FN Thomson Reuters Web of Science™
VR 1.0
PT J
AU Ekstrom, AB
Hakenas-Plate, L
Samuelsson, L
Tulinius, M
Wentz, E
AF Ekstrom, Anne-Berit
Hakenas-Plate, Louise
Samuelsson, Lena
Tulinius, Mar
Wentz, Elisabet
TI Autism spectrum conditons in myotonic dystrophy type 1: A study on 57
individuals with congenital and childhood forms
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article; Proceedings Paper
CT 6th European-Pediatric-Neurology-Society Congress
CY SEP 14-17, 2005
CL Goteborg, SWEDEN
SP European Pediat Neurol Soc
DE neuropsychiatry; mental retardation; ADI-R; genetics; adolescence
ID PERVASIVE DEVELOPMENTAL DISORDERS; FRAGILE-X-SYNDROME;
ASPERGER-SYNDROME; CTG-REPEAT; FOLLOW-UP; DIAGNOSTIC-INTERVIEW;
UNTRANSLATED REGION; MUSCULAR-DYSTROPHY; CHILDREN; BRAIN
AB Myotonic dystrophy type 1 (DM1) is an autosomal dominant disorder, caused by an expansion of a CTG triplet repeat in the DMPK gene. The aims of the present study were to classify a cohort of children with DM1, to describe their neuropsychiatric problems and cognitive level, to estimate the size of the CTG expansion, and to correlate the molecular findings with the neuropsychiatric problems. Fifty-seven children and adolescents (26 females; 31 males) with DM1 (CTG repeats > 40) were included in the study. The following instruments were used: Autism Diagnostic Interview-Revised (ADI-R), 5-15, Griffiths Mental Development Scales, and the Wechsler Scales. Based on age at onset and presenting symptoms, the children were divided into four DM1 groups; severe congenital (n = 19), mild congenital (n = 18), childhood (n = 18), and classical DM1 (n = 2). Forty-nine percent had an autism spectrum disorder (ASD) and autistic disorder was the most common diagnosis present in 35% of the subjects. Eighty-six percent of the individuals with DM1 had mental retardation (MR), most of them moderate or severe MR. ASD was significantly correlated with the DM1 form; the more severe the form of DM1, the higher the frequency of ASD. The frequency of ASD increased with increasing CTG repeat expansions. ASD and/or other neuropsychiatric disorders such as attention deficit hyperactivity disorder, and Tourette's disorder were found in 54% of the total DM1. group. In conclusion, awareness of ASD comorbidity in DM1. is essential. Further studies are warranted to elucidate the molecular etiology causing neurodevelopmental symptoms such as ASD and MR in DM1. (c) 2008 Wiley-Liss, Inc.
C1 [Ekstrom, Anne-Berit] No Alvsborg Cty Hosp, Dept Pediat, S-46185 Trollhattan, Sweden.
[Ekstrom, Anne-Berit; Tulinius, Mar] Univ Gothenburg, Sahlgrenska Acad, Inst Clin Sci, Dept Pediat, Gothenburg, Sweden.
[Hakenas-Plate, Louise] Sahlgrens Univ Hosp, Child Neuropsychiat Clin, Gothenburg, Sweden.
[Wentz, Elisabet] Univ Gothenburg, Sahlgrenska Acad, Inst Neurosci & Physiol, Gothenburg, Sweden.
[Wentz, Elisabet] Swedish Inst Hlth Sci, Vardal Inst, Lund, Sweden.
[Samuelsson, Lena] Univ Gothenburg, Sahlgrenska Acad, Dept Clin Sci, Gothenburg, Sweden.
RP Ekstrom, AB (reprint author), No Alvsborg Cty Hosp, Dept Pediat, S-46185 Trollhattan, Sweden.
EM anne-berit.ekstrom@vgregion.se
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NR 82
TC 22
Z9 25
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD SEP 5
PY 2008
VL 147B
IS 6
BP 918
EP 926
DI 10.1002/ajmg.b.30698
PG 9
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 346EB
UT WOS:000259050100034
PM 18228241
ER
PT J
AU Kakinuma, H
Ozaki, M
Sato, H
Takahashi, H
AF Kakinuma, Hiroaki
Ozaki, Mamoru
Sato, Hitoshi
Takahashi, Hiroaki
TI Variation in GABA-A subunit gene copy number in an autistic patient with
mosaic 4 p duplication (p12p16)
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE 4p duplication; autism; FISH; CABA-A receptor subunit gene
ID DISORDER; FAMILY
AB Autism has been associated with chromosomal aberrations, including duplications at chromosome 4, and the identification of genetic factors contributing to the etiology of this disease is the focus of much research. Here we report a Japanese girl with mosaic of chromosome 4p duplication, mos 46,XX,dup(4)(p12p16)[54]/46,XX [6], who was diagnosed with autism at 3 years of age. Fluorescence in situ hybridization (FISH) with probes covering the region spanning a cluster of the gamma aminobutyric acid A (GABA-A) receptor subunit genes in the proximal short arm of chromosome 4 demonstrated total three signals for the GABRG1, GABPLA4, and GABRA2 genes, but only two signals for GABRB1. This suggests that aberrant copy number of the GABA-A receptor subunit genes may contribute to the etiology of autism in this patient. (c) 2007 Wiley-Liss, Inc.
C1 [Kakinuma, Hiroaki; Sato, Hitoshi; Takahashi, Hiroaki] Kanazawa Med Univ, Dept Pediat, Uchinada, Ishikawa 9200293, Japan.
[Kakinuma, Hiroaki; Ozaki, Mamoru] Kanazawa Med Univ, Med Res Inst, Div Human Genet, Uchinada, Ishikawa 9200293, Japan.
RP Kakinuma, H (reprint author), Kanazawa Med Univ, Dept Pediat, 1-1 Daigaku, Uchinada, Ishikawa 9200293, Japan.
EM p-kaki@kanazawa-med.ac.jp
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NR 14
TC 15
Z9 15
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD SEP 5
PY 2008
VL 147B
IS 6
BP 973
EP 975
DI 10.1002/ajmg.b.30663
PG 3
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 346EB
UT WOS:000259050100041
PM 18163449
ER
PT J
AU Kent, L
Gallagher, L
Elliott, HR
Mowbray, C
Chinnery, PF
AF Kent, Lindsey
Gallagher, Louise
Elliott, Hannah R.
Mowbray, Catherine
Chinnery, Patrick F.
TI An investigation of mitochondrial haplogroups in autism
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Article
DE child psychiatry; molecular genetics; autism spectrum disorders
ID CARRIER SLC25A12 GENE; SPECTRUM DISORDERS; DNA HAPLOGROUPS; CHILDREN;
POLYMORPHISMS; ASSOCIATION; PHENOTYPE; PARENTS; DISEASE
AB Family and twin studies provide strong evidence of a major genetic influence in autism, but the underlying gene defects have yet to be characterized. The mothers of boys with autism share autistic traits, raising the possibility of a maternally inherited factor. Mitochondrial DNA (mtDNA) is almost exclusively inherited down the maternal line. We therefore explored the possibility that a particular mtDNA lineage contributes to the risk of developing autism. The mtDNA haplogroup was determined in 162 autism probands, and compared to two sets of population controls. Results show no compelling evidence of an association of any mitochondrial haplogroup in autism. (c) 2007 Wiley-Liss, Inc.
C1 [Kent, Lindsey] Univ Cambridge, Cambridge, England.
[Gallagher, Louise] Trinity Coll Dublin, Dept Psychiat, Dublin, Ireland.
[Elliott, Hannah R.; Mowbray, Catherine; Chinnery, Patrick F.] Univ Newcastle Upon Tyne, Sch Med, Mitochondrial Res Grp, Newcastle Upon Tyne NE1 7RU, Tyne & Wear, England.
[Elliott, Hannah R.; Mowbray, Catherine; Chinnery, Patrick F.] Univ Newcastle Upon Tyne, Sch Med, Inst Human Genet, Newcastle Upon Tyne NE1 7RU, Tyne & Wear, England.
RP Kent, L (reprint author), Univ St Andrews, Bute Med Sch, St Andrews KY16 9TS, Fife, Scotland.
EM lsk8@st-andrews.ac.uk
FU Wellcome Trust; Nancy Lurie Marks Family Foundation
FX This study was funded by the Wellcome Trust and the Nancy Lurie Marks
Family Foundation. PFC is a Wellcome Trust Senior Fellow in Clinical
Science.
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NR 20
TC 7
Z9 7
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD SEP 5
PY 2008
VL 147B
IS 6
BP 987
EP 989
DI 10.1002/ajmg.b.30687
PG 3
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 346EB
UT WOS:000259050100044
PM 18161860
ER
PT J
AU Hornig, M
Briese, T
Buie, T
Bauman, ML
Lauwers, G
Siemetzki, U
Hummel, K
Rota, PA
Bellini, WJ
O'Leary, JJ
Sheils, O
Alden, E
Pickering, L
Lipkin, WI
AF Hornig, Mady
Briese, Thomas
Buie, Timothy
Bauman, Margaret L.
Lauwers, Gregory
Siemetzki, Ulrike
Hummel, Kimberly
Rota, Paul A.
Bellini, William J.
O'Leary, John J.
Sheils, Orla
Alden, Errol
Pickering, Larry
Lipkin, W. Ian
TI Lack of Association between Measles Virus Vaccine and Autism with
Enteropathy: A Case-Control Study
SO PLOS ONE
LA English
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDERS; MUMPS-RUBELLA VACCINATION; LYMPHOCYTE
CYTOKINE PROFILES; INFLAMMATORY-BOWEL-DISEASE; SPECTRUM DISORDER; NO
EVIDENCE; GASTROINTESTINAL SYMPTOMS; NODULAR HYPERPLASIA; CAUSAL
ASSOCIATION; REGRESSIVE AUTISM
AB Background: The presence of measles virus (MV) RNA in bowel tissue from children with autism spectrum disorders (ASD) and gastrointestinal (GI) disturbances was reported in 1998. Subsequent investigations found no associations between MV exposure and ASD but did not test for the presence of MV RNA in bowel or focus on children with ASD and GI disturbances. Failure to replicate the original study design may contribute to continued public concern with respect to the safety of the measles, mumps, and rubella (MMR) vaccine.
Methodology/Principal Findings: The objective of this case-control study was to determine whether children with GI disturbances and autism are more likely than children with GI disturbances alone to have MV RNA and/or inflammation in bowel tissues and if autism and/or GI episode onset relate temporally to receipt of MMR. The sample was an age-matched group of US children undergoing clinically-indicated ileocolonoscopy. Ileal and cecal tissues from 25 children with autism and GI disturbances and 13 children with GI disturbances alone (controls) were evaluated by real-time reverse transcription (RT)-PCR for presence of MV RNA in three laboratories blinded to diagnosis, including one wherein the original findings suggesting a link between MV and ASD were reported. The temporal order of onset of GI episodes and autism relative to timing of MMR administration was examined. We found no differences between case and control groups in the presence of MV RNA in ileum and cecum. Results were consistent across the three laboratory sites. GI symptom and autism onset were unrelated to MMR timing. Eighty-eight percent of ASD cases had behavioral regression.
Conclusions/Significance: This study provides strong evidence against association of autism with persistent MV RNA in the GI tract or MMR exposure. Autism with GI disturbances is associated with elevated rates of regression in language or other skills and may represent an endophenotype distinct from other ASD.
C1 [Hornig, Mady; Briese, Thomas; Siemetzki, Ulrike; Lipkin, W. Ian] Columbia Univ, Mailman Sch Publ Hlth, Ctr Infect & Immun, New York, NY 10027 USA.
[Buie, Timothy] Massachusetts Gen Hosp, Div Pediat Gastroenterol & Nutr, Boston, MA USA.
[Bauman, Margaret L.] Massachusetts Gen Hosp, Dept Neurol, Dept Pediat, LADDERS, Boston, MA USA.
[Lauwers, Gregory] Massachusetts Gen Hosp, Harvard Med Sch, Dept Pathol, Boston, MA USA.
[Hummel, Kimberly; Rota, Paul A.; Bellini, William J.] Ctr Dis Control & Prevent, Measles Mumps Rubella & Herpesvirus Lab Branch, Atlanta, GA USA.
[O'Leary, John J.; Sheils, Orla] Trinity Coll Dublin, Dept Histopathol, Dublin, Ireland.
[Alden, Errol] American Acad Pediat, Elk Grove Village, IL USA.
[Pickering, Larry] Ctr Dis Control & Prevent, Natl Ctr Immunizat & Resp Dis, Atlanta, GA USA.
RP Hornig, M (reprint author), Columbia Univ, Mailman Sch Publ Hlth, Ctr Infect & Immun, New York, NY 10027 USA.
EM mady.hornig@columbia.edu; wil2001@columbia.edu
FU CDC [U50 CCU522351]; National Institutes of Health [AI57158, HL083850,
NS47537]
FX This work was supported by CDC grant U50 CCU522351 to AAP and by
National Institutes of Health awards AI57158 (Northeast Biodefense
Center-Lipkin), HL083850, and NS47537. Role of Study Sponsors: Members
of the funding organization (AAP) and its sponsor (CDC) participated
along with experts in virology and neurovirology, autism pathogenesis,
and vaccine design and safety; representatives of the autism advocacy
community; and study collaborators in an Oversight Committee that
reviewed and agreed to all aspects of study design prior to data
collection. The final decision to submit for publication was the
responsibility of all study collaborators.
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NR 49
TC 45
Z9 46
PU PUBLIC LIBRARY SCIENCE
PI SAN FRANCISCO
PA 185 BERRY ST, STE 1300, SAN FRANCISCO, CA 94107 USA
SN 1932-6203
J9 PLOS ONE
JI PLoS One
PD SEP 4
PY 2008
VL 3
IS 9
AR e3140
DI 10.1371/journal.pone.0003140
PG 8
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 422HU
UT WOS:000264419100008
PM 18769550
ER
PT J
AU Mueller, BR
Bale, TL
AF Mueller, Bridget R.
Bale, Tracy L.
TI Sex-specific programming of offspring emotionality after stress early in
pregnancy
SO JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE placenta; neurodevelopment; DNA methylation; prenatal stress;
depression; affective disorders
ID CORTICOTROPIN-RELEASING-FACTOR; TAIL SUSPENSION TEST; FORCED SWIM TEST;
PRENATAL STRESS; EPIGENETIC MECHANISMS; GENE-EXPRESSION; LOCUS-CERULEUS;
ANIMAL-MODEL; DEPRESSION; SCHIZOPHRENIA
AB Prenatal stress is associated with an increased vulnerability to neurodevelopmental disorders, including autism and schizophrenia. To determine the critical time window when fetal antecedents may induce a disease predisposition, we examined behavioral responses in offspring exposed to stress during early, mid, and late gestation. We found that male offspring exposed to stress early in gestation displayed maladaptive behavioral stress responsivity, anhedonia, and an increased sensitivity to selective serotonin reuptake inhibitor treatment. Long-term alterations in central corticotropin-releasing factor (CRF) and glucocorticoid receptor (GR) expression, as well as increased hypothalamic-pituitary-adrenal (HPA) axis responsivity, were present in these mice and likely contributed to an elevated stress sensitivity. Changes in CRF and GR gene methylation correlated with altered gene expression, providing important evidence of epigenetic programming during early prenatal stress. In addition, we found the core mechanism underlying male vulnerability may involve sex-specific placenta responsivity, where stress early in pregnancy significantly increased expression of PPAR alpha( peroxisome proliferator-activated receptor alpha), IGFBP-1 ( insulin-like growth factor binding protein 1), HIF3 alpha(hypoxia-inducible factor 3a), and GLUT4 ( glucose transporter 4) in male placentas but not females. Examination of placental epigenetic machinery revealed basal sex differences, providing further evidence that sex-specific programming begins very early in pregnancy, and may contribute to the timing and vulnerability of the developing fetus to maternal perturbations. Overall, these results indicate that stress experience early in pregnancy may contribute to male neurodevelopmental disorders through impacts on placental function and fetal development.
C1 [Mueller, Bridget R.; Bale, Tracy L.] Univ Penn, Sch Vet Med, Dept Anim Biol, Philadelphia, PA 19104 USA.
RP Bale, TL (reprint author), Univ Penn, Sch Vet Med, Dept Anim Biol, 201E,3800 Spruce St, Philadelphia, PA 19104 USA.
EM tbale@vet.upenn.edu
FU University of Pennsylvania Research Foundation; March of Dimes
[5FY03-133]; National Institutes of Health Behavioral/Cognitive
Neuroscience Training Grant [T32 MH-017168]
FX This work was supported by the University of Pennsylvania Research
Foundation and March of Dimes 5FY03-133. B. R. M. was supported by the
National Institutes of Health Behavioral/Cognitive Neuroscience Training
Grant T32 MH-017168. We thank K. Carlin and C. Welle for technical
support and D. Pankevich for editorial assistance.
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NR 51
TC 308
Z9 312
PU SOC NEUROSCIENCE
PI WASHINGTON
PA 11 DUPONT CIRCLE, NW, STE 500, WASHINGTON, DC 20036 USA
SN 0270-6474
J9 J NEUROSCI
JI J. Neurosci.
PD SEP 3
PY 2008
VL 28
IS 36
BP 9055
EP 9065
DI 10.1523/JNEUROSCI.1424-08.2008
PG 11
WC Neurosciences
SC Neurosciences & Neurology
GA 343XY
UT WOS:000258890900023
PM 18768700
ER
PT J
AU von Knorring, AL
Soderberg, A
Austin, L
Uvnas-Moberg, K
AF von Knorring, Anne-Liis
Soderberg, Anna
Austin, Lena
Uvnas-Moberg, Kerstin
TI Massage decreases aggression in preschool children: a long-term study
SO ACTA PAEDIATRICA
LA English
DT Article
DE aggression; deviant behaviour; long-term treatment; massage; preschool
children
ID BEHAVIOR; THERAPY; OXYTOCIN; CORTISOL; AUTISM
AB Aim: To evaluate the effects of massage in 4- to 5-year-old children with aggression and deviant behaviour at day-care centres.
Method: The children received daily massage in preschool at the midday rest (n = 60). The controls were listening to a story (n = 50). The Child Behaviour Checklist (CBCL) was used to rate the children's behaviour by parents and staff before the treatment started, and after 3 and 6 months. A long-term evaluation was also carried out. It included all massaged children still in daycare after 12 months (n = 34).
Results: Children with high scores of behaviour problems, receiving massage and/or extra attention showed significant decrease in aggression scores after 3 months, but after 6 months significantly lowered scores were only found in massage-treated deviant children. Parents of the children receiving massage rated a significant decrease of somatic problems of their children. Staff rated that the massaged children's social problems decreased, compared to the control children. Attention problems tended to decrease, especially at home. A continuous decrease in aggressive behaviour and somatic problems over a 12-month period was observed in the children receiving massage.
Conclusion: Daily touching by massage lasting for 5-10 min could be an easy and inexpensive way to decrease aggression among preschool children.
C1 [von Knorring, Anne-Liis] Uppsala Univ, Dept Neurosci, Uppsala, Sweden.
[Soderberg, Anna; Austin, Lena] Axelsons Gymnas Inst, Stockholm, Sweden.
[Uvnas-Moberg, Kerstin] Univ Agr, Dept Anim Hlth & Welf, Skara, Sweden.
RP von Knorring, AL (reprint author), Univ Uppsala Hosp, SE-75185 Uppsala, Sweden.
EM anne-liis.von_knorring@bupinst.uu.se
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TC 6
Z9 6
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0803-5253
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BP 1265
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PM 18782279
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AU Fisch, GS
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TI Syndromes and epistemology II: Is autism a polygenic disorder?
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
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ID TRANSPORTER GENE SLC6A4; QUANTITATIVE TRAIT LOCI; EARLY
INFANTILE-AUTISM; SPECTRUM DISORDERS; SEROTONIN TRANSPORTER; LINKAGE
DISEQUILIBRIUM; LANGUAGE IMPAIRMENT; 22Q11.2 DELETION; PROXIMAL 15Q;
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NR 117
TC 4
Z9 4
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD SEP 1
PY 2008
VL 146A
IS 17
BP 2203
EP 2212
DI 10.1002/ajmg.a.32438
PG 10
WC Genetics & Heredity
SC Genetics & Heredity
GA 347GP
UT WOS:000259128900003
PM 18666231
ER
PT J
AU Jones, JR
Skinner, C
Friez, MJ
Schwartz, CE
Stevenson, RE
AF Jones, Julie R.
Skinner, Cindy
Friez, Michael J.
Schwartz, Charles E.
Stevenson, Roger E.
TI Hypothesis: Dysregulation of methylation of brain-expressed genes on the
X chromosome and autism spectrum disorders
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Review
DE autism; methylation; X chromosome; autism spectrum disorders;
epigenetics
ID PERVASIVE DEVELOPMENTAL DISORDERS; SEVERE MENTAL-RETARDATION;
OF-THE-LITERATURE; DNA METHYLATION; NEUROANATOMICAL ABNORMALITIES;
PLATELET SEROTONIN; IMPRINTED GENES; SEX-DIFFERENCES; HUMAN HOMOLOG;
DUPLICATION
AB The hypothesis is set forth that dysregulation of brain-expressed genes on the X chromosome constitutes the major predisposition to autism spectrum disorders (ASDs). This dysregulation, mediated by hypomethylation or hypermethylation of CpG sites within gene promoters, leads to overexpression or partial silencing of one or more brain-expressed genes, which in turn results in an unbalanced production of the proteins responsible for brain structure and function. This hypothesis accommodates the predominantly sporadic occurrence (95%), the male excess (4:1), and the usual absence of malformations or other syndromic manifestations in ASDs. (C) 2008 Wiley-Liss, Inc.
C1 [Jones, Julie R.; Skinner, Cindy; Friez, Michael J.; Schwartz, Charles E.; Stevenson, Roger E.] Greenwood Genet Ctr, Greenwood, SC USA.
RP Stevenson, RE (reprint author), Greenwood Genet Ctr, 101 Gregor Mendel Circle, Greenwood, SC USA.
EM res@ggc.org
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NR 104
TC 14
Z9 14
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD SEP 1
PY 2008
VL 146A
IS 17
BP 2213
EP 2220
DI 10.1002/ajmg.a.32396
PG 8
WC Genetics & Heredity
SC Genetics & Heredity
GA 347GP
UT WOS:000259128900004
PM 18698615
ER
PT J
AU Croen, LA
Matevia, M
Yoshida, CK
Grether, JK
AF Croen, Lisa A.
Matevia, Marilyn
Yoshida, Cathleen K.
Grether, Judith K.
TI Maternal Rh D status, anti-D immune globulin exposure during pregnancy,
and risk of autism spectrum disorders
SO AMERICAN JOURNAL OF OBSTETRICS AND GYNECOLOGY
LA English
DT Article
DE autism spectrum disorders; perinatal risk factors; RhoGam; thimerosal
ID PERVASIVE DEVELOPMENTAL DISORDERS; THIMEROSAL; METHYLMERCURY;
ASSOCIATION; POPULATION; CHILDREN; OUTCOMES
AB OBJECTIVE: The objective of the study was to investigate the association between maternal Rh D status, prenatal exposure to anti-D immune globulin, and the risk of autism in the offspring. STUDY DESIGN: Case-control study among children born from 1995 to 1999 at Kaiser Permanente Northern California hospitals. Cases (n = 400) were children with an autism diagnosis; controls (n = 410) were children without autism, randomly sampled and frequency matched to cases on sex, birth year, and birth hospital. Maternal Rh D status and anti-D immune globulin exposure were ascertained from prenatal medical records. RESULTS: No case-control differences were observed for maternal Rh negative status (11.5% vs 10.0%, P =.5) or prenatal anti-D immune globulin exposure (10.0% vs. 9.3%, P =.7). Risk of autism remained unassociated with maternal Rh status or prenatal exposure to anti-D immune globulins after adjustment for covariates. CONCLUSION: These data support previous findings that prenatal exposure to thimerosal-containing anti-D immune globulins does not increase the risk of autism.
C1 [Croen, Lisa A.; Matevia, Marilyn; Yoshida, Cathleen K.] Kaiser Permanente No Calif, Div Res, Oakland, CA USA.
[Grether, Judith K.] Calif Dept Publ Hlth, Environm Hlth Invest Branch, Richmond, CA USA.
RP Croen, LA (reprint author), Kaiser Permanente, Div Res, 2000 Broadway, Oakland, CA 94612 USA.
EM lisa.a.croen@kp.org
CR American Psychiatric Association, 2000, TASK FORC DSM 4 DIAG
American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 32
TC 1
Z9 1
PU MOSBY-ELSEVIER
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0002-9378
J9 AM J OBSTET GYNECOL
JI Am. J. Obstet. Gynecol.
PD SEP
PY 2008
VL 199
IS 3
DI 10.1016/j.ajog.2008.04.044
PG 6
WC Obstetrics & Gynecology
SC Obstetrics & Gynecology
GA 344VM
UT WOS:000258955400006
ER
PT J
AU DeFranco, E
Gross, G
Shanks, A
Johnson, T
Shen, T
AF DeFranco, Emily
Gross, Gilad
Shanks, Anthony
Johnson, Traci
Shen, Tammy
TI Prenatal exposure to anti-D immune globulin and autism risk: Croen et al
SO AMERICAN JOURNAL OF OBSTETRICS AND GYNECOLOGY
LA English
DT Editorial Material
C1 [Gross, Gilad] Washington Univ, Sch Med, Dept Obstet & Gynecol, Div Maternal Fetal Med, St Louis, MO 63110 USA.
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
*CDCP, AUT INF CTR FREQ ASK
Fombonne E, 2002, MOL PSYCHIATR, V7, pS4, DOI 10.1038/sj.mp.4001162
NR 3
TC 0
Z9 0
PU MOSBY-ELSEVIER
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0002-9378
J9 AM J OBSTET GYNECOL
JI Am. J. Obstet. Gynecol.
PD SEP
PY 2008
VL 199
IS 3
BP 322
EP 323
DI 10.1016/j.ajog.2008.07.025
PG 2
WC Obstetrics & Gynecology
SC Obstetrics & Gynecology
GA 344VM
UT WOS:000258955400043
PM 18772000
ER
PT J
AU DeFranco, E
Gross, G
Shanks, A
Johnson, T
Shen, T
AF DeFranco, Emily
Gross, Gilad
Shanks, Anthony
Johnson, Traci
Shen, Tammy
TI Discussion: Prenatal exposure to anti-D immune globulin and autism risk
by Croen et al
SO AMERICAN JOURNAL OF OBSTETRICS AND GYNECOLOGY
LA English
DT Editorial Material
C1 [Gross, Gilad] Washington Univ, Sch Med, Dept Obstet & Gynecol, Div Maternal Fetal Med, St Louis, MO 63110 USA.
CR *CDCP, AUT INF CTR FREQ ASK
Fombonne E, 2002, MOL PSYCHIATR, V7, pS4, DOI 10.1038/sj.mp.4001162
NR 2
TC 0
Z9 0
PU MOSBY-ELSEVIER
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0002-9378
J9 AM J OBSTET GYNECOL
JI Am. J. Obstet. Gynecol.
PD SEP
PY 2008
VL 199
IS 3
BP E1
EP E4
DI 10.1016/j.ajog.2008.07.026
PG 4
WC Obstetrics & Gynecology
SC Obstetrics & Gynecology
GA 344VM
UT WOS:000258955400047
PM 18771966
ER
PT J
AU Silva, LMT
Ayres, R
Schalock, M
AF Silva, Louisa M. T.
Ayres, Robert
Schalock, Mark
TI Outcomes of a pilot training program in a qigong massage intervention
for young children with autism
SO AMERICAN JOURNAL OF OCCUPATIONAL THERAPY
LA English
DT Article
DE autism; early intervention; pediatrics; qigong massage; Qigong Sensory
Training; sensory impairment
ID EQUIVALENCE; EFFICACY; TESTS
AB Sensory impairment is a common and significant feature of children on the autism spectrum. In 2005, a qigong massage intervention based on Chinese medicine and delivered by a doctor of Chinese medicine was shown to improve sensory impairment and adaptive behavior in a small controlled study of young children with autism. In 2006, the Qigong Sensory Training (QST) program was developed to train early intervention professionals to provide the QST intervention. This article describes the preliminary evaluation of the QST program as piloted with 15 professionals and 26 children and outcomes testing using standardized tests of sensory impairment and adaptive behavior. Results of outcomes comparing delivery by QST-trained therapists with delivery by a doctor of Chinese medicine showed that both groups improved and that there was no difference in outcome between the two groups. The intervention and training program are described, and implications for future research are discussed.
C1 [Ayres, Robert; Schalock, Mark] Western Oregon Univ, Teaching Res Inst, Monmouth, OR USA.
RP Silva, LMT (reprint author), Western Oregon Univ, Teaching Res Inst, POB 688, Salem, OR 97308 USA.
EM lmtsilvaqigong@comcast.net
CR ARICK JR, 2003, FOCUS AUTISM OTHER D, V18, P74
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YANG J, 2005, QIGONG MASSAGE
NR 30
TC 8
Z9 8
PU AMER OCCUPATIONAL THERAPY ASSOC, INC
PI BETHESDA
PA 4720 MONTGOMERY LANE, BETHESDA, MD 20814-3425 USA
SN 0272-9490
J9 AM J OCCUP THER
JI Am. J. Occup. Ther.
PD SEP-OCT
PY 2008
VL 62
IS 5
BP 538
EP 546
PG 9
WC Rehabilitation
SC Rehabilitation
GA 348ZK
UT WOS:000259248400006
PM 18826014
ER
PT J
AU Shoener, RF
Kinnealey, M
Koenig, KP
AF Shoener, Rachel Freret
Kinnealey, Moya
Koenig, Kristie P.
TI You can know me now if you listen: Sensory, motor, and communication
issues in a nonverbal person with autism
SO AMERICAN JOURNAL OF OCCUPATIONAL THERAPY
LA English
DT Article
DE autism; communication; effectiveness; movement; sensory processing
ID PROCESSING DISORDERS; CHILDREN; INTERVENTION; INTEGRATION
AB This case report describes an intensive approach to treating autism and provides an intersection between a first-person narrative paired with intervention and outcomes. In-depth conversations between a person with autism and an occupational therapist provide insight into understanding differences and difficulties in sensory processing and regulation, praxis, and communication. Individuals with autism may be intellectually and emotionally intact but hampered by deficits that interfere with the ability to move the body efficiently. These sensorimotor deficits underlie the ability to communicate with others and to develop relationships. This article illustrates the benefits of an intensive therapeutic program designed to address sensory and motor differences underlying communication, as well as the vital role the occupational therapist plays in addressing these underlying differences to improve functional communication and social participation.
C1 [Shoener, Rachel Freret] TALK Inst & Sch, Newtown Sq, PA 19073 USA.
[Kinnealey, Moya] Temple Univ, Philadelphia, PA 19122 USA.
[Koenig, Kristie P.] New York Univ Steinhardt, Dept Occupat Therapy, New York, NY USA.
RP Shoener, RF (reprint author), TALK Inst & Sch, 395H Bishop Hollow Rd, Newtown Sq, PA 19073 USA.
EM rach530@gmail.com
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Teitelbaum P, 2002, J DEV LEARNING DISOR, V6, P15
NR 31
TC 5
Z9 5
PU AMER OCCUPATIONAL THERAPY ASSOC, INC
PI BETHESDA
PA 4720 MONTGOMERY LANE, BETHESDA, MD 20814-3425 USA
SN 0272-9490
J9 AM J OCCUP THER
JI Am. J. Occup. Ther.
PD SEP-OCT
PY 2008
VL 62
IS 5
BP 547
EP 553
PG 7
WC Rehabilitation
SC Rehabilitation
GA 348ZK
UT WOS:000259248400007
PM 18826015
ER
PT J
AU Hilton, CL
Crouch, MC
Israel, H
AF Hilton, Claudia List
Crouch, Mary Catherine
Israel, Heidi
TI Out-of-school participation patterns in children with high-functioning
autism spectrum disorders
SO AMERICAN JOURNAL OF OCCUPATIONAL THERAPY
LA English
DT Article
DE autism; interpersonal relations; leisure activities; pediatrics;
socialization
ID RECIPROCAL SOCIAL-BEHAVIOR; PERVASIVE DEVELOPMENTAL DISORDERS;
ASPERGER-SYNDROME; LEISURE ACTIVITIES; LEARNING-DISABILITIES;
GENERAL-POPULATION; MOTOR IMPAIRMENT; TRAITS; ADOLESCENTS; VALIDATION
AB PURPOSE. This study examined differences in out-of-school activity participation between typically developing children and those with high-functioning autism spectrum disorders (HFASD).
METHOD. Children with HFASD, ages 6 to 12 (N = 52), and a control group (N = 53) were assessed using the Children's Assessment of Participation and Enjoyment and the Social Responsiveness Scale.
RESULTS. Significant differences were seen in participation between typical and HFASD groups in number of activities in which children participate, the numbers of individuals with whom they participate, and the variety of environments in which they participate.
CONCLUSIONS. These findings indicate that out-of-school participation is significantly different for children with HFASD than for typically developing peers. Findings suggest that social impairment is related to some aspects of participation and that addressing social skills in intervention could contribute to increased participation in out-of-school activities by children with HFASD, which would contribute to their long-term mental and physical health.
C1 [Crouch, Mary Catherine] Barnes Jewish Hosp, Occupat Therapy Dept, St Louis, MO 63110 USA.
[Israel, Heidi] St Louis Univ, Dept Orthopaed Surg, St Louis, MO 63103 USA.
EM hiltonc@psychiatry.wustl.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 38
TC 32
Z9 32
PU AMER OCCUPATIONAL THERAPY ASSOC, INC
PI BETHESDA
PA 4720 MONTGOMERY LANE, BETHESDA, MD 20814-3425 USA
SN 0272-9490
J9 AM J OCCUP THER
JI Am. J. Occup. Ther.
PD SEP-OCT
PY 2008
VL 62
IS 5
BP 554
EP 563
PG 10
WC Rehabilitation
SC Rehabilitation
GA 348ZK
UT WOS:000259248400008
PM 18826016
ER
PT J
AU Ashburner, J
Ziviani, J
Rodger, S
AF Ashburner, Jill
Ziviani, Jenny
Rodger, Sylvia
TI Sensory processing and classroom emotional, behavioral, and educational
outcomes in children with autism spectrum disorder
SO AMERICAN JOURNAL OF OCCUPATIONAL THERAPY
LA English
DT Article; Proceedings Paper
CT 14th World Federation of Occupational Therapists Congress
CY JUL 25-28, 2006
CL Sydney, AUSTRALIA
SP Greek Natl Scholarship Fdn, 2nd Div, Overseas Scholarships Sect
DE pediatrics; pervasive development disorders; school; sensory
ID YOUNG-CHILDREN; DEVELOPMENTAL DISORDERS; TYPICAL DEVELOPMENT; ATTENTION;
INDIVIDUALS; RECOGNITION; DEFICITS; PERFORMANCE; DYSFUNCTION; CEREBELLAR
AB OBJECTIVE. We explored the associations between sensory processing and classroom emotional, behavioral, and educational outcomes of children with autism spectrum disorder (ASD).
METHOD. Twenty-eight children with ASD (with average-range 10) were compared with 51 age- and gender-matched typically developing peers on sensory processing and educational outcomes.
RESULTS. For children with ASD, the Short Sensory Profile scores Underresponsive/Seeks Sensation and Auditory Filtering explained 47% of the variance in academic performance, yet estimated intelligence was not a significant predictor of academic performance. Significant negative correlations were found between (1) auditory filtering and inattention to cognitive tasks, (2) tactile hypersensitivity and hyperactivity and inattention, and (3) movement sensitivity and oppositional behavior.
CONCLUSION. A pattern of auditory filtering difficulties, sensory underresponsiveness, and sensory seeking was associated with academic underachievement in the children with ASD. Children who have difficulty processing verbal instructions in noisy environments and who often focus on sensory-seeking behaviors appear more likely to underachieve academically.
C1 [Ashburner, Jill] Autism Queensland, Sunnybank, Qld 4109, Australia.
[Ziviani, Jenny; Rodger, Sylvia] Univ Queensland, Sch Hlth & Rehabil Sci, Div Occupat Therapy, Brisbane, Qld 4072, Australia.
RP Ashburner, J (reprint author), Autism Queensland, POB 354, Sunnybank, Qld 4109, Australia.
EM jilla@autismqld.com.au
RI Rodger, Sylvia/F-8738-2010; Ziviani, Jenny/C-1708-2010
CR Achenbach TM, 2001, MANUAL ASEBA SCH AGE
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NR 59
TC 46
Z9 48
PU AMER OCCUPATIONAL THERAPY ASSOC, INC
PI BETHESDA
PA 4720 MONTGOMERY LANE, BETHESDA, MD 20814-3425 USA
SN 0272-9490
J9 AM J OCCUP THER
JI Am. J. Occup. Ther.
PD SEP-OCT
PY 2008
VL 62
IS 5
BP 564
EP 573
PG 10
WC Rehabilitation
SC Rehabilitation
GA 348ZK
UT WOS:000259248400009
PM 18826017
ER
PT J
AU Hagerman, R
AF Hagerman, Randi
TI Clinical manual for the treatment of autism
SO AMERICAN JOURNAL OF PSYCHIATRY
LA English
DT Book Review
CR HOLLANDER E, 2007, CLINICAL MANUAL TREA
NR 1
TC 0
Z9 0
PU AMER PSYCHIATRIC PUBLISHING, INC
PI ARLINGTON
PA 1000 WILSON BOULEVARD, STE 1825, ARLINGTON, VA 22209-3901 USA
SN 0002-953X
J9 AM J PSYCHIAT
JI Am. J. Psychiat.
PD SEP
PY 2008
VL 165
IS 9
BP 1210
EP 1211
DI 10.1176/appi.ajp.2008.08040492
PG 3
WC Psychiatry
SC Psychiatry
GA 344BF
UT WOS:000258899500035
ER
PT J
AU Smith, LE
Greenberg, JS
Seltzer, MM
Hong, J
AF Smith, Leann E.
Greenberg, Jan S.
Seltzer, Marsha Mailick
Hong, Jinkuk
TI Symptoms and behavior problems of adolescents and adults with autism:
Effects of mother-child relationship quality, warmth, and praise
SO AMERICAN JOURNAL ON MENTAL RETARDATION
LA English
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDERS; EXPRESSED EMOTION; SPECTRUM
DISORDERS; MENTAL-HEALTH; INTELLECTUAL DISABILITIES; DIAGNOSTIC
INTERVIEW; PSYCHIATRIC-PATIENTS; DOWN-SYNDROME; SCHIZOPHRENIA; FAMILY
AB Using a cross-lagged panel design, we investigated the impact of positive family processes on change in autism symptoms and behaviors. A sample of 149 co-residing mothers and their adolescent or adult child with autism was drawn from a large, longitudinal study. Maternal warmth and praise were measured using coded speech samples in which mothers talked about their son or daughter. A high level of relationship quality was associated with subsequent reductions in internalizing and externalizing problems as well as reductions in impairments in social reciprocity and repetitive behaviors. Maternal warmth and praise were also related to symptom abatement in the repetitive behaviors domain.
C1 [Smith, Leann E.; Greenberg, Jan S.; Seltzer, Marsha Mailick; Hong, Jinkuk] Univ Wisconsin, Waisman Ctr, Madison, WI 53705 USA.
RP Smith, LE (reprint author), Univ Wisconsin, Waisman Ctr, 1500 Highland Ave, Madison, WI 53705 USA.
EM lsmith@waisman.wisc.edu
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Luckasson R., 2002, MENTAL RETARDATION D
MAGANA AB, 1986, PSYCHIAT RES, V17, P203, DOI 10.1016/0165-1781(86)90049-1
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Rutter M., 2003, ADI R AUTISM DIAGNOS
Ryff CD, 2006, PSYCHOTHER PSYCHOSOM, V75, P85, DOI 10.1159/000090892
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NR 53
TC 28
Z9 29
PU AMER ASSOC MENTAL RETARDATION
PI WASHINGTON
PA 444 N CAPITOL ST, NW, STE 846, WASHINGTON, DC 20001-1512 USA
SN 0895-8017
J9 AM J MENT RETARD
JI Am. J. Ment. Retard.
PD SEP
PY 2008
VL 113
IS 5
BP 387
EP 402
DI 10.1352/2008.113:387-402
PG 16
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 351JQ
UT WOS:000259421000006
PM 18702558
ER
PT J
AU Coben, R
AF Coben, Robert
TI Assessment guided neurofeedback for ASD: EEG analyses
SO APPLIED PSYCHOPHYSIOLOGY AND BIOFEEDBACK
LA English
DT Meeting Abstract
DE neurofeedback; autism; EEG
C1 [Coben, Robert] Long Isl Univ, Massapequa Pk, NY 11762 USA.
EM drcoben@thebrainlabs.com
NR 0
TC 2
Z9 2
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1090-0586
J9 APPL PSYCHOPHYS BIOF
JI Appl. Psychophysiol. Biofeedback
PD SEP
PY 2008
VL 33
IS 3
BP 176
EP 176
PG 1
WC Psychology, Clinical
SC Psychology
GA 351GC
UT WOS:000259411600015
ER
PT J
AU Sokhadze, T
Tasman, A
Baruth, J
Sears, L
Mathai, G
Casanova, M
AF Sokhadze, Tato
Tasman, Allan
Baruth, Joshua
Sears, Lonnie
Mathai, Grace
Casanova, Manuel
TI Dense-array event-related potential study of novelty processing
abnormalities in autism spectrum disorders
SO APPLIED PSYCHOPHYSIOLOGY AND BIOFEEDBACK
LA English
DT Meeting Abstract
DE EEG; ERP; autism
C1 [Sokhadze, Tato] Univ Louisville, Dept Psychiat, Louisville, KY 40292 USA.
EM emsokh01@louisville.edu
NR 0
TC 0
Z9 0
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1090-0586
J9 APPL PSYCHOPHYS BIOF
JI Appl. Psychophysiol. Biofeedback
PD SEP
PY 2008
VL 33
IS 3
BP 179
EP 179
PG 1
WC Psychology, Clinical
SC Psychology
GA 351GC
UT WOS:000259411600025
ER
PT J
AU Cass, H
Gringras, P
March, J
McKendrick, I
O'Hare, AE
Owen, L
Pollin, C
AF Cass, H.
Gringras, P.
March, J.
McKendrick, I.
O'Hare, A. E.
Owen, L.
Pollin, C.
TI Absence of urinary opioid peptides in children with autism
SO ARCHIVES OF DISEASE IN CHILDHOOD
LA English
DT Article
ID DISORDERS
AB Objective: It has been claimed for a number of years that the urine of children with autism contains exogenously derived opioid peptides. This finding is said to reflect a disturbance in the integrity of the gut epithelium, act as a diagnostic marker for autism and predict treatment response to a diet excluding gluten and casein. The aim of the present study was to determine whether exogenous or endogenous peptides were present in the urine of children with autism or of control children.
Design: Case-control study
Setting: Cases were recruited from two tertiary referral centres specialising in autistic spectrum disorders, while controls were recruited from mainstream primary and secondary schools in the same geographical area.
Participants: 65 boys with autism, mean age 7.4 years (range 5-11) and 158 control boys, mean age 7.8 years (range 4.2-11).
Investigations: Urine samples were examined by high pressure liquid chromatography (HPLC) and matrix assisted laser desorbtion ionisation-time of flight mass spectrometry (MALDI-TOF MS) for the presence of a number of putative opioid peptides.
Outcomes: There were no significant differences between the HPLC urinary profiles of the children affected by autism and the typically developing controls. In those cases where HPLC showed peaks in the locations at which opioid peptides might be expected to be found, MALDI-TOF established that these peaks did not, in fact, represent opioid peptides.
Conclusions: Given the lack of evidence for any opioid peptiduria in children with autism, opioid peptides can neither serve as a biomedical marker for autism nor be employed to predict or monitor response to a casein- and gluten-free diet.
C1 [Cass, H.] Great Ormond St Hosp Sick Children, London WC1N 3JH, England.
[Gringras, P.] Evelina Childrens Hosp, London, England.
[March, J.; Pollin, C.] Moredun Res Inst, Penicuik, Midlothian, Scotland.
[McKendrick, I.] JCMB, Biomathemat & Stat Scotland, Edinburgh, Midlothian, Scotland.
[O'Hare, A. E.] Univ Edinburgh, Coll Med & Vet Med, Edinburgh, Midlothian, Scotland.
[Owen, L.] City Univ London, Dept Psychol, Family & Child Psychol Res Ctr, London EC1V 0HB, England.
RP Cass, H (reprint author), Great Ormond St Hosp Sick Children, Great Ormond St, London WC1N 3JH, England.
EM cassh@gosh.nhs.uk
RI McKendrick, Iain/J-5687-2013
FU RD Fund; RHSC, Edinburgh; Chief Scientist Office, Edinburgh;
Ortho-Clinical Diagnostics
FX This research was funded by the R&D Fund, RHSC, Edinburgh and the Chief
Scientist Office, Edinburgh. Dr Lucy Owen's post was funded by
Ortho-Clinical Diagnostics, a UK based diagnostics arm of Johnson and
Johnson.
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NR 22
TC 23
Z9 24
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0003-9888
J9 ARCH DIS CHILD
JI Arch. Dis. Child.
PD SEP
PY 2008
VL 93
IS 9
BP 745
EP 750
DI 10.1136/adc.2006.114389
PG 6
WC Pediatrics
SC Pediatrics
GA 340UB
UT WOS:000258669400008
PM 18337276
ER
PT J
AU Zwaigenbaum, L
Stone, W
AF Zwaigenbaum, Lonnie
Stone, Wendy
TI Untitled
SO AUTISM
LA English
DT Editorial Material
ID AUTISM
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Zwaigenbaum L, 2007, J AUTISM DEV DISORD, V37, P466, DOI 10.1007/s10803-006-0179-x
NR 8
TC 4
Z9 4
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 427
EP 432
DI 10.1177/1362361308097836
PG 6
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000001
PM 18935699
ER
PT J
AU Brian, J
Bryson, SE
Garon, N
Roberts, W
Smith, IM
Szatmari, P
Zwaigenbaum, L
AF Brian, J.
Bryson, S. E.
Garon, N.
Roberts, W.
Smith, I. M.
Szatmari, P.
Zwaigenbaum, L.
TI Clinical assessment of autism in high-risk 18-month-olds
SO AUTISM
LA English
DT Article
DE assessment; autism; early identification; infant; siblings
ID PERVASIVE DEVELOPMENTAL DISORDERS; DIAGNOSTIC OBSERVATION SCHEDULE;
EARLY IDENTIFICATION; SPECTRUM DISORDERS; YOUNG-CHILDREN; EARLY
RECOGNITION; HOME VIDEOTAPES; FOLLOW-UP; AGE; POPULATION
AB Earlier intervention improves outcomes for children with autism spectrum disorders (ASDs), but existing identification tools are at the limits of standardization with 18-month-olds. We assessed potential behavioural markers of ASD at 18 months in a high-risk cohort of infant siblings of children with ASD. Prospective data were collected using the Autism Diagnostic Observation Schedule (ADOS) and Autism Observation Scale for Infants (AOSI) on 155 infant siblings and 73 low-risk controls at 18 months. Infants were classified into three groups (ASD sibs, non-ASD sibs, controls) based on blind best-estimate diagnosis at age 3. Fisher's exact tests, followed by discriminant function analyses, revealed that the majority of informative ADOS items came from the social and behavioural domains, and AOSI items measuring behavioural reactivity and motor control contributed additional information. Findings highlight the importance of considering not only social-communication deficits, but also basic dimensions of temperament including state regulation and motor control when assessing toddlers with suspected ASD.
C1 [Brian, J.] Univ Toronto, Autism Res Unit, Hosp Sick Children, Toronto, ON M5G 1X8, Canada.
[Bryson, S. E.; Garon, N.; Smith, I. M.] IWK Hlth Ctr, Halifax, NS, Canada.
[Szatmari, P.] McMaster Univ, Hamilton, ON, Canada.
[Zwaigenbaum, L.] Univ Alberta, Edmonton, AB, Canada.
RP Brian, J (reprint author), Univ Toronto, Autism Res Unit, Hosp Sick Children, 555 Univ Ave, Toronto, ON M5G 1X8, Canada.
EM jbrian@sickkids.ca
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NR 53
TC 45
Z9 48
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
EI 1461-7005
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 433
EP 456
DI 10.1177/1362361308094500
PG 24
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000002
PM 18805941
ER
PT J
AU Ozonoff, S
Macari, S
Young, GS
Goldring, S
Thompson, M
Rogers, SJ
AF Ozonoff, Sally
Macari, Suzanne
Young, Gregory S.
Goldring, Stacy
Thompson, Meagan
Rogers, Sally J.
TI Atypical object exploration at 12 months of age is associated with
autism in a prospective sample
SO AUTISM
LA English
DT Article
DE autism; diagnosis; early identification; repetitive behavior
ID YOUNG-CHILDREN; SPECTRUM DISORDERS; SPATIAL ATTENTION; HOME VIDEOTAPES;
JOINT ATTENTION; VALIDITY; BEHAVIOR; INFANTS; COMMUNICATION;
QUESTIONNAIRE
AB This prospective study examined object exploration behavior in 66 12-month-old infants, of whom nine were subsequently diagnosed with an autism spectrum disorder. Previous investigations differ on when the repetitive behaviors characteristic of autism are first present in early development. A task was developed that afforded specific opportunities for a range of repetitive uses of objects and was coded blind to outcome status. The autism/ASD outcome group displayed significantly more spinning, rotating, and unusual visual exploration of objects than two comparison groups. The average unusual visual exploration score of the autism/ASD group was over four standard deviations above the mean of the group with no concerns at outcome. Repetitive behaviors at 12 months were significantly related to cognitive and symptomatic status at 36 month outcome. These results suggest that repetitive or stereotyped behaviors may be present earlier than initially thought in very young children developing the autism phenotype.
C1 [Ozonoff, Sally] Univ Calif Davis Hlth Syst, MIND Inst, Sacramento, CA 95817 USA.
[Macari, Suzanne] Yale Univ, New Haven, CT 06520 USA.
RP Ozonoff, S (reprint author), Univ Calif Davis Hlth Syst, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM sjozonoff@ucdavis.edu
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Wetherby AM, 2004, J AUTISM DEV DISORD, V34, P473, DOI 10.1007/s10803-004-2544-y
Zwaigenbaum L, 2005, INT J DEV NEUROSCI, V23, P143, DOI 10.1016/j.ijdevneu.2004.05.001
NR 35
TC 75
Z9 76
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 457
EP 472
DI 10.1177/1362361308096402
PG 16
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000003
PM 18805942
ER
PT J
AU Ibanez, LV
Messinger, DS
Newell, L
Lambert, B
Sheskin, M
AF Ibanez, Lisa V.
Messinger, Daniel S.
Newell, Lisa
Lambert, Brittany
Sheskin, Mark
TI Visual disengagement in the infant siblings of children with an autism
spectrum disorder (ASD)
SO AUTISM
LA English
DT Article
DE at risk; autism spectrum disorders; disengagement; early deficits;
siblings
ID PERVASIVE DEVELOPMENTAL DISORDERS; JOINT ATTENTION; STIMULUS
OVERSELECTIVITY; HOME VIDEOTAPES; COMMUNICATION; AGE; RECOGNITION;
CAREGIVERS; BEHAVIORS; DEFICITS
AB Children with autism spectrum disorders (ASDs) are impaired in visually disengaging attention in both social and non-social contexts. These impairments may, in subtler form, also affect the infant siblings of children with ASD (ASD-sibs). We investigated patterns of visual attention (gazing) in 6-month-old ASD-sibs (n = 17) and the siblings of typically developing children (COMP-sibs: n = 17) during the Face-to-Face/Still-Face Protocol (FFSF), in which parents are sequentially responsive, non-responsive, and responsive to their infants. Throughout the protocol, ASD-sibs shifted their gaze to and from their parents' faces less frequently than did COMP-sibs. The mean durations of ASD-sibs' gazes away from their parents' faces were longer than those of COMP-sibs. ASD-sibs and COMP-sibs did not differ in the mean durations of gazes at their parents' faces. In sum, ASD-sibs showed no deficits in visual interest to their parents' faces, but greater interest than COMP-sibs in non-face stimuli.
C1 [Ibanez, Lisa V.] Univ Miami, Dept Psychol, Coral Gables, FL 33146 USA.
[Newell, Lisa] Indiana Univ Penn, Indiana, PA 15705 USA.
RP Messinger, DS (reprint author), Univ Miami, Dept Psychol, Flipse Bldg Rm 341,5665 Ponce De Leon Blvd, Coral Gables, FL 33146 USA.
EM dmessinger@miami.edu
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NR 39
TC 20
Z9 21
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 473
EP 485
DI 10.1177/1362361308094504
PG 13
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000004
PM 18805943
ER
PT J
AU Wetherby, AM
Brosnan-Maddox, S
Peace, V
Newton, L
AF Wetherby, Amy M.
Brosnan-Maddox, Susan
Peace, Vickie
Newton, Laura
TI Validation of the Infant-Toddler Checklist as a broadband screener for
autism spectrum disorders from 9 to 24 months of age
SO AUTISM
LA English
DT Article
DE autism; diagnosis; Infant-Toddler Checklist; screening
ID DEVELOPMENTAL PROFILE; FOLLOW-UP; 2ND YEAR; CHILDREN; COMMUNICATION;
DIAGNOSIS; IDENTIFICATION; POPULATION; VALIDITY; LIFE
AB There is an urgent requirement for the improvement of early detection of ASDs. This article provides a brief review of research on the accuracy of screeners for children with ASD that have been administered to general pediatric samples and then present results of a population-based study with a broadband screener to detect children with communication delays including children with ASD.
C1 [Wetherby, Amy M.] Florida State Univ, FIRST WORDS Project, Tallahassee, FL 32301 USA.
RP Wetherby, AM (reprint author), Florida State Univ, FIRST WORDS Project, 625B N Adams St, Tallahassee, FL 32301 USA.
EM amy.wetherby@med.fsu.edu
CR Baird G, 2000, J AM ACAD CHILD PSY, V39, P694, DOI 10.1097/00004583-200006000-00007
BARONCOHEN S, 1992, BRIT J PSYCHIAT, V161, P839, DOI 10.1192/bjp.161.6.839
BARONCOHEN S, 1996, BRIT J PSYCHIAT, V168, P158, DOI DOI 10.1192/BJP.168.2.158
Centers for Disease Control and Prevention, 2007, MMWR-MORBID MORTAL W, V56, P1
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CHAWARSKA K, J AUTISM DE IN PRESS
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NR 38
TC 49
Z9 51
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
EI 1461-7005
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 487
EP 511
DI 10.1177/1362361308094501
PG 25
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000005
PM 18805944
ER
PT J
AU Pandey, J
Verbalis, A
Robins, DL
Boorstein, H
Klin, A
Babitz, T
Chawarska, K
Volkmar, F
Green, J
Barton, M
Fein, D
AF Pandey, Juhi
Verbalis, Alyssa
Robins, Diana L.
Boorstein, Hilary
Klin, Ami
Babitz, Tammy
Chawarska, Katarzyna
Volkmar, Fred
Green, James
Barton, Marianne
Fein, Deborah
TI Screening for autism in older and younger toddlers with the Modified
Checklist for Autism in Toddlers
SO AUTISM
LA English
DT Article
DE autism spectrum disorders; early detection; early identification;
pediatric screening
ID PERVASIVE DEVELOPMENTAL DISORDERS; INTENSIVE BEHAVIORAL TREATMENT;
SPECTRUM DISORDERS; EARLY RECOGNITION; HOME VIDEOTAPES; FIELD TRIAL;
CHILDREN; DIAGNOSIS; AGE; REGRESSION
AB The Modified Checklist for Autism in Toddlers (M-CHAT) was used to screen younger (16-23 months) versus older (24-30 months) high- and low-risk toddlers. Refusal rates for follow-up interview showed no group differences, but parents of younger/low-risk children were more likely to refuse evaluation than parents of high-risk children. PPP for an ASD diagnosis was: younger/high-risk 0.79, older/high-risk 0.74, younger/low-risk 0.28, and older/low-risk 0.61, with PPP differing by age within the low-risk group. Most of the children in all groups, however, were diagnosed with a developmental disorder. Symptom severity generally did not differ among groups. Cognitive and adaptive measures showed minimal group differences. Therefore, older and younger toddlers had similar symptomatology and developmental delays; PPP for ASD is better at 24 than 18 months for low-risk children; however, these children are still highly likely to show a developmental disorder. Clinical decision making should balance early identification against the lower specificity of M-CHAT screening for the younger/low-risk group.
C1 [Pandey, Juhi; Verbalis, Alyssa; Boorstein, Hilary; Green, James; Barton, Marianne; Fein, Deborah] Univ Connecticut, Storrs, CT 06269 USA.
[Robins, Diana L.] Georgia State Univ, Atlanta, GA 30303 USA.
[Klin, Ami; Babitz, Tammy; Chawarska, Katarzyna; Volkmar, Fred] Yale Univ, Sch Med, Yale Child Study Ctr, New Haven, CT 06520 USA.
RP Pandey, J (reprint author), Univ Connecticut, Storrs, CT 06269 USA.
EM juhi_pandey@hotmail.com
RI Robins, Diana/D-9959-2011
CR *AM AC PED, 2006, PEDIATRICS, V118, P404
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 51
TC 43
Z9 43
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 513
EP 535
DI 10.1177/1362361308094503
PG 23
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000006
PM 18805945
ER
PT J
AU Robins, DL
AF Robins, Diana L.
TI Screening for autism spectrum disorders in primary care settings
SO AUTISM
LA English
DT Article
DE autism; M-CHAT; screening; toddlers
ID 6-YEAR FOLLOW-UP; YOUNG-CHILDREN; MODIFIED CHECKLIST; DEVELOPMENTAL
DISORDERS; EARLY IDENTIFICATION; EARLY INTERVENTION; EARLY-CHILDHOOD;
AGE; TODDLERS; DIAGNOSIS
AB The need for autism-specific screening during pediatric well-child visits has been established. However, additional support for specific screening instruments is needed. The current study used the Modified Checklist for Autism in Toddlers (M-CHAT) and the M-CHAT Follow-Up Interview to screen 4797 children during toddler checkups. Of the 4797 cases, 466 screened positive on the M-CHAT; of the 362 who completed the follow-up interview, 61 continued to show risk for autism spectrum disorders (ASDs). A total of 41 children have been evaluated; 21 children have been diagnosed with ASD, 17 were classified with non-ASD delays, and three were typically developing. The PPV of M-CHAT plus interview was .57. It is notable that only four of the 21 cases of ASD were flagged by their pediatrician. These findings suggest that the M-CHAT is effective in identifying ASD in primary care settings. Future research will follow this sample longitudinally.
C1 Georgia State Univ, Dept Psychol, Atlanta, GA 30302 USA.
RP Robins, DL (reprint author), Georgia State Univ, Dept Psychol, POB 5010, Atlanta, GA 30302 USA.
EM drobins@gsu.edu
RI Robins, Diana/D-9959-2011
CR ALLISON C, 2006, INT M AUT RES MONTR
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Yoder P, 2006, J CONSULT CLIN PSYCH, V74, P426, DOI 10.1037/0022-006X.74.3.426
NR 62
TC 53
Z9 54
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
EI 1461-7005
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 537
EP 556
DI 10.1177/1362361308094502
PG 20
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000007
PM 18805946
ER
PT J
AU Stone, WL
McMahon, CR
Henderson, LM
AF Stone, Wendy L.
McMahon, Caitlin R.
Henderson, Lynnette M.
TI Use of the Screening Tool for Autism in Two-Year-Olds (STAT) for
children under 24 months
SO AUTISM
LA English
DT Article
DE autism; early identification; infant; sensitivity; specificity
ID PERVASIVE DEVELOPMENTAL DISORDERS; INTENSIVE BEHAVIORAL TREATMENT;
TRAITS QUESTIONNAIRE ESAT; SPECTRUM DISORDER; DIAGNOSTIC INTERVIEW;
EARLY RECOGNITION; MENTAL-RETARDATION; EARLY INTERVENTION; MODIFIED
CHECKLIST; YOUNGER SIBLINGS
AB The study examined the properties of the Screening Tool for Autism in Two-Year-Olds (STAT) for children under 24 months. The STAT provides a standard context for observing social-communicative behavior in play, imitation, and communication. Seventy-one children received the STAT between 12 and 23 months of age and a follow-up diagnostic evaluation after 24 months. All had an older sibling with an autism spectrum diagnosis (n = 59) or had been referred for evaluation for concerns about autism (n = 12). Signal detection analysis resulted in a cut score of 2.75 for this sample, which yielded a sensitivity of 0.95, specificity of 0.73, positive predictive value of 0.56, and negative predictive value of 0.97. False positives were highest for the 12- to 13-month-old age group; STAT screening properties were improved when the sample was limited to children 14 months and older. Implications for using the STAT with children under 24 months are discussed.
C1 [Stone, Wendy L.; McMahon, Caitlin R.; Henderson, Lynnette M.] Vanderbilt Univ, Nashville, TN USA.
RP Stone, WL (reprint author), Vanderbilt Kennedy Ctr TRIAD, Peabody Box 74,230 Appleton Pl, Nashville, TN 37203 USA.
EM wendy.stone@vanderbilt.edu
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NR 63
TC 27
Z9 28
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 557
EP 573
DI 10.1177/1362361308096403
PG 17
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000008
PM 18805947
ER
PT J
AU Wachtel, K
Carter, AS
AF Wachtel, Karen
Carter, Alice S.
TI Reaction to diagnosis and parenting styles among mothers of young
children with ASDs
SO AUTISM
LA English
DT Article
DE autism spectrum disorder; parenting styles; resolution to diagnosis;
parent-child interaction
ID MATERNAL SENSITIVITY; AUTISM; COMMUNICATION; SPECIFICITY; ATTACHMENT;
DISORDERS; CHILDHOOD; BEHAVIORS; INTERVIEW; LANGUAGE
AB When a child is diagnosed with an autism spectrum disorder (ASD) parents often experience a range of difficult feelings, which typically are not addressed in child-focused interventions. This study examined the relationship between a mother's acceptance of and sense of resolution regarding her child's diagnosis of an ASD and maternal interaction style, controlling for child competence, autism symptoms and maternal depression. Participants included 63 children with an ASD between 20 and 50 months of age and their mothers. Mothers who were more emotionally resolved were rated as higher in Cognitive Engagement and Supportive Engagement in play interactions, reflecting greater verbal and nonverbal scaffolding to enhance the child's play and attention to activities and greater reciprocity and mutual enjoyment. This study highlights the importance of considering a mother's resolution about her child's diagnosis, suggesting that maternal emotions and cognitions associated with the diagnosis may be potential targets for intervention.
C1 [Carter, Alice S.] Univ Massachusetts, Dept Psychol, Boston, MA 02125 USA.
RP Carter, AS (reprint author), Univ Massachusetts, Dept Psychol, 100 Morrissey Blvd, Boston, MA 02125 USA.
EM alicescarter@umb.edu
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NR 36
TC 27
Z9 29
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD SEP
PY 2008
VL 12
IS 5
BP 575
EP 594
DI 10.1177/1362361308094505
PG 20
WC Psychology, Developmental
SC Psychology
GA 350YY
UT WOS:000259391000009
PM 18805948
ER
PT J
AU Thompson, T
AF Thompson, Travis
TI Self-Awareness: Behavior Analysis and Neuroscience
SO BEHAVIOR ANALYST
LA English
DT Article; Proceedings Paper
CT 34th Annual Meeting of the Association-for-Behavior-Analysis
CY MAY, 2008
CL Chicago, IL
SP Assoc Behav Anal
DE self-awareness; autoclitics; neuroplasticity
ID AUTISM SPECTRUM DISORDERS; MOTOR CORTEX; CHILDREN; LEVEL; GYRUS; MIND
AB Self-awareness is a specific type of autoclitic discriminative behavior and inferential generalization to similar performances exhibited by other people. Brain imaging findings take on special importance within behavior analysis when they indicate that dysfunctions in these areas are related to differential effects of our interventions, with some acquiring Substantially typical self-awareness skills and others failing to do so. It appears that those individuals whose brain dysfunctions are limited to these areas, and are not part of more generalized brain abnormalities, are amenable to Substantial acquisition of those most. basic of human skills called self-awareness, whereas individuals with more generalized brain dysfunction are not so disposed. Through a combination of less or more effective reaching contingencies during childhood, and degrees of dysfunction of those brain structures, some children grow up kicking self-reflective abilities and self-insight, whereas others are extraordinarily astute at those capacities. Among children with autism spectrum disorders who lack those skills due to abnormal brain development, approximately half of them can acquire those skills, at least to some degree through the use of effective, intensive, early behavior therapy methods.
C1 [Thompson, Travis] Univ Minnesota, Sch Med, Minneapolis, MN 55455 USA.
RP Thompson, T (reprint author), 2187 Ferris Lane, Roseville, MN 55113 USA.
EM thomp199@umn.edu
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NR 48
TC 1
Z9 1
PU SOC ADVANCEMENT BEHAVIOR ANALYSIS
PI KALAMAZOO
PA WESTERN MICHIGAN UNIV, 260 WOOD HALL, KALAMAZOO, MI 49008-5052 USA
SN 0738-6729
J9 BEHAV ANALYST
JI Behav. Anal.
PD FAL
PY 2008
VL 31
IS 2
BP 137
EP 144
PG 8
WC Psychology, Clinical
SC Psychology
GA 380IO
UT WOS:000261459600005
PM 22478507
ER
PT J
AU Kupfer, J
AF Kupfer, Jeff
TI Destination Unknown: A Behavior Analysis Journey into Autism Education
using Sulzer-Azaroff and Associates' Applying Behavior Analysis across
the Autism Spectrum: A Field Guide for Practitioners
SO BEHAVIOR ANALYST
LA English
DT Editorial Material
RP Kupfer, J (reprint author), POB 85, Erie, CO 80516 USA.
EM JKupf@aol.com
CR Holland J, 1961, ANAL BEHAV
Johnston J. M., 1993, STRATEGIES TACTICS B, V2nd
Sidman M., 1960, TACTICS SCI RES
SULZERAZAROFF B, 2008, SLOAN CENTURY SERIES
2007, DEP DEFENSE REPORT P
NR 5
TC 0
Z9 0
PU SOC ADVANCEMENT BEHAVIOR ANALYSIS
PI KALAMAZOO
PA WESTERN MICHIGAN UNIV, 260 WOOD HALL, KALAMAZOO, MI 49008-5052 USA
SN 0738-6729
J9 BEHAV ANALYST
JI Behav. Anal.
PD FAL
PY 2008
VL 31
IS 2
BP 205
EP 211
PG 7
WC Psychology, Clinical
SC Psychology
GA 380IO
UT WOS:000261459600009
ER
PT J
AU Prendergast, A
Barry, C
Foley, A
Regan, C
AF Prendergast, A.
Barry, C.
Foley, A.
Regan, C.
TI Treatment with histone deacetylase inhibitors SAHA and MS-275
ameliorates the core symptoms of asocialty and cognitive impairment in
an animal model of autism
SO BEHAVIOURAL PHARMACOLOGY
LA English
DT Meeting Abstract
CT European-Behavioural-Pharmacology-Society Workshop on Behavioural
Genertics and Neuropsychiatric Disorders
CY AUG 25-27, 2008
CL Cork, IRELAND
SP European Behavioural Pharmacol Soc
C1 [Prendergast, A.; Barry, C.; Foley, A.; Regan, C.] Berand Neuropharmacol, Dublin 4, Ireland.
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0955-8810
J9 BEHAV PHARMACOL
JI Behav. Pharmacol.
PD SEP
PY 2008
VL 19
IS 5-6
MA S58
BP 667
EP 668
PG 2
WC Behavioral Sciences; Neurosciences; Pharmacology & Pharmacy
SC Behavioral Sciences; Neurosciences & Neurology; Pharmacology & Pharmacy
GA 342DD
UT WOS:000258763900081
ER
PT J
AU McLellan, AS
Wynne, F
Ning, Z
Ball, M
Moore, T
AF McLellan, A. S.
Wynne, F.
Ning, Z.
Ball, M.
Moore, T.
TI Sexual antagonism and autism susceptibility in the imprinted XQ/YQ
pseudoautosomal region (PAR2)
SO BEHAVIOURAL PHARMACOLOGY
LA English
DT Meeting Abstract
CT European-Behavioural-Pharmacology-Society Workshop on Behavioural
Genertics and Neuropsychiatric Disorders
CY AUG 25-27, 2008
CL Cork, IRELAND
SP European Behavioural Pharmacol Soc
C1 [McLellan, A. S.; Wynne, F.; Ning, Z.; Ball, M.; Moore, T.] Natl Univ Ireland Univ Coll Cork, Dept Biochem, Biosci Inst, Cork, Ireland.
EM t.moore@ucc.ie
RI Moore, Tom/B-1471-2015
NR 0
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0955-8810
J9 BEHAV PHARMACOL
JI Behav. Pharmacol.
PD SEP
PY 2008
VL 19
IS 5-6
MA S59
BP 668
EP 668
PG 1
WC Behavioral Sciences; Neurosciences; Pharmacology & Pharmacy
SC Behavioral Sciences; Neurosciences & Neurology; Pharmacology & Pharmacy
GA 342DD
UT WOS:000258763900082
ER
PT J
AU Greene, CM
Braet, W
Johnson, KA
Bellgrove, MA
AF Greene, Ciara M.
Braet, Wouter
Johnson, Katherine A.
Bellgrove, Mark A.
TI Imaging the genetics of executive function
SO BIOLOGICAL PSYCHOLOGY
LA English
DT Review
DE neuroimaging; executive functions; dopamine; genetics
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER;
DEFICIT-HYPERACTIVITY-DISORDER; CATECHOL-O-METHYLTRANSFERASE; SPATIAL
WORKING-MEMORY; DORSOLATERAL PREFRONTAL CORTEX;
DOPAMINE-BETA-HYDROXYLASE; VAL(108/158) MET GENOTYPE;
RESPONSE-INHIBITION TASK; CARD SORTING TEST; SUSTAINED ATTENTION
AB Recent advances in neuroimaging technologies have allowed ever more detailed studies of the human brain. The combination of neuroimaging techniques with genetics may provide a more sensitive measure of the influence of genetic variants on cognitive function than behavioural measures alone. Here we present a review of functional magnetic resonance imaging (fMRI) studies of genetic links to executive functions, focusing on sustained attention, working memory and response inhibition. In addition to studies in the normal population, we also address findings from three clinical populations: schizophrenia, ADHD and autism spectrum disorders. While the findings in the populations studied do not always converge, they all point to the usefulness of neuroimaging techniques such as fMRI as potential endophenotypes for parsing the genetic aetiology of executive function. (C) 2007 Elsevier B.V. All rights reserved.
C1 [Greene, Ciara M.; Braet, Wouter; Johnson, Katherine A.] Univ Dublin Trinity Coll, Trinity Coll, Inst Neurosci, Dublin 2, Ireland.
[Greene, Ciara M.; Johnson, Katherine A.] Univ Dublin Trinity Coll, Dept Psychiat, Neuropsychiat Genet Grp, Dublin 2, Ireland.
[Bellgrove, Mark A.] Univ Queensland, Sch Psychol, Brisbane, Qld 4072, Australia.
[Bellgrove, Mark A.] Univ Queensland, Queensland Brain Inst, Brisbane, Qld 4072, Australia.
RP Greene, CM (reprint author), Univ Dublin Trinity Coll, Trinity Coll, Inst Neurosci, Dublin 2, Ireland.
EM cmgreene@gmail.com
RI Braet, Wouter/A-5061-2009
FU Irish Health Research Board, Science Foundation Ireland; Irish Higher
Education Authority's Programme
FX This work was supported by grants from the Irish Health Research Board,
Science Foundation Ireland and the Irish Higher Education Authority's
Programme for Research in Third-Level Institutions. KAJ is supported by
the Health Research Board of Ireland.
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NR 177
TC 30
Z9 30
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0301-0511
J9 BIOL PSYCHOL
JI Biol. Psychol.
PD SEP
PY 2008
VL 79
IS 1
BP 30
EP 42
DI 10.1016/j.biopsycho.2007.11.009
PG 13
WC Psychology, Biological; Behavioral Sciences; Psychology; Psychology,
Experimental
SC Psychology; Behavioral Sciences
GA 341TU
UT WOS:000258738800004
PM 18178303
ER
PT J
AU Silverman, C
AF Silverman, Chloe
TI Fieldwork on Another Planet: Social Science Perspectives on the Autism
Spectrum
SO BIOSOCIETIES
LA English
DT Review
DE Autism Spectrum Disorders; Disability; Neurodiversity; Social Science
AB The autism spectrum disorders are a group of neurodevelopmental syndromes of communication, behavior and social cognition. Over the past decade, they have received increasing attention from scholars in the social sciences. This research has been motivated by the prospect of critiquing and improving support services and therapies, by self-advocates who have argued that autism should be tolerated as a form of difference rather than treated as a disorder, and by the interest inherent in syndromes that seem to affect many of the attributes that we use to define personhood. In this commentary, I review social science research on the autism spectrum. I identify some key approaches in the work, including the idea of autism as a culture, transcultural comparisons, studies based on treatment strategies, investigations of subjectivity and interpersonal relations, and research on social movements. In the process, I suggest some further directions for this area of research. I also consider some reasons why the autism spectrum disorders are a particularly interesting site for studies of the ways that biomedical information is used to craft individual and group identities.
C1 Penn State Univ, STS Program, University Pk, PA 16802 USA.
RP Silverman, C (reprint author), Penn State Univ, STS Program, 201 Old Bot, University Pk, PA 16802 USA.
EM cbs14@psu.edu
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NR 104
TC 20
Z9 20
PU PALGRAVE MACMILLAN LTD
PI BASINGSTOKE
PA BRUNEL RD BLDG, HOUNDMILLS, BASINGSTOKE RG21 6XS, HANTS, ENGLAND
SN 1745-8552
J9 BIOSOCIETIES
JI BioSocieties
PD SEP
PY 2008
VL 3
IS 3
BP 325
EP 341
DI 10.1017/S1745855208006236
PG 17
WC Social Sciences, Biomedical
SC Biomedical Social Sciences
GA V14OJ
UT WOS:000207743400007
ER
PT J
AU Thakkar, KN
Polli, FE
Joseph, RM
Tuch, DS
Hadjikhani, N
Barton, JJS
Manoach, DS
AF Thakkar, Katharine N.
Polli, Frida E.
Joseph, Robert M.
Tuch, David S.
Hadjikhani, Nouchine
Barton, Jason J. S.
Manoach, Dara S.
TI Response monitoring, repetitive behaviour and anterior cingulate
abnormalities in autism spectrum disorders (ASD)
SO BRAIN
LA English
DT Review
DE autism; anterior cingulate cortex; response monitoring; functional MRI;
diffusion tensor imaging
ID OBSESSIVE-COMPULSIVE DISORDER; EVENT-RELATED FMRI; HIGH-FUNCTIONING
AUTISM; EMOTIONAL PROCESSING DISORDER; ERROR-RELATED NEGATIVITY;
SURFACE-BASED ANALYSIS; CORTICAL SURFACE; ANTISACCADE TASK;
ANTI-SACCADES; PRO-SACCADES
AB Autism spectrum disorders (ASD) are characterized by inflexible and repetitive behaviour. Response monitoring involves evaluating the consequences of behaviour and making adjustments to optimize outcomes. Deficiencies in this function, and abnormalities in the anterior cingulate cortex (ACC) on which it relies, have been reported as contributing factors to autistic disorders. We investigated whether ACC structure and function during response monitoring were associated with repetitive behaviour in ASD. We compared ACC activation to correct and erroneous antisaccades using rapid presentation event-related functional MRI in 14 control and ten ASD participants. Because response monitoring is the product of coordinated activity in ACC networks, we also examined the microstructural integrity of the white matter (WM) underlying this brain region using diffusion tensor imaging (DTI) measures of fractional anisotropy (FA) in 12 control and 12 adult ASD participants. ACC activation and FA were examined in relation to Autism Diagnostic Interview-Revised ratings of restricted and repetitive behaviour. Relative to controls, ASD participants: (i) made more antisaccade errors and responded more quickly on correct trials; (ii) showed reduced discrimination between error and correct responses in rostral ACC (rACC), which was primarily due to (iii) abnormally increased activation on correct trials and (iv) showed reduced FA in WM underlying ACC. Finally, in ASD (v) increased activation on correct trials and reduced FA in rACC WM were related to higher ratings of repetitive behaviour. These findings demonstrate functional and structural abnormalities of the ACC in ASD that may contribute to repetitive behaviour. rACC activity following errors is thought to reflect affective appraisal of the error. Thus, the hyperactive rACC response to correct trials can be interpreted as a misleading affective signal that something is awry, which may trigger repetitive attempts at correction. Another possible consequence of reduced affective discrimination between error and correct responses is that it might interfere with the reinforcement of responses that optimize outcomes. Furthermore, dysconnection of the ACC, as suggested by reduced FA, to regions involved in behavioural control might impair on-line modulations of response speed to optimize performance (i.e. speed-accuracy trade-off) and increase error likelihood. These findings suggest that in ASD, structural and functional abnormalities of the ACC compromise response monitoring and thereby contribute to behaviour that is rigid and repetitive rather than flexible and responsive to contingencies. Illuminating the mechanisms and clinical significance of abnormal response monitoring in ASD represents a fruitful avenue for further research.
C1 [Thakkar, Katharine N.; Polli, Frida E.; Manoach, Dara S.] Harvard Univ, Sch Med, Massachusetts Gen Hosp, Dept Psychiat, Boston, MA 02215 USA.
[Polli, Frida E.] Suffolk Univ, Dept Psychol, Boston, MA 02114 USA.
[Joseph, Robert M.] Boston Univ, Sch Med, Dept Anat & Neurobiol, Boston, MA 02118 USA.
[Tuch, David S.; Hadjikhani, Nouchine] Massachusetts Gen Hosp, Dept Radiol, Boston, MA 02114 USA.
[Tuch, David S.; Hadjikhani, Nouchine; Manoach, Dara S.] Athinoula A Martinos Ctr Biomed Imaging, Charlestown, MA 02129 USA.
[Barton, Jason J. S.] Univ British Columbia, Dept Neurol, Vancouver, BC V5Z 1M9, Canada.
[Barton, Jason J. S.] Univ British Columbia, Dept Ophthalmol & Visual Sci, Vancouver, BC V5Z 1M9, Canada.
RP Manoach, DS (reprint author), 149 13th St,Room 2608, Charlestown, MA 02129 USA.
EM dara@nmr.mgh.harvard.edu
RI Hadjikhani, Nouchine/C-2018-2008; Barton, Jason/A-6362-2012; Joseph,
Roy/D-8530-2015
OI Hadjikhani, Nouchine/0000-0003-4075-3106;
FU National Institute for Mental Health [R01 MH67720]; Mental Illness
Neuroscience Discovery (MIND) Institute [DOE DE-FG02-99ER62764];
National Center for Research Resources [P41RR14075]; National Institute
for Mental Health NRSA [MH72120]
FX The authors wish to thank Mark Vangel for statistical consultation,
Robert Levy, Kim Ono, Christopher Sherman and Jeremy Young for help with
manuscript preparation, and Matt Cain and Jay Edelman for technical
assistance. Some of these data were presented at the annual meeting of
the Society for Neuroscience in Washington, DC, in November, 2005. This
study was supported by National Institute for Mental Health [R01 MH67720
(D. S. M.); MH72120 (F. E. P.)]; Mental Illness Neuroscience Discovery
(MIND) Institute (DOE DE-FG02-99ER62764): The National Center for
Research Resources (P41RR14075). Funding to pay the Open Access
publication charges for this article was provided by the National
Institute for Mental Health NRSA MH72120.
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Belmonte, Matthew K.
Bullmore, Edward T.
Bernard, Frederic A.
Baron-Cohen, Simon
TI Brain hyper-reactivity to auditory novel targets in children with
high-functioning autism
SO BRAIN
LA English
DT Article
DE autism; novelty; fMRI; auditory; oddball
ID EVENT-RELATED FMRI; SPECTRUM QUOTIENT AQ; SELECTIVE ATTENTION; ODDBALL
TASK; GENERAL-POPULATION; PARIETAL CORTEX; FRONTAL-CORTEX; ERP;
POTENTIALS; P300
AB Although communication and social difficulties in autism have received a great deal of research attention, the other key diagnostic feature, extreme repetitive behaviour and unusual narrow interests, has been addressed less often. Also known as resistance to change this may be related to atypical processing of infrequent, novel stimuli. This can be tested at sensory and neural levels. Our aims were to (i) examine auditory novelty detection and its neural basis in children with autism spectrum conditions (ASC) and (ii) test for brain activation patterns that correlate quantitatively with number of autistic traits as a test of the dimensional nature of ASC. The present study employed event-related fMRI during a novel auditory detection paradigm. Participants were twelve 10- to 15-year-old children with ASC and a group of 12 age-, IQ- and sex-matched typical controls. The ASC group responded faster to novel target stimuli. Group differences in brain activity mainly involved the right prefrontalpremotor and the left inferior parietal regions, which were more activated in the ASC group than in controls. In both groups, activation of prefrontal regions during target detection was positively correlated with Autism Spectrum Quotient scores measuring the number of autistic traits. These findings suggest that target detection in autism is associated not only with superior behavioural performance (shorter reaction time) but also with activation of a more widespread network of brain regions. This pattern also shows quantitative variation with number of autistic traits, in a continuum that extends to the normal population. This finding may shed light on the neurophysiological process underlying narrow interests and what clinically is called need for sameness.
C1 [Gomot, Marie] Univ Tours, CHRU, CHU Bretonneau, INSERM,U930,Ctr Pedopsychiat, F-37044 Tours 9, France.
[Gomot, Marie; Belmonte, Matthew K.; Baron-Cohen, Simon] Univ Cambridge, Dept Psychiat, Autism Res Ctr, Cambridge CB2 1TN, England.
[Belmonte, Matthew K.] Cornell Univ, Dept Human Dev, Ithaca, NY USA.
[Bullmore, Edward T.; Bernard, Frederic A.] Univ Cambridge, Dept Psychiat, Brain Mapping Unit, Cambridge CB2 1TN, England.
[Bernard, Frederic A.] Ecole Normale Super, Dept Etude Cognit, F-75231 Paris, France.
RP Gomot, M (reprint author), Univ Tours, CHRU, CHU Bretonneau, INSERM,U930,Ctr Pedopsychiat, 2 Bd Tonnelle, F-37044 Tours 9, France.
EM m.gomot@chu-tours.fr
RI Bernard, Frederic/B-7860-2008; Bullmore, Edward/C-1706-2012
OI Bullmore, Edward/0000-0002-8955-8283
FU Fondation pour la Recherche Medicale; 'Region Centre' and the 'Fondation
d'Entreprise France Telecom'; UK Medical Research Council; The Wolfson
Brain Imaging Centre
FX This study was supported by grants from the Fondation pour la Recherche
Medicale, the 'Region Centre' and the 'Fondation d'Entreprise France
Telecom' (M. G.), Cure Autism Now (M. K. B.), Merck Sharp and Dohme
Post-doctoral Fellowship award (F. A. B.) and the UK Medical Research
Council (S. B.-C., E. T. B.). The Wolfson Brain Imaging Centre is
supported by an MRC Cooperative Group grant. The authors thank Jose
Alcantara and Matt Davis and most of all the participants and their
parents for their contributions to this study.
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NR 69
TC 51
Z9 53
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0006-8950
J9 BRAIN
JI Brain
PD SEP
PY 2008
VL 131
BP 2479
EP 2488
DI 10.1093/brain/awn172
PN 9
PG 10
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 343OA
UT WOS:000258862200022
PM 18669482
ER
PT J
AU Hambrook, D
Tchanturia, K
Schmidt, U
Russell, T
Treasure, J
AF Hambrook, David
Tchanturia, Kate
Schmidt, Ulrike
Russell, Tamara
Treasure, Janet
TI Empathy, systemizing, and autistic traits in anorexia nervosa: A pilot
study
SO BRITISH JOURNAL OF CLINICAL PSYCHOLOGY
LA English
DT Article
ID HIGH-FUNCTIONING AUTISM; NORMAL SEX-DIFFERENCES; ASPERGER-SYNDROME;
QUOTIENT; ADULTS
AB Objectives. This pilot study examined whether patients with anorexia nervosa (AN) would display an empathizing-systemizing psychometric profile similar to that found in autism spectrum disorders (ASD), and whether people with AN would score highly on a measure of autistic traits.
Method. Self-report measures of empathy, systemizing, and autistic traits were administered to 22 female AN patients and 45 female healthy controls (HC).
Results. AN patients and HCs did not differ significantly in their self-reported empathy and systemizing. AN patients scored significantly higher than HCs on the autism-spectrum quotient.
Conclusions. Replication of these findings is required with larger samples and more sensitive measures.
C1 [Hambrook, David; Tchanturia, Kate; Schmidt, Ulrike; Russell, Tamara; Treasure, Janet] Kings Coll London, Sect Eating Disorders, Inst Psychiat, Div Psychol Med & Psychiat, London SE5 8AF, England.
RP Tchanturia, K (reprint author), Kings Coll London, Sect Eating Disorders PO59, Inst Psychiat, London SE5 8AF, England.
EM k.tchanturia@iop.kcl.ac.uk
RI Tchanturia, Kate/H-1474-2011
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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ZUCKER NL, 2007, PSYCHOL BULL, V113, P976
NR 13
TC 42
Z9 42
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0144-6657
J9 BRIT J CLIN PSYCHOL
JI Br. J. Clin. Psychol.
PD SEP
PY 2008
VL 47
BP 335
EP 339
DI 10.1348/014466507X272475
PN 3
PG 5
WC Psychology, Clinical
SC Psychology
GA 333XC
UT WOS:000258185900007
PM 18208640
ER
PT J
AU Cramer, H
Carlin, J
AF Cramer, Helen
Carlin, Jeanne
TI Family-based short breaks (respite) for disabled children: Results from
the fourth national survey
SO BRITISH JOURNAL OF SOCIAL WORK
LA English
DT Article
DE family-based short breaks; respite; disabled children; services
ID SERVICES; NEEDS; CARE; PARENTS
AB A survey of family-based short breaks for disabled children in the UK was undertaken in order to investigate the state of current services. The research builds on the work of previous surveys and takes the perspective of the service co-ordinators. The findings show that family-based short breaks are rapidly changing and diversifying. The number of schemes with contract carers has gone up and the number of schemes with additional sitting and befriending services has gone down. The reasons for the growth in contract carers are understood better than the reasons for the decline in sitting and befriending. The changing population of disabled children and the increased number of children with complex needs wanting a service are key contributory factors in these trends. The most common profile of children waiting the longest for a service is still teenage boys with autism or 'challenging behaviour'. Some improvements have been observable in the numbers of families and children from ethnic minorities accessing services. The introduction of Care Standards has received a mixed response. The challenge for short breaks is to be flexible and responsive to the local needs of children and families.
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RP Cramer, H (reprint author), Univ Bristol, Acad Unit Primary Hlth Care, 25-27 Belgrave Rd, Bristol BS8 2AA, Avon, England.
EM Helen.Cramer@bristol.ac.uk
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NR 34
TC 7
Z9 7
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0045-3102
J9 BRIT J SOC WORK
JI Br. J. Soc. Work
PD SEP
PY 2008
VL 38
IS 6
BP 1060
EP 1075
DI 10.1093/bjsw/bcl393
PG 16
WC Social Work
SC Social Work
GA 356OG
UT WOS:000259786900003
ER
PT J
AU Fatemi, SH
Reutiman, TJ
Folsom, TD
Sidwell, RW
AF Fatemi, S. Hossein
Reutiman, Teri J.
Folsom, Timothy D.
Sidwell, Robert W.
TI The role of cerebellar genes in pathology of autism and schizophrenia
SO CEREBELLUM
LA English
DT Review
DE Psychiatric; infection; cerebellar; gene
ID VESICULAR GLUTAMATE TRANSPORTER-1; CAUSES DIFFERENTIAL EXPRESSION;
PRENATAL VIRAL-INFECTION; ACID DECARBOXYLASE 65; ALZHEIMERS-DISEASE;
GABA(A) RECEPTOR; ALTERED EXPRESSION; PREFRONTAL CORTEX;
BIPOLAR-DISORDER; SYNAPTIC PATHOLOGY
AB Schizophrenia and autism are neurodevelopmental diseases that have genetic as well as environmental etiologies. Both disorders have been associated with prenatal viral infection. Brain imaging and postmortem studies have found alterations in the structure of the cerebellum as well as changes in gene expression. Our laboratory has developed an animal model using prenatal infection of mice with human influenza virus that has demonstrated changes in behavior, pharmacology, structure, and gene expression in the brains of exposed offspring. In the current communication we describe altered expression of cerebellar genes associated with development of brain disorder in a mouse model for schizophrenia and autism and correlate these changes with those involved in the pathology of these two disorders.
C1 [Fatemi, S. Hossein; Reutiman, Teri J.; Folsom, Timothy D.] Univ Minnesota, Sch Med, Dept Psychiat, Div Neurosci Res, Minneapolis, MN 55455 USA.
[Fatemi, S. Hossein] Univ Minnesota, Sch Med, Dept Pharmacol, Minneapolis, MN 55455 USA.
[Fatemi, S. Hossein] Univ Minnesota, Sch Med, Dept Neurosci, Minneapolis, MN 55455 USA.
[Sidwell, Robert W.] Utah State Univ, Dept Anim Dairy & Vet Sci, Inst Antiviral Res, Logan, UT 84322 USA.
RP Fatemi, SH (reprint author), Univ Minnesota, Sch Med, Dept Psychiat, Div Neurosci Res, MMC 392,420 Delaware St SE, Minneapolis, MN 55455 USA.
EM fatem002@umn.edu
FU National Institute of Child Health and Human Development [1R01
HD046589-01A2]; Jonty Foundation; NIH [N01-AI-15435]
FX Grant support by National Institute of Child Health and Human
Development (#1R01 HD046589-01A2) to and Jonty Foundation SHF and from
the virology branch NIAID, NIH (N01-AI-15435) to RWS is gratefully
acknowledged.
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NR 140
TC 22
Z9 23
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1473-4222
J9 CEREBELLUM
JI Cerebellum
PD SEP
PY 2008
VL 7
IS 3
BP 279
EP 294
DI 10.1007/s12311-008-0017-0
PG 16
WC Neurosciences
SC Neurosciences & Neurology
GA 359OI
UT WOS:000259996900006
PM 18418686
ER
PT J
AU Whitney, ER
Kemper, TL
Bauman, ML
Rosene, DL
Blatt, GJ
AF Whitney, Elizabeth R.
Kemper, Thomas L.
Bauman, Margaret L.
Rosene, Douglas L.
Blatt, Gene J.
TI Cerebellar Purkinje Cells are Reduced in a Subpopulation of Autistic
Brains: A Stereological Experiment Using Calbindin-D28k
SO CEREBELLUM
LA English
DT Article
DE Purkinje cells; Autism; Calbindin-D28k
ID COGNITIVE-AFFECTIVE SYNDROME; RHESUS-MONKEY; BASIS PONTIS;
CEREBROCEREBELLAR SYSTEM; MENTAL-RETARDATION; PREFRONTAL CORTEX;
PHENYTOIN THERAPY; COUNTING METHODS; PROJECTIONS; DEGENERATION
AB Although a decreased number of cerebellar Purkinje cells (PCs) in the autistic brain has been widely reported with a variety of qualitative and quantitative methods, the more accurate method of cell counting with modern stereology has not yet been employed. An additional possible problem with prior reports is the use of Nissl staining to identify the PCs, as this can miss cells due to staining irregularities. In the present study, PCs were immunostained for calbindin-D28k (CB), as this has been shown to be a more reliable marker for PCs than the Nissl stain, with more than 99% of the PCs immunopositive (Whitney, Kemper, Rosene, Bauman, Blatt, J Neurosci Methods 168:42-47, 2008). Using stereology and CB immunostaining, the density of PCs was determined in serial sections from a consistently defined area of the cerebellar hemisphere in four control and six autistic brains, with the density of PCs then correlated with the clinical severity of autism. Overall, there was no significant difference in the density of PCs between the autistic and control groups. However, three of six autistic brains had PC numbers that fell within the control range, whereas the remaining three autistic brains revealed a reduction compared with the control brains. These data demonstrate that a reduction in cerebellar PCs was not a consistent feature of these autistic brains and that it occurred without discernible correlation between their density and the clinical features or severity of autism.
C1 [Whitney, Elizabeth R.; Kemper, Thomas L.; Bauman, Margaret L.; Rosene, Douglas L.; Blatt, Gene J.] Boston Univ, Sch Med, Dept Anat & Neurobiol, Boston, MA 02118 USA.
[Bauman, Margaret L.] Massachusetts Gen Hosp, Dept Neurol, Boston, MA 02114 USA.
RP Whitney, ER (reprint author), Boston Univ, Sch Med, Dept Anat & Neurobiol, L-1004,715 Albany St, Boston, MA 02118 USA.
EM ewhitney@bu.edu
RI Rosene, Douglas/G-1973-2015
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 43
TC 62
Z9 63
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1473-4222
J9 CEREBELLUM
JI Cerebellum
PD SEP
PY 2008
VL 7
IS 3
BP 406
EP 416
DI 10.1007/s12311-008-0043-y
PG 11
WC Neurosciences
SC Neurosciences & Neurology
GA 359OI
UT WOS:000259996900018
PM 18587625
ER
PT J
AU Skirrow, P
AF Skirrow, Paul
TI Counselling People on the Autism Spectrum: A Practical Manual
SO CHILD AND ADOLESCENT MENTAL HEALTH
LA English
DT Book Review
C1 [Skirrow, Paul] Asperger Team, Liverpool, Merseyside, England.
RP Skirrow, P (reprint author), Asperger Team, Liverpool, Merseyside, England.
CR Paxton K., 2007, COUNSELLING PEOPLE A
NR 1
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1475-357X
J9 CHILD ADOL MENT H-UK
JI Child Adolesc. Ment. Health
PD SEP
PY 2008
VL 13
IS 3
BP 153
EP 153
DI 10.1111/j.1475-3588.2008.00500_1.x
PG 1
WC Psychology, Clinical; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA V16FA
UT WOS:000207854300009
ER
PT J
AU Makhoul, S
Le Couteur, A
AF Makhoul, Samer
Le Couteur, Ann
TI Clinical Manual for the Treatment of Autism
SO CHILD AND ADOLESCENT MENTAL HEALTH
LA English
DT Book Review
C1 [Makhoul, Samer] Newcastle Univ, Newcastle Upon Tyne, Tyne & Wear, England.
Fleming Nuffield Unit, Newcastle Upon Tyne, Tyne & Wear, England.
RP Makhoul, S (reprint author), Newcastle Univ, Newcastle Upon Tyne, Tyne & Wear, England.
CR Anagnostou E., 2007, CLIN MANUAL TREATMEN
NR 1
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1475-357X
J9 CHILD ADOL MENT H-UK
JI Child Adolesc. Ment. Health
PD SEP
PY 2008
VL 13
IS 3
BP 154
EP 155
DI 10.1111/j.1475-3588.2008.00500_6.x
PG 2
WC Psychology, Clinical; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA V16FA
UT WOS:000207854300014
ER
PT J
AU Akhondzadeh, S
Tajdar, H
Mohammadi, MR
Mohammadi, M
Nouroozinejad, GH
Shabstari, OL
Ghelichnia, HA
AF Akhondzadeh, Shahin
Tajdar, Hamid
Mohammadi, Mohammad-Reza
Mohammadi, Mohammad
Nouroozinejad, Gholam-Hossein
Shabstari, Omid L.
Ghelichnia, Hossein-Ali
TI A double-blind placebo controlled trial of piracetam added to
risperidone in patients with autistic disorder
SO CHILD PSYCHIATRY & HUMAN DEVELOPMENT
LA English
DT Article
DE AMPA; autism; glutamate; piracetam
ID GLUTAMATE HYPOTHESIS; SPECTRUM DISORDERS; RATING-SCALE; SCHIZOPHRENIA;
CHILDREN
AB It has been reported that autism is a hypoglutamatergic disorder. Therefore, it was of interest to assess the efficacy of piracetam, a positive modulator of AMPA-sensitive glutamate receptors in autistic disorder. About 40 children between the ages three and 11 years (inclusive) with a DSM IV clinical diagnosis of autism and who were outpatients from a specialty clinic for children were recruited. The children presented with a chief complaint of severely disruptive symptoms related to autistic disorder. Patients were randomly allocated to piracetam + risperidone (Group A) or placebo + risperidone (Group B) for a 10-week, double-blind, placebo-controlled study. The dose of risperidone was titrated up to 2 mg/day for children between 10 and 40 kg and 3 mg/day for children weighting above 40 kg. The dose of piracetam was titrated up to 800 mg/day. Patients were assessed at baseline and after 2, 4, 6, 8 and 10 weeks of starting medication. The measure of the outcome was the Aberrant Behavior Checklist-Community (ABC-C) Rating Scale (total score). The ABC-C Rating Scale scores improved with piracetam. The difference between the two protocols was significant as indicated by the effect of group, the between subjects factor (F = 5.85, d.f. = 1, P = 0.02). The changes at the endpoint compared with baseline were: -11.90 +/- 3.79 (mean +/- SD) and -5.15 +/- 3.04 for group A and B respectively. A significant difference was observed on the change in scores in the ABC-C Rating Scale in week 10 compared with baseline in the two groups (t = 6.017, d.f. = 38, P < 0.0001). The results suggest that a combination of atypical antipsychotic medications and a glutamate agent such as piracetam, might have increase synergistic effects in the treatment of autism.
C1 [Akhondzadeh, Shahin; Tajdar, Hamid; Mohammadi, Mohammad-Reza] Univ Tehran Med Sci, Roozbeh Psychiat Hosp, Psychiat Res Ctr, Tehran 13337, Iran.
[Mohammadi, Mohammad] Azad Univ, Fac Med, Tehran, Iran.
[Nouroozinejad, Gholam-Hossein] Jondi Shapour Univ Med Sci, Dept Psychiat, Ahwaz, Iran.
[Shabstari, Omid L.] Avesina Res Inst, Reprod Biol Biotechnol & Infertil Res Ctr, Tehran, Iran.
[Ghelichnia, Hossein-Ali] Univ Tehran Med Sci, Fac Med, Dept Neurol, Tehran, Iran.
RP Akhondzadeh, S (reprint author), Univ Tehran Med Sci, Roozbeh Psychiat Hosp, Psychiat Res Ctr, S Kargar St, Tehran 13337, Iran.
EM s.akhond@neda.net
CR Akhondzadeh S, 1998, J CLIN PHARM THER, V23, P243
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*WORLD MED ASS DEC, 2000, ETH PRINC MED RES IN
NR 28
TC 20
Z9 20
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0009-398X
J9 CHILD PSYCHIAT HUM D
JI Child Psychiat. Hum. Dev.
PD SEP
PY 2008
VL 39
IS 3
BP 237
EP 245
DI 10.1007/s10578-007-0084-3
PG 9
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 312WD
UT WOS:000256702200002
PM 17929164
ER
PT J
AU Meguid, NA
Atta, HM
Gouda, AS
Khalil, RO
AF Meguid, Nagwa A.
Atta, Hazem M.
Gouda, Amr S.
Khalil, Rehab O.
TI Role of polyunsaturated fatty acids in the management of Egyptian
children with autism
SO CLINICAL BIOCHEMISTRY
LA English
DT Article
DE autism; polyunsaturated fatty acids; fish oil; Efalex (R); tandem mass
spectrometry
ID DISORDERS; SCHIZOPHRENIA
AB Objective: Estimation of free polyunsaturated fatty acids (PUFAs) in blood and evaluation of behavior of autistic children before and after taking fish oil (Efalex (R))) were performed.
Design and methods: 30 autistic children (18 males and 12 females) aged 3-11 years and 30 healthy children as control group were included in this study. Tandem mass spectrometry and CARS were used to estimate the free PUFAs from dried blood spot and to evaluate the autistic behavior respectively.
Results: Before taking Efalex (R), linolenic acid showed a significant reduction (71%), followed by docosahexaenoic acid (65%) and arachidonic acid (45%), while linoleic acid was the least affected PUFA (32%). After taking Efalex (R), 66% of autistic children showed clinical and biochemical improvement, linolenic acid and docosahexaenoic acid showed the highest levels after Efalex,11 supplementation.
Conclusion: PUFA supplementation may play an important role in ameliorating the autistic behavior. (C) 2008 The Canadian Society of Clinical Chemists. Published by Elsevier Inc. All rights reserved.
C1 [Atta, Hazem M.] Cairo Univ, Fac Med, Dept Med Biochem, Unit Biochem & Mol Biol, Cairo 11562, Egypt.
[Meguid, Nagwa A.; Khalil, Rehab O.] Natl Res Ctr, Dept Res Children Special Needs, Cairo, Egypt.
[Gouda, Amr S.] Natl Res Ctr, Dept Biochem Genet, Cairo, Egypt.
RP Atta, HM (reprint author), Cairo Univ, Fac Med, Dept Med Biochem, Unit Biochem & Mol Biol, Cairo 11562, Egypt.
EM hazemmahmoudatta@yahoo.ie
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NR 23
TC 27
Z9 28
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0009-9120
J9 CLIN BIOCHEM
JI Clin. Biochem.
PD SEP
PY 2008
VL 41
IS 13
BP 1044
EP 1048
DI 10.1016/j.clinbiochem.2008.05.013
PG 5
WC Medical Laboratory Technology
SC Medical Laboratory Technology
GA 342WO
UT WOS:000258814500004
PM 18582451
ER
PT J
AU Nowak, C
Heinrichs, N
AF Nowak, Christoph
Heinrichs, Nina
TI A comprehensive meta-analysis of triple p-positive parenting program
using hierarchical linear modeling: Effectiveness and moderating
variables
SO CLINICAL CHILD AND FAMILY PSYCHOLOGY REVIEW
LA English
DT Review
DE parenting; review; behavioral family intervention; moderator; child
behavior
ID BEHAVIORAL FAMILY INTERVENTION; RANDOMIZED CONTROLLED-TRIALS; ONSET
CONDUCT PROBLEMS; DEVELOPMENTAL-DISABILITIES;
PHARMACOLOGICAL-TREATMENTS; METHODOLOGICAL QUALITY; EXTERNALIZING
BEHAVIOR; DISRUPTIVE BEHAVIOR; MARITAL ADJUSTMENT; AUTISM-SPECTRUM
AB A meta-analysis encompassing all studies evaluating the impact of the Triple P-Positive Parenting Program on parent and child outcome measures was conducted in an effort to identify variables that moderate the program's effectiveness. Hierarchical linear models (HLM) with three levels of data were employed to analyze effect sizes. The results (N = 55 studies) indicate that Triple P causes positive changes in parenting skills, child problem behavior and parental well-being in the small to moderate range, varying as a function of the intensity of the intervention. The most salient findings of variables moderating the interventions' impact were larger effects found on parent report as compared to observational measures and more improvement associated with more intensive formats and initially more distressed families. Sample characteristics (e.g., child's age, being a boy) and methodological features (e.g., study quality) exhibited different degrees of predictive power. The analysis clearly identified several strengths of the Triple P system, most importantly its ability to effect meaningful improvement in parents and children. Some limitations pertain to the small evidence-base of certain formats of Triple P and the lack of follow-up data beyond 3 years after the intervention. Many of the present findings may be relevant to other evidence-based parenting programs.
C1 [Nowak, Christoph] Tech Univ Carolo Wilhelmina Braunschweig, Inst Psychol, D-38106 Braunschweig, Germany.
[Heinrichs, Nina] Univ Bielefeld, Fac Psychol & Sports Sci, Dept Psychol, D-33501 Bielefeld, Germany.
RP Nowak, C (reprint author), Tech Univ Carolo Wilhelmina Braunschweig, Inst Psychol, Humboldtstr 33, D-38106 Braunschweig, Germany.
EM nowakchr@googlemail.com; nina.heinrichs@uni-bielefeld.de
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NR 166
TC 141
Z9 141
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1096-4037
J9 CLIN CHILD FAM PSYCH
JI Clin. Child Fam. Psychol. Rev.
PD SEP
PY 2008
VL 11
IS 3
BP 114
EP 144
DI 10.1007/s10567-008-0033-0
PG 31
WC Psychology, Clinical
SC Psychology
GA 343DA
UT WOS:000258831600002
PM 18509758
ER
PT J
AU Magnee, MJCM
de Gelder, B
van Engeland, H
Kemner, C
AF Magnee, Maurice J. C. M.
de Gelder, Beatrice
van Engeland, Herman
Kemner, Chantal
TI A typical processing of fearful face-voice pairs in Pervasive
Developmental Disorder: An ERP study
SO CLINICAL NEUROPHYSIOLOGY
LA English
DT Article
DE autism; facial expression; multisensory processing; voice prosody; EEG
ID TIME-COURSE; EMOTION RECOGNITION; BRAIN RESPONSES; VISUAL-CORTEX;
AUTISM; PERCEPTION; EXPRESSIONS; BINDING; HUMANS; INFORMATION
AB Objective: An important premise for successful social-affective communication is rapid perception of visual and auditory emotional cues, as well as their multisensory integration (MSI). We investigated to what extent a deficit in recognition of emotions in individuals with Pervasive Developmental Disorder (PDD) may have its roots in abnormal MSI of emotional cues provided by the sight of a facial expression and an emotional tone of voice.
Methods: In twelve high-functioning, adult PDD individuals and thirteen age- and IQ-matched controls, (1) the processing of fearful faces was compared with that of happy faces; (2) MSI was assessed by characterizing the interaction effects of crossmodal presentation, using EEG.
Results: Increased PI and N170 amplitudes were seen in response to fearful faces compared with happy faces in both groups. However, PDD individuals differed from healthy controls in NISI of fearful information from visual and auditory cues.
Conclusions: Both groups show a similar pattern as concerns the early components of visual emotion processing, but there are anomalies in processing of fearful face-voice combinations in the PDD group. Significance: Because of the importance of rapid MSI for social competence, MSI anomalies in PDD may be linked to the observed deficits in their emotional behavior. (c) 2008 International Federation of Clinical Neurophysiology. Published by Elsevier Ireland Ltd. All rights reserved.
C1 [Magnee, Maurice J. C. M.; van Engeland, Herman; Kemner, Chantal] Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, NL-3584 CX Utrecht, Netherlands.
[Magnee, Maurice J. C. M.; de Gelder, Beatrice] Tilburg Univ, Lab Cognit & Affect Neurosci, NL-5000 LE Tilburg, Netherlands.
[Kemner, Chantal] Maastricht Univ, Fac Psychol, Sect Biol Dev Psychol, NL-6200 MD Maastricht, Netherlands.
[de Gelder, Beatrice] Massachusetts Gen Hosp, Martinos Ctr Biomed Imaging, Charlestown, MA 02129 USA.
[de Gelder, Beatrice] Harvard Univ, Sch Med, Charlestown, MA 02129 USA.
RP Magnee, MJCM (reprint author), Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, B01-201,Heidelberglaan 100, NL-3584 CX Utrecht, Netherlands.
EM M.J.C.M.Magnec@umcutrecht.nl
FU Netherlands Organization for Scientific Research (NWO) [402-01-094]
FX The work described was supported by an Innovational Research Incentives
Grant (VIDI-scheme, 402-01-094) of the Netherlands Organization for
Scientific Research (NWO) to Chantal Kemner.
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NR 34
TC 10
Z9 12
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 1388-2457
J9 CLIN NEUROPHYSIOL
JI Clin. Neurophysiol.
PD SEP
PY 2008
VL 119
IS 9
BP 2004
EP 2010
DI 10.1016/j.clinph.2008.05.005
PG 7
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 342QS
UT WOS:000258799200011
ER
PT J
AU Davidoff, J
Fonteneau, E
Fagot, J
AF Davidoff, Jules
Fonteneau, Elisabeth
Fagot, Joel
TI Local and global processing: Observations from a remote culture
SO COGNITION
LA English
DT Article
DE Local/global processing; Faces; Navon; Cross-culture
ID WEAK CENTRAL COHERENCE; UPSIDE-DOWN FACES; HEMISPHERIC-SPECIALIZATION;
HIERARCHICAL PATTERNS; CHILDRENS ANALYSIS; VISUAL-PERCEPTION; INVERTED
FACES; HOMO-SAPIENS; AUTISM; PROSOPAGNOSIA
AB In Experiment 1, a normal adult population drawn from a remote culture (Himba) in northern Namibia made similarity matches to [Navon, D. (1977). Forest before trees: The precedence of global features in visual perception. Cognitive Psychology, 9, 353-383] hierarchical figures. The Himba showed a local bias stronger than that has been previously observed in any other non-clinical human population. However, in Experiment 2, their recognition of normal or distorted ("Thatcherized") faces did not appear to have been affected by their attention to detail as has been suggested for autistic populations. The data are consistent with a cultural/experiential origin for population differences in local processing and suggest that attention to the local and global properties of stimuli may differ for hierarchical figures and faces. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Davidoff, Jules; Fonteneau, Elisabeth] Univ London Goldsmiths Coll, Dept Psychol, Ctr Cognit Computat & Culture, London SW14 6NW, England.
[Fagot, Joel] Mediterranean Inst Cognit Neurosci, CNRS, Marseille, France.
[Fagot, Joel] Univ Aix Marseille 2, Marseille, France.
RP Davidoff, J (reprint author), Univ London Goldsmiths Coll, Dept Psychol, Ctr Cognit Computat & Culture, Lewisham Way, London SW14 6NW, England.
EM j.davidoff@gold.ac.uk
RI fagot, joel/H-6412-2011; van wolputte, steven/I-3930-2014
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NR 66
TC 24
Z9 24
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0010-0277
J9 COGNITION
JI Cognition
PD SEP
PY 2008
VL 108
IS 3
BP 702
EP 709
DI 10.1016/j.cognition.2008.06.004
PG 8
WC Psychology, Experimental
SC Psychology
GA 357ND
UT WOS:000259851600010
PM 18662813
ER
PT J
AU Brock, J
Norbury, C
Einav, S
Nation, K
AF Brock, Jon
Norbury, Courtenay
Einav, Shiri
Nation, Kate
TI Do individuals with autism process words in context? Evidence from
language-mediated eye-movements
SO COGNITION
LA English
DT Article
DE Autism; Central coherence; Eye-movements; Language impairment; Semantic
processing
ID CENTRAL COHERENCE; SPOKEN-LANGUAGE; SPECTRUM DISORDERS;
SPEECH-PERCEPTION; CHILDREN; COMPREHENSION; COMMUNICATION; INFORMATION;
INTEGRATION; IMPAIRMENT
AB it is widely argued that people with autism have difficulty processing ambiguous linguistic information in context. To investigate this claim, we recorded the eye-movements of 24 adolescents with autism spectrum disorder and 24 language-matched peers as they monitored spoken sentences for words corresponding to objects on a computer display, Following a target word, participants looked more at a competitor object sharing the same onset than at phonologically unrelated objects. This effect was, however, mediated by the sentence context such that participants looked less at the phonological competitor if it was semantically incongruous with the preceding verb. Contrary to predictions, the two groups evidenced similar effects of context on eye-movements. Instead, across both groups, the effect of sentence context was reduced in individuals with relatively poor language skills. Implications for the weak central coherence account of autism are discussed. (c) 2008 Elsevier B.V. All rights reserved.
C1 [Brock, Jon] Macquarie Univ, Macquarie Ctr Cognit Sci, Sydney, NSW 2109, Australia.
[Brock, Jon] Univ Oxford, Dept Expt Psychol, Oxford OX1 3UD, England.
[Norbury, Courtenay] Univ London, Dept Psychol, Egham TW20 0EX, Surrey, England.
RP Brock, J (reprint author), Macquarie Univ, Macquarie Ctr Cognit Sci, Sydney, NSW 2109, Australia.
EM jbrock@maccs.mq.edu.au
RI Nation, Kate/F-8228-2014
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NR 38
TC 42
Z9 42
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0010-0277
J9 COGNITION
JI Cognition
PD SEP
PY 2008
VL 108
IS 3
BP 896
EP 904
DI 10.1016/j.cognition.2008.06.007
PG 9
WC Psychology, Experimental
SC Psychology
GA 357ND
UT WOS:000259851600032
PM 18692181
ER
PT J
AU Matsuura, H
Tateno, K
Aou, S
AF Matsuura, Hirotsune
Tateno, Katsumi
Aou, Shuji
TI Dynamical properties of the two-process model for sleep-wake cycles in
infantile autism
SO COGNITIVE NEURODYNAMICS
LA English
DT Article
DE Autism; Two-process model; Sleep cycle
ID DEPRESSION; CHILDHOOD; CHILDREN; EEG
AB The two-process model is a scheme for the timing of sleep that consists of homeostatic (Process S) and circadian (Process C) variables. The two-process model exhibits abnormal sleep patterns such as internal desynchronization or sleep fragmentation. Early infants with autism often experience sleep difficulties. Large day-by-day changes are found in the sleep onset and waking times in autistic children. Frequent night waking is a prominent property of their sleep. Further, the sleep duration of autistic children is often fragmented. These sleep patterns in infants with autism are not fully understood yet. In the present study, the sleep patterns in autistic children were reproduced by a modified two-process model using nonlinear analysis. A nap term was introduced into the original two-process model to reproduce the sleep patterns in early infants. The nap term and the time course of Process S are mentioned in the present study. Those parameters led to bifurcation of the sleep-wake cycle in the modified two-process model. In a certain range of these parameter sets, a small external noise was amplified, and an irregular sleep-wake cycle appeared. The short duration of sleep led to another irregular sleep onset or waking. Consequently, an irregular sleep-wake cycle appeared in early infantile autism.
C1 [Matsuura, Hirotsune; Tateno, Katsumi; Aou, Shuji] Kyushu Inst Technol, Dept Brain Sci & Engn, Grad Sch Life Sci & Syst Engn, Wakamatsu Ku, Kitakyushu, Fukuoka 8080196, Japan.
RP Tateno, K (reprint author), Kyushu Inst Technol, Dept Brain Sci & Engn, Grad Sch Life Sci & Syst Engn, Wakamatsu Ku, 2-4 Hibikino, Kitakyushu, Fukuoka 8080196, Japan.
EM tateno@brain.kyutech.ac.jp
FU 21th Century COE Program in the Kyushu Institute of Technology
FX The authors thank Dr. Kunio Inanuma for support and discussion about his
data. This work was supported by the 21th Century COE Program in the
Kyushu Institute of Technology entitled "World of brain computing
interwoven out of animals and robots".
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NR 25
TC 7
Z9 8
PU SPRINGER
PI DORDRECHT
PA VAN GODEWIJCKSTRAAT 30, 3311 GZ DORDRECHT, NETHERLANDS
SN 1871-4080
J9 COGN NEURODYNAMICS
JI Cogn. Neurodynamics
PD SEP
PY 2008
VL 2
IS 3
BP 221
EP 228
DI 10.1007/s11571-008-9051-3
PG 8
WC Neurosciences
SC Neurosciences & Neurology
GA 450PJ
UT WOS:000266413000005
PM 19003487
ER
PT J
AU Jones, SR
de-Wit, L
Fernyhough, C
Meins, E
AF Jones, Simon R.
de-Wit, Lee
Fernyhough, Charles
Meins, Elizabeth
TI A new spin on the Wheel of Fortune: Priming of action-authorship
judgements and relation to psychosis-like experiences
SO CONSCIOUSNESS AND COGNITION
LA English
DT Article
DE agency; conscious will; delusions of control; forward model;
hallucinations; passivity
ID SELF-EFFICACY; SCHIZOPHRENIA; RELIABILITY; INFORMATION; ASCRIPTION;
AWARENESS; SYMPTOMS; IDEATION; ABILITY; AUTISM
AB The proposal that there is an illusion of conscious will has been supported by findings that priming of stimulus location in a task requiring judgements of action-authorship can enhance participants' experience of agency. We attempted to replicate findings from the 'Wheel of Fortune' task [Aarts, H., Custers, R., & Wegner, D. M. (2005). On the inference of personal authorship: enhancing experienced agency by priming effect information. Consciousness and Cognition, 14, 439-458]. We also examined participants' performance on this task in relation to self-reported passivity experiences and hallucination-proneness. We found a significant effect of priming, with primes being found to increase the experience of agency. An interaction between gender and priming was also found, with priming enhancing feelings of agency in women but not in men. There were no significant correlations between levels of self-reported passivity experiences or hallucination-proneness and participants' susceptibility to the priming effect on ratings of agency. Implications of these findings are discussed with regard to a prominent model of passivity experiences. (C) 2007 Elsevier Inc. All rights reserved.
C1 [Jones, Simon R.; de-Wit, Lee; Fernyhough, Charles; Meins, Elizabeth] Univ Durham, Dept Psychol, Durham DH1 3LE, England.
RP Jones, SR (reprint author), Univ Durham, Dept Psychol, South Rd, Durham DH1 3LE, England.
EM s.r.jones@durham.ac.uk
RI Fernyhough, Charles/A-1057-2010
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NR 36
TC 11
Z9 11
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1053-8100
J9 CONSCIOUS COGN
JI Conscious. Cogn.
PD SEP
PY 2008
VL 17
IS 3
BP 576
EP 586
DI 10.1016/j.concog.2007.08.008
PG 11
WC Psychology, Experimental
SC Psychology
GA 334ZD
UT WOS:000258259500003
PM 17949998
ER
PT J
AU Zaja, RH
Rojahn, J
AF Zaja, Rebecca H.
Rojahn, Johannes
TI Facial emotion recognition in intellectual disabilities
SO CURRENT OPINION IN PSYCHIATRY
LA English
DT Article
DE Down syndrome; emotion recognition; facial processing; intellectual
disability; Williams syndrome
ID MENTAL-RETARDATION; WILLIAMS-SYNDROME; EXPRESSIONS; CHILDREN; PEOPLE;
INDIVIDUALS; MILD
AB Purpose of review
Interpreting facial emotion is a requisite skill that enables us to navigate our social environment. Autism spectrum disorder is characterized by shortcomings in socio-cognitive abilities in general, and in emotion recognition in particular, and much has been written on this subject. Less research, however, has been conducted on individuals with intellectual disabilities. This review discusses recent emotion recognition research in this population.
Recent findings
Facial emotion recognition research in individuals with intellectual disabilities can be divided into two broad categories: studies on the causes of emotion recognition deficits (i.e. primary deficits or secondary phenomena) and studies on the effects of emotion recognition deficits (behavioral implications). Recent research on causes has not yet produced definitive conclusions and current research on specific effects has been limited to aggression and self-reported anger.
Summary
Some evidence exists that individuals with intellectual disability of heterogeneous etiology (excluding autism) have facial affect recognition deficits that cannot be fully accounted for by cognitive-intellectual abilities. In addition, cognitive processing strategies and genetic syndrome-specific differences in facial affect recognition have been discovered but further research is needed. We found no evidence that emotion recognition deficits contribute to the emergence of later antisocial behavior.
C1 [Zaja, Rebecca H.; Rojahn, Johannes] George Mason Univ, Fairfax, VA 22030 USA.
RP Rojahn, J (reprint author), George Mason Univ, 10340 Democracy Lane,Ste 202, Fairfax, VA 22030 USA.
EM jrojahn@gmu.edu
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NR 14
TC 7
Z9 7
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0951-7367
J9 CURR OPIN PSYCHIATR
JI Curr. Opin. Psychiatr.
PD SEP
PY 2008
VL 21
IS 5
BP 441
EP 444
PG 4
WC Psychiatry
SC Psychiatry
GA 336MP
UT WOS:000258368300003
PM 18650683
ER
PT J
AU Decety, J
Meyer, M
AF Decety, Jean
Meyer, Meghan
TI From emotion resonance to empathic understanding: A social developmental
neuroscience account
SO DEVELOPMENT AND PSYCHOPATHOLOGY
LA English
DT Review
ID AUTISM SPECTRUM DISORDERS; MIRROR-NEURON SYSTEM; TEMPORO-PARIETAL
JUNCTION; CEREBRAL-BLOOD-FLOW; COGNITIVE NEUROSCIENCE;
INDIVIDUAL-DIFFERENCES; PERSPECTIVE-TAKING; FACIAL EXPRESSIONS;
EXECUTIVE FUNCTION; BEHAVIOR PROBLEMS
AB The psychological construct of empathy refers to an intersubjective induction process by which positive and negative emotions are shared, without losing sight of whose feelings belong to whom. Empathy can lead to personal distress or to empathic concern (sympathy). The goal of this paper is to address the underlying cognitive processes and their neural underpinnings that constitute empathy within a developmental neuroscience perspective. In addition, we focus on how these processes go awry in developmental disorders marked by impairments in social cognition, such as autism spectrum disorder, and conduct disorder. We argue that empathy involves both bottom-up and top-down information processing, underpinned by specific and interacting neural systems. We discuss data from developmental psychology as well as cognitive neuroscience in support of such a model, and highlight the impact of neural dysfunctions on social cognitive developmental behaviour. Altogether, bridging developmental science and cognitive neuroscience helps approach a more complete understanding of social cognition. Synthesizing these two domains also contributes to a better characterization of developmental psychopathologies that impacts the development of effective treatment strategies.
C1 [Decety, Jean] Univ Chicago, Dept Psychol, Chicago, IL 60637 USA.
[Meyer, Meghan] Stanford Univ, Sch Med, Stanford, CA 94305 USA.
RP Decety, J (reprint author), Univ Chicago, Dept Psychol, 5848 S Univ Ave, Chicago, IL 60637 USA.
EM decety@uchicago.edu
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NR 201
TC 106
Z9 107
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0954-5794
EI 1469-2198
J9 DEV PSYCHOPATHOL
JI Dev. Psychopathol.
PD FAL
PY 2008
VL 20
IS 4
SI SI
BP 1053
EP 1080
DI 10.1017/S0954579408000503
PG 28
WC Psychology, Developmental
SC Psychology
GA 373SU
UT WOS:000260993800003
PM 18838031
ER
PT J
AU Pelphrey, KA
Carter, EJ
AF Pelphrey, Kevin A.
Carter, Elizabeth J.
TI Charting the typical and atypical development of the social brain
SO DEVELOPMENT AND PSYCHOPATHOLOGY
LA English
DT Review
ID AUTISM SPECTRUM DISORDER; SUPERIOR TEMPORAL SULCUS; HIGH-FUNCTIONING
AUTISM; CEREBRAL-BLOOD-FLOW; HUMAN VISUAL-CORTEX; BIOLOGICAL-MOTION;
JOINT ATTENTION; FACIAL EXPRESSIONS; HUMAN AMYGDALA; CORTICAL ACTIVATION
AB We describe recent progress in our program of research that aims to use functional magnetic resonance imaging (fMRI) to identify and delineate the brain systems involved in social perception and to chart the development of those systems and their roles as mechanisms supporting the development of social cognition in children, adolescents, and adults with and without autism. This research program was initiated with the intention of further specifying the role of the posterior superior temporal sulcus (STS) region in the network of neuroanatomical structures comprising the social brain. Initially, this work focused on evaluating STS function when typically developing adults were engaged in the visual analysis of other people's actions and intentions. We concluded that that the STS region plays all important role in social perception via its involvement in representing and predicting the actions and social intentions of other people from an analysis of biological-motion cues. These studies of typically developing people a set of core findings and a methodologicalapproach that informed a set of fMRI studies of social perception dysfunction in autism. The work has established that dysfunction in the STS region, as well as reduced connectivity between this region and other social brain structures including the fusiform gyrus and amygdala, play a role in the pathophysiology of social perception derficits in autism. Most recently, this research program has incorporated a developmental perspective in beginning to chart the development of the STS region in children with and without autism.
C1 [Pelphrey, Kevin A.] Carnegie Mellon Univ, Dept Psychol, Pittsburgh, PA 15213 USA.
RP Pelphrey, KA (reprint author), Carnegie Mellon Univ, Dept Psychol, Baker Hall 342C,5000 Forbes Ave, Pittsburgh, PA 15213 USA.
EM kpelphrey@emu.edu
RI Carter, Elizabeth/G-6958-2012
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NR 117
TC 52
Z9 55
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0954-5794
J9 DEV PSYCHOPATHOL
JI Dev. Psychopathol.
PD FAL
PY 2008
VL 20
IS 4
BP 1081
EP 1102
DI 10.1017/S0954579408000515
PG 22
WC Psychology, Developmental
SC Psychology
GA 373SU
UT WOS:000260993800004
PM 18838032
ER
PT J
AU O'Hearn, K
Asato, M
Ordaz, S
Luna, B
AF O'Hearn, Kirsten
Asato, Miya
Ordaz, Sarah
Luna, Beatriz
TI Neurodevelopment and executive function in autism
SO DEVELOPMENT AND PSYCHOPATHOLOGY
LA English
DT Review
ID SPATIAL WORKING-MEMORY; OBSESSIVE-COMPULSIVE DISORDER; DEFICIT
HYPERACTIVITY DISORDER; POSITRON-EMISSION-TOMOGRAPHY; WHITE-MATTER
DEVELOPMENT; SACCADIC EYE-MOVEMENTS; DEVELOPMENTAL CEREBELLAR
ABNORMALITY; ANTISACCADE TASK-PERFORMANCE; RESONANCE-IMAGING EVIDENCE;
HUMAN PREFRONTAL CORTEX
AB Autisms is a neurodevelopmental disorder characterized by social and communication deficits, and repititive behavior. Studies investigating the integrity of brain systems in autism suggest a wide range of gray and whited matter abnormalities that are present early in life and change with development. These abnormalities predominantly affect association areas and undermine functional integration. Executive function, which has a protracted developement into adolescence and reflects the integration of complex widely distributed brain function, is also affected in autism. Evidence from studies probing response inhibition and working memory indicate impairments in these core components of executive function, as well as compensatory mechanisms that permit normative function in autism. Studies also demonstrate age-related improvements in executive function from childhood to adolescence in autism, indicating the presence of plasticity and suggesting a prolonged window for effective treatment. Despite developmental gains, mature executive funcitoning is limited in autism, reflecting abnormalities abnormalities in wide-spread brain networks that may lead to impaired processing of complex information across all domains.
C1 [O'Hearn, Kirsten; Asato, Miya; Ordaz, Sarah; Luna, Beatriz] Univ Pittsburgh, Pittsburgh, PA 15213 USA.
RP O'Hearn, K (reprint author), Univ Pittsburgh, OHearn Loeffler Bldg,Room 112,121 Meyran Ave, Pittsburgh, PA 15213 USA.
EM ohearn@upmc.edu
RI Luna, Beatriz/F-1201-2010
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NR 262
TC 56
Z9 56
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0954-5794
J9 DEV PSYCHOPATHOL
JI Dev. Psychopathol.
PD FAL
PY 2008
VL 20
IS 4
BP 1103
EP 1132
DI 10.1017/S0954579408000527
PG 30
WC Psychology, Developmental
SC Psychology
GA 373SU
UT WOS:000260993800005
PM 18838033
ER
PT J
AU Monk, CS
AF Monk, Christopher S.
TI The development of emotion-related neural circuitry in health and
psychopathology
SO DEVELOPMENT AND PSYCHOPATHOLOGY
LA English
DT Review
ID MAJOR DEPRESSIVE DISORDER; GENERALIZED ANXIETY DISORDER; MEDIAL
PREFRONTAL CORTEX; FEAR-POTENTIATED STARTLE; TREATMENT-RESISTANT
DEPRESSION; CEREBRAL BLOOD-FLOW; FACIAL EXPRESSIONS;
BEHAVIORAL-INHIBITION; INCREASED AMYGDALA; ORBITOFRONTAL CORTEX
AB Disturbances in the detection of response to, and interpretation of emotion are common in many forms of psychopathology, The amygdala, striatum, and structures within the prefrontal cortex and highly involved in mediating these stages of emotion processing, and evidence indicates that these regions show structural and functional alterations in different types of psychopathology, including anxiety, depression, and autism spectrum disorders. However, we do not know how genes and the environment interact to alter development of these brain regions in ways that give rise to emotion-related psychopathology, This review discusses the current understanding of brain regions that are involved in emotional funcitoning, how they develop, and how they are altered in three forms of psychopathology: anxiety, depression, and autism spectrum disorders. Following this, a framework is described that may facilitate the integration of investigations of genetic variation and brain function with symptom and diagnostic measures. The framework involves three components: (a) a greater emphasis on simultaneously analyzing multiple levels (genes, brain function, behavior, symptoms, and diagnoses); (b) further integration of developmental considerations, including timing of environmental events, adaptations (or maladaptations). and disorder-related trajectories that guide some children toward atypical experiences; and (c) greater cross-talk between animal and human investigators to take advantage of biological measures that cannot be acquired in humans.
C1 Univ Michigan, Dept Psychol, Ann Arbor, MI 48109 USA.
RP Monk, CS (reprint author), Univ Michigan, Dept Psychol, 2000 E Hall,530 Church St, Ann Arbor, MI 48109 USA.
EM esmonk@umich.edu
RI Monk, Christopher/J-1805-2014
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 156
TC 55
Z9 55
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0954-5794
J9 DEV PSYCHOPATHOL
JI Dev. Psychopathol.
PD FAL
PY 2008
VL 20
IS 4
BP 1231
EP 1250
DI 10.1017/S095457940800059X
PG 20
WC Psychology, Developmental
SC Psychology
GA 373SU
UT WOS:000260993800012
PM 18838040
ER
PT J
AU Yeargin-Allsopp, M
AF Yeargin-Allsopp, Marshalyn
TI The prevalence and characteristics of autism spectrum disorders in the
ALSPAC cohort
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Editorial Material
ID CHILDREN
C1 Ctr Dis Control & Prevent, Atlanta, GA USA.
RP Yeargin-Allsopp, M (reprint author), Ctr Dis Control & Prevent, Atlanta, GA USA.
CR *CDCP, 2007, MMWR 0209, P56
Fombonne E, 2003, J AUTISM DEV DISORD, V33, P365, DOI 10.1023/A:1025054610557
Golding J, 2001, PAEDIATR PERINAT EP, V15, P74
Posserud MB, 2006, J CHILD PSYCHOL PSYC, V47, P167, DOI 10.1111/j.1469-7610.2005.01462.x
NR 4
TC 1
Z9 1
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD SEP
PY 2008
VL 50
IS 9
BP 646
EP 646
PG 1
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 337NF
UT WOS:000258442100004
PM 18754911
ER
PT J
AU Williams, E
Thomas, K
Sidebotham, H
Emond, A
AF Williams, Emma
Thomas, Kate
Sidebotham, Helen
Emond, Alan
TI Prevalence and characteristics of autistic spectrum disorders in the
ALSPAC cohort
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Article
ID ASPERGER-SYNDROME; DIAGNOSTIC INTERVIEW; TOTAL POPULATION; CHILDREN;
EPIDEMIOLOGY
AB The aim of this study was to determine the prevalence of autistic spectrum disorder (ASD) within a large representative population sample: the Avon Longitudinal Study of Parents and Children (ALSPAC). Cases of ASD were identified from the clinical notes of children in the ALSPAC with a suspected developmental disorder and from the Pupil Level Annual Schools Census (PLASC) for England in 2003. Seventy-one cases of ASD diagnosed after a multidisciplinary assessment were identified from health records. There were an additional 15 cases from PLASC data in which ASD was mentioned as a principal difficulty, thus giving a total of 86 children diagnosed by the age of 11 years. Prevalence of ASD per 10 000 population at 11 years was 51.1 for those with a multi-professional diagnosis, and 61.9 if cases from education were included, made up of 21.6 for childhood autism, 10.8 for atypical autism, 16.6 for Asperger syndrome, and 13.0 for unspecified ASD. The male:female ratio was 6.8:1. Median age at diagnosis ranged from 45 months in childhood autism to 116 months in Asperger syndrome. A comorbid developmental disorder was recorded in 33.8% of cases, including learning disability* in 14.7%, epilepsy in 10.3%, and mixed developmental disorder in 4.4%. We conclude that the prevalence of ASD diagnosed at 11 years in a UK representative population-based sample is at least 51.1/10 000.
C1 [Williams, Emma; Sidebotham, Helen; Emond, Alan] Univ Bristol, Ctr Child & Adolescent Hlth, Bristol BS6 6JS, Avon, England.
[Thomas, Kate] Univ Bristol, ALSPAC, Bristol BS6 6JS, Avon, England.
RP Emond, A (reprint author), Univ Bristol, Ctr Child & Adolescent Hlth, Hampton House, Bristol BS6 6JS, Avon, England.
EM alan.emond@bristol.ac.uk
FU University of Bristol; Wellcome Trust [59579]
FX We are grateful to all the families who took part in this study, the
midwives for help in recruiting them, and the whole ALSPAC team, which
includes interviewers, computer and laboratory technicians, clerical
workers, research scientists, volunteers, managers, receptionists, and
nurses. The UK Medical Research Council, the Wellcome Trust, and the
University of Bristol provide core support for ALSPAC. This study was
supported by a steering group consisting of the authors and Dr Jon
Pollock, Ms Sue Bonnell, Ms Karen Birmingham, and Professor Jean
Golding. We thank Andy Boyd and Mike Crawford for their help with data
management and linkage, and Debbie Johnson for her help with data
extraction from notes. This study was funded by the Wellcome Trust, (
grant 59579).
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 24
TC 68
Z9 70
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD SEP
PY 2008
VL 50
IS 9
BP 672
EP 677
DI 10.1111/j.1469-8749.2008.03042.x
PG 6
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 337NF
UT WOS:000258442100009
PM 18754916
ER
PT J
AU Tasker, SL
Schmidt, LA
AF Tasker, Susan L.
Schmidt, Louis A.
TI The "dual usage problem" in the explanations of "joint attention" and
children's socioemotional development: A reconceptualization
SO DEVELOPMENTAL REVIEW
LA English
DT Article
DE joint attention; mother-child interaction; early socio-emotional
development; children; toddlers
ID BLIND INFANTS; MOTHER-INFANT; DEAF; COMMUNICATION; LANGUAGE; HEARING;
AUTISM; PEOPLE; SKILL; MODEL
AB The term "joint attention", which first gained currency in the early 1960s in studies of the development of language and symbolic thought, remains significant in the developmental literature. However, its meaning is unclear. A definitional problem exists similar to what Patterson [Patterson, M. L. (1982). A sequential functional model of nonverbal exchange. Psychological Review, 89, 231-249] described as the "dual usage" problem in the study of behaviour. The dual usage problem manifests when the behaviours of interest are used interchangeably with the function served by those behaviours. Similarly, the behaviours or skills that are taken to show joint attention are used interchangeably with the functions served by these behaviours. Scant attention is given to how the behaviours are generated and regulated and how they contribute to development. The purpose of the present position paper was: (1) to identify and illustrate the ways in which the behaviours and functions of joint attention have been confounded in the literature; (2) to provide a revised operationalization of joint attention incorporating the idea of Consummative Joint Attention, which is defined as a process variable that integrates these two theoretically different and complementary aspects of "joint attention;" and (3) to test this revised definition with evidence from a sample of hearing mothers and their 18- to 36-month-old hearing or deaf toddlers. We suggest that a reconceptualization and revised definition of joint attention as a process served by a particular sequence of complementary and well-timed events in the behaviour of mother-child interaction are particularly well-suited to testing joint attention as an early prelingual mechanism and to detecting early problems with psychosocial and adaptive development created by ineffective patterns of social interaction. (c) 2007 Elsevier Inc. All rights reserved.
C1 [Tasker, Susan L.; Schmidt, Louis A.] McMaster Univ, Dept Psychol Neurosci & Behav, Hamilton, ON L8S 4K1, Canada.
RP Schmidt, LA (reprint author), McMaster Univ, Dept Psychol Neurosci & Behav, Hamilton, ON L8S 4K1, Canada.
EM schmidtl@mcmaster.ca
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NR 74
TC 14
Z9 14
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0273-2297
J9 DEV REV
JI Dev. Rev.
PD SEP
PY 2008
VL 28
IS 3
BP 263
EP 288
DI 10.1016/j.dr.2007.07.001
PG 26
WC Psychology, Developmental
SC Psychology
GA 351NG
UT WOS:000259430400001
ER
PT J
AU Begeer, S
Koot, HM
Rieffe, C
Terwogt, MM
Stegge, H
AF Begeer, Sander
Koot, Hans M.
Rieffe, Carolien
Terwogt, Mark Meerum
Stegge, Hedy
TI Emotional competence in children with autism: Diagnostic criteria and
empirical evidence
SO DEVELOPMENTAL REVIEW
LA English
DT Review
DE autism; mental retardation; intelligence; emotion; emotional
development; theory of mind; diagnostic assessment; DSM
ID HIGH-FUNCTIONING CHILDREN; PERVASIVE DEVELOPMENTAL DISORDERS;
FACE-PROCESSING DEFICIT; DOWN-SYNDROME; SPECTRUM DISORDER;
ASPERGER-SYNDROME; FACIAL EXPRESSIONS; YOUNG-CHILDREN; JOINT ATTENTION;
MENTAL-RETARDATION
AB The diagnostic criteria of autism spectrum disorders (ASD) include emotional impairments. However, scientific evidence for these impairments is varied and subtle. In this contribution, recent empirical studies that examined the emotional competence in children and adolescents with ASD are reviewed. Four aspects of emotional competence that are important to children's daily social functioning (expression, perception, responding, and understanding) are discussed, differentiating between mentally retarded and normally intelligent children and adolescents with and without ASD in natural and structured contexts. On various accounts, the emotional impairments of children with ASD that are found in scientific studies provide a more differentiated view on the impairments suggested by the diagnostic literature. Consistent empirical findings and gaps in the field are discussed. Theoretical and clinical recommendations for assessment procedures are suggested. (c) 2007 Elsevier Inc. All rights reserved.
C1 [Begeer, Sander; Koot, Hans M.; Terwogt, Mark Meerum; Stegge, Hedy] Vrije Univ Amsterdam, Dept Dev Psychol, Fac Psychol & Educ, NL-1081 BT Amsterdam, Netherlands.
[Rieffe, Carolien] Leiden Univ, Fac Social & Behav Sci, Leiden, Netherlands.
RP Begeer, S (reprint author), Vrije Univ Amsterdam, Dept Dev Psychol, Fac Psychol & Educ, Van der Boechorststr 1, NL-1081 BT Amsterdam, Netherlands.
EM S.Begeer@psy.vu.nl
RI Koot, Hans/D-9466-2012; Begeer, Sander/I-3383-2012
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NR 217
TC 39
Z9 40
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0273-2297
J9 DEV REV
JI Dev. Rev.
PD SEP
PY 2008
VL 28
IS 3
BP 342
EP 369
DI 10.1016/j.dr.2007.09.001
PG 28
WC Psychology, Developmental
SC Psychology
GA 351NG
UT WOS:000259430400004
ER
PT J
AU Shinawi, M
Cheung, SW
AF Shinawi, Marwan
Cheung, Sau Wai
TI The array CGH and its clinical applications
SO DRUG DISCOVERY TODAY
LA English
DT Review
ID COMPARATIVE GENOMIC HYBRIDIZATION; MULTIPLE CONGENITAL-ANOMALIES;
IDIOPATHIC MENTAL-RETARDATION; HIGH-RESOLUTION ANALYSIS; FOCAL DERMAL
HYPOPLASIA; COPY NUMBER; MICRODELETION SYNDROME; CHROMOSOMAL
REARRANGEMENTS; CYTOGENETIC DIAGNOSIS; MICROARRAY ANALYSIS
AB Array comparative genomic hybridization (aCGH) is a technique enabling high-resolution, genome-wide screening of segmental genomic copy number variations (CNVs). It is becoming an essential and a routine clinical diagnostic tool and is gradually replacing cytogenetic methods. Most of the clinically available aCGH platforms are designed to detect aneuploidies, well-characterized microdeletion/ microduplication syndromes and subtelomeric or other unbalanced chromosomal rearrangements. In addition, aCGH can uncover numerous CNVs of unclear significance scattered throughout the human genome. But this technology is not able to identify balanced chromosomal imbalances such as translocations and inversions and some ploidies. aCGH increased the ability to detect segmental genomic CNVs in patients with global developmental delay, mental retardation, autism, multiple congenital anomalies and dysmorphism, and is becoming a powerful tool in disease gene discovery and prenatal diagnostics. This tool is also showing promising data in cancer research. and in the diagnosis, classification and prognostification of different malignancies.
C1 [Shinawi, Marwan; Cheung, Sau Wai] Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
[Cheung, Sau Wai] Baylor Coll Med, Med Genet Labs, Houston, TX USA.
RP Cheung, SW (reprint author), Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
EM scheung@bcm.edu
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NR 81
TC 80
Z9 87
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1359-6446
J9 DRUG DISCOV TODAY
JI Drug Discov. Today
PD SEP
PY 2008
VL 13
IS 17-18
BP 760
EP 770
DI 10.1016/j.drudis.2008.06.007
PG 11
WC Pharmacology & Pharmacy
SC Pharmacology & Pharmacy
GA 356PP
UT WOS:000259790400004
PM 18617013
ER
PT J
AU Banda, DR
Grimmett, E
AF Banda, Devender R.
Grimmett, Eric
TI Enhancing social and transition behaviors of persons with autism through
activity schedules: A review
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID PHOTOGRAPHIC ACTIVITY SCHEDULES; PICTURE ACTIVITY SCHEDULES;
MENTAL-RETARDATION; TASK ENGAGEMENT; CHILDREN; SKILLS; ADULTS;
MAINTENANCE; PRESCHOOL; PLAY
AB Thirteen studies were reviewed that were conducted using activity schedules with persons with autism to improve social interaction skills and decrease problem behaviors. Results across studies indicate that activity schedules enhanced social interactions and on-task and transition behaviors. Also, investigators in some studies used activity schedules to decrease students' tantrums and other problem behaviors during transitions. furthermore, researchers in several studies that reported generalization indicated that behaviors learned through activity schedules generalized across settings and persons. Implications for practitioners and for future researchers are discussed.
C1 [Banda, Devender R.] Texas Tech Univ, Coll Educ, Dept Educ Psychol & Leadership, Lubbock, TX 79416 USA.
RP Banda, DR (reprint author), Texas Tech Univ, Coll Educ, Dept Educ Psychol & Leadership, POB 41701, Lubbock, TX 79416 USA.
EM devender.banda@ttu.edu
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NR 31
TC 15
Z9 15
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD SEP
PY 2008
VL 43
IS 3
BP 324
EP 333
PG 10
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 335GA
UT WOS:000258277600004
ER
PT J
AU Whalon, K
Hanline, MF
AF Whalon, Kelly
Hanline, Mary Frances
TI Effects of a reciprocal questioning intervention on the question
generation and responding of children with autism spectrum disorder
SO EDUCATION AND TRAINING IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID SCRIPT-FADING PROCEDURE; COOPERATIVE LEARNING GROUPS; SOCIAL-INTERACTION
SKILLS; SELF-MANAGEMENT; KNOWLEDGE CONSTRUCTION; ACADEMIC-ACHIEVEMENT;
INTEGRATION STRATEGY; BEGINNING READERS; TEACHING-CHILDREN; STUDENTS
AB A multiple baseline design across participants was used to investigate the effects of reciprocal questioning strategy instruction delivered in cooperative pairs on the question generation and responding of children with autism spectrum disorder. Three children with autism spectrum disorder and nine general education peers participated in the study. Following intervention, children with autism increased frequency of question generation and responding using a story map framework. Social validity data indicated children with autism spectrum disorder and their general education peers found the intervention helpful, and parents perceived a change in their child's reading and language skills.
C1 [Whalon, Kelly] Coll William & Mary, Williamsburg, VA 23187 USA.
[Hanline, Mary Frances] Florida State Univ, Tallahassee, FL 32306 USA.
RP Whalon, K (reprint author), Coll William & Mary, POB 8795, Williamsburg, VA 23187 USA.
EM kjwhal@wm.edu
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NR 60
TC 10
Z9 10
PU COUNCIL EXCEPTIONAL CHILDREN
PI ARLINGTON
PA 1110 N GLEBE RD, ARLINGTON, VA 22201-5704 USA
SN 1547-0350
J9 EDUC TRAIN DEV DISAB
JI Educ. Train. Dev. Disabil.
PD SEP
PY 2008
VL 43
IS 3
BP 367
EP 387
PG 21
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 335GA
UT WOS:000258277600008
ER
PT J
AU Kakooza-Mwesige, A
Wachtel, LE
Dhossche, DM
AF Kakooza-Mwesige, Angelina
Wachtel, Lee E.
Dhossche, Dirk M.
TI Catatonia in autism: implications across the life span
SO EUROPEAN CHILD & ADOLESCENT PSYCHIATRY
LA English
DT Article
DE autism; benzodiazepines; catatonia; classification; electroconvulsive
therapy; pervasive developmental disorders; psychosis; treatment
ID CHILDHOOD-ONSET SCHIZOPHRENIA; ELECTROCONVULSIVE-THERAPY; SPECTRUM
DISORDERS; RELAPSE PREVENTION; ECT; CONTINUATION; INDIVIDUALS;
ADOLESCENCE; EXPRESSION; LORAZEPAM
AB Background There is increasing evidence that catatonia is an important source of impairment in adolescents and adults with autism. Aim Review of the evaluation, diagnosis, differential diagnosis, and treatment of catatonia in autism. Method Presentation and discussion of a case-vignette spanning early childhood to adulthood. Results Autistic and catatonic symptoms overlap, yet catatonia is diagnosable in about one of seven adolescents and young adults with autism. Case-reports suggest that benzodiazepines and electroconvulsive therapy are effective treatments in the acute and maintenance phase for people with autism who develop catatonia. Conclusions Catatonia should be assessed in people with autism when there is an obvious and marked deterioration in movement, vocalizations, pattern of activities, self-care, and practical skills. Benzodiazepines and electroconvulsive therapy are favored options for acute and maintenance treatment in these cases. Further studies on the possible biological- genetic overlap between autism and catatonia would be helpful.
C1 [Wachtel, Lee E.] Johns Hopkins Sch Med, Kennedy Krieger Inst, Baltimore, MD 21205 USA.
[Kakooza-Mwesige, Angelina] Makerere Univ, Sch Med, Dept Pediat & Child Hlth, Kampala, Uganda.
[Dhossche, Dirk M.] Univ Mississippi, Med Ctr, Dept Psychiat & Human Behav, Jackson, MS 39216 USA.
RP Dhossche, DM (reprint author), Univ Mississippi, Med Ctr, Dept Psychiat & Human Behav, 2500 N State St, Jackson, MS 39216 USA.
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NR 61
TC 25
Z9 25
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1018-8827
J9 EUR CHILD ADOLES PSY
JI Eur. Child Adolesc. Psych.
PD SEP
PY 2008
VL 17
IS 6
BP 327
EP 335
DI 10.1007/s00787-008-0676-x
PG 9
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 356MM
UT WOS:000259782300001
PM 18427869
ER
PT J
AU Matson, JL
Dempsey, T
Wilkins, J
AF Matson, Johnny L.
Dempsey, Timothy
Wilkins, Jonathan
TI Rett syndrome in adults with severe intellectual disability: Exploration
of behavioral characteristics
SO EUROPEAN PSYCHIATRY
LA English
DT Article
DE Rett syndrome; adults; behavioral characteristics
ID SEVERE RETARDATION MESSIER; MATSON EVALUATION; SOCIAL-SKILLS; AUTISM;
CHILDREN; MECP2; EXPRESSION; GENE; COMMUNICATION; INDIVIDUALS
AB Rett syndrome is a genetically linked form of autism spectrum disorder (ASD) accompanied by intellectual disability (ID). The disorder is also characterized by cardiorespiratory dysregulation, disturbance in muscle tone, reduced brain growth and scoliosis. Over 300 studies have been published on the disorder, most of which has focused on identification of causative factors, which appears to be the result of mutations of gene MECP,. Rarely have adults with Rett syndrome been studied, and behavioral characteristics in these individuals are largely unknown. The present study aimed to extend what little is known about behavioral characteristics of Rett syndrome in adults, with particular emphasis on social, communicative, and adaptive behavior. Rett syndrome adults with severe ID were matched to autistic adults with ID and ID only controls. The implications of these data for more fully describing and diagnosing the condition in adults are discussed. (C) 2007 Elsevier Masson SAS. All rights reserved.
C1 [Matson, Johnny L.; Dempsey, Timothy; Wilkins, Jonathan] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, 324 Audubon Hall, Baton Rouge, LA 70803 USA.
EM johnmatson@aol.com
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 41
TC 15
Z9 16
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0924-9338
J9 EUR PSYCHIAT
JI Eur. Psychiat.
PD SEP
PY 2008
VL 23
IS 6
BP 460
EP 465
DI 10.1016/j.eurpsy.2007.11.008
PG 6
WC Psychiatry
SC Psychiatry
GA 350ZD
UT WOS:000259391500013
PM 18207372
ER
PT J
AU Cihak, DF
Foust, JL
AF Cihak, David F.
Foust, Jennifer L.
TI Comparing Number Lines and Touch Points to Teach Addition Facts to
Students With Autism
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism; mathematics; computation; elementary
AB Three elementary students with autism were taught single-digit addition problem-solving skills using number and touch-point strategies. Prior to the study, all students were unable to correctly calculate single-digit addition problems. An alternating-treatments design was used to compare the acquisition performance of single-digit addition problem-solving skills. The results indicated that the touch-point strategy was more effective in teaching single-digit addition skills. The touch-point strategy was then replicated using the nonpreferred strategy's content, improving all students' addition skills.
C1 [Cihak, David F.] Univ Tennessee, Dept Theory & Practice Teacher Educ, Knoxville, TN 37996 USA.
RP Cihak, DF (reprint author), Univ Tennessee, Dept Theory & Practice Teacher Educ, A412 Bailey Educ Complex, Knoxville, TN 37996 USA.
EM dcihak@utk.edu
CR Adams G., 1996, RES DIRECT INSTRUCTI
ALPERN G, 1988, DEV PROFILE 2
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NR 23
TC 11
Z9 11
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD SEP
PY 2008
VL 23
IS 3
BP 131
EP 137
DI 10.1177/1088357608318950
PG 7
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10GA
UT WOS:000207451300001
ER
PT J
AU Goin-Kochel, RP
Cohen, R
AF Goin-Kochel, Robin P.
Cohen, Robert
TI Screening Cases Within a Statewide Autism Registry A Comparison of
Parental Reports Using DSM-IV-TR Criteria Versus the SCQ
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism spectrum disorders; research registry; Social Communication
Questionnaire; diagnostic
AB Parents and caregivers of 70 children enrolled in a university-based, statewide autism registry (M age = 9.5 years) completed two questionnaires, one generated from criteria outlined in the Diagnostic and Statistical Manual of Mental Disorders-Fourth Edition-Text Revision (DSM-IV-TR) and the other the Social Communication Questionnaire (SCQ), to determine the number of cases likely to have autism. Based on DSM-IV-TR questionnaire (DSM-Q) results, 94.3% met criteria for probable autism. In contrast, 88.6% met criteria for possible autism with the SCQ. The two instruments agreed on 89% of the cases, and the frequencies of cases meeting criteria on each measure were not statistically different (p > .05). Results are discussed in terms of their application to the creation of local autism registries.
C1 [Goin-Kochel, Robin P.] Texas Childrens Hosp, Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
[Cohen, Robert] Virginia Commonwealth Univ, Dept Psychiat, Richmond, VA USA.
RP Goin-Kochel, RP (reprint author), Texas Childrens Hosp, Baylor Coll Med, Dept Mol & Human Genet, 6621 Fannin St,CC1560, Houston, TX 77030 USA.
EM kochel@bcm.tmc.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Berument SK, 1999, BRIT J PSYCHIAT, V175, P444, DOI 10.1192/bjp.175.5.444
Brooke E, 1974, CURRENT FUTURE USE R
COHEN IL, 2005, PERVASIVE DEV DISORD
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Rice C., 2007, PREVALENCE AUTISM SP
Rutter M., 2003, AUTISM DIAGNOSTIC IN
Rutter M., 2003, SOCIAL COMMUNICATION
NR 11
TC 1
Z9 1
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD SEP
PY 2008
VL 23
IS 3
BP 148
EP 154
DI 10.1177/1088357608316270
PG 7
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10GA
UT WOS:000207451300003
ER
PT J
AU Hoffman, CD
Sweeney, DP
Lopez-Wagner, MC
Hodge, D
Nam, CY
Botts, BH
AF Hoffman, Charles D.
Sweeney, Dwight P.
Lopez-Wagner, Muriel C.
Hodge, Danelle
Nam, Cindy Y.
Botts, Betsy H.
TI Children With Autism Sleep Problems and Mothers' Stress
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism; sleep problems; maternal stress
AB Parenting a child with autism has been associated with maternal stress. The present investigation examined children's sleep difficulties and severity of autism along with mothers' sleep problems in relation to stress levels reported by mothers (N = 72). Mothers' reports of their children's sleep problems were related to mothers' reports of their own sleep difficulties and to the severity of children's autistic symptoms. Severity of autism was predictive of mothers' stress. After controlling for child age and gender, mothers' sleep, and severity of autism, children's sleep was a significant predictor (p < .001) of maternal stress. Findings suggest including children's sleep problems along with their symptomatic behavior when examining factors that contribute to stress in mothers of children with autism.
C1 [Hoffman, Charles D.] Calif State Univ San Bernardino, Dept Psychol, San Bernardino, CA 92407 USA.
[Nam, Cindy Y.] Loma Linda Univ, Loma Linda, CA 92350 USA.
[Botts, Betsy H.] Univ W Florida, Special Educ Program, Pensacola, FL 32514 USA.
RP Hoffman, CD (reprint author), Calif State Univ San Bernardino, Dept Psychol, San Bernardino, CA 92407 USA.
EM choffman@csusb.edu
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NR 80
TC 19
Z9 20
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD SEP
PY 2008
VL 23
IS 3
BP 155
EP 165
DI 10.1177/1088357608316271
PG 11
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10GA
UT WOS:000207451300004
ER
PT J
AU Bird, EKR
Cleave, PL
Curia, J
Dunleavy, M
AF Bird, Elizabeth Kay-Raining
Cleave, Patricia L.
Curia, Joanne
Dunleavy, Michelle
TI Parental Talk About Internal States to Their Child With Autism
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism; language development; internal state language; parental input;
theory of mind
AB In this case study, all parental talk directed to a young child with autism at home over a 3-day period was analyzed for internal state (IS) language, which explicitly focuses upon the thoughts, feelings, and perceptions of animate beings. The mother and father used IS terms in 33% and 24% of their utterances, respectively, with sensory and desire categories occurring most frequently. Rarely did either parent elaborate on the causes or consequences of the IS terms they used. Comparison of these data to the literature on typically developing children with the same language age suggests similarities in the pattern of IS language input. However, the overall frequency appeared to be lower. Implications of these findings are discussed.
C1 [Bird, Elizabeth Kay-Raining] Dalhousie Univ, Sch Human Commun Disorders, Halifax, NS B3H 1R2, Canada.
RP Bird, EKR (reprint author), Dalhousie Univ, Sch Human Commun Disorders, 5599 Fenwick St, Halifax, NS B3H 1R2, Canada.
EM rainbird@dal.ca
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Chapman R., 1991, SYSTEMATIC ANAL LANG
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NR 24
TC 1
Z9 1
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD SEP
PY 2008
VL 23
IS 3
BP 166
EP 175
DI 10.1177/1088357608319530
PG 10
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10GA
UT WOS:000207451300005
ER
PT J
AU Barnhill, GP
AF Barnhill, Gena P.
TI Autism spectrum disorders: Applied behavioral analysis, evidence, and
practice
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Book Review
C1 [Barnhill, Gena P.] Lynchburg Coll, Sch Educ & Human Dev, Lynchburg, VA 24501 USA.
RP Barnhill, GP (reprint author), Lynchburg Coll, Sch Educ & Human Dev, 1501 Lakeside Dr, Lynchburg, VA 24501 USA.
EM barnhill@lynchburg.edu
CR Lovaas O. I., 1981, TEACHING DEV DISABLE
Lovaas O. I., 2003, TEACHING INDIVIDUALS
Skinner B. F, 1953, SCI HUMAN BEHAV
Sturmey P., 2007, AUTISM SPECTRUM DISO
NR 4
TC 0
Z9 0
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD SEP
PY 2008
VL 23
IS 3
BP 186
EP 187
DI 10.1177/1088357608316272
PG 2
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10GA
UT WOS:000207451300007
ER
PT J
AU Darley, SD
AF Darley, Sharon D.
TI That's what's different about me! Helping children understand autism
spectrum disorders
SO FOCUS ON AUTISM AND OTHER DEVELOPMENTAL DISABILITIES
LA English
DT Book Review
RP Darley, SD (reprint author), Ficquett Elementary, 2207 Williams St, Covington, GA 30014 USA.
EM kitdkitd@aol.com
CR MCCRAKEN H, 2006, THATS WHATS DIFFEREN
NR 1
TC 0
Z9 0
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1088-3576
J9 FOCUS AUTISM DEV DIS
JI Focus Autism Dev. Disabil.
PD SEP
PY 2008
VL 23
IS 3
BP 188
EP 189
PG 2
WC Education, Special; Psychology, Developmental; Rehabilitation
SC Education & Educational Research; Psychology; Rehabilitation
GA V10GA
UT WOS:000207451300009
ER
PT J
AU Gantert, M
Hurter, HE
van Kooten, IAJ
Garnier, Y
Mallmann, P
Schmitz, C
AF Gantert, M.
Huerter, H. E.
van Kooten, I. A. J.
Garnier, Y.
Mallmann, P.
Schmitz, C.
TI Increased number of cerebral granular cells after LSP injection in
sheep's fete a possible animal model for autism investigation
SO GEBURTSHILFE UND FRAUENHEILKUNDE
LA German
DT Meeting Abstract
C1 [Gantert, M.; Huerter, H. E.; Garnier, Y.; Mallmann, P.] Klinikum Univ Koln, Univ Frauenklin Koln, Cologne, Germany.
[van Kooten, I. A. J.] Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, Utrecht, Netherlands.
[Schmitz, C.] Univ Maastricht, Dept Neurosci, Maastricht, Netherlands.
[Schmitz, C.] Univ Maastricht, European Grad Sch Neurosci EURON, Maastricht, Netherlands.
RI Mallmann, Peter/B-1220-2010
NR 0
TC 0
Z9 0
PU GEORG THIEME VERLAG KG
PI STUTTGART
PA RUDIGERSTR 14, D-70469 STUTTGART, GERMANY
SN 0016-5751
J9 GEBURTSH FRAUENHEILK
JI Geburtshilfe Frauenheilkd.
PD SEP
PY 2008
VL 68
SU 1
BP S4
EP S4
PG 1
WC Obstetrics & Gynecology
SC Obstetrics & Gynecology
GA 353OO
UT WOS:000259577500017
ER
PT J
AU Buchanan, JA
Scherer, SW
AF Buchanan, Janet A.
Scherer, Stephen W.
TI Contemplating effects of genomic structural variation
SO GENETICS IN MEDICINE
LA English
DT Review
DE structural variation; copy number variants; CNV; genome scanning;
autism; complex traits
ID COPY-NUMBER-VARIATION; CEREBRAL AMYLOID ANGIOPATHY; FAMILIAL
PARKINSONS-DISEASE; AUTISM SPECTRUM DISORDERS; MICRODELETION SYNDROME;
SOTOS-SYNDROME; CHROMOSOMAL REARRANGEMENTS; SEGMENTAL DUPLICATIONS;
LOCUS DUPLICATION; SPORADIC DISEASE
AB Two developments have sparked new directions in the genetics-to-genomics transition for research and medical applications: the advance of whole-genome assays by array or DNA sequencing technologies, and the discovery among human genomes of extensive submicroscopic genomic structural variation, including copy number variation. For health care to benefit from interpretation of genomic data, we need to know how these variants contribute to the phenotype of the individual. Research is revealing the spectrum, both in size and complexity, of structural genotypic variation, and its association with a broad range of human phenotypes. Genomic disorders associated with relatively large, recurrent contiguous variants have been recognized for some time, as have certain Mendelian traits associated with functional disruption of single genes by structural variation. More recent examples from phenotype- and genotype-driven studies demonstrate a greater level of complexity, with evidence of incremental dosage effects, gene interaction networks, buffering and modifiers, and position effects. Mechanisms underlying such variation are emerging to provide a handle on the bulk of human variation, which is associated with complex traits and adaptive potential. Interpreting genotypes for personalized health care and communicating knowledge to the individual will be significant challenges for genomics professionals.
C1 [Buchanan, Janet A.; Scherer, Stephen W.] Hosp Sick Children, Ctr Appl Genom, Toronto, ON M5G 1L7, Canada.
[Buchanan, Janet A.; Scherer, Stephen W.] Hosp Sick Children, Program Genet & Genom Biol, Toronto, ON M5G 1L7, Canada.
[Scherer, Stephen W.] Univ Toronto, Dept Mol Genet, Toronto, ON, Canada.
RP Scherer, SW (reprint author), Hosp Sick Children, Ctr Appl Genom, 14th Floor,Toronto Med Discovery Tower MaRS Disco, Toronto, ON M5G 1L7, Canada.
EM steve@genet.sickkids.on.ca
RI Howe, Jennifer/I-9013-2012; Scherer, Stephen /B-3785-2013
OI Scherer, Stephen /0000-0002-8326-1999
FU Centre for Applied Genomics; Genome Canada/Ontario Genomics Institute;
the Canadian Institutes for Health Research (CIHR); the Canadian
Institutes for Advanced Research; McLaughlin Centre for Molecular
Medicine; Canadian Foundation For Innovation; Ontario Ministry of
Research and Innovation; Hospital for Sick Children Foundation
FX Supported by The Centre for Applied Genomics, Genome Canada/Ontario
Genomics Institute, the Canadian Institutes for Health Research (CIHR),
the Canadian Institutes for Advanced Research, the McLaughlin Centre for
Molecular Medicine, the Canadian Foundation For Innovation, the Ontario
Ministry of Research and Innovation, and the Hospital for Sick Children
Foundation.We thank Andrew Carson, Lars Feuk, Jeff MacDonald, Christian
Marshall, and Dalila Pinto for theoretical ideas and contributions to
the display items. S.W.S. holds the GlaxoSmith Kline/CIHR Chair in
Genetics and Genomics at the University of Toronto and the Hospital for
Sick Children.
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NR 107
TC 33
Z9 35
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 1098-3600
J9 GENET MED
JI Genet. Med.
PD SEP
PY 2008
VL 10
IS 9
BP 639
EP 647
DI 10.1097/GIM.0b013e318183f848
PG 9
WC Genetics & Heredity
SC Genetics & Heredity
GA 353VN
UT WOS:000259596800001
PM 18978673
ER
PT J
AU Sonne, T
AF Sonne, Thorkil
TI A conversation with Thorkil Sonne
SO HARVARD BUSINESS REVIEW
LA English
DT Editorial Material
AB This Danish entrepreneur shares widely applicable lessons from his experience managing employees with autism. More than two-thirds of workers at his software-testing firm, Specialisterne, have the condition and are thriving in their jobs.
NR 0
TC 0
Z9 0
PU HARVARD BUSINESS SCHOOL PUBLISHING CORPORATION
PI WATERTOWN
PA 300 NORTH BEACON STREET, WATERTOWN, MA 02472 USA
SN 0017-8012
J9 HARVARD BUS REV
JI Harv. Bus. Rev.
PD SEP
PY 2008
VL 86
IS 9
BP 32
EP +
PG 2
WC Business; Management
SC Business & Economics
GA 340DW
UT WOS:000258627300007
ER
PT J
AU McPhie-Lalmansingh, AA
Tejada, LD
Weaver, JL
Rissman, EF
AF McPhie-Lalmansingh, Anika A.
Tejada, Lucia D.
Weaver, Jessica L.
Rissman, Emilie F.
TI Sex chromosome complement affects social interactions in mice
SO HORMONES AND BEHAVIOR
LA English
DT Article
DE affective disorders; autism; depression; anxiety; sexual
differentiation; X inactivation; cognition; pain
ID SEXUALLY DIMORPHIC EXPRESSION; X-CHROMOSOME; Y-CHROMOSOME; MOUSE-BRAIN;
PSEUDOAUTOSOMAL REGION; INTERMALE AGGRESSION; GENE-EXPRESSION;
NERVOUS-SYSTEM; DIFFERENTIATION; FEMALE
AB Sex differences in behavior can be attributed to differences in steroid hormones. Sex chromosome complement can also influence behavior, independent of gonadal differentiation. The mice used for this work combined a spontaneous mutation of the Sty gene with a transgene for Sty that is incorporated into an autosome thus disassociating gonad differentiation from sex chromosome complement. The resulting genotypes are XX and XY- females (ovary-bearing) along with XXSry and XY(-)Sry males (testes-bearing). Here we report results of basic behavioral phenotyping conducted with these mice. Motor coordination, use of olfactory cues to find a food item, general activity, foot shock threshold, and behavior in an elevated plus maze were not affected by gonadal sex or sex chromosome complement. In a one-way active avoidance learning task females were faster to escape an electric shock than males. In addition, sex chromosome complement differences were noted during social interactions with submissive intruders. Female XY- mice were faster to follow an intruder than XX female mice. All XY- mice spent more time sniffing and grooming the intruder than the XX mice, with XY- females spending the most amount of time in this activity. Finally, XX females were faster to display an asocial behavior, digging, and engaged in more digging than XXSry male mice. All of these behaviors were tested in gonadectomized adults, thus, differences in circulating levels of gonadal steroids cannot account for these effects. Taken together, these data show that sex chromosome complement affects social interaction style in mice. (C) 2008 Published by Elsevier Inc.
C1 [McPhie-Lalmansingh, Anika A.; Tejada, Lucia D.; Weaver, Jessica L.; Rissman, Emilie F.] Univ Virginia, Sch Med, Dept Biochem & Mol Genet, Program Neurosci, Charlottesville, VA 22908 USA.
RP Rissman, EF (reprint author), Univ Virginia, Sch Med, Dept Biochem & Mol Genet, Program Neurosci, POB 800733, Charlottesville, VA 22908 USA.
EM Rissman@virginia.edu
FU [T32 DK007646]; [R01 NS55218]; [T32 GM008328]
FX The authors would like to thank Aileen Wills for care of the animals and
Savera Shetty for genotyping. We thank Dr. Marcia McDuffie for
verification of our crosses into C57BL/6j. This work was supported by
T32 DK007646, R01 NS55218, and T32 GM008328.
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NR 41
TC 29
Z9 29
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0018-506X
J9 HORM BEHAV
JI Horm. Behav.
PD SEP
PY 2008
VL 54
IS 4
BP 565
EP 570
DI 10.1016/j.yhbeh.2008.05.016
PG 6
WC Behavioral Sciences; Endocrinology & Metabolism
SC Behavioral Sciences; Endocrinology & Metabolism
GA 347RR
UT WOS:000259159200013
PM 18590732
ER
PT J
AU Field, T
Diego, M
AF Field, Tiffany
Diego, Miguel
TI Vagal activity, early growth and emotional development
SO INFANT BEHAVIOR & DEVELOPMENT
LA English
DT Article
DE vagal activity; emotional development
ID AUTONOMIC NERVOUS-SYSTEM; HEART-RATE-VARIABILITY; BIRTH-WEIGHT INFANTS;
PRETERM INFANTS; DEPRESSED MOTHERS; MASSAGE THERAPY; TONE; STABILITY;
TEMPERAMENT; REACTIVITY
AB A review of the research on infant vagal tone suggests that vagal activity is associated with both infant growth and infant socioemotional development. Vagal activity has been noted to increase following the stimulation of pressure receptors as in massage therapy. Vagal activity, in turn, stimulates gastric motility which mediates weight gain in infants. Vagal activity has also been notably elevated during synchronous mother-infant interactions and positive affect, providing confirmatory data for the Porges "social engagement system" model. In contrast. low vagal activity has been noted in prenatally depressed mothers (and prenatally angry and anxious mothers) and their infants, as well as in children with autism. These studies highlight the relations between vagal activity and the social behaviors of attentiveness, facial expressions and vocalizations. (c) 2008 Elsevier Inc. All rights reserved.
C1 [Field, Tiffany; Diego, Miguel] Univ Miami, Sch Med, Touch Res Inst, Miami, FL 33101 USA.
[Field, Tiffany] Fielding Grad Univ, Santa Barbara, CA USA.
RP Field, T (reprint author), Univ Miami, Sch Med, Touch Res Inst, POB 016820, Miami, FL 33101 USA.
EM tfield@med.miami.edu
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NR 60
TC 27
Z9 27
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0163-6383
J9 INFANT BEHAV DEV
JI Infant Behav. Dev.
PD SEP
PY 2008
VL 31
IS 3
BP 361
EP 373
DI 10.1016/j.infbeh.2007.12.008
PG 13
WC Psychology, Developmental
SC Psychology
GA 341UQ
UT WOS:000258741000003
PM 18295898
ER
PT J
AU Liu, CC
Conn, K
Sarkar, N
Stone, W
AF Liu, Changchun
Conn, Karla
Sarkar, Nilanjan
Stone, Wendy
TI Physiology-based affect recognition for computer-assisted intervention
of children with Autism Spectrum Disorder
SO INTERNATIONAL JOURNAL OF HUMAN-COMPUTER STUDIES
LA English
DT Article
DE human-computer Interaction; Autism intervention; physiological sensing;
support vector machines; affect recognition
ID EMOTION RECOGNITION; AUTONOMIC RESPONSE; SOCIAL-INTERACTION; PEOPLE;
SKILLS; DEFICITS; ANXIETY; GAME
AB Generally, an experienced therapist continuously monitors the affective cues of the children with Autism Spectrum Disorders (ASD) and adjusts the course of the intervention accordingly. In this work, we address the problem of how to make the computer-based ASD intervention tools affect-sensitive by designing therapist-like affective models of the children with ASD based on their physiological responses. Two computer-based cognitive tasks are designed to elicit the affective states of liking, anxiety, and engagement that are considered important in autism intervention. A large set of physiological indices are investigated that may correlate with the above affective states of children with ASD. In order to have reliable reference points to link the physiological data to the affective states, the subjective reports of the affective states from a therapist, a parent, and the child himself/herself were collected and analyzed. A support vector machines (SVM)-based affective model yields reliable prediction with approximately 82.9% success when using the therapist's reports. This is the first time, to our knowledge, that the affective states of children with ASD have been experimentally detected via physiology-based affect recognition technique. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Liu, Changchun; Conn, Karla; Sarkar, Nilanjan] Vanderbilt Univ, Dept Elect Engn & Comp Sci, Nashville, TN 37235 USA.
[Sarkar, Nilanjan] Vanderbilt Univ, Dept Mech Engn, Nashville, TN 37235 USA.
[Stone, Wendy] Vanderbilt Treatment & Res Inst Autism Spectrum D, Nashville, TN 37212 USA.
[Stone, Wendy] Vanderbilt Kennedy Ctr, Dept Pediat, Nashville, TN 37212 USA.
RP Liu, CC (reprint author), Vanderbilt Univ, Dept Elect Engn & Comp Sci, VU Stn B 351679,2301 Vanderbilt Pl, Nashville, TN 37235 USA.
EM changchun.liu@vanderbilt.edu; karla.g.conn@vanderbilt.edu;
nilanjan.sarkar@vanderbilt.edu; wendy.stone@vanderbilt.edu
RI Liu, Changchun/D-2256-2012; Welch, Karla/B-3425-2013
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NR 68
TC 24
Z9 25
PU ACADEMIC PRESS LTD- ELSEVIER SCIENCE LTD
PI LONDON
PA 24-28 OVAL RD, LONDON NW1 7DX, ENGLAND
SN 1071-5819
J9 INT J HUM-COMPUT ST
JI Int. J. Hum.-Comput. Stud.
PD SEP
PY 2008
VL 66
IS 9
BP 662
EP 677
DI 10.1016/j.ijhcs.2008.04.003
PG 16
WC Computer Science, Cybernetics; Ergonomics; Psychology, Multidisciplinary
SC Computer Science; Engineering; Psychology
GA 342TZ
UT WOS:000258807700002
ER
PT J
AU Ng, F
Berk, M
Dean, O
Bush, AI
AF Ng, Felicity
Berk, Michael
Dean, Olivia
Bush, Ashley I.
TI Oxidative stress in psychiatric disorders: evidence base and therapeutic
implications
SO INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY
LA English
DT Review
DE antioxidant; mechanisms; oxidative stress; pathophysiology; psychiatric
disorders
ID N-ACETYL-CYSTEINE; ANTIOXIDANT ENZYME-ACTIVITIES; PLACEBO-CONTROLLED
TRIAL; OBSESSIVE-COMPULSIVE DISORDER; POLYUNSATURATED FATTY-ACIDS;
MEMBRANE-LIPID PEROXIDATION; RADICAL SCAVENGING ENZYMES; BIPOLAR
AFFECTIVE-DISORDER; MAJOR DEPRESSIVE DISORDER; CEREBRAL CORTICAL-CELLS
AB Oxidative stress has been implicated in the pathogenesis of diverse disease states, and may be a common pathogenic mechanism underlying many major psychiatric disorders, as the brain has comparatively greater vulnerability to oxidative damage. This review aims to examine the current evidence for the role of oxidative stress in psychiatric disorders, and its academic and clinical implications. A literature search was conducted using the Medline, Pubmed, PsycINFO, CINAHL PLUS, BIOSIS Previews, and Cochrane databases, with a time-frame extending to September 2007. The broadest data for oxidative stress mechanisms have been derived from studies conducted in schizophrenia, where evidence is available from different areas of oxidative research, including oxidative marker assays, psychopharmacology studies, and clinical trials of antioxidants. For bipolar disorder and depression, a solid foundation for oxidative stress hypotheses has been provided by biochemical, genetic, pharmacological, preclinical therapeutic studies and one clinical trial. Oxidative pathophysiology in anxiety disorders is strongly supported by animal models, and also by human biochemical data. Pilot studies have suggested efficacy of N-acetylcysteine in cocaine dependence, while early evidence is accumulating for oxidative mechanisms in autism and attention deficit hyperactivity disorder. In conclusion, multi-dimensional data support the role of oxidative stress in diverse psychiatric disorders. These data not only suggest that oxidative mechanisms may form unifying common pathogenic pathways in psychiatric disorders, but also introduce new targets for the development of therapeutic interventions.
C1 [Ng, Felicity; Berk, Michael] Univ Melbourne, Dept Clin & Biomed Sci, Geelong, Vic, Australia.
[Berk, Michael; Dean, Olivia; Bush, Ashley I.] Mental Hlth Res Inst Victoria, Parkville, Vic, Australia.
[Berk, Michael] ORYGEN Res Ctr, Parkville, Vic, Australia.
RP Ng, F (reprint author), Swanston Ctr, POB 281, Geelong, Vic 3220, Australia.
EM felicitn@barwonhealth.org.au
RI Berk, Michael/M-7891-2013; Bush, Ashley/A-1186-2007
OI Berk, Michael/0000-0002-5554-6946; Bush, Ashley/0000-0001-8259-9069
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Yanik M, 2004, ACTA NEUROPSYCHIATR, V16, P200, DOI 10.1111/j.0924-2708.2004.00090.x
Yao JK, 2004, SCHIZOPHRENIA BULL, V30, P923
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Yao JK, 1998, SCHIZOPHR RES, V32, P1, DOI 10.1016/S0920-9964(98)00030-9
Yao JK, 2006, DIS MARKERS, V22, P83
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Yorbik O, 2002, PROSTAG LEUKOTR ESS, V67, P341, DOI 10.1054/plef.439
Young J, 2007, PROSTAG LEUKOTR ESS, V76, P73, DOI 10.1016/j.plefa.2006.11.003
Young LT, 2007, J PSYCHIATR NEUROSCI, V32, P160
Zhang XY, 2001, J CLIN PSYCHIAT, V62, P878
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Zhang XY, 2003, J CLIN PSYCHOPHARM, V23, P128, DOI 10.1097/00004714-200304000-00004
Zhou DF, 1999, CHINESE MED J-PEKING, V112, P1093
ZHOU W, 2007, BIOL PSYCHIAT
Zingg JM, 2004, CURR MED CHEM, V11, P1113
Zoroglu SS, 2004, EUR ARCH PSY CLIN N, V254, P143, DOI 10.1007/s00406-004-0456-7
Zoroglu SS, 2002, J PSYCHIAT RES, V36, P309
NR 190
TC 271
Z9 275
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1461-1457
J9 INT J NEUROPSYCHOPH
JI Int. J. Neuropsychopharmacol.
PD SEP
PY 2008
VL 11
IS 6
BP 851
EP 876
DI 10.1017/S1461145707008401
PG 26
WC Clinical Neurology; Neurosciences; Pharmacology & Pharmacy; Psychiatry
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Psychiatry
GA 355KC
UT WOS:000259706800010
PM 18205981
ER
PT J
AU Martineau, J
Hernandez, N
Andersson, F
Destrieux, C
AF Martineau, J.
Hernandez, N.
Andersson, F.
Destrieux, C.
TI Visual scanning, brain activations and emotional face perception in
autism
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
C1 [Martineau, J.; Hernandez, N.; Destrieux, C.] INSERM, Paedopsychiat Dept, U 930, Tours, France.
[Andersson, F.] Dept Psychol Cognit Sci Unit, IFR135, Helsinki, Finland.
RI Destrieux, Christophe/K-1190-2012
NR 0
TC 0
Z9 0
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 175
EP 175
DI 10.1016/j.ijpsycho.2008.05.457
PG 1
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700129
ER
PT J
AU Orekhova, EV
Stroganova, TA
Prokofiev, AO
Nygren, G
Gillberg, C
Elam, M
AF Orekhova, E. V.
Stroganova, T. A.
Prokofiev, A. O.
Nygren, G.
Gillberg, C.
Elam, M.
TI Right hemispheric failure to respond to temporal novelty in autism:
Evidence from an event-related potential study
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
C1 [Orekhova, E. V.; Elam, M.] Sahlgrens Univ Hosp, Dept Clin Neurophysiol, S-41345 Gothenburg, Sweden.
[Stroganova, T. A.; Prokofiev, A. O.] Moscow Univ Psychol & Educ, Moscow 103051, Russia.
[Nygren, G.; Gillberg, C.] Sahlgrens Univ Hosp, Dept Child & Adolescent Psychiat, S-41119 Gothenburg, Sweden.
NR 0
TC 1
Z9 1
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 186
EP 186
DI 10.1016/j.ijpsycho.22008.05.496
PG 1
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700168
ER
PT J
AU Murias, M
AF Murias, M.
TI EEG connectivity differences at different ages in autism spectrum
disorder
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
C1 [Murias, M.] Univ Washington, Seattle, WA 98195 USA.
NR 0
TC 0
Z9 0
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 202
EP 202
DI 10.1016/j.ijpsycho.2008.05.552
PG 1
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700224
ER
PT J
AU Innocenti, GM
AF Innocenti, G. M.
TI Stimulus dependent interaction between the visual areas of the two
hemispheres
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
ID EEG COHERENCE; PHASE SYNCHRONIZATION; CONNECTIVITY; INDIVIDUALS;
NETWORKS; AUTISM; BRAIN
C1 [Innocenti, G. M.] Karolinska Inst, Dept Neurosci, Stockholm, Sweden.
CR AJDARI RA, 2008, CHAOS, V18, P2008
Bernier R, 2007, BRAIN COGNITION, V64, P228, DOI 10.1016/j.bandc.2007.03.004
Carmeli C, 2007, PLOS ONE, V2, DOI 10.1371/journal.pone.0001287
Carmeli C, 2005, NEUROIMAGE, V25, P339, DOI 10.1016/j.neuroimage.2004.11.049
Innocenti GM, 2005, NAT REV NEUROSCI, V6, P955, DOI 10.1038/nrn1790
Ito J, 2005, BIOL CYBERN, V92, P54, DOI 10.1007/s00422-004-0533-z
Jalili M, 2007, INT J CIRC THEOR APP, V35, P611, DOI 10.1002/cta.436
Jalili M, 2007, PLOS ONE, V2, DOI 10.1371/journal.pone.0001059
Knyazeva MG, 2001, BRAIN RES REV, V36, P119, DOI 10.1016/S0165-0173(01)00087-X
Knyazeva MG, 2006, J NEUROPHYSIOL, V96, P259, DOI 10.1152/jn.00687.2005
Knyazeva MG, 2006, NEUROIMAGE, V29, P593, DOI 10.1016/j.neuroimage.2005.07.045
MAKAROV VA, 2007, CEREB CORTEX 1207
Murias M, 2007, CEREB CORTEX, V17, P1788, DOI 10.1093/cercor/bhl089
Murias M, 2007, BIOL PSYCHIAT, V62, P270, DOI 10.1016/j.biopsych.2006.11.012
Nikolaev AR, 2005, CLIN NEUROPHYSIOL, V116, P2403, DOI 10.1016/j.clinph.2005.07.003
NIKOLAEV AR, 2005, PSYCHOL RES 1008, P1
STERLING L, 2008, J AUTISM DEV DI 0228
NR 17
TC 0
Z9 0
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 203
EP 203
DI 10.1016/j.ijpsycho.2008.05.554
PG 1
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700226
ER
PT J
AU Stroganova, TA
Orekhova, EV
Tsetlin, MM
Posikera, IN
Nygren, G
Gillberg, C
Elam, M
AF Stroganova, T. A.
Orekhova, E. V.
Tsetlin, M. M.
Posikera, I. N.
Nygren, G.
Gillberg, C.
Elam, M.
TI EEG evidences of aberrant brain functioning in young children with
autism
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
C1 [Stroganova, T. A.; Tsetlin, M. M.] Moscow State Univ Psychol & Educ, Moscow, Russia.
[Orekhova, E. V.; Elam, M.] Sahlgrens Univ Hosp, Dept Clin Neurophysiol, Gothenburg, Sweden.
[Posikera, I. N.] Russian Acad Educ, Inst Psychol, Moscow, Russia.
Sahlgrens Univ Hosp, Dept Child & Adolescent Psychiat, Gothenburg, Sweden.
NR 0
TC 4
Z9 4
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 203
EP 204
DI 10.1016/j.ijpsycho.2008.05.556
PG 2
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700228
ER
PT J
AU Tsetlin, MM
Pushina, NP
Galuta, IA
Stroganova, TA
AF Tsetlin, M. M.
Pushina, N. P.
Galuta, I. A.
Stroganova, T. A.
TI Spatial asymmetry of frontal tasks performance in young boys with autism
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
C1 [Tsetlin, M. M.; Galuta, I. A.] Moscow City Univ Psychol & Educ, Moscow, Russia.
[Pushina, N. P.; Stroganova, T. A.] Russian Acad Educ, Inst Psychol, Lab Behav Genet, Moscow, Russia.
NR 0
TC 2
Z9 2
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 267
EP 267
DI 10.1016/j.ijpsycho.2008.05.209
PG 1
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700432
ER
PT J
AU Kim, SK
Kim, HJ
Park, HJ
Ban, JY
Yim, SV
Kim, JW
Hong, SJ
Choe, BK
Park, HK
Chung, JH
AF Kim, S. K.
Kim, H. J.
Park, H. J.
Ban, J. Y.
Yim, S. V.
Kim, J. W.
Hong, S. J.
Choe, B. K.
Park, H. K.
Chung, J. H.
TI Polymorphisms of piccolo (PCLO), bassoon (BSN), and OBOE (RIMS2) in
schizophrenia and autism spectrum disorders in the Korean population
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
C1 [Kim, S. K.; Kim, H. J.; Park, H. J.; Ban, J. Y.; Yim, S. V.; Kim, J. W.; Hong, S. J.; Choe, B. K.; Chung, J. H.] Kyung Hee Univ, Dept Pharmacol, Seoul, South Korea.
[Park, H. K.] Kyung Hee Univ, Dept Biomed Engn, Seoul, South Korea.
NR 0
TC 0
Z9 0
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 272
EP 272
DI 10.1016/j.ijpsycho.2008.05.226
PG 1
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700449
ER
PT J
AU Vorsanov, SG
Lourov, IY
Demidova, IA
Beresheva, AK
Ulas, VYV
Kravets, VS
Kolotii, AD
Monakhov, VV
Kurinnaya, OS
Soloviev, IV
Gorbachevskaya, NL
Yurov, YB
AF Vorsanov, S. G.
Lourov, I. Y.
Demidova, I. A.
Beresheva, A. K.
Ulas, V. Y. Voinova
Kravets, V. S.
Kolotii, A. D.
Monakhov, V. V.
Kurinnaya, O. S.
Soloviev, I. V.
Gorbachevskaya, N. L.
Yurov, Y. B.
TI Interindividual and intercellular genomic variations significantly
contribute to pathophysiology of autism
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
C1 [Vorsanov, S. G.; Demidova, I. A.; Beresheva, A. K.; Ulas, V. Y. Voinova; Kravets, V. S.; Kolotii, A. D.; Kurinnaya, O. S.] Rosmedtechnol, Inst Pediat & Children Surg, Moscow, Russia.
[Lourov, I. Y.; Monakhov, V. V.; Soloviev, I. V.; Gorbachevskaya, N. L.; Yurov, Y. B.] RAMS, Natl Res Ctr Mental Hlth, Lab Cytogenet, Moscow, Russia.
NR 0
TC 0
Z9 0
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 288
EP 289
DI 10.1016/j.ijpsycho.2008.05.264
PG 2
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700492
ER
PT J
AU Pop-Jordanova, N
AF Pop-Jordanova, N.
TI Psychophysiological characteristics of autism in children
SO INTERNATIONAL JOURNAL OF PSYCHOPHYSIOLOGY
LA English
DT Meeting Abstract
CT 14th World Congress of Psychophysiology the Olympics of the Brain
CY SEP 08-13, 2008
CL St Petersburg, RUSSIA
SP Int Org Psychophysiol
C1 [Pop-Jordanova, N.] Univ Skopje, Fac Med, Pediat Clin, Dept Psychophysiol, Skopje, Macedonia.
NR 0
TC 1
Z9 1
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0167-8760
J9 INT J PSYCHOPHYSIOL
JI Int. J. Psychophysiol.
PD SEP
PY 2008
VL 69
IS 3
BP 309
EP 309
DI 10.1016/j.ijpsycho.2008.05.320
PG 1
WC Psychology, Biological; Neurosciences; Physiology; Psychology;
Psychology, Experimental
SC Psychology; Neurosciences & Neurology; Physiology
GA 336VU
UT WOS:000258393700548
ER
PT J
AU Spencer, VG
Simpson, CG
Lynch, SA
AF Spencer, Vicky G.
Simpson, Cynthia G.
Lynch, Sharon A.
TI Using social stories to increase positive behaviors for children with
autism spectrum disorders
SO INTERVENTION IN SCHOOL AND CLINIC
LA English
DT Article
ID ASPERGER-SYNDROME; IMPROVE
C1 [Spencer, Vicky G.] George Mason Univ, Heller Inst Human Disabil, Fairfax, VA 22030 USA.
[Simpson, Cynthia G.; Lynch, Sharon A.] Sam Houston State Univ, Dept Language Literacy & Special Populat, Huntsville, TX 77340 USA.
RP Spencer, VG (reprint author), Sam Houston State Univ, Teacher Educ Ctr, 1908 Bobby K Marks Dr, Huntsville, TX 77340 USA.
EM vspencer@shsu.edu
CR Agosta E, 2004, INTERV SCH CLIN, V39, P276, DOI 10.1177/10534512040390050401
BARRY LM, 2004, FOCUS AUTISM OTHER D, V19, P43
Crozier S., 2005, FOCUS AUTISM OTHER D, V20, P150, DOI DOI 10.1177/10883576050200030301
Crozier S., 2005, TEACHING EXCEPTIONAL, V37, P26
Gray C., 2000, WRITING SOCIAL STORI
Gray C. A., 1993, FOCUS AUTISTIC BEHAV, V8, P1, DOI DOI 10.1177/108835769300800101
Kuoch H., 2003, FOCUS AUTISM OTHER D, V18, P219, DOI DOI 10.1177/10883576030180040301
Kuttler S., 1998, FOCUS AUTISM OTHER D, V13, P176, DOI DOI 10.1177/108835769801300306
Lorimer PA, 2002, J POSIT BEHAV INTERV, V4, P53, DOI 10.1177/109830070200400109
MIRENDA P, 1989, J SPEECH HEAR DISORD, V54, P131
Myles B. S., 1999, FOCUS AUTISM OTHER D, V14, P82, DOI 10.1177/108835769901400203
Norris C, 1999, FOCUS AUTISM OTHER D, V14, P180, DOI DOI 10.1177/108835769901400307
O'Neill R. E., 1997, FUNCTIONAL ASSESSMEN, V2nd
Rogers MF, 2001, INTERV SCH CLIN, V36, P310
Sansosti FJ, 2006, J POSIT BEHAV INTERV, V8, P43, DOI 10.1177/10983007060080010601
Scattone D, 2002, J AUTISM DEV DISORD, V32, P535, DOI 10.1023/A:1021250813367
Smith C., 2001, ED PSYCHOL PRACTICE, V17, P337, DOI 10.1080/02667360127230
Swaggart B. L., 1995, FOCUS AUTISTIC BEHAV, V10, P1
Theimann K., 2001, J APPL BEHAV ANAL, V34, P425
NR 19
TC 2
Z9 2
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1053-4512
J9 INTERV SCH CLIN
JI Interv. Sch. Clin.
PD SEP
PY 2008
VL 44
IS 1
BP 58
EP 61
DI 10.1177/1053451208318876
PG 4
WC Education, Special
SC Education & Educational Research
GA 345HS
UT WOS:000258987700009
ER
PT J
AU Nazni, P
Wesely, EG
Nishadevi, V
AF Nazni, Peerkhan
Wesely, Edward Gnanaraj
Nishadevi, Veerappan
TI Impact of casein and gluten free dietary intervention on selected
autistic children
SO IRANIAN JOURNAL OF PEDIATRICS
LA English
DT Article
DE autism; gluten; casein; opioid peptide; children; dietary intervention
ID SPECTRUM DISORDER; BRAIN
AB Objective: Autism is a life long developmental disorder that emerges in early childhood and results in significant lifelong disability. The goal of treatment is to promote the child's social and language development and minimize behaviors that interfere with the child's functioning and learning. This study evaluated the impact of casein and gluten free diet among selected autistic children.
Methods: Three private special schools in Salem District, Tamilnadu, India were selected. A total number of 50 autistic children 10 from SIMEC, 10 from MMIC and 30 from CSI comprised the study sample. Background information, clinical history and nutritional status, dietary pattern were collected from the 50 selected autistic children. Out of 50 autistic children 30 autistic children were selected for the dietary intervention. Diet counseling regarding casein free diet was imparted to Group I (n=10), gluten free diet to Group 11 (n=10) and both casein and gluten free diet for Group III (n=10). The diet was followed for a period of 2 months. The efficacy of the dietary exclusion of casein and gluten was evaluated using a food and behavior diary on a day to day basis, using observation method.
Findings: Results about Group I autistic children who followed dietary exclusion of casein free diet showed that the mean scores before and after casein free dietary intervention depiticted these improvements as, 1 to 1.2 for attention, 2.8 to 2.9 for sleep, 1.1 to 1.3 for hyperactivity, 1.1 to 1.2 for anxiety/compulsion. For Group 11 autistic children who followed dietary exclusion of gluten free diet showed the improvements as 1.1 to 1.4 for attention 2.5 to 3 for sleep, 1.7 to 1.9 for hyperactivity, 1.1 to 1.2 for anxiety/compulsion. About Group III autistic children who followed dietary exclusion of both casein and gluten free diet showed the improvements as 1.1 to 1.3 for attention, 2.5 to 2.7 for sleep, 1.3 to 1.7 for hyperactivity, and 1.1 to 1.2 for anxiety/compulsion.
Conclusion:The impact of dietary intervention made using dietary guide books found to be useful to reduce various behavior symptoms among the selected autistic children.
C1 [Nazni, Peerkhan; Nishadevi, Veerappan] Periyar Univ, Dept Food Sci, Salem, Tamil Nadu, India.
[Wesely, Edward Gnanaraj] Muthayammal Coll Arts & Sci, Dept Biotechnol, Rasipuram, Tamil Nadu, India.
RP Nazni, P (reprint author), Periyar Univ, Dept Food Sci, Salem, Tamil Nadu, India.
EM naznip@gmail.com
FU Tamil Nadu State Council for Science and Technology (TNSCST), Chennai
FX The authors are immensely thankful to the Tamil Nadu State Council for
Science and Technology (TNSCST), Chennai, for approving a grant towards
the conduct of the study.
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Whiteley P, 2001, Expert Opin Pharmacother, V2, P1191
NR 18
TC 1
Z9 1
PU IRANIAN SCIENTIFIC SOCIETY MEDICAL ENTOMOLOGY
PI TEHRAN
PA SCHOOL PUBLIC HEALTH & INST HEALTH RESEARCH, TEHRAN UNIV MEDICAL
SCIENCES, P O BOX 6446-14155, TEHRAN, 00000, IRAN
SN 2008-2142
J9 IRAN J PEDIATR
JI Iran. J. Pediatr.
PD SEP
PY 2008
VL 18
IS 3
BP 244
EP 250
PG 7
WC Pediatrics
SC Pediatrics
GA 354XL
UT WOS:000259673100005
ER
PT J
AU Volkert, VM
Lerman, DC
Trosclair, N
Addison, L
Kodak, T
AF Volkert, Valerie M.
Lerman, Dorothea C.
Trosclair, Nicole
Addison, Laura
Kodak, Tiffany
TI An exploratory analysis of task-interspersal procedures while teaching
object labels to children with autism
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; behavioral momentum; high-probability instructional sequence;
interspersal procedures; reinforcer potency
ID BEHAVIORAL MOMENTUM; STUDENTS; DISABILITIES; ACQUISITION; RETENTION;
REINFORCEMENT; PERCEPTIONS; PERFORMANCE; ASSIGNMENT; DISORDERS
AB Research has demonstrated that interspersing mastered tasks with new tasks facilitates learning under certain conditions; however, little is known about factors that influence the effectiveness of this treatment strategy. The initial purpose of the current investigation was to evaluate the effects of similar versus dissimilar interspersed tasks while teaching object labels to children diagnosed with autism or developmental delays. We then conducted a series of exploratory analyses involving the type of reinforcer delivered for correct responses on trials with unknown or known object labels. Performance was enhanced under the interspersal condition only when either brief praise was delivered for all correct responses or presumably more preferred reinforcers were provided for performance on known trials rather than on unknown trials.
C1 [Volkert, Valerie M.; Trosclair, Nicole; Addison, Laura; Kodak, Tiffany] Louisiana State Univ, Baton Rouge, LA 70803 USA.
[Lerman, Dorothea C.] Univ Houston Clear Lake, Houston, TX 77058 USA.
RP Volkert, VM (reprint author), 900 Farnam St,Apt 813, Omaha, NE 68102 USA.
EM vvolkert@unmc.edu
CR Browder DM, 1996, J SPEC EDUC, V29, P400
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NR 22
TC 10
Z9 10
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD FAL
PY 2008
VL 41
IS 3
BP 335
EP 350
DI 10.1901/jaba.2008.41-335
PG 16
WC Psychology, Clinical
SC Psychology
GA 345AU
UT WOS:000258969200003
PM 18816973
ER
PT J
AU Roscoe, EM
Carreau, A
MacDonald, J
Pence, ST
AF Roscoe, Eileen M.
Carreau, Abbey
MacDonald, Jackie
Pence, Sacha T.
TI Further evaluation of leisure items in the attention condition of
functional analyses
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE assessment; autism; functional analysis; problem behavior
ID MAINTAINED DESTRUCTIVE BEHAVIOR; SELF-INJURIOUS-BEHAVIOR; NONCONTINGENT
DELIVERY; ALTERNATIVE STIMULI; ABERRANT BEHAVIOR; ASSESSMENTS
AB Research suggests that including leisure items in the attention condition of a functional analysis may produce engagement that masks sensitivity to attention. In this study, 4 individuals' initial functional analyses indicated that behavior was maintained by nonsocial variables (n = 3) or by attention (n = 1). A preference assessment was used to identify items for subsequent functional analyses. Four conditions were compared, attention with and without leisure items and control with and without leisure items. Following this, either high- or low-preference items were included in the attention condition. Problem behavior was more probable during the attention condition when no leisure items or low-preference items were included, and lower levels of problem behavior were observed during the attention condition when high-preference leisure items were included. These findings suggest how preferred items may hinder detection of behavioral function.
C1 [Roscoe, Eileen M.; Carreau, Abbey; MacDonald, Jackie; Pence, Sacha T.] NE Univ, New England Ctr Children, Southborough, MA 01772 USA.
RP Roscoe, EM (reprint author), NE Univ, New England Ctr Children, 33 Turnpike Rd, Southborough, MA 01772 USA.
EM eroscoe@necc.org
CR Berg WK, 2000, J APPL BEHAV ANAL, V33, P463, DOI 10.1901/jaba.2000.33-463
DERBY KM, 1992, J APPL BEHAV ANAL, V25, P713, DOI 10.1901/jaba.1992.25-713
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Fisher WW, 2000, J APPL BEHAV ANAL, V33, P79, DOI 10.1901/jaba.2000.33-79
Hanley GP, 1997, J APPL BEHAV ANAL, V30, P229, DOI 10.1901/jaba.1997.30-229
Hanley GP, 2003, J APPL BEHAV ANAL, V36, P147, DOI 10.1901/jaba.2003.36-147
IWATA BA, 1994, J APPL BEHAV ANAL, V27, P215, DOI 10.1901/jaba.1994.27-215
IWATA BA, 1994, J APPL BEHAV ANAL, V27, P197, DOI 10.1901/jaba.1994.27-197
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MICHAEL J, 1982, J EXP ANAL BEHAV, V37, P149, DOI 10.1901/jeab.1982.37-149
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O'Reilly MF, 2006, J APPL BEHAV ANAL, V39, P239, DOI 10.1901/jaba.2006.160-04
Piazza CC, 1996, J APPL BEHAV ANAL, V29, P137, DOI 10.1901/jaba.1996.29-137
Ringdahl JE, 2002, J APPL BEHAV ANAL, V35, P407, DOI 10.1901/jaba.2002.35-407
Smith RG, 1995, J APPL BEHAV ANAL, V28, P515, DOI 10.1901/jaba.1995.28-515
Vollmer TR, 1995, J APPL BEHAV ANAL, V28, P561, DOI 10.1901/jaba.1995.28-561
NR 16
TC 11
Z9 12
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD FAL
PY 2008
VL 41
IS 3
BP 351
EP 364
DI 10.1901/jaba.2008.41-351
PG 14
WC Psychology, Clinical
SC Psychology
GA 345AU
UT WOS:000258969200004
PM 18816974
ER
PT J
AU Taylor, BA
Hoch, H
AF Taylor, Bridget A.
Hoch, Hannah
TI Teaching children with autism to respond to and initiate bids for joint
attention
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; joint attention; social skills; teaching language
ID INTERVENTION; BEHAVIOR; COMMUNICATION; LANGUAGE; INFANT
AB A multiple baseline design across 3 children with autism was used to assess the effects of prompting and social reinforcement to teach participants to respond to an adult's bid for joint attention and to initiate bids for joint attention. Participants were taught to respond to an adult's bid for joint attention by looking in the direction of an object at which the adult pointed, by making a comment about the object, and by looking back at the adult. Additional training and reinforcement were needed to teach the participants to initiate bids for joint attention. Findings are discussed in terms of the social relevance of reaching children with autism to respond to and initiate bids for joint attention.
C1 [Taylor, Bridget A.; Hoch, Hannah] Alpine Learning Grp, Paramus, NJ 07652 USA.
RP Taylor, BA (reprint author), Alpine Learning Grp, 777 Paramus Rd, Paramus, NJ 07652 USA.
EM btaylor@alpinelearninggroup.org
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NR 19
TC 24
Z9 25
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD FAL
PY 2008
VL 41
IS 3
BP 377
EP 391
DI 10.1901/jaba.2008.41-377
PG 15
WC Psychology, Clinical
SC Psychology
GA 345AU
UT WOS:000258969200006
PM 18816976
ER
PT J
AU Chan, JM
O'Reilly, MF
AF Chan, Jeffrey M.
O'Reilly, Mark F.
TI A Social Stories (TM) intervention package for students with autism in
inclusive classroom settings
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; inclusion classroom; role play; social skills training; Social
Stories
ID SOCIAL STORIES; CHILDREN; FEEDBACK; BEHAVIOR
AB A Social Stories (TM) intervention package was used to teach 2 Students with autism to read Social Stories, answer comprehension questions, and engage in role plays. Appropriate social behaviors increased and inappropriate behaviors decreased for both participants, and the effects were maintained for up to 10 months. This intervention package appears to be useful in inclusive classroom environments and does not require intensive Supervision of the child's behavior.
C1 [Chan, Jeffrey M.] Univ Texas Austin, Dept Special Educ, Austin, TX 78712 USA.
RP Chan, JM (reprint author), Univ Texas Austin, Dept Special Educ, 1 Univ Stn,D5300, Austin, TX 78712 USA.
EM jeffchan@mail.utexas.edu
CR Delano M, 2006, J POSIT BEHAV INTERV, V8, P29, DOI 10.1177/10983007060080010501
Digennaro FD, 2007, J APPL BEHAV ANAL, V40, P447, DOI 10.1901/jaba.2007.40-447
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HAYES SC, 2001, RELATIONAL FRAME THE
Lorimer PA, 2002, J POSIT BEHAV INTERV, V4, P53, DOI 10.1177/109830070200400109
Reynhout G, 2006, J AUTISM DEV DISORD, V36, P445, DOI 10.1007/s10803-006-0086-1
Sansosti FJ, 2006, J POSIT BEHAV INTERV, V8, P43, DOI 10.1177/10983007060080010601
Swaggart B. L., 1995, FOCUS AUTISTIC BEHAV, V10, P1
Thiemann KS, 2001, J APPL BEHAV ANAL, V34, P425, DOI 10.1901/jaba.2001.34-425
NR 12
TC 12
Z9 12
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD FAL
PY 2008
VL 41
IS 3
BP 405
EP 409
DI 10.1901/jaba.2008.41-405
PG 5
WC Psychology, Clinical
SC Psychology
GA 345AU
UT WOS:000258969200008
PM 18816978
ER
PT J
AU Ingvarsson, ET
Kahng, S
Hausman, NL
AF Ingvarsson, Einar T.
Kahng, SungWoo
Hausman, Nicole L.
TI Some effects of noncontingent positive reinforcement on multiply
controlled problem behavior and compliance in a demand context
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; escape behavior; noncontingent reinforcement; problem behavior;
reinforcement density
ID MAINTAINED DESTRUCTIVE BEHAVIOR; SELF-INJURY; ESCAPE; DISABILITIES
AB Functional analysis suggested that the problem behavior of an 8-year-old girl with autism was maintained by escape from demands and access to edible items. Noncontingent delivery of an edible item was sufficient to increase compliance and reduce the rate of problem behavior without the use of escape extinction in a demand context. Leaner and richer schedules of noncontingent reinforcement were equally effective, and there were minimal differences between noncontingent reinforcement and differential reinforcement of compliance.
C1 [Ingvarsson, Einar T.] Youngstown State Univ, Dept Psychol, Youngstown, OH 44555 USA.
[Kahng, SungWoo] Johns Hopkins Univ, Sch Med, Baltimore, MD 21218 USA.
[Kahng, SungWoo; Hausman, Nicole L.] Kennedy Krieger Inst, Baltimore, MD 21205 USA.
RP Ingvarsson, ET (reprint author), Youngstown State Univ, Dept Psychol, DeBartolo Hall,1 Univ Plaza, Youngstown, OH 44555 USA.
EM etingvarsson@ysu.edu
RI Kahng, SungWoo/A-9994-2009
CR CAUTELA JR, 1984, J BEHAV THER EXP PSY, V15, P109, DOI 10.1016/0005-7916(84)90004-1
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GOH HL, 1994, J APPL BEHAV ANAL, V27, P173, DOI 10.1901/jaba.1994.27-173
HAGOPIAN LP, 1994, J APPL BEHAV ANAL, V27, P317, DOI 10.1901/jaba.1994.27-317
IWATA BA, 1994, J APPL BEHAV ANAL, V27, P215, DOI 10.1901/jaba.1994.27-215
IWATA BA, 1994, J APPL BEHAV ANAL, V27, P197, DOI 10.1901/jaba.1994.27-197
Lalli JS, 1999, J APPL BEHAV ANAL, V32, P285, DOI 10.1901/jaba.1999.32-285
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Wilder DA, 2005, J APPL BEHAV ANAL, V38, P549, DOI 10.1901/jaba.2005.132-04
NR 10
TC 15
Z9 15
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD FAL
PY 2008
VL 41
IS 3
BP 435
EP 440
DI 10.1901/jaba.2008.41-435
PG 6
WC Psychology, Clinical
SC Psychology
GA 345AU
UT WOS:000258969200013
PM 18816983
ER
PT J
AU Lang, R
O'Reilly, M
Machalicek, W
Lancioni, G
Rispoli, M
Chan, JM
AF Lang, Russell
O'Reilly, Mark
Machalicek, Wendy
Lancioni, Giulio
Rispoli, Mandy
Chan, Jeffrey M.
TI A preliminary comparison of functional analysis results when conducted
in contrived versus natural settings
SO JOURNAL OF APPLIED BEHAVIOR ANALYSIS
LA English
DT Article
DE autism; contrived settings; functional analysis; natural settings;
assessment
ID DESTRUCTIVE BEHAVIOR
AB A preliminary evaluation of the correspondence between functional analysis outcomes across setting; was conducted with 2 children who had been diagnosed with autism and who engaged in challenging behavior. Differences across settings (a therapy room and a classroom) were demonstrated in ABAB reversal designs. Three potential patterns of results that may occur when comparing functional analyses across environments are described, and one possible explanation for the occurrence of discrepancies between environments (differing learning histories within separate environments) is offered.
C1 [Lang, Russell] Univ Texas Austin, Dept Special Educ, Austin, TX 78712 USA.
[Lancioni, Giulio] Univ Bari, I-70121 Bari, Italy.
RP Lang, R (reprint author), Univ Texas Austin, Dept Special Educ, 1 Univ Stn D5300, Austin, TX 78712 USA.
EM russlang@mail.utexas.edu
CR Bowman LG, 1997, J APPL BEHAV ANAL, V30, P251, DOI 10.1901/jaba.1997.30-251
Carr EG, 1997, J APPL BEHAV ANAL, V30, P673, DOI 10.1901/jaba.1997.30-673
Fisher WW, 1998, J APPL BEHAV ANAL, V31, P513, DOI 10.1901/jaba.1998.31-513
Hanley GP, 2003, J APPL BEHAV ANAL, V36, P147, DOI 10.1901/jaba.2003.36-147
Iwata B A, 1994, J Appl Behav Anal, V27, P131, DOI 10.1901/jaba.1994.27-131
IWATA BA, 1994, J APPL BEHAV ANAL, V27, P197, DOI 10.1901/jaba.1994.27-197
Ringdahl JE, 2000, J APPL BEHAV ANAL, V33, P247, DOI 10.1901/jaba.2000.33-247
Tiger J. H., 2006, ED TREATMENT CHILDRE, V29, P107
Van Camp CM, 2000, J APPL BEHAV ANAL, V33, P207, DOI 10.1901/jaba.2000.33-207
NR 9
TC 11
Z9 11
PU JOURNAL APPL BEHAV ANAL
PI LAWRENCE
PA DEPT HUMAN DEVELOPMENT, UNIV KANSAS, LAWRENCE, KS 66045 USA
SN 0021-8855
J9 J APPL BEHAV ANAL
JI J. Appl. Behav. Anal.
PD FAL
PY 2008
VL 41
IS 3
BP 441
EP 445
DI 10.1901/jaba.2008.41-441
PG 5
WC Psychology, Clinical
SC Psychology
GA 345AU
UT WOS:000258969200014
PM 18816984
ER
PT J
AU Nyden, A
Myren, KJ
Gillberg, C
AF Nyden, Agneta
Myren, Karl-Johan
Gillberg, Christopher
TI Long-Term Psychosocial and Health Economy Consequences of ADHD, Autism,
and Reading-Writing Disorder A Prospective Service Evaluation Project
SO JOURNAL OF ATTENTION DISORDERS
LA English
DT Article
DE ADHD; autism; costs; health economy; psychosocial outcome
AB Objective: The study aims to evaluate psychosocial, societal, and family cost consequences of a psychoeducational intervention program. Methods: Sixty boys with ADHD, Asperger syndrome/high-functioning autism (AS/HFA), and reading and writing disorder (RD/WD) were allocated to participate in a service evaluation project. Every other boy in each diagnostic group was randomly allocated to receive either (a) a special education program (clinical index group) or (b) follow-up without the special education program (clinical comparison group). Nine years after initial assessments the stability of the psychosocial and economic resource consequences over time was studied. Results: ADHD, AS/HFA, and RD/WD all had severe impact on family life quality. The societal costs were high, but no significant differences in resource use or in total costs were found between the clinical index and the comparison groups. Conclusions: The results underscore the very long-term need for support including individually tailored reevaluations and carefully monitored intervention programs adapted to family needs and severity of child disorder. (J. of Att. Dis. 2008; 12(2) 141-148)
C1 [Gillberg, Christopher] Univ Gothenburg, S-41119 Gothenburg, Sweden.
RP Nyden, A (reprint author), Univ Gothenburg, Kungsgatan 12, S-41119 Gothenburg, Sweden.
EM agneta.nyden@vgregion.se
FU Eli Lilly, Sweden AB; Swedish Inheritance Fund for Christopher Gillberg
FX This study was supported by grants from the Eli Lilly, Sweden AB, Box
721, 169 27 Solna, Sweden. Disclosures: The study was partly funded by
grant from the Swedish Inheritance Fund for Christopher Gillberg, and
grants for Agneta Nyden and Karl Johan Myren by Eli Lilly, Sweden.
Address correspondence to Agneta Nyden, Child and Adolescent Psychiatry,
University of Goteborg, Kungsgatan 12, 411 19 Goteborg, Sweden; e-mail:
agneta.nyden@vgregion.se.
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NR 27
TC 5
Z9 5
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1087-0547
J9 J ATTEN DISORD
JI J. Atten. Disord.
PD SEP
PY 2008
VL 12
IS 2
BP 141
EP 148
DI 10.1177/1087054707306116
PG 8
WC Psychology, Developmental; Psychiatry
SC Psychology; Psychiatry
GA V11GT
UT WOS:000207520800004
PM 17968030
ER
PT J
AU Goldberg, MC
Mostow, AJ
Vecera, SP
Larson, JCG
Mostofsky, SH
Mahone, EM
Denckla, MB
AF Goldberg, Melissa C.
Mostow, Allison J.
Vecera, Shaun P.
Larson, Jennifer C. Gidley
Mostofsky, Stewart H.
Mahone, E. Mark
Denckla, Martha B.
TI Evidence for impairments in using static line drawings of eye gaze cues
to orient visual-spatial attention in children with high functioning
autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE eye gaze; high functioning autism; orienting; attention; cueing
ID FRONTAL-LOBE DAMAGE; EXECUTIVE FUNCTIONS; CHILDHOOD AUTISM; SOCIAL
ATTENTION; DIRECTION CUES; DEFICITS; SHIFTS; PERCEPTION; INFANTS; ARROW
AB We examined the ability to use static line drawings of eye gaze cues to orient visual-spatial attention in children with high functioning autism (HFA) compared to typically developing children (TD). The task was organized such that on valid trials, gaze cues were directed toward the same spatial location as the appearance of an upcoming target, while on invalid trials gaze cues were directed to an opposite location. Unlike TD children, children with HFA showed no advantage in reaction time (RT) on valid trials compared to invalid trials (i.e., no significant validity effect). The two stimulus onset asynchronies (200 ms, 700 ms) did not differentially affect these findings. The results suggest that children with HFA show impairments in utilizing static line drawings of gaze cues to orient visual-spatial attention.
C1 [Goldberg, Melissa C.; Larson, Jennifer C. Gidley; Mostofsky, Stewart H.; Mahone, E. Mark; Denckla, Martha B.] Kennedy Krieger Inst, Baltimore, MD 21205 USA.
[Mostow, Allison J.] Kenney Krieger Inst, Baltimore, MD 21231 USA.
[Goldberg, Melissa C.; Mostofsky, Stewart H.; Mahone, E. Mark; Denckla, Martha B.] Johns Hopkins Univ, Sch Med, Baltimore, MD USA.
[Vecera, Shaun P.] Univ Iowa, Iowa City, IA USA.
RP Goldberg, MC (reprint author), Kennedy Krieger Inst, 707 N Broadway,Room 232, Baltimore, MD 21205 USA.
EM goldbergm@kennedykrieger.org
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NR 50
TC 14
Z9 14
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1405
EP 1413
DI 10.1007/s10803-007-0506-x
PG 9
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400001
PM 18074212
ER
PT J
AU Allison, C
Baron-Cohen, S
Wheelwright, S
Charman, T
Richler, J
Pasco, G
Brayne, C
AF Allison, Carrie
Baron-Cohen, Simon
Wheelwright, Sally
Charman, Tony
Richler, Jennifer
Pasco, Greg
Brayne, Carol
TI The Q-CHAT (Quantitative CHecklist for autism in toddlers): A normally
distributed quantitative measure of autistic traits at 18-24 months of
age: Preliminary report
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE Autism spectrum conditions; Q-CHAT; childhood screening
ID PERVASIVE DEVELOPMENTAL DISORDERS; SPECTRUM QUOTIENT AQ;
PRESCHOOL-CHILDREN; SCREENING QUESTIONNAIRE; FUNCTIONING AUTISM;
GENERAL-POPULATION; ASPERGER-SYNDROME; FOLLOW-UP; PREVALENCE;
2-YEAR-OLDS
AB We report a major revision of the CHecklist for Autism in Toddlers (CHAT). This quantitative CHAT (Q-CHAT) contains 25 items, scored on a 5 point scale (0-4). The Q-CHAT was completed by parents of n = 779 unselected toddlers (mean age 21 months) and n = 160 toddlers and preschoolers (mean age 44 months) with an Autism Spectrum Condition (ASC). The ASC group (mean (SD) = 51.8 (14.3)) scored higher on the Q-CHAT than controls (26.7 (7.8)). Boys in the control group (27.5 (7.8)) scored higher than girls (25.8 (7.7)). The intraclass correlation for test-retest reliability was 0.82 (n = 330). The distribution in the control group was close to normal. Full examination of the clinical validity of the Q-CHAT and test properties is underway.
C1 [Allison, Carrie; Baron-Cohen, Simon; Wheelwright, Sally; Richler, Jennifer; Pasco, Greg] Univ Cambridge, Dept Psychiat, Autism Res Ctr, Cambridge CB2 8AH, England.
[Charman, Tony] UCL Inst Child Hlth, London, England.
[Brayne, Carol] Univ Cambridge, Dept Publ Hlth & Primary Care, Cambridge, England.
RP Allison, C (reprint author), Univ Cambridge, Dept Psychiat, Autism Res Ctr, Douglas House,18B Trumpington Rd, Cambridge CB2 8AH, England.
EM cla29@cam.ac.uk
RI Charman, Tony/A-2085-2014
OI Charman, Tony/0000-0003-1993-6549
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
AUYEUNG B, J AUTISM DE IN PRESS
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NR 56
TC 56
Z9 56
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
EI 1573-3432
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1414
EP 1425
DI 10.1007/s10803-007-0509-7
PG 12
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400002
PM 18240013
ER
PT J
AU Luyster, RJ
Kadlec, MB
Carter, A
Tager-Flusberg, H
AF Luyster, Rhiannon J.
Kadlec, Mary Beth
Carter, Alice
Tager-Flusberg, Helen
TI Language assessment and development in toddlers with autism spectrum
disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE language; toddlers; early childhood; development; assessment
ID DIAGNOSTIC OBSERVATION SCHEDULE; JOINT ATTENTION; YOUNG-CHILDREN;
PRESCHOOL-CHILDREN; COMMUNICATIVE DEVELOPMENT; PRELINGUISTIC PREDICTORS;
MOTOR CORTEX; 2ND YEAR; IMITATION; PLAY
AB One of the primary diagnostic criteria for the diagnosis of autism spectrum disorders (ASD) is the presence of a language delay or impairment. Children with ASD are now being identified at significantly younger ages, and prior research has consistently found that early language skills in this population are heterogeneous and an important predictor for later outcome. The goal of this study was to systematically investigate language in toddlers with ASD and to identify early correlates of receptive and expressive language in this population. The study included 164 toddlers with ASD between the ages of 18 and 33 months who were evaluated on several cognitive, language and behavioral measures. Results suggested good agreement among different measures of early language, including direct assessment and parent report measures. Significant concurrent predictors of receptive language included gestures, non-verbal cognitive ability and response to joint attention. For expressive language, the most significant predictors were non-verbal cognitive ability, gestures and imitation. These findings have important implications for intervention programs targeting this population.
C1 [Kadlec, Mary Beth; Tager-Flusberg, Helen] Boston Univ, Sch Med, Dept Anat & Neurobiol, Boston, MA 02118 USA.
[Luyster, Rhiannon J.] Autism Consortium, Boston, MA USA.
[Carter, Alice] Univ Massachusetts, Boston, MA 02125 USA.
RP Tager-Flusberg, H (reprint author), Boston Univ, Sch Med, Dept Anat & Neurobiol, 715 Albany St,L-814, Boston, MA 02118 USA.
EM htagerf@bu.edu
RI Tager-Flusberg, Helen/D-5265-2009
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NR 83
TC 78
Z9 79
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1426
EP 1438
DI 10.1007/s10803-007-0510-1
PG 13
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400003
PM 18188685
ER
PT J
AU Honey, E
McConachie, H
Randle, V
Shearer, H
Le Couteur, AS
AF Honey, Emma
McConachie, Helen
Randle, Val
Shearer, Heather
Le Couteur, Ann S.
TI One-year change in repetitive behaviours in young children with
communication disorders including autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE longitudinal; repetitive behaviour; autism spectrum disorder; ability;
early identification; ADI-R
ID PERVASIVE DEVELOPMENTAL DISORDERS; FOLLOW-UP; SPECTRUM DISORDERS;
PRESCHOOL-CHILDREN; ASPERGER-SYNDROME; INFANTILE-AUTISM; DIAGNOSIS; AGE;
ABNORMALITIES; CHILDHOOD
AB Repetitive behaviours are a relatively neglected area of study in autism. Previous research has concluded that repetitive behaviour is inversely related to ability and that it tends to increase over the preschool years. One-hundred and four children ages 24-48 months, with autism, autism spectrum disorder (ASD) or other disorders, were followed for 13 months. Twelve items from the Autism Diagnostic Interview (ADI-R) were analysed, as well as diagnostic algorithm scores. Ability was related to degree of repetitive behaviours, except for one cluster of relatively able children. ADI-R repetitive behaviour algorithm scores increased over time; however, when all 12 behaviours were considered, there was a general decrease in impact upon the child's and family's activities. Reasons for this decrease are discussed.
C1 [McConachie, Helen; Randle, Val; Le Couteur, Ann S.] Univ Newcastle, Royal Victoria Infirm, Sir James Spence Inst, Sch Clin Med Sci, Newcastle NE1 4LP, NSW, England.
[Honey, Emma; Shearer, Heather] Univ Durham, Dept Psychol, Durham DH1 3LE, England.
RP McConachie, H (reprint author), Univ Newcastle, Royal Victoria Infirm, Sir James Spence Inst, Sch Clin Med Sci, Queen Victoria Rd, Newcastle NE1 4LP, NSW, England.
EM h.r.mcconachie@ncl.ac.uk
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NR 50
TC 16
Z9 16
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1439
EP 1450
DI 10.1007/s10803-006-0191-1
PG 12
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400004
PM 16900401
ER
PT J
AU Shamay-Tsoory, SG
AF Shamay-Tsoory, Simone G.
TI Recognition of 'fortune of others' emotions in asperger syndrome and
high functioning autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE asperger syndrome; fortune of others emotions; envy; schadenfreude
ID PREFRONTAL CORTEX; COMPLEX EMOTIONS; EMPATHY DEFICITS; JOINT ATTENTION;
REVISED VERSION; NORMAL-CHILDREN; NORMAL ADULTS; MIND; ENVY;
SCHADENFREUDE
AB 'Fortune of others' emotions, such as envy and gloating over the other's misfortune, are complex emotions experienced in situations where events are presumed to be desirable or undesirable for another person. The present paper explores the notion that individuals with AS and HFA are impaired in understanding of envy and gloating. We tested the ability of adults with AS/HFA to understand envy and gloating and compared their performance to that of age-matched healthy controls. The 'fortune of others' emotion task and an additional theory-of-mind (ToM) task were based on a task designed to assess ToM on the basis of eye gaze direction. Individuals with AS and HFA showed no difficulty on basic ToM conditions, but were impaired in their ability to identify envy and gloating. Furthermore, the ability to recognize these emotions was related to scores on a self-rating scale of perspective-taking ability and the ToM task.
C1 Univ Haifa, Dept Psychol, IL-31905 Haifa, Israel.
RP Shamay-Tsoory, SG (reprint author), Univ Haifa, Dept Psychol, IL-31905 Haifa, Israel.
EM sshamay@psy.haifa.ac.il
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NR 66
TC 18
Z9 19
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1451
EP 1461
DI 10.1007/s10803-007-0515-9
PG 11
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400005
PM 18161015
ER
PT J
AU Horrocks, JL
White, G
Roberts, L
AF Horrocks, Judy L.
White, George
Roberts, Laura
TI Principals' attitudes regarding inclusion of children with autism in
Pennsylvania public schools
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; principals; inclusion
ID HIGH-FUNCTIONING CHILDREN; SPECTRUM DISORDERS; EDUCATION; STUDENTS;
DISABILITIES; LONELINESS; TEACHERS
AB This study sought to identify the attitudes that principals held regarding the inclusion of students with disabilities, and the relationship between their attitudes and their placement recommendations for children with autism and to identify the relationship between specific demographic factors and attitudes toward inclusion and placement. A stratified random sample was drawn from the active list of 3,070 principals in the Pennsylvania public schools. From 1,500 surveys, 571 principal responses were received. The most significant factor in predicting both a positive attitude toward inclusion of children with disabilities and higher recommendations of placements for children with autism was the principal's belief that children with autism could be included in a regular education classroom.
C1 [Horrocks, Judy L.] Timothy Sch, Berwyn, PA 19312 USA.
[White, George] Lehigh Univ, Coll Educ, Bethlehem, PA 18015 USA.
[Roberts, Laura] Roberts Educ Res, Lansdale, PA USA.
RP Horrocks, JL (reprint author), Timothy Sch, 973 Old Lancaster Rd, Berwyn, PA 19312 USA.
EM jhorrocks@timothyschool.com
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NR 24
TC 13
Z9 13
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1462
EP 1473
DI 10.1007/s10803-007-0522-x
PG 12
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400006
PM 18256916
ER
PT J
AU Solomon, M
Ozonoff, S
Carter, C
Caplan, R
AF Solomon, Marjorie
Ozonoff, Sally
Carter, Cameron
Caplan, Rochelle
TI Formal thought disorder and the autism spectrum: Relationship with
symptoms, executive control, and anxiety
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism spectrum disorders; schizophrenia; thought disorder; executive
functions; anxiety
ID COMMUNICATION IMPAIRMENTS; 1ST-EPISODE SCHIZOPHRENIA; DEVELOPMENTAL
DISORDERS; AFFECTIVE REACTIVITY; ASPERGER-SYNDROME; CHILDREN; CHILDHOOD;
LANGUAGE; MIND; SYMPTOMATOLOGY
AB This study investigated whether children with autism spectrum disorders (ASDs) exhibit formal thought disorder (FTD), and whether this is related to ASD symptoms, executive control, and anxiety. Participants aged 8-17 with ASDs exhibited significantly more illogical thinking and loose associations than matched typically developing control subjects. In participants with ASDs, illogical thinking was related to aspects of cognitive functioning and to executive control. Loose associations were related to autism communication symptoms and to parent reports of stress and anxiety. When FTD is present in ASDs, it generally is not a co-morbid schizophrenia symptom, but is related to pragmatic language abnormalities found in ASDs. The clinical and neurobiological significance of this work is discussed.
C1 [Solomon, Marjorie; Ozonoff, Sally] MIND Inst, Sacramento, CA 95817 USA.
[Solomon, Marjorie; Ozonoff, Sally; Carter, Cameron] Univ Calif Davis, Dept Psychiat & Behav Sci, Sacramento, CA 95817 USA.
[Solomon, Marjorie; Carter, Cameron] UC Davis Imaging Res Ctr, Sacramento, CA 95817 USA.
[Caplan, Rochelle] Univ Calif Los Angeles, Semel Inst Neurosci & Human Behav, Los Angeles, CA USA.
RP Solomon, M (reprint author), MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM marjorie.solomon@ucdmc.ucdavis.edu; sally.ozonoff@ucdmc.ucdavis.edu;
cameron.carter@ucdmc.ucdavis.edu
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NR 63
TC 29
Z9 29
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1474
EP 1484
DI 10.1007/s10803-007-0526-6
PG 11
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400007
PM 18297385
ER
PT J
AU Minshew, NJ
Hobson, JA
AF Minshew, Nancy J.
Hobson, Jessica A.
TI Sensory sensitivities and performance on sensory perceptual tasks in
high-functioning individuals with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; sensory sensitivities; sensory perception; sensory neglect
ID CHILDHOOD AUTISM; INFANTILE-AUTISM; DEVELOPMENTAL DISORDERS; SENTENCE
COMPREHENSION; SPECTRUM DISORDERS; DISTURBED-CHILDREN; MOTOR
INTEGRATION; ASPERGER-SYNDROME; ABNORMALITIES; CONNECTIVITY
AB Most reports of sensory symptoms in autism are second hand or observational, and there is little evidence of a neurological basis. Sixty individuals with high-functioning autism and 61 matched typical participants were administered a sensory questionnaire and neuropsychological tests of elementary and higher cortical sensory perception. Thirty-two percent of autism participants endorsed more sensory sensitivity items than any control participants. Both groups made few errors on elementary sensory perception items. Controls made few errors on higher cortical sensory perception items, but 30% of the autism participants made high numbers of errors. These findings support the common occurrence of sensory symptoms in high functioning autism based on first person report, and the presence of neurological abnormalities in higher cortical sensory perception.
C1 [Minshew, Nancy J.] Univ Pittsburgh, Sch Med, NICHD, Collaborat Program Excellence Autism, Pittsburgh, PA 15213 USA.
[Minshew, Nancy J.] Univ Pittsburgh, Sch Med, Dept Psychiat, Pittsburgh, PA 15213 USA.
[Minshew, Nancy J.] Univ Pittsburgh, Sch Med, Dept Neurol, Pittsburgh, PA 15213 USA.
[Hobson, Jessica A.] UCL, Inst Child Hlth, London, England.
RP Minshew, NJ (reprint author), Univ Pittsburgh, Sch Med, NICHD, Collaborat Program Excellence Autism, 3811 OHara St,Suite 300 Webster Hall, Pittsburgh, PA 15213 USA.
EM minshewnj@upmc.edu
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NR 85
TC 21
Z9 21
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1485
EP 1498
DI 10.1007/s10803-007-0528-4
PG 14
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400008
PM 18302014
ER
PT J
AU Shane, HC
Albert, PD
AF Shane, Howard C.
Albert, Patti Ducoff
TI Electronic screen media for persons with autism spectrum disorders:
Results of a survey
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; autism spectrum disorders; media; video modeling; observational
learning
ID CHILDREN; ACQUISITION; SPEECH; SKILLS
AB Social and anecdotal reports suggest a predilection for visual media among individuals on the autism spectrum, yet no formal investigation has explored the extent of that use. Using a distributed questionnaire design, parents and caregivers report on time allotted toward media, including observable behaviors and communicative responses. More time was spent engaged with electronic screen media (ESM) than any other leisure activity. Television and movie viewing was more popular than computer usage. Across media platforms, animated programs were more highly preferred. Prevalent verbal and physical imitation was reported to occur during and following exposure to ESM. Clinical implications to strategically incorporate ESM into learning approaches for children with autism spectrum disorders (ASD) are provided.
C1 [Albert, Patti Ducoff] Childrens Hosp Boston, Hartford, CT USA.
[Shane, Howard C.] Harvard Univ, Sch Med, Boston, MA 02115 USA.
[Shane, Howard C.] MGH Inst Hlth Profess, Boston, MA USA.
[Shane, Howard C.] Childrens Hosp, Ctr Commun Enhancement, Boston, MA 02115 USA.
RP Albert, PD (reprint author), Childrens Hosp Boston, Hartford, CT USA.
EM pdalbertslp@yahoo.com
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NR 28
TC 38
Z9 39
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1499
EP 1508
DI 10.1007/s10803-007-0527-5
PG 10
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400009
PM 18293074
ER
PT J
AU Bitterman, A
Daley, TC
Misra, S
Carlson, E
Markowitz, J
AF Bitterman, Amy
Daley, Tamara C.
Misra, Sunil
Carlson, Elaine
Markowitz, Joy
TI A national sample of preschoolers with autism spectrum disorders:
Special education services and parent satisfaction
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; ASD; IDEA; preschoolers; services; parent satisfaction
ID YOUNG-CHILDREN; SOCIAL VALIDATION; DOWN-SYNDROME; INTERVENTION;
PROGRAMS; BEHAVIOR
AB The Pre-Elementary Education Longitudinal Study (PEELS) examines the preschool and early elementary school experiences of a nationally representative sample of 3,104 children ages 3-5 with disabilities from 2004 through 2009. This paper describes the special education and related services received by a subsample of 186 preschoolers with autism spectrum disorders (ASD) in 2003-2004 and parental satisfaction with those services. Past research and patterns of litigation suggest that parents of children with ASD are not wholly satisfied with the special education and related services their children receive. In the current study, the authors found many similarities between children with ASD and children with other disabilities in the type of services received under IDEA and in parent satisfaction with these services. Still, some significant differences emerged in the number of services received, the amount of time children with ASD spent in special education settings, and parent satisfaction with the amount of time children spent with typically developing peers. Implications about the importance of parent satisfaction and social validity measures are discussed.
C1 [Bitterman, Amy; Daley, Tamara C.; Misra, Sunil; Carlson, Elaine; Markowitz, Joy] Westat Corp, Rockville, MD 20850 USA.
RP Carlson, E (reprint author), Westat Corp, 1650 Res Blvd, Rockville, MD 20850 USA.
EM elainecarlson@westat.com
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NR 37
TC 34
Z9 34
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1509
EP 1517
DI 10.1007/s10803-007-0531-9
PG 9
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400010
PM 18228122
ER
PT J
AU Watt, N
Wetherby, AM
Barber, A
Morgan, L
AF Watt, Nola
Wetherby, Amy M.
Barber, Angie
Morgan, Lindee
TI Repetitive and stereotyped behaviors in children with autism spectrum
disorders in the second year of life
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE repetitive stereotyped behavior; autism spectrum disorder; second year
ID NORMAL HUMAN INFANTS; PREDICTIVE-VALIDITY; YOUNG-CHILDREN; DEVELOPMENTAL
PROFILE; DIAGNOSTIC INTERVIEW; FOLLOW-UP; COMMUNICATION; AGE;
RELIABILITY; GENERALIZABILITY
AB This study examined repetitive and stereotyped behaviors (RSB) in children with autism spectrum disorders (ASD, n = 50), developmental delays without ASD (DD; n = 25) and typical development (TD, n = 50) between 18 and 24 months of age. Children with ASD demonstrated significantly higher frequency and longer duration of RSB with objects, body, and sensory behaviors during a systematic behavior sample than both the DD and TD groups. RSB with objects were related to concurrent measures of symbolic capacity and social competence in the second year and predicted developmental outcomes as well as severity of autism symptoms at 3 years in children with communication delays. RSB in the second year appear to be important for early identification and prediction of developmental outcomes.
C1 [Watt, Nola] Univ Witwatersrand, Sch Human & Community Dev, Discipline Speech Pathol & Audiol, ZA-2050 Johannesburg, Gauteng, South Africa.
[Wetherby, Amy M.; Barber, Angie; Morgan, Lindee] Florida State Univ, Dept Commun Disorders, Tallahassee, FL 32306 USA.
RP Watt, N (reprint author), Univ Witwatersrand, Sch Human & Community Dev, Discipline Speech Pathol & Audiol, 1 Jan Smuts Ave,Private Bag 3, ZA-2050 Johannesburg, Gauteng, South Africa.
EM nola.watt@wits.ac.za
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NR 48
TC 44
Z9 46
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1518
EP 1533
DI 10.1007/s10803-007-0532-8
PG 16
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400011
PM 18266099
ER
PT J
AU Golan, O
Baron-Cohen, S
Golan, Y
AF Golan, Ofer
Baron-Cohen, Simon
Golan, Yael
TI The 'Reading the Mind in Films' task [child version]: Complex emotion
and mental state recognition in children with and without autism
spectrum conditions
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE emotion recognition; complex emotions; empathy; social cognition;
childhood; autism spectrum
ID ASPERGER-SYNDROME; DEVELOPMENTAL-CHANGE; FACIAL EXPRESSION; NORMAL
ADULTS; FACE; INFORMATION; PERCEPTION; DISORDERS; ABILITIES; DECEPTION
AB Children with autism spectrum conditions (ASC) have difficulties recognizing others' emotions. Research has mostly focused on basic emotion recognition, devoid of context. This study reports the results of a new task, assessing recognition of complex emotions and mental states in social contexts. An ASC group (n = 23) was compared to a general population control group (n = 24). Children with ASC performed lower than controls on the task. Using task scores, more than 87% of the participants were allocated to their group. This new test quantifies complex emotion and mental state recognition in life-like situations. Our findings reveal that children with ASC have residual difficulties in this aspect of empathy. The use of language-based compensatory strategies for emotion recognition is discussed.
C1 [Golan, Ofer] Bar Ilan Univ, Dept Psychol, IL-52900 Ramat Gan, Israel.
[Golan, Ofer; Baron-Cohen, Simon] Univ Cambridge, Dept Psychiat, Autism Res Ctr, Cambridge, England.
[Golan, Yael] Univ Cambridge, Fac Educ, Cambridge, England.
RP Golan, O (reprint author), Bar Ilan Univ, Dept Psychol, IL-52900 Ramat Gan, Israel.
EM golano1@mail.biu.ac.il
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NR 60
TC 37
Z9 38
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1534
EP 1541
DI 10.1007/s10803-007-0533-7
PG 8
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400012
PM 18311514
ER
PT J
AU Wallace, AE
Anderson, GM
Dubrow, R
AF Wallace, Anna E.
Anderson, George M.
Dubrow, Robert
TI Obstetric and parental psychiatric variables as potential predictors of
autism severity
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; preeclampsia; psychiatric; obstetric; hypertension; depression
ID PERVASIVE DEVELOPMENTAL DISORDER; RIGID-COMPULSIVE BEHAVIORS; SEROTONIN
TRANSPORTER GENE; DIAGNOSTIC INTERVIEW; INFANTILE-AUTISM; EPIDEMIOLOGIC
SURVEY; POSTNATAL FACTORS; RISK-FACTORS; CHILDREN; ASSOCIATION
AB Associations between obstetric and parental psychiatric variables and subjects' Autism Diagnostic Interview-Revised (ADI-R) and Autism Diagnostic Observation Schedule (ADOS) domain scores were examined using linear mixed effects models. Data for the 228 families studied were provided by the Autism Genetic Resource Exchange. Hypertension (P = 0.002), preeclampsia (P = 0.021) and generalized edema (P = 0.011) were associated with higher ADI-R communication scores. Hypertension (P = 0.011), albuminuria (P = 0.039) and generalized edema (P = 0.009) were associated with higher ADI-R repetitive behaviors scores. Parent depression was associated with higher ADI-R repetitive behaviors scores (P = 0.005), and parent anxiety with lower ADOS social/communication composite scores (P = 0.025). The associations between hypertension-related obstetric conditions and autistic severity warrant further investigation and raise intriguing questions regarding potential causal and modifying factors in autism.
C1 [Wallace, Anna E.; Dubrow, Robert] Yale Univ, Sch Publ Hlth, New Haven, CT USA.
[Anderson, George M.] Yale Univ, Sch Med, Ctr Child Study, New Haven, CT 06510 USA.
RP Wallace, AE (reprint author), HealthCore Inc, 800 Delaware Ave,5th Floor, Wilmington, DE 19801 USA.
EM awallace@healthcore.com
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NR 47
TC 13
Z9 13
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1542
EP 1554
DI 10.1007/s10803-007-0536-4
PG 13
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400013
PM 18324467
ER
PT J
AU Hoekstra, RA
Bartels, M
Cath, DC
Boomsma, DI
AF Hoekstra, Rosa A.
Bartels, Meike
Cath, Danielle C.
Boomsma, Dorret I.
TI Factor structure, reliability and criterion validity of the
autism-spectrum quotient (AQ): A study in dutch population and patient
groups
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; factor analysis; validity; reliability; autism phenotype
ID PERVASIVE DEVELOPMENTAL DISORDERS; GENERAL-POPULATION; COMPULSIVE
BEHAVIORS; FAMILY HISTORY; PHENOTYPE; TRAITS; IMPAIRMENT; CHILDREN;
PARENTS; VERSION
AB The factor structure of the Dutch translation of the Autism-Spectrum Quotient (AQ; a continuous, quantitative measure of autistic traits) was evaluated with confirmatory factor analyses in a large general population and student sample. The criterion validity of the AQ was examined in three matched patient groups (autism spectrum conditions (ASC), social anxiety disorder, and obsessive-compulsive disorder). A two factor model, consisting of a "Social interaction" factor and "Attention to detail" factor could be identified. The internal consistency and test-retest reliability of the AQ were satisfactory. High total AQ and factor scores were specific to ASC patients. Men scored higher than women and science students higher than non-science students. The Dutch translation of the AQ is a reliable instrument to assess autism spectrum conditions.
C1 [Hoekstra, Rosa A.] Univ Cambridge, Dept Dev Psychiat, Autism Res Ctr, Cambridge CB2 8AH, England.
[Hoekstra, Rosa A.; Bartels, Meike; Boomsma, Dorret I.] Vrije Univ Amsterdam, Dept Biol Psychol, Amsterdam, Netherlands.
[Cath, Danielle C.] GGZ Buitenamstel, Dept Psychiat, Amsterdam, Netherlands.
RP Hoekstra, RA (reprint author), Univ Cambridge, Dept Dev Psychiat, Autism Res Ctr, Douglas House,18B Trumpington Rd, Cambridge CB2 8AH, England.
EM rah58@medschl.cam.ac.uk
RI Hoekstra, Rosa/G-2410-2011; Bartels, Meike/D-4492-2014
OI Bartels, Meike/0000-0002-9667-7555
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 37
TC 127
Z9 127
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1555
EP 1566
DI 10.1007/s10803-008-0538-x
PG 12
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400014
PM 18302013
ER
PT J
AU Kopra, K
von Wendt, L
Wendt, TNV
Paavonen, EJ
AF Kopra, Kristiina
von Wendt, Lennart
Wendt, Taina Nieminen-von
Paavonen, E. Juulia
TI Comparison of diagnostic methods for asperger syndrome
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE asperger syndrome; diagnostic criteria; sensitivity; specificity
ID DSM-IV; TOTAL POPULATION; AUTISM; DISORDER; CHILDREN; CRITERIA
AB Several different diagnostic sets of criteria exist for Asperger syndrome (AS), but there is no agreement on a gold standard. The aim of this study was to compare four diagnostic sets of criteria for AS: the ICD-10, the DSM-IV, the Gillberg & Gillberg, and the Szatmari criteria. The series consists of 36 children who had been referred to two centers with a tentative diagnosis of AS. The best agreement was between the ICD-10 and the DSM-IV criteria (Kappa coefficient 0.48), and the lowest between the Gillberg & Gillberg and Szatmari criteria (Kappa coefficient -0.21). The poor agreement between these sets of diagnostic criteria compromises the comparability of studies on AS.
C1 [Paavonen, E. Juulia] Natl Res & Dev Ctr Welf & Hlth STAKES, Helsinki 00531, Finland.
[Kopra, Kristiina; von Wendt, Lennart] HUCH Hosp Children & Adolescents, Dept Child Neurol, Helsinki, Finland.
[Wendt, Taina Nieminen-von] Neuropsychiat Rehabil Ctr Neuromental, Helsinki, Finland.
[Wendt, Taina Nieminen-von] Jorvi Hosp, Dept Adolescent Psychiat, SF-02740 Espoo, Finland.
[Paavonen, E. Juulia] Univ Helsinki, Dept Child Psychiat, Helsinki, Finland.
RP Paavonen, EJ (reprint author), Natl Res & Dev Ctr Welf & Hlth STAKES, POB 220,Lintulahdenkuja 4, Helsinki 00531, Finland.
EM juulia.paavonen@helsinki.fi
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NR 21
TC 9
Z9 10
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1567
EP 1573
DI 10.1007/s10803-008-0537-y
PG 7
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400015
PM 18324466
ER
PT J
AU Saalasti, S
Lepistoe, T
Toppila, E
Kujala, T
Laakso, M
Wendt, TNV
von Wendt, L
Jansson-Verkasalo, E
AF Saalasti, Satu
Lepistoe, Tuulia
Toppila, Esko
Kujala, Teija
Laakso, Minna
Wendt, Taina Nieminen-von
von Wendt, Lennart
Jansson-Verkasalo, Eira
TI Language abilities of children with asperger syndrome
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE asperger syndrome; language; comprehension of instructions; executive
dysfunction
ID HIGH-FUNCTIONING AUTISM; SCHOOL-AGE-CHILDREN; EVENT-RELATED POTENTIALS;
INDIVIDUALS; DISORDERS; SPECTRUM; ADULTS; IMPAIRMENTS; PERCEPTION;
SPEECH
AB Current diagnostic taxonomies (ICD-10, DSM-IV) emphasize normal acquisition of language in Asperger syndrome (AS). Although many linguistic sub-skills may be fairly normal in AS there are also contradictory findings. There are only few studies examining language skills of children with AS in detail. The aim of this study was to study language performance in children with AS and their age, sex and IQ matched controls. Children with AS had significantly lower scores in the subtest of Comprehension of Instructions. Results showed that although many linguistic skills may develop normally, comprehension of language may be affected in children with AS. The results suggest that receptive language processes should be studied in detail in children with AS.
C1 [Saalasti, Satu; Laakso, Minna] Univ Helsinki, Dept Speech Sci, FIN-00014 Helsinki, Finland.
[Saalasti, Satu; Lepistoe, Tuulia; Kujala, Teija; Jansson-Verkasalo, Eira] Univ Helsinki, Dept Psychol, Cognit Brain Res Unit, FIN-00014 Helsinki, Finland.
[Saalasti, Satu; Lepistoe, Tuulia; von Wendt, Lennart] Univ Helsinki, Cent Hosp, Dept Child Neurol, FIN-00014 Helsinki, Finland.
[Toppila, Esko] Finnish Inst Occupat Hlth, Helsinki, Finland.
[Wendt, Taina Nieminen-von] Jorvi Hosp, Dept Adolescent Psychiat, SF-02740 Espoo, Finland.
[Wendt, Taina Nieminen-von] Med Ctr Dextra, Helsinki, Finland.
[Jansson-Verkasalo, Eira] Univ Oulu, Fac Humanities, Oulu, Finland.
RP Saalasti, S (reprint author), Univ Helsinki, Dept Speech Sci, POB 9, FIN-00014 Helsinki, Finland.
EM satu.saalasti@helsinki.fi
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World Health Organization, 1993, INT CLASS DIS ICD 10
NR 46
TC 21
Z9 24
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1574
EP 1580
DI 10.1007/s10803-008-0540-3
PG 7
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400016
PM 18324464
ER
PT J
AU Nordahl, CW
Simon, TJ
Zierhut, C
Solomon, M
Rogers, SJ
Amaral, DG
AF Nordahl, Christine Wu
Simon, Tony J.
Zierhut, Cynthia
Solomon, Marjorie
Rogers, Sally J.
Amaral, David G.
TI Brief report: Methods for acquiring structural MRI data in very young
children with autism without the use of sedation
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE MRI; autism; natural sleep; sedation; children; toddlers
ID DIAGNOSTIC OBSERVATION SCHEDULE; NORMAL BRAIN-DEVELOPMENT; HEAD
CIRCUMFERENCE; NIH MRI; AGE; SPECTRUM; DISORDER
AB We describe a protocol with which we achieved a 93% success rate in acquiring high quality MRI scans without the use of sedation in 2.5-4.5 year old children with autism, developmental delays, and typical development. Our main strategy was to conduct MRIs during natural nocturnal sleep in the evenings after the child's normal bedtime. Alternatively, with some older and higher functioning children, the MRI was conducted while the child was awake and watching a video. Both strategies relied heavily on the creation of a child and family friendly MRI environment and the involvement of parents as collaborators in the project. Scanning very young children with autism, typical development, and developmental delays without the use of sedation or anesthesia was possible in the majority of cases.
C1 [Nordahl, Christine Wu; Simon, Tony J.; Zierhut, Cynthia; Solomon, Marjorie; Rogers, Sally J.; Amaral, David G.] Univ Calif Davis, MIND Inst, Sacramento, CA 95817 USA.
[Nordahl, Christine Wu; Simon, Tony J.; Solomon, Marjorie; Rogers, Sally J.; Amaral, David G.] Univ Calif Davis, Dept Psychiat & Behav Sci, Sacramento, CA 95817 USA.
RP Nordahl, CW (reprint author), Univ Calif Davis, MIND Inst, 2805 50th St, Sacramento, CA 95817 USA.
EM crswu@ucdavis.edu
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NR 19
TC 22
Z9 22
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1581
EP 1590
DI 10.1007/s10803-007-0514-x
PG 10
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400017
PM 18157624
ER
PT J
AU Mitchell, P
O'Keefe, K
AF Mitchell, Peter
O'Keefe, Kelly
TI Brief report: Do individuals with autism spectrum disorder think they
know their own minds?
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE self knowledge; theory of mind; estimations; subjectivity
ID CHILDREN; SELF
AB How much do individuals with autism spectrum disorder (ASD) think they know about their inner states? To find out, we asked 24 participants with ASD and 24 non-clinical participants to rate how well they knew about six topics of self knowledge; they also rated how well a comparison individual knew these things about them. Participants with ASD differed from the non-clinical participants in assigning about the same amount of knowledge to the comparison individual as to themselves. Non-clinical participants, in contrast, assigned relatively more knowledge to themselves. The findings are consistent with the possibility that individuals with ASD do not appreciate the value of having first-person privileged access to their own inner states.
C1 [Mitchell, Peter; O'Keefe, Kelly] Univ Nottingham, Sch Psychol, Nottingham NG7 2RD, England.
RP Mitchell, P (reprint author), Univ Nottingham, Sch Psychol, Univ Pk, Nottingham NG7 2RD, England.
EM peter.mitchell@nottingham.ac.uk
CR Banerjee R, 2006, SOC DEV, V15, P418, DOI 10.1111/j.1467-9507.2006.00349.x
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WRIGHT C, 2000, KNOWING OUR OWN MIND, P13
NR 16
TC 23
Z9 24
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1591
EP 1597
DI 10.1007/s10803-007-0530-x
PG 7
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400018
PM 18311515
ER
PT J
AU Campbell, JM
AF Campbell, Jonathan M.
TI Brief report: Reliability and validity of the shared activities
questionnaire as a measure of middle school students' attitudes toward
autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; peers; attitudes; validity; measurement
ID BEHAVIORAL INTENTIONS; CHILDRENS ATTITUDES; PEERS
AB The Shared Activities Questionnaire (SAQ) is a self-report measure of children's behavioral intentions towards peers with disabilities. The SAQ has been validated as a measure of elementary school students' attitudes towards peers with disabilities. In the present study, psychometric properties of the SAQ as a measure of middle school students' attitudes toward autism were examined in a sample of 1,007 students (M age = 12.95 years). Confirmatory factor analysis supported the three-factor structure found for elementary school students. Internal consistency reliability was excellent. Criterion-related validity was established by demonstrating strong and positive relationships with a measure of cognitive attitudes. The SAQ is a reliable and valid measure of middle school students' behavioral intentions towards autism.
C1 Univ Georgia, Dept Educ Psychol & Instruct Technol, Athens, GA 30602 USA.
RP Campbell, JM (reprint author), Univ Georgia, Dept Educ Psychol & Instruct Technol, 325-J Aderhold Hall, Athens, GA 30602 USA.
EM jmcmpbll@uga.edu
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NR 19
TC 5
Z9 5
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD SEP
PY 2008
VL 38
IS 8
BP 1598
EP 1604
DI 10.1007/s10803-007-0534-6
PG 7
WC Psychology, Developmental
SC Psychology
GA 338TI
UT WOS:000258531400019
PM 18293073
ER
PT J
AU Ward, LM
Pinto, T
Lawrence, S
Lawson, M
Bastepe, M
Juppner, H
Rauch, F
AF Ward, L. M.
Pinto, T.
Lawrence, S.
Lawson, M.
Bastepe, M.
Jueppner, H.
Rauch, F.
TI Hypothyroidism and Autism Combined with Pseudohypoparathyroidism in the
Absence of Albright's Hereditary Osteodystrophy and GNAS Imprinting
Changes: A Novel Clinical Syndrome?
SO JOURNAL OF BONE AND MINERAL RESEARCH
LA English
DT Meeting Abstract
CT 30th Annual Meeting of the
American-Society-for-Bone-and-Mineral-Research
CY SEP 12-16, 2008
CL Montreal, CANADA
SP Amer Soc Bone & Mineral Res
C1 [Ward, L. M.; Pinto, T.; Lawrence, S.; Lawson, M.] Childrens Hosp Eastern Ontario, Div Endocrinol, Ottawa, ON K1H 8L1, Canada.
[Bastepe, M.; Jueppner, H.] Massachusetts Gen Hosp, Endocrine Unit, Boston, MA 02114 USA.
[Rauch, F.] Shriners Hosp Children, Genet Unit, Montreal, PQ, Canada.
NR 0
TC 0
Z9 0
PU AMER SOC BONE & MINERAL RES
PI WASHINGTON
PA 2025 M ST, N W, STE 800, WASHINGTON, DC 20036-3309 USA
SN 0884-0431
J9 J BONE MINER RES
JI J. Bone Miner. Res.
PD SEP
PY 2008
VL 23
BP S183
EP S183
PG 1
WC Endocrinology & Metabolism
SC Endocrinology & Metabolism
GA 351FX
UT WOS:000259411001056
ER
PT J
AU Frye, RE
Zimmerman, AW
Shoffner, JN
AF Frye, Richard E.
Zimmerman, Andrew W.
Shoffner, John N.
TI Conflict of interest statement concerning "Developmental Regression and
Mitochondrial Dysfunction in a Child With Autism"
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Letter
C1 [Frye, Richard E.] Univ Texas Hlth Sci Ctr, Dept Pediat & Neurol, Houston, TX USA.
[Zimmerman, Andrew W.] Johns Hopkins Univ, Sch Med, Dept Neurol, Baltimore, MD 21205 USA.
[Zimmerman, Andrew W.] Johns Hopkins Univ, Sch Med, Dept Psychiat & Behav Sci, Baltimore, MD 21205 USA.
[Shoffner, John N.] LLC, Med Neurogenet, Atlanta, GA USA.
RP Frye, RE (reprint author), Univ Texas Hlth Sci Ctr, Dept Pediat & Neurol, Houston, TX USA.
CR Poling JS, 2006, J CHILD NEUROL, V21, P170, DOI 10.2310/7010.2006.00032
NR 1
TC 2
Z9 2
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD SEP
PY 2008
VL 23
IS 9
BP 1089
EP 1090
DI 10.1177/0883073808322336
PG 2
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 343GR
UT WOS:000258841800024
ER
PT J
AU Poling, JS
AF Poling, Jon S.
TI Correspondence on "Developmental Regression and Mitochondrial
Dysfunction in a Child with Autism"
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Letter
CR Poling JS, 2006, J CHILD NEUROL, V21, P170, DOI 10.2310/7010.2006.00032
NR 1
TC 1
Z9 1
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD SEP
PY 2008
VL 23
IS 9
BP 1089
EP 1089
DI 10.1177/0883073808322330
PG 1
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 343GR
UT WOS:000258841800023
ER
PT J
AU Brumback, RA
AF Brumback, Roger A.
TI The appalling Poling Saga
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Letter
ID AUTISM; VACCINES
RI Brumback, Roger/A-2404-2008
CR Frye RE, 2008, J CHILD NEUROL, V23, P1089, DOI 10.1177/0883073808322336
Offit PA, 2008, NEW ENGL J MED, V358, P2089, DOI 10.1056/NEJMp0802904
Offit PA, 2008, NEW ENGL J MED, V359, P656
Poling JS, 2008, J CHILD NEUROL, V23, P1089, DOI 10.1177/0883073808322330
Poling JS, 2008, NEW ENGL J MED, V359, P655, DOI 10.1056/NEJMc086269
Poling JS, 2006, J CHILD NEUROL, V21, P170, DOI 10.2310/7010.2006.00032
NR 6
TC 3
Z9 3
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD SEP
PY 2008
VL 23
IS 9
BP 1090
EP 1091
DI 10.1177/0883073808322333
PG 2
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 343GR
UT WOS:000258841800025
PM 18827280
ER
PT J
AU Magnee, MJCM
de Gelder, B
van Engeland, H
Kemner, C
AF Magnee, Maurice J. C. M.
de Gelder, Beatrice
van Engeland, Herman
Kemner, Chantal
TI Audiovisual speech integration in pervasive developmental disorder:
evidence from event-related potentials
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE multisensory integration; language and communication; autism; EEG;
visual; auditory
ID AUDITORY-VISUAL INTEGRATION; AUTISM; RECOGNITION; INFORMATION
AB Background: Integration of information from multiple sensory sources is an important prerequisite for successful social behavior, especially during face-to-face conversation. It has been suggested that communicative impairments among individuals with pervasive developmental disorders (PDD) might be caused by an inability to integrate synchronously presented visual and auditory cues. Method: We investigated audiovisual integration of speech stimuli among a group of high-functioning adult PDD individuals and age- and IQ-matched controls using electroencephalography, measuring both early pre-phonological, as well as late phonologically driven integration. Results: Pre-phonological AV interactions are intact, while AV interactions corresponding to more complex phonological processes are impaired in individuals with PDD. Conclusions: The present findings argue for a pattern of impairments on tasks related to complex audiovisual integration combined with relative sparing of low-level integrational abilities. This combination may very well contribute to the communicative disabilities which are typical for the disorder.
C1 [Magnee, Maurice J. C. M.; van Engeland, Herman; Kemner, Chantal] Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, NL-3584 CX Utrecht, Netherlands.
[Magnee, Maurice J. C. M.; de Gelder, Beatrice] Tilburg Univ, Lab Cognit & Affect Neurosci, NL-5000 LE Tilburg, Netherlands.
[Kemner, Chantal] Maastricht Univ, Fac Psychol, Sect Biol Dev Psychol, Maastricht, Netherlands.
[de Gelder, Beatrice] Massachusetts Gen Hosp, Martinos Ctr Biomed Imaging, Charlestown, MA USA.
[de Gelder, Beatrice] Harvard Univ, Sch Med, Charlestown, MA USA.
RP Magnee, MJCM (reprint author), Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Rudolf Magnus Inst Neurosci, B01-201,Heidelberglaan 100, NL-3584 CX Utrecht, Netherlands.
EM M.J.C.M.Magnee@umcutrecht.nl
FU Netherlands Organization for Scientific Research [402-01-094]
FX This research was funded by an Innovational Research Incentives grant of
the Netherlands Organization for Scientific Research (NWO; VIDI-scheme,
402-01-094) to Chantal Kemner.
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Besle J, 2004, EUR J NEUROSCI, V20, P2225, DOI 10.1111/j.1460-9568.2004.03670.x
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van der Smagt MJ, 2007, J AUTISM DEV DISORD, V37, P2014, DOI 10.1007/s10803-006-0346-0
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van Wassenhove V, 2005, P NATL ACAD SCI USA, V102, P1181, DOI 10.1073/pnas.0408949102
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NR 20
TC 27
Z9 27
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD SEP
PY 2008
VL 49
IS 9
BP 995
EP 1000
DI 10.1111/j.1469-7610.2008.01902.x
PG 6
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 342KL
UT WOS:000258782900012
PM 18492039
ER
PT J
AU Dykens, EM
Roof, E
AF Dykens, Elisabeth M.
Roof, Elizabeth
TI Behavior in Prader-Willi syndrome: relationship to genetic subtypes and
age
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE Prader-Willi syndrome; genetic subtypes; age; CYFIP1
ID MATERNAL UNIPARENTAL DISOMY; COMPULSIVE BEHAVIOR; MALADAPTIVE BEHAVIOR;
DELETION; PEOPLE; EXPRESSION; PHENOTYPE; ABILITIES; SYMPTOMS; AUTISM
AB Background: Some behavioral features of Prader-Willi syndrome (PWS) are associated with the major genetic subtypes of this disorder. While most agree that those with maternal uniparental disomy (UPD) have a distinctive cognitive and psychiatric profile, findings are more controversial regarding possible differences among persons who vary in paternal deletion size. Methods: Caregivers of 88 persons with PWS aged 5 to 51 years (M = 22 years) were administered measures of problem behavior, compulsivity, hyperphagia, and adaptive skills. The sample was well characterized as having relatively large, Type I (n = 26) or smaller, Type II (n = 29) deletions, or UPD (n = 33). Results: No significant behavioral differences were found between the Type I versus Type II deletion groups. Within each genetic subtype, however, differences emerged in how advancing age related to behavior. Although age did not emerge as a significant correlate of behavior in the Type II or UPD groups, in the Type I group age was consistently associated with lower problem behaviors, adaptive skills, and externalizing symptoms. Conclusion: Although differences between deletion subtypes were not found, significant within-subtype differences emerged in relationships between age and behavior. Negative associations between age and behavior in the Type I group only may relate to non-imprinted genes that are deleted in Type I but not Type II cases, including CYFIP1. Altered expression of CYFIP1 is seen in other developmental disabilities, including 15q disorders, and haploinsufficiency of CYFIP1 in Type I PWS cases may be associated with age-related phenotypic effects. Findings underscore the importance of a life-span perspective in phenotypic research.
C1 [Dykens, Elisabeth M.] Vanderbilt Univ, Vanderbilt Kennedy Ctr Res Human Dev, Dept Psychol & Human Dev, Nashville, TN 37203 USA.
RP Dykens, EM (reprint author), Vanderbilt Univ, Vanderbilt Kennedy Ctr Res Human Dev, Dept Psychol & Human Dev, 230 Appleton Pl, Nashville, TN 37203 USA.
EM elisabeth.dykens@vanderbilt.edu
FU NICHD [R01HD135681, P30HD15052]; Vanderbilt General Clinical Research
Center; Prader-Willi Syndrome Association (USA)
FX This research was supported by NICHD grant R01HD135681 to EMD, and
P30HD15052 to the Vanderbilt Kennedy Center, the Vanderbilt General
Clinical Research Center, and a pilot grant from the Prader-Willi
Syndrome Association (USA). The authors thank the families for their
enthusiastic participation, Merlin Butler, M. D., and Douglas Bittel,
Ph. D., for their subtyping of deletion cases, Elizabeth Pantino and
Rebecca Kossler for assistance in data collection, and Robert Hodapp,
Ph. D, for his helpful comments on previous drafts of this manuscript.
CR Achenbach TM, 2001, MANUAL CHILD BEHAV C
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Whittington J, 2004, PRADER WILLI SYNDROM
Zarcone J, 2007, J INTELL DISABIL RES, V51, P478, DOI 10.1111/j.1365-2788.2006.00916.x
NR 36
TC 33
Z9 34
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD SEP
PY 2008
VL 49
IS 9
BP 1001
EP 1008
DI 10.1111/j.1469-7610.2008.01913.x
PG 8
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 342KL
UT WOS:000258782900013
PM 18665884
ER
PT J
AU Tanguay, PE
AF Tanguay, Peter E.
TI The science and fiction of autism
SO JOURNAL OF CLINICAL PSYCHIATRY
LA English
DT Book Review
C1 [Tanguay, Peter E.] Univ Louisville, Sch Med, Louisville, KY 40292 USA.
RP Tanguay, PE (reprint author), Univ Louisville, Sch Med, Louisville, KY 40292 USA.
CR Schreibman L., 2007, SCI FICTION AUTISM
NR 1
TC 0
Z9 0
PU PHYSICIANS POSTGRADUATE PRESS
PI MEMPHIS
PA P O BOX 240008, MEMPHIS, TN 38124 USA
SN 0160-6689
J9 J CLIN PSYCHIAT
JI J. Clin. Psychiatry
PD SEP
PY 2008
VL 69
IS 9
BP 1503
EP 1504
PG 2
WC Psychology, Clinical; Psychiatry
SC Psychology; Psychiatry
GA 355NV
UT WOS:000259716500023
ER
PT J
AU Leslie, LK
Lambros, KM
Aarons, GA
Haine, RA
Hough, RL
AF Leslie, Laurel K.
Lambros, Katina M.
Aarons, Gregory A.
Haine, Rachel A.
Hough, Richard L.
TI School-Based Service Use by Youth With ADHD in Public-Sector Settings
SO JOURNAL OF EMOTIONAL AND BEHAVIORAL DISORDERS
LA English
DT Article
DE ADD/ADHD/autism disorders/disabilities; community services; mental
health; school-based services; mental health access issues; cross-system
collaboration
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; DEFICIT-HYPERACTIVITY
DISORDER; DIAGNOSTIC INTERVIEW SCHEDULE; MEDICATION ALGORITHM PROJECT;
CLINICAL-PRACTICE GUIDELINE; SPECIAL-EDUCATION SERVICES; CONSENSUS
CONFERENCE PANEL; TEST-RETEST RELIABILITY; MENTAL-HEALTH-PROGRAMS;
NATIONAL PERSPECTIVE
AB This study investigates rates and predictors of school-based services (SBSs) for 390 youth meeting criteria for Attention Deficit Hyperactivity Disorder and served in the San Diego public sectors. Only 60% of youth had received an Attention Deficit Hyperactivity Disorder diagnosis; these youth were younger, male, Caucasian (versus Latino), and active to public mental health and special education (Emotional Disturbance category) at enumeration of study participants. Higher rates of SBSs (64%) were revealed than in community samples. Only 26% accessed multimodal treatment including SBSs, medication, and mental health. In multivariate modeling, SBSs displayed a curvilinear relationship with age, which may explain previously conflicting results regarding that relationship. Youth with private insurance or receiving mental health or medication were more likely to receive SBSs. Gender, race/ethnicity, and caregiver education, health, and mental health were not related to SBSs use. These findings may reflect sample characteristics. Further exploration of factors influencing SBSs use in different populations is warranted.
C1 [Leslie, Laurel K.] Tufts Univ, Boston, MA 02111 USA.
[Lambros, Katina M.; Haine, Rachel A.] Rady Childrens Hosp, Res Ctr, Child & Adolescent Serv, San Diego, CA USA.
[Aarons, Gregory A.] Univ Calif San Diego, San Diego, CA 92103 USA.
[Hough, Richard L.] Univ New Mexico, Albuquerque, NM 87131 USA.
RP Leslie, LK (reprint author), Tufts Univ, Boston, MA 02111 USA.
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*US DEP ED, 1999, CHILDR ADD ADHD TOP
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NR 83
TC 5
Z9 5
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1063-4266
J9 J EMOT BEHAV DISORD
JI J. Emot. Behav. Disord.
PD SEP
PY 2008
VL 16
IS 3
BP 163
EP 177
DI 10.1177/1063426608314290
PG 15
WC Education, Special; Psychology, Educational; Psychology,
Multidisciplinary
SC Education & Educational Research; Psychology
GA 394AA
UT WOS:000262416400003
ER
PT J
AU Lopez-Wagner, MC
Hoffman, CD
Sweeney, DP
Hodge, D
Gilliam, JE
AF Lopez-Wagner, Muriel C.
Hoffman, Charles D.
Sweeney, Dwight P.
Hodge, Danelle
Gilliam, James E.
TI Sleep problems of parents of typically developing children and parents
of children with autism
SO JOURNAL OF GENETIC PSYCHOLOGY
LA English
DT Article
DE autism; parents; sleep problems
ID SEVERE LEARNING-DISABILITIES; QUALITY INDEX; SCHOOL PERFORMANCE; MENTAL
HANDICAP; RATING-SCALE; STRESS; DISORDERS; FAMILIES; DISTURBANCES;
INSTRUMENT
AB Few researchers have investigated the relation of children's sleep problems to their parents' sleep problems. Children with autism have been reported to evidence greater sleep problems than do typically developing children (C. D. Hoffman, D. P. Sweeney, J. E. Gilliam, & M. C. Lopez-Wagner, 2006: P. G. William, L. L. Sears, & A. Allard, 2004). 111 the present study, parents (N = 106) of children independently diagnosed with autism (4-16 years of age; M = 8.20 years, SD = 2.69 years) reported greater sleep problems for themselves than did parents (N = 168) of typically developing children (4-15 years of age; M = 8.62 years, SD = 3.28 years). Children's sleep problems were related to parents' sleep problems for both groups; in the autism group, children's level of symptomatology was not related to their parents' sleep. The authors suggest areas for further research oil the sleep problems of children and their parents, the potential interaction of these problems with children's symptomatic behavior, and the relations of these factors to child, parent, and family functioning.
C1 [Lopez-Wagner, Muriel C.] Calif State Univ San Bernardino, Off Inst Res, San Bernardino, CA 92407 USA.
RP Lopez-Wagner, MC (reprint author), Calif State Univ San Bernardino, Off Inst Res, 5500 Univ Pkwy AD170, San Bernardino, CA 92407 USA.
EM mclopez@csusb.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
American Sleep Disorder Association, 1990, INT CLASS SLEEP DIS
Backhaus J, 2002, J PSYCHOSOM RES, V53, P737, DOI 10.1016/S0022-3999(02)00330-6
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NR 67
TC 18
Z9 18
PU HELDREF PUBLICATIONS
PI WASHINGTON
PA 1319 EIGHTEENTH ST NW, WASHINGTON, DC 20036-1802 USA
SN 0022-1325
J9 J GENET PSYCHOL
JI J. Genet. Psychol.
PD SEP
PY 2008
VL 169
IS 3
BP 245
EP 259
DI 10.3200/GNTP.169.3.245-260
PG 15
WC Psychology, Developmental; Psychology; Psychology, Multidisciplinary
SC Psychology
GA 341EG
UT WOS:000258696400003
PM 18788326
ER
PT J
AU Barber, JCK
Ozgen, HM
van Daalen, E
Bolton, P
Maloney, VK
Huang, S
Lachlan, KL
Cresswell, L
de Boogaard, MJV
Eleveld, MJ
van t'Slot, R
Hochstenbach, R
Barrow, M
Beerner, FA
Poot, M
AF Barber, John C. K.
Ozgen, H. M.
van Daalen, E.
Bolton, P.
Maloney, V. K.
Huang, S.
Lachlan, K. L.
Cresswell, L.
de Boogaard, M. J. van
Eleveld, M. J.
van t'Slot, R.
Hochstenbach, R.
Barrow, M.
Beerner, F. A.
Poot, M.
TI Microcephalin (MCPH1); a candidate gene for autism in 8p23.1 with
variable penetrance
SO JOURNAL OF MEDICAL GENETICS
LA English
DT Meeting Abstract
CT British Human Genetics Conference
CY SEP 15-17, 2008
CL York, ENGLAND
HO Univ York
C1 [Barber, John C. K.; Maloney, V. K.; Huang, S.] Salisbury Dist Hosp, Salisbury NHS Fdn Trust, Wessex Reg Genet Lab, Salisbury, Wilts, England.
[Ozgen, H. M.; van Daalen, E.] Univ Med Ctr, Dept Child & Adolescent Psychiat, Utrecht, Netherlands.
[Ozgen, H. M.; van Daalen, E.] Kings Coll London, Inst Psychiat, Dept Child & Adolescent Psychiat, London, England.
[Lachlan, K. L.] Princess Anne Hosp, Southampton Univ Hosp Trust, Wessex Clin Genet Serv, Southampton, Hants, England.
[Cresswell, L.; Barrow, M.] Leicester Royal Infirm, Leicester Genet Ctr, Leicester, Leics, England.
[de Boogaard, M. J. van; Eleveld, M. J.; van t'Slot, R.; Hochstenbach, R.; Beerner, F. A.; Poot, M.] Univ Med Ctr, Dept Med Genet, Utrecht, Netherlands.
EM john.barber@salisbury.nhs.uk
RI Bolton, Patrick/E-8501-2010
OI Bolton, Patrick/0000-0002-5270-6262
NR 0
TC 0
Z9 0
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0022-2593
J9 J MED GENET
JI J. Med. Genet.
PD SEP
PY 2008
VL 45
BP S93
EP S93
PG 1
WC Genetics & Heredity
SC Genetics & Heredity
GA 351PM
UT WOS:000259436400202
ER
PT J
AU Miedzybrodzka, Z
Neves-Pereira, M
Muller, B
Massie, D
Williams, JHG
O'Brien, PCM
Hughes, A
Shen, S
StClair, D
AF Miedzybrodzka, Zosia
Neves-Pereira, M.
Muller, B.
Massie, D.
Williams, J. H. G.
O'Brien, P. C. M.
Hughes, A.
Shen, S.
StClair, D.
TI Disruption of the EIF4E Gene in Non-Syndromic Autism
SO JOURNAL OF MEDICAL GENETICS
LA English
DT Meeting Abstract
CT British Human Genetics Conference
CY SEP 15-17, 2008
CL York, ENGLAND
HO Univ York
C1 [Miedzybrodzka, Zosia; Neves-Pereira, M.; Muller, B.; Williams, J. H. G.; Hughes, A.; Shen, S.] Univ Aberdeen, Aberdeen, Scotland.
NHS Grampian, Aberdeen, Scotland.
Univ Cambridge, Cytogenet Grp, Dept Vet Med, Cambridge CB2 1TN, England.
EM zosia@abdn.ac.uk
RI Shen, Sanbing/E-3750-2015
NR 0
TC 0
Z9 0
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0022-2593
J9 J MED GENET
JI J. Med. Genet.
PD SEP
PY 2008
VL 45
BP S16
EP S16
PG 1
WC Genetics & Heredity
SC Genetics & Heredity
GA 351PM
UT WOS:000259436400006
ER
PT J
AU Williams, J
Williams, S
AF Williams, Jill
Williams, S.
TI Rate of Abnormality in Paediatric Referrals with autism- a retrospective
study (The Sheffield Experience)
SO JOURNAL OF MEDICAL GENETICS
LA English
DT Meeting Abstract
CT British Human Genetics Conference
CY SEP 15-17, 2008
CL York, ENGLAND
HO Univ York
C1 [Williams, Jill; Williams, S.] Sheffield Reg Cytogenet Serv, Sheffield, S Yorkshire, England.
EM jill.williams@sch.nhs.uk
NR 0
TC 0
Z9 0
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0022-2593
J9 J MED GENET
JI J. Med. Genet.
PD SEP
PY 2008
VL 45
BP S99
EP S99
PG 1
WC Genetics & Heredity
SC Genetics & Heredity
GA 351PM
UT WOS:000259436400224
ER
PT J
AU Losh, M
Sullivan, PF
Trembath, D
Piven, J
AF Losh, Molly
Sullivan, Patrick F.
Trembath, Dimitri
Piven, Joseph
TI Current developments in the genetics of autism: From phenome to genome
SO JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY
LA English
DT Review
DE autism; copy number variation (CNV); endophenotype; genetic
ID TUBEROUS SCLEROSIS COMPLEX; SEROTONIN TRANSPORTER GENE; QUANTITATIVE
TRAIT LOCUS; RIGID-COMPULSIVE BEHAVIORS; WEAK CENTRAL COHERENCE;
SPECTRUM DISORDERS; BROADER PHENOTYPE; FAMILY-HISTORY; COGNITIVE
PHENOTYPE; SUSCEPTIBILITY GENE
AB Despite compelling evidence from twin and family studies indicating a strong genetic involvement in the etiology of autism, the unequivocal detection of autism susceptibility genes remains an elusive goal. The purpose of this review is to evaluate the current state of autism genetics research, with attention focused on new techniques and analytic approaches. We first present a brief overview of evidence for the genetic basis of autism, followed by an appraisal of linkage and candidate Cleric study findings and consideration of new analytic approaches to the study of complex psychiatric conditions, namely, genome-wide association studies, assessment of structural variation within the genome, and the incorporation of endophenotypes in genetic analysis.
C1 [Losh, Molly] Univ N Carolina, Dept Allied Hlth Sci, Chapel Hill, NC 27599 USA.
[Losh, Molly; Sullivan, Patrick F.; Piven, Joseph] Univ N Carolina, Dept Psychiat, Chapel Hill, NC 27599 USA.
[Trembath, Dimitri] Univ N Carolina, Dept Pathol & Lab Med, Chapel Hill, NC 27599 USA.
[Sullivan, Patrick F.] Karolinska Inst, Dept Med Epidemiol & Biostat, Stockholm, Sweden.
RP Losh, M (reprint author), Univ N Carolina, Dept Allied Hlth Sci, CB 7190, Chapel Hill, NC 27599 USA.
EM losh@med.unc.edu
FU [K12RR023248]; [R03MH079998]; [Autism Speaks]
FX ML was supported by K12RR023248, R03MH079998, and a grant from Autism
Speaks.
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NR 143
TC 49
Z9 49
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0022-3069
J9 J NEUROPATH EXP NEUR
JI J. Neuropathol. Exp. Neurol.
PD SEP
PY 2008
VL 67
IS 9
BP 829
EP 837
DI 10.1097/NEN.0b013e318184482d
PG 9
WC Clinical Neurology; Neurosciences; Pathology
SC Neurosciences & Neurology; Pathology
GA 344PA
UT WOS:000258938600001
PM 18716561
ER
PT J
AU Taber, KH
Hurley, RA
AF Taber, Katherine H.
Hurley, Robin A.
TI Mercury Exposure: Effects Across the Lifespan
SO JOURNAL OF NEUROPSYCHIATRY AND CLINICAL NEUROSCIENCES
LA English
DT Article
ID RANDOMIZED CLINICAL-TRIAL; MINAMATA-DISEASE; DENTAL AMALGAM; PRENATAL
EXPOSURE; HAIR MERCURY; METHYLMERCURY; CHILDREN; AUTISM; BRAIN; BLOOD
C1 [Taber, Katherine H.; Hurley, Robin A.] WG Hefner Vet Affairs Med Ctr, Vet Affairs Midatlantic Mental Illness Res Educ &, Salisbury, NC USA.
[Taber, Katherine H.; Hurley, Robin A.] WG Hefner Vet Affairs Med Ctr, Mental Hlth Serv Line, Salisbury, NC USA.
[Taber, Katherine H.] Virginia Coll Osteopath Med, Div Biomed Sci, Blacksburg, VA USA.
[Taber, Katherine H.] Baylor Coll Med, Dept Phys Med & Rehabil, Houston, TX 77030 USA.
[Hurley, Robin A.] Wake Forest Univ, Sch Med, Dept Psychiat, Winston Salem, NC 27109 USA.
[Hurley, Robin A.] Wake Forest Univ, Sch Med, Dept Radiol, Winston Salem, NC 27109 USA.
[Hurley, Robin A.] Baylor Coll Med, Menninger Dept Psychiat & Behav Sci, Houston, TX 77030 USA.
RP Hurley, RA (reprint author), Henfer VA Med Ctr, 1601 Brenner Ave, Salisbury, NC 28144 USA.
EM Robin.Hurley@va.gov
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NR 48
TC 0
Z9 0
PU AMER PSYCHIATRIC PUBLISHING, INC
PI ARLINGTON
PA 1000 WILSON BOULEVARD, STE 1825, ARLINGTON, VA 22209-3901 USA
SN 0895-0172
J9 J NEUROPSYCH CLIN N
JI J. Neuropsychiatr. Clin. Neurosci.
PD FAL
PY 2008
VL 20
IS 4
BP IV
EP 389
DI 10.1176/appi.neuropsych.20.2.iv
PG 6
WC Clinical Neurology; Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 399CV
UT WOS:000262778900001
PM 18451184
ER
PT J
AU Williams, K
MacDermott, S
Ridley, G
Glasson, EJ
Wray, JA
AF Williams, Katrina
MacDermott, Sarah
Ridley, Greta
Glasson, Emma J.
Wray, John A.
TI The prevalence of autism in Australia. Can it be established from
existing data?
SO JOURNAL OF PAEDIATRICS AND CHILD HEALTH
LA English
DT Article
DE autism spectrum disorders; autism; Asperger disorder; pervasive
developmental disorder - not otherwise specified; prevalence; data
collection
ID PERVASIVE DEVELOPMENTAL DISORDERS; SPECTRUM DISORDERS;
PRESCHOOL-CHILDREN; CHILDHOOD AUTISM; FOLLOW-UP; DIAGNOSIS;
EPIDEMIOLOGY; POPULATION; COMMUNITY; STATES
AB Aim: To assess whether existing data collection mechanisms can provide accurate and sufficient information about the prevalence of autism in Australia.
Methods: Summary data about the number of children aged 0-16 years known to have an autism spectrum disorder (ASD) were gathered from State and Territory health, disability, education sources and autism associations. Summary data were also provided by national sources. Initial contact was made by letter, and follow-up was undertaken by telephone or email.
Results: For the years 2003-2004, the estimated prevalence of autism for 6- to 12-year-olds ranged from 9.6 to 40.8/10 000 for the State and Territory data, and from 12.1 to 35.7/10 000 for the national data. There was a similar variation in prevalence estimates for children aged 0-5 and 13-16 years. There was also a variation in prevalence estimates between age groups.
Conclusion: Inconsistencies in autism prevalence estimates calculated from existing data sources suggest that further efforts are needed to ensure the collection of reliable information about the prevalence of ASD for national, State and Territory use. Existing data systems need to be improved or additional data systems need to be developed to ensure the collection of reliable information. Reliable and consistent ASD prevalence data would ensure that services are being provided to those who need them and would enhance the opportunities to undertake meaningful population-based research.
C1 [Williams, Katrina] Sydney Childrens Hosp, Sydney, NSW, Australia.
[Williams, Katrina] Univ New S Wales, Sydney, NSW, Australia.
[MacDermott, Sarah; Wray, John A.] Univ Western Australia, State Child Dev Ctr, Children & Adolescent Hlth Serv, Perth, WA 6009, Australia.
[MacDermott, Sarah; Glasson, Emma J.] Univ Western Australia, Telethon Inst Child Hlth Res, Ctr Child Hlth Res, Perth, WA 6009, Australia.
[Ridley, Greta] Childrens Hosp Westmead, Sydney, NSW, Australia.
[Glasson, Emma J.] Univ Western Australia, Sch Populat Hlth, Perth, WA, Australia.
RP Williams, K (reprint author), Sydney Childrens Community Hlth Ctr Child Hlth, Cnr Avoca & Barker St, Randwick, NSW 2031, Australia.
EM katrina.williams@sesiahs.health.nsw.gov.au
RI Glasson, Emma/H-5339-2013; Williams, Katrina/B-6828-2015
OI Glasson, Emma/0000-0003-3996-9049; Williams, Katrina/0000-0002-1686-4458
FU Australian Advisory Boards on Autism Spectrum Disorders; Commonwealth
Department of Department of Family; Community Services and Indigenous
Affairs (FaCSIA)
FX The authors of the study would like to acknowledge the support of the
Australian Advisory Boards on Autism Spectrum Disorders (formerly the
Autism Council of Australia, Ltd) who funded this study through a grant
from the Commonwealth Department of Department of Family, Community
Services and Indigenous Affairs (FaCSIA).We also wish to acknowledge the
support of the following individuals and organizations who assisted in
providing the data for the study;ACT: Lindy Abbott (DET), Therapy ACT;
NSW: Adrien Ford & Trevor Clarke (ASPECT), Brian Smyth King (DET),
Brendan O'Reilly (DADHC); NT: Carolyn Borci (DEET), Gail Bowker (TEMHS),
Alison Bird (AANT); QLD: Michael Keates (DSQ), Penny Beeston & Frances
Scodellaro (AQ), Andres Toro (QH); SA: Piet Crosby (DECS), Claire
Wittwer-Smith (DFC), Susan Deeprose & Lyn Zeidler (IDSC), Jon Martin
(ASA); TAS: Brodrick Smith (DHHS), Lynne James (DE); VIC: Geoff Riley
(DET), Amanda Golding (AV), Justin McDermott (DHS); WA: David Hounsome &
Anne Lawson (DSC), Val McKelvey (Cath Ed), Karen Lee Clark & Bernadette
Long (DET), Susan Rosendorff (AISWA), Joan McKenna Kerr (AAWA);
Centrelink: Cris Trevillian, Malinda Kirley & BI Frontdoor team: AIHW:
Timothy Beard & Xing-yan Wen, and Andrew Hayen for providing advice
about calculation of 95% confidence intervals.This report was presented,
on invitation, to the Board of the Autism Council of Australia Ltd by
one of the authors (John Wray) on September 2nd 2006.
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NR 48
TC 19
Z9 19
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1034-4810
J9 J PAEDIATR CHILD H
JI J. Paediatr. Child Health
PD SEP
PY 2008
VL 44
IS 9
BP 504
EP 510
DI 10.1111/j.1440-1754.2008.01331.x
PG 7
WC Pediatrics
SC Pediatrics
GA 339SI
UT WOS:000258597200007
PM 18564076
ER
PT J
AU Bourke, J
Ricciardo, B
Bebbington, A
Alberti, K
Jacoby, P
Dyke, P
Msall, M
Bower, C
Leonard, H
AF Bourke, Jenny
Ricciardo, Bernadette
Bebbington, Ami
Alberti, Karina
Jacoby, Peter
Dyke, Paula
Msall, Michael
Bower, Carol
Leonard, Helen
TI Physical and mental health in mothers of children with Down syndrome
SO JOURNAL OF PEDIATRICS
LA English
DT Article
ID INTELLECTUAL DISABILITY; PRESCHOOL-CHILDREN; BEHAVIOR PROBLEMS;
CEREBRAL-PALSY; PARENTS; STRESS; ADOLESCENTS; ADAPTATION;
PSYCHOPATHOLOGY; AUTISM
AB Objective To identify the relationship between characteristics of the child with Down syndrome and the health of their mother.
Study design Families with a child/young adult with Down syndrome (< 25 years) provided information related to the health of the child, functioning and behavior, and the health and well-being of the mother (n = 250).
Results The mean physical health score of mothers was 50.2 (SD = 9.6). Factors associated with lower mean physical health scores were as follows: child having a current heart problem (P = .036), a higher body mass index (P = .006), and higher (poorer) scores on the Developmental Behavior Checklist. Better physical health scores were seen in mothers whose children required no help/supervision in learning new skills (P = .008) and domestic tasks (P = .014). The mean mental health score of mothers was 45.2 (SD = 10.6), significantly lower than the norm of 50 (P < .0001). Associated child factors included current car problems (P = .079), muscle/bone problems (P = .004), > 4 episodes of illness in past year (P = .016), and higher scores on the DBC (P < .0001).
Conclusions The most important predictors of maternal health were children's behavioral difficulties, everyday functioning and current health status. Mothers of children with Down syndrome appear to experience poorer mental health and may require greater support and services to improve behavior management skills for their child and their own psychological well-being.
C1 [Bourke, Jenny; Bebbington, Ami; Alberti, Karina; Jacoby, Peter; Dyke, Paula; Bower, Carol; Leonard, Helen] Univ Western Australia, Ctr Child Hlth Res, Telethon Inst Child Hlth Res, Perth, WA 6009, Australia.
[Ricciardo, Bernadette] Princess Margaret Hosp Children, Perth, WA, Australia.
[Msall, Michael] Univ Chicago, Pritzker Sch Med, Kennedy Mental Retardat Ctr, Chicago, IL 60637 USA.
[Msall, Michael] Comer Childrens Hosp, Dept Dev & Behav Pediat, Chicago, IL USA.
[Msall, Michael] La Rabida Childrens Hosp & Res Ctr, Chicago, IL USA.
RP Leonard, H (reprint author), Telethon Inst Child Hlth Res, POB 855, Perth, WA 6872, Australia.
RI Leonard, Helen/A-1010-2013
OI Leonard, Helen/0000-0001-6405-5834
FU National Institutes of Health [5 R01 HD43100-04]; NHMRC [353514, 353628]
FX Supported by the National Institutes of Health (5 R01 HD43100-04) and
NHMRC program grant 353514, and CB by NHMRC Fellowship 353628.
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NR 35
TC 27
Z9 27
PU MOSBY-ELSEVIER
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0022-3476
J9 J PEDIATR-US
JI J. Pediatr.
PD SEP
PY 2008
VL 153
IS 3
BP 320
EP 326
DI 10.1016/j.jpeds.2008.02.047
PG 7
WC Pediatrics
SC Pediatrics
GA 348JV
UT WOS:000259207700007
PM 18534233
ER
PT J
AU Roza, SJ
Govaert, PP
Lequin, MH
Jaddoe, VWV
Moll, HA
Steegers, EAP
Hofman, A
Verhulst, FC
Tiemeier, H
AF Roza, Sabine J.
Govaert, Paul P.
Lequin, Maarten H.
Jaddoe, Vincent W. V.
Moll, Henriette A.
Steegers, Eric A. P.
Hofman, Albert
Verhulst, Frank C.
Tiemeier, Henning
TI Cerebral ventricular volume and temperamental difficulties in infancy.
The generation R study
SO JOURNAL OF PSYCHIATRY & NEUROSCIENCE
LA English
DT Article
DE attention deficit and disruptive behaviour disorders; fetal development;
child development
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; DEFICIT HYPERACTIVITY
DISORDER; SPECTRUM DISORDER; PLACENTAL INSUFFICIENCY;
PSYCHIATRIC-DISORDERS; PRENATAL EXPOSURE; BRAIN-DEVELOPMENT;
SCHIZOPHRENIA; AUTISM; CHILD
AB Background: Numerous studies have provided evidence for subtle deviations in brain morphology in children with psychiatric disorders, but much less is known about the onset and developmental trajectory of these deviations early in life. We sought to determine whether variances in cerebral ventricular size in fetuses and newborns are associated with temperamental difficulties in infants. Methods: Within a population-based cohort study, we measured the size of the lateral ventricle of the fetus' brain twice during pregnancy. We used 3-dimensional cranial ultrasound to measure the cerebral ventricular volume of infants at age 6 weeks. We then related the size of the cerebral ventricular system to temperamental dimensions at age 3 months using the Mother and Baby Scales, and at age 6 months using the Infant Behavior Questionnaire for a total of 1028 infants. Results: The size of the lateral ventricle of the fetuses in mid-pregnancy was not related to temperamental difficulties in infants; however, smaller lateral ventricles in late pregnancy were associated with higher activity levels at the age of 6 months. Infants with smaller ventricular volumes at age 6 weeks experienced higher activity levels, more anger or irritability and poorer orienting later in infancy. Children with the lowest ventricular volumes scored on average 0.15 (95% confidence interval 0.06-0.23, p = 0.001) points higher (23%) on activity levels than children with the highest ventricular volumes. Conclusion: Variations in ventricular size before and shortly after birth are associated with temperamental difficulties. Some of the morphologic differences between children with and without psychiatric disorders may develop very early in life.
C1 [Roza, Sabine J.; Verhulst, Frank C.; Tiemeier, Henning] Erasmus MC, Dept Child & Adolescent Psychiat, NL-3000 CB Rotterdam, Netherlands.
[Jaddoe, Vincent W. V.; Hofman, Albert; Tiemeier, Henning] Erasmus MC, Dept Epidemiol, NL-3000 CB Rotterdam, Netherlands.
[Steegers, Eric A. P.] Erasmus MC, Dept Obstet & Gynecol, NL-3000 CB Rotterdam, Netherlands.
[Govaert, Paul P.; Jaddoe, Vincent W. V.; Moll, Henriette A.] Erasmus MC, Dept Pediat, NL-3000 CB Rotterdam, Netherlands.
[Roza, Sabine J.; Jaddoe, Vincent W. V.] Erasmus MC, Generat R Study Grp, NL-3000 CB Rotterdam, Netherlands.
[Lequin, Maarten H.] Erasmus MC, Dept Radiol, NL-3000 CB Rotterdam, Netherlands.
RP Tiemeier, H (reprint author), Erasmus MC, Dept Child & Adolescent Psychiat, POB 2060, NL-3000 CB Rotterdam, Netherlands.
EM h.tiemeier@erasmusmc.nl
FU Erasmus Medical Center Rotterdam; Erasmus University Rotterdam;
Netherlands Organization for Health Research and Development (ZonMw);
Netherlands Organization for Health Research and Development
[10.000.1003]
FX The Generation R Study is conducted by the Erasmus Medical Center
Rotterdam ill close collaboration with the Faculty Of Social Sciences Of
the Erasmus University Rotterdam, the Municipal Health Service Rotterdam
area, the Rotterdam Homecare Foundation and the Stichting Trombosedienst
& Artsenlabortorium Rijnmond (STAR), Rotterdam, the Netherlands. We
gratefully acknowledge the contribution of general practitioners,
hospitals, mid-wives and pharmacies ill Rotterdam. The first phase of
the Generation R Study is made possible by financial support from the
Erasmus Medical Center Rotterdam, the Erasmus University Rotterdam and
the Netherlands Organization for Health Research and Development
(ZonMw). This study was supported by an additional grant from the
Netherlands Organization for Health Research and Development (ZonMw,
'Geestkraht' programme 10.000.1003).
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TC 5
Z9 6
PU CMA-CANADIAN MEDICAL ASSOC
PI OTTAWA
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EP 439
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TC 0
Z9 0
PU SLACK INC
PI THOROFARE
PA 6900 GROVE RD, THOROFARE, NJ 08086 USA
SN 0279-3695
EI 1938-2413
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Christianity; qi; locus of control; psychotherapy; Bible; self
psychology; psychology
ID SPECTRUM DISORDERS; ASPERGER-SYNDROME; AUTISM; ADULTS
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LA English
DT Article
DE Sibling Relations; Relations with Handicap Sibling; Attitudes Tended to
Handicap Child
ID CHILDHOOD
AB This study investigated the relationships of healthy children with their handicap siblings and analyzed their acceptance of the siblings. The study was conducted in 16 different special education and rehabilitation centers in Ankara. Two hundred twenty eight healthy children between the ages of 10 to 17 years voluntarily participated in the investigation. All participating children had siblings diagnosed with one of the six disabilities: Learning disability (LD), mental retardation (MR), cerebral palsy (CP), mental-motor retardation (MMR), Down syndrome (DS), or autism (A). The attitudes of the healthy children towards their handicap siblings (EKYTO) and towards other handicap people (EBYTO) were studied. Results show that healthy children dispay positive attitudes towards their own handicap siblings whereas less positive attitudes towards other handicapped people. The investigation takes into consideration diagnosis and degree of the handicap. In general, it is observed that the healthy children's attitudes towards their handicap sibling are clearly changing according to diagnosis and level of handicap whereas the same factors do not make any difference in terms of attitudes towards other handicapped people.
C1 [Aksoy, Ayse B.] Gazi Univ, Fac Vocat Educ, Main Discipline Children Dev & Educ, Ankara, Turkey.
RP Aksoy, AB (reprint author), Gazi Univ, Fac Vocat Educ, Main Discipline Children Dev & Educ, Ankara, Turkey.
EM ayse1961@gmail.com
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NR 18
TC 2
Z9 2
PU EDAM
PI ISTANBUL
PA KISIKLI MH ALEMDAG CD YAN YOL SK, SBK IS MERKEZI NO 5, KAT 1 USKUDAR,
ISTANBUL, 81190, TURKEY
SN 1303-0485
J9 KURAM UYGULAMA EGI
JI Kuram Uygulamada Egit. Bilim.
PD SEP
PY 2008
VL 8
IS 3
BP 769
EP 779
PG 11
WC Education & Educational Research
SC Education & Educational Research
GA 453LP
UT WOS:000266612300004
ER
PT J
AU Rogers, EJ
AF Rogers, Eugene J.
TI Has enhanced folate status during pregnancy altered natural selection
and possibly Autism prevalence? A closer look at a possible link
SO MEDICAL HYPOTHESES
LA English
DT Article
ID RECURRENT MISCARRIAGES; REDUCTASE POLYMORPHISM; HOMOCYSTEINE; OUTCOMES;
WOMEN
AB The inverse association between maternal folate status and incidence of infants born with neural tube defects (NTD's) was recognized over twenty years ago and led the US health agencies in the early 1990s to recommend that women of childbearing age consume 400 mu g of folic acid each day. The FDA followed by mandating that certain foods be fortified with folic acid and this has resulted in a significant enhancement of maternal folate status to levels that are often difficult to otherwise achieve naturally. At Least one study indicates that this has decreased the incidence of NTD's. However, this same time period directly coincides with what many feet is the apparent beginning and continuous increase in the prevalence of Autism and related Autism Spectrum Disorders (ASD's) in the US. Are these similar time frames of changes in maternal folate status and possible Autism prevalence a random event or has improved maternal (and fetal) folate status during pregnancy played a role? It is not only plausible but highly likely. A particular polymorphic form to a key enzyme required to activate folate for methylation in neurodevelopment, 5-methylenetetrahydrofolate reductase (MTHFR), demonstrates reduced activity under low or normal folate Levels but normal activity under conditions of higher folate nutritional status. A consequence of the presence of the polymorphic form of this enzyme during normal or reduced folate status are higher plasma homocysteine levels than noncarriers and the combination of these factors have been shown in several studies to result in an increase rate of miscarriage via thrombotic events. However, the incidence of hyperhomocysteinemia in the presence of the polymorphism is reduced under the common condition of enhanced folate status and thereby masks the latent adverse effects of the presence of this enzyme form during pregnancy. Of great importance is that this polymorphism, although common in the normal population, is found in significantly higher frequency in Autisic individuals. It is hypothesized here that the enhancement of maternal folate status before and during pregnancy in the last 15 years has altered natural selection by increasing survival rates during pregnancy of infants possessing the MTHFR C677T polymorphism, via reduction in hyperhomocysteinemia associated with this genotype and thereby miscarriage rates. This also points directly to an increased rate of births of infants with higher postnatal requirements for folic acid needed for normal methylation during this critical neurodevelopmental period. If these numbers have increased then so have the absolute number of infants that after birth fait to maintain the higher folate status experienced in utero thus leading to an increased number of cases of developmental disorders such as Autism. Detection of the C677T polymorphism as well as other methionine cycle enzymes related to folate metabolism and methylation at birth as part of newborn screening programs could determine which newborns need be monitored and maintained on diets or supplements that ensure adequate folate status during this critical postnatal neurodevelopment period. (C) 2008 Elsevier Ltd. All rights reserved.
C1 Univ Massachusetts, Clin Lab & Nutr Sci, Lowell, MA 01854 USA.
RP Rogers, EJ (reprint author), Univ Massachusetts, Clin Lab & Nutr Sci, 3 Solomont Way,Suite 4, Lowell, MA 01854 USA.
EM Eugene_Rogers@uml.edu
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NR 14
TC 20
Z9 21
PU CHURCHILL LIVINGSTONE
PI EDINBURGH
PA JOURNAL PRODUCTION DEPT, ROBERT STEVENSON HOUSE, 1-3 BAXTERS PLACE,
LEITH WALK, EDINBURGH EH1 3AF, MIDLOTHIAN, SCOTLAND
SN 0306-9877
J9 MED HYPOTHESES
JI Med. Hypotheses
PD SEP
PY 2008
VL 71
IS 3
BP 406
EP 410
DI 10.1016/j.mehy.2008.04.013
PG 5
WC Medicine, Research & Experimental
SC Research & Experimental Medicine
GA 340JL
UT WOS:000258641800015
PM 18514430
ER
PT J
AU Barshop, BA
Summar, ML
AF Barshop, Bruce A.
Summar, Marshall L.
TI Attitudes regarding vaccination among practitioners of clinical
biochemical genetics
SO MOLECULAR GENETICS AND METABOLISM
LA English
DT Editorial Material
ID MITOCHONDRIAL DISEASE; AUTISM
C1 [Barshop, Bruce A.] Univ Calif San Diego, Sch Med, Dept Pediat, Rady Childrens Hosp San Diego, La Jolla, CA 92093 USA.
[Summar, Marshall L.] Vanderbilt Univ, Dept Pediat, Nashville, TN USA.
RP Barshop, BA (reprint author), Univ Calif San Diego, Sch Med, Dept Pediat, Rady Childrens Hosp San Diego, 9500 Gilman Dr,MC 0830, La Jolla, CA 92093 USA.
EM bbarshop@ucsd.edu
CR [Anonymous], 1993, MMWR RECOMM REP, V42, P1
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Poling JS, 2006, J CHILD NEUROL, V21, P170, DOI 10.2310/7010.2006.00032
NR 8
TC 0
Z9 0
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1096-7192
J9 MOL GENET METAB
JI Mol. Genet. Metab.
PD SEP-OCT
PY 2008
VL 95
IS 1-2
BP 1
EP 2
DI 10.1016/j.ymgme.2008.08.001
PG 2
WC Biochemistry & Molecular Biology; Genetics & Heredity; Medicine,
Research & Experimental
SC Biochemistry & Molecular Biology; Genetics & Heredity; Research &
Experimental Medicine
GA 358TS
UT WOS:000259941000001
PM 18816884
ER
PT J
AU Kreutz, G
Schubert, E
Mitchell, LA
AF Kreutz, Gunter
Schubert, Emery
Mitchell, Laura A.
TI Cognitive styles of music listening
SO MUSIC PERCEPTION
LA English
DT Article
DE music cognition; empathizer-systemizer-theory; sex differences;
musicians; music performance
ID SEX-DIFFERENCES; GENDER-DIFFERENCES; BRAIN; RESPONSES; CHILDREN; AUTISM
AB BARON-COHEN'S EMPATHIZER-SYSTEMIZER-THEORY (E-S theory, Baron-Cohen, Knickmeyer, & Belmonte, 2005) distinguishes two general cognitive styles. Empathizing is characterized as the capacity to respond to feeling states of other individuals, whereas systemizing is characterized as the capacity to respond to regularities of objects and events. To investigate these traits within the music domain, a questionnaire study (N = 442) was conducted. Construct validity and reliability of the measurement instrument were assessed by factor analysis procedures. A simplified unit weighting (SUW) scale was used to determine individual differences in music empathizing (ME) and music systemizing (MS). Significant effects of sex and of music performance experience were observed. A highly similar pattern of results emerged from a replicating survey (N = 155) using a short-version of the questionnaire. These results suggest that the ME and MS traits corroborate and extend the general E-S theory.
C1 [Kreutz, Gunter] Carl von Ossietzky Univ Oldenburg, Dept Mus, D-26129 Oldenburg, Germany.
[Schubert, Emery] Univ New S Wales, Sydney, NSW, Australia.
[Mitchell, Laura A.] Glasgow Caledonian Univ, Glasgow G4 0BA, Lanark, Scotland.
RP Kreutz, G (reprint author), Carl von Ossietzky Univ Oldenburg, Dept Mus, Ammerlander Heerstr 114-118, D-26129 Oldenburg, Germany.
EM gunter.kreutz@uni-oldenburg.de
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NR 31
TC 26
Z9 26
PU UNIV CALIFORNIA PRESS
PI BERKELEY
PA C/O JOURNALS DIVISION, 2000 CENTER ST, STE 303, BERKELEY, CA 94704-1223
USA
SN 0730-7829
J9 MUSIC PERCEPT
JI Music Percept.
PD SEP
PY 2008
VL 26
IS 1
BP 57
EP 73
DI 10.1525/MP.2008.26.1.57
PG 17
WC Music; Psychology, Experimental
SC Music; Psychology
GA 340AR
UT WOS:000258618900004
ER
PT J
AU Materna, S
Dicke, PW
Thier, P
AF Materna, Simone
Dicke, Peter W.
Thier, Peter
TI The posterior superior temporal sulcus is involved in social
communication not specific for the eyes
SO NEUROPSYCHOLOGIA
LA English
DT Article
DE functional magnetic resonance imaging (fMRI); social cognition; social
attention; gaze following; temporal lobe
ID BIOLOGICAL MOTION PERCEPTION; JOINT ATTENTION; GAZE DIRECTION; AUTISM;
CORTEX; CUES; SENSITIVITY; MOVEMENTS; NEURONS; CELLS
AB Neuroimaging and lesion studies suggest that the superior temporal sulcus (STS) region is involved in eye gaze processing. Hence, the STS region is suggested to be the location of the "eye-direction detector", a key element in the "mindreading model" proposed by Baron-Cohen [Baron-Cohen, S. (1995). Mindblindness: An essay on autism and theory of mind. Cambridge: The MIT Press]. Not only the eyes, but also a pointing finger of another person can inform us about the direction of attention of the other one. In an event-related functional magnetic resonance imaging experiment, healthy human subjects actively followed a directional cue provided either by the eyes or. alternatively, the pointing finger of another person to make an eye movement toward an object in space. Our results show clearly that the posterior STS region is equally involved in processing directional information from either source. The only difference between the two cues was found in the lingual gyrus, in which a stronger blood-oxygen-level-dependent (BOLD) response was observed during the finger pointing compared to the gaze following task. We suggest that different structures might be involved in the initial processing of directional information coming from the eyes or the pointing finger. These different streams of information may then converge in the posterior STS region, orchestrating the usage of a wider range of socially relevant directional cues able to inform us about the direction of attention and the intentions of another person. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Materna, Simone; Dicke, Peter W.; Thier, Peter] Univ Tubingen, Dept Cognit Neurol, Hertie Inst Clin Brain Res, Tubingen, Germany.
RP Materna, S (reprint author), Univ Tubingen, Dept Cognit Neurol, Hertie Inst Clin Brain Res, Tubingen, Germany.
EM simone.kamphuis@uni-tuebingen.de; thier@uni-tuebingen.de
FU European Union; German Federal Ministry of Education and Research [01
GA0503]
FX This work was supported by the European Union research program 'PERACT
and a grant from the German Federal Ministry of Education and Research
(grant 01 GA0503).
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NR 37
TC 37
Z9 38
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0028-3932
J9 NEUROPSYCHOLOGIA
JI Neuropsychologia
PD SEP
PY 2008
VL 46
IS 11
BP 2759
EP 2765
DI 10.1016/j.neuropsychologia.2008.05.016
PG 7
WC Behavioral Sciences; Neurosciences; Psychology, Experimental
SC Behavioral Sciences; Neurosciences & Neurology; Psychology
GA 339YB
UT WOS:000258612100015
PM 18585742
ER
PT J
AU Enticott, PG
Johnston, PJ
Herring, SE
Hoy, KE
Fitzgerald, PB
AF Enticott, Peter G.
Johnston, Patrick J.
Herring, Sally E.
Hoy, Kate E.
Fitzgerald, Paul B.
TI Mirror neuron activation is associated with facial emotion processing
SO NEUROPSYCHOLOGIA
LA English
DT Article
DE mirror neurons; premotor cortex; facial emotion processing; facial
cognition; transcranial magnetic stimulation
ID AUTISM SPECTRUM DISORDERS; MOTOR FACILITATION; SOCIAL COGNITION; SYSTEM;
EMPATHY; EXPRESSIONS; DYSFUNCTION; MIMICRY; OTHERS; RECOGNITION
AB Theoretical accounts suggest that mirror neurons play a crucial role in social cognition. The current study used transcranial magnetic stimulation (TMS) to investigate the association between mirror neuron activation and facial emotion processing, a fundamental aspect of social cognition, among healthy adults (n = 20). Facial emotion processing of static (but not dynamic) images correlated significantly with an enhanced motor response, proposed to reflect mirror neuron activation. These correlations did not appear to reflect general facial processing or pattern recognition, and provide support to current theoretical accounts linking the mirror neuron system to aspects of social cognition. We discuss the mechanism by which mirror neurons might facilitate facial emotion recognition. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Enticott, Peter G.; Herring, Sally E.; Hoy, Kate E.; Fitzgerald, Paul B.] Monash Univ, Sch Psychol Psychiat & Psychol Med, Alfred Psychiat Res Ctr, Melbourne, Vic 3004, Australia.
[Johnston, Patrick J.] Swinburne Univ Technol, Brain Sci Inst, Hawthorn, Vic 3122, Australia.
RP Enticott, PG (reprint author), Monash Univ, Sch Psychol Psychiat & Psychol Med, Alfred Psychiat Res Ctr, Level 1,Old Baker Bldg, Melbourne, Vic 3004, Australia.
EM peter.enticott@med.monash.edu.au
RI Fitzgerald, Paul/A-1225-2008; Johnston, Patrick/P-3158-2014
OI Fitzgerald, Paul/0000-0003-4217-8096; Johnston,
Patrick/0000-0001-7703-1073
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Uddin LQ, 2007, TRENDS COGN SCI, V11, P153, DOI 10.1016/j.tics.2007.01.001
Wicker B, 2003, NEURON, V40, P655, DOI 10.1016/S0896-6273(03)00679-2
Williams JHG, 2001, NEUROSCI BIOBEHAV R, V25, P287, DOI 10.1016/S0149-7634(01)00014-8
NR 30
TC 43
Z9 45
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0028-3932
J9 NEUROPSYCHOLOGIA
JI Neuropsychologia
PD SEP
PY 2008
VL 46
IS 11
BP 2851
EP 2854
DI 10.1016/j.neuropsychologia.2008.04.022
PG 4
WC Behavioral Sciences; Neurosciences; Psychology, Experimental
SC Behavioral Sciences; Neurosciences & Neurology; Psychology
GA 339YB
UT WOS:000258612100027
PM 18554670
ER
PT J
AU Riby, DM
Hancock, PJB
AF Riby, Deborah M.
Hancock, Peter J. B.
TI Viewing it differently: Social scene perception in Williams syndrome and
Autism
SO NEUROPSYCHOLOGIA
LA English
DT Article
DE eye-tracking; Williams syndrome; autism; social cognition
ID REAL-WORLD SCENES; FACE; CHILDREN; HYPERSOCIABILITY; SCHIZOPHRENIA;
PREDICTORS; ATTENTION; FIXATION; PATTERNS; THREAT
AB The genetic disorder Williams syndrome (WS) is associated with a propulsion towards social stimuli and interactions with people. In contrast, the neuro-developmental disorder autism is characterised by social withdrawal and lack of interest in socially relevant information. Using eye-tracking techniques we investigate how individuals with these two neuro-developmental disorders associated with distinct social characteristics view scenes containing people. The way individuals with these disorders view social stimuli may impact upon successful social interactions and communication. Whilst individuals with autism spend less time than is typical viewing people and faces in static pictures of social interactions, the opposite is apparent for those with WS whereby exaggerated fixations are prevalent towards the eyes. The results suggest more attention should be drawn towards understanding the implications of atypical social preferences in WS, in the same way that attention has been drawn to the social deficits associated with autism. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Riby, Deborah M.] Newcastle Univ, Sch Psychol, Newcastle Upon Tyne NE1 7RU, Tyne & Wear, England.
[Hancock, Peter J. B.] Univ Stirling, Dept Psychol, Stirling FK9 4LA, Scotland.
RP Riby, DM (reprint author), Newcastle Univ, Sch Psychol, Ridley Bldg 1,Framlington Pl, Newcastle Upon Tyne NE1 7RU, Tyne & Wear, England.
EM D.M.Riby@newcastle.ac.uk
RI Hancock, Peter/A-4633-2009
OI Hancock, Peter/0000-0001-6025-7068
FU Economic and Social Research Council [R000222030]
FX This research was entirely supported by a grant from the Economic and
Social Research Council (R000222030)to PJBH and DMR.
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NR 35
TC 111
Z9 117
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0028-3932
J9 NEUROPSYCHOLOGIA
JI Neuropsychologia
PD SEP
PY 2008
VL 46
IS 11
BP 2855
EP 2860
DI 10.1016/j.neuropsychologia.2008.05.003
PG 6
WC Behavioral Sciences; Neurosciences; Psychology, Experimental
SC Behavioral Sciences; Neurosciences & Neurology; Psychology
GA 339YB
UT WOS:000258612100028
PM 18561959
ER
PT J
AU Barnes, KA
Howard, DV
Howard, JH
Gilotty, L
Kenworthy, L
Gaillard, WD
Vaidya, CJ
AF Barnes, Kelly Anne
Howard, Darlene V.
Howard, James H., Jr.
Gilotty, Lisa
Kenworthy, Lauren
Gaillard, William D.
Vaidya, Chandan J.
TI Intact implicit learning of spatial context and temporal sequences in
childhood autism spectrum disorder
SO NEUROPSYCHOLOGY
LA English
DT Article
DE frontostriatal circuits; medial temporal lobes; spatial attention;
sequence learning; developmental disorders
ID HIGH-FUNCTIONING AUTISM; DEVELOPMENTAL DIFFERENCES; EXECUTIVE
DYSFUNCTION; CENTRAL COHERENCE; MEMORY; CHILDREN; BRAIN; ADOLESCENTS;
HIPPOCAMPUS; EXPLICIT
AB Autism spectrum disorder (ASD) is defined by atypicalities in domains that are posited to rely on implicit learning processes Such as social communication, language, and motor behavior. The authors examined 2 forms of implicit learning in 14 children with high-functioning ASD (10 of whom were diagnosed with Asperger's syndrome) and 14 control children, learning of spatial context known to be mediated by the medial temporal lobes (using the contextual cueing task) and of sequences known to be mediated by frontal-striatal and frontal-cerebellar circuits (using the alternating serial reaction time task). Both forms of learning were unimpaired in ASD. Spatial contextual implicit learning was spared in ASD despite slower visual search of spatial displays. The present findings provide evidence for the integrity of learning processes dependent on integration of spatial and sequential contextual information in high-functioning children with ASD.
C1 [Barnes, Kelly Anne; Howard, Darlene V.; Vaidya, Chandan J.] Georgetown Univ, Dept Psychol, Washington, DC 20057 USA.
[Howard, James H., Jr.] Catholic Univ Amer, Dept Psychol, Washington, DC 20064 USA.
[Howard, James H., Jr.; Gaillard, William D.] Georgetown Univ, Dept Neurol, Washington, DC 20057 USA.
[Gilotty, Lisa; Kenworthy, Lauren; Gaillard, William D.; Vaidya, Chandan J.] Childrens Natl Med Ctr, Dept Neurol, Washington, DC 20010 USA.
RP Barnes, KA (reprint author), Georgetown Univ, Dept Psychol, 306L White Gravenor, Washington, DC 20057 USA.
EM kab69@georgetown.edu
FU Georgetown University; National Alliance for Autism Research; National
Institute of Mental Health [MH 065395, NIA R37AG15450]; Frederick and
Elizabeth Singer Foundation
FX Funding support came from Georgetown University, National Alliance for
Autism Research, National Institute of Mental Health Grants MH 065395,
NIA R37AG15450, and the Frederick and Elizabeth Singer Foundation. We
thank Jennifer Foss-Feig and Margaret Benner for assistance with data
collection, and Sunbin Song and Marvin Chun for providing the ASRT and
CC tasks, respectively. Lisa Gilotty is now with the National Institute
of Mental Health, the National Institutes of Health.
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NR 50
TC 39
Z9 41
PU AMER PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST NE, WASHINGTON, DC 20002-4242 USA
SN 0894-4105
J9 NEUROPSYCHOLOGY
JI Neuropsychology
PD SEP
PY 2008
VL 22
IS 5
BP 563
EP 570
DI 10.1037/0894-4105.22.5.563
PG 8
WC Psychology, Clinical; Neurosciences; Psychology
SC Psychology; Neurosciences & Neurology
GA 345KC
UT WOS:000258994300002
PM 18763876
ER
PT J
AU Yoo, HJ
Cho, IH
Park, M
Cho, E
Cho, SC
Kim, BN
Kim, JW
Kim, SA
AF Yoo, Hee Jeong
Cho, In Hee
Park, Mira
Cho, Eunchung
Cho, Soo Churl
Kim, Bung Nyun
Kim, Jae Won
Kim, Soon Ae
TI Association between PTGS2 polymorphism and autism spectrum disorders in
Korean trios
SO NEUROSCIENCE RESEARCH
LA English
DT Article
DE autism spectrum disorders (ASD); cyclooxygenase 2; transmission
disequilibrium test (TDT); association study
ID FAMILY-BASED ASSOCIATION; CYCLOOXYGENASE-2 EXPRESSION; CANCER; RISK;
GENE
AB Cyclooxygenase-2 (Cox-2) is an inducible enzyme involved in neuroplasticity and the neuropathology of the central nervous system. This study evaluated the relationship between autism spectrum disorders (ASDs) and polymorphisms of PTGS2 (the gene encoding Cox-2) with 151 Korean family trios including children with ASDs. We found that the A allele of rs2745557 was preferentially transmitted in ASDs (p < 0.01) and that the GAAA haplotype was significantly associated with ASDs (p < 0.01). We also observed statistically significant associations between each genotype and the specific symptom domain scores of ADOS and ADI-R, including communication, qualitative abnormalities in reciprocal social interaction, and overactivity/agitation. (C) 2008 Published by Elsevier Ireland Ltd and the Japan Neuroscience Society.
C1 [Kim, Soon Ae] Eulji Univ, Sch Med, Dept Pharmacol, Taejon 301832, South Korea.
[Yoo, Hee Jeong; Cho, Eunchung] Seoul Natl Univ, Bundang Hosp, Dept Psychiat, Songnam, Kyeonggi, South Korea.
[Cho, In Hee] Gachon Univ Med & Sci, Gil Med Ctr, Dept Psychiat, Inchon, South Korea.
[Park, Mira] Eulji Univ, Sch Med, Dept Prevent Med, Taejon 301832, South Korea.
[Cho, Soo Churl; Kim, Bung Nyun; Kim, Jae Won] Seoul Natl Univ Hosp, Coll Med, Dept Child & Adolescent Psychiat, Seoul 110744, South Korea.
RP Kim, SA (reprint author), Eulji Univ, Sch Med, Dept Pharmacol, 143-5 Youngdu Dong, Taejon 301832, South Korea.
EM sakim@eulji.ac.kr
RI Kim, Jae Won/J-5430-2012; Kim, Boong Nyun/J-5397-2012; Cho, Soo
Churl/J-5667-2012; Yoo, Hee Jeong/J-5555-2012
FU Korean Government (MOEHRD, Basic Research Promotion Fund)
[KRF-2006-311-E00266]; Korean Government (MOEHRD)
[R14-2003-002-01001-0]; Gachon University of Medicine and Science
FX This work was supported by the Korea Research Foundation Grant funded by
the Korean Government (MOEHRD, Basic Research Promotion Fund)
(KRF-2006-311-E00266). Mira Park was supported by a Korea Research
Foundation Grant funded by the Korean Government (MOEHRD)
(R14-2003-002-01001-0). In Hee Cho was supported by a Grant for Young
Scientists funded by the Gachon University of Medicine and Science.
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Zhang XM, 2005, GASTROENTEROLOGY, V129, P565, DOI 10.1053/j.gastro.2005.05.003
NR 21
TC 7
Z9 7
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0168-0102
J9 NEUROSCI RES
JI Neurosci. Res.
PD SEP
PY 2008
VL 62
IS 1
BP 66
EP 69
DI 10.1016/j.neures.2008.05.008
PG 4
WC Neurosciences
SC Neurosciences & Neurology
GA 342XT
UT WOS:000258817600010
PM 18579107
ER
PT J
AU Doja, A
AF Doja, Asif
TI Genetics and the myth of vaccine encephalopathy
SO PAEDIATRICS & CHILD HEALTH
LA English
DT Editorial Material
ID DE-NOVO MUTATIONS; AUTISM; CHILDREN; IMMUNIZATIONS; PERTUSSIS; SEIZURES;
RECEIPT; RUBELLA; MEASLES; MUMPS
C1 Childrens Hosp Eastern Ontario, Div Neurol, Ottawa, ON K1H 8L1, Canada.
RP Doja, A (reprint author), Childrens Hosp Eastern Ontario, Div Neurol, 401 Smyth Rd, Ottawa, ON K1H 8L1, Canada.
EM adoja@cheo.on.ca
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HARRIS G, DEAL AUTISM CASE FUE
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*PUBL HLTH AG CAN, 2006, CAN NAT REP IMM
*PUBL HLTH AG CAN, DIS INF MEASL
Smith PJ, 2004, PEDIATRICS, V114, P187, DOI 10.1542/peds.114.1.187
NR 16
TC 0
Z9 0
PU PULSUS GROUP INC
PI OAKVILLE
PA 2902 S SHERIDAN WAY, OAKVILLE, ONTARIO L6J 7L6, CANADA
SN 1205-7088
J9 PAEDIATR CHILD HEALT
JI Paediatr. Child Health
PD SEP
PY 2008
VL 13
IS 7
BP 597
EP 599
PG 3
WC Pediatrics
SC Pediatrics
GA 352YB
UT WOS:000259532200004
PM 19436557
ER
PT J
AU Marshall, J
Sheller, B
Mancl, L
Williams, BJ
AF Marshall, Jennifer
Sheller, Barbara
Mancl, Lloyd
Williams, Bryan J.
TI Parental Attitudes Regarding Behavior Guidance of Dental Patients with
Autism
SO PEDIATRIC DENTISTRY
LA English
DT Article
DE PEDIATRIC DENTISTRY; BEHAVIOR GUIDANCE; PARENTAL ATTITUDES; AUTISM
AB Purposes: The purposes of this study were to evaluate: (1) parents' ability to predict dental treatment cooperation by their autistic child; (2) behavior guidance techniques (BGTs) used during treatment; and (3) parental attitudes regarding basic and advanced BGTs. Methods: Data were collected from 85 parent/autistic child pairs and their dentists using surveys and treatment records. Results: Parents most accurately predicted If their child would permit an examination in the dental choir (>= 88%) and would cooperate for radiographs (>= 84%). BGTs utilized most often (>50%) were positive verbal reinforcement (PVR), tell-show-do (TSD), mouthprops, and rewards. In general, basic BGTs were more acceptable (>81%) than advanced BGTs (>54%). The most acceptable techniques (>90%) in order were: PVR, TSD, distraction, rewards, general anesthesia, hand-holding by parent, and mouthprops. When parents evaluated only BGTs used for their child, all BGTs, including a stabilization device, were highly acceptable (>91%), except for staff restraint (74%). Conclusions: Parents were accurate in predicting cooperation for some procedures. The most acceptable and efficacious BGTs in order were: PVR, TSD, distraction, rewords, and hand-holding by parent. Parental perceptions of BGTs were influenced by whether or not they had been used for their child. (Pediatr Dent 2008;30:400-7) Received June 22, 2007 / Last Revision October 10, 2007 / Revision Accepted October 15, 2007
C1 [Sheller, Barbara] Childrens Hosp & Reg Med Ctr, Dept Dent Med, Seattle, WA USA.
[Mancl, Lloyd] Univ Washington, Sch Dent, Dept Dent Publ Hlth Sci, Seattle, WA 98195 USA.
EM mammelon@hotmail.com
CR *AAPD, GUID US AN CAR PROV
*AAPD, GUID BEH GUID PED DE
Adair SM, 2004, PEDIATR DENT, V26, P167
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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*US BUR CENS, CENS 2000 DAT STAT W
NR 37
TC 10
Z9 11
PU AMER ACAD PEDIATRIC DENTISTRY
PI CHICAGO
PA 211 E CHICAGO AVENUE SUITE 1036, CHICAGO, IL 60611-2616 USA
SN 0164-1263
J9 PEDIATR DENT
JI Pediatr. Dent.
PD SEP-OCT
PY 2008
VL 30
IS 5
BP 400
EP 407
PG 8
WC Dentistry, Oral Surgery & Medicine; Pediatrics
SC Dentistry, Oral Surgery & Medicine; Pediatrics
GA V13JR
UT WOS:000207663600005
PM 18942599
ER
PT J
AU Kuwaik, G
Roberts, W
Brian, J
Bryson, S
Smith, IM
Szatmari, P
Zwaigenbaum, L
AF Kuwaik, Ghassan
Roberts, Wendy
Brian, Jessica
Bryson, Susan
Smith, Isabel M.
Szatmari, Peter
Zwaigenbaum, Lonnie
TI Immunization uptake in siblings of children with autism
SO PEDIATRICS
LA English
DT Letter
C1 [Kuwaik, Ghassan] Bloorview Kids Rehab, Child Dev Program, Toronto, ON M4G 1R8, Canada.
[Kuwaik, Ghassan; Roberts, Wendy; Brian, Jessica] Hosp Sick Children, Autism Res Unit, Toronto, ON M5G 1X8, Canada.
[Roberts, Wendy] Univ Toronto, Dept Pediat, Toronto, ON M5S 1A8, Canada.
[Bryson, Susan; Smith, Isabel M.] Dalhousie Univ, Dept Pediat, Halifax B3K 6R8, NS, Canada.
[Bryson, Susan; Smith, Isabel M.] Dalhousie Univ, Dept Psychol, Halifax B3K 6R8, NS, Canada.
[Bryson, Susan; Smith, Isabel M.] IWK Hlth Ctr, Toronto M4G 1R8, ON, Canada.
[Szatmari, Peter] McMaster Univ, Dept Psychiat & Behav Neurosci, Hamilton, ON L8N 3Z5, Canada.
[Zwaigenbaum, Lonnie] Univ Alberta, Glenrose Rehabil Hosp, Dept Pediat, Edmonton, AB T5G 0B7, Canada.
RP Kuwaik, G (reprint author), Bloorview Kids Rehab, Child Dev Program, Toronto, ON M4G 1R8, Canada.
CR SMITH MJ, 2008, PEDIATRICS, V121
*TOR PUBL HLTH, MEASL CONT CIRC GREA
*WHO UN CHILDR FUN, REV NAT IMM COV 1980
NR 3
TC 3
Z9 3
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD SEP
PY 2008
VL 122
IS 3
BP 684
EP 685
DI 10.1542/peds.2008-1624
PG 5
WC Pediatrics
SC Pediatrics
GA 342ZR
UT WOS:000258822600044
PM 18762548
ER
PT J
AU Smith, MJ
Ellenberg, SS
Bell, LM
Rubin, DM
AF Smith, Michael J.
Ellenberg, Susan S.
Bell, Louis M.
Rubin, David M.
TI Immunization uptake in siblings of children with autism - Reply
SO PEDIATRICS
LA English
DT Letter
ID PARENTS
C1 [Smith, Michael J.] Univ Louisville, Div Pediat Infect Dis, Louisville, KY 40205 USA.
[Ellenberg, Susan S.; Rubin, David M.] Univ Penn, Ctr Clin Epidemiol & Biostat, Philadelphia, PA 19104 USA.
[Bell, Louis M.] Childrens Hosp Philadelphia, Div Infect Dis, Philadelphia, PA 19104 USA.
[Bell, Louis M.; Rubin, David M.] Childrens Hosp Philadelphia, Div Gen Pediat, Philadelphia, PA 19104 USA.
RP Smith, MJ (reprint author), Univ Louisville, Div Pediat Infect Dis, Louisville, KY 40205 USA.
CR Gellin BG, 2000, PEDIATRICS, V106, P1097, DOI 10.1542/peds.106.5.1097
Salmon DA, 2005, ARCH PEDIAT ADOL MED, V159, P470, DOI 10.1001/archpedi.159.5.470
SMITH MJ, 2008, PEDIATRICS, V121
Woo EJ, 2004, AM J PUBLIC HEALTH, V94, P990, DOI 10.2105/AJPH.94.6.990
NR 4
TC 0
Z9 0
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD SEP
PY 2008
VL 122
IS 3
BP 685
EP 686
DI 10.1542/peds.2008-1957
PG 5
WC Pediatrics
SC Pediatrics
GA 342ZR
UT WOS:000258822600045
ER
PT J
AU Cukier, HN
Perez, AM
Collins, AL
Zhou, ZL
Zoghbi, HY
Botas, J
AF Cukier, Holly N.
Perez, Alma M.
Collins, Ann L.
Zhou, Zhaolan
Zoghbi, Huda Y.
Botas, Juan
TI Genetic Modifiers of MeCP2 Function in Drosophila
SO PLOS GENETICS
LA English
DT Article
ID SEVERE MENTAL-RETARDATION; CPG-BINDING PROTEIN-2; RETT-SYNDROME BRAIN;
METHYL-CPG; DNA METHYLATION; NEUROLOGICAL SYMPTOMS; SYNDROME PHENOTYPE;
ANGELMAN-SYNDROME; UBE3A EXPRESSION; MOUSE MODEL
AB The levels of methyl-CpG-binding protein 2 (MeCP2) are critical for normal post-natal development and function of the nervous system. Loss of function of MeCP2, a transcriptional regulator involved in chromatin remodeling, causes classic Rett syndrome (RTT) as well as other related conditions characterized by autism, learning disabilities, or mental retardation. Increased dosage of MeCP2 also leads to clinically similar neurological disorders and mental retardation. To identify molecular mechanisms capable of compensating for altered MeCP2 levels, we generated transgenic Drosophila overexpressing human MeCP2. We find that MeCP2 associates with chromatin and is phosphorylated at serine 423 in Drosophila, as is found in mammals. MeCP2 overexpression leads to anatomical (i.e., disorganized eyes, ectopic wing veins) and behavioral (i.e., motor dysfunction) abnormalities. We used a candidate gene approach to identify genes that are able to compensate for abnormal phenotypes caused by MeCP2 increased activity. These genetic modifiers include other chromatin remodeling genes (Additional sex combs, corto, osa, Sex combs on midleg, and trithorax), the kinase tricornered, the UBE3A target pebble, and Drosophila homologues of the MeCP2 physical interactors Sin3a, REST, and N-CoR. These findings demonstrate that anatomical and behavioral phenotypes caused by MeCP2 activity can be ameliorated by altering other factors that might be more amenable to manipulation than MeCP2 itself.
C1 [Cukier, Holly N.; Perez, Alma M.; Collins, Ann L.; Zoghbi, Huda Y.] Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
[Zhou, Zhaolan] Childrens Hosp, Neurobiol Program, Boston, MA 02115 USA.
[Zhou, Zhaolan] Harvard Univ, Sch Med, Dept Neurol, Boston, MA 02115 USA.
[Zhou, Zhaolan] Harvard Univ, Sch Med, Dept Neurobiol, Boston, MA 02115 USA.
[Zoghbi, Huda Y.] Baylor Coll Med, Dept Neurosci, Houston, TX 77030 USA.
[Zoghbi, Huda Y.] Baylor Coll Med, Dept Pediat, Houston, TX 77030 USA.
[Zoghbi, Huda Y.] Baylor Coll Med, Howard Hughes Med Inst, Houston, TX 77030 USA.
[Botas, Juan] Baylor Coll Med, Dept Mol & Cellular Biol, Houston, TX 77030 USA.
RP Cukier, HN (reprint author), Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
EM jbotas@bcm.edu
FU Rett Syndrome Research Foundation; National Institute of General Medical
Sciences [1 F31 GM067501-01A1]; National Institute for Neurological
Disorders and Stroke [NS042179, NS057819]; National Institute of Child
Health and Human Development; Developmental Disabilities Research Center
[HD024064]
FX This work was supported by a G.E.A.R. award from the Rett Syndrome
Research Foundation to JB, and by the National Institute of General
Medical Sciences predoctoral fellowship to HNC (1 F31 GM067501-01A1),
National Institute for Neurological Disorders and Stroke grants to JB
(NS042179) and to HYZ. (NS057819), and National Institute of Child
Health and Human Development grant to the Baylor College of Medicine
Mental Retardation and Developmental Disabilities Research Center
(HD024064). HYZ is a Howard Hughes Medical Institute investigator.
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NR 58
TC 24
Z9 24
PU PUBLIC LIBRARY SCIENCE
PI SAN FRANCISCO
PA 185 BERRY ST, STE 1300, SAN FRANCISCO, CA 94107 USA
SN 1553-7390
J9 PLOS GENET
JI PLoS Genet.
PD SEP
PY 2008
VL 4
IS 9
AR e1000179
DI 10.1371/journal.pgen.1000179
PG 12
WC Genetics & Heredity
SC Genetics & Heredity
GA 365MR
UT WOS:000260411200030
PM 18773074
ER
PT J
AU Da Fonseca, D
Viellard, M
Fakra, E
Bastard-Rosset, D
Deruelle, C
Poinso, F
AF Da Fonseca, David
Viellard, Marine
Fakra, Eric
Bastard-Rosset, Delphine
Deruelle, Christine
Poinso, Francois
TI Schizophrenia or Asperger syndrome?
SO PRESSE MEDICALE
LA French
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDER; CHILDHOOD-ONSET SCHIZOPHRENIA; AUTISM;
CLASSIFICATION; CHILDREN
AB Patients with Asperger syndrome ore often diagnosed late or ore wrongly considered to hove schizophrenia.
Misdiagnosing Asperger syndrome creates serious problems by preventing effective therapy.
Several clinical signs described in Asperger syndrome could also be considered as clinical signs of schizophrenia, including impaired social interactions, disabilities in communication, restricted interests, and delusions of persecution.
A number of clinical features may facilitate the differential diagnosis: younger age at onset, family history of pervasive developmental disorder, recurring conversations on the some topic, pragmatic aspects of language use, oddities of intonation and pitch, lock of imagination, and incomprehension of social rules are more characteristic of Asperger syndrome.
Accurate distinction between Asperger syndrome and schizophrenia would make it possible to offer more treatment appropriate to the patient's functioning.
C1 [Da Fonseca, David; Viellard, Marine; Bastard-Rosset, Delphine; Poinso, Francois] Hop St Marguerite, Serv Pedopsychiat, Ctr Ressources Autisme PACA, F-13009 Marseille, France.
[Da Fonseca, David; Bastard-Rosset, Delphine; Deruelle, Christine; Poinso, Francois] CNRS, UMR 6193, Inst Neurosci Cognit Mediterranee, F-13000 Marseille, France.
[Da Fonseca, David; Bastard-Rosset, Delphine; Deruelle, Christine; Poinso, Francois] Univ Aix Marseille 2, F-13000 Marseille, France.
[Fakra, Eric] Hop St Marguerite, F-13009 Marseille, France.
RP Da Fonseca, D (reprint author), 270 Blvd St Marguerite, F-13009 Marseille, France.
EM david.dafonseca@ap-hm.fr
RI deruelle, christine/E-2130-2015
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TC 3
Z9 3
PU MASSON EDITEUR
PI MOULINEAUX CEDEX 9
PA 21 STREET CAMILLE DESMOULINS, ISSY, 92789 MOULINEAUX CEDEX 9, FRANCE
SN 0755-4982
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JI Presse Med.
PD SEP
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VL 37
IS 9
BP 1268
EP 1273
DI 10.1016/j.lpm.2008.01.020
PG 6
WC Medicine, General & Internal
SC General & Internal Medicine
GA 356YP
UT WOS:000259813800014
PM 18417316
ER
PT J
AU Davis, E
Saeed, SA
Antonacci, DJ
AF Davis, Ervin
Saeed, Sy Atezaz
Antonacci, Diana J.
TI Anxiety disorders in persons with developmental disabilities:
Empirically informed diagnosis and treatment - Reviews literature on
anxiety disorders in DD population with practical take-home messages for
the clinician
SO PSYCHIATRIC QUARTERLY
LA English
DT Article
DE developmental disability; anxiety; diagnosis; treatment
ID OBSESSIVE-COMPULSIVE DISORDER; SEROTONIN REUPTAKE INHIBITORS;
POSTTRAUMATIC-STRESS-DISORDER; SEVERE MENTAL-RETARDATION; INTELLECTUAL
DISABILITY; PSYCHIATRIC-DISORDERS; AUTISTIC DISORDER; RETARDED ADULTS;
NORMAL-CHILDREN; DUAL DIAGNOSIS
AB Anxiety disorders are common in individuals with developmental disabilities (DDs), although they may not be diagnosed and treated as often as they are in patients without DDs. Patients with mental retardation, autism, and other pervasive developmental disorders may exhibit comorbid anxiety disorders, such as generalized anxiety disorder (GAD), obsessive-compulsive disorder (OCD), phobias, and other anxiety symptoms at much higher rates than in the general population, but identification of these comorbid anxiety disorders may be made more difficult by the presence of the DD and concurrent difficulties with communication, other behavior problems, the lack of standardized assessments specific to diagnosing patients with DDs and psychiatric comorbidities, and the need for greater collateral sources of assessment information. In addition, systematic study of the treatment of anxiety in patients with DD is limited to a relatively small number of empirical studies done specifically in these patients along with case reports and theoretical reviews on the extension and modification of more well-studied treatments used for anxiety in patients without DDs. The present article reviews the literature on the prevalence, features, assessment and diagnosis of anxiety disorders in individuals with DDs, and also reviews empirical studies of pharmacological and psychological treatment of patients with comorbid anxiety and DD and summarizes the findings. Recommendations are made to guide treatment and further research in this area.
C1 [Davis, Ervin] E Carolina Univ, Dept Psychol, Greenville, NC 27858 USA.
[Saeed, Sy Atezaz; Antonacci, Diana J.] E Carolina Univ, Dept Psychiat Med, Brody Sch Med, Greenville, NC 27858 USA.
RP Davis, E (reprint author), E Carolina Univ, Dept Psychol, Rawl Bldg, Greenville, NC 27858 USA.
EM daviscl@ecu.edu
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NR 82
TC 19
Z9 19
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0033-2720
J9 PSYCHIAT QUART
JI Psychiatr. Q.
PD SEP
PY 2008
VL 79
IS 3
BP 249
EP 263
DI 10.1007/s11126-008-9081-3
PG 15
WC Psychiatry
SC Psychiatry
GA 346QR
UT WOS:000259084600007
PM 18726156
ER
PT J
AU Lahuis, BE
Durston, S
Nederveen, H
Zeegers, M
Palmen, SJMC
Van Engeland, H
AF Lahuis, B. E.
Durston, S.
Nederveen, H.
Zeegers, M.
Palmen, S. J. M. C.
Van Engeland, H.
TI MRI-based morphometry in children with multiple complex developmental
disorder, a phenotypically defined subtype of pervasive developmental
disorder not otherwise specified
SO PSYCHOLOGICAL MEDICINE
LA English
DT Article
DE autism; MCDD; MRI; PPD-NOS
ID AUTISM DIAGNOSTIC INTERVIEW; BORDERLINE SYNDROME; SPECTRUM DISORDERS;
HEAD CIRCUMFERENCE; BRAIN; CHILDHOOD; CLASSIFICATION; ABNORMALITIES;
EPIDEMIOLOGY; VOLUME
AB Background. The DSM-IV-R classification Pervasive Developmental Disorder - Not otherwise Specified (PDD-NOS) is based on the symptoms for autism and includes a wide variety of phenotypes that do not meet full criteria for autism. As such, PDD-NOS is a broad and poorly defined residual category of the autism spectrum disorders. In order to address the heterogeneity in this residual category it may be helpful to define clinical and neurobiological subtypes. Multiple complex developmental disorder (MCDD) may constitute such a subtype. In order to study the neurobiological specificity of MCDD in comparison with other autism spectrum disorders, we investigated brain morphology in children (age 7-15 years) with MCDD compared to children with autism and typically developing controls.
Method. Structural MRI measures were compared between 22 high-functioning subjects with MCDD and 21 high-functioning subjects with autism, and 21 matched controls.
Results. Subjects with MCDD showed an enlarged cerebellum and a trend towards larger grey-matter volume compared to control subjects. Compared to subjects with autism, subjects with MCDD had smaller intracranial volume.
Conclusions. We report a pattern of volumetric changes in the brains of subjects with MCDD, similar to that seen in autism. However, no enlargement in head size was found. This suggests that although some of the neurobiological changes associated with MCDD overlap with those in autism, others do not. These neurobiological changes may reflect differences in the developmental trajectories associated with these two subtypes of autism spectrum disorders.
C1 Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Utrecht, Netherlands.
Univ Med Ctr Utrecht, Rudolf Magnus Inst Neurosci, Utrecht, Netherlands.
RP Lahuis, BE (reprint author), Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, B 01201,POB 85500, Utrecht, Netherlands.
EM b.e.lahuis@umcutrecht.nl
FU Korczak Foundation
FX The authors acknowledge Rutger Jan van der Gaag, Margreet Scherpenisse,
and Jacqueline Jansen, for their contribution to subject recruitment and
screening. Research support was provided by the Korczak Foundation.
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NR 28
TC 5
Z9 5
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0033-2917
J9 PSYCHOL MED
JI Psychol. Med.
PD SEP
PY 2008
VL 38
IS 9
BP 1361
EP 1367
DI 10.1017/S0033291707001481
PG 7
WC Psychology, Clinical; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 344FF
UT WOS:000258911400015
PM 17825125
ER
PT J
AU Santaella, ML
Varela, Y
Linares, N
Disdier, OM
AF Santaella, Maria L.
Varela, Yessica
Linares, Nicolas
Disdier, Orville M.
TI Prevalence of autism spectrum disorders in relatives of patients with
selective immunoglobulin A deficiency
SO PUERTO RICO HEALTH SCIENCES JOURNAL
LA English
DT Article
DE autism; autism specitrum disorders; selective; IgA deficiency
ID UNITED-STATES; CHILDREN; ASSOCIATION; PROTEIN
AB Background: An association of selective IgA (immunoglobulin A) deficiency in individuals with autism has been previously described. The objective of this study was to examine the incidence of autism spectrum disorders (ASD) in children and siblings of selective IgA deficient patients. Objective: to assess the likelihood of parents with the most common type of primary immunodeficiency (selective IgA deficiency) having children with ASD and to investigate the occurrence of ASD in siblings of the immunodeficient patients.
Methods: A study was conducted in 31 selective IgA deficient patients and 62 age and gender-matched controls. Children and siblings of IgA deficient patients and controls were screened for an ASD (autism spectrum disorder) using a standard questionnaire.
Results: Only one patient in the IgA deficient group had classical autism. Three children in that group (10.3%) had an ASD compared to only one in the control group (1.6%) and this difference was statistically significant. In terms of siblings, there was a higher occurrence of an ASD in the IgA deficient group than in the control group, but the difference was not statistically significant. A high incidence of allergies (71%) was documented in IgA deficient patients. All individuals with allergies had food sensitivities. There was a predominance of the male gender in cases identified with an ASD in all groups.
Conclusions: A lower prevalence of ASD was observed in the IgA deficient group, as compared to other reports. The study suggests that screening for an ASD seems appropriate for children of IgA deficient patients.
C1 [Santaella, Maria L.; Varela, Yessica; Linares, Nicolas] Univ Puerto Rico, FILIUS Inst, San Juan, PR 00926 USA.
RP Santaella, ML (reprint author), Univ Puerto Rico, FILIUS Inst, 1187 Calle Flamboyan, San Juan, PR 00926 USA.
EM lousant@prtc.net
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NR 20
TC 3
Z9 3
PU UNIV PUERTO RICO MEDICAL SCIENCES CAMPUS
PI SAN JUAN
PA OFFICE DEAN ACADEMIC AFFAIRS BOX 365067, SAN JUAN, PR 00936-5067 USA
SN 0738-0658
J9 P R HEALTH SCI J
JI P. R. Health Sci. J.
PD SEP
PY 2008
VL 27
IS 3
BP 204
EP 208
PG 5
WC Public, Environmental & Occupational Health
SC Public, Environmental & Occupational Health
GA 349BX
UT WOS:000259254900003
PM 18782963
ER
PT J
AU Friesen, E
AF Friesen, Eric
TI Oliver Sacks Interviewed
SO QUEENS QUARTERLY
LA English
DT Editorial Material
AB Dr Oliver Sacks was born in London to a family of physicians and scientists. He earned his medical degree at Oxford University, and has lived in New York since 1965. As a neurologist, he has written extensively on the struggles of human beings afflicted by Tourette's Syndrome, autism, parkinsonism, musical hallucination, Alzheimer's Disease, and many other conditions. His latest book examines the deep and complex relationship between music and the human brain. CBC Radio's Eric Friesen, host of Studio Sparks, recently interviewed him in Ottawa.
NR 0
TC 0
Z9 0
PU QUEENS QUARTERLY
PI KINGSTON
PA QUEENS UNIV, KINGSTON, ON K7L 3N6, CANADA
SN 0033-6041
J9 QUEENS QUART
JI Queens Q.
PD FAL
PY 2008
VL 115
IS 3
BP 443
EP 454
PG 12
WC Literary Reviews
SC Literature
GA 372DF
UT WOS:000260881200009
ER
PT J
AU Kliewer, C
AF Kliewer, Christopher
TI Joining the Literacy Flow: Fostering Symbol and Written Language
Learning in Young Children With Significant Developmental Disabilities
Through the Four Currents of Literacy
SO RESEARCH AND PRACTICE FOR PERSONS WITH SEVERE DISABILITIES
LA English
DT Article
DE early childhood inclusion; literacy; severe disability
ID MULTIPLE DISABILITIES; MENTAL-RETARDATION; EMERGENT LITERACY;
COMMUNICATION; INDIVIDUALS; INSTRUCTION; AWARENESS; STUDENTS; AUTISM
AB This article is both an ethnographic and an action-based description of how excellent early, childhood teachers in seven inclusive preschool and kindergarten classrooms fostered the developing literacy profiles of young children with significant developmental disabilities alongside their typically developing peers through active, engaging, social means. I have developed four broad themes, described here as currents, that support the meaning-based literacy-learning of children still commonly cast as intrinsically incapable of literate citizenship, using participant observation; in-depth interviews with teachers, therapists, and parents; the implementation of increasingly responsive, systematic literacy-based themes, opportunities, and activities into certain inclusive classrooms; and the development of process-oriented portfolio documentation. Important findings include the following: (a) children with significant developmental disabilities can join the early childhood literacy flow; (b) they do so through interactive means; and (c) spoken language need not serve as the foundation of written language learning.
C1 Univ No Iowa, Dept Special Educ, Cedar Falls, IA 50614 USA.
RP Kliewer, C (reprint author), Univ No Iowa, Dept Special Educ, Cedar Falls, IA 50614 USA.
EM kliewer@uni.edu
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NR 73
TC 3
Z9 3
PU TASH
PI WASHINGTON
PA 1025 VERMONT AVE, NW 7TH FLR, WASHINGTON, DC 20005 USA
SN 0274-9483
J9 RES PRACT PERS SEV D
JI Res. Pract. Pers. Sev. Disabil.
PD FAL
PY 2008
VL 33
IS 3
BP 103
EP 121
PG 19
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 501ZH
UT WOS:000270422900005
ER
PT J
AU Dukes, C
Wade, D
AF Dukes, Charles
Wade, Dorett
TI Autism & the Transition to Adulthood-Success Beyond the Classroom.
SO RESEARCH AND PRACTICE FOR PERSONS WITH SEVERE DISABILITIES
LA English
DT Book Review
ID BEHAVIOR
C1 [Dukes, Charles; Wade, Dorett] Florida Atlantic Univ, Boca Raton, FL 33431 USA.
[Dukes, Charles; Wade, Dorett] Broward Cty Publ Sch, Ft Lauderdale, FL USA.
RP Dukes, C (reprint author), Florida Atlantic Univ, Boca Raton, FL 33431 USA.
CR Carothers D. E., 2004, FOCUS AUTISM OTHER D, V19, P102, DOI 10.1177/10883576040190020501
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Maurice C., 1996, BEHAV INTERVENTIONS
Maurice C, 2001, MAKING DIFFERENCE BE
SCHMITZ T, 2008, EXCEPTIONAL PARENT, V38, P37
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WEHMAN P, AUTISM TRANSITION A
Zhang D., 2005, CAREER DEV EXCEPTION, V28, P15
NR 10
TC 0
Z9 0
PU TASH
PI WASHINGTON
PA 1025 VERMONT AVE, NW 7TH FLR, WASHINGTON, DC 20005 USA
SN 0274-9483
J9 RES PRACT PERS SEV D
JI Res. Pract. Pers. Sev. Disabil.
PD FAL
PY 2008
VL 33
IS 3
BP 170
EP 171
PG 2
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 501ZH
UT WOS:000270422900016
ER
PT J
AU Peters-Scheffer, N
Didden, R
Green, VA
Sigafoos, J
Korzilius, H
Pituch, K
O'Reilly, MF
Lancioni, G
AF Peters-Scheffer, Nienke
Didden, Robert
Green, Vanessa A.
Sigafoos, Jeff
Korzilius, Hubert
Pituch, Keenan
O'Reilly, Mark F.
Lancioni, Giulio
TI The behavior flexibility rating scale-revised (BFRS-R): Factor analysis,
internal consistency, inter-rater and intra-rater reliability, and
convergent validity
SO RESEARCH IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE behavior flexibility rating scale-revised (BFRS-R); children with
developmental disabilities; factor analysis; internal consistency;
inter-rater reliability; intra-rater reliability; convergent validity
ID AUTISM; DISORDERS
AB We examined the psychometric properties of the behavior flexibility rating scale-revised (BFRS-R), a new scale intended for assessing behavioral flexibility in individuals with developmental disabilities. Seventy-six direct care staff members and 56 parents completed the BFRS-R for 70 children with developmental disabilities. Factor analysis revealed three factors (i.e., Flexibility towards objects, Flexibility towards the environment, and Flexibility towards persons) and results of several analyses indicated an excellent internal consistency and good intra-rater and inter-rater reliability of the total scale. These data suggest that the BFRS-R may provide a reliable rating of behavioral flexibility when used by direct-care staff and parents of children with developmental disabilities. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Peters-Scheffer, Nienke; Didden, Robert; Korzilius, Hubert] Radboud Univ Nijmegen, Dept Special Educ, NL-6500 HE Nijmegen, Netherlands.
[Green, Vanessa A.; Sigafoos, Jeff] Univ Tasmania, Sch Educ, Hobart, Tas 7001, Australia.
[Pituch, Keenan; O'Reilly, Mark F.] Univ Texas Austin, Dept Special Educ, Austin, TX 78712 USA.
[Lancioni, Giulio] Univ Bari, Dept Psychol, I-70121 Bari, Italy.
RP Didden, R (reprint author), Radboud Univ Nijmegen, Dept Special Educ, POB 9104, NL-6500 HE Nijmegen, Netherlands.
EM R.Didden@pwo.ru.nl
RI Peters-Scheffer, Nienke`/F-1012-2012
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NR 9
TC 15
Z9 15
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0891-4222
J9 RES DEV DISABIL
JI Res. Dev. Disabil.
PD SEP-OCT
PY 2008
VL 29
IS 5
BP 398
EP 407
DI 10.1016/j.ridd.2007.07.004
PG 10
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 347QK
UT WOS:000259155900002
PM 17826945
ER
PT J
AU Ben Itzchak, E
Lahat, E
Burgin, R
Zachor, AD
AF Ben Itzchak, Esther
Lahat, Eliezer
Burgin, Ruth
Zachor, A. Ditza
TI Cognitive, behavior and intervention outcome in young children with
autism
SO RESEARCH IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE autism spectrum disorder; developmental disabilities; cognition; ADOS;
IQ
ID PERVASIVE DEVELOPMENTAL DISORDER; 6-YEAR FOLLOW-UP; HEAD CIRCUMFERENCE;
MENTAL-RETARDATION; AGE; IQ; PRESCHOOLERS; PREDICTORS; DIAGNOSIS; BIRTH
AB The relations between cognition and autism severity, head size and intervention outcome, were examined. Change in cognitive level with intervention was measured in children with autism and compared to children with developmental disabilities (DD). Eighty-one children (mean age 25.9 months) with autism (n = 44) and DD (n = 37) were assessed at pre- and post I year of intervention. Cognitive abilities and autism severity were measured by standardized tests. Three pre-intervention cognitive level groups: normal (IQ > 90), borderline (70 < IQ < 89) and impaired (50 < IQ < 69) were examined. The impaired group had more severe autism symptoms than the borderline and the normal cognitive groups. However, following intervention the groups did not differ in the change in core autism symptoms. IQ scores increased significantly more in the autism group than in the DD group. IQ improvements correlated significantly with reduction in autism symptoms and mostly in stereotyped behaviors.
Conclusions: Cognitive ability in autism is associated with autism severity. Two distinct subtypes based on cognitive level are identified. However, baseline cognitive level cannot predict the progress rate in autism symptoms with intervention. Improvement of social-communicative behaviors and the intensive intervention are related to significant cognitive increments in autism. (c) 2007 Elsevier Ltd. All fights reserved.
C1 [Ben Itzchak, Esther] Asaf Harofeh Med Ctr, Autism Ctr, Univ Ctr Samaria, Dept Commun Disorders, Zerifin, Israel.
[Lahat, Eliezer] Tel Aviv Univ, Sch Med, Asaf Harofeh Med Ctr, Child Dev Ctr, Tel Aviv, Israel.
[Zachor, A. Ditza] Tel Aviv Univ, Sch Med, Asaf Harofeh Med Ctr, Autism Ctr, Tel Aviv, Israel.
RP Ben Itzchak, E (reprint author), Asaf Harofeh Med Ctr, Autism Ctr, Univ Ctr Samaria, Dept Commun Disorders, Zerifin, Israel.
EM benitze@mail.biu.ac.il
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NR 50
TC 56
Z9 56
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0891-4222
J9 RES DEV DISABIL
JI Res. Dev. Disabil.
PD SEP-OCT
PY 2008
VL 29
IS 5
BP 447
EP 458
DI 10.1016/j.ridd.2007.08.003
PG 12
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 347QK
UT WOS:000259155900007
PM 17923388
ER
PT J
AU Hsieh, HC
AF Hsieh, Hsieh-Chun
TI Effects of ordinary and adaptive toys on pre-school children with
developmental disabilities
SO RESEARCH IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE adaptive toys; early intervention; play; developmental disabilities;
single-subject design
ID YOUNG-CHILDREN; PLAY; AUTISM
AB Toys help children in mastering developmental tasks. This study investigated toy effect on children with developmental disabilities as they engage in using ordinary and adaptive toys. A single-subject design was used to identify the effects on their toy play abilities. Differences in toy effects between playing ordinary and adaptive toys were examined. Three special education teachers chose ordinary toys and modified ordinary toys. Modified ordinary toys, i.e., adaptive toys, were designed according to the individual disabilities of participating children, treatment goals, and the toy types. Three children with developmental disabilities from pre-schools in Taiwan were enrolled. Appropriate participation of three pre-schoolers increased dramatically in playing adaptive toys during intervention phase. The toy effects demonstrate that when using adaptive toys, children with developmental disabilities may response better during toy play sessions. (c) 2007 Elsevier Ltd. All rights reserved.
C1 Natl Hsinchu Univ Educ, Dept Special Educ, Hsinchu 300, Taiwan.
RP Hsieh, HC (reprint author), Natl Hsinchu Univ Educ, Dept Special Educ, 521 Nanda Rd, Hsinchu 300, Taiwan.
EM elsajj@mail.nhcue.edu.tw
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NR 22
TC 7
Z9 9
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0891-4222
J9 RES DEV DISABIL
JI Res. Dev. Disabil.
PD SEP-OCT
PY 2008
VL 29
IS 5
BP 459
EP 466
DI 10.1016/j.ridd.2007.08.004
PG 8
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 347QK
UT WOS:000259155900008
PM 17936580
ER
PT J
AU Gallup, GG
Frederick, MJ
Pipitone, RN
AF Gallup, Gordon G., Jr.
Frederick, Michael J.
Pipitone, R. Nathan
TI Morphology and behavior: Phrenology revisited
SO REVIEW OF GENERAL PSYCHOLOGY
LA English
DT Article
DE fluctuating asymmetry; facial attractiveness; body configuration;
reproductive competition; evolution and behavior
ID 4TH DIGIT RATIO; TO-HIP RATIO; DEFICIT HYPERACTIVITY DISORDER; FEMALE
PHYSICAL ATTRACTIVENESS; FLUCTUATING ASYMMETRY; FACIAL ATTRACTIVENESS;
SEXUAL-BEHAVIOR; FINGER-LENGTH; DEVELOPMENTAL INSTABILITY; COLLEGE
SAMPLE
AB Research conducted by evolutionary psychologists and biologists shows that Subtle individual differences in body morphology can be related to Surprising and important differences in human behavior and reproductive Success. The authors summarize recent work on these effects as they relate to fluctuating asymmetry, facial attractiveness, finger digit morphology, sexually dimorphic differences in body configuration, and head circumference. Examples include the discovery that women who have sex with bilaterally symmetrical men report more orgasms; men with attractive faces have higher quality sperm; the length of the index finger in relation to the ring finger is related to verbal fluency, spatial ability, and the risk of autism; women with all hourglass figure have more regular menstrual cycles and are more fertile; and the Sound of a person's voice predicts his or her sexual behavior.
C1 [Gallup, Gordon G., Jr.; Frederick, Michael J.; Pipitone, R. Nathan] SUNY Albany, Dept Psychol, Albany, NY 12222 USA.
RP Gallup, GG (reprint author), SUNY Albany, Dept Psychol, 1400 Washington Ave, Albany, NY 12222 USA.
EM gallup@albany.edu
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NR 75
TC 6
Z9 6
PU EDUCATIONAL PUBLISHING FOUNDATION-AMERICAN PSYCHOLOGICAL ASSOC
PI WASHINGTON
PA 750 FIRST ST, NE, WASHINGTON, DC 20002-4242 USA
SN 1089-2680
J9 REV GEN PSYCHOL
JI Rev. Gen. Psychol.
PD SEP
PY 2008
VL 12
IS 3
BP 297
EP 304
DI 10.1037/1089-2680.12.3.297
PG 8
WC Psychology, Multidisciplinary
SC Psychology
GA 343NS
UT WOS:000258861400006
ER
PT J
AU Marteleto, MRF
Menezes, CGDE
Tamanaha, AC
Chiari, BM
Perissinoto, J
AF Fumagalli Marteleto, Marcia Regina
de Lima e Menezes, Camila Gioconda
Tamanaha, Ana Carina
Chiari, Brasilia Maria
Perissinoto, Jacy
TI Administration of the Autism Behavior Checklist: agreement between
parents and professionals' observations in two intervention contexts
SO REVISTA BRASILEIRA DE PSIQUIATRIA
LA English
DT Article
DE autistic disorder; behavior; evaluation studies; questionnaires;
reproducibility of results
ID CHILDHOOD DEPRESSION; RELIABILITY; DISORDERS; SYMPTOMS; VALIDITY;
INDIVIDUALS; PERCEPTIONS; ADOLESCENT; DIAGNOSES; MOTHERS
AB Objective: To determine the agreement between responses on the Autism Behavior Checklist by mothers and healthcare professionals. Method: Twenty-three mothers of children from the autism spectrum (DSM-IV-TR, 2002) were interviewed. The children were part of an educational program of the Autistic Friends Association-SP The healthcare professionals in charge of the children at the institution filled out a questionnaire regarding the method for observing behavior For comparison purposes, an additional 15 mothers of children from the autism spectrum were interviewed at the speech therapy clinic of Universidade Federal de Sao Paulo, and the speech therapists responsible for the children filled out the questionnaire on the observation method. The Kappa index was employed to obtain the frequency of agreement between mothers and healthcare professionals' observations on the overall Autism Behavior Checklist, as well as its individual components. The Student's t-test was used to assess differences or similarities between the Kappa results. Results: The agreement index was low for the total score, with a statistically significant difference between both groups (p < 0.001). Agreement was also low for each Autism Behavior Checklist components, but there was greater agreement in the group treated at the public service in the Language, Body and Use of Objects components (p < 0.001). Conclusion: There was disagreement between the observations in both intervention contexts, but agreement was better at the public service than at Autistic Friends Association.
C1 [Fumagalli Marteleto, Marcia Regina; de Lima e Menezes, Camila Gioconda] Univ Fed Sao Paulo, Dept Fonoaudiol, Posgrad Program Human Commun Disorders, BR-04023900 Sao Paulo, Brazil.
[Tamanaha, Ana Carina; Chiari, Brasilia Maria; Perissinoto, Jacy] Univ Fed Sao Paulo, Dept Speech & Language Therapy, BR-04023900 Sao Paulo, Brazil.
RP Marteleto, MRF (reprint author), Univ Fed Sao Paulo, Dept Fonoaudiol, Posgrad Program Human Commun Disorders, Rua Botucatu,802 Vila Clementino, BR-04023900 Sao Paulo, Brazil.
EM marcia.marteleto@terra.com.br
RI Tamanaha, Ana Carina/J-5942-2012
OI Tamanaha, Ana Carina/0000-0001-9915-6299
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NR 38
TC 4
Z9 8
PU ASSOC BRASILEIRA PSIQUIATRIA
PI SAO PAULO
PA SUBSCRIPTION DEPARTMENT, RUA PEDRO DE TOLEDO, 967 - CASA 01, SAO PAULO,
SP 04039-032 A, BRAZIL
SN 1516-4446
J9 REV BRAS PSIQUIATR
JI Rev. Bras. Psiquiatr.
PD SEP
PY 2008
VL 30
IS 3
BP 203
EP 208
DI 10.1590/S1516-44462008000300005
PG 6
WC Psychiatry
SC Psychiatry
GA 348FS
UT WOS:000259197000006
PM 18833419
ER
PT J
AU Gomez, SL
Torres, RMR
Ares, EMT
AF Lopez Gomez, Santiago
Rivas Torres, Rosa Ma.
Taboada Ares, Eva Ma.
TI Detection of the perinatal maternal risks in the pervasive developmental
disorders
SO SALUD MENTAL
LA Spanish
DT Article
DE Pervasive Developmental Disorders (PDD); perinatal risk
ID INFANTILE-AUTISM; NEONATAL FACTORS; COMPLICATIONS; EPIDEMIOLOGY;
POPULATION; PREDICTORS; CALIFORNIA; PREGNANCY; CHILDREN; MOTHERS
AB Pervasive Developmental Disorders (PDD) refer to a group of severe neuropsychologic alterations. Symptoms affect three development components: social-interaction skills, language and communication skills and a set of behaviours and activities that become restricted and stereotyped. PDDs include the following disorders: Autistic, Rett's, Infantile Disintegrative, Asperger's and Generalized Non-specific Development Disorder.
Regarding to its unknown causes, several explanations have been gathered as a challenging task. They highlight the idea of generalised alterations in the Central Nervous System (CNS). However, the strongest thesis defines a multicausal etiology, with different factors associated to PDDs.
Never the less, over the past few years, the review of problems associated with pregnancy and tabour have been stressed. This perspective is complemented by other elements that point towards genetic alterations and CNS deficits as causes behind PDD. It has been suggested that pregnancy, labour and even neonatal complications can act on different fronts: increasing the risk of autism or any other PDD, or interacting along with genetic determinants to increase the potential risk of a critical moment in the perinatal development process.
The goal of this paper is to study the presence of perinatal risk in mothers of children with and without PDD.
A total of 259 mothers took part in the study; 95 were used as an experimental group: they all hod a PDD-diagnosed. child, according to DSM-IV-TR criteria (68 had autistic disorder, six had Asperger's disorder, one had Rett's disorder and 19 had non-specific PDD). The remaining 165 women had children with a normal evolutive development and were selected as a control group.
In order to collect information about perinatal risk, a Maternal Perinatal Risk Questionnaire (MPRQ) was used. This is a structured and specifically-designed autoreport that evaluates the presence or absence of 40 pregestational and perigestational risk factors annalysed from six perspectives: pregestational, perigestational, intrapartum, neonatal, psychosocial and sociodemographic.
For every factor evaluated in the MPRQ, an analysis of the average scores and typical deviations was made, along with a frequency and percentage study. Furthermore, a comparative of the frequencies in the control and experimental groups was carried out for every MPRQ item.
By means of descriptive analysis, both groups were classified according to the children's age and birth order, the mother's age during pregnancy, current parent's age and their educational and professional levels.
When comparing the experimental group's frequencies to those of the control group in the pregestational stage, two significative items were found in Chi-square: the number of previous spontaneous abortions and the use of contraceptive methods. As a result, the control group had fewer spontaneous abortions than the experimental group (10.9% and 22.4% respectively). The use of contraceptive methods previous to pregnancy described the control group's superiority both for hormonal methods and intrauterine devices (IUD). The experimental group was defined by the absence of IUD and the scarce use of hormonal contraceptives (4.3%).
In the perigestational dimension, the three significative items in Chi-square were: pharmacological consumption and presence of edema during pregnancy, and premature rupture of amniotic sac.
Pharmacological consumption during pregnancy stresses the consumption of medicine or vitamines and iron in control group (81.2%), compared to the group of mothers of children with PDD (60.6%).
The presence of gestational edema has been conclusively linked to the control group.
As for the premature rupture of waters, a significantly higher presence of amniotic rupture was found in the experimental group compared to the control group.
In the intrapartum dimension, the experimental group confirmed higher frequencies in situations that imply a higher perinatal risk such as: a very quick tabour or one lasting over 12 hours.
In the neonatal dimension, the control group showed with higher percentages (87.9%), the absence of blue coloration-which would be indicative of cyanosis-, when compared to the experimental group (79.8%).
The psycho-social dimension included two significant items: the desired gender for the newborn and the desired pregnancy. The desired gender item confirmed that situations of happiness about finding out the baby's gender were higher in the control group (68.4%) than in the experimental group. The desired pregnancy item proved that situations of desired pregnancy were higher in the control group (91.5%) compared to the experimental group (84.0%).
Finally, in the socio-demographic dimension, two siginificative items were identified when comparing both groups: the mother's profession and the baby's gender.
In one hand, regarding the mother's profession, it was observed that mothers of PDD children were mainly found within home enviroment (37.2%) or unqualfied worker categories (18.1%). On the other hand, in the control group, the mothers who adscribed to the qualified professional category was notably higher (33.3%).
Regarding the child's gender, a higher risk is detected in males, at a proportion of 1 to 3.
The results of this study showed that there are significative differences between PDD children who developed perinatal risks, compared to children who have a regular evolutive development. Children with PDD will thus show significant differences compared to non-PDD children: they have an unequal perinatal development and developed perinatal risks. Therefore, many risks are present in a higher measure in PDD children when compared to the control group. An innovative contribution is also made, by strongly suggesting that physical risks define the presence of perinatal risks in PDD. However, the psychosocial and sociodemographic dimensions must also be taken into account.
C1 [Lopez Gomez, Santiago; Rivas Torres, Rosa Ma.; Taboada Ares, Eva Ma.] Univ Santiago de Compostela, Fac Psicol, Dpto Psicol Evolut & Educ, Santiago De Compostela 15782, Spain.
RP Torres, RMR (reprint author), Univ Santiago de Compostela, Fac Psicol, Dpto Psicol Evolut & Educ, Campus Sur, Santiago De Compostela 15782, Spain.
EM perivas@usc.es
RI Lopez Gomez, Santiago/D-8916-2011
OI Lopez Gomez, Santiago/0000-0001-9519-4722
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NR 42
TC 3
Z9 3
PU INST MEX PSIQUIATRIA
PI MEXICO CITY
PA CALZ MEXICO-XOCHIMILCO #101, MEXICO CITY 22 DF, MEXICO
SN 0185-3325
J9 SALUD MENT
JI Salud Ment.
PD SEP-OCT
PY 2008
VL 31
IS 5
BP 371
EP 379
PG 9
WC Psychiatry
SC Psychiatry
GA 372ZZ
UT WOS:000260942100005
ER
PT J
AU Fatemi, SH
Folsom, TD
Reutiman, TJ
Pandian, T
Braun, NN
Haug, K
AF Fatemi, S. Hossein
Folsom, Timothy D.
Reutiman, Teri J.
Pandian, Twinkle
Braun, Natalie N.
Haug, Kari
TI Chronic psychotropic drug treatment causes differential expression of
connexin 43 and GFAP in frontal cortex of rats
SO SCHIZOPHRENIA RESEARCH
LA English
DT Article
DE connexin 43; GFAP; rat; prefrontal cortex; schizophrenia; autism
ID FIBRILLARY ACIDIC PROTEIN; CENTRAL-NERVOUS-SYSTEM; GAP-JUNCTIONS;
PREFRONTAL CORTEX; BIPOLAR DISORDER; NEONATAL MICE; SCHIZOPHRENIA;
BRAIN; ASTROCYTES; AUTISM
AB Astrocytic markers glial fibrillary acidic protein (GFAP) and connexin 43 (CX43) are known to have altered expression ill brains of subjects with psychiatric disorders including autism and major depression. The current study investigated whether GFAP and CX43 expressions are affected by several commonly used psychotropic medications (clozapine, fluoxetine, haloperidol, lithium, olanzapine, and valproic acid). Using SDS-PAGE and western blotting technique, we observed that CX43 protein expression in prefrontal cortex was significantly increased following chronic treatment with fluoxetine and clozapine, while it was significantly decreased by haloperidol and lithium. GFAP protein expression was significantly decreased following chronic treatment with clozapine and valproic acid. These results suggest that astroglial markers GFAP and CX43 could be potential targets for therapeutic intervention. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Fatemi, S. Hossein; Folsom, Timothy D.; Reutiman, Teri J.; Pandian, Twinkle; Braun, Natalie N.; Haug, Kari] Univ Minnesota, Sch Med, Dept Psychiat, Div Neurosci Res, Minneapolis, MN 55455 USA.
[Fatemi, S. Hossein] Univ Minnesota, Sch Med, Dept Pharmacol, Minneapolis, MN 55455 USA.
[Fatemi, S. Hossein] Univ Minnesota, Sch Med, Dept Neurosci, Minneapolis, MN 55455 USA.
[Pandian, Twinkle] Mayo Clin & Mayo Fdn, Mayo Med Sch, Rochester, MN 55905 USA.
RP Fatemi, SH (reprint author), 420 Delaware St SE,MMC 392, Minneapolis, MN 55455 USA.
EM fatem002@umn.edu; folso013@umn.edu; reuti003@umn.edu;
Pandian.Twinkle@mayo.edu; brau0291@umn.edu; haugx037@umn.edu
FU Stanley Medical Research Institute [02R-232]
FX Grant Support by Stanley Medical Research Institute (02R-232) to SHF is
acknowledged. Additionally. we would like to acknowledge the kind gift
of olanzapine from Eli Lilly and Company and provided by Dr. L. Jaton.
We wish to thank L. Iversen for secretarial assistance.
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NR 40
TC 18
Z9 18
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0920-9964
J9 SCHIZOPHR RES
JI Schizophr. Res.
PD SEP
PY 2008
VL 104
IS 1-3
BP 127
EP 134
DI 10.1016/j.schres.2008.05.016
PG 8
WC Psychiatry
SC Psychiatry
GA 357XE
UT WOS:000259879700014
PM 18585900
ER
PT J
AU Esterberg, ML
Trotman, HD
Brasfield, JL
Compton, MT
Walker, EF
AF Esterberg, Michelle L.
Trotman, Hanan D.
Brasfield, Joy L.
Compton, Michael T.
Walker, Elaine F.
TI Childhood and current autistic features in adolescents with schizotypal
personality disorder
SO SCHIZOPHRENIA RESEARCH
LA English
DT Article
DE Asperger's disorder; autistic disorder; schizophrenia; schizotypal
personality disorder
ID SPECTRUM DISORDERS; DELETION SYNDROME; SCHIZOPHRENIA; CHILDREN;
PSYCHOSIS; SYMPTOMS; EXPRESSION; INTERVIEW; CAPACITY; DEFICITS
AB The diagnostic boundaries between autistic- and schizophrenia-spectrum disorders have varied over the years, and some overlap in diagnostic criteria persists. The present study examined childhood and current Signs of autistic disorder (AD) in adolescents with schizotypal personality disorder (SPD) or other personality disorders, as well as healthy controls. A structured interview was administered to rate participants' current symptoms. Participants' guardians were interviewed with the Autism Diagnostic Inventory-Revised (ADI-R). a clinical assessment of childhood and Current autistic signs. Compared to both the other personality-disordered and healthy groups, adolescents with SPD were rated as having significantly more impairment on childhood and current social functioning, and having more unusual interests and behaviors. For the entire sample. impaired childhood social functioning and unusual interests and behaviors were associated with higher negative symptom scores. Current impairments in social functioning, unusual interests and behaviors, and communication were also linked with greater negative symptoms. However, neither childhood nor current autistic features significantly predicted later conversion to an Axis I psychotic disorder over the course of three years of follow-up. The findings indicate that past and current autistic signs are more common in adolescents with SPD, but neither current nor childhood autistic features are linked with conversion to psychosis. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Esterberg, Michelle L.; Trotman, Hanan D.; Brasfield, Joy L.; Walker, Elaine F.] Emory Univ, Grad Sch Arts & Sci, Dept Psychol, Atlanta, GA 30322 USA.
[Compton, Michael T.] Emory Univ, Sch Med, Dept Psychiat & Behav Sci, Atlanta, GA 30303 USA.
RP Esterberg, ML (reprint author), Emory Univ, Grad Sch Arts & Sci, Dept Psychol, 1462 Clifton Rd,Suite 235, Atlanta, GA 30322 USA.
EM mesterb@emory.edu; htrotma@emory.edu; jbrasfi@emory.edu;
mcompto@emory.edu; psyefw@emory.edu
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NR 42
TC 27
Z9 27
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0920-9964
J9 SCHIZOPHR RES
JI Schizophr. Res.
PD SEP
PY 2008
VL 104
IS 1-3
BP 265
EP 273
DI 10.1016/j.schres.2008.04.029
PG 9
WC Psychiatry
SC Psychiatry
GA 357XE
UT WOS:000259879700029
PM 18554872
ER
PT J
AU Clarke, CE
AF Clarke, Christopher E.
TI A question of balance - The autism-vaccine controversy in the British
and American elite press
SO SCIENCE COMMUNICATION
LA English
DT Article
DE vaccination; autism; media; ethics; risk communication
ID ANTHROPOGENIC CLIMATE-CHANGE; RISK COMMUNICATION; MEDIA REPRESENTATIONS;
RUBELLA VACCINATION; NEWSPAPER COVERAGE; MMR VACCINATION; UNITED-STATES;
SCIENCE; MEASLES; MUMPS
AB Although balance is a well-known and arguably important journalistic norm, how should journalists adhere to this norm when the bulk of scientific evidence clearly favors one (presumably accurate) perspective? Should balance be defined in terms of the quantity of information or the quality of viewpoints presented? Using British and American newspaper coverage of the autism-vaccine controversy as a case study, this article explores whether balanced reporting on scientific claims produced a discourse at odds with the scientific consensus that there was no autism-vaccine link. Implications for journalism ethics and risk communication are discussed.
C1 Cornell Univ, Dept Commun, Ithaca, NY 14853 USA.
RP Clarke, CE (reprint author), Cornell Univ, Dept Commun, Ithaca, NY 14853 USA.
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NR 75
TC 23
Z9 23
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1075-5470
J9 SCI COMMUN
JI Sci. Commun.
PD SEP
PY 2008
VL 30
IS 1
BP 77
EP 107
DI 10.1177/1075547008320262
PG 31
WC Communication
SC Communication
GA 336LC
UT WOS:000258364300003
ER
PT J
AU Kayser, MA
AF Kayser, Michael A.
TI Inherited Metabolic Diseases in Neurodevelopmental and Neurobehavioral
Disorders
SO SEMINARS IN PEDIATRIC NEUROLOGY
LA English
DT Review
AB In the past few years, there has been a veritable explosion in the discovery of "new" inborn errors of metabolism. These new conditions are involved in complex pathways of intermediary metabolism affecting processes heretofore unknown. The phenotypes of these new conditions are in many ways milder than the classically described metabolic disorders. Several of these conditions present as nonsyndromic neurodevelopmental and/or neurobehavioral disorders. As such, these conditions should be considered in the differential diagnosis of conditions such as mental retardation, autism spectrum disorders, movement disorders, and cerebral palsy. This article reviews several of these recently described conditions including the clinical presentation, the biochemical profile, the diagnostic approach, and therapeutic options. Semin Pediatr Neurol 15:127-131 (C) 2008 Elsevier Inc. All rights reserved.
C1 [Kayser, Michael A.] Warren Clin, Tulsa, OK USA.
RP Kayser, MA (reprint author), Ctr Genet Testing St Francis, Warren Clin Ctr Genet, 6465 S Yale Ave,Suite 1015, Tulsa, OK 74136 USA.
EM MAKayser@saintfrancis.com
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NR 37
TC 10
Z9 10
PU W B SAUNDERS CO-ELSEVIER INC
PI PHILADELPHIA
PA 1600 JOHN F KENNEDY BOULEVARD, STE 1800, PHILADELPHIA, PA 19103-2899 USA
SN 1071-9091
J9 SEMIN PEDIATR NEUROL
JI Semin. Pediatr. Neurol.
PD SEP
PY 2008
VL 15
IS 3
BP 127
EP 131
DI 10.1016/j.spen.2008.05.006
PG 5
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA V15GE
UT WOS:000207789700005
PM 18708003
ER
PT J
AU Oberman, LM
Ramachandran, VS
AF Oberman, Lindsay M.
Ramachandran, Vilayanur S.
TI Preliminary evidence for deficits in multisensory integration in autism
spectrum disorders: The mirror neuron hypothesis
SO SOCIAL NEUROSCIENCE
LA English
DT Article
ID CORTICAL ACTIVATION; MOTOR FACILITATION; SPEECH PRODUCTION; CHILDHOOD
AUTISM; INFANTILE-AUTISM; SOCIAL COGNITION; FRONTAL-CORTEX; ACTION
WORDS; IMITATION; DYSFUNCTION
AB Autism is a complex disorder, characterized by social, cognitive, communicative, and motor symptoms. One suggestion, proposed in the current study, to explain the spectrum of symptoms is an underlying impairment in multisensory integration (MSI) systems such as a mirror neuron-like system. The mirror neuron system, thought to play a critical role in skills such as imitation, empathy, and language can be thought of as a multisensory system, converting sensory stimuli into motor representations. Consistent with this, we report preliminary evidence for deficits in a task thought to tap into MSI-"the bouba-kiki task" in children with ASD. The bouba-kiki effect is produced when subjects are asked to pair nonsense shapes with nonsense "words". We found that neurotypical children chose the nonsense "word" whose phonemic structure corresponded with the visual shape of the stimuli 88% of the time. This is presumably because of mirror neuron-like multisensory systems that integrate the visual shape with the corresponding motor gestures used to pronounce the nonsense word. Surprisingly, individuals with ASD only chose the corresponding name 56% of the time. The poor performance by the ASD group on this task suggests a deficit in MSI, perhaps related to impaired MSI brain systems. Though this is a behavioral study, it provides a testable hypothesis for the communication impairments in children with ASD that implicates a specific neural system and fits well with the current findings suggesting an impairment in the mirror systems in individuals with ASD.
C1 [Oberman, Lindsay M.; Ramachandran, Vilayanur S.] Univ Calif San Diego, La Jolla, CA 92093 USA.
RP Oberman, LM (reprint author), 9500 Gilman Dr, La Jolla, CA 92093 USA.
EM loberman@ucsd.edu
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NR 50
TC 30
Z9 30
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 1747-0919
J9 SOC NEUROSCI
JI Soc. Neurosci.
PD SEP-DEC
PY 2008
VL 3
IS 3-4
BP 348
EP 355
DI 10.1080/17470910701563681
PG 8
WC Neurosciences; Psychology
SC Neurosciences & Neurology; Psychology
GA 382FE
UT WOS:000261589800013
PM 18979385
ER
PT J
AU Jones, MC
Okere, K
AF Jones, Melissa C.
Okere, Kennedy
TI Treatment of hypersexual behavior with oral estrogen in an autistic male
SO SOUTHERN MEDICAL JOURNAL
LA English
DT Article
DE autism; hypersexual behavior; hormonal agents
AB Hypersexual behavior can be exhibited by patients with autism. Several medications have been used in hypersexual paraphiliac male and elderly patient populations, including antiandrogens, estrogen, gonadotropin-releasing hormone analogues, and selective serotonin reuptake inhibitors. Due to limited research in autistic patients exhibiting hypersexuality, physicians must base their medication selection on outcomes seen in patients with other conditions.
C1 [Jones, Melissa C.; Okere, Kennedy] Mem Family Med Ctr, Savannah, GA USA.
RP Jones, MC (reprint author), S Univ, Sch Pharm, 709 Mall Blvd, Savannah, GA 31406 USA.
EM mjones@southuniversity.edu
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NR 5
TC 3
Z9 3
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0038-4348
J9 SOUTH MED J
JI South.Med.J.
PD SEP
PY 2008
VL 101
IS 9
BP 959
EP 960
PG 2
WC Medicine, General & Internal
SC General & Internal Medicine
GA 353QL
UT WOS:000259583200024
PM 18708975
ER
PT J
AU Popovic-Deusic, S
Pejovic-Milovancevic, M
Draganic-Gajic, S
Aleksic-Hil, O
Lecic-Tosevski, D
AF Popovic-Deusic, Smiljka
Pejovic-Milovancevic, Milica
Draganic-Gajic, Saveta
Aleksic-Hil, Olivera
Lecic-Tosevski, Dusica
TI PSYCHOTIC SPECTRUM DISORDERS IN CHILDHOOD
SO SRPSKI ARHIV ZA CELOKUPNO LEKARSTVO
LA Serbian
DT Review
DE early onset psychoses; pervasive developmental disorders; psychotic
spectrum in childhood; classification systems
ID AUTISM
AB For a long time, there was a strong belief of existing continuity between childhood-onset psychoses and adult psychoses. Important moment in understanding psychotic presentations during infancy and childhood is Kanner's description of early infantile autism. Later studies of Rutter and Kolvin, as well as new classification systems, have delineated pervasive developmental disorders from all other psychotic disorders in childhood. But clinical experience is showing that in spite of existence of the group of pervasive developmental disorders with subgroups within it and necessary diagnostic criteria - there are children with pervasive symptoms, who are not fulfilling all necessary diagnostic criteria for pervasive developmental disorder. Therefore, in this paper we are discussing and pointing at psychotic spectrum presentations in children, which have not the right place in any existing classification system (ICID-10, DSM-IV).
C1 [Popovic-Deusic, Smiljka; Pejovic-Milovancevic, Milica; Draganic-Gajic, Saveta; Aleksic-Hil, Olivera; Lecic-Tosevski, Dusica] Inst Mental Hlth, Belgrade 11000, Serbia.
RP Popovic-Deusic, S (reprint author), Inst Mental Hlth, Palmoticeva 37, Belgrade 11000, Serbia.
EM s.popovicdeusic@imh.org.yu
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NR 22
TC 0
Z9 0
PU SRPSKO LEKARSKO DRUSTVO
PI BEOGRAD
PA UREDNISTVO CASOPISA SRPSKI ARHIV, UL DZORDZA VASINGTONA 19, BEOGRAD,
11000, SERBIA
SN 0370-8179
J9 SRP ARK CELOK LEK
JI Srp. Ark. Celok. Lek.
PD SEP-OCT
PY 2008
VL 136
IS 9-10
BP 555
EP 558
DI 10.2298/SARH0810555P
PG 4
WC Medicine, General & Internal
SC General & Internal Medicine
GA 376RQ
UT WOS:000261201100017
ER
PT J
AU Chevreuil, C
Reymann, JM
Fremaux, T
Polard, E
Seveno, T
Bentue-Ferrer, D
AF Chevreuil, Claire
Reymann, Jean-Michel
Fremaux, Tanya
Polard, Elisabeth
Seveno, Tanguy
Bentue-Ferrer, Daniele
CA Grp Etud Interdisciplinaire Bretag
TI Risperidone Use in Child and Adolescent Psychiatric Patients.
SO THERAPIE
LA French
DT Review
DE risperidone; child; adolescent; psychiatric somatic therapies; psychotic
disorders; conduct disorders
ID DISRUPTIVE BEHAVIOR DISORDERS; PERVASIVE DEVELOPMENTAL DISORDERS;
LONG-TERM SAFETY; PLACEBO-CONTROLLED TRIAL; INDUCED WEIGHT-GAIN;
OPEN-LABEL TRIAL; DOUBLE-BLIND; YOUNG-CHILDREN; AUTISTIC DISORDER;
BIPOLAR DISORDER
AB Risperidone Use in Child and Adolescent Psychiatric Patients. In a plural and multidisciplinary process of care, it would be fruitful to ally complementary, pharmacologic and psychodynamic approaches. We have done a review of the literature on the effectiveness and the cautions for prescription of risperidone, a second generation antipsychotic drug. Risperidone has proved helpful in treating children and adolescents with autism spectrum, conduct and bipolar disorders, Tourette's syndrome, and schizophrenia. The principal side effects are sedation, weight gain, and metabolic disturbances. Extrapyramidal symptoms, QTc prolongation, and hyperprolactemia with clinical signs are infrequent and not clinically significant. The benefit/risk is clearly in favor of the prescription when it is accompanied with the precautions and with the adequate monitoring.
C1 [Reymann, Jean-Michel; Bentue-Ferrer, Daniele] Fac Med, Pharmacol Lab, CS 34317, F-35043 Rennes, France.
[Chevreuil, Claire; Fremaux, Tanya; Seveno, Tanguy] CH Guillaume Regnier, Rennes, France.
[Polard, Elisabeth] CHU Pontchaillou, Ctr Pharmacovigilance, Rennes, France.
RP Bentue-Ferrer, D (reprint author), Fac Med, Pharmacol Lab, CS 34317, 2 Ave Prof Leon Bernard, F-35043 Rennes, France.
EM daniele.bentue-ferrer@univ-rennes1.fr
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NR 106
TC 7
Z9 7
PU EDP SCIENCES S A
PI LES ULIS CEDEX A
PA 17, AVE DU HOGGAR, PA COURTABOEUF, BP 112, F-91944 LES ULIS CEDEX A,
FRANCE
SN 0040-5957
J9 THERAPIE
JI Therapie
PD SEP-OCT
PY 2008
VL 63
IS 5
BP 359
EP 375
DI 10.2515/therapie:2008059
PG 17
WC Pharmacology & Pharmacy
SC Pharmacology & Pharmacy
GA 404LS
UT WOS:000263154400002
PM 19154706
ER
PT J
AU Baykara, B
Gencer, O
Ilkin, Z
Miral, S
AF Baykara, Burak
Gencer, Oezlem
Ilkin, Zeynep
Miral, Sueha
TI Neurocognitive Features of the Frontal Lobe in Parents of Autistic
Children
SO TURK PSIKIYATRI DERGISI
LA Turkish
DT Article
DE Autism; parent; neurocognitive test; executive function; intelligence
ID EXECUTIVE FUNCTION; FAMILY HISTORY; PHENOTYPE; DEFICITS; CLASSIFICATION;
DYSFUNCTION; PROFILES; SIBLINGS; GENETICS; STANDARD
AB Objective: The aim of this study was to examine the neurocognitive functions of the frontal lobe in parents of autistic children.
Method: The study group included 64 parents of children (aged 4-18 years) diagnosed with autism, according to DSM-IV criteria, that were followed-up at the child and adolescent psychiatry outpatient clinic. Parents of children with Down syndrome (n = 60) were selected as the control group. We administered the Wisconsin Card Sorting Test (WCST), Stroop Test, and Wechsler Adult Intelligence Test (WAIS) to both groups in order to evaluate executive functions, attention, inhibition, and intelligence.
Results: Mothers of children with autism performed better than the control group mothers on the executive function measures of WCST There were no group differences in Stroop Test measures of attention and inhibition, or in the verbal and performance intelligence subtests of WAIS. Fathers of children with severe autistic symptoms performed better on some WAIS subtests compared to other, however there were no significant differences in IQ between the parents in both groups.
Conclusion: The results suggest that parents of autistic children could display different cognitive styles, but we did not observe any distinctive cognitive profile pertaining to frontal lobe functions. The cognitive ability of parents of autistic children and its neurobiological basis should be further investigated.
C1 [Baykara, Burak; Gencer, Oezlem; Ilkin, Zeynep; Miral, Sueha] Dokuz Eylul Univ, Tip Fak, Cocuk Psikiyatrisi AD, Izmir, Turkey.
RP Baykara, B (reprint author), Dokuz Eylul Univ, Tip Fak, Cocuk Psikiyatrisi AD, Izmir, Turkey.
EM burak.baykara@deu.edu.tr
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NR 45
TC 1
Z9 1
PU TURKIYE SINIR VE RUH SAGLIGI DERNEGI
PI YENISEHIR
PA PK 401, YENISEHIR, 06442, TURKEY
SN 1300-2163
J9 TURK PSIKIYATR DERG
JI Turk. Psikiyatr. Derg.
PD FAL
PY 2008
VL 19
IS 3
BP 225
EP 234
PG 10
WC Psychiatry
SC Psychiatry
GA 359NF
UT WOS:000259993100002
PM 18791875
ER
PT J
AU de Smith, AJ
Walters, RG
Coin, LJM
Steinfeld, I
Yakhini, Z
Sladek, R
Froguel, P
Blakemore, AIF
AF de Smith, Adam J.
Walters, Robin G.
Coin, Lachlan J. M.
Steinfeld, Israel
Yakhini, Zohar
Sladek, Rob
Froguel, Philippe
Blakemore, Alexandra I. F.
TI Small Deletion Variants Have Stable Breakpoints Commonly Associated with
Alu Elements
SO PLOS ONE
LA English
DT Article
AB Copy number variants (CNVs) contribute significantly to human genomic variation, with over 5000 loci reported, covering more than 18% of the euchromatic human genome. Little is known, however, about the origin and stability of variants of different size and complexity. We investigated the breakpoints of 20 small, common deletions, representing a subset of those originally identified by array CGH, using Agilent microarrays, in 50 healthy French Caucasian subjects. By sequencing PCR products amplified using primers designed to span the deleted regions, we determined the exact size and genomic position of the deletions in all affected samples. For each deletion studied, all individuals carrying the deletion share identical upstream and downstream breakpoints at the sequence level, suggesting that the deletion event occurred just once and later became common in the population. This is supported by linkage disequilibrium (LD) analysis, which has revealed that most of the deletions studied are in moderate to strong LD with surrounding SNPs, and have conserved long-range haplotypes. Analysis of the sequences flanking the deletion breakpoints revealed an enrichment of microhomology at the breakpoint junctions. More significantly, we found an enrichment of Alu repeat elements, the overwhelming majority of which intersected deletion breakpoints at their poly-A tails. We found no enrichment of LINE elements or segmental duplications, in contrast to other reports. Sequence analysis revealed enrichment of a conserved motif in the sequences surrounding the deletion breakpoints, although whether this motif has any mechanistic role in the formation of some deletions has yet to be determined. Considered together with existing information on more complex inherited variant regions, and reports of de novo variants associated with autism, these data support the presence of different subgroups of CNV in the genome which may have originated through different mechanisms.
C1 [de Smith, Adam J.; Walters, Robin G.; Froguel, Philippe; Blakemore, Alexandra I. F.] Univ London Imperial Coll Sci Technol & Med, Hammersmith Hosp, Sect Genom Med, London, England.
[Coin, Lachlan J. M.] Imperial Coll London, Dept Epidemiol & Publ Hlth, London, England.
[Steinfeld, Israel; Yakhini, Zohar] Agilent Lab, Petah Tiqwa, Israel.
[Sladek, Rob] McGill Univ, Genome Quebec Innovat Ctr, Dept Human Genet, Montreal, PQ H3A 2T5, Canada.
[Sladek, Rob] McGill Univ, Genome Quebec Innovat Ctr, Dept Med, Montreal, PQ H3A 2T5, Canada.
[Froguel, Philippe] Inst Pasteur, Inst Biol, CNRS 8090, Lille, France.
RP de Smith, AJ (reprint author), Univ London Imperial Coll Sci Technol & Med, Hammersmith Hosp, Sect Genom Med, London, England.
EM a.blakemore@imperial.ac.uk
RI Walters, Robin/E-6069-2010; Coin, Lachlan/A-9001-2014
OI Walters, Robin/0000-0002-9179-0321; Coin, Lachlan/0000-0002-4300-455X
FU Hammersmith Hospital Trust; Genome Quebec
FX This work was supported by the Hammersmith Hospital Trust, and SNP
genotyping was funded by Genome Quebec.
CR [Anonymous], DATABASE GENOMIC VAR
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NR 42
TC 27
Z9 28
PU PUBLIC LIBRARY SCIENCE
PI SAN FRANCISCO
PA 185 BERRY ST, STE 1300, SAN FRANCISCO, CA 94107 USA
SN 1932-6203
J9 PLOS ONE
JI PLoS One
PD AUG 29
PY 2008
VL 3
IS 8
AR e3104
DI 10.1371/journal.pone.0003104
PG 11
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 427RQ
UT WOS:000264796800009
PM 18769679
ER
PT J
AU Kuehn, BM
AF Kuehn, Bridget M.
TI Autism genes
SO JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION
LA English
DT News Item
CR Morrow EM, 2008, SCIENCE, V321, P218, DOI 10.1126/science.1157657
NR 1
TC 0
Z9 0
PU AMER MEDICAL ASSOC
PI CHICAGO
PA 515 N STATE ST, CHICAGO, IL 60654-0946 USA
SN 0098-7484
J9 JAMA-J AM MED ASSOC
JI JAMA-J. Am. Med. Assoc.
PD AUG 27
PY 2008
VL 300
IS 8
BP 891
EP 891
PG 1
WC Medicine, General & Internal
SC General & Internal Medicine
GA 341AR
UT WOS:000258687100008
ER
PT J
AU Scattoni, ML
Gandhy, SU
Ricceri, L
Crawley, JN
AF Scattoni, Maria Luisa
Gandhy, Shruti U.
Ricceri, Laura
Crawley, Jacqueline N.
TI Unusual Repertoire of Vocalizations in the BTBR T plus tf/J Mouse Model
of Autism
SO PLOS ONE
LA English
DT Article
ID BEHAVIORAL TASKS RELEVANT; ULTRASONIC VOCALIZATIONS; MATERNAL-BEHAVIOR;
INBRED STRAINS; LABORATORY RATS; DEVELOPMENTAL INFLUENCES; COMMUNICATION
CALLS; MENTAL-RETARDATION; SOCIAL-INTERACTION; SPECTRUM DISORDER
AB BTBR T+ tf/J (BTBR) is an inbred mouse strain that displays social abnormalities and repetitive behaviors analogous to the first and third diagnostic symptoms of autism. Here we investigate ultrasonic vocalizations in BTBR, to address the second diagnostic symptom of autism, communication deficits. As compared to the commonly used C57BL/6J (B6) strain, BTBR pups called more loudly and more frequently when separated from their mothers and siblings. Detailed analysis of ten categories of calls revealed an unusual pattern in BTBR as compared to B6. BTBR emitted high levels of harmonics, two-syllable, and composite calls, but minimal numbers of chevron-shaped syllables, upward, downward, and short calls. Because body weights were higher in BTBR than B6 pups, one possible explanation was that larger thoracic size was responsible for the louder calls and different distribution of syllable categories. To test this possibility, we recorded separation calls from FVB/NJ, a strain with body weights similar to BTBR, and 129X1/SvJ, a strain with body weights similar to B6. BTBR remained the outlier on number of calls, displaying low numbers of complex, upward, chevron, short, and frequency steps calls, along with high harmonics and composites. Further, developmental milestones and growth rates were accelerated in BTBR, indicating an unusual neurodevelopmental trajectory. Overall, our findings demonstrate strain-specific patterns of ultrasonic calls that may represent different lexicons, or innate variations in complex vocal repertoires, in genetically distinct strains of mice. Particularly intriguing is the unusual pattern of vocalizations and the more frequent, loud harmonics evident in the BTBR mouse model of autism that may resemble the atypical vocalizations seen in some autistic infants.
C1 [Scattoni, Maria Luisa; Gandhy, Shruti U.; Crawley, Jacqueline N.] NIMH, Lab Behav Neurosci, Bethesda, MD 20892 USA.
[Scattoni, Maria Luisa; Ricceri, Laura] Ist Superiore Sanita, Dept Cell Biol & Neurosci, Behav Neurosci Sect, Rome, Italy.
RP Scattoni, ML (reprint author), NIMH, Lab Behav Neurosci, Bethesda, MD 20892 USA.
EM marialuisa.scattoni@iss.it
FU National Institute of Mental Health Intramural Research Program
[Z01-MH-002498-17, ISS-NIH 0F14]
FX Supported by the National Institute of Mental Health Intramural Research
Program (Z01-MH-002498-17, JNC), and ISS-NIH 0F14 Neurobehavioral
phenotyping of genetically modified mouse models of mental
retardation(LR). The funders had no role in study design, data
collection and analysis, decision to publish, or preparation of the
manuscript. The views expressed in this article do not necessarily
represent the views of the NIMH, NIH, or the United States Government.
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NR 78
TC 126
Z9 128
PU PUBLIC LIBRARY SCIENCE
PI SAN FRANCISCO
PA 185 BERRY ST, STE 1300, SAN FRANCISCO, CA 94107 USA
SN 1932-6203
J9 PLOS ONE
JI PLoS One
PD AUG 27
PY 2008
VL 3
IS 8
AR e3067
DI 10.1371/journal.pone.0003067
PG 15
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 427RM
UT WOS:000264796400007
PM 18728777
ER
PT J
AU Bailey, DB
Raspa, M
Olmsted, M
Holiday, DB
AF Bailey, Donald B., Jr.
Raspa, Melissa
Olmsted, Murrey
Holiday, David B.
TI Co-occurring conditions associated with FMR1 gene variations: Findings
from a national parent survey
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Article
DE fragile X syndrome; co-occuring conditions
ID FRAGILE-X-SYNDROME; TREMOR/ATAXIA SYNDROME FXTAS; SECONDARY CONDITIONS;
FULL MUTATION; CGG REPEATS; PREMUTATION; MALES; PHENOTYPE; CHILDREN;
EPIDEMIOLOGY
AB Parents enrolling in a national survey of families of children with fragile X (FX) reported whether each of their children had been diagnosed or treated for developmental delay or eight conditions frequently associated with FX: attention problems, hyperactivity, aggressiveness, self-injury, autism seizures, anxiety, or depression. This article reports results for 976 full mutation males, 259 full mutation females. 57 premutation males, and 199 premutation females. Co-occuring conditions were frequently reported for all FMR1 gene variations. The number of co-occurring conditions experienced was strongly associated with parent reports of their child's ability to learn, adaptability, and quality of life. Most individuals with the full mutation experienced multiple co-occuring conditions, with a modal number of 4 for males and 2 for females. Most (>80%) full mutation males and females had been diagnosed or treated for attention problems. Premutation males, when compared with a matched group of non-FX males, were more likely to have been diagnosed or treated for developmental delay, attention problems, aggression. seizures, autism, and anxiety. Premutation females were more likely to have been diagnosed or treated for attention problems, anxiety, depression, and developmental delay. Clusters of conditions were identified, seeming to occur in an additive fashion. Self-injury autism, and seizures rarely occured in isolation, but were more likely in individuals who also had problems with attention, anxiety, and hyperactivity. The findings provide a reference point for future studies on the prevalence and nature of co-occurring conditions in FX; suggest the possibility that certain conditions cluster together; provide evidence that male and female carriers experience elevated rates of co-occuring conditions compared with matched groups of non-carrier children: and emphasize the importance of including an assessment of co-occuring conditions in any clinical evaluation of individuals with abnormal variation in the FMR1 gene. (C) 2008 Wiley-Liss, Inc.
C1 [Bailey, Donald B., Jr.; Raspa, Melissa; Olmsted, Murrey; Holiday, David B.] RTI Int, Res Triangle Pk, NC 27709 USA.
RP Bailey, DB (reprint author), RTI Int, 3040 Cornwallis Rd, Res Triangle Pk, NC 27709 USA.
EM dbailey@rti.org
FU Centers for Disease Control and Prevention (CDC); Association for
Prevention Teaching and Research (APTR) [U50/CCU300860, TS-1380]
FX Preparation of this article was supported in part by the Centers for
Disease Control and Prevention (CDC) and the Association for Prevention
Teaching and Research (APTR) Cooperative Agreement No. U50/CCU300860,
Project TS-1380. The authors express their appreciation to the research
collaborators and organizations who supported the recruitment of study
participants, the families who completed the survey, and the RTI staff
who assisted in survey programming (Venkat Yetuktiri), data management
(Anne Kenyon and Kirstin Miller), and call center services (Ellen Fay).
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NR 43
TC 119
Z9 120
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD AUG 15
PY 2008
VL 146A
IS 16
BP 2060
EP 2069
DI 10.1002/ajmg.a.32439
PG 10
WC Genetics & Heredity
SC Genetics & Heredity
GA 338AL
UT WOS:000258477100003
PM 18570292
ER
PT J
AU Molina, J
Carmona-Mora, P
Chrast, J
Krall, PM
Canales, CP
Lupski, JR
Reymond, A
Walz, K
AF Molina, Jessica
Carmona-Mora, Paulina
Chrast, Jacqueline
Krall, Paola M.
Canales, Cesar P.
Lupski, James R.
Reymond, Alexandre
Walz, Katherina
TI Abnormal social behaviors and altered gene expression rates in a mouse
model for Potocki-Lupski syndrome
SO HUMAN MOLECULAR GENETICS
LA English
DT Article
ID SMITH-MAGENIS-SYNDROME; RETT-SYNDROME; CHROMOSOMAL REARRANGEMENTS;
AUTISM; MICE; RAI1; DUP(17)(P11.2P11.2); 17P11.2; DUPLICATION; MUTATIONS
AB The Potocki-Lupski syndrome (PTLS) is associated with a microduplication of 17p11.2. Clinical features include multiple congenital and neurobehavioral abnormalities and autistic features. We have generated a PTLS mouse model, Dp(11)17/+, that recapitulates some of the physical and neurobehavioral phenotypes present in patients. Here, we investigated the social behavior and gene expression pattern of this mouse model in a pure C57BL/6-Tyr(c-Brd) genetic background. Dp(11)17/+ male mice displayed normal home-cage behavior but increased anxiety and increased dominant behavior in specific tests. A subtle impairment in the preference for a social target versus an inanimate target and abnormal preference for social novelty (the preference to explore an unfamiliar mouse versus a familiar one) was also observed. Our results indicate that these animals could provide a valuable model to identify the specific gene(s) that confer abnormal social behaviors and that map within this delimited genomic deletion interval. In a first attempt to identify candidate genes and for elucidating the mechanisms of regulation of these important phenotypes, we directly assessed the relative transcription of genes within and around this genomic interval. In this mouse model, we found that candidates genes include not only most of the duplicated genes, but also normal-copy genes that flank the engineered interval; both categories of genes showed altered expression levels in the hippocampus of Dp(11)17/+ mice.
C1 [Molina, Jessica; Carmona-Mora, Paulina; Krall, Paola M.; Canales, Cesar P.; Walz, Katherina] Ctr Estudios Cient, Ctr Estudios Cientificos, Valdivia, Chile.
[Molina, Jessica; Carmona-Mora, Paulina; Krall, Paola M.; Canales, Cesar P.] Univ Austral Chile, Valdivia, Chile.
[Chrast, Jacqueline; Reymond, Alexandre] Univ Lausanne, Ctr Integrat Gen, Lausanne, Switzerland.
[Lupski, James R.] Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
[Lupski, James R.] Baylor Coll Med, Dept Pediat, Houston, TX 77030 USA.
[Lupski, James R.] Texas Childrens Hosp, Houston, TX 77030 USA.
RP Walz, K (reprint author), Avda Prat 514, Valdivia 5110246, Chile.
EM kwalz@cecs.cl
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NR 38
TC 41
Z9 41
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0964-6906
J9 HUM MOL GENET
JI Hum. Mol. Genet.
PD AUG 15
PY 2008
VL 17
IS 16
BP 2486
EP 2495
DI 10.1093/hmg/ddn148
PG 10
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 330SL
UT WOS:000257963000008
PM 18469339
ER
PT J
AU Maussion, G
Carayol, J
Lepagnol-Bestel, AM
Tores, F
Loe-Mie, Y
Milbreta, U
Rousseau, F
Fontaine, K
Renaud, J
Moalic, JM
Philippi, A
Chedotal, A
Gorwood, P
Ramoz, N
Hager, J
Simonneau, M
AF Maussion, Gilles
Carayol, Jerome
Lepagnol-Bestel, Aude-Marie
Tores, Frederic
Loe-Mie, Yann
Milbreta, Ulla
Rousseau, Francis
Fontaine, Karine
Renaud, Julie
Moalic, Jean-Marie
Philippi, Anne
Chedotal, Alain
Gorwood, Philip
Ramoz, Nicolas
Hager, Joerg
Simonneau, Michel
TI Convergent evidence identifying MAP/microtubule affinity-regulating
kinase 1 (MARK1) as a susceptibility gene for autism
SO HUMAN MOLECULAR GENETICS
LA English
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDERS; NEURONAL POLARITY; UNIFIED APPROACH;
PROTEIN; ASSOCIATION; COMPLEX; EVOLUTION; LINKAGE; FAMILY; BRAIN
AB Autism spectrum disorders (ASDs) are common, heritable, but genetically heterogeneous neurodevelopmental conditions. We recently defined a susceptibility locus for ASDs on chromosome 1q41-q42. High-resolution single-nucleotide polymorphisms (126 SNPs) genotyping across the chromosome 1q41-q42 region, followed by a MARK1 (microtubule affinity-regulating kinase 1)-tagged-SNP association study in 276 families with autism from the Autism Genetic Research Exchange, showed that several SNPs within the MARK1 gene were significantly associated with ASDs by transmission disequilibrium tests. Haplotype rs12740310*C-rs3737296*G-rs12410279*A was overtransmitted (Pcorrected= 0.0016), with a relative risk for autism of 1.8 in homozygous carriers. Furthermore, ASD-associated SNP rs12410279 modulates the level of transcription of MARK1. We found that MARK1 was overexpressed in the prefrontal cortex (BA46) but not in cerebellar granule cells, on postmortem brain tissues from patients. MARK1 displayed an accelerated evolution along the lineage leading to humans, suggesting possible involvement of this gene in cognition. MARK1 encodes a kinase-regulating microtubule-dependent transport in axons and dendrites. Both overexpression and silencing of MARK1 resulted in significantly shorter dendrite length in mouse neocortical neurons and modified dendritic transport speed. As expected for a gene encoding a key polarity determinant Par-1 protein kinase, MARK1 is involved in axon-dendrite specification. Thus, MARK1 overexpression in humans may be responsible for subtle changes in dendritic functioning.
C1 [Maussion, Gilles; Lepagnol-Bestel, Aude-Marie; Loe-Mie, Yann; Moalic, Jean-Marie; Gorwood, Philip; Ramoz, Nicolas; Simonneau, Michel] Univ Paris 07, INSERM, U675, IFR2,Fac Med Xavier Bichat, F-75018 Paris, France.
[Carayol, Jerome; Tores, Frederic; Rousseau, Francis; Fontaine, Karine; Philippi, Anne; Hager, Joerg] IntegraGen SA Genopole, Evry, France.
[Renaud, Julie; Chedotal, Alain] Univ Paris 06, UMRS 592, F-75005 Paris, France.
[Renaud, Julie; Chedotal, Alain] Inst Vis, UMRS 592, INSERM, F-75012 Paris, France.
RP Simonneau, M (reprint author), Univ Paris 07, INSERM, U675, IFR2,Fac Med Xavier Bichat, 16 Rue Henri Huchard, F-75018 Paris, France.
EM michel.simonneau@inserm.fr
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NR 55
TC 21
Z9 23
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0964-6906
J9 HUM MOL GENET
JI Hum. Mol. Genet.
PD AUG 15
PY 2008
VL 17
IS 16
BP 2541
EP 2551
DI 10.1093/hmg/ddn154
PG 11
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 330SL
UT WOS:000257963000013
PM 18492799
ER
PT J
AU Young, HA
Geier, DA
Geier, MR
AF Young, Heather A.
Geier, David A.
Geier, Mark R.
TI Thimerosal exposure in infants and neurodevelopmental disorders: An
assessment of computerized medical records in the Vaccine Safety
Datalink
SO JOURNAL OF THE NEUROLOGICAL SCIENCES
LA English
DT Article
DE ADD; ADHD autistic disorder; ASD; ethylmercury; merthiolate; thiomersal
ID DEVELOPMENTAL DISORDERS; AUTISTIC DISORDERS; SPECTRUM DISORDERS; CAUSAL
ASSOCIATION; MERCURY EXPOSURE; UNITED-KINGDOM; BLOOD MERCURY; CHILDREN;
COHORT; METHYLMERCURY
AB The study evaluated possible associations between neurodevelopmental disorders (NDs) and exposure to mercury (Hg) from Thimerosal-containing vaccines (TCVs) by examining the automated Vaccine Safety Datalink (VSD). A total of 278,624 subjects were identified in birth cohorts from 1990-1996 that had received their first oral polio vaccination by 3 months of age in the VSD. The birth cohort prevalence rate of medically diagnosed International Classification of Disease, 9th revision (ICD-9) specific NDs and control outcomes were calculated. Exposures to Hg from TCVs were calculated by birth cohort for specific exposure windows from birth-7 months and birth-13 months of age. Poisson regression analysis was used to model the association between the prevalence of outcomes and Hg doses from TCVs. Consistent significantly increased rate ratios were observed for autism, autism spectrum disorders, tics, attention deficit disorder, and emotional disturbances with Hg exposure from TCVs. By contrast, none of the control outcomes had significantly increased rate ratios with Hg exposure from TCVs. Routine childhood vaccination should be continued to help reduce the morbidity and mortality associated with infectious diseases, but efforts should be undertaken to remove Hg from vaccines. Additional studies should be conducted to further evaluate the relationship between Hg exposure and NDs. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Young, Heather A.] George Washington Univ, Sch Publ Hlth, Washington, DC 20052 USA.
[Young, Heather A.] George Washington Univ, Hlth Serv, Dept Epidemiol & Biostat, Washington, DC 20052 USA.
[Geier, David A.] Inst Chron Illnesses Inc, Silver Spring, MD 20905 USA.
RP Geier, MR (reprint author), 14 Redgate Ct, Silver Spring, MD 20905 USA.
EM mgeier@comcast.net
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NR 53
TC 62
Z9 63
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0022-510X
J9 J NEUROL SCI
JI J. Neurol. Sci.
PD AUG 15
PY 2008
VL 271
IS 1-2
BP 110
EP 118
DI 10.1016/j.jns.2008.04.002
PG 9
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 330RA
UT WOS:000257959300017
PM 18482737
ER
PT J
AU Dutta, S
Sinha, S
Ghosh, S
Chatterjee, A
Ahmed, S
Usha, R
AF Dutta, Shruti
Sinha, Swagata
Ghosh, Saurabh
Chatterjee, Anindita
Ahmed, Shabina
Usha, Rajamma
TI Genetic analysis of reelin gene (RELN) SNPs: No association with autism
spectrum disorder in the Indian population
SO NEUROSCIENCE LETTERS
LA English
DT Article
DE autism; PDD-NOS; case-control analysis; family-based analyses; linkage
disequilibrium
ID VULNERABILITY FACTOR; POLYMORPHISMS; ALLELES; RISK; SUSCEPTIBILITY;
SCHIZOPHRENIA; HAPLOTYPES; PROTEINS; REPEAT; TESTS
AB Involvement of reelin with Autism spectrum disorder (ASD) has been implicated through several biochemical as well as genetic studies. Reelin is an extracellular signaling protein, which plays a significant role in cytoarchitectonic pattern formation of different brain areas during development. Reelin gene (RELN) is located on chromosome 7q22; an important autism critical region identified through several genome-wide scans. A number of genetic studies have been carried out to investigate the association of reelin with autism. Recently we reported possible paternal effect in the transmission of CGG repeat alleles of RELN in the susceptibility towards autism. Further analysis on other polymorphisms is warranted to validate the status of RELN as a candidate for autism. Therefore in the present study, we have investigated six more SNPs (rs727531, rs2072403, rs2072402, rs362691, rs362719, rs736707) in 102 patients, 182 parents and 101 healthy controls. We have followed DSM-IV criteria and the screening for autism was carried out using CARS. Genomic DNA isolated from blood was used for PCR and subsequent RFLP analysis. Finally, case-control and family-based association studies were carried out to examine the genetic association of these SNP markers with ASD in the Indian population. But, we failed to detect either preferential parental transmission of any alleles of the markers to affected offspring or any biased allelic or genotypic distribution between the cases and controls. Thus the present study suggests that these SNPs of RELN are unlikely to be associated with ASO in the Indian population. (C) 2008 Elsevier Ireland Ltd. All rights reserved.
C1 [Dutta, Shruti; Sinha, Swagata; Chatterjee, Anindita; Usha, Rajamma] Manovikas Kendra Rehabil & Res Inst Handicapped, Manovikas Biomed Res & Diagnost Ctr, Out Patients Dept, Kolkata 700107, India.
[Ghosh, Saurabh] Indian Stat Inst, Human Genet Unit, Kolkata 700108, India.
[Ahmed, Shabina] Assam Autism Fdn, Christian Basti 781005, Guwahati, India.
RP Usha, R (reprint author), Manovikas Kendra Rehabil & Res Inst Handicapped, Manovikas Biomed Res & Diagnost Ctr, Out Patients Dept, 482 Madudah,Plot 1-24,Sector J,EM Bypass, Kolkata 700107, India.
EM ushamvk@yahoo.co.in
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NR 33
TC 17
Z9 18
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0304-3940
EI 1872-7972
J9 NEUROSCI LETT
JI Neurosci. Lett.
PD AUG 15
PY 2008
VL 441
IS 1
BP 56
EP 60
DI 10.1016/j.neulet.2008.06.022
PG 5
WC Neurosciences
SC Neurosciences & Neurology
GA 333TD
UT WOS:000258174400012
PM 18597938
ER
PT J
AU Fecteau, S
Pascual-Leone, A
Theoret, H
AF Fecteau, Shirley
Pascual-Leone, Alvaro
Theoret, Hugo
TI Psychopathy and the mirror neuron system: Preliminary findings from a
non-psychiatric sample
SO PSYCHIATRY RESEARCH
LA English
DT Article
DE psychopathy; empathy; mirror neuron system; pain; motor cortex
ID TRANSCRANIAL MAGNETIC STIMULATION; PERSONALITY-INVENTORY; MOTOR CORTEX;
EMPATHY; VALIDITY; AUTISM; EXCITABILITY; POPULATIONS; DISORDERS;
IMITATION
AB Recent advances in social neuroscience suggest a link between empathy and the mirror neuron system (MNS). Impaired empathy is one of the core diagnostic features of psychopathic personality disorder. In the present study, we investigated whether psychopathic personality traits in a non-psychiatric sample were related to MNS function. Healthy participants viewed short videos known to activate the sensorimotor MNS for pain (a needle penetrating a human hand) while transcranial magnetic stimulation (TMS)-induced motor evoked potentials (MEP) were recorded as a measure of motor cortex excitability. Individual psychopathic personality traits were assessed using the Psychopathic Personality Inventory (PPI) and correlated with the MEP findings. Consistent with previous data, observation of the painful stimulus was associated with a significant reduction in the amplitude of the TMS-induced MEP. Interestingly, the level of corticospinal excitability modulation was positively correlated with individual scores on the coldheartedness subscale of the PPI, such that individuals with the greatest MEP reduction were the ones scoring highest on the coldheartedness measure. These data suggest the existence of a functional link between 'motor empathy' and psychopathy. (C) 2007 Elsevier Ireland Ltd. All rights reserved.
C1 [Theoret, Hugo] Univ Montreal, Dept Psychol, Montreal, PQ H3C 3J7, Canada.
[Theoret, Hugo] Univ Montreal, Hop St Justine, Montreal, PQ H3C 3J7, Canada.
[Fecteau, Shirley; Pascual-Leone, Alvaro] Beth Israel Deaconess Med Ctr, Ctr Noninvas Brain Stimulat, Boston, MA 02215 USA.
[Fecteau, Shirley; Pascual-Leone, Alvaro] Harvard Univ, Sch Med, Boston, MA 02215 USA.
RP Theoret, H (reprint author), Univ Montreal, Dept Psychol, CP 6128,Succ Ctr Ville, Montreal, PQ H3C 3J7, Canada.
EM hugo.theoret@umontreal.ca
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NR 30
TC 46
Z9 46
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0165-1781
J9 PSYCHIAT RES
JI Psychiatry Res.
PD AUG 15
PY 2008
VL 160
IS 2
BP 137
EP 144
DI 10.1016/j.psychres.2007.08.022
PG 8
WC Psychiatry
SC Psychiatry
GA 336LN
UT WOS:000258365400002
PM 18599127
ER
PT J
AU Geschwind, DH
AF Geschwind, Daniel H.
TI Autism - Family connections
SO NATURE
LA English
DT Editorial Material
ID SPECTRUM DISORDERS; GENE-EXPRESSION
C1 [Geschwind, Daniel H.] Univ Calif Los Angeles, Program Neurogenet, Dept Neurol, Los Angeles, CA 90095 USA.
[Geschwind, Daniel H.] Univ Calif Los Angeles, David Geffen Sch Med, Semel Inst, Los Angeles, CA 90095 USA.
RP Geschwind, DH (reprint author), Univ Calif Los Angeles, Program Neurogenet, Dept Neurol, 710 Westwood Plaza, Los Angeles, CA 90095 USA.
EM dhg@mednet.ucla.edu
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Strauss KA, 2006, NEW ENGL J MED, V354, P1370, DOI 10.1056/NEJMoa052773
NR 12
TC 6
Z9 6
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 0028-0836
J9 NATURE
JI Nature
PD AUG 14
PY 2008
VL 454
IS 7206
BP 838
EP 839
DI 10.1038/454838a
PG 2
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 336XH
UT WOS:000258398600024
PM 18704077
ER
PT J
AU Baron-Cohen, S
AF Baron-Cohen, Simon
TI Representing autism: Culture, narrative, fascination
SO NATURE
LA English
DT Book Review
C1 [Baron-Cohen, Simon] Univ Cambridge, Autism Res Ctr, Cambridge CB2 8AH, England.
RP Baron-Cohen, S (reprint author), Univ Cambridge, Autism Res Ctr, Cambridge CB2 8AH, England.
EM sb205@hermes.cam.ac.uk
CR Murray Stuart, 2008, REPRESENTING AUTISM
NR 1
TC 0
Z9 0
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 0028-0836
J9 NATURE
JI Nature
PD AUG 7
PY 2008
VL 454
IS 7205
BP 695
EP 696
DI 10.1038/454695a
PG 2
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 334NH
UT WOS:000258228000021
ER
PT J
AU Poling, JS
AF Poling, Jon S.
TI Vaccines and autism revisited
SO NEW ENGLAND JOURNAL OF MEDICINE
LA English
DT Letter
C1 Athens Neurol Assoc, Athens, GA 30606 USA.
RP Poling, JS (reprint author), Athens Neurol Assoc, Athens, GA 30606 USA.
EM jpoling@athensneuro.com
CR Brady MT, 2006, PEDIATRICS, V118, P810, DOI 10.1542/peds.2006-0846
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Offit PA, 2008, NEW ENGL J MED, V358, P2089, DOI 10.1056/NEJMp0802904
Oliveira G, 2007, DEV MED CHILD NEUROL, V49, P726
NR 4
TC 3
Z9 3
PU MASSACHUSETTS MEDICAL SOC
PI WALTHAM
PA WALTHAM WOODS CENTER, 860 WINTER ST,, WALTHAM, MA 02451-1413 USA
SN 0028-4793
J9 NEW ENGL J MED
JI N. Engl. J. Med.
PD AUG 7
PY 2008
VL 359
IS 6
BP 655
EP 655
PG 1
WC Medicine, General & Internal
SC General & Internal Medicine
GA 334IY
UT WOS:000258216700026
PM 18687652
ER
PT J
AU Offit, PA
AF Offit, Paul A.
TI Vaccines and autism revisited - Reply
SO NEW ENGLAND JOURNAL OF MEDICINE
LA English
DT Letter
ID UNITED-STATES; MEASLES
C1 [Offit, Paul A.] Childrens Hosp Philadelphia, Philadelphia, PA 19104 USA.
RP Offit, PA (reprint author), Childrens Hosp Philadelphia, Philadelphia, PA 19104 USA.
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NR 4
TC 1
Z9 1
PU MASSACHUSETTS MEDICAL SOC
PI WALTHAM
PA WALTHAM WOODS CENTER, 860 WINTER ST,, WALTHAM, MA 02451-1413 USA
SN 0028-4793
J9 NEW ENGL J MED
JI N. Engl. J. Med.
PD AUG 7
PY 2008
VL 359
IS 6
BP 656
EP 656
PG 1
WC Medicine, General & Internal
SC General & Internal Medicine
GA 334IY
UT WOS:000258216700027
ER
PT J
AU Montgomery, KS
Mackey, J
Thuett, K
Ginestra, S
Bizon, JL
Abbott, LC
AF Montgomery, Karienn S.
Mackey, Jessica
Thuett, Kerry
Ginestra, Stephanie
Bizon, Jennifer L.
Abbott, Louise C.
TI Chronic, low-dose prenatal exposure to methylmercury impairs motor and
mnemonic function in adult C57/B6 mice
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Article
DE locomotor activity; Morris water maze; open field activity; rotarod;
spatial learning; cue learning
ID DEVELOPMENTAL EXPOSURE; LACTATIONAL EXPOSURE; FISH CONSUMPTION; WATER
MAZE; MERCURY; CHILDREN; BENEFITS; LESIONS; RISKS; BRAIN
AB Methylmercury (MeHg) has cytotoxic effects on animals and humans, and a major target organ for MeHg is the central nervous system (CNS). It is well known that the developing CNS is extremely vulnerable to MeHg-induced changes in comparison to the mature brain. Most studies have concentrated on the direct effects of high levels of prenatal MeHg exposure. Surprisingly, behavioral outcomes found in adult offspring exposed developmentally to the neurotoxic effects of chronic, low-dose mercury more akin to ingestion in humans are not well characterized. The objective of this study was to determine whether such exposure produces deleterious effects on behavior in adult mice, including motor/coordination abilities, overall activity and mnemonic function. Developing mouse fetuses were exposed in utero during gestational days 8-18 by giving pregnant C57B1/6j female mice food containing MeHg at a daily dose of 0.01 mg/kg body weight. Adult mice prenatally exposed to MeHg exhibited significant deficits in motor abilities, coordination, and overall activity, as measured by rotarod, footprint analysis and open field. In addition, MeHg-exposed mice were impaired with respect to reference memory but not in a visible, cued version of the Morris water maze task. These results indicate that prenatal exposure to the lowest dose of MeHg examined to date can have long-lasting motor and cognitive consequences on adult offspring. These findings have far reaching implications related to putative safe levels of MeHg ingestion, particularly during pregnancy, and increasing rates of cognitive and psychological disorders (e.g. attention hyperactivity deficit disorder, autism) in our society. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Thuett, Kerry; Ginestra, Stephanie; Abbott, Louise C.] Texas A&M Univ, Coll Vet Med & Biomed Sci, Dept Vet Integrat Biosci, College Stn, TX 77843 USA.
[Bizon, Jennifer L.; Abbott, Louise C.] Texas A&M Univ, Fac Neurosci, College Stn, TX 77843 USA.
[Montgomery, Karienn S.; Bizon, Jennifer L.] Texas A&M Univ, Dept Psychol, College Stn, TX 77843 USA.
[Mackey, Jessica] Louisiana State Univ, Hlth Sci Ctr, Sch Med, Shreveport, LA 71101 USA.
RP Abbott, LC (reprint author), Texas A&M Univ, Coll Vet Med & Biomed Sci, Dept Vet Integrat Biosci, College Stn, TX 77843 USA.
EM labbott@cvm.tamu.edu
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NR 39
TC 29
Z9 30
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD AUG 5
PY 2008
VL 191
IS 1
BP 55
EP 61
DI 10.1016/j.bbr.2008.03.008
PG 7
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 314CP
UT WOS:000256786700008
PM 18436314
ER
PT J
AU Saverino, C
Gerlai, R
AF Saverino, Cristina
Gerlai, Robert
TI The social zebrafish: Behavioral responses to conspecific,
heterospecific, and computer animated fish
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Article
DE behavioral phenotyping; Dania rerio; shoaling; social behavior;
zebrafish
ID DANIO-RERIO; SHOALING BEHAVIOR; TRANSGENIC MICE; GENETIC MODEL;
HUMAN-DISEASE; AUTISM; SIZE; QUANTIFICATION; PREFERENCES; FAMILIARITY
AB Zebrafish has been in the forefront of developmental biology and genetics, but only recently has interest in their behavior increased. Zebrafish are small and prolific, which lends this species to high throughput screening applications. A typical feature of zebrafish is its propensity to aggregate in groups, a behavior known as shoaling. Thus, zebrafish has been proposed as a possible model organism appropriate for the analysis of the genetics of vertebrate social behavior. However, shoaling behavior is not well characterized in zebrafish. Here, using a recently developed software application, we first investigate how zebrafish respond to conspecific and heterospecific fish species that differ in coloration and/or shoaling tendencies. We found that zebrafish shoaled with their own species but not with two heterospecific species, one of which was a shoaling the other a non-shoaling species. In addition, we have started the analysis of visual stimuli that zebrafish may utilize to determine whether to shoal with a fish or not. We systematically modified the color, the location, the pattern, and the body shape of computer animated zebrafish images and presented them to experimental zebrafish. The subjects responded differentially to some of these stimuli showing preference for yellow and avoidance of elongated zebrafish images. Our results suggest that computerized stimulus presentation and automated behavioral quantification of zebrafish responses are feasible, which in turn implies that high throughput forward genetic mutation or drug screening will be possible in the analysis of social behavior with this model organism. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Saverino, Cristina; Gerlai, Robert] Univ Toronto, Dept Psychol, Mississauga, ON L5L 1C6, Canada.
RP Gerlai, R (reprint author), Univ Toronto, Dept Psychol, Room 3035,3359 Mississauga Rd, Mississauga, ON L5L 1C6, Canada.
EM robert.gerlai@utoronto.ca
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NR 45
TC 104
Z9 105
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD AUG 5
PY 2008
VL 191
IS 1
BP 77
EP 87
DI 10.1016/j.bbr.2008.03.013
PG 11
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 314CP
UT WOS:000256786700012
PM 18423643
ER
PT J
AU Moy, SS
Nadler, JJ
Young, NB
Nonneman, RJ
Segall, SK
Andrade, GM
Crawley, JN
Magnuson, TR
AF Moy, Sheryl S.
Nadler, Jessica J.
Young, Nancy B.
Nonneman, Randal J.
Segall, Samantha K.
Andrade, Gabriela M.
Crawley, Jacqueline N.
Magnuson, Terry R.
TI Social approach and repetitive behavior in eleven inbred mouse strains
SO BEHAVIOURAL BRAIN RESEARCH
LA English
DT Article
DE autism; morris water maze; reversal learning; sociability; social
preference; spectrum disorders; stereotypy; T-maze
ID AUTISM SPECTRUM DISORDERS; FMR1 KNOCKOUT MICE; EXECUTIVE FUNCTIONS;
MODEL; GENE; PHENOTYPE; RELEVANT; SOCIABILITY; ENVIRONMENT; CHILDREN
AB Core symptoms of autism include deficits in social interaction, impaired communication, and restricted, repetitive behaviors. The repetitive behavior domain encompasses abnormal motoric stereotypy, an inflexible insistence on sameness, and resistance to change. In recent years, many genetic mouse models of autism and related disorders have been developed, based on candidate genes for disease susceptibility. The present studies are part ofan ongoing initiative to develop appropriate behavioral tasks for the evaluation of mouse models relevant to autism. We have previously reported profiles for sociability, preference for social novelty, and resistance to changes in a learned pattern of behavior, as well as other functional domains, for 10 inbred mouse strains of divergent genetic backgrounds. The present studies extend this multi-component behavioral characterization to several additional strains: C58/J, NOD/LtJ, NZB/B1NJ, PL/J, SJL/J, SWR/J, and the wild-derived PERA/EiJ. C58/J, NOD/LEJ, NZB/B1NJ, SJL/J, and PERA/EiJ demonstrated low sociability, measured by time spent in proximity to an unfamiliar conspecific, with 30-60% of mice from these strains showing social avoidance. In the Morris water maze, NZB/B1NJ had a persistent bias for the quadrant where the hidden platform was located during acquisition, even after 9 days of reversal training. A particularly interesting profile was found for C58/J, which had low social preference, poor performance in the T-maze, and overt motoric stereotypy. Overall, this set of tasks and observational methods provides a strategy for evaluating novel mouse models in behavioral domains relevant to the autism phenotype. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Moy, Sheryl S.; Nadler, Jessica J.; Young, Nancy B.; Nonneman, Randal J.; Andrade, Gabriela M.; Crawley, Jacqueline N.; Magnuson, Terry R.] Univ N Carolina, Sch Med, Neurodev Disorders Res Ctr, Chapel Hill, NC 27599 USA.
[Moy, Sheryl S.; Crawley, Jacqueline N.] Univ N Carolina, Sch Med, Dept Psychiat, Chapel Hill, NC 27599 USA.
[Nadler, Jessica J.; Segall, Samantha K.; Magnuson, Terry R.] Univ N Carolina, Dept Genet, Chapel Hill, NC 27599 USA.
[Crawley, Jacqueline N.] NIMH, Lab Behav Neurosci, Intramural Res Program, Bethesda, MD 20892 USA.
RP Moy, SS (reprint author), Univ N Carolina, Sch Med, Neurodev Disorders Res Ctr, CB 7146, Chapel Hill, NC 27599 USA.
EM ssmoy@med.unc.edu
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NR 50
TC 91
Z9 91
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0166-4328
J9 BEHAV BRAIN RES
JI Behav. Brain Res.
PD AUG 5
PY 2008
VL 191
IS 1
BP 118
EP 129
DI 10.1016/j.bbr.2008.03.015
PG 12
WC Behavioral Sciences; Neurosciences
SC Behavioral Sciences; Neurosciences & Neurology
GA 314CP
UT WOS:000256786700017
PM 18440079
ER
PT J
AU Spitzer, NC
AF Spitzer, Nicholas C.
TI A Rosetta stone for analysis of human membrane protein function
SO PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF
AMERICA
LA English
DT Editorial Material
ID ACETYLCHOLINE-RECEPTORS; XENOPUS OOCYTES; MESSENGER-RNA; AUTISM; BRAIN;
MECHANISMS; CELLS
C1 [Spitzer, Nicholas C.] Univ Calif San Diego, Ctr Mol Genet, Div Biol Sci, Neurobiol Sect, La Jolla, CA 92093 USA.
[Spitzer, Nicholas C.] Univ Calif San Diego, Kavli Inst Brain & Mind, La Jolla, CA 92093 USA.
RP Spitzer, NC (reprint author), Univ Calif San Diego, Ctr Mol Genet, Div Biol Sci, Neurobiol Sect, La Jolla, CA 92093 USA.
EM nspitzer@ucsd.edu
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NR 16
TC 1
Z9 1
PU NATL ACAD SCIENCES
PI WASHINGTON
PA 2101 CONSTITUTION AVE NW, WASHINGTON, DC 20418 USA
SN 0027-8424
J9 P NATL ACAD SCI USA
JI Proc. Natl. Acad. Sci. U. S. A.
PD AUG 5
PY 2008
VL 105
IS 31
BP 10641
EP 10642
DI 10.1073/pnas.0806110105
PG 2
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 335RT
UT WOS:000258308500004
PM 18669649
ER
PT J
AU Limon, A
Reyes-Ruiz, JM
Miledi, R
AF Limon, Agenor
Reyes-Ruiz, Jorge Mauricio
Miledi, Ricardo
TI Microtransplantation of neurotransmitter receptors from postmortem
autistic brains to Xenopus oocytes
SO PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF
AMERICA
LA English
DT Article
DE autism; human brain; GABA receptors; glutamate receptors
ID TEMPORAL-LOBE EPILEPSY; MESSENGER-RNA; FROG OOCYTES; NEURODEVELOPMENTAL
DISORDERS; A RECEPTORS; RAT-BRAIN; EXPRESSION; MEMBRANES; GLUTAMATE;
CHANNELS
AB Autism is a complex disorder that arises from the pervasive action of genetic and epigenetic factors that alter synaptic connectivity of the brain. Although GABA and glutamate receptors seem to be two of those factors, very little is known about the functional properties of the autistic receptors. Autistic tissue samples stored in brain banks usually have relatively long postmortem times, and it is highly desirable to know whether neurotransmitter receptors in such tissues are still functional. Here we demonstrate that native receptors microtransplanted from autistic brains, as well as de novo mRNA-expressed receptors, are still functional and susceptible to detailed electrophysiological characterization even after long postmortem intervals. The opportunity to study the properties of human receptors present in diseased brains not only opens new avenues toward understanding autism and other neurological disorders, but it also makes the microtransplantation method a useful translational system to evaluate and develop novel medicinal drugs.
C1 [Limon, Agenor; Reyes-Ruiz, Jorge Mauricio; Miledi, Ricardo] Univ Calif Irvine, Dept Neurobiol & Behav, Irvine, CA 92697 USA.
[Miledi, Ricardo] Univ Nacl Autonoma Mexico, Inst Neurobiol, Juriquilla 76230, Queretaro, Mexico.
RP Miledi, R (reprint author), 2205 McGaugh Hall, Irvine, CA 92697 USA.
EM rmiledi@uci.edu
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NR 39
TC 11
Z9 11
PU NATL ACAD SCIENCES
PI WASHINGTON
PA 2101 CONSTITUTION AVE NW, WASHINGTON, DC 20418 USA
SN 0027-8424
J9 P NATL ACAD SCI USA
JI Proc. Natl. Acad. Sci. U. S. A.
PD AUG 5
PY 2008
VL 105
IS 31
BP 10973
EP 10977
DI 10.1073/pnas.0804386105
PG 5
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 335RT
UT WOS:000258308500063
PM 18645182
ER
PT J
AU Sinzig, J
Bruning, N
Morsch, D
Lehmkuhl, G
AF Sinzig, Judith
Bruning, Nicole
Morsch, Dagmar
Lehmkuhl, Gerd
TI Attention profiles in autistic children with and without comorbid
hyperactivity and attention problems
SO ACTA NEUROPSYCHIATRICA
LA English
DT Article
DE ADHD; attention; autism; neuropsychology
ID PERVASIVE DEVELOPMENTAL DISORDER; EXECUTIVE FUNCTION DEFICITS;
HIGH-FUNCTIONING AUTISM; DEFICIT/HYPERACTIVITY DISORDER; COEXISTING
PSYCHOPATHOLOGY; PSYCHIATRIC-DISORDERS; SUSTAINED ATTENTION; SPECTRUM
DISORDERS; BEHAVIOR DOMAINS; GENOMEWIDE SCAN
AB Objective: Psychopathological, neuropsychological and genetic findings indicate an association between ASD Spectrum Disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD). The goal of this study was to compare the neuropsychological profiles of attention functions in children with ADHD and with ASD and without comorbid ADHD. The hypothesis was that either ADHD and autistic children with comorbid ADHD symptoms were more impaired in inhibition and sustained attention performance and that all individuals with ASD show more deficits in divided attention.
Method: Children aged 6 to 18 years old with ADHD (n = 30) or ASD with (n = 21) and without comorbid ADHD (n = 20) and 30 healthy children were included consecutively. Psychopathology was evaluated using the KIDDIE-SADS and symptom checklists for ADHD and ASD according to DSM-IV. Assessed neuropsychological functioning included inhibition, sustained as well as divided attention and alertness tasks.
Results: Age and IQ-corrected z-scores were used. Statistically significant group effects were found for the variables sustained attention median (F = 3.2, = .02), hits (F = 3.3, p = .02) and false alarms (F = 3.9, p = .01), divided attention hits (F = 3.3, p = .02), errors (F = 3.1, p = .03) and false alarms (F = 3.3, p = .03) and alertness false alarms (F = 2.9, p = .04). Pearson Correlations revealed associations between ADHD symptoms and sustained attention in the ADHD group and between ADHD symptoms and inhibition in the ASD+ group.
Conclusion: Our hypothesis was partly confirmed as ADHD children showed more deficits in sustained attention and ASD children in divided attention tasks. However there was no evidence that children with ASD and comorbid ADHD symptoms have a specific profile in comparison to pure ASD children.
C1 [Sinzig, Judith; Bruning, Nicole; Morsch, Dagmar; Lehmkuhl, Gerd] Univ Cologne, Dept Child & Adolescent Psychiat, D-50931 Cologne, Germany.
RP Sinzig, J (reprint author), Univ Cologne, Dept Child & Adolescent Psychiat, Robert Koch Str 10, D-50931 Cologne, Germany.
EM judith.sinzig@uk-koeln.de
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NR 47
TC 17
Z9 18
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0924-2708
J9 ACTA NEUROPSYCHIATR
JI Acta Neuropsychiatr.
PD AUG
PY 2008
VL 20
IS 4
BP 207
EP 215
DI 10.1111/j.1601-5215.2008.00292.x
PG 9
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 327UG
UT WOS:000257753400006
PM 25385656
ER
PT J
AU Kondziella, D
Lycke, J
AF Kondziella, Daniel
Lycke, Jan
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a role?
SO ACTA NEUROPSYCHIATRICA
LA English
DT Editorial Material
ID ASYMMETRY
C1 [Kondziella, Daniel; Lycke, Jan] Gothenburg Univ, Sahlgrenska Hosp, Dept Neurol, Gothenburg, Sweden.
RP Kondziella, D (reprint author), Sahlgrens Univ Hosp, Dept Neurol, SE-41345 Gothenburg, Sweden.
EM daniel_kondziella@yahoo.com
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TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0924-2708
J9 ACTA NEUROPSYCHIATR
JI Acta Neuropsychiatr.
PD AUG
PY 2008
VL 20
IS 4
BP 227
EP U1
DI 10.1111/j.1601-5215.2008.00298.x
PG 2
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 327UG
UT WOS:000257753400011
ER
PT J
AU Garcia-Nonell, C
Ratera, ER
Harris, S
Hessl, D
Ono, MY
Tartaglia, N
Marvin, E
Tassone, F
Hagerman, RJ
AF Garcia-Nonell, Catalina
Ratera, Euienia Rigau
Harris, Susan
Hessl, David
Ono, Michele Y.
Tartaglia, Nicole
Marvin, Emily
Tassone, Flora
Hagerman, Randi J.
TI Secondary medical diagnosis in fragile X syndrome with and without
autism spectrum disorder
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Article
DE fragile X syndrome; autism; PDDNOS; autism spectrum disorder; seizures;
Prader-Willi like phenotype
ID PERVASIVE DEVELOPMENTAL DISORDERS; WILLI-LIKE PHENOTYPE;
BEHAVIORAL-PHENOTYPE; MENTAL-RETARDATION; YOUNG-CHILDREN; EEG FINDINGS;
MALES; EPILEPSY; FMRP; COMMUNICATION
AB This study examine whether secondary medical diagnoses that affects CNS function (i.e., seizures, malformations, or genetic disorders), are more likely to occur in individuals with fragile X syndrome (FXS) and autism spectrum disorder (FXS + ASD) or FXS alone. Ninety males (3-25 years) with FXS or FXS + ASD were evaluated for secondary medical diagnoses by medical history and examination. A significant difference in the incidence of medical problems was found between patients with FXS + ASD (38.6%) and FXS alone (18.2%, P < 0.05). Medical problems that affect the CNS are more likely to occur in those with FXS + ASD and it is probable that additional brain dysfunction associated with these medical problems enhance the risk of autism in those with FXS. (c) 2008 Wiley-Liss, Inc.
C1 [Hagerman, Randi J.] Univ Calif Davis Hlth Syst, Dept Pediat, Endowed Chair Fragile X Res, MIND Inst, Sacramento, CA 95817 USA.
[Garcia-Nonell, Catalina] PSYNCRON, Barcelona, Spain.
[Ratera, Euienia Rigau] Hosp Sabadell, Corp Sanit Parc Tauli, GIRMOGEN, Unidad Neuropediat, Sabadell, Spain.
[Hessl, David] Univ Calif Davis Hlth Syst, Dept Psychiat & Behav Sci, Sacramento, CA 95817 USA.
[Tartaglia, Nicole] Univ Colorado, Hlth Sci Ctr, Childrens Hosp, Child Dev Unit, Denver, CO USA.
[Tassone, Flora] Univ Calif Davis, Sch Med, Dept Biochem & Mol Med, Sacramento, CA 95817 USA.
RP Hagerman, RJ (reprint author), Univ Calif Davis Hlth Syst, Dept Pediat, Endowed Chair Fragile X Res, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM randi.hagerman@ucdmc.ucdavis.edu
FU NICHD [HD36071, HD02274]; NIH Pediatric Research LRP
FX This study was partially supported by NICHD HD36071 and HD02274, and the
NIH Pediatric Research LRP for Dr. Tartaglia. We would also like to
thank the M.I.N.D. Institute and Dr. Josep Artigas for providing the
opportunity for Catalina Garcia Nonell and Eugenia Rigau Ratera to study
at the M.I.N.D Institute.
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NR 55
TC 38
Z9 38
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD AUG 1
PY 2008
VL 146A
IS 15
BP 1911
EP 1916
DI 10.1002/ajmg.a.32290
PG 6
WC Genetics & Heredity
SC Genetics & Heredity
GA 338AI
UT WOS:000258476800002
PM 18627038
ER
PT J
AU Calounova, G
Hedvicakova, P
Silhanova, E
Kreckova, G
Sedlacek, Z
AF Calounova, Gabriela
Hedvicakova, Petra
Silhanova, Eva
Kreckova, Gabriela
Sedlacek, Zdenek
TI Molecular and clinical characterization of two patients with
Prader-Willi syndrome and atypical deletions of proximal chromosome 15q
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Article
DE Prader-Willi syndrome; atypical deletions; methylation-specific MLPA;
microarray CGH; deletion breakpoints; breakpoint cloning
ID AUTISM-SPECTRUM DISORDERS; INTERSTITIAL DELETION; UNIPARENTAL DISOMY;
GENETIC SUBTYPES; ARRAY-CGH; PHENOTYPE; DISEASE; REGION; REARRANGEMENTS;
EXPRESSION
AB Prader-Willi syndrome (PWS) is caused by the disturbed expression of genes from the imprinted region of 15q11-q13, but the specific contributions of individual genes remain unknown. Most paternal PWS deletions are bracketed by recurrent breakpoints BP1 or BP2 and BP3. Atypical deletions are very rare. In the present work, we describe the molecular analysis of two patients with atypical deletions using microsatellite analysis, methylation-specific MLPA, and microarray CGH. A deletion of about 2 Mb in Patient 1 started at BP2 and ended in the middle of the typically deleted region within the UBE3A gene. The deletion in Patient 2 started 1.3 Mb distal from BP2 within the C15ORF2 gene, extended over 9.5 Mb, and ended within the AVEN gene in proximal 15q14. In Patient 1 both deletion breakpoints involved repetitive regions, which precluded cloning of the junction and pointed to non-allelic homologous recombination as a possible mechanism of this rearrangement. The breakpoints in Patient 2 were sequenced, and their structure suggested non-homologous end joining as the most likely cause of this deletion. The phenotype of both patients did not depart significantly from the typical clinical picture of PWS, although some symptoms in Patient 2 were also reminiscent of the phenotype of individuals with the recently described 15q13.3 microdeletion syndrome. Our findings support previous observations of relatively mild phenotypic effects resulting from deletions that extend distally from the PWS region and observations of the modest effects of different types of genetic defects on the spectrum and severity of symptoms in PWS. (c) 2008 Wiley-Liss, Inc.
C1 [Calounova, Gabriela; Hedvicakova, Petra; Sedlacek, Zdenek] Charles Univ Prague, Sch Med 2, Dept Biol & Med Genet, Prague 15006 5, Czech Republic.
[Calounova, Gabriela; Hedvicakova, Petra; Sedlacek, Zdenek] Univ Hosp Motol, Prague 15006 5, Czech Republic.
[Silhanova, Eva] Univ Ostrava, Univ Hosp Ostrava, Dept Med Genet, Ostrava, Czech Republic.
[Kreckova, Gabriela] Gennet, Liberec, Czech Republic.
RP Sedlacek, Z (reprint author), Charles Univ Prague, Sch Med 2, Dept Biol & Med Genet, Uvalu 84, Prague 15006 5, Czech Republic.
EM zdenek.sedlacek@ifmotol.cuni.cz
FU Ministry of Health of the Czech Republic [NR/9457-3]
FX Grant sponsor Ministry of Health of the Czech Republic; Grant number;
NR/9457-3
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TC 6
Z9 6
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD AUG 1
PY 2008
VL 146A
IS 15
BP 1955
EP 1962
DI 10.1002/ajmg.a.32416
PG 8
WC Genetics & Heredity
SC Genetics & Heredity
GA 338AI
UT WOS:000258476800007
PM 18627056
ER
PT J
AU Williams, CA
Dagli, A
Battaglia, A
AF Williams, Charles A.
Dagli, Aditi
Battaglia, Agatino
TI Genetic disorders associated with macrocephaly
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Review
DE macrocephaly syndromes; familial macrocephaly; autism; megalencephaly;
genetics; macrocephaly
ID FRAGILE-X-SYNDROME; MARMORATA-TELANGIECTATICA-CONGENITA; PERVASIVE
DEVELOPMENTAL DISORDERS; GENOTYPE-PHENOTYPE CORRELATION; AUTISM SPECTRUM
DISORDERS; CAUSE NOONAN-SYNDROME; FRONTAL SUBARACHNOID SPACE;
FACIO-CUTANEOUS SYNDROME; MUTATIONS CAUSE NOONAN; OF-FUNCTION MUTATIONS
AB Macrocephaly is associated with many genetic disorders and is a frequent cause of referral to the clinical geneticist. In this review we classify the commonly encountered macrocephaly disorders into useful categories and summarize recent genetic advances. Conditions where macrocephaly is a predominant aspect of the clinical presentation are discussed and a diagnostic approach to the common macrocephaly disorders is provided. Some emphasis is placed on familial macrocephaly (sometimes referred to as benign external hydrocephalus) and on the macrocephaly associated with autism spectrum disorders. The more recent conditions associated with the leukodystrophies and the organic acidurias are reviewed, but the well known conditions involving storage disorders and bone dysplasias are mentioned but not discussed. The genetic macrocephaly conditions cover a broad spectrum of gene disorders and their related proteins have diverse biological functions. As of yet it is not clear what precise biological pathways lead to generalized brain overgrowth. (c) 2008 Wiley-Liss, Inc.
C1 [Williams, Charles A.; Dagli, Aditi] Univ Florida, Dept Pediat, Div Genet, Raymond C Philips Res & Educ Unit, Gainesville, FL 32610 USA.
[Battaglia, Agatino] Stella Maris Clin Res Inst Child & Adolescent Neu, Pisa, Italy.
RP Williams, CA (reprint author), Univ Florida, Dept Pediat, Div Genet, Raymond C Philips Res & Educ Unit, POB 100296, Gainesville, FL 32610 USA.
EM willicx@peds.ufl.edu
FU Raymond C. Philips Research and Education Contract, State of Florida,
USA.
FX The authors declare that they have no competing or financial interests
related to this scientific work. This study was funded in part by the
Raymond C. Philips Research and Education Contract, State of Florida,
USA.
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Z9 39
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD AUG 1
PY 2008
VL 146A
IS 15
BP 2023
EP 2037
DI 10.1002/ajmg.a.32434
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SC Genetics & Heredity
GA 338AI
UT WOS:000258476800021
PM 18629877
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SO AMERICAN JOURNAL OF PSYCHIATRY
LA English
DT Article
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HIGH-FUNCTIONING AUTISM; PERINATAL FACTORS; CHILDREN; DISABILITIES;
INTERVIEW
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PM 18676600
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LA English
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BP 1350
EP 1350
DI 10.2105/AJPH.2008.138776
PG 1
WC Public, Environmental & Occupational Health
SC Public, Environmental & Occupational Health
GA 330KU
UT WOS:000257940800003
PM 18556596
ER
PT J
AU Schlosser, RW
Wendt, O
AF Schlosser, Ralf W.
Wendt, Oliver
TI Effects of augmentative and alternative communication intervention on
speech production in children with autism: A systematic review
SO AMERICAN JOURNAL OF SPEECH-LANGUAGE PATHOLOGY
LA English
DT Review
DE augmentative and alternative communication; autism spectrum disorders;
speech improvement; systematic review
ID SINGLE-SUBJECT RESEARCH; DEVELOPMENTAL-DISABILITIES; SIGN LANGUAGE;
PHASE-III; PECS; INDIVIDUALS; ACQUISITION; BEHAVIOR; OUTPUT; DISORDERS
AB Purpose: This systematic review aimed to determine the effects of augmentative and alternative communication (AAC) intervention on speech production in children with autism or pervasive developmental disorder-not otherwise specified.
Method: A systematic review methodology was utilized to limit bias in searching, selecting, coding, and synthesizing relevant treatment studies. This involved a multifaceted search for studies written between 1975 and May 2007 using various bibliographic databases, dissertation databases, hand searches of selected journals and published compilations of AAC theses and dissertations, and ancestry searches. To be included, studies had to meet stringent criteria. A coding manual and form facilitated data extraction in terms of participant characteristics, treatment characteristics, design and measurement, and outcomes.
Results: Nine single-subject experimental design (27 participants) and 2 group studies (98 participants) were included. Results indicated that AAC interventions do not impede speech production. In fact, most studies reported an increase in speech production. However, in-depth analyses revealed. that the gains were rather modest.
Conclusions: Although AAC interventions do not appear to impede speech production and may result in increased speech production, the modest gains observed require realistic expectations among clinicians and other stakeholders. Future research should be more hypothesis driven and aim to identify predictive child characteristics, such as prior speech imitation and object exploration skills.
C1 [Schlosser, Ralf W.] Northeastern Univ, Dept Speech Language Pathol & Audiol, Boston, MA 02115 USA.
[Wendt, Oliver] Purdue Univ, W Lafayette, IN 47907 USA.
RP Schlosser, RW (reprint author), Northeastern Univ, Dept Speech Language Pathol & Audiol, 151B Forsyth, Boston, MA 02115 USA.
EM r.schlosser@neu.edu
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STUDIES WERE INCLUDE
NR 77
TC 52
Z9 52
PU AMER SPEECH-LANGUAGE-HEARING ASSOC
PI ROCKVILLE
PA 10801 ROCKVILLE PIKE, ROCKVILLE, MD 20852-3279 USA
SN 1058-0360
EI 1558-9110
J9 AM J SPEECH-LANG PAT
JI Am. J. Speech-Lang. Pathol.
PD AUG
PY 2008
VL 17
IS 3
BP 212
EP 230
DI 10.1044/1058-0360(2008/021)
PG 19
WC Audiology & Speech-Language Pathology; Linguistics; Rehabilitation
SC Audiology & Speech-Language Pathology; Linguistics; Rehabilitation
GA 334DB
UT WOS:000258201400002
PM 18663107
ER
PT J
AU Reichle, J
Dropik, PL
Alden-Anderson, E
Haley, T
AF Reichle, Joe
Dropik, Patricia L.
Alden-Anderson, Elizabeth
Haley, Tom
TI Teaching a young child with autism to request assistance conditionally:
A preliminary study
SO AMERICAN JOURNAL OF SPEECH-LANGUAGE PATHOLOGY
LA English
DT Article
DE autism; children; intervention; communication; conditional
discrimination; generalized requesting
ID FUNCTIONAL COMMUNICATION; RESPONSE EFFICIENCY; REINFORCEMENT;
ACQUISITION; BEHAVIORS; MAND
AB Purpose: Investigators taught a 5-year-old boy with autistic disorder and severe language delay to conditionally use requests for assistance.
Method: A within-participant multiple-probe design across 3 functional tasks was implemented in order to evaluate the child's acquisition and conditional use of requests for assistance during intervention with each task.
Results: Results indicated initial acquisition of requests for assistance followed by a brief period of overgeneralization. As independence in completing a task increased, requests for assistance correspondingly decreased. The participant's conditional use of requests for assistance and independent task completion were sustained across time.
Conclusion: This study highlights the need to assess conditional use of newly taught communicative behavior.
C1 [Reichle, Joe] Univ Minnesota, Dept Speech Language Hearing Sci, Minneapolis, MN 55455 USA.
[Haley, Tom] Minnesota Autism Ctr, Minnetonka, MN USA.
RP Reichle, J (reprint author), Univ Minnesota, Dept Speech Language Hearing Sci, Minneapolis, MN 55455 USA.
EM reich001@umn.edu
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NR 27
TC 4
Z9 4
PU AMER SPEECH-LANGUAGE-HEARING ASSOC
PI ROCKVILLE
PA 10801 ROCKVILLE PIKE, ROCKVILLE, MD 20852-3279 USA
SN 1058-0360
J9 AM J SPEECH-LANG PAT
JI Am. J. Speech-Lang. Pathol.
PD AUG
PY 2008
VL 17
IS 3
BP 231
EP 240
DI 10.1044/1058-0360(2008/022)
PG 10
WC Audiology & Speech-Language Pathology; Linguistics; Rehabilitation
SC Audiology & Speech-Language Pathology; Linguistics; Rehabilitation
GA 334DB
UT WOS:000258201400003
PM 18663108
ER
PT J
AU Nadon, G
Feldman, DE
Gisel, E
AF Nadon, G.
Feldman, D. Ehrmann
Gisel, E.
TI Review of assessment methods used to evaluate feeding for children with
pervasive developmental disorder
SO ARCHIVES DE PEDIATRIE
LA French
DT Article
ID TREAT FOOD SELECTIVITY; ANOREXIA-NERVOSA; YOUNG-CHILDREN; AUTISM;
SYMPTOMS; REINFORCEMENT; INTERVENTION; DISABILITIES; RELIABILITY;
CONSUMPTION
AB Current evaluations used by occupational therapists to assess and treat feeding problems derive mainly from the domain of dysphagia. The purpose of this article is to familiarize the reader with tools used, in research, for children with pervasive developmental disorders (PDD) and to determine if any of these meet the needs of occupational therapists. The following data bases were searched: Medline, CINAHL, HAPI and PsyINFO, using the terms pervasive developmental disorder, autism, Asperger syndrome, pervasive developmental disorder not otherwise specified, eating behavior, eating disorder, food preference, food selectivity, feeding disorders, Picky eater and child. All articles published between 1980 and 2006 (n = 27) were reviewed. A total of 20 studies met our selection criteria. Assessment methods are compared using the Disability Creation Model (DCP). The DCP is the Quebec alternative to the International Classification of Functioning, Disability and Health (ICF). None of the evaluation tools reviewed met all factors that may influence eating in children with PDD. Implications for research and practice in occupational therapy are discussed. (C) 2008 Elsevier Masson SAS. All rights reserved.
C1 [Nadon, G.; Feldman, D. Ehrmann] Univ Montreal, Ecole Readaptat, Fac Med, Montreal, PQ H3N 1X7, Canada.
[Gisel, E.] McGill Univ, Fac Med, Ecole Physiotherapie & Ergotherapie, Montreal, PQ H3G 1Y5, Canada.
RP Nadon, G (reprint author), CR La Myriade, 1280 Blvd St Henri, Mascouche, PQ J7K 2N1, Canada.
EM gen_nadon@hotmail.com
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NR 50
TC 1
Z9 1
PU ELSEVIER FRANCE-EDITIONS SCIENTIFIQUES MEDICALES ELSEVIER
PI PARIS
PA 23 RUE LINOIS, 75724 PARIS, FRANCE
SN 0929-693X
J9 ARCH PEDIATRIE
JI Arch. Pediatr.
PD AUG
PY 2008
VL 15
IS 8
BP 1332
EP 1348
DI 10.1016/j.arcped.2008.04.022
PG 17
WC Pediatrics
SC Pediatrics
GA 335TC
UT WOS:000258312600014
PM 18562184
ER
PT J
AU Jones, W
Carr, K
Klin, A
AF Jones, Warren
Carr, Katelin
Klin, Ami
TI Absence of preferential looking to the eyes of approaching adults
predicts level of social disability in 2-year-old toddlers with autism
spectrum disorder
SO ARCHIVES OF GENERAL PSYCHIATRY
LA English
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDERS; CHIMPANZEES PAN-TROGLODYTES;
HIGH-FUNCTIONING AUTISM; HUMAN AMYGDALA; BIOLOGICAL MOTION; GAZE;
CHILDREN; RECOGNITION; FACE; MOVEMENTS
AB Context: Within the first week of life, typical human newborns give preferential attention to the eyes of others. Similar findings in other species suggest that attention to the eyes is a highly conserved phylogenetic mechanism of social development. For children with autism, however, diminished and aberrant eye contact is a lifelong hallmark of disability.
Objective: To quantify preferential attention to the eyes of others at what is presently the earliest point of diagnosis in autism.
Design: We presented the children with 10 videos. Each video showed an actress looking directly into the camera, playing the role of caregiver, and engaging the viewer ( playing pat- a- cake, peek- a- boo, etc). Children's visual fixation patterns were measured by eye tracking.
Participants: Fifteen 2- year- old children with autism were compared with 36 typically developing children and with 15 developmentally delayed but nonautistic children.
Main Outcome Measure: Preferential attention was measured as percentage of visual fixation time to 4 regions of interest: eyes, mouth, body, and object. Level of social disability was assessed by the Autism Diagnostic Observation Schedule.
Results: Looking at the eyes of others was significantly decreased in 2- year- old children with autism ( P <. 001), while looking at mouths was increased ( P <. 01) in comparison with both control groups. The 2 control groups were not distinguishable on the basis of fixation patterns. In addition, fixation on eyes by the children with autism correlated with their level of social disability; less fixation on eyes predicted greater social disability ( r=- 0.669, P <. 01).
Conclusions: Looking at the eyes of others is important in early social development and in social adaptation throughout one's life span. Our results indicate that in 2- year- old children with autism, this behavior is already derailed, suggesting critical consequences for development but also offering a potential biomarker for quantifying syndrome manifestation at this early age.
C1 [Klin, Ami] Yale Univ, Sch Med, Yale Child Study Ctr, Interdepartmental Neurosci Program, New Haven, CT 06520 USA.
RP Klin, A (reprint author), Yale Univ, Sch Med, Yale Child Study Ctr, Interdepartmental Neurosci Program, 230 S Frontage Rd, New Haven, CT 06520 USA.
EM ami.klin@yale.edu
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NR 64
TC 119
Z9 122
PU AMER MEDICAL ASSOC
PI CHICAGO
PA 515 N STATE ST, CHICAGO, IL 60610-0946 USA
SN 0003-990X
J9 ARCH GEN PSYCHIAT
JI Arch. Gen. Psychiatry
PD AUG
PY 2008
VL 65
IS 8
BP 946
EP 954
DI 10.1001/archpsyc.65.8.946
PG 9
WC Psychiatry
SC Psychiatry
GA 334MH
UT WOS:000258225400010
PM 18678799
ER
PT J
AU Sutcliffe, JS
AF Sutcliffe, James S.
TI Heterogeneity and the Design of Genetic Studies in Autism
SO AUTISM RESEARCH
LA English
DT Editorial Material
C1 [Sutcliffe, James S.] Vanderbilt Univ, Vanderbilt Kennedy Ctr Res Human Dev, Nashville, TN 37235 USA.
[Sutcliffe, James S.] Vanderbilt Univ, Ctr Human Genet Res, Nashville, TN USA.
RP Sutcliffe, JS (reprint author), Vanderbilt Univ, Vanderbilt Kennedy Ctr Res Human Dev, Nashville, TN 37235 USA.
EM james.s.sutcliffe@Vanderbilt.edu
RI Sutcliffe, James/C-1348-2012
OI Sutcliffe, James/0000-0001-5200-6007
NR 0
TC 2
Z9 2
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD AUG
PY 2008
VL 1
IS 4
BP 205
EP 206
DI 10.1002/aur.37
PG 2
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CL
UT WOS:000270031900001
PM 19360669
ER
PT J
AU Bailey, AJ
AF Bailey, Anthony J.
TI The Neuroscience of Autism Education
SO AUTISM RESEARCH
LA English
DT Editorial Material
EM anthony.bailey@psych.ox.ac.uk
RI Bailey, Anthony/J-2860-2014
OI Bailey, Anthony/0000-0003-4257-972X
NR 0
TC 0
Z9 0
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD AUG
PY 2008
VL 1
IS 4
BP 207
EP 207
DI 10.1002/aur.36
PG 1
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CL
UT WOS:000270031900002
PM 19360670
ER
PT J
AU Tek, S
Jaffery, G
Fein, D
Naigles, LR
AF Tek, Saime
Jaffery, Gul
Fein, Deborah
Naigles, Letitia R.
TI Do Children With Autism Spectrum Disorders Show a Shape Bias in Word
Learning?
SO AUTISM RESEARCH
LA English
DT Article
DE shape bias; word learning; language; development
ID YOUNG-CHILDREN; LANGUAGE-ACQUISITION; LATE TALKERS; CATEGORIZATION;
COMPREHENSION; INDIVIDUALS; IMPAIRMENT; BEHAVIOR; SYNTAX; TASK
AB Many children with autism spectrum disorders (ASD) acquire a sizeable lexicon. However, these children also seem to understand and/or store the meanings of words differently from typically developing children. One of the mechanisms that helps typically developing children learn novel words is the shape bias, in which the referent of a noun is mapped onto the shape of an object, rather than onto its color, texture, or size. We hypothesized that children with autistic disorder would show reduced or absent shape bias. Using the intermodal preferential looking paradigm, we compared the performance of young children with ASD and typically developing children (TYP), across four time points, in their use of shape bias. Neither group showed a shape bias at Visit 1, when half of the children in both groups produced fewer than 50 count nouns. Only the TYP group showed a shape bias at Visits 2, 3, and 4. According to the growth curve analyses, the rate of increase in the shape bias scores over time was significant for the TYP children. The fact that the TYP group showed a shape bias at 24 months of age, whereas children with ASD did not demonstrate a shape bias despite a sizeable vocabulary, supports a dissociation between vocabulary size and principles governing acquisition in ASD children from early in language development.
C1 [Tek, Saime] Univ Connecticut, Dept Psychol, Unit 1020, Storrs, CT 06269 USA.
RP Tek, S (reprint author), Univ Connecticut, Dept Psychol, Unit 1020, 406 Babbidge Rd, Storrs, CT 06269 USA.
EM saime.tek@uconn.edu
FU National Institute on Deafness and Other Communication Disorders [R01
DC07428]
FX Grant sponsor: National Institute on Deafness and Other Communication
Disorders; Grant number: R01 DC07428.
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NR 48
TC 24
Z9 24
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD AUG
PY 2008
VL 1
IS 4
BP 208
EP 222
DI 10.1002/aur.38
PG 15
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CL
UT WOS:000270031900003
PM 19360671
ER
PT J
AU Tannan, V
Holden, JK
Zhang, Z
Baranek, GT
Tommerdahl, MA
AF Tannan, Vinay
Holden, Jameson K.
Zhang, Zheng
Baranek, Grace T.
Tommerdahl, Mark A.
TI Perceptual Metrics of Individuals With Autism Provide Evidence for
Disinhibition
SO AUTISM RESEARCH
LA English
DT Article
DE autism; inhibition; adaptation; habituation; amplitude discrimination;
sensory discrimination
ID PRIMARY VISUAL-CORTEX; VIBROTACTILE ADAPTATION; RECEPTIVE-FIELD; SI
CORTEX; MINICOLUMNAR ORGANIZATION; AMPLITUDE DISCRIMINATION; SPATIAL
LOCALIZATION; DYNAMIC CHANGES; STIMULATION; CAT
AB Adults with autism exhibit inhibitory deficits that are often manifested in behavioral modifications, such as repetitive behaviors, and/or sensory hyper-responsiveness. If such behaviors are the result of a generalized deficiency in inhibitory neurotransmission, then it stands to reason that deficits involving localized cortical-cortical interactions-such as in sensory discrimination tasks-could be detected and quantified. This study exemplifies a newly developed method for quantifying sensory testing metrics. Our novel sensory discrimination tests may provide (a) an effective means for biobehavioral assessment of deficits specific to autism and (b) an efficient and sensitive measure of change following treatment. The sensory discriminative capacity of ten Subjects with autism and ten controls was compared both before and after short duration adapting stimuli. Specifically, vibrotactile amplitude discriminative capacity was obtained both in the presence and absence of I sec adapting stirmili that were delivered 1 sec prior to the comparison stimuli. Although adaptation had a pronounced effect on the amplitude discriminative capacity of the control subjects, little or no impact was observed on the sensory discriminative capacity of the subjects with autism. This lack of impact of the adapting stimuli on the responses of the subjects with autism was interpreted to be consistent with the reduced GABAergic-mediated inhibition described in previous reports. One significant aspect of this Study is that the methods could prove to be a useful and efficient way to detect specific neural deficits and monitor the efficacy of pharmacological or behavioral treatments in autism.
C1 [Tannan, Vinay; Holden, Jameson K.; Zhang, Zheng; Baranek, Grace T.; Tommerdahl, Mark A.] Univ N Carolina, Dept Biomed Engn, Chapel Hill, NC 27599 USA.
RP Tommerdahl, MA (reprint author), Univ N Carolina, Dept Biomed Engn, Chapel Hill, NC 27599 USA.
EM tommerda@med.unc.edu
FU Cure Autism Now Foundation; Department of Defense [W81XWH-07-1-0287]
FX Grant sponsor: Cure Autism Now Foundation; Grant sponsor: Department of
Defense; Grant number: W81XWH-07-1-0287.
CR (APA) APA, 2000, DIAGN STAT MAN MENT
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NR 57
TC 21
Z9 21
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD AUG
PY 2008
VL 1
IS 4
BP 223
EP 230
DI 10.1002/aur.34
PG 8
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CL
UT WOS:000270031900004
PM 19360672
ER
PT J
AU Dalton, KM
Holsen, L
Abbeduto, L
Davidson, RJ
AF Dalton, Kim M.
Holsen, Laura
Abbeduto, Leonard
Davidson, Richard J.
TI Brain Function and Gaze Fixation During Facial-Emotion Processing in
Fragile X and Autism
SO AUTISM RESEARCH
LA English
DT Article
DE fragile X syndrome; autism; face processing; brain function; fMRI
ID PREFRONTAL CORTEX; SOCIAL COGNITION; ACTIVATION; SPECTRUM; AMYGDALA;
FACE; UNDERCONNECTIVITY; COMMUNICATION; EXPRESSION; DISORDERS
AB Fragile X syndrome (FXS) is the most commonly known genetic disorder associated with autism spectrum disorder (ASD). Overlapping features in these populations include gaze aversion, Communication deficits, and social withdrawal. Although the association between FXS and ASD has been well documented at the behavioral level, the underlying neural mechanisms associated with the social/emotional deficits in these groups remain unclear. We collected functional brain images and eye-gaze fixations from 9 individuals with FXS and 14 individuals with idiopathic ASD, as well as 15 typically developing (TD) individuals, while they performed a facial-emotion discrimination task. The FXS group showed a similar yet less aberrant pattern of gaze fixations compared with the ASD group. The FXS group also showed fusiform gyrus (FG) hypoactivation compared with the TD control group. Activation in FG was strongly and positively associated with average eye fixation and negatively associated with ASD characteristics in the FXS group. The FXS group displayed significantly greater activation than both the TD control and ASD groups in the left hippocampus (HIPP), left superior temporal gyrus (STG), right insula (INS), and left postcentral gyrus (PCG). These group differences in brain activation are important as they suggest unique underlying face-processing neural circuitry in FXS versus idiopathic ASD, largely supporting the hypothesis that ASD characteristics in FXS and idiopathic ASD reflect partially divergent impairments at the neural level, at least in FXS individuals without a co-morbid diagnosis of ASD.
C1 [Dalton, Kim M.; Holsen, Laura; Davidson, Richard J.] Univ Wisconsin, Waisman Ctr, Madison, WI 53705 USA.
[Holsen, Laura] Harvard Univ, Sch Med, Dept Psychiat & Med, Boston, MA USA.
[Holsen, Laura] Brigham & Womens Hosp, Boston, MA 02115 USA.
[Davidson, Richard J.] Univ Wisconsin, Dept Psychol, Madison, WI 53706 USA.
[Davidson, Richard J.] Univ Wisconsin, Dept Psychiat, Madison, WI 53706 USA.
[Abbeduto, Leonard] Univ Wisconsin, Dept Educ Psychol, Madison, WI 53706 USA.
RP Dalton, KM (reprint author), Univ Wisconsin, Waisman Ctr T 127, Madison, WI 53705 USA.
EM kmdalton@wisc.edu
FU National Institute of Mental Health (NIMH) Studies [U54MH066398];
National Alliance for Research on Schizophrenia and Affective Disorders
(NARSAD); NIH [P30 HD03352]; NICHD [T32 HD07489]
FX Grant sponsor: National Institute of Mental Health (NIMH) Studies to
Advance Autism Research and Treatment (STAART); Grant number:
U54MH066398; Grant sponsor: National Alliance for Research on
Schizophrenia and Affective Disorders (NARSAD); Grant sponsor: NIH;
Grant number: P30 HD03352; Grant sponsor: NICHD; Grant number: T32
HD07489.
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NR 34
TC 21
Z9 22
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD AUG
PY 2008
VL 1
IS 4
BP 231
EP 239
DI 10.1002/aur.32
PG 9
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CL
UT WOS:000270031900005
PM 19360673
ER
PT J
AU Talebizadeh, Z
Butler, MG
Theodoro, MF
AF Talebizadeh, Zohreh
Butler, Merlin G.
Theodoro, Mariana F.
TI Feasibility and Relevance of Examining Lymphoblastoid Cell Lines to
Study Role of microRNAs in Autism
SO AUTISM RESEARCH
LA English
DT Article
DE microRNA; autism; lymphoblastoid cell lines; differential expression
ID X MENTAL-RETARDATION; NONCODING RNAS; MAMMALIAN MICRORNAS;
BRAIN-DEVELOPMENT; GENE-EXPRESSION; NERVOUS-SYSTEM; REVEALS; TARGETS;
PROTEIN; IDENTIFICATION
AB To assess the feasibility and relevance of using lymphoblastoid cell lines to study the role of noncoding RNAs in the etiology of autism, we evaluated global expression profiling of 470 mature human microRNAs from six subjects with autism compared with six matched controls. Differential expression (either higher or lower) for 9 of the 470 microRNAs was observed in our autism samples compared with controls. Potential target genes for these microRNAs were identified using computer tools, which included several autism susceptibility genes. Our preliminary results indicate microRNAs should be considered and evaluated in the etiology of autism. In addition, analysis of this class of noncoding RNAs in lymphoblastoid cells has the potential to reveal at least a subset of brain-related microRNAs implicated in autism. Subsequently, this model system should allow for detection of complex subtle changes in susceptibility genes/pathways contributing to autism.
C1 [Talebizadeh, Zohreh; Butler, Merlin G.; Theodoro, Mariana F.] Childrens Mercy Hosp & Clin, Sect Med Genet & Mol Med, Kansas City, MO 64108 USA.
[Talebizadeh, Zohreh; Butler, Merlin G.; Theodoro, Mariana F.] Univ Missouri, Sch Med, Kansas City, MO 64108 USA.
RP Talebizadeh, Z (reprint author), Childrens Mercy Hosp & Clin, Sect Med Genet & Mol Med, 2401 Gillham Rd, Kansas City, MO 64108 USA.
EM ztalebi@cmh.edu
FU Children's Mercy Hospital (CMH) [01.3905]; Cure Autism Now (CAN)
Foundation [01.3956]
FX Grant sponsor: Children's Mercy Hospital (CMH) Physician Scientist
Award; Grant number: 01.3905; Grant sponsor: Cure Autism Now (CAN)
Foundation; Grant number: 01.3956.
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NR 59
TC 39
Z9 42
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD AUG
PY 2008
VL 1
IS 4
BP 240
EP 250
DI 10.1002/aur.33
PG 11
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CL
UT WOS:000270031900006
PM 19360674
ER
PT J
AU Sakurai, T
Reichert, J
Hoffman, EJ
Cai, GQ
Jones, HB
Faham, M
Buxbaum, JD
AF Sakurai, Takeshi
Reichert, Jennifer
Hoffman, Ellen J.
Cai, Guiqing
Jones, Hywel B.
Faham, Malek
Buxbaum, Joseph D.
TI A Large-Scale Screen for Coding Variants in SERT/SLC6A4 in Autism
Spectrum Disorders
SO AUTISM RESEARCH
LA English
DT Article
DE resequencing; rare variants; serotonin; rigid-compulsive behavior
ID OBSESSIVE-COMPULSIVE DISORDER; SEROTONIN TRANSPORTER GENE; SLC6A4;
ASSOCIATION; SUSCEPTIBILITY; PHENOTYPE; LOCUS
AB In the current study we explored the hypothesis that rare variants in SLC6A4 contribute to autism susceptibility and to rigid-compulsive behaviors in autism. We made use of a large number of unrelated cases with autism spectrum disorders (similar to 350) and controls (similar to 420) and screened for rare exonic variants in SLC6A4 by a high-throughput method followed by sequencing. We observed no difference in the frequency of such variants in the two groups, irrespective of how we defined the rare variants. Furthermore, we did not observe an association of rare coding variants in SLC6A4 with rigid-compulsive traits scores in the cases. These results do not Support a significant role for rare coding variants in SLC6A4 in autism spectrum disorders, nor do they support a significant role for SLC6A4 in rigid-compulsive traits in these disorders.
C1 [Sakurai, Takeshi; Reichert, Jennifer; Hoffman, Ellen J.; Cai, Guiqing; Buxbaum, Joseph D.] Mt Sinai Sch Med, Dept Psychiat, New York, NY 10029 USA.
[Sakurai, Takeshi; Reichert, Jennifer; Hoffman, Ellen J.; Cai, Guiqing; Buxbaum, Joseph D.] Mt Sinai Sch Med, Lab Mol Neuropsychiat, Seaver Autism Res Ctr, New York, NY 10029 USA.
[Jones, Hywel B.; Faham, Malek] ParAllele Biosci Inc, San Francisco, CA USA.
RP Buxbaum, JD (reprint author), Mt Sinai Sch Med, Dept Psychiat, 1 Gustave L Levy Pl,Box 1668, New York, NY 10029 USA.
EM joseph.buxbaum@mssm.edu
FU New York State Spinal Cord Injur grant; Stanley Medical Research
Institute; Beatrice and Samuel A. Seaver Foundation; Milton & Miriam
Handler Foundation; Autism Speaks; National Institutes of Health
[MH-066673, NS-042165]
FX Grant sponsors: New York State Spinal Cord Injury grant, Stanley Medical
Research Institute, Beatrice and Samuel A. Seaver Foundation; Milton &
Miriam Handler Foundation; Autism Speaks; Grant sponsor: National
Institutes of Health; Grant numbers: MH-066673, NS-042165.
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NR 18
TC 12
Z9 12
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1939-3792
J9 AUTISM RES
JI Autism Res.
PD AUG
PY 2008
VL 1
IS 4
BP 251
EP 257
DI 10.1002/aur.30
PG 7
WC Behavioral Sciences; Psychology, Developmental
SC Behavioral Sciences; Psychology
GA 497CL
UT WOS:000270031900007
PM 19360675
ER
PT J
AU Swain, JE
Swain, JD
AF Swain, James E.
Swain, John D.
TI Creativity or mental illness: Possible errors of relational priming in
neural networks of the brain
SO BEHAVIORAL AND BRAIN SCIENCES
LA English
DT Editorial Material
AB If connectionist computational models explain the acquisition of complex cognitive skills, errors in such models would also help explain unusual brain activity such as in creativity - as well as in mental illness, including childhood onset problems with social behaviors in autism, the inability to maintain focus in attention deficit and hyperactivity disorder (ADHD), and the lack of motivation of depression disorders.
C1 [Swain, James E.] Yale Univ, Sch Med, Ctr Child Study, New Haven, CT 06520 USA.
[Swain, John D.] Northeastern Univ, Dept Phys, Boston, MA 02115 USA.
RP Swain, JE (reprint author), Yale Univ, Sch Med, Ctr Child Study, 333 Cedar St, New Haven, CT 06520 USA.
EM james.swain@yale.edu; John.swain@cern.ch
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NR 9
TC 1
Z9 1
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 0140-525X
J9 BEHAV BRAIN SCI
JI Behav. Brain Sci.
PD AUG
PY 2008
VL 31
IS 4
BP 398
EP +
DI 10.1017/S0140525X08004688
PG 8
WC Psychology, Biological; Behavioral Sciences; Neurosciences
SC Psychology; Behavioral Sciences; Neurosciences & Neurology
GA 370SP
UT WOS:000260783700023
ER
PT J
AU Bird, A
AF Bird, Adrian
TI The methyl-CpG-binding protein MeCP2 and neurological disease
SO BIOCHEMICAL SOCIETY TRANSACTIONS
LA English
DT Article
DE DNA methylation; MeCP2; methyl-CpG-binding protein; Rett syndrome; sin3a
ID DEPENDENT TRANSCRIPTIONAL REPRESSOR; RETT-SYNDROME; DNA METHYLATION;
MOUSE MODEL; HISTONE DEACETYLASE; CHROMOSOMAL-PROTEIN;
MENTAL-RETARDATION; BDNF TRANSCRIPTION; GENE-EXPRESSION; CHROMATIN
AB The methyl-CpG-binding protein MeCP2 was discovered over 15 years ago as part of a search for proteins that selectively bind methylated DNA. It is a nuclear protein that is largely chromatin-bound and has a strong preference for binding to methylated DNA sequences in vivo. Evidence from model systems shows that MeCP2 can recruit the Sin3a co-repressor complex to promoters leading to transcriptional repression, therefore suggesting that MeCP2 can interpret the DNA methylation signal to bring about gene silencing. Mutations in the human MECP2 gene cause the autism spectrum disorder Rett Syndrome. MeCP2 is most highly expressed in neurons, and mice lacking this protein show symptoms that strikingly parallel those of Rett patients. Surprisingly, these symptoms are efficiently reversed by delayed activation of a 'stopped' Mecp2 gene, raising hopes that human Rett syndrome may also be reversible. Future studies of MeCP2 promise to shed light upon brain function, neurological disease and the biology of DNA methylation.
C1 Univ Edinburgh, Wellcome Trust Ctr Cell Biol, Edinburgh EH9 3JR, Midlothian, Scotland.
RP Bird, A (reprint author), Univ Edinburgh, Wellcome Trust Ctr Cell Biol, Kings Bldg, Edinburgh EH9 3JR, Midlothian, Scotland.
EM a.bird@ed.ac.uk
FU MRC (medical Research Council); CRUK (Cancer Research UK); Rett Syndrome
Research Foundation; Rett Syndrome UK/Jeans for Genes
FX I am indebted to members of my laboratory, past and present, who have
helped to understand MBPs. Structural studies of MeCP2 have depended on
long-term collaborations with Professor Paul Barlow and Professor
Malcolm Walkinshaw at The University of Edinburgh. The Wellcome Trust
have funded much of our research, with significant contributions from
MRC (medical Research Council), CRUK (Cancer Research UK), the Rett
Syndrome Research Foundation and Rett Syndrome UK/Jeans for Genes.
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NR 92
TC 29
Z9 30
PU PORTLAND PRESS LTD
PI LONDON
PA THIRD FLOOR, EAGLE HOUSE, 16 PROCTER STREET, LONDON WC1V 6 NX, ENGLAND
SN 0300-5127
J9 BIOCHEM SOC T
JI Biochem. Soc. Trans.
PD AUG
PY 2008
VL 36
BP 575
EP 583
DI 10.1042/BST0360575
PN 4
PG 9
WC Biochemistry & Molecular Biology
SC Biochemistry & Molecular Biology
GA 338QI
UT WOS:000258522200003
PM 18631120
ER
PT J
AU Bello, KD
Goharpey, N
Crewther, SG
Crewther, DP
AF Bello, Katrina D.
Goharpey, Nahal
Crewther, Sheila G.
Crewther, David P.
TI A puzzle form of a non-verbal intelligence test gives significantly
higher performance measures in children with severe intellectual
disability
SO BMC PEDIATRICS
LA English
DT Article
ID COLORED-PROGRESSIVE-MATRICES; DOWN-SYNDROME; MENTAL-RETARDATION; AUTISM;
ATTENTION; INDIVIDUALS; RELIABILITY; PREVALENCE; ENGAGEMENT; PROFILE
AB Background: Assessment of 'potential intellectual ability' of children with severe intellectual disability (ID) is limited, as current tests designed for normal children do not maintain their interest. Thus a manual puzzle version of the Raven's Coloured Progressive Matrices (RCPM) was devised to appeal to the attentional and sensory preferences and language limitations of children with ID. It was hypothesized that performance on the book and manual puzzle forms would not differ for typically developing children but that children with ID would perform better on the puzzle form.
Methods: The first study assessed the validity of this puzzle form of the RCPM for 76 typically developing children in a test-retest crossover design, with a 3 week interval between tests. A second study tested performance and completion rate for the puzzle form compared to the book form in a sample of 164 children with ID.
Results: In the first study, no significant difference was found between performance on the puzzle and book forms in typically developing children, irrespective of the order of completion. The second study demonstrated a significantly higher performance and completion rate for the puzzle form compared to the book form in the ID population.
Conclusion: Similar performance on book and puzzle forms of the RCPM by typically developing children suggests that both forms measure the same construct. These findings suggest that the puzzle form does not require greater cognitive ability but demands sensory-motor attention and limits distraction in children with severe ID. Thus, we suggest the puzzle form of the RCPM is a more reliable measure of the non-verbal mentation of children with severe ID than the book form.
C1 [Bello, Katrina D.; Goharpey, Nahal; Crewther, David P.] Swinburne Univ Technol, Brain Sci Inst, Hawthorn, Vic 3122, Australia.
[Crewther, Sheila G.] La Trobe Univ, Sch Psychol Sci, Bundoora, Vic 3086, Australia.
RP Bello, KD (reprint author), Swinburne Univ Technol, Brain Sci Inst, Hawthorn, Vic 3122, Australia.
EM katrinabello@hotmail.com; goharpey@hotmail.com;
s.crewther@latrobe.edu.au; dcrewther@swin.edu.au
FU ARC [LP0346984]
FX This research was supported by an ARC Linkage grant LP0346984. We thank
Bella Irlicht, Dr. Carl Parsons, as well as the staff, parents, students
of Port Phillip Specialist School, Bulleen Heights School, and St. Pius
X Primary School, along with Dr. Sue Cotton.
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NR 40
TC 3
Z9 3
PU BIOMED CENTRAL LTD
PI LONDON
PA 236 GRAYS INN RD, FLOOR 6, LONDON WC1X 8HL, ENGLAND
SN 1471-2431
J9 BMC PEDIATR
JI BMC Pediatr.
PD AUG 1
PY 2008
VL 8
AR 30
DI 10.1186/1471-2431-8-30
PG 8
WC Pediatrics
SC Pediatrics
GA 420OW
UT WOS:000264300100001
PM 18671882
ER
PT J
AU Rojas, DC
Maharajh, K
Teale, P
Rogers, SJ
AF Rojas, Donald C.
Maharajh, Keeran
Teale, Peter
Rogers, Sally J.
TI Reduced neural synchronization of gamma-band MEG oscillations in
first-degree relatives of children with autism
SO BMC PSYCHIATRY
LA English
DT Article
ID MAGNETIC-FIELDS; CEREBRAL LATERALIZATION; LINKAGE-DISEQUILIBRIUM;
NETWORK OSCILLATIONS; INTERNEURON NETWORKS; AUDITORY M100; IN-VITRO;
SCHIZOPHRENIA; BRAIN; RESPONSES
AB Background: Gamma-band oscillations recorded from human electrophysiological recordings, which may be associated with perceptual binding and neuronal connectivity, have been shown to be altered in people with autism. Transient auditory gamma-band responses, however, have not yet been investigated in autism or in the first-degree relatives of persons with the autism.
Methods: We measured transient evoked and induced magnetic gamma-band power and intertrial phase-locking consistency in the magnetoencephalographic recordings of 16 parents of children with autism, 11 adults with autism and 16 control participants. Source space projection was used to separate left and right hemisphere transient gamma-band measures of power and phase-locking.
Results: Induced gamma-power at 40 Hz was significantly higher in the parent and autism groups than in controls, while evoked gamma-band power was reduced compared to controls. The phaselocking factor, a measure of phase consistency of neuronal responses with external stimuli, was significantly lower in the subjects with autism and the autism parent group, potentially explaining the difference between the evoked and induced power results.
Conclusion: These findings, especially in first degree relatives, suggest that gamma-band phase consistency and changes in induced versus induced power may be potentially useful endophenotypes for autism, particularly given emerging molecular mechanisms concerning the generation of gamma-band signals.
C1 [Rojas, Donald C.; Maharajh, Keeran; Teale, Peter] Univ Colorado, Hlth Sci Ctr, Denver, CO USA.
[Rogers, Sally J.] Univ Calif Davis, MIND Inst, Sacramento, CA 95817 USA.
RP Rojas, DC (reprint author), Univ Colorado, Hlth Sci Ctr, 4200 E 9th Ave, Denver, CO USA.
EM don.rojas@uchsc.edu; keeran.maharajh@uchsc.edu; peter.teale@uchsc.edu;
sally.rogers@ucdmc.ucdavis.edu
RI Rojas, Don/F-4296-2012
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NR 57
TC 40
Z9 42
PU BIOMED CENTRAL LTD
PI LONDON
PA 236 GRAYS INN RD, FLOOR 6, LONDON WC1X 8HL, ENGLAND
SN 1471-244X
J9 BMC PSYCHIATRY
JI BMC Psychiatry
PD AUG 1
PY 2008
VL 8
AR 66
DI 10.1186/1471-244X-8-66
PG 9
WC Psychiatry
SC Psychiatry
GA 342OW
UT WOS:000258794400001
PM 18673566
ER
PT J
AU Diehl, JJ
Bennetto, L
Watson, D
Gunlogson, C
McDonough, J
AF Diehl, Joshua J.
Bennetto, Loisa
Watson, Duane
Gunlogson, Christine
McDonough, Joyce
TI Resolving ambiguity: A psycholinguistic approach to understanding
prosody processing in high-functioning autism
SO BRAIN AND LANGUAGE
LA English
DT Article
DE autism; prosody; language; comprehension; intonation; syntax;
pragmatics; high-functioning
ID SPECTRUM DISORDERS; CORPUS-CALLOSUM; ASPERGER-SYNDROME; YOUNG-CHILDREN;
LANGUAGE; SPEECH; ADULTS; COMPREHENSION; CONSTRAINTS; CONTEXT
AB Individuals with autism exhibit significant impairments in prosody production, yet there is a paucity of research on prosody comprehension in this population. The current study adapted a psycholinguistic paradigm to examine whether individuals with autism are able to use prosody to resolve syntactically ambiguous sentences. Participants were 21 adolescents with high-functioning autism (HFA), and 22 typically developing controls matched on age, 10, receptive language, and gender. The HFA group was significantly less likely to use prosody to disambiguate syntax, but scored comparably to controls when syntax alone or both prosody and syntax indicated the correct response. These findings indicate that adolescents with HFA have difficulty using prosody to disambiguate syntax in comparison to typically developing controls, even when matched on chronological age, IQ, and receptive language. The implications of these findings for how individuals with autism process language are discussed. (C) 2008 Elsevier Inc. All rights reserved.
C1 [Diehl, Joshua J.] Yale Univ, Sch Med, Ctr Child Study, New Haven, CT 06510 USA.
[Diehl, Joshua J.; Bennetto, Loisa] Univ Rochester, Dept Clin & Social Sci Psychol, Rochester, NY USA.
[Watson, Duane] Univ Rochester, Dept Brain & Cognit Sci, Rochester, NY USA.
[Watson, Duane] Univ Illinois, Dept Psychol, Champaign, IL USA.
[Gunlogson, Christine; McDonough, Joyce] Univ Rochester, Dept Linguist, Rochester, NY USA.
RP Diehl, JJ (reprint author), Yale Univ, Sch Med, Ctr Child Study, 230 S Frontage Rd,POB 207900, New Haven, CT 06510 USA.
EM joshua.diehl@yale.edu
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NR 61
TC 14
Z9 14
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0093-934X
J9 BRAIN LANG
JI Brain Lang.
PD AUG
PY 2008
VL 106
IS 2
BP 144
EP 152
DI 10.1016/j.bandl.2008.04.002
PG 9
WC Audiology & Speech-Language Pathology; Linguistics; Neurosciences;
Psychology, Experimental
SC Audiology & Speech-Language Pathology; Linguistics; Neurosciences &
Neurology; Psychology
GA 337MC
UT WOS:000258439200006
PM 18502497
ER
PT J
AU Martin, LA
Ashwood, P
Braunschweig, D
Cabanlit, M
Van de Water, J
Amaral, DG
AF Martin, Loren A.
Ashwood, Paul
Braunschweig, Daniel
Cabanlit, Maricel
Van de Water, Judy
Amaral, David G.
TI Stereotypies and hyperactivity in rhesus monkeys exposed to IgG from
mothers of children with autism
SO BRAIN BEHAVIOR AND IMMUNITY
LA English
DT Article
DE repetitive; primate; macaque; macaca mulatta; activity; Asperger
syndrome
ID ARTHROGRYPOSIS MULTIPLEX CONGENITA; NEONATAL AMYGDALA LESIONS; MATERNAL
MYASTHENIA; ANTIBODIES; AUTOANTIBODIES; BEHAVIOR; DISORDERS; ATTENTION;
DISEASE; ANTIGEN
AB Autism together with Asperger syndrome and pervasive developmental disorder not otherwise specified form a spectrum of conditions (autism spectrum disorders or ASD) that is characterized by disturbances in social behavior, impaired communication and the presence of stereotyped behaviors or circumscribed interests. Recent estimates indicate a prevalence of ASD of 1 per 150 (Kuehn, 2007). The cause(s) of most cases of ASD are unknown but there is an emerging consensus that ASD have multiple etiologies. One proposed cause of ASD is exposure of the fetal brain to maternal autoantibodies during pregnancy [Dalton, P., Deacon, R., Blamire, A., Pike, M., McKinlay, I., Stein, J., Styles, P., Vincent, A., 2003. Maternal neuronal antibodies associated with autism and a language disorder. Ann. Neurol. 53, 533-537]. To provide evidence for this hypothesis, four rhesus monkeys were exposed prenatally to human IgG collected from mothers of multiple children diagnosed with ASD. Four control rhesus monkeys were exposed to human IgG collected from mothers of multiple typically developing children. Five additional monkeys were untreated controls. Monkeys were observed in a variety of behavioral paradigms involving unique social situations. Behaviors were scored by trained observers and overall activity was monitored with actimeters. Rhesus monkeys gestationally exposed to IgG class antibodies from mothers of children with ASD consistently demonstrated increased whole-body stereotypies across multiple testing paradigms. These monkeys were also hyperactive compared to controls. Treatment with IgG purified from mothers of typically developing children did not induce stereotypical or hyperactive behaviors. These findings support the potential for an autoimmune etiology in a subgroup of patients with neurodevelopmental disorders. This research raises the prospect of prenatal evaluation for neurodevelopmental risk factors and the potential for preventative therapeutics. (c) 2008 Elsevier Inc. All rights reserved.
C1 [Martin, Loren A.; Ashwood, Paul; Amaral, David G.] Univ Calif Davis, MIND Inst, Sacramento, CA 95817 USA.
[Martin, Loren A.; Amaral, David G.] Univ Calif Davis, Calif Natl Primate Res Ctr, Ctr Neurosci, Dept Psychiat & Behav Sci, Sacramento, CA 95817 USA.
[Ashwood, Paul] Univ Calif Davis, Dept Med Microbiol & Immunol, Sacramento, CA 95817 USA.
[Ashwood, Paul; Van de Water, Judy; Amaral, David G.] Univ Calif Davis, NIEHS Ctr Childrens Environm Hlth, Sacramento, CA 95817 USA.
[Braunschweig, Daniel; Cabanlit, Maricel; Van de Water, Judy] Univ Calif Davis, Div Rheumatol Allergy & Clin Immunol, Sacramento, CA 95817 USA.
RP Amaral, DG (reprint author), Univ Calif Davis, MIND Inst, 2825 50th St, Sacramento, CA 95817 USA.
EM dgamaral@ucdavis.edu
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NR 50
TC 83
Z9 86
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0889-1591
J9 BRAIN BEHAV IMMUN
JI Brain Behav. Immun.
PD AUG
PY 2008
VL 22
IS 6
BP 806
EP 816
DI 10.1016/j.bbi.2007.12.007
PG 11
WC Immunology; Neurosciences
SC Immunology; Neurosciences & Neurology
GA 325RL
UT WOS:000257605900003
PM 18262386
ER
PT J
AU Ming, X
Gordon, E
Kang, N
Wagner, GC
AF Ming, Xue
Gordon, Emily
Kang, Ning
Wagner, George C.
TI Use of clonidine in children with autism spectrum disorders
SO BRAIN & DEVELOPMENT
LA English
DT Article
DE autism spectrum disorders; clonidine; sleep disorders; attention deficit
hyperactivity disorders; aggression; mood instability
ID DEFICIT HYPERACTIVITY DISORDER; ATTENTION-DEFICIT/HYPERACTIVITY
DISORDER; PERVASIVE DEVELOPMENTAL DISORDERS; SLEEP PROBLEMS;
DOUBLE-BLIND; INTELLECTUAL DISABILITIES; CONDUCT DISORDER; CONTROLLED
TRIAL; PLACEBO; SYMPTOMS
AB Children with autism spectrum disorders (ASD) often exhibit sleep and behavioral disorders. Treatment of sleep disorders can be difficult in these children. Clonidine, an alpha 2-adrenergic receptor agonist, has been shown to be effective in reducing impulsivity, inattention, and hyperactivity, as well as in serving as a sedative for medial procedures. An open labeled retrospective study of clonidine in treatment of insomnia, and/or hyperactivity, inattention, mood disorder, and aggressive behaviors was conducted using parent reports of sleep initiation and maintenance, as well as behaviors prior and during clonidine treatment. Clonidine was effective in reducing sleep initiation latency and night awakening, to a less degree in improving attention deficits hyperactivity, mood instability and aggressiveness in this cohort of 19 children with ASD. The side,effects were largely tolerable. Further evaluation with placebo-controlled double-blind clinical trial of clonidine use in ASD will provide more insight into the clinical efficacy and safety of the medicine in ASD. (C) 2007 Elsevier B.V. All rights reserved.
C1 [Ming, Xue] Univ Med & Dent New Jersey, New Jersey Med Sch, Dept Neurosci & Pediat, Newark, NJ 07103 USA.
[Gordon, Emily] Univ Med & Dent New Jersey, New Jersey Med Sch, Newark, NJ 07103 USA.
[Kang, Ning] Univ Calif Los Angeles, Sch Publ Hlth, Dept Biostat, Los Angeles, CA 90024 USA.
[Wagner, George C.] Rutgers State Univ, Dept Psychol, New Brunswick, NJ 08903 USA.
RP Ming, X (reprint author), Univ Med & Dent New Jersey, New Jersey Med Sch, Dept Neurosci & Pediat, 90 Bergen St,Doctors Off Ctr 8100, Newark, NJ 07103 USA.
EM mingxu@umdnj.edu
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NR 47
TC 26
Z9 27
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0387-7604
J9 BRAIN DEV-JPN
JI Brain Dev.
PD AUG
PY 2008
VL 30
IS 7
BP 454
EP 460
DI 10.1016/j.braindev.2007.12.007
PG 7
WC Clinical Neurology
SC Neurosciences & Neurology
GA 329CB
UT WOS:000257843400003
PM 18280681
ER
PT J
AU Zannolli, R
Buoni, S
de Santi, M
Miracco, C
Vonella, G
Tassini, M
Vivi, A
Viviano, M
Rossi, T
Orsi, A
Scarinci, R
D'Ambrosio, A
Livi, W
Volterrani, L
Fois, A
Willems, P
Hayek, J
AF Zannolli, Raffaella
Buoni, Sabrina
de Santi, Margherita
Miracco, Clelia
Vonella, Giuseppina
Tassini, Maria
Vivi, Antonio
Viviano, Massimo
Rossi, Tiziana
Orsi, Alessandra
Scarinci, Renato
D'Ambrosio, Alfonso
Livi, Walter
Volterrani, Luca
Fois, Alberto
Willems, Patrick
Hayek, Joseph
TI New neurocutaneous syndrome with defect in cell trafficking and
melanosome pathway: The future challenge
SO BRAIN & DEVELOPMENT
LA English
DT Article
DE cell trafficking; intracellular vesicular compartment; melanosomes;
mental delay; neurocutaneous syndrome
ID ECTODERMAL DYSPLASIA SYNDROME; INBORN-ERRORS; MENTAL DELAY;
SPECTROSCOPY; OSTEOPENIA; METABOLISM; PTOSIS; CHILD
AB Objective: Case study of a CNS impairment lacking in presumptive cause; case presents with a clinical phenotype encompassing multiple differently expressed and combined symptoms, as well as a subtle skin defect. Materials and methods: A 6-year-old male with apparently isolated mental delay, speech delay, attention deficit/hyperactivity disorder, epilepsy, and subtle and insignificant skin dyschromias. The patient underwent a systematic evaluation, including clinical history; medical, neurological and ophthalmologic examinations. Skin, teeth, nails, hair and sudation were examined for defects, Routine laboratory tests for blood, urine, were performed. The proband had thyroid function tests, electrocardiography, genitourinary system and abdominal examinations. Special examinations pertaining to mental performance, biochemistry, chromosome studies, imaging and electrodiagnostic studies, and skin biopsy were also performed. Results: Investigators ruled out genetic syndromes, congenital infections, fetal deprivation, perinatal insults, intrauterine exposure to drug abuse, and postnatal events such as CNS infections as possible common causes of brain impairment. Being all further test negative, the patient exhibited an ultrastructural defect of the skin, identical to that previously described [Buoni S, Zannolli R, de Santi MM, Macucci F, Hayek J, Orsi A et al. Neurocutaneous syndrome with mental delay, autism, blockage in intracellular vesicular trafficking and melanosome defects. Eur J Neurol 2006;13:842-51], suggesting that some cell compartments, such as rough endoplasmic reticulum, lysosomes, Golgi apparatus, and the vesicular zone (racket) of Birbeck granules, sharing similar components, can be altered, resulting in a common defect in cell trafficking, associated to melanosome defects. Conclusions: This new devasting, ultrastructural phenotype accompanied by apparently unspecific and mixed neurological symptoms should represent a future challenge to finally discover the pathogenesis of many childhood CNS symptoms, that currently seem to lack any apparent cause. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Zannolli, Raffaella; Scarinci, Renato; D'Ambrosio, Alfonso; Fois, Alberto] Univ Siena, Policlin Le Scotte, Azienda Osped Univ Senese, Sect Pediat Neurol,Dept Pediat, I-53100 Siena, Italy.
[Zannolli, Raffaella; Scarinci, Renato; D'Ambrosio, Alfonso; Fois, Alberto] Univ Siena, Policlin Le Scotte, Azienda Osped Univ Senese, Pediat Neuropsychiat Unit, I-53100 Siena, Italy.
[de Santi, Margherita; Miracco, Clelia; Rossi, Tiziana] Univ Siena, Policlin Le Scotte, Sect Pathol Anat & Istol, Dept Human Pathol & Oncol, I-53100 Siena, Italy.
[Viviano, Massimo] Univ Siena, Policlin Le Scotte, Dept Odontostomatol Sci, I-53100 Siena, Italy.
[Livi, Walter] Univ Siena, Policlin Le Scotte, Dept ENT, I-53100 Siena, Italy.
[Volterrani, Luca] Univ Siena, Policlin Le Scotte, Sect Radiol & Radiotherapy, Dept Human Pathol & Oncol, I-53100 Siena, Italy.
[Willems, Patrick] GENDIA Network, B-2000 Antwerp, Belgium.
[Tassini, Maria; Vivi, Antonio] Univ Siena, Policlin Le Scotte, Azienda Osped Univ Senese, Magnet Resonance Unit,Dept Neurosci, I-53100 Siena, Italy.
RP Zannolli, R (reprint author), Univ Siena, Policlin Le Scotte, Azienda Osped Univ Senese, Sect Pediat Neurol,Dept Pediat, Viale Bracci, I-53100 Siena, Italy.
EM zannolli@unisi.it
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NR 19
TC 1
Z9 1
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0387-7604
J9 BRAIN DEV-JPN
JI Brain Dev.
PD AUG
PY 2008
VL 30
IS 7
BP 461
EP 468
DI 10.1016/j.braindev.2007.12.008
PG 8
WC Clinical Neurology
SC Neurosciences & Neurology
GA 329CB
UT WOS:000257843400004
PM 18226865
ER
PT J
AU Tavares, P
Lawrence, AD
Barnard, PJ
AF Tavares, Paula
Lawrence, Andrew D.
Barnard, Philip J.
TI Paying attention to social meaning: An fMRI study
SO CEREBRAL CORTEX
LA English
DT Article
DE amygdala; animacy; autism; dorsomedial prefrontal cortex; social
cognition
ID FUNCTIONAL NEUROANATOMY; VISUAL-CORTEX; MIRROR SYSTEM; HUMAN BRAIN;
MEMORY; AUTISM; PERCEPTION; BEHAVIOR; MOTION; ATTRIBUTION
AB Animations of simple geometric shapes are readily interpreted as animate agents engaged in meaningful social interactions. Such animations have been shown to activate brain regions implicated in the detection of animate motion, in understanding the intentions of others as well as areas commonly linked to the processing of social and emotional information. However, attribution of animacy does not occur under all circumstances and the precise conditions under which specific regions are activated remains unclear. In a functional magnetic resonance imaging study we manipulated viewers' perspective to assess the part played by selective attention. Participants were cued to attend either to spatial properties of the movements or to the kind of social behavior it could represent. Activations that occurred to the initial cue, while observing the animations themselves and while responding to a postpresentation probe, were analyzed separately. Results showed that activity in the social brain network was strongly influenced by selective attention, and that remarkably similar activations were seen during film viewing and in response to probe questions. Our use of stimuli supporting rich and diverse social narratives likely enhanced the influence of top-down processes on neural activity in the social brain.
C1 [Tavares, Paula] Univ Lisbon, Fac Ciencias, Inst Biofis & Engn Biomed, P-1749016 Lisbon, Portugal.
[Lawrence, Andrew D.; Barnard, Philip J.] MRC Cognit & Brain Sci Unit, Cambridge CB2 7EF, England.
[Lawrence, Andrew D.] Cardiff Univ, Sch Psychol, Wales Inst Cognit Neurosci, Cardiff CF10 3AT, S Glam, Wales.
RP Tavares, P (reprint author), Univ Lisbon, Fac Ciencias, Inst Biofis & Engn Biomed, Campo Grande, P-1749016 Lisbon, Portugal.
EM ptavares@fc.ul.pt
RI Lawrence, Andrew/A-1673-2010
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NR 73
TC 29
Z9 29
PU OXFORD UNIV PRESS INC
PI CARY
PA JOURNALS DEPT, 2001 EVANS RD, CARY, NC 27513 USA
SN 1047-3211
J9 CEREB CORTEX
JI Cereb. Cortex
PD AUG
PY 2008
VL 18
IS 8
BP 1876
EP 1885
DI 10.1093/cercor/bhm212
PG 10
WC Neurosciences
SC Neurosciences & Neurology
GA 328GV
UT WOS:000257787300013
PM 18065722
ER
PT J
AU Sadoni, N
Targosz, BS
Englmann, A
Fesser, S
Koch, J
Schindelhauer, D
Zink, D
AF Sadoni, Nicolas
Targosz, Bianca-Sabrina
Englmann, Andreas
Fesser, Stephanie
Koch, Jeannette
Schindelhauer, Dirk
Zink, Daniele
TI Transcription-dependent spatial arrangements of CFTR and conserved
adjacent loci are not conserved in human and murine nuclei
SO CHROMOSOMA
LA English
DT Article
ID INTERPHASE CHROMOSOME TERRITORIES; CYSTIC-FIBROSIS; CELL-NUCLEI;
DNA-REPLICATION; GENE DENSITY; GLOBIN GENES; CHROMATIN; MOUSE;
ORGANIZATION; AUTISM
AB The human genes CFTR, ASZ1/GASZ, and CTTNBP2/CORTBP2 map to adjacent loci on chromosome 7q31 and display characteristic patterns of nuclear positioning, which strictly correlate with the state of activity. To address the evolutionary conservation of gene positioning, we investigated transcriptional activity and nuclear positioning of the highly conserved murine orthologs and of additional murine genes mapping to the region of conserved synteny on mouse chromosome 6. The results showed that all murine loci investigated constitutively localized in the nuclear interior irrespective of their functional state. Silenced loci did not display preferential association with the nuclear periphery or with chromocenters, respectively, and no differential positioning with respect to the chromosome 6 territory could be observed. This positional behavior of the murine loci was in striking contrast to the positioning of the human orthologs, and the results show that the transcription-dependent positioning of CFTR and adjacent loci has not been conserved. The findings reveal that the nuclear organization of conserved chromosomal regions can change rapidly during evolution and is not always as highly conserved as other features of chromosome organization. Furthermore, the results suggest that the way how nuclear positioning contributes to the regulation of conserved loci can be different in different vertebrate species.
C1 [Sadoni, Nicolas; Targosz, Bianca-Sabrina; Englmann, Andreas; Fesser, Stephanie; Koch, Jeannette; Zink, Daniele] Univ Munich, Dept Biol 2, Bioctr, D-82152 Planegg Martinsried, Germany.
[Schindelhauer, Dirk] Tech Univ Munich, Life Sci Ctr Weihenstephan, D-85354 Freising Weihenstephan, Germany.
RP Zink, D (reprint author), Inst Bioengn & Nanotechnol, 31 Biopolis Way,Nanos 04-01, Singapore 138669, Singapore.
EM dzink@ibn.a-star.edu.sg
RI Zink, Daniele/A-8181-2015
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NR 67
TC 13
Z9 13
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0009-5915
J9 CHROMOSOMA
JI Chromosoma
PD AUG
PY 2008
VL 117
IS 4
BP 381
EP 397
DI 10.1007/s00412-008-0157-5
PG 17
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 322IU
UT WOS:000257369200008
PM 18408947
ER
PT J
AU Qiao, Y
Liu, X
Harvard, C
Hildebrand, MJ
Rajcan-Separovic, E
Holden, JJA
Lewis, MES
AF Qiao, Y.
Liu, X.
Harvard, C.
Hildebrand, M. J.
Rajcan-Separovic, E.
Holden, J. J. A.
Lewis, M. E. S.
TI Autism-associated familial microdeletion of Xp11.22
SO CLINICAL GENETICS
LA English
DT Article
DE array comparative genomic hybridization; autism spectrum disorder; cleft
lip/palate; microdeletion of Xp11.22; X-linked mental retardation
ID LINKED MENTAL-RETARDATION; CLEFT LIP/CLEFT PALATE; PERVASIVE
DEVELOPMENTAL DISORDERS; CHROMOSOMAL REARRANGEMENTS; PHF8 GENE;
MUTATIONS; SPECTRUM; IDENTIFICATION; INACTIVATION; DEFICITS
AB We describe two brothers with autistic disorder, intellectual disability (ID) and cleft lip/palate with a microdeletion of Xp11.22 detected through screening individuals with autism spectrum disorders (ASDs) for microdeletions and duplications using 1-Mb resolution array comparative genomic hybridization. The deletion was confirmed by fluorescence in situ hybridization/real-time quantitative polymerase chain reaction (RT-qPCR) and shown to be inherited from their unaffected mother who had skewed (100%) X inactivation of the aberrant chromosome. RT-qPCR characterization of the del(X)(p11.22) region (similar to 53,887,000-54,359,000 bp) revealed complete deletion of the plant homeodomain finger protein 8 (PHF8) gene as well as deletions of the FAM120C and WNK lysine-deficient protein kinase 3 (WNK3) genes, for which a definitive phenotype has not been previously characterized. Xp11.2 is a gene-rich region within the critical linkage interval for several neurodevelopmental disorders. Rare interstitial microdeletions of Xp11.22 have been recognized with ID, craniofacial dysmorphism and/or cleft lip/palate and truncating mutations of the PHF8 gene within this region. Despite evidence implicating genes within Xp11.22 with language and cognitive development that could contribute to an ASD phenotype, their involvement with autism has not been systematically evaluated. Population screening of 481 (319 males/81 females) and 282 X chromosomes (90 males/96 females) in respective ASD and control cohorts did not identify additional subjects carrying this deletion. Our findings show that in addition to point mutations, a complete deletion of the PHF8 gene is associated with the X-linked mental retardation Siderius-Hamel syndrome (OMIM 300263) and further suggest that the larger size of the Xp11.22 deletion including genes FAM120C and WNK3 may be involved in the pathogenesis of autism.
C1 [Lewis, M. E. S.] Univ British Columbia, Child & Family Res Inst, BC Childrens & Womens Hlth Center, Dept Med Genet, Vancouver, BC V6H 3N1, Canada.
[Qiao, Y.; Harvard, C.; Rajcan-Separovic, E.] Univ British Columbia, Child & Family Res Inst, Dept Pathol, Vancouver, BC V6H 3N1, Canada.
[Liu, X.; Holden, J. J. A.] Queens Univ, Dept Psychiat, Kingston, ON K7L 3N6, Canada.
[Holden, J. J. A.] Queens Univ, Dept Physiol, Kingston, ON K7L 3N6, Canada.
[Holden, J. J. A.] Autism Res Program, Kingston, ON, Canada.
RP Lewis, MES (reprint author), Univ British Columbia, Child & Family Res Inst, BC Childrens & Womens Hlth Center, Dept Med Genet, C234-4500 Oak St, Vancouver, BC V6H 3N1, Canada.
EM sume@interchange.ubc.ca
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NR 31
TC 39
Z9 40
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0009-9163
J9 CLIN GENET
JI Clin. Genet.
PD AUG
PY 2008
VL 74
IS 2
BP 134
EP 144
DI 10.1111/j.1399-0004.2008.01028.x
PG 11
WC Genetics & Heredity
SC Genetics & Heredity
GA 323VJ
UT WOS:000257476200005
PM 18498374
ER
PT J
AU Galbraith, GC
AF Galbraith, Gary Cloyd
TI Deficient brainstem encoding in autism
SO CLINICAL NEUROPHYSIOLOGY
LA English
DT Editorial Material
ID FREQUENCY-FOLLOWING RESPONSE; AUDITORY-EVOKED-POTENTIALS; SELECTIVE
ATTENTION; LEARNING-PROBLEMS; TIMING DEFICITS; SPEECH; CORTEX; PITCH;
EXPERIENCE; CHILDREN
C1 Univ Calif Los Angeles, David Geffen Sch Med, Mental Retardat Res Ctr, Dept Psychiat & Biobehav Sci, Los Angeles, CA 90024 USA.
RP Galbraith, GC (reprint author), Univ Calif Los Angeles, David Geffen Sch Med, Mental Retardat Res Ctr, Dept Psychiat & Biobehav Sci, Los Angeles, CA 90024 USA.
EM garyg@ucla.edu
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NR 48
TC 7
Z9 7
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 1388-2457
J9 CLIN NEUROPHYSIOL
JI Clin. Neurophysiol.
PD AUG
PY 2008
VL 119
IS 8
BP 1697
EP 1700
DI 10.1016/j.clinph.2008.04.012
PG 4
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 331PD
UT WOS:000258023300001
PM 18562247
ER
PT J
AU Russo, NM
Skoe, E
Trommer, B
Nicol, T
Zecker, S
Bradlow, A
Kraus, N
AF Russo, N. M.
Skoe, E.
Trommer, B.
Nicol, T.
Zecker, S.
Bradlow, A.
Kraus, N.
TI Deficient brainstem encoding of pitch in children with Autism Spectrum
Disorders
SO CLINICAL NEUROPHYSIOLOGY
LA English
DT Article
DE auditory brainstem; autism; pitch tracking; prosody
ID FREQUENCY-FOLLOWING RESPONSE; DIAGNOSTIC OBSERVATION SCHEDULE;
ASPERGER-SYNDROME; INFERIOR COLLICULUS; AFFERENT PATTERNS; CHILDHOOD
AUTISM; EVOKED-RESPONSES; SPEECH SOUNDS; HEARING-LOSS; LANGUAGE
AB Objective: Deficient prosody is a hallmark of the pragmatic (socially contextualized) language impairment in Autism Spectrum Disorders (ASD). Prosody communicates emotion and intention and is conveyed through acoustic cues such as pitch contour. Thus, the objective of this study was to examine the subcortical representations of prosodic speech in children with ASD.
Methods: Using passively evoked brainstem responses to speech syllables with descending and ascending pitch contours, we examined sensory encoding of pitch in children with ASD who had normal intelligence and hearing and were age-matched with typically developing (TD) control children.
Results: We found that some children on the autism spectrum show deficient pitch tracking (evidenced by increased Frequency and Slope Errors and reduced phase locking) compared with TD children.
Conclusions: This is the first demonstration of subcortical involvement in prosody encoding deficits in this population of children.
Significance: Our findings may have implications for diagnostic and remediation strategies in a subset of children with ASD and open up an avenue for future investigations. Published by Elsevier Ireland Ltd. on behalf of International Federation of Clinical Neurophysiology.
C1 [Russo, N. M.; Skoe, E.; Nicol, T.; Zecker, S.; Kraus, N.] Northwestern Univ, Roxelyn & Richard Pepper Dept Commun Sci, Evanston, IL 60208 USA.
[Russo, N. M.; Trommer, B.; Bradlow, A.; Kraus, N.] Northwestern Univ, Interdept Neurosci Program, Evanston, IL 60208 USA.
[Trommer, B.] Northwestern Univ, Feinberg Sch Med, Dept Pediat, Evanston, IL 60208 USA.
[Trommer, B.] Northwestern Univ, Feinberg Sch Med, Dept Neurol, Evanston, IL 60208 USA.
[Trommer, B.] Evanston NW Healthcare, Evanston, IL USA.
[Bradlow, A.] Northwestern Univ, Dept Linguist, Evanston, IL 60208 USA.
[Kraus, N.] Northwestern Univ, Dept Neurobiol & Physiol, Evanston, IL 60208 USA.
[Kraus, N.] Northwestern Univ, Dept Otolaryngol, Evanston, IL 60208 USA.
RP Russo, NM (reprint author), Northwestern Univ, Roxelyn & Richard Pepper Dept Commun Sci, 2240 Campus Dr, Evanston, IL 60208 USA.
EM nicole_russo@rush.edu; nkraus@northwestern.edu
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NR 68
TC 54
Z9 56
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 1388-2457
J9 CLIN NEUROPHYSIOL
JI Clin. Neurophysiol.
PD AUG
PY 2008
VL 119
IS 8
BP 1720
EP 1731
DI 10.1016/j.clinph.2008.01.108
PG 12
WC Clinical Neurology; Neurosciences
SC Neurosciences & Neurology
GA 331PD
UT WOS:000258023300005
PM 18558508
ER
PT J
AU Senju, A
Csibra, G
Johnson, MH
AF Senju, Atsushi
Csibra, Gergely
Johnson, Mark H.
TI Understanding the referential nature of looking: Infants' preference for
object-directed gaze
SO COGNITION
LA English
DT Article
DE eye gaze; face; infant; looking time; social cognition
ID VISUAL-ATTENTION; NEURAL BASIS; EYE CONTACT; PERCEPTION; SHIFTS;
ATTRIBUTION; HYPOTHESIS; AUTISM
AB In four experiments, we investigated whether 9-month-old infants are sensitive to the relationship between gaze direction and object location and whether this sensitivity depends on the presence of communicative cues like eye contact. Infants observed a face, which repeatedly shifted its eyes either toward, or away from, unpredictably appearing objects. We found that they looked longer at the face when the gaze shifts were congruent with the location of the object. A second experiment ruled out that this effect was simply due to spatial congruency, while a third and a fourth experiment revealed that a preceding period of eye contact is required to elicit the gaze-object congruency effect. These results indicate that infants at this age can encode eye direction in referential terms in the presence of communication cues and are biased to attend to scenes with object-directed gaze. (c) 2008 Elsevier B.V. All rights reserved.
C1 [Senju, Atsushi; Csibra, Gergely; Johnson, Mark H.] Univ London, Birkbeck Coll, Sch Psychol, Ctr Brain & Cognit Dev, London WC1E 7HX, England.
RP Senju, A (reprint author), Univ London, Birkbeck Coll, Sch Psychol, Ctr Brain & Cognit Dev, Malet St, London WC1E 7HX, England.
EM a.senju@bbk.ac.uk
RI Senju, Atsushi/C-4097-2008; Csibra, Gergely/C-4345-2008
OI Senju, Atsushi/0000-0002-8081-7170; Csibra, Gergely/0000-0002-7044-3056
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NR 35
TC 37
Z9 37
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0010-0277
J9 COGNITION
JI Cognition
PD AUG
PY 2008
VL 108
IS 2
BP 303
EP 319
DI 10.1016/j.cognition.2008.02.009
PG 17
WC Psychology, Experimental
SC Psychology
GA 330ZQ
UT WOS:000257981900001
PM 18371943
ER
PT J
AU Passerino, LM
Santarosa, LAC
AF Passerino, Liliana M.
Santarosa, Lucila A. Costi
TI Autism and digital learning environments: Processes of interaction and
mediation
SO COMPUTERS & EDUCATION
LA English
DT Article
DE autism; digital learning environments; social interaction; mediation
process
AB Using a socio-historical perspective to explain social interaction and taking advantage of information and communication technologies (ICTs) currently available for creating digital learning environments (DLEs), this paper seeks to redress the absence of empirical data concerning technology-aided social interaction between autistic individuals. In terms of social interaction, autism can be likened to the negative of a photograph and it is currently one of the most disturbing and challenging syndromes. This study puts forward another viewpoint on the processes of interaction and mediation established in digital environments. Based on a 3-year multi-case study, which observed groups of autistic individuals in DLE-aided interaction activities, it identifies relevant technological categories that have an important contribution to make in the areas involved (Psychology, Education and Computer Science) in the development of a digital learning environment that takes the results achieved into account. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Passerino, Liliana M.] UFRGs, NIEE, PGIE, Porto Alegre, RS, Brazil.
[Santarosa, Lucila A. Costi] UFRGs, NIEE, Res Ctr Special Educ Needs & Comp Educ, Porto Alegre, RS, Brazil.
RP Passerino, LM (reprint author), UFRGs, NIEE, PGIE, Porto Alegre, RS, Brazil.
EM liliana@cinted.ufrgs.br; lucila.santarosa@ufrgs.br
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PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0360-1315
J9 COMPUT EDUC
JI Comput. Educ.
PD AUG
PY 2008
VL 51
IS 1
BP 385
EP 402
DI 10.1016/j.compedu.2007.05.015
PG 18
WC Computer Science, Interdisciplinary Applications; Education &
Educational Research
SC Computer Science; Education & Educational Research
GA 318PG
UT WOS:000257103500026
ER
PT J
AU Pexman, PM
AF Pexman, Penny M.
TI It's fascinating research - The cognition of verbal irony
SO CURRENT DIRECTIONS IN PSYCHOLOGICAL SCIENCE
LA English
DT Article
DE verbal irony; sarcasm; figurative language; parallel constraint
satisfaction; communicative intent; humor
ID DEVELOPING-CHILDREN; COMPREHENSION; AUTISM; MIND
AB Verbal irony is nonliteral language that makes salient a discrepancy between expectations and reality. For researchers who study verbal irony, a critical question is: How do we grasp the meaning of ironic language? The parallel-constraint-satisfaction approach holds promise as an answer to this question. By this account, multiple cues to ironic intent, such as tone of voice, incongruity, and knowledge of the speaker, are processed rapidly and in parallel and this information is coordinated with the utterance itself in order to construct a coherent interpretation that is the best fit for the activated information. Recently, research with individuals who struggle with irony comprehension (typically developing children, individuals with autism-spectrum disorder, individuals with brain injury) has provided new clues about the complex process by which ironic meaning is inferred.
C1 Univ Calgary, Dept Psychol, Calgary, AB T2N 1N4, Canada.
RP Pexman, PM (reprint author), Univ Calgary, Dept Psychol, 2500 Univ Dr NW, Calgary, AB T2N 1N4, Canada.
EM pexman@ucalgary.ca
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NR 21
TC 17
Z9 18
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0963-7214
J9 CURR DIR PSYCHOL SCI
JI Curr. Dir. Psychol.
PD AUG
PY 2008
VL 17
IS 4
BP 286
EP 290
DI 10.1111/j.1467-8721.2008.00591.x
PG 5
WC Psychology, Multidisciplinary
SC Psychology
GA 331BK
UT WOS:000257987500010
ER
PT J
AU Gatto, CL
Broadie, K
AF Gatto, Cheryl L.
Broadie, Kendal
TI Temporal requirements of the fragile X mental retardation protein in the
regulation of synaptic structure
SO DEVELOPMENT
LA English
DT Article
DE Drosophila; gene-switch; neuromuscular junction; bouton; futsch
ID METABOTROPIC GLUTAMATE-RECEPTOR; PRESYNAPTIC TRANSMITTER RELEASE;
AMYLOID PRECURSOR PROTEIN; MOUSE MODEL; NEUROMUSCULAR-JUNCTION;
DEPENDENT TRANSLATION; MICROTUBULE STABILITY; RETROGRADE SIGNAL;
DENDRITIC SPINES; CIRCADIAN OUTPUT
AB Fragile X syndrome (FraX), caused by the loss-of-function of one gene ( FMR1), is the most common inherited form of both mental retardation and autism spectrum disorders. The FMR1 product ( FMRP) is an mRNA-binding translation regulator that mediates activity-dependent control of synaptic structure and function. To develop any FraX intervention strategy, it is essential to define when and where FMRP loss causes the manifestation of synaptic defects, and whether the reintroduction of FMRP can restore normal synapse properties. In the Drosophila FraX model, dFMRP loss causes neuromuscular junction (NMJ) synapse over-elaboration ( overgrowth, overbranching, excess synaptic boutons), accumulation of development-arrested satellite boutons, and altered neurotransmission. We used the Gene-Switch method to conditionally drive dFMRP expression to define the spatiotemporal requirements in synaptic mechanisms. Constitutive induction of targeted neuronal dFMRP at wild-type levels rescues all synaptic architectural defects in Drosophila Fmr1 (dfmr1)-null mutants, demonstrating a presynaptic requirement for synapse structuring. By contrast, presynaptic dFMRP expression does not ameliorate functional neurotransmission defects, indicating a postsynaptic dFMRP requirement. Strikingly, targeted early induction of dFMRP effects nearly complete rescue of synaptic structure defects, showing a primarily early-development role. In addition, acute dFMRP expression at maturity partially alleviates dfmr1-null defects, although rescue is not as complete as either early or constitutive dFMRP expression, showing a modest capacity for late-stage structural plasticity. We conclude that dFMRP predominantly acts early in synaptogenesis to modulate architecture, but that late dFMRP introduction at maturity can weakly compensate for early absence of dFMRP function.
C1 [Gatto, Cheryl L.; Broadie, Kendal] Vanderbilt Univ, Dept Biol Sci, Kennedy Ctr Res Human Dev, Nashville, TN 37232 USA.
RP Broadie, K (reprint author), Vanderbilt Univ, Dept Biol Sci, Kennedy Ctr Res Human Dev, 221 Kirkland Hall, Nashville, TN 37232 USA.
EM kendal.broadie@vanderbilt.edu
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NR 67
TC 51
Z9 51
PU COMPANY OF BIOLOGISTS LTD
PI CAMBRIDGE
PA BIDDER BUILDING CAMBRIDGE COMMERCIAL PARK COWLEY RD, CAMBRIDGE CB4 4DL,
CAMBS, ENGLAND
SN 0950-1991
J9 DEVELOPMENT
JI Development
PD AUG 1
PY 2008
VL 135
IS 15
BP 2637
EP 2648
DI 10.1242/dev.022244
PG 12
WC Developmental Biology
SC Developmental Biology
GA 324ZF
UT WOS:000257557200015
PM 18579676
ER
PT J
AU McDougle, CJ
AF McDougle, Christopher J.
TI Serotonin and dopamine transporter binding in children with autism
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Editorial Material
C1 Indiana Univ, Sch Med, Dept Psychiat, Bloomington, IN 47405 USA.
RP McDougle, CJ (reprint author), Indiana Univ, Sch Med, Dept Psychiat, Bloomington, IN 47405 USA.
CR McDougle C. J., 2003, AUTISM SPECTRUM DISO, P199
MCDOUGLE CJ, 2008, CLIN HDB OBSESSIVE C, P238
McDougle CJ, 2005, J CLIN PSYCHIAT, V66, P9
NR 3
TC 2
Z9 2
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD AUG
PY 2008
VL 50
IS 8
BP 565
EP 565
PG 1
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 333LQ
UT WOS:000258154400003
PM 18754890
ER
PT J
AU Makkonen, I
Riikonen, R
Kokki, H
Airaksinen, MM
Kuikka, JT
AF Makkonen, Ismo
Riikonen, Raili
Kokki, Hannu
Airaksinen, Mauno M.
Kuikka, Jyrki T.
TI Serotonin and dopamine transporter binding in children with autism
determined by SPECT
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Article
ID NOR-BETA-CIT; DEVELOPMENTAL-CHANGES; BRAIN; DISORDER; TOMOGRAPHY; GENE
AB Disturbances in the serotonergic system have been recognized in autism. To investigate the association between serotonin and dopamine transporters and autism, we studied 15 children (14 males, one female; mean age 8y 8mo [SD 3y 10mo]) with autism and 10 non-autistic comparison children (five males, five females; mean age 9y 10mo [SD 2y 8mo]) using single-photon emission computed tomography (SPECT) with [I-123] nor-beta-CIT. The children, with autism were studied during light sedation. They showed reduced serotonin transporter (SERT) binding capacity in the medial frontal cortex, midbrain, and temporal lobe areas. However, after correction due to the estimated effect of sedation, the difference remained significant only in the medial frontal cortex area (p=0.002). In the individuals with autism dopamine transporter (DAT) binding did not differ from that of the comparison group. The results indicate that SERT binding capacity is disturbed in autism. The reduction is more evident in adolescence than in earlier childhood. The low SERT binding reported here and the low serotonin synthesis capacity shown elsewhere may indicate maturation of a lesser number of serotonergic nerve terminals in individuals with autism.
C1 [Makkonen, Ismo] Kuopio Univ Hosp, Dept Paediat, Unit Child Neurol, FI-70211 Kuopio, Finland.
[Kokki, Hannu] Kuopio Univ Hosp, Dept Anaesthesiol & Intens Care, SF-70210 Kuopio, Finland.
[Airaksinen, Mauno M.] Univ Kuopio, Dept Pharmacol & Toxicol, FIN-70211 Kuopio, Finland.
[Kuikka, Jyrki T.] Kuopio Univ Hosp, Dept Clin Physiol & Nucl Med, SF-70210 Kuopio, Finland.
RP Makkonen, I (reprint author), Kuopio Univ Hosp, Dept Paediat, Unit Child Neurol, POB 1777, FI-70211 Kuopio, Finland.
EM ismo.makkonen@kuh.fi
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NR 24
TC 64
Z9 67
PU WILEY-BLACKWELL
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN 07030-5774, NJ USA
SN 0012-1622
EI 1469-8749
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD AUG
PY 2008
VL 50
IS 8
BP 593
EP 597
DI 10.1111/j.1469-8749.2008.03027.x
PG 5
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 333LQ
UT WOS:000258154400009
PM 18754896
ER
PT J
AU Barnevik-Olsson, M
Gillberg, C
Fernell, E
AF Barnevik-Olsson, Martina
Gillberg, Christopher
Fernell, Elisabeth
TI Prevalence of autism in children born to Somali parents living in
Sweden: a brief report
SO DEVELOPMENTAL MEDICINE AND CHILD NEUROLOGY
LA English
DT Article
ID IMMIGRANTS; GENES
AB In a geographical area of Stockholm, with a relatively large Somali immigrant population, parents as well as teachers in special schools and staff at habilitation centres have raised concerns over whether children with a Somali background are over-represented in the total group of children with autism. The aim of the study was, therefore, to investigate the prevalence of autism in children with parents from Somalia, living in Stockholm county, and to compare the prevalence in children of Somali background with that in the non-Somali group. We reviewed the records of 17 children (13 males, four females), born between 1988 and 1998 (age range 7-17y) and with a Somali background, who had a diagnosis of autistic disorder or pervasive developmental disorder not otherwise specified (PDDNOS) and were registered at either of the two autism habilitation centres for school-aged children. The prevalence of autistic disorder or PDDNOS was found to be three to four times higher than in the non-Somali group (0.7% vs 0.19%). All children also had learning disability.* Our findings warrant further investigations of possible aetiological factors behind the increased prevalence of autistic disorders in children of Somali origin found in this area in Sweden.
C1 [Fernell, Elisabeth] Habilitat Ctr Sch Children Autism, S-10462 Stockholm, Sweden.
[Barnevik-Olsson, Martina] Jarva BUP, Dept Child & Adolescent Psychiat, Stockholm, Sweden.
[Gillberg, Christopher] Univ Gothenburg, Dept Child & Adolescent Psychiat, Gothenburg, Sweden.
RP Fernell, E (reprint author), Habilitat Ctr Sch Children Autism, S-10462 Stockholm, Sweden.
EM elisabeth.fernell@karolinska.se
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NR 19
TC 24
Z9 24
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0012-1622
J9 DEV MED CHILD NEUROL
JI Dev. Med. Child Neurol.
PD AUG
PY 2008
VL 50
IS 8
BP 598
EP 601
DI 10.1111/j.1469-8749.2008.03036.x
PG 4
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 333LQ
UT WOS:000258154400010
PM 18754897
ER
PT J
AU Galanopoulou, AS
AF Galanopoulou, Aristea S.
TI Sexually dimorphic expression of KCC2 and GABA function
SO EPILEPSY RESEARCH
LA English
DT Review
DE GABA; substantia nigra; hippocampus; sex; autism; epilepsy
ID GAMMA-AMINOBUTYRIC-ACID; RAT SUBSTANTIA-NIGRA; RECEPTOR SUBUNIT GENES;
CHILDHOOD ABSENCE EPILEPSY; IDIOPATHIC GENERALIZED EPILEPSIES;
CATION-CHLORIDE COTRANSPORTERS; GRAMICIDIN PERFORATED-PATCH; RECURRENT
NEONATAL SEIZURES; CA3 HIPPOCAMPAL-NEURONS; SEX-RELATED DIFFERENCES
AB GABA(A) receptors have an age-adapted function in the brain. During early development, they mediate depolarizing effects, which result in activation of calcium-sensitive signaling processes that are important for the differentiation of the brain. In more mature stages of development and in adults, GABA(A) receptors acquire their classical hyperpolarizing signaling. The switch from depolarizing to hyperpolarizing GABA(A)-ergic signaling is triggered through the developmental shift in the balance of chloride cotransporters that either increase (i.e. NKCC1) or decrease (i.e. KCC2) intracellular chloride. The maturation of GABA(A) signaling follows sex-specific patterns, which correlate with the developmental expression profiles of chloride cotransporters. This has first been demonstrated in the substantia nigra, where the switch occurs earlier in females than in mates. As a result, there are sensitive periods during development when drugs or conditions that activate GABA(A) receptors mediate different transcriptional effects in mates and females. Furthermore, neurons with depolarizing or hyperpolarizing GABA(A)-ergic signaling respond differently to neurotrophic factors like estrogens. Consequently, during sensitive developmental periods, GABA(A) receptors may act as broadcasters of sexually differentiating signals, promoting gender-appropriate brain development. This has particular implications in epilepsy, where both the pathophysiology and treatment of epileptic seizures involve GABA(A) receptor activation. It is important therefore to study separately the effects of these factors not only on the course of epilepsy but also design new treatments that may not necessarily disturb the gender-appropriate brain development. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Galanopoulou, Aristea S.] Albert Einstein Coll Med, Kennedy Ctr, Saul R Korey Dept Neurol, Bronx, NY 10461 USA.
[Galanopoulou, Aristea S.] Albert Einstein Coll Med, Dominick P Purpura Dept Neurosci, Bronx, NY 10461 USA.
RP Galanopoulou, AS (reprint author), Albert Einstein Coll Med, Kennedy Ctr, Saul R Korey Dept Neurol, 1410 Pelham Pkwy S, Bronx, NY 10461 USA.
EM agalan@aecom.yu.edu
FU NIH NINDS [45243]; Rett Syndrome Research Foundation
FX I would like to acknowledge the grant support from NIH NINDS (grant
45243) and the Rett Syndrome Research Foundation.
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NR 159
TC 35
Z9 35
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0920-1211
J9 EPILEPSY RES
JI Epilepsy Res.
PD AUG
PY 2008
VL 80
IS 2-3
BP 99
EP 113
DI 10.1016/j.eplepsyres.2008.04.013
PG 15
WC Clinical Neurology
SC Neurosciences & Neurology
GA 341QE
UT WOS:000258728900001
PM 18524541
ER
PT J
AU Skovgaard, AM
Houmann, T
Christiansen, E
Olsen, EM
Landorph, SL
Lichtenberg, A
Jorgensen, T
AF Skovgaard, Anne Mette
Houmann, Tine
Christiansen, Eva
Olsen, Else Marie
Landorph, Susanne Lassen
Lichtenberg, Anne
Jorgensen, Torben
TI Can a general health surveillance between birth and 10 months identify
children with mental disorder at 1 1/2 year? A case-control study nested
in cohort CCC 2000
SO EUROPEAN CHILD & ADOLESCENT PSYCHIATRY
LA English
DT Article
DE screening; mental health; infant-toddler; psychopathology; birth cohort;
risks
ID ADOLESCENT PSYCHIATRIC-DISORDERS; PSYCHOPATHOLOGY; AGE; EPIDEMIOLOGY;
AUTISM; DESIGN
AB Mental health surveillance in infancy was studied in an existing child health surveillance programme with child psychiatric disorder at 1 1/2 year as the outcome. Methods Children considered of concern by community health nurses were cases in a case control study nested in the Copenhagen Child Cohort (CCC 2000). Outcome was mental health status at 1 1/2 year assessed by clinical and standardised strategies, including videotape recordings, parent interviews and the instruments: CBCL 1 1/2-5, ITSCL, CHAT, Bayley Scales of Infant Development II, PC ERA and PIRGAS. Results The positive predictive value of concern in the first 10 months of living was 24% (CI 17.0-31.9), the negative predictive value was 85% (CI 77.9-89.6) and the sensitivity was 56% (CI 42.4-69.0). Concern about development was significantly associated with the child having a neuro-developmental disorder at 1 1/2 year, and concern about mother-child relationship was associated with emotional, behavioural, eating, and sleeping disturbances. Conclusions A general health surveillance program seems to have potentials to identify infants at risk for mental health problems provided standardised measures and specific training of the involved health professionals.
C1 [Skovgaard, Anne Mette; Houmann, Tine; Christiansen, Eva; Olsen, Else Marie; Landorph, Susanne Lassen] Univ Copenhagen Hosp, Child & Adolescent Psychiat Ctr, DK-2600 Glostrup, Denmark.
[Olsen, Else Marie; Jorgensen, Torben] Univ Copenhagen Hosp, Res Ctr Prevent & Hlth, DK-2600 Glostrup, Denmark.
[Lichtenberg, Anne] Dept Res & Dev Hlth Care, Copenhagen, Capital Region, Denmark.
RP Skovgaard, AM (reprint author), Univ Copenhagen Hosp, Child & Adolescent Psychiat Ctr, DK-2600 Glostrup, Denmark.
EM ames@glo.regionH.dk
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NR 35
TC 6
Z9 6
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1018-8827
J9 EUR CHILD ADOLES PSY
JI Eur. Child Adolesc. Psych.
PD AUG
PY 2008
VL 17
IS 5
BP 290
EP 298
DI 10.1007/s00787-007-0666-4
PG 9
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 356MJ
UT WOS:000259782000005
PM 18301939
ER
PT J
AU Leboyer, M
AF Leboyer, M.
TI The first autism disease genes: role of synapse formation
SO EUROPEAN NEUROPSYCHOPHARMACOLOGY
LA English
DT Meeting Abstract
CT 21st Congress of the European-College-of-Neuropsychopharmacology
CY AUG 30-SEP 03, 2008
CL Barcelona, SPAIN
SP European Coll Neuropsychopharmacol
ID SPECTRUM DISORDERS; MUTATIONS
C1 [Leboyer, M.] Univ Paris 12, Dept Psychiat & Psychiat Genet, INSERM Unit, Creteil, France.
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NR 3
TC 0
Z9 0
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0924-977X
J9 EUR NEUROPSYCHOPHARM
JI Eur. Neuropsychopharmacol.
PD AUG
PY 2008
VL 18
SU 4
BP S173
EP S173
DI 10.1016/S0924-977X(08)70161-5
PG 1
WC Clinical Neurology; Neurosciences; Pharmacology & Pharmacy; Psychiatry
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Psychiatry
GA 353UK
UT WOS:000259593800052
ER
PT J
AU Walker, MA
AF Walker, Michael A.
TI Treatment of autism spectrum disorders: neurotransmitter signaling
pathways involved in motivation and reward as therapeutic targets
SO EXPERT OPINION ON THERAPEUTIC TARGETS
LA English
DT Review
DE acetylcholine; Asperger; dopamine; gamma-aminobutyric acid; nicotinic
acid; oxyrocin; Rett; vasopressin
ID PERVASIVE DEVELOPMENTAL DISORDERS; AMINO-ACID DECARBOXYLASE;
PEPTIDE-HORMONE-OXYTOCIN; VASOPRESSIN RECEPTOR; SOCIAL RECOGNITION;
GABA(A) RECEPTORS; MESSENGER-RNA; DOUBLE-BLIND; ASSOCIATION ANALYSIS;
CEREBROSPINAL-FLUID
AB Background: There is a growing body of literature describing the etiology of autism spectrum disorders (ASD). Some of the targets suggested belong to neurochemical transmitter pathways implicated in the behavior and motivation reward pathway. Objective: To examine data linking potential targets to ASD and the feasibility of developing drugs targeting these pathways. While the inhibitors are mostly being developed for other indications, it is beneficial to examine them to determine the responsiveness of the targets to small-molecule modulation. Methods: A search in Medline and Scifinder(R) for articles concerning relevant targets in the context of ASD and their relation to the reward signaling pathway. Results: There is evidence suggesting that behaviors controlled by these targets are related to behaviors exhibited by individuals with ASD. The targets appear to be involved in neurotransmitter pathways controlling motivation and reward, further implicating this system in ASD. Sufficient research has been conducted to identify lead compounds for discovering agents for treatment of ASD.
C1 Bristol Myers Squibb Co, Med Chem, Res & Dev, Wallingford, CT 06492 USA.
RP Walker, MA (reprint author), Bristol Myers Squibb Co, Med Chem, Res & Dev, 5 Res Pkwy, Wallingford, CT 06492 USA.
EM michael.a.walker@bms.com
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NR 133
TC 2
Z9 2
PU INFORMA HEALTHCARE
PI LONDON
PA TELEPHONE HOUSE, 69-77 PAUL STREET, LONDON EC2A 4LQ, ENGLAND
SN 1472-8222
J9 EXPERT OPIN THER TAR
JI Expert Opin. Ther. Targets
PD AUG
PY 2008
VL 12
IS 8
BP 949
EP 967
DI 10.1517/14728220802198140
PG 19
WC Pharmacology & Pharmacy
SC Pharmacology & Pharmacy
GA 330CL
UT WOS:000257916900004
PM 18620518
ER
PT J
AU Kumbier, E
Haack, K
Herpertz, SC
AF Kumbier, E.
Haack, K.
Herpertz, S. C.
TI Autism: Exploring historical psychiatric and psychological concepts
SO FORTSCHRITTE DER NEUROLOGIE PSYCHIATRIE
LA German
DT Article
DE autism; Bleuler; history of psychiatry; psychopathology; schizophrenia
ID ASPERGERS-SYNDROME; PSYCHOPATHY
AB Autism today is a widely used term, yet what is understood by autism has changed considerably since first being introduced in scientific discourse almost 100 years ago. Autism is one example for the influence of the psychoanalytic school of Sigmund Freud on scientific psychiatry at the beginning of the 20th century. In particular psychoanalysis had an impact on Eugen Bleuler's concept of schizophrenia. The Swiss psychiatrist did not only acknowledge and follow a biological, but also a psychological approach to psychiatry and thus opened up his subject to psychoanalytic thoughts. This paper provides insights into the term's conceptual history - or, more specifically and precisely - sheds light on the expansion of the term's scope, which got to be used for more and more symptoms and phenomena. When Bleuler first presented the term autism, he used it to refer to a classical schizophrenic symptom. Since, however, Bleuler was not very specific and exclusive in his definition, the term was soon used for other phenomena as well, such as to describe a schizoid symptom in the sense of today's schizoid personality disorder (schizoid autism). The concepts of autistic hebephrenia and depressive autism are further examples how the term was used and give insight into how the contents behind the term changed, got less and less specific and widened its scope. Due to its growing vagueness its suitability and usability as a psychopathological term decreased. This process further was strengthened when the word autism got more and more widely used in colloquial language for different aspects of day-to-day routine and thinking. Thus in psychiatry today, autism is exclusively used in connection with the so-called autism spectrum disorders, but has, as other formerly exclusively technical terms, different and rather unspecific meanings in everyday communication.
C1 [Kumbier, E.; Herpertz, S. C.] Univ Rostock, Klin & Poliklin Psychiat & psychotherapie, Zentrum Nervenheilkunde, D-18147 Rostock, Germany.
RP Kumbier, E (reprint author), Univ Rostock, Klin & Poliklin Psychiat & psychotherapie, Zentrum Nervenheilkunde, Gehlsheimer Str 20, D-18147 Rostock, Germany.
EM ekkehardt.kumbier@medizin.uni-rostock.de
RI Kumbier, Ekkehardt/G-6228-2012
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NR 60
TC 2
Z9 2
PU GEORG THIEME VERLAG KG
PI STUTTGART
PA RUDIGERSTR 14, D-70469 STUTTGART, GERMANY
SN 0720-4299
J9 FORTSCHR NEUROL PSYC
JI Forschritte Neurol. Psychiatr.
PD AUG
PY 2008
VL 76
IS 8
BP 484
EP 490
DI 10.1055/s-2008-1038229
PG 7
WC Clinical Neurology; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 338JL
UT WOS:000258503900006
PM 18677680
ER
PT J
AU Slatkin, M
AF Slatkin, Montgomery
TI Exchangeable models of complex inherited diseases
SO GENETICS
LA English
DT Article
ID GENOME-WIDE ASSOCIATION; RISK LOCI; MULTIPLE-SCLEROSIS; COMMON VARIANT;
SUSCEPTIBILITY; RELATIVES; LINKAGE; PATTERN; FAMILY; GENES
AB A model of unlinked diallelic loci affecting the risk of a complex inherited disease is explored. The goal is to determine what assumptions about dependence of disease risk on genotype are consistent with data for diseases such as schizophrenia, bipolar disorder, autism, and multiple sclerosis that are relatively common (0.1-2% prevalence) and that have high concordance rates for monozygotic twins (30-50%) and high risks to first-degree relatives of affected individuals (risk ratios exceeding 4). These observations are consistent with a variety of models, including generalized additive, multiplicative, and threshold models, provided that disease risk increases rapidly for a narrow range of numbers of causative alleles. If causative alleles are in relatively high frequency, then the combined effects of numerous causative loci are necessary to substantially elevate disease risk.
C1 Univ Calif Berkeley, Dept Integrat Biol, Berkeley, CA 94720 USA.
RP Slatkin, M (reprint author), Univ Calif Berkeley, Dept Integrat Biol, VLSB 3060, Berkeley, CA 94720 USA.
EM slatkin@berkeley.edu
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NR 30
TC 20
Z9 21
PU GENETICS
PI BALTIMORE
PA 428 EAST PRESTON ST, BALTIMORE, MD 21202 USA
SN 0016-6731
J9 GENETICS
JI Genetics
PD AUG
PY 2008
VL 179
IS 4
BP 2253
EP 2261
DI 10.1534/genetics.107.077719
PG 9
WC Genetics & Heredity
SC Genetics & Heredity
GA 339QD
UT WOS:000258591200042
PM 18689899
ER
PT J
AU Kourtidou-Papadeli, C
Papadelis, CL
Vernikos, J
Pd, B
Hitoglou-Antoniadou, M
Perantoni, E
Vlachogiannis, E
AF Kourtidou-Papadeli, C.
Papadelis, C. L.
Vernikos, J.
PD, Bamidis
Hitoglou-Antoniadou, M.
Perantoni, E.
Vlachogiannis, E.
TI The therapeutic benefits of gravity in space and on earth
SO HIPPOKRATIA
LA English
DT Article; Proceedings Paper
CT 5th International Meeting of Aerospace Medicine
CY AUG 30-SEP 02, 2007
CL Skiathos Isl, GREECE
DE hypergravity; neural plasticity; gravity sensors; autism; brain damage
ID SPACEFLIGHT; ADAPTATION; MISSION; WEIGHTLESSNESS; MICROGRAVITY;
EXERCISE; MUSCLE; BONE; RATS
AB The traditional scientific approach of investigating the role of a variable on a living organism is to remove it or the ability of the organism to sense it. Gravity is no exception. Access to space has made it possible for us to begin the exploration of how gravity has influenced our evolution, our genetic make-up and our physiology. Identifying the thresholds at which each body system perceives, how much, how often, how long the gravity stimulus is needed and in which direction should it be presented for maximum effectiveness, is fundamental knowledge required for using artificial gravity as a therapeutic or maintenance countermeasure treatment in exploration missions. Here on earth, although Surrounded by gravity we are negligent in using gravity as it was intended, to maintain the level of health that is appropriate to living in 1G. These, changes in lifestyle or pathologies caused by various types of injury call benefit as well from artificial gravity ill Much the same way as we are now considering for astronauts in space.
C1 [Kourtidou-Papadeli, C.; Papadelis, C. L.; Vernikos, J.; PD, Bamidis; Perantoni, E.] Greek Aerosp Med Assoc & Space Res, Thessaloniki, Greece.
[PD, Bamidis] Aristotle Univ Thessaloniki, Sch Med, Lab Med Informat, GR-54006 Thessaloniki, Greece.
[Hitoglou-Antoniadou, M.] Aristotle Univ Thessaloniki, Sch Med, AHEPA Hosp, Lab Speech & Commun Disorders,ENT Clin 1, GR-54006 Thessaloniki, Greece.
[Vlachogiannis, E.] St Paul Gen Hosp, Dept Internal Med, Thessaloniki, Greece.
RP Kourtidou-Papadeli, C (reprint author), Erythreas 2, Thessaloniki 55132, Greece.
EM papadeli@koz.forthnet.gr
RI Papadelis, Christos/E-5985-2010
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NR 30
TC 0
Z9 0
PU LITHOGRAPHIA
PI THESSALONIKI
PA ANTONIADIS I-PSARRAS TH G P, NEA REDESTOS, THESSALONIKI, 00000, GREECE
SN 1108-4189
J9 HIPPOKRATIA
JI Hippokratia
PD AUG
PY 2008
VL 12
SU 1
BP 28
EP 31
PG 4
WC Medicine, General & Internal
SC General & Internal Medicine
GA 353PC
UT WOS:000259579000006
PM 19050751
ER
PT J
AU Noriega, G
AF Noriega, Gerardo
TI A neural model for compensation of sensory abnormalities in autism
through feedback from a measure of global perception
SO IEEE TRANSACTIONS ON NEURAL NETWORKS
LA English
DT Article
DE autism; self-organizing maps (SOMs); sensory abnormalities; weak central
coherence (WCC)
ID SELF-ORGANIZING MAP; COHERENCE
AB Sensory abnormalities and weak central coherence (WCC), a processing bias for features and local information, are important characteristics associated with autism. This paper introduces a self-organizing map (SOM)-based computational model of sensory abnormalities in autism, and of a feedback system to compensate for them. Feedback relies on a measure of balance of coverage over four (sensory) domains. Different methods to compute this measure are discussed, as is the flexibility to configure the system using different control mechanisms. Statistically significant improvements throughout training are demonstrated for compensation of a simple (i.e., monotonically decreasing) hypersensitivity in one of the domains. Fine-tuning control parameters can lead to further gains, but a standard setup results in good performance. Significant improvements are also shown for complex hypersensitivities (i.e., increasing and decreasing through time) in two domains. Although naturally best suited to compensate hypersensitivities-stimuli filtering may mitigate neuron migration to a hypersensitive domain-the system is also shown to perform effectively when compensating hyposensitivities. With poor coverage balance in the model akin to poor global perception, WCC would be consistent with inadequate feedback, resulting in uncontrolled hyper- and/or hyposensitivities characteristic of autism, as seen in the topologies of the resulting SOMs.
C1 RMS Instruments Ltd, Mississauga, ON L4V 1L9, Canada.
RP Noriega, G (reprint author), RMS Instruments Ltd, Mississauga, ON L4V 1L9, Canada.
EM g.noriega@ieee.org
CR BALKENIUS C, 2004, P 4 INT WORKSH EP RO, P27
Bogdashina O, 2003, SENSORY PERCEPTUAL I
Cheung YM, 2007, IEEE T NEURAL NETWOR, V18, P289, DOI 10.1109/TNN.2006.885039
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NR 31
TC 2
Z9 2
PU IEEE-INST ELECTRICAL ELECTRONICS ENGINEERS INC
PI PISCATAWAY
PA 445 HOES LANE, PISCATAWAY, NJ 08855-4141 USA
SN 1045-9227
J9 IEEE T NEURAL NETWOR
JI IEEE Trans. Neural Netw.
PD AUG
PY 2008
VL 19
IS 8
BP 1402
EP 1414
DI 10.1109/TNN.2008.2000447
PG 13
WC Computer Science, Artificial Intelligence; Computer Science, Hardware &
Architecture; Computer Science, Theory & Methods; Engineering,
Electrical & Electronic
SC Computer Science; Engineering
GA 338KD
UT WOS:000258505700007
PM 18701370
ER
PT J
AU Liu, CC
Conn, K
Sarkar, N
Stone, W
AF Liu, Changchun
Conn, Karla
Sarkar, Nilanjan
Stone, Wendy
TI Online affect detection and robot behavior adaptation for intervention
of children with autism
SO IEEE TRANSACTIONS ON ROBOTICS
LA English
DT Article; Proceedings Paper
CT 16th IEEE International Symposium on Robot and Human Interactive
Communication
CY AUG 26-29, 2007
CL Cheju Isl, SOUTH KOREA
SP IEEE
DE autism intervention; closed-loop human-robot interaction (HRI);
physiological sensing
ID HUMAN-COMPUTER INTERACTION; EMOTION RECOGNITION; SOCIAL-SKILLS; ANXIETY;
PEOPLE; TASK
AB Investigation into robot-assisted intervention for children with autism spectrum disorder (ASD) has gained momentum in recent years. Therapists involved in interventions must overcome the communication impairments generally exhibited by children with ASD by adeptly inferring the affective cues of the children to adjust the intervention accordingly. Similarly, a robot must also be able to understand the affective needs of these children-an ability that the current robot-assisted ASD intervention systems lack-to achieve effective interaction that addresses the role of affective states in human-robot interaction and intervention practice. In this paper, we present a physiology-based affect-inference mechanism for robot-assisted intervention where the robot can detect the affective states of a child with ASD as discerned by a therapist and adapt its behaviors accordingly. This paper is the first step toward developing "understanding" robots for use in future ASD intervention. Experimental results with six children with ASD from a proof-of-concept experiment (i.e., a robot-based basketball game) are presented. The robot learned the individual liking level of each child with regard to the game configuration and selected appropriate behaviors to present the task at his/her preferred liking level. Results show that the robot automatically predicted individual liking level in real time with 81.1 % accuracy. This is the first time, to our knowledge, that the affective states of children with ASD have been detected via a physiology-based affect recognition technique in real time. This is also the first time that the impact of affect-sensitive closed-loop interaction between a robot and a child with ASD has been demonstrated experimentally.
C1 [Liu, Changchun; Conn, Karla; Sarkar, Nilanjan] Vanderbilt Univ, Dept Elect Engn & Comp Sci, Nashville, TN 37235 USA.
[Sarkar, Nilanjan] Vanderbilt Univ, Dept Mech Engn, Nashville, TN 37235 USA.
[Stone, Wendy] TRIAD, Nashville, TN 37203 USA.
[Stone, Wendy] Vanderbilt Kennedy Ctr, MARI, Nashville, TN 37203 USA.
[Stone, Wendy] Vanderbilt Univ, Dept Pediat, Nashville, TN 37203 USA.
[Stone, Wendy] Vanderbilt Univ, Dept Psychol & Human Dev, Nashville, TN 37203 USA.
RP Liu, CC (reprint author), Vanderbilt Univ, Dept Elect Engn & Comp Sci, 221 Kirkland Hall, Nashville, TN 37235 USA.
EM changchun.liu@vanderbilt.edu; karla.g.conn@vanderbilt.edu;
nilanjan.sarkar@vanderbilt.edu; wendy.stone@vanderbilt.edu
RI Liu, Changchun/D-2256-2012; Welch, Karla/B-3425-2013
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NR 52
TC 33
Z9 33
PU IEEE-INST ELECTRICAL ELECTRONICS ENGINEERS INC
PI PISCATAWAY
PA 445 HOES LANE, PISCATAWAY, NJ 08855 USA
SN 1552-3098
J9 IEEE T ROBOT
JI IEEE Trans. Robot.
PD AUG
PY 2008
VL 24
IS 4
BP 883
EP 896
DI 10.1109/TRO.2008.2001362
PG 14
WC Robotics
SC Robotics
GA 340AH
UT WOS:000258617900011
ER
PT J
AU Mazur-Kolecka, B
Cohen, IL
Jenkins, EC
Brown, WT
Frackowiak, J
AF Mazur-Kolecka, Bozena
Cohen, Ira L.
Jenkins, Edmund C.
Brown, W. Ted
Frackowiak, Janusz
TI Application of neuronal progenitors to study pathomechanisms of autism
in vitro
SO IN VITRO CELLULAR & DEVELOPMENTAL BIOLOGY-ANIMAL
LA English
DT Meeting Abstract
C1 [Mazur-Kolecka, Bozena; Cohen, Ira L.; Jenkins, Edmund C.; Brown, W. Ted; Frackowiak, Janusz] New York State Inst Basic Res Dev Disabil, Staten Isl, NY 10314 USA.
EM mazur-kolecka@omr.state.ny.us
NR 0
TC 0
Z9 0
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1071-2690
J9 IN VITRO CELL DEV-AN
JI In Vitro Cell. Dev. Biol.-Anim.
PD AUG
PY 2008
VL 44
IS 7
BP 298
EP 299
PG 2
WC Cell Biology; Developmental Biology
SC Cell Biology; Developmental Biology
GA 341HK
UT WOS:000258704600022
ER
PT J
AU Robledo, JA
Donnellan, AM
AF Robledo, Jodi A.
Donnellan, Anne M.
TI Properties of supportive relationships from the perspective of
academically successful individuals with autism
SO INTELLECTUAL AND DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID SOCIAL SUPPORT; STUDENTS; CHILDREN; LIFE
AB This qualitative Study explored supportive relationships from the perspective of 5 academically successful individuals with autism. To ensure that data were rich and based on personal experience, participants with autism identified between 2 and 4 people with whom they had a successful supportive relationship. The participants in this study identified and described properties within these relationships. Analysis of in-depth interviews and documents using the constant comparative method revealed 6 properties of the Successful supportive relationships: trust, intimacy, the presumption of competence, understanding, shared vision of independence, and good communication. Implications for research and practice are discussed.
C1 [Robledo, Jodi A.; Donnellan, Anne M.] Univ San Diego, Sch Leadership & Educ Sci, Autism Inst, San Diego, CA 92110 USA.
RP Robledo, JA (reprint author), Univ San Diego, Sch Leadership & Educ Sci, Autism Inst, 5998 Alcala Pk, San Diego, CA 92110 USA.
EM jrobledo@sandiego.edu
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NR 55
TC 4
Z9 4
PU AMER ASSOC INTELLECTUAL DEVELOPMENTAL DISABILITIES
PI WASHINGTON
PA 444 N CAPITOL ST, NW STE 846, WASHINGTON, DC 20001-1512 USA
SN 1934-9491
J9 INTELLECT DEV DISAB
JI Intellect. Dev. Disabil.
PD AUG
PY 2008
VL 46
IS 4
BP 299
EP 310
DI 10.1352/2008.46:299-310
PG 12
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 339WX
UT WOS:000258609100004
PM 18671444
ER
PT J
AU Lim, MA
Stack, CM
Cuasay, K
Stone, MM
McFarlane, HG
Waschek, JA
Hill, JM
AF Lim, Maria A.
Stack, Conor M.
Cuasay, Katrina
Stone, Madeleine M.
McFarlane, Hewlet G.
Waschek, James A.
Hill, Joanna M.
TI Regardless of genotype, offspring of VIP-deficient female mice exhibit
developmental delays and deficits in social behavior
SO INTERNATIONAL JOURNAL OF DEVELOPMENTAL NEUROSCIENCE
LA English
DT Article
DE vasoactive intestinal peptide; developmental milestones; developmental
delays; social behavior; neurodevelopmental disorders; autism;
neuropeptide; maternal attachment; play behavior
ID VASOACTIVE-INTESTINAL-PEPTIDE; DEPENDENT NEUROTROPHIC FACTOR;
CYCLASE-ACTIVATING POLYPEPTIDE; EMBRYONIC GROWTH; NEURITE OUTGROWTH;
ENVELOPE PROTEIN; GENE-EXPRESSION; IGF-I; MOUSE; ANTAGONIST
AB Pharmacological studies indicate that vasoactive intestinal peptide (VIP) may be necessary for normal embryonic development in the mouse. For example, VIP antagonist treatment before embryonic day I I resulted in developmental delays, growth restriction, modified adult brain chemistry and reduced social behavior. Here, developmental milestones, growth, and social behaviors of neonates of VIP-deficient mothers (VIP +/-) mated to VIP +/- males were compared with the offspring of wild type mothers (VIP +/+) mated to VIP +/+ and +/- males, to assess the contributions of both maternal and offspring VIP genotype. Regardless of their own genotype, all offsprings of VIP-deficient mothers exhibited developmental delays. No delays were seen in the offspring of wild type mothers, regardless of their own genotype. Body weights were significantly reduced in offspring of VIP-deficient mothers, with VIP null (-/-) the most affected. Regardless of genotype, all offspring of VIP-deficient mothers expressed reduced maternal affiliation compared with wild type offspring of wild type mothers; +/- offspring of wild type mothers did not differ in maternal affiliation from their wild type littermates. Play behavior was significantly reduced in all offsprings of VIP-deficient mothers. Maternal behavior did not differ between wild type and VIP-deficient mothers, and cross-fostering of litters did not change offspring development, indicating that offspring deficits were induced prenatally. This study illustrated that the VIP status of a pregnant mouse had a greater influence on the growth, development and behavior of her offspring than the VIP genotype of the offspring themselves. Deficiencies were apparent in +/+, +/- and -/- offspring born to VIP-deficient mothers; no deficiencies were apparent in +/- offspring born to normal mothers. These results underscore the significant contribution of the uterine environment to normal development and indicate a potential usefulness of the VIP knockout mouse in furthering the understanding of neurodevelopmental disorders with social behavior deficits such as autism. Published by Elsevier Ltd on behalf of ISDN.
C1 [Lim, Maria A.; Stack, Conor M.; Cuasay, Katrina; Stone, Madeleine M.; McFarlane, Hewlet G.; Hill, Joanna M.] NIMH, Behav Sci Lab, NIH, Bethesda, MD 20892 USA.
[McFarlane, Hewlet G.] Kenyon Coll, Dept Psychol, Gambier, OH 43022 USA.
[Waschek, James A.] Univ Calif Los Angeles, Dept Psychiat & Biobehav Sci, Los Angeles, CA 90024 USA.
RP Hill, JM (reprint author), NIMH, Behav Sci Lab, NIH, Bldg 35,Room 1C903,35 Convent Dr, Bethesda, MD 20892 USA.
EM hilljoa@mail.nih.gov
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NR 61
TC 10
Z9 10
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0736-5748
J9 INT J DEV NEUROSCI
JI Int. J. Dev. Neurosci.
PD AUG
PY 2008
VL 26
IS 5
BP 423
EP 434
DI 10.1016/j.ijdevneu.2008.03.002
PG 12
WC Developmental Biology; Neurosciences
SC Developmental Biology; Neurosciences & Neurology
GA 312EC
UT WOS:000256650900006
PM 18423945
ER
PT J
AU Weiss, HR
Liu, X
Chi, OZ
AF Weiss, Harvey R.
Liu, Xia
Chi, Oak Z.
TI Cerebral O-2 consumption in young Eker rats, effects of GABA blockade:
implications for autism
SO INTERNATIONAL JOURNAL OF DEVELOPMENTAL NEUROSCIENCE
LA English
DT Article
DE autism spectrum disorder; gamma-aminobutyric acid; GABA(A) receptors;
cerebral blood flow; cerebral O-2 consumption
ID RECEPTOR SUBUNIT GENES; TUBEROUS SCLEROSIS; SPECTRUM DISORDERS;
OXYGEN-CONSUMPTION; BICUCULLINE; METABOLISM; ISCHEMIA; SYSTEM
AB Since there is a strong correlation between tuberous sclerosis and autism, we used a tuberous sclerosis model (Eker rat) to test the hypothesis that the increased regional cerebral O-2 consumption in the Eker rat might be associated with autism. We also examined whether this increased cerebral O-2 consumption was related to changes in the activity of the gamma-aminobutyric acid (GABA) inhibitory system. Young (4 weeks) male control Long Evans (n = 14) and Eker (n = 14) rats (70-100 g) were divided into control and bicuculline (1 mg/kg/min for 2 min then 0.1 mg/kg/min for 13 min, GABA(A) receptor antagonist) treated animals. Cerebral regional blood flow (C-14-iodoantipyrine) and O-2 consumption (cryomicrospectrophotometry) were determined in isoflurane anesthetized rats. We found significantly increased basal O-2 consumption in the cortex (6.3 +/- 0.7 ml O-2/min/100 g Eker vs. 5.1 +/- 0.2 ml O-2/min/100 g control), hippocampus and cerebellum, but not the pons. Regional cerebral blood flow was also elevated in the cortex and hippocampus in Eker rats at baseline, but cerebral O-2 extractions were similar. Bicuculline significantly increased O-2 consumption in the cortex (6.5 +/- 0.3) and all other regions of the control rats, but had no effect on cortex (5.9 +/- 1.5) or other regions of the Eker rats. Cerebral blood flow followed a similar pattern. In conclusion, Eker rats had significantly elevated cerebral O-2 consumption and blood flow, but this was not affected by GABA receptor blockade. This suggested a reduced activity of the GABA(A) receptor in the brains of Eker rats. This may have important implications in the treatment of autism. (C) 2008 ISDN. Published by Elsevier Ltd. All rights reserved.
C1 [Weiss, Harvey R.] Univ Med & Dent New Jersey, Robert Wood Johnson Med Sch, Dept Physiol & Biophys, Piscataway, NJ 08854 USA.
[Liu, Xia; Chi, Oak Z.] Univ Med & Dent New Jersey, Robert Wood Johnson Med Sch, Dept Anesthesia, Piscataway, NJ 08854 USA.
RP Weiss, HR (reprint author), Univ Med & Dent New Jersey, Robert Wood Johnson Med Sch, Dept Physiol & Biophys, 675 Hoes Lane W, Piscataway, NJ 08854 USA.
EM hweiss@umdnj.edu
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NR 35
TC 5
Z9 5
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0736-5748
J9 INT J DEV NEUROSCI
JI Int. J. Dev. Neurosci.
PD AUG
PY 2008
VL 26
IS 5
BP 517
EP 521
DI 10.1016/j.ijdevneu.2008.01.002
PG 5
WC Developmental Biology; Neurosciences
SC Developmental Biology; Neurosciences & Neurology
GA 312EC
UT WOS:000256650900015
PM 18282678
ER
PT J
AU Grynszpan, O
Martin, JC
Nadelc, J
AF Grynszpan, Ouriel
Martin, Jean-Claude
Nadelc, Jacqueline
TI Multimedia interfaces for users with high functioning autism: An
empirical investigation
SO INTERNATIONAL JOURNAL OF HUMAN-COMPUTER STUDIES
LA English
DT Article
DE multimodality; autism; design; animated conversational agent; facial
expression; training
ID ASPERGER-SYNDROME; EXECUTIVE DYSFUNCTION; NORMAL-CHILDREN; ADULTS;
VOCABULARY
AB This article focuses on issues relevant to human-computer interaction in the case of autism. We designed training software that target specific communicative disorders attributed to autism and defined an empirical protocol to test this software. The experimental software platform that we developed manages each game's interface modalities and logs users' actions, for the purpose of exploring the impact of various human-computer interfaces, which involve text, speech and images. Ten adolescents diagnosed with autism used this software during 13 sessions, at the rate of one session per week. The first and last sessions were dedicated for evaluating participants' skills. The experiment was also performed by a group of 10 typically developing children matched on developmental age and academic level. Results show that participants with autism had poorer performances on the richer multimedia interfaces. They seemed to lack the initiative of organizing the available multimodal sources of information. In this article, we specifically discuss the impact of executive disorders on the use of multimodal interfaces with an emphasis on Animated Conversational Agents. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Grynszpan, Ouriel] CNRS, LIMSI, F-91403 Orsay, France.
[Martin, Jean-Claude] Univ Paris 08, CNRS, LIMSI, LINC, F-91403 Orsay, France.
[Grynszpan, Ouriel; Nadelc, Jacqueline] Hop La Pitie Salpetriere, CNRS, UMR 7593, F-75013 Paris, France.
RP Grynszpan, O (reprint author), Hop La Pitie Salpetriere, CNRS, UMR 7593, 47 Bd Hop, F-75013 Paris, France.
EM ouriel@chups.jussieu.fr; martin@limsi.fr; jnadel@extjussieu.fr
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NR 37
TC 21
Z9 21
PU ACADEMIC PRESS LTD ELSEVIER SCIENCE LTD
PI LONDON
PA 24-28 OVAL RD, LONDON NW1 7DX, ENGLAND
SN 1071-5819
J9 INT J HUM-COMPUT ST
JI Int. J. Hum.-Comput. Stud.
PD AUG
PY 2008
VL 66
IS 8
BP 628
EP 639
DI 10.1016/j.ijhcs.2008.04.001
PG 12
WC Computer Science, Cybernetics; Ergonomics; Psychology, Multidisciplinary
SC Computer Science; Engineering; Psychology
GA 325GU
UT WOS:000257577900004
ER
PT J
AU Ivarsson, T
Melin, K
AF Ivarsson, Tord
Melin, Karin
TI Autism spectrum traits in children and adolescents with
obsessive-compulsive disorder (OCD)
SO JOURNAL OF ANXIETY DISORDERS
LA English
DT Article
DE obsessive-compulsive disorder; autistic traits; autism spectrum
disorders; co-morbidity; childhood; adolescence
ID SCHOOL-AGE-CHILDREN; SCREENING QUESTIONNAIRE; DIAGNOSTIC INTERVIEW;
ASPERGER-SYNDROME; TIC DISORDERS; PREVALENCE; FAMILY; EPIDEMIOLOGY;
METAANALYSIS; RELIABILITY
AB Objective: Assess the prevalence of autistic traits (AST) in pediatric obsessive-compulsive disorder (OCD) and relate them to OCD co-morbidity and compare them with published normative data.
Methods: Pediatric patients with obsessive-compulsive disorder (n = 109) according to the DSM-IV were studied using parent ratings of the Autistic Symptom/Syndrome Questionnaire to assess AST symptoms as a continuous rather than categorical trait. The KSADS, a semi-structured psychiatric interview, was used for the psychiatric diagnostic evaluation. Also, the Children's Yale-Brown Obsessive-Compulsive Scale was used to assess OCD severity and other clinical features.
Results: AST was common among our patients. Symptom scores were highest in cases with co-morbid Autistic Spectrum Disorders, but cases with other co-morbidities as tics/Tourette and attention/behavioral disorders also scored higher. All sub-groups, including OCD without these co-morbidities scored higher than the Swedish normative group. Using ANOVA, co-morbid ASD and tics/Tourette (plus a term for gender by tic interaction indicating that girls with tics scored high, otherwise low) and pathological doubt contributed (R-2 = .41) to the AST-traits, while OCD severity and co-morbid anxiety- and depressive disorders did not.
Conclusion: AST traits are prevalent in OCD and seem to be intricately associated with the co-morbidities as well as the OCD syndrome itself. The findings might have implication for our nosological understanding of OCD which currently is discussed. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Ivarsson, Tord] Ctr Child & Adolescent Mental Hlth, N-0484 Oslo, Norway.
[Ivarsson, Tord] Univ Gothenburg, Dept Child & Adolescent Psychiat, SE-41119 Gothenburg, Sweden.
[Ivarsson, Tord; Melin, Karin] Queen Silvia Childrens Hosp, Dept Child & Adolescent Psychiat, Gothenburg, Sweden.
RP Ivarsson, T (reprint author), R BUR, Postboks 4623 Nydalen, N-0405 Oslo, Norway.
EM tord.ivarsson@r-bup.no
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NR 35
TC 38
Z9 40
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0887-6185
J9 J ANXIETY DISORD
JI J. Anxiety Disord.
PD AUG
PY 2008
VL 22
IS 6
BP 969
EP 978
DI 10.1016/j.janxdis.2007.10.003
PG 10
WC Psychology, Clinical; Psychiatry
SC Psychology; Psychiatry
GA 330CQ
UT WOS:000257917500005
PM 18053683
ER
PT J
AU Tsermentseli, S
O'Brien, JM
Spencer, JV
AF Tsermentseli, Stella
O'Brien, Justin M.
Spencer, Janine V.
TI Comparison of form and motion coherence processing in autistic spectrum
disorders and dyslexia
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE high-functioning autism; Asperger's disorder; dyslexia; visual system;
motion processing; form processing
ID HIGH-FUNCTIONING AUTISM; MAGNOCELLULAR DEFICIT THEORY;
ASPERGER-SYNDROME; EYE-MOVEMENTS; DORSAL-STREAM; DEVELOPMENTAL DYSLEXIA;
CONTRAST SENSITIVITY; VISUAL-ATTENTION; LEARNING-DISABILITIES;
REACTION-TIME
AB A large body of research has reported visual perception deficits in both people with dyslexia and autistic spectrum disorders. In this study, we compared form and motion coherence detection between a group of adults with high-functioning autism, a group with Asperger's disorder, a group with dyslexia, and a matched control group. It was found that motion detection was intact in dyslexia and Asperger. Individuals with high-functioning autism showed a general impaired ability to detect coherent form and motion. Participants with Asperger's syndrome showed lower form coherence thresholds than the dyslexic and normally developing adults. The results are discussed with respect to the involvement of the dorsal and ventral pathways in developmental disorders.
C1 [Tsermentseli, Stella; O'Brien, Justin M.; Spencer, Janine V.] Brunel Univ, Ctr Cognit & Neuroimaging, Uxbridge UB8 3PH, Middx, England.
[O'Brien, Justin M.; Spencer, Janine V.] Brunel Univ, Ctr Res Infant Behav, Inst Psychol, Uxbridge UB8 3PH, Middx, England.
RP Tsermentseli, S (reprint author), Brunel Univ, Ctr Cognit & Neuroimaging, Uxbridge UB8 3PH, Middx, England.
EM stella.tsermentseli@brunel.ac.uk
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NR 71
TC 39
Z9 39
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1201
EP 1210
DI 10.1007/s10803-007-0500-3
PG 10
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100001
PM 18034294
ER
PT J
AU Bauminger, N
Solomon, M
Aviezer, A
Heung, K
Brown, J
Rogers, SJ
AF Bauminger, Nirit
Solomon, Marjorie
Aviezer, Anat
Heung, Kelly
Brown, John
Rogers, Sally J.
TI Friendship in high-functioning children with autism spectrum disorder:
Mixed and non-mixed dyads
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE high-functioning children with ASD; Asperger syndrome; friendship;
social-emotional functioning
ID PEER RELATIONSHIPS; MIDDLE CHILDHOOD; SOCIAL RELATIONSHIPS; EMOTION
REGULATION; SUPPORT NETWORKS; ATTACHMENT; COMPETENCE; ADJUSTMENT;
ADOLESCENCE; PRESCHOOL
AB Friendships containing a child with autism and a friend with typical development ("mixed" friendships, n = 26) and those of children with autism and a friend with a disability ("non-mixed," n = 16) were contrasted with friendships of typically developing subjects and their friends (n = 31). Measures included dyadic interaction samples, and interview and questionnaire data from subjects, friends, and parents. Mixed friendship interactions resembled typical friendships. Participants in mixed friendships were more responsive to one another, had stronger receptive language skills, exhibited greater positive social orientation and cohesion, and demonstrated more complex coordinated play than in the non-mixed dyads. Exposure to typical peers appears to have significant effects on friendship behaviors.
C1 [Bauminger, Nirit; Aviezer, Anat] Bar Ilan Univ, Sch Educ, IL-52900 Ramat Gan, Israel.
[Solomon, Marjorie; Heung, Kelly; Brown, John; Rogers, Sally J.] Univ Calif Davis, Mind Inst, Sacramento, CA 95817 USA.
RP Bauminger, N (reprint author), Bar Ilan Univ, Sch Educ, IL-52900 Ramat Gan, Israel.
EM bauminn@mail.biu.ac.il
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NR 90
TC 31
Z9 32
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1211
EP 1229
DI 10.1007/s10803-007-0501-2
PG 19
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100002
PM 18058212
ER
PT J
AU Auyeung, B
Baron-Cohen, S
Wheelwright, S
Allison, C
AF Auyeung, Bonnie
Baron-Cohen, Simon
Wheelwright, Sally
Allison, Carrie
TI The Autism Spectrum Quotient: Children's Version (AQ-Child)
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE Autism Spectrum Quotient-Children's Version; Autism; sex differences
ID ASPERGER; PHENOTYPE; PERSONALITY; DISORDERS; PARENTS
AB The Autism Spectrum Quotient-Children's Version (AQ-Child) is a parent-report questionnaire that aims to quantify autistic traits in children 4-11 years old. The range of scores on the AQ-Child is 0-150. It was administered to children with an autism spectrum condition (ASC) (n = 540) and a general population sample (n = 1,225). Results showed a significant difference in scores between those with an ASC diagnosis and the general population. Receiver-operating-characteristic analyses showed that using a cut-off score of 76, the AQ-Child has high sensitivity (95%) and specificity (95%). The AQ-Child showed good test-retest reliability and high internal consistency. Factor analysis provided support for four of the five AQ-Child design subscales. Future studies should evaluate how the AQ-C performs in population screening.
C1 [Auyeung, Bonnie; Baron-Cohen, Simon; Wheelwright, Sally; Allison, Carrie] Univ Cambridge, Dept Psychiat, Autism Res Ctr, Cambridge, England.
RP Auyeung, B (reprint author), Univ Cambridge, Dept Psychiat, Autism Res Ctr, Douglas House,18B Trumpington Rd, Cambridge, England.
EM ba251@cam.ac.uk
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Woodbury-Smith MR, 2005, J AUTISM DEV DISORD, V35, P331, DOI 10.1007/s10803-005-3300-7
NR 23
TC 80
Z9 82
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1230
EP 1240
DI 10.1007/s10803-007-0504-z
PG 11
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100003
PM 18064550
ER
PT J
AU Harris, JM
Best, CS
Moffat, VJ
Spencer, MD
Philip, RCM
Power, MJ
Johnstone, EC
AF Harris, Jonathan M.
Best, Catherine S.
Moffat, Vivien J.
Spencer, Michael D.
Philip, Ruth C. M.
Power, Michael J.
Johnstone, Eve C.
TI Autistic traits and cognitive performance in young people with mild
intellectual impairment
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autistic spectrum; intellectual impairment; central coherence; theory of
mind; executive performance
ID HIGH-FUNCTIONING AUTISM; EXECUTIVE FUNCTION; MENTAL-RETARDATION; NORMAL
ADULTS; MIND; CHILDREN; INDIVIDUALS; ADOLESCENTS; DISORDERS; TASK
AB Cognitive performance and the relationship between theory of mind (TOM), weak central coherence and executive function were investigated in a cohort of young people with additional learning needs. Participants were categorized by social communication questionnaire score into groups of 10 individuals within the autistic spectrum disorder (ASD) range, 14 within the pervasive developmental disorder range and 18 with few autistic traits. The ASD group were significantly poorer than the other groups on a test of cognitive flexibility. In the ASD group only, there was a strong relationship between executive performance and TOM which remained after controlling for IQ. Our findings suggest that the relationship between cognitive traits may more reliably distinguish autism than the presence of individual deficits alone.
C1 [Harris, Jonathan M.; Best, Catherine S.; Moffat, Vivien J.; Spencer, Michael D.; Philip, Ruth C. M.; Power, Michael J.; Johnstone, Eve C.] Univ Edinburgh, Royal Edinburgh Hosp, Div Psychiat, Edinburgh EH10 5HF, Midlothian, Scotland.
RP Harris, JM (reprint author), Univ Edinburgh, Royal Edinburgh Hosp, Div Psychiat, Kennedy Tower,Morningside Pk, Edinburgh EH10 5HF, Midlothian, Scotland.
EM jharris1@staffmail.ed.ac.uk
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NR 36
TC 7
Z9 8
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1241
EP 1249
DI 10.1007/s10803-007-0502-1
PG 9
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100004
PM 18064551
ER
PT J
AU Stel, M
van den Heuvel, C
Smeets, RC
AF Stel, Marielle
van den Heuvel, Claudia
Smeets, Raymond C.
TI Facial feedback mechanisms in Autistic Spectrum Disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; facial feedback; mimicry; imitation; emotions; facial
expressions
ID HIGH-FUNCTIONING CHILDREN; ASPERGERS-SYNDROME; NEWBORN-INFANTS;
IMITATION; INDIVIDUALS; EXPRESSIONS; ADOLESCENTS; PERCEPTION;
ACTIVATION; EMOTIONS
AB Facial feedback mechanisms of adolescents with Autistic Spectrum Disorders (ASD) were investigated utilizing three studies. Facial expressions, which became activated via automatic (Studies 1 and 2) or intentional (Study 2) mimicry, or via holding a pen between the teeth (Study 3), influenced corresponding emotions for controls, while individuals with ASD remained emotionally unaffected. Thus, individuals with ASD do not experience feedback from activated facial expressions as controls do. This facial feedback-impairment enhances our understanding of the social and emotional lives of individuals with ASD.
C1 [Stel, Marielle; van den Heuvel, Claudia] Leiden Univ, Dept Social & Org Psychol, NL-2300 RB Leiden, Netherlands.
[Smeets, Raymond C.] Radboud Univ Nijmegen, Dept Social Psychol, NL-6525 ED Nijmegen, Netherlands.
RP Stel, M (reprint author), Leiden Univ, Dept Social & Org Psychol, POB 9555, NL-2300 RB Leiden, Netherlands.
EM mastel@fsw.leidenuniv.nl
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NR 50
TC 21
Z9 22
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1250
EP 1258
DI 10.1007/s10803-007-0505-y
PG 9
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100005
PM 18293075
ER
PT J
AU Boucher, J
Bigham, S
Mayes, A
Muskett, T
AF Boucher, Jill
Bigham, Sally
Mayes, Andrew
Muskett, Tom
TI Recognition and language in low functioning autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; language; memory
ID INFANTILE-AUTISM; DECLARATIVE/PROCEDURAL MODEL; ASPERGER-SYNDROME;
EPISODIC MEMORY; CHILDREN; INDIVIDUALS; PERFORMANCE; DISORDERS;
SUBGROUPS; SPECTRUM
AB The hypothesis that a pervasive impairment of declarative memory contributes to language impairment in low functioning autism (LFA) was tested. Participants with LFA, high functioning autism (HFA), intellectual disability (ID) without autism, and typical development (TD) were given two recognition tests and four tests of lexical understanding. It was predicted that recognition would be impaired in the LFA group relative to the HFA and TD groups but not the ID group, and that recognition would correlate with lexical knowledge in the LFA group but none of the other groups. These predictions were supported except that the HFA group performed more similarly to the LFA group than expected, a finding interpreted in terms of selectively impaired episodic memory.
C1 [Boucher, Jill; Bigham, Sally] Univ Warwick, Dept Psychol, Coventry CV7 4AL, W Midlands, England.
[Mayes, Andrew] Univ Manchester, Manchester, England.
[Muskett, Tom] Univ Sheffield, Sheffield, S Yorkshire, England.
RP Boucher, J (reprint author), Univ Warwick, Dept Psychol, Coventry CV7 4AL, W Midlands, England.
EM J.Boucher@warwick.ac.uk
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NR 50
TC 8
Z9 8
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1259
EP 1269
DI 10.1007/s10803-007-0508-8
PG 11
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100006
PM 18064549
ER
PT J
AU Sanchez-Marin, FJ
Padilla-Medina, JA
AF Sanchez-Marin, Francisco J.
Padilla-Medina, Jose A.
TI A psychophysical test of the visual pathway of children with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autistic spectrum; visual pathway; visual detection; signal detection
theory; 2AFC
ID CORTICAL MAGNIFICATION FACTOR; CONTRAST SENSITIVITY; PERFORMANCE;
COHERENCE; BRAIN; TASK
AB Signal detection psychophysical experiments were conducted to investigate the visual path of children with autism. Computer generated images with Gaussian noise were used. Simple signals, still and in motion were embedded in the background noise. The computer monitor was linearized to properly display the contrast changes. To our knowledge, this is the first time that experiments of this type have been done with observers with autism. Our results show that the visual capabilities of typically developed children were superior to those of the children with autism, and that the related problems of children with autism are sensation related only in a reduced proportion. There is no evidence that they are disabled in detecting simple visual stimuli, still and in motion.
C1 [Sanchez-Marin, Francisco J.] Ctr Invest Opt, Guanajuato 37150, Mexico.
[Padilla-Medina, Jose A.] Inst Technol Celaya, Guanajuato 38010, Mexico.
RP Sanchez-Marin, FJ (reprint author), Ctr Invest Opt, Loma Bosque 115 Col,Lomas Campestre, Guanajuato 37150, Mexico.
EM sanchez@cio.mx
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NR 44
TC 12
Z9 12
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1270
EP 1277
DI 10.1007/s10803-007-0507-9
PG 8
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100007
PM 18058009
ER
PT J
AU Davis, NO
Carter, AS
AF Davis, Naomi Ornstein
Carter, Alice S.
TI Parenting stress in mothers and fathers of toddlers with autism spectrum
disorders: Associations with child characteristics
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE toddlers; parenting stress; autism spectrum disorders; mothers and
fathers
ID EMOTIONAL ASSESSMENT ITSEA; BEHAVIOR PROBLEMS; YOUNG-CHILDREN;
DIAGNOSTIC INTERVIEW; 3-YEAR-OLD CHILDREN; PRESCHOOL-CHILDREN; MATERNAL
STRESS; FAMILY STRESS; DISABILITIES; PERCEPTIONS
AB Elevated parenting stress is observed among mothers of older children with autism spectrum disorders (ASD), but little is known about parents of young newly-diagnosed children. Associations between child behavior and parenting stress were examined in mothers and fathers of 54 toddlers with ASD (mean age = 26.9 months). Parents reported elevated parenting stress. Deficits/delays in children's social relatedness were associated with overall parenting stress, parent-child relationship problems, and distress for mothers and fathers. Regulatory problems were associated with maternal stress, whereas externalizing behaviors were associated with paternal stress. Cognitive functioning, communication deficits, and atypical behaviors were not uniquely associated with parenting stress. Clinical assessment of parental stress, acknowledging differences in parenting experiences for mothers and fathers of young children with ASD, is needed.
C1 [Davis, Naomi Ornstein; Carter, Alice S.] Univ Massachusetts, Dept Psychol, Boston, MA 02125 USA.
[Davis, Naomi Ornstein; Carter, Alice S.] Boston Univ, Sch Med, Dept Anat & Neurobiol, Boston, MA 02118 USA.
RP Carter, AS (reprint author), Univ Massachusetts, Dept Psychol, 100 Morrissey Blvd, Boston, MA 02125 USA.
EM alice.carter@umb.edu
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NR 50
TC 150
Z9 155
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1278
EP 1291
DI 10.1007/s10803-007-0512-z
PG 14
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100008
PM 18240012
ER
PT J
AU Pan, CY
AF Pan, Chien-Yu
TI Objectively measured physical activity between children with autism
spectrum disorders and children without disabilities during inclusive
recess settings in Taiwan
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE physical activity; autism; children; recess; accelerometry
ID YOUNG-CHILDREN; ADOLESCENTS; BEHAVIOR; PATTERNS; YOUTH; MOTOR
AB The purpose of this study was to compare the percent time children with and without autism spectrum disorders (ASD) spent in moderate-to-vigorous physical activity (MVPA) during inclusive recess settings. Forty-eight children (ASD, 23 boys and 1 girl; Non-ASD, 23 boys and 1 girl) aged 7-12 years from 14 schools had their physical activity during recess quantified using a uniaxial accelerometer over a 5-day in school period. Children with ASD were less active during overall recess, lunchtime, first and second morning recess compared to those without disabilities (p < .01). All children in this study did not achieve 40% of recess time in physical activity, suggesting that interventions for increasing physical activity of children during inclusive recess settings are warranted.
C1 Natl Kaohsiung Normal Univ, Dept Phys Educ, Kaohsiung 802, Taiwan.
RP Pan, CY (reprint author), Natl Kaohsiung Normal Univ, Dept Phys Educ, 116,He Ping 1st Rd, Kaohsiung 802, Taiwan.
EM chpan@nknucc.nknu.edu.tw
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NR 41
TC 20
Z9 20
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1292
EP 1301
DI 10.1007/s10803-007-0518-6
PG 10
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100009
PM 18157623
ER
PT J
AU Gadow, KD
DeVincent, CJ
Drabick, DAG
AF Gadow, Kenneth D.
DeVincent, Carla J.
Drabick, Deborah A. G.
TI Oppositional defiant disorder as a clinical phenotype in children with
autism spectrum disorder
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE oppositional defiant disorder; autism spectrum disorder; autism;
Asperger's syndrome; PDDNOS; pervasive developmental disorder;
attention-deficit/hyperactivity disorder; DSM-IV; diagnosis
ID PERVASIVE DEVELOPMENTAL DISORDER; DEFICIT HYPERACTIVITY DISORDER; ADHD
SYMPTOM SUBTYPES; PAST 10 YEARS; PRESCHOOL-CHILDREN; CONDUCT DISORDER;
PSYCHIATRIC-SYMPTOMS; AGE-CHILDREN; ODD; COMORBIDITY
AB To examine the validity of oppositional defiant disorder (ODD) as a clinical phenotype distinct from attention-deficit hyperactivity disorder (ADHD), parents and teachers completed a DSM-IV-referenced rating scale and a background questionnaire for 608 children (ages 3-12 years) with autism spectrum disorder (ASD). The ASD sample was separated into four groups: ODD, ADHD, ODD + ADHD, and neither (NONE). Comparison samples were non-ASD clinic (n = 326) and community (n > 800) controls. In the ASD sample, all three ODD/ADHD groups were clearly differentiated from the NONE group, and the ODD + ADHD group had the most severe co-occurring symptoms, medication use, and environmental disadvantage. There were few differences between ASD + ODD and ASD + ADHD groups. Findings for ASD and control samples were similar, supporting overlapping mechanisms in the pathogenesis of ODD.
C1 [Gadow, Kenneth D.] SUNY Stony Brook, Dept Psychiat & Behav Sci, Stony Brook, NY 11794 USA.
[DeVincent, Carla J.] SUNY Stony Brook, Dept Pediat, New York, NY USA.
[Drabick, Deborah A. G.] Temple Univ, Dept Psychol, Philadelphia, PA 19122 USA.
RP Gadow, KD (reprint author), SUNY Stony Brook, Dept Psychiat & Behav Sci, Putnam Hall S Campus, Stony Brook, NY 11794 USA.
EM kenneth.gadow@stonybrook.edu
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 30
TC 34
Z9 34
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1302
EP 1310
DI 10.1007/s10803-007-0516-8
PG 9
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100010
PM 18188684
ER
PT J
AU Reichow, B
Volkmar, FR
Cicchetti, DV
AF Reichow, Brian
Volkmar, Fred R.
Cicchetti, Domenic V.
TI Development of the evaluative method for evaluating and determining
evidence-based practices in autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; evidence-based practice
ID EVIDENCE-BASED INTERVENTIONS; SPECIAL-EDUCATION; CHILDREN; RELIABILITY;
PSYCHOLOGY; DIAGNOSIS; QUALITY; SOCIETY
AB Although research in autism has grown more sophisticated, the gap between research knowledge and applicability of research in real world settings has grown. There have been a number of different reviews of evidence-based practices of treatments for young children with autism. Reviews which have critically evaluated the empirical evidence have not found any treatments that can be considered evidence-based. Reasons for this shortcoming are explored, and a new method for the evaluation of empirical evidence is provided. Future uses of this evaluative method are provided as well as a discussion of how this tool might aid in narrowing the research to practice gap.
C1 [Reichow, Brian] Vanderbilt Univ, Peabody Coll, Dept Special Educ, Nashville, TN 37203 USA.
[Volkmar, Fred R.; Cicchetti, Domenic V.] Yale Univ, Sch Med, Ctr Child Study, New Haven, CT 06510 USA.
RP Reichow, B (reprint author), Vanderbilt Univ, Peabody Coll, Dept Special Educ, Box 328, Nashville, TN 37203 USA.
EM breichow@alumni.unc.edu
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NR 36
TC 70
Z9 70
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
EI 1573-3432
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1311
EP 1319
DI 10.1007/s10803-007-0517-7
PG 9
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100011
PM 18095149
ER
PT J
AU Chambres, P
Auxiette, C
Vansingle, C
Gil, S
AF Chambres, Patrick
Auxiette, Catherine
Vansingle, Carole
Gil, Sandrine
TI Adult attitudes toward behaviors of a six-year-old boy with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; problematic and non-problematic behaviors; attitudes; standard
of comparison
ID PERVASIVE DEVELOPMENTAL DISORDERS; CHILDRENS ATTITUDES; DISABILITY;
INDIVIDUALS; INTERVENTION; PERCEPTIONS; INFORMATION; INTENTIONS;
KNOWLEDGE; STUDENTS
AB Parents report that their children with autism are often judged as undisciplined and rude (e.g., Peeters, Autism: From theoretical understanding to educational intervention, 1997). The phenomenon of a negative view of individuals with autism was studied here. Four behaviors (two problematic and two non-problematic) produced by a six-year-old child with autism were assessed on social, emotional, and cognitive dimensions by 88 adults in an "informed" or "uninformed" condition. The child was perceived more positively when identified as having autism. However, this effect was dependent on the type of behavior and the evaluative dimension used. The results indicate that the mere fact of being informed of a child's disability triggers the use of a different standard of comparison than that employed to evaluate typical children (Mussweiler and Strack, J Pers Soc Psychol 78:1038-1052, 2000).
C1 [Chambres, Patrick; Auxiette, Catherine; Vansingle, Carole; Gil, Sandrine] Univ Clermont Ferrand, CNRS, Lab Psychol Sociale & Cognit, UMR 6024, F-63037 Clermont Ferrand, France.
RP Chambres, P (reprint author), Univ Clermont Ferrand, CNRS, Lab Psychol Sociale & Cognit, UMR 6024, 34 Ave Carnot, F-63037 Clermont Ferrand, France.
EM Patrick.Chambres@univ-bpclermont.fr
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
[Anonymous], 1992, INT STAT CLASS DIS R
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NR 30
TC 8
Z9 8
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1320
EP 1327
DI 10.1007/s10803-007-0519-5
PG 8
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100012
PM 18297387
ER
PT J
AU Jarvinen-Pasley, A
Peppe, S
King-Smith, G
Heaton, P
AF Jarvinen-Pasley, Anna
Peppe, Susan
King-Smith, Gavin
Heaton, Pamela
TI The relationship between form and function level receptive prosodic
abilities in autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; language; perception; prosody; speech
ID SPECTRUM DISORDERS; ASPERGER-SYNDROME; CHILDREN; SPEECH; INTONATION;
DISCRIMINATION; COMMUNICATION; IMPAIRMENT; PERCEPTION; SPEAKERS
AB Prosody can be conceived as having form (auditory-perceptual characteristics) and function (pragmatic/linguistic meaning). No known studies have examined the relationship between form- and function-level prosodic skills in relation to the effects of stimulus length and/or complexity upon such abilities in autism. Research in this area is both insubstantial and inconclusive. Children with autism and controls completed the receptive tasks of the Profiling Elements of Prosodic Systems in Children (PEPS-C) test, which examines both form- and function-level skills, and a sentence-level task assessing the understanding of intonation. While children with autism were unimpaired in both form and function tasks at the single-word level, they showed significantly poorer performance in the corresponding sentence-level tasks than controls. Implications for future research are discussed.
C1 [Peppe, Susan; King-Smith, Gavin] Queen Margaret Univ, Edinburgh, Midlothian, Scotland.
[Jarvinen-Pasley, Anna; Heaton, Pamela] Univ London, Goldsmiths Coll, London, England.
RP Jarvinen-Pasley, A (reprint author), Salk Inst Biol Studies, Cognit Neurosci Lab, 10010 N Torrey Pines Rd, La Jolla, CA 92037 USA.
EM pasley@salk.edu
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NR 53
TC 17
Z9 18
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1328
EP 1340
DI 10.1007/s10803-007-0520-z
PG 13
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100013
PM 18172749
ER
PT J
AU Joosten, AV
Bundy, AC
AF Joosten, Annette V.
Bundy, Anita C.
TI The motivation of stereotypic and repetitive behavior: Examination of
construct validity of the motivation assessment scale
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE motivation; stereotypic; repetitive; validity; Rasch analysis
ID SELF-INJURIOUS-BEHAVIOR; MENTAL-RETARDATION; FUNCTIONAL-ANALYSIS;
AUTISM; RELIABILITY; DISORDERS; CHILDREN
AB Construct validity of the Motivation Assessment Scale (MAS) (Durand, Crimmins, The Motivation Assessment Scale 1988) was studied using Rasch analysis data from 67 children (246 MASs), with dual diagnosis of autism and intellectual disability or with intellectual disability only. Results failed to support the proposed unidimensional construct or the original 4-factor structure. Some motivators appear to form a unidimensional construct: "to gain attention", "to gain a tangible object", and "to escape". There was evidence that sensory stimulation represents a different construct. Children with intellectual disability were more apt to be motivated by desire to gain a tangible item or attention. Children with the dual diagnoses were more apt to have sensory stimulation or escape from task demand as a motivator for stereotypic and repetitive behavior.
C1 [Joosten, Annette V.; Bundy, Anita C.] Univ Sydney, Fac Hlth Sci, Sydney, NSW 2165, Australia.
RP Joosten, AV (reprint author), Ballarat Specialist Sch, 25 Gillies St, Ballarat, VIC 3350, Australia.
EM ajoo5942@usyd.edu.au
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NR 40
TC 5
Z9 5
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1341
EP 1348
DI 10.1007/s10803-007-0523-9
PG 8
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100014
PM 18172748
ER
PT J
AU Mongillo, EA
Irwin, JR
Whalen, DH
Klaiman, C
Carter, AS
Schultz, RT
AF Mongillo, Elizabeth A.
Irwin, Julia R.
Whalen, D. H.
Klaiman, Cheryl
Carter, Alice S.
Schultz, Robert T.
TI Audiovisual processing in children with and without autism spectrum
disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE audiovisual processing; visual influence; McGurk effect
ID PERVASIVE DEVELOPMENTAL DISORDERS; RECIPROCAL SOCIAL-BEHAVIOR; LISTENING
PREFERENCES; SPEECH-PERCEPTION; ASPERGER-SYNDROME; INFANTS; INDIVIDUALS;
INTEGRATION; STIMULI; HEARING
AB Fifteen children with autism spectrum disorders (ASD) and twenty-one children without ASD completed six perceptual tasks designed to characterize the nature of the audiovisual processing difficulties experienced by children with ASD. Children with ASD scored significantly lower than children without ASD on audiovisual tasks involving human faces and voices, but scored similarly to children without ASD on audiovisual tasks involving nonhuman stimuli (bouncing balls). Results suggest that children with ASD may use visual information for speech differently from children without ASD. Exploratory results support an inverse association between audiovisual speech processing capacities and social impairment in children with ASD.
C1 [Mongillo, Elizabeth A.; Carter, Alice S.] Univ Massachusetts, Dept Psychol, Boston, MA 02125 USA.
[Irwin, Julia R.; Whalen, D. H.] Haskins Labs Inc, New Haven, CT USA.
[Klaiman, Cheryl; Schultz, Robert T.] Yale Univ, Ctr Child Study, Yale Sch Med, New Haven, CT 06520 USA.
RP Mongillo, EA (reprint author), Univ Massachusetts, Dept Psychol, 100 Morrissey Blvd, Boston, MA 02125 USA.
EM elizabeth.mongillo@aya.yale.edu
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NR 51
TC 43
Z9 44
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1349
EP 1358
DI 10.1007/s10803-007-0521-y
PG 10
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100015
PM 18307027
ER
PT J
AU VanBergeijk, E
Klin, A
Volkmar, F
AF VanBergeijk, Ernst
Klin, Ami
Volkmar, Fred
TI Supporting more able students on the autism spectrum: College and beyond
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism spectrum disorder; Asperger syndrome; college; young adults
ID ASPERGERS SYNDROME; FOLLOW-UP; DISORDER; VIOLENCE; INDIVIDUALS;
ADOLESCENCE; POPULATION; PREVALENCE; CHILDHOOD; OUTCOMES
AB In the 1990's a surge of children were diagnosed with autism spectrum disorders (ASDs) and are now approaching college age. Through early diagnosis and intervention many of these children are now able to consider post secondary education. However, these students will need specific interventions and supports in order to be successful. This article reviews the nosology of ASDs, the legal basis for providing accommodations to students on the autism spectrum, and the incidence and prevalence of ASDs. The authors provide specific recommendations regarding the academic, independent living, social, vocational and counseling needs of college students who are on the autism spectrum. With a carefully planned transition, appropriate accommodations, and support, ASD students can be successful academically and socially in college.
C1 [VanBergeijk, Ernst] New York Inst Technol, Vocat Independence Program, Cent Islip, NY 11722 USA.
[Klin, Ami; Volkmar, Fred] Yale Univ, Yale Child Study Ctr, New Haven, CT USA.
RP VanBergeijk, E (reprint author), New York Inst Technol, Vocat Independence Program, 300 Carleton Ave,Independence Hall, Cent Islip, NY 11722 USA.
EM evanberg@nyit.edu
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NR 64
TC 30
Z9 30
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1359
EP 1370
DI 10.1007/s10803-007-0524-8
PG 12
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100016
PM 18172747
ER
PT J
AU Rutherford, MD
Towns, AM
AF Rutherford, M. D.
Towns, Ashley M.
TI Scan path differences and similarities during emotion perception in
those with and without autism spectrum disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE emotion perception; face perception; scan path; eye tracking
ID FACIAL EXPRESSIONS; FACE RECOGNITION; ASPERGER-SYNDROME; CHILDREN;
INFORMATION; FEATURES; EYES; DISCRIMINATION; ADOLESCENTS; INVERSION
AB Typical adults use predictable scan patterns while observing faces. Some research suggests that people with autism spectrum disorders (ASD) instead attend to eyes less, and perhaps to the mouth more. The current experiment was designed as a direct measure of scan paths that people with and without ASD use when identifying simple and complex emotions. Participants saw photos of emotions and chose emotion labels. Scan paths were measured via infrared corneal reflectance. Both groups looked significantly longer at eyes than mouth, and neither overall looking time at eyes nor first fixations distinguished the groups. These results are contrary to suggestions that those with ASD attend preferentially to the mouth and avoid the eyes. Furthermore, there was no interaction between group and area of the face: the ratio of attention between eyes and mouth did not differ between the ASD and control groups. However, those with ASD looked at the eyes less than the control group when viewing complex emotions.
C1 [Rutherford, M. D.; Towns, Ashley M.] McMaster Univ, Dept Psychol Neurosci & Behav, Hamilton, ON L8S 4K1, Canada.
RP Rutherford, MD (reprint author), McMaster Univ, Dept Psychol Neurosci & Behav, 1280 Main St W, Hamilton, ON L8S 4K1, Canada.
EM rutherm@mcmaster.ca
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NR 49
TC 68
Z9 68
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1371
EP 1381
DI 10.1007/s10803-007-0525-7
PG 11
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100017
PM 18297386
ER
PT J
AU McGonigle-Chalmers, M
Bodner, K
Fox-Pitt, A
Nicholson, L
AF McGonigle-Chalmers, Margaret
Bodner, Kimberly
Fox-Pitt, Alicia
Nicholson, Laura
TI Size sequencing as a window on executive control in children with autism
and Asperger's Syndrome
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE executive functioning; autism; Asperger's syndrome; working memory;
sequencing
ID PREFRONTAL COGNITIVE-PROCESSES; WORKING-MEMORY; INDIVIDUALS;
DYSFUNCTION; DISORDERS; SERIATION; MIND
AB A study is reported in which size sequencing on a touch screen is used as a measure of executive control in 20 high-functioning children with Autistic Spectrum Disorders (ASD). The data show a significant and age-independent effect of the length of sequence that can be executed without errors by these children, in comparison with a chronologically age-matched group of children with normal development. Error data and reaction times are analysed and are interpreted as revealing a constraint on the prospective component of working memory in children on the autistic spectrum even when there is no change in goal or perceptual set. It is concluded that the size sequencing paradigm is an effective measure of executive difficulties associated with autism.
C1 [McGonigle-Chalmers, Margaret; Bodner, Kimberly; Fox-Pitt, Alicia; Nicholson, Laura] Univ Edinburgh, PPLS, Sch Philosophy Psychol & Language Sci, Dept Psychol, Edinburgh EH8 9JZ, Midlothian, Scotland.
RP McGonigle-Chalmers, M (reprint author), Univ Edinburgh, PPLS, Sch Philosophy Psychol & Language Sci, Dept Psychol, 7 George Sq, Edinburgh EH8 9JZ, Midlothian, Scotland.
EM M.McGonigle@ed.ac.uk
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NR 38
TC 9
Z9 9
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1382
EP 1390
DI 10.1007/s10803-007-0396-y
PG 9
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100018
PM 17594137
ER
PT J
AU Messmer, RL
Nader, R
Craig, KD
AF Messmer, Rosemary L.
Nader, Rami
Craig, Kenneth D.
TI Brief report: Judging pain intensity in children with autism undergoing
venepuncture: The influence of facial activity
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; pain; facial expression; observer report; biasing information
ID CODING SYSTEM; EXPRESSION; INFANTS
AB The biasing effect of pain sensitivity information and the impact of facial activity on observers' judgements of pain intensity of children with autism were examined. Observers received information that pain experience in children with autism is either the same as, more intense than, or less intense than children without autism. After viewing six video clips of children with autism undergoing venepuncture, observers estimated pain intensity using a visual analogue scale. Facial activity as coded by Chambers et al. (Child Facial Action Coding System Revised Manual, 1996) had a significant impact on observers' estimates of pain intensity; pain sensitivity information did not. These results have important implications for the assessment and management of pain in children with autism.
C1 [Messmer, Rosemary L.; Nader, Rami; Craig, Kenneth D.] Univ British Columbia, Fac Educ, Vancouver, BC V6T 1Z4, Canada.
RP Messmer, RL (reprint author), Univ British Columbia, Fac Educ, 2125 Main Mall, Vancouver, BC V6T 1Z4, Canada.
EM rmessmer@interchange.ubc.ca
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Breau LM, 2001, CLIN J PAIN, V17, P178, DOI 10.1097/00002508-200106000-00011
Chambers CT, 1996, CHILD FACIAL CODING
Craig KD, 1996, CLIN J PAIN, V12, P232, DOI 10.1097/00002508-199609000-00011
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WALCO GA, 1994, NEW ENGL J MED, V331, P541, DOI 10.1056/NEJM199408253310812
NR 14
TC 9
Z9 9
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1391
EP 1394
DI 10.1007/s10803-007-0511-0
PG 4
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100019
PM 18161016
ER
PT J
AU Walenski, M
Mostofsky, SH
Gidley-Larson, JC
Ullman, MT
AF Walenski, Matthew
Mostofsky, Stewart H.
Gidley-Larson, Jennifer C.
Ullman, Michael T.
TI Brief report: Enhanced picture naming in autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; language; memory; sex difference
ID NEUROTROPHIC FACTOR; VAL66MET POLYMORPHISM; WORD PRODUCTION; LEXICAL
ACCESS; HUMAN-MEMORY; LANGUAGE; ESTROGEN; SYSTEM; MODEL; BDNF
AB Language and communication deficits are key diagnostic criteria for autism. However, not all aspects of language are equally affected. Here we present evidence of enhanced performance of a critical aspect of language-word processing-in children with autism. The results have implications for explanatory theories of autism and language, and for the development of therapeutic approaches.
C1 [Walenski, Matthew; Ullman, Michael T.] Georgetown Univ, Dept Neurosci, Brain & Language Lab, Washington, DC 20057 USA.
[Mostofsky, Stewart H.; Gidley-Larson, Jennifer C.] Kennedy Krieger Inst, Baltimore, MD USA.
[Mostofsky, Stewart H.] Johns Hopkins Univ, Sch Med, Dept Neurol, Baltimore, MD 21205 USA.
[Mostofsky, Stewart H.] Johns Hopkins Univ, Sch Med, Dept Psychiat, Baltimore, MD 21205 USA.
RP Ullman, MT (reprint author), Georgetown Univ, Dept Neurosci, Brain & Language Lab, POB 571464, Washington, DC 20057 USA.
EM michael@georgetown.edu
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NR 32
TC 23
Z9 23
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1395
EP 1399
DI 10.1007/s10803-007-0513-y
PG 5
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100020
PM 18163206
ER
PT J
AU Manev, H
Manev, R
AF Manev, Hari
Manev, Radmila
TI Pharmacological probing of type 1 autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Letter
ID CONTACTIN-ASSOCIATED-PROTEIN-LIKE-2; DISORDERS; CNTNAP2
C1 [Manev, Hari; Manev, Radmila] Univ Illinois, Inst Psychiat, Dept Psychiat, Chicago, IL 60612 USA.
RP Manev, H (reprint author), Univ Illinois, Inst Psychiat, Dept Psychiat, 1601 W Taylor St,MC912, Chicago, IL 60612 USA.
EM hmanev@psych.uic.edu
CR Alarcon M, 2008, AM J HUM GENET, V82, P150, DOI 10.1016/j.ajhg.2007.09.005
Arking DE, 2008, AM J HUM GENET, V82, P160, DOI 10.1016/j.ajhg.2007.09.015
Bakkaloglu B, 2008, AM J HUM GENET, V82, P165, DOI 10.1016/j.ajhg.2007.09.017
Manev R, 2001, BMC PSYCHIATRY, V1, DOI 10.1186/1471-244X-1-5
Stephan DA, 2008, AM J HUM GENET, V82, P7, DOI 10.1016/j.ajhg.2007.12.003
Strauss KA, 2006, NEW ENGL J MED, V354, P1370, DOI 10.1056/NEJMoa052773
Welch EM, 2007, NATURE, V447, P87, DOI 10.1038/nature05756
NR 7
TC 0
Z9 0
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1400
EP 1401
DI 10.1007/s10803-008-0594-2
PG 2
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100021
PM 18512134
ER
PT J
AU Talbot, C
AF Talbot, Catherine
TI Building social relationships: A systematic approach to teaching social
interaction skills to children and adolescents with autism spectrum
disorders and other social difficulties (Textbook edition, 1st ed.)
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Book Review
C1 Yale Univ, Sch Med, Yale Child Study Ctr, New Haven, CT 06520 USA.
RP Talbot, C (reprint author), Yale Univ, Sch Med, Yale Child Study Ctr, 230 S Frontage Rd, New Haven, CT 06520 USA.
EM cat38@email.med.yale.edu
CR Bellini S, 2008, BUILDING SOCIAL RELA
NR 1
TC 0
Z9 0
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD AUG
PY 2008
VL 38
IS 7
BP 1402
EP 1403
DI 10.1007/s10803-008-0565-7
PG 2
WC Psychology, Developmental
SC Psychology
GA 330IU
UT WOS:000257935100022
ER
PT J
AU Mandy, WPL
Skuse, DH
AF Mandy, William P. L.
Skuse, David H.
TI Research Review: What is the association between the
social-communication element of autism and repetitive interests,
behaviours and activities?
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Review
ID PERVASIVE DEVELOPMENTAL DISORDERS; OBSESSIVE-COMPULSIVE BEHAVIORS;
DIAGNOSTIC INTERVIEW; SPECTRUM DISORDERS; ASPERGER-SYNDROME; CHILDHOOD
AUTISM; SYMPTOM DOMAINS; FAMILY HISTORY; RATING-SCALE; CHILDREN
AB Autism is currently conceptualised as a unitary disorder, in which social-communication impairments are found alongside repetitive interests, behaviours and activities (RIBAs). This relies upon the validity of the assumption that social-communication impairments and RIBAs co-occur at an above chance level as a result of sharing underlying causes. In the current review it is argued that the evidence for this assumption is scarce: the very great majority of RIBA research has not been intended for or suited to its examination. In fact only three studies are fit to address directly the question of the relationship between social-communication impairment and RIBAs, and these contradict each other. In consequence, further relevant evidence was sought in the behavioural and genetic literature. This approach suggested that the correlation between social-communication impairments and RIBAs has been exaggerated in the current consensus about the autism syndrome, and that these aspects of autism may well share largely independent underlying causes. Some clinical and research implications are discussed.
C1 [Mandy, William P. L.] UCL, Sub Dept Clin Hlth Psychol, London, England.
[Mandy, William P. L.; Skuse, David H.] Great Ormond St Hosp Sick Children, Dept Child & Adolescent Mental Hlth, London WC1N 3JH, England.
[Skuse, David H.] UCL, Inst Child Hlth, Behav & Brain Sci Unit, London, England.
RP Mandy, WPL (reprint author), UCL, Sub Dept Clin Hlth Psychol, Gower St, London, England.
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NR 77
TC 75
Z9 76
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD AUG
PY 2008
VL 49
IS 8
BP 795
EP 808
DI 10.1111/j.1469-7610.2008.01911.x
PG 14
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 331CA
UT WOS:000257989100002
PM 18564070
ER
PT J
AU Boomsma, A
Van Lang, NDJ
De Jonge, MV
De Bildt, AA
Van Engeland, H
Minderaa, RB
AF Boomsma, A.
Van Lang, N. D. J.
De Jonge, M. V.
De Bildt, A. A.
Van Engeland, H.
Minderaa, R. B.
TI A new symptom model for autism cross-validated in an independent sample
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE autism spectrum disorder; symptom model; cross-validation
ID PERVASIVE DEVELOPMENTAL DISORDERS; DIAGNOSTIC INTERVIEW; SPECTRUM
DISORDER; CHILDREN; DOMAINS; ADOLESCENCE; COMPONENTS
AB Background: Results from several studies indicated that a symptom model other than the DSM triad might better describe symptom domains of autism. The present study focused on a) investigating the stability of a new symptom model for autism by cross-validating it in an independent sample and b) examining the invariance of the model regarding three covariates: symptom severity, intelligence, and age. Method: The validity of the symptom model was examined in an independent sample of N = 263 children and adolescents with autism spectrum disorders, and model invariance was studied in a larger sample of N = 356 children and adolescents with autism spectrum disorders. The fit of the symptom model to the sample data was compared to that of alternative models (including the DSM triad), and the invariance of the new model was investigated for each covariate by multiple-group comparisons. Results: The fit of the new symptom model was better than that of two alternative models. It could not be compared to that of the DSM triad, because the latter encountered empirical identification problems. There were no significant or substantive differences between the estimated model in each of the dichotomised groups for any of the three covariates, which indicated factorial invariance of both structural form and factor loadings. Conclusions: The symptom model appeared to be relatively stable: It could be cross-validated in the independent sample and factorial invariance was shown between the dichotomised groups for each covariate. Further model validation with instruments other than the Autism Diagnostic Interview-Revised (ADI-R) is recommended.
C1 [Van Lang, N. D. J.] Leiden Univ, Med Ctr, Dept Child & Adolescents Psychiat, NL-2300 AA Leiden, Netherlands.
[Boomsma, A.] Univ Groningen, Dept Stat & Measurement Theory, NL-9700 AB Groningen, Netherlands.
[De Jonge, M. V.; Van Engeland, H.] Univ Med Ctr Utrecht, Dept Child & Adolescent Psychiat, Utrecht, Netherlands.
[De Bildt, A. A.; Minderaa, R. B.] Univ Med Ctr Groningen, Dept Child & Adolescent Psychiat, Groningen, Netherlands.
RP Van Lang, NDJ (reprint author), Leiden Univ, Med Ctr, Dept Child & Adolescents Psychiat, POB 15, NL-2300 AA Leiden, Netherlands.
EM N.D.J.van_Lang@Curium.nl
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van Lang NDJ, 2006, J CHILD PSYCHOL PSYC, V47, P37, DOI 10.1111/j.1469-7610.2005.01434.x
NR 31
TC 18
Z9 18
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD AUG
PY 2008
VL 49
IS 8
BP 809
EP 816
DI 10.1111/j.1469-7610.2008.01897.x
PG 8
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 331CA
UT WOS:000257989100003
PM 18492042
ER
PT J
AU Ben-Sasson, A
Cermak, SA
Orsmond, GI
Tager-Flusberg, H
Kadlec, MB
Carter, AS
AF Ben-Sasson, A.
Cermak, S. A.
Orsmond, G. I.
Tager-Flusberg, H.
Kadlec, M. B.
Carter, A. S.
TI Sensory clusters of toddlers with autism spectrum disorders: differences
in affective symptoms
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE autism; clusters; sensory profile; affective; toddlers; anxiety;
depression
ID PERVASIVE DEVELOPMENTAL DISORDERS; YOUNG-CHILDREN; DIAGNOSIS;
ABNORMALITIES; RESPONSIVITY; INTEGRATION; MODULATION; SAMPLES; PDD; AGE
AB Background: Individuals with autism spectrum disorders (ASDs) show variability in their sensory behaviors. In this study we identified clusters of toddlers with ASDs who shared sensory profiles and examined differences in affective symptoms across these clusters. Method: Using cluster analysis 170 toddlers with ASDs were grouped based on parent rating of the Infant Toddler Sensory Profile (Dunn, 2002) under-responsivity, over-responsivity, and seeking scales. Affective symptoms were evaluated with the Infant Toddler Social Emotional Assessment (Carter & Briggs-Gowan, 2005). Results: Three clusters were identified: (1) low frequency of sensory symptoms (n = 44); (2) high frequency of symptoms (n = 49); and (3) mixed (n = 77); high frequency of under-and over-responsivity and low frequency of seeking). Relative to the low frequency cluster, parents rated toddlers in the high frequency and mixed clusters (both characterized by high frequencies of sensory under- and over-responsivity) as higher on negative emotionality, depression, and anxiety symptoms. Sensory and affective differences among clusters remained after co-varying severity of ASD symptoms. Conclusions: Interdisciplinary assessments are recommended for toddlers with ASDs in order to identify the interplay of sensory and affective symptoms.
C1 [Ben-Sasson, A.] Univ Haifa, Dept Occupat Therapy, IL-31905 Haifa, Israel.
[Cermak, S. A.; Orsmond, G. I.] Boston Univ, Sargent Coll Hlth & Rehabil Sci, Boston, MA 02215 USA.
[Kadlec, M. B.; Carter, A. S.] Boston Univ, Sch Med, Studies Adv Autism Res & Treatment Ctr, Boston, MA 02215 USA.
[Carter, A. S.] Univ Massachusetts, Amherst, MA 01003 USA.
RP Ben-Sasson, A (reprint author), Univ Haifa, Dept Occupat Therapy, Eshkol Bldg,Mt Carmel, IL-31905 Haifa, Israel.
EM ayelet@bu.edu
RI Tager-Flusberg, Helen/D-5265-2009
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NR 42
TC 53
Z9 53
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD AUG
PY 2008
VL 49
IS 8
BP 817
EP 825
DI 10.1111/j.1469-7610.2008.01899.x
PG 9
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 331CA
UT WOS:000257989100004
PM 18498344
ER
PT J
AU Morgan, L
Wetherby, AM
Barber, A
AF Morgan, Lindee
Wetherby, Amy M.
Barber, Angie
TI Repetitive and stereotyped movements in children with autism spectrum
disorders late in the second year of life
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE assessment; autism spectrum disorders; developmental delay; infancy;
repetitive and stereotyped behavior
ID SYMBOLIC BEHAVIOR SCALES; PREDICTIVE-VALIDITY; DEVELOPMENTAL PROFILE;
DIAGNOSTIC INTERVIEW; YOUNG-CHILDREN; FOLLOW-UP; 2ND YEAR; AGE;
COMMUNICATION; RELIABILITY
AB Objectives: The purpose of this study was to examine group differences and relationships with later developmental level and autism symptoms using a new clinical tool developed to measure repetitive and stereotyped movements (RSM) in young children. Method: Videotaped behavior samples using the Communication and Symbolic Behavior Scales Developmental Profile (CSBS; Wetherby & Prizant, 2002) were coded for children with autism spectrum disorders (ASD; n = 50), developmental delays without ASD (DD; n = 25), and typical development (TD; n = 50) between 18 and 24 months of age. Results: Children with ASD demonstrated significantly higher rate and larger inventory of RSM with objects and body during a systematic behavior sample than both the DD and TD groups. Measures of RSM were related to concurrent measures of social communication and predicted developmental outcomes and autism symptoms in the fourth year for the ASD group. None of the correlations between RSM and autism symptoms remained significant when controlling for CSBS Symbolic level. RSM with objects predicted unique variance in the severity of autism symptoms in the fourth year beyond that predicted by social communication measures alone. Conclusions: This study provides support for the diagnostic significance of RSM in children under 24 months of age and documents the utility of this RSM measurement tool as a companion to the CSBS.
C1 [Morgan, Lindee; Wetherby, Amy M.; Barber, Angie] Florida State Univ, Tallahassee, FL 32306 USA.
RP Morgan, L (reprint author), 625 B N Adams St, Tallahassee, FL 32301 USA.
EM lindee.morgan@med.fsu.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Bakeman R, 1997, OBSERVING INTERACTIO
Baranek GT, 1999, J AUTISM DEV DISORD, V29, P213, DOI 10.1023/A:1023080005650
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Wetherby AM, 2004, J AUTISM DEV DISORD, V34, P473, DOI 10.1007/s10803-004-2544-y
Wetherby AM, 2003, INFANT YOUNG CHILD, V16, P161
NR 40
TC 32
Z9 34
PU WILEY-BLACKWELL
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN 07030-5774, NJ USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD AUG
PY 2008
VL 49
IS 8
BP 826
EP 837
DI 10.1111/j.1469-7610.2008.01904.x
PG 12
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 331CA
UT WOS:000257989100005
PM 18503532
ER
PT J
AU Scherf, KS
Behrmann, M
Minshew, N
Luna, B
AF Scherf, K. Suzanne
Behrmann, Marlene
Minshew, Nancy
Luna, Beatriz
TI Atypical development of face and greeble recognition in autism
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE autism; visual processing; configural processing; face recognition;
greebles; perceptual development; expertise; adolescence; child
development; cognition
ID UPSIDE-DOWN FACES; OBJECT RECOGNITION; SPECTRUM DISORDERS;
ASPERGER-SYNDROME; CHILDREN; INVERSION; INDIVIDUALS; ADOLESCENTS;
PERCEPTION; ACTIVATION
AB Background: Impaired face processing is a widely documented deficit in autism. Although the origin of this deficit is unclear, several groups have suggested that a lack of perceptual expertise is contributory. We investigated whether individuals with autism develop expertise in visuoperceptual processing of faces and whether any deficiency in such processing is specific to faces, or extends to other objects, too. Method: Participants performed perceptual discrimination tasks, including a face inversion task and a classification-level task, which requires especially fine-grained discriminations, on three classes of stimuli: socially-laden faces, perceptually homogenous novel objects, greebles, and perceptually heterogeneous common objects. Results: We found that children with autism develop typical levels of expertise for recognition of common objects. However, they evince poorer recognition for perceptually homogenous objects, including faces and, most especially, greebles. Conclusions: Documenting the atypical recognition abilities for greebles in children with autism has provided an important insight into the potential origin of the relatively poor face recognition skills. Our findings suggest that, throughout development, individuals with autism have a generalized deficit in visuoperceptual processing that may interfere with their ability to undertake configural processing, and that this, in turn, adversely impacts their recognition of within-class perceptually homogenous objects.
C1 [Scherf, K. Suzanne; Behrmann, Marlene] Carnegie Mellon Univ, Dept Psychol, Pittsburgh, PA 15213 USA.
[Minshew, Nancy; Luna, Beatriz] Univ Pittsburgh, Dept Psychiat, Pittsburgh, PA 15260 USA.
[Minshew, Nancy] Univ Pittsburgh, Dept Neurol, Pittsburgh, PA 15260 USA.
[Luna, Beatriz] Univ Pittsburgh, Dept Psychol, Pittsburgh, PA 15260 USA.
RP Scherf, KS (reprint author), Carnegie Mellon Univ, Dept Psychol, Baker Hall 330,5000 Forbes Ave, Pittsburgh, PA 15213 USA.
EM scherf@pitt.edu
RI Luna, Beatriz/F-1201-2010
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NR 40
TC 22
Z9 22
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD AUG
PY 2008
VL 49
IS 8
BP 838
EP 847
DI 10.1111/j.1469-7610.2008.01903.x
PG 10
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 331CA
UT WOS:000257989100006
PM 18422548
ER
PT J
AU Geurts, HM
Luman, M
van Meel, CS
AF Geurts, Hilde M.
Luman, Mariolein
van Meel, Catharina S.
TI What's in a game: the effect of social motivation on interference
control in boys with ADHD and autism spectrum disorders
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE autism; ADHD; motivation; cognitive control
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; ERROR-RELATED NEGATIVITY;
DEFICIT HYPERACTIVITY DISORDER; ANTERIOR CINGULATE CORTEX; BEHAVIORAL
TREATMENT; SUSTAINED ATTENTION; EXECUTIVE FUNCTION; FRONTAL-CORTEX;
CHILDREN; AMYGDALA
AB Background: Children with attention deficit hyperactivity disorder (ADHD) and with autism spectrum disorders (ASD) are known to have cognitive control deficits. Some studies suggest that such deficits may be reduced when motivation is increased through tangible reinforcers. Whether these deficits can also be modulated by non-tangible reinforcers has hardly been studied. Methods: Therefore, the effect of social motivation on the ability to suppress irrelevant information (i.e., interference control) was investigated in 22 ADHD boys, 22 ASD boys, and 33 typically developing (TD) boys. An adapted Eriksen Flanker task was administered under a motivational condition in which the boys were told that they were competing with peers, and under a neutral condition in which standard instructions were given. Results: In comparison with TD boys, boys with ADHD were impaired even when no interference was present, while this was not the case for the ASD boys. All groups benefited from the motivation manipulation, i.e., their performance increased when they thought they were competing with peers. Although the boys with ADHD were still slower than TD boys when motivated, they performed as accurately as TD boys. Children with ASD also improved slightly in accuracy and response speed, but this did not reach significance. Conclusion: Children with ADHD are able to exert sufficient cognitive control when they are motivated, which is in line with the current models of ADHD. However, motivation seems to have a general effect on performance and is not solely related to cognitive control abilities. In contrast, this effect was not obtained in children with ASD.
C1 [Geurts, Hilde M.] Univ Amsterdam, Div Psychon, NL-1018 WB Amsterdam, Netherlands.
[Luman, Mariolein] Vrije Univ Amsterdam, Dept Clin Neuropsychol, Amsterdam, Netherlands.
[van Meel, Catharina S.] Leiden Univ, Leiden Inst Psychol Res, NL-2300 RA Leiden, Netherlands.
[van Meel, Catharina S.] Leiden Univ, Leiden Inst Brain & Cognit, NL-2300 RA Leiden, Netherlands.
RP Geurts, HM (reprint author), Univ Amsterdam, Div Psychon, Roetersstraat 15, NL-1018 WB Amsterdam, Netherlands.
EM h.m.geurts@uva.nl
RI Luman, Marjolein/C-7752-2009
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NR 56
TC 30
Z9 30
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD AUG
PY 2008
VL 49
IS 8
BP 848
EP 857
DI 10.1111/j.1469-7610.2008.01916.x
PG 10
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 331CA
UT WOS:000257989100007
PM 18564068
ER
PT J
AU Howlin, P
AF Howlin, Patricia
TI Autism spectrum disorders: Psychological theory and research
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Book Review
CR BOWLER D, 2006, AUTISM SPECTRUM DISO
NR 1
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD AUG
PY 2008
VL 49
IS 8
BP 894
EP 894
DI 10.1111/j.1469-7610.2008.01895.x
PG 1
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 331CA
UT WOS:000257989100012
ER
PT J
AU Ray, T
Tobias, JD
AF Ray, Theresa
Tobias, Joseph D.
TI Dexmedetomidine for sedation during electroencephalographic analysis in
children with autism, pervasive developmental disorders, and seizure
disorders
SO JOURNAL OF CLINICAL ANESTHESIA
LA English
DT Article
DE dexmedetomidine; EEG analysis; procedural sedation; autism
ID INTRAVENOUS DEXMEDETOMIDINE; ADRENOCEPTOR AGONISTS; CHLORAL HYDRATE;
ANESTHESIA; THRESHOLD; RATS; VENTILATION; VOLUNTEERS; EXPERIENCE;
DECREASES
AB Study Objective: To assess the efficacy of dexmedetomidine in providing sedation during electroencephalographic (EEG) analysis in children with autism, seizure disorders, or pervasive developmental disorders (PDDs).
Design: Retrospective chart review.
Setting: University medical center.
Measurements: The charts of 42 children, aged two to 11 years, who received dexmedetomidine for sedation during EEG analysis, were studied. Information collected included route of administration of dexmedetomidine (oral and/or intravenous [IV]), loading dose, and infusion rate. Heart rate, blood pressure, respiratory rate, and level of sedation were monitored every 5 minutes, and oxygen saturation was monitored continuously during the procedure. Interventions (administration of fluid or use of an anticholinergic agent) for hypotension or bradycardia were identified.
Main Results: 18 children received oral dexmedetomidine (range, 2.9-4.4 mu g/kg) before placement of an TV. Forty patients received an IV loading dose of dexmedetomidine (2.1 +/- 0.8 mu g/kg), which was given in increments of 0.5 to one mu g/kg every three to 5 minutes until a sedation score of 3 to 4 was achieved. Effective sedation was eventually achieved in all patients. An IV infusion of dexmedetomidine was started (1.5 +/- 0.2 mu g kg(-1) hr(-1)) in all patients. During performance of the EEG, adjustments in the infusion rate (increase or decrease) or additional bolus doses were necessary in 25 patients. No significant hemodynamic or respiratory effects were noted.
Conclusions: Dexmedetomidine provides effective sedation during EEG analysis in children with autism or PDD. (C) 2008 Elsevier Inc. All rights reserved.
C1 [Tobias, Joseph D.] Univ Missouri, Dept Anesthesiol, Sch Med, Div Pediat Anesthesiol, Columbia, MO 65212 USA.
[Tobias, Joseph D.] Univ Missouri, Dept Pediat, Sch Med, Russell & Mary Shelden Chair Pediat Intens Care M, Columbia, MO 65212 USA.
RP Tobias, JD (reprint author), Univ Missouri, Dept Anesthesiol, Sch Med, Div Pediat Anesthesiol, 3W-27G HSC,1 Hosp Dr, Columbia, MO 65212 USA.
EM tobiasj@health.missouri.edu
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NR 30
TC 14
Z9 18
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0952-8180
J9 J CLIN ANESTH
JI J. Clin. Anesth.
PD AUG
PY 2008
VL 20
IS 5
BP 364
EP 368
DI 10.1016/j.jclinane.2008.03.004
PG 5
WC Anesthesiology
SC Anesthesiology
GA 349GH
UT WOS:000259268800009
PM 18761245
ER
PT J
AU Handen, BL
Sahl, R
Hardan, AY
AF Handen, Benjamin L.
Sahl, Robert
Hardan, Antonio Y.
TI Guanfacine in children with autism and/or intellectual disabilities
SO JOURNAL OF DEVELOPMENTAL AND BEHAVIORAL PEDIATRICS
LA English
DT Article
DE autism; intellectual disability; guanfacine
ID DEFICIT HYPERACTIVITY DISORDER; PERVASIVE DEVELOPMENTAL DISORDERS;
MENTALLY-RETARDED CHILDREN; OPEN TRIAL; METHYLPHENIDATE; CLONIDINE;
PLACEBO; RETARDATION; EFFICACY; ADHD
AB Objective: Attention-deficit/hyperactivity disorder (ADHD) affects 3%-5% of typical school-age children. However, considerably higher rates of ADHD (15%-25%) are observed in children with intellectual disability and autism. Studies of psychostimulants in the latter two populations have found poorer response rates compared to typically developing children. In addition, evidence suggests that children with developmental disabilities experience higher rates of adverse events. Guanfacine, an alpha(2)-adrenergic receptor agonist, has shown some promise as an alternative to psychostimulants. Methods: The present study involved a double-blind, placebo-controlled, crossover trial of guanfacine in 11 children (ages 5-9 years) with developmental disabilities and symptoms of inattention/overactivity. The 6-week trial involved a maximum dose of 3 mg/day of guanfacine. Results: Significant benefits were observed on the Hyperactivity subscale of the parent and teacher Aberrant Behavior Checklist (ABC) and Global Improvement Ratings. No gains were noted on other ABC subscales. Five of the 11 subjects (45%) were judged to be responders based on a 50% decrease in the ABC Hyperactivity subscale score between the placebo and guanfacine conditions. Several side effects were reported, including drowsiness and irritability. Conclusion: While guanfacine appears to be an alternative to psychostimulants among children with developmental disabilities, clinicians need to remain vigilant to the possibility of side effects.
C1 [Handen, Benjamin L.] Univ Pittsburgh, Sch Med, Pittsburgh, PA USA.
[Sahl, Robert] Inst Living, Hartford, CT USA.
[Hardan, Antonio Y.] Stanford Univ, Stanford, CA 94305 USA.
RP Handen, BL (reprint author), 1011 Bingham St, Pittsburgh, PA 15203 USA.
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American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 32
TC 35
Z9 35
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0196-206X
J9 J DEV BEHAV PEDIATR
JI J. Dev. Behav. Pediatr.
PD AUG
PY 2008
VL 29
IS 4
BP 303
EP 308
DI 10.1097/DBP.0b013e3181739b9d
PG 6
WC Behavioral Sciences; Psychology, Developmental; Pediatrics
SC Behavioral Sciences; Psychology; Pediatrics
GA 337CA
UT WOS:000258411900012
PM 18552703
ER
PT J
AU Krishnamurthy, V
AF Krishnamurthy, Vibha
TI A clinical experience of autism in India
SO JOURNAL OF DEVELOPMENTAL AND BEHAVIORAL PEDIATRICS
LA English
DT Editorial Material
C1 [Krishnamurthy, Vibha] Ummeed Child Dev Ctr, Bombay 400011, Maharashtra, India.
[Krishnamurthy, Vibha] Jaslok Hosp & Res Ctr, Bombay, Maharashtra, India.
RP Krishnamurthy, V (reprint author), Ummeed Child Dev Ctr, Bombay 400011, Maharashtra, India.
EM vibha_krish@hotmail.com
CR [Anonymous], 2007, MMWR SURVEILL SUMM, V56, P12
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KUMAR D, 2006, PUBLIC HLTH, V20, P705
*OFF REG GEN CENS, CENS IND DAT 2001
NR 6
TC 7
Z9 7
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0196-206X
J9 J DEV BEHAV PEDIATR
JI J. Dev. Behav. Pediatr.
PD AUG
PY 2008
VL 29
IS 4
BP 331
EP 333
DI 10.1097/DBP.0b013e3181829f1f
PG 3
WC Behavioral Sciences; Psychology, Developmental; Pediatrics
SC Behavioral Sciences; Psychology; Pediatrics
GA 337CA
UT WOS:000258411900016
PM 18698197
ER
PT J
AU Matson, JL
Wilkins, J
AF Matson, Johnny L.
Wilkins, Jonathan
TI Reliability of the Autism Spectrum Disorders-Comorbid for Children
(ASD-CC)
SO JOURNAL OF DEVELOPMENTAL AND PHYSICAL DISABILITIES
LA English
DT Article
DE reliability; comorbidity; autism spectrum disorders
ID PREVALENCE; BEHAVIORS; COMMUNITY; SAMPLE
AB Autism spectrum disorders (ASD) in children are a serious and complex set of conditions that researchers have been attempting to quantify for many decades. In addition to the core symptoms of ASD, comorbid psychopathology is often present as well. Despite this fact and despite the many attempts to develop scaling methods for ASD, little has been done to develop tests specific to childhood psychopathology in ASD children. In this study we describe the initial psychometric properties of a new scale designed specifically to fill this assessment need, the Autism Spectrum Disorders-Comorbid for Children (ASD-CC). One-hundred and thirteen children with ASD between the ages of 2 and 16 years were assessed in order to determine interrater, test-retest, and internal reliabilities of this new scale. Implications of these promising findings with respect to further development of the ASD-DC are discussed.
C1 [Matson, Johnny L.; Wilkins, Jonathan] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
EM johnmatson@aol.com
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American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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Matson JL, 2007, RES AUTISM SPECT DIS, V1, P28, DOI 10.1016/j.rasd.2006.07.003
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Matson JL, 2007, RES DEV DISABIL, V28, P341, DOI 10.1016/j.ridd.2005.12.004
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Reynolds C. R., 2004, MANUAL BEHAV ASSESSM
Siklos S, 2007, RES DEV DISABIL, V28, P9, DOI 10.1016/j.ridd.2005.09.003
WOODWARD C, 2005, J AUTISM DEV DISORD, V9, P11
World Health Organization, 1992, INT CLASS DIS
NR 33
TC 20
Z9 20
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1056-263X
J9 J DEV PHYS DISABIL
JI J. Dev. Phys. Disabil.
PD AUG
PY 2008
VL 20
IS 4
BP 327
EP 336
DI 10.1007/s10882-008-9100-1
PG 10
WC Rehabilitation
SC Rehabilitation
GA 312WK
UT WOS:000256702900002
ER
PT J
AU Ramaekers, V
Sequeira, J
Blau, N
Quadros, E
AF Ramaekers, V
Sequeira, J.
Blau, N.
Quadros, E.
TI Asperger autism associated with folate receptor autoantibodies and
cerebral folate deficiency
SO JOURNAL OF INHERITED METABOLIC DISEASE
LA English
DT Meeting Abstract
C1 [Ramaekers, V] CHU Liege, Dept Ped Neurol, Liege, Belgium.
[Sequeira, J.; Quadros, E.] SUNY, Dept Cell Biol, Brooklyn, NY USA.
[Blau, N.] Univ Child Hosp, Div Clin Chem, Zurich, Switzerland.
NR 0
TC 0
Z9 0
PU SPRINGER
PI DORDRECHT
PA VAN GODEWIJCKSTRAAT 30, 3311 GZ DORDRECHT, NETHERLANDS
SN 0141-8955
J9 J INHERIT METAB DIS
JI J. Inherit. Metab. Dis.
PD AUG
PY 2008
VL 31
SU 1
MA 290P
BP 73
EP 73
PG 1
WC Endocrinology & Metabolism; Genetics & Heredity
SC Endocrinology & Metabolism; Genetics & Heredity
GA 340PB
UT WOS:000258656400290
ER
PT J
AU Njenga, F
AF Njenga, F.
TI Autism in Africa: A challenge in the diagnosis and management of an
important disorder
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Njenga, F.] Upper Hill Med Ctr, Nairobi 00200, Kenya.
EM fnjenga@africaonline.co.ke
NR 0
TC 1
Z9 1
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 639
EP 639
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700005
ER
PT J
AU Brown, WT
Kuchna, I
Nowicki, K
Wegiel, J
Wisniewski, T
Wegiel, J
AF Brown, W. T.
Kuchna, I.
Nowicki, K.
Wegiel, J.
Wisniewski, T.
Wegiel, J.
TI Enhanced accumulation of AB neurons in autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Brown, W. T.; Kuchna, I.; Nowicki, K.; Wegiel, J.; Wisniewski, T.; Wegiel, J.] New York State Inst Basic Res Dev Disabil, Staten Isl, NY 10314 USA.
EM Ted.Brown@OMR.State.NY.US
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 659
EP 659
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700117
ER
PT J
AU Mukherjee, RAS
Patton, M
Morgan, E
Turk, J
Rossiter, R
AF Mukherjee, R. A. S.
Patton, M.
Morgan, E.
Turk, J.
Rossiter, R.
TI Identical triplets with different types of autism: An exploration of the
aetiological causes of difference
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Mukherjee, R. A. S.; Patton, M.; Morgan, E.; Turk, J.; Rossiter, R.] Surrey & Borders Partnership NHS Trust, Surrey, England.
EM rmukherj@sgul.ac.uk
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 660
EP 660
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700126
ER
PT J
AU Senechal, C
Larivee, S
AF Senechal, C.
Larivee, S.
TI The link between MMR vaccine and autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Senechal, C.; Larivee, S.] Univ Ottawa, Ottawa, ON K1N 6N, Canada.
EM carolesenechal@aol.com
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 660
EP 660
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700125
ER
PT J
AU McClean, B
AF McClean, B.
TI Rapport-based intervention for people with severe intellectual
disability and autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
EM bmcclean@indigo.ie
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 670
EP 670
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700182
ER
PT J
AU Esposito, G
de Falco, S
Venuti, P
AF Esposito, G.
de Falco, S.
Venuti, P.
TI Analysis of gait during the second year of life in children with autism
spectrum disorders and evidence for early diagnosis
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Esposito, G.; de Falco, S.; Venuti, P.] Univ Trent, I-38100 Trento, Italy.
EM gianluca.esposito@unitn.it
RI Esposito, Gianluca/B-1374-2012
OI Esposito, Gianluca/0000-0002-9442-0254
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 672
EP 672
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700187
ER
PT J
AU Turk, J
Amin, P
Bax, M
Gillberg, C
AF Turk, J.
Amin, P.
Bax, M.
Gillberg, C.
TI Autistic disorder and epilepsy in children: Implications for the autism
construct
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Turk, J.; Amin, P.; Bax, M.; Gillberg, C.] Univ London, London SW17 0RE, England.
EM jturk@sgul.ac.uk
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 673
EP 673
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700191
ER
PT J
AU Lyng, K
AF Lyng, K.
TI Autonomy as explained by autism diagnosis, age, cohort, language skills
and challenging behaviour
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Lyng, K.] Molde Univ Coll, N-6402 Molde, Norway.
EM kolbein.lyng@himolde.no
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 674
EP 674
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700198
ER
PT J
AU Liefhebber-van der Veer, T
AF Liefhebber-van der Veer, T.
TI Autism and coping behaviour
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
EM t.liefhebber@philadelphia.nl
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 678
EP 678
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700225
ER
PT J
AU Senechal, C
Larivee, S
AF Senechal, Carole
Larivee, Serge
TI Autism: Another intelligence?
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Senechal, Carole; Larivee, Serge] Univ Ottawa, Ottawa, ON K1N 6N5, Canada.
EM carolesenechal@aol.com
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 679
EP 679
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700226
ER
PT J
AU Wenke, A
Mophosho, M
AF Wenke, A.
Mophosho, M.
TI The role of speech and language therapists as perceived by neurologists,
paediatricians and child psychiatrists before and after a child is
diagnosed with autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Wenke, A.; Mophosho, M.] Univ Witwatersrand, Dept Speech Pathol & Audiol, ZA-2050 Wits, South Africa.
EM Munyane.mophosho@wits.ac.za
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 679
EP 679
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700228
ER
PT J
AU Keen, D
AF Keen, D.
TI Seizing the moment: Enhancing engagement and learning opportunities for
children with autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Keen, D.] Griffith Univ, Sch Educ & Profess Studies, Nathan, Qld 4111, Australia.
EM d.keen@griffith.edu.au
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 684
EP 684
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700252
ER
PT J
AU Graves, P
AF Graves, P.
TI Attention deficit disorders and autism spectrum disorders:
Contributions, challenges and opportunities
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Graves, P.] So Hlth Australia, Oakleigh, Vic 3166, Australia.
EM gravespj@connexus.net.au
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 687
EP 687
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700268
ER
PT J
AU Blacher, J
Howell, E
AF Blacher, J.
Howell, E.
TI Meeting the needs of families and children with high functioning autism
and asperger syndrome in schools
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Blacher, J.; Howell, E.] Univ Calif Riverside, Riverside, CA 92521 USA.
EM jan.blacher@ucr.edu
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 713
EP 713
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700411
ER
PT J
AU Hines, M
Balandin, S
Togher, L
AF Hines, M.
Balandin, S.
Togher, L.
TI Gender and the meaning of parenting an adult with autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Hines, M.; Balandin, S.; Togher, L.] Univ Sydney, Sydney, NSW 2006, Australia.
EM mhin0228@usyd.edu.au
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 715
EP 715
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700419
ER
PT J
AU Hines, M
Balandin, S
Togher, L
AF Hines, M.
Balandin, S.
Togher, L.
TI Blaming the autism: Older parents' perceptions of their adult sons and
daughters with autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Hines, M.; Balandin, S.; Togher, L.] Univ Sydney, Sydney, NSW 2006, Australia.
EM mhin0228@usyd.edu.au
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 715
EP 715
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700421
ER
PT J
AU Tait, K
AF Tait, K.
TI The impact of children with autism on the family system in Negara Brunei
Darussalam
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Tait, K.] Univ New England, Sch Educ, Armidale, NSW 2351, Australia.
EM kathleen.tait@une.edu.au
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 715
EP 715
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700424
ER
PT J
AU Orsmond, GI
Kuo, HS
Seltzer, MM
AF Orsmond, G. I.
Kuo, H. S.
Seltzer, M. M.
TI Father involvement with adolescents and adults with autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Orsmond, G. I.; Kuo, H. S.; Seltzer, M. M.] Boston Univ, Boston, MA 02215 USA.
EM gorsmond@bu.edu
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 716
EP 716
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700427
ER
PT J
AU Baum, N
AF Baum, N.
TI Can a monthly programme for South Asian children with autism and a
skills programme for their parents affect family quality of life (FQOL)?
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Baum, N.] MukiBaum Treatment Ctr, Toronto, ON, Canada.
EM nehama@mukibaum.com
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 724
EP 724
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700470
ER
PT J
AU Jegatheesan, B
AF Jegatheesan, B.
TI From symptom recognition to services: How South Asian muslim immigrant
families navigate autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Jegatheesan, B.] Univ Washington, Seattle, WA 98195 USA.
EM binda@u.washington.edu
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 724
EP 724
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700471
ER
PT J
AU Seidel, M
AF Seidel, M.
TI The International Classification of Functioning, Disability and Health
(ICF) - Its heuristic potential for a better understanding of autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Seidel, M.] Bodelschwinghsche Anstalten Bethel, D-33617 Bielefeld, Germany.
EM Michael.seidel@bethel.de
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 726
EP 726
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700482
ER
PT J
AU Symalla, R
AF Symalla, R.
TI An ICF based core set for autism
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Symalla, R.] Bodelschwingsche Anstalen Bethel, Fachdienst Autismus, D-33617 Bielefeld, Germany.
EM autismusprogramm@bethel.de
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 726
EP 726
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700484
ER
PT J
AU Seidel, M
AF Seidel, M.
TI Chronic catatonic courses vs autism spectrum disorders - An outstanding
diagnostic challenge
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Seidel, M.] Bodelschwinghsche Anstalten Bethel, D-33617 Bielefeld, Germany.
EM michael.seidel@bethel.de
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 727
EP 727
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700491
ER
PT J
AU Gobrial, E
Raghavan, R
AF Gobrial, E.
Raghavan, R.
TI Anxiety disorders in children and young people with autism and ID
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 Northumbria Univ, Sch Hlth, Newcastle Upon Tyne NE7 7XA, Tyne & Wear, England.
Zagazig Univ, Zagazig, Egypt.
EM ereny.gobrial@unn.ac.uk
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 731
EP 731
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700513
ER
PT J
AU Drahota, A
Wood, JJ
AF Drahota, A.
Wood, J. J.
TI Intervening with the adaptive functioning of children with autism and
concurrent anxiety disorders
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Drahota, A.; Wood, J. J.] Univ Calif Los Angeles, Los Angeles, CA 90095 USA.
EM adrahota@ucla.edu
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 733
EP 733
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700524
ER
PT J
AU Gobrial, E
AF Gobrial, E.
TI Consensus model of management strategies for anxiety disorders for
children with autism and ID
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Gobrial, E.] Northumbria Univ, Sch Hlth, Newcastle Upon Tyne NE7 7XA, Tyne & Wear, England.
[Gobrial, E.] Zagazig Univ, Zagazig, Egypt.
EM ereny.gobrial@unn.ac.uk
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 733
EP 733
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700526
ER
PT J
AU Jegatheesan, B
Braun, K
AF Jegatheesan, B.
Braun, K.
TI Knowledge of autism and diagnostic practices in health care settings:
Asian immigrant families in the USA
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Jegatheesan, B.; Braun, K.] Univ Washington, Seattle, WA 98195 USA.
EM brinda@u.washington.edu
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 761
EP 761
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700680
ER
PT J
AU Felce, D
Lowe, K
Jones, E
AF Felce, D.
Lowe, K.
Jones, E.
TI The impact of autism or severe challenging behaviour on lifestyle
outcome in community housing
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Felce, D.; Lowe, K.; Jones, E.] Cardiff Univ, Sch Med, Welsh Ctr Learning Disabil, Cardiff CF14 4YS, S Glam, Wales.
EM felce@cf.ac.uk
RI turton, miranda/F-4682-2011
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 784
EP 784
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700808
ER
PT J
AU Baum, N
AF Baum, N.
TI An assessment of the effect a well designed transition process has on
individuals with Autism and other intellectual disabilities
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Baum, N.] MukiBaum Treatment Ctr, Toronto, ON M6A 1J6, Canada.
EM nehama@mukibaum.com
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 789
EP 789
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700836
ER
PT J
AU Baum, N
AF Baum, N.
TI The affect architecture and building design have on the quality of life
of people with autism and other intellectual disabilities
SO JOURNAL OF INTELLECTUAL DISABILITY RESEARCH
LA English
DT Meeting Abstract
C1 [Baum, N.] Mukibaum Treatment Ctr, Toronto, ON M6A 1J6, Canada.
EM nehama@mukibaum.com
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0964-2633
J9 J INTELL DISABIL RES
JI J. Intell. Disabil. Res.
PD AUG
PY 2008
VL 52
BP 790
EP 790
PN 8-9
PG 1
WC Education, Special; Genetics & Heredity; Clinical Neurology; Psychiatry;
Rehabilitation
SC Education & Educational Research; Genetics & Heredity; Neurosciences &
Neurology; Psychiatry; Rehabilitation
GA 328KQ
UT WOS:000257797700840
ER
PT J
AU Kent, L
Emerton, J
Bhadravathi, V
Weisblatt, E
Pasco, G
Willatt, LR
McMahon, R
Yates, JRW
AF Kent, L.
Emerton, J.
Bhadravathi, V.
Weisblatt, E.
Pasco, G.
Willatt, L. R.
McMahon, R.
Yates, J. R. W.
TI X-linked ichthyosis (steroid sulfatase deficiency) is associated with
increased risk of attention deficit hyperactivity disorder, autism and
social communication deficits
SO JOURNAL OF MEDICAL GENETICS
LA English
DT Article
ID CONTIGUOUS-GENE-SYNDROME; SCHOOL-AGE-CHILDREN; MENTAL-RETARDATION;
VCX-A; INTERSTITIAL DELETION; VARIABLE PHENOTYPE; TURNER-SYNDROME;
XP22.3; FAMILY; NLGN4
AB Background: X-linked ichthyosis (XLI) ( steroid sulfatase deficiency) is caused by deletions or point mutations of the steroid sulfatase (STS) gene on chromosome Xp22.32. Deletions of this region can be associated with cognitive behavioural difficulties including autism. Animal work suggests the STS gene may be involved in attentional processes. We have therefore undertaken a systematic study of autism and attention deficit hyperactivity disorder (ADHD) in boys with XLI.
Methods: Cases of XLI were recruited from families originally ascertained when pregnancies with STS deficiency were identified through a routine maternal screening programme. Boys with XLI were assessed for ADHD and autism using standardised questionnaires and interviews. Deletions of the STS gene were identified and characterised by analysis of genomic DNA and/or fluorescent in situ hybridisation.
Results: 25 boys with XLI were assessed for autism and ADHD. 40% fulfilled DSM-IV criteria for a diagnosis of ADHD, 80% of which were inattentive subtype. ADHD diagnoses were present in those with both deletions and presumed point mutations of STS. Additionally, five boys, from three unrelated families, fulfilled criteria for an autistic spectrum disorder or related language/communication difficulty, and all had an unusually large deletion of the STS gene with loss of the neuroligin 4 (NLGN4) gene. None of the boys with the typical deletion or presumed point mutations of STS demonstrated autistic difficulties.
Conclusions: STS deficiency may be a risk factor for ADHD with predominantly inattentive symptoms. Boys with XLI and large deletions encompassing STS and NLGN4 are at increased risk of developing autism and related disorders.
C1 [Weisblatt, E.] Univ Cambridge, Dept Expt Psychol, Cambridge CB2 1TN, England.
[Pasco, G.] Univ Cambridge, Autism Res Ctr, Cambridge CB2 1TN, England.
[Willatt, L. R.; McMahon, R.; Yates, J. R. W.] Addenbrookes Hosp, Med Genet Serv, Cambridge, England.
[Yates, J. R. W.] Univ Cambridge, Dept Med Genet, Cambridge CB2 1TN, England.
RP Kent, L (reprint author), Univ St Andrews, Bute Med Sch, St Andrews KY16 9TS, Fife, Scotland.
EM lsk8@st-andrews.ac.uk
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NR 29
TC 56
Z9 56
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0022-2593
J9 J MED GENET
JI J. Med. Genet.
PD AUG
PY 2008
VL 45
IS 8
BP 519
EP 524
DI 10.1136/jmg.2008.057729
PG 6
WC Genetics & Heredity
SC Genetics & Heredity
GA 333HK
UT WOS:000258143400006
PM 18413370
ER
PT J
AU He, Y
Casaccia-Bonnefil, P
AF He, Ye
Casaccia-Bonnefil, Patrizia
TI The Yin and Yang of YY1 in the nervous system
SO JOURNAL OF NEUROCHEMISTRY
LA English
DT Article
DE brain; chromatin; histone deacetylase; neuron; oligodendrocyte
ID TRANSCRIPTION FACTOR YY1; AMYLOID PRECURSOR PROTEIN; DNA-BINDING
PROTEIN; OLIGODENDROCYTE PROGENITOR DIFFERENTIATION;
AUTISM-SPECTRUM-DISORDER; BETA-HYDROXYLASE GENE; POLYCOMB GROUP GENE;
FACTOR YIN-YANG-1; NEURAL CREST; ALZHEIMERS-DISEASE
AB The transcription factor Yin Yang 1 (YY1) is a multifunctional protein that can activate or repress gene expression depending on the cellular context. YY1 is ubiquitously expressed and highly conserved between species. However, its role varies in diverse cell types and includes proliferation, differentiation, and apoptosis. This review will focus on the function of YY1 in the nervous system including its role in neural development, neuronal function, developmental myelination, and neurological disease. The multiple functions of YY1 in distinct cell types are reviewed and the possible mechanisms underlying the cell specificity for these functions are discussed.
C1 [He, Ye; Casaccia-Bonnefil, Patrizia] Univ Med & Dent New Jersey, Dept Neurosci & Cell Biol, Robert Wood Johnson Med Sch, Piscataway, NJ 08854 USA.
RP He, Y (reprint author), Mt Sinai Sch Med, Dept Neurosci & Genet & Genom, 1 Gustave Levy Pl Box 1065, New York, NY 10029 USA.
EM heye@umdnj.edu; patrizia.casaccia@mssm.edu
RI He, Ye/G-7500-2011
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NR 95
TC 42
Z9 44
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0022-3042
J9 J NEUROCHEM
JI J. Neurochem.
PD AUG
PY 2008
VL 106
IS 4
BP 1493
EP 1502
DI 10.1111/j.1471-4159.2008.05486.x
PG 10
WC Biochemistry & Molecular Biology; Neurosciences
SC Biochemistry & Molecular Biology; Neurosciences & Neurology
GA 333RQ
UT WOS:000258170400002
PM 18485096
ER
PT J
AU Harrison, NA
Martino, BD
Knafo, S
Bird, G
Critchley, HD
Dolan, RJ
AF Harrison, N. A.
Martino, B. D.
Knafo, S.
Bird, G.
Critchley, H. D.
Dolan, R. J.
TI Hyper-rationality and behavioural invariance in autism
SO JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY
LA English
DT Meeting Abstract
CT 21st Annual Meeting of the British-Neuropsychiatry-Association
CY FEB 07-08, 2008
CL London, ENGLAND
SP British Neuropsychiat Assoc
HO Inst Child Health
EM n.harrison@fil.ion.ucl.ac.uk
RI critchley, hugo/G-9267-2011
NR 0
TC 0
Z9 0
PU B M J PUBLISHING GROUP
PI LONDON
PA BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND
SN 0022-3050
J9 J NEUROL NEUROSUR PS
JI J. Neurol. Neurosurg. Psychiatry
PD AUG
PY 2008
VL 79
IS 8
MA 15
BP 972
EP 972
PG 1
WC Clinical Neurology; Psychiatry; Surgery
SC Neurosciences & Neurology; Psychiatry; Surgery
GA 328KF
UT WOS:000257796600045
ER
PT J
AU Moore, C
AF Moore, Charlotte
TI Thoughts About the Autism Label: A Parental View
SO JOURNAL OF PHILOSOPHY OF EDUCATION
LA English
DT Article
AB The number of people diagnosed with autism has risen exponentially in recent years. Are the diagnostic labels currently in use adequate to describe such a vast range of symptoms? Should we reconsider the appropriateness of the language we use to discuss autism? A mother of two autistic sons describes what the autism label has meant for her and her family.
EM csmoore@csmoore.freeserve.co.uk
CR Baron-Cohen S., 2003, ESSENTIAL DIFFERENCE
NR 1
TC 0
Z9 0
PU WILEY-BLACKWELL PUBLISHING, INC
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0309-8249
J9 J PHILOS EDUC
JI J. Philos. Educ.
PD AUG-NOV
PY 2008
VL 42
IS 3-4
BP 493
EP 498
DI 10.1111/j.1467-9752.2008.00652.x
PG 6
WC Education & Educational Research; History Of Social Sciences
SC Education & Educational Research; Social Sciences - Other Topics
GA 397PD
UT WOS:000262673700012
ER
PT J
AU Simonoff, E
Pickles, A
Charman, T
Chandler, S
Loucas, T
Baird, G
AF Simonoff, Emily
Pickles, Andrew
Charman, Tony
Chandler, Susie
Loucas, Tom
Baird, Gillian
TI Psychiatric disorders in children with autism spectrum disorders:
Prevalence, comorbidity, and associated factors in a population-derived
sample
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Article
DE autism; child psychiatric disorders; prevalence; Special Needs and
Autism Project
ID PERVASIVE DEVELOPMENTAL DISORDERS; DEFICIT-HYPERACTIVITY DISORDER;
INTELLECTUAL DISABILITY; YOUNG-ADULTS; DIAGNOSTIC INTERVIEW;
MENTAL-RETARDATION; PSYCHOPATHOLOGY; SYMPTOMS; ADOLESCENTS; PEOPLE
AB Objective: Autism spectrum disorders are now recognized to occur in up to 1% of the population and to be a major public health concern because of their early onset, lifelong persistence, and high levels of associated impairment. Little is known about the associated psychiatric disorders that may contribute to impairment. We identify the rates and type of psychiatric comorbidity associated with ASDs and explore the associations with variables identified as risk factors for child psychiatric disorders. Method: A subgroup of 112 ten- to 14-year old children from a population-derived cohort was assessed for other child psychiatric disorders (3 months' prevalence) through parent interview using the Child and Adolescent Psychiatric Assessment. DSM-IV diagnoses for childhood anxiety disorders, depressive disorders, oppositional defiant and conduct disorders, attention-deficit/hyperactivity disorder, tic disorders, trichotillomania, enuresis, and encopresis were identified. Results: Seventy percent of participants had at least one comorbid disorder and 41% had two or more. The most common diagnoses were social anxiety disorder (29.2%, 95% confidence interval [CI)] 13.2-45.1), attention-deficit/hyperactivity disorder (28.2%, 95% CI 13.3-43.0), and oppositional defiant disorder (28.1%, 95% CI 13.9-42.2). Of those with attention/deficit/hyperactivity disorder, 84% received a second comorbid diagnosis. There were few associations between putative risk factors and psychiatric disorder. Conclusions: Psychiatric disorders are common and frequently multiple in children with autism spectrum disorders. They may provide targets for intervention and should be routinely evaluated in the clinical assessment of this group.
C1 [Simonoff, Emily] Kings Coll London, Dept Child & Adolescent Psychiat, Inst Psychiat, London SE5 8AF, England.
[Pickles, Andrew] Univ Manchester, Div Epidemiol & Hlth Sci, Manchester M13 9PL, Lancs, England.
[Charman, Tony; Chandler, Susie] UCL, Inst Child Hlth, London WC1E 6BT, England.
[Loucas, Tom] Univ Reading, Sch Psychol & Clin Language Sci, Reading RG6 2AH, Berks, England.
RP Simonoff, E (reprint author), Kings Coll London, Dept Child & Adolescent Psychiat, Inst Psychiat, Crespigay Pk, London SE5 8AF, England.
EM e.simonoff@iop.kcl.ac.uk
RI Pickles, Andrew/A-9625-2011; Simonoff, Emily/B-7593-2011; Charman,
Tony/A-2085-2014
OI Pickles, Andrew/0000-0003-1283-0346; Charman, Tony/0000-0003-1993-6549
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NR 49
TC 473
Z9 474
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD AUG
PY 2008
VL 47
IS 8
BP 921
EP 929
DI 10.1097/CHI.0b013e318179964f
PG 9
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 331WR
UT WOS:000258043800013
PM 18645422
ER
PT J
AU Goodlin-Jones, BL
Tang, K
Liu, JY
Anders, TF
AF Goodlin-Jones, Beth L.
Tang, Karen
Liu, Jingyi
Anders, Thomas F.
TI Sleep patterns in preschool-age children with autism, developmental
delay, and typical development
SO JOURNAL OF THE AMERICAN ACADEMY OF CHILD AND ADOLESCENT PSYCHIATRY
LA English
DT Article
DE sleep; preschool children; autism; developmental disability; actigraphy;
neurodevelopment
ID HABITS QUESTIONNAIRE; ASPERGERS-DISORDER; PREVALENCE; ACTIGRAPHY;
SEVERITY; ISSUES
AB Objective: A prominent noncore symptom of autistic disorder is disturbed sleep, but relatively few studies have investigated this symptom. Method: A multimethod approach assessed the quantity and quality of sleep in 194 children (68 with autism [AUT], 57 with developmental delay without autism [DD], 69 with typical development) recorded over 1 week. Parent perceptions, structured questionnaires, and actigraphy were compared. In addition, problem sleep as defined by parents was compared with research diagnostic criteria for behavioral insomnia obtained from actigraph recordings. Results: On actigraphy, children in the DID group, after sleep onset, exhibited more and longer awakenings than the other two groups. In contrast, children in the AUT group exhibited less total sleep time in 24 hours than the other two groups. Parent reports of sleep problems were higher in the AUT and DD groups than the typical development group, but parent reports did not concur with more objective RDC for behavioral insomnia. Parent reports of sleep problems in all of the groups were significantly associated with increased self-reports of stress. Total 24-hour sleep durations for all of the groups were shorter than recommended for preschool-age children. Conclusions: Our study provides objective evidence that sleep patterns are different in preschool children across the categories of AUT, DD, or typical development.
C1 [Goodlin-Jones, Beth L.; Tang, Karen; Anders, Thomas F.] Univ Calif Davis, Dept Psychiat & Behav Sci, Davis MIND Inst, Davis, CA 95616 USA.
[Liu, Jingyi] Univ Calif Davis, Dept Stat, Davis, CA 95616 USA.
RP Anders, TF (reprint author), 2825 50th St, Sacramento, CA USA.
EM tfanders@ucdavis.edu
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NR 48
TC 41
Z9 41
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0890-8567
J9 J AM ACAD CHILD PSY
JI J. Am. Acad. Child Adolesc. Psychiatr.
PD AUG
PY 2008
VL 47
IS 8
BP 930
EP 938
DI 10.1097/CHI.0b013e3181799f7c
PG 9
WC Psychology, Developmental; Pediatrics; Psychiatry
SC Psychology; Pediatrics; Psychiatry
GA 331WR
UT WOS:000258043800014
PM 18596550
ER
PT J
AU Lockner, DW
Crowe, TK
Skipper, BJ
AF Lockner, Donna W.
Crowe, Terry K.
Skipper, Betty J.
TI Dietary intake and parents' perception of mealtime behaviors in
preschool-age children with autism spectrum disorder and in typically
developing children
SO JOURNAL OF THE AMERICAN DIETETIC ASSOCIATION
LA English
DT Article
ID NUTRIENT INTAKE; UNITED-STATES; FOOD; PREVALENCE; TODDLERS; INFANTS
AB Parents of children with autism spectrum disorder (ASD) frequently report that their children have selective eating behaviors and refuse many foods, which could result in inadequate nutrient intake. This preliminary cross-sectional descriptive study investigated dietary intake and parents' reported perception of food behaviors of 20 3- to 5-year-old children with ASD. Twenty typically developing children matched for sex, age, and ethnicity were also studied as a case-control comparison. Nutrient intake determined from 3-day food records was adjusted for day-to-day variation to determine the estimate of usual intake distribution for the two groups. This distribution was compared with the Estimated Average Requirement or Adequate Intake recommendations. The reported food behaviors and use of vitamin or mineral supplements were compared for matched pairs using the exact McNemar test. Nutrient intake was similar for both groups of children, with the majority of children consuming more than the recommended amounts for most nutrients. Nutrients least likely to be consumed in recommended amounts were vitamin A, vitamin E, fiber, and calcium. Children with ASD were more likely to consume vitamin/mineral supplements than typically developing children. Compared with parents of typically developing children, parents of children with ASD were more likely to report that their children were picky eaters and resisted trying new foods, and they were less likely to describe their children as healthy eaters or that they eat a variety of foods. Despite the similar and generally adequate nutrient intake for the 40 children in this study, parents of children with ASD had more negative perceptions of their children's dietary behaviors.
C1 [Lockner, Donna W.] Univ New Mexico, Coll Educ, Nutr Program, Dept Individual Family & Community Educ, Albuquerque, NM 87131 USA.
[Skipper, Betty J.] Univ New Mexico, Sch Med, Dept Family & Community Med, Albuquerque, NM 87131 USA.
[Crowe, Terry K.] Univ New Mexico, Sch Med, Dept Pediat, Div Occupat Therapy, Albuquerque, NM 87131 USA.
RP Lockner, DW (reprint author), Univ New Mexico, Coll Educ, Nutr Program, Dept Individual Family & Community Educ, MSC05 3040, Albuquerque, NM 87131 USA.
EM dlockner@unm.edu
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NR 20
TC 20
Z9 21
PU AMER DIETETIC ASSOC
PI CHICAGO
PA 216 W JACKSON BLVD #800, CHICAGO, IL 60606-6995 USA
SN 0002-8223
J9 J AM DIET ASSOC
JI J. Am. Diet. Assoc.
PD AUG
PY 2008
VL 108
IS 8
BP 1360
EP 1363
DI 10.1016/j.jada.2008.05.003
PG 4
WC Nutrition & Dietetics
SC Nutrition & Dietetics
GA 335KZ
UT WOS:000258290800016
PM 18656577
ER
PT J
AU Seeman, C
AF Seeman, Carey
TI The way I see it: A personal look at autism and asperger's
SO LIBRARY JOURNAL
LA English
DT Book Review
C1 [Seeman, Carey] Univ Michigan, Kresge Business Adm Lib, Ann Arbor, MI 48109 USA.
RP Seeman, C (reprint author), Univ Michigan, Kresge Business Adm Lib, Ann Arbor, MI 48109 USA.
CR Grandin T., 2008, WAY SEE IT PERSONAL
NR 1
TC 0
Z9 0
PU REED BUSINESS INFORMATION
PI NEW YORK
PA 360 PARK AVENUE SOUTH, NEW YORK, NY 10010 USA
SN 0363-0277
J9 LIBR J
JI Libr. J.
PD AUG
PY 2008
VL 133
IS 13
BP 104
EP 104
PG 1
WC Information Science & Library Science
SC Information Science & Library Science
GA 341LB
UT WOS:000258714900245
ER
PT J
AU Chamak, B
AF Chamak, Brigitte
TI Autism and social movements: French parents' associations and
international autistic individuals' organizations
SO M S-MEDECINE SCIENCES
LA French
DT Article
RP Chamak, B (reprint author), Univ Paris 05, CESAMES Ctr Rech Psychotropes, CNRS, UMR 8136,Inserm U611, 45 Rue St Peres, F-75270 Paris 06, France.
EM brigitte.chamak@paris5.sorbonne.fr
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NR 14
TC 1
Z9 1
PU MASSON EDITEUR
PI MOULINEAUX CEDEX 9
PA 21 STREET CAMILLE DESMOULINS, ISSY, 92789 MOULINEAUX CEDEX 9, FRANCE
SN 0767-0974
J9 M S-MED SCI
JI M S-Med. Sci.
PD AUG-SEP
PY 2008
VL 24
IS 8-9
BP 768
EP 770
DI 10.1051/medsci/20082489768
PG 3
WC Medicine, Research & Experimental
SC Research & Experimental Medicine
GA 353LL
UT WOS:000259569100025
PM 18789226
ER
PT J
AU Rout, UK
Dhossche, DM
AF Rout, Ujjwal K.
Dhossche, Dirk M.
TI A pathogenetic model of autism involving Purkinje cell loss through
anti-GAD antibodies
SO MEDICAL HYPOTHESES
LA English
DT Article
ID GLUTAMIC-ACID DECARBOXYLASE; STIFF-MAN SYNDROME; SPECTRUM DISORDERS;
DIABETES-MELLITUS; BRAIN; CEREBELLAR; CHILDREN; AUTOANTIBODIES;
ACTIVATION; EPILEPSY
AB Autism is a medical enigma, lacking truly effective treatments. Both genetics and environmental factors are recognized as players in the development of autism spectrum disorders (ASDs). Nevertheless, the exact mechanism(s) for the development of ASDs is (are) not known primarily because current understanding about the etiology of the disease is limited. Selective loss of Purkinje cells and the cerebellar atrophies are the neurological abnormalities most consistently found in persons diagnosed with autism. Because Purkinje cells are involved in motor coordination, working memory and learning, toss of these cells are likely to cause symptoms defining behavioral parameters of ASD. Currently the mechanism(s) for the loss of Purkinje cells in the cerebella of autistic individual is (are) not understood. Here we postulate a hypothesis for the development of autistic symptoms, severity of which is based on the extent of Purkinje cell loss triggered by Glutamate acid decarboxylase antibody (GAD-Ab). This model accommodates any genetic basis of autism and immunogenic triggers resulting GAD-Ab in the blood of the mother while pregnant with the child diagnosed autistic after birth or of an individual diagnosed with autism some time in the life time. Identification and characterization of GAD-Abs from pregnant mothers with a family history of autism, from children with autistic siblings, and individuals diagnosed with autism may allow find preventive and new therapeutic avenues. (C) 2007 Published by Elsevier Ltd.
C1 [Rout, Ujjwal K.] Univ Mississippi, Med Ctr, Dept Surg, Ctr Psychiat Neurosci, Jackson, MS 39216 USA.
[Rout, Ujjwal K.] Univ Mississippi, Med Ctr, Dept Physiol & Biophys, Ctr Psychiat Neurosci, Jackson, MS 39216 USA.
[Dhossche, Dirk M.] Univ Mississippi, Med Ctr, Dept Psychiat & Human Behav, Jackson, MS 39216 USA.
RP Rout, UK (reprint author), Univ Mississippi, Med Ctr, Dept Surg, Ctr Psychiat Neurosci, Jackson, MS 39216 USA.
EM urout@umsmed.edu
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NR 31
TC 13
Z9 13
PU CHURCHILL LIVINGSTONE
PI EDINBURGH
PA JOURNAL PRODUCTION DEPT, ROBERT STEVENSON HOUSE, 1-3 BAXTERS PLACE,
LEITH WALK, EDINBURGH EH1 3AF, MIDLOTHIAN, SCOTLAND
SN 0306-9877
J9 MED HYPOTHESES
JI Med. Hypotheses
PD AUG
PY 2008
VL 71
IS 2
BP 218
EP 221
DI 10.1016/j.mehy.2007.11.012
PG 4
WC Medicine, Research & Experimental
SC Research & Experimental Medicine
GA 331LN
UT WOS:000258013800009
PM 18514431
ER
PT J
AU Holst, MI
Maercker, C
Pintea, B
Masseroli, M
Liebig, C
Jankowski, J
Miething, A
Martini, J
Schwaller, B
Oberdick, J
Schilling, K
Baader, SL
AF Holst, Martin I.
Maercker, Christian
Pintea, Bogdan
Masseroli, Marco
Liebig, Christian
Jankowski, Jakob
Miething, Andreas
Martini, Julia
Schwaller, Beat
Oberdick, John
Schilling, Karl
Baader, Stephan L.
TI Engrailed-2 regulates genes related to vesicle formation and transport
in cerebellar Purkinje cells
SO MOLECULAR AND CELLULAR NEUROSCIENCE
LA English
DT Article
DE cell polarity; cerebellum; cholecystokinin; Mtss1/mim; polarity;
tetraspanin; autism
ID DIFFERENTIALLY EXPRESSED GENES; HOMEOBOX-TRANSCRIPTION-FACTOR;
AUTISM-SPECTRUM-DISORDER; MICROARRAY DATA; STATISTICAL-ANALYSIS;
NERVOUS-SYSTEM; PROTEIN; OXYTOCIN; NEURONS; MICE
AB Engrailed transcription factors regulate survival, cell fate decisions and axon pathfinding in central neurons. En-2 can also attenuate Purkinje cell (PC) maturation. Here, we use array analysis to scrutinize gene expression in developing PCs overexpressing Engrailed-2 (L7En-2). The majority (70%) of regulated genes was found down-regulated in L7En-2 cerebella, consistent with the known repressive function of Engrailed-2. Differential gene expression, verified by in situ hybridization or Western blotting, was particularly evident during the first postnatal week, when L7En-2 PCs display conspicuous deficits in dendritogenesis. Functional classification revealed clusters of genes linked to vesicle formation and transport. Consistently, Golgi stacks located at the axonal pole of wild type PC somata were rarely detected in L7En-2 PCs. In addition, long continuous stretches of endoplasmic reticulum typically found around the axonal pole of wild type PCs were less frequently observed in transgenic cells. Engrailed-2 might therefore orchestrate PC survival and process formation as a regulator of subcellular organization. (C) 2008 Elsevier Inc. All rights reserved.
C1 [Holst, Martin I.; Pintea, Bogdan; Liebig, Christian; Jankowski, Jakob; Martini, Julia; Schilling, Karl; Baader, Stephan L.] Univ Bonn, Inst Anat Anat & Cell Biol, D-53115 Bonn, Germany.
[Maercker, Christian] German Canc Res Ctr, Genom & Prote Core Facil, D-6900 Heidelberg, Germany.
[Maercker, Christian] Mannheim Univ Appl Sci, Mannheim, Germany.
[Masseroli, Marco] Politecn Milan, Dept Elect & Informat, I-20133 Milan, Italy.
[Miething, Andreas] Univ Bonn, Inst Anat, D-53115 Bonn, Germany.
[Schwaller, Beat] Univ Fribourg, Unit Anat, Dept Med, CH-1700 Fribourg, Switzerland.
[Oberdick, John] Ohio State Univ, Coll Med, Dept Neurosci, Columbus, OH 43210 USA.
[Oberdick, John] Ohio State Univ, Coll Med, Ctr Mol Neurobiol, Columbus, OH 43210 USA.
[Baader, Stephan L.] Univ Jena, Inst Anat 1, D-07743 Jena, Germany.
RP Baader, SL (reprint author), Univ Bonn, Inst Anat Anat & Cell Biol, Nussallee 10, D-53115 Bonn, Germany.
EM s.baader@uni-bonn.de
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NR 77
TC 9
Z9 10
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1044-7431
J9 MOL CELL NEUROSCI
JI Mol. Cell. Neurosci.
PD AUG
PY 2008
VL 38
IS 4
BP 495
EP 504
DI 10.1016/j.mcn.2008.04.010
PG 10
WC Neurosciences
SC Neurosciences & Neurology
GA 336EO
UT WOS:000258347300005
PM 18562208
ER
PT J
AU Howlin, P
AF Howlin, Patricia
TI Redressing the balance in autism research
SO NATURE CLINICAL PRACTICE NEUROLOGY
LA English
DT Editorial Material
C1 Kings Coll London, Inst Psychiat, London WC2R 2LS, England.
RP Howlin, P (reprint author), Kings Coll London, Inst Psychiat, London WC2R 2LS, England.
RI Howlin, Patricia/A-7622-2011
NR 0
TC 5
Z9 5
PU NATURE PUBLISHING GROUP
PI NEW YORK
PA 75 VARICK STREET, 9TH FLOOR, NEW YORK, NY 10013-1917 USA
SN 1745-834X
J9 NAT CLIN PRACT NEURO
JI Nat. Clin. Pract. Neurol.
PD AUG
PY 2008
VL 4
IS 8
BP 407
EP 407
DI 10.1038/ncpneuro0860
PG 1
WC Clinical Neurology
SC Neurosciences & Neurology
GA 332TM
UT WOS:000258106000001
PM 18679169
ER
PT J
AU Ehninger, D
Han, S
Shilyansky, C
Zhou, Y
Li, WD
Kwiatkowski, DJ
Ramesh, V
Silva, AJ
AF Ehninger, Dan
Han, Sangyeul
Shilyansky, Carrie
Zhou, Yu
Li, Weidong
Kwiatkowski, David J.
Ramesh, Vijaya
Silva, Alcino J.
TI Reversal of learning deficits in a Tsc2(+/-) mouse model of tuberous
sclerosis
SO NATURE MEDICINE
LA English
DT Article
ID SYNAPTIC PLASTICITY; COGNITIVE DEFICITS; RAT MODEL; FRAGILE-X; COMPLEX;
MEMORY; HIPPOCAMPUS; TSC1; TRANSLATION; IDENTIFICATION
AB Tuberous sclerosis is a single-gene disorder caused by heterozygous mutations in the TSC1 (9q34) or TSC2 (16p13.3) gene(1,2) and is frequently associated with mental retardation, autism and epilepsy. Even individuals with tuberous sclerosis and a normal intelligence quotient (approximately 50%)(3-5) are commonly affected with specific neuropsychological problems, including long-term and working memory deficits(6,7). Here we report that mice with a heterozygous, inactivating mutation in the Tsc2 gene (Tsc2(+/-) mice)(8) show deficits in learning and memory. Cognitive deficits in Tsc2(+/-) mice emerged in the absence of neuropathology and seizures, demonstrating that other disease mechanisms are involved(5,9-11). We show that hyperactive hippocampal mammalian target of rapamycin (mTOR) signaling led to abnormal long-term potentiation in the CA1 region of the hippocampus and consequently to deficits in hippocampal-dependent learning. These deficits included impairments in two spatial learning tasks and in contextual discrimination. Notably, we show that a brief treatment with the mTOR inhibitor rapamycin in adult mice rescues not only the synaptic plasticity, but also the behavioral deficits in this animal model of tuberous sclerosis. The results presented here reveal a biological basis for some of the cognitive deficits associated with tuberous sclerosis, and they show that treatment with mTOR antagonists ameliorates cognitive dysfunction in a mouse model of this disorder.
C1 [Ehninger, Dan; Shilyansky, Carrie; Zhou, Yu; Li, Weidong; Silva, Alcino J.] Univ Calif Los Angeles, Dept Neurobiol, Los Angeles, CA 90095 USA.
[Ehninger, Dan; Shilyansky, Carrie; Zhou, Yu; Li, Weidong; Silva, Alcino J.] Univ Calif Los Angeles, Dept Psychiat & Biobehav Sci, Los Angeles, CA 90095 USA.
[Ehninger, Dan; Shilyansky, Carrie; Zhou, Yu; Li, Weidong; Silva, Alcino J.] Univ Calif Los Angeles, Dept Psychol, Los Angeles, CA 90095 USA.
[Ehninger, Dan; Shilyansky, Carrie; Zhou, Yu; Li, Weidong; Silva, Alcino J.] Univ Calif Los Angeles, Brain Res Inst, Los Angeles, CA 90095 USA.
[Han, Sangyeul; Ramesh, Vijaya] Harvard Univ, Sch Med, Massachusetts Gen Hosp, Ctr Human Genet Res,Richard B Simches Res Ctr, Boston, MA 02114 USA.
[Kwiatkowski, David J.] Harvard Univ, Brigham & Womens Hosp, Sch Med, Genet Lab,Div Translat Med, Boston, MA 02115 USA.
RP Silva, AJ (reprint author), Univ Calif Los Angeles, Dept Neurobiol, 695 Charles E Young Dr S, Los Angeles, CA 90095 USA.
EM silvaa@mednet.ucla.edu
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NR 30
TC 323
Z9 333
PU NATURE PUBLISHING GROUP
PI NEW YORK
PA 75 VARICK ST, 9TH FLR, NEW YORK, NY 10013-1917 USA
SN 1078-8956
J9 NAT MED
JI Nat. Med.
PD AUG
PY 2008
VL 14
IS 8
BP 843
EP 848
DI 10.1038/nm1788
PG 6
WC Biochemistry & Molecular Biology; Cell Biology; Medicine, Research &
Experimental
SC Biochemistry & Molecular Biology; Cell Biology; Research & Experimental
Medicine
GA 334QY
UT WOS:000258237500031
PM 18568033
ER
PT J
AU Hrdlicka, M
AF Hrdlicka, Michal
TI EEG abnormalities, epilepsy and regression in autism: A review
SO NEUROENDOCRINOLOGY LETTERS
LA English
DT Review
DE autism; EEG abnormalities; epilepsy; autistic regression, mental
retardation
ID PERVASIVE DEVELOPMENTAL DISORDERS; SPECTRUM DISORDERS; CHILDHOOD AUTISM;
INFANTILE-AUTISM; HOME VIDEOTAPES; ETIOLOGIC YIELD; CHILDREN;
ELECTROENCEPHALOGRAMS; SYMPTOMS
AB Autism is associated with a high frequency of epileptiform EEG abnormalities (prevalence range 10.3-72.4%) and epilepsy (prevalence range 0-44.5%). A significant subgroup of autistic children (20-49%) experience autistic regression. The relationship among EEG abnormalities, epilepsy, and regression in autistic patients is not yet well understood. In this review, the current knowledge of the relationship is summarized. The evidence from clinical studies does not support the view that EEG abnormalities play a role in autistic regression. The majority of studies also failed to find any significant relationship between epilepsy and autistic regression. However, some results indicated that the higher the prevalence of epilepsy in the sample, the greater the probability of there being a significant association between epilepsy and autistic regression. Further research on the topic is needed.
C1 [Hrdlicka, Michal] Charles Univ Prague, Fac Med 2, Dept Child Psychiat, Prague 15006, Czech Republic.
[Hrdlicka, Michal] Charles Univ Prague, Fac Med 1, Prague 12108, Czech Republic.
RP Hrdlicka, M (reprint author), Charles Univ Prague, Fac Med 2, Dept Child Psychiat, V Uvalu 84, Prague 15006, Czech Republic.
EM michal.hrdlicka@lfmotol.cuni.cz
FU [MSM 0021620849]; [FNM MZ000064203]
FX Supported by grants MSM 0021620849 and FNM MZ000064203.
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NR 48
TC 6
Z9 6
PU MAGHIRA & MAAS PUBLICATIONS
PI STOCKHOLM
PA PO BOX 26132, S-100 41 STOCKHOLM, SWEDEN
SN 0172-780X
J9 NEUROENDOCRINOL LETT
JI Neuroendocrinol. Lett.
PD AUG
PY 2008
VL 29
IS 4
BP 405
EP 409
PG 5
WC Endocrinology & Metabolism; Neurosciences
SC Endocrinology & Metabolism; Neurosciences & Neurology
GA 356JJ
UT WOS:000259774200003
PM 18766157
ER
PT J
AU Kulisek, R
Hrncir, Z
Hrdlicka, M
Faladova, L
Sterbova, K
Krsek, P
Vymlatilova, E
Palus, M
Zumrova, A
Komarek, V
AF Kulisek, Robert
Hrncir, Zbynek
Hrdlicka, Michal
Faladova, Ludvika
Sterbova, Katalin
Krsek, Pavel
Vymlatilova, Eva
Palus, Milan
Zumrova, Alena
Komarek, Vladimir
TI Nonlinear analysis of the sleep EEG in children with pervasive
developmental disorder
SO NEUROENDOCRINOLOGY LETTERS
LA English
DT Article
DE autism; EEG analysis; sleep; synchronization; connectivity
ID CORPUS-CALLOSUM; NEURAL SYSTEMS; CORTICAL CONNECTIVITY; SPECTRUM
DISORDERS; ASPERGER-SYNDROME; AUTISM PHENOTYPE; BRAIN; SYNCHRONIZATION;
RECORDINGS; ARCHITECTURE
AB OBJECTIVES: Autism is a severe neurodevelopmental disorder with a high rate of epilepsy and subclinical epileptiform activity. High physical connectivity on a microcolumnar level leading to epileptiform activity and low functional informational connectivity are assumed in autism. The aim of this study was to investigate nonlinear EEG brain dynamics in terms of synchronization in a group of children with autism spectrum disorders compared to a control group. We expected a lower degree of synchronization in autistic subjects.
METHODS: The autistic group consisted of 27 patients with autism spectrum disorders diagnosed according to ICD-10. The mean age of the sample was 7.1 (SD 3.6) years, 14 of them were mentally retarded. Normal EEG was found in 9 patients, epileptiform EEG in 18 autistic patients. Four patients had a history of epileptic seizures, fully compensated in long term. The control group consisted of 20 children (mean age of 8.4, SD 2.3 years) with normal intelligence, without an epileptic history, investigated within the frame of the research program for cochlear implantation. They had normal neurological examination and suffered from perceptive deafness. Normal EEG was found in 17 of the control subjects, epileptiform EEG was in 3 control subjects. We analyzed night sleep EEG recordings from 10 channels (F3, F4, F7, F8, C3, C4, T3, T4, P3 and P4) with the inclusion of sleep stages NREM 2, 3 and 4 in the subsequent analyses. Coarse-grained entropy information rates between neighbouring electrodes were computed, expressing the synchronization between 11 selected electrode couples.
RESULTS: Synchronization was significantly lower in the autistic group in all three examined NREM stages even when age and intelligence were taken into account as covariates.
CONCLUSIONS: The results of the study confirmed the validity of the underconnectivity model in autism.
C1 [Kulisek, Robert; Hrdlicka, Michal] Charles Univ Prague, Fac Med 2, Dept Child Psychiat, Prague 15006, Czech Republic.
[Hrncir, Zbynek; Faladova, Ludvika; Sterbova, Katalin; Krsek, Pavel; Zumrova, Alena; Komarek, Vladimir] Charles Univ Prague, Fac Med 2, Dept Child Neurol, Prague 15006, Czech Republic.
[Vymlatilova, Eva] Charles Univ Prague, Fac Med 2, Dept Otorhinolaryngol, Prague 15006, Czech Republic.
[Palus, Milan] Acad Sci Czech Republic, Inst Comp Sci, Prague, Czech Republic.
[Hrdlicka, Michal] Charles Univ Prague, Fac Med 1, Prague 12108, Czech Republic.
RP Kulisek, R (reprint author), Charles Univ Prague, Fac Med 2, Dept Child Psychiat, V Uvalu 84, Prague 15006, Czech Republic.
EM robertkulisek@seznam.cz
RI Palus, Milan/I-6899-2012
OI Palus, Milan/0000-0001-8474-1436
FU Faculty Hospital Motol [FNM V2 MZO 000 64203-6504]; MSM [0021620849]
FX This study was supported by the Research project of the Faculty Hospital
Motol FNM V2 MZO 000 64203-6504 and by the Research project MSM
0021620849.
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NR 46
TC 9
Z9 9
PU MAGHIRA & MAAS PUBLICATIONS
PI STOCKHOLM
PA PO BOX 26132, S-100 41 STOCKHOLM, SWEDEN
SN 0172-780X
J9 NEUROENDOCRINOL LETT
JI Neuroendocrinol. Lett.
PD AUG
PY 2008
VL 29
IS 4
BP 512
EP 517
PG 6
WC Endocrinology & Metabolism; Neurosciences
SC Endocrinology & Metabolism; Neurosciences & Neurology
GA 356JJ
UT WOS:000259774200022
PM 18766147
ER
PT J
AU Oslejskova, H
Dusek, L
Makovska, Z
Pejcochova, J
Autrata, R
Slapak, I
AF Oslejskova, Hana
Dusek, Ladislav
Makovska, Zuzana
Pejcochova, Jana
Autrata, Rudolf
Slapak, Ivo
TI Complicated relationship between autism with regression and epilepsy
SO NEUROENDOCRINOLOGY LETTERS
LA English
DT Article
DE pervasive developmental disorders; autism with regression; epilepsy;
epileptiform abnormalities in EEG; IQ; functionality of autism;
Landau-Kleffner syndrome
ID SPECTRUM DISORDERS; PEDIATRIC EPILEPSY; CHILDHOOD AUTISM;
SEX-DIFFERENCES; RISK-FACTORS; CHILDREN; PSYCHOPATHOLOGY;
CLASSIFICATION; ABNORMALITIES; DIAGNOSIS
AB We retrospectively evaluated a set of 205 children with autism and compared it to the partial sub-set of 71 (34.6%) children with a history of regression. From 71 children with regression, signs of epileptic processes were present in 43 (60.6%), 28 (65.12%) suffered clinical epileptic seizures, and 15 (34.9%) just had an epileptiform abnormality on the EEG. In our analysis, autistic regression is substantially more associated with epileptic process symptoms than in children with autism and no history of regression. More than 90% of children with a history of regression also show IQ < 70 and reduced functionality. Functionality and IQ further worsens with the occurrence of epileptic seizures (98% of children with regression and epilepsy have IQ < 70). We proved that low IQ and reduced functionality significantly correlate rather with epileptic seizures than just sub-clinical epileptiform abnormality on EEG.
Clinical epileptic seizures associated with regression significantly influence the age of regression and its clinical type. The age of regression is higher compared to children with regression without epileptic seizures (in median: 35 months of age in patients with seizures while only 24 months in other patients). Patients with seizures revealed regression after 24(th) months of age in 68% of cases, while patients without seizures only in 27%. However, coincidence with epilepsy also increased the occurrence of regression before the 18(th) month of age (23% of patients), while only 4% of patients without epilepsy revealed regression before the 18th month. Epileptic seizures are significantly associated especially with behaviour regression rather than speech regression or regression in both behaviour and speech. Also epileptic seizures diagnosed before correct diagnosis of autism were significantly associated with delayed regression (both behavioural and speech regression).
C1 [Oslejskova, Hana] Univ Hosp, Childrens Med Ctr, Dept Paediat Neurol, Brno 62500, Czech Republic.
[Dusek, Ladislav] Masaryk Univ, Inst Biostat & Anal, Brno 62500, Czech Republic.
[Autrata, Rudolf] Univ Hosp, Dept Paediat Ophthalmol, Brno 62500, Czech Republic.
[Slapak, Ivo] Univ Hosp, Dept Paediat Otorhinolaryngol, Brno 62500, Czech Republic.
RP Oslejskova, H (reprint author), Univ Hosp, Childrens Med Ctr, Dept Paediat Neurol, Brno 62500, Czech Republic.
EM hoslej@fnbrno.cz
RI Dusek, Ladislav/G-8794-2013
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NR 58
TC 9
Z9 9
PU MAGHIRA & MAAS PUBLICATIONS
PI STOCKHOLM
PA PO BOX 26132, S-100 41 STOCKHOLM, SWEDEN
SN 0172-780X
J9 NEUROENDOCRINOL LETT
JI Neuroendocrinol. Lett.
PD AUG
PY 2008
VL 29
IS 4
BP 558
EP 570
PG 13
WC Endocrinology & Metabolism; Neurosciences
SC Endocrinology & Metabolism; Neurosciences & Neurology
GA 356JJ
UT WOS:000259774200029
PM 18766162
ER
PT J
AU Schulte-Ruther, M
Markowitsch, HJ
Shah, NJ
Fink, GR
Piefke, M
AF Schulte-Ruether, Martin
Markowitsch, Hans J.
Shah, N. Jon
Fink, Gereon R.
Piefke, Martina
TI Gender differences in brain networks supporting empathy
SO NEUROIMAGE
LA English
DT Article
ID EMOTIONAL FACIAL EXPRESSIONS; SEX-DIFFERENCES; INDIVIDUAL-DIFFERENCES;
NEURAL MECHANISMS; MIRROR NEURON; SPECTRUM DISORDERS; ASPERGER-SYNDROME;
FUNCTIONAL MRI; MIND; AUTISM
AB Females frequently score higher on standard tests of empathy, social sensitivity, and emotion recognition than do males. It remains to be clarified, however, whether these gender differences; are associated with gender specific neural mechanisms of emotional social cognition. We investigated gender differences in an emotion attribution task using functional magnetic resonance imaging. Subjects either focused on their own emotional response to emotion expressing faces (SELF-task) or evaluated the emotional state expressed by the faces (OTHER-task). Behaviorally, females rated SELF-related emotions significantly stronger than males. Across the sexes, SELF-and OTHER-related processing of facial expressions activated a network of medial and lateral prefrontal, temporal, and parietal brain regions involved in emotional perspective taking. During SELF-related processing, females recruited the right inferior frontal cortex and superior temporal sulcus stronger than males. In contrast, there was increased neural activity in the left temporoparietal junction in males (relative to females). When performing the OTHER-task, females showed increased activation of the right inferior frontal cortex while there were no differential activations in males. The data suggest that females recruit areas containing mirror neurons to a higher degree than males during both SELF- and OTHER-related processing in empathic face-to-face interactions. This may underlie facilitated emotional "contagion" in females. Together with the observation that males differentially rely on the left temporoparietal junction (an area mediating the distinction between the SELF and OTHERS) the data suggest that females and males rely on different strategies when assessing their own emotions in response to other people. (c) 2008 Elsevier Inc. All rights reserved.
C1 [Schulte-Ruether, Martin] Res Ctr Julich, Inst Neurosci & Biophys INB3 Med, Inst Med, Cognit Neurol Sect, D-52425 Julich, Germany.
[Schulte-Ruether, Martin] Rhein Westfal TH Aachen, Univ Hosp Aachen, Dept Child & Adolescent Psychiat, Child Neuropsychol Sect, Aachen, Germany.
[Markowitsch, Hans J.; Piefke, Martina] Univ Bielefeld, D-4800 Bielefeld, Germany.
[Fink, Gereon R.] Univ Hosp Cologne, Dept Neurol, Cologne, Germany.
RP Schulte-Ruther, M (reprint author), Res Ctr Julich, Inst Neurosci & Biophys INB3 Med, Inst Med, Cognit Neurol Sect, Leo Brand Str 5, D-52425 Julich, Germany.
EM m.schulte@fz-juelich.de
RI Fink, Gereon/E-1616-2012; Schulte-Ruther, Martin /F-4784-2013; Shah, N.
Jon /H-5849-2013
OI Fink, Gereon/0000-0002-8230-1856; Schulte-Ruther, Martin
/0000-0002-7198-9923; Shah, N. Jon /0000-0002-8151-6169
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NR 72
TC 106
Z9 111
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1053-8119
J9 NEUROIMAGE
JI Neuroimage
PD AUG 1
PY 2008
VL 42
IS 1
BP 393
EP 403
DI 10.1016/j.neuroimage.2008.04.180
PG 11
WC Neurosciences; Neuroimaging; Radiology, Nuclear Medicine & Medical
Imaging
SC Neurosciences & Neurology; Radiology, Nuclear Medicine & Medical Imaging
GA 332TC
UT WOS:000258105000039
PM 18514546
ER
PT J
AU Pellicano, E
Gibson, LY
AF Pellicano, Elizabeth
Gibson, Lisa Y.
TI Investigating the functional integrity of the dorsal visual pathway in
autism and dyslexia
SO NEUROPSYCHOLOGIA
LA English
DT Article
DE dorsal-stream functioning; autism; dyslexia; developmental disorders;
visual perception
ID MOTION PERCEPTION; MAGNOCELLULAR DEFICIT; DISABLED READERS; STREAM
FUNCTION; COHERENCE; IMPAIRMENTS; CHILDREN
AB Numerous reports of elevated global motion thresholds across a variety of neurodevelopmental disorders have prompted researchers to suggest that abnormalities in global motion perception are a result of a general deficiency in the dorsal visual pathway. To test this hypothesis, we assessed the integrity of the dorsal visual pathway at lower subcortical (sensitivity to flicker contrast) and higher cortical (sensitivity to global motion) levels in children with autism, children with dyslexia, and typically developing children, of similar age and ability. While children with autism demonstrated intact lower-level, but impaired higher-level dorsal-stream functioning, children with dyslexia displayed abnormalities at both lower and higher levels of the dorsal visual stream. These findings suggest that these disorders can be dissociated according to the origin of the impairment along the dorsal-stream pathway. implications for general cross-syndrome accounts are discussed. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Pellicano, Elizabeth] Univ Bristol, Dept Expt Psychol, Bristol BS8 1TU, Avon, England.
[Pellicano, Elizabeth] Univ Western Australia, Sch Psychol, Nedlands, WA 6009, Australia.
[Gibson, Lisa Y.] Univ Bristol, Ctr Child Hlth Res, Telethon Inst Child Hlth Res, Bristol BS8 1TU, Avon, England.
RP Pellicano, E (reprint author), Univ Bristol, Dept Expt Psychol, 12A Priory Rd, Bristol BS8 1TU, Avon, England.
EM liz.pellicano@bristol.ac.uk
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Wechsler D, 1999, WECHSLER ABBREVIATED
White S, 2006, COGN NEUROPSYCHOL, V23, P748, DOI 10.1080/02643290500438607
NR 30
TC 32
Z9 32
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0028-3932
J9 NEUROPSYCHOLOGIA
JI Neuropsychologia
PD AUG
PY 2008
VL 46
IS 10
BP 2593
EP 2596
DI 10.1016/j.neuropsychologia.2008.04.008
PG 4
WC Behavioral Sciences; Neurosciences; Psychology, Experimental
SC Behavioral Sciences; Neurosciences & Neurology; Psychology
GA 326SL
UT WOS:000257679100015
PM 18501932
ER
PT J
AU Miocinovic, LP
Nad, KB
Dugandzija, GP
AF Miocinovic, Ljiljana Popovic
Nad, Katarina Bosnjak
Dugandzija, Gordana Paskov
TI THE RELATIONSHIP BETWEEN EPILEPTIC AND NON-EPILEPTIC SEIZURES IN
CHILDHOOD
SO PAEDIATRIA CROATICA
LA Croatian
DT Article
DE EPILEPSY, diagnosis; SEIZURES, diagnosis; VIDEO-ELECTROENCEPHALOGRAPHY,
methods
ID PAROXYSMAL EVENTS; EEG ABNORMALITIES; NORMAL-CHILDREN; VIDEO-EEG;
ADOLESCENTS; DISORDERS; DIAGNOSIS; FEATURES; SPECTRUM; AUTISM
AB This paper illustrates the difficulty in distinguishing non-epileptic and epileptic attacks in children who have abnormal epileptiform interictal EEG. We describe our own experience with long-term video EEG monitoring (VEEG), including the recording technique and objective and subjective monitoring difficulties specific to children. The usefulness of VEEG was demonstrated in cases where non-epileptic and epileptic seizures occurred frequently), however VEEG was not useful when seizures were infrequent.
In practice it is recommended to combine neurological methods to differentiate non-epileptic and epileptic seizures and to use VEEG more frequently, especially prior to initiating potentially toxic anti-epileptic pharmacotherapy This is particularly important in children with autism, mental retardation, other neurological dysfunctions and those with refractory epilepsies. Timely diagnosis of non-epileptic seizures with initiation of appropriate therapy will certainly improve the final outcome. Clinical assessment and developmental follow up continue to be crucial steps because the child's growth (aid development can impact the clinical manifestation of the disorder.
C1 [Miocinovic, Ljiljana Popovic; Dugandzija, Gordana Paskov] Specijalna Boln Zastitu Djece Neurorazvojnim & Mo, Zagreb 2, Goljak, Croatia.
RP Miocinovic, LP (reprint author), Specijalna Boln Zastitu Djece Neurorazvojnim & Mo, Zagreb 2, Goljak, Croatia.
EM ljiljana.miocino-vic@hi.t-com.hr
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NR 38
TC 0
Z9 0
PU CHILDRENS HOSPITAL ZAGREB
PI ZAGREB
PA KLAICEVA 16, ZAGREB, 10000, CROATIA
SN 1330-1403
J9 PAEDIATR CROAT
JI Paediatr. Croat.
PD AUG-SEP
PY 2008
VL 52
IS 3
BP 171
EP 175
PG 5
WC Pediatrics
SC Pediatrics
GA 389WB
UT WOS:000262124100007
ER
PT J
AU Brenton, JN
Devries, SP
Barton, C
Minnich, H
Sokol, DK
AF Brenton, James N.
Devries, Seth P.
Barton, Christine
Minnich, Heike
Sokol, Deborah K.
TI Absolute pitch in a four-year-old boy with autism
SO PEDIATRIC NEUROLOGY
LA English
DT Article
ID SEROTONIN; CHILDREN
AB Absolute pitch is the ability to identify the pitch of an isolated tone. We report on a 4-year-old boy with autism and absolute pitch, one of the youngest reported in the literature. Absolute pitch is thought to be attributable to a single gene, transmitted in an autosomal-dominant fashion. The association of absolute pitch with autism raises the speculation that this talent could be linked to a genetically distinct subset of children with autism. Further, the identification of absolute pitch in even young children with autism may lead to a lifelong skill. (C) 2008 by Elsevier Inc. All rights reserved.
C1 [Brenton, James N.; Devries, Seth P.; Barton, Christine; Minnich, Heike; Sokol, Deborah K.] Indiana Univ, Sch Med, Pediat Sect, Dept Neurol, Indianapolis, IN 46202 USA.
RP Sokol, DK (reprint author), Indiana Univ, Sch Med, Pediat Sect, Dept Neurol, 575 West Dr,XE 40, Indianapolis, IN 46202 USA.
EM dksokol@iupui.edu
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NR 15
TC 8
Z9 8
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0887-8994
EI 1873-5150
J9 PEDIATR NEUROL
JI Pediatr. Neurol.
PD AUG
PY 2008
VL 39
IS 2
BP 137
EP 138
DI 10.1016/j.pediatrneurol.2008.05.004
PG 2
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 336LO
UT WOS:000258365500015
PM 18639762
ER
PT J
AU Grigorenko, EL
Han, SS
Yrigollen, CM
Leng, L
Mizue, Y
Anderson, GM
Mulder, EJ
de Bildt, A
Minderaa, RB
Volkmar, FR
Chang, JT
Bucala, R
AF Grigorenko, Elena L.
Han, Summer S.
Yrigollen, Carolyn M.
Leng, Lin
Mizue, Yuka
Anderson, George M.
Mulder, Erik J.
de Bildt, Annelies
Minderaa, Ruud B.
Volkmar, Fred R.
Chang, Joseph T.
Bucala, Richard
TI Macrophage migration inhibitory factor and autism spectrum disorders
SO PEDIATRICS
LA English
DT Article
DE autism spectrum disorder; ASD; MIF; neurodevelopmental disorders;
genetic association; genetic polymorphisms; immunologic insult
ID PERVASIVE DEVELOPMENTAL DISORDERS; GENOTYPE-PHENOTYPE ASSOCIATIONS;
AUTOIMMUNE-DISEASE; INNATE IMMUNITY; FACTOR MIF; CHILDREN; BRAIN;
POLYMORPHISM; ANTIBODIES; LINKAGE
AB OBJECTIVE. Autistic spectrum disorders are childhood neurodevelopmental disorders characterized by social and communicative impairment and repetitive and stereotypical behavior. Macrophage migration inhibitory factor (MIF) is an upstream regulator of innate immunity that promotes monocyte/macrophage-activation responses by increasing the expression of Toll-like receptors and inhibiting activation-induced apoptosis. On the basis of results of previous genetic linkage studies and reported altered innate immune response in autism spectrum disorder, we hypothesized that MIF could represent a candidate gene for autism spectrum disorder or its diagnostic components.
METHODS. Genetic association between autism spectrum disorder and MIF was investigated in 2 independent sets of families of probands with autism spectrum disorder, from the United States (527 participants from 152 families) and Holland (532 participants from 183 families). Probands and their siblings, when available, were evaluated with clinical instruments used for autism spectrum disorder diagnoses. Genotyping was performed for 2 polymorphisms in the promoter region of the MIF gene in both samples sequentially. In addition, MIF plasma analyses were conducted in a subset of Dutch patients from whom plasma was available.
RESULTS. There were genetic associations between known functional polymorphisms in the promoter for MIF and autism spectrum disorder - related behaviors. Also, probands with autism spectrum disorder exhibited higher circulating MIF levels than did their unaffected siblings, and plasma MIF concentrations correlated with the severity of multiple autism spectrum disorder symptoms.
CONCLUSIONS. These results identify MIF as a possible susceptibility gene for autism spectrum disorder. Additional research is warranted on the precise relationship between MIF and the behavioral components of autism spectrum disorder, the mechanism by which MIF contributes to autism spectrum disorder pathogenesis, and the clinical use of MIF genotyping.
C1 [Grigorenko, Elena L.; Yrigollen, Carolyn M.; Anderson, George M.; Volkmar, Fred R.] Yale Univ, Ctr Child Study, New Haven, CT 06519 USA.
[Grigorenko, Elena L.; Volkmar, Fred R.] Yale Univ, Dept Psychol, New Haven, CT 06519 USA.
[Grigorenko, Elena L.; Leng, Lin; Bucala, Richard] Yale Univ, Dept Epidemiol & Publ Hlth, New Haven, CT 06519 USA.
[Chang, Joseph T.] Yale Univ, Dept Stat, New Haven, CT 06519 USA.
[Leng, Lin; Bucala, Richard] Yale Univ, Dept Internal Med, New Haven, CT 06519 USA.
[Leng, Lin; Bucala, Richard] Yale Univ, Dept Pathol, New Haven, CT 06519 USA.
[Han, Summer S.] Moscow MV Lomonosov State Univ, Dept Psychol, Moscow, Russia.
[Mizue, Yuka] Sapporo Immuno Diagnost Lab, Sapporo, Hokkaido, Japan.
[Mulder, Erik J.; de Bildt, Annelies; Minderaa, Ruud B.] Univ Groningen, Univ Med Ctr Groningen, Univ Ctr Child & Adolescent Psychiat, Accare, Groningen, Netherlands.
RP Grigorenko, EL (reprint author), Yale Univ, Ctr Child Study, 230 S Frontage Rd, New Haven, CT 06519 USA.
EM elena.grigorenko@yale.edu
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NR 60
TC 40
Z9 41
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD AUG
PY 2008
VL 122
IS 2
BP E438
EP E445
DI 10.1542/peds.2007-3604
PG 8
WC Pediatrics
SC Pediatrics
GA 333HB
UT WOS:000258142500064
PM 18676531
ER
PT J
AU Philippe, A
Boddaert, N
Vaivre-Douret, L
Robel, L
Danon-Boileau, L
Malan, V
de Blois, MC
Heron, D
Colleaux, L
Golse, B
Zilbovicius, M
Munnich, A
AF Philippe, Anne
Boddaert, Nathalie
Vaivre-Douret, Laurence
Robel, Laurence
Danon-Boileau, Laurent
Malan, Valerie
de Blois, Marie-Christine
Heron, Delphine
Colleaux, Laurence
Golse, Bernard
Zilbovicius, Monica
Munnich, Arnold
TI Neurobehavioral profile and brain imaging study of the 22q13.3 deletion
syndrome in childhood
SO PEDIATRICS
LA English
DT Article
DE 22q13.3 deletion; pervasive developmental disorders; autism; regression;
language deficit; neuromotor disturbances; sensory abnormalities; thin
corpus callosum
ID PERVASIVE DEVELOPMENTAL DISORDERS; MOLECULAR CHARACTERIZATION;
YOUNG-CHILDREN; CHROMOSOME 22Q13.3; MENTAL-RETARDATION; AUTISM SPECTRUM;
FEATURES; FISH; TRANSLOCATIONS; OVERGROWTH
AB OBJECTIVE. The 22q13.3 deletion syndrome (Online Mendelian Inheritance in Man No. 606232) is a neurodevelopmental disorder that includes hypotonia, severely impaired development of speech and language, autistic-like behavior, and minor dysmorphic features. Although the number of reported cases is increasing, the 22q13.3 deletion remains underdiagnosed because of failure in recognizing the clinical phenotype and detecting the 22qter deletion by routine chromosome analyses. Our goal is to contribute to the description of the neurobehavioral phenotype and brain abnormalities of this microdeletional syndrome.
METHODS. We assessed neuromotor, sensory, language, communication, and social development and performed cerebral MRI and study of regional cerebral blood flow measured by positron emission tomography in 8 children carrying the 22q13.3 deletion.
RESULTS. Despite variability in expression and severity, the children shared a common developmental profile characterized by hypotonia, sleep disorders, and poor response to their environment in early infancy; expressive language deficit contrasting with emergence of social reciprocity from ages similar to 3 to 5 years; sensory processing dysfunction; and neuromotor disorders. Brain MRI findings were normal or showed a thin or morphologically atypical corpus callosum. Positron emission tomography study detected a localized dysfunction of the left temporal polar lobe and amygdala hypoperfusion.
CONCLUSIONS. The developmental course of the 22q13.3 deletion syndrome belongs to pervasive developmental disorders but is distinct from autism. An improved description of the natural history of this syndrome should help in recognizing this largely underdiagnosed condition.
C1 [Philippe, Anne; Malan, Valerie; de Blois, Marie-Christine; Colleaux, Laurence; Munnich, Arnold] Hop Necker Enfants Malad, Assistance Publ Hop Paris, Natl Inst Hlth & Med Res, Paris, France.
[Philippe, Anne; de Blois, Marie-Christine; Colleaux, Laurence; Munnich, Arnold] Hop Necker Enfants Malad, Assistance Publ Hop Paris, Dept Genet, Paris, France.
[Boddaert, Nathalie] Natl Inst Hlth & Med Res, Mixed Unit Res 0205, Orsay, France.
[Vaivre-Douret, Laurence; Robel, Laurence; Golse, Bernard] Hop Necker Enfants Malad, Assistance Publ Hop Paris, Dept Psychiat, Paris, France.
[Vaivre-Douret, Laurence] Univ Paris 10, Mixed Unit Res S0669, Univ Paris 05, Univ Paris 11, Paris 10, France.
[Vaivre-Douret, Laurence] Assistance Publ Hop Paris, Dept Obstet & Gynaecol, Paris, France.
[Danon-Boileau, Laurent] Natl Ctr Sci Res, Mixed Unit Res 7114, Paris, France.
[Heron, Delphine] Hop La Pitie Salpetriere, Assistance Publ Hop Paris, Dept Genet, Paris, France.
RP Philippe, A (reprint author), Hop Necker Enfants Malad, INSERM, U781, 149 Rue Sevres, F-75015 Paris, France.
EM anne.philippe@necker.fr
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NR 38
TC 27
Z9 27
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD AUG
PY 2008
VL 122
IS 2
BP E376
EP E382
DI 10.1542/peds.2007-2584
PG 7
WC Pediatrics
SC Pediatrics
GA 333HB
UT WOS:000258142500056
PM 18625665
ER
PT J
AU Nataf, R
Skorupka, C
Lam, A
Springbett, A
Lathe, R
AF Nataf, Robert
Skorupka, Corinne
Lam, Alain
Springbett, Anthea
Lathe, Richard
TI Porphyrinuria in childhood autistic disorder is not associated with
urinary creatinine deficiency
SO PEDIATRICS INTERNATIONAL
LA English
DT Article
DE autism; creatinine; pervasive developmental disorder; porphyrin; urine
ID PERVASIVE DEVELOPMENTAL DISORDERS; OCCUPATIONAL MERCURY EXPOSURE;
POSTPRANDIAL ALKALINE TIDE; OXIDASE CPOX POLYMORPHISM; HEME
BIOSYNTHETIC-PATHWAY; BODY-MASS INDEX; HEYER ET-AL.; OXIDATIVE STRESS;
SPECTRUM DISORDERS; CASCADE ANALYSIS
AB Background: Urinary metabolite measurements are often normalized to levels of the ubiquitous metabolite creatinine (CRT) to take account of variations in fluid export. Following CRT normalization, excesses of porphyrins and isoprostanes have been reported in the urines of children with neurodevelopmental disorders. It was suggested (Whiteley et al., 2006, Pediatr. Int. 2006; 48: 292-297) that urinary CRT levels may be depressed in children with autism spectrum disorders. This prompted re-evaluation of CRT levels in such children.
Methods: First matinal urinary CRT levels were compared between subjects in different diagnostic categories including autistic disorder, pervasive developmental disorder not otherwise specified (PDD-NOS) and hyperactivity, before and after correction for age and gender. A larger reference group, consisting of subjects with unrelated disorders and Asperger disorder, with no reported porphyrin excess, was also compared to the group with autistic disorder, both for CRT and for porphyrin (coproporphyrin, COPRO) excess.
Results: No significant difference in CRT was observed between any of the categories analyzed, also when corrected for age and gender. In contrast, urinary COPRO levels were significantly higher in autistic disorder versus reference groups, either when expressed as absolute values (independent of CRT levels) or when normalized to CRT.
Conclusions: These data do not support a systematic reduction in urinary CRT levels in subjects with autism spectrum disorders including autistic disorder and PDD-NOS. Urinary COPRO excess in autistic disorder was not associated with or consequent upon urinary CRT deficiency. Differences between affected and control subjects in age and sampling time, as reported by Whiteley et al., may underlie the apparent CRT reduction.
C1 [Lathe, Richard] Pieta Res, Edinburgh EH10 5YW, Midlothian, Scotland.
[Nataf, Robert; Lam, Alain] Philippe Auguste Lab, Paris, France.
[Skorupka, Corinne] Assoc ARIANE, Clichy, France.
[Springbett, Anthea] Roslin Inst, Dept Stat, Roslin EH25 9PS, Midlothian, Scotland.
RP Lathe, R (reprint author), Pieta Res, POB 27069, Edinburgh EH10 5YW, Midlothian, Scotland.
EM rlathe@pieta-research.org
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NR 48
TC 9
Z9 9
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1328-8067
J9 PEDIATR INT
JI Pediatr. Int.
PD AUG
PY 2008
VL 50
IS 4
BP 528
EP 532
DI 10.1111/j.1442-200X.2008.02621.x
PG 5
WC Pediatrics
SC Pediatrics
GA 341RK
UT WOS:000258732300023
PM 19143977
ER
PT J
AU Mouridsen, SE
Rich, B
Isager, T
AF Mouridsen, Svend E.
Rich, Bente
Isager, Torben
TI Body mass index in male and female children with pervasive developmental
disorders
SO PEDIATRICS INTERNATIONAL
LA English
DT Article
DE Asperger syndrome; atypical autism; body mass index; developmental
disorder; endophenotype; pervasive
ID ASPERGERS-DISORDER; INFANTILE-AUTISM; ADOLESCENTS; WEIGHT; PSYCHIATRY;
CURVES; BIRTH; AGE
AB Background: The aim of the present study was to evaluate body mass index (BMI) of children with a pervasive developmental disorder (PDD) attending two university clinics during the 1960-84 period.
Methods: BMI derived from medical records of 83 consecutively admitted children with atypical autism and 115 children with Asperger syndrome were compared with the corresponding BMI percentiles in an age- and sex-matched reference population.
Results: The BMI distribution of the boys, but not the girls, in both diagnostic categories was significantly lower than those of the age-matched reference populations. Approximately 15% of the boys had a BMI below the fifth percentile.
Conclusions: The present findings are comparable to the results of other studies. Particular attention is given to low BMI as a potential endophenotype in boys with PDD.
C1 [Mouridsen, Svend E.] Bispebjerg Hosp, Dept Child & Adolescent Psychiat, DK-2400 Copenhagen, Denmark.
[Rich, Bente] Odense Univ Hosp, Child Adolescent Psychiat Dept, DK-5000 Odense, Denmark.
[Isager, Torben] Glostrup Univ Hosp, Ctr Child & Adolescent Psychiat, Glostrup, Denmark.
RP Mouridsen, SE (reprint author), Bispebjerg Hosp, Dept Child & Adolescent Psychiat, DK-2400 Copenhagen, Denmark.
EM sem01@bbh.hosp.dk
FU Mindelegat
FX The authors wish to thank C. Hermansen's Mindelegat for financial
support and Dr Bernadette Buhl-Nielsen for linguistic revision of this
paper.
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NR 18
TC 7
Z9 7
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1328-8067
J9 PEDIATR INT
JI Pediatr. Int.
PD AUG
PY 2008
VL 50
IS 4
BP 569
EP 571
DI 10.1111/j.1442-200X.2008.02618.x
PG 3
WC Pediatrics
SC Pediatrics
GA 341RK
UT WOS:000258732300031
PM 19143984
ER
PT J
AU Liss, M
Mailloux, J
Erchull, MJ
AF Liss, Miriam
Mailloux, Jennifer
Erchull, Mindy J.
TI The relationships between sensory processing sensitivity, alexithymia,
autism, depression, and anxiety
SO PERSONALITY AND INDIVIDUAL DIFFERENCES
LA English
DT Article
DE sensory processing sensitivity; highly sensitive people; alexithymia;
autism; anxiety; depression
ID SPECTRUM QUOTIENT AQ; SOCIAL ANXIETY; DISORDER; STIMULATION;
RELIABILITY; INVENTORY; VALIDITY; STRESS; SCALE
AB The goal of the current investigation was to better understand the relationships between the three recently-developed factors of sensory processing sensitivity (ease of excitation - EOE, low sensory threshold - LST, and aesthetic sensitivity - AES) and alexithymia, autism symptoms, anxiety, and depression. Two hundred and one college students completed the highly sensitive person scale, as well as measures of anxiety, depression, alexithymia, and autism symptoms. EOE and LST were related to autism symptoms, alexithymia, anxiety, and depression. AES was related to attention to details (a symptom of autism) and anxiety but not to depression. It was also negatively related to externally-oriented thinking (a symptom of alexithymia). Results indicate that AES is conceptually distinct from LST and EOE. Furthermore, EOE interacted with difficulty identifying feelings in predicting anxiety, indicating that being both easily excited by stimuli and unable to identify one's feelings is particularly anxiety provoking. (C) 2008 Elsevier Ltd. All rights reserved.
C1 [Liss, Miriam; Mailloux, Jennifer; Erchull, Mindy J.] Univ Mary Washington, Dept Psychol, Fredericksburg, VA 22401 USA.
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NR 36
TC 13
Z9 13
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0191-8869
J9 PERS INDIV DIFFER
JI Pers. Individ. Differ.
PD AUG
PY 2008
VL 45
IS 3
BP 255
EP 259
DI 10.1016/j.paid.2008.04.009
PG 5
WC Psychology, Social
SC Psychology
GA 320SZ
UT WOS:000257256600010
ER
PT J
AU Kakinuma, H
Sato, H
AF Kakinuma, Hiroaki
Sato, Hitoshi
TI Copy-number variations associated with autism spectrum disorder
SO PHARMACOGENOMICS
LA English
DT Review
DE array comparative genomic hybridization; autism spectrum disorder; ASD;
copy-number variation; CNV; de novo CNV; Inherited CNV; multiplex
family; recurrent CNV; simplex family; SNP; SNP genotyping array
ID PERVASIVE DEVELOPMENTAL DISORDERS; COMPARATIVE GENOMIC HYBRIDIZATION;
MENTAL-RETARDATION; CHROMOSOMAL REARRANGEMENTS; VELOCARDIOFACIAL
SYNDROME; STRUCTURAL VARIATION; GENETIC DISORDER; CANDIDATE GENES;
YOUNG-CHILDREN; HIGH-FREQUENCY
AB Autism spectrum disorder (ASD) is a clinically heterogeneous developmental disorder with a strong genetic component. Rare genetic disorders and various chromosomal abnormalities are thought to account for approximately 10% of people with ASD. The etiology of the remaining cases remains unknown. Recent advances in array-based technology have increased the resolution in detecting submicroscopic deletions and duplications, referred to as copy-number variations. ASD-associated copy-number variations, which are considered to be present in individuals with ASD but not in unaffected individuals, have been extensively investigated. These data will provide us with an opportunity not only to search for genes causing or contributing to ASDs but also to understand the genetics of ASD.
C1 [Kakinuma, Hiroaki; Sato, Hitoshi] Kanazawa Med Univ, Dept Pediat, Kanazawa, Ishikawa 920293, Japan.
RP Kakinuma, H (reprint author), Kanazawa Med Univ, Dept Pediat, 1-1 Uchinada, Kanazawa, Ishikawa 920293, Japan.
EM p-kaki@kanazawa-med.ac.jp
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NR 103
TC 7
Z9 7
PU FUTURE MEDICINE LTD
PI LONDON
PA UNITEC HOUSE, 3RD FLOOR, 2 ALBERT PLACE, FINCHLEY CENTRAL, LONDON, N3
1QB, ENGLAND
SN 1462-2416
J9 PHARMACOGENOMICS
JI Pharmacogenomics
PD AUG
PY 2008
VL 9
IS 8
BP 1143
EP 1154
DI 10.2217/14622416.9.8.1143
PG 12
WC Pharmacology & Pharmacy
SC Pharmacology & Pharmacy
GA 339FI
UT WOS:000258563100023
PM 18681787
ER
PT J
AU Bourgeois, JP
AF Bourgeois, J. -P.
TI Normal, pathological and mendable synaptogenesis in the cerebral cortex
SO PSN-PSYCHIATRIE SCIENCES HUMAINES NEUROSCIENCES
LA French
DT Article
DE cerebral cortex; synaptogenesis; synaptic pathologies; plasticity
ID PREFRONTAL CORTEX; GENE-EXPRESSION; DENDRITIC SPINES; CRITICAL PERIOD;
NERVOUS-SYSTEM; VISUAL-CORTEX; PLASTICITY; BRAIN; SCHIZOPHRENIA; AUTISM
AB In the cerebral cortex, different phases of synaptogenesis coincide with the main maturation stages of sensory, motor, and cognitive abilities. Early in development, synapses are generated and differentiated under the control of robust mechanisms governed by genes. Then, during multiple critical periods, extending from the end of gestation to the end of puberty, the way in which the synaptic structure develops is highly dependent upon the quality of the environment. The duration of these critical periods increases significantly through evolution. This "epigenetic opening'' of synaptogenesis to the environment is maximal in the human cerebral cortex. It is the source of the exceptional cognitive adaptability and cultural creativity of our species. It is also, possibly, one of our multiple psychological fragilities. Analysis of the human genome reveals that mutations in genes related to synaptic structure and signalisation are linked to many psychiatric disorders. Neurobiologists are able to manipulate the critical periods of synaptogenesis during development and have begun to do so in the adult cerebral cortex. These epigenetic manipulations might allow us to restore synaptic plasticity and, potentially, repair the disorganised synaptic circuits observed in psychiatric pathologies. You said epigenetic manipulations?
C1 Inst Pasteur, CNRS URA2182 Gene Synapse & Cognit, Lab Genet Humaine & Fonct Cognit, Dept Neurosci, F-75724 Paris 15, France.
RP Bourgeois, JP (reprint author), Inst Pasteur, CNRS URA2182 Gene Synapse & Cognit, Lab Genet Humaine & Fonct Cognit, Dept Neurosci, 25 Rue Dr Roux, F-75724 Paris 15, France.
EM jpbourg@pasteur.fr
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NR 39
TC 0
Z9 0
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1639-8319
J9 PSN-PSYCHIAT SCI HUM
JI PSN-Psychiatr. Sci. Hum. Neurosc.
PD AUG
PY 2008
VL 6
IS 3
BP 124
EP 136
DI 10.1007/s11836-008-0065-z
PG 13
WC Neurosciences
SC Neurosciences & Neurology
GA 338WC
UT WOS:000258539100003
ER
PT J
AU [Anonymous]
AF [Anonymous]
TI Researchers Link Early Stem Cell Mutation to Autism
SO PSYCHIATRIC ANNALS
LA English
DT News Item
NR 0
TC 0
Z9 0
PU SLACK INC
PI THOROFARE
PA 6900 GROVE RD, THOROFARE, NJ 08086 USA
SN 0048-5713
EI 1938-2456
J9 PSYCHIAT ANN
JI Psychiatr. Ann.
PD AUG
PY 2008
VL 38
IS 8
BP 497
EP 497
PG 1
WC Psychiatry
SC Psychiatry
GA 364QC
UT WOS:000260349200003
ER
PT J
AU Yan, J
Feng, JN
Schroer, R
Li, WY
Skinner, C
Schwartz, CE
Cook, EH
Sommer, SS
AF Yan, Jin
Feng, Jinong
Schroer, Richard
Li, Wenyan
Skinner, Cindy
Schwartz, Charles E.
Cook, Edwin H., Jr.
Sommer, Steve S.
TI Analysis of the neuroligin 4Y gene in patients with autism
SO PSYCHIATRIC GENETICS
LA English
DT Article
DE autism; mutation detection; neuroligin 4Y
ID MUTATIONS; NLGN4; SCHIZOPHRENIA; CHROMOSOME; BINDING
AB Frameshift and missense mutations in the X-linked neuroligin 4 (NLGN4, MIM# 300427) and neuroligin 3 (NLGN3, MIM# 300336) genes have been identified in patients with autism, Asperger syndrome and mental retardation. We hypothesize that sequence variants in NLGN4Y are associated with autism or mental retardation. The coding sequences and splice junctions of the NLGN4Y gene were analyzed in 335 male samples (290 with autism and 45 with mental retardation). A total of 1.1 Mb of genomic DNA was sequenced. One missense variant, p.1679V, was identified in a patient with autism, as well as his father with learning disabilities. The 1679 residue is highly conserved in three members of the neuroligin family. The absence of p.1679V in 2986 control Y chromosomes and the high similarity of NLGN4 and NLGN4Y are consistent with the hypothesis that p.1679V contributes to the etiology of autism. The presence of only one structural variant in our population of 335 males with autism/mental retardation, the unavailability of significant family cosegregation and an absence of functional assays are, however, important limitations of this study.
C1 [Yan, Jin; Feng, Jinong; Li, Wenyan; Sommer, Steve S.] City Hope Natl Med Ctr, Dept Mol Genet, Duarte, CA 91010 USA.
[Schroer, Richard; Skinner, Cindy; Schwartz, Charles E.] Greenwood Genet Ctr, Greenwood, SC 29646 USA.
[Cook, Edwin H., Jr.] Univ Illinois, Dept Psychiat, Inst Juvenile Res, Chicago, IL 60612 USA.
RP Sommer, SS (reprint author), City Hope Natl Med Ctr, Dept Mol Genet, 1500 E Duarte Rd, Duarte, CA 91010 USA.
EM sommerlab@coh.org
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NR 15
TC 38
Z9 39
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0955-8829
J9 PSYCHIAT GENET
JI Psychiatr. Genet.
PD AUG
PY 2008
VL 18
IS 4
BP 204
EP 207
PG 4
WC Genetics & Heredity; Neurosciences
SC Genetics & Heredity; Neurosciences & Neurology
GA 331OF
UT WOS:000258020800009
PM 18628683
ER
PT J
AU Koyama, T
Inada, N
Tsujii, H
Kurita, H
AF Koyama, Tomonori
Inada, Naoko
Tsujii, Hiromi
Kurita, Hiroshi
TI Predicting children with pervasive developmental disorders using the
Wechsler Intelligence Scale for Children-Third Edition
SO PSYCHIATRY AND CLINICAL NEUROSCIENCES
LA English
DT Article
DE pervasive developmental disorders; screening; sensitivity; specificity;
Wechsler Intelligence Scale for Children-Third Edition
ID WISC-III; PROFILES; AUTISM
AB An original combination score (i.e. the sum of Vocabulary and Comprehension subtracted from the sum of Block Design and Digit Span) was created from the four Wechsler Intelligence Scale for Children-Third Edition (WISC-III) subtests identified by discriminant analysis on WISC-III data from 139/129 children with/without pervasive developmental disorders (PDD; mean, 8.3/8.1 years) and its utility examined for predicting PDD. Its best cut-off was 2/3, with sensitivity, specificity, positive and negative predictive values of 0.68, 0.61, 0.65 and 0.64, respectively. The score seems useful, so long as clinicians are aware of its limitations and use it only as a supplemental measure in PDD diagnosis.
C1 [Koyama, Tomonori; Inada, Naoko; Tsujii, Hiromi] Natl Ctr Neurol & Psychiat, Dept Child & Adolescent Mental Hlth, Natl Inst Mental Hlth, Kodaira, Tokyo, Japan.
[Kurita, Hiroshi] Univ Tokyo, Grad Sch Med, Dept Mental Hlth, Tokyo, Japan.
RP Koyama, T (reprint author), Natl Ctr Neurol & Psychiat, Dept Child & Adolescent Mental Hlth, Natl Inst Mental Hlth, 4-1-1 Ogawa Higashi, Tokyo 1878553, Japan.
EM tomok@ncnp.go.jp
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NR 10
TC 1
Z9 1
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1323-1316
J9 PSYCHIAT CLIN NEUROS
JI Psychiatry Clin. Neurosci.
PD AUG
PY 2008
VL 62
IS 4
BP 476
EP 478
DI 10.1111/j.1440-1819.2008.01826.x
PG 3
WC Clinical Neurology; Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 332RB
UT WOS:000258099700015
PM 18778447
ER
PT J
AU Turner, M
AF Turner, Mark
TI Robots, mirror, neurons, virtual reality and autism
SO PSYCHOLOGIST
LA English
DT Article
ID SPECTRUM DISORDERS; IMITATION; DYSFUNCTION; PREMOTOR; CHILDREN; FMRI
AB To address a growing public awareness and apparent increase in prevalence of autism (Prior, 2003), psychologists need to devise improved treatment options based on the latest scientific discoveries. One popular treatment option is the use of 'psychoeducational' intervention strategies. The use of such strategies by educationists might be more fruitful in helping children with autism with the added application of scientific methods that seem to monitor what has been called a 'mirror neuron' (MN) dysfunction in autism. So why do MN system dysfunction and difficulties in imitation apply to children with autism, and how can educationists facilitate such discoveries for the benefit of those affected?
EM markalext@hotmail.com
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NR 21
TC 0
Z9 0
PU BRITISH PSYCHOLOGICAL SOC
PI LEICESTER
PA ST ANDREWS HOUSE, 48 PRINCESS RD EAST, LEICESTER LE1 7DR, LEICS, ENGLAND
SN 0952-8229
J9 PSYCHOLOGIST
JI Psychologist
PD AUG
PY 2008
VL 21
IS 8
BP 674
EP 676
PG 3
WC Psychology, Multidisciplinary
SC Psychology
GA 341ZF
UT WOS:000258753700014
ER
PT J
AU Stanton-Chapman, TL
Denning, CB
Jamison, KR
AF Stanton-Chapman, Tina L.
Denning, Christopher B.
Jamison, Kristen Roorbach
TI Exploring the effects of a social communication intervention for
improving requests and word diversity in preschoolers with disabilities
SO PSYCHOLOGY IN THE SCHOOLS
LA English
DT Article
ID LANGUAGE IMPAIRMENT; YOUNG-CHILDREN; HEAD-START; SKILLS; AUTISM; SCHOOL;
COMPETENCE; DISCOURSE; BEHAVIOR; ACCESS
AB This study examined the effects of a multicomponent social communication intervention to promote language learning and peer-directed social interactions in preschool children with disabilities. Participants were eight children with developmental disabilities who met the specified Criteria for the study. The intervention consisted of three components: (a) a planning, period for instructional purposes. (b) a play session to practice skills. and (c) a brief reporting period to review skill performance. A Multiple baseline design across two dyads replicated across two additional dyads was used. Results indicated an increase in peer-directed requests. verbal requests. and word diversity for 6 of the 8 participants postintervention. Implications of the results are discussed. (C) 2008 Wiley Periodicals. Inc.
C1 [Stanton-Chapman, Tina L.] Univ Virginia, Curry Sch Educ, Charlottesville, VA 22903 USA.
RP Stanton-Chapman, TL (reprint author), Univ Virginia, Curry Sch Educ, 405 Emmet St,POB 400273, Charlottesville, VA 22903 USA.
EM stantonchapman@virginia.edu
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NR 63
TC 7
Z9 7
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 0033-3085
J9 PSYCHOL SCHOOLS
JI Psychol. Schools
PD AUG
PY 2008
VL 45
IS 7
BP 644
EP 664
DI 10.1002/pits.20315
PG 21
WC Psychology, Educational
SC Psychology
GA 334WM
UT WOS:000258252100006
ER
PT J
AU Woodgate, RL
Ateah, C
Secco, L
AF Woodgate, Roberta L.
Ateah, Christine
Secco, Loretta
TI Living in a world of our own: The experience of parents who have a child
with autism
SO QUALITATIVE HEALTH RESEARCH
LA English
DT Article
DE autism; children; families, caregiving; parenting; phenomenology
ID SPECTRUM DISORDER; COPING STRATEGIES; MOTHERS; FAMILIES; LIFE;
INTERVENTION; MANAGEMENT; DIAGNOSIS; STIGMA; STRESS
AB In this article, we discuss findings of a hermeneutic phenomenological study that sought to describe the experiences of parents who have a child with autism. Qualitative interviews were conducted with parents from 16 families of children with autism residing in a western Canadian province. "Living in a world of our own" emerged as the essence of the parents' experiences. In "living in a world of our own," parents described a world of isolation. Three themes representing the essential challenging elements of the parents' experiences included vigilant parenting, sustaining the self and family, and fighting all the way. Although much is known about the fundamental importance of support to parents of children with chronic conditions and/or disabilities, findings from this study indicate that knowledge has not been adequately transferred to the care of children with autism.
C1 [Woodgate, Roberta L.] Univ Manitoba, Fac Nursing, Winnipeg, MB, Canada.
[Ateah, Christine] Univ Manitoba, Fac Nursing, Winnipeg, MB, Canada.
[Secco, Loretta] Cape Breton Univ, Dept Nursing, Sydney, NS, Canada.
RP Woodgate, RL (reprint author), Univ Manitoba, Fac Nursing, Winnipeg, MB, Canada.
RI Woodgate, Roberta/B-9038-2014
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NR 27
TC 48
Z9 48
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1049-7323
J9 QUAL HEALTH RES
JI Qual. Health Res.
PD AUG
PY 2008
VL 18
IS 8
BP 1075
EP 1083
DI 10.1177/1049732308320112
PG 9
WC Health Policy & Services
SC Health Care Sciences & Services
GA 329QM
UT WOS:000257884300006
PM 18650563
ER
PT J
AU Thompson, JC
Hardee, JE
AF Thompson, James C.
Hardee, Jillian E.
TI The first time ever I saw your face
SO TRENDS IN COGNITIVE SCIENCES
LA English
DT Article
ID BIOLOGICAL MOTION; PERCEPTION; AUTISM; RECOGNITION; CHILDREN; INFANTS;
PEOPLE
AB The perception of social information is crucial for the survival of most animal species. Two recent studies demonstrate the joint contribution of innate mechanisms and perceptual experience to two aspects of social perception - faces and biological motion. Together, they highlight how important it is to consider faces and biological motion as different visual properties used by a broader social perception system.
C1 [Thompson, James C.] George Mason Univ, Dept Psychol, Fairfax, VA 22030 USA.
[Hardee, Jillian E.] W Virginia Univ, Ctr Adv Imaging, Dept Radiol, Morgantown, WV 26506 USA.
[Hardee, Jillian E.] W Virginia Univ, Ctr Adv Imaging, Dept Neurobiol & Anat, Morgantown, WV 26506 USA.
RP Thompson, JC (reprint author), George Mason Univ, Dept Psychol, 4400 Univ Dr,MS3F5, Fairfax, VA 22030 USA.
EM jthompsz@gmu.edu
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NR 21
TC 9
Z9 9
PU ELSEVIER SCIENCE LONDON
PI LONDON
PA 84 THEOBALDS RD, LONDON WC1X 8RR, ENGLAND
SN 1364-6613
J9 TRENDS COGN SCI
JI TRENDS COGN. SCI.
PD AUG
PY 2008
VL 12
IS 8
BP 283
EP 284
DI 10.1016/j.tics.2008.05.002
PG 2
WC Behavioral Sciences; Neurosciences; Psychology, Experimental
SC Behavioral Sciences; Neurosciences & Neurology; Psychology
GA 337JU
UT WOS:000258433200002
PM 18606560
ER
PT J
AU Theoharides, TC
Doyle, R
Francis, K
Conti, P
Kalogeromitros, D
AF Theoharides, Theoharis C.
Doyle, Robert
Francis, Konstantinos
Conti, Pio
Kalogeromitros, Dimitris
TI Novel therapeutic targets for autism
SO TRENDS IN PHARMACOLOGICAL SCIENCES
LA English
DT Review
ID HUMAN MAST-CELLS; CORTICOTROPIN-RELEASING-HORMONE; BLOOD-BRAIN-BARRIER;
INFLAMMATORY-BOWEL-DISEASE; ENDOTHELIAL GROWTH-FACTOR; SPECTRUM
DISORDERS; OXIDATIVE STRESS; GASTROINTESTINAL SYMPTOMS;
AUTOIMMUNE-DISEASES; FOOD ALLERGY
AB Autism spectrum disorders (ASDs) are pervasive neuro-developmental disorders, diagnosed in early childhood when acquired skills are lost or the acquisition of new skills becomes delayed. ASDs are associated with varying degrees of dysfunctional communication and social skills, in addition to repetitive and stereotypic behaviors. The diagnosis has increased considerably to approximately one in 180 people, but it is not clear whether this is because of a higher prevalence of the disorder, improved awareness by clinicians or a combination of both. There are no defined mechanisms of pathogenesis or curative therapy presently available. Oxidative stress, overactivation of the hypothalamic-pituitary-adrenal axis and increased gut-blood-brain-barrier permeability might be involved. The scope of this article is to integrate these findings and present the opinion that non-allergic activation of gastrointestinal and brain mast cells could contribute to many of the pathologic findings and provide unique targets for ASD therapy. We make suggestions for new research directives and possible novel therapies from readily available molecules.
C1 [Theoharides, Theoharis C.] Tufts Univ, Sch Med, Dept Pharmacol & Expt Therapeut,Tufts Med Ctr, Lab Mol Immunopharmacol & Drug Discovery, Boston, MA 02111 USA.
[Theoharides, Theoharis C.] Tufts Univ, Sch Med, Tufts Med Ctr, Dept Internal Med, Boston, MA 02111 USA.
[Theoharides, Theoharis C.] Tufts Univ, Sch Med, Tufts Med Ctr, Dept Biochem, Boston, MA 02111 USA.
[Theoharides, Theoharis C.] Tufts Univ, Sch Med, Tufts Med Ctr, Dept Psychiat, Boston, MA 02111 USA.
[Theoharides, Theoharis C.; Kalogeromitros, Dimitris] Attikon Gen Hosp, Athens Med Sch, Allergy Unit, Allergy Clin Res Ctr, Athens 12462, Greece.
[Doyle, Robert] Harvard Univ, Sch Med, Massachusetts Gen Hosp, Dept Pediat Psychopharmacol, Boston, MA 02114 USA.
[Francis, Konstantinos] Attikon Gen Hosp, Athens Med Sch, Dept Psychiat 2, Athens 12462, Greece.
[Conti, Pio] Univ G dAnnunzio, Sch Med, Dept Canc & Neurosci, Div Immunol, I-66013 Chieti, Italy.
RP Theoharides, TC (reprint author), Tufts Univ, Sch Med, Dept Pharmacol & Expt Therapeut,Tufts Med Ctr, Lab Mol Immunopharmacol & Drug Discovery, 136 Harrison Ave, Boston, MA 02111 USA.
EM theoharis.theoharides@tufts.edu
FU NIH [NS38326, AR47652]; Mastocytosis Society (USA); Theta Biomedical
Consulting and Development (Brookline, MA)
FX Aspects of this work were funded in part by NIH grants NS38326 and
AR47652, the Mastocytosis Society (USA) and Theta Biomedical Consulting
and Development (Brookline, MA) to T.C.T. Many thanks to Jessica
Christian for her word-processing skills.
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NR 69
TC 30
Z9 31
PU ELSEVIER SCIENCE LONDON
PI LONDON
PA 84 THEOBALDS RD, LONDON WC1X 8RR, ENGLAND
SN 0165-6147
J9 TRENDS PHARMACOL SCI
JI Trends Pharmacol. Sci.
PD AUG
PY 2008
VL 29
IS 8
BP 375
EP 382
DI 10.1016/j.tips.2008.06.002
PG 8
WC Pharmacology & Pharmacy
SC Pharmacology & Pharmacy
GA 341WQ
UT WOS:000258746600001
PM 18606459
ER
PT J
AU Hu, HL
Qin, Y
Bochorishvili, G
Zhu, YH
van Aelst, L
Zhu, JJ
AF Hu, Hailan
Qin, Yi
Bochorishvili, Genrieta
Zhu, Yinghua
van Aelst, Linda
Zhu, J. Julius
TI Ras signaling mechanisms underlying impaired GluR1-dependent plasticity
associated with fragile X syndrome
SO JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE autism; AMPA receptor trafficking; Ras-PI3K signaling; cancer risk;
mental retardation; dendritic spine dysmorphogenesis; facial dysmorphism
ID MENTAL-RETARDATION PROTEIN; AMPA-RECEPTOR TRAFFICKING; FMR1 KNOCKOUT
MICE; LONG-TERM POTENTIATION; NEOCORTICAL PYRAMIDAL NEURONS;
BETA-GAMMA-SUBUNITS; SYNAPTIC PLASTICITY; MOUSE MODEL; DENDRITIC SPINES;
PHOSPHOINOSITIDE 3-KINASE
AB Fragile X syndrome, caused by the loss of FMR1 gene function and loss of fragile X mental retardation protein (FMRP), is the most commonly inherited form of mental retardation. The syndrome is characterized by associative learning deficits, reduced risk of cancer, dendritic spine dysmorphogenesis, and facial dysmorphism. However, the molecular mechanism that links loss of function of FMR1 to the learning disability remains unclear. Here, we report an examination of small GTPase Ras signaling and synaptic AMPA receptor (AMPA-R) trafficking in cultured slices and intact brains of wild-type and FMR1 knock-out mice. In FMR1 knock-out mice, synaptic delivery of GluR1-, but not GluR2L- and GluR4-containing AMPA-Rs is impaired, resulting in a selective loss of GluR1- dependent long-term synaptic potentiation (LTP). Although Ras activity is upregulated, its downstream MEK (extracellular signal-regulated kinase kinase)-ERK (extracellular signal-regulated kinase) signaling appears normal, and phosphoinositide 3-kinase (PI3K)-protein kinase B (PKB; or Akt) signaling is compromised in FMR1 knock-out mice. Enhancing Ras-PI3K-PKB signaling restores synaptic delivery of GluR1-containing AMPA-Rs and normal LTP in FMR1 knock-out mice. These results suggest aberrant Ras signaling as a novel mechanism for fragile X syndrome and indicate manipulating Ras-PI3K-PKB signaling to be a potentially effective approach for treating patients with fragile X syndrome.
C1 [Hu, Hailan; Qin, Yi; Bochorishvili, Genrieta; Zhu, Yinghua; Zhu, J. Julius] Univ Virginia, Sch Med, Dept Pharmacol, Charlottesville, VA 22908 USA.
[Zhu, J. Julius] Univ Virginia, Sch Med, Dept Neurosci, Charlottesville, VA 22908 USA.
[Qin, Yi; van Aelst, Linda] Cold Spring Harbor Lab, Ctr Canc Res, Cold Spring Harbor, NY 11724 USA.
RP Zhu, JJ (reprint author), Univ Virginia, Sch Med, Dept Pharmacol, 1300 Jefferson Pk Ave, Charlottesville, VA 22908 USA.
EM jjzhu@virginia.edu
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NR 89
TC 73
Z9 74
PU SOC NEUROSCIENCE
PI WASHINGTON
PA 11 DUPONT CIRCLE, NW, STE 500, WASHINGTON, DC 20036 USA
SN 0270-6474
J9 J NEUROSCI
JI J. Neurosci.
PD JUL 30
PY 2008
VL 28
IS 31
BP 7847
EP 7862
DI 10.1523/JNEUROSCI.1496-08.2008
PG 16
WC Neurosciences
SC Neurosciences & Neurology
GA 332DG
UT WOS:000258061600014
PM 18667617
ER
PT J
AU Ge, JQ
Han, SH
AF Ge, Jianqiao
Han, Shihui
TI Distinct Neurocognitive Strategies for Comprehensions of Human and
Artificial Intelligence
SO PLOS ONE
LA English
DT Article
ID NEURAL BASIS; SELF; MIND; BRAIN; PERSPECTIVE; DEACTIVATE; SIMULATION;
THINKING; AUTISM; OTHERS
AB Although humans have inevitably interacted with both human and artificial intelligence in real life situations, it is unknown whether the human brain engages homologous neurocognitive strategies to cope with both forms of intelligence. To investigate this, we scanned subjects, using functional MRI, while they inferred the reasoning processes conducted by human agents or by computers. We found that the inference of reasoning processes conducted by human agents but not by computers induced increased activity in the precuneus but decreased activity in the ventral medial prefrontal cortex and enhanced functional connectivity between the two brain areas. The findings provide evidence for distinct neurocognitive strategies of taking others' perspective and inhibiting the process referenced to the self that are specific to the comprehension of human intelligence.
C1 [Ge, Jianqiao; Han, Shihui] Peking Univ, Dept Psychol, Beijing 100871, Peoples R China.
[Han, Shihui] Peking Univ, Funct Imaging Ctr, Beijing, Peoples R China.
[Han, Shihui] Peking Univ, Key Lab Machine Percept, Beijing, Peoples R China.
RP Han, SH (reprint author), Peking Univ, Dept Psychol, Beijing 100871, Peoples R China.
EM shan@pku.edu.cn
FU National Natural Science Foundation of China [30630025]
FX This work was supported by the National Natural Science Foundation of
China (Project 30630025).
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JI PLoS One
PD JUL 30
PY 2008
VL 3
IS 7
AR e2797
DI 10.1371/journal.pone.0002797
PG 8
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 420QM
UT WOS:000264304300016
PM 18665211
ER
PT J
AU Kleinhans, NM
Muller, RA
Cohen, DN
Courchesne, E
AF Kleinhans, Natalia M.
Mueller, Ralph-Axel
Cohen, David N.
Courchesne, Eric
TI Atypical functional lateralization of language in autism spectrum
disorders
SO BRAIN RESEARCH
LA English
DT Article; Proceedings Paper
CT 13th Conference of the International-Linear-Algebra-Society
CY JUL 18-21, 2006
CL Amsterdam, NETHERLANDS
SP Int Linear Algebra Soc
HO VU Univ
DE fMRI; asymmetry; letter fluency; category fluency; frontal lobe
ID VERBAL FLUENCY; BRAIN ACTIVATION; DEVELOPMENTAL DISORDERS; SENTENCE
COMPREHENSION; ASPERGERS-DISORDER; HEAD CIRCUMFERENCE; PREFRONTAL
CORTEX; CHILDHOOD AUTISM; SEMANTIC FLUENCY; FRONTAL-CORTEX
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C1 [Kleinhans, Natalia M.] Univ Washington, Dept Radiol, Seattle, WA 98195 USA.
[Mueller, Ralph-Axel] San Diego State Univ, Dept Psychol, Sari Diego, CA 92120 USA.
[Cohen, David N.] George Washington Univ, Sch Med, Washington, DC 20037 USA.
[Mueller, Ralph-Axel] Univ Calif San Diego, Dept Cognit Sci, San Diego, CA 92093 USA.
[Courchesne, Eric] Univ Calif San Diego, Dept Neurosci, San Diego, CA 92093 USA.
[Courchesne, Eric] Childrens Hosp Res Ctr, San Diego, CA 92093 USA.
RP Kleinhans, NM (reprint author), Univ Washington, Dept Radiol, Box 357115, Seattle, WA 98195 USA.
EM nkleinha@u.washington.edu
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NR 76
TC 66
Z9 66
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0006-8993
J9 BRAIN RES
JI Brain Res.
PD JUL 24
PY 2008
VL 1221
BP 115
EP 125
DI 10.1016/j.brainres.2008.04.080
PG 11
WC Neurosciences
SC Neurosciences & Neurology
GA 334XF
UT WOS:000258254300014
PM 18555209
ER
PT J
AU Adolphs, R
Spezio, ML
Parlier, M
Piven, J
AF Adolphs, Ralph
Spezio, Michael L.
Parlier, Morgan
Piven, Joseph
TI Distinct face-processing strategies in parents of autistic children
SO CURRENT BIOLOGY
LA English
DT Article
ID AMYGDALA VOLUME; GAZE-FIXATION; RECOGNITION; INFORMATION; INDIVIDUALS;
PHENOTYPE; FAMILIES; BUBBLES; BRAIN
AB In his original description of autism, Kanner [1] noted that the parents of autistic children often exhibited unusual social behavior themselves, consistent with what we now know about the high heritability of autism [2]. We investigated this so-called Broad Autism Phenotype in the parents of children with autism, who themselves did not receive a diagnosis of any psychiatric illness. Building on recent quantifications of social cognition in autism [3], we investigated face processing by using the "bubbles" method [4] to measure how viewers make use of information from specific facial features in order to judge emotions. Parents of autistic children who were assessed as socially aloof (N = 15), a key component of the phenotype [5], showed a remarkable reduction in processing the eye region in faces, together with enhanced processing of the mouth, compared to a control group of parents of neurotypical children (N = 20), as well as to nonaloof parents of autistic children (N = 27, whose pattern of face processing was intermediate). The pattern of face processing seen in the Broad Autism Phenotype showed striking similarities to that previously reported to occur in autism [3] and for the first time provides a window into the endophenotype that may result from a subset of the genes that contribute to social cognition.
C1 [Parlier, Morgan; Piven, Joseph] Univ N Carolina, Chapel Hill, NC 27599 USA.
[Adolphs, Ralph; Spezio, Michael L.] CALTECH, Pasadena, CA 91125 USA.
[Spezio, Michael L.] Scripps Coll, Claremont, CA 91711 USA.
RP Piven, J (reprint author), Univ N Carolina, Chapel Hill, NC 27599 USA.
EM jpiven@med.unc.edu
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NR 25
TC 41
Z9 44
PU CELL PRESS
PI CAMBRIDGE
PA 600 TECHNOLOGY SQUARE, 5TH FLOOR, CAMBRIDGE, MA 02139 USA
SN 0960-9822
J9 CURR BIOL
JI Curr. Biol.
PD JUL 22
PY 2008
VL 18
IS 14
BP 1090
EP 1093
DI 10.1016/j.cub.2008.06.073
PG 4
WC Biochemistry & Molecular Biology; Cell Biology
SC Biochemistry & Molecular Biology; Cell Biology
GA 336PW
UT WOS:000258378100034
PM 18635351
ER
PT J
AU Skogstrand, K
Ekelund, CK
Thorsen, P
Vogel, I
Jacobsson, B
Norgaard-Pedersen, B
Hougaard, DM
AF Skogstrand, Kristin
Ekelund, Charlotte K.
Thorsen, Poul
Vogel, Ida
Jacobsson, Bo
Norgaard-Pedersen, Bent
Hougaard, David M.
TI Effects of blood sample handling procedures on measurable inflammatory
markers in plasma, serum and dried blood spot samples
SO JOURNAL OF IMMUNOLOGICAL METHODS
LA English
DT Article
DE blood spots; plasma; storage; inflammatory markers; Luminex
ID NEONATAL CYTOKINES; CEREBRAL-PALSY; WHOLE-BLOOD; NEUROTROPHINS;
CHILDREN; NEUROPEPTIDES; AUTISM
AB The interests in monitoring inflammation by immunoassay determination of blood inflammatory markers call for information on the stability of these markers in relation to the handling of blood samples. The increasing use of stored biobank samples for such ventures that may have been collected and stored for other purposes, justifies the study hereof. Blood samples were stored for 0, 4, 24, and 48 h at 4 degrees C, room temperature (RT), and at 35 degrees C, respectively, before they were separated into serum or plasma and frozen. Dried blood spot samples (DBSS) were stored for 0, 1, 2, 3, 7, and 30 days at the same temperatures. 27 inflammatory markers in serum and plasma and 25 markers in DBSS were measured by a previously validated multiplex sandwich immumoassay using Luminex xMAP technology. The measurable concentrations of several cytokines in serum and plasma were significantly increased when blood samples were stored for a period of time before the centrifugation, for certain cytokines more than 1000 fold compared to serum and plasma isolated and frozen immediately after venepuncture. The concentrations in serum generally increased more than in plasma. The measurable concentrations of inflammatory markers also changed in DBSS stored under various conditions compared to controls frozen immediately after preparation, but to a much lesser degree than in plasma or serum.
The study demonstrates that trustworthy measurement of several inflammatory markers relies on handling of whole blood samples at low temperatures and rapid isolation of plasma and serum. Effects of different handling procedures for all markers studied are given. DBSS proved to be a robust and convenient way to handle samples for immunoassay analysis of inflammatory markers in whole blood. (C) 2008 Elsevier B.V. All rights reserved.
C1 [Skogstrand, Kristin; Norgaard-Pedersen, Bent; Hougaard, David M.] Statens Serum Inst, Dept Clin Biochem, DK-2300 Copenhagen S, Denmark.
[Skogstrand, Kristin; Ekelund, Charlotte K.; Thorsen, Poul; Vogel, Ida; Jacobsson, Bo] Univ Aarhus, NANEA, Inst Publ Hlth, Aarhus, Denmark.
[Ekelund, Charlotte K.] Copenhagen Univ Hosp, Dept Fetal Med & Ultrasound, Rigshosp, Copenhagen, Denmark.
[Vogel, Ida] Aarhus Univ Hosp, Dept Clin Genet, Aarhus, Denmark.
[Jacobsson, Bo] Univ Gothenburg, Perinatal Ctr, Dept Obstet & Gynecol, Gothenburg, Sweden.
RP Skogstrand, K (reprint author), Statens Serum Inst, Dept Clin Biochem, Artillerivej 5, DK-2300 Copenhagen S, Denmark.
EM ksk@ssi.dk
CR CALAM RR, 2004, PROCEDURES HANDLING, V24, pH18
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NR 15
TC 43
Z9 43
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0022-1759
J9 J IMMUNOL METHODS
JI J. Immunol. Methods
PD JUL 20
PY 2008
VL 336
IS 1
BP 78
EP 84
DI 10.1016/j.jim.2008.04.006
PG 7
WC Biochemical Research Methods; Immunology
SC Biochemistry & Molecular Biology; Immunology
GA 317TY
UT WOS:000257044200011
PM 18495149
ER
PT J
AU Stokstad, E
AF Stokstad, Erik
TI Medicine - Stalled trial for autism highlights dilemma of alternative
treatments
SO SCIENCE
LA English
DT News Item
CR 2001, SCIENCE 1005, P37
NR 1
TC 2
Z9 2
PU AMER ASSOC ADVANCEMENT SCIENCE
PI WASHINGTON
PA 1200 NEW YORK AVE, NW, WASHINGTON, DC 20005 USA
SN 0036-8075
J9 SCIENCE
JI Science
PD JUL 18
PY 2008
VL 321
IS 5887
BP 326
EP 326
DI 10.1126/science.321.5887.326
PG 1
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 327FB
UT WOS:000257713900005
PM 18635766
ER
PT J
AU Wadman, M
AF Wadman, Meredith
TI Autism study panned by critics
SO NATURE
LA English
DT News Item
CR Stangle DE, 2007, ENVIRON HEALTH PERSP, V115, P201, DOI 10.1289/ehp.9263
NR 1
TC 2
Z9 2
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 0028-0836
J9 NATURE
JI Nature
PD JUL 17
PY 2008
VL 454
IS 7202
BP 259
EP 259
DI 10.1038/454259a
PG 1
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 326ND
UT WOS:000257665300006
PM 18633377
ER
PT J
AU Yamagata, T
Urano, H
Weeber, EJ
Nelson, DL
Nishijima, I
AF Yamagata, T.
Urano, H.
Weeber, E. J.
Nelson, D. L.
Nishijima, I.
TI Impaired hippocampal synaptic function in secretin deficient mice
SO NEUROSCIENCE
LA English
DT Article
DE peptide hormone; knock-out mice; hippocampus; synapse; autism;
beta-galactosidase
ID STEM-CELLS; AUTISM; RECEPTORS; TRANSMISSION; CEREBELLUM; ACTIVATION;
CORTEX; BRAIN
AB Secretin is a gut peptide hormone that is also expressed in the CNS. To explore the potential neuroactive role of secretin in the brain, we have generated secretin deficient mice. Secretin deficient mice demonstrated impairment in synaptic plasticity (significant reduction in long term potentiation (LTP) induction and LTP maintenance) in the CA1 area of the hippocampus. Using a beta-galactosidase (lacZ) reporter in the targeted allele and secretin antibody staining, we have detected secretin gene expression in the hippocampus, cerebellum, and the brain stem in adult mouse brain. In the hippocampus, secretin was expressed in the dentate gyrus, the hilus, and the molecular layer. These findings suggest that secretin is involved in synaptic function in the adult brain. (C) 2008 IBRO. Published by Elsevier Ltd. All rights reserved.
C1 [Urano, H.; Nishijima, I.] Ohio State Univ, Dept Pediat, Nationwide Childrens Hosp, Ctr Mol & Human Genet,Res Inst, Columbus, OH 43205 USA.
[Yamagata, T.] Jichi Med Univ, Dept Pediat, Shimotsuke, Tochigi, Japan.
[Weeber, E. J.] Univ S Florida, Dept Mol Pharmacol & Physiol, Tampa, FL 33612 USA.
[Nelson, D. L.] Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
RP Nishijima, I (reprint author), Ohio State Univ, Dept Pediat, Nationwide Childrens Hosp, Ctr Mol & Human Genet,Res Inst, 700 Childrens Dr, Columbus, OH 43205 USA.
EM nishijii@pediatrics.ohio-state.edu
RI Weeber, Edwin/A-5396-2012
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NR 25
TC 14
Z9 14
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0306-4522
J9 NEUROSCIENCE
JI Neuroscience
PD JUL 17
PY 2008
VL 154
IS 4
BP 1417
EP 1422
DI 10.1016/j.neuroscience.2008.04.037
PG 6
WC Neurosciences
SC Neurosciences & Neurology
GA 326RZ
UT WOS:000257677900024
PM 18534766
ER
PT J
AU Plon, M
AF Plon, Michel
TI The child who was stopped on the threshold of language. Understanding
autism
SO QUINZAINE LITTERAIRE
LA French
DT Book Review
CR REYFLAUD H, ENFANT QUI SEST ARRE
NR 1
TC 0
Z9 0
PU QUINZAINE LITTERAIRE
PI PARIS
PA 135 RUE SAINT-MARTIN, 75194 PARIS, FRANCE
SN 0048-6493
J9 QUINZAINE LITTERAIRE
JI Quinz. Litt.
PD JUL 16
PY 2008
IS 973
BP 23
EP 24
PG 2
WC Literature
SC Literature
GA 329FI
UT WOS:000257851900026
ER
PT J
AU Catani, M
Jones, DK
Daly, E
Embiricos, N
Deeley, Q
Pugliese, L
Curran, S
Robertson, D
Murphy, DGM
AF Catani, Marco
Jones, Derek K.
Daly, Eileen
Embiricos, Nitzia
Deeley, Quinton
Pugliese, Luca
Curran, Sarah
Robertson, Dene
Murphy, Declan G. M.
TI Altered cerebellar feedback projections in Asperger syndrome
SO NEUROIMAGE
LA English
DT Article
DE Asperger; cerebellum; white matter; diffusion tensor; tractography;
autism
ID POSTERIOR-FOSSA STRUCTURES; AUTISM SPECTRUM DISORDER; CEREBRAL
WHITE-MATTER; DIFFUSION TENSOR MRI; INFANTILE-AUTISM; HUMAN BRAIN; FIBER
TRACTOGRAPHY; PREFRONTAL CORTEX; ABNORMALITIES; CHILDREN
AB It has been proposed that the biological basis of autism spectrum disorder includes cerebellar 'disconnection'. However, direct in vivo evidence in support of this is lacking. Here, the microstructural integrity of cerebellar white matter in adults with Asperger syndrome was studied using diffusion tensor magnetic resonance tractography. Fifteen adults with Asperger syndrome and 16 age-IQ-gender-matched healthy controls underwent diffusion tensor magnetic resonance imaging. For each subject, tract-specific measurements of mean diffusivity and fractional anisotropy were made within the inferior, middle, superior cerebellar peduncles and short intracerebellar fibres. No group differences were observed in mean diffusivity. However, people with Asperger syndrome had significantly lower fractional anisotropy in the short intracerebellar fibres (p < 0.001) and right superior cerebellar (output) peduncle (p < 0.001) compared to controls; but no difference in the input tracts. Severity of social impairment, as measured by the Autistic Diagnostic Interview, was negatively correlated with diffusion anisotropy in the fibres of the left superior cerebellar peduncle. These findings suggest a vulnerability of specific cerebellar neural pathways in people with Asperger syndrome. The localised abnormalities in the main cerebellar outflow pathway may prevent the cerebral cortex from receiving those cerebellar feedback inputs necessary for a successful adaptive social behaviour. (c) 2008 Elsevier Inc. All rights reserved.
C1 [Catani, Marco; Daly, Eileen; Embiricos, Nitzia; Deeley, Quinton; Pugliese, Luca; Curran, Sarah; Robertson, Dene; Murphy, Declan G. M.] Kings Coll London, Sect Brain Maturat, Dept Psychol Med & Psychiat, Inst Psychiat, London SE5 8AF, England.
[Jones, Derek K.] CUBRIC Cardiff Univ, Cardiff CF10 3AT, Wales.
RP Catani, M (reprint author), Kings Coll London, Sect Brain Maturat, Dept Psychol Med & Psychiat, Inst Psychiat, Crespigny Pk, London SE5 8AF, England.
EM m.catani@iop.kcl.ac.uk
RI Jones, Derek/D-1460-2009; daly, eileen/B-6716-2011; Catani,
Marco/H-7801-2012
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NR 77
TC 94
Z9 96
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1053-8119
J9 NEUROIMAGE
JI Neuroimage
PD JUL 15
PY 2008
VL 41
IS 4
BP 1184
EP 1191
DI 10.1016/j.neuroimage.2008.03.041
PG 8
WC Neurosciences; Neuroimaging; Radiology, Nuclear Medicine & Medical
Imaging
SC Neurosciences & Neurology; Radiology, Nuclear Medicine & Medical Imaging
GA 311SR
UT WOS:000256620400002
PM 18495494
ER
PT J
AU Vanderwal, T
Hunyadi, E
Grupe, DW
Connors, CM
Schultz, RT
AF Vanderwal, Tamara
Hunyadi, Elinora
Grupe, Daniel W.
Connors, Caitlin M.
Schultz, Robert T.
TI Self, mother and abstract other: An fMRI study of reflective social
processing
SO NEUROIMAGE
LA English
DT Article
DE self; self reference; theory of mind; social attribution; dorsal medial
prefrontal cortex; temporal pole
ID MEDIAL PREFRONTAL CORTEX; COGNITIVE NEUROSCIENCE; REPRESENTATION;
METAANALYSIS; PERSPECTIVE; JUDGMENTS; KNOWLEDGE; BEHAVIOR; SEARCH;
AUTISM
AB Using fMRI, we studied the neural correlates of self-referential processing by comparing BOLD signal changes during self and mother conditions of a self-reference effect (SRE) task. Conjunction analysis of these two conditions showed several common areas of significant activation, including the medial aspects of the superior frontal gyri, left inferior frontal gyrus, bilateral temporal poles, left superior temporal sulcus and left precuneus. The locations of the 7 strongest peak activations for the self condition and for the mother condition were compared on a subject-by-subject basis in native space. Of the 119 pairs of peaks explored, 87% were located within 2 voxels of each other, demonstrating the commonality of the brain regions subserving both self- and mother-referential processing within an individual subject. In group analyses of the self-referential vs. mother-referential contrast, small differences in activation strength were observed in the left superior frontal sulcus, right cingulate gyrus and the left fusiform gyrus. Greater activation for mother than for self was observed in bilateral temporal lobes. Subjects also performed a social attribution task (SAT) in which they inferred mental states about interacting geometric shapes. Activations from this visual theory of mind task were compared with the activations demonstrated during self-referential processing. Striking similarities were found, including overlapping activations in bilateral medial prefrontal cortices, left inferior frontal gyrus and precuneus. These data suggest that reflective analysis of self, mother and abstract others relies predominantly on the same neural architecture. (c) 2008 Elsevier Inc. All rights reserved.
C1 [Vanderwal, Tamara; Grupe, Daniel W.] Yale Univ, Sch Med, Yale Child Study Ctr, New Haven, CT 06520 USA.
[Connors, Caitlin M.] London Sch Econ, London, England.
[Hunyadi, Elinora; Schultz, Robert T.] Childrens Hosp Philadelphia, Dept Pediat, Ctr Austism Res, Philadelphia, PA 19104 USA.
RP Vanderwal, T (reprint author), Yale Univ, Sch Med, Yale Child Study Ctr, 230 S Frontage Rd, New Haven, CT 06520 USA.
EM tamara.vanderwal@yale.edu
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NR 38
TC 50
Z9 51
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1053-8119
J9 NEUROIMAGE
JI Neuroimage
PD JUL 15
PY 2008
VL 41
IS 4
BP 1437
EP 1446
DI 10.1016/j.neuroimage.2008.03.058
PG 10
WC Neurosciences; Neuroimaging; Radiology, Nuclear Medicine & Medical
Imaging
SC Neurosciences & Neurology; Radiology, Nuclear Medicine & Medical Imaging
GA 311SR
UT WOS:000256620400024
PM 18486489
ER
PT J
AU Reekie, YL
Braesicke, K
Man, MS
Roberts, AC
AF Reekie, Y. L.
Braesicke, K.
Man, M. S.
Roberts, A. C.
TI Uncoupling of behavioral and autonomic responses after lesions of the
primate orbitofrontal cortex
SO PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF
AMERICA
LA English
DT Article
DE behavioral inhibition; emotion; reversal learning
ID PREFRONTAL CORTICAL PROJECTIONS; CENTRAL NUCLEUS FUNCTION; BASOLATERAL
AMYGDALA; INFRALIMBIC CORTEX; MACAQUE MONKEYS; FRONTAL-CORTEX;
RHESUS-MONKEYS; LONGITUDINAL COLUMNS; PERIAQUEDUCTAL GRAY; EXTINCTION
AB Successful adaptation to changes in an animal's emotional and motivational environment depends on behavioral flexibility accompanied by changes in bodily responses, e.g., autonomic and endocrine, which support the change in behavior. Here, we identify the orbitofrontal cortex (OFC) as pivotal in the flexible regulation and coordination of behavioral and autonomic responses during adaptation. Using an appetitive Pavlovian task, we demonstrate that OFC lesions in the marmoset (i) impair an animal's ability to rapidly suppress its appetitive cardiovascular arousal upon termination of a conditioned stimulus and (ii) cause an uncoupling of the behavioral and autonomic components of the adaptive response after reversal of the reward contingencies. These findings highlight the role of the OFC in emotional regulation and are highly relevant to our understanding of disorders such as schizophrenia and autism in which uncoupling of emotional responses may contribute to the experiential distress and disadvantageous behavior associated with these disorders.
C1 [Reekie, Y. L.; Braesicke, K.; Man, M. S.; Roberts, A. C.] Univ Cambridge, Dept Physiol Dev & Neurosci, Cambridge CB2 3DY, England.
[Braesicke, K.; Man, M. S.; Roberts, A. C.] Univ Cambridge, Behav & Clin Neurosci Inst, Cambridge CB2 3EB, England.
RP Roberts, AC (reprint author), Univ Cambridge, Dept Physiol Dev & Neurosci, Downing St, Cambridge CB2 3DY, England.
EM acr4@cam.ac.uk
RI Roberts, Angela/C-1313-2012
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NR 55
TC 21
Z9 21
PU NATL ACAD SCIENCES
PI WASHINGTON
PA 2101 CONSTITUTION AVE NW, WASHINGTON, DC 20418 USA
SN 0027-8424
J9 P NATL ACAD SCI USA
JI Proc. Natl. Acad. Sci. U. S. A.
PD JUL 15
PY 2008
VL 105
IS 28
BP 9787
EP 9792
DI 10.1073/pnas.0800417105
PG 6
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 328FV
UT WOS:000257784700060
PM 18621690
ER
PT J
AU Bora, E
Gokcen, S
Veznedaroglu, B
AF Bora, Emre
Goekcen, Sezen
Veznedaroglu, Baybars
TI Empathic abilities in people with schizophrenia
SO PSYCHIATRY RESEARCH
LA English
DT Article
DE empathy; schizophrenia; social cognition; empathy quotient
ID HIGH-FUNCTIONING AUTISM; EMOTION RECOGNITION; SPECTRUM DISORDERS;
ASPERGER-SYNDROME; SOCIAL COGNITION; MIND; REMEDIATION; ADULTS
AB Although the existence of empathy deficits in schizophrenia is generally accepted, very few studies have directly investigated the issue. The nature of empathy deficits in healthy subjects and psychiatric patients is an understudied subject. The performances of the 30 outpatients with schizophrenia on a psychometric measure, the Empathy Quotient (EQ), were compared with those of 30 control subjects matched for age, duration of education and gender. The relatives or spouses of the patients also filled out the EQ. A neuropsychological battery, including emotion recognition, emotional reasoning and theory of mind tasks, was also administered. Schizophrenia patients had severe empathy dysfunction based on their relative EQ ratings. There was a serious discrepancy between the self and relative/spouse assessments of the empathic skills of schizophrenia patients. Consistent with the previous findings schizophrenia patients were impaired in nearly all cognitive tasks. The empathy deficits of schizophrenia patients were associated with their impairments in other social cognitive tasks. Studies focusing on dysfunctional brain networks underlying empathy deficits and studies using more experimental measures of empathy should be helpful to unravel the true nature of the empathic failure in patients with schizophrenia. (C) 2007 Elsevier Ireland Ltd. All rights reserved.
C1 [Bora, Emre] Univ Melbourne, Dept Psychiat, Melbourne Neuropsychiat Ctr, Melbourne, Vic 3010, Australia.
RP Bora, E (reprint author), Univ Melbourne, Dept Psychiat, Melbourne Neuropsychiat Ctr, Melbourne, Vic 3010, Australia.
EM emrebora@hotmail.com
RI bora, emre/D-4123-2009
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NR 34
TC 48
Z9 52
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0165-1781
J9 PSYCHIAT RES
JI Psychiatry Res.
PD JUL 15
PY 2008
VL 160
IS 1
BP 23
EP 29
DI 10.1016/j.psychres.2007.05.017
PG 7
WC Psychiatry
SC Psychiatry
GA 326GE
UT WOS:000257645800004
PM 18514324
ER
PT J
AU Hardan, AY
Minshew, NJ
Melhem, NM
Srihari, S
Jo, B
Bansal, R
Keshavan, MS
Stanley, JA
AF Hardan, Antonio Y.
Minshew, Nancy J.
Melhem, Nadine M.
Srihari, Sumana
Jo, Booil
Bansal, Rahul
Keshavan, Matcheri S.
Stanley, Jeffrey A.
TI An MRI and proton spectroscopy study of the thalamus in children with
autism
SO PSYCHIATRY RESEARCH-NEUROIMAGING
LA English
DT Article
DE sensory profile questionnaire (SPQ); sensory abnormalities;
N-acetylaspartate (NAA)
ID SENSORY PROFILE; BRAIN SIZE; AGE; INFANCY; MOTOR; ABNORMALITIES;
INTERVENTIONS; INDIVIDUALS; PERFORMANCE; METABOLITES
AB Thalamic alterations have been reported in autism, but the relationships between these abnormalities and clinical symptoms, specifically sensory features, have not been elucidated. The goal of this investigation is to combine two neuroimaging methods to examine further the pathophysiology of thalamic anomalies in autism and to identify any association with sensory deficits. Structural MRI and multi-voxel, short echo-time proton magnetic resonance spectroscopy (H-1 MRS) measurements were collected from 18 male children with autism and 16 healthy children. Anatomical measurements of thalamic nuclei and absolute concentration levels of key H-1 MRS metabolites were obtained. Sensory abnormalities were assessed using a sensory profile questionnaire. Lower levels of N-acetylaspartate (NAA), phosphocreatine and creatine, and choline-containing metabolites were observed on the left side in the autism group compared with controls. No differences in thalamic volumes were observed between the two groups. Relationships, although limited, were observed between measures of sensory abnormalities and H-1 MRS metabolites. Findings from this study support the role of the thalamus in the pathophysiology of autism and more specifically in the sensory abnormalities observed in this disorder. Further investigations of this structure are warranted, since it plays an important role in information processing as part of the cortico-thalamo-cortical pathways. (c) 2008 Elsevier Ireland Ltd. All rights reserved.
C1 [Hardan, Antonio Y.; Jo, Booil] Stanford Univ, Dept Psychiat & Behav Sci, Stanford, CA 94305 USA.
[Minshew, Nancy J.; Melhem, Nadine M.; Srihari, Sumana] Univ Pittsburgh, Western Psychiat Inst & Clin, Dept Psychiat, Pittsburgh, PA 15213 USA.
[Bansal, Rahul; Keshavan, Matcheri S.; Stanley, Jeffrey A.] Wayne State Univ, Dept Psychiat & Behav Neurosci, Detroit, MI USA.
RP Hardan, AY (reprint author), 401 Quarry Rd, Stanford, CA 94305 USA.
EM hardanay@stanford.edu
RI Melhem, Nadine/G-1510-2013
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 47
TC 46
Z9 52
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0925-4927
J9 PSYCHIAT RES-NEUROIM
JI Psychiatry Res. Neuroimaging
PD JUL 15
PY 2008
VL 163
IS 2
BP 97
EP 105
DI 10.1016/j.pscychresns.2007.12.002
PG 9
WC Clinical Neurology; Neuroimaging; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 325HY
UT WOS:000257581200001
PM 18508243
ER
PT J
AU Vidal, CN
Nicolson, R
Boire, JY
Barra, V
DeVito, TJ
Hayashi, KM
Geage, JA
Drost, DJ
Williamson, PC
Rajakumar, N
Toga, AW
Thompson, PM
AF Vidal, Christine N.
Nicolson, Rob
Boire, Jean-Yves
Barra, Vincent
DeVito, Timothy J.
Hayashi, Kiralee M.
Geage, Jennifer A.
Drost, Dick J.
Williamson, Peter C.
Rajakumar, Nagalingam
Toga, Arthur W.
Thompson, Paul M.
TI Three-dimensional mapping of the lateral ventricles in autism
SO PSYCHIATRY RESEARCH-NEUROIMAGING
LA English
DT Article; Proceedings Paper
CT 11th Annual Meeting of the Organization-for-Human-Brain-Mapping
CY JUN 12-16, 2005
CL Toronto, CANADA
SP Org Human Brain Mapping
DE autism; lateral ventricles; brain; MRI
ID CEREBRAL-CORTEX; BRAIN STRUCTURE; REGIONAL BRAIN; MATTER VOLUME;
ABNORMALITIES; DISORDERS; SCHIZOPHRENIA; CHILDREN; SIZE; MRI
AB In this study, a computational mapping technique was used to examine the three-dimensional profile of the lateral ventricles in autism. T1-weighted three-dimensional magnetic resonance images of the brain were acquired from 20 males with autism (age: 10.1 +/- 3.5 years) and 22 male control subjects (age: 10.7 +/- 2.5 years). The lateral ventricles were delineated manually and ventricular volumes were compared between the two groups. Ventricular traces were also converted into statistical three-dimensional maps, based on anatomical surface meshes. These maps were used to visualize regional morphological differences in the thickness of the lateral ventricles between patients and controls. Although ventricular volumes measured using traditional methods did not differ significantly between groups, statistical surface maps revealed subtle, highly localized reductions in ventricular size in patients with autism in the left frontal and occipital horns. These localized reductions in the lateral ventricles may result from exaggerated brain growth early in life. (c) 2007 Elsevier Ireland Ltd. All rights reserved.
C1 [Nicolson, Rob] Univ Western Ontario, Dept Psychiat, London Hlth Sci Ctr, London, ON N6C 2V5, Canada.
[Vidal, Christine N.; Hayashi, Kiralee M.; Geage, Jennifer A.; Toga, Arthur W.; Thompson, Paul M.] Univ Calif Los Angeles, Sch Med, Dept Neurol, Lab Neuro Imaging,Brain Mapping Div, Los Angeles, CA 90024 USA.
[Nicolson, Rob; DeVito, Timothy J.; Drost, Dick J.; Williamson, Peter C.] Univ Western Ontario, Dept Med Biophys, London, ON N6C 2V5, Canada.
[Boire, Jean-Yves] INSERM, Fac Med, Clermont Ferrand, France.
[Barra, Vincent] Dept Comp Sci, Aubiere, France.
RP Nicolson, R (reprint author), Univ Western Ontario, Dept Psychiat, London Hlth Sci Ctr, 800 Commissioners Rd E,E2-601, London, ON N6C 2V5, Canada.
EM Rnicolso@uwo.ca
RI Nicolson, Robert/E-4797-2011; Williamson, Peter/F-7462-2010
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NR 51
TC 13
Z9 13
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0925-4927
J9 PSYCHIAT RES-NEUROIM
JI Psychiatry Res. Neuroimaging
PD JUL 15
PY 2008
VL 163
IS 2
BP 106
EP 115
DI 10.1016/j.pscychresns.2007.11.002
PG 10
WC Clinical Neurology; Neuroimaging; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 325HY
UT WOS:000257581200002
PM 18502618
ER
PT J
AU Sutcliffe, JS
AF Sutcliffe, James S.
TI Genetics - Insights into the pathogenesis of autism
SO SCIENCE
LA English
DT Editorial Material
ID SPECTRUM DISORDERS; DIAGNOSIS; 16P11.2; RISK
C1 [Sutcliffe, James S.] Vanderbilt Univ, Dept Mol Physiol & Biophys, Nashville, TN 37232 USA.
RP Sutcliffe, JS (reprint author), Vanderbilt Univ, Dept Mol Physiol & Biophys, Nashville, TN 37232 USA.
EM james.s.sutcliffe@vanderbilt.edu
RI Sutcliffe, James/C-1348-2012
OI Sutcliffe, James/0000-0001-5200-6007
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NR 12
TC 31
Z9 31
PU AMER ASSOC ADVANCEMENT SCIENCE
PI WASHINGTON
PA 1200 NEW YORK AVE, NW, WASHINGTON, DC 20005 USA
SN 0036-8075
J9 SCIENCE
JI Science
PD JUL 11
PY 2008
VL 321
IS 5886
BP 208
EP 209
DI 10.1126/science.1160555
PG 2
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 324IS
UT WOS:000257512900026
PM 18621658
ER
PT J
AU Morrow, EM
Yoo, SY
Flavell, SW
Kim, TK
Lin, YX
Hill, RS
Mukaddes, NM
Balkhy, S
Gascon, G
Hashmi, A
Al-Saad, S
Ware, J
Joseph, RM
Greenblatt, R
Gleason, D
Ertelt, JA
Apse, KA
Bodell, A
Partlow, JN
Barry, B
Yao, H
Markianos, K
Ferland, RJ
Greenberg, ME
Walsh, CA
AF Morrow, Eric M.
Yoo, Seung-Yun
Flavell, Steven W.
Kim, Tae-Kyung
Lin, Yingxi
Hill, Robert Sean
Mukaddes, Nahit M.
Balkhy, Soher
Gascon, Generoso
Hashmi, Asif
Al-Saad, Samira
Ware, Janice
Joseph, Robert M.
Greenblatt, Rachel
Gleason, Danielle
Ertelt, Julia A.
Apse, Kira A.
Bodell, Adria
Partlow, Jennifer N.
Barry, Brenda
Yao, Hui
Markianos, Kyriacos
Ferland, Russell J.
Greenberg, Michael E.
Walsh, Christopher A.
TI Identifying autism loci and genes by tracing recent shared ancestry
SO SCIENCE
LA English
DT Article
ID SPECTRUM DISORDERS; CHROMOSOMAL REARRANGEMENTS; TRANSCRIPTION FACTORS;
MUTATIONS; LINKAGE; PROTEIN; MEMBER; ASSOCIATION; SUPERFAMILY;
INHERITANCE
AB To find inherited causes of autism- spectrum disorders, we studied families in which parents share ancestors, enhancing the role of inherited factors. We mapped several loci, some containing large, inherited, homozygous deletions that are likely mutations. The largest deletions implicated genes, including PCDH10 ( protocadherin 10) and DIA1 ( deleted in autism1, or c3orf58), whose level of expression changes in response to neuronal activity, a marker of genes involved in synaptic changes that underlie learning. A subset of genes, including NHE9 (Na(+)/H(+) exchanger 9), showed additional potential mutations in patients with unrelated parents. Our findings highlight the utility of "homozygosity mapping" in heterogeneous disorders like autism but also suggest that defective regulation of gene expression after neural activity may be a mechanism common to seemingly diverse autism mutations.
C1 [Morrow, Eric M.; Yoo, Seung-Yun; Hill, Robert Sean; Greenblatt, Rachel; Gleason, Danielle; Ertelt, Julia A.; Apse, Kira A.; Bodell, Adria; Partlow, Jennifer N.; Barry, Brenda; Yao, Hui; Markianos, Kyriacos; Walsh, Christopher A.] Childrens Hosp, Div Genet, Boston, MA 02115 USA.
[Morrow, Eric M.; Yoo, Seung-Yun; Hill, Robert Sean; Greenblatt, Rachel; Gleason, Danielle; Ertelt, Julia A.; Apse, Kira A.; Bodell, Adria; Partlow, Jennifer N.; Barry, Brenda; Yao, Hui; Markianos, Kyriacos; Walsh, Christopher A.] Harvard Univ, Sch Med, Boston, MA 02115 USA.
[Morrow, Eric M.; Yoo, Seung-Yun; Hill, Robert Sean; Greenblatt, Rachel; Gleason, Danielle; Ertelt, Julia A.; Apse, Kira A.; Bodell, Adria; Partlow, Jennifer N.; Barry, Brenda; Walsh, Christopher A.] Beth Israel Deaconess Med Ctr, Dept Neurol, Boston, MA 02115 USA.
[Morrow, Eric M.; Yoo, Seung-Yun; Hill, Robert Sean; Greenblatt, Rachel; Gleason, Danielle; Ertelt, Julia A.; Apse, Kira A.; Bodell, Adria; Partlow, Jennifer N.; Barry, Brenda; Walsh, Christopher A.] Beth Israel Deaconess Med Ctr, Howard Hughes Med Inst, Boston, MA 02115 USA.
[Morrow, Eric M.] Massachusetts Gen Hosp, Dept Psychiat, Boston, MA 02114 USA.
[Morrow, Eric M.; Yoo, Seung-Yun; Hill, Robert Sean; Walsh, Christopher A.] MIT, Program Med & Populat Genet, Broad Inst, Cambridge, MA 02142 USA.
[Morrow, Eric M.; Yoo, Seung-Yun; Hill, Robert Sean; Walsh, Christopher A.] Harvard Univ, Cambridge, MA 02142 USA.
[Morrow, Eric M.; Yoo, Seung-Yun; Flavell, Steven W.; Kim, Tae-Kyung; Lin, Yingxi; Hill, Robert Sean; Ware, Janice; Joseph, Robert M.; Apse, Kira A.; Greenberg, Michael E.; Walsh, Christopher A.] Autism Consortium, Boston, MA 02115 USA.
[Flavell, Steven W.; Kim, Tae-Kyung; Lin, Yingxi; Greenberg, Michael E.] Childrens Hosp, FM Kirby Neurobiol Ctr, Boston, MA 02115 USA.
[Flavell, Steven W.; Kim, Tae-Kyung; Lin, Yingxi; Greenberg, Michael E.] Harvard Univ, Sch Med, Dept Neurol, Boston, MA 02115 USA.
[Flavell, Steven W.; Kim, Tae-Kyung; Lin, Yingxi; Greenberg, Michael E.] Harvard Univ, Sch Med, Dept Neurobiol, Boston, MA 02115 USA.
[Mukaddes, Nahit M.] Istanbul Univ, Istanbul Fac Med, Dept Child Psychiat, Istanbul, Turkey.
[Balkhy, Soher; Gascon, Generoso] King Faisal Specialist Hosp & Res Ctr, Dept Neurosci & Pediat, Jeddah, Saudi Arabia.
[Gascon, Generoso] Brown Univ, Sch Med, Providence, RI 02912 USA.
[Hashmi, Asif] Combined Mil Hosp, Dept Neurol, Lahore, Pakistan.
[Al-Saad, Samira] Kuwait Ctr Autism, Kuwait, Kuwait.
[Ware, Janice] Childrens Hosp, Dev Med Ctr, Boston, MA 02115 USA.
[Joseph, Robert M.] Boston Univ, Sch Med, Dept Anat & Neurobiol, Boston, MA 02118 USA.
[Ferland, Russell J.] Rensselaer Polytech Inst, Dept Biol, Troy, NY 12180 USA.
RP Walsh, CA (reprint author), Childrens Hosp, Div Genet, 300 Longwood Ave, Boston, MA 02115 USA.
EM christopher.walsh@childrens.harvard.edu
RI Ferland, Russell/E-3500-2010; Morrow, Eric/J-2767-2013; Joseph,
Roy/D-8530-2015
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NR 43
TC 335
Z9 343
PU AMER ASSOC ADVANCEMENT SCIENCE
PI WASHINGTON
PA 1200 NEW YORK AVE, NW, WASHINGTON, DC 20005 USA
SN 0036-8075
J9 SCIENCE
JI Science
PD JUL 11
PY 2008
VL 321
IS 5886
BP 218
EP 223
DI 10.1126/science.1157657
PG 6
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 324IS
UT WOS:000257512900031
PM 18621663
ER
PT J
AU Kurian, JR
Bychowski, ME
Forbes-Lorman, RM
Auger, CJ
Auger, AP
AF Kurian, Joseph R.
Bychowski, Meaghan E.
Forbes-Lorman, Robin M.
Auger, Catherine J.
Auger, Anthony P.
TI Mecp2 organizes juvenile social behavior in a sex-specific manner
SO JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE Mecp2; siRNA; amygdala; sex difference; behavior; epigenetic
ID RETT-SYNDROME; PLAY; EXPRESSION; RATS; TESTOSTERONE; AUTISM; PERIOD;
MUTATIONS; RELEVANT; PROTEINS
AB Methyl-CpG-binding protein 2 (MeCP2) binds methylated DNA and recruits corepressor proteins to modify chromatin and alter gene transcription. Mutations of the MECP2 gene can cause Rett syndrome, whereas subtle reductions of MeCP2 expression may be associated with male-dominated social and neurodevelopmental disorders. We report that transiently decreased amygdala Mecp2 expression during a sensitive period of brain sexual differentiation disrupts the organization of sex differences in juvenile social play behavior. Interestingly, neonatal treatment with Mecp2 small interfering RNA within the developing amygdala reduced juvenile social play behavior in males but not females. Reduced Mecp2 expression did not change juvenile sociability or anxiety-like behavior, suggesting that this disruption is associated with subtle behavioral modification. This suggests that Mecp2 may have an overlooked role in the organization of sexually dimorphic behaviors and that male juvenile behavior is particularly sensitive to Mecp2 disruption during this period of development.
C1 [Kurian, Joseph R.; Forbes-Lorman, Robin M.; Auger, Catherine J.; Auger, Anthony P.] Univ Wisconsin, Dept Psychol, Madison, WI 53706 USA.
[Bychowski, Meaghan E.; Auger, Catherine J.; Auger, Anthony P.] Univ Wisconsin, Neurosci Training Program, Madison, WI 53706 USA.
RP Auger, AP (reprint author), Univ Wisconsin, Dept Psychol, 1202 W Johnson St, Madison, WI 53706 USA.
EM apauger@wisc.edu
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NR 28
TC 42
Z9 42
PU SOC NEUROSCIENCE
PI WASHINGTON
PA 11 DUPONT CIRCLE, NW, STE 500, WASHINGTON, DC 20036 USA
SN 0270-6474
J9 J NEUROSCI
JI J. Neurosci.
PD JUL 9
PY 2008
VL 28
IS 28
BP 7137
EP 7142
DI 10.1523/JNEUROSCI.1345-08.2008
PG 6
WC Neurosciences
SC Neurosciences & Neurology
GA 323TT
UT WOS:000257472000015
PM 18614683
ER
PT J
AU Li, H
Radford, JC
Ragusa, MJ
Shea, KL
McKercher, SR
Zaremba, JD
Soussou, W
Nie, Z
Kang, YJ
Nakanishi, N
Okamoto, SI
Roberts, AJ
Schwarz, JJ
Lipton, SA
AF Li, Hao
Radford, Jonathan C.
Ragusa, Michael J.
Shea, Katherine L.
McKercher, Scott R.
Zaremba, Jeffrey D.
Soussou, Walid
Nie, Zhiguo
Kang, Yeon-Joo
Nakanishi, Nobuki
Okamoto, Shu-ichi
Roberts, Amanda J.
Schwarz, John J.
Lipton, Stuart A.
TI Transcription factor MEF2C influences neural stem/progenitor cell
differentiation and maturation in vivo
SO PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF
AMERICA
LA English
DT Article
DE neurogenesis; synaptogenesis; autism; Rett syndrome
ID RETT-SYNDROME; MOUSE EMBRYOGENESIS; CEREBRAL-CORTEX; MECP2; MICE;
NEURONS; PROTEIN; GENE; DEFICIENCY; EXPRESSION
AB Emerging evidence suggests that myocyte enhancer factor 2 (MEF2) transcription factors act as effectors of neurogenesis in the brain, with MEF2C the predominant isoform in developing cerebrocortex. Here, we show that conditional knockout of Mef2c in nestin-expressing neural stem/progenitor cells (NSCs) impaired neuronal differentiation in vivo, resulting in aberrant compaction and smaller somal size. NSC proliferation and survival were not affected. Conditional null mice surviving to adulthood manifested more immature electrophysiological network properties and severe behavioral deficits reminiscent of Rett syndrome, an autism-related disorder. Our data support a crucial role for MEF2C in programming early neuronal differentiation and proper distribution within the layers of the neocortex.
C1 [Li, Hao; Radford, Jonathan C.; McKercher, Scott R.; Zaremba, Jeffrey D.; Soussou, Walid; Nie, Zhiguo; Kang, Yeon-Joo; Nakanishi, Nobuki; Okamoto, Shu-ichi; Lipton, Stuart A.] Burnham Inst Med Res, Ctr Neurosci Aging & Stem Cell Res, La Jolla, CA 92037 USA.
[Ragusa, Michael J.; Shea, Katherine L.; Schwarz, John J.] Albany Med Ctr, Ctr Cardiovasc Sci, Albany, NY 12208 USA.
[Roberts, Amanda J.; Lipton, Stuart A.] Scripps Res Inst, Mol & Integrat Neurosci Dept, La Jolla, CA 92037 USA.
RP Lipton, SA (reprint author), Burnham Inst Med Res, Ctr Neurosci Aging & Stem Cell Res, La Jolla, CA 92037 USA.
EM slipton@burnham.org
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NR 28
TC 73
Z9 77
PU NATL ACAD SCIENCES
PI WASHINGTON
PA 2101 CONSTITUTION AVE NW, WASHINGTON, DC 20418 USA
SN 0027-8424
J9 P NATL ACAD SCI USA
JI Proc. Natl. Acad. Sci. U. S. A.
PD JUL 8
PY 2008
VL 105
IS 27
BP 9397
EP 9402
DI 10.1073/pnas.0802876105
PG 6
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 326GA
UT WOS:000257645400048
PM 18599437
ER
PT J
AU Hettinger, JA
Liu, XD
Schwartz, CE
Michaelis, RC
Holden, JJA
AF Hettinger, Joe A.
Liu, Xudong
Schwartz, Charles E.
Michaelis, Ron C.
Holden, Jeanette J. A.
TI A DRD1 haplotype is associated with risk for autism spectrum disorders
in male-only affected sib-pair families
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART B-NEUROPSYCHIATRIC GENETICS
LA English
DT Review
DE autism spectrum disorders; dopamine D1 receptor; family-based
association tests; case-control study; candidate gene
ID PERVASIVE DEVELOPMENTAL DISORDERS; DOPAMINE-RECEPTOR GENE;
ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; OBSESSIVE-COMPULSIVE DISORDER;
LYMPHOBLASTOID CELL-LINES; PRIMATE PREFRONTAL CORTEX; SPATIAL
WORKING-MEMORY; CARD SORTING TEST; BETA-HYDROXYLASE; EXECUTIVE
DYSFUNCTION
AB Individuals with autism spectrum disorders (ASDs) have impairments in executive function and social cognition, with males generally being more severely affected in these areas than females. Because the dopamine D1 receptor (encoded by DRD1) is integral to the neural circuitry mediating these processes, we examined the DRD1 gene for its role in susceptibility to ASDs by performing single marker and haplotype case-control comparisons, family-based association tests, and genotype-phenotype assessments (quantitative transmission disequilibrium. tests: QTDT) using three DRD1 polymorphisms, rs265981C/T, rs4532A/G, and rs686T/C. Our previous findings suggested that the dopaminergic system may be more integrally involved in families with affected males only than in other families. We therefore restricted our study to families with two or more affected males (N = 112). There was over-transmission of rs265981-C and rs4532-A in these families (P = 0.040, P = 0.038), with haplotype TDT analysis showing over-transmission of the C-A-T haplotype (P=0.022) from mothers to affected sons (P = 0.013). In addition, haplotype case-control comparisons revealed an increase of this putative risk haplotype in affected individuals relative to a comparison group (P = 0.004). QTDT analyses showed associations of the rs265981-C, rs4532-A, rs686-T alleles, and the C-A-T haplotype with more severe problems in social interaction, greater difficulties with nonverbal communication and increased stereotypies compared to individuals with other haplotypes. Preferential haplotype transmission of markers at the DRD1 locus and an increased frequency of a specific haplotype support the DRD1 gene as a risk gene for core symptoms of ASD in families having only affected males. (C) 2008 Wiley-Liss, Inc.
C1 [Hettinger, Joe A.; Liu, Xudong; Holden, Jeanette J. A.] Ongwanada Resource Ctr, Autism Res Program, Kingston, ON K7M 8A6, Canada.
[Hettinger, Joe A.; Holden, Jeanette J. A.] Queens Univ, Dept Psychol, Kingston, ON K7L 3N6, Canada.
[Liu, Xudong; Holden, Jeanette J. A.] Queens Univ, Dept Psychiat, Kingston, ON K7L 3N6, Canada.
[Schwartz, Charles E.] Greenwood Genet Ctr, Ctr Mol Studies, Greenwood, SC 29646 USA.
[Michaelis, Ron C.] Western Carolina Univ, Dept Biol, Cullowhee, NC 28723 USA.
[Holden, Jeanette J. A.] Autism Spectrum Disorders Canadian American Res C, Kingston, ON, Canada.
[Holden, Jeanette J. A.] Queens Univ, Ctr Neurosci Studies, Kingston, ON, Canada.
RP Holden, JJA (reprint author), Ongwanada Resource Ctr, Autism Res Program, 191 Portsmouth Ave, Kingston, ON K7M 8A6, Canada.
EM holdenj@queensu.ca
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NR 107
TC 31
Z9 35
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1552-4841
J9 AM J MED GENET B
JI Am. J. Med. Genet. B
PD JUL 5
PY 2008
VL 147B
IS 5
BP 628
EP 636
DI 10.1002/ajmg.b.30655
PG 9
WC Genetics & Heredity; Psychiatry
SC Genetics & Heredity; Psychiatry
GA 323LG
UT WOS:000257447000013
PM 18205172
ER
PT J
AU Rommelse, NNJ
Arias-Vasquez, A
Altink, ME
Buschgens, CJM
Fliers, E
Asherson, P
Faraone, SV
Buitelaar, JK
Sergeant, JA
Osterlaan, J
Franke, B
AF Rommelse, Nanda N. J.
Arias-Vasquez, Alejandro
Altink, Marieke E.
Buschgens, Cathelijne J. M.
Fliers, Ellen
Asherson, Philip
Faraone, Stephen V.
Buitelaar, Jan K.
Sergeant, Joseph A.
Osterlaan, Jaap
Franke, Barbara
TI Neuropsychological endophenotype approach to genome-wide linkage
analysis identifies susceptibility loci for ADHD on 2q21.1 and 13q12.11
SO AMERICAN JOURNAL OF HUMAN GENETICS
LA English
DT Article
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; CHILDREN; AUTISM; ASSOCIATION;
DYSLEXIA; ADOLESCENTS; SIBLINGS; DEFICITS; ADULTS; SCAN
AB ADHD linkage findings have not all been consistently replicated, suggesting that other approaches to linkage analysis in ADHD might be necessary, such as the use of (quantitative) endophenotypes (heritable traits associated with an increased risk for ADHD). Genome-wide linkage analyses were performed in the Dutch subsample of the International Multi-Center ADHD Genetics (IMAGE) study comprising 238 DSM-IV combined-type ADHD probands and their 112 affected and 195 nonaffected siblings. Eight candidate neuropsychological ADHD endophenotypes with heritabilities > 0.2 were used as quantitative traits. In addition, an overall component score of neuropsychological functioning was used. A total of 5407 autosomal single-nucleotide polymorphisms (SNPs) were used to run multipoint regression-based linkage analyses. Two significant genome-wide linkage signals were found, one for Motor Timing on chromosome 2q21.1 (LOD score: 3.944) and one for Digit Span on 13q12.11 (LOD score: 3.959). Ten suggestive linkage signals were found (LOD scores > 2) on chromosomes 2p, 2q, 3p, 4q, 8q, 12p, 12q, 14q, and 17q. The suggestive linkage signal for the component score that was found at 2q14.3 (LOD score: 2.878) overlapped with the region significantly linked to Motor Timing. Endophenotype approaches may increase power to detect susceptibility loci in ADHD and possibly in other complex disorders.
C1 [Rommelse, Nanda N. J.; Arias-Vasquez, Alejandro; Altink, Marieke E.; Buschgens, Cathelijne J. M.; Fliers, Ellen; Buitelaar, Jan K.; Franke, Barbara] Radboud Univ Nijmegen, Dept Psychiat, Donders Ctr Neurosci, Med Ctr, NL-6525 ED Nijmegen, Netherlands.
[Rommelse, Nanda N. J.; Sergeant, Joseph A.; Osterlaan, Jaap] Vrije Univ Amsterdam, Dept Clin Neuropsychol, Amsterdam, Netherlands.
[Arias-Vasquez, Alejandro; Franke, Barbara] Radboud Univ Nijmegen, Dept Human Genet, Med Ctr, NL-6525 ED Nijmegen, Netherlands.
[Arias-Vasquez, Alejandro] Radboud Univ Nijmegen, Dept Epidemiol & Biostat, Med Ctr, NL-6525 ED Nijmegen, Netherlands.
[Asherson, Philip] Kings Coll London, MRC Social Genet & Dev Psychiat Ctr, London SE5 A87, England.
[Faraone, Stephen V.] SUNY Upstate Med Univ, Dept Psychiat & Neurosci, Syracuse, NY USA.
[Faraone, Stephen V.] SUNY Upstate Med Univ, Dept Physiol, Syracuse, NY USA.
RP Franke, B (reprint author), Radboud Univ Nijmegen, Dept Psychiat, Donders Ctr Neurosci, Med Ctr, NL-6525 ED Nijmegen, Netherlands.
EM b.franke@antrg.umcn.nl
RI Franke, Barbara/D-4836-2009; Arias Vasquez, Alejandro/E-4762-2012;
Buitelaar, Jan/E-4584-2012; Rommelse, Nanda/D-4872-2009
OI Franke, Barbara/0000-0003-4375-6572; Arias Vasquez,
Alejandro/0000-0002-4786-0169; Buitelaar, Jan/0000-0001-8288-7757;
Rommelse, Nanda/0000-0002-1711-0359
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NR 35
TC 41
Z9 41
PU CELL PRESS
PI CAMBRIDGE
PA 600 TECHNOLOGY SQUARE, 5TH FLOOR, CAMBRIDGE, MA 02139 USA
SN 0002-9297
J9 AM J HUM GENET
JI Am. J. Hum. Genet.
PD JUL
PY 2008
VL 83
IS 1
BP 99
EP 105
DI 10.1016/j.ajhg.2008.06.006
PG 7
WC Genetics & Heredity
SC Genetics & Heredity
GA 328FO
UT WOS:000257784000012
PM 18599010
ER
PT J
AU Case-Smith, J
Arbesman, M
AF Case-Smith, Jane
Arbesman, Marian
TI Evidence-based review of interventions for autism used in or of
relevance to occupational therapy
SO AMERICAN JOURNAL OF OCCUPATIONAL THERAPY
LA English
DT Article
DE autism spectrum disorder; pediatrics; systematic review
ID PERVASIVE DEVELOPMENTAL DISORDERS; YOUNG-CHILDREN; PRESCHOOL-CHILDREN;
INTEGRATION; BEHAVIOR; PROGRAM; PARENTS; RESPONSIVITY; DISABILITIES;
BENEFITS
AB Occupational therapy practitioners are among the professionals who provide services to children and adults with autism spectrum disorder (ASD), embracing both leadership and supportive roles in service delivery. The study's primary aims were as follows: (1) to identify, evaluate, and synthesize the research literature on interventions for ASD of relevance to occupational therapy and (2) to interpret and apply the research literature to occupational therapy. A total of 49 articles met the authors' criteria and were included in the review. Six categories of research topics were identified, the first 3 of which are most closely related to occupational therapy: (1) sensory integration and sensory-based interventions, (2) relationship-based, interactive interventions, (3) developmental skill-based programs, (4) social cognitive skill training, (5) parent-directed or parent-mediated approaches; and (6) intensive behavioral intervention. Under each category, themes supported by research evidence and applicable to occupational therapy were defined. The findings have implications for intervention methods, communication regarding efficacious practices to professionals and consumers, and future occupational therapy research.
C1 [Case-Smith, Jane] Ohio State Univ, Div Occupat Therapy, Sch Allied Med Profess, Columbus, OH 43210 USA.
[Arbesman, Marian] Arbesldeas Inc, Williamsville, NY USA.
[Arbesman, Marian] AOTA Evidence Based Literature Review Project, Williamsville, NY USA.
RP Case-Smith, J (reprint author), Ohio State Univ, Div Occupat Therapy, Sch Allied Med Profess, Columbus, OH 43210 USA.
EM jane.case-smith@osumc.edu
RI Case-smith, Jane/E-2849-2011
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NR 65
TC 36
Z9 37
PU AMER OCCUPATIONAL THERAPY ASSOC, INC
PI BETHESDA
PA 4720 MONTGOMERY LANE, BETHESDA, MD 20814-3425 USA
SN 0272-9490
J9 AM J OCCUP THER
JI Am. J. Occup. Ther.
PD JUL-AUG
PY 2008
VL 62
IS 4
BP 416
EP 429
PG 14
WC Rehabilitation
SC Rehabilitation
GA 330MB
UT WOS:000257944600006
PM 18712004
ER
PT J
AU Moss, JF
Oliver, C
Berg, K
Kaur, G
Jephcott, L
Cornish, K
AF Moss, Joanna F.
Oliver, Chris
Berg, Katy
Kaur, Gurmeash
Jephcott, Lesley
Cornish, Kim
TI Prevalence of Autism spectrum phenomenology in Cornelia de Lange and Cri
du Chat syndromes
SO AMERICAN JOURNAL ON MENTAL RETARDATION
LA English
DT Article
ID DE-LANGE-SYNDROME; GENOTYPE-PHENOTYPE CORRELATIONS;
BEHAVIORAL-PHENOTYPE; MENTAL-RETARDATION; PROGNOSTIC INDICATORS;
DELANGE-SYNDROME; LANGUAGE-SKILLS; COHEN-SYNDROME; NIPPED-B; INDIVIDUALS
AB Autism spectrum disorder characteristics have not been evaluated in Cornelia de Lange and Cri du Chat syndromes using robust assessments. The Autism Diagnostic Observation Schedule and Social Communication Questionnaire were administered to 34 participants with Cornelia de Lange syndrome and a comparison group of 23 participants with Cri du Chat syndrome (M ages 12.4 [SD = 3.8] and 10.3 years [SD = 3.6], respectively). Twenty-one participants with Cornelia de Lange syndrome (61.8%) scored above the autism cutoff on the Autism Diagnostic Observation Schedule compared to 9 with Cri du Chat syndrome (39.2%). Prevalence of autism spectrum disorder characteristics is heightened in Cornelia de Lange syndrome. The profile of characteristics is atypical to that of idiopathic autism.
C1 [Moss, Joanna F.; Oliver, Chris; Berg, Katy; Kaur, Gurmeash; Jephcott, Lesley] Univ Birmingham, Sch Psychol, Ctr Neurodev Disorders, Birmingham B15 2TT, W Midlands, England.
[Cornish, Kim] McGill Univ, Montreal, PQ, Canada.
RP Oliver, C (reprint author), Univ Birmingham, Sch Psychol, Ctr Neurodev Disorders, Birmingham B15 2TT, W Midlands, England.
EM c.oliver@bham.ac.uk
RI Moss, Jo/C-8812-2009
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World Health Organisation, 1992, ICD 10 CLASS MENT BE
NR 60
TC 28
Z9 28
PU AMER ASSOC MENTAL RETARDATION
PI WASHINGTON
PA 444 N CAPITOL ST, NW, STE 846, WASHINGTON, DC 20001-1512 USA
SN 0895-8017
J9 AM J MENT RETARD
JI Am. J. Ment. Retard.
PD JUL
PY 2008
VL 113
IS 4
BP 278
EP 291
DI 10.1352/0895-8017(2008)113[278:POASPI]2.0.CO;2
PG 14
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 327VU
UT WOS:000257757400004
PM 18564888
ER
PT J
AU Barisnikov, K
Hippolyte, L
Van der Linden, M
AF Barisnikov, Koviijka
Hippolyte, Loyse
Van der Linden, Martial
TI Face processing and facial emotion recognition in adults with Down
syndrome
SO AMERICAN JOURNAL ON MENTAL RETARDATION
LA English
DT Article
ID FRAGILE-X-SYNDROME; MENTAL-RETARDATION; NONRETARDED-CHILDREN;
WILLIAMS-SYNDROME; EXPRESSIONS; AUTISM; PERCEPTION; ADOLESCENTS;
PERFORMANCE; MEMORY
AB Face processing and facial expression recognition was investigated in 17 adults with Down syndrome, and results were compared with those of a child control group matched for receptive vocabulary. On the tasks involving faces without emotional content, the adults with Down syndrome performed significantly worse than did the controls. However, their performance was good on the tests with complete faces. On the facial expression tasks, participants with Down syndrome exhibited particular difficulties with the neutral and surprised expressions. Analysis of their error pattern suggest they had a tendency to judge faces more positively than did the controls. Finally, there were significant relationships among emotional processing, receptive vocabulary, and inhibition measures; nonverbal reasoning ability was not related to any of the tasks.
C1 Univ Geneva, Child Clin Neuropsychol Unit, CH-1211 Geneva, Switzerland.
RP Barisnikov, K (reprint author), Univ Geneva, Child Clin Neuropsychol Unit, CH-1211 Geneva, Switzerland.
EM Koviljka.Barisnikov@pse.unige.ch
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NR 49
TC 12
Z9 12
PU AMER ASSOC MENTAL RETARDATION
PI WASHINGTON
PA 444 N CAPITOL ST, NW, STE 846, WASHINGTON, DC 20001-1512 USA
SN 0895-8017
J9 AM J MENT RETARD
JI Am. J. Ment. Retard.
PD JUL
PY 2008
VL 113
IS 4
BP 292
EP 306
DI 10.1352/0895-8017(2008)113[292:FPAFER]2.0.CO;2
PG 15
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 327VU
UT WOS:000257757400005
ER
PT J
AU Bitsika, V
AF Bitsika, Vicki
TI Including an analysis of difficult behaviour in the assessment of
children with an autism spectrum disorder: Implications for school
psychologists
SO AUSTRALIAN JOURNAL OF GUIDANCE AND COUNSELLING
LA English
DT Article
DE ASD; assessment; behaviour; functional assessment
ID FUNCTIONAL-ANALYSIS; EPIDEMIOLOGY; FUTURE
AB School psychologists who contribute to the assessment of children with an autism spectrum disorder (ASD) are required by various Australian state government authorities to use standardised testing of cognitive skills, adaptive behaviour and some autism-based Symptomatology to demonstrate the eligibility of those children for support funding in the school environment. However, this process does not always address the needs of parents and teachers for detailed and particular analysis of the reasons for the pervasive behavioural difficulties often shown by children with an ASD. Such difficult behaviour has been shown to contribute to high levels of stress among caregivers and often prevents the child with an ASD from successful school inclusion. Therefore, it would be advantageous for parents and teachers to have access to information obtained from an analysis of the child's behavioural difficulties as one outcome of the formal testing process that is conducted to examine the possible presence of an ASD. This paper presents, a brief example of how a basic behavioural investigation might be incorporated into the assessment of children with a suspected ASD.
C1 Bond Univ, Fac Humanities & Social Sci, Gold Coast, Qld 4229, Australia.
RP Bitsika, V (reprint author), Bond Univ, Fac Humanities & Social Sci, Gold Coast, Qld 4229, Australia.
EM vbitsika@kond.edu.au
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NR 40
TC 1
Z9 1
PU AUSTRALIAN ACAD PRESS
PI BOWEN HILLS
PA 32 JEAYS ST, BOWEN HILLS, QLD 4006, AUSTRALIA
SN 1037-2911
J9 AUST J GUID COUNS
JI Aust. J. Guid. Couns.
PD JUL
PY 2008
VL 18
IS 1
BP 1
EP 14
DI 10.1375/ajgc.18.1.1
PG 14
WC Education & Educational Research; Social Work
SC Education & Educational Research; Social Work
GA 350WS
UT WOS:000259384900002
ER
PT J
AU Dahlgren, S
Sandberg, AD
AF Dahlgren, Svenolof
Sandberg, Annika Dahlgren
TI Referential communication in children with autism spectrum disorder
SO AUTISM
LA English
DT Article
DE autism spectrum disorder; memory; referential communication; theory of
mind
ID ASPERGERS-SYNDROME; LANGUAGE; MEMORY; MIND; DEFICITS; SPEAKERS; RECALL;
DELAY
AB Referential communication was studied in children with autism spectrum disorder (ASD) including children with autism and Asperger syndrome. The aim was to study alternative explanations for the children's communicative problems in such situations. Factors studied were theory of mind, IQ, verbal ability and memory. The main results demonstrated diminished performance in children with autism spectrum disorder, mirroring performance in everyday life, in comparison to verbal IQ and mental age matched typically developing children. Among children with autism spectrum disorders, there was a positive relationship between performance in referential communication and theory of mind. Memory capacity also proved to play a role in success in the task.
C1 [Dahlgren, Svenolof] Stockholm Cty Council, Stockholm, Sweden.
[Dahlgren, Svenolof; Sandberg, Annika Dahlgren] Univ Gothenburg, Gothenburg, Sweden.
RP Dahlgren, S (reprint author), Stockholm Cty Council, Stockholm, Sweden.
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NR 25
TC 5
Z9 5
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
EI 1461-7005
J9 AUTISM
JI Autism
PD JUL
PY 2008
VL 12
IS 4
BP 335
EP 348
DI 10.1177/1362361308091648
PG 14
WC Psychology, Developmental
SC Psychology
GA 331AS
UT WOS:000257985700002
PM 18579643
ER
PT J
AU Salter, G
Seigal, A
Claxton, M
Lawrence, K
Skuse, D
AF Salter, Gemma
Seigal, Anna
Claxton, Melanie
Lawrence, Kate
Skuse, David
TI Can autistic children read the mind of an animated triangle?
SO AUTISM
LA English
DT Article
DE animations; Asperger syndrome; autism; mentalizing; theory of mind
ID HIGHER-FUNCTIONING AUTISM; ASPERGER-SYNDROME; DIAGNOSTIC INTERVIEW;
SPECTRUM DISORDERS; REVISED VERSION; ATTRIBUTION; INDIVIDUALS;
PERCEPTION; CAUSALITY; INTENTION
AB Are children with an autism spectrum disorder (ASD), but normal-range intelligence, impaired on theory of mind skills measured by responses to abstract animations in the form of a computerized cartoon? Fifty-six cases and closely matched comparisons were tested. We rated verbal responses according to the length of their descriptions, their appropriateness and the children's use of 'mentalizing' terms. Children with ASD used 'mentalizing' language to describe the animations as well as comparisons, although the content of their descriptions was significantly less appropriate. Performance on this task was not well correlated with standardized measures of parent-reported behaviour or the child's interactions with an observer. The implications of our results are discussed in relation to previous studies that have used this methodology.
C1 [Salter, Gemma; Seigal, Anna; Claxton, Melanie; Lawrence, Kate; Skuse, David] Inst Child Hlth, London, England.
RP Salter, G (reprint author), Inst Child Hlth, London, England.
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NR 26
TC 12
Z9 13
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD JUL
PY 2008
VL 12
IS 4
BP 349
EP 371
DI 10.1177/1362361308091654
PG 23
WC Psychology, Developmental
SC Psychology
GA 331AS
UT WOS:000257985700003
PM 18579644
ER
PT J
AU Hutton, J
Goode, S
Murphy, M
Le Couteur, A
Rutter, M
AF Hutton, Jane
Goode, Susan
Murphy, Margaret
Le Couteur, Ann
Rutter, Michael
TI New-onset psychiatric disorders in individuals with autism
SO AUTISM
LA English
DT Article
DE affective disorder; autism; catatonia; obsessive-compulsive disorder;
schizophrenia
ID OBSESSIVE-COMPULSIVE DISORDER; SPECTRUM DISORDERS; ASPERGER-SYNDROME;
CATATONIA; CHILDREN; ADULTS; DEPRESSION; BEHAVIOR; PEOPLE; ECT
AB A follow-up study to at least the age of 21 years of 135 individuals with an autism spectrum disorder diagnosed in childhood and an IQ of over 30 was conducted. The study is distinctive in its large size, low attrition rate and use of systematic interviews to obtain clinical information. Questionnaires completed by caregivers asked about the development of new psychiatric disorders. For the 39 individuals with a possible new disorder, a detailed psychiatric assessment was undertaken through parental interview. Of all participants, 16 percent developed a definite new psychiatric disorder. A further 6 percent developed a possible new disorder. Five individuals developed an obsessive-compulsive disorder and/or catatonia; eight an affective disorder with marked obsessional features; three complex affective disorders; four more straightforward affective disorders; one a bipolar disorder; and one an acute anxiety state complicated by alcohol excess. There was no case of schizophrenia.
C1 [Hutton, Jane] Inst Psychiat, London, England.
[Goode, Susan] CAMHS, Croydon, England.
[Murphy, Margaret] Ida Darwin Hosp, Cambridge, England.
[Le Couteur, Ann] Royal Victoria Infirm, Newcastle upon Tyne NE1 4LP, Tyne & Wear, England.
[Rutter, Michael] Kings Coll London, Inst Psychiat, London WC2R 2LS, England.
RP Hutton, J (reprint author), Inst Psychiat, London, England.
RI Rutter, Michael/C-8570-2013
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World Health Organisation, 1992, ICD 10 CLASS MENT BE
Zaw FKM, 1999, DEV MED CHILD NEUROL, V41, P843, DOI 10.1017/S001216229900167X
NR 30
TC 36
Z9 36
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD JUL
PY 2008
VL 12
IS 4
BP 373
EP 390
DI 10.1177/1362361308091650
PG 18
WC Psychology, Developmental
SC Psychology
GA 331AS
UT WOS:000257985700004
PM 18579645
ER
PT J
AU Williamson, S
Craig, J
Slinger, R
AF Williamson, Sara
Craig, Jaime
Slinger, Richard
TI Exploring the relationship between measures of self-esteem and
psychological adjustment among adolescents with Asperger Syndrome
SO AUTISM
LA English
DT Article
DE adolescence; Asperger syndrome; psychological adjustment; self-esteem
ID HIGH-FUNCTIONING CHILDREN; AUTISM; COMPETENCE; ANXIETY
AB This exploratory study examines the relationships between self-esteem and psychological adjustment among 19 adolescents with Asperger syndrome and 19 typically developing adolescents using a model developed by Harter. The groups were matched for age (mean 13 years), sex (M: F 16: 3) and ethnicity (white British). Participants completed four quantitative measures examining self-competencies, social approval, anxiety, depression and self-worth. Findings revealed significant group differences: the adolescents with Asperger syndrome perceived themselves to be less competent in social and athletic domains, and to receive less peer approval. Logistic regression analyses indicated that the variables studied were particularly relevant in distinguishing group differences and worked together in predicting group membership. Indeed the variables in the final model accurately predicted group membership for all individuals in the sample. Exploratory hierarchical multiple regression analyses suggested that the two groups had different patterns of relationships between measures of self-competencies, social approval and psychological outcomes.
C1 [Williamson, Sara; Craig, Jaime] Univ Lancaster, Lancaster LA1 4YW, England.
RP Williamson, S (reprint author), Univ Lancaster, Lancaster LA1 4YW, England.
CR Attwood T., 1998, ASPERGERS SYNDROME G
Barnhill G. P., 2001, FOCUS AUTISM OTHER D, V16, P46, DOI 10.1177/108835760101600112
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NR 24
TC 14
Z9 14
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD JUL
PY 2008
VL 12
IS 4
BP 391
EP 402
DI 10.1177/1362361308091652
PG 12
WC Psychology, Developmental
SC Psychology
GA 331AS
UT WOS:000257985700005
PM 18579646
ER
PT J
AU Mouridsen, SE
Hansen, HB
Rich, B
Isager, T
AF Mouridsen, Svend Erik
Hansen, Henrik Bronnum
Rich, Bente
Isager, Torben
TI Mortality and causes of death in autism spectrum disorders - An update
SO AUTISM
LA English
DT Article
DE autism spectrum disorders; cause of death; mortality; standardized
mortality ratio
ID ADOLESCENT PSYCHIATRIC-PATIENTS; CHILD; POPULATION; REGISTER
AB This study compared mortality among Danish citizens with autism spectrum disorders (ASDs) with that of the general population. A clinical cohort of 341 Danish individuals with variants of ASD, previously followed over the period 1960-93, now on average 43 years of age, were updated with respect to mortality and causes of death. Standardized mortality ratios (SMRs) were calculated for various times after diagnosis. In all, 26 persons with ASD had died, whereas the expected number of deaths was 13.5. Thus the mortality risk among those with ASD was nearly twice that of the general population. The SMR was particularly high in females. The excess mortality risk has remained unchanged since our first study in 1993. Eight of the 26 deaths were associated with epilepsy and four died from epilepsy. Future staff education should focus on better managing of the complex relationships between ASD and physical illness to prevent avoidable deaths.
C1 [Mouridsen, Svend Erik] Bispebjerg Hosp, Copenhagen, Denmark.
[Hansen, Henrik Bronnum] Natl Inst Publ Hlth, Copenhagen, Denmark.
[Rich, Bente] Odense Univ Hosp, DK-5000 Odense, Denmark.
[Isager, Torben] Glostrup Univ Hosp, Glostrup, Denmark.
RP Mouridsen, SE (reprint author), Bispebjerg Hosp, Copenhagen, Denmark.
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World Health Organisation, 1992, ICD 10 CLASS MENT BE
World Health Organization (WHO), 1978, INT CLASS DIS MENT D
NR 24
TC 34
Z9 34
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD JUL
PY 2008
VL 12
IS 4
BP 403
EP 414
DI 10.1177/1362361308091653
PG 12
WC Psychology, Developmental
SC Psychology
GA 331AS
UT WOS:000257985700006
PM 18579647
ER
PT J
AU Hudry, K
AF Hudry, Kristelle
TI Interactive play for children with autism
SO AUTISM
LA English
DT Book Review
ID INTERVENTION; DISORDERS
C1 [Hudry, Kristelle] UCL, Inst Child Hlth, London, England.
RP Hudry, K (reprint author), UCL, Inst Child Hlth, London, England.
CR Aldred C, 2004, J CHILD PSYCHOL PSYC, V45, P1420, DOI 10.1111/j1469-7610.2004.00338.x
Baird G, 2001, ARCH DIS CHILD, V84, P468, DOI 10.1136/adc.84.6.468
Drew A, 2002, EUR CHILD ADOLES PSY, V11, P266, DOI 10.1007/s00787-002-0299-6
Hobson R. Peter, 1993, AUTISM DEV MIND
Kasari C, 2008, J CONSULT CLIN PSYCH, V76, P125, DOI 10.1037/0022-006X.76.1.125
MCCONACHIE H, 2005, J PEDIATR, V146, P335
Seach D., 2007, INTERACTIVE PLAY CHI
Wetherby AM, 2006, TOP EARLY CHILD SPEC, V26, P67, DOI 10.1177/02711214060260020201
NR 8
TC 0
Z9 0
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 1362-3613
J9 AUTISM
JI Autism
PD JUL
PY 2008
VL 12
IS 4
BP 415
EP 417
DI 10.1177/1362361308092100
PG 3
WC Psychology, Developmental
SC Psychology
GA 331AS
UT WOS:000257985700007
ER
PT J
AU Lechago, SA
Carr, JE
AF Lechago, Sarah A.
Carr, James E.
TI Recommendations for reporting independent variables in outcome studies
of early and intensive behavioral intervention for autism
SO BEHAVIOR MODIFICATION
LA English
DT Article
DE autism; early intervention; behavioral treatment; clinical trials
ID SPECTRUM DISORDERS; MAINTENANCE TASKS; CHILDREN; INTERSPERSAL; PROJECT;
SETTINGS; PROGRAMS
AB Early and intensive behavioral intervention (EIBI) has been established as an effective treatment for autism. However, the complexity and intensity of EIBI programs make it difficult to fully report all critical aspects of the independent variable. Consequently, scientific reports of EIBI outcomes have been criticized for providing less than comprehensive treatment descriptions. In an effort to address this problem, the present article provides a template to aid outcome researchers in (a) clearly reporting each participant's curricular targets and (b) describing critical aspects of treatment.
C1 [Carr, James E.] Western Michigan Univ, Dept Psychol, Kalamazoo, MI 49008 USA.
[Lechago, Sarah A.; Carr, James E.] Western Michigan Univ, Behav Anal Program, Kalamazoo, MI 49008 USA.
RP Carr, JE (reprint author), Western Michigan Univ, Dept Psychol, 1903 W Michigan Ave, Kalamazoo, MI 49008 USA.
EM jim.carr@wmich.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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Sundberg M. L., 1999, AUTISM BEHAV ANAL PE, P139
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NR 44
TC 6
Z9 6
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0145-4455
J9 BEHAV MODIF
JI Behav. Modificat.
PD JUL
PY 2008
VL 32
IS 4
BP 489
EP 503
DI 10.1177/0145445507309034
PG 15
WC Psychology, Clinical
SC Psychology
GA 312NF
UT WOS:000256677400004
PM 18525063
ER
PT J
AU Friedman, A
Luiselli, JK
AF Friedman, Abby
Luiselli, James K.
TI Excessive daytime sleep - Behavioral assessment and intervention in a
child with autism
SO BEHAVIOR MODIFICATION
LA English
DT Article
DE excessive daytime sleep; autism; sleep problems; behavioral intervention
AB Some children with autism have excessive daytime sleep but intervention research for this problem has not been conducted. The present study evaluated procedures with a 13 year old boy who had autism and slept for prolonged periods during the day. Classroom staff at a specialized school implemented procedures with the boy according to an ABAB experimental design. Intervention eliminated daytime sleep through a 6-month follow-up assessment. The study adds to the literature concerning sleep problems in children with autism and extends intervention to excessive daytime sleep.
C1 [Friedman, Abby; Luiselli, James K.] May Inst, Randolph, MA 02368 USA.
RP Friedman, A (reprint author), May Inst, 41 Pacella Pk Dr, Randolph, MA 02368 USA.
EM jluiselli@mayinstitute.org
CR Allik H, 2006, BMC PSYCHIATRY, V6, P6
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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Chokroverty S, 1999, SLEEP DISORDERS MED
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Liu XC, 2006, CHILD PSYCHIAT HUM D, V37, P179, DOI 10.1007/s10578-006-0028-3
Richdale A., 2001, SLEEP DISTURBANCE CH, P181
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Thorpy M., 1990, HDB SLEEP DISORDERS
Williams PG, 2004, J SLEEP RES, V13, P265
NR 14
TC 6
Z9 6
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0145-4455
J9 BEHAV MODIF
JI Behav. Modificat.
PD JUL
PY 2008
VL 32
IS 4
BP 548
EP 555
DI 10.1177/0145445507312187
PG 8
WC Psychology, Clinical
SC Psychology
GA 312NF
UT WOS:000256677400008
PM 18258994
ER
PT J
AU Anan, RM
Warner, LJ
McGillivary, JE
Chong, IM
Hines, SJ
AF Anan, Ruth M.
Warner, Lori J.
McGillivary, Jamie E.
Chong, Ivy M.
Hines, Stefani J.
TI Group intensive family training (gift) for preschoolers with autism
spectrum disorders
SO BEHAVIORAL INTERVENTIONS
LA English
DT Article
ID PERVASIVE DEVELOPMENTAL DISORDERS; BEHAVIORAL TREATMENT; YOUNG-CHILDREN;
EARLY INTERVENTION; PARENTS; DISABILITIES; PROGRAM; TRIAL; SKILLS
AB Early, intensive behavioral intervention is effective in treating children with autism spectrum disorders (ASDs), but can be cost prohibitive. Expenses may be defrayed if children can benefit from parents acting as therapists. This quantitative case series examines the efficacy of the Group Intensive Family Training (GIFT) program, a 12-week (180h, delivered 3h each weekday) parent-training for preschoolers with ASDs. Parents were individually mentored in the hands-on application of behavior analytic techniques, implementing these skills in vivo within a group of six parent-child dyads. Seventy-two parents and children (ages 25-68 months) with ASDs participated in this study. Children's cognitive and adaptive functioning was assessed before and after the intervention program. Analyses revealed average gains of eight standard score points on the Mullen Early Learning Composite and five standard score points on the Vineland Adaptive Behavior Composite after 12 weeks of treatment. Additionally, 14% and 11% of the children moved from the 'impaired' to 'non-impaired' range on Mullen and Vineland composite scores, respectively. This preliminary investigation suggests that GIFT's behavioral, group parent-training can lead to significant, yet cost- and time-efficient gains for children with ASDs. Results must be interpreted with caution because of the absence of a control group. Copyright (C) 2008 John Wiley & Sons, Ltd.
C1 [Anan, Ruth M.; Warner, Lori J.; McGillivary, Jamie E.; Chong, Ivy M.; Hines, Stefani J.] William Beaumont Hosp, Dept Pediat, Div Dev Behav Pediat, Royal Oak, MI 48072 USA.
RP Anan, RM (reprint author), William Beaumont Hosp, 1695 W 12 Mile,Suite 120, Berkley, MI 48072 USA.
EM ranan@beaumont.edu
CR Barbera M. L., 2007, VERBAL BEHAV APPROAC
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NR 56
TC 16
Z9 17
PU JOHN WILEY & SONS LTD
PI CHICHESTER
PA THE ATRIUM, SOUTHERN GATE, CHICHESTER PO19 8SQ, W SUSSEX, ENGLAND
SN 1072-0847
J9 BEHAV INTERVENT
JI Behav. Intervent.
PD JUL
PY 2008
VL 23
IS 3
BP 165
EP 180
DI 10.1002/bin.262
PG 16
WC Psychology, Clinical
SC Psychology
GA 332BC
UT WOS:000258055600002
ER
PT J
AU Schreck, KA
Mazur, A
AF Schreck, Kimberly A.
Mazur, Alison
TI Behavior analyst use of and beliefs in treatments for people with autism
SO BEHAVIORAL INTERVENTIONS
LA English
DT Article
AB With the increase in the numbers of children diagnosed with autism and scientific support solely for Applied Behavior Analysis (ABA) treatment, a proliferation of professionals have sought ABA training to provide services to this population. However, not all have been reportedly providing solely ABA services. This study surveyed Board Certified Behavior Analysts (BCBAs; N = 469) concerning their beliefs, endorsement, and use of a variety of scientifically supported and unsupported treatments for people with autism. Although not all endorsed using ABA, BCBA professionals most widely reported using ABA and ABA-related treatments. Surprisingly, BCBAs endorsed and used all types of treatments, despite their beliefs that the treatments were difficult to implement, not cost effective, and not Supported by research. Copyright (C) 2008 John Wiley & Sons, Ltd.
C1 Penn State Univ, Middletown, PA 17057 USA.
Penn State Univ, Harrisburg, PA USA.
[Mazur, Alison] Pathway Sch, Norristown, PA USA.
RP Schreck, KA (reprint author), Penn State Univ, 777 W Harrisburg Pike, Middletown, PA 17057 USA.
EM kas24@psu.edu
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Metz B, 2005, CONTROVERSIAL THERAPIES FOR DEVELOPMENTAL DISABILITES: FAD, FASHION, AND SCIENCE IN PROFESSIONAL PRACTICE, P237
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Richdale AL, 2008, PRACT RESOUR MENT, P3, DOI 10.1016/B978-012373606-2.50003-6
U. S. Department of Health and Human Services, 1999, MENT HLTH REP SURG G
NR 5
TC 9
Z9 10
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1072-0847
J9 BEHAV INTERVENT
JI Behav. Intervent.
PD JUL
PY 2008
VL 23
IS 3
BP 201
EP 212
DI 10.1002/bin.264
PG 12
WC Psychology, Clinical
SC Psychology
GA 332BC
UT WOS:000258055600004
ER
PT J
AU Sze, KM
Wood, JJ
AF Sze, Karen M.
Wood, Jeffrey J.
TI Enhancing CBT for the Treatment of Autism Spectrum Disorders and
Concurrent Anxiety
SO BEHAVIOURAL AND COGNITIVE PSYCHOTHERAPY
LA English
DT Article
DE Anxiety; Asperger syndrome; autism; cognitive-behavioural therapy
ID ASPERGER-SYNDROME; CONTROLLED-TRIAL; CHILDREN; THERAPY
AB Because anxiety and other concurrent psychological problems are common among children with high-functioning autism and Asperger syndrome. research initiatives have been devoted to the development of efficacious treatments to address the multifaceted needs of youth with this presentation. Emerging research indicates that when carefully adapted to accommodate for the unique needs of children with autism Spectrum disorders (ASD), cognitive-behavioural therapy (CBT) may be a viable treatment modality. Because inherent features of ASD may compromise the efficacy of traditional CBT, our preliminary work suggests that it may be important to gear intervention efforts towards directly addressing core autism symptoms to promote optimal treatment response. This article describes,in evidence-based CBT treatment manual modified and expanded to address core ASD features above and beyond anxiety symptomatology. A case example of a 10-year-old boy with Asperger syndrome is presented with supporting data to illustrate the treatment modifications and their rationale. The positive treatment response attained in this Case suggests that in enhanced variant of a standard CBT programme may be an efficacious treatment approach for children with ASD and concurrent anxiety.
C1 [Wood, Jeffrey J.] Univ Calif Los Angeles, Grad Sch Educ & Informat Studies, Los Angeles, CA 90095 USA.
RP Wood, JJ (reprint author), Univ Calif Los Angeles, Grad Sch Educ & Informat Studies, Moore Hall,Box 951521, Los Angeles, CA 90095 USA.
EM jeffwood@ucla.edu
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NR 18
TC 18
Z9 18
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1352-4658
J9 BEHAV COGN PSYCHOTH
JI Behav. Cognit. Psychther.
PD JUL
PY 2008
VL 36
IS 4
BP 403
EP 409
DI 10.1017/S1352465808004384
PG 7
WC Psychology, Clinical
SC Psychology
GA 363XF
UT WOS:000260300100004
ER
PT J
AU Pinkston, JW
Madden, GJ
Fowler, SC
AF Pinkston, Jonathan W.
Madden, Gregory J.
Fowler, Stephen C.
TI Effects of white and infrared lighting on apomorphine-induced pecking in
pigeons
SO BEHAVIOURAL PHARMACOLOGY
LA English
DT Article
DE apomorphine; force; infrared light; peck; pigeon; stereotypy; white
light
ID INDUCED STEREOTYPED BEHAVIOR; ANIMAL-MODELS; SENSITIZATION; AMPHETAMINE;
DOPAMINE; DYNAMICS; AUTISM; FORM
AB This experiment was concerned with the role of the environment in the production and form of apomorphine-induced pecking of pigeons. Earlier literature has suggested that the pecking occurs even when pigeons are placed in complete darkness, but there are no systematic or quantitative reports of such pecking. Six pigeons were tested with doses of 0.1, 0.3, and 1.0 mg/kg apomorphine. Tests were made in conditions of white and infrared light. The apparatus used novel force transduction measures that provided for both the detection of a peck as well as its peak forcefulness. At the lowest dose tested, apornorphine elicited pecking when the pigeon was placed in white light, but not when the dose was examined under infrared lighting. As the dose increased, however, pecking was observed regardless of lighting condition. No consistent differences were found in forcefulness of pecking as a function of lighting condition or dose. Though response output was seemingly unaffected by the lighting condition at higher doses, videotaped analysis revealed important changes in the formal characteristics of pecking. In white light, apomorphine elicited pecking at stimuli in the chamber (e.g. screw heads or the pigeon's own toes), whereas in infrared light pecking was directed at the floor directly in front of the pigeon. Such differences may be attributable to shifts in control to other stimulus modalities when vision is limited. Additionally, apomorphine may have direct effects on retinal dopamine function modulating the expression of pecking in the dark. Behavioural Pharmacology 19:347-352 (c) 2008 Wolters Kluwer Health vertical bar Lippincott Williams & Wilkins.
C1 [Pinkston, Jonathan W.; Fowler, Stephen C.] Schiefelbusch Inst Life Span Studies, Lawrence, KS 66049 USA.
[Madden, Gregory J.] Univ Kansas, Dept Appl Behav Sci, Lawrence, KS 66045 USA.
[Fowler, Stephen C.] Univ Kansas, Dept Pharmacol & Toxicol, Lawrence, KS 66045 USA.
RP Pinkston, JW (reprint author), Schiefelbusch Inst Life Span Studies, 1000 Sunnyside Ave,1000 Sunnyside Ave,Dole HDC,Rm, Lawrence, KS 66049 USA.
EM pinkston@ku.edu
RI Madden, Gregory/E-8524-2010
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NR 27
TC 3
Z9 3
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0955-8810
J9 BEHAV PHARMACOL
JI Behav. Pharmacol.
PD JUL
PY 2008
VL 19
IS 4
BP 347
EP 352
PG 6
WC Behavioral Sciences; Neurosciences; Pharmacology & Pharmacy
SC Behavioral Sciences; Neurosciences & Neurology; Pharmacology & Pharmacy
GA 328TV
UT WOS:000257821600010
PM 18622183
ER
PT J
AU Senju, A
Kikuchi, Y
Hasegawa, T
Tojo, Y
Osanai, H
AF Senju, Atsushi
Kikuchi, Yukiko
Hasegawa, Toshikazu
Tojo, Yoshikuni
Osanai, Hiroo
TI Is anyone looking at me? Direct gaze detection in children with and
without autism
SO BRAIN AND COGNITION
LA English
DT Article
DE autism; autism spectrum disorder; gaze; direct gaze; face processing;
visual search; search asymmetry; face inversion effect
ID UPSIDE-DOWN FACES; EYE-GAZE; VISUAL-SEARCH; REACTION-TIME; EARLY
RECOGNITION; SOCIAL ATTENTION; HOME VIDEOTAPES; COGNITIVE-STYLE;
YOUNG-CHILDREN; CONTACT
AB Atypical processing of eye contact is one of the significant characteristics of individuals with autism, but the mechanism underlying atypical direct gaze processing is still unclear. This study used a visual search paradigm to examine whether the facial context would affect direct gaze detection in children with autism. Participants were asked to detect target gazes presented among distracters with different gaze directions. The target gazes were either direct gaze or averted gaze, which were either presented alone (Experiment 1) or within facial context (Experiment 2). As with the typically developing children, the children with autism, were faster and more efficient to detect direct gaze than averted gaze, whether or not the eyes were presented alone or within faces. In addition, face inversion distorted efficient direct gaze detection in typically developing children, but not in children with autism. These results suggest that children with autism use featural information to detect direct gaze, whereas typically developing children use configural information to detect direct gaze. (C) 2007 Elsevier Inc. All rights reserved.
C1 [Senju, Atsushi] Univ London Birkbeck Coll, Dept Psychol, Ctr Brain & Cognit Dev, London WC1E 7HX, England.
[Kikuchi, Yukiko; Hasegawa, Toshikazu] Univ Tokyo, Tokyo, Japan.
[Tojo, Yoshikuni] Ibaraki Univ, Ibaraki, Japan.
[Osanai, Hiroo] Musashino Higashi Gakuen, Tokyo, Japan.
RP Senju, A (reprint author), Univ London Birkbeck Coll, Dept Psychol, Ctr Brain & Cognit Dev, Henry Wellcome Bldg,Torrington Sq, London WC1E 7HX, England.
EM a.senju@bbk.ac.uk
RI Senju, Atsushi/C-4097-2008
OI Senju, Atsushi/0000-0002-8081-7170
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NR 74
TC 26
Z9 28
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 0278-2626
J9 BRAIN COGNITION
JI Brain Cogn.
PD JUL
PY 2008
VL 67
IS 2
BP 127
EP 139
DI 10.1016/j.bandc.2007.12.001
PG 13
WC Neurosciences; Psychology, Experimental
SC Neurosciences & Neurology; Psychology
GA 323YN
UT WOS:000257484600001
PM 18226847
ER
PT J
AU Dougall, A
Fiske, J
AF Dougall, A.
Fiske, J.
TI Access to special care dentistry, part 2. Communication
SO BRITISH DENTAL JOURNAL
LA English
DT Article
ID INTERNATIONAL SURVEY; AUTISM; IMPAIRMENT; DYSARTHRIA; DISEASE; APHASIA
AB This article considers what communication is, its elements, what helps and what hinders it, and why it matters. It also considers managing people with communication differences and when communication is affected in special care dentistry (SCD). The article focuses on patients with hearing and visual impairments and considers how communication is affected and what techniques can be used to improve the situation. It offers recommendations for communicating with patients with neurological impairments typically seen after stroke, such as aphasia and dysarthria, with tips for the listener including the use of communication aids where appropriate. Finally it will consider communicating with patients who have autistic spectrum conditions and discuss how effective techniques and a tailored approach to their specific needs and anxieties can increase the likelihood of a successful dental visit.
C1 [Fiske, J.] Kings Coll London, Inst Dent, Dept Sedat & Special Care Dent, London SE1 9RT, England.
[Dougall, A.] Dublin Dent Sch & Hosp, Div Special Care Dent 1, Dublin 2, Ireland.
RP Fiske, J (reprint author), Kings Coll London, Inst Dent, Dept Sedat & Special Care Dent, Floor 26,Guys Tower, London SE1 9RT, England.
EM Janice.Fiske@gstt.nhs.uk
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WANLESS M, 2007, ORAL HLTH REPORT, V1, P2
NR 38
TC 6
Z9 7
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 0007-0610
J9 BRIT DENT J
JI Br. Dent. J.
PD JUL
PY 2008
VL 205
IS 1
BP 11
EP 21
DI 10.1038/sj.bdj.2008.533
PG 11
WC Dentistry, Oral Surgery & Medicine
SC Dentistry, Oral Surgery & Medicine
GA 324JR
UT WOS:000257515400009
PM 18617935
ER
PT J
AU Japundza-Milisavljevic, M
Macesic-Petrovic, D
AF Japundza-Milisavljevic, Mirjana
Macesic-Petrovic, Dragana
TI Executive functions in children with intellectual disabilities
SO BRITISH JOURNAL OF DEVELOPMENTAL DISABILITIES
LA English
DT Article
ID YOUNG-CHILDREN; AUTISM; MEMORY; HANOI
C1 [Japundza-Milisavljevic, Mirjana; Macesic-Petrovic, Dragana] Fac Special Educ & Rehabil, Belgrade, Serbia.
RP Japundza-Milisavljevic, M (reprint author), Fac Special Educ & Rehabil, Visokog Stevana Br 2, Belgrade, Serbia.
EM japundza@eunet.yu
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NR 17
TC 4
Z9 4
PU BRITISH SOC DEVELOPMENTAL DISABILITIES
PI MODLING
PA WEYPRECHTGASSE 10, MODLING A-2340, AUSTRIA
SN 0969-7950
J9 BRIT J DEV DISABIL
JI Br. J. Dev. Disabil.
PD JUL
PY 2008
VL 54
IS 107
BP 113
EP 121
PN 2
PG 9
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 326YK
UT WOS:000257695400005
ER
PT J
AU Simonsen, H
AF Simonsen, Helen
TI Autism and loss
SO BRITISH JOURNAL OF DEVELOPMENTAL DISABILITIES
LA English
DT Book Review
CR JONES RF, 2007, AUTISM LOSS
NR 1
TC 0
Z9 0
PU BRITISH SOC DEVELOPMENTAL DISABILITIES
PI MODLING
PA WEYPRECHTGASSE 10, MODLING A-2340, AUSTRIA
SN 0969-7950
J9 BRIT J DEV DISABIL
JI Br. J. Dev. Disabil.
PD JUL
PY 2008
VL 54
IS 107
BP 133
EP 134
PN 2
PG 2
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 326YK
UT WOS:000257695400008
ER
PT J
AU Bryson, SE
Bradley, EA
Thompson, A
Wainwright, A
AF Bryson, Susan E.
Bradley, Elspeth A.
Thompson, Ann
Wainwright, Ann
TI Prevalence of autism among adolescents with intellectual disabilities
SO CANADIAN JOURNAL OF PSYCHIATRY-REVUE CANADIENNE DE PSYCHIATRIE
LA English
DT Article
DE intellectual disabilities; autism; prevalence
ID PERVASIVE DEVELOPMENTAL DISORDERS; MENTAL-RETARDATION; DIAGNOSTIC
INTERVIEW; PSYCHIATRIC-DISORDERS; EPIDEMIOLOGIC ASPECTS; SPECTRUM
DISORDERS; PRESCHOOL-CHILDREN; TEENAGERS; BEHAVIOR; CLASSIFICATION
AB Objective: To estimate the prevalence of autism in an epidemiologically-derived population of adolescents with intellectual disabilities (ID).
Method: The prevalence of autism was examined using the Autism Diagnostic Interview-Revised, with appropriate care taken in assessing lower functioning individuals and those with additional physical and sensory impairments. Individual assessment during psychological evaluation, and consensus classification of complex cases, involving clinicians experienced in the assessment of autism, contributed to the identification of autism.
Results: Overall, 28% of individuals, or 2.0 of the 7.1/1000 with ID in the target population (as we have previously identified in another study), were identified with autism. Autism rates did not differ significantly across severe ID (32.0%) and mild ID (24.1%); males predominated (2.3 males to 1 female), but less so for severe ID (2 males to 1 female, compared with 2.8 males to 1 female for mild ID). Socioeconomic status did not distinguish the groups with and without autism. Less than one-half of the adolescents who met diagnostic criteria for autism were previously diagnosed as such.
Conclusions: Our overall prevalence estimate for autism is in the higher range of estimates reported in previous studies of ID (more so for mild ID). This likely reflects the changes in diagnostic criteria for autism that have subsequently occurred. Discussion focuses on the identification of autism in the population with ID, and on the implications for service delivery and clinical training.
C1 [Bryson, Susan E.] Dalhousie Univ, IWK Hlth Ctr, Dept Pediat, Halifax, NS B3K 6R8, Canada.
[Bradley, Elspeth A.] Surrey Pl Ctr, Med Serv, Toronto, ON M5S 2C2, Canada.
[Bradley, Elspeth A.] Univ Toronto, Dept Psychiat, Toronto, ON, Canada.
[Thompson, Ann] McMaster Univ, Offord Ctr Child Studies, Hamilton, ON, Canada.
[Wainwright, Ann] Seneca Coll, Toronto, ON, Canada.
RP Bryson, SE (reprint author), Dalhousie Univ, IWK Hlth Ctr, Dept Pediat, 5850-5980 Univ Ave,POB 9700, Halifax, NS B3K 6R8, Canada.
EM susan.bryson@iwk.nshealth.ca
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NR 54
TC 27
Z9 28
PU CANADIAN PSYCHIATRIC ASSOC
PI OTTAWA
PA 141 LAURIER AVENUE WEST, STE 701, OTTAWA, ONTARIO K1P 5J3, CANADA
SN 0706-7437
J9 CAN J PSYCHIAT
JI Can. J. Psychiat.-Rev. Can. Psychiat.
PD JUL
PY 2008
VL 53
IS 7
BP 449
EP 459
PG 11
WC Psychiatry
SC Psychiatry
GA 335DJ
UT WOS:000258270700010
PM 18674403
ER
PT J
AU Berthoz, S
Wessa, M
Kedia, G
Wicker, B
Grezes, J
AF Berthoz, Sylvie
Wessa, Michele
Kedia, Gayannee
Wicker, Bruno
Grezes, Julie
TI Cross-cultural validation of the Empathy Quotient in a French-speaking
sample
SO CANADIAN JOURNAL OF PSYCHIATRY-REVUE CANADIENNE DE PSYCHIATRIE
LA English
DT Article
DE empathizing; sex differences; autism spectrum disorders; reliability
ID INDIVIDUAL-DIFFERENCES; SPECTRUM DISORDER; AUTISM; SELF; ADULTS;
VENTURESOMENESS; IMPULSIVENESS; PERSONALITY; STABILITY; ABILITIES
AB Objective: The Empathy Quotient (EQ) is a self-report that was developed to measure the cognitive and affective aspects of empathy. We further evaluated its validity in 2 studies.
Method: The psychometric qualities of the French version of the EQ, and its correspondence with 2 other measures of empathy (Interpersonal Reactivity Index and the Empathy Scale of the Impulsiveness-Venturesomeness-Empathy Questionnaire), and with dimensions of the emotional state (depression and anxiety), were evaluated in a sample of 410 students (201 men and 209 women). Second, the clinical validity of the EQ was investigated in participants expected to have dysfunctional empathy. For this purpose, EQ scores of 16 people with autistic spectrum disorder (ASD) were collected.
Results: The EQ showed satisfying internal, convergent, test-retest and discriminant validity. The confirmatory factorial analyses suggested a 3-factor structure offered a good fit to the data. The women's superiority in empathy was replicated. As expected, the ASD EQ scores were very low.
Conclusion: This study provides further evidence that the EQ is reliable in this population and should be recommended to estimate empathy problems, notably in individuals with troubled interpersonal interaction patterns.
C1 [Berthoz, Sylvie] Univ Paris 05, Serv Psychiat Adolescent & Jeune Adulte, Inst Mutualiste Montsouris, Dept Psychiat Adolescents & Young Adults, F-75674 Paris 14, France.
[Wessa, Michele] Univ Heidelberg, Cent Inst Mental Hlth, Dept Clin & Cognit Neurosci, D-6800 Mannheim, Germany.
[Kedia, Gayannee] CEA, Hosp Dept Frederic Joliot, IFR49, Inserm U Neuroimaging Psychiat 797, Orsay, France.
[Wicker, Bruno] Univ Aix Marseille 2, CNRS, Inst Neurosci Cognit Mediterranee, Marseille, France.
[Grezes, Julie] Coll France, CNRS, Lab Physiol Percept & Act, F-75231 Paris, France.
[Kedia, Gayannee] Univ Toulouse, Dept Social Psychol, Toulouse, France.
RP Berthoz, S (reprint author), Univ Paris 05, Serv Psychiat Adolescent & Jeune Adulte, Inst Mutualiste Montsouris, Dept Psychiat Adolescents & Young Adults, 42 Bd Jourdan, F-75674 Paris 14, France.
EM sylvie.berthoz@imm.fr
RI Wessa, Michele/F-7010-2014
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NR 46
TC 26
Z9 27
PU CANADIAN PSYCHIATRIC ASSOC
PI OTTAWA
PA 141 LAURIER AVENUE WEST, STE 701, OTTAWA, ONTARIO K1P 5J3, CANADA
SN 0706-7437
J9 CAN J PSYCHIAT
JI Can. J. Psychiat.-Rev. Can. Psychiat.
PD JUL
PY 2008
VL 53
IS 7
BP 469
EP 477
PG 9
WC Psychiatry
SC Psychiatry
GA 335DJ
UT WOS:000258270700012
PM 18674405
ER
PT J
AU Hazell, P
Williams, R
AF Hazell, Philip
Williams, Richard
TI Editorial review: shifting views on juvenile bipolar disorder and
pervasive developmental disorder
SO CURRENT OPINION IN PSYCHIATRY
LA English
DT Editorial Material
DE child; culture; diagnosis; mental disorders; prevalence
ID SEVERE MOOD DYSREGULATION; CHILD-BEHAVIOR CHECKLIST; TREATMENT
GUIDELINES; COMMUNITY SAMPLE; AUTISM; ONSET; PREVALENCE; PHENOTYPE;
EPIDEMIOLOGY; ADOLESCENTS
AB Purpose of review
To examine the changes in prevalence estimates and concepts of core disorder in two child mental disorders that were once considered rare, and to place these changes in a cultural context.
Recent findings
Up to one-quarter of people with bipolar disorder may have experienced onset of symptoms prior to puberty, but the precision of the diagnosis in children is uncertain. An ongoing challenge is differentiating bipolar disorder from other child mental disorders. Reliable markers of persistent bipolarity have yet to be identified. Despite a popular perception, pervasive developmental disorder is unlikely to have increased in the population, but recognition rates have increased as much as ten-fold since 1980. The increase is largely accounted for by shifts in diagnostic practice and in attitudes towards the condition.
Summary
Changes in diagnostic practice and in clinician and public attitude account for much of the apparent variation in the prevalence of child mental disorders.
C1 [Hazell, Philip] Univ Newcastle, Discipline Psychiat, Callaghan, NSW 2308, Australia.
[Hazell, Philip] Univ Sydney, Discipline Psychol Med, Sydney, NSW 2006, Australia.
[Williams, Richard] Univ Glamorgan, Pontypridd CF37 1DL, M Glam, Wales.
RP Hazell, P (reprint author), Thomas Walker Hosp, Hosp Rd, Concord W, NSW 2138, Australia.
EM philip.hazell@sswahs.nsw.gov.au
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NR 30
TC 0
Z9 0
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0951-7367
J9 CURR OPIN PSYCHIATR
JI Curr. Opin. Psychiatr.
PD JUL
PY 2008
VL 21
IS 4
BP 328
EP 331
DI 10.1097/YCO.0b013e328305b6f9
PG 4
WC Psychiatry
SC Psychiatry
GA 318ZB
UT WOS:000257130200007
PM 18520735
ER
PT J
AU Soh, NL
Walter, G
AF Soh, Nerissa L.
Walter, Garry
TI Complementary medicine for psychiatric disorders in children and
adolescents
SO CURRENT OPINION IN PSYCHIATRY
LA English
DT Article
DE attention deficit hyperactivity disorder; bipolar disorder; depression;
omega-3 fatty acids; St John's wort
ID ST-JOHNS-WORT; PLACEBO-CONTROLLED TRIALS; AUTISM SPECTRUM DISORDERS;
ALTERNATIVE MEDICINE; GINKGO-BILOBA; DOUBLE-BLIND; FOOD COLORINGS;
ANXIETY DISORDERS; SOUTH-AUSTRALIA; FATTY-ACIDS
AB Purpose of review
To examine recent empirical studies of herbal and dietary treatments for psychiatric disorders in children and adolescents.
Recent findings
Very few quality studies into the use of omega-3 fatty acids, St John's wort, dietary manipulations, kava, gingko and lemon balm in managing psychiatric disorders have been conducted in children and adolescents.
Summary
A number of herbal treatments show promise, but much more empirical research is required to establish their efficacy in the paediatric population.
C1 No Sydney Cent Coast Area Hlth, Child & Adolescent Mental Hlth Serv, Sydney, NSW, Australia.
Univ Sydney, Discipline Psychol Med, Sydney, NSW 2006, Australia.
RP Soh, NL (reprint author), Coral Tree Family Serv, POB 142, N Ryde, NSW 1670, Australia.
EM nsoh@nsccahs.health.nsw.gov.au
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Wong HHL, 2006, J AUTISM DEV DISORD, V36, P901, DOI 10.1007/s10803-006-0131-0
Wozniak J, 2007, EUR NEUROPSYCHOPHARM, V17, P440, DOI 10.1016/j.euroneuro.2006.11.006
NR 59
TC 5
Z9 5
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0951-7367
J9 CURR OPIN PSYCHIATR
JI Curr. Opin. Psychiatr.
PD JUL
PY 2008
VL 21
IS 4
BP 350
EP 355
DI 10.1097/YCO.0b013e328303672f
PG 6
WC Psychiatry
SC Psychiatry
GA 318ZB
UT WOS:000257130200011
PM 18520739
ER
PT J
AU Moon, J
Ota, KT
Driscoll, LL
Levitsky, DA
Strupp, BJ
AF Moon, J.
Ota, K. T.
Driscoll, L. L.
Levitsky, D. A.
Strupp, B. J.
TI A mouse model Fragile X syndrome exhibits heightened arousal and/or
emotion following errors or reversal of contingencies
SO DEVELOPMENTAL PSYCHOBIOLOGY
LA English
DT Article
DE Fragile X; autism; arousal; emotion; attention; learning set; reversal
learning; fmr1 knockout mouse; mouse; fmr1 gene; transfer of learning;
Fragile X mental retardation protein (FMRP)
ID FMR1 KNOCKOUT MOUSE; MENTAL-RETARDATION; BEHAVIORAL-PHENOTYPE; YOUNG
MALES; MICE; ATTENTION; GENE; CHILDREN; DEFICITS; PROFILE
AB This study was designed to further assess cognitive and affective functioning in a mouse model of Fragile X syndrome (FXS), the Fmr1(tm1Cgr) "knockout" (KO) mouse. Male KO mice and wild-type littermate controls were tested on learning set and reversal learning tasks. The KO mice were not impaired in associative learning, transfer of learning, or reversal learning, based on measures of learning rate. Analyses of videotapes of the reversal learning task revealed that both groups of mice exhibited higher levels of activity and wall-climbing during the initial sessions of the task than during the final sessions, a pattern also seen for trials following all error relative to those following a correct response. Notably, the increase in both behavioral measures seen early in the task was significantly more pronounced for the KO mice than for controls, as was the error-induced increase in activity level. This pattern of effects suggests that the KO mice reacted more strongly than controls to the reversal of contingencies and pronounced drop in reinforcement rate, and to errors in general. This pattern of affects is consistent with the heightened emotional reactivity frequently described for humans with FXS. (C) 2008 Wiley Periodicals, Inc.
C1 Cornell Univ, Dept Psychol, Div Nutr Sci, Ithaca, NY 14853 USA.
RP Strupp, BJ (reprint author), Cornell Univ, Dept Psychol, Div Nutr Sci, 109 Savage Hall, Ithaca, NY 14853 USA.
EM bjs13@cornell.edu
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NR 69
TC 10
Z9 10
PU JOHN WILEY & SONS INC
PI HOBOKEN
PA 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 0012-1630
J9 DEV PSYCHOBIOL
JI Dev. Psychobiol.
PD JUL
PY 2008
VL 50
IS 5
BP 473
EP 485
DI 10.1002/dev.20308
PG 13
WC Developmental Biology; Psychology
SC Developmental Biology; Psychology
GA 322GM
UT WOS:000257363200005
PM 18551464
ER
PT J
AU Whitehouse, AJO
Bishop, DVM
AF Whitehouse, Andrew J. O.
Bishop, Dorothy V. M.
TI Do children with autism 'switch off' to speech sounds? An investigation
using event-related potentials
SO DEVELOPMENTAL SCIENCE
LA English
DT Article
ID LISTENING PREFERENCES; ATTENTION; COMMUNICATION; IMPAIRMENTS;
INHIBITION; STANDARD; LANGUAGE; SPECTRUM; DEFICITS; STIMULI
AB Autism is a disorder characterized by a core impairment in social behaviour. A prominent component of this social deficit is poor orienting to speech. It is unclear whether this deficit involves an impairment in allocating attention to speech sounds, or a sensory impairment in processing phonetic information. In this study, event-related potentials of 15 children with high functioning autism (mean nonverbal IQ = 109.87) and 15 typically developing children (mean nonverbal IQ = 115.73) were recorded in response to sounds in two oddball conditions. Participants heard two stimulus types: vowels and complex tones. In each condition, repetitive 'standard' sounds (condition 1: vowel; condition 2: complex tone) were replaced by a within stimulus-type 'deviant' sound and a between stimulus-type 'novel' sound. Participants' level of attention was also varied between conditions. Children with autism had significantly diminished obligatory components in response to the repetitive speech sound, but not to the repetitive nonspeech sound. This difference disappeared when participants were required to allocate attention to the sound stream. Furthermore, the children with autism showed reduced orienting to novel tones presented in a sequence of speech sounds, but not to novel speech sounds presented in a sequence of tones. These findings indicate that high functioning children with autism can allocate attention to novel speech sounds. However, they use top-down inhibition to attenuate responses to repeated streams of speech. This suggests that problems with speech processing in this population involve efferent pathways.
C1 [Whitehouse, Andrew J. O.; Bishop, Dorothy V. M.] Univ Oxford, Dept Expt Psychol, Oxford OX1 3UD, England.
RP Whitehouse, AJO (reprint author), Univ Oxford, Dept Expt Psychol, S Parks Rd, Oxford OX1 3UD, England.
EM andrew.whitehouse@psy.ox.ac.uk
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NR 36
TC 36
Z9 37
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1363-755X
J9 DEVELOPMENTAL SCI
JI Dev. Sci.
PD JUL
PY 2008
VL 11
IS 4
BP 516
EP 524
DI 10.1111/j.1467-7687.2008.00697.x
PG 9
WC Psychology, Developmental; Psychology, Experimental
SC Psychology
GA 313DK
UT WOS:000256721100010
PM 18576959
ER
PT J
AU Marin, JCM
Moura, PJ
Cysneiros, RM
Colugnati, DB
Cavalheiro, EA
Scorza, FA
Xavier, GF
Zilbovicius, M
Mercadante, MT
AF Marin, Joao Carlos M.
Moura, Paula J.
Cysneiros, Roberta M.
Colugnati, Diego B.
Cavalheiro, Esper A.
Scorza, Fulvio A.
Xavier, Gilberto F.
Zilbovicius, Monica
Mercadante, Marcos T.
TI Temporal lobe epilepsy and social behavior: An animal model
SO EPILEPSY & BEHAVIOR
LA English
DT Article
DE temporal lobe epilepsy; social behavior; social inemory; social
discrimination paradigm; autism animal model; pilocarpine
ID PERVASIVE DEVELOPMENTAL DISORDERS; AUTISM SPECTRUM DISORDERS;
PILOCARPINE MODEL; AMYGDALA; RECOGNITION; MIND; HIPPOCAMPUS; PERCEPTION;
DEFICITS; RAT
AB Social behavior depends on the integrity of social brain circuitry. The temporal lobe is an important part of the social brain, and manifests morphological and functional alterations in autism spectrum disorders (ASD). Rats with temporal lobe epilepsy (TLE), induced with pilocarpine, were subjected to a social discrimination test that has been used to investigate potential animal models of ASD, and the results were compared with those for the control group. Rats with TLE exhibited fewer social behaviors than controls. No differences were observed in nonsocial behavior between groups. The results suggest an important role for the temporal lobe in regulating social behaviors. This animal model might be used to explore some questions about ASD pathophysiology. (c) 2008 Elsevier Inc. All rights reserved.
C1 [Marin, Joao Carlos M.; Mercadante, Marcos T.] Univ Fed Sao Paulo, Escola Paulista Med, Dept Psychiat, BR-04023900 Sao Paulo, Brazil.
[Moura, Paula J.; Xavier, Gilberto F.] Univ Sao Paulo, Dept Physiol, Sao Paulo, Brazil.
[Cysneiros, Roberta M.; Colugnati, Diego B.; Cavalheiro, Esper A.; Scorza, Fulvio A.] Univ Sao Paulo, Dept Neurol, Sao Paulo, Brazil.
[Zilbovicius, Monica] CEA, Serv Hosp Frederic Joliot, INSERM, URM Brain Imaging Psychiat 0205, F-91406 Orsay, France.
RP Mercadante, MT (reprint author), Univ Fed Sao Paulo, Escola Paulista Med, Dept Psychiat, Rua Botucatu 740-3 Andar, BR-04023900 Sao Paulo, Brazil.
EM mt.mercadante@uol.com.br
RI Xavier, Gilberto/B-9464-2013; Neurociencia, Inct/I-1011-2013; Scorza,
Fulvio/C-7048-2013; COLUGNATI, DIEGO/G-1954-2012; Cysneiros,
Roberta/H-6224-2012
OI Xavier, Gilberto/0000-0002-8567-7031; Scorza,
Fulvio/0000-0002-0694-8674; Cysneiros, Roberta/0000-0002-3191-9146
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NR 26
TC 8
Z9 8
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1525-5050
J9 EPILEPSY BEHAV
JI Epilepsy Behav.
PD JUL
PY 2008
VL 13
IS 1
BP 43
EP 46
DI 10.1016/j.yebeh.2008.03.004
PG 4
WC Behavioral Sciences; Clinical Neurology; Psychiatry
SC Behavioral Sciences; Neurosciences & Neurology; Psychiatry
GA 317HA
UT WOS:000257009400009
PM 18439879
ER
PT J
AU Slifer, KJ
Avis, KT
Frutchey, RA
AF Slifer, Keith J.
Avis, Kristin T.
Frutchey, Robin A.
TI Behavioral intervention to increase compliance with
electroencephalographic procedures in children with developmental
disabilities
SO EPILEPSY & BEHAVIOR
LA English
DT Article
DE electroencephalogram; seizure; pediatric; developmental disability;
sleep; behavioral intervention; compliance; anxiety; tactile
defensiveness
ID CHLORAL HYDRATE; YOUNG-CHILDREN; SEDATION; EFFICACY; AUTISM
AB The EEG, or electroencephalogram, is a neurophysiological technique used to detect and record electrical activity in the brain. It is critical to the diagnosis and management of seizure disorders, such as epilepsy, as well as other neurological conditions. The EEG procedure is often not well tolerated by children with developmental disabilities because of anxiety about unfamiliar equipment, difficulty inhibiting motion, and tactile defensiveness. The inability of children with developmental disabilities to tolerate an EEG procedure is especially problematic because the incidence of epilepsy is considerably higher in children with disabilities. This clinical outcome study sought to determine the efficacy of using behavioral intervention to teach children with developmental disorders to cooperate with an EEG procedure. The behavioral training employed modeling, counterconditioning, escape extinction, and differential reinforcement-based shaping procedures. Results indicated that behavioral training is successful in promoting EEG compliance without restraint, anesthesia, or sedation. (c) 2008 Elsevier Inc. All rights reserved.
C1 [Slifer, Keith J.; Frutchey, Robin A.] Kennedy Krieger Inst, Dept Behav Psychol, Baltimore, MD 21205 USA.
[Slifer, Keith J.] Johns Hopkins Univ, Sch Med, Dept Psychiat & Behav Sci, Baltimore, MD 21205 USA.
[Slifer, Keith J.] Johns Hopkins Univ, Sch Med, Dept Pediat, Baltimore, MD 21205 USA.
[Avis, Kristin T.] Univ Alabama, Dept Pediat, Birmingham, AL USA.
RP Frutchey, RA (reprint author), Kennedy Krieger Inst, Dept Behav Psychol, 707 N Broadway, Baltimore, MD 21205 USA.
EM frutcheyr@kennedykrieger.org
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NR 23
TC 11
Z9 12
PU ACADEMIC PRESS INC ELSEVIER SCIENCE
PI SAN DIEGO
PA 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA
SN 1525-5050
J9 EPILEPSY BEHAV
JI Epilepsy Behav.
PD JUL
PY 2008
VL 13
IS 1
BP 189
EP 195
DI 10.1016/j.yebeh.2008.01.013
PG 7
WC Behavioral Sciences; Clinical Neurology; Psychiatry
SC Behavioral Sciences; Neurosciences & Neurology; Psychiatry
GA 317HA
UT WOS:000257009400033
PM 18348911
ER
PT J
AU Zhu, JL
Vestergaard, M
Madsen, KM
Olsen, J
AF Zhu, Jin Liang
Vestergaard, Mogens
Madsen, Kreesten M.
Olsen, Jorn
TI Paternal age and mortality in children
SO EUROPEAN JOURNAL OF EPIDEMIOLOGY
LA English
DT Article
DE epidemiology; mortality; paternal age
ID POPULATION-BASED COHORT; PARENTAL AGE; RISK-FACTORS;
SPONTANEOUS-ABORTION; CHILDHOOD CANCERS; PRETERM BIRTH; DEATH; AUTISM;
SCHIZOPHRENIA; ASSOCIATION
AB Background Since paternal age correlates with some diseases that have a high case-fatality, a paternal age effect on offspring's survival is expected but unsettled. We examined the association between paternal age and mortality in children in a large population-based cohort taking maternal age and socioeconomic factors into account. Methods From the Danish Fertility Database (1980-1996), we identified 102,879 couples and their firstborn singleton children. Information on childhood death (N = 831) was obtained by linking the cohort to the nationwide register on cause of death (1980-1998). Results We observed a U-shaped association between paternal age and the overall mortality rate in children up to 18 years of age. Adjustment for maternal age and other confounders reduced the mortality rate ratio (MRR) for children of younger fathers but not for children of older fathers. Compared with children of fathers aged between 25 and 29 years, the adjusted MRR was 1.77 (95% confidence interval 1.28-2.45) for children of fathers aged between 45 and 49 years and 1.59 (1.03-2.46) for children of fathers aged 50 years or more. The cause-specific MRRs were highest for congenital malformations [2.35 (1.42-3.88)] and injury or poisoning [3.43 (1.49-7.92)] for children of fathers aged 45 years or more. Conclusion Our data revealed a higher mortality in offspring of fathers aged 45 years or more that lasted into adulthood. This adds to the cumulating evidence on adverse effects of advanced paternal age in procreation.
C1 [Zhu, Jin Liang; Vestergaard, Mogens; Madsen, Kreesten M.] Univ Aarhus, Danish Epidemiol Sci Ctr, DK-8000 Aarhus C, Denmark.
[Vestergaard, Mogens] Univ Aarhus, Inst Publ Hlth, Dept Gen Practice, DK-8000 Aarhus C, Denmark.
[Olsen, Jorn] Univ Calif Los Angeles, Sch Publ Hlth, Dept Epidemiol, Los Angeles, CA 90095 USA.
RP Zhu, JL (reprint author), Univ Aarhus, Danish Epidemiol Sci Ctr, Vennelyst Blvd 6, DK-8000 Aarhus C, Denmark.
EM zjl@soci.au.dk
RI Vestergaard, Mogens/M-9333-2014; Olsen, Jorn/F-8801-2015
OI Vestergaard, Mogens/0000-0001-8830-2174; Olsen, Jorn/0000-0001-7462-5140
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NR 31
TC 21
Z9 21
PU SPRINGER
PI DORDRECHT
PA VAN GODEWIJCKSTRAAT 30, 3311 GZ DORDRECHT, NETHERLANDS
SN 0393-2990
J9 EUR J EPIDEMIOL
JI Eur. J. Epidemiol.
PD JUL
PY 2008
VL 23
IS 7
BP 443
EP 447
DI 10.1007/s10654-008-9253-3
PG 5
WC Public, Environmental & Occupational Health
SC Public, Environmental & Occupational Health
GA 316DB
UT WOS:000256928000001
PM 18437509
ER
PT J
AU Vaglio, A
Milunsky, A
Huang, XL
Quadrelli, A
Mechoso, B
Maher, TA
Quadrelli, R
AF Vaglio, Alicia
Milunsky, Aubrey
Huang, Xin-Li
Quadrelli, Andrea
Mechoso, Burix
Maher, Thomas A.
Quadrelli, Roberto
TI A 21 years follow-up of a girl patient with a pseudodicentric
bisatellited chromosome 22 associated with partial trisomy 22pter ->
22q12.1: Clinical, cytogenetic and molecular observations
SO EUROPEAN JOURNAL OF MEDICAL GENETICS
LA English
DT Article
DE chromosome 22; pseudodicentric; partial trisomy 22
ID CAT-EYE SYNDROME; DELETION SYNDROME; AUTISM SPECTRUM; 22Q13 DELETION;
DUPLICATION; SEGMENT; DISORDERS; INVERSION; SHANK3; CHILD
AB We present clinical and developmental data on a patient with a de novo recombinant pseudodicentric bisatellited chromosome 22 associated with a partial trisomy 22pter-22q12.1. The patient was evaluated at birth and followed-up until 21 years of age. Clinical findings include facial and digital dysmorphism, hydrocephalus and postnatal-onset growth deficiency. The patient showed bilateral microphthalmia with severe palpebral ptosis and coloboma of the iris and left optic nerve. She also has skeletal and neurological abnormalities, cholesteatoma and seizures. She had absence of speech, poor mobility, poor vision and required help with all daily living skills. Conventional chromosome GTG banded analysis showed that the proband had an abnormal karyotype:46,XX,add(22)(q13). Fluorescence in situ hybridization (FISH) analyses and microsatellite markers for DNA polymorphism study ascertained the karyotype as 46,XX,add(22)(q13.3).ish psu dic(22;22)(q13.3;q12.1)(D14Z1/D22Z1++, N25++, ARSA+, PCP22q+). The recombinant chromosome was stable and present in all cells examined. The paternal origin of the psu dic(22;22) chromosome was determined by using five highly polymorphic microsatellite markers located to the region of chromosome 22q11.2-22q13.33. A 22q13.3 monosomy was ruled out with 22q13.3 cosmid probes covering the terminal 22q-140 Kb. The proband carried a recombinant pseudodicentric bisatellited chromosome psu dic(22;22)(q13.3;q12.1). To our knowledge, this is the first report of such rearrangement resulting in partial trisomy 22pter-22q12.1. (C) 2008 Elsevier Masson SAS. All rights reserved.
C1 [Vaglio, Alicia; Quadrelli, Andrea; Mechoso, Burix; Quadrelli, Roberto] Hosp Italiano, Inst Med Genet, Montevideo 11600, Uruguay.
[Milunsky, Aubrey; Huang, Xin-Li; Maher, Thomas A.] Boston Univ, Sch Med, Ctr Human Genet, Boston, MA 02118 USA.
RP Vaglio, A (reprint author), Hosp Italiano, Inst Med Genet, Bulevar Artigas 1632, Montevideo 11600, Uruguay.
EM rquadr@dedicado.net.uy
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NR 24
TC 0
Z9 0
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 1769-7212
J9 EUR J MED GENET
JI Eur. J. Med. Genet.
PD JUL-AUG
PY 2008
VL 51
IS 4
BP 332
EP 342
DI 10.1016/j.ejmg.2008.01.001
PG 11
WC Genetics & Heredity
SC Genetics & Heredity
GA 339CR
UT WOS:000258556200007
PM 18316257
ER
PT J
AU Vlamings, PHJM
Jonkman, LM
Hoeksma, MR
van Engeland, H
Kemner, C
AF Vlamings, Petra H. J. M.
Jonkman, Lisa M.
Hoeksma, Marco R.
van Engeland, Herman
Kemner, Chantal
TI Reduced error monitoring in children with autism spectrum disorder: an
ERP study
SO EUROPEAN JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE ACC; ERN; ERP; Pe; self-monitoring
ID ANTERIOR CINGULATE CORTEX; PERVASIVE DEVELOPMENTAL DISORDERS;
EVENT-RELATED POTENTIALS; MEDIAL-FRONTAL-CORTEX;
FUNCTIONAL-SIGNIFICANCE; NEURAL BASIS; NEGATIVITY; PERFORMANCE;
DYSFUNCTION; COMPONENTS
AB This study investigated self-monitoring in children with autism spectrum disorder (ASD) with event-related potentials looking at both the error-related negativity (ERN) and error-related positivity (Pe). The ERN is related to early error/conflict detection, and the Pe has been associated with conscious error evaluation or attention allocation. In addition, post-error slowing in reaction times (RTs) was measured. Children with ASD and age- and IQ- matched controls were administered an easy and a hard version of an auditory decision task. Results showed that the ERN was smaller in children with ASD but localized in the anterior cingulate cortex (ACC) in both groups. In addition we found a negativity on correct trials (CRN) that did not differ between the groups. Furthermore, a reduced Pe and a lack of post-error slowing in RTs were found in children with ASD. The reduced ERN in children with ASD, in the presence of an intact CRN, might suggest a specific insensitivity to detect situations in which the chance of making errors is enhanced. This might in turn lead to reduced error awareness/attention allocation to the erroneous event (reduced Pe) and eventually in a failure in change of strategy to deal with a situation, as becomes evident from the lack of post-error slowing in the ASD group. This relates well to the perseverative behaviour that is seen in children with ASD. We discuss these results in terms of a general deficit in self-monitoring, underlying social disturbance in ASD and the involvement of the ACC.
C1 [Vlamings, Petra H. J. M.; Jonkman, Lisa M.; Kemner, Chantal] Univ Maastricht, Fac Psychol, Dept Neurocognit, Maastricht, Netherlands.
[Hoeksma, Marco R.; van Engeland, Herman; Kemner, Chantal] Univ Med Ctr, Dept Child & Adolescent Psychiat, Utrecht, Netherlands.
RP Vlamings, PHJM (reprint author), Univ Maastricht, Fac Psychol, Dept Neurocognit, Maastricht, Netherlands.
EM p.vlamings@psychology.unimaas.nl
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NR 52
TC 37
Z9 37
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0953-816X
J9 EUR J NEUROSCI
JI Eur. J. Neurosci.
PD JUL
PY 2008
VL 28
IS 2
BP 399
EP 406
DI 10.1111/j.1460-9568.2008.06336.x
PG 8
WC Neurosciences
SC Neurosciences & Neurology
GA 327FU
UT WOS:000257715800017
PM 18702711
ER
PT J
AU Wong, TKW
Fung, PCW
Chua, SE
McAlonan, GM
AF Wong, Teresa K. W.
Fung, Peter C. W.
Chua, Siew E.
McAlonan, Grainne M.
TI Abnormal spatiotemporal processing of emotional facial expressions in
childhood autism: dipole source analysis of event-related potentials
SO EUROPEAN JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE BESA; brain; electroencephalography; emotion; faces
ID FUSIFORM FACE AREA; SPECTRUM DISORDER; YOUNG-CHILDREN;
ASPERGERS-SYNDROME; DIAGNOSTIC INTERVIEW; EXECUTIVE FUNCTION; TEMPORAL
CORTEX; FUNCTIONAL MRI; TACTILE INPUT; BRAIN
AB Previous studies of face processing in autism suggest abnormalities in anatomical development, functioning and connectivity/coordination of distributed brain systems involved in social cognition, but the spatial sequence and time course of rapid (sub-second) neural responses to emotional facial expressions have not been examined in detail. Source analysis of high-density event-related potentials (ERPs) is an optimal means to examine both the precise temporal profile and spatial location of early electrical brain activity in response to emotionally salient stimuli. Therefore, we recorded 128-channel ERPs from high-functioning males with autism (aged 6-10 years), and age-, sex- and IQ-matched typically developing controls during explicit and implicit processing of emotion from pictures showing happy, angry, fearful, sad and neutral facial expressions. Children with autism showed normal patterns of behavioural and ERP (P1, N170 and P2) responses. However, dipole source analysis revealed that ERP responses relating to face detection (visual cortex) and configural processing of faces (fusiform gyrus), as well as mental state decoding (medial prefrontal lobe), were significantly weaker and/or slower in autism compared with controls during both explicit and implicit emotion-processing tasks. Slower- and larger-amplitude ERP source activity in the parietal somatosensory cortices possibly reflected more effortful compensatory analytical strategies used by the autism group to process facial gender and emotion. Such aberrant neurophysiological processing of facial emotion observed in children with autism within the first 300 ms of stimulus presentation suggests abnormal cortical specialization within social brain networks, which would likely disrupt the development of normal social-cognitive skills.
C1 [Wong, Teresa K. W.; Fung, Peter C. W.; Chua, Siew E.; McAlonan, Grainne M.] Univ Hong Kong, Dept Psychiat, Pokfulam, Hong Kong, Peoples R China.
[Fung, Peter C. W.] Univ Hong Kong, Dept Med, Pokfulam, Hong Kong, Peoples R China.
[Chua, Siew E.; McAlonan, Grainne M.] Univ Hong Kong, State Key Lab Brain & Cognit Sci, Hong Kong, Hong Kong, Peoples R China.
RP Wong, TKW (reprint author), 10-64 Lab Block,21 Sassoon Rd, Pokfulam, Hong Kong, Peoples R China.
EM wongkwt@gmail.com
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NR 72
TC 30
Z9 31
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0953-816X
J9 EUR J NEUROSCI
JI Eur. J. Neurosci.
PD JUL
PY 2008
VL 28
IS 2
BP 407
EP 416
DI 10.1111/j.1460-9568.2008.06328.x
PG 10
WC Neurosciences
SC Neurosciences & Neurology
GA 327FU
UT WOS:000257715800018
PM 18702712
ER
PT J
AU Bureau, A
Labbe, A
Croteau, J
Merette, C
AF Bureau, Alexandre
Labbe, Aurelie
Croteau, Jordie
Merette, Chantal
TI Using disease symptoms to improve detection of linkage under genetic
heterogeneity
SO GENETIC EPIDEMIOLOGY
LA English
DT Article
DE familial correlation; gene mapping; latent class models; penetrance
function
ID LATENT CLASS ANALYSIS; DISORDERS; TESTS; MODELS; SCHIZOPHRENIA;
DIAGNOSIS; FAMILIES; CANCER
AB A major reason for the slow progress in identifying susceptibility genes for complex diseases may be that the clinical diagnoses used as phenotypes are genetically heterogeneous. This has led researchers to collect various phenotypes related to the diagnosis, such as detailed symptoms, in the hope that these measurements define more homogeneous disease sub-types, influenced by a smaller number of genes that will thus be more easily detectable. Latent class analysis can be used to define disease sub-types from multivariate symptoms under the assumption that the subjects are independent, an assumption that does not hold between members of the same family. We have recently developed a latent class model allowing dependence between the latent disease class status of relatives within nuclear families. In this paper, we propose approaches to use the resulting latent class probabilities in linkage analysis. We present results from a simulation study showing that the latent class approach can provide a substantial gain in power to detect disease genes over the standard heterogeneity approach of Smith and identity-by-descent sharing methods applied to the disease diagnosis. Taking into account familial dependence in the latent class model generally provides greater power than assuming independence. In an analysis of autism symptoms in families from the Autism Genetics Research Exchange, linkage signals obtained with latent class-derived phenotypes were stronger than those obtained using the original autism spectrum disorder diagnosis.
C1 [Bureau, Alexandre; Labbe, Aurelie; Croteau, Jordie; Merette, Chantal] Univ Laval Robert Giffard, Ctr Rech, Quebec City, PQ G1J 2G3, Canada.
[Bureau, Alexandre] Univ Laval, Dept Social & Prevent Med, Quebec City, PQ, Canada.
[Labbe, Aurelie] Univ Laval, Dept Math & Stat, Quebec City, PQ G1K 7P4, Canada.
[Merette, Chantal] Univ Laval, Dept Psychiat, Quebec City, PQ, Canada.
RP Bureau, A (reprint author), Univ Laval Robert Giffard, Ctr Rech, 2601 Canardiere,Suite F-4561-1, Quebec City, PQ G1J 2G3, Canada.
EM alexandre.bureau@msp.ulaval.ca
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NR 27
TC 8
Z9 8
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 0741-0395
J9 GENET EPIDEMIOL
JI Genet. Epidemiol.
PD JUL
PY 2008
VL 32
IS 5
BP 476
EP 486
DI 10.1002/gepi.20320
PG 11
WC Genetics & Heredity; Public, Environmental & Occupational Health
SC Genetics & Heredity; Public, Environmental & Occupational Health
GA 324VR
UT WOS:000257548000009
PM 18330904
ER
PT J
AU Iossifov, I
Zheng, T
Baron, M
Gilliam, TC
Rzhetsky, A
AF Iossifov, Ivan
Zheng, Tian
Baron, Miron
Gilliam, T. Conrad
Rzhetsky, Andrey
TI Genetic-linkage mapping of complex hereditary disorders to a
whole-genome molecular-interaction network
SO GENOME RESEARCH
LA English
DT Article
ID FALSE DISCOVERY RATE; SEROTONIN TRANSPORTER; ALZHEIMERS-DISEASE; BIPOLAR
DISORDER; AUTISTIC DISORDER; CANDIDATE GENES; SCHIZOPHRENIA;
ASSOCIATION; POLYMORPHISM; EXPRESSION
AB Common hereditary neurodevelopmental disorders such as autism, bipolar disorder, and schizophrenia are most likely both genetically multifactorial and heterogeneous. Because of these characteristics traditional methods for genetic analysis fail when applied to such diseases. To address the problem we propose a novel probabilistic framework that combines the standard genetic linkage formalism with whole-genome molecular-interaction data to predict pathways or networks of interacting genes that contribute to common heritable disorders. We apply the model to three large genotype-phenotype data sets, identify a small number of significant candidate genes for autism ( 24), bipolar disorder ( 21), and schizophrenia ( 25), and predict a number of gene targets likely to be shared among the disorders.
C1 [Gilliam, T. Conrad; Rzhetsky, Andrey] Univ Chicago, Dept Human Genet, Chicago, IL 60637 USA.
[Iossifov, Ivan] Columbia Univ, Dept Biomed Informat, Ctr Computat Biol & Bioinformat, New York, NY 10032 USA.
[Zheng, Tian] Columbia Univ, Dept Stat, New York, NY 10027 USA.
[Baron, Miron] Columbia Univ, Dept Psychiat, New York, NY 10032 USA.
[Rzhetsky, Andrey] Univ Chicago, Dept Med, Computat Inst, Chicago, IL 60637 USA.
RP Rzhetsky, A (reprint author), Univ Chicago, Dept Human Genet, Chicago, IL 60637 USA.
EM arzhetsky@uchicago.edu
RI rzhetsky, andrey/B-6118-2012
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NR 52
TC 34
Z9 35
PU COLD SPRING HARBOR LAB PRESS, PUBLICATIONS DEPT
PI COLD SPRING HARBOR
PA 1 BUNGTOWN RD, COLD SPRING HARBOR, NY 11724 USA
SN 1088-9051
J9 GENOME RES
JI Genome Res.
PD JUL
PY 2008
VL 18
IS 7
BP 1150
EP 1162
DI 10.1101/gr.075622.107
PG 13
WC Biochemistry & Molecular Biology; Biotechnology & Applied Microbiology;
Genetics & Heredity
SC Biochemistry & Molecular Biology; Biotechnology & Applied Microbiology;
Genetics & Heredity
GA 320QC
UT WOS:000257249100015
PM 18417725
ER
PT J
AU Hatton, S
AF Hatton, Sue
TI Autism and loss
SO HEALTH & SOCIAL CARE IN THE COMMUNITY
LA English
DT Book Review
CR Forrester-Jones R., 2007, AUTISM LOSS
NR 1
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0966-0410
J9 HEALTH SOC CARE COMM
JI Health Soc. Care Community
PD JUL
PY 2008
VL 16
IS 4
BP 446
EP 446
DI 10.1111/j.1365-2524.2008.798_10.x
PG 1
WC Public, Environmental & Occupational Health; Social Work
SC Public, Environmental & Occupational Health; Social Work
GA 316SS
UT WOS:000256970600021
ER
PT J
AU Birn, RM
Murphy, K
Bandettini, PA
AF Birn, Rasmus M.
Murphy, Kevin
Bandettini, Peter A.
TI The effect of respiration variations on independent component analysis
results of resting state functional connectivity
SO HUMAN BRAIN MAPPING
LA English
DT Article
DE default-mode network; respiration; independent component analysis;
functional connectivity; rest
ID DEFAULT-MODE; HUMAN BRAIN; FMRI DATA; ACTIVITY FLUCTUATIONS;
PHYSIOLOGICAL NOISE; HEALTHY-SUBJECTS; BOLD SIGNAL; MRI; AUTISM;
HYPOTHESIS
AB The analysis of functional connectivity in fMRI can be severely affected by cardiac and respiratory fluctuations. While some of these artifactual signal changes can be reduced by physiological noise correction routines, signal fluctuations induced by slower breath-to-breath changes in the depth and rate of breathing are typically not removed. These slower respiration-induced signal changes occur at low frequencies and spatial locations similar to the fluctuations used to infer functional connectivity, and have been shown to significantly affect seed-ROI or seed-voxel based functional connectivity analysis, particularly in the default mode network. In this study, we investigate the effect of respiration variations on functional connectivity maps derived from independent component analysis (ICA) of resting-state data. Regions of the default mode network were identified by deactivations during a lexical decision task. Variations in respiration were measured independently and correlated with the MRI time series data. ICA appears to separate the default mode network and the respiration-related changes in most cases. In some cases, however, the component automatically identified as the default mode network was the same as the component identified as respiration-related. Furthermore, in most cases the time series associated with the default mode network component was still significantly correlated with changes in respiration volume per time, suggesting that current methods of ICA may not completely separate respiration from the default mode network. An independent measure of the respiration provides valuable information to help distinguish the default mode network from respiration-related signal changes, and to assess the degree of residual respiration related effects.
C1 [Birn, Rasmus M.; Murphy, Kevin; Bandettini, Peter A.] NIMH, Sect Funct Imaging Methods, Lab Brain & Cognit, Bethesda, MD 20892 USA.
RP Birn, RM (reprint author), NIMH, Sect Funct Imaging Methods, Lab Brain & Cognit, 10 Ctr Dr,Bldg 10,Rm 1D80, Bethesda, MD 20892 USA.
EM rbirn@mail.nih.gov
RI Murphy, Kevin/A-1581-2010
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NR 53
TC 117
Z9 119
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1065-9471
J9 HUM BRAIN MAPP
JI Hum. Brain Mapp.
PD JUL
PY 2008
VL 29
IS 7
BP 740
EP 750
DI 10.1002/hbm.20577
PG 11
WC Neurosciences; Neuroimaging; Radiology, Nuclear Medicine & Medical
Imaging
SC Neurosciences & Neurology; Radiology, Nuclear Medicine & Medical Imaging
GA 312MB
UT WOS:000256674400002
PM 18438886
ER
PT J
AU Calhoun, VD
Kiehl, KA
Pearlson, GD
AF Calhoun, Vince D.
Kiehl, Kent A.
Pearlson, Godfrey D.
TI Modulation of temporally coherent brain networks estimated using ICA at
rest and during cognitive tasks
SO HUMAN BRAIN MAPPING
LA English
DT Article
DE fMRI; auditory oddball; independent component analysis; P3;
schizophrenia
ID INDEPENDENT COMPONENT ANALYSIS; AUDITORY ODDBALL TASK; LOW-FREQUENCY
FLUCTUATIONS; FUNCTIONAL MRI DATA; DEFAULT-MODE; BLIND SEPARATION; FMRI
ANALYSIS; BOLD SIGNAL; CONNECTIVITY; SCHIZOPHRENIA
AB Brain regions which exhibit temporally coherent fluctuations, have been increasingly studied using functional magnetic resonance imaging (fMRI). Such networks are often identified in the context of an fMRI scan collected during rest (and thus are called "resting state networks"); however, they are also present during (and modulated by) the performance of a cognitive task. In this article, we will refer to such networks as temporally coherent networks (TCNs). Although there is still some debate over the physiological source of these fluctuations, TCNs are being studied in a variety of ways. Recent studies have examined ways TCNs can be used to identify patterns associated with various brain disorders (e.g. schizophrenia, autism or Alzheimer's disease). Independent component analysis (ICA) is one method being used to identify TCNs. ICA is a data driven approach which is especially useful for decomposing activation during complex cognitive tasks where multiple operations occur simultaneously. In this article we review recent TCN studies with emphasis on those that use ICA. We also present new results showing that TCNs are robust, and can be consistently identified at rest and during performance of a cognitive task in healthy individuals and in patients with schizophrenia. In addition, multiple TCNs show temporal and spatial modulation during the cognitive task versus rest. In summary, TCNs show considerable promise as potential imaging biological markers of brain diseases, though each network needs to be studied in more detail.
C1 [Calhoun, Vince D.; Kiehl, Kent A.] MIND Inst, Albuquerque, NM 87131 USA.
[Calhoun, Vince D.; Kiehl, Kent A.; Pearlson, Godfrey D.] Inst Living, Olin Neuropsychiat Res Ctr, Hartford, CT 06106 USA.
[Calhoun, Vince D.; Kiehl, Kent A.; Pearlson, Godfrey D.] Yale Univ, Sch Med, Dept Psychiat, New Haven, CT 06520 USA.
[Calhoun, Vince D.] Univ New Mexico, Dept ECE, Albuquerque, NM 87131 USA.
[Kiehl, Kent A.] Univ New Mexico, Dept Psychol, Albuquerque, NM 87131 USA.
RP Calhoun, VD (reprint author), MIND Inst, 1101 Yale Blvd NE, Albuquerque, NM 87131 USA.
EM vcalhoun@unm.edu
RI Calhoun, Vince/H-7146-2013
OI Calhoun, Vince/0000-0001-9058-0747
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NR 41
TC 224
Z9 225
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 1065-9471
J9 HUM BRAIN MAPP
JI Hum. Brain Mapp.
PD JUL
PY 2008
VL 29
IS 7
BP 828
EP 838
DI 10.1002/hbm.20581
PG 11
WC Neurosciences; Neuroimaging; Radiology, Nuclear Medicine & Medical
Imaging
SC Neurosciences & Neurology; Radiology, Nuclear Medicine & Medical Imaging
GA 312MB
UT WOS:000256674400011
PM 18438867
ER
PT J
AU Allan, AM
Liang, XM
Luo, YP
Pak, CH
Li, XK
Szulwach, KE
Chen, DH
Jin, P
Zhao, XY
AF Allan, Andrea M.
Liang, Xiaomin
Luo, Yuping
Pak, ChangHui
Li, Xuekun
Szulwach, Keith E.
Chen, Dahua
Jin, Peng
Zhao, Xinyu
TI The loss of methyl-CpG binding protein 1 leads to autism-like behavioral
deficits
SO HUMAN MOLECULAR GENETICS
LA English
DT Article
ID SEROTONIN 5-HT2C RECEPTORS; RIGID-COMPULSIVE BEHAVIORS; WET-DOG SHAKES;
DNA METHYLATION; RETT-SYNDROME; KNOCKOUT MICE; TRANSCRIPTIONAL
REPRESSION; SUSCEPTIBILITY LOCI; GENE POLYMORPHISMS; GENOMEWIDE SCREEN
AB Methyl-CpG binding proteins (MBDs) are central components of DNA methylation-mediated epigenetic gene regulation. Alterations of epigenetic pathways are known to be associated with several neurodevelopmental disorders, particularly autism. Our previous studies showed that the loss of Mbd1 led to reduced hippocampal neurogenesis and impaired learning in mice. However, whether MBD1 regulates the autism-related cognitive functions remains unknown. Here we show that Mbd1 mutant (Mbd1(-/-)) mice exhibit several core deficits frequently associated with autism, including reduced social interaction, learning deficits, anxiety, defective sensory motor gating, depression and abnormal brain serotonin activity. Furthermore, we find that Mbd1 can directly regulate the expression of Htr2c, one of the serotonin receptors, by binding to its promoter, and the loss of Mbd1 led to elevated expression of Htr2c. Our results, therefore, demonstrate the importance of epigenetic regulation in mammalian brain development and cognitive functions. Understanding how the loss of Mbd1 could lead to autism-like behavioral phenotypes would reveal much-needed information about the molecular pathogenesis of autism.
C1 [Allan, Andrea M.; Liang, Xiaomin; Luo, Yuping; Li, Xuekun; Zhao, Xinyu] Univ New Mexico, Dept Neurosci, Albuquerque, NM 87131 USA.
[Pak, ChangHui; Szulwach, Keith E.; Jin, Peng] Emory Univ, Sch Med, Dept Human Genet, Atlanta, GA 30322 USA.
[Pak, ChangHui; Szulwach, Keith E.; Jin, Peng] Emory Univ, Sch Med, Grad Program Genet & Mol Biol, Atlanta, GA 30322 USA.
[Chen, Dahua] Chinese Acad Sci, Inst Zool, State Key Lab Biomembrane & Membrane Biotechnol, Beijing 100101, Peoples R China.
RP Zhao, XY (reprint author), Univ New Mexico, Dept Neurosci, Albuquerque, NM 87131 USA.
EM xzhao@salud.unm.edu
RI Jin, Peng/B-5977-2012
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NR 69
TC 45
Z9 46
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0964-6906
J9 HUM MOL GENET
JI Hum. Mol. Genet.
PD JUL 1
PY 2008
VL 17
IS 13
BP 2047
EP 2057
DI 10.1093/hmg/ddn102
PG 11
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 316VQ
UT WOS:000256978200017
PM 18385101
ER
PT J
AU Borck, G
Molla-Herman, A
Boddaert, N
Encha-Razavi, F
Philippe, A
Robel, L
Desguerre, I
Brunelle, F
Benmerah, A
Munnich, A
Colleaux, L
AF Borck, Guntram
Molla-Herman, Anahi
Boddaert, Nathalie
Encha-Razavi, Ferechte
Philippe, Anne
Robel, Laurence
Desguerre, Isabelle
Brunelle, Francis
Benmerah, Alexandre
Munnich, Arnold
Colleaux, Laurence
TI Clinical, cellular, and neuropathologlical consequences of AP1S2
mutations: Further delineation of a recognizable X-linked mental
retardation syndrome
SO HUMAN MUTATION
LA English
DT Article
DE mental retardation; clathrin; adaptor protein complex 1; AP-1; cerebral
calcifications; elevated CSF protein levels; AP1S2
ID ADAPTER PROTEIN-1 COMPLEX; CLATHRIN ADAPTER; COATED VESICLES;
GAMMA-ADAPTIN; BASAL GANGLIA; AP-1 BINDING; HIV-1 NEF; MEMBRANE;
IDENTIFICATION; HEMICOMPLEXES
AB Mutations in the AP1S2 gene, encoding the sigma 1B subunit of the clathrin-associated adaptor protein complex (AP)-1, have been recently identified in five X-linked mental retardation (XLMR) families, including the original family with Fried syndrome. Studying four patients in two unrelated families in which AP1S2 nonsense and splice-site mutations segregated, we found that affected individuals presented, in addition to previously described features, with elevated protein levels in cerebrospinal fluid (CSF). Moreover, computed tomography scans demonstrated that the basal ganglia calcifications associated with AP1S2 mutations appeared during childhood and might be progressive. Based on these observations, we propose that AP1S2 mutations are responsible for a clinically recognizable XLMR and autism syndrome associating hypotonia, delayed walking, speech delay, aggressive behavior, brain calcifications, and elevated CSF protein levels. Using the AP-2 complex, in which the sigma subunit is encoded by one single gene, as a model system, we demonstrated that a subunits are essential for the stability of human AP complexes. By contrast, no major alteration of the stability, subcellular localization, and function of the AP-1 complex was observed in fibroblasts derived from a patient carrying an AP1S2 mutation. Similarly, neither macro- nor microscopic defects were observed in the brain of an affected fetus. Altogether, these data suggest that the absence of an AP-1 defect in peripheral tissues is due to functional redundancy among AP-1 sigma subunits (sigma 1A, sigma 1B, and sigma 1C) and that the phenotype observed in our patients results from a subtle and brain-specific defect of the AP-1-dependent intracellular protein traffic.
C1 [Borck, Guntram; Philippe, Anne; Munnich, Arnold; Colleaux, Laurence] Univ Paris 05, Fac Med, Hop Necker Enfants Malad, INSERM U781,AP HP, F-75015 Paris, France.
[Borck, Guntram; Philippe, Anne; Munnich, Arnold; Colleaux, Laurence] Univ Paris 05, Fac Med, Hop Necker Enfants Malad, Dept Med Genet,AP HP, F-75015 Paris, France.
[Boddaert, Nathalie; Brunelle, Francis] Univ Paris 05, Fac Med, Hop Necker Enfants Malad, INSERM U797,AP HP, F-75015 Paris, France.
[Boddaert, Nathalie; Brunelle, Francis] Univ Paris 05, Fac Med, Hop Necker Enfants Malad, Dept Pediat Radiol,AP HP, F-75015 Paris, France.
[Encha-Razavi, Ferechte] Univ Paris 05, Fac Med, Hop Necker Enfants Malad, Dept Genet,Embryo Fetal Pathol Unit,AP HP, F-75015 Paris, France.
[Robel, Laurence] Univ Paris 05, Fac Med, Hop Necker Enfants Malad, Dept Child Psychiat,AP HP, F-75015 Paris, France.
[Desguerre, Isabelle] Univ Paris 05, Fac Med, Hop Necker Enfants Malad, Dept Neuropediat,AP HP, F-75015 Paris, France.
[Benmerah, Alexandre] INSERM, U567, Paris, France.
[Benmerah, Alexandre] Univ Paris 05, CNRS UMR 8104, Inst Cochin, Paris, France.
RP Colleaux, L (reprint author), Univ Paris 05, Fac Med, Hop Necker Enfants Malad, INSERM U781,AP HP, Tour Lavoisier,149 Rue Sevres, F-75015 Paris, France.
EM colleaux@necker.fr
RI Borck, Guntram/F-1868-2015
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NR 38
TC 26
Z9 26
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1059-7794
J9 HUM MUTAT
JI Hum. Mutat.
PD JUL
PY 2008
VL 29
IS 7
BP 966
EP 974
DI 10.1002/humu.20531
PG 9
WC Genetics & Heredity
SC Genetics & Heredity
GA 323WR
UT WOS:000257479600009
PM 18428203
ER
PT J
AU Ghosh, M
Shah, AH
Dhir, K
Merchant, KF
AF Ghosh, Madhumita
Shah, Amita H.
Dhir, Kiran
Merchant, Kaneez Fatima
TI Behavior in children with Down syndrome
SO INDIAN JOURNAL OF PEDIATRICS
LA English
DT Article
DE Down syndrome (DS); Autism Spectrum Disorder (ASD); socialization
skills; communication skills; repetitive stereotyped behaviors
ID AUTISM
AB Objective. To highlight the differences in behaviors in children with diagnosis of down syndrome.
Method. Eight children with Down syndrome who displayed autistic features were compared with eight Down syndrome children without autistic features. These children were randomly selected and were matched for age and level of retardation. Standardized Psychological tests were administered to tap the behavioral differences. Mann-Whitney U test was used for significance of difference between both the groups.
Results. Down syndrome children without Autism Spectrum Disorder had better communication and socialization skills than children with Down syndrome with Autism Spectrum Disorder. Down syndrome children with Autism Spectrum Disorder displayed more restricted repetitive and stereotyped patterns of behaviors, interests and activities.
Conclusion. Our findings indicate that Autism Spectrum Disorder manifests as a distinct behavioral phenomenon in Down syndrome. Hence it is important for professionals to consider the possibility of a dual diagnosis which will entitle the child to a more specialized and effective educational and intervention services.
C1 [Ghosh, Madhumita] Res Soc Care Treatment & Training Children Specia, Dept Psychol, Smt Motibai Thackersey Inst Res Field Mental Reta, Bombay 400033, Maharashtra, India.
RP Ghosh, M (reprint author), Res Soc Care Treatment & Training Children Specia, Dept Psychol, Smt Motibai Thackersey Inst Res Field Mental Reta, Sewri Rd, Bombay 400033, Maharashtra, India.
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NR 16
TC 1
Z9 1
PU ALL INDIA INST MEDICAL SCIENCES
PI NEW DELHI
PA ANSARI NAGAR, NEW DELHI 110 029, INDIA
SN 0019-5456
J9 INDIAN J PEDIATR
JI Indian J. Pediatr.
PD JUL
PY 2008
VL 75
IS 7
BP 685
EP 689
DI 10.1007/s12098-008-0129-z
PG 5
WC Pediatrics
SC Pediatrics
GA 340PU
UT WOS:000258658300003
PM 18716736
ER
PT J
AU Tyminski, RF
Moore, PJ
AF Tyminski, Robert F.
Moore, Philip J.
TI The impact of group psychotherapy on social development in children with
pervasive developmental disorders
SO INTERNATIONAL JOURNAL OF GROUP PSYCHOTHERAPY
LA English
DT Article
ID AUTISM SPECTRUM DISORDERS; HIGH-FUNCTIONING CHILDREN; ADAPTIVE-BEHAVIOR
SCALES; PREVALENCE; SKILLS
AB Thirty-nine children with a diagnosed pervasive developmental disorder (PDD) participated in homogeneous psychotherapy groups. Their social development was assessed at home and at school both before and after treatment. Significant improvements in social functioning were observed in both settings, and these gains were not attributable to demographic variables or cognitive development. This research provides psychometric validation for the Vineland Adaptive Behavior Scale (VABS; Sparrow, Balla, & Cicchetti, 1984) and the Tyminski Social Skills Checklist (SSC), a new index designed to assess children's social functioning in educational settings. The results suggest that group therapy may be an important modality for better understanding and developing crucial social skills among the growing number of children diagnosed with a pervasive developmental disorder.
C1 [Tyminski, Robert F.] Univ Calif San Francisco, Dept Psychiat, San Francisco, CA 94143 USA.
[Moore, Philip J.] George Washington Univ, Dept Psychol, Washington, DC 20052 USA.
RP Tyminski, RF (reprint author), 3529 Sacramento St, San Francisco, CA 94118 USA.
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NR 27
TC 1
Z9 1
PU GUILFORD PUBLICATIONS INC
PI NEW YORK
PA 72 SPRING STREET, NEW YORK, NY 10012 USA
SN 0020-7284
J9 INT J GROUP PSYCHOTH
JI Int. J. Group Psychother.
PD JUL
PY 2008
VL 58
IS 3
BP 363
EP 379
DI 10.1521/ijgp.2008.58.3.363
PG 17
WC Psychology, Clinical
SC Psychology
GA 317RN
UT WOS:000257037400005
PM 18573027
ER
PT J
AU Vojdani, A
AF Vojdani, A.
TI ANTIBODIES AS PREDICTORS OF COMPLEX AUTOIMMUNE DISEASES AND CANCER
SO INTERNATIONAL JOURNAL OF IMMUNOPATHOLOGY AND PHARMACOLOGY
LA English
DT Article
DE predictive antibodies; neuroimmune disorders; autoimmune diseases;
cancer; environmental triggers; polyreactive antibodies;
ELISAIntroduction
ID BLOOD-BRAIN-BARRIER; CENTRAL-NERVOUS-SYSTEM; MULTIPLE-SCLEROSIS;
PROSTATE-CANCER; B-CELLS; AUTOANTIBODIES; ACTIVATION; RECEPTORS;
EPITOPES; PROTEIN
AB The pathologic role of autoantibodies in many autoimmune diseases is widely accepted. An enzyme immunoassay was used for measurement of antibodies against disease-specific antigens and etiologic agents for cross-reactive antigens associated with them. This antibody assay was applied to a panel of antigens for the detection of different neuroautoimmune diseases that included multiple sclerosis, motor peripheral neuropathies, multifocal motor neuropathy, amyotrophic lateral sclerosis and pediatric autoimmune neuropsychiatric disorder associated with streptococcal infection. We studied women with pregnancies complicated by neural tube defect, neuroborreliosis, autism and patients with possible somatic hypermutation. Antibodies were also measured against antigens and etiologic agents associated with primary biliary cirrhosis and chronic obstructive pulmonary disease. And, finally, antibodies were measured against several tumor antigens or peptides which are expressed in prostatic, breast and colon tissues. This panel of different autoantibodies was applied to 290 patients with neuroautoimmune disorders, cancer, and possible somatic hypermutation. The levels of these antibodies against different tissue-specific antigens and etiologic agents associated with them were significantly elevated in patients versus controls. We hope that this novel 96 antigen-specific ELISA will be used in additional studies that will prove its clinical efficacy, not only for the early diagnosis of many neuroautoimmune, liver and lung autoimmune disorders, but also for prognosis and the implementation of preventive steps for many complex diseases.
C1 [Vojdani, A.] Immunisci Lab Inc, Beverly Hills, CA USA.
RP Vojdani, A (reprint author), 8693 Wilshire Blvd,Suite 200, Beverly Hills, CA 90211 USA.
EM immunsci@ix.netcom.com
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NR 40
TC 11
Z9 11
PU BIOLIFE SAS
PI SILVA MARINA (TE)
PA VIA S STEFANO 39 BIS, 64029 SILVA MARINA (TE), ITALY
SN 0394-6320
J9 INT J IMMUNOPATH PH
JI Int. J. Immunopathol. Pharmacol.
PD JUL-SEP
PY 2008
VL 21
IS 3
BP 553
EP 566
PG 14
WC Immunology; Pathology; Pharmacology & Pharmacy
SC Immunology; Pathology; Pharmacology & Pharmacy
GA 359KR
UT WOS:000259986100008
PM 18831922
ER
PT J
AU Kakooza-Mwesige, A
Wachtel, L
Dhossche, D
AF Kakooza-Mwesige, A.
Wachtel, L.
Dhossche, D.
TI Catatonia in autism: Implications across the life span
SO INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY
LA English
DT Meeting Abstract
CT 26th Collegium Internationale Neuro-Psychopharmacologicum Congress
(CINP)
CY JUL 13-17, 2008
CL Munich, GERMANY
C1 [Dhossche, D.] Univ Mississippi, Jackson, MS USA.
NR 0
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1461-1457
J9 INT J NEUROPSYCHOPH
JI Int. J. Neuropsychopharmacol.
PD JUL
PY 2008
VL 11
SU 1
BP 90
EP 91
PG 2
WC Clinical Neurology; Neurosciences; Pharmacology & Pharmacy; Psychiatry
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Psychiatry
GA 343LO
UT WOS:000258855500381
ER
PT J
AU Fatemi, SH
Reutiman, T
Folsom, T
Thuras, P
AF Fatemi, S. H.
Reutiman, T.
Folsom, T.
Thuras, P.
TI Global reductions in levels of GABAA and GARAB receptors in brains of
subjects with autism
SO INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY
LA English
DT Meeting Abstract
CT 26th Collegium Internationale Neuro-Psychopharmacologicum Congress
(CINP)
CY JUL 13-17, 2008
CL Munich, GERMANY
C1 [Fatemi, S. H.; Reutiman, T.; Folsom, T.; Thuras, P.] Univ Minnesota, Minneapolis, MN USA.
NR 0
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1461-1457
J9 INT J NEUROPSYCHOPH
JI Int. J. Neuropsychopharmacol.
PD JUL
PY 2008
VL 11
SU 1
BP 233
EP 233
PG 1
WC Clinical Neurology; Neurosciences; Pharmacology & Pharmacy; Psychiatry
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Psychiatry
GA 343LO
UT WOS:000258855501250
ER
PT J
AU Dhossche, D
Kakooza, A
Wachtel, L
AF Dhossche, D.
Kakooza, A.
Wachtel, L.
TI Catatonia in autism: Implications across the life span
SO INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY
LA English
DT Meeting Abstract
CT 26th Collegium Internationale Neuro-Psychopharmacologicum Congress
(CINP)
CY JUL 13-17, 2008
CL Munich, GERMANY
C1 [Dhossche, D.] Univ Mississippi, Jackson, MS USA.
[Kakooza, A.] Makerere Univ, Kampala, Uganda.
[Wachtel, L.] Kennedy Krieger Inst, Baltimore, MD USA.
NR 0
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1461-1457
J9 INT J NEUROPSYCHOPH
JI Int. J. Neuropsychopharmacol.
PD JUL
PY 2008
VL 11
SU 1
BP 293
EP 293
PG 1
WC Clinical Neurology; Neurosciences; Pharmacology & Pharmacy; Psychiatry
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Psychiatry
GA 343LO
UT WOS:000258855501491
ER
PT J
AU Martenkoysky, I
Bikshaeva, Y
Martsenkovska, I
Butenko, L
AF Martenkoysky, I.
Bikshaeva, Y.
Martsenkovska, I.
Butenko, L.
TI Divalproex sodium and risperidone for the treatment of preschool
children with autism
SO INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY
LA English
DT Meeting Abstract
CT 26th Collegium Internationale Neuro-Psychopharmacologicum Congress
(CINP)
CY JUL 13-17, 2008
CL Munich, GERMANY
C1 [Martenkoysky, I.; Bikshaeva, Y.; Martsenkovska, I.; Butenko, L.] URI SF PDA, Kiev, Ukraine.
NR 0
TC 0
Z9 0
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1461-1457
J9 INT J NEUROPSYCHOPH
JI Int. J. Neuropsychopharmacol.
PD JUL
PY 2008
VL 11
SU 1
BP 293
EP 293
PG 1
WC Clinical Neurology; Neurosciences; Pharmacology & Pharmacy; Psychiatry
SC Neurosciences & Neurology; Pharmacology & Pharmacy; Psychiatry
GA 343LO
UT WOS:000258855501492
ER
PT J
AU Wilkinson, N
Ang, RP
Goh, DH
AF Wilkinson, Nathan
Ang, Rebecca P.
Goh, Dion H.
TI Online video game therapy for mental health concerns: A review
SO INTERNATIONAL JOURNAL OF SOCIAL PSYCHIATRY
LA English
DT Article
DE online video games; therapy; mental health; review
ID OBSESSIVE-COMPULSIVE DISORDER; AUTISTIC SPECTRUM DISORDERS;
VIRTUAL-REALITY; COMPUTER GAMES; COGNITIVE REHABILITATION; EXPOSURE
THERAPY; IN-VIVO; ADOLESCENTS; CHILDREN; ENVIRONMENTS
AB Background: There has been research on the use of offline video games for therapeutic purposes but online video game therapy is still fairly under-researched. Online therapeutic interventions have only recently included a gaming component. Hence, this review represents a timely first step toward taking advantage of these recent technological and cultural innovations, particularly for the treatment of special-needs groups such as the young, the elderly and people with various conditions such as ADHD, anxiety and autism spectrum disorders.
Material: A review integrating research findings on two technological advances was conducted: the home computer boom of the 1980s, which triggered a flood of research on therapeutic video games for the treatment of various mental health conditions; and the rise of the internet in the 1990s, which caused computers to be seen as conduits for therapeutic interaction rather than replacements for the therapist.
Discussion: We discuss how video games and the internet can now be combined in therapeutic interventions, as attested by a consideration of pioneering studies.
Conclusion: Future research into online video game therapy for mental health concerns might focus on two broad types of game: simple society games, which are accessible and enjoyable to players of all ages, and online worlds, which offer a unique opportunity for narrative content and immersive remote interaction with therapists and fellow patients. Both genres might be used for assessment and training purposes, and provide an unlimited platform for social interaction. The mental health community can benefit from more collaborative efforts between therapists and engineers, making such innovations a reality.
C1 [Ang, Rebecca P.] Nanyang Technol Univ, Sch Humanities & Social Sci, Div Psychol, Singapore, Singapore.
[Goh, Dion H.] Nanyang Technol Univ, Wee Kim Kee Sch Commun & Informat, Div Informat Studies, Singapore, Singapore.
RP Ang, RP (reprint author), Nanyang Technol Univ, Sch Humanities & Social Sci, Div Psychol, Singapore, Singapore.
EM rpang@ntu.edu.sg
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NR 70
TC 31
Z9 31
PU SAGE PUBLICATIONS LTD
PI LONDON
PA 1 OLIVERS YARD, 55 CITY ROAD, LONDON EC1Y 1SP, ENGLAND
SN 0020-7640
EI 1741-2854
J9 INT J SOC PSYCHIATR
JI Int. J. Soc. Psychiatr.
PD JUL
PY 2008
VL 54
IS 4
BP 370
EP 382
DI 10.1177/0020764008091659
PG 13
WC Psychiatry
SC Psychiatry
GA 331SE
UT WOS:000258032100009
PM 18720897
ER
PT J
AU Toriello, HV
AF Toriello, Helga V.
TI Intellectual disability and genetic influences
SO INTERNATIONAL JOURNAL ON DISABILITY AND HUMAN DEVELOPMENT
LA English
DT Review
DE Comparative genomic hybridization; genetic counseling; genetics
evaluation; human development
ID AUTISM SPECTRUM DISORDERS; PRADER-WILLI-SYNDROME; MOWAT-WILSON-SYNDROME;
MENTAL-RETARDATION; DELETION SYNDROME; MICRODELETION SYNDROME; GENOMIC
HYBRIDIZATION; CONGENITAL DISORDERS; FAMILIAL DELETION; MUTATIONS
AB Clearly, chromosome abnormalities and single gene mutations are a significant cause of intellectual disability (113). In many cases, however, the cause of the ID is unknown. Recently developed techniques and molecular discoveries can provide a means of diagnosing the cause of that individual's ID, thus providing that person and his/her family with prognostic and management information.
C1 [Toriello, Helga V.] Spectrum Hlth Hosp, Grand Rapids, MI USA.
[Toriello, Helga V.] Michigan State Univ, Dept Pediat & Human Dev, E Lansing, MI 48824 USA.
RP Toriello, HV (reprint author), Genet Serv, 21 Michigan St,Suite 465, Grand Rapids, MI 49503 USA.
EM Heiga.toriello@spectrum-health.org
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NR 46
TC 1
Z9 1
PU FREUND PUBLISHING HOUSE LTD
PI TEL AVIV
PA PO BOX 35010, TEL AVIV 61350, ISRAEL
SN 1565-012X
J9 INT J DISABIL HUM DE
JI Int. J. Disabil. Hum. Dev.
PD JUL-SEP
PY 2008
VL 7
IS 3
SI SI
BP 349
EP 354
PG 6
WC Public, Environmental & Occupational Health
SC Public, Environmental & Occupational Health
GA 377VV
UT WOS:000261280800017
ER
PT J
AU Gray, KM
Tonge, BJ
Sweeney, DJ
Einfeld, SL
AF Gray, K. M.
Tonge, B. J.
Sweeney, D. J.
Einfeld, S. L.
TI Screening for autism in young children with developmental delay: An
evaluation of the developmental behaviour checklist: Early screen
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE Screening; Preschool children; dvelopmental delay
ID SOCIAL COMMUNICATION QUESTIONNAIRE; PRESCHOOL-CHILDREN; DISORDERS;
SPECTRUM; POPULATION; PREVALENCE; INSTRUMENT; VALIDITY; ESAT
AB The ability to identify children who require specialist assessment for the possibility of autism at as early an age as possible has become a growing area of research. A number of measures have been developed as potential screening tools for autism. The reliability and validity of one of these measures for screening for autism in young children with developmental problems was evaluated. The parents of 207 children aged 20-51 months completed the Developmental Checklist-Early Screen (DBC-ES), prior to their child undergoing assessment. Good interrater agreement and internal consistency was found, along with significant correlations with a clinician completed measure of autism symptomatology. High sensitivity was found, with lower specificity for the originally proposed 17-item screening tool and a five-item version.
C1 [Gray, K. M.] Monash Med Ctr, Child & Adolescent Mental Hlth Serv, Ctr Dev Psychiat & Psychol, Clayton, Vic 3168, Australia.
[Gray, K. M.; Tonge, B. J.; Sweeney, D. J.] Monash Univ, Sch Psychol Psychiat & Psychol Med, Ctr Dev Psychiat & Psychol, Clayton, Vic, Australia.
[Einfeld, S. L.] Univ New S Wales, Sch Psychiat, Sydney, NSW, Australia.
RP Gray, KM (reprint author), Monash Med Ctr, Child & Adolescent Mental Hlth Serv, Ctr Dev Psychiat & Psychol, 246 Clayton Rd, Clayton, Vic 3168, Australia.
EM kylie.gray@med.monash.edu.au
RI Gray, Kylie/H-3345-2014
OI Gray, Kylie/0000-0001-6518-4240
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Wechsler D., 2002, WECHSLER PRESCHOOL P, V3rd
NR 28
TC 13
Z9 13
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
EI 1573-3432
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1003
EP 1010
DI 10.1007/s10803-007-0473-2
PG 8
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700001
PM 17972169
ER
PT J
AU Sterling, L
Dawson, G
Estes, A
Greenson, J
AF Sterling, Lindsey
Dawson, Geraldine
Estes, Annette
Greenson, Jessica
TI Characteristics associated with presence of depressive symptoms in
adults with autism spectrum disorder
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; depression; anxiety; comorbidity; associated psychopathology;
cognitive ability
ID PERVASIVE DEVELOPMENTAL DISORDERS; OBSESSIVE-COMPULSIVE DISORDER;
HIGH-FUNCTIONING CHILDREN; ASPERGER-SYNDROME; LIFE EVENTS;
PSYCHIATRIC-DISORDERS; YOUNG-PEOPLE; ADOLESCENTS; PREVALENCE;
INDIVIDUALS
AB Evidence suggests that individuals with autism spectrum disorders (ASD) often exhibit associated psychiatric symptoms, particularly related to depression. The current study investigated whether individual characteristics, specifically, severity of ASD symptoms, level of cognitive ability, and/or presence of other psychiatric disorders, are associated with occurrence of depressive symptoms in adults with ASD. Forty-six adults with ASD were administered a standardized psychiatric history interview. Twenty participants (43%) endorsed depressive symptoms. It was found that individuals with less social impairment, higher cognitive ability, and higher rates of other psychiatric symptoms, were more likely to report depressive symptoms. These characteristics may be vulnerability factors for the development of depression, and should be considered when screening and treating adults with ASD.
C1 [Sterling, Lindsey; Dawson, Geraldine; Greenson, Jessica] Univ Washington, Dept Psychol, UW Autism Ctr, Seattle, WA 98195 USA.
[Dawson, Geraldine; Estes, Annette] Univ Washington, Dept Psychiat, UW Autism Ctr, Seattle, WA 98195 USA.
[Dawson, Geraldine; Estes, Annette] Univ Washington, Dept Behav Sci, UW Autism Ctr, Seattle, WA 98195 USA.
RP Dawson, G (reprint author), Univ Washington, Dept Psychol, UW Autism Ctr, Seattle, WA 98195 USA.
EM lsterlng@u.washington.edu; dawson@u.washington.edu
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NR 51
TC 43
Z9 43
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1011
EP 1018
DI 10.1007/s10803-007-0477-y
PG 8
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700002
PM 17975722
ER
PT J
AU Goin-Kochel, RP
Mazefsky, CA
Riley, BP
AF Goin-Kochel, Robin P.
Mazefsky, Carla A.
Riley, Brien P.
TI Level of functioning in autism spectrum disorders: Phenotypic congruence
among affected siblings
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE AGRE; broad spectrum; twins; IQ; adaptive behavior
ID PERVASIVE DEVELOPMENTAL DISORDERS; BEHAVIORAL TREATMENT;
ADAPTIVE-BEHAVIOR; SYMPTOM DOMAINS; INDIVIDUALS; CHILDREN; MULTIPLEX;
FAMILIES; GENETICS; TWIN
AB Little evidence supports that siblings with autism exhibit the same behaviors; however, some findings suggest that level of functioning shows familial aggregation. We tested this notion among multiplex families participating with the Autism Genetic Resource Exchange (AGRE) Consortium, using scores on the Peabody Picture Vocabulary Test-Third Edition (N = 204 families), the Ravens Colored Progressive Matrices (N = 226 families), and the Vineland Adaptive Behavior Scales (N = 348 families). Intraclass Correlation Coefficients revealed that siblings with autism/autism spectrum disorders (ASD) were more similar on measures of verbal and nonverbal IQ and adaptive functioning than were unrelated children with autism/ASD. Preliminary twin correlations indicated strong genetic effects for some skill domains and the influence of shared environmental factors for others.
C1 [Goin-Kochel, Robin P.] Baylor Univ, Texas Childrens Hosp, Houston, TX 77030 USA.
[Mazefsky, Carla A.] Univ Pittsburgh, Dept Pediat, Pittsburgh, PA 15260 USA.
[Mazefsky, Carla A.] Univ Pittsburgh, Dept Psychiat, Pittsburgh, PA 15260 USA.
[Riley, Brien P.] Virginia Commonwealth Univ, Virginia Inst Psychiat & Behav Genet, Richmond, VA USA.
RP Goin-Kochel, RP (reprint author), Baylor Univ, Texas Childrens Hosp, 6621 Fannin St,CC1560, Houston, TX 77030 USA.
EM kochel@bcm.tmc.edu
CR *AUT GEN RES EXCH, 2006, AGRE AFF STAT CAT
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NR 33
TC 7
Z9 7
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1019
EP 1027
DI 10.1007/s10803-007-0476-z
PG 9
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700003
PM 17968643
ER
PT J
AU Knickmeyer, RC
Wheelwright, S
Baron-Cohen, SB
AF Knickmeyer, Rebecca C.
Wheelwright, Sally
Baron-Cohen, Simon B.
TI Sex-typical play: Masculinization/Defeminization in girls with an autism
spectrum condition
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; sex differences; play; fetal testosterone
ID CONGENITAL ADRENAL-HYPERPLASIA; GENDER-ROLE DEVELOPMENT; FETAL
TESTOSTERONE; SYMBOLIC PLAY; TUMBLE PLAY; SCHOOL-AGE; MALE BRAIN;
BEHAVIOR; CHILDREN; ANDROGENS
AB We tested the hypothesis that prenatal masculinization of the brain by androgens increases risk of developing an autism spectrum condition (ASC). Sex-typical play was measured in n = 66 children diagnosed with an ASC and n = 55 typically developing age-matched controls. Consistent with the hypothesis, girls with autism did not show the female-typical play preferences, though this was only seen on non-pretence items. Boys with autism showed a preference for male play on non-pretence items, in keeping with their sex. Girls with autism engaged in more pretend play than boys with autism, suggesting that pretence is relatively more protected in females with autism. We conclude that play preference studies in ASC provide partial support for the fetal androgen theory.
C1 [Knickmeyer, Rebecca C.] Univ N Carolina, Dept Psychiat, Chapel Hill, NC 27599 USA.
[Knickmeyer, Rebecca C.; Wheelwright, Sally; Baron-Cohen, Simon B.] Univ Cambridge, Dept Psychiat, Autism Res Ctr, Cambridge, England.
RP Knickmeyer, RC (reprint author), Univ N Carolina, Dept Psychiat, CB 7160,7023 Neurosci Hosp, Chapel Hill, NC 27599 USA.
EM rebecca_knickmeyer@med.unc.edu
RI Knickmeyer, Rebecca/G-8128-2014
OI Knickmeyer, Rebecca/0000-0001-7708-1388
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NR 40
TC 19
Z9 21
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1028
EP 1035
DI 10.1007/s10803-007-0475-0
PG 8
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700004
PM 17985222
ER
PT J
AU Coo, H
Ouellette-Kuntz, H
Lloyd, JEV
Kasmara, L
Holden, JJA
Lewis, MES
AF Coo, Helen
Ouellette-Kuntz, Helene
Lloyd, Jennifer E. V.
Kasmara, Liza
Holden, Jeanette J. A.
Lewis, M. E. Suzanne
TI Trends in autism prevalence: Diagnostic substitution revisited
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; autistic disorder; diagnostic substitution; prevalence; Edudata
Canada; British Columbia Ministry of Education
ID SPECTRUM DISORDERS; CHANGING PREVALENCE; SPECIAL-EDUCATION; CALIFORNIA;
STATES
AB There has been little evidence to support the hypothesis that diagnostic substitution may contribute to increases in the administrative prevalence of autism. We examined trends in assignment of special education codes to British Columbia (BC) school children who had an autism code in at least 1 year between 1996 and 2004, inclusive. The proportion of children with an autism code increased from 12.3/10,000 in 1996 to 43.1/10,000 in 2004; 51.9% of this increase was attributable to children switching from another special education classification to autism (16.0/10,000). Taking into account the reverse situation (children with an autism code switching to another special education category (5.9/10.000)), diagnostic substitution accounted for at least one-third of the increase in autism prevalence over the study period.
C1 [Coo, Helen; Ouellette-Kuntz, Helene] Queens Univ, Ongwanada Resource Ctr, Dept Community Hlth & Epidemiol, Kingston, ON K7M 8A6, Canada.
[Ouellette-Kuntz, Helene; Holden, Jeanette J. A.] Queens Univ, Dept Psychiat, Kingston, ON K7M 8A6, Canada.
[Lloyd, Jennifer E. V.] Univ British Columbia, Fac Educ, Vancouver, BC V5Z 1M9, Canada.
[Kasmara, Liza; Lewis, M. E. Suzanne] Univ British Columbia, Dept Med Sci, Vancouver, BC V5Z 1M9, Canada.
[Holden, Jeanette J. A.] Queens Univ, Dept Physiol, Kingston, ON K7M 8A6, Canada.
[Lewis, M. E. Suzanne] Child & Family Res Inst, BC Childrens & Womens Hlth Ctr, Dept Med Genet, Vancouver, BC, Canada.
RP Ouellette-Kuntz, H (reprint author), Queens Univ, Ongwanada Resource Ctr, Dept Community Hlth & Epidemiol, 191 Portsmouth Ave, Kingston, ON K7M 8A6, Canada.
EM oullette@post.queensu.ca
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Blaxill MF, 2004, PUBLIC HEALTH REP, V119, P536, DOI 10.1016/j.phr.2004.09.003
BLAXILL MF, 2003, CALIFORNIA J AUTISM, V33, P4920
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NR 29
TC 38
Z9 39
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1036
EP 1046
DI 10.1007/s10803-007-0478-x
PG 11
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700005
PM 17975721
ER
PT J
AU Thorsen, KL
Goldberg, WA
Osann, K
Spence, MA
AF Thorsen, Kara L.
Goldberg, Wendy A.
Osann, Kathryn
Spence, M. Anne
TI Birthday and non-birthday videotapes: The importance of context for the
behavior of young children with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; videotapes; methodology; birthday; adaptive functioning; sensory
ID SPECTRUM DISORDER; HOME MOVIES; EARLY IDENTIFICATION; EARLY RECOGNITION;
INFANTS; AGE; LIFE; 1ST; DIAGNOSIS; SYMPTOMS
AB The present study examines whether children display different frequencies of behaviors at birthday party as compared to non-birthday party settings, and elucidates in which setting behavior is more predictive of later child functioning. Behavior in birthday and non-birthday contexts was examined at 12 and 24 months of age for 56 children with autism spectrum disorder (ASD). The results of this study indicate that context does matter for young children's behavior and leads to a different picture of behavioral functioning. For children with ASD, behaviors from non-birthday videotapes are more predictive of functioning later in childhood. The findings suggest that close attention must be paid to contextual factors that may influence young children's behavior.
C1 [Thorsen, Kara L.; Goldberg, Wendy A.] Univ Calif Irvine, Sch Social Ecol 2 3375, Dept Psychol & Social Behav, Irvine, CA 92697 USA.
[Osann, Kathryn] Univ Calif Irvine, Dept Med, Irvine, CA 92697 USA.
[Spence, M. Anne] Univ Calif Irvine, Dept Pediat, Irvine, CA 92697 USA.
RP Goldberg, WA (reprint author), Univ Calif Irvine, Sch Social Ecol 2 3375, Dept Psychol & Social Behav, Irvine, CA 92697 USA.
EM wagoldbe@uci.edu
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NR 34
TC 3
Z9 3
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1047
EP 1058
DI 10.1007/s10803-007-0479-9
PG 12
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700006
PM 17985221
ER
PT J
AU Mandell, DS
AF Mandell, David S.
TI Psychiatric hospitalization among children with autism spectrum
disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autistic disorder; hospitalization; health services
ID DEVELOPMENTAL-DISABILITIES; MULTISYSTEMIC THERAPY; CHALLENGING BEHAVIOR;
RESPITE CARE; PEOPLE; ADOLESCENTS; INPATIENT; DIAGNOSIS; COSTS;
INTERVENTIONS
AB This study examined predictors of psychiatric hospitalization among children with autism spectrum disorders (ASD). Data were collected from 760 caregivers of children with ASD. Cox regression was used to determine factors associated with hospitalization. Almost 11% were hospitalized. Youth in single parent homes were more likely to be hospitalized (OR = 2.54), as were youth diagnosed at a later age (OR = 1.10). Engaging in self-injurious behavior (OR = 2.14), aggressive behavior (OR = 4.83), and being diagnosed with depression (OR = 2.48) or obsessive compulsive disorder (OR = 2.35) increased the odds of hospitalization. Risk for hospitalization increased with age and over time. The results suggest early diagnosis and community-based interventions for aggressive and self-injurious behaviors may reduce hospitalizations.
C1 Univ Penn, Sch Med, Ctr Mental Hlth Policy & Serv Res, Philadelphia, PA 19104 USA.
RP Mandell, DS (reprint author), Univ Penn, Sch Med, Ctr Mental Hlth Policy & Serv Res, 3535 Market St,3rd Floor, Philadelphia, PA 19104 USA.
EM mandelld@mail.med.upenn.edu
RI Mandell, David/H-2730-2012
OI Mandell, David/0000-0001-8240-820X
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NR 45
TC 26
Z9 26
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1059
EP 1065
DI 10.1007/s10803-007-0481-2
PG 7
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700007
PM 17975720
ER
PT J
AU Robertson, MA
Sigalet, DL
Holst, JJ
Meddings, JB
Wood, J
Sharkey, KA
AF Robertson, Marli A.
Sigalet, David L.
Holst, Jens J.
Meddings, Jon B.
Wood, Julie
Sharkey, Keith A.
TI Intestinal permeability and glucagon-like peptide-2 in children with
autism: A controlled pilot study
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; nutrition; intestinal permeability; glucagon-like peptide 2
ID PERVASIVE DEVELOPMENTAL DISORDER; LYMPHOID-NODULAR HYPERPLASIA;
CROHNS-DISEASE; SPECTRUM DISORDERS; GASTROINTESTINAL SYMPTOMS;
CELIAC-DISEASE; GLP-2 LEVELS; PREVALENCE; ADAPTATION; LACTULOSE
AB We measured small intestinal permeability using a lactulose:mannitol sugar permeability test in a group of children with autism, with current or previous gastrointestinal complaints. Secondly, we examined whether children with autism had an abnormal glucagon-like peptide-2 (GLP-2) response to feeding. Results were compared with sibling controls and children without developmental disabilities. We enrolled 14 children with autism, 7 developmentally normal siblings of these children and 8 healthy, developmentally normal, unrelated children. Our study did not detect differences in these measures of gastrointestinal function in a group of children with autism.
C1 [Robertson, Marli A.; Wood, Julie] Alberta Childrens Prov Gen Hosp, Calgary, AB T3B 6A8, Canada.
[Sigalet, David L.; Sharkey, Keith A.] Univ Calgary, Inst Infect Immun & Inflammat, Calgary, AB, Canada.
[Sigalet, David L.] Univ Calgary, Dept Surg, Calgary, AB, Canada.
[Holst, Jens J.] Univ Copenhagen, Dept Med Physiol, Copenhagen, Denmark.
[Meddings, Jon B.] Univ Alberta, Dept Med, Edmonton, AB, Canada.
[Sharkey, Keith A.] Univ Calgary, Dept Phys & Biophys, Calgary, AB, Canada.
[Robertson, Marli A.] Univ Calgary, Dept Pediat, Calgary, AB T2N 1N4, Canada.
RP Robertson, MA (reprint author), Alberta Childrens Prov Gen Hosp, 2888 Shaganappi Trail NW, Calgary, AB T3B 6A8, Canada.
EM marli.robertson@calgaryhealthregion.ca
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NR 39
TC 19
Z9 19
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1066
EP 1071
DI 10.1007/s10803-007-0482-1
PG 6
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700008
PM 18311517
ER
PT J
AU Corden, B
Chilvers, R
Skuse, D
AF Corden, Ben
Chilvers, Rebecca
Skuse, David
TI Emotional modulation of perception in Asperger's syndrome
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE social cognition; amygdala; autism; fear; attentional blink; emotion
ID PERVASIVE DEVELOPMENTAL DISORDERS; AUTISM SPECTRUM DISORDERS;
HIGH-FUNCTIONING AUTISM; FUSIFORM FACE AREA; HUMAN AMYGDALA; DIAGNOSTIC
INTERVIEW; FACIAL EXPRESSIONS; ATTENTIONAL BLINK; IMPAIRED RECOGNITION;
FEAR RECOGNITION
AB Using an attentional blink paradigm, we show that the typical enhancement of perception for emotionally arousing events is significantly reduced in Asperger's syndrome (AS) at short inter-target intervals. Control experiments demonstrate that this finding cannot be attributed to differences in the perceived arousal of the stimuli, or to a global impairment affecting any type of modulation of perceptual encoding. Because a functioning amygdala is critical for emotional modulation of the attentional blink, the findings support a role for the amygdala in the pathophysiology of AS. More specifically, they suggest there is a fundamental failure of the amygdala to modulate processing in cortex, a concept at the heart of some recent theories of amygdala involvement in the aetiology of autistic-spectrum disorders.
C1 [Corden, Ben; Chilvers, Rebecca; Skuse, David] UCL, Inst Child Hlth, Behav & Brain Sci Unit, London, England.
RP Corden, B (reprint author), Univ Cambridge Wolfson Coll, Barton Rd, Cambridge CB3 9BB, England.
EM bc291@cam.ac.uk
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NR 51
TC 10
Z9 10
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1072
EP 1080
DI 10.1007/s10803-007-0485-y
PG 9
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700009
PM 17990090
ER
PT J
AU Moss, J
Magiati, I
Charman, T
Howlin, P
AF Moss, Jo
Magiati, Iliana
Charman, Tony
Howlin, Patricia
TI Stability of the autism diagnostic interview-revised from pre-school to
elementary school age in children with autism spectrum disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism diagnostic interview-revised; autism spectrum disorder; autism;
longitudinal study; symptom severity
ID FOLLOW-UP; ADOLESCENCE; SYMPTOMS; CHILDHOOD; ADULTS; TIME
AB This study examined the stability of scores on the ADI-R from pre-school to elementary school age in children with autism spectrum disorders (ASD). Participants were 35 children who, at T1, all had a clinical diagnosis of ASD. On initial assessment (mean age 3.5 years; SD 0.6 years), all met ADI-R algorithm criteria for autism. ADI-R assessments were repeated at follow up (FU; mean age 10.5 years; SD 0.8 years). Changes in ADI-R total, domain and ADI-R algorithm item scores were assessed. Twenty-eight children continued to score above the ADI-R cut-off for autism at FU, although significant decreases in ADI-R domain and item scores were also found. In conclusion while classification of children according to ADI-R criteria generally remained stable between pre-school and elementary school age, many children demonstrated significant improvements in symptom severity.
C1 [Moss, Jo; Howlin, Patricia] St Georges Univ London, Dept Community Hlth Sci, London, England.
[Charman, Tony] UCL, Inst Child Hlth, London, England.
RP Moss, J (reprint author), Kings Coll London, Inst Psychiat, Dept Psychol, Henry Wellcome Bldg,PO 77,De Crespigny Pk, London SE5 8AF, England.
EM jo.moss@iop.kcl.ac.uk
RI Howlin, Patricia/A-7622-2011; Moss, Jo/C-8812-2009; Charman,
Tony/A-2085-2014
OI Charman, Tony/0000-0003-1993-6549
CR Bayley N, 1993, BAYLEY SCALES INFANT
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NR 34
TC 20
Z9 20
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1081
EP 1091
DI 10.1007/s10803-007-0487-9
PG 11
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700010
PM 18058215
ER
PT J
AU Elder, LM
Dawson, G
Toth, K
Fein, D
Munson, J
AF Elder, Lauren M.
Dawson, Geraldine
Toth, Karen
Fein, Deborah
Munson, Jeff
TI Head circumference as an early predictor of autism symptoms in younger
siblings of children with autism spectrum disorder
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; head circumference; infant siblings
ID PERVASIVE DEVELOPMENTAL DISORDERS; EARLY INTERVENTION; EARLY
RECOGNITION; INFANTILE-AUTISM; BRAIN OVERGROWTH; HOME VIDEOTAPES;
TODDLERS; AGE; 1ST; DIAGNOSIS
AB Siblings of children with autism have an increased risk for autism spectrum disorders (ASD). As children with autism often exhibit an atypical trajectory of head circumference (HC) growth, HC may be an indicator of vulnerability to autism. This study investigated whether infant siblings of children with ASD (n = 77) with an atypical trajectory of HC growth were more likely than those without an atypical HC trajectory to develop autism symptoms. Results showed that infants who had larger HC at 12 months, and whose HC growth rate decelerated more rapidly between 12 and 24 months were more likely to exhibit autism symptoms than infants with more typical HC trajectories. Among infant siblings of children with autism, atypical HC growth might alert pediatricians to provide screening and/or referral for further evaluation.
C1 [Elder, Lauren M.; Dawson, Geraldine] Univ Washington, Dept Psychol, Autism Ctr, Seattle, WA 98195 USA.
[Toth, Karen] Childrens Hosp, Seattle, WA USA.
[Fein, Deborah] Univ Connecticut, Dept Psychol, Storrs, CT USA.
[Dawson, Geraldine; Munson, Jeff] Univ Washington, Dept Psychiat, Autism Ctr, Seattle, WA 98195 USA.
RP Dawson, G (reprint author), Univ Washington, Dept Psychol, Autism Ctr, Box 357920, Seattle, WA 98195 USA.
EM dawson@u.washington.edu
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 53
TC 41
Z9 42
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1104
EP 1111
DI 10.1007/s10803-007-0495-9
PG 8
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700012
PM 18058011
ER
PT J
AU Kaland, N
Callesen, K
Moller-Nielsen, A
Mortensen, EL
Smith, L
AF Kaland, Nils
Callesen, Kirsten
Moller-Nielsen, Annette
Mortensen, Erik Lykke
Smith, Lars
TI Performance of children and adolescents with Asperger syndrome or
high-functioning autism on advanced theory of mind tasks
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE Asperger syndrome; autism spectrum disorders; eyes Task; story tasks;
perceptual-affective; social-cognitive
ID THEORY-OF-MIND; PERVASIVE DEVELOPMENTAL DISORDERS; STRANGE STORIES TEST;
NONRETARDED-CHILDREN; SOCIAL-BEHAVIOR; FALSE BELIEF; ADULTS;
ATTRIBUTION; COGNITION; INDIVIDUALS
AB Although a number of advanced theory of mind tasks have been developed, there is a dearth of information on whether performances on different tasks are associated. The present study examined the performance of 21 children and adolescents with diagnoses of Asperger syndrome (AS) and 20 typically developing controls on three advanced theory of mind tasks: The Eyes Task, the Strange Stories, and the Stories from Everyday Life. The participants in the clinical group demonstrated lower performance than the controls on all the three tasks. The pattern of findings, however, indicates that these tasks may share different information-processing requirements in addition to tapping different mentalizing abilities.
C1 [Kaland, Nils] Lillehammer Univ Coll, Fac Social Sci, N-2604 Lillehammer, Norway.
[Callesen, Kirsten; Moller-Nielsen, Annette] Danish Univ Educ, Dept Psychol & Special Educ, Copenhagen NV, Denmark.
[Mortensen, Erik Lykke] Univ Copenhagen, Inst Publ Hlth, Dept Hlth Psychol, Copenhagen NV, Denmark.
[Smith, Lars] Univ Oslo, Natl Network Study Infant Mental Hlth Oslo, Oslo, Norway.
[Smith, Lars] Univ Oslo, Inst Psychol, Oslo, Norway.
RP Kaland, N (reprint author), Lillehammer Univ Coll, Fac Social Sci, Postboks 952, N-2604 Lillehammer, Norway.
EM nils@kaland.net
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NR 65
TC 40
Z9 42
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1112
EP 1123
DI 10.1007/s10803-007-0496-8
PG 12
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700013
PM 18058213
ER
PT J
AU Loftin, RL
Odom, SL
Lantz, JF
AF Loftin, Rachel L.
Odom, Samuel L.
Lantz, Johanna F.
TI Social interaction and repetitive motor behaviors
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; social skills; initiations; self-monitoring; repetitive motor
behavior; peer training; stereotypic behavior
ID SELF-STIMULATORY-BEHAVIOR; MANAGEMENT TREATMENT PACKAGE; STEREOTYPIC
BEHAVIOR; COMPONENT ANALYSIS; AUTISTIC-CHILDREN; SEVERE DISABILITIES;
TEACHING-CHILDREN; REINFORCEMENT; SKILLS; STUDENTS
AB Students with autism have difficulty initiating social interactions and may exhibit repetitive motor behavior (e.g., body rocking, hand flapping). Increasing social interaction by teaching new skills may lead to reductions in problem behavior, such as motor stereotypies. Additionally, self-monitoring strategies can increase the maintenance of skills. A multiple baseline design was used to examine whether multi-component social skills intervention (including peer training, social initiation instruction, and self-monitoring) led to a decrease in repetitive motor behavior. Social initiations for all participants increased when taught to initiate, and social interactions continued when self-monitoring was introduced. Additionally, participants' repetitive motor behavior was reduced. Changes in social behavior and in repetitive motor behavior maintained more than one month after the intervention ended.
C1 [Loftin, Rachel L.] Yale Univ, Sch Med, Yale Child Study Ctr, New Haven, CT USA.
[Odom, Samuel L.] Univ N Carolina, Frank Porter Graham Child Dev Inst, Chapel Hill, NC USA.
[Lantz, Johanna F.] Columbia Univ, Med Ctr, New York, NY USA.
RP Loftin, RL (reprint author), Univ Illinois, Inst Juvenile Res, 1747 W Roosevelt,M-C 747, Chicago, IL 60608 USA.
EM rloftin@uic.edu
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NR 56
TC 32
Z9 32
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1124
EP 1135
DI 10.1007/s10803-007-0499-5
PG 12
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700014
PM 18064552
ER
PT J
AU Goldberg, WA
Thorsen, KL
Osann, K
Spence, MA
AF Goldberg, Wendy A.
Thorsen, Kara L.
Osann, Kathryn
Spence, M. Anne
TI Use of home videotapes to confirm parental reports of regression in
autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; regression; language; videotapes; parent report; methodology
ID SPECTRUM DISORDER; LANGUAGE-DEVELOPMENT; PRESCHOOL-CHILDREN; EARLY
RECOGNITION; AGE; TEMPERAMENT; VALIDITY; 1ST; COMMUNICATION;
QUESTIONNAIRE
AB The current study examined consistency between parental reports on early language development and behaviors in non-language domains and observer-coded videotapes of young children with and without autism spectrum disorder (ASD) and autistic regression. Data are reported on 56 children (84% male) with ASD (early onset or autistic regression) and 14 typically developing children (57% male) who had home videotapes. Unique to the current study is the independent identification of loss/no loss for each child by both parental report and observer-coded home videotapes and the examination of agreement between these two methods. Results indicate substantial concordance between parental report and observer codes for onset and loss of expressive language, but minimal concordance for loss in non-language domains, suggesting a need for supplementation of parental reports in these areas.
C1 [Goldberg, Wendy A.; Thorsen, Kara L.] Univ Calif Irvine, Sch Social Ecol 2 3389, Dept Psychol & Social Behav, Irvine, CA 92697 USA.
[Osann, Kathryn] Univ Calif Irvine, Dept Med, Irvine, CA 92697 USA.
[Spence, M. Anne] Univ Calif Irvine, Dept Pediat, Irvine, CA 92697 USA.
RP Goldberg, WA (reprint author), Univ Calif Irvine, Sch Social Ecol 2 3389, Dept Psychol & Social Behav, Irvine, CA 92697 USA.
EM wendy.goldberg@uci.edu
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WERNER EB, 2001, BIANN M SOC RES CHIL
NR 42
TC 15
Z9 16
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1136
EP 1146
DI 10.1007/s10803-007-0498-6
PG 11
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700015
PM 18058010
ER
PT J
AU Lee, LC
Harrington, RA
Louie, BB
Newschaffer, CJ
AF Lee, Li-Ching
Harrington, Rebecca A.
Louie, Brian B.
Newschaffer, Craig J.
TI Children with autism: Quality of life and parental concerns
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; ADD; ADHD; quality of life; parental concern
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; PERVASIVE DEVELOPMENTAL
DISORDERS; INTELLECTUAL DISABILITIES; SPECTRUM DISORDERS; BEHAVIOR
PROBLEMS; YOUNG-CHILDREN; FAMILY STRESS; MENTAL-HEALTH; ADOLESCENTS;
CHILDHOOD
AB Past research has shown that children with autism and their families have compromised quality of life (QOL) in several domains. This study examined QOL and parental concerns in children with autism during early childhood, childhood, and adolescence compared to children with Attention Deficit Disorder/Attention Deficit Hyperactivity Disorder (ADD/ADHD) and to typical controls from a US national sample. Families with children diagnosed with autism reported more profound QOL effects than families of children with ADD/ADHD or unaffected controls. Children with autism were significantly less likely to attend religious services, more likely to miss school, and less likely to participate in organized activities. Parental concerns over learning difficulty, being bullied, stress-coping, and achievement were overwhelming in the autism group relative to the comparison groups.
C1 [Lee, Li-Ching; Harrington, Rebecca A.; Louie, Brian B.; Newschaffer, Craig J.] Johns Hopkins Bloomberg Sch Publ Hlth, Dept Epidemiol, Baltimore, MD 21205 USA.
RP Lee, LC (reprint author), Johns Hopkins Bloomberg Sch Publ Hlth, Dept Epidemiol, 615 N Wolfe St,Suite E6032, Baltimore, MD 21205 USA.
EM llee2@jhsph.edu
CR Allik H, 2006, HEALTH QUAL LIFE OUT, V4, DOI 10.1186/1477-7525-4-1
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Blumberg SJ, 2005, DESIGN OPERATION NAT
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Yirmiya N, 2005, J CHILD PSYCHOL PSYC, V46, P69, DOI 10.1111/j.1469-7610.2004.00334.x
NR 42
TC 51
Z9 53
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1147
EP 1160
DI 10.1007/s10803-007-0491-0
PG 14
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700016
PM 18058214
ER
PT J
AU Kaland, N
Smith, L
Mortensen, EL
AF Kaland, Nils
Smith, Lars
Mortensen, Erik Lykke
TI Brief report: Cognitive flexibility and focused attention in children
and adolescents with asperger syndrome or high-functioning autism as
measured on the computerized version of the Wisconsin Card Sorting Test
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE Asperger syndrome; high-functioning autism; executive function;
Wisconsin Card Sorting Test; attention problems
ID EXECUTIVE FUNCTION DEFICITS; DEVELOPMENTAL DISORDERS; MIND DEVELOPMENT;
INDIVIDUALS; PERFORMANCE; DYSFUNCTION
AB The aim of the present study was to assess mental flexibility and set maintenance of a group of individuals with Asperger syndrome (AS) or high-functioning autism (HFA) (N = 13; mean age 16,4), as compared with a matched group of typically developing children and adolescents (N = 13; mean age 15,6) on the computerized version of the Wisconsin Card Sorting Test (WCST). The participants in the AS/HFA group performed less well than the controls on all categories of the WCST, but the differences did not reach conventional statistical significance on most categories of the WCST. On the category failure to maintain set, however, the AS/HFA participants performed significantly less well than the controls, suggesting a deficit of focused attention.
C1 [Kaland, Nils] Lillehammer Univ Coll, Fac Social Sci, N-2624 Lillehammer, Norway.
[Smith, Lars] Univ Oslo, Inst Psychol, Oslo, Norway.
[Mortensen, Erik Lykke] Univ Copenhagen, Inst Publ Hlth, Dept Hlth Psychol, Copenhagen, Denmark.
RP Kaland, N (reprint author), Lillehammer Univ Coll, Fac Social Sci, Gudbrandsdalsvegen 350, N-2624 Lillehammer, Norway.
EM nils@kaland.net
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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LORD C, 1989, J AUTISM DEV DISORD, V19, P185, DOI 10.1007/BF02211841
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WHO, 1993, ICD 10 CLASS MENT BE
NR 32
TC 25
Z9 27
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1161
EP 1165
DI 10.1007/s10803-007-0474-1
PG 5
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700017
PM 17965928
ER
PT J
AU Overton, T
Fielding, C
de Alba, RG
AF Overton, Terry
Fielding, Cheryl
de Alba, Roman Garcia
TI Brief report: Exploratory analysis of the ADOS revised algorithm:
Specificity and predictive value with hispanic children referred for
autism spectrum disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; ADOS; algorithm; diagnostic specificity
AB This study compared Autism diagnostic observation schedule (ADOS) algorithm scores of a sample of 26 children who were administered modules 1-3 of the ADOS with the scores obtained applying the revised ADOS algorithm proposed by Gotham et al. (2007). Results of this application were inconsistent, yielding slightly more accurate results for module 1. New algorithm scores on modules 2 and 3 remained consistent with the original algorithm scores. The Mann-Whitney U was applied to compare revised algorithm and clinical levels of social impairment to determine if significant differences were evident. Results of Mann-Whitney U analyses were inconsistent and demonstrated less specificity for children with milder levels of social impairment. The revised algorithm demonstrated accuracy for the more severe autistic group.
C1 [Overton, Terry] Univ Texas Brownsville, Dept School Specialties, Brownsville, TX 78520 USA.
[Fielding, Cheryl] Univ Texas Pan Amer, Dept Educ Psychol, Edinburg, TX 78541 USA.
[de Alba, Roman Garcia] Brownsville ISD, Brownsville, TX USA.
RP Overton, T (reprint author), Univ Texas Brownsville, Dept School Specialties, 80 Ft Brown, Brownsville, TX 78520 USA.
EM overtont@mac.com
CR Corsello C., 2005, HDB AUTISM PERVASIVE, V2, P730
de Bildt A, 2004, J AUTISM DEV DISORD, V34, P129
GOTHAM G, 2007, J AUTISM DEV DISORD, V37, P613
Lord C, 2000, J AUTISM DEV DISORD, V30, P205, DOI 10.1023/A:1005592401947
Overton T, 2007, J AUTISM DEV DISORD, V37, P1996, DOI 10.1007/s10803-006-0349-x
*REG ON ED SERV CT, 2005, REG 1 PUBL ED INF MA
NR 6
TC 10
Z9 10
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1166
EP 1169
DI 10.1007/s10803-007-0488-8
PG 4
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700018
PM 18026872
ER
PT J
AU Moretti, P
Peters, SU
del Gaudio, D
Sahoo, T
Hyland, K
Bottiglieri, T
Hopkin, RJ
Peach, E
Min, SH
Goldman, D
Roa, B
Bacino, CA
Scaglia, F
AF Moretti, Paolo
Peters, Sarika U.
del Gaudio, Daniela
Sahoo, Trilochan
Hyland, Keith
Bottiglieri, Teodoro
Hopkin, Robert J.
Peach, Elizabeth
Min, Sang Hee
Goldman, David
Roa, Benjamin
Bacino, Carlos A.
Scaglia, Fernando
TI Brief report: Autistic symptoms, developmental regression, mental
retardation, epilepsy, and dyskinesias in CNS folate deficiency
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE folic Acid; 5-methyltetrahydrofolate; 5-MTHF; cerebral folate
deficiency; folate transporters; autism
ID FOLINIC ACID; CEREBROSPINAL-FLUID; OXIDATIVE STRESS; MALABSORPTION;
CHILDREN
AB We studied seven children with CNS folate deficiency (CFD). All cases exhibited psychomotor retardation, regression, cognitive delay, and dyskinesia; six had seizures; four demonstrated neurological abnormalities in the neonatal period. Two subjects had profound neurological abnormalities that precluded formal behavioral testing. Five subjects received ADOS and ADI-R testing and met diagnostic criteria for autism or autism spectrum disorders. They exhibited difficulties with transitions, insistence on sameness, unusual sensory interests, and repetitive behaviors. Those with the best language skills largely used repetitive phrases. No mutations were found in folate transporter or folate enzyme genes. These findings demonstrate that autistic features are salient in CFD and suggest that a subset of children with developmental regression, mental retardation, seizures, dyskinesia, and autism may have CNS folate abnormalities.
C1 [Moretti, Paolo; del Gaudio, Daniela; Sahoo, Trilochan; Bacino, Carlos A.; Scaglia, Fernando] Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
[Moretti, Paolo] Baylor Coll Med, Dept Neurol, Houston, TX 77030 USA.
[Peters, Sarika U.] Baylor Coll Med, Dept Pediat, Houston, TX 77030 USA.
[Hyland, Keith] Horizon Mol Med, Atlanta, GA USA.
[Bottiglieri, Teodoro] Baylor Univ, Med Ctr, Inst Metab Dis, Dallas, TX USA.
[Hopkin, Robert J.; Peach, Elizabeth] Cincinnati Childrens Hosp, Med Ctr, Div Human Genet, Cincinnati, OH USA.
[Min, Sang Hee; Goldman, David] Albert Einstein Coll Med, Dept Med, Bronx, NY 10467 USA.
[Min, Sang Hee; Goldman, David] Albert Einstein Coll Med, Dept Mol Pharmacol, Bronx, NY 10467 USA.
[Roa, Benjamin] Myriad Genet Labs, Salt Lake City, UT USA.
RP Scaglia, F (reprint author), Baylor Coll Med, Dept Mol & Human Genet, 1 Baylor Plaza, Houston, TX 77030 USA.
EM fscaglia@bcm.tmc.edu
CR Blau N, 2003, NEUROLOGY, V61, P642
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Hansen FJ, 2005, MOL GENET METAB, V84, P371, DOI 10.1016/j.ymgme.2004.12.001
HYLAND K, 1992, PTERIDINES, V3, P149
James SJ, 2004, AM J CLIN NUTR, V80, P1611
James SJ, 2006, AM J MED GENET B, V141B, P947, DOI 10.1002/ajmg.b.30366
Mercimek-Mahmutoglu S, 2007, TOHOKU J EXP MED, V211, P95, DOI 10.1620/tjem.211.95
Moretti P, 2005, NEUROLOGY, V64, P1088
Pineda M, 2006, ANN NEUROL, V59, P394, DOI 10.1002/ana.20746
Qiu AD, 2006, CELL, V127, P917, DOI 10.1016/j.cell.2006.09.041
Ramaekers VT, 2002, NEUROPEDIATRICS, V33, P301, DOI 10.1055/s-2002-37082
Ramaekers VT, 2004, DEV MED CHILD NEUROL, V46, P843, DOI 10.1017/S0012162204001471
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Ramaekers VT, 2003, NEUROLOGY, V61, P506
ROSENBLATT DS, 2000, METABOLIC MOL BASES
WEVERS RA, 1994, J NEUROL NEUROSUR PS, V57, P223, DOI 10.1136/jnnp.57.2.223
NR 16
TC 30
Z9 31
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1170
EP 1177
DI 10.1007/s10803-007-0492-z
PG 8
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700019
PM 18027081
ER
PT J
AU Hubel, M
Hagell, P
Sivberg, B
AF Hubel, Marie
Hagell, Peter
Sivberg, Bengt
TI Brief report: Development and initial testing of a questionnaire version
of the Environmental Rating Scale (ERS) for assessment of residential
programs for individuals with autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE autism; Environmental Rating Scale; assessment; validity; reliability
ID QUALITY; ADULTS
AB There is a lack of validated autism-specific outcome measures for large-scale evaluation of the effectiveness of psycho-educational programmes. To fill this gap the Environment Rating Scale (ERS) was adapted from an interview version to a staff-completed questionnaire version (ERS-Q). The ERS-Q was tested regarding data quality, validity, reliability and ease of understanding amongst 18 residential staff members. The ERS-Q and ERS showed comparable reliability (alpha = 0.89 and 0.93, respectively) and their correlation was 0.73. These observations support that the ERS interview can be adapted into a questionnaire without substantial loss of conceptual meaning. However, further evaluations in larger samples are needed to more firmly evaluate the measurement properties.
C1 [Hubel, Marie; Hagell, Peter; Sivberg, Bengt] Lund Univ, Fac Med, Dept Hlth Sci, S-22100 Lund, Sweden.
RP Hubel, M (reprint author), Lund Univ, Fac Med, Dept Hlth Sci, POB 157, S-22100 Lund, Sweden.
EM marie.hubel@med.lu.se
RI Hagell, Peter/F-5915-2010; Hagell, Peter/G-9559-2014
OI Hagell, Peter/0000-0003-2174-372X; Hagell, Peter/0000-0003-2174-372X
CR BLAND JM, 1995, BRIT MED J, V310, P170
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Mesibov G. B., 1997, AUTISM, V1, P25, DOI 10.1177/1362361397011005
PERSSON B, 2004, FOCUS AUTISM RES, P1
Schopler E., 1995, LEARNING COGNITION A, P2243
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Streiner D, 1995, HLTH MEASUREMENT SCA
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NR 14
TC 1
Z9 1
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1178
EP 1183
DI 10.1007/s10803-007-0493-y
PG 6
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700020
PM 17987373
ER
PT J
AU Webster, S
Potter, DD
AF Webster, Simon
Potter, Douglas D.
TI Brief report: Eye direction detection improves with development in
autism
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE gaze perception; development; autism
ID JOINT ATTENTION
AB Eye direction detection has been claimed to be intact in autism, but the development of this skill has not been investigated. Eleven children with autism and 11 typically developing children performed a demanding face-to-face eye direction detection task. Younger children with autism demonstrated a deficit in this skill, relative to younger control participants. Older children with autism were as accurate as older control participants on this task. In autism, eye direction detection is deficient in late childhood but is typically accurate by adolescence. The implications of this finding for models of social cognitive development in autism are considered.
C1 [Webster, Simon; Potter, Douglas D.] Univ Dundee, Sch Psychol, Dundee DD1 4HN, Scotland.
RP Potter, DD (reprint author), Univ Dundee, Sch Psychol, Dundee DD1 4HN, Scotland.
EM d.d.potter@dundee.ac.uk
CR BARONCOHEN S, 1989, BRIT J DEV PSYCHOL, V7, P113
Baron-Cohen Simon, 1995, MINDBLINDNESS ESSAY
Berument SK, 1999, BRIT J PSYCHIAT, V175, P444, DOI 10.1192/bjp.175.5.444
Howard MA, 2000, NEUROREPORT, V11, P2931, DOI 10.1097/00001756-200009110-00020
Leekam S, 1997, BRIT J DEV PSYCHOL, V15, P77
Morales M, 2000, J APPL DEV PSYCHOL, V21, P283, DOI 10.1016/S0193-3973(99)00040-4
SWETTENHAM J, 2001, CHILDREN AUTISM SHOW
WEBSTER S, 2004, THESIS U DUNDEE
NR 8
TC 11
Z9 11
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1184
EP 1186
DI 10.1007/s10803-008-0539-9
PG 3
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700021
PM 18324465
ER
PT J
AU Begeer, S
Banerjee, R
Lunenburg, P
Terwogt, MM
Stegge, H
Rieffe, C
AF Begeer, Sander
Banerjee, Robin
Lunenburg, Patty
Terwogt, Mark Meerum
Stegge, Hedy
Rieffe, Carolien
TI Brief report: Self-presentation of children with autism spectrum
disorders
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Article
DE self-presentation; theory of mind; autism; high functioning
ID MIND; EMBARRASSMENT; INDIVIDUALS; ABILITIES; EMOTION
AB The self-presentational behaviour of 43 6- to 12-year-old children with high functioning autism spectrum disorders (HFASD) and normal intelligence and 43 matched comparisons was investigated. Children were prompted to describe themselves twice, first in a baseline condition and then in a condition where they were asked to convince others to select them for a desirable activity (self-promotion). Even after controlling for theory of mind skills, children with HFASD used fewer positive self-statements at baseline, and were less goal-directed during self-promotion than comparison children. Children with HFASD alter their self-presentation when seeking personal gain, but do this less strategically and convincingly than typically-developing children.
C1 [Begeer, Sander; Lunenburg, Patty; Terwogt, Mark Meerum; Stegge, Hedy] Vrije Univ Amsterdam, Dept Dev Psychol, NL-1081 BT Amsterdam, Netherlands.
[Banerjee, Robin] Univ Sussex, Falmer, England.
[Rieffe, Carolien] Leiden Univ, Leiden, Netherlands.
RP Begeer, S (reprint author), Vrije Univ Amsterdam, Dept Dev Psychol, Van der Boechorststr 1, NL-1081 BT Amsterdam, Netherlands.
EM S.Begeer@psy.vu.nl
RI Begeer, Sander/I-3383-2012
CR ALOISEYOUNG PA, 1993, SOC COGNITION, V11, P201, DOI 10.1521/soco.1993.11.2.201
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 18
TC 6
Z9 7
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1187
EP 1191
DI 10.1007/s10803-007-0503-0
PG 5
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700022
PM 18311516
ER
PT J
AU Scahill, L
AF Scahill, Lawrence
TI How do I decide whether or not to use medication for my child with
autism? Should I try behavior therapy first?
SO JOURNAL OF AUTISM AND DEVELOPMENTAL DISORDERS
LA English
DT Letter
ID RISPERIDONE
C1 Yale Univ, Ctr Child Study, New Haven, CT 06520 USA.
RP Scahill, L (reprint author), Yale Univ, Ctr Child Study, 230 S Frontage Rd,POB 207900, New Haven, CT 06520 USA.
EM lawrence.scahill@yale.edu
CR Mccracken JT, 2005, AM J PSYCHIAT, V162, P1361
McCracken JT, 2002, NEW ENGL J MED, V347, P314, DOI 10.1056/NEJMoa013171
Tyrer P, 2008, LANCET, V371, P57, DOI 10.1016/S0140-6736(08)60072-0
NR 3
TC 3
Z9 3
PU SPRINGER/PLENUM PUBLISHERS
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 0162-3257
J9 J AUTISM DEV DISORD
JI J. Autism Dev. Disord.
PD JUL
PY 2008
VL 38
IS 6
BP 1197
EP 1198
DI 10.1007/s10803-008-0573-7
PG 2
WC Psychology, Developmental
SC Psychology
GA 310II
UT WOS:000256521700024
PM 18463973
ER
PT J
AU Brumback, RA
AF Brumback, Roger A.
TI A never ending saga: The mercury and autism articles by Ip et al
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Editorial Material
ID EXPOSURE; CHILDREN
RI Brumback, Roger/A-2404-2008
CR Brumback RA, 2007, J CHILD NEUROL, V22, P1321, DOI 10.1177/0883073807308149
DeSoto MC, 2008, J CHILD NEUROL, V23, P463, DOI 10.1177/0883073808314718
Desoto M Catherine, 2007, J Child Neurol, V22, P1308, DOI 10.1177/0883073807307111
IP P, 2004, HK J PAEDIAT NEW SER, V9, P103
Ip P, 2007, J CHILD NEUROL, V22, P1324, DOI 10.1177/0883073807308148
Ip P, 2004, PEDIATR INT, V46, P715, DOI 10.1111/j.1442-200x.2004.01972.x
Ip P, 2004, J CHILD NEUROL, V19, P431
NR 7
TC 3
Z9 3
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD JUL
PY 2008
VL 23
IS 7
BP 725
EP 725
DI 10.1177/0883073808320530
PG 1
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 330GP
UT WOS:000257929000001
PM 18658071
ER
PT J
AU Gabis, L
Huang, W
Azizian, A
DeVincent, C
Tudorica, A
Kesner-Baruch, Y
Roche, P
Pomeroy, J
AF Gabis, Lidia
Huang, Wei
Azizian, Allen
DeVincent, Carla
Tudorica, Alina
Kesner-Baruch, Yael
Roche, Patricia
Pomeroy, John
TI H-1-magnetic resonance spectroscopy markers of cognitive and language
ability in clinical subtypes of autism spectrum disorders
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Article
DE language; brain imaging; H-1-magnetic resonance spectroscopy; autism
spectrum disorders
ID ASPERGER-SYNDROME; BRAIN; MRI; HIPPOCAMPUS; CEREBELLUM; ANOMALIES;
AMYGDALA
AB This study assessed metabolic functioning of regional brain areas to address whether there is a neurometabolic profile reflecting the underlying neuropathology in individuals with autism spectrum disorders, and if varied profiles correlate with the clinical subtypes. Thirteen children (7-16 years) with autism spectrum disorders and 8 typically developing children were compared on H-1-magnetic resonance spectroscopy data collected from hippocampus-amygdala and cerebellar regions. The autism spectrum disorder group had significantly lower N-acetyl-aspartate/creatine ratios bilaterally in the hippocampus-amygdala but not cerebellum, whereas myo-inositol/creatine was significantly increased in all measured regions. Choline/creatine was also significantly elevated in the left hippocampus-amygdala and cerebellar regions of children with autism spectrum disorder. Comparisons within the autism spectrum disorder group when clinically subdivided by history of speech delay revealed significant metabolic ratio differences. Magnetic resonance spectroscopy can provide important information regarding abnormal brain metabolism and clinical classification in autism spectrum disorders.
C1 [Gabis, Lidia; Kesner-Baruch, Yael] Safra Childrens Hosp, Weinberg Child Dev Ctr, IL-52621 Tel Hashomer, Israel.
[Gabis, Lidia; Kesner-Baruch, Yael] Tel Aviv Univ, Sackler Sch Med, IL-69978 Tel Aviv, Israel.
[Huang, Wei] Mem Sloan Kettering Canc Ctr, Dept Med Phys, New York, NY 10021 USA.
[Azizian, Allen] Univ Calif Los Angeles, Neuropsychiat Inst & Hosp, Los Angeles, CA 90024 USA.
[DeVincent, Carla; Pomeroy, John] SUNY Stony Brook, Dept Pediat, Stony Brook, NY 11794 USA.
[Tudorica, Alina; Roche, Patricia] SUNY Stony Brook, Dept Radiol, Stony Brook, NY 11794 USA.
RP Gabis, L (reprint author), Safra Childrens Hosp, Weinberg Child Dev Ctr, IL-52621 Tel Hashomer, Israel.
EM gabis@post.tau.ac.il
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 33
TC 26
Z9 26
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD JUL
PY 2008
VL 23
IS 7
BP 766
EP 774
DI 10.1177/0883073808315423
PG 9
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 330GP
UT WOS:000257929000010
PM 18487520
ER
PT J
AU Galasso, C
Lo-Castro, A
Lalli, C
Nardone, AM
Gullotta, F
Curatolo, P
AF Galasso, Cinzia
Lo-Castro, Adriana
Lalli, Cristina
Nardone, Anna Maria
Gullotta, Francesca
Curatolo, Paolo
TI Deletion 2q37: An identifiable clinical syndrome with mental retardation
and autism
SO JOURNAL OF CHILD NEUROLOGY
LA English
DT Article
DE chromosome 2q syndrome; 2q deletion syndrome; monosomy 2q; mental
retardation; autism
ID ALBRIGHT HEREDITARY OSTEODYSTROPHY; TERMINAL DELETION; LONG ARM;
CANDIDATE REGION; NEUROMEDIN-U; CHROMOSOME-2; PHENOTYPE; GENE;
46,XY,DEL(2)(Q37); TRANSLOCATION
AB Terminal deletion of the long arm of chromosome 2 is a rare chromosomal disorder characterized by low birth weight, delayed somatic and mental development, craniofacial defects, short neck, heart and lung congenital defects, and autistic features. We report on a girl with 46,XX.ish del(2)(q37.1) de novo karyotype, mental retardation, dysmorphic features, gastrointestinal anomalies, and autistic traits and compare her clinical manifestations with patients with the same deletion previously described in literature.
C1 [Galasso, Cinzia; Lo-Castro, Adriana; Lalli, Cristina; Curatolo, Paolo] Univ Roma Tor Vergata, Pediat Neurol Unit, Dept Neurosci, I-00133 Rome, Italy.
[Nardone, Anna Maria; Gullotta, Francesca] Univ Roma Tor Vergata, Dept Med Genet, I-00133 Rome, Italy.
RP Galasso, C (reprint author), Univ Roma Tor Vergata, Pediat Neurol Unit, Dept Neurosci, Via Montpellier 1, I-00133 Rome, Italy.
EM cinzia.galasso@uniroma2.it
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NR 31
TC 15
Z9 19
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 0883-0738
J9 J CHILD NEUROL
JI J. Child Neurol.
PD JUL
PY 2008
VL 23
IS 7
BP 802
EP 806
DI 10.1177/0883073808314150
PG 5
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 330GP
UT WOS:000257929000014
PM 18658079
ER
PT J
AU Beaumont, R
Sofronoff, K
AF Beaumont, Renae
Sofronoff, Kate
TI A multi-component social skills intervention for children with Asperger
syndrome: The junior detective training program
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Article
DE Asperger syndrome; social skills
ID HIGH-FUNCTIONING AUTISM; RANDOMIZED CONTROLLED-TRIAL; TEACHING THEORY;
EMOTIONS; MIND; RECOGNIZE; ADULTS; BELIEF
AB Background: The study aimed to investigate the effectiveness of a new multi-component social skills intervention for children with Asperger syndrome (AS): The Junior Detective Training Program. This 7-week program included a computer game, small group sessions, parent training sessions and teacher handouts. Method: Forty-nine children with AS were recruited to participate and randomly assigned to intervention (n = 26) or wait-list control (n = 23) conditions. Results: Relative to children in the wait-list group, program participants showed greater improvements in social skills over the course of the intervention, as indicated by parent-report measures. Teacher-report data also confirmed that children receiving the intervention made significant improvements in social functioning from pre- to post-treatment. Treatment group participants were better able to suggest appropriate emotion-management strategies for story characters at post-intervention than at pre-intervention, whereas control participants were not. However, there was no difference in the improvements made by children in the intervention and control conditions on facial expression and body-posture recognition measures. Follow-up data suggested that treatment gains were maintained by children at 5-months post-intervention. Conclusions: The Junior Detective Training Program appeared to be effective in enhancing the social skills and emotional understanding of children with AS. Limitations and suggestions for future research are discussed.
C1 [Sofronoff, Kate] Univ Queensland, Sch Psychol, Brisbane, Qld, Australia.
RP Sofronoff, K (reprint author), Univ Queensland, Sch Psychol, Brisbane, Qld, Australia.
EM kate@psy.uq.edu.au
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NR 25
TC 50
Z9 51
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD JUL
PY 2008
VL 49
IS 7
BP 743
EP 753
DI 10.1111/j.1469-7610.2008.01920.x
PG 11
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 315CH
UT WOS:000256855000008
PM 18503531
ER
PT J
AU Berger, M
AF Berger, Michael
TI Understanding autism: From basic neuroscience to treatment
SO JOURNAL OF CHILD PSYCHOLOGY AND PSYCHIATRY
LA English
DT Book Review
CR Moldin S. O., 2006, UNDERSTANDING AUTISM
NR 1
TC 0
Z9 1
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0021-9630
J9 J CHILD PSYCHOL PSYC
JI J. Child Psychol. Psychiatry
PD JUL
PY 2008
VL 49
IS 7
BP 792
EP 792
DI 10.1111/j.1469-7610.2007.01767.x
PG 1
WC Psychology, Developmental; Psychiatry; Psychology
SC Psychology; Psychiatry
GA 315CH
UT WOS:000256855000013
ER
PT J
AU Whitehouse, AJO
Barry, JG
Bishop, DVM
AF Whitehouse, Andrew J. O.
Barry, Johanna G.
Bishop, Dorothy V. M.
TI Further defining the language impairment of autism: Is there a specific
language impairment subtype?
SO JOURNAL OF COMMUNICATION DISORDERS
LA English
DT Article
ID SUSCEPTIBILITY GENE; DEFICITS; CHILDREN; TWIN; PHENOTYPES; SIBLINGS;
SPECTRUM; DISORDER; FOXP2; RISK
AB Some children with autism demonstrate poor nonword repetition-a deficit considered to be a psycholinguistic marker of specific language impairment (SLI). The present study examined whether there is an SLI subtype among children with autism. We compared the language abilities of children with SLI (n = 34, M age = 11; 10 S.D. = 2;3), and children with autism with (Apoor, n = 18, M age = 10; 11 S.D. = 3; 1) and without (Aapp, n = 16, M age = 10;8 S.D. = 2;7) structural language difficulties. Participants were administered battery of standardized language and memory tests. Although there were some similarities in the language profile of the SLI and Apoor groups, the two groups differed on the tests of oromotor ability and verbal short-term memory and showed a different pattern of errors on the nonword repetition task. These findings providing evidence against the idea of an SLI subtype in autism. Further analyses suggested that the nonword repetition deficits experienced by some children with autism may arise when there is substantial impairment in multiple autistic domains.
Learning outcomes: Readers will be introduced to (a) the current state of behavioral, cognitive and genetic research that has investigated the relation between SLI and autism, and (b) three hypotheses of why there exists similarity in the language characteristics of children with SLI and autism. Readers will then be taken through a detailed comparison of the language and memory abilities of group of children with each diagnosis. A theoretical model that seeks to explain the relation between these two disorders will be discussed. (C) 2008 Elsevier Inc. All rights reserved.
C1 [Whitehouse, Andrew J. O.; Barry, Johanna G.; Bishop, Dorothy V. M.] Univ Oxford, Dept Expt Psychol, Oxford Study Childrens Commun Impairments, Oxford OX1 3UD, England.
RP Whitehouse, AJO (reprint author), Univ Oxford, Dept Expt Psychol, Oxford Study Childrens Commun Impairments, S Parks Rd, Oxford OX1 3UD, England.
EM andrew.whitehouse@psy.ox.ac.uk
CR Alarcon M, 2002, AM J HUM GENET, V70, P60, DOI 10.1086/338241
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Whitehouse AJO, 2007, J CHILD PSYCHOL PSYC, V48, P822, DOI 10.1111/j.1469-7610.2007.01765.x
NR 37
TC 47
Z9 47
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0021-9924
J9 J COMMUN DISORD
JI J. Commun. Disord.
PD JUL-AUG
PY 2008
VL 41
IS 4
BP 319
EP 336
DI 10.1016/j.jcomdis.2008.01.002
PG 18
WC Audiology & Speech-Language Pathology; Linguistics; Rehabilitation
SC Audiology & Speech-Language Pathology; Linguistics; Rehabilitation
GA 304MZ
UT WOS:000256114900002
PM 18295779
ER
PT J
AU Gargus, JJ
AF Gargus, J. Jay
TI Calcium signaling abnormalities in familial hemiplegic migraine type 2
(FHM2), a window into autism and polygenic neuropsychiatric
channelopathies
SO JOURNAL OF GENERAL PHYSIOLOGY
LA English
DT Meeting Abstract
CT 62nd Annual Meeting of the Society-of-General-Physiologists
CY SEP 03-07, 2008
CL Woods Hole, MA
SP Soc Gen Physiol
HO Marine Biol Lab
C1 [Gargus, J. Jay] Univ Calif Irvine, Dept Physiol & Biophys, Orange, CA 92868 USA.
[Gargus, J. Jay] Univ Calif Irvine, Dept Pediat, Div Human Genet, Orange, CA 92868 USA.
NR 0
TC 2
Z9 2
PU ROCKEFELLER UNIV PRESS
PI NEW YORK
PA 1114 FIRST AVE, 4TH FL, NEW YORK, NY 10021 USA
SN 0022-1295
J9 J GEN PHYSIOL
JI J. Gen. Physiol.
PD JUL
PY 2008
VL 132
IS 1
MA 42
BP 19A
EP 20A
PG 2
WC Physiology
SC Physiology
GA 354JC
UT WOS:000259634900046
ER
PT J
AU Xu, Y
Xue, Y
Asan
Daly, A
Wu, LJ
Tyler-Smith, C
AF Xu, Yang
Xue, Yali
Asan
Daly, Allan
Wu, Lijie
Tyler-Smith, Chris
TI Variation of the oxytocin/neurophysin I (OXT) gene in four human
populations
SO JOURNAL OF HUMAN GENETICS
LA English
DT Article
DE oxytocin; OXT; human genetic variation; HapMap populations; HKA test
ID HUMAN GENOME; STATISTICAL-METHOD; DNA POLYMORPHISM; OXYTOCIN; AUTISM
AB Oxytocin is a short peptide with multiple functions in human biology and has been implicated in autism. We aimed to determine the normal pattern of variation around the oxytocin gene and resequenced it and its flanking regions in 91 individuals from four HapMap populations and one chimpanzee. We identified 14 single nucleotide polymorphisms (SNPs), all noncoding, including eight that were novel. Population genetic analyses were largely consistent with a neutral evolutionary history, but an Hudson-Kreitman-Aguade (HKA) test revealed more variation within the human population than expected from the level of chimpanzee-human divergence.
C1 [Xu, Yang; Wu, Lijie] Harbin Med Coll, Dept Childrens & Adolescent Hlth, Coll Publ Hlth, Harbin 150086, Peoples R China.
[Xu, Yang; Xue, Yali; Asan; Daly, Allan; Tyler-Smith, Chris] Wellcome Trust Sanger Inst, Hinxton CB10 1SA, Cambs, England.
RP Wu, LJ (reprint author), Harbin Med Coll, Dept Childrens & Adolescent Hlth, Coll Publ Hlth, 157 Baojian Rd, Harbin 150086, Peoples R China.
EM wljhyd@ems.hrbmu.edu.cn; cts@sanger.ac.uk
CR Akey JM, 2004, PLOS BIOL, V2, P1591, DOI 10.1371/journal.pbio.0020286
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Carter CS, 2007, BEHAV BRAIN RES, V176, P170, DOI 10.1016/j.bbr.2006.08.025
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NR 21
TC 2
Z9 2
PU NATURE PUBLISHING GROUP
PI NEW YORK
PA 75 VARICK ST, 9TH FLR, NEW YORK, NY 10013-1917 USA
SN 1434-5161
J9 J HUM GENET
JI J. Hum. Genet.
PD JUL
PY 2008
VL 53
IS 7
BP 637
EP 643
DI 10.1007/s10038-008-0292-0
PG 7
WC Genetics & Heredity
SC Genetics & Heredity
GA 320BH
UT WOS:000257208200008
PM 18566739
ER
PT J
AU Sadakata, T
Furuichi, T
AF Sadakata, T.
Furuichi, T.
TI Analysis of autism-related gene CAPS2/CADPS2
SO JOURNAL OF NEUROCHEMISTRY
LA English
DT Meeting Abstract
CT 8th Biennial Meeting of the Asia-Pacific-Society-of-Neurochemistry
CY JUN 23-26, 2008
CL Shanghai, PEOPLES R CHINA
SP Asia Pacific Soc Neurochem
C1 [Sadakata, T.; Furuichi, T.] RIKEN, Brain Sci Inst, Lab Mol Neurogenesis, Saitama, Japan.
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0022-3042
J9 J NEUROCHEM
JI J. Neurochem.
PD JUL
PY 2008
VL 106
SU 1
BP 31
EP 31
PG 1
WC Biochemistry & Molecular Biology; Neurosciences
SC Biochemistry & Molecular Biology; Neurosciences & Neurology
GA 307WY
UT WOS:000256351500077
ER
PT J
AU Chauhan, A
Essa, MM
Muthaiyah, B
Brown, WT
Chauhan, V
AF Chauhan, A.
Essa, M. M.
Muthaiyah, B.
Brown, W. T.
Chauhan, V
TI Increased oxidative damage and free radical generation in lymphoblasts
from autism
SO JOURNAL OF NEUROCHEMISTRY
LA English
DT Meeting Abstract
CT 8th Biennial Meeting of the Asia-Pacific-Society-of-Neurochemistry
CY JUN 23-26, 2008
CL Shanghai, PEOPLES R CHINA
SP Asia Pacific Soc Neurochem
C1 [Chauhan, A.; Essa, M. M.; Muthaiyah, B.; Brown, W. T.; Chauhan, V] NYS Inst Basic Res Dev Disabil, New York, NY USA.
NR 0
TC 0
Z9 0
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0022-3042
J9 J NEUROCHEM
JI J. Neurochem.
PD JUL
PY 2008
VL 106
SU 1
BP 44
EP 44
PG 1
WC Biochemistry & Molecular Biology; Neurosciences
SC Biochemistry & Molecular Biology; Neurosciences & Neurology
GA 307WY
UT WOS:000256351500104
ER
PT J
AU Vasconcelos, MM
Brito, AR
Domingues, RC
da Cruz, LCH
Gasparetto, EL
Werner, J
Goncalves, JPS
AF Vasconcelos, Marcio Moacyr
Brito, Adriana Rocha
Domingues, Romeu Cortes
Hygino da Cruz, Luiz Celso, Jr.
Gasparetto, Emerson L.
Werner, Jairo
Sevalho Goncalves, Joao Pedro
TI Proton magnetic resonance spectroscopy in school-aged autistic children
SO JOURNAL OF NEUROIMAGING
LA English
DT Article
DE magnetic resonance spectroscopy; autistic disorder; diagnostic imaging;
brain chemistry
ID CEREBRAL WHITE-MATTER; BRAIN-METABOLITES; YOUNG-CHILDREN; DISORDER; BOYS
AB PURPOSE
This study aims to assess cerebral metabolites in school-aged autistic patients through proton magnetic resonance spectroscopy.
METHODS
This case-control study included 10 right-handed mate children (median age, 9.53 years +/- 1.80) with autism according to DSM-IV criteria, and 10 healthy age- and sex-matched healthy controls (median age, 8.52 years +/- 1.42). Imaging was performed on a 1.5-T scanner utilizing a single voxel point-resolved spectroscopy (PRESS) technique (TR = 1,500 ms, TE = 30 ms). Four cerebral areas were evaluated: bilateral anterior cingulate, left striatum, left cerebellar hemisphere, and left frontal lobe. Peak areas and ratios to creatine (Cr) of N-acetylaspartate (NAA), choline (Cho), and myo-inositol (mI) were analyzed.
RESULTS
Compared with controls, autistic children showed a significant increase in ml (P = .021) and Cho (P = .042) peak areas in anterior cingulate and in mI/Cr ratio in anterior cingulate (P = .037) and left striatum (P = .035). The remaining metabolites and ratios were not significantly different between the 2 groups.
CONCLUSIONS
This study found a statistically significant increase in myo-inositol and choline in anterior cingulate and left striatum of autistic children compared with controls. In contrast to previous studies, NAA peak area and NAA/Cr and NAA/Cho ratios had, no statistically significant decrease in any of the 4brain regions.
C1 [Vasconcelos, Marcio Moacyr; Brito, Adriana Rocha; Werner, Jairo] Univ Fed Fluminense, Hosp Univ Antonio Pedro, Dept Materno Infantil, Rio De Janeiro, Brazil.
[Domingues, Romeu Cortes; Hygino da Cruz, Luiz Celso, Jr.; Sevalho Goncalves, Joao Pedro] Clin Multi Imagem, Rio De Janeiro, Brazil.
[Gasparetto, Emerson L.] Univ Fed Rio de Janeiro, Dept Radiol, Clin CDPI, Rio De Janeiro, Brazil.
RP Vasconcelos, MM (reprint author), Av Amer,700 Sala 229 Bloco 6, BR-22640100 Rio De Janeiro, Brazil.
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, P66
BRANDAO LA, 2003, MR SPECTROSCOPY BRAI, P2
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Levitt JG, 2003, BIOL PSYCHIAT, V54, P1355, DOI 10.1016/S0006-3223(03)00688-7
McDougle CJ, 2005, J CLIN PSYCHIAT, V66, P9
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van der Knaap MS, 2005, MAGNETIC RESONANCE M, P859
Zeegers M, 2007, J NEURAL TRANSM, V114, P289, DOI 10.1007/s00702-006-0501-y
NR 22
TC 12
Z9 12
PU BLACKWELL PUBLISHING
PI OXFORD
PA 9600 GARSINGTON RD, OXFORD OX4 2DQ, OXON, ENGLAND
SN 1051-2284
J9 J NEUROIMAGING
JI J. Neuroimaging
PD JUL
PY 2008
VL 18
IS 3
BP 288
EP 295
DI 10.1111/j.1552-6569.2007.00200.x
PG 8
WC Clinical Neurology; Neuroimaging; Radiology, Nuclear Medicine & Medical
Imaging
SC Neurosciences & Neurology; Radiology, Nuclear Medicine & Medical Imaging
GA 331BY
UT WOS:000257988900011
PM 18304036
ER
PT J
AU Sansosti, FJ
Powell-Smith, KA
AF Sansosti, Frank J.
Powell-Smith, Kelly A.
TI Using Computer-Presented Social Stories and Video Models to Increase the
Social Communication Skills of Children With High-Functioning Autism
Spectrum Disorders
SO JOURNAL OF POSITIVE BEHAVIOR INTERVENTIONS
LA English
DT Article
DE autism; Asperger Syndrome; Social Stories; video modeling; social skills
ID PERVASIVE DEVELOPMENTAL DISORDERS; TEACH PERSPECTIVE-TAKING;
ASPERGER-SYNDROME; ASSISTED-INSTRUCTION; INTERVENTION; BEHAVIORS;
FEEDBACK; PLAY
AB The purpose of this study was to investigate the effects of computer-presented Social Stories and video models on the social communication skills of three children with High-Functioning Autism/Asperger's Syndrome (HFA/AS). Using a multiple-baseline across-participants design, computer-presented Social Stories and video models were implemented and direct observations of the participants' identified target behaviors were collected two times per week during unstructured school activities (e.g., recess). Overall, data demonstrated that the combined treatment package was effective for improving the rates of social communication for the participants, although modifications to allow access to social reinforcement were needed in two cases. In addition, all three participants demonstrated maintenance of skills at a 2-week follow-up. However, generalization of skills was only observed for one participant. This research adds evidence that a combined intervention presented via computer may be a beneficial method for remediating social skill difficulties for individuals with HFA/AS.
C1 [Sansosti, Frank J.] Kent State Univ, Educ Fdn & Special Serv, Kent, OH 44242 USA.
[Powell-Smith, Kelly A.] Dynam Measurement Grp, Eugene, OR USA.
RP Sansosti, FJ (reprint author), Kent State Univ, Educ Fdn & Special Serv, 405 White Hall, Kent, OH 44242 USA.
EM fsansost@kent.edu
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NR 67
TC 48
Z9 48
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1098-3007
J9 J POSIT BEHAV INTERV
JI J. Posit. Behav. Interv.
PD JUL
PY 2008
VL 10
IS 3
BP 162
EP 178
DI 10.1177/1098300708316259
PG 17
WC Psychology, Clinical; Education, Special
SC Psychology; Education & Educational Research
GA 390MS
UT WOS:000262169000004
ER
PT J
AU Kraemer, BR
Cook, CR
Browning-Wright, D
Mayer, GR
Wallace, MD
AF Kraemer, Bonnie R.
Cook, Clayton R.
Browning-Wright, Diana
Mayer, G. Roy
Wallace, Michele D.
TI Effects of Training on the Use of the Behavior Support Plan Quality
Evaluation Guide With Autism Educators
SO JOURNAL OF POSITIVE BEHAVIOR INTERVENTIONS
LA English
DT Article
DE autism; PDD; functional assessment; behavior(s); training
ID FUNCTIONAL-ANALYSIS METHODOLOGY; YOUNG-CHILDREN; ACQUISITION;
STRATEGIES; PROVISIONS; TEACHERS; STUDENTS; BURNOUT
AB Positive behavior support (PBS) plans are required practice for students whose behaviors impede their learning or that of others. Educators of children and youth with autism and other developmental disorders represent a subgroup of special educators who are frequently involved in the development of PBS plans. The goal of this research was to assess the effect of a specific, brief training delivered to improve the substantive, evidence-based quality of PBS plans developed by autism educators in a graduate-level university program. Intra-individual tests of significance revealed that the training significantly improved the quality of PBS plans. The plan components with the highest ratings were predictors of problem behavior and behavioral definition, whereas the components with the lowest ratings were behavioral goals/objectives and team communication. The implications for delivering brief trainings to improve evidence-based practice, as well as limitations and future directions, are discussed.
C1 [Kraemer, Bonnie R.] San Diego State Univ, Dept Special Educ, San Diego, CA 92182 USA.
[Cook, Clayton R.] Univ Calif Riverside, Sch Psychol Program, Riverside, CA 92521 USA.
[Wallace, Michele D.] Calif State Univ Los Angeles, Div Special Educ & Counseling, Los Angeles, CA 90032 USA.
RP Kraemer, BR (reprint author), San Diego State Univ, Dept Special Educ, San Diego, CA 92182 USA.
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NR 28
TC 8
Z9 8
PU SAGE PUBLICATIONS INC
PI THOUSAND OAKS
PA 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA
SN 1098-3007
J9 J POSIT BEHAV INTERV
JI J. Posit. Behav. Interv.
PD JUL
PY 2008
VL 10
IS 3
BP 179
EP 189
DI 10.1177/1098300708318796
PG 11
WC Psychology, Clinical; Education, Special
SC Psychology; Education & Educational Research
GA 390MS
UT WOS:000262169000005
ER
PT J
AU Bornhofen, C
Mcdonald, S
AF Bornhofen, Cristina
Mcdonald, Skye
TI Emotion perception deficits following traumatic brain injury: A review
of the evidence and rationale for intervention
SO JOURNAL OF THE INTERNATIONAL NEUROPSYCHOLOGICAL SOCIETY
LA English
DT Review
DE rehabilitation; facial expression; social interaction; social behavior;
affect; recovery of function; cortical contusion; traumatic brain injury
ID CLOSED-HEAD-INJURY; FACIAL AFFECT RECOGNITION; HIGH-FUNCTIONING AUTISM;
ADULT SQUIRREL-MONKEYS; PRIMARY MOTOR CORTEX; SOCIAL-PERCEPTION;
ASPERGERS-SYNDROME; AMYGDALA DAMAGE; MOVEMENT REPRESENTATIONS; COGNITIVE
REHABILITATION
AB While the cognitive disturbances that frequently follow severe traumatic brain injury (TBI) are relatively well understood, the ways in which these affect the psychosocial functioning of people with TBI are yet to be determined and have thus received little attention in treatment research. Growing evidence indicates that a significant proportion of individuals with TBI demonstrate an inability to recognize affective information from the face, voice, bodily movement, and posture. Because accurate interpretation of emotion in others is critical for the successful negotiation of social interactions, effective treatments are necessary. Until recently, however, there have been no rehabilitation efforts in this area. The present review examines the literature on emotion perception deficits in TBI and presents a theoretical rationale for targeted intervention. Several lines of research relevant to the remediation of emotion perception in people with TBI are considered. These include work on emotion perception remediation with other cognitively impaired populations, current neuropsychological models of emotion perception and underlying neural systems, and recent conceptualizations of remediation processes. The article concludes with a discussion of the importance of carrying out efforts to improve emotion perception within a contextualized framework in which the day-to-day relevance of training is clear to all recipients.
C1 [Bornhofen, Cristina; Mcdonald, Skye] Univ New S Wales, Sch Psychol, Sydney, NSW 2052, Australia.
RP Bornhofen, C (reprint author), Univ New S Wales, Sch Psychol, Sydney, NSW 2052, Australia.
EM cristinab@iinet.net.au
RI McDonald, Skye/G-4118-2014
OI McDonald, Skye/0000-0003-0723-6094
FU National Medical and Research Council of Australia
FX The information contained in this manuscript is based on parts of a PhD
thesis completed by the first author and has not previously been
published. This research was facilitated by a project grant from the
National Medical and Research Council of Australia
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NR 223
TC 40
Z9 40
PU CAMBRIDGE UNIV PRESS
PI NEW YORK
PA 32 AVENUE OF THE AMERICAS, NEW YORK, NY 10013-2473 USA
SN 1355-6177
J9 J INT NEUROPSYCH SOC
JI J. Int. Neuropsychol. Soc.
PD JUL
PY 2008
VL 14
IS 4
BP 511
EP 525
DI 10.1017/S1355617708080703
PG 15
WC Clinical Neurology; Neurosciences; Psychiatry; Psychology
SC Neurosciences & Neurology; Psychiatry; Psychology
GA 338VL
UT WOS:000258537400001
PM 18577280
ER
PT J
AU Quincozes-Santos, A
Abib, RT
Leite, MC
Bobermin, D
Bambini-Junior, V
Goncalves, CA
Riesgo, R
Gottfried, C
AF Quincozes-Santos, Andre
Abib, Renata Torres
Leite, Marina Concli
Bobermin, Daiane
Bambini-Junior, Victorio
Goncalves, Carlos-Alberto
Riesgo, Rudimar
Gottfried, Carmem
TI Effect of the atypical neuroleptic risperidone on morphology and S100B
secretion in C6 astroglial lineage cells
SO MOLECULAR AND CELLULAR BIOCHEMISTRY
LA English
DT Article
DE risperidone; atypical antipsychotic; astrocyte; C6 cells; S100B;
morphology; autism
ID GLIOMA-CELLS; GLUTAMATE UPTAKE; GLIAL-CELLS; RHO GTPASES; AUTISM;
ORGANIZATION; PROTEINS; BRAIN
AB We investigated the effect of the atypical neuroleptic risperidone on morphology and S100B secretion in C6 glioma cells, considering the putative involvement of astroglial cells in neuropsychiatric disorders. In the presence of high experimental doses of risperidone, C6 cells become stellate, with process-bearing cells and partial retraction of the cell body followed by detachment from the adhesion surface with practically no cell death. These results indicate that risperidone is able to interfere with C6 cell adhesion without toxic effects. RhoA activator LPA prevented the effects of risperidone on cell morphology. From 6 h risperidone induced a statistically significant increment of about 80% in S100B secretion. These data contribute to the proposal that glial cells are targets of risperidone, which could be involved in the therapeutic response of risperidone to improve autism symptoms.
C1 [Quincozes-Santos, Andre; Abib, Renata Torres; Leite, Marina Concli; Bobermin, Daiane; Bambini-Junior, Victorio; Goncalves, Carlos-Alberto; Gottfried, Carmem] Univ Fed Rio Grande do Sul, Inst Ciencias Basicas Saude, Dept Bioquim, BR-90035003 Porto Alegre, RS, Brazil.
[Riesgo, Rudimar; Gottfried, Carmem] Univ Fed Rio Grande do Sul, Hosp Clin Porto Alegre, Unidade Neurol Pediat, Pervas Dev Disorders Program ProTID, BR-90035003 Porto Alegre, RS, Brazil.
RP Gottfried, C (reprint author), Univ Fed Rio Grande do Sul, Inst Ciencias Basicas Saude, Dept Bioquim, Rua Ramiro Barcelos 2600 Anexo, BR-90035003 Porto Alegre, RS, Brazil.
EM cgottfried@ufrgs.br
RI Riesgo, Rudimar/F-5332-2010; Goncalves, Carlos/K-1851-2014;
Bambini-Junior, Victorio/O-7563-2014
OI Riesgo, Rudimar/0000-0001-9888-7532;
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PA VAN GODEWIJCKSTRAAT 30, 3311 GZ DORDRECHT, NETHERLANDS
SN 0300-8177
J9 MOL CELL BIOCHEM
JI Mol. Cell. Biochem.
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PY 2008
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IS 1-2
BP 59
EP 63
DI 10.1007/s11010-008-9765-x
PG 5
WC Cell Biology
SC Cell Biology
GA 313RH
UT WOS:000256757200008
PM 18421423
ER
PT J
AU Burmeister, M
McInnis, MG
Zollner, S
AF Burmeister, Margit
McInnis, Melvin G.
Zollner, Sebastian
TI Psychiatric genetics: progress amid controversy
SO NATURE REVIEWS GENETICS
LA English
DT Review
ID GENOME-WIDE ASSOCIATION; OBSESSIVE-COMPULSIVE DISORDER; DEFICIT
HYPERACTIVITY DISORDER; MOLECULAR CYTOGENETIC ANALYSIS; BIPOLAR
AFFECTIVE-DISORDER; AUTISM SPECTRUM DISORDERS; SUSCEPTIBILITY LOCI;
RECEPTOR GENE; SCHIZOAFFECTIVE DISORDER; POLYMORPHISM 5-HTTLPR
AB Several psychiatric disorders-such as bipolar disorder, schizophrenia and autism-are highly heritable, yet identifying their genetic basis has been challenging, with most discoveries failing to be replicated. However, inroads have been made by the incorporation of intermediate traits (endophenotypes) and of environmental factors into genetic analyses, and through the identification of rare inherited variants and novel structural mutations. Current efforts aim to increase sample sizes by gathering larger samples for case-control studies or through meta-analyses of such studies. More attention on unique families, rare variants, and on incorporating environment and the emerging knowledge of biological function and pathways into genetic analysis is warranted.
C1 [Burmeister, Margit] Univ Michigan, Mol & Behav Neurosci Inst, Ann Arbor, MI 48109 USA.
[Burmeister, Margit; McInnis, Melvin G.; Zollner, Sebastian] Univ Michigan, Dept Psychiat, Ann Arbor, MI 48109 USA.
[Burmeister, Margit; McInnis, Melvin G.; Zollner, Sebastian] Univ Michigan, Depress Ctr, Ann Arbor, MI 48109 USA.
[Burmeister, Margit] Dept Human Genet, Ann Arbor, MI 48109 USA.
[Zollner, Sebastian] Univ Michigan, Dept Biostat, Ann Arbor, MI 48109 USA.
RP Burmeister, M (reprint author), Univ Michigan, Mol & Behav Neurosci Inst, 5061 BSRB,109 Zina Pitcher Pl, Ann Arbor, MI 48109 USA.
EM margit@umich.edu
RI McInnis, Melvin/F-6963-2012; Burmeister, Margit/A-3157-2013
OI McInnis, Melvin/0000-0002-0375-6247; Burmeister,
Margit/0000-0002-1914-2434
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NR 161
TC 182
Z9 188
PU NATURE PUBLISHING GROUP
PI LONDON
PA MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND
SN 1471-0056
J9 NAT REV GENET
JI Nat. Rev. Genet.
PD JUL
PY 2008
VL 9
IS 7
BP 527
EP 540
DI 10.1038/nrg2381
PG 14
WC Genetics & Heredity
SC Genetics & Heredity
GA 314TX
UT WOS:000256832900013
PM 18560438
ER
PT J
AU Abu-Elneel, K
Liu, T
Gazzaniga, FS
Nishimura, Y
Wall, DP
Geschwind, DH
Lao, KQ
Kosik, KS
AF Abu-Elneel, Kawther
Liu, Tsunglin
Gazzaniga, Francesca S.
Nishimura, Yuhei
Wall, Dennis P.
Geschwind, Daniel H.
Lao, Kaiqin
Kosik, Kenneth S.
TI Heterogeneous dysregulation of microRNAs across the autism spectrum
SO NEUROGENETICS
LA English
DT Article
DE autism spectrum disorder; miRNA; postmortem tissue; cerebellum
ID LEMLI-OPITZ-SYNDROME; TUBEROUS SCLEROSIS; ATYPICAL AUTISM; HUMAN GENOME;
COPY-NUMBER; DISORDERS; GENE; ASSOCIATION; MUTATIONS; LINKAGE
AB microRNAs (miRNAs) are similar to 21 nt transcripts capable of regulating the expression of many mRNAs and are abundant in the brain. miRNAs have a role in several complex diseases including cancer as well as some neurological diseases such as Tourette's syndrome and Fragile x syndrome. As a genetically complex disease, dysregulation of miRNA expression might be a feature of autism spectrum disorders (ASDs). Using multiplex quantitative polymerase chain reaction (PCR), we compared the expression of 466 human miRNAs from postmortem cerebellar cortex tissue of individuals with ASD (n=13) and a control set of non-autistic cerebellar samples (n=13). While most miRNAs levels showed little variation across all samples suggesting that autism does not induce global dysfunction of miRNA expression, some miRNAs among the autistic samples were expressed at significantly different levels compared to the mean control value. Twenty-eight miRNAs were expressed at significantly different levels compared to the non-autism control set in at least one of the autism samples. To validate the finding, we reversed the analysis and compared each non-autism control to a single mean value for each miRNA across all autism cases. In this analysis, the number of dysregulated miRNAs fell from 28 to 9 miRNAs. Among the predicted targets of dysregulated miRNAs are genes that are known genetic causes of autism such Neurexin and SHANK3. This study finds that altered miRNA expression levels are observed in postmortem cerebellar cortex from autism patients, a finding which suggests that dysregulation of miRNAs may contribute to autism spectrum phenotype.
C1 [Abu-Elneel, Kawther; Liu, Tsunglin; Gazzaniga, Francesca S.; Kosik, Kenneth S.] Univ Calif Santa Barbara, Neurosci Res Inst, Santa Barbara, CA 93106 USA.
[Abu-Elneel, Kawther; Liu, Tsunglin; Gazzaniga, Francesca S.; Kosik, Kenneth S.] Univ Calif Santa Barbara, Dept Mol Cellular & Dev Biol, Santa Barbara, CA 93106 USA.
[Nishimura, Yuhei; Geschwind, Daniel H.] Univ Calif Los Angeles, David Geffen Sch Med, Dept Neurol, Program Neurogenet & Neurobehav Genet, Los Angeles, CA 90095 USA.
[Wall, Dennis P.] Harvard Univ, Sch Med, Ctr Biomed Informat, Boston, MA 02115 USA.
[Wall, Dennis P.] Harvard Univ, Sch Med, Dept Syst, Boston, MA 02115 USA.
[Lao, Kaiqin] Appl Biosyst Inc, Foster City, CA 94404 USA.
RP Kosik, KS (reprint author), Univ Calif Santa Barbara, Neurosci Res Inst, Santa Barbara, CA 93106 USA.
EM kosik@lifesci.ucsb.edu
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TARGETSCAN PREDICTIO
AUTISM TISSUE PROGRA
NR 54
TC 92
Z9 98
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1364-6745
J9 NEUROGENETICS
JI Neurogenetics
PD JUL
PY 2008
VL 9
IS 3
BP 153
EP 161
DI 10.1007/s10048-008-0133-5
PG 9
WC Genetics & Heredity; Clinical Neurology
SC Genetics & Heredity; Neurosciences & Neurology
GA 320EJ
UT WOS:000257216200001
PM 18563458
ER
PT J
AU Higgins, JJ
Hao, J
Kosofsky, BE
Rajadhyaksha, AM
AF Higgins, Joseph J.
Hao, Jin
Kosofsky, Barry E.
Rajadhyaksha, Anjali M.
TI Dysregulation of large-conductance Ca(2+)-activated K(+) channel
expression in nonsyndromal mental retardation due to a cereblon p.R419X
mutation
SO NEUROGENETICS
LA English
DT Article
DE large-conductance calcium-activated potassium channel alpha subunits;
alternative splicing; cereblon protein; human; mental retardation;
nonsyndromic; autosomal recessive; 2A protein; human; reverse
transcriptase polymerase chain reaction
ID POTASSIUM CHANNEL; CALCIUM-CHANNEL; PROTEIN-KINASE; BKCA CHANNEL; GENE;
AUTISM; SLO; PHOSPHORYLATION; FAMILIES; SIBLINGS
AB A nonsense mutation (R419X) in the human cereblon gene [mutation (mut) CRBN] causes a mild type of autosomal recessive nonsyndromal mental retardation (ARNSMR). CRBN, a cytosolic protein, regulates the assembly and neuronal surface expression of large-conductance Ca(2+)-activated K(+) channels (BK(Ca)) in brain regions involved in memory and learning. Using the real-time quantitative polymerase chain reaction, we show that mut CRBN disturbs the development of adult brain BKCa isoforms. These changes are predicted to result in BKCa channels with a higher intracellular Ca2+ sensitivity, faster activation, and slower deactivation kinetics. Such alterations may contribute to cognitive impairments in patients with mild ARNSMR.
C1 [Higgins, Joseph J.; Hao, Jin] New York Presbyterian Hosp, Weill Cornell Med Coll, Dept Pediat, Div Pediatr Neurol, New York, NY 10065 USA.
[Kosofsky, Barry E.; Rajadhyaksha, Anjali M.] New York Presbyterian Hosp, Weill Cornell Med Coll, Dept Pediat, Div Pediatr Neurol Neurol & Neurosci, New York, NY 10065 USA.
RP Higgins, JJ (reprint author), New York Presbyterian Hosp, Weill Cornell Med Coll, Dept Pediat, Div Pediatr Neurol, New York, NY 10065 USA.
EM joh2016@med.cornell.edu
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NR 30
TC 24
Z9 27
PU SPRINGER
PI NEW YORK
PA 233 SPRING ST, NEW YORK, NY 10013 USA
SN 1364-6745
J9 NEUROGENETICS
JI Neurogenetics
PD JUL
PY 2008
VL 9
IS 3
BP 219
EP 223
DI 10.1007/s10048-008-0128-2
PG 5
WC Genetics & Heredity; Clinical Neurology
SC Genetics & Heredity; Neurosciences & Neurology
GA 320EJ
UT WOS:000257216200009
PM 18414909
ER
PT J
AU Yiu, EM
Kornberg, AJ
AF Yiu, Eppie M.
Kornberg, Andrew J.
TI Duchenne muscular dystrophy
SO NEUROLOGY INDIA
LA English
DT Review
DE Continuous positive airway pressure; corticosteroids; creatine kinase;
Duchenne muscular dystrophy; gene therapy; muscle disease; pediatric
ID BONE-MINERAL DENSITY; HOME NOCTURNAL VENTILATION; AUTISM SPECTRUM
DISORDER; BLIND CONTROLLED TRIAL; SKELETAL-MUSCLE CELLS; DMD GENE;
CORTICOSTEROID TREATMENT; NEUROMUSCULAR DISORDERS; MALIGNANT
HYPERTHERMIA; NONSENSE MUTATION
AB Duchenne muscular dystrophy (DMD), an X-linked disorder, is the most common muscular dystrophy in children, presenting in early childhood and characterized by proximal muscle weakness and calf hypertrophy in affected boys. Patients usually become wheelchair-bound by the age of 12 years, and die of cardiorespiratory complications in their late teens to early twenties. Advances in the management of DMD, including treatment with corticosteroids and the use of intermittent positive pressure ventilation have provided improvements in function, ambulation, quality of life and life expectancy, although novel therapies still aim to provide a cure for this devastating disorder. The clinical features, investigations, and management of DMD are reviewed, as well as the latest in some of the novel therapies.
C1 [Yiu, Eppie M.; Kornberg, Andrew J.] Royal Childrens Hosp Melbourne, Childrens Neurosci Ctr, Parkville, Vic 3052, Australia.
[Kornberg, Andrew J.] Univ Melbourne, Dept Pediat, Murdoch Childrens Res Inst, Melbourne, Vic 3010, Australia.
RP Yiu, EM (reprint author), Royal Childrens Hosp Melbourne, Childrens Neurosci Ctr, Flemington Rd, Parkville, Vic 3052, Australia.
EM andrew.kornberg@rch.org.au
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NR 191
TC 18
Z9 19
PU MEDKNOW PUBLICATIONS
PI MUMBAI
PA A-108-109 KANARA BUSINESS CENTRE, GHAKTOPAR, MUMBAI, 400075, INDIA
SN 0028-3886
J9 NEUROL INDIA
JI Neurol. India
PD JUL-SEP
PY 2008
VL 56
IS 3
BP 236
EP 247
PG 12
WC Neurosciences
SC Neurosciences & Neurology
GA 371YJ
UT WOS:000260868300004
PM 18974549
ER
PT J
AU Hermann, EJ
Rittierodt, M
Krauss, JK
AF Hermann, Elvis J.
Rittierodt, Marion
Krauss, Joachim K.
TI Combined transventricular and supracerebellar infratentorial approach
preserving the vermis in giant pediatric posterior fossa midline tumors
SO NEUROSURGERY
LA English
DT Article
DE cerebellomedullary fissure approach; infratentorial supracerebellar
approach; pediatric midline tumors; vermis
ID COGNITIVE-AFFECTIVE SYNDROME; CEREBELLOMEDULLARY FISSURE; CEREBELLAR
MUTISM; RESECTION; CHILDREN; PATHOPHYSIOLOGY; 4TH-VENTRICLE; ANATOMY;
AUTISM; VEINS
AB OBJECTIVE: Giant pediatric midline tumors of the posterior fossa involving the fourth ventricle and the tectal region are difficult to approach and present a high risk of postoperative neurological deficits. Children with sequelae such as cerebellar mutism and ataxia experience a compromise in their quality of life. Here, we present our combined transventricular and supracerebellar infratentorial approach to avoid complications of vermian splitting.
METHODS:The combined transventricular and supracerebellar infratentorial approach described here was used in a total of four pediatric patients. A medial suboccipital craniotomy with opening of the foramen magnum and resection of the C1 lamina was performed with the patient in the semisitting position. The tumor mass filling the fourth ventricle was removed via a transventricular telovelar route through the foramen of Magendie, preserving the vermis. The rostral tumor portions in the peritectal region extruding up to the thalami were exposed and resected via an infratentorial supracerebellar route to preserve the venous drainage of the cerebellum.
RESULTS: There were no new neurological deficits postoperatively. Two patients had low-grade astrocytomas, and two patients had malignant tumors. Complete tumor resection was achieved in two patients, and near-total tumor removal in the two others.
CONCLUSION: The combined transventricular and supracerebellar infratentorial approach offers a unique possibility of safely removing giant pediatric midline tumors. Splitting of the cerebellar vermis is not necessary for removal of such tumors.
C1 [Hermann, Elvis J.; Rittierodt, Marion; Krauss, Joachim K.] Hannover Med Sch, Dept Neurosurg, D-30625 Hannover, Germany.
RP Krauss, JK (reprint author), Hannover Med Sch, Dept Neurosurg, Carl Neuberg Str 1, D-30625 Hannover, Germany.
EM Krauss.Joachim@mh-hannover.de
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NR 35
TC 2
Z9 4
PU LIPPINCOTT WILLIAMS & WILKINS
PI PHILADELPHIA
PA 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA
SN 0148-396X
J9 NEUROSURGERY
JI Neurosurgery
PD JUL
PY 2008
VL 63
IS 1
SU S
BP 30
EP 35
DI 10.1227/01.NEU.0000316853.47780.B9
PG 6
WC Clinical Neurology; Surgery
SC Neurosciences & Neurology; Surgery
GA 341LN
UT WOS:000258716100009
ER
PT J
AU Grigg-Domberger, M
AF Grigg-Domberger, Madeleine
TI Evaluating children who seize during sleep
SO PEDIATRIC ANNALS
LA English
DT Review
ID FRONTAL-LOBE EPILEPSY; ELECTRICAL STATUS EPILEPTICUS; AUTISM SPECTRUM
DISORDERS; LANDAU-KLEFFNER-SYNDROME; BENIGN CHILDHOOD EPILEPSY; COMPLEX
PARTIAL SEIZURES; ROLANDIC EPILEPSY; CENTROTEMPORAL SPIKES; SLOW SLEEP;
PANAYIOTOPOULOS-SYNDROME
C1 [Grigg-Domberger, Madeleine] Univ New Mexico, Clin Neurophysiol Lab, Albuquerque, NM 87131 USA.
[Grigg-Domberger, Madeleine] Univ New Mexico, Sleep Disorders Ctr, Pediat Sleep Med Serv, Albuquerque, NM 87131 USA.
RP Grigg-Domberger, M (reprint author), 915 Camino Salud NE, Albuquerque, NM 87131 USA.
EM mgriggd@solud.unm.edu
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2007, PRESCRIRE INT, V16, P200
NR 120
TC 0
Z9 0
PU SLACK INC
PI THOROFARE
PA 6900 GROVE RD, THOROFARE, NJ 08086 USA
SN 0090-4481
J9 PEDIATR ANN
JI Pediatr. Annu.
PD JUL
PY 2008
VL 37
IS 7
BP 472
EP 480
PG 9
WC Pediatrics
SC Pediatrics
GA 330SF
UT WOS:000257962400006
PM 18710137
ER
PT J
AU Do, SB
Napalinga, K
Pinninti, N
Carvalho, KS
Valencia, I
Legido, A
Kothare, SV
AF Do, Shaila Bokkala
Napalinga, Katherine
Pinninti, Narsimha
Carvalho, Karen S.
Valencia, Ignacio
Legido, Agustin
Kothare, Sanjeev V.
TI Correlates of periodic limb movements of sleep in the pediatric
population
SO PEDIATRIC NEUROLOGY
LA English
DT Article
ID RESTLESS LEGS SYNDROME; PARKINSONS-DISEASE; BEHAVIOR DISORDER; IRON
STATUS; REM-SLEEP; CHILDREN; DISTURBANCE; PREVALENCE; CHILDHOOD; EEG
AB Periodic limb movements of sleep are clinically underdiagnosed in children. Polysomnography is the most accurate diagnostic test. There is a paucity of information regarding polysomnography findings in children. We evaluated the prevalence and correlates of pediatric periodic limb movements detected by polysomnography. Periodic limb movements of sleep were identified in 77 of 982 polysomnograms, with a prevalence of 7.8% and male predominance (47 boys; 30 girls). Mean age was 9.4 +/- 4.2 years (1-19 years) (body mass index, 24.1 +/- 12.3). Mean sleep time was 395.4 +/- 73.4 minutes, of which rapid eye movement sleep constituted 16.6% +/- 6.7%, and slow-wave sleep, 22% +/- 10%. Sleep efficiency was 93.8 +/- 9.83, periodic limb movement index, 9.78 +/- 7.9; periodic limb movement arousal, 4.5 +/- 8.4; arousal index, 27.8 +/- 12.4; and peak end-tidal CO2, 48.9 +/- 10.5 mm Hg. Associated diagnoses included obstructive sleep apnea in 36 (46.8%), attention deficit hyperactivity disorder in 10 (13%), migraine in 7 (9.1%), seizures in 7 (9.1%), autism spectrum disorders in 5 (6.5%), and narcolepsy in 7 (9.1%). Serum ferritin was decreased (mean, 26.1 mu g/L) in 29 (96.6%). Prospective studies may clarify the significance of incidental pediatric periodic limb movements in sleep detected on polysomnograms. (C) 2008 by Elsevier Inc. All rights reserved.
C1 [Do, Shaila Bokkala; Napalinga, Katherine; Pinninti, Narsimha; Carvalho, Karen S.; Valencia, Ignacio; Legido, Agustin; Kothare, Sanjeev V.] Drexel Univ, Coll Med, Dept Pediat, St Christophers Hosp Children,Dept Neurol, Philadelphia, PA 19104 USA.
[Do, Shaila Bokkala] Drexel Univ, Coll Med, Dept Neurol, Hahnemann Hosp, Philadelphia, PA 19104 USA.
RP Kothare, SV (reprint author), Harvard Univ, Sch Med, Childrens Hosp Boston, Dept Neurol, Fegan 9,300 Longwood Ave, Boston, MA 02115 USA.
EM sanjeev.kothare@childrens.harvard.edu
CR American Academy of Sleep Medicine, 2005, INT CLASS SLEEP DIS, V2nd
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NR 43
TC 0
Z9 0
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0887-8994
EI 1873-5150
J9 PEDIATR NEUROL
JI Pediatr. Neurol.
PD JUL
PY 2008
VL 39
IS 1
BP 33
EP 39
DI 10.1016/j.pediatrneuro1.2008.03.008
PG 7
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 318PU
UT WOS:000257104900006
ER
PT J
AU Burusnukul, P
de los Ryes, EC
Yinger, J
Boue, DR
AF Burusnukul, Prinyarat
de los Ryes, Emily C.
Yinger, Jacqueline
Boue, Daniel R.
TI Danon disease: An unusual presentation of autism
SO PEDIATRIC NEUROLOGY
LA English
DT Article
ID LINKED VACUOLAR CARDIOMYOPATHY; LAMP-2 GENE MUTATION; HYPERTROPHIC
CARDIOMYOPATHY; MYOPATHY
AB Danon disease is an X-linked cardioskeletal myopathy, originally reported as "lysosomal glycogen storage disease with normal acid maltase," resulting from a primary deficiency of lysosome-associated membrane protein-2 because of mutations in the lysosome-associated membrane protein-2 gene. Classic clinical features in males include cardiomyopathy (100%, eventually), myopathy (90%), and mental retardation (70%), but mostly of a mild degree. We report on an unusual presentation in a patient with autism, motor delay, and a normal cardiac evaluation. The presence of multiorgan involvement, including elevated liver enzymes, abnormal cranial magnetic resonance imaging, and diffuse hypotonia with swallowing difficulties, prompted a muscle biopsy. A quadriceps muscle biopsy was performed, and the findings were most suspicious for a glycogen storage-type disease. Subsequently, a pathogenic lysosome-associated membrane protein-2 mutation was found. To our knowledge, there are no previous clinical reports of autism in children with Danon disease. (C) 2008 by Elsevier Inc. All rights reserved.
C1 [Burusnukul, Prinyarat; de los Ryes, Emily C.; Yinger, Jacqueline] Nationwide Childrens Hosp, Dept Pediat & Neurol, Columbus, OH 43205 USA.
[Boue, Daniel R.] Nationwide Childrens Hosp, Dept Lab Med & Pathol, Columbus, OH 43205 USA.
Ohio State Univ, Columbus, OH 43205 USA.
RP Burusnukul, P (reprint author), Nationwide Childrens Hosp, Dept Pediat & Neurol, 700 Childrens Dr, Columbus, OH 43205 USA.
EM Prinyarat.Burusnukul@nationwidechildrens.org
CR Arad M, 2005, NEW ENGL J MED, V352, P362, DOI 10.1056/NEJMoa033349
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Nishino I, 2000, NATURE, V406, P906, DOI 10.1038/35022604
Sugie K, 2002, NEUROLOGY, V58, P1773
Yang Z, 2005, CIRCULATION, V112, P1612, DOI 10.1161/CIRCULATIONAHA.105.546481
NR 8
TC 8
Z9 8
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0887-8994
J9 PEDIATR NEUROL
JI Pediatr. Neurol.
PD JUL
PY 2008
VL 39
IS 1
BP 52
EP 54
DI 10.1016/j.pediatrneuro1.2008.03.011
PG 3
WC Clinical Neurology; Pediatrics
SC Neurosciences & Neurology; Pediatrics
GA 318PU
UT WOS:000257104900010
PM 18555174
ER
PT J
AU Jangaard, KA
Fell, DB
Dodds, L
Allen, AC
AF Jangaard, Krista A.
Fell, Deshayne B.
Dodds, Linda
Allen, Alexander C.
TI Outcomes in a population of healthy term and near-term infants with
serum bilirubin levels of >= 325 mu mol/l (>= 19 mg/dL) who were born in
Nova Ccotia, Canada, between 1994 and 2000
SO PEDIATRICS
LA English
DT Article
DE hyperbilirubinemia; outcomes; population-based
ID DEVELOPMENT CLINICAL-TRIAL; BREAST-FED TERM; FOLLOW-UP; NEONATAL
HYPERBILIRUBINEMIA; NONHEMOLYTIC HYPERBILIRUBINEMIA; NATIONAL-INSTITUTE;
CHILD-HEALTH; HEARING-LOSS; KERNICTERUS; PHOTOTHERAPY
AB OBJECTIVE. The goal was to study the incidence of kernicterus, developmental delay, autism, cerebral palsy, and hearing loss in infants with peak total serum bilirubin levels of >= 325 mu mol/ L ( >= 19 mg/ dL), compared with infants with less- severe or no hyperbilirubinemia, in a population of healthy term and late preterm infants.
METHODS. Prospectively gathered, standardized, maternal and neonatal data for infants at >= 35 weeks of gestation who were born between January 1, 1994, and December 31, 2000, were extracted from the Nova Scotia Atlee Perinatal Database. Infants with Rh factor isoimmunization, significant congenital or chromosomal abnormalities, or severe peripartum asphyxia were excluded. Comparisons were made on the basis of peak total serum bilirubin levels. Diagnoses were obtained through data linkage with the Medical Services Insurance Database for office visits and the Canadian Institute for Health Information Database for hospital admissions. The registration file provided information allowing calculation of follow-up times, which were determined for each separate outcome. Follow-up periods ranged from 2 to 9 years, with the end point being the first time the diagnostic code was encountered in either database. Cox proportional-hazards regression analyses were used to examine the relationships between outcomes and total serum bilirubin levels.
RESULTS. Of 61 238 infants included in the study cohort, 4010 ( 6.7%) did not have linkage data, which left 56 019 infants for analysis. There were no cases of kernicterus and no significant differences in rates of cerebral palsy, deafness, developmental delay, or visual abnormalities between the groups. There were suggestions of associations with attention-deficit disorder in the severe hyperbilirubinemia group and with autism in the combined moderate and severe hyperbilirubinemia group.
CONCLUSIONS. There was no increase in adverse effects reported previously to be associated with bilirubin toxicity. Associations with developmental delay, attention-deficit disorder, and autism were observed.
C1 [Jangaard, Krista A.; Allen, Alexander C.] Dalhousie Univ, Dept Neonatal Perinatal Med, Halifax, NS B3K 6R8, Canada.
[Fell, Deshayne B.; Dodds, Linda; Allen, Alexander C.] Dalhousie Univ, Perinatal Epidemiol Res Unit, Halifax, NS B3K 6R8, Canada.
RP Jangaard, KA (reprint author), Dalhousie Univ, Dept Neonatal Perinatal Med, 5850-5980 Univ Ave,POB 9700, Halifax, NS B3K 6R8, Canada.
EM krista.jangaard@dal.ca
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Centers for Disease Control and Prevention (CDC), 2001, MMWR-MORBID MORTAL W, V50, P491
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Wong V, 2006, J CHILD NEUROL, V21, P309, DOI 10.2310/7010.2006.00058
NR 25
TC 33
Z9 34
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD JUL
PY 2008
VL 122
IS 1
BP 119
EP 124
DI 10.1542/peds.2007-0967
PG 6
WC Pediatrics
SC Pediatrics
GA 320YN
UT WOS:000257271200016
PM 18595994
ER
PT J
AU Harrington, JW
AF Harrington, John W.
TI Are we overmedicating our children?
SO PEDIATRICS
LA English
DT Letter
ID AUTISM
C1 Childrens Hosp Kings Daughters, Eastern Virginia Med Sch, Div Gen Pediat, Norfolk, VA 23507 USA.
RP Harrington, JW (reprint author), Childrens Hosp Kings Daughters, Eastern Virginia Med Sch, Div Gen Pediat, Norfolk, VA 23507 USA.
CR Kolevzon A, 2006, J CLIN PSYCHIAT, V67, P407
MANDELL DS, 2008, PEDIATRICS, V121
Myers SM, 2007, PEDIATRICS, V120, P1162, DOI 10.1542/peds.2007-2362
SHEA S, 2004, PEDIATRICS, V114
NR 4
TC 1
Z9 1
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD JUL
PY 2008
VL 122
IS 1
BP 211
EP 212
DI 10.1542/peds.2008-0879
PG 8
WC Pediatrics
SC Pediatrics
GA 320YN
UT WOS:000257271200034
PM 18596010
ER
PT J
AU Indredavik, MS
Vik, T
Skranes, J
Brubakk, AM
AF Indredavik, Marit S.
Vik, Torstein
Skranes, Jon
Brubakk, Ann-Mari
TI Positive screening results for autism in ex-preterm infants
SO PEDIATRICS
LA English
DT Letter
ID LOW-BIRTH-WEIGHT; PSYCHIATRIC-SYMPTOMS; ADOLESCENTS; ABNORMALITIES
C1 [Indredavik, Marit S.] Norwegian Univ Sci & Technol, Dept Neurosci, NO-7489 Trondheim, Norway.
[Indredavik, Marit S.] St Olavs Univ Hosp, Dept Child & Adolescent Psychiat, NO-7433 Trondheim, Norway.
[Vik, Torstein] Norwegian Univ Sci & Technol, Dept Publ Hlth & Gen Practice, NO-7489 Trondheim, Norway.
[Skranes, Jon; Brubakk, Ann-Mari] Norwegian Univ Sci & Technol, Dept Lab Med, NO-7489 Trondheim, Norway.
[Skranes, Jon; Brubakk, Ann-Mari] St Olavs Univ Hosp, Childrens Clin, NO-7433 Trondheim, Norway.
RP Indredavik, MS (reprint author), Norwegian Univ Sci & Technol, Dept Neurosci, NO-7489 Trondheim, Norway.
CR Indredavik MS, 2004, ARCH DIS CHILD, V89, pF445, DOI 10.1136/adc.2003.038943
Indredavik MS, 2005, PEDIATR NEUROL, V33, P259, DOI 10.1016/j.pediatrneurol.2005.05.002
Limperopoulos C, 2008, PEDIATRICS, V121, P758, DOI 10.1542/peds.2007-2158
Skranes J, 2007, BRAIN, V130, P654, DOI 10.1093/brain/awm001
NR 4
TC 8
Z9 8
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD JUL
PY 2008
VL 122
IS 1
BP 222
EP 222
DI 10.1542/peds.2008-1044
PG 1
WC Pediatrics
SC Pediatrics
GA 320YN
UT WOS:000257271200050
PM 18596018
ER
PT J
AU Limperopoulos, C
AF Limperopoulos, Catherine
TI Positive screening results for autism in ex-preterm infants - In reply
SO PEDIATRICS
LA English
DT Letter
ID INTERVENTION
C1 McGill Univ, Montreal Childrens Hosp, Sch Phys & Occupat Therapy, Dept Neurol & Neurosurg, Montreal, PQ H3H 1P3, Canada.
RP Limperopoulos, C (reprint author), McGill Univ, Montreal Childrens Hosp, Sch Phys & Occupat Therapy, Dept Neurol & Neurosurg, Montreal, PQ H3H 1P3, Canada.
CR Ben-Itzchak E, 2007, RES DEV DISABIL, V28, P287, DOI 10.1016/j.ridd.2006.03.002
Bryson SE, 2003, CAN J PSYCHIAT, V48, P506
National Research Council Committee on Interventions for Children with Autism, 2001, ED CHILDR AUT
NR 3
TC 0
Z9 0
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD JUL
PY 2008
VL 122
IS 1
BP 222
EP 223
DI 10.1542/peds.2008-1288
PG 8
WC Pediatrics
SC Pediatrics
GA 320YN
UT WOS:000257271200051
ER
PT J
AU Montes, G
Halterman, JS
AF Montes, Guillermo
Halterman, Jill S.
TI Child care problems and employment among families with preschool-aged
children with autism in the United States
SO PEDIATRICS
LA English
DT Article
ID SPECTRUM DISORDERS; IDENTIFYING CHILDREN; DEVELOPMENTAL STATUS; PARENTS
EVALUATION; HEALTH
AB BACKGROUND. The impact of childhood autism on parental employment is largely unknown.
OBJECTIVE. The purpose of this work was to describe the child care arrangements of children with autism and to determine whether families of preschool-aged children with autism are more likely to report that child care arrangements affected employment compared with typically developing children and children at high risk for developmental problems.
METHODS. Parents of 16 282 preschool-aged children were surveyed by the National Survey of Children's Health. An autism spectrum disorder was defined as an affirmative response to the question, "Has a doctor or health professional ever told you that [child] has any of the following conditions? Autism?" There were 82 children with autism spectrum disorder in the sample, and 1955 children at high risk on the basis of the Parent's Evaluation of Developmental Status. We used chi(2) and multivariate logistic regression analyses.
RESULTS. Ninety-seven percent of preschool-aged children diagnosed with autism spectrum disorder were cared for in community settings, particularly preschool and Head Start, with only 3% in exclusive parental care. Thirty-nine percent of the parents of children with autism spectrum disorder reported that child care problems had greatly affected their employment decisions, compared with 16% of the children at high risk and 9% of those who were typically developing. In multivariate analyses, families with a child with autism spectrum disorder were 7 times more likely to state that child care problems affected employment than other families, after controlling for household and child covariates. This effect was 3 times larger than the effect of poverty.
CONCLUSIONS. Developmental problems and autism spectrum disorder are associated with higher use of child care services and higher probability that child care problems will greatly affect employment. These findings warrant evaluation of the community resources available to families with children with special needs.
C1 [Montes, Guillermo] Childrens Inst, Rochester, NY 14607 USA.
Univ Rochester, Sch Med & Dent, Dept Pediat, Rochester, NY 14642 USA.
RP Montes, G (reprint author), Childrens Inst, 271 N Goodman St,Suite D103, Rochester, NY 14607 USA.
EM gmontes@childrensinstitute.net
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NR 28
TC 19
Z9 20
PU AMER ACAD PEDIATRICS
PI ELK GROVE VILLAGE
PA 141 NORTH-WEST POINT BLVD,, ELK GROVE VILLAGE, IL 60007-1098 USA
SN 0031-4005
J9 PEDIATRICS
JI Pediatrics
PD JUL
PY 2008
VL 122
IS 1
BP E202
EP E208
DI 10.1542/peds.2007-3037
PG 7
WC Pediatrics
SC Pediatrics
GA 320YN
UT WOS:000257271200086
PM 18595965
ER
PT J
AU Kim, J
Jung, SY
Lee, YK
Park, S
Choi, JS
Lee, CJ
Kim, HS
Choi, YB
Scheiffele, P
Bailey, CH
Kandel, ER
Kim, JH
AF Kim, Juhyun
Jung, Sang-Yong
Lee, Yeon Kyung
Park, Sangki
Choi, June-Seek
Lee, C. Justin
Kim, Hye-Sun
Choi, Yun-Beom
Scheiffele, Peter
Bailey, Craig H.
Kandel, Eric R.
Kim, Joung-Hun
TI Neuroligin-1 is required for normal expression of LTP and associative
fear memory in the amygdala of adult animals
SO PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF
AMERICA
LA English
DT Article
DE synaptic plasticity; neuroligin; autism
ID LONG-TERM POTENTIATION; SYNAPTIC-TRANSMISSION; INHIBITORY SYNAPSES;
ADHESION MOLECULE; LATERAL AMYGDALA; AUTISM; PLASTICITY; MODULATION;
MUTATION; PATHWAY
AB Neuroligin-1 is a potent trigger for the de novo formation of synaptic connections, and it has recently been suggested that it is required for the maturation of functionally competent excitatory synapses. Despite evidence for the role of neuroligin-1 in specifying excitatory synapses, the underlying molecular mechanisms and physiological consequences that neuroligin-1 may have at mature synapses of normal adult animals remain unknown. By silencing endogenous neuroligin-1 acutely in the amygdala of live behaving animals, we have found that neuroligin-1 is required for the storage of associative fear memory. Subsequent cellular physiological studies showed that suppression of neuroligin-1 reduces NMDA receptor-mediated currents and prevents the expression of long-term potentiation without affecting basal synaptic connectivity at the thalamo-amygdala pathway. These results indicate that persistent expression of neuroligin-1 is required for the maintenance of NMDAR-mediated synaptic transmission, which enables normal development of synaptic plasticity and long-term memory in the amygdala of adult animals.
C1 [Choi, Yun-Beom; Scheiffele, Peter; Bailey, Craig H.; Kandel, Eric R.] Columbia Univ, Coll Phys & Surg, Dept Neurosci, New York, NY 10032 USA.
[Kim, Juhyun; Jung, Sang-Yong; Park, Sangki; Kim, Joung-Hun] Pohang Univ Sci & Technol, Dept Life Sci, Pohang 790784, Gyungbuk, South Korea.
[Lee, Yeon Kyung; Choi, June-Seek] Korea Univ, Dept Psychol, Seoul 136701, South Korea.
[Lee, C. Justin] Korea Inst Sci & Technol, Ctr Neural Sci, Seoul 136791, South Korea.
[Kim, Hye-Sun] Seoul Natl Univ, Coll Med, Dept Pharmacol, Seoul 110799, South Korea.
RP Kandel, ER (reprint author), Columbia Univ, Coll Phys & Surg, Dept Neurosci, New York, NY 10032 USA.
EM erk5@columbia.edu; joungkim@postech.ac.kr
RI Kim, Hye Sun/J-2752-2012; yu, yan/C-2322-2012
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NR 33
TC 64
Z9 65
PU NATL ACAD SCIENCES
PI WASHINGTON
PA 2101 CONSTITUTION AVE NW, WASHINGTON, DC 20418 USA
SN 0027-8424
J9 P NATL ACAD SCI USA
JI Proc. Natl. Acad. Sci. U. S. A.
PD JUL 1
PY 2008
VL 105
IS 26
BP 9087
EP 9092
DI 10.1073/pnas.0803448105
PG 6
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 322DC
UT WOS:000257354400052
PM 18579781
ER
PT J
AU Park, CC
AF Park, Clara C.
TI Growing up with autism: Working with school-age children and adolescents
SO PSYCHIATRIC SERVICES
LA English
DT Book Review
C1 [Park, Clara C.] Williams Coll, Williamstown, MA 01267 USA.
RP Park, CC (reprint author), Williams Coll, Williamstown, MA 01267 USA.
CR GABRIELS RL, 2007, GROWING UP AUTISM WO
NR 1
TC 0
Z9 0
PU AMER PSYCHIATRIC PUBLISHING, INC
PI ARLINGTON
PA 1000 WILSON BOULEVARD, STE 1825, ARLINGTON, VA 22209-3901 USA
SN 1075-2730
J9 PSYCHIAT SERV
JI Psychiatr. Serv.
PD JUL
PY 2008
VL 59
IS 7
BP 818
EP 818
DI 10.1176/appi.ps.59.7.818
PG 1
WC Health Policy & Services; Public, Environmental & Occupational Health;
Psychiatry
SC Health Care Sciences & Services; Public, Environmental & Occupational
Health; Psychiatry
GA 319YV
UT WOS:000257201800029
ER
PT J
AU Schneider, T
Roman, A
Basta-Kaim, A
Kubera, M
Budziszewska, B
Schneider, K
Przewtocki, R
AF Schneider, Tomasz
Roman, Adam
Basta-Kaim, Agnieszka
Kubera, Marta
Budziszewska, Bogustawa
Schneider, Karolina
Przewtocki, Ryszard
TI Gender-specific behavioral and immunological alterations in an animal
model of autism induced by prenatal exposure to valproic acid
SO PSYCHONEUROENDOCRINOLOGY
LA English
DT Review
DE animal model; autism; gender-specific; alterations; rats; valproic acid
ID PERVASIVE DEVELOPMENTAL DISORDERS; CENTRAL-NERVOUS-SYSTEM;
PITUITARY-ADRENAL HORMONES; NITRIC-OXIDE PRODUCTION; ELEVATED PLUS-MAZE;
SEX-DIFFERENCES; ESTROUS-CYCLE; FEMALE RATS; BRAIN-STEM;
PERITONEAL-MACROPHAGES
AB Autism is a severe behavioral disorder characterized by pervasive impairments in social interactions, deficits in verbal and non-verbal communication, and stereotyped behaviors, with a four times higher incidence in boys than in girls. The core symptoms are frequently accompanied by a spectrum of neurobehavioral and immunological derangements, including: aberrant sensitivity to sensory stimulation, anxiety, and decreased cellular immune capacity. Recently, a new potential rodent model of autism induced by prenatal exposure to valproic acid (VPA rats) has been proposed. In order to determine if gender has an influence on alterations observed in VPA rats, male and female rats have been evaluated in a battery of behavioral, immunological, and endocrinological tests. A plethora of aberrations has been found in male VPA rats: lower sensitivity to pain, increased repetitive/stereotypic-like activity, higher anxiety, decreased level of social interaction, increased basal level of corticosterone, decreased weight of the thymus, decreased splenocytes proliferative response to concanavaline A, lower IFN-gamma/IL-10 ratio, and increased production of NO by peritoneal macrophages. Female VPA rats exhibited only increased repetitive/stereotypic-like activity and decreased IFN-gamma/IL-10 ratio. Sexual dimorphism characteristics for measured parameters have been observed in both groups of animals, except social interaction in VPA rats. Our results confirm existence of similarities between the observed pattern of aberrations in VPA rats and features of disturbed behavior and immune function in autistic patients, and suggest that they are gender-specific, which is intriguing in light of disproportion in boys to girls ratio in autism. (c) 2008 Elsevier Ltd. All rights reserved.
C1 [Schneider, Tomasz; Roman, Adam; Basta-Kaim, Agnieszka; Kubera, Marta; Budziszewska, Bogustawa; Schneider, Karolina; Przewtocki, Ryszard] Polish Acad Sci, Inst Pharmacol, PL-31343 Krakow, Poland.
RP Schneider, T (reprint author), Kings Coll London, Inst Psychiat P049, De Crespigny Pk, London SE5 8AF, England.
EM Tomasz.Schneider@iop.kcl.ac.uk
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NR 109
TC 54
Z9 61
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0306-4530
J9 PSYCHONEUROENDOCRINO
JI Psychoneuroendocrinology
PD JUL
PY 2008
VL 33
IS 6
BP 728
EP 740
DI 10.1016/j.psyneuen.2008.02.011
PG 13
WC Endocrinology & Metabolism; Neurosciences; Psychiatry
SC Endocrinology & Metabolism; Neurosciences & Neurology; Psychiatry
GA 336HZ
UT WOS:000258356200004
PM 18396377
ER
PT J
AU Birmingham, E
Bischof, WF
Kingstone, A
AF Birmingham, Elina
Bischof, Walter F.
Kingstone, Alan
TI Social attention and real-world scenes: The roles of action, competition
and social content
SO QUARTERLY JOURNAL OF EXPERIMENTAL PSYCHOLOGY
LA English
DT Article
ID EYE-MOVEMENTS; EARLY RECOGNITION; AUTISM; GAZE; PERCEPTION; INFANTS;
SHIFTS; DIRECTION; CHILDREN; STIMULI
AB The present study examined how social attention is influenced by social content and the presence of items that are available for attention. We monitored observers' eye movements while they freely viewed real-world social scenes containing either 1 or 3 people situated among a variety of objects. Building from the work of Yarbus (1965/1967) we hypothesized that observers would demonstrate a preferential bias to fixate the eyes of the people in the scene, although other items would also receive attention. In addition, we hypothesized that fixations to the eyes would increase as the social content (i.e., number of people) increased. Both hypotheses were supported by the data, and we also found that the level of activity in the scene influenced attention to eyes when social content was high. The present results provide support for the notion that the eyes are selected by others in order to extract social information. Our study also suggests a simple and surreptitious methodology for studying social attention to real-world stimuli in a range of populations, such as those with autism spectrum disorders.
C1 [Birmingham, Elina] Univ British Columbia, Dept Psychol, Vancouver, BC V6T 1Z4, Canada.
[Bischof, Walter F.] Univ Alberta, Edmonton, AB, Canada.
RP Birmingham, E (reprint author), Univ British Columbia, Dept Psychol, 2136 W Mall, Vancouver, BC V6T 1Z4, Canada.
EM ebirmingham2@yahoo.ca
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NR 33
TC 46
Z9 46
PU PSYCHOLOGY PRESS
PI HOVE
PA 27 CHURCH RD, HOVE BN3 2FA, EAST SUSSEX, ENGLAND
SN 1747-0218
J9 Q J EXP PSYCHOL
JI Q. J. Exp. Psychol.
PD JUL
PY 2008
VL 61
IS 7
BP 986
EP 998
DI 10.1080/17470210701410375
PG 13
WC Psychology, Biological; Physiology; Psychology; Psychology, Experimental
SC Psychology; Physiology
GA 318RY
UT WOS:000257111100004
PM 18938281
ER
PT J
AU Bebko, JM
Schroeder, JH
Weiss, JA
Wells, K
Mcfee, K
Goldstein, GM
AF Bebko, James M.
Schroeder, Jessica H.
Weiss, Jonathan A.
Wells, Kerry
McFee, Kristen
Goldstein, Gayle M.
TI The face of Autism research as reflected in the IMFAR looking glass
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Review
DE Autism; Trends; Young children; Autism spectrum disorders; Pervasive
developmental disorders
AB Recent overviews of autism research have yielded it number of suggestions, including: additional research with very young, and with lower functioning samples, and renewed emphasis on appropriate comparison/control groups. We reviewed the abstracts from a major autism conference (IMFAR) from 2004 to 2006 to examine these trends. We found an increase in the proportion of studies with preschool or infant participants. However, there was a decrease in studies using lower functioning samples, and an increase in Studies using Mixed samples. The use of control groups generally decreased, and the use of cognitively impaired comparison groups remains low. We also found the use of the more generic term, ASD, versus DSM categories as sample descriptors increasing at IMFAR, The potential impact of these trends on limiting the generalizability of results is discussed. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Bebko, James M.; Schroeder, Jessica H.; Weiss, Jonathan A.; Wells, Kerry; McFee, Kristen; Goldstein, Gayle M.] York Univ, Toronto, ON M3J 1P3, Canada.
RP Bebko, JM (reprint author), York Univ, Toronto, ON M3J 1P3, Canada.
EM bebkolab@yorku.ca
CR Autism Spectrum Disorders, AUTISM SPECTRUM DISO
Fombonne E, 2005, J APPL RES INTELLECT, V18, P281, DOI 10.1111/j.1468-3148.2005.00266.x
*INT SOC AUT RES, 2004, INT M AUT RES PROGR
*INT SOC AUT RES, 2006, INT M AUT RES PROGR
*INT SOC AUT RES, 2005, INT M AUT RES PROGR
La Malfa G, 2004, J INTELL DISABIL RES, V48, P262, DOI 10.1111/j.1365-2788.2003.00567.x
Mottron L, 2004, J AUTISM DEV DISORD, V34, P19, DOI 10.1023/B:JADD.0000018070.88380.83
Volkmar FR, 2004, J CHILD PSYCHOL PSYC, V45, P135, DOI 10.1046/j.0021-9630.2003.00317.x
WHAT ARE AUTISM SPEC
NR 9
TC 2
Z9 2
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 385
EP 394
DI 10.1016/j.rasd.2007.06.005
PG 10
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900001
ER
PT J
AU Machalicek, W
O'Reilly, MF
Beretvas, N
Sigafoos, J
Lancioni, G
Sorrells, A
Lang, R
Rispoli, M
AF Machalicek, Wendy
O'Reilly, Mark F.
Beretvas, Natasha
Sigafoos, Jeff
Lancioni, Guilio
Sorrells, Audrey
Lang, Russell
Rispoli, Mandy
TI A review of school-based instructional interventions for students with
autism spectrum disorders
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Review
DE Instruction; School; Autism spectrum disorders
ID SOCIAL-INTERACTION SKILLS; DEVELOPMENTAL-DISABILITIES;
COMMUNICATION-SKILLS; YOUNG-CHILDREN; TEACHING-CHILDREN;
GENERAL-EDUCATION; SELF-MANAGEMENT; PLAY SKILLS; PERSPECTIVE; BEHAVIORS
AB This review evaluates school-based instructional research for students with autism spectrum disorders (ASD). Electronic database searches identified 45 studies (n = 118 participants) published between 1995 and 2005. These studies were classified into five curricular areas: (a) academic skills, (b) communication skills, (c) functional life skills, (d) play, and (c) social skills. The results of the reviewed studies indicated effective instructional methods and several trends across curricular areas, Fewer than half of the studies (n = 20) assessed the generalization of skills to different setting,. and stimuli. A minority Of the studies (n = 19) assessed the maintenance of skills. Very few studies reported student characteristics, such as cultural background. Additionally, many studies (lid not address the social validity of instructional interventions or have used inadequate procedures to judge the perceptions of stakeholders. In light of these findings, several relevant areas for future research tire proposed. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Machalicek, Wendy; O'Reilly, Mark F.; Beretvas, Natasha; Sorrells, Audrey; Lang, Russell; Rispoli, Mandy] Univ Texas Austin, Dept Special Educ, Austin, TX 78712 USA.
[Sigafoos, Jeff] Univ Tasmania, Hobart, Tas 7001, Australia.
[Lancioni, Guilio] Univ Bari, Bari, Italy.
RP O'Reilly, MF (reprint author), Univ Texas Austin, Dept Special Educ, 1 Univ Stn,D5300, Austin, TX 78712 USA.
EM markoreilly@mail.utexas.edu
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NR 70
TC 27
Z9 27
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 395
EP 416
DI 10.1016/j.rasd.2007.07.001
PG 22
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900002
ER
PT J
AU Schoen, SA
Miller, LJ
Brett-Green, B
Hepburn, SL
AF Schoen, Sarah A.
Miller, Lucy Jane
Brett-Green, Barbara
Hepburn, Susan L.
TI Psychophysiology of children with autism spectrum disorder
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Psychophysiology; Sensory processing; Asperger Syndrome; High
functioning autism
ID FRAGILE-X-SYNDROME; ASPERGER-SYNDROME; ELECTRODERMAL ACTIVITY;
SENSORY-MODULATION; DEVELOPMENTAL DISORDERS; AUTONOMIC RESPONSES;
ORIENTING RESPONSE; INFANTILE-AUTISM; VISUAL-STIMULI; SCHIZOPHRENIA
AB This study (1) explored the feasibility Of using electrodermal activity (EDA) to characterize the arousal and sensory reactivity of children with high functioning autism (HFA) and Asperger's Syndrome (AS), (2) determined the reliability of electrodermal measures and (3) described the variability of EDA in this sample, Forty children with HFA and AS participated. All participants received a diagnostic psychological assessment and a physiological evaluation. Fourteen participated in the retest study on the physiological measures. Results indicated psychophysiologic testing was feasible with this sample. Seventy-three percent of the variables had reliability coefficients greater than .33, with a median variable reliability of .45. No significant differences were detected between HFA and AS groups, Visual inspection of skin conductance level (SCL) suggested two patterns: (1) high SCL (high arousal), with higher EDA magnitudes, faster latencies and slower habituation and (2) low SCL (low arousal), with lower EDA magnitudes, slower latencies and faster habituation, The presence of two EDA patterns applied equally when medications were eliminated. The previous inconsistency in studies of EDA in children with HFA and AS may be due to the presence of a high arousal groups and a low arousal group. Hence, this population should not be assumed to be homogeneous. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Schoen, Sarah A.; Miller, Lucy Jane; Brett-Green, Barbara] SPD Fdn, Greenwood Village, CO USA.
[Schoen, Sarah A.; Miller, Lucy Jane; Brett-Green, Barbara; Hepburn, Susan L.] Univ Colorado, Denver & Hlth Sci Ctr, Boulder, CO 80309 USA.
RP Schoen, SA (reprint author), SPD Fdn, 5655 S Yosemite St,Suite 305, Greenwood Village, CO USA.
EM schoen@SPDFoundation.net
CR Baranek GT, 1997, OCCUP THER J RES, V17, P173
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NR 63
TC 21
Z9 21
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 417
EP 429
DI 10.1016/j.rasd.2007.09.002
PG 13
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900003
ER
PT J
AU Angermeier, K
Schlosser, RW
Luiselli, JK
Harrington, C
Carter, B
AF Angermeier, Katie
Schlosser, Ralf W.
Luiselli, James K.
Harrington, Caroline
Carter, Beth
TI Effects of iconicity on requesting with the Picture Exchange
Communication System in children with autism spectrum disorder
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Augmentative and alternative communication; Autism spectrum disorders;
Iconicity; Picture Exchange Communication System; Requesting; Symbol
selection
ID ACQUISITION; INTERVENTIONS; TRANSPARENCY; PRESCHOOLERS; DISABILITIES;
INDIVIDUALS
AB Research on graphic symbol learning suggests that symbols with a greater visual resemblance to their referents (greater iconicity) are more easily learned. The iconicity hypothesis has not yet been explored within the intervention protocol of the Picture Exchange Communication System (PECS). Within the PECS protocol, participants do not point to a symbol but exchange the symbol for an object. The purpose of this study was to examine whether children learn to request more readily with PECS when the symbols involved are highly iconic versus symbols that are low in iconicity. An adapted alternating treatments design combined with a multiple baseline design across subjects was used to evaluate the effectiveness and efficiency of symbol learning under two conditions: high iconicity and low iconicity. Four students with A autism or pervasive developmental disorders between the ages of six and nine years participated. Results indicated that students learned to request desired objects under both conditions, lending further support for the effectiveness of PECS. There was little to no difference, however, in the effectiveness and efficiency of requesting between the two conditions during Phases I and II of PECS training. Thus learners do not benefit A from symbols that bear more resemblance with their referents during the first two phases of PECS instruction. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Schlosser, Ralf W.; Carter, Beth] Northeastern Univ, Boston, MA 02115 USA.
RP Schlosser, RW (reprint author), Northeastern Univ, Boston, MA 02115 USA.
EM r.schlosser@neu.edu
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Beukelman D., 2005, AUGMENTATIVE ALTERNA, V3rd
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NR 29
TC 15
Z9 15
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 430
EP 446
DI 10.1016/j.rasd.2007.09.004
PG 17
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900004
ER
PT J
AU Wallace, GL
Happe, F
AF Wallace, Gregory L.
Happe, Francesca
TI Time perception in autism spectrum disorders
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism; Asperger disorder; Temporal cognition; Duration judgment;
Perception; Time perception
ID ATTENTION-DEFICIT/HYPERACTIVITY-DISORDER; DEFICIT HYPERACTIVITY
DISORDER; ENHANCED DISCRIMINATION; BEHAVIORAL-INHIBITION; CHILDREN;
ADHD; MEMORY; SAVANT; ADULTS; REPRODUCTION
AB Duration judgment has not been comprehensively examined in autism spectrum disorders (ASD), despite reports of perceptual idiosyncrasies in these individuals. Time estimation, production, and reproduction were tested in 25 individuals with ASD and 25 controls matched group-wise on age and IQ. Individuals with ASD performed comparably to matched controls in time estimation and production, but showed a tendency to outperform controls oil a task of time reproduction, Time perception of large units is intact for children and adolescents with ASD. Time reproduction may represent a cognitive asset in ASD, likely reflecting strengths in eidetic-type imagery in which a pure recording of a stimulus is less affected by top-down modulation. published by Elsevier Ltd.
C1 [Wallace, Gregory L.; Happe, Francesca] Kings Coll London, Inst Psychiat, Social Genet & Dev Psychiat Ctr, London WC2R 2LS, England.
RP Wallace, GL (reprint author), NIMH, Lab Brain & Cognit, 10 Ctr Dr,Room 4C104,MSC 1366, Bethesda, MD 20892 USA.
EM gregwallace@mail.nih.gov
RI Happe, Francesca/D-5544-2012
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
Barkley RA, 2001, NEUROPSYCHOLOGY, V15, P351, DOI 10.1037//0894-4105.15.3.351
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GOLOMBOK S, 1992, MANUAL WECHSLER INTE
Happe F, 2006, J AUTISM DEV DISORD, V36, P5, DOI 10.1007/s10803-005-0039-0
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NR 34
TC 16
Z9 16
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 447
EP 455
DI 10.1016/j.rasd.2007.09.005
PG 9
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900005
ER
PT J
AU Perra, O
Williams, JHG
Whiten, A
Fraser, L
Benzie, H
Perrett, DI
AF Perra, Oliver
Williams, Justin H. G.
Whiten, Andrew
Fraser, Lesley
Benzie, Helen
Perrett, David I.
TI Imitation and 'theory of mind' competencies in discrimination of autism
from other neurodevelopmental disorders
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Imitation; Autistic Spectrum Disorder; Theory of mind; Discriminant
function analysis
ID PERVASIVE DEVELOPMENTAL DISORDERS; SPECTRUM DISORDERS; YOUNG-CHILDREN;
QUESTIONNAIRE; STANDARD
AB Several studies have reported imitative deficits in autism spectrum disorder (ASD). However, it is still debated if imitative deficits are specific to ASD or shared with clinical groups with similar mental impairment and motor difficulties. We investigated whether imitative tasks can be used to discriminate ASD children from typically developing children (TD) and children with general developmental delay (GDD). We applied discriminant function analyses to the performance of these groups on three imitation tasks and tests of dexterity, motor planning, verbal skills, theory of mind (ToM). Analyses revealed two significant dimensions. The first represented impairment of dexterity and verbal ability, and discriminated TD from GDD children. Once these differences were accounted for, differences in ToM and the three imitation tasks accounted for a significant proportion of the remaining intergroup variance and discriminated the ASD group from other groups. Further analyses revealed that inclusion of imitative tasks increased the specificity and sensitivity of ASD classification and that imitative tasks considered alone were able to reliably discriminate ASD, TD and GDD. The results suggest that imitation and theory of mind impairment in autism may stem from a common domain of origin separate from general cognitive and motor skill. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Perra, Oliver] Cardiff Univ, Sch Psychol, Cardiff CF10 3AT, S Glam, Wales.
[Williams, Justin H. G.; Benzie, Helen] Univ Aberdeen, Royal Aberdeen Childrens Hosp, Aberdeen AB9 1FX, Scotland.
[Whiten, Andrew; Fraser, Lesley; Perrett, David I.] Univ St Andrews, Sch Psychol, St Andrews KY16 9AJ, Fife, Scotland.
RP Perra, O (reprint author), Cardiff Univ, Sch Psychol, Tower Block, Cardiff CF10 3AT, S Glam, Wales.
EM perrao@cardiff.ac.uk
CR Bekkering H, 2000, Q J EXP PSYCHOL-A, V53, P153, DOI 10.1080/027249800390718
BERGERSE.FJ, 1965, AUST J BIOL SCI, V18, P1
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Toth K, 2007, J AUTISM DEV DISORD, V37, P145, DOI 10.1007/s10803-006-0336-2
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NR 29
TC 11
Z9 11
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 456
EP 468
DI 10.1016/j.rasd.2007.09.007
PG 13
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900006
ER
PT J
AU Cunningham, AB
Schreibman, L
AF Cunningham, Allison B.
Schreibman, Laura
TI Stereotypy in autism: The importance of function
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism; Stereotypy; Self-stimulatory behavior; Functional analysis;
Treatment
ID SELF-STIMULATORY-BEHAVIOR; REPETITIVE BEHAVIOR; MATCHED STIMULATION;
SENSORY EXTINCTION; CHILDREN; REINFORCEMENT; OVERCORRECTION; ANNOTATION;
PREDICTORS
AB We argue for the utility of a functional definition of stereotypy based on evidence of both sensory automatic and socially mediated reinforcement contingencies in the occurrence of stereotypy in children with autism. A predetermined sensory function of stereotypy is often invoked in the behavioral literature and the term "self-stimulatory behavior" is commonly misused as interchangeable with "stereotypy." We discuss evidence for a variety of potential functional properties of stereotypy. Diagnostic definitions are reviewed and support for both sensory and social functions is outlined, We argue that stereotypies should be described and categorized according to their function, rather than form. Furthermore, treatment decisions should lie based on a functional interpretation of stereotypy, which acknowledges its operant and heterogeneous quality in autism. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Cunningham, Allison B.; Schreibman, Laura] Univ Calif San Diego, Dept Psychol, La Jolla, CA 92093 USA.
RP Cunningham, AB (reprint author), Univ Calif San Diego, Dept Psychol, 9500 Gilman Dr,MC 0109, La Jolla, CA 92093 USA.
EM abcunnin@ucsd.edu
CR Ahearn WH, 2005, J APPL BEHAV ANAL, V38, P247, DOI 10.1901/jaba.2005.36-04
Ahearn WH, 2003, J APPL BEHAV ANAL, V36, P439, DOI 10.1901/jaba.2003.36-439
American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Bodfish JW, 2000, J AUTISM DEV DISORD, V30, P237, DOI 10.1023/A:1005596502855
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NR 43
TC 48
Z9 48
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 469
EP 479
DI 10.1016/j.rasd.2007.09.006
PG 11
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900007
ER
PT J
AU Brown, JL
Krantz, PJ
McClannahan, LE
Poulson, CL
AF Brown, John L.
Krantz, Patricia J.
McClannahan, Lynn E.
Poulson, Claire L.
TI Using script fading to promote natural environment stimulus control of
verbal interactions among youths with autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism; Script fading; Natural environments; Stimulus control;
Conversation
ID SOCIAL-INTERACTION SKILLS; TEACHING-CHILDREN
AB Script fading was used to teach three youths with autism to initiate and sustain verbal interactions under stimulus control consistent with the natural environment. The youths learned to engage in verbal interactions during simulated shopping trips and during visits to community stores. The effectiveness of script fading was examined using a multiple-baseline-across-settings experimental design. During pre-test trips to community stores and during baseline sessions, all three youths demonstrated near zero rates of verbal interactions. With the introduction of the script-fading procedure, all three youths successfully learned to use the scripted statements in conversations during a series of simulated shopping trips. As the scripts were faded, from last word to first word, rates of unscripted statements systematically increased. All three participants also demonstrated generalization of their newly acquired conversation skills to untrained stimuli. In addition, all three youths demonstrated generalization of their conversation skills during community shopping trips to local retail stores. These results suggest that the script-fading procedure brought verbal interactions under the stimulus control of environmental, as opposed to teacher-controlled stimuli. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Brown, John L.; Krantz, Patricia J.; McClannahan, Lynn E.] Princeton Child Dev Inst, Princeton, NJ USA.
[Brown, John L.; Poulson, Claire L.] CUNY, Grad Ctr, New York, NY USA.
[Brown, John L.; Poulson, Claire L.] CUNY Queens Coll, New York, NY USA.
RP Brown, JL (reprint author), REED Acad, 85 Summit Ave, Garfield, NJ 07026 USA.
EM johnlloydbrown@yahoo.com
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
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NR 23
TC 9
Z9 9
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 480
EP 497
DI 10.1016/j.rasd.2007.08.006
PG 18
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900008
ER
PT J
AU Ludlow, AK
Wilkins, AJ
Heaton, P
AF Ludlow, A. K.
Wilkins, A. J.
Heaton, P.
TI Colored overlays enhance visual perceptual performance in children with
autism spectrum disorders
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Autism spectrum disorders; Visual stress; Color filters; Magnocellular
deficits; Cortical hyperexcitability
ID MEARES-IRLEN-SYNDROME; MIGRAINE; DYSLEXIA; FILTERS; SENSITIVITY;
CLASSROOM; TARGETS; READ
AB Children with autism spectrum disorders (ASD), together with controls matched for age and ability participated in three experiments that assessed the therapeutic benefit of colored overlays. The findings from the first experiment showed that a significantly greater proportion of children with ASD, than controls, increased reading speed when using a colored overlay. This finding was replicated in the second experiment which also showed that therapeutic benefits were only observed when participants were instructed to select colors that improved textual clarity and not when colors were selected on the basis simply of preference. In the final experiment, children were required to discriminate between pictorially presented objects with and without overlays self-selected for improvements in clarity. Participants with ASD, both with and without concurrent intellectual impairment, showed significant gains in performance when using an overlay. The beneficial effects of color overlays and the implications of these results for current neuropsychological models of ASD are discussed. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Ludlow, A. K.] Anglia Ruskin Univ, Dept Psychol, Cambridge CB1 1PT, England.
[Wilkins, A. J.] Univ Essex, Dept Psychol, Colchester CO4 3SQ, Essex, England.
[Heaton, P.] Univ London, Univ London Goldsmiths Coll, Dept Psychol, London SE14 6NW, England.
RP Ludlow, AK (reprint author), Anglia Ruskin Univ, Dept Psychol, East Rd, Cambridge CB1 1PT, England.
EM a.ludlow@anglia.ac.uk
CR ATTWOOD T, 1994, WHY DOES CHRIS DO UN
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Williams M C, 1992, J Am Optom Assoc, V63, P411
WRIGHT BN, J NEUROLOGY IN PRESS
NR 66
TC 2
Z9 2
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 498
EP 515
DI 10.1016/j.rasd.2007.10.001
PG 18
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900009
ER
PT J
AU Matson, JL
Gonzalez, ML
Wilkins, J
Rivet, TT
AF Matson, Johnny L.
Gonzalez, Melissa L.
Wilkins, Jonathan
Rivet, Tessa T.
TI Reliability of the Autism Spectrum Disorder-Diagnostic For Children
(ASD-DC)
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Asperger's Syndrome; PDD-NOS; Autism; Test; ASD-DC
ID PERVASIVE DEVELOPMENTAL DISORDER; HIGH-FUNCTIONING AUTISM;
OF-THE-LITERATURE; DIFFERENTIAL-DIAGNOSIS; BEHAVIORAL INTERVENTION;
INTERRATER RELIABILITY; YOUNG-CHILDREN; DISABILITIES; ISSUES; SKILLS
AB The reliability of a new scale to assess Autistic Disorder, Pervasive Developmental Disorder, Not Otherwise Specified (PI)D-NOS), and Asperger's Disorder in children was examined. Parents or other caregivers rated symptoms of 207 children between 2 and 16 years of age. The scale, which had 40 items in the final version, correlated highly with DSM-IV-TR and ICD-10 criteria and proved to have good inter-rater reliability and, excellent test-retest, and internal consistency reliability. These data are considered to have clinical utility given the need to establish data-based distinctions between these three subtypes of ASD. Furthermore, the measure is part of a more extensive battery measuring comorbid psychopathology and challenging behaviors. Implications for future research of this comprehensive assessment battery are discussed. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Matson, Johnny L.; Gonzalez, Melissa L.; Wilkins, Jonathan; Rivet, Tessa T.] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, 236 Audubon Hall, Baton Rouge, LA 70803 USA.
EM johnmatson@aol.com
CR American Psychiatric Association, 2000, DIAGN STAT MAN MENT
Baird G, 2006, LANCET, V368, P210, DOI 10.1016/S0140-6736(06)69041-7
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World Health Organization, 1992, INT CLASS DIS
NR 48
TC 43
Z9 43
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 533
EP 545
DI 10.1016/j.rasd.2007.11.001
PG 11
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900011
ER
PT J
AU Goin-Kochel, RP
Peters, SU
Treadwell-Deering, D
AF Goin-Kochel, Robin P.
Peters, Sarika U.
Treadwell-Deering, Diane
TI Parental reports on the prevalence of co-occurring intellectual
disability among children with autism spectrum disorders
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Mental retardation; Cognition; IQ; Diagnosis; Assessment; PDD-NOS
ID PERVASIVE DEVELOPMENTAL DISORDERS; MENTAL-RETARDATION;
PRESCHOOL-CHILDREN; DISCLOSURE; DIAGNOSIS; COMMUNICATION; DETERMINANTS;
SATISFACTION; STABILITY; LEVEL
AB Limited evidence suggests that the prevalence of mental retardation (MR) - now called "intellectual disability" (ID) - in ASD ranges between 25.8% and 58.5%, with co-occurrence in 66-70% of children who meet strict criteria for autism. However, parents and professionals differ significantly in their understanding of or opinions about intellectual functioning among children with ASD. The current study analyzed archival data to calculate the prevalence of parent-reported MR among children with ASD (N = 498). Collectively, 9.2% of families affirmed MR; when analyzed by type of ASD, the rates for those with autism and PDD-NOS were 12.6% and 7.3%, respectively. A variety of explanations for these low rates are posited alongside implications for clinical practice and families' receipt of services. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Goin-Kochel, Robin P.] Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
[Peters, Sarika U.] Baylor Coll Med, Dept Pediat, Houston, TX 77030 USA.
[Treadwell-Deering, Diane] Baylor Coll Med, Dept Psychiat & Behav Sci, Houston, TX 77030 USA.
RP Goin-Kochel, RP (reprint author), Texas Childrens Hosp, 6621 Fannin St,CC1560, Houston, TX 77030 USA.
EM kochel@bcm.tmc.edu
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NR 36
TC 3
Z9 3
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 546
EP 556
DI 10.1016/j.rasd.2007.11.002
PG 11
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900012
ER
PT J
AU Pineda, JA
Brang, D
Hecht, E
Edwards, L
Carey, S
Bacon, M
Futagaki, C
Suk, D
Tom, J
Birnbaum, C
Rork, A
AF Pineda, J. A.
Brang, D.
Hecht, E.
Edwards, L.
Carey, S.
Bacon, M.
Futagaki, C.
Suk, D.
Tom, J.
Birnbaum, C.
Rork, A.
TI Positive behavioral and electrophysiological changes following
neurofeedback training in children with autism
SO RESEARCH IN AUTISM SPECTRUM DISORDERS
LA English
DT Article
DE Mu rhythm; Mirror neurons; Imitation; TOVA; ATEC
ID MIRROR NEURON DYSFUNCTION; INFERIOR FRONTAL-CORTEX; SPECTRUM DISORDERS;
MU-RHYTHM; DEVELOPMENTAL DISORDERS; CORTICAL ACTIVATION; ACTION
RECOGNITION; BIOLOGICAL MOTION; PREMOTOR CORTEX; MOTOR IMAGERY
AB Two electrophysiological studies tested the hypothesis that operant conditioning of mu rhythms via neuro-feedback training can renormalize mu suppression,an index of mirror neuron activity, and improve behavior in children diagnosed with autism spectrum disorders (ASD). In Study 1, eight high-functioning ASD participants were assigned to placebo or experimental groups before 10 weeks of training of the mu frequency band (8-13 Hz). Following training, experimental participants showed decreased mu power and coherence, increased sustained attention ability, and improved scores on subscales of the ATEC compared to the placebo group. Both groups showed improvement in imitation ability. In Study 2, 19 high-functioning ASD children underwent a similar procedure with verified diagnoses, a modified double-blind protocol, and training of the high mu band (10-13 Hz). The results showed decreases in amplitude but increases in phase coherence in mu rhythms and normalization of mu rhythm suppression in experimental participants compared to placebo. Furthermore, like Study 1, participants showed improvements in sustained attention and in ATEC scores but no improvements in imitation following training. This suggests that training of the mu rhythm can be effective in producing changes in EEG and behavior in high-functioning ASD children, but does not affect imitation behavior per se. (c) 2007 Elsevier Ltd. All rights reserved.
C1 [Pineda, J. A.; Brang, D.; Hecht, E.; Edwards, L.; Carey, S.; Bacon, M.; Futagaki, C.; Suk, D.; Tom, J.; Birnbaum, C.; Rork, A.] Univ Calif San Diego, Dept Cognit Sci, La Jolla, CA 92093 USA.
[Pineda, J. A.] Univ Calif San Diego, Dept Neurosci, La Jolla, CA 92093 USA.
RP Pineda, JA (reprint author), Univ Calif San Diego, Dept Cognit Sci, 9500 Gilman Dr, La Jolla, CA 92093 USA.
EM pineda@cogsci.ucsd.edu
RI Brang, David/G-5326-2012
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NR 66
TC 30
Z9 30
PU ELSEVIER SCI LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND
SN 1750-9467
J9 RES AUTISM SPECT DIS
JI Res. Autism Spectr. Disord.
PD JUL-SEP
PY 2008
VL 2
IS 3
BP 557
EP 581
DI 10.1016/j.rasd.2007.12.003
PG 25
WC Education, Special; Psychology, Developmental; Psychiatry;
Rehabilitation
SC Education & Educational Research; Psychology; Psychiatry; Rehabilitation
GA 408DK
UT WOS:000263413900013
ER
PT J
AU Matson, JL
Dempsey, T
LoVullo, SV
Wilkins, J
AF Matson, Johnny L.
Dempsey, Timothy
LoVullo, Santino V.
Wilkins, Jonathan
TI The effects of intellectual functioning on the range of core symptoms of
autism spectrum disorders
SO RESEARCH IN DEVELOPMENTAL DISABILITIES
LA English
DT Article
DE IQ; adults; behavioral characteristics
ID SEVERE RETARDATION MESSIER; OF-THE-LITERATURE; ADAPTIVE-BEHAVIOR;
YOUNG-CHILDREN; DIFFERENTIAL-DIAGNOSIS; SOCIAL-SKILLS; CHALLENGING
BEHAVIORS; MENTAL-RETARDATION; MATSON EVALUATION; AGE
AB Autism spectrum disorders (ASD) are a class of conditions categorized by communication problems, ritualistic behaviors, and inappropriate social behaviors. While there is much evidence to support a genetic link for ASD, an identified genetic marker remains elusive. As such, practitioners place considerable emphasis on traditional measures of intelligence and adaptive behavior to aid in diagnosis. Despite the fact that these measures are commonplace, little research has been conducted to shed light on whether deficits in intellectual functioning affect the range of core symptoms for ASD. This study represents a first attempt to determine whether level of IQ has an effect on the expression of ASD symptoms in adults with intellectual disability (ID). Three hundred and six adults, 151 with both ASD and ID and 155 with ID alone, were evaluated with respect to the nature and extent of their ASD symptoms and intellectual functioning. Individuals with autism displayed a higher number of symptoms than those with pervasive developmental disorder-not otherwise specified (PDD-NOS) on all three domains of impairment (social, communication, repetitive behaviors). As expected, persons with ID alone evinced far fewer symptoms than both these groups. IQ level was found to be a moderator for expression of ASD symptoms for the entire sample but not for the autism group. (C) 2007 Elsevier Ltd. All rights reserved.
C1 Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, Baton Rouge, LA 70803 USA.
EM Johnmatson@aol.com
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SPARROW SS, 1985, J PEDIATR PSYCHOL, V10, P215, DOI 10.1093/jpepsy/10.2.215
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World Health Organization, 1992, INT CLASS DIS
NR 51
TC 77
Z9 77
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0891-4222
J9 RES DEV DISABIL
JI Res. Dev. Disabil.
PD JUL-AUG
PY 2008
VL 29
IS 4
BP 341
EP 350
DI 10.1016/j.ridd.2007.06.006
PG 10
WC Education, Special; Rehabilitation
SC Education & Educational Research; Rehabilitation
GA 314OU
UT WOS:000256819500005
PM 17646082
ER
PT J
AU Alvarez, LAV
Cruz, LP
Thompson, GG
Olvera, FD
AF Vargas Alvarez, Luis Alberto
Cruz, Lino Palacios
Thompson, Guillermo Gonzalez
Olvera, Francisco de la Pena
TI Obsessive-compulsive in child and adolescents: An update. Part two
SO SALUD MENTAL
LA Spanish
DT Article
DE obsessive-compulsive disorder; children and adolescents; comorbidity;
assessment; treatment
ID ATTENTION-DEFICIT/HYPERACTIVITY DISORDER; PLACEBO-CONTROLLED TRIAL;
SPECTRUM DISORDERS; BIPOLAR COMORBIDITY; REFERRED CHILDREN;
DOUBLE-BLIND; MULTICENTER; FLUOXETINE; FAMILY; ONSET
AB During the last years obsessive-compulsive disorder (OCD) has been reported with increased prevalence in pediatric population; this is due to the development of more specific assessment methods. This evolution in the evaluation tools has given rise to the possibility of characterizing OCD presentation in children and adolescents. In childhood, OCD is a chronic and distressing disorder that can lead to severe impairments in social, academic and family functioning. Current diagnosis criteria for pediatric OCD are the same than those used in adults. During all life span, obsessive and compulsive symptoms are necessary to establish the presence of the disorder. There are several different clinical manifestations among age groups, different evolution among children, adolescents and adults; all these represent a diagnostic and therapeutic challenge for the clinician.
Several classifications incorporate pediatric OCD, especially those related to the familiar presentation form and patterns of comorbidity, mainly with tics disorders. At least 50% of children and adolescents with Gilles de la Tourette syndrome develop obsessive-compulsive symptoms or OCD in adulthood and almost a half of early-onset OCD subjects have a tics history. These findings support the notion that tics disorders are the comorbidity more closely related with early-onset OCD, giving elements to consider this association as a specific pediatric OCD subtype.
In this age group population, comorbidity has been reported as high as in adulthood; some diagnoses are especially prevalent during childhood and others during adolescence. On the whole, anxiety disorders are frequent with OCD, generalized anxiety disorder, panic attack, social phobia and anxiety separation disorder.
Comorbidity related with affective disorders is high too. The OCD association with major depressive disorder (MDD) in childhood is low but increases in adolescence; MDD reaches similar adult comorbidity rates in adolescence. Higher comorbidity prevalence of MDD has been found more related to the duration of OCD-illness than early-onset.
Bipolar disorder (BD) is another frequent comorbid entity with great clinical relevance. When BD is the main diagnosis, comorbidity with OCD shows a prevalence of 16%; when OCD is the main diagnosis, comorbidity with BID shows a prevalence of 44%, showing an unidirectional relation. Some studies have shown even higher comorbidity prevalence of BD when considering bipolar spectra dimension as hypomania and cyclothymic disorder (30% and 50%, respectively) in OCD samples. Adults with OCD and BD comorbidity have more frequent episodic form, a greater number of concurrent mayor depressive episodes and a higher rate of religious or sexual obsessions. Adults with OCD without BID comorbidity show more rituals and compulsions.
A recent study in pediatric population with BD and OCD found that BD type II was the must common related diagnosis, when age was considered, subjects with bipolar disorder resulted to have an earlier onset of OCD.
Other comorbid diagnoses frequently reported in this early-onset OCD population are externalizing disorders as attention deficit hyperactivity disorder (ADHD) and oppositional defiant disorder (ODD). Children and adolescents with OCD have high rates of commorbid ADHD; this co-occurrence seems to be bidirectional.
There is a consistent preponderance of males in most epidemiological studies. The onset of ADHD preceded the onset of OCD and the onset of OCD was earlier when ADHD was comorbid. Children with OCD plus ADHD compared with peers with OCD without ADHD show higher attentional and social problems, as well as aggressive high scores. ADHD is a risk factor for ODD.
A valid and reliable clinical interview is needed to establish differential diagnosis among OCD and other compulsive behaviors and intrusive thoughts present in disorders like anorexia nervosa, body dysmorphic disorder, hypochondrias, tics disorders and impulse control disorders. All these categories have been considered as part of the obsessive-compulsive disorders spectrum.
It is important to establish the difference between obsessions with poor insight common in early-onset OCD and overvalued ideas or delusions. Pervasive disorders as autism and Asperger syndrome frequently show stereotyped behaviors which may be considered as obsessive-compulsive symptoms.
The diagnostic evaluation of children and adolescents with OCD includes a careful assessment and review of current and post obsessive-compulsive symptoms and comorbid conditions. This evaluation requires interviewing both child/adolescent and parents and usually requires more than one session. For children who do not regard their symptoms as excessive, information from parents, and if possible from teachers, is essential to identify the range of symptoms, severity and context. Many children and adolescents feel confused and embarrassed with their symptoms. It is important to dedicate time to build a true clinical alliance to elicit the story of their symptoms, as well as the impact on a child's thoughts and feelings.
There are several useful instruments to establish OCD diagnosis and severity in children and adolescents. Self-report questionnaires have been used to identify the presence and severity of OCD symptoms. The most used self-rated measures for pediatric OCD are the 44-item Leyton Obsession Inventory-Child version (LOI-CV) and its shorter version, the 20-itern LOI-CV Survey Form, and the Maudsley Obsession-Compulsion Inventory (MOCI). Clinician-administered interviews may be a more reliable method to identify obsessive-compulsive disorders in youth. The Childs Yale-Brown Obsessive-Compulsive Scale (CY-BOCS) is a commonly used Clinician-Rated measure of OCD symptoms derived from the Adult Yale-Brown Obsessive-Compulsive Scale.
CY-BOCS Spanish version was translated in Mexico and as the original version it must be applied to parents and children/adolescents separately; the clinician establishes then the best clinical information with all the data. The initial CY-BOCS section consists of a symptom checklist covering a comprehensive array of obsessions and compulsions. The severity score is derived from the second section of the measure in which global rating of time spent, interference, distress, resistance and control associated with obsessions and compulsions are generated. Separate scores are obtained for obsessions and compulsions, which, when combined, yield a total severity score of a maximum 40 points. Scores greater than or equal to 16 indicate clinically significant OCD in children and adolescents.
The knowledge we now have about pediatric OCD pharmacotherapy is better. Several studies have demonstrated the efficacy of clorimipramine. This was the first agent approved for use in pediatric populations with OCD. Subsequent multisite randomized, placebo-controlled trials of selective reuptake inhibitors (SSRIs) have also demonstrated significant efficacy in pediatric population. Almost all meta-analysis with SSRIs studies in children and adolescents with OCD have proved their efficacy. The most common adverse effects of SSRIs are nausea, insomnia, activation and headache. These effects are transient and most children tolerate them.
The availability and effectiveness of SSRI have changed dramatically the OCD treatment, and neurobiological and neuroimaging advances have supported their use.
Many children and adolescents with OCD need multiple treatments including cognitive behavior therapy (CBT), pharmacologic treatment, parental, family and teachers training. These interventions need to be applied by experts in order to be effective. CBT is a well-documented and effective intervention for adults with OCD. The potential usefulness of CBT for pediatric OCD has been valued and the results report that combined CBT and pharmacotherapy have proved high and sustained response in children and adolescents with OCD.
C1 [Vargas Alvarez, Luis Alberto] Inst Nacl Psiquiatria Ramon Fuente, Clin Trastornos Obsesivos Compulsivos, Direcc Serv Clin, Mexico City, DF, Mexico.
[Cruz, Lino Palacios; Thompson, Guillermo Gonzalez] Inst Nacl Psiquiatria Ramon Fuente, Clin Adolescencia, Direcc Serv Clin, Mexico City 14370, DF, Mexico.
[Olvera, Francisco de la Pena] Inst Nacl Psiquiatria Ramon Fuente, Dept Fomento Invest, Direcc Serv Clin, Mexico City, DF, Mexico.
RP Alvarez, LAV (reprint author), Inst Nacl Psiquiatria Ramon Fuente, Clin Adolescencia, Direcc Serv Clin, Calz Mexico Xochimilco 101, Mexico City 14370, DF, Mexico.
EM drluisvargasalvarez@yahoo.com.mx
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NR 36
TC 0
Z9 0
PU INST MEX PSIQUIATRIA
PI MEXICO CITY
PA CALZ MEXICO-XOCHIMILCO #101, MEXICO CITY 22 DF, MEXICO
SN 0185-3325
J9 SALUD MENT
JI Salud Ment.
PD JUL-AUG
PY 2008
VL 31
IS 4
BP 283
EP 289
PG 7
WC Psychiatry
SC Psychiatry
GA 349IY
UT WOS:000259275700005
ER
PT J
AU Pinkham, AE
Hopfinger, JB
Ruparel, K
Penn, DL
AF Pinkham, Amy E.
Hopfinger, Joseph B.
Ruparel, Kosha
Penn, David L.
TI An investigation of the relationship between activation of a social
cognitive neural network and social functioning
SO SCHIZOPHRENIA BULLETIN
LA English
DT Article
DE schizophrenia; paranoia; amygdala; fusiform; gyrus; trustworthiness;
fMRI
ID PERSECUTORY DELUSIONS; FACE PERCEPTION; HUMAN AMYGDALA; SCHIZOPHRENIA;
AUTISM; BRAIN; RECOGNITION; COMPETENCE; RESPONSES; FEAR
AB Previous work examining the neurobiological substrates of social cognition in healthy individuals has reported modulation of a social cognitive network such that increased activation of the amygdala, fusiform gyrus, and superior temporal sulcus are evident when individuals judge a face to be untrustworthy as compared with trustworthy. We examined whether this pattern would be present in individuals with schizophrenia who are known to show reduced activation within these same neural regions when processing faces. Additionally, we sought to determine how modulation of this social cognitive network may relate to social functioning. Neural activation was measured using functional magnetic resonance imaging with blood oxygenation level dependent contrast in 3 groups of individuals-nonparanoid individuals with schizophrenia, paranoid individuals with schizophrenia, and healthy controls-while they rated faces as either trustworthy or untrustworthy. Analyses of mean percent signal change extracted from a priori regions of interest demonstrated that both controls and nonparanoid individuals with schizophrenia showed greater activation of this social cognitive network when they rated a face as untrustworthy relative to trustworthy. In contrast, paranoid individuals did not show a significant difference in levels of activation based on how they rated faces. Further, greater activation of this social cognitive network to untrustworthy faces was significantly and positively correlated with social functioning. These findings indicate that impaired modulation of neural activity while processing social stimuli may underlie deficits in social cognition and social dysfunction in schizophrenia.
C1 [Pinkham, Amy E.; Ruparel, Kosha] Univ Penn, Sch Med, Dept Psychiat, Brain Behav Lab, Philadelphia, PA 19104 USA.
[Hopfinger, Joseph B.; Penn, David L.] Univ N Carolina, Dept Psychol, Chapel Hill, NC USA.
[Penn, David L.] Univ N Carolina, Dept Psychiat, Chapel Hill, NC USA.
RP Pinkham, AE (reprint author), Univ Penn, Sch Med, Dept Psychiat, Brain Behav Lab, 10th Floor,Gates Bldg, Philadelphia, PA 19104 USA.
EM amypi@mail.med.upenn.edu
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NR 51
TC 30
Z9 30
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0586-7614
J9 SCHIZOPHRENIA BULL
JI Schizophr. Bull.
PD JUL
PY 2008
VL 34
IS 4
BP 688
EP 697
DI 10.1093/schbul/sbn031
PG 10
WC Psychiatry
SC Psychiatry
GA 323AV
UT WOS:000257417300015
PM 18477583
ER
PT J
AU Enticott, PG
Hoy, KE
Herring, SE
Johnston, PJ
Daskalakis, ZJ
Fitzgerald, PB
AF Enticott, Peter G.
Hoy, Kate E.
Herring, Sally E.
Johnston, Patrick J.
Daskalakis, Zafiris J.
Fitzgerald, Paul B.
TI Reduced motor facilitation during action observation in schizophrenia: A
mirror neuron deficit?
SO SCHIZOPHRENIA RESEARCH
LA English
DT Article
DE schizophrenia; social cognition; mirror neurons; transcranial magnetic
stimulation; primary motor cortex; premotor cortex
ID TRANSCRANIAL MAGNETIC STIMULATION; CORTICAL INHIBITION; CORTEX;
EXCITABILITY; AUTISM; INDIVIDUALS; RESPONSES
AB impairments in social cognitive functioning are well documented in schizophrenia, however the neural basis of these deficits is unclear. A recent explanatory model of social cognition centers upon the activity of mirror neurons, which are cortical brain cells that become active during both the performance and observation of behavior. Here, we test for the first time whether mirror neuron functioning is reduced in schizophrenia. Fifteen individuals with schizophrenia or schizoaffective disorder and fifteen healthy controls completed a transcranial magnetic stimulation (TMS) experiment designed to assess mirror neuron activation. While patients demonstrated no abnormalities in cortical excitability, motor facilitation during action observation, putatively reflecting mirror neuron activity, was reduced in schizophrenia. Dysfunction within the mirror neuron system may contribute to the pathophysiology of schizophrenia. (c) 2008 Elsevier B.V. All rights reserved.
C1 [Enticott, Peter G.; Hoy, Kate E.; Herring, Sally E.; Fitzgerald, Paul B.] The Alfred, Alfred Psychiat Res Ctr, Sch Psychol Psychiat & Psychol Med, Melbourne, Vic 3004, Australia.
[Enticott, Peter G.; Hoy, Kate E.; Herring, Sally E.; Fitzgerald, Paul B.] Monash Univ, Alfred Psychiat Res Ctr, Sch Psychol Psychiat & Psychol Med, Clayton, Vic 3800, Australia.
[Johnston, Patrick J.] Swinburne Univ Technol, Brain Sci Inst, Hawthorn, Vic 3122, Australia.
[Daskalakis, Zafiris J.] Univ Toronto, Ctr Addict & Mental Hlth, Toronto, ON M5S 1A1, Canada.
RP Enticott, PG (reprint author), The Alfred, Alfred Psychiat Res Ctr, Sch Psychol Psychiat & Psychol Med, Level 1,Old Baker Bldg, Melbourne, Vic 3004, Australia.
EM peter.enticott@med.monash.edu.au
RI Daskalakis, Zafiris/J-5503-2013; Fitzgerald, Paul/A-1225-2008; Johnston,
Patrick/P-3158-2014
OI Daskalakis, Zafiris/0000-0001-9502-0538; Fitzgerald,
Paul/0000-0003-4217-8096; Johnston, Patrick/0000-0001-7703-1073
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NR 26
TC 37
Z9 37
PU ELSEVIER SCIENCE BV
PI AMSTERDAM
PA PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS
SN 0920-9964
J9 SCHIZOPHR RES
JI Schizophr. Res.
PD JUL
PY 2008
VL 102
IS 1-3
BP 116
EP 121
DI 10.1016/j.schres.2008.04.001
PG 6
WC Psychiatry
SC Psychiatry
GA 328ZW
UT WOS:000257837700014
PM 18485674
ER
PT J
AU Fatemi, SH
Folsom, TD
Reutiman, TJ
Lee, S
AF Fatemi, S. Hossein
Folsom, Timothy D.
Reutiman, Teri J.
Lee, Susanne
TI Expression of Astrocytic markers aquaporin 4 and connexin 43 is altered
in brains of subjects with autism
SO SYNAPSE
LA English
DT Article
DE astroglia; aquaporin 4; connexin 43; cerebellum; BA9; BA40
ID FIBRILLARY ACIDIC PROTEIN; GAP-JUNCTIONS; INFANTILE-AUTISM;
VIRAL-INFECTION; FRONTAL-CORTEX; RAT-BRAIN; ABNORMALITIES; CHILDREN;
MICE; IMMUNOREACTIVITY
AB Neuroanatomical studies have revealed extensive structural brain abnormalities in subjects with autism. Recently, studies have provided evidence of neuroglial responses and neuroinflammation in autism. The current study investigated whether two astrocytic markers, aquaporin 4 and connexin 43, are altered in brains from subjects with autism. Postmortem brain tissues from Brodmann's Area 40 (BA40, parietal cortex), Brodmann's Area 9 (BA9, superior frontal cortex), and cerebella of subjects with autism and matched controls were subject to SDS-PAGE and western blotting. Connexin 43 expression was increased significantly in BA9. Aquaporin 4 expression was decreased significantly in cerebellum. These data suggest that changes are apparent in markers for abnormal glial-neuronal communication (connexin 43 and aquaporin 4) in brains of subjects with autism.
C1 [Fatemi, S. Hossein; Folsom, Timothy D.; Reutiman, Teri J.; Lee, Susanne] Univ Minnesota, Dept Psychiat, Div Neurosci Res, Minneapolis, MN 55455 USA.
[Fatemi, S. Hossein] Univ Minnesota, Dept Pharmacol, Minneapolis, MN 55455 USA.
[Fatemi, S. Hossein] Univ Minnesota, Dept Neurosci, Minneapolis, MN 55455 USA.
RP Fatemi, SH (reprint author), 420 Delaware St SE,MMC 392, Minneapolis, MN 55455 USA.
EM fatem002@umn.edu
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NR 43
TC 37
Z9 39
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 0887-4476
J9 SYNAPSE
JI Synapse
PD JUL
PY 2008
VL 62
IS 7
BP 501
EP 507
DI 10.1002/syn.20519
PG 7
WC Neurosciences
SC Neurosciences & Neurology
GA 311LB
UT WOS:000256600600003
PM 18435417
ER
PT J
AU Mittleman, G
Goldowitz, D
Heck, DH
Blaha, CD
AF Mittleman, Guy
Goldowitz, Daniel
Heck, Detlef H.
Blaha, Charles D.
TI Cerebellar modulation of frontal cortex dopamine efflux in mice:
Relevance to autism and schizophrenia
SO SYNAPSE
LA English
DT Article
DE cerebellum; dentate nucleus; prefrontal cortex; amperometry; lurcher
mice
ID VENTRAL TEGMENTAL AREA; RAT NUCLEUS-ACCUMBENS; MOUSE MODEL SYSTEM;
PREFRONTAL CORTEX; IN-VIVO; GLUTAMATE RECEPTORS; SPECTRUM DISORDERS;
DEEP CEREBELLAR; STIMULATION; PURKINJE
AB Cerebellar and frontal cortical pathologies have been commonly reported in schizophrenia, autism, and other developmental disorders. Whether there is a relationship between prefrontal and cerebellar pathologies is unknown. Using fixed potential amperometry, dopamine (DA) efflux evoked by cerebellar or, dentate nucleus electrical stimulation (50 Hz, 200 mu A) was recorded in prefrontal cortex of urethane anesthetized lurcher (Lc/+) mice with 100% loss of cerebellar Purkinje cells and wildtype (+/+) control mice. Cerebellar stimulation with 25 and 100 pulses evoked prefrontal cortex DA efflux in +/+ mice that persisted for 12 and 25 s poststimulation, respectively. In contrast, 25 pulse cerebellar stimulation failed to evoke prefrontal cortex DA efflux in Lc/+ mice indicating a dependency on cerebellar Purkinje cell outputs. Dentate nucleus stimulation (25 pulses) evoked a comparable but briefer (baseline recovery within 7 s) increase in prefrontal cortex DA efflux compared to similar cerebellar stimulation in +/+ mice. However, in Lc/+ mice 25 pulse dentate nucleus evoked prefrontal cortex DA efflux was attenuated by 60% with baseline recovery within 4 s suggesting that dentate nucleus outputs to prefrontal cortex remain partially functional. DA reuptake blockade enhanced 100 pulse stimulation evoked prefrontal cortex responses, while serotonin or norepinephrine reuptake blockade were without effect indicating the specificity of the amperometric recordings to DA. Results provide neurochemical evidence that the cerebellum can modulate DA efflux in the prefrontal cortex. Together, these findings may explain why cerebellar and frontal cortical pathologies co-occur, and may provide a mechanism that accounts for the diversity of symptoms common to multiple developmental disorders.
C1 [Mittleman, Guy; Blaha, Charles D.] Univ Memphis, Dept Psychol, Memphis, TN 38152 USA.
[Goldowitz, Daniel; Heck, Detlef H.] Univ Tennessee, Hlth Sci Ctr, Dept Anat & Neurobiol, Memphis, TN 38163 USA.
RP Blaha, CD (reprint author), Univ Memphis, Dept Psychol, Memphis, TN 38152 USA.
EM cblaha@memphis.edu
RI Sia, Michelle/A-1756-2010
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NR 43
TC 28
Z9 30
PU WILEY-LISS
PI HOBOKEN
PA DIV JOHN WILEY & SONS INC, 111 RIVER ST, HOBOKEN, NJ 07030 USA
SN 0887-4476
J9 SYNAPSE
JI Synapse
PD JUL
PY 2008
VL 62
IS 7
BP 544
EP 550
DI 10.1002/syn.20525
PG 7
WC Neurosciences
SC Neurosciences & Neurology
GA 311LB
UT WOS:000256600600008
PM 18435424
ER
PT J
AU Becchio, C
Bertone, C
Castiello, U
AF Becchio, Cristina
Bertone, Cesare
Castiello, Umberto
TI How the gaze of others influences object processing
SO TRENDS IN COGNITIVE SCIENCES
LA English
DT Review
ID EYE GAZE; SOCIAL COGNITION; ATTENTION; DIRECTION; CUES; AUTISM
AB An aspect of gaze processing, which so far has been given little attention, is the influence that intentional gaze processing can have on object processing. Converging evidence from behavioural neuroscience and developmental psychology strongly suggests that objects failing under the gaze of others acquire properties that they would not display if not looked at. Specifically, observing another person gazing at an object enriches that object of motor, affective and status properties that go beyond its chemical or physical structure. A conceptual analysis of available evidence leads to the conclusion that gaze has the potency to transfer to the object the intentionality of the person looking at it.
C1 [Castiello, Umberto] Univ Padua, Dipartimento Psicol Gen, I-35131 Padua, Italy.
[Becchio, Cristina] Univ Turin, Dipartimento Psicol, Ctr Cognit Sci, I-10123 Turin, Italy.
[Bertone, Cesare] Univ Turin, Ctr Theoret & Appl Ontol, I-10124 Turin, Italy.
[Castiello, Umberto] Univ London Royal Holloway & Bedford New Coll, Dept Psychol, Egham TW20 0EX, Surrey, England.
RP Castiello, U (reprint author), Univ Padua, Dipartimento Psicol Gen, Via Venezia 8, I-35131 Padua, Italy.
EM umberto.castiello@unipd.it
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NR 32
TC 34
Z9 34
PU ELSEVIER SCIENCE LONDON
PI LONDON
PA 84 THEOBALDS RD, LONDON WC1X 8RR, ENGLAND
SN 1364-6613
J9 TRENDS COGN SCI
JI TRENDS COGN. SCI.
PD JUL
PY 2008
VL 12
IS 7
BP 254
EP 258
DI 10.1016/j.tics.2008.04.005
PG 5
WC Behavioral Sciences; Neurosciences; Psychology, Experimental
SC Behavioral Sciences; Neurosciences & Neurology; Psychology
GA 329AC
UT WOS:000257838300006
PM 18555735
ER
PT J
AU Ozdemir, S
AF Ozdemir, Selda
TI Using multimedia social stories to increase appropriate social
engagement in young children with autism
SO TURKISH ONLINE JOURNAL OF EDUCATIONAL TECHNOLOGY
LA English
DT Article
DE autism; multimedia social stories; social engagement; intervention;
young children
AB The purpose of this study was to examine the effectiveness of the multimedia social stories on the duration of appropriate social engagement of 3 young children with autism. Using a multiple-baseline-across-participants design, the multimedia social stories were implemented, and observations of 10-min play sessions were conducted three times per week. Study results showed an increase in the duration of appropriate social engagement for all participants and two participants also showed generalization to a classroom setting. The results from the study provide support for the use of the multimedia social stories without additional behavioral management techniques in increasing the social engagement of children with autism. Recommendations for future research and potential benefits of the multimedia social story interventions are discussed.
C1 Gazi Univ, Gazi Egitim Fak, Ozel Egitim Bolumu, Ankara, Turkey.
RP Ozdemir, S (reprint author), Gazi Univ, Gazi Egitim Fak, Ozel Egitim Bolumu, Ankara, Turkey.
EM seldaunaldi@yahoo.com
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NR 40
TC 3
Z9 3
PU TURKISH ONLINE JOURNAL EDUCATIONAL TECH-TOJET
PI SAKARYA
PA SAKARYA UNIV, ESENTEPE KAMPUSU, SAKARYA, 54187, TURKEY
SN 1303-6521
J9 TURK ONLINE J EDUC T
JI Turk. Online J. Educ. Technol.
PD JUL
PY 2008
VL 7
IS 3
BP 80
EP 88
PG 9
WC Education & Educational Research
SC Education & Educational Research
GA 356WW
UT WOS:000259809300010
ER
PT J
AU Nakahachi, T
Yamashita, K
Iwase, M
Ishigami, W
Tanaka, C
Toyonaga, K
Maeda, S
Hirotsune, H
Tei, Y
Yokoi, K
Okajima, S
Shirnizu, A
Takeda, M
AF Nakahachi, Takayuki
Yamashita, Ko
Iwase, Masao
Ishigami, Wataru
Tanaka, Chitaru
Toyonaga, Koji
Maeda, Shizuyo
Hirotsune, Hideto
Tei, Yosyo
Yokoi, Koichi
Okajima, Shoji
Shirnizu, Akira
Takeda, Masatoshi
TI Disturbed holistic processing in autism spectrum disorders verified by
two cognitive tasks requiring perception of complex visual stimuli
SO PSYCHIATRY RESEARCH
LA English
DT Article
DE autistic disorder; Asperger's disorder; central coherence; visual
perception; memory; facial inversion effect; Thatcher's illusion
ID HUMAN EXTRASTRIATE CORTEX; CENTRAL COHERENCE THEORY; HIGH-FUNCTIONING
ADULTS; HUMAN FUSIFORM GYRUS; UPSIDE-DOWN FACES; ASPERGER-SYNDROME;
RECOGNITION; CHILDREN; DISCRIMINATION; INVERSION
AB Central coherence is a key concept in research on autism spectrum disorders (ASD). It refers to the process in which diverse information is integrated and higher meaning is constructed in context. A malfunction in this process could result in abnormal attention to partial information in preference to the whole. To verify this hypothesis, we studied the performance of two visual tasks by 10 patients with autistic disorder or Asperger's disorder and by 26 (experiment 1) or 25 (experiment 2) normal subjects. In experiment 1, the subjects memorized pictures, some pictures with a change related to the main theme (D1) and others with a change not related to the main theme (D2); then the same pictures were randomly presented to the subjects who were asked to find the change. In experiment 2, the subjects were presented pictures of a normal (N) or a Thatcherized (T) face arranged side by side inversely (I) or uprightly (U) and to judge them as the same or different. In experiment 1, ASD subjects exhibited significantly lower rates of correct responses in D1 but not in D2. In experiment 2, ASD subjects exhibited significantly longer response times in NT-U but not in TN-I. These results showed a deficit in holistic processing, which is consistent with weak central coherence in ASD. (C) 2005 Elsevier Ireland Ltd. All rights reserved.
C1 [Nakahachi, Takayuki; Yamashita, Ko; Iwase, Masao; Hirotsune, Hideto; Takeda, Masatoshi] Osaka Univ, Grad Sch Med, Dept Clin Neurosci, Osaka 5650871, Japan.
[Ishigami, Wataru] Osakafu Eiseikai Clin, Osaka, Japan.
[Tanaka, Chitaru] Tanaka Mental Clin, Osaka, Japan.
[Toyonaga, Koji; Maeda, Shizuyo; Tei, Yosyo] Osaka City Gen Hosp, Dept Child & Adolescence Psychiat, Osaka, Japan.
[Okajima, Shoji] Mizuma Hosp, Osaka, Japan.
RP Nakahachi, T (reprint author), Osaka Univ, Grad Sch Med, Dept Clin Neurosci, D3 2-2 Yamadaoka, Osaka 5650871, Japan.
EM nakaha@psy.med.osaka-u.ac.jp
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NR 55
TC 9
Z9 10
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0165-1781
J9 PSYCHIAT RES
JI Psychiatry Res.
PD JUN 30
PY 2008
VL 159
IS 3
BP 330
EP 338
DI 10.1016/j.psychres.2005.08.028
PG 9
WC Psychiatry
SC Psychiatry
GA 313MD
UT WOS:000256743800008
PM 18417223
ER
PT J
AU Yan, J
Noltner, K
Feng, JN
Li, WY
Schroer, R
Skinner, C
Zeng, WQ
Schwartz, CE
Sommer, SS
AF Yan, Jin
Noltner, Katie
Feng, Jinong
Li, Wenyan
Schroer, Richard
Skinner, Cindy
Zeng, Wenqi
Schwartz, Charles E.
Sommer, Steve S.
TI Neurexin 1 alpha structural variants associated with autism
SO NEUROSCIENCE LETTERS
LA English
DT Article
DE autism; alpha-neurexin genes; mutation detection
ID PCR AMPLIFICATION; ALPHA-NEUREXINS; BETA-NEUREXINS; MUTATIONS; GENES;
SCHIZOPHRENIA; POLYMORPHISMS; NEUROLIGIN-1; ALLELES; NLGN4
AB Neurexins are presynaptic membrane cell-adhesion molecules which bind to neuroligins, a family of proteins that are associated with autism. To explore the possibility that structural variants in the neurexin alpha genes predispose to autism, the coding regions and associated splice junctions of the neurexin 1 alpha gene were sequenced in 116 Caucasian patients with autism and 192 Caucasian controls. Five ultra-rare structural variants including a predicted splicing mutation were found in patients with autism and absent in 10,000 control alleles. Only one ultra-rare structural variant was found in controls (5/116 vs. 1/192; P= 0.03, Fisher's exact test, one-sided). In the context of all available data, the ultra-rare structural variants of the neurexin 1 alpha gene are consistent with mutations predisposing to autism. (c) 2008 Elsevier Ireland Ltd. All rights reserved
C1 [Yan, Jin; Noltner, Katie; Feng, Jinong; Li, Wenyan; Sommer, Steve S.] City Hope Natl Med Ctr, Dept Mol Genet, Duarte, CA 91010 USA.
[Zeng, Wenqi; Sommer, Steve S.] City Hope Natl Med Ctr, Dept Mol Diag, Duarte, CA 91010 USA.
[Schroer, Richard; Skinner, Cindy; Schwartz, Charles E.] Greenwood Genet Ctr, Greenwood, SC 29646 USA.
RP Sommer, SS (reprint author), City Hope Natl Med Ctr, Dept Mol Genet, 1500 E Duarte Rd, Duarte, CA 91010 USA.
EM sommerlab@coh.org
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NR 23
TC 76
Z9 77
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0304-3940
J9 NEUROSCI LETT
JI Neurosci. Lett.
PD JUN 27
PY 2008
VL 438
IS 3
BP 368
EP 370
DI 10.1016/j.neulet.2008.04.074
PG 3
WC Neurosciences
SC Neurosciences & Neurology
GA 322RN
UT WOS:000257393100022
PM 18490107
ER
PT J
AU Petrovic, P
Kalisch, R
Singer, T
Dolan, RJ
AF Petrovic, Predrag
Kalisch, Raffael
Singer, Tania
Dolan, Raymond J.
TI Oxytocin attenuates affective evaluations of conditioned faces and
amygdala activity
SO JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE oxytocin; amygdala; fusiform gyrus; face stimuli; gaze; affective value
ID SOCIAL RECOGNITION; WILLIAMS-SYNDROME; NEURAL CIRCUITRY; HUMAN BRAIN;
VASOPRESSIN; HUMANS; AUTISM; RESPONSES; COGNITION; FEAR
AB Social relations between humans critically depend on our affective experiences of others. Oxytocin enhances prosocial behavior, but its effect on humans' affective experience of others is not known. We tested whether oxytocin influences affective ratings, and underlying brain activity, of faces that have been aversively conditioned. Using a standard conditioning procedure, we induced differential negative affective ratings in faces exposed to an aversive conditioning compared with nonconditioning manipulation. This differential negative evaluative effect was abolished by treatment with oxytocin, an effect associated with an attenuation of activity in anterior medial temporal and anterior cingulate cortices. In amygdala and fusiform gyrus, this modulation was stronger for faces with direct gaze, relative to averted gaze, consistent with a relative specificity for socially relevant cues. The data suggest that oxytocin modulates the expression of evaluative conditioning for socially relevant faces via influences on amygdala and fusiform gyrus, an effect that may explain its prosocial effects.
C1 [Petrovic, Predrag; Kalisch, Raffael; Singer, Tania; Dolan, Raymond J.] UCL, Wellcome Trust Funct Imaging Lab, London WC1N 3BG, England.
RP Petrovic, P (reprint author), UCL, Wellcome Trust Funct Imaging Lab, 12 Queen Sq, London WC1N 3BG, England.
EM predrag.petrovic@ki.se
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NR 50
TC 198
Z9 202
PU SOC NEUROSCIENCE
PI WASHINGTON
PA 11 DUPONT CIRCLE, NW, STE 500, WASHINGTON, DC 20036 USA
SN 0270-6474
J9 J NEUROSCI
JI J. Neurosci.
PD JUN 25
PY 2008
VL 28
IS 26
BP 6607
EP 6615
DI 10.1523/JNEUROSCI.4572-07.2008
PG 9
WC Neurosciences
SC Neurosciences & Neurology
GA 318RW
UT WOS:000257110800008
PM 18579733
ER
PT J
AU Donaldson, ZR
Kondrashov, FA
Putnam, A
Bai, Y
Stoinski, TL
Hammock, EAD
Young, LJ
AF Donaldson, Zoe R.
Kondrashov, Fyodor A.
Putnam, Andrea
Bai, Yaohui
Stoinski, Tara L.
Hammock, Elizabeth A. D.
Young, Larry J.
TI Evolution of a behavior-linked microsatellite-containing element in the
5 ' flanking region of the primate AVPR1A gene
SO BMC EVOLUTIONARY BIOLOGY
LA English
DT Article
ID POPULATION-STRUCTURE; SEQUENCE ALIGNMENT; PROMOTER REGION;
SOCIAL-BEHAVIOR; MESSENGER-RNA; DNA-SEQUENCES; V-1A RECEPTOR;
VASOPRESSIN; AUTISM; ASSOCIATION
AB Background: The arginine vasopressin V1a receptor (V1aR) modulates social cognition and behavior in a wide variety of species. Variation in a repetitive microsatellite element in the 5' flanking region of the V1aR gene (AVPR1A) in rodents has been associated with variation in brain V1aR expression and in social behavior. In humans, the 5' flanking region of AVPR1A contains a tandem duplication of two similar to 350 bp, microsatellite-containing elements located approximately 3.5 kb upstream of the transcription start site. The first block, referred to as DupA, contains a polymorphic (GT)(25) microsatellite; the second block, DupB, has a complex (CT)(4)-(TT)-(CT)(8)-(GT)(24) polymorphic motif, known as RS3. Polymorphisms in RS3 have been associated with variation in sociobehavioral traits in humans, including autism spectrum disorders. Thus, evolution of these regions may have contributed to variation in social behavior in primates. We examined the structure of these regions in six ape, six monkey, and one prosimian species.
Results: Both tandem repeat blocks are present upstream of the AVPR1A coding region in five of the ape species we investigated, while monkeys have only one copy of this region. As in humans, the microsatellites within DupA and DupB are polymorphic in many primate species. Furthermore, both single (lacking DupB) and duplicated alleles (containing both DupA and DupB) are present in chimpanzee (Pan troglodytes) populations with allele frequencies of 0.795 and 0.205 for the single and duplicated alleles, respectively, based on the analysis of 47 wild-caught individuals. Finally, a phylogenetic reconstruction suggests two alternate evolutionary histories for this locus.
Conclusion: There is no obvious relationship between the presence of the RS3 duplication and social organization in primates. However, polymorphisms identified in some species may be useful in future genetic association studies. In particular, the presence of both single and duplicated alleles in chimpanzees provides a unique opportunity to assess the functional role of this duplication in contributing to variation in social behavior in primates. While our initial studies show no signs of directional selection on this locus in chimps, pharmacological and genetic association studies support a potential role for this region in influencing V1aR expression and social behavior.
C1 [Donaldson, Zoe R.; Young, Larry J.] Emory Univ, Neurosci Program, Atlanta, GA 30322 USA.
[Donaldson, Zoe R.; Young, Larry J.] Emory Univ, Ctr Behav Neurosci, Atlanta, GA 30322 USA.
[Donaldson, Zoe R.; Bai, Yaohui; Young, Larry J.] Emory Univ, Yerkes Natl Primate Res Ctr, Atlanta, GA 30322 USA.
[Kondrashov, Fyodor A.; Putnam, Andrea] Univ Calif San Diego, Div Biol Sci, La Jolla, CA 92093 USA.
[Stoinski, Tara L.] Zoo Atlanta, Atlanta, GA USA.
[Hammock, Elizabeth A. D.] Vanderbilt Kennedy Ctr Res Human Dev, Nashville, TN USA.
[Young, Larry J.] Emory Univ, Dept Psychiat & Behav Sci, Atlanta, GA 30322 USA.
RP Donaldson, ZR (reprint author), Emory Univ, Neurosci Program, Atlanta, GA 30322 USA.
EM zdonald@emory.edu; fkondrashov@ucsd.edu; asputnam@biomail.ucsd.edu;
ybai01@emory.edu; tstoinski@zooatlanta.org;
elizabeth.hammock@vanderbilt.edu; lyoun03@emory.edu
RI Hammock, Elizabeth/G-1897-2011
FU NSF [IBN-9876754, NIH RR00165, NIMH56897, MH64692]; Howard Hughes
Predoctoral Fellowship
FX We thank the caretakers at Zoo Atlanta and Yerkes National Primate
Center for help with procuring specimens. Additional DNA samples were
supplied by Bill Hopkins, Emory University (chimpanzee), Allyson Bennet,
Wake Forest University (chimpanzee, rhesus macaque, bonnet macaque), Mar
Sanchez, Emory University (rhesus macaque), and Anne Yoder, Duke
University (galago). Susan Lambeth, M. D. Anderson Cancer Center, and
Katie Chace, Yerkes National Primate Center, helped provide records
regarding the origins of wild born chimps at these centers. We would
like to thank Dr Lisa McGraw and two anonymous reviewers for their
comments on this manuscript. This work was supported by NSF IBN-9876754,
NIH RR00165, NIMH56897 (LJY), MH64692 (LJY) and a Howard Hughes
Predoctoral Fellowship (ZRD).
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2008, US FISH WILDLIFE END
NR 45
TC 28
Z9 29
PU BIOMED CENTRAL LTD
PI LONDON
PA 236 GRAYS INN RD, FLOOR 6, LONDON WC1X 8HL, ENGLAND
SN 1471-2148
J9 BMC EVOL BIOL
JI BMC Evol. Biol.
PD JUN 23
PY 2008
VL 8
AR 180
DI 10.1186/1471-2148-8-180
PG 12
WC Evolutionary Biology; Genetics & Heredity
SC Evolutionary Biology; Genetics & Heredity
GA 346UX
UT WOS:000259096200001
PM 18573213
ER
PT J
AU Meyer, U
Engler, A
Weber, L
Schedlowskic, M
Feldon, J
AF Meyer, U.
Engler, A.
Weber, L.
Schedlowskic, M.
Feldon, J.
TI Preliminary evidence for a modulation of fetal dopaminergic development
by maternal immune activation during pregnancy
SO NEUROSCIENCE
LA English
DT Article
DE cytokines; dopamine; infection; Polyl : C; pregnancy; schizophrenia
ID NEURODEVELOPMENTAL ANIMAL-MODEL; PRENATAL INFECTION; BRAIN-DEVELOPMENT;
GENE-EXPRESSION; PHARMACOLOGICAL CHANGES; SCHIZOPHRENIA; NEURONS;
CHALLENGE; LEADS; MICE
AB Maternal infection during pregnancy is an environmental risk factor for the offspring to develop severe brain disorders, including schizophrenia and autism. However, only little is known about the neurodevelopmental mechanisms underlying the association between prenatal exposure to infection and the emergence of brain and behavioral dysfunctions in later life. Using a mouse model of prenatal immune challenge by the viral mimic polyriboinosinic-polyribocytidilic acid (Polyl:C), we explored the acute effects of maternal immune activation during pregnancy on the development of the fetal dopaminergic system, a neurotransmitter system known to be affected in schizophrenia and related disorders. We found that maternal immunological stimulation in early/middle pregnancy increased the number of mesencephalic dopamine neurons in the fetal brain at middle/late and late gestation. This effect was paralleled by changes in fetal expression of several genes known to be involved in dopamine neuron development, including the inductive signals sonic hedgehog (Shh) and fibroblast growth factor 8 (Fgf8), as well as transcription factors Nurr1 and Pitx3. These findings provide initial in vivo evidence for a modulation of fetal dopaminergic development by maternal immune activation during pregnancy. Additional investigations of the neurodevelopmental effects of prenatal immune challenge are thus clearly warranted in order to further validate whether abnormal dopaminergic development may be a critical neuropathological mechanism underlying the precipitation of schizophrenia-like brain and behavioral dysfunctions emerging after in utero exposure to infection. (C) 2008 IBRO. Published by Elsevier Ltd. All rights reserved.
C1 [Meyer, U.; Engler, A.; Weber, L.; Feldon, J.] ETH, Lab Behav Neurobiol, CH-8603 Schwerzenbach, Switzerland.
[Engler, A.] ETH, Lab Psychol & Behav Immunobiol, CH-8092 Zurich, Switzerland.
[Schedlowskic, M.] Univ Duisburg Essen, Fac Med, Inst Med Psychol & Behav Immunobiol, D-45122 Essen, Germany.
RP Meyer, U (reprint author), ETH, Lab Behav Neurobiol, Schorenstr 16, CH-8603 Schwerzenbach, Switzerland.
EM urmeyer@ethz.ch; andrea.engler@ifv.gess.ethz.ch;
weber@behav.biol.ethz.ch; manfred.schedlowski@uk-essen.de;
feldon@behav.biol.ethz.ch
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NR 46
TC 57
Z9 59
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0306-4522
J9 NEUROSCIENCE
JI Neuroscience
PD JUN 23
PY 2008
VL 154
IS 2
BP 701
EP 709
DI 10.1016/j.neuroscience.2008.04.031
PG 9
WC Neurosciences
SC Neurosciences & Neurology
GA 319OB
UT WOS:000257172200029
PM 18495356
ER
PT J
AU Fujimoto, M
Uchida, S
Watanuki, T
Wakabayashi, Y
Otsuki, K
Matsubara, T
Suetsugi, M
Funato, H
Watanabe, Y
AF Fujimoto, Michiko
Uchida, Shusaku
Watanuki, Toshio
Wakabayashi, Yusuke
Otsuki, Koji
Matsubara, Toshio
Suetsugi, Masatomo
Funato, Hiromasa
Watanabe, Yoshifumi
TI Reduced expression of glyoxalase-1 mRNA in mood disorder patients
SO NEUROSCIENCE LETTERS
LA English
DT Article
DE mood disorder; major depression; bipolar disorder; glyoxalase-1;
oxidative stress
ID MAJOR DEPRESSION; PANIC DISORDER; ANTIOXIDANT ENZYME; ANXIETY DISORDERS;
COMORBIDITY; ILLNESS; BRAIN; POLYMORPHISM; RECEPTORS; DISEASE
AB Glyoxalase-1 (Glo1) is an antioxidant enzyme which detoxifies alpha-ketoaldehydes to prevent the accumulation of pro-oxidant compounds, such as methylglyoxal, in all cell types. Glo1 has been suggested to be involved in anxiety disorders, autism, and Alzheimer's disease. Mood disorders have a high rate of comorbidity with anxiety disorders although, to date, little is known of the involvement of Glo1 in the pathophysiology of these conditions. In the present study, we examined the expression levels of Glo1 mRNA in peripheral white blood cells of mood disorder patients to understand the role of Glo1 in mood disorders. Quantitative real-time polymerase chain reaction experiments revealed that reduced expression of Glo1 mRNA was observed in major depressive and bipolar disorder patients in a current depressive state, as compared with healthy control subjects. In contrast, the expression of Glo1 mRNA in major depressive and bipolar patients, in a remissive state, showed no significant alteration when compared with healthy control subjects. These results suggest that the aberrant expression of Glo1 might be involved in the pathophysiology of mood disorders. (C) 2008 Elsevier Ireland Ltd. All rights reserved.
C1 [Fujimoto, Michiko; Uchida, Shusaku; Watanuki, Toshio; Wakabayashi, Yusuke; Otsuki, Koji; Matsubara, Toshio; Suetsugi, Masatomo; Funato, Hiromasa; Watanabe, Yoshifumi] Yamaguchi Univ, Grad Sch Med, Dept Neurosci, Div Neuropsychiat, Ube, Yamaguchi 7558505, Japan.
RP Watanabe, Y (reprint author), Yamaguchi Univ, Grad Sch Med, Dept Neurosci, Div Neuropsychiat, 1-1-1 Minamikogushi, Ube, Yamaguchi 7558505, Japan.
EM yoshiwat@yamaguchi-u.ac.jp
CR American Psychiatric Association, 1994, DIAGN STAT MAN MENT, V4th
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NR 32
TC 36
Z9 43
PU ELSEVIER IRELAND LTD
PI CLARE
PA ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000,
IRELAND
SN 0304-3940
J9 NEUROSCI LETT
JI Neurosci. Lett.
PD JUN 20
PY 2008
VL 438
IS 2
BP 196
EP 199
DI 10.1016/j.neulet.2008.04.024
PG 4
WC Neurosciences
SC Neurosciences & Neurology
GA 321AQ
UT WOS:000257276900014
PM 18455873
ER
PT J
AU Weber, P
Isler, E
Schmeck, K
AF Weber, P.
Isler, E.
Schmeck, K.
TI Childhood autism: an interdisciplinary approach
SO SWISS MEDICAL WEEKLY
LA English
DT Meeting Abstract
C1 [Weber, P.] Univ Childrens Hosp Basel, Dept Neuropediat, Basel, Switzerland.
[Isler, E.] Off Child & Adolescents Psychiat Basel Land, Basel, Switzerland.
[Isler, E.] Univ Psychiat Clin, Clin Child & Adolescents Psychiat, Basel, Switzerland.
NR 0
TC 0
Z9 0
PU E M H SWISS MEDICAL PUBLISHERS LTD
PI MUTTENZ
PA FARNSBURGERSTR 8, CH-4132 MUTTENZ, SWITZERLAND
SN 1424-7860
J9 SWISS MED WKLY
JI Swiss Med. Wkly.
PD JUN 20
PY 2008
VL 138
SU 164
BP 7S
EP 7S
PG 1
WC Medicine, General & Internal
SC General & Internal Medicine
GA 318RB
UT WOS:000257108300014
ER
PT J
AU Kreibich, AS
Reyes, BAS
Curtis, AL
Ecke, L
Chavkin, C
Van Bockstaele, EJ
Valentino, RJ
AF Kreibich, Arati S.
Reyes, Beverly A. S.
Curtis, Andre L.
Ecke, Laurel
Chavkin, Charles
Van Bockstaele, Elisabeth J.
Valentino, Rita J.
TI Presynaptic inhibition of diverse afferents to the locus ceruleus by
kappa-opiate receptors: A novel mechanism for regulating the central
norepinephrine system
SO JOURNAL OF NEUROSCIENCE
LA English
DT Article
DE locus ceruleus; kappa-opiate receptor; dynorphin;
corticotropin-releasing factor; glutamate; morphine withdrawal
ID CORTICOTROPIN-RELEASING-FACTOR; COERULEUS-NORADRENERGIC SYSTEM; INDUCED
BEHAVIORAL-RESPONSES; MEDIAL PREFRONTAL CORTEX; ELEMENT-BINDING PROTEIN;
NUCLEUS-ACCUMBENS SHELL; FREELY MOVING CATS; REACTION-TIME-TASK; FORCED
SWIM TEST; OPIOID-RECEPTOR
AB The norepinephrine nucleus, locus ceruleus (LC), is activated by diverse stimuli and modulates arousal and behavioral strategies in response to these stimuli through its divergent efferent system. Afferents communicating information to the LC include excitatory amino acids (EAAs), corticotropin-releasing factor (CRF), and endogenous opioids acting at mu-opiate receptors. Because the LC is also innervated by the endogenous kappa-opiate receptor (kappa-OR) ligand dynorphin and expresses kappa-ORs, this study investigated kappa-OR regulation of LC neuronal activity in rat. Immunoelectron microscopy revealed a prominent localization of kappa-ORs in axon terminals in the LC that also contained either the vesicular glutamate transporter or CRF. Microinfusion of the kappa-OR agonist (trans)-3,4-dichloro-N-methyl-N-[2-1-pyrrolidinyl)-cyclo-hexyl] benzeneacetamide (U50488) into the LC did not alter LC spontaneous discharge but attenuated phasic discharge evoked by stimuli that engage EAA afferents to the LC, including sciatic nerve stimulation and auditory stimuli and the tonic activation associated with opiate withdrawal. Inhibitory effects of the kappa-OR agonist were not restricted to EAA afferents, as U50488 also attenuated tonic LC activation by hypotensive stress, an effect mediated by CRF afferents. Together, these results indicate that kappa-ORs are poised to presynaptically inhibit diverse afferent signaling to the LC. This is a novel and potentially powerful means of regulating the LC-norepinephrine system that can impact on forebrain processing of stimuli and the organization of behavioral strategies in response to environmental stimuli. The results implicate kappa-ORs as a novel target for alleviating symptoms of opiate withdrawal, stress-related disorders, or disorders characterized by abnormal sensory responses, such as autism.
C1 [Kreibich, Arati S.] Univ Penn, Translat Res Labs, Philadelphia, PA 19104 USA.
[Kreibich, Arati S.; Curtis, Andre L.; Valentino, Rita J.] Childrens Hosp Philadelphia, Philadelphia, PA 19104 USA.
[Reyes, Beverly A. S.; Van Bockstaele, Elisabeth J.] Thomas Jefferson Univ, Philadelphia, PA 19108 USA.
[Chavkin, Charles] Univ Washington, Seattle, WA 98195 USA.
RP Kreibich, AS (reprint author), Univ Penn, Translat Res Labs, 125 S 31st St, Philadelphia, PA 19104 USA.
EM aratik@mail.med.upenn.edu
RI Chavkin, Charles/G-2797-2010
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NR 75
TC 32
Z9 32
PU SOC NEUROSCIENCE
PI WASHINGTON
PA 11 DUPONT CIRCLE, NW, STE 500, WASHINGTON, DC 20036 USA
SN 0270-6474
J9 J NEUROSCI
JI J. Neurosci.
PD JUN 18
PY 2008
VL 28
IS 25
BP 6516
EP 6525
DI 10.1523/JNEUROSCI.0390-08.2008
PG 10
WC Neurosciences
SC Neurosciences & Neurology
GA 315OT
UT WOS:000256890000026
PM 18562623
ER
PT J
AU Tartaglia, N
Davis, S
Hench, A
Nitnishakavi, S
Beauregard, R
Reynolds, A
Fenton, L
Albrecht, L
Ross, J
Visootsak, J
Hansen, R
Hagerman, R
AF Tartaglia, Nicole
Davis, Shanlee
Hench, Alison
Nitnishakavi, Sheela
Beauregard, Renee
Reynolds, Ann
Fenton, Laura
Albrecht, Lindsey
Ross, Judith
Visootsak, Jeannie
Hansen, Robin
Hagerman, Randi
TI A new look at XXYY syndrome: Medical and psychological features
SO AMERICAN JOURNAL OF MEDICAL GENETICS PART A
LA English
DT Article
DE XXYY syndrome; Klinefelter syndrome; sex chromosome abnormality; autism
spectrum disorder; tremor; ADHD
ID SEX-CHROMOSOME ANEUPLOIDIES; KLINEFELTERS-SYNDROME; 48,XXYY SYNDROME;
ESSENTIAL TREMOR; Y-CHROMOSOME; SHORT STATURE; TURNER-SYNDROME;
PHENOTYPE; GROWTH; 47,XXY
AB XXYY syndrome occurs in approximately 1:18,000-1:40,000 males. Although the physical phenotype is similar to 47,XXY (tall stature, hypergonadotropic hypogonadism, and infertility), XXYY is associated with additional medical problems and more significant neurodevelopmental and psychological features. We report on the results of a cross-sectional, multicenter Study of 95 males age 1-55 with XXYY syndrome (mean age 14.9 years), describing diagnosis, physical features, medical problems, medications, and psychological features stratified by age groups. The mean age of diagnosis was 7.7 years. Developmental delays and behavioral problems were the most common primary indication for genetic testing (68.4%). Physical and facial features varied with age, although hypertelorism, clinodactyly, pes planus, and dental problems were common across all age groups. Tall statute was present in adolescents and adults, with a mean adult statute of 192.4 cm (SD 7.5; n = 22). Common medical problems included allergies and asthma (>50%), congenital heart defects (19.4%), radioulnar synostosis (17.2%), inguinal hernia and/or cryptorchidism (16.1%), and Seizures (15%). Medical features in adulthood included hypogonadism (100%), DVT (18.2%), intention tremor (71%) and type 11 diabetes (18.2%). Brain MRI (n = 35) showed white matter abnormalities in 45.7% of patients and enlarged ventricles in 22.8%. Neurodevelopmental and psychological difficulties were a significant component of the behavioral phenotype, with developmental delays and learning disabilities universal but variable in severity. Twenty-six percent had full-scale IQs in the range of intellectual disability (MR), and adaptive functioning was significantly impacted with 68% with adaptive composite scores <70. Rates of neurodevelopmental disorders, including ADHD (72.2%), autism spectrum disorders (28.3%), mood disorders (46.8%), and tic disorders (18.9%), were elevated with 55.9% on psychopharmacologic medication overall. Recommendations for evaluation and treatment are summarized. (c) 2008 Wiley-Liss, Inc.
C1 [Tartaglia, Nicole; Reynolds, Ann] Univ Colorado Denver, Sch Med, Dept Pediat, Aurora, CO USA.
[Davis, Shanlee; Hench, Alison; Nitnishakavi, Sheela; Hansen, Robin; Hagerman, Randi] Univ Calif Davis, Med Ctr, MIND Inst, Sacramento, CA 95817 USA.
[Beauregard, Renee] Colorado Nonprofit Dev Ctr, XXYY Project, Aurora, CO USA.
[Fenton, Laura] Univ Colorado Denver, Sch Med, Dept Radiol, Aurora, CO USA.
[Albrecht, Lindsey; Hansen, Robin; Hagerman, Randi] Univ Calif Davis, Med Ctr, Dept Pediat, Sacramento, CA 95817 USA.
[Ross, Judith] Thomas Jefferson Univ, Dept Pediat, Philadelphia, PA 19107 USA.
[Visootsak, Jeannie] Emory Univ, Dept Human Genet & Pediat, Atlanta, GA 30322 USA.
RP Tartaglia, N (reprint author), 13123 E 16th Ave,B-140, Aurora, CO 80045 USA.
EM tartaglia.nicole@tchden.org
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NR 73
TC 49
Z9 56
PU WILEY-BLACKWELL
PI MALDEN
PA COMMERCE PLACE, 350 MAIN ST, MALDEN 02148, MA USA
SN 1552-4825
J9 AM J MED GENET A
JI Am. J. Med. Genet. A
PD JUN 15
PY 2008
VL 146A
IS 12
BP 1509
EP 1522
DI 10.1002/ajmg.a.32366
PG 14
WC Genetics & Heredity
SC Genetics & Heredity
GA 307PB
UT WOS:000256329700002
PM 18481271
ER
PT J
AU Christian, SL
Brune, CW
Sudi, J
Kumar, RA
Liu, S
Karamohamed, S
Badner, JA
Matsui, S
Conroy, J
McQuaid, D
Gergel, J
Hatchwell, E
Gilliam, TC
Gershon, ES
Nowak, NJ
Dobyns, WB
Cook, EH
AF Christian, Susan L.
Brune, Camille W.
Sudi, Jyotsna
Kumar, Ravinesh A.
Liu, Shaung
Karamohamed, Samer
Badner, Judith A.
Matsui, Seiichi
Conroy, Jeffrey
McQuaid, Devin
Gergel, James
Hatchwell, Eli
Gilliam, T. Conrad
Gershon, Elliot S.
Nowak, Norma J.
Dobyns, William B.
Cook, Edwin H., Jr.
TI Novel submicroscopic chromosomal abnormalities detected in autism
spectrum disorder
SO BIOLOGICAL PSYCHIATRY
LA English
DT Article
DE array comparative genomic hybridization; autism; microdeletions;
microduplications
ID PERVASIVE DEVELOPMENTAL DISORDERS; 22Q11.2 DUPLICATION; ARRAY-CGH;
MICRODELETIONS; FEATURES
AB Background: One genetic mechanism known to be associated with autism spectrum disorders (ASD) is chromosomal abnormalities. The identification of copy number variants (CNV), i.e., microdeletions and microduplications that are undetectable at the level of traditional cytogenetic analysis, allows the potential association of submicroscopic chromosomal imbalances and human disease.
Methods: We performed array comparative genomic hybridization (aCGH) utilizing a 19K whole genome tiling path bacterial artificial chromosome (BAC) microarray on 397 unrelated subjects with autism spectrum disorder. Common CNV were excluded using a control group comprised of 372 individuals from the National Institute of Mental Health (NIMH) Genetics Initiative Control samples. Confirmation studies were performed on all remaining CNV using fluorescence in situ hybridization (FISH), microsatellite analysis, and/or quantitative polymerase chain reaction (PCR) analysis.
Results: A total of 51 CNV were confirmed in 46 ASD subjects. Three maternal interstitial duplications of 15q11-q13 known to be associated with AS D were identified. The other 48 CNV ranged in size from 189 kilobase (kb) to 5.5 megabase (Mb) and contained from 0 to similar to 40 National Center for Biotechnology Information (NCBI) Reference Sequence (RefSeq) genes. Seven CNV were de novo and 44 were inherited.
Conclusions: Fifty-one autism-specific CNV were identified in 46 of 397 ASD patients using a 19K BAC microarray for an overall rate of 11.6% . These microdeletions and microduplications cause gene dosage imbalance in 272 genes, many of which could be considered as candidate genes for autism.
C1 [Christian, Susan L.; Sudi, Jyotsna; Kumar, Ravinesh A.; Karamohamed, Samer; Gilliam, T. Conrad; Dobyns, William B.] Univ Chicago, Dept Human Genet, Chicago, IL 60637 USA.
[Brune, Camille W.; Cook, Edwin H., Jr.] Univ Illinois, Dept Psychiat, Inst Juvenile Res, Chicago, IL 60680 USA.
[Liu, Shaung; Badner, Judith A.; Gershon, Elliot S.] Univ Chicago, Dept Psychiat, Chicago, IL 60637 USA.
[Gergel, James] Oak Ridge Natl Lab, Oak Ridge, TN USA.
[Matsui, Seiichi; Conroy, Jeffrey; McQuaid, Devin; Nowak, Norma J.] Roswell Pk Canc Inst, Dept Canc Genet, Buffalo, NY 14263 USA.
[Nowak, Norma J.] Roswell Pk Canc Inst, Dept Canc Prevent & Populat Sci, Buffalo, NY 14263 USA.
[Hatchwell, Eli] SUNY Stony Brook, Stony Brook, NY 11794 USA.
[Nowak, Norma J.] SUNY Buffalo, Dept Biochem, Buffalo, NY 14214 USA.
RP Christian, SL (reprint author), Univ Chicago, Dept Human Genet, 920 E 58th St,CLSC 319,MC0077, Chicago, IL 60637 USA.
EM schrist@bsd.uchicago.edu
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NR 22
TC 165
Z9 171
PU ELSEVIER SCIENCE INC
PI NEW YORK
PA 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA
SN 0006-3223
J9 BIOL PSYCHIAT
JI Biol. Psychiatry
PD JUN 15
PY 2008
VL 63
IS 12
BP 1111
EP 1117
DI 10.1016/j.biopsych.2008.01.009
PG 7
WC Neurosciences; Psychiatry
SC Neurosciences & Neurology; Psychiatry
GA 309WU
UT WOS:000256491700003
PM 18374305
ER
PT J
AU Samaco, RC
Fryer, JD
Ren, J
Fyffe, S
Chao, HT
Sun, YL
Greer, JJ
Zoghbi, HY
Neul, JL
AF Samaco, Rodney C.
Fryer, John D.
Ren, Jun
Fyffe, Sharyl
Chao, Hsiao-Tuan
Sun, Yaling
Greer, John J.
Zoghbi, Huda Y.
Neul, Jeffrey L.
TI A partial loss of function allele of Methyl-CpG-binding protein 2
predicts a human neurodevelopmental syndrome
SO HUMAN MOLECULAR GENETICS
LA English
DT Article
ID LINKED MENTAL-RETARDATION; CPG-BINDING PROTEIN-2; RETT-SYNDROME; MECP2
GENE; NEUROLOGICAL SYMPTOMS; COPY NUMBER; MOUSE MODEL; MUTATION;
DUPLICATION; EXPRESSION
AB Rett Syndrome, an X-linked dominant neurodevelopmental disorder characterized by regression of language and hand use, is primarily caused by mutations in methyl-CpG-binding protein 2 (MECP2 ). Loss of function mutations in MECP2 are also found in other neurodevelopmental disorders such as autism, Angelman-like syndrome and non-specific mental retardation. Furthermore, duplication of the MECP2 genomic region results in mental retardation with speech and social problems. The common features of human neurodevelopmental disorders caused by the loss or increase of MeCP2 function suggest that even modest alterations of MeCP2 protein levels result in neurodevelopmental problems. To determine whether a small reduction in MeCP2 level has phenotypic consequences, we characterized a conditional mouse allele of Mecp2 that expresses 50% of the wild-type level of MeCP2. Upon careful behavioral analysis, mice that harbor this allele display a spectrum of abnormalities such as learning and motor deficits, decreased anxiety, altered social behavior and nest building, decreased pain recognition and disrupted breathing patterns. These results indicate that precise control of MeCP2 is critical for normal behavior and predict that human neurodevelopmental disorders will result from a subtle reduction in MeCP2 expression.
C1 [Samaco, Rodney C.; Fryer, John D.; Fyffe, Sharyl; Sun, Yaling] Baylor Coll Med, Dept Mol & Human Genet, Houston, TX 77030 USA.
[Chao, Hsiao-Tuan; Zoghbi, Huda Y.] Baylor Coll Med, Dept Neurosci, Houston, TX 77030 USA.
[Ren, Jun; Greer, John J.] Univ Alberta, Dept Physiol, Edmonton, AB T6G 2M7, Canada.
[Zoghbi, Huda Y.; Neul, Jeffrey L.] Baylor Coll Med, Dept Pediat, Neurol Sect, Houston, TX 77030 USA.
RP Neul, JL (reprint author), 1 Baylor Plaza,MS 225, Houston, TX 77030 USA.
EM jneul@bcm.tmc.edu
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NR 47
TC 76
Z9 77
PU OXFORD UNIV PRESS
PI OXFORD
PA GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND
SN 0964-6906
J9 HUM MOL GENET
JI Hum. Mol. Genet.
PD JUN 15
PY 2008
VL 17
IS 12
BP 1718
EP 1727
DI 10.1093/hmg/ddn062
PG 10
WC Biochemistry & Molecular Biology; Genetics & Heredity
SC Biochemistry & Molecular Biology; Genetics & Heredity
GA 306VJ
UT WOS:000256275600002
PM 18321864
ER
PT J
AU Officer, R
AF Officer, Rachel
TI Autism is not a disease
SO NEW SCIENTIST
LA English
DT Letter
NR 0
TC 0
Z9 0
PU REED BUSINESS INFORMATION LTD
PI SUTTON
PA QUADRANT HOUSE THE QUADRANT, SUTTON SM2 5AS, SURREY, ENGLAND
SN 0262-4079
J9 NEW SCI
JI New Sci.
PD JUN 14
PY 2008
VL 198
IS 2660
BP 20
EP 20
DI 10.1016/S0262-4079(08)61473-6
PG 1
WC Multidisciplinary Sciences
SC Science & Technology - Other Topics
GA 314XR
UT WOS:000256843000014
ER
PT J
AU Matson, JL
Ancona, MN
Wilkins, J
AF Matson, Johnny L.
Ancona, Martin N.
Wilkins, Jonathan
TI Sleep Disturbances in Adults with Autism Spectrum Disorders and Severe
Intellectual Impairments
SO JOURNAL OF MENTAL HEALTH RESEARCH IN INTELLECTUAL DISABILITIES
LA English
DT Article
DE intellectual disability; sleep disturbance; PDD-NOS; autism
AB Sleep disturbances are a significant problem for persons with developmental disabilities. These problems occur at a higher rate than what is observed in the typically developing population, and persons with Autism Spectrum Disorders (ASD) appear to be at a higher risk than individuals with other developmental disabilities. However, another major risk is intellectual disability (ID). These two groups of disorders overlap to a substantial degree. Thus, persons with ASD and ID appear to be particularly susceptible to sleep disturbances. These sleep problems can have serious consequences beyond sleep, particularly with respect to increased challenging behaviors and as an impediment to learning. Despite these concerns, adults with ASD and ID have been largely neglected with respect to the study of these nocturnal difficulties. In this report, 168 adults with ASD and ID were compared to 166 adults with ID alone in regard to sleep disturbance and related difficulties. Individuals in the ASD group presented with much higher rates of sleep disturbances, and greater severity of sleep disorder was related to the expression of more serious challenging behaviors. The implications of our results are discussed for future assessment and treatment of these disorders.
C1 [Matson, Johnny L.; Ancona, Martin N.; Wilkins, Jonathan] Louisiana State Univ, Dept Psychol, Baton Rouge, LA 80803 USA.
RP Matson, JL (reprint author), Louisiana State Univ, Dept Psychol, Baton Rouge, LA 80803 USA.
EM johnmatson@aol.com
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Wiggs L., 1996, J AUTISM DEV DISORD, V30, P127
NR 33
TC 7
Z9 7
PU ROUTLEDGE JOURNALS, TAYLOR & FRANCIS LTD
PI ABINGDON
PA 4 PARK SQUARE, MILTON PARK, ABINGDON OX14 4RN, OXFORDSHIRE, ENGLAND
SN 1931-5864
EI 1931-5872
J9 J MENT HEALTH RES IN
JI J. Ment. Health Res. Intellect. Disabil.
PD JUN 12
PY 2008
VL 1
IS 3
BP 129
EP 139
DI 10.1080/19315860801988210
PG 11
WC Education, Special; Psychiatry; Rehabilitation
SC Education & Educational Research; Psychiatry; Rehabilitation
GA V37ZY
UT WOS:000209314900001
ER
PT J
AU Muller, CM
Nussbeck, S
AF Mueller, Christoph M.
Nussbeck, Susanne
TI Do Children with Autism Spectrum Disorders Prefer to Match Pictures
Based on their Physical Details or their Meaning?
SO JOURNAL OF MENTAL HEALTH RESEARCH IN INTELLECTUAL DISABILITIES
LA English
DT Article
DE local-global processing; perceptual-conceptual processing; autism;
central coherence
AB This study investigated whether children with high-functioning autism/Asperger's syndrome have a different spontaneous processing style than typically developing children, that is, a style where they prefer details over meaning. Participants were 25 children with autism spectrum disorders (ASD) and 25 typically developing children matched by age, IQ, and sex. In the first 2 experiments, the children were given a picture and asked to choose which of 2 other pictures best matched with it. Pictures could be matched based either on congruence for details or meaning but not both. Hence, a choice for either detail or meaning had to be made. It was ensured that all participants had understood the meaningful relations between the pictures prior to testing. Children with ASD chose to match the pictures by detail significantly more often than the typically developing children. In a second test session, naming aloud the presented pictures prior to matching them was required. This additional manipulation led most children with ASD to match pictures according to meaning. It is concluded that children with ASD tend to spontaneously focus on details despite access to meaningful connections. Naming seems to support the use of meaning in spontaneous processing.
C1 [Mueller, Christoph M.; Nussbeck, Susanne] Univ Cologne, Dept Special Educ & Rehabil, D-50931 Cologne, Germany.
RP Nussbeck, S (reprint author), Univ Cologne, Humanwissensch Fak, Dept Heilpadagog & Rehabil, Klosterstr 79b, D-50931 Cologne, Germany.
EM Susanne.Nussbeck@uni-koeln.de
FU Stiftung der Deutschen Wirtschaft
FX We thank participants, parents, and staff from Heilpadagogisches Zentrum
"Die Gute Hand," Autismus Therapie Zentrum Koln, Autismus Therapie
Zentrum Aachen, and all participating schools. We also thank Catherine
Best from the University of Edinburgh for her helpful comments on this
article. Christoph M. Muller was supported by a doctoral grant from the
Stiftung der Deutschen Wirtschaft.
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Caron M. J., 2006, COGNITIVE MECH SPECI, P1787
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World Health Organization, 1992, INT CLASS DIS
NR 49
TC 3
Z9 3
PU ROUTLEDGE JOURNALS, TAYLOR & FRANCIS LTD
PI ABINGDON
PA 4 PARK SQUARE, MILTON PARK, ABINGDON OX14 4RN, OXFORDSHIRE, ENGLAND
SN 1931-5864
EI 1931-5872
J9 J MENT HEALTH RES IN
JI J. Ment. Health Res. Intellect. Disabil.
PD JUN 12
PY 2008
VL 1
IS 3
BP 140
EP 155
DI 10.1080/19315860801988244
PG 16
WC Education, Special; Psychiatry; Rehabilitation
SC Education & Educational Research; Psychiatry; Rehabilitation
GA V37ZY
UT WOS:000209314900002
ER
PT J
AU Ashworth, M
Martin, L
Hirdes, JP
AF Ashworth, Melody
Martin, Lynn
Hirdes, John P.
TI Prevalence and Correlates of Pica Among Adults with Intellectual
Disability in Institutions
SO JOURNAL OF MENTAL HEALTH RESEARCH IN INTELLECTUAL DISABILITIES
LA English
DT Article
DE interRAI Intellectual Disability; adults; challenging behavior; pica;
self-injury; intellectual disability
AB Increased knowledge of complex behaviors such as pica is needed to improve the support and services in the community for individuals with intellectual disability (ID). Though the prevalence of pica has been documented extensively in institutionalized settings, few studies have explored its etiology. The aim of this study is to explore the correlates of pica among institutionalized adults with ID. Secondary data analysis was performed on census-level data on 1,008 persons with ID residing in Ontario's remaining specialized institutions. All persons had been assessed using the interRAI Intellectual Disability (interRAI ID; Martin, Hirdes, Fries, & Smith, 2007) assessment instrument-a comprehensive and standardized instrument that supports service planning. Bivariate and multivariate analyses were used to explore the relation between pica and demographic, functional, and clinical characteristics. The overall prevalence of pica was 21.8%. Logistic regression analysis showed that being male, a diagnosis of autism, and use of nonverbal means of communication were associated with a higher likelihood of pica, whereas impairment in activities of daily living reduced that likelihood. A curvilinear relation was observed between cognitive functioning and pica. The prevalence of pica is quite high in Ontario's institutions, and nonverbal communication emerged as the strongest correlate of the behavior. The implications for education, training, and interventions are discussed.
C1 [Ashworth, Melody] Univ Toronto, Dept Human Dev & Appl Psychol, Toronto, ON M5S 1V6, Canada.
[Martin, Lynn] Lakehead Univ, Dept Publ Hlth, Thunder Bay, ON P7B 5E1, Canada.
[Hirdes, John P.] Univ Waterloo, Dept Hlth Studies & Gerontol, Waterloo, ON N2L 3G1, Canada.
RP Ashworth, M (reprint author), Univ Toronto, Dept Human Dev & Appl Psychol, OISE, 252 Bloor St West, Toronto, ON M5S 1V6, Canada.
EM mashworth@oise.utoronto.ca
FU Ontario Ministry of Community and Social Services
FX This study was supported through a contract with the Ontario Ministry of
Community and Social Services. We also thank Dr. Alison Pedlar for her
support and helpful suggestions in this research.
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NR 38
TC 7
Z9 7
PU ROUTLEDGE JOURNALS, TAYLOR & FRANCIS LTD
PI ABINGDON
PA 4 PARK SQUARE, MILTON PARK, ABINGDON OX14 4RN, OXFORDSHIRE, ENGLAND
SN 1931-5864
EI 1931-5872
J9 J MENT HEALTH RES IN
JI J. Ment. Health Res. Intellect. Disabil.
PD JUN 12
PY 2008
VL 1
IS 3
BP 176
EP 190
DI 10.1080/19315860802029154
PG 15
WC Education, Special; Psychiatry; Rehabilitation
SC Education & Educational Research; Psychiatry; Rehabilitation
GA V37ZY
UT WOS:000209314900004
ER
EF