses by students with ASD. Unexpectedly, untrained peers also showed increased initiations. Future research directions are discussed, including characteristics of the peers selected for training ( e. g., gender, popularity) and measurement of qualitative changes in social relationships and opportunities.
C1 [Owen-DeSchryver, Jamie S.] Grand Valley State Univ, Allendale, MI 49401 USA.
[Carr, Edward G.] SUNY Stony Brook, Dept Psychol, Stony Brook, NY 11794 USA.
[Cale, Sanja I.] Long Isl Univ, Long Isl City, NY USA.
[Cale, Sanja I.] Inst Children Autism, Long Isl City, NY USA.
[Blakeley-Smith, Audrey] Univ Colorado Denver, Hlth Sci Ctr, Dept Psychiat, Denver, CO USA.
RP Owen-DeSchryver, JS (reprint author), Grand Valley State Univ, 2195 Au Sable Hall, Allendale, MI 49401 USA.
EM owendesj@gvsu.edu
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NR 39
TC 32
Z9 33
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD MAR
PY 2008
VL 23
IS 1
BP 15
EP 28
DI 10.1177/1088357608314370
PG 14
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10FY
UT WOS:000207451100002
ER
PT J
AU Sulzer-Azaroff, B
Fleming, R
Tupa, M
Bass, R
Hamad, C
AF Sulzer-Azaroff, Beth
Fleming, Richard
Tupa, Megan
Bass, Robert
Hamad, Charles
TI Choosing Objectives for a Distance Learning Behavioral Intervention in
Autism Curriculum
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE applied behavior analysis; autism spectrum disorders; instruction;
personnel preparation
AB The national need for individuals trained in applied behavior analysis (ABA) helped persuade the U. S. Department of Education to fund the development of a four-course, undergraduate-level curriculum on the topic. In this article, the authors report the results and application of a four-phase Delphi survey procedure designed to identify the most critical topics to cover in the curriculum. Thirty-four specialists in behavioral intervention participated and identified 74 items, 36 of which eventually emerged as strongly recommended, 27 as recommended and 11 as not recommended. This information was used to select topics and practices to cover in the courses. Other ABA scholars, researchers, and practitioners may find this information useful in designing instructional programs for adults.
C1 [Fleming, Richard; Bass, Robert; Hamad, Charles] Univ Massachusetts, Sch Med, EK Shriver Ctr, Waltham, MA USA.
[Tupa, Megan] SchoolWorks Inc, Beverly, MA USA.
RP Sulzer-Azaroff, B (reprint author), 5555 Heron Pt Dr,Unit 1102, Naples, FL 34108 USA.
EM bazaroff@comcast.net
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Comish E., 1977, STUDY FUTURE
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National Research Council, 2001, ED CHILDR AUT
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2002, J AUTISM DEV DISORDE, V32
NR 20
TC 1
Z9 2
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD MAR
PY 2008
VL 23
IS 1
BP 29
EP 36
DI 10.1177/1088357607311442
PG 8
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10FY
UT WOS:000207451100003
ER
PT J
AU Meadan, H
Halle, J
Ostrosky, MM
DeStefano, L
AF Meadan, Hedda
Halle, James
Ostrosky, Michaelene M.
DeStefano, Lizanne
TI Communicative Behavior in the Natural Environment Case Studies of Two
Young Children With Autism and Limited Expressive Language
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE communicative functions; communication; autism spectrum disorders; young
children; nonverbal
AB Many individuals with developmental disabilities show delays in the development of sophisticated communication skills. Listeners often misunderstand their unconventional means of communication, and breakdowns occur. A communication repair is a second attempt to communicate a message when a breakdown is encountered. Qualitative methods were employed to assess and describe the communication repairs used by two young children with autism spectrum disorders who had limited expressive language. During natural observations, only one of the two participating children was observed to repair his initial requests. The results suggest that in contrast to the emphasis in the literature that development is the primary determinant of repair behavior, variables in the children's social and physical environment influenced their communication repair behavior. Implications for practice and research are described.
C1 [Meadan, Hedda] Illinois State Univ, Normal, IL 61790 USA.
[DeStefano, Lizanne] Univ Illinois, Bur Educ Res, Urbana, IL 61801 USA.
RP Meadan, H (reprint author), Illinois State Univ, Campus Box 5910, Normal, IL 61790 USA.
EM hmeadan@ilstu.edu
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NR 41
TC 2
Z9 2
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD MAR
PY 2008
VL 23
IS 1
BP 37
EP 48
DI 10.1177/1088357607311444
PG 12
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10FY
UT WOS:000207451100004
ER
PT J
AU Simpson, RL
AF Simpson, Richard L.
TI Children and Youth with Autism Spectrum Disorders: The Search for
Effective Methods
SO FOCUS ON EXCEPTIONAL CHILDREN
LA English
DT Article
ID FACILITATED COMMUNICATION; INTERVENTION; THIMEROSAL; EDUCATION; TEACHERS
C1 Univ Kansas, Dept Special Educ, Lawrence, KS 66045 USA.
RP Simpson, RL (reprint author), Univ Kansas, Dept Special Educ, Lawrence, KS 66045 USA.
CR Biklen D., 1993, COMMUNICATION UNBOUN
BROPHY J, 1988, TEACH TEACH EDUC, V4, P1, DOI 10.1016/0742-051X(88)90020-0
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*CDCP, 2008, AUT INF CTR
Christie K, 2008, PHI DELTA KAPPAN, V89, P629
CHRISTIE K, 2008, PHI DELTA KAPPAN, V89, P703
*COUNC EXC CHILDR, 2008, NEW HOP AUT
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FIEDLER C, 2007, PARENTS FAMILIES CHI
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National Research Council, 2001, ED CHILDR AUT
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THIEMANN K, 2008, ED CHILDREN YOUTH AU, P267
*US DEP ED I ED SC, 2008, WHAT WORKS CLEAR
Volkmar Fred, 1999, Journal of the American Academy of Child and Adolescent Psychiatry, V38, p32S
WALLIS C, 2006, TIME 0515, P43
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WHEELER DL, 1993, MENT RETARD, V31, P49
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NR 56
TC 11
Z9 11
PU LOVE PUBLISHING COMPANY
PI DENVER
PA 9101 EAST KENYON AVENUE, STE 2200, DENVER, CO 80237 USA
SN 0015-511X
J9 FOCUS EXCEPT CHILD
JI Focus Except. Child
PD MAR
PY 2008
VL 40
IS 7
BP 1
EP 14
PG 14
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 400MW
UT WOS:000262873300001
ER
PT J
AU Huerter, HE
Gantert, M
van Kooten, IAJ
Steinbusch, HWM
van Engeland, H
Garnier, Y
Hof, PR
Kramer, BW
Mallmann, P
Schmitz, C
AF Huerter, H. E.
Gantert, M.
van Kooten, I. A. J.
Steinbusch, H. W. M.
van Engeland, H.
Garnier, Y.
Hof, P. R.
Kramer, B. W.
Mallmann, P.
Schmitz, C.
TI Additional number of cerebellar granular cells after prenatal
LPS-application for rest after amniocentesis - A possible new partial
model for study of autism
SO GEBURTSHILFE UND FRAUENHEILKUNDE
LA German
DT Meeting Abstract
C1 [Huerter, H. E.] Klinikum Univ Koln, Geburtshilfe & Frauenheilkunde, Cologne, Germany.
[Gantert, M.; Garnier, Y.; Mallmann, P.] Klinikum Univ Koln, Klin & Poliklin Frauenheilkunde & Geburstshilfe, Cologne, Germany.
[van Kooten, I. A. J.; van Engeland, H.] Univ Utrecht, Med Ctr, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, Utrecht, Netherlands.
[Steinbusch, H. W. M.; Schmitz, C.] Univ Maastricht, Dept Neurosci, European Grad Sch Neurosci, Maastricht, Netherlands.
[Hof, P. R.] Mt Sinai Sch Med, Dept Neurosci, New York, NY USA.
[Kramer, B. W.] Univ Hosp Maastricht, Dept Pediat & Neonatol, Maastricht, Netherlands.
RI Mallmann, Peter/B-1220-2010
NR 0
TC 0
Z9 0
PU GEORG THIEME VERLAG KG
PI STUTTGART
PA RUDIGERSTR 14, D-70469 STUTTGART, GERMANY
SN 0016-5751
J9 GEBURTSH FRAUENHEILK
JI Geburtshilfe Frauenheilkd.
PD MAR
PY 2008
VL 68
IS 3
BP 301
EP 302
PG 2
WC Obstetrics & Gynecology
SC Obstetrics & Gynecology
GA 290LI
UT WOS:000255123900016
ER
PT J
AU McFarlane, HG
Kusek, GK
Yang, M
Phoenix, JL
Bolivar, VJ
Crawley, JN
AF McFarlane, H. G.
Kusek, G. K.
Yang, M.
Phoenix, J. L.
Bolivar, V. J.
Crawley, J. N.
TI Autism-like behavioral phenotypes in BTBR T+tf/J mice
SO GENES BRAIN AND BEHAVIOR
LA English
DT Article
DE anxiety; autism; gene; grooming; inbred strain; kynurenic acid;
olfaction; repetitive behaviors; social behaviors; social communication;
single nucleotide polymorphism
ID INBRED MOUSE STRAINS; SOCIAL APPROACH BEHAVIORS; CORPUS-CALLOSUM;
KYNURENIC ACID; DEVELOPMENTAL DISORDERS; SPECTRUM DISORDERS; TRANSGENIC
MICE; TASKS RELEVANT; KNOCKOUT MICE; GENE
AB Autism is a behaviorally defined neurodevelopmental disorder of unknown etiology. Mouse models with face validity to the core symptoms offer an experimental approach to test hypotheses about the causes of autism and translational tools to evaluate potential treatments. We discovered that the inbred mouse strain BTBR T+tf/J (BTBR) incorporates multiple behavioral phenotypes relevant to all three diagnostic symptoms of autism. BTBR displayed selectively reduced social approach, low reciprocal social interactions and impaired juvenile play, as compared with C57BL/6J (B6) controls. Impaired social transmission of food preference in BTBR suggests communication deficits. Repetitive behaviors appeared as high levels of self-grooming by juvenile and adult BTBR mice. Comprehensive analyses of procedural abilities confirmed that social recognition and olfactory abilities were normal in BTBR, with no evidence for high anxiety-like traits or motor impairments, supporting an interpretation of highly specific social deficits. Database comparisons between BTBR and B6 on 124 putative autism candidate genes showed several interesting single nucleotide polymorphisms (SNPs) in the BTBR genetic background, including a nonsynonymous coding region polymorphism in Kmo. The Kmo gene encodes kynurenine 3-hydroxylase, an enzyme-regulating metabolism of kynurenic acid, a glutamate antagonist with neuroprotective actions. Sequencing confirmed this coding SNP in Kmo, supporting further investigation into the contribution of this polymorphism to autism-like behavioral phenotypes. Robust and selective social deficits, repetitive self-grooming, genetic stability and commercial availability of the BTBR inbred strain encourage its use as a research tool to search for background genes relevant to the etiology of autism, and to explore therapeutics to treat the core symptoms.
C1 [McFarlane, H. G.; Yang, M.; Crawley, J. N.] NIMH, Lab Behav Neurosci, Intramural Res Program, NIH, Bethesda, MD 20892 USA.
[McFarlane, H. G.] Kenyon Coll, Dept Psychol, Gambier, OH 43022 USA.
[Kusek, G. K.; Phoenix, J. L.] New York State Dept Hlth, Wadsworth Ctr, Troy, NY USA.
[Bolivar, V. J.] SUNY Albany, Sch Publ Hlth, Dept Biomed Sci, Albany, NY 12222 USA.
[Crawley, J. N.] Univ N Carolina, Neurodev Disorders Res Ctr, Chapel Hill, NC USA.
RP Crawley, JN (reprint author), NIMH, Lab Behav Neurosci, Intramural Res Program, NIH, Bldg 35 Room 1C-903, Bethesda, MD 20892 USA.
EM crawleyj@intra.nimh.nih.gov
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NR 81
TC 198
Z9 200
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1601-1848
J9 GENES BRAIN BEHAV
JI Genes Brain Behav.
PD MAR
PY 2008
VL 7
IS 2
BP 152
EP 163
DI 10.1111/j.1601-183X.2007.00330.x
PG 12
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 270ZN
UT WOS:000253758800002
PM 17559418
ER
PT J
AU Leskovec, TJ
Rowles, BM
Findling, RL
AF Leskovec, Thomas J.
Rowles, Brieana M.
Findling, Robert L.
TI Pharmacological treatment options for autism spectrum disorders in
children and adolescents
SO HARVARD REVIEW OF PSYCHIATRY
LA English
DT Review
DE autism; pervasive developmental disorder; treatment
ID PERVASIVE DEVELOPMENTAL DISORDERS; ATTENTION-DEFICIT/HYPERACTIVITY
DISORDER; OBSESSIVE-COMPULSIVE DISORDER; PLACEBO-CONTROLLED CROSSOVER;
OPEN-LABEL TRIAL; DEFICIT HYPERACTIVITY DISORDER; RANDOMIZED
CONTROLLED-TRIAL; SELF-INJURIOUS-BEHAVIOR; DOUBLE-BLIND; SCHIZOPHRENIC
CHILDREN
AB Autism and other pervasive developmental disorders (PDDs) are frequently associated with dysfunctional behaviors and are characterized by deficits in socialization, communication, and behavioral rigidity. Despite the absence of a pharmacological cure for PDDs, many of the dysfunctional, coinciding behaviors may be treated pharmacologically. This article reviews what is known about the efficacy and tolerability of pharmacological interventions for the treatment of children and adolescents suffering from autistic spectrum disorders.
C1 [Leskovec, Thomas J.; Rowles, Brieana M.; Findling, Robert L.] Case Western Reserve Univ, Sch Med, Dept Psychiat, Cleveland, OH 44106 USA.
[Findling, Robert L.] Univ Hosp, Case Med Ctr, Cleveland, OH USA.
RP Rowles, BM (reprint author), Case Western Reserve Univ, Sch Med, Dept Psychiat, 11400 Euclid Ave, Cleveland, OH 44106 USA.
EM Brieana.Rowles@UHhospitals.org
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NR 116
TC 31
Z9 32
PU TAYLOR & FRANCIS INC
PI PHILADELPHIA
PA 325 CHESTNUT ST, SUITE 800, PHILADELPHIA, PA 19106 USA
SN 1067-3229
J9 HARVARD REV PSYCHIAT
JI Harv. Rev. Psychiatr.
PD MAR-APR
PY 2008
VL 16
IS 2
BP 97
EP 112
DI 10.1080/10673220802075852
PG 16
WC Psychiatry
SC Psychiatry
GA 288QH
UT WOS:000255000400004
PM 18415882
ER
PT J
AU Dworzynski, K
Ronald, A
Hayiou-Thomas, ME
McEwan, F
Happe, F
Bolton, P
Plomin, R
AF Dworzynski, Katharina
Ronald, Angelica
Hayiou-Thomas, Marianna E.
McEwan, Fiona
Happe, Francesca
Bolton, Patrick
Plomin, Robert
TI Developmental path between language and autistic-like impairments: A
twin study
SO INFANT AND CHILD DEVELOPMENT
LA English
DT Article
DE autism spectrum disorders; early language development
ID CAST CHILDHOOD ASPERGER; GENERAL-POPULATION; BEHAVIOR PROBLEMS; GENETIC
ETIOLOGY; COMORBIDITY; TRAITS; MULTIVARIATE; MATHEMATICS; DISABILITY;
COGNITION
AB Autism spectrum disorders (ASDs) are diagnosed when individuals show impairments in three behavioural domains: communication, social interactions, and repetitive, restrictive behaviours and interests (RRBIs). Recent data suggest that these three sets of behaviours are genetically heterogeneous. Early language delay is strongly associated with ASD, but the basis for this association and the relationship with individual sub-domains of ASD has not been systematically investigated. In the present study, data came from a population-based twin sample with language development data at 2-4 years, measured by the MacArthur Communicative Development Inventory (MCDI), and data at 8 years using the Childhood Asperger Syndrome Test (CAST). For the total CAST and the three subscales at 8 years, approximately 300 same-sex twin pairs were selected as showing extreme autistic-like traits (ALTs), defined here as pairs in which at least one member of the twin pair scored in the highest 5% of the distribution. Phenotypic analyses indicated that children showing extreme social and communication ALTs (but not the RRBI subscale) at 8 years were below average in language development at 2-4 years. A regression model for selected twin data suggested that genetic influences account for this overlap, but that these effects are only in part mediated by genes that are shared between language and extreme autistic traits. Copyright (C) 2008 John Wiley & Sons, Ltd.
C1 [Dworzynski, Katharina; Ronald, Angelica; McEwan, Fiona; Happe, Francesca; Bolton, Patrick; Plomin, Robert] Kings Coll London, Inst Psychiat, London, England.
[Hayiou-Thomas, Marianna E.] Univ York, Dept Psychol, York YO10 5DD, N Yorkshire, England.
RP Dworzynski, K (reprint author), Newcomen Ctr, Inst Psychiat, St Thomas St, London SE1 9RT, England.
EM k.dworzynski@iop.kcl.ac.uk
RI Hayiou-Thomas, Marianna/B-6465-2009; Happe, Francesca/D-5544-2012;
Ronald, Angelica/C-7812-2009; McEwen, Fiona/H-8966-2012; Bolton,
Patrick/E-8501-2010; Plomin, Robert/B-8911-2008
OI Ronald, Angelica/0000-0002-9576-2176; McEwen, Fiona/0000-0002-5562-481X;
Bolton, Patrick/0000-0002-5270-6262;
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TC 8
Z9 9
PU JOHN WILEY & SONS LTD
PI CHICHESTER
PA THE ATRIUM, SOUTHERN GATE, CHICHESTER PO19 8SQ, W SUSSEX, ENGLAND
SN 1522-7227
J9 INFANT CHILD DEV
JI Infant Child Dev.
PD MAR-APR
PY 2008
VL 17
IS 2
BP 121
EP 136
DI 10.1002/icd.536
PG 16
WC Psychology, Developmental
SC Psychology
GA 288IX
UT WOS:000254980900003
ER
PT J
AU Lopez, C
Tchanturia, K
Stahl, D
Booth, R
Holliday, J
Treasure, J
AF Lopez, Carolina
Tchanturia, Kate
Stahl, Daniel
Booth, Rhonda
Holliday, Joanna
Treasure, Janet
TI An examination of the concept of central coherence in women with
anorexia nervosa
SO INTERNATIONAL JOURNAL OF EATING DISORDERS
LA English
DT Article
DE central coherence; anorexia; cognition; information processing; local
processing; global processing; neuropsychology
ID OBSESSIVE-COMPULSIVE DISORDER; EATING-DISORDERS; SPECTRUM DISORDERS;
NEURAL MECHANISMS; MEMORY IMPAIRMENT; ASPERGER-SYNDROME; AUTISM
SPECTRUM; PHENOTYPE; SCALE; HETEROGENEITY
AB Objective: To examine central coherence (local and global processing) in women with anorexia nervosa (AN).
Method: 42 women with AN and 42 healthy women (HC) completed neuropsychological testing measuring visuospatial and verbal aspects of central coherence: Rey-Osterrieth Complex Figure (RCFT), Embedded Figures Test (EFT), Homograph Reading Test (HRT), and Sentence Completion Task (SCT).
Results: People with AN displayed superior performance on the EFT and poorer performance in RCFT with the exception of accuracy in the copy trial. Long hesitations in the SCT were observed. Verbal coherence tasks were not sensitive enough to detect coherence anomalies in AN.
Conclusion: Women with AN have strengths in tasks requiring local processing (EFT) and weaknesses on tasks benefited by global processing (RCFT and SCT). These results are consistent with the weak central coherence account. This trait might play a role in the maintenance of AN and can be addressed in specific clinical interventions. (c) 2007 by Wiley Periodicals, Inc.
C1 [Lopez, Carolina; Tchanturia, Kate] Kings Coll London, Inst Psychiat, Div Psychol Med, London, England.
[Stahl, Daniel] Kings Coll London, Dept Biostat & Comp, London, England.
[Booth, Rhonda] Kings Coll London, SGDP Ctr, London, England.
[Holliday, Joanna] Warneford Hosp, Isis Educ Ctr, Oxford Doctoral Course Clin Psychol, Oxford OX3 7JX, England.
[Treasure, Janet] Guys Kings & St Thomass Med Sch, Dept Acad Psychiat, London, England.
RP Lopez, C (reprint author), Guys Hosp, Dept Acad Psychiat, 5th Floor,Thomas Guy House, London SE1 9RT, England.
EM c.lopez@iop.kcl.ac.uk
RI Stahl, Daniel/B-9713-2011; Tchanturia, Kate/H-1474-2011
OI Stahl, Daniel/0000-0001-7987-6619;
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NR 62
TC 78
Z9 79
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 0276-3478
J9 INT J EAT DISORDER
JI Int. J. Eating Disord.
PD MAR
PY 2008
VL 41
IS 2
BP 143
EP 152
DI 10.1002/eat.20478
PG 10
WC Psychology, Clinical; Nutrition & Dietetics; Psychiatry; Psychology
SC Psychology; Nutrition & Dietetics; Psychiatry
GA 263OL
UT WOS:000253226300006
PM 17937420
ER
PT J
AU Endo, A
AF Endo, Al
TI Intervention to change guidance provided by a teacher in a special
education classroom : Feedback to avoid the teacher's resistance
SO JAPANESE JOURNAL OF EDUCATIONAL PSYCHOLOGY
LA Japanese
DT Article
DE teacher's guidance; teacher's resistance to feedback; indirect feedback;
junior high school special education classroom; students with autism
ID CONSULTATION; IMPLEMENTATION; PERFORMANCE; SUPPORT
AB The present paper examines methods for providing effective feedback to a junior high school special education teacher, and the effect of those methods on how the teacher provided guidance to students with autistic disorders. In order to avoid the teacher developing a negative attitude toward the feedback, he was given 2 kinds of indirect feedback : written and videotaped. This evaluation was conducted not on the guidance provided to his students by the teacher, but rather by graduates in after-school care programs, an environment similar to the teacher's work environment. After the teacher received the feedback, it was observed that he provided better guidance to his students, and the students' behavior also improved. The teacher planned guidance for a specific student, however, other students in the class who were not directly involved in the guidance also participated positively in the class.
C1 Rikkyo Univ, Grad Sch Contemporary Psychol, Tokyo, Japan.
RP Endo, A (reprint author), Rikkyo Univ, Grad Sch Contemporary Psychol, Tokyo, Japan.
EM sister-moon@keh.biglobe.ne.jp
CR ALPER S, 2001, ALTERNATIVE ASSESSME
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NR 16
TC 1
Z9 1
PU JAPANESE ASSOC EDUCATIONAL PSYCHOLOGY
PI TOKYO
PA 5-24-6-7F HONGO, BUMKYO-KU, TOKYO, 113-0033, JAPAN
SN 0021-5015
J9 JPN J EDUC PSYCHOL
JI Jpn. J. Educ. Psychol.
PD MAR
PY 2008
VL 56
IS 1
BP 116
EP 126
PG 11
WC Psychology, Educational
SC Psychology
GA 296PM
UT WOS:000255558300011
ER
PT J
AU Zavaliy, AG
AF Zavaliy, Andrei G.
TI Absent, full and partial responsibility of the psychopaths
SO JOURNAL FOR THE THEORY OF SOCIAL BEHAVIOUR
LA English
DT Article
ID AUTISM; EMPATHY
AB The research into the typical behavioral pattern, motivational structure, and the value system of psychopaths can shed light on at least three aspects related to the analysis of the moral agency. First, it can help elucidating the emotive and cognitive conditions necessary for moral performance. Secondly, it can provide empirical evidence supporting the externalist theories of moral motivation. Finally, it can bring into greater focus our intuitive notion of the limits of moral responsibility. In this paper I shall concentrate on the last one-the question of responsibility of the amoralists, but the discussion will have an indirect bearing on the other two themes as well. My main reason for holding psychopaths morally responsible breaks down into two claims: the assumption that most ordinary people are morally responsible for their intentional actions (i.e., the rejection of hard determinism) and the denial that the psychopaths are qualitatively different from the non-psychopaths. This thesis is further defended against two objections. First, I am arguing that the genetically based emotive deficiency of the psychopaths cannot be seen the factor condemning them to amoral existence, and thus cannot be cited as an exempting condition. Secondly, my position is defended against the claim that psychopaths are partly responsible for their actions. It is argued that the notion of partial responsibility is either incoherent or else rests on a false empirical premise. My conclusion is in agreement with, and provides a theoretical justification for, the position of most classifications of the persons with antisocial personality disorder in the DSM IV.
C1 CUNY Hunter Coll, Dept Philosophy, New York, NY 10065 USA.
RP Zavaliy, AG (reprint author), CUNY Hunter Coll, Dept Philosophy, 695 Pk Ave, New York, NY 10065 USA.
EM azavaliy@hunter.cuny.edu
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NR 39
TC 2
Z9 2
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 0021-8308
J9 J THEOR SOC BEHAV
JI J. Theory Soc. Behav.
PD MAR
PY 2008
VL 38
IS 1
BP 87
EP +
DI 10.1111/j.1468-5914.2008.00358.x
PG 19
WC Psychology, Social
SC Psychology
GA 259TW
UT WOS:000252964800005
ER
PT J
AU Allam, H
Eldine, NG
Helmy, G
AF Allam, Hemat
Eldine, Nirvana Gamal
Helmy, Ghada
TI Scalp acupuncture effect on language development in children with
autism: A pilot study
SO JOURNAL OF ALTERNATIVE AND COMPLEMENTARY MEDICINE
LA English
DT Article; Proceedings Paper
CT International Congress on Complementary Medicine Research
CY MAY 11-13, 2007
CL Munich, GERMANY
AB Background and objectives: Autism is a neurodevelopmental disorder that manifests in delays in social interaction, language used in social communication, and symbolic or imaginative play, with an onset prior to age 3 years. Language therapy (LT) for children with autism is the main form of rehabilitation, because it emphasizes its major presenting symptom (i.e., language impairment). Scalp acupuncture (scalp AP) is a modality based on the physiologic function of different brain areas, where different scalp zones are stimulated with needles so as to stimulate the reflexively related nervous tissue. This study aimed to evaluate the role of scalp AP as a complementary modality to LT in rehabilitation of children with autism.
Subjects and design: The study involved 20 children (divided into 2 equal groups: A and B), diagnosed as autistic according to DSM IV classification. Their ages ranged between 4 and 7 years old. All subjects underwent LT twice weekly, aiming at stimulation of cognitive and verbal abilities. Group B only was subjected to scalp AP sessions-twice weekly-as a rehabilitation complementary tool during the 9-month period of the study. The acupoints used were: Du 20, 26, GV17; three temple needles; and Yamamoto's New Scalp Acupuncture cerebrum and aphasia points (acupuncture needles 0.3 X 30 mm). A language test was performed before and after therapy to monitor cognition and expression (an Arabic test was included).
Results: Both groups, whose mean age range was 5.5 years +/- 1.22 years, showed a significant improvement in cognitive and expressive language skills pre- and post-therapy, which was highly significant among group B children treated with scalp AP (attention 2.8 +/- 0.8 in group A versus 3.5 +/- 0.8 in group B; receptive semantics were 7 +/- 3.8 in group A versus 9.4 +/- 3.1 in group B). Expressive semantics significantly improved in both groups.
Conclusions: Scalp AP is a safe complementary modality when combined with LT and has a significantly positive effect on language development in children with autism.
C1 [Allam, Hemat; Helmy, Ghada] Natl Res Ctr, Complementary Med Dept, Cairo 11571, Egypt.
[Eldine, Nirvana Gamal] Ain Shams Univ, Fac Med, Phoniatr Unit, Cairo, Egypt.
RP Allam, H (reprint author), Natl Res Ctr, Complementary Med Dept, Mukattam POB 93, Cairo 11571, Egypt.
EM hematallam@yahoo.com
CR [Anonymous], 1998, NEUROL RES S1, V20, P28
FIALA P, 2005, ICMART, P107
Filipek PA, 2000, NEUROLOGY, V55, P468
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NR 19
TC 13
Z9 13
PU MARY ANN LIEBERT INC
PI NEW ROCHELLE
PA 140 HUGUENOT STREET, 3RD FL, NEW ROCHELLE, NY 10801 USA
SN 1075-5535
J9 J ALTERN COMPLEM MED
JI J. Altern. Complement Med.
PD MAR
PY 2008
VL 14
IS 2
BP 109
EP 114
DI 10.1089/acm.2007.0508
PG 6
WC Integrative & Complementary Medicine
SC Integrative & Complementary Medicine
GA 282PU
UT WOS:000254580500003
PM 18315511
ER
PT J
AU Grow, LL
Kelley, ME
Roane, HS
Shillingsburg, MA
AF Grow, Laura L.
Kelley, Michael E.
Roane, Henry S.
Shillingsburg, M. Alice
TI Utility of extinction-induced response variability for the selection of
mands
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE extinction; functional analysis; functional communication training;
manding
ID FUNCTIONAL COMMUNICATION; BEHAVIOR PROBLEMS; REINFORCEMENT; ACQUISITION;
MAINTENANCE; PUNISHMENT; EFFICIENCY; SETTINGS; AUTISM
AB Functional communication training (FCT; Carr & Durand, 1985) is a commonly used differential reinforcement procedure for replacing problem behavior with socially acceptable alternative responses. Most studies in the FCT literature consist of demonstrations of the maintenance of responding when various treatment components (e.g., extinction, punishment) are present and absent (e.g., Fisher et al., 1993; Wacker et al., 1990). Relatively little research on FCT has (a) evaluated the conditions under which alternative responses are acquired or (b) described procedures with technological precision. Thus, additional research on a cogent technology for response acquisition appears to be warranted. In the current study, we evaluated the efficacy of exposing problem behavior to extinction for inducing response variability as a tool for selecting an alternative response during FCT. Once participants engaged in appropriate alternative responses, the reinforcer identified in the functional analysis as maintaining problem behavior was delivered contingent on the alternative behavior. Results showed that exposing problem behavior to extinction was a useful method for producing alternative behaviors during FCT.
C1 Emory Univ, Sch Med, Atlanta, GA 30322 USA.
RP Kelley, ME (reprint author), Univ So Maine, Dept Human Resource Dev, 407 Bailey Hall, Gorham, ME 04038 USA.
EM mekelley@usm.maine.edu
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NR 28
TC 19
Z9 19
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD SPR
PY 2008
VL 41
IS 1
BP 15
EP 24
DI 10.1901/jaba.2008.41-15
PG 10
WC Psychology, Clinical
SC Psychology
GA 280LU
UT WOS:000254429100005
PM 18468276
ER
PT J
AU Borrero, CSW
Borrero, JC
AF Borrero, Carrie S. W.
Borrero, John C.
TI Descriptive and experimental analyses of potential precursors to problem
behavior
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article; Proceedings Paper
CT 32nd Annual Meeting of the Association-for-Behavior-Analysis
CY 2006
CL Atlanta, GA
SP Assoc Behav Anal
DE descriptive assessment; functional analysis; lag-sequential analysis;
precursors; problem behavior; response-class hierarchies
ID FUNCTIONAL-ANALYSIS; IDENTIFICATION
AB We conducted descriptive observations of severe problem behavior for 2 individuals with autism to identify precursors to problem behavior. Several comparative probability analyses were conducted in addition to lag-sequential analyses using the descriptive data. Results of the descriptive analyses showed that the probability of the potential precursor was greater given problem behavior compared to the unconditional probability of the potential precursor. Results of the lag-sequential analyses showed a marked increase in the probability of a potential precursor in the 1-s intervals immediately preceding an instance of problem behavior, and that the probability of problem behavior was highest in the 1-s intervals immediately following an instance of the precursor. We then conducted separate functional analyses of problem behavior and the precursor to identify respective operant functions. Results of the functional analyses showed that both problem behavior and the precursor served the same operant functions. These results replicate prior experimental analyses on the relation between problem behavior and precursors and extend prior research by illustrating a quantitative method to identify precursors to more severe problem behavior.
C1 [Borrero, John C.] Univ Pacific, Stockton, CA 95211 USA.
RP Borrero, CSW (reprint author), Kennedy Krieger Inst, 707 N Broadway, Baltimore, MD 21205 USA.
EM Borrero@kennedykrieger.org
CR Bakeman R, 1997, OBSERVING INTERACTIO
Borrero JC, 2007, J APPL BEHAV ANAL, V40, P463, DOI 10.1901/jaba.2007.40-463
CARR EG, 1977, PSYCHOL BULL, V84, P800, DOI 10.1037//0033-2909.84.4.800
Hagopian LP, 2005, RES DEV DISABIL, V26, P393, DOI 10.1016/j.ridd.2003.09.002
Hanley GP, 2003, J APPL BEHAV ANAL, V36, P147, DOI 10.1901/jaba.2003.36-147
Iwata B A, 1994, J Appl Behav Anal, V27, P131, DOI 10.1901/jaba.1994.27-131
Iwata B. A., 2000, HDB APPL BEHAV ANAL, P61
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IWATA BA, 1994, J APPL BEHAV ANAL, V27, P413, DOI 10.1901/jaba.1994.27-413
Kern L., 2004, ED TREATMENT CHILDRE, V27, P440
Lalli JS, 1995, J APPL BEHAV ANAL, V28, P551, DOI 10.1901/jaba.1995.28-551
Magee SK, 2000, J APPL BEHAV ANAL, V33, P313, DOI 10.1901/jaba.2000.33-313
Richman DM, 1999, J APPL BEHAV ANAL, V32, P269, DOI 10.1901/jaba.1999.32-269
Roane HS, 1998, J APPL BEHAV ANAL, V31, P605, DOI 10.1901/jaba.1998.31-605
SAMAHA AL, IN PRESS J APPL BEHA
Sloman KN, 2005, J APPL BEHAV ANAL, V38, P373, DOI 10.1901/jaba.2005.118-04
Smith RG, 2002, J APPL BEHAV ANAL, V35, P125, DOI 10.1901/jaba.2002.35-125
Vollmer TR, 2001, J APPL BEHAV ANAL, V34, P269, DOI 10.1901/jaba.2001.34-269
Yoder PJ, 2000, BEHAVIORAL OBSERVATION, P317
NR 19
TC 30
Z9 30
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD SPR
PY 2008
VL 41
IS 1
BP 83
EP 96
DI 10.1901/jaba.2008.41-83
PG 14
WC Psychology, Clinical
SC Psychology
GA 280LU
UT WOS:000254429100010
PM 18468281
ER
PT J
AU Anglesea, MM
Hoch, H
Taylor, BA
AF Anglesea, Melissa M.
Hoch, Hannah
Taylor, Bridget A.
TI Reducing rapid eating in teenagers with autism: Use of a pager prompt
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; pager prompts; rapid eating; stimulus control
ID REDUCTION
AB This study assessed the effects of a vibrating pager for increasing the duration of meal consumption in 3 teenagers with autism who were observed to eat too quickly. Participants were taught to take a bite only when the pager vibrated at predetermined intervals. A reversal design indicated that the vibrating pager successfully increased the total duration of mealtime, thereby slowing the pace of consumption for all 3 participants.
C1 [Anglesea, Melissa M.; Hoch, Hannah; Taylor, Bridget A.] Alpine Learning Grp, Paramus, NJ 07652 USA.
RP Hoch, H (reprint author), Alpine Learning Grp, 777 Paramus Rd, Paramus, NJ 07652 USA.
EM hhoch@alpinelearninggroup.org
CR FAVELL JE, 1980, BEHAV MODIF, V4, P481, DOI 10.1177/014544558044004
KEDESKY JH, 1998, CHILDHOOD FEEDING DI
LENNOX DB, 1987, J APPL BEHAV ANAL, V20, P279, DOI 10.1901/jaba.1987.20-279
Shabani DB, 2002, J APPL BEHAV ANAL, V35, P79, DOI 10.1901/jaba.2002.35-79
Taylor BA, 2004, J APPL BEHAV ANAL, V37, P79, DOI 10.1901/jaba.2004.37-79
Taylor K, 1998, J CARDIOVASC PHARM, V31, P654, DOI 10.1097/00005344-199805000-00002
Wright CS, 2002, J APPL BEHAV ANAL, V35, P89, DOI 10.1901/jaba.2002.35-89
NR 7
TC 14
Z9 14
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD SPR
PY 2008
VL 41
IS 1
BP 107
EP 111
DI 10.1901/jaba.2008.41-107
PG 5
WC Psychology, Clinical
SC Psychology
GA 280LU
UT WOS:000254429100012
PM 18468283
ER
PT J
AU Baghdadli, A
Picot, MC
Pry, R
Michelon, C
Burzstejn, C
Lazartigues, A
Aussilloux, C
AF Baghdadli, A.
Picot, M. C.
Pry, R.
Michelon, C.
Burzstejn, C.
Lazartigues, A.
Aussilloux, C.
TI What factors are related to a negative outcome of self-injurious
behaviour during childhood in pervasive developmental disorders?
SO JOURNAL OF APPLIED RESEARCH IN INTELLECTUAL DISABILITIES
LA English
DT Article
DE children; longitudinal study; outcome; pervasive developmental
disorders; risk factors; self-injurious behaviour
ID YOUNG-CHILDREN; FOLLOW-UP; AUTISM; AGE; DISABILITIES; PREVALENCE; PEOPLE
AB Aim To explore the factors related to the outcome of self-injurious behaviour (SIB) in children with pervasive developmental disorders (PDD).
Method Children with PDD were assessed on individual and environmental variables at time 1 and 3 years later. A questionnaire about the presence or absence of SIB was also administered at both times in order to examine the early course of SIB.
Results Our findings suggest an association between a negative outcome and several aspects of the children's behaviour. Children with a negative outcome differed from the others in having greater speech impairment, more cognitive and adaptive deficits, and more severe signs of autism. When these variables were entered into a multivariate model for predicting the outcome of SIB, only speech deficits (ORa 3.5, CI 95% 1.13-13.4) and autism severity (ORa 1.1, CI 95% 1.03-1.18) were significant risk factors.
Discussion The importance of these findings for improving our understanding of SIB is discussed.
C1 [Baghdadli, A.; Pry, R.; Michelon, C.; Aussilloux, C.] Child & Adolescent Psychiat Dept, Montpellier, France.
[Picot, M. C.] Dept Stat, Montpellier, France.
[Burzstejn, C.] Child & Adolescent Psychiat Dept, Strasbourg, France.
[Lazartigues, A.] Child & Adolescent Psychiat Dept, Brest, France.
RP Baghdadli, A (reprint author), Ctr Hosp Univ, Clin Peyre Plantade, Autism Resources Ctr, 291 Ave Doyen Giraud, F-34295 Montpellier 5, France.
EM cent-ress-autisme@chu-montpellier.fr
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World Health Organisation, 1992, ICD 10 CLASS MENT BE
NR 31
TC 6
Z9 6
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1360-2322
J9 J APPL RES INTELLECT
JI J. Appl. Res. Intellect. Disabil.
PD MAR
PY 2008
VL 21
IS 2
BP 142
EP 149
DI 10.1111/j.1468-3148.2007.00389.x
PG 8
WC Psychology, Educational; Rehabilitation
SC Psychology; Rehabilitation
GA 259HK
UT WOS:000252930200005
ER
PT J
AU Fukumoto, A
Hashimoto, T
Ito, H
Nishimura, M
Tsuda, Y
Miyazaki, M
Mori, K
Arisawa, K
Kagami, S
AF Fukumoto, Aya
Hashimoto, Toshiaki
Ito, Hiromichi
Nishimura, Mio
Tsuda, Yoshimi
Miyazaki, Masahito
Mori, Kenji
Arisawa, Kokichi
Kagami, Shoji
TI Growth of head circumference in autistic infants during the first year
of life
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; head circumference; body length; body weight; "Mother-and-baby"
notebook; infant physical growth
ID PERVASIVE DEVELOPMENTAL DISORDERS; BRAIN OVERGROWTH; CHILDREN;
CONNECTIVITY; ADULTS; VOLUME; SIZE; MRI
AB This study analyzed the increase in head circumference (HC) of 85 autistic infants (64 boys and 21 girls) during their first year of life. The data were collected from their "mother-and-baby" notebooks. This notebook is a medical record of the baby's growth and development delivered to the parents of all babies born in Japan. This is a retrospective study which gathered the data from the notebooks after the diagnosis of autism. However, none of the babies were known to have autism at the time the records were made. The head circumference at birth of these autistic children was similar to that of the average found in a Japanese Government Study of 14,115 children. However, it showed a marked increase at 1 month after birth. The discrepancy reached a peak at 6 months, while the difference became smaller at 12 months. Body length (BL) and body weight (BW) began to increase at 3 months, although at a rate smaller than the head circumference increase.
C1 [Fukumoto, Aya; Hashimoto, Toshiaki; Ito, Hiromichi; Nishimura, Mio; Tsuda, Yoshimi; Miyazaki, Masahito; Mori, Kenji; Kagami, Shoji] Univ Tokushima, Grad Sch, Dept Pediat, Div Human Dev & Hlth Sci Subdiv Human Dev, Tokushima 7708503, Japan.
[Hashimoto, Toshiaki; Tsuda, Yoshimi] Naruto Univ Educ, Dept Special Support Educ, Naruto, Tokushima 772, Japan.
[Nishimura, Mio] Hinomine Med Ctr, Dept Pediat, Komatsushima, Tokushima, Japan.
[Arisawa, Kokichi] Univ Tokushima, Grad Sch, Inst Hlth Biosci, Dept Prevent Med, Tokushima 770, Japan.
RP Fukumoto, A (reprint author), Univ Tokushima, Grad Sch, Dept Pediat, Div Human Dev & Hlth Sci Subdiv Human Dev, 3-18-15 Kuramoto Cho, Tokushima 7708503, Japan.
EM aya11@mb.tcn.ne.jp
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NR 36
TC 30
Z9 30
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD MAR
PY 2008
VL 38
IS 3
BP 411
EP 418
DI 10.1007/s10803-007-0405-1
PG 8
WC Psychology, Developmental
SC Psychology
GA 268HW
UT WOS:000253571900001
PM 17647099
ER
EF